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// +build csall proto // Code generated by protoc-gen-grpc-gateway. DO NOT EDIT. // source: service.proto /* Package store is a reverse proxy. It translates gRPC into RESTful JSON APIs. */ package store import ( "context" "io" "net/http" "github.com/gogo/protobuf/types" "github.com/golang/protobuf/proto" "github.com/grpc-ecosystem/grpc-gateway/runtime" "github.com/grpc-ecosystem/grpc-gateway/utilities" "google.golang.org/grpc" "google.golang.org/grpc/codes" "google.golang.org/grpc/grpclog" "google.golang.org/grpc/status" ) var _ codes.Code var _ io.Reader var _ status.Status var _ = runtime.String var _ = utilities.NewDoubleArray func request_StoreService_IsAllowedStoreID_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoIsAllowedStoreIDRequest var metadata runtime.ServerMetadata newReader, berr := utilities.IOReaderFactory(req.Body) if berr != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", berr) } if err := marshaler.NewDecoder(newReader()).Decode(&protoReq); err != nil && err != io.EOF { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.IsAllowedStoreID(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_DefaultStoreView_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq types.Empty var metadata runtime.ServerMetadata msg, err := client.DefaultStoreView(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_DefaultStoreID_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoRunModeRequest var metadata runtime.ServerMetadata newReader, berr := utilities.IOReaderFactory(req.Body) if berr != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", berr) } if err := marshaler.NewDecoder(newReader()).Decode(&protoReq); err != nil && err != io.EOF { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.DefaultStoreID(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_StoreIDbyCode_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoStoreIDbyCodeRequest var metadata runtime.ServerMetadata newReader, berr := utilities.IOReaderFactory(req.Body) if berr != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", berr) } if err := marshaler.NewDecoder(newReader()).Decode(&protoReq); err != nil && err != io.EOF { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.StoreIDbyCode(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_AllowedStores_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoRunModeRequest var metadata runtime.ServerMetadata newReader, berr := utilities.IOReaderFactory(req.Body) if berr != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", berr) } if err := marshaler.NewDecoder(newReader()).Decode(&protoReq); err != nil && err != io.EOF { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.AllowedStores(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_AddWebsite_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq StoreWebsite var metadata runtime.ServerMetadata newReader, berr := utilities.IOReaderFactory(req.Body) if berr != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", berr) } if err := marshaler.NewDecoder(newReader()).Decode(&protoReq); err != nil && err != io.EOF { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.AddWebsite(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } var ( filter_StoreService_DeleteWebsite_0 = &utilities.DoubleArray{Encoding: map[string]int{}, Base: []int(nil), Check: []int(nil)} ) func request_StoreService_DeleteWebsite_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoIDRequest var metadata runtime.ServerMetadata if err := runtime.PopulateQueryParameters(&protoReq, req.URL.Query(), filter_StoreService_DeleteWebsite_0); err != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.DeleteWebsite(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } var ( filter_StoreService_WebsiteByID_0 = &utilities.DoubleArray{Encoding: map[string]int{}, Base: []int(nil), Check: []int(nil)} ) func request_StoreService_WebsiteByID_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoIDRequest var metadata runtime.ServerMetadata if err := runtime.PopulateQueryParameters(&protoReq, req.URL.Query(), filter_StoreService_WebsiteByID_0); err != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.WebsiteByID(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_ListWebsites_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq types.Empty var metadata runtime.ServerMetadata msg, err := client.ListWebsites(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_AddGroup_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq StoreGroup var metadata runtime.ServerMetadata newReader, berr := utilities.IOReaderFactory(req.Body) if berr != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", berr) } if err := marshaler.NewDecoder(newReader()).Decode(&protoReq); err != nil && err != io.EOF { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.AddGroup(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } var ( filter_StoreService_DeleteGroup_0 = &utilities.DoubleArray{Encoding: map[string]int{}, Base: []int(nil), Check: []int(nil)} ) func request_StoreService_DeleteGroup_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoIDRequest var metadata runtime.ServerMetadata if err := runtime.PopulateQueryParameters(&protoReq, req.URL.Query(), filter_StoreService_DeleteGroup_0); err != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.DeleteGroup(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } var ( filter_StoreService_GroupByID_0 = &utilities.DoubleArray{Encoding: map[string]int{}, Base: []int(nil), Check: []int(nil)} ) func request_StoreService_GroupByID_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoIDRequest var metadata runtime.ServerMetadata if err := runtime.PopulateQueryParameters(&protoReq, req.URL.Query(), filter_StoreService_GroupByID_0); err != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.GroupByID(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_ListGroups_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq types.Empty var metadata runtime.ServerMetadata msg, err := client.ListGroups(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_AddStore_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq Store var metadata runtime.ServerMetadata newReader, berr := utilities.IOReaderFactory(req.Body) if berr != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", berr) } if err := marshaler.NewDecoder(newReader()).Decode(&protoReq); err != nil && err != io.EOF { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.AddStore(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } var ( filter_StoreService_DeleteStore_0 = &utilities.DoubleArray{Encoding: map[string]int{}, Base: []int(nil), Check: []int(nil)} ) func request_StoreService_DeleteStore_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoIDRequest var metadata runtime.ServerMetadata if err := runtime.PopulateQueryParameters(&protoReq, req.URL.Query(), filter_StoreService_DeleteStore_0); err != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.DeleteStore(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } var ( filter_StoreService_StoreByID_0 = &utilities.DoubleArray{Encoding: map[string]int{}, Base: []int(nil), Check: []int(nil)} ) func request_StoreService_StoreByID_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq ProtoIDRequest var metadata runtime.ServerMetadata if err := runtime.PopulateQueryParameters(&protoReq, req.URL.Query(), filter_StoreService_StoreByID_0); err != nil { return nil, metadata, status.Errorf(codes.InvalidArgument, "%v", err) } msg, err := client.StoreByID(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } func request_StoreService_ListStores_0(ctx context.Context, marshaler runtime.Marshaler, client StoreServiceClient, req *http.Request, pathParams map[string]string) (proto.Message, runtime.ServerMetadata, error) { var protoReq types.Empty var metadata runtime.ServerMetadata msg, err := client.ListStores(ctx, &protoReq, grpc.Header(&metadata.HeaderMD), grpc.Trailer(&metadata.TrailerMD)) return msg, metadata, err } // RegisterStoreServiceHandlerFromEndpoint is same as RegisterStoreServiceHandler but // automatically dials to "endpoint" and closes the connection when "ctx" gets done. func RegisterStoreServiceHandlerFromEndpoint(ctx context.Context, mux *runtime.ServeMux, endpoint string, opts []grpc.DialOption) (err error) { conn, err := grpc.Dial(endpoint, opts...) if err != nil { return err } defer func() { if err != nil { if cerr := conn.Close(); cerr != nil { grpclog.Infof("Failed to close conn to %s: %v", endpoint, cerr) } return } go func() { <-ctx.Done() if cerr := conn.Close(); cerr != nil { grpclog.Infof("Failed to close conn to %s: %v", endpoint, cerr) } }() }() return RegisterStoreServiceHandler(ctx, mux, conn) } // RegisterStoreServiceHandler registers the http handlers for service StoreService to "mux". // The handlers forward requests to the grpc endpoint over "conn". func RegisterStoreServiceHandler(ctx context.Context, mux *runtime.ServeMux, conn *grpc.ClientConn) error { return RegisterStoreServiceHandlerClient(ctx, mux, NewStoreServiceClient(conn)) } // RegisterStoreServiceHandlerClient registers the http handlers for service StoreService // to "mux". The handlers forward requests to the grpc endpoint over the given implementation of "StoreServiceClient". // Note: the gRPC framework executes interceptors within the gRPC handler. If the passed in "StoreServiceClient" // doesn't go through the normal gRPC flow (creating a gRPC client etc.) then it will be up to the passed in // "StoreServiceClient" to call the correct interceptors. func RegisterStoreServiceHandlerClient(ctx context.Context, mux *runtime.ServeMux, client StoreServiceClient) error { mux.Handle("POST", pattern_StoreService_IsAllowedStoreID_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_IsAllowedStoreID_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_IsAllowedStoreID_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("POST", pattern_StoreService_DefaultStoreView_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_DefaultStoreView_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_DefaultStoreView_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("POST", pattern_StoreService_DefaultStoreID_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_DefaultStoreID_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_DefaultStoreID_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("POST", pattern_StoreService_StoreIDbyCode_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_StoreIDbyCode_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_StoreIDbyCode_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("POST", pattern_StoreService_AllowedStores_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_AllowedStores_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_AllowedStores_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("POST", pattern_StoreService_AddWebsite_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_AddWebsite_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_AddWebsite_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("DELETE", pattern_StoreService_DeleteWebsite_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_DeleteWebsite_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_DeleteWebsite_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("GET", pattern_StoreService_WebsiteByID_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_WebsiteByID_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_WebsiteByID_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("GET", pattern_StoreService_ListWebsites_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_ListWebsites_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_ListWebsites_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("POST", pattern_StoreService_AddGroup_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_AddGroup_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_AddGroup_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("DELETE", pattern_StoreService_DeleteGroup_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_DeleteGroup_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_DeleteGroup_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("GET", pattern_StoreService_GroupByID_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_GroupByID_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_GroupByID_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("GET", pattern_StoreService_ListGroups_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_ListGroups_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_ListGroups_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("POST", pattern_StoreService_AddStore_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_AddStore_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_AddStore_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("DELETE", pattern_StoreService_DeleteStore_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_DeleteStore_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_DeleteStore_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("GET", pattern_StoreService_StoreByID_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_StoreByID_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_StoreByID_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) mux.Handle("GET", pattern_StoreService_ListStores_0, func(w http.ResponseWriter, req *http.Request, pathParams map[string]string) { ctx, cancel := context.WithCancel(req.Context()) defer cancel() inboundMarshaler, outboundMarshaler := runtime.MarshalerForRequest(mux, req) rctx, err := runtime.AnnotateContext(ctx, mux, req) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } resp, md, err := request_StoreService_ListStores_0(rctx, inboundMarshaler, client, req, pathParams) ctx = runtime.NewServerMetadataContext(ctx, md) if err != nil { runtime.HTTPError(ctx, mux, outboundMarshaler, w, req, err) return } forward_StoreService_ListStores_0(ctx, mux, outboundMarshaler, w, req, resp, mux.GetForwardResponseOptions()...) }) return nil } var ( pattern_StoreService_IsAllowedStoreID_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "isAllowedStoreID"}, "")) pattern_StoreService_DefaultStoreView_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "defaultStoreView"}, "")) pattern_StoreService_DefaultStoreID_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "defaultStoreID"}, "")) pattern_StoreService_StoreIDbyCode_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "storeIDByCode"}, "")) pattern_StoreService_AllowedStores_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "allowedStores"}, "")) pattern_StoreService_AddWebsite_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "website"}, "")) pattern_StoreService_DeleteWebsite_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "website"}, "")) pattern_StoreService_WebsiteByID_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "website"}, "")) pattern_StoreService_ListWebsites_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "websites"}, "")) pattern_StoreService_AddGroup_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "group"}, "")) pattern_StoreService_DeleteGroup_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "group"}, "")) pattern_StoreService_GroupByID_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "group"}, "")) pattern_StoreService_ListGroups_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "groups"}, "")) pattern_StoreService_AddStore_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "store"}, "")) pattern_StoreService_DeleteStore_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "store"}, "")) pattern_StoreService_StoreByID_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 3}, []string{"api", "v1", "stores", "store"}, "")) pattern_StoreService_ListStores_0 = runtime.MustPattern(runtime.NewPattern(1, []int{2, 0, 2, 1, 2, 2, 2, 2}, []string{"api", "v1", "stores"}, "")) ) var ( forward_StoreService_IsAllowedStoreID_0 = runtime.ForwardResponseMessage forward_StoreService_DefaultStoreView_0 = runtime.ForwardResponseMessage forward_StoreService_DefaultStoreID_0 = runtime.ForwardResponseMessage forward_StoreService_StoreIDbyCode_0 = runtime.ForwardResponseMessage forward_StoreService_AllowedStores_0 = runtime.ForwardResponseMessage forward_StoreService_AddWebsite_0 = runtime.ForwardResponseMessage forward_StoreService_DeleteWebsite_0 = runtime.ForwardResponseMessage forward_StoreService_WebsiteByID_0 = runtime.ForwardResponseMessage forward_StoreService_ListWebsites_0 = runtime.ForwardResponseMessage forward_StoreService_AddGroup_0 = runtime.ForwardResponseMessage forward_StoreService_DeleteGroup_0 = runtime.ForwardResponseMessage forward_StoreService_GroupByID_0 = runtime.ForwardResponseMessage forward_StoreService_ListGroups_0 = runtime.ForwardResponseMessage forward_StoreService_AddStore_0 = runtime.ForwardResponseMessage forward_StoreService_DeleteStore_0 = runtime.ForwardResponseMessage forward_StoreService_StoreByID_0 = runtime.ForwardResponseMessage forward_StoreService_ListStores_0 = runtime.ForwardResponseMessage )
Low
[ 0.514734774066797, 32.75, 30.875 ]
~ ~~ An action of Occupy Wall Street Good Riddance CEO Cooper! CEO Cooper has handed in his letter of resignation! That is a success for everybody who participated in occupying Trinity Wall Street Church. Congratulations everyone — we have ousted a corrupt CEO! Now, CEO Cooper being the greedy bastard that he is, won’t actually resign for two more years — at least, that’s his plan. However, we’ll be back in the Spring to occupy his front steps again, and push him into leaving sooner than he wants. He doesn’t deserve two more years of a million dollar salary. What does he actually do for that money anyway? With less than 50 parishoners his leadership is worse than the smallest and poorest parish of his born-in-woman’s-blood Church of England 1%’er religion. Seems to me he is a total failure. It is the height of stupidity to let him stay on for two more years simply so he can milk the church for more millions. It has come out recently that the vestry’s voting procedures are a total sham, and the financial reporting is hidden from everyone’s view except those who are stealing it. Only CEO Cooper, the vestry and the church wardens are now allowed to see the financial records. All of them should be fired, arrested, tried and jailed — not only for the financial crimes they are engaged in, but also for the crimes of humanity they have perpetrated against Occupiers and the people of New York City. We will be back, Trinity. Expect us. A two year delay in Cooper’s resignation is not acceptable. Donations Gratefully Accepted! We desperately need monetary donations for food, clothing, medical and informational supplies. The NYPD keeps stealing our property and trying to torture us with lack of sleep, stress and daily-all-day-all-night harassment. Please go to our WePay page to donate and help us change the world for the better.
Mid
[ 0.6036036036036031, 33.5, 22 ]
A diverse bloc of defectors has emerged in the House GOP majority — and it’s not those who rode into office on the tea party wave last fall. The real rabble-rousers in the Republican Conference are mostly moderates and seasoned lawmakers, not the freshmen who many predicted would be a problem caucus. Those who have voted against their party at least 20 percent of the time include Tuesday Group members, those hailing from the Northeast and several appropriators, according to vote rankings by Congressional Quarterly as of April 15. Rep. Dave Reichert leads the pack, voting against the party position a quarter of the time. “What a rebel,” the Washington state Republican deadpanned when asked about his record. A four-term Member, Reichert has never won re-election with more than 53 percent and he hails from a Seattle suburban district that President Barack Obama carried with 57 percent in 2008. Reichert has always boasted a moderate voting record, but he only emerged as his party’s top defector this year. The former King County sheriff suggested the Conference’s shift to the right might explain his new status. “I think there are certain factions within the Conference that certainly have presented themselves more toward the right end of the spectrum,” he said. “You can tell that by comments being made at our Conference meetings.” Earlier this year, Reichert said, “there was a little more pressure applied to try to get me to maybe change my mind” on votes, but leadership has largely let him vote his own way. Several other lawmakers interviewed agreed, noting that Speaker John Boehner (Ohio) has so far made good on his promise to let Members vote their conscience. Rep. Peter King, who rebelled against his party earlier this year on a proposal to “retrieve” $178 million in funds paid to the United Nations, said Boehner’s even-handed style has made a difference in his own voting record, even though the New York Republican has voted against his party 13 percent of the time. The veteran lawmaker has served under three Republican Speakers, including former Reps. Dennis Hastert (Ill.) and Newt Gingrich (Ga.). “To me, everything is personal. For me, I had a pretty hard time voting against Hastert because he’s a nice guy, and Boehner’s a nice guy,” King said. “But for me,” he said with a grin, “voting against Gingrich was fun.” Boehner spokesman Michael Steel noted that his boss “has made it clear that he wants the House to work its will, and for every Member to have the opportunity to participate fully as legislators in as open a process as possible.” Leaders have held listening sessions and tutorials for Members as a way to keep an open process and buy goodwill among the rank and file. Aides note the tactic has helped during contentious debates, although it’s currently being tested as leaders struggle to unify the party behind any position on the debt limit. King’s votes against the party position were mostly on amendments to the Republicans’ yearlong continuing resolution that sought $61 billion in cuts. Boehner allowed an open debate process on the floor that led to votes on nearly 100 amendments. King did join his party to vote for final passage and later for the CR that eventually became law after heated negotiations between House Republicans and Senate Democrats. “I don’t think on any key vote I’ve been off,” King said. “But if I have to be, I will be.” Rep. Michael Grimm, another New York Republican, is just one of a dozen freshmen to make the list of top defectors in the GOP Conference. The outspoken lawmaker, however, is part of Majority Whip Kevin McCarthy’s (R-Calif.) whip team and in an interview said, “I’m certainly not doing it just to be a thorn in the side of leadership.” Rep. Greg Walden, a close Boehner ally, was quick to debunk the “perception that it’s the freshmen against the leadership.” Instead, the Oregon Republican noted that conservatives have required the most courting this year. Asked if that was a surprise, Walden said, “Nope, and here’s why: This is the biggest majority we’ve had in a very, very long time, so that brings with it a diversity of philosophy.” Just six of the party’s top 40 defectors voted against the long-term CR that passed in March and drew the ire of many conservatives, prompting 54 of them to vote against the measure. Republicans had to rely on significant Democratic turnout for that contentious floor vote, and some suggest a similar situation will arise when the House considers a debt limit increase. Republican Study Committee members have called for sweeping cuts that would likely be watered down in negotiations with Senate Democrats and the White House, and while details are far from settled, Boehner could end up calling on Minority Whip Steny Hoyer (D-Md.) for assistance. The Democrats’ top vote counter acknowledged the challenge Boehner and GOP leaders have in the debt limit debate and in managing a diverse caucus that includes 87 freshmen. The one-time Majority Leader said, “In the majority, the consequences of not getting to 218 are much greater than the consequences of not getting 193.” “In the majority, you have to govern, you have to take responsibility,” Hoyer said. “And hopefully our Republican colleagues will agree to do that, and especially on the debt limit.” Rep. Charlie Dent (R-Pa.) agreed. Even though the Tuesday Group leader has voted against his party 13 percent of the time this year, he said sticking together on the key votes will amount to more leverage, particularly when it comes to the debt limit.&nbsp; “This is not rocket science,” Dent said. “If the goal is to get spending restraint and spending cuts, the more of our folks who defect on these consequential votes simply emboldens Nancy Pelosi and the Democrats.” Editor's Note: The online version of this article has been corrected to show that Rep. Charlie Dent (R-Pa.) has voted against his party 13 percent of the time.
Low
[ 0.503787878787878, 33.25, 32.75 ]
/* fixme disabled in BATS because of gcfiles TEST_CONFIG OS=macosx BATS=0 TEST_BUILD cp $DIR/gcfiles/$C{ARCH}-aso gcenforcer-app-aso.exe END TEST_RUN_OUTPUT .*No Info\.plist file in application bundle or no NSPrincipalClass in the Info\.plist file, exiting END */
Low
[ 0.38725490196078405, 19.75, 31.25 ]
9th Annual Pharmaceutical Chemical Analysis Congress October 02-03, 2017 Vienna, Austria Theme: Innovations in Pharmaceutical Chemical Analysis and Quality Analytical Chemistry Analytical chemistry studies and uses instruments and methods used to separate, identify, and quantify matter. In practice separation, identification or quantification may constitute the entire analysis or be combined with another method. Separation isolates analyses. Qualitative analysis identifies analyses, while quantitative analysis determines the numerical amount or concentration. Analytical chemistry has been important since the early days of chemistry, providing methods for determining which elements and chemicals are present in the object in question. During this period significant contributions to analytical chemistry include the development of systematic elemental analysis by Justus von Liebig and systematized organic analysis based on the specific reactions of functional groups. Be a member and support us Follow us Authorization Policy By registering for the conference you grant permission to Conferenceseries Ltd to photograph, film or record and use your name, likeness, image, voice and comments and to publish, reproduce, exhibit, distribute, broadcast, edit and/or digitize the resulting images and materials in publications, advertising materials, or in any other form worldwide without compensation. Taking of photographs and/or videotaping during any session is prohibited. Contact us for any queries.
Mid
[ 0.570175438596491, 32.5, 24.5 ]
Building an RC Car using Raspberry Pi, an Xbox Controller and Lego - zephod http://blog.zephod.com/post/37120089376/raspberry-pi ====== joss82 I wonder why this very interesting article is not picking up on HN. It did very well on reddit/r/programming though. EDIT: Here is the link to reddit discussion thread: [http://www.reddit.com/r/programming/comments/149ib2/building...](http://www.reddit.com/r/programming/comments/149ib2/building_an_rc_car_using_lego_an_xbox_controller/)
High
[ 0.669410150891632, 30.5, 15.0625 ]
About Me I was a reporter and columnist for 40 years for a chain of newspapers in the suburbs of Chicago. I'm a military veteran having served in the United States Army Combat Engineers (Cpl. E-4) and a Korean War veteran with an Honorable Discharge from the Armed Forces of the United States of America Monday, August 4, 2008 The printing presses are rolling and one book after another attacking Barack Obama are hitting the nation's bookstores. The smears of Barack Obama have just begun and this is one of them. Bill Corcoran, Editor, CORKSPHERE Unfit for Publication: Corsi's The Obama Nation filled with falsehoods Summary: In its preface, Jerome Corsi compares his new book, The Obama Nation, to his 2004 book Unfit for Command. The comparison seems apt: Just as Unfit for Command contains false attacks on Sen. John Kerry's military service, a Media Matters review finds that The Obama Nation similarly contains numerous falsehoods about Sen. Barack Obama. In the preface of his recently released book, The Obama Nation: Leftist Politics and the Cult of Personality, author Jerome Corsi writes: "My intent in writing this book, as was the case in coauthoring Unfit for Command, is to fully document all arguments and contentions I make, extensively footnoting all references, so readers can determine for themselves the truth and validity of the factual claims." Indeed, Corsi's comparison of the two books seems quite apt: Just as Unfit for Command: Swift Boat Veterans Speak Out Against John Kerry contains false and baseless attacks on Sen. John Kerry's military service, a Media Matters for America review finds that Obama Nation similarly contains numerous falsehoods about Sen. Barack Obama. Media Matters previously documented false and baseless charges Corsi made in Obama Nation about Obama's positions on the Global Poverty Act of 2007 and nuclear weapons. Media Matters also pointed out false statements Corsi made while discussing the book with Fox News host Sean Hannity, concerning Obama's position on abortion and Obama's memoirDreams From My Father (Crown, 1995). Below are additional falsehoods from Obama Nation, listed in the order in which they appear in the book:The divorceCorsi baselessly suggests that Obama's father, Barack Obama Sr., may have divorced his mother, Stanley Ann Dunham, "following the prescripts of Islamic sharia law." Corsi's sole source for this statement is a blogger who made the claim in a March 20 post, which featured the false headline, "BARACK HUSSEIN OBAMA WAS MUSLIM FOR 31 YEARS" and misstated Obama's mother's name as "Shirley Ann." The blogger Corsi cited, "Majalah Bulanan Kumunitas Indonesia Di Edmonton," provided no substantiation for the claim that "the senior Obama divorced Dunham according to Sharia law." On page 44, Corsi writes:The circumstances of exactly how or why Stanley Ann ended her marriage with Obama's father, like much else in the narrative, remain unclear. One version is that Stanley Ann divorced Obama Senior in 1964, when Obama Junior was three years old and Stanley Ann realized Obama Senior had returned to Africa to rejoin the previously undisclosed wife he had abandoned in Kenya. The other version is that Obama Senior, following the prescripts of Islamic sharia law, divorced Stanley Ann when he returned to Africa in 1963 to begin his work as a bureaucrat in the Kenyatta government.4The endnote for Corsi's claim reads as follows:The first version, that Stanley Ann Dunham Obama divorced Obama Senior, is the version commonly found. The version that Obama Senior divorced Stanley Ann under sharia law is told in sources such as the following: Majalah Bulanan Kumunitas Indonesia Di Edmonton, "Barack Hussein Obama was Muslim for 31 Years," March 20, 2008, at http://www.indonesiaedmonton.org/berita/2008/03/20/barack-hussein-obama-was-muslim-for-31-years/. In the blog post that Corsi cites, the author asserted:The senior Obama married a fellow student, eighteen-year old Shirley Ann (Anna) Dunham of Witchita, Kansas. Dunham was an anthropology student. Obama Jr. was born on August 4, 1961, in Honolulu. Two years later, the senior Obama divorced Dunham according to Sharia law and returned to Kenya the same year that Kenyatta took control as the first prime minister of a self-governing Kenya (June 1963). In Dreams from My Father, Obama quotes his mother stating of Barack Obama Sr., "I divorced him":The teapot whistled, and I stamped my envelope. Then, without any prompting, my mother began to retell an old story, in a distant voice, as if she were telling it to herself. "It wasn't your father's fault that he left, you know. I divorced him. When the two of us got married, your grandparents weren't happy with the idea. But they said okay -- they probably couldn't have stopped us anyway, and they eventually came around to the idea that it was the right thing to do. Then Barack's father -- your grandfather Hussein wrote Gramps this long, nasty letter saying that he didn't approve of the marriage. He didn't want the Obama blood sullied by a white woman, he said. Well, you can imagine how Gramps reacted to that. And then there was a problem with your father's first wife ... he had told me they were separated, but it was a village wedding, so there was no legal document that could show a divorce ..." [Pages 125-126] The dedicationOn Pages 49-50, Corsi falsely claims that Obama did not dedicate Dreams from My Father to his mother or his grandparents. Corsi writes:Interestingly, Obama did not dedicate Dreams from My Father to his mother, or to his father, Barack Senior, or to his Indonesian stepfather. Missing from the dedication are the grandparents who raised him in Hawaii, especially during the years his mother abandoned him to return to Indonesia to be with Lolo [Soetoro, Obama's stepfather]. In fact, while the 2004 edition of Dreams -- the version Corsi cites in Obama Nation (Part One, endnote 2, Page 306) -- does not contain a separate dedication page, on Page xvii, the last page of the book's introduction, Obama writes:It is to my family, though -- my mother, my grandparents, my siblings, stretched across oceans and continents -- that I owe the deepest gratitude and to whom I dedicated this book. Without their constant love and support, without their willingness to let me sing their song and their toleration of the occasional wrong note, I could never have hoped to finish. If nothing else, I hope that the love and respect I feel for them shines through on every page [emphasis added].Obama's sister Corsi falsely claims that Obama does not mention the birth of his half-sister, Maya Soetoro-Ng, during the chapter in Dreams in which he discusses his time living in Indonesia. Corsi writes:In the midst of the personal drama being played against the background of this Indonesian turmoil, on August 15, 1970, Obama's half-sister, Maya Soetoro-Ng, was born to his mother and stepfather. Obama devotes the entire second chapter of his autobiography to his time in Indonesia, but remarkably, he makes no reference to Maya's birth [Page 48].
Low
[ 0.510869565217391, 29.375, 28.125 ]
The exon-junction complex proteins, Y14 and MAGOH regulate STAT3 activation. Signal transducer and activator of transcription 3 (STAT3), which is activated by cytokines and growth factors, mediates biological actions in many physiological processes. In a previous study, we found that Y14, a core component of the exon-junction complex (EJC) bound to STAT3 and upregulated the transcriptional activity of STAT3 by influencing its DNA-binding activity. In the present study, we demonstrate that STAT3 endogenously interacts with Y14. In addition, we found that MAGOH, a Y14 partner in the EJC, inhibits the STAT3-Y14 complex formation. Furthermore, small-interfering RNA-mediated reduction of MAGOH expression enhanced interleukin-6-induced gene expression. These results indicate that MAGOH regulates the transcriptional activation of STAT3 by interfering complex formation between STAT3 and Y14.
High
[ 0.657963446475195, 31.5, 16.375 ]
<?php /** * Copyright © Magento, Inc. All rights reserved. * See COPYING.txt for license details. */ declare(strict_types=1); namespace Magento\QuoteGraphQl\Model\CartItem\DataProvider\CustomizableOptionValue; use Magento\Catalog\Model\Product\Option; use Magento\Catalog\Model\Product\Option\Type\DefaultType; use Magento\Quote\Model\Quote\Item as QuoteItem; use Magento\Quote\Model\Quote\Item\Option as SelectedOption; use Magento\QuoteGraphQl\Model\CartItem\DataProvider\CustomizableOptionValueInterface; /** * Multiple Option Value Data provider */ class Multiple implements CustomizableOptionValueInterface { /** * @var PriceUnitLabel */ private $priceUnitLabel; /** * @param PriceUnitLabel $priceUnitLabel */ public function __construct( PriceUnitLabel $priceUnitLabel ) { $this->priceUnitLabel = $priceUnitLabel; } /** * @inheritdoc */ public function getData( QuoteItem $cartItem, Option $option, SelectedOption $selectedOption ): array { $selectedOptionValueData = []; $optionIds = explode(',', $selectedOption->getValue()); if (0 === count($optionIds)) { return $selectedOptionValueData; } foreach ($optionIds as $optionId) { $optionValue = $option->getValueById($optionId); $priceValueUnits = $this->priceUnitLabel->getData($optionValue->getPriceType()); $selectedOptionValueData[] = [ 'id' => $selectedOption->getId(), 'label' => $optionValue->getTitle(), 'value' => $optionId, 'price' => [ 'type' => strtoupper($optionValue->getPriceType()), 'units' => $priceValueUnits, 'value' => $optionValue->getPrice(), ], ]; } return $selectedOptionValueData; } }
Low
[ 0.536203522504892, 34.25, 29.625 ]
49 F.Supp.2d 1081 (1999) Stephen PRICE, Plaintiff, v. INTERSTATE WAREHOUSING, INC., Defendant. No. 97 C 3618. United States District Court, N.D. Illinois, Eastern Division. May 19, 1999. Thomas J. Canna, John F. Canna, Dawn M. Hinkle, Canna and Canna, Ltd., Orland Park, IL, for plaintiff. Christopher E. Hoyme, Nancy Wood, Berens & Tate, P.C., Omaha, NE, Thomas S. Moore, Anderson & Moore, P.C., Chicago, IL, for defendant. MEMORANDUM OPINION AND ORDER MORTON DENLOW, United States Magistrate Judge. On April 19, 1999, a jury returned a $20,000 verdict in favor of Plaintiff, Stephen Price ("Price"), and against Defendant, Interstate Warehousing, Inc., ("Interstate"), for a violation of the Americans with Disabilities Act (the "ADA"), 42 U.S.C. § 12101 et seq. The case is now before the Court on the issues of back pay, prejudgment interest and front pay. I. BACKGROUND FACTS Price was employed by Interstate as a warehouse worker from October 31, 1994 through November 29, 1995, when he was terminated. Price was a good worker and received a positive ninety-day review and *1082 raise on January 31, 1995. After a second raise on August 6, 1995, along with all other warehouse workers, he was making $9.50 per hour. Price suffered a heart attack on September 1, 1995, and was placed on medical leave for twelve weeks. On November 20, 1995, Price's doctor gave him a letter stating that he could return to work; however, he could not lift more than 20 pounds. Following a meeting between Price and Interstate's Director of Human Resources, Interstate terminated Price effective November 29, 1995, and notified him via a telephone call and confirming letter. Price retained counsel who sent Interstate a letter dated December 14, 1995 requesting reinstatement. Interstate did reconsider the matter by having Price examined by its company doctor, having him submit to a functional capacity assessment and retaining a consultant to perform a job site analysis. On April 29, 1996, Interstate wrote to Price and offered to reinstate him on the condition he obtain a letter from his doctor releasing him to return to work without any restriction. In the meantime, in January 1996, Price took a job as a janitor with Heritage Inn Best-Western in the St. Louis area. He has continued to reside in the St. Louis area. In February 1996, he left Best-Western and began work at Datastor, Inc. (now Pierce Leahy Corporation). At about the same time, he also began working for Schnuck's Food Market. At the time of trial, Plaintiff was earning $8.50 per hour at Pierce-Leahy Corporation for 40 hours per week and $7.75 per hour at Schnuck's Food Market for 32 hours per week. At the time of trial, a warehouse worker at Interstate was earning $11.50 per hour. The jury found Interstate violated the ADA and awarded Price $20,000 in compensatory damages and no punitive damages. The issues of back pay and front pay were left to the Court. During the trial, the Court heard additional evidence and arguments on these issues outside the presence of the jury. II. BACK PAY Title VII remedies apply to ADA cases. See 42 U.S.C. § 12117(a). In the event the jury returned a verdict for Plaintiff, the parties agreed a full back pay award would include $1,801.48 for Cobra insurance paid for by Price, $200 in job search costs and $21,420.75 in back pay. The only disputed issue was whether Price's back pay should be cut off by reason of Interstate's letter of April 29, 1996. If so, Price's back pay award would be reduced to $13,702. Interstate relies upon Ford Motor Company v. EEOC, 458 U.S. 219, 102 S.Ct. 3057, 73 L.Ed.2d 721 (1982). In Ford, the Court held an employer can toll the accrual of back pay liability by unconditionally offering claimant the job, thereby providing claimant the opportunity to minimize damages. 458 U.S. at 232, 102 S.Ct. at 3066. The Ford decision is distinguishable because the offer to Price was conditioned upon returning to work with no weight lifting restriction. Accordingly, Price is awarded full back pay as follows: Cobra Premiums $ 1,801.48 Job Search Costs 200.00 Back Pay $21,420.75 __________ Total: $23,422.23 III. INTEREST Prejudgment interest on back pay is awarded to compensate Price for the loss of use of money. Downes v. Volkswagen of America, Inc., 41 F.3d 1132, 1143 (7th Cir.1994). Interest in the amount of $3,052.58 is awarded. The parties did not dispute the computation of interest. IV. FRONT PAY Front pay represents the equitable equivalent of reinstatement and is available when reinstatement is not appropriate. Williams v. Pharmacia, Inc., 137 F.3d 944, 951 (7th Cir.1998). Front pay is defined as "a lump sum ... representing *1083 the discounted present value of the difference between the earnings [an employee] would have received in his old employment and earnings he can be expected to receive in his present and future, and by hypothesis inferior, employment." McKnight v. General Motors Corp., 908 F.2d 104, 116 (7th Cir.1990), cert. denied, 499 U.S. 919, 111 S.Ct. 1306, 113 L.Ed.2d 241 (1991). Given the hostility between the parties and the likelihood of an appeal, reinstatement is not appropriate. Price seeks a front pay award of five years based on the difference between the $11.50 per hour currently being paid at Interstate to warehouse workers and the $8.50 per hour he is currently earning at Pierce Leahy. Interstate argues no front pay should be awarded because Price is currently working two jobs and making more in total than he would have earned at Interstate. The front pay award is designed to approximate the benefit Price would have received had he been able to return to Interstate. Williams, 137 F.3d at 953. The Court concludes that a two-year front pay award based on the difference between the rate of pay at Interstate ($11.50 per hour) and the rate of pay at Pierce Leahy Corp. ($8.50 per hour) for a forty-hour week ($12,480) discounted to present value at the prime rate of interest (7.75%), which equals $11,980, is appropriate. It is highly unlikely Price could find an equivalent paying job to his Interstate job. He has been unable to do so for over three years. Price's employment history demonstrates a propensity to change jobs. Given the nature and difficulty of the work and the high turnover rates, it is reasonable to expect that if reinstated, Price would leave Interstate at the end of two years. In addition, given the downturn in Interstate's Joliet business, it is not unreasonable to expect Price might have been laid off at the end of two years. However, Price should not be punished for taking a second job to make as much or more than he was making at Interstate. Facing the same dire financial predicament, he would seek overtime or a second job while at Interstate. Accordingly, Price is awarded $11,980 in front pay. V. CONCLUSION The Court directs the entry of judgment in favor of Plaintiff, Stephen Price, and against Defendant, Interstate Warehousing, Inc., in the amount of $157,875.32, computed as follows: Jury Award for Compensatory Damages $ 20,000.00 Cobra Premium $ 1,801.48 Job Search Costs $ 200.00 Back Pay $ 21,420.75 Interest $ 3,052.58 Front Pay $ 11,980.00 Attorney's Fees $ 95,750.00[1] Court Costs and Expenses $ 3,670.51 ____________ Total: $157,875.32 NOTES [1] The Court's award of attorney's fees, court costs and expenses was made during the hearing held on May 19, 1999.
Mid
[ 0.558823529411764, 30.875, 24.375 ]
Q: Entity Framework - How to get Standard Deviation using GroupBy? I'm using entity framework 5, code first fluent API. I've built a simple query with a GroupBy for aggregation: var query = context.Table1 .GroupBy(g => new { Prop1= g.Prop1, Prop2 = g.Prop2 }) .Select(s => new { Prop1= s.Key.Prop1, Prop2= s.Key.Prop2, Avg = s.Average(m => m.Minutes), Min = s.Min(m => m.Minutes), Max = s.Max(m => m.Minutes), StdDev = //How to get standard deviation of Minutes here? }) .ToList(); Along with other aggregation (average, min, max), how can I get standard deviation of Minutes in my select? I've heard there is a canonical function for StdDev but have been unable to figure out how to apply it in this case. Any help or a nudge in the right direction would be greatly appreciated! A: You should be able to use EntityFunctions.StandardDeviation in System.Data.Objects like this: StdDev = EntityFunctions.StandardDeviation(s.Select(m => m.Minutes)) (If you were using EF 6, it would be System.Data.Entity.DbFunctions.StandardDeviation())
High
[ 0.696117804551539, 32.5, 14.1875 ]
Q: Problems when using chemscheme/chemstyle with Palatino and T1 encoding Automatic generation of compound numbers by chemscheme (part of chemstyle) works: In Latin Modern (lmodern) with T1 or OT1 encoding In Palatino (mathpazo) with OT1 encoding, but not with T1 encoding - instead, the numbers simply vanish (see example below). This is presumably down to Palatino's use of bitmap fonts, when switched to T1. I'd like to find a way to carry on using Palatino (or similar), but be able to use the automatic compound numbering features of chemstyle (or rather the underlying chemcompounds) Fully working version Taking, as my MWE, a version of the example document from the chemstyle documentation which works as it's supposed to: \documentclass[a4paper]{article} \usepackage[T1]{fontenc} \usepackage[runs=2]{auto-pst-pdf} \usepackage[journal=rsc]{chemstyle} \usepackage{lmodern} \begin{document} \begin{scheme}[ht] \includegraphics{chemscheme} \caption{A scheme with temporary compound numbers.} \end{scheme} \begin{scheme}[ht] \schemeref{IMesHCl} \schemeref{IMes} \includegraphics{chemscheme} \caption{A scheme with automated compound numbers.} \end{scheme} \end{document} The above uses the eps file from the chemstyle manual, i.e. chemscheme.eps . As best I can tell, that eps doesn't seem to be available as-is in the chemstyle documentation – rather, there's a chemscheme.cdx (a ChemDrawfile) that (for those with ChemDraw) can be saved as eps. Edit: Alternatively, I've uploaded a copy at http://dur.ac.uk/o.w.j.burnham/stackexchange/chemscheme.eps. When run, with shell-escape enabled, via pdflatex.exe -synctex=1 -interaction=nonstopmode --shell-escape %.tex I get, as expected, i.e. in the second scheme, the placeholders (TMP1 and TMP2) from the eps have been replaced by automatically-generated numbers. The error However, if I disable Latin Modern, i.e. if the document reads \documentclass[a4paper]{article} \usepackage[T1]{fontenc} \usepackage[runs=2]{auto-pst-pdf} \usepackage[journal=rsc]{chemstyle} %\usepackage{lmodern} %Latin Modern disabled. Now using Computer Modern. \begin{document} \begin{scheme}[ht] \includegraphics{chemscheme} \caption{A scheme with temporary compound numbers.} \end{scheme} \begin{scheme}[ht] \schemeref{IMesHCl} \schemeref{IMes} \includegraphics{chemscheme} \caption{A scheme with automated compound numbers.} \end{scheme} \end{document} and, therefore, the document compiles using the default font (Computer Modern), the automated compounds numbers vanish. The problem also occurs if I replace Latin Modern with Palatino, i.e. if I replace lmodern with mathpazo. In either case I get something like this, where the auto-generated numbers are missing: Happily (?) this can be fixed – using Computer Modern or Palatino – by removing \usepackage[T1]{fontenc}, so as to return to the default (OT1) font-encoding: So, in summary, as best I can tell, automatic generation of compound numbers works: In Latin Modern with T1 or OT1 encoding In Computer Modern with OT1 encoding (but not with T1 encoding) In Palatino with OT1 encoding (but not with T1 encoding) As best I understand it, this correlates with Computer Modern and Palatino's use of bitmap fonts when switched to T1 encoding. Intuitively it makes some sense to me that the eps – being a vector format – works with vector fonts but not with bitmaps, and so the use of bitmap fonts here could lead to problems. Possible solutions that have occurred to me: Use Latin Modern – I would prefer not to, as I like Palatino Use a Palatino-like font that has a vector form in T1 – however, I don't know of any such font! Use Palatino except in the context of compound numbering – however, I don't know how I'd go about doing this If anyone has any other ideas for possible fixes but without knowing how they would implement them, I'll happily make edits and add any such suggestions to this list, in case someone else can them work out how to implement them. A: You have a bitmap (pk-)font in your log (ecrm1000.pk) which is the source of the problem. Install the cm-super package (large package, can take some time and in miktex you will perhaps have to call updmap after the installation to activate the map-file) and then try again.
Low
[ 0.5339366515837101, 29.5, 25.75 ]
Fulminating Staphylococcus epidermidis bacteremia. We have reported a case of disseminated Staphylococcus epidermidis infection in a patient with leukemia and examined the relation between an acute respiratory arrest and the infection. Plasmid profiles of five isolates of S epidermidis cultured from this patient's blood, bone marrow, and lung before and after the arrest indicate that all isolates were derived from a single strain. This strain was also isolated by culture of the tip of the central venous catheter that was removed from the patient suggesting that the indwelling catheter was the source of infection. Because this patient had rigors during an infusion through the catheter just before the acute respiratory arrest, we suspect that infusion through the colonized catheter precipitated the respiratory arrest.
Mid
[ 0.5804195804195801, 31.125, 22.5 ]
Bodo Igesz Bodo Igesz (February 7, 1935 in Amsterdam – December 25, 2014 in New York City) was a Dutch stage director who had an active career staging operas around the world during the second half of the 20th century. He was particularly known for his work with the Metropolitan Opera where he worked for 25 years on the staging staff. He also staged operas for the Salzburg Festival, and staged numerous operas for the Santa Fe Opera; including the United States premieres of Hindemith's Cardillac (1967), Schoenberg's Die Jakobsleiter (1968), Henze's The Bassarids (1968) and Aribert Reimann's Melusine (1972). A particular triumph for Igesz at the Met was his staging of Georges Bizet's Carmen which premiered in 1972 with Marilyn Horne as the title heroine and Leonard Bernstein conducting. The staging remained in the Met repertory for several years, and was notably the staging used for the first Met Carmens of Régine Crespin (1975) and Elena Obraztsova(1978). Another Met career highlight was his 1988 staging of Ariadne auf Naxos by Richard Strauss which starred Jessye Norman, Kathleen Battle, James King, Tatiana Troyanos, and Stephen Dickson. This production was Broadcast nationwide on the television program The Metropolitan Opera Presents on PBS. References External links Interview with Bodo Igesz by Bruce Duffie, February 13, 1988 Category:1935 births Category:2014 deaths Category:Dutch opera directors
High
[ 0.701854493580599, 30.75, 13.0625 ]
The townland of Keelkyle comprised of 3 dwellings in 1911. All 3 are recorded as being private dwellings. They were all constructed of stone, brick or concrete walls. House 1 had slate, iron or tiles for roofing and houses 2 and 3 had thatch, wood or other perishable material. House 1 was a 1st class house with 5 rooms and 7 windows in the front. Houses 2 and 3 were 3rd class dwellings with 2 rooms and 2 windows. There were a total of 14 out buildings consisting of 2 stables, a coach house, 3 cow houses, 2 calf houses, 2 piggeries, a fowl house, a barn, a turf house and a shed. A total of 13 people lived there, 6 male and 7 female. Const. John Hickey was the enumerator. House 1 was the home of the Leigh family. Head of the family was Edward Leonard Holmes (65) and his wife Blanche Isabel (34) who had been married for 4 years and had had 1 child. In the house with them at that time were their son Edward ?[i] Patrick (3), a visitor Emily Longbottom (52) and a servant Ellen Connolly (19). Ellen is recorded as speaking Irish and all, with the exception of Edward ? Patrick could read and write. Edward ? Patrick and Ellen were born in Co. Galway with the rest being born in England. Ellen was a Roman Catholic and the rest were Church of England. Edward Leonard Holmes is recorded as a retired farmer, Emily was an artist and Ellen was a general servant domestic. The house they shared was a 1st class, 5 roomed dwelling with a stable, coach house, cow house, calf house, piggery, fowl house, barn, turf house and a shed. Hector R. L. Graham was the landholder. House 2 was the Joyce household and the head of the family was the widow Margret (sic) (80) who had given birth to 9 children with 8 surviving. 2 of those children lived with her Philip (38) and Maria (36). All 3 could speak both Irish and English but only Philip could read and write. All were born in Co. Galway and were Roman Catholic. Maria is recorded as a farmer’s daughter and Philip a farmer’s son. They lived in a 2 roomed, 3rd class dwelling with a cow house and piggery. Margret Joyce was the landholder. The third house was home to the Walsh family. The head of the family was Peter (74) and his wife Honor (63) who had been married for 40 years and had had 10 children all surviving. 2 of those children lived with them and were John (22) and Stephen (17) and peter’s moth-in-law also lived there and she was Ellen Daly (90). All could speak both Irish and English but only John and Stephen could read and write. Peter was a farmer and John and Stephen were farmer’s sons. Their house was a 2 roomed, 3rd class dwelling with a stable, cow house and calf house. The landholder was Peter Walsh. There were 4 houses in Keelkyle according to the 1901 census. All were classified as private dwellings and all had stone, brick or concrete walls. Houses 1 and 2 had slate, iron or tiles for roofing and houses 3 and4 had thatch, wood or other perishable material. Houses 1 and 2 were 1st class dwellings and houses 2 and 3 were 3rd class. House 1 had 5 rooms and 7 windows in the front, house 2 had 4 rooms and 7 windows, house 3 had 2 rooms and 2 windows and house 4 had 1 room and 2 windows. There were a total of 15 out buildings, those being 2 stables, 2 coach houses, 6 cow houses, a piggery, 2 fowl houses and 2 stores. There were a total of 29 people, 16 male and 13 female. The enumerator was Const. Edward Robison. The head of the Graham family was Francis John (67). With him in the house that time were a cook Eliza Laird (32) and 3 servants, George Laird, John Mannion (32) and Thoms (sic) McDonnell (19). John and Thoms (sic) could speak both Irish and English and the others on English. They could all read and write with the exception of John Mannion. John and Thoms (sic) were born in Co. Galway and were Roman Catholic. Francis John and George were born in Co. Fermanagh and Eliza was born in Co. Tyrone, these thre were members of the Presbyterian Church. Francis John’s occupation is recorded as Lanstord D.L. (sic) Eliza was a cook domestic st (sic), George was a gardener, John a gamekeeper and Thoms a farm labourer. The house they lived in was a 5 roomed, 1st class dwelling with a stable, coach house, 2 cow houses, a fowl house and a store. The landholder was Francis J. Graham. The head of the McDonnell family was the widow Mary (74). In the house with her at that time were her children Joseph (42), Josephine (40) and Barry (38), her daughter-in-law Mary (32), her grandchildren Charles Edward (6), Josephine Mary (2) and Joseph (8mths). Also in the house were 3 servants Michael Conneely (35), Mary Maninan (sic)[ii] and Hanoria Lydon (sic). Mary, Charles Edward, Josephine Mary and Honoria could speak only English. All the rest, with the exception 8 month old Joseph, could speak both Irish and English. Josephine Mary, Joseph and Mary could not read but all the rest were able to read and write. Joseph (42) is listed as being a staff surgeon R.N., Barry was a farmer J.P., Charles Edward was a scholar, Michael was a farm servant, Mary (36) was a domestic servant cook and Honoria was a domestic servant. All were Roman Catholic, Mary (32) was born in England, Charles Edward was born in Lamarkshire (sic)[iii], Josephine Mary was born in Hampshire and the rest were born in Co. Galway. The house they shared was a 4 roomed, 1st class dwelling with a stable, coach house, cow house, fowl house and a store. The landholder was Mary McDonnell. The head of the Walsh family was Peter (60) and his wife Honor (50). Living with them at that time were their children Thomas (23), Annie (15), Ellen (14), John (12), Peter (8) and Steph (6) along with Peter’s mother-in-law Ellen McLoughlin (80). All except Peter (8) and Stephen could speak both Irish and English and all except Peter(60) and Ellen McLoughlin could read and write. All were born in Co. Galway and were Roman Catholic. Peter (60) was a farmer, Thomas was a farmer’s son, Annie and Ellen (14) were farmer’s daughters and John Peter (8) and Stephen were scholars. The house they shared was a 2 roomed, 2nd class dwelling with 2 cow houses and a piggery. The landholder was Peter Walsh. The Joyce family consisted of 4 members with the head of the fanliy being Patrick (80) with his wife Margret (sic) (70). Living with them were their children Philip (30) and Maria (24). All were born in Co. Galway and were Roman Catholic. All 4 could speak both Irish and English and Philip and Maria could read and write. Patrick is recorded as a farmer, Margret as a farmer’s wife, Philip as a farmer’s son and Maria as a farmer’s daughter. They shared a 1 roomed, 3rd class house with cow house. Patrick Joyce was the landholder. Robert Graham owned most of the land in this area according to Griffith’s Valuation (1847-1864) and he leased tenements to a number of people. William Yates leased an area of land of 1 Acre and 24 Perches from Robert Graham at an annual rate of 8s. William then leased a house and office to Michael King for £1 annually. Francis J Graham owned 2 lots of land in this area. The first being an area 31 Acres on which he had a house and offices and paid £10 10s for the land and 10s for the buildings. In addition to that he also had 816 Acres, 2 Roods and 10 Perches of mountain and bog land for which he paid an annual rate of £6 10s. Patrick Heaney and John Manning jointly leased 10 Acres 2 Roods and 38 Perches of land from Robert Graham. Patrick Heaney had a house and John Manning had a house and offices on that land. Patrick paid 15s for his share of the land and 5s for the house, John paid £1 8s for his part of the land and 7s for the buildings. James Ellis leased 2 Acres of land from Robert Graham for 15s. Patrick Kilkoyne leased 12 Acres, 3 Roods and 10 Perches of land on which there were a house and office. For this he paid £2 15s for the land and 15s for the buildings. Patrick Heaney, John Manning, Patrick Kilcoyne, John Mealy, John Lyden and Thaddeus Walsh jointly leased 7 Acres an 3 Roods of land from Robert Graham at a rate of 1s each annually. John Lydon and Bartholomew Conroy leaded 11 Acres and 13 Perches of land that had houses on and paid £1 10s for the land and 5s for the buildings each. Thaddeus Walsh leased 12 Acres and 10 Perches of land and buildings from Robert Graham for £2 for the land and 10s for the buildings. John Mealy leased 12 Acres of land and a house from Robert Graham for which he paid £3 10s for the land and 10s for the house. James Lydon and Martin Coyne leased 31 Acres, 2 Roods and 11 Perches of land and buildings from Robert Graham for which James Lydon paid £2 7s for the land and 8s for the house and Martin Coyne paid £1 3s for the land and 7s for the house. Robert Graham had an area of land of 5 Acres, 1 Rood and 20 Perches of land which he paid an annual ratable valuation of 12s. Thomas Heaney leased 21 Acres, 3 Roods and 13 Perches of land from Robert Graham that included a house for which he paid £3 10s for the land and 5s for the house. Lastly, Patrick Coyne leased 30 Acres, 3 Roods and 25 Perches of land and buildings from Robaert Graham for £4 for the land and 10s for the buildings. The name for this area in the 1670 Down Survey was Cossekillarie. The 1641 owner was the Catholic Edmund O’Flaharty. In 1670 the owner was Sir Thomas Meredith, a Protestant. There were 2077 plantation acres of unprofitable land, 292 plantation acres of profitable land and 293 plantation acres were forfeited. No Comments Your email address will not be published. Required fields are marked * Your comment: * Name * Email * Website I consent to my name and e-mail address being stored along with this comment, and to the website editors communicating with me by e-mail about the comment if necessary. My name may be published alongside the comment on the website, but my e-mail address will not be published. My information will not be shared with any third party (see our Privacy Statement - opens in a new window). *
Mid
[ 0.5766871165644171, 35.25, 25.875 ]
Carol Reed’s Night Train to Munich is a twisting, turning, cloak-and-dagger delight, combining comedy, romance, and thrills with the greatest of ease. Paced like an out-of-control locomotive, Night Train takes viewers on a journey from Prague to England to the Swiss Alps as Nazis pursue a Czech scientist and his daughter (Margaret Lockwood), who are being aided by a debonair British undercover agent, played by Rex Harrison. This captivating, long-overlooked adventure—which features Paul Henreid and a clever screenplay by Frank Launder and Sidney Gilliat, best known for writing Alfred Hitchcock’s The Lady Vanishes—is a deftly concocted spy game that could give the Master of Suspense a run for his money.
High
[ 0.678048780487804, 34.75, 16.5 ]
The NCAA men’s basketball tournament will cost U.S. companies an estimated $134 million in “lost wages” this week. But do employers care? Not really. A survey released last week by job outplacement firm Challenger, Gray & Christmas found that the men’s college basketball tournament – which lasts three weeks – will cost $134 million in just the first two days (Thursday and Friday) of the tournament. An estimated 3 million U.S. employees will spend one to three hours at work watching the games, and two-thirds of all workers will follow the tournament at some point during work hours. A few decades ago, the idea that employees would be able to spend hours watching a sporting event during normal working hours would’ve been unthinkable. But our work and personal lives have become completely tangled, and today most bosses are not only fine with employees who watch a few games or set up an office pool, they almost encourage it. According to a separate survey by staffing firm OfficeTeam, when office managers and executives were asked whether the NCAA basketball tournament had a negative effect on employee productivity, 75% said there was no impact, and 16% said there was either a very positive or somewhat positive impact. And one-fifth of employers said those activities elicited a positive impact on employee morale. That’s despite the fact the tournament likely takes more than $100 million out of productivity in just two days. “That’s a big number,” says John Challenger of Challenger, Gray & Christmas, referring to how much the tournament is expected to cost businesses. “And yet, I think companies that allow their employees freedom and autonomy recognize that the workplace brings people’s personal lives in. It’s the way the modern workplace works.” The acceptance of March Madness into the workplace is of course just one example of how the workplace has changed. Today, employees check Facebook, share YouTube videos, or instant message with friends throughout the work day. But more employees are also expected to check email in the evenings and sometimes be available on weekends. The Internet has kept us perpetually connected to our personal lives at work and tied to our work lives at home. Watching the NCAA tournament in the workplace is just an extension of that, and most employers have accepted it. “As an employer, while hours may be 9 to 5 or 9 to 6, a great number of employees are doing things after hours they don’t even recognize or realize,” says Daryl Pigat, a spokesman for OfficeTeam. “An extended lunch or letting employees check in on games here and there can mean a lot for overall morale.” Within the last couple years employers seem to have given in even more to workplace tournament watching. In 2010, 22% of employers said March Madness adversely affected employee output whereas only 9% said there were negative impacts this year, according to the OfficeTeam survey. Pigat believes that’s largely because managers are more confident about the economy these days, as opposed to the more shaky recovery of a few years ago, and aren’t as inclined to micromanage every minute of their employees’ work days. Still, there are some employers who try to limit workers’ participation in the tournament. Some try to cut off streaming video to employees because it can slow a company’s servers. For example, employees at Los Angeles City Hall were told not to watch the London Olympics on their work computers last summer over concern it could crash their systems. But with the growth of online streaming video, mobile apps, and the brilliant “Boss Button” on cbssports.com (which quickly brings up a fake Excel spreadsheet to hide live video of the tournament), each year it gets easier for employees to follow the games and harder for employers to stop them, if they even try. “I think most employers do look the other way,” says Challenger. “There may be some work lost when there’s urgency to work, but a lot of it gets done later. Employees tend to catch up.”
Mid
[ 0.6090712742980561, 35.25, 22.625 ]
The present disclosure relates broadly to a multi-purpose pet utility vest. In exemplary embodiments discussed herein, the pet utility vest may dispense plastic waste bags for collection of pet waste, and may incorporate at least one utility pocket assembly for carry the pet waste until it can be disposed of properly. The exemplary utility vest may also be used for enhancing visibility of the pet during the day and night, and for covering the pet in cold and inclement weather conditions.
High
[ 0.681067344345616, 33.5, 15.6875 ]
Leonid Borodin Leonid Ivanovich Borodin (; 14 April 1938 in Irkutsk – 24 November 2011 in Moscow) was a Russian novelist and journalist. Biography Born in Irkutsk, Borodin was a Russian Orthodox Christian and a Soviet dissident. In the 1960s he belonged to the anti-Communist All-Russian Social-Christian Union for the Liberation of the People (VSHSON). He was arrested and imprisoned in the 'strict regime' Camp 17 in 1967, and went on hunger strike there with Yuli Daniel and Aleksandr Ginzburg in 1969. After his release in 1973, Borodin’s works were smuggled out of the Soviet Union. The publication of an English translation of The Story of a Strange Time led to his arrest in 1982 on charges of 'anti-Soviet propaganda'. He was sentenced to 10 years of hard labour in Perm-36 Maximum Security Camp (ITK-6), as well as five years' internal exile. Released after four years, in the perestroika era, Borodin was allowed to visit the West with his wife. Borodin was the subject and first-person narrator of the 2001 film Leonid Borodin: Looking through the Years. A winner of many literary prizes, including the 2002 Solzhenitsyn Prize, Borodin was editor-in-chief of Moskva, a popular literary magazine. In 2005 he was appointed to the first convocation of the Public Chamber of Russia. Works in English translation Partings, The Harvill Press, 1988. The Year of Miracle and Grief, Quartet Books, 1988. The Third Truth, Harpercollins, 1992. The Story of a Strange Time, Harpercollins, 1993. References External links 2002 interview after receiving the Solzhenitsyn Prize Moskva Journal New York Times review of ''Partings New York Times review of The Year of Miracle and Grief Category:1938 births Category:2011 deaths Category:People from Irkutsk Category:Irkutsk State University alumni Category:Buryat State University alumni Category:Russian journalists Category:Russian male novelists Category:Soviet novelists Category:Soviet male writers Category:20th-century male writers Category:Soviet dissidents Category:Soviet prisoners and detainees Category:Members of the Civic Chamber of the Russian Federation Category:Inmates of Vladimir Central Prison Category:Solzhenitsyn Prize winners
High
[ 0.6636771300448431, 27.75, 14.0625 ]
Jegbefumere Albert Jegbefumere Albert (born 27 July 1981) is a Nigerian boxer who competed in the light heavyweight division. At the 2000 Summer Olympics, Albert was defeated by Rudolf Kraj from the Czech Republic during the quarterfinal match. In 2002 Commonwealth Games Jegbefumere Albert defeated Joseph Lubega (Ugandan) in the final to win a gold medal for Nigeria. References Category:1981 births Category:Living people Category:Olympic boxers of Nigeria Category:Boxers at the 2000 Summer Olympics Category:Middleweight boxers Category:Nigerian male boxers Category:Boxers at the 2002 Commonwealth Games Category:Commonwealth Games medallists in boxing Category:Commonwealth Games gold medallists for Nigeria
High
[ 0.6694329183955741, 30.25, 14.9375 ]
2536 Sw 43rd Court, Redmond $449,990 <div><b>Don't Miss Out! 3 Homes remain in this sought after community!</b><span style=font-size: 10pt;=>Welcome to the Gem of Redmond. Emerald Estates boasts spectacular Cascade Mountain Views with large home sites & conveniently located to shopping & entertainment. Only a 5 minute drive to Downtown Redmond & 15 minutes to Downtown Bend. The stylish and versatile Talent plan is designed to fulfill all of your must-haves. A wonderful open kitchen with dining area open to a spacious great room perfect for entertainment or relaxation. Upstairs offers 3 </span><span style=font-size: 13.3333px;=>additional</span><span style=font-size: 10pt;=>bedrooms and master suite that has an </span><span style=font-size: 13.3333px;=>oversize</span><span style=font-size: 10pt;=>walk-in closet and a vaulted ceiling. There is plenty of storage in this award winning home. 3rd car garage w/ mountain views!*Finishes in photos may be different. Personalize your home today &amp; Make sure to check out the Virtual Tour!</span></div><div><b>We are here to safely assist you in purchasing your new Hayden Home. Currently, we are operating on an appointment basis while offering virtual tours and virtual appointments.&nbsp;</b><span style=font-size: 10pt;=><br></span></div>
Mid
[ 0.5647558386411891, 33.25, 25.625 ]
Thessaly Railways Thessaly Railways () was a railway company in Greece, which owned and operated the metre gauge railway network of Thessaly and Pelion railway from 1884 to 1955, when the company was absorbed by the Hellenic State Railways. Today the term usually refers to the section of mainline between Domokos and Rapsani and its two branches, the West Thessaly branch to Kalambaka and the Volos branch. Network and stations The network of Thessaly Railways consisted of the following lines: Volos—Velestinon. The line extended from Volos station to the city centre along Dimitriados Street. Velestinon—Kalampaka, connecting with the Athens-Larissa-Thessaloniki standard gauge mainline at Palaiofarsalos. This section had a maximum gradient of 3% between Velestinon and Aerinon. Velestinon—Larissa, terminating to the Thessaly Railways station, next to the mainline (standard gauge) station. Volos—Mileai (Pelion railway) Construction started in 1882 under the general management of chief engineer Evaristo de Chirico. The section from Volos to Larissa, 61 km long, was inaugurated on 22 April 1884 by King George I. The 142 km section from Velestinon to Kalambaka was completed on 16 June 1886. Rolling stock Steam locomotives Thessaly railways used 46 metre gauge steam locomotives of various types: All of them were tank locomotives, without tenders. Railcars - Diesel multiple units In addition, three Breda railcars of a different type were transferred to Volos from Messolongi-Agrinion line in 1976. Three Linke-Hofmann DMU-2 were transferred in 1978 from the Peloponnese network. Four class 9401 Mitsumbishi diesel locomotives (numbers 9416 to 9419) were also used in Thessaly after 1973 and were used for shunting and as a replacement of steam locomotives for freight trains. The Thessaly network after 1955 The Hellenic State Railways (OSE) absorbed Thessaly Railways in 1955. In 1960 the line from Larissa to Volos was converted to standard gauge and was connected at Larissa with the mainline from Athens to Thessaloniki. For the section between Latomeio and Volos the standard gauge line follows a different route with an additional halt at Melissiatika. The standard gauge line is physically connected to the Athens-Thessaloniki mainline, allowing OSE to run through services to Volos from Athens and Thessaloniki. Volos station was converted to dual gauge, in order to accommodate trains of the two branches. Parts of the station and the track towards the city centre were at this period of a unique triple-gauge system: standard gauge for Larissa trains, metre gauge for Kalambaka trains and gauge for Pelion trains. In 1970 the network was taken over by the new Hellenic Railways Organisation, successor of the Hellenic State Railways. In 2001 the section between Palaiofarsalos and Kalampaka was converted to standard gauge and physically connected at Palaiofarsalos with the mainline from Athens to Thessaloniki. Volos station was converted to exclusively standard gauge. The section to the city centre was abandoned and covered with asphalt. The remaining metre gauge section (Volos-Velestino-Palaiofarsalos) was closed in 1999. Freight traffic declined sharply when the state-imposed monopoly of OSE for the transport of agricultural products and fertilisers ended in the early 1990s. Many small stations of the network with little passenger traffic were closed down, especially on the mainline section and between Karditsa and Kalampaka. However, travel times improved and the unification of rail gauge allowed direct services, even InterCity services, to link Volos and Kalambaka with Athens and Thessaloniki. Recently (August 2009) TrainOSE S.A. proceeded to a drastic cutback of passenger services on Thessaly lines. There are only six local trains on each direction on Larissa-Volos line and just four on Palaiofarsalos-Kalambaka. In addition, there is one train from Athens to Kalambana and back (884/885) and one InterCity train from Athens to Volos and back (IC40/IC41). The mainline of Thessaly was electrified and some services are now using HellasSprinter electric locomotives and Desiro EMUs. There are proposals to electrify the branch lines as well, in order to allow through services from Volos and Kalampaka to Thessaloniki. Preservation When the Hellenic Railways Organisation converted the section Palaiofarsalos-Kalambaka to standard gauge as a branch of the mainline, the metre gauge section from Velestino to Palaiofarsalos was cut off. This section is maintained by the Museum Railways Company or EMOS ( or ΕΜΟΣ). EMOS operates on this section a former SPAP Linke-Hofmann DMU, usually between Velestino and Aerino. EMOS preserves a variety of rolling stock owned by the Hellenic Railways Organisation under long term loan. Most significant is a vintage Linke-Hofmann DMU-2 (1937), formerly of Piraeus, Athens and Peloponnese Railways. They also have a Nippon Sharyo diesel locomotive, formerly of the Aliveri Coalmines of the Public Power Corporation and there are plans to restore a Jung steam locomotive in storage at Volos. In addition, the old single line passing through Tempi Valley, which was cut off from the mainline when new tunnels were constructed, is used as a tourist attraction with light railway vehicles. See also References Further reading Thessaly Museum Railway: Web site of EMOS. Category:Defunct railway companies of Greece Category:Railway companies established in 1884 Category:1884 establishments in Greece Category:Railway companies disestablished in 1955 Category:1955 disestablishments in Greece Category:Rail transport in Thessaly
Mid
[ 0.592452830188679, 39.25, 27 ]
namespace foo vm config filesystem $images/minicccfs vm config hostname foo vm config uuid a5e980ca-3958-48cc-92e9-93d50c0157ad vm launch container foo vm start all namespace bar vm config filesystem $images/minicccfs vm config hostname bar vm config uuid d67f9fed-adea-415a-a66b-301d275a2383 vm launch container bar vm start all clear namespace # wait for clients to start shell sleep 10 .columns uuid,hostname namespace foo cc clients .columns uuid,hostname namespace bar cc clients # run command in one namespace namespace foo cc exec hostname namespace bar cc exec hostname shell sleep 10 # check responses namespace foo cc responses all namespace bar cc responses all # make sure that commands ended up being attributed correctly .columns id,responses,filter namespace foo cc commands .columns id,responses,filter namespace bar cc commands # clear commands in one namespace namespace foo clear cc commands namespace foo .columns id,responses,filter cc commands namespace bar .columns id,responses,filter cc commands
High
[ 0.65625, 36.75, 19.25 ]
Q: Show that $A_{11}$ is invertible $\iff $ $A_{22}$ is invertible. Let $A$ be orthogonal matrix such that $A=$\begin{bmatrix} A_{11} & A_{12}\\A_{21} & A_{22}\end{bmatrix} Show that $A_{11}$ is invertible $\iff $ $A_{22}$ is invertible. Note :$A_{11}$ is square matrix. Attempt: Suppose that $A_{11}$ is non-invertible then there exists $v_0\neq 0$ such that $A_{11}v=0$ Since $A$ is othogonal $\implies A_{11}A_{21}+A_{12}A_{22}=0;A_{11}A_{12}+A_{21}A_{22}=0;A_{11}^2+A_{12}^2=I$ For any $v\neq 0$ we have $(A_{11}^2+A_{12}^2)v=v\neq 0\implies A_{12}v_0\neq 0$ Similarly $A_{21}v_0\neq 0$ But I could not proceed further.Will you please give some hints. A: If $A_{11}$ is singular, $A_{11}v=0$ for some $v\neq 0$. Then $$ Bv:=\begin{bmatrix} A_{11}\\A_{21} \end{bmatrix} v =: \begin{bmatrix} 0\\w \end{bmatrix}, $$ where $w\neq 0$ since $B$ has orthonormal columns. The orthogonality of $A$ gives $$ A_{22}^TA_{21}=-A_{12}^TA_{11} $$ and hence multiplying with $v$ from right gives $$ A_{22}^Tw=0. $$ Since $w\neq 0$, $A_{22}$ is singular.
High
[ 0.706849315068493, 32.25, 13.375 ]
Nobiskrug shipyard on the river Eider used wood supplied by a Burmese government-controlled firm which was sanctioned earlier this year for exporting teak from threatened forests, lawyer for the state Axel Bieler said. Timber experts analysed samples of wood used in the construction of Sailing Yacht A, a 143-metre sail-assisted motor yacht believed to be among the largest in the world, and determined from examining the composition and ring structure that it did not come from a legal plantation. Instead, it is likely the tropical hardwood came from forests in Myanmar, which are being cut down at an alarming rate despite a national ban on exporting raw teak, Mr Bieler told German news agency DPA. Since 2010, Myanmar – one of only four countries in the world where teak trees grow naturally – has lost more than 1.3 million acres of forest on average each year, according to a report by the UN’s Food and Agriculture Organisation. Mr Bieler said it was unclear whether the illegal origin of the wood, which was sold by Myanmar Timber Enterprise (MTE) through Spanish supplier Teak Solutions, was known to the shipyard. Allegations that the teak was logged illegally were first raised by the London-based NGO the Environmental Investigation Agency in October 2016. After a two-month investigation, the EIA accused Teak Solutions, along with eight other European suppliers, of willful ignorance on the source of their timber. The organisation said at the time: “EIA identified a shipment of 1,278 pieces of teak costing 174,750 euros ($186,000) being used aboard the yacht. This was one of many shipments from Myanmar by Teak Solutions, imported in direct violation of the European Union Timber Regulation (EUTR). “All nine companies failed to identify or verify the source of the teak, a clear right to harvest or information that might mitigate the risks of harvesting in violation of relevant forestry provisions." Teak Solutions bought the timber from Myanmar Timber Enterprise (MTE), a state-run wood trading company that was recently sanctioned by Denmark for supplying teak of dubious-origin. View photos The remains of a forest where teak trees once grew in Bago, Burma (Getty) At the time, Teak Solutions CEO Mike Kimble told German newspaper Der Spiegel his Burmese imports were legal and met EU regulations, saying dealing with MTE was the “safest” way to source the wood. The shipyard told local newspaper Kieler Nachrichten: “We support the investigations of Kiel's public prosecutor and we are cooperating fully with the authorities. We have a great interest in quickly disproving the allegations, especially since the Federal Agency for Agriculture and Food halted similar investigations in the same case against us last year.” The criminal investigation led by Mr Bieler is ongoing. Almost two per cent of the Myanmar's forest cover was lost each year from 2010 to 2015, or eight-and-a-half per cent over the five years. In March, MTE released a statement saying it would take steps to address illegal logging and its associated trade. The tropical hardwood is prized by boat builders for decking because it is strong, easy to work with, resistant to rot and does not shrink. Sailing Yacht A, which is estimated to have cost more than £350 million, was built for Russian billionaire Andrey Igorevich Melnichenko, who owns energy and fertiliser companies. Mr Melnichenko, who has an estimated net worth of $13.4 billion (£10.8 billion), is said to favour naming his vessels 'A' so that they are listed first in shipping registers.Sailing Yacht A, which was has three 300ft masts that are taller than Big Ben's tower, is Mr Melnichenko's second yacht by French designer Philippe Starck — his first is Motor Yacht A. The German shipbuilder's website described the eight-deck yacht as “the most advanced sail-assisted superyacht ever built”. The luxury sea-cruiser features a helipad, onboard swimming pool, and underwater observation pod. A spokesman for the owner of Sailing Yacht A said: "We are extremely disappointed that the high profile of Sailing Yacht A is being used by others to raise an issue which has nothing to do with the yacht or the owner. Virtually every superyacht in the world uses Burmese teak. This issue is for the shipyard and supplier of the teak."
Mid
[ 0.5633187772925761, 32.25, 25 ]
1. Technical Field The present disclosure relates to electrosurgical devices suitable for use in tissue ablation applications and, more particularly, to ablation devices with adjustable radiating section lengths, electrosurgical systems including the same, and methods of adjusting ablation fields using the same. 2. Discussion of Related Art Treatment of certain diseases requires the destruction of malignant tissue growths, e.g., tumors. Electromagnetic radiation can be used to heat and destroy tumor cells. Treatment may involve inserting ablation probes into tissues where cancerous tumors have been identified. Once the probes are positioned, electromagnetic energy is passed through the probes into surrounding tissue. In the treatment of diseases such as cancer, certain types of tumor cells have been found to denature at elevated temperatures that are slightly lower than temperatures normally injurious to healthy cells. Known treatment methods, such as hyperthermia therapy, heat diseased cells to temperatures above 41° C. while maintaining adjacent healthy cells below the temperature at which irreversible cell destruction occurs. These methods involve applying electromagnetic radiation to heat, ablate and/or coagulate tissue. Microwave energy is sometimes utilized to perform these methods. Other procedures utilizing electromagnetic radiation to heat tissue also include coagulation, cutting and/or ablation of tissue. Electrosurgical devices utilizing electromagnetic radiation have been developed for a variety of uses and applications. A number of devices are available that can be used to provide high bursts of energy for short periods of time to achieve cutting and coagulative effects on various tissues. There are a number of different types of apparatus that can be used to perform ablation procedures. Typically, microwave apparatus for use in ablation procedures include a microwave generator that functions as an energy source, and a microwave surgical instrument (e.g., microwave ablation probe) having an antenna assembly for directing the energy to the target tissue. The microwave generator and surgical instrument are typically operatively coupled by a cable assembly having a plurality of conductors for transmitting microwave energy from the generator to the instrument, and for communicating control, feedback and identification signals between the instrument and the generator. There are several types of microwave probes in use, e.g., monopole, dipole and helical, which may be used in tissue ablation applications. In monopole and dipole antenna assemblies, microwave energy generally radiates perpendicularly away from the axis of the conductor. Monopole antenna assemblies typically include a single, elongated conductor. A typical dipole antenna assembly includes two elongated conductors that are linearly aligned and positioned end-to-end relative to one another with an electrical insulator placed therebetween. Helical antenna assemblies include helically-shaped conductor configurations of various dimensions, e.g., diameter and length. The main modes of operation of a helical antenna assembly are normal mode (broadside), in which the field radiated by the helix is maximum in a perpendicular plane to the helix axis, and axial mode (end fire), in which maximum radiation is along the helix axis. A microwave transmission line typically includes a long, thin inner conductor that extends along the longitudinal axis of the transmission line and is surrounded by a dielectric material and is further surrounded by an outer conductor around the dielectric material such that the outer conductor also extends along the transmission line axis. In one variation of an antenna, a waveguiding structure, such as a length of transmission line or coaxial cable, is provided with a plurality of openings through which energy “leaks” or radiates away from the guiding structure. This type of construction is typically referred to as a “leaky coaxial” or “leaky wave” antenna. During certain procedures, it can be difficult to assess the extent to which the microwave energy will radiate into the surrounding tissue, making it difficult to determine the area or volume of surrounding tissue that will be ablated. Ablation volume is correlated with antenna design, antenna performance, antenna impedance, ablation time and wattage, and tissue characteristics, e.g., tissue impedance. The particular type of tissue ablation procedure may dictate a particular ablation volume in order to achieve a desired surgical outcome. By way of example and without limitation, a spinal ablation procedure may call for a longer, narrower ablation volume, whereas in a prostate ablation procedure a more spherical ablation volume may be required. Treatment of certain tumors may involve probe repositioning during the ablation procedure, such as where the tumor is larger than the probe or has a shape that does not correspond with available probe geometry or radiation pattern. Ablation procedures may be improved by avoiding inadvertent application of ablative energy to tissue structures, such as large vessels, healthy organs, sensitive neural structures, or vital membrane barriers. Tissue ablation devices capable of influencing ablation volume may enable more precise ablation treatments, which may lead to shorter patient recovery times, fewer complications from undesired tissue damage, and improved patient outcomes.
Mid
[ 0.5955882352941171, 30.375, 20.625 ]
Q: Including and excluding globbing patterns in bash I am trying to use the PHPCompatibility standard for PHP CodeSniffer to test a specific set of files. I'm currently using find to do this. find ./path1 ./path2 ./path3 ./path4 \ -type f \ -name '*.php' \ -not -path './path4/dontwantthis/*' \ -exec ./vendor/bin/phpcs \ --standard=PHPCompatibility --runtime-set testVersion 5.6 {} \; However, this is really slow and inefficient because the startup for the script runs for every single file. The phpcs script takes in a path like ./vendor/bin/phpcs --standard=PHPCompatibility --runtime-set testVersion 5.6 <path-of-your-php-files> and I'd like to find a way to replicate the find stuff with a globbing pattern in place of the <path-of-your-php-files> One of the major problems I was having was including ./path4/*.php while also excluding ./path4/dontwantthis/* A: End your -exec option with + instead of \;. That tells find to run the command once with all the filenames, rather than separately for each filename. find ./path1 ./path2 ./path3 ./path4 \ -type f \ -name '*.php' \ -not -path './path4/dontwantthis/*' \ -exec ./vendor/bin/phpcs \ --standard=PHPCompatibility --runtime-set testVersion 5.6 {} +
Mid
[ 0.645569620253164, 31.875, 17.5 ]
<!DOCTYPE HTML> <html lang="en"> <head> <title>three.js - hatching - postprocessing - webgl</title> <meta charset="utf-8"> <style type="text/css"> body { color: #fff; font-family:Monospace; font-size:13px; text-align:center; font-weight: bold; background-color: #000; margin: 0px; overflow: hidden; } #info { color:#fff; position: absolute; top: 0px; width: 100%; padding: 5px; } #oldie { font-family:monospace; font-size:13px; text-align:center; background:#eee; color:#000; padding:1em; width:475px; margin:5em auto 0; display:none; } a { color: red; } </style> </head> <body> <div id="container"></div> <center> <div id="oldie"> Sorry, your browser doesn't support <a href="hxxp://khronos.org/webgl/wiki/Getting_a_WebGL_Implementation">WebGL</a> and <a href="hxxp://www.whatwg.org/specs/web-workers/current-work/">Web Workers</a>.<br/> <br/> Please try in <a href="hxxp://www.chromium.org/getting-involved/dev-channel">Chrome 9+</a> / <a href="hxxp://www.mozilla.com/en-US/firefox/all-beta.html">Firefox 4+</a> / <a href="hxxp://nightly.webkit.org/">Safari OSX 10.6+</a> </div> </center> <script type="text/javascript" src="js/ThreeExtras.js"></script> <script type="text/javascript" src="js/Stats.js"></script> <!-- ----------------------------------------------------------------- Convolution shader - ported from o3d sample to WebGL / GLSL http://o3d.googlecode.com/svn/trunk/samples/convolution.html ----------------------------------------------------------------- -> <!-- Convolution fragment shader --> <script id="fs-convolution" type="x-shader/x-fragment"> varying vec2 vUv; uniform sampler2D tDiffuse; uniform vec2 uImageIncrement; #define KERNEL_SIZE 25 uniform float cKernel[KERNEL_SIZE]; void main(void) { vec2 imageCoord = vUv; vec4 sum = vec4( 0.0, 0.0, 0.0, 0.0 ); for( int i=0; i<KERNEL_SIZE; ++i ) { sum += texture2D( tDiffuse, imageCoord ) * cKernel[i]; imageCoord += uImageIncrement; } gl_FragColor = sum; } </script> <!-- Convolution vertex shader --> <script id="vs-convolution" type="x-shader/x-vertex"> varying vec2 vUv; uniform vec2 uImageIncrement; #define KERNEL_SIZE 25.0 void main(void) { //vUv = vec2( uv.x, 1.0 - uv.y ) - ((KERNEL_SIZE - 1.0) / 2.0) * uImageIncrement; vUv = uv - ((KERNEL_SIZE - 1.0) / 2.0) * uImageIncrement; gl_Position = projectionMatrix * modelViewMatrix * vec4( position, 1.0 ); } </script> <!-- Render parameter modulated texture fragment shader --> <script id="fs-screen" type="x-shader/x-fragment"> varying vec2 vUv; uniform sampler2D tDiffuse; uniform float opacity; void main(void) { vec4 texel = texture2D( tDiffuse, vUv ); gl_FragColor = opacity * texel; } </script> <!-- Generic vertex shader --> <script id="vs-generic" type="x-shader/x-vertex"> varying vec2 vUv; void main() { vUv = vec2( uv.x, 1.0 - uv.y ); gl_Position = projectionMatrix * modelViewMatrix * vec4( position, 1.0 ); } </script> <script type="text/javascript"> if ( !is_browser_compatible() ) { document.getElementById( "oldie" ).style.display = "block"; } var ShaderTest = { 'hatching' : { uniforms: { "uDirLightPos": { type: "v3", value: new THREE.Vector3() }, "uDirLightColor": { type: "c", value: new THREE.Color( 0xeeeeee ) }, "uAmbientLightColor": { type: "c", value: new THREE.Color( 0x050505 ) }, "uBaseColor": { type: "c", value: new THREE.Color( 0xffffff ) }, "uLineColor1": { type: "c", value: new THREE.Color( 0x000000 ) }, "uLineColor2": { type: "c", value: new THREE.Color( 0x000000 ) }, "uLineColor3": { type: "c", value: new THREE.Color( 0x000000 ) }, "uLineColor4": { type: "c", value: new THREE.Color( 0x000000 ) } }, vertex_shader: [ "varying vec3 vNormal;", "void main() {", "gl_Position = projectionMatrix * modelViewMatrix * vec4( position, 1.0 );", "vNormal = normalize( normalMatrix * normal );", "}" ].join("\n"), fragment_shader: [ "uniform vec3 uBaseColor;", "uniform vec3 uLineColor1;", "uniform vec3 uLineColor2;", "uniform vec3 uLineColor3;", "uniform vec3 uLineColor4;", "uniform vec3 uDirLightPos;", "uniform vec3 uDirLightColor;", "uniform vec3 uAmbientLightColor;", "varying vec3 vNormal;", "void main() {", "float directionalLightWeighting = max( dot( normalize(vNormal), uDirLightPos ), 0.0);", "vec3 lightWeighting = uAmbientLightColor + uDirLightColor * directionalLightWeighting;", "gl_FragColor = vec4( uBaseColor, 1.0 );", "if ( length(lightWeighting) < 1.00 ) {", "if ( mod(gl_FragCoord.x + gl_FragCoord.y, 10.0) == 0.0) {", "gl_FragColor = vec4( uLineColor1, 1.0 );", "}", "}", "if ( length(lightWeighting) < 0.75 ) {", "if (mod(gl_FragCoord.x - gl_FragCoord.y, 10.0) == 0.0) {", "gl_FragColor = vec4( uLineColor2, 1.0 );", "}", "}", "if ( length(lightWeighting) < 0.50 ) {", "if (mod(gl_FragCoord.x + gl_FragCoord.y - 5.0, 10.0) == 0.0) {", "gl_FragColor = vec4( uLineColor3, 1.0 );", "}", "}", "if ( length(lightWeighting) < 0.3465 ) {", "if (mod(gl_FragCoord.x - gl_FragCoord.y - 5.0, 10.0) == 0.0) {", "gl_FragColor = vec4( uLineColor4, 1.0 );", "}", "}", "}" ].join("\n") } }; var container, stats; var cameraOrtho, cameraPerspective, sceneRTT, sceneScreen, sceneBG, renderer, mesh, directionalLight; var windowHalfX = window.innerWidth / 2; var windowHalfY = window.innerHeight / 2; var rtTexture, materialScreen, materialConvolution, blurx, blury, quadBG, quadScreen; init(); setInterval( loop, 1000 / 60 ); function init() { container = document.getElementById( 'container' ); cameraOrtho = new THREE.Camera(); cameraOrtho.projectionMatrix = THREE.Matrix4.makeOrtho( window.innerWidth / - 2, window.innerWidth / 2, window.innerHeight / 2, window.innerHeight / - 2, -10000, 10000 ); cameraOrtho.position.z = 100; cameraPerspective = new THREE.Camera( 50, window.innerWidth / window.innerHeight, 1, 10000 ); cameraPerspective.position.z = 900; sceneRTT = new THREE.Scene(); sceneScreen = new THREE.Scene(); sceneBG = new THREE.Scene(); directionalLight = new THREE.DirectionalLight( 0xffffff ); directionalLight.position.x = 0; directionalLight.position.y = 0; directionalLight.position.z = 1; directionalLight.position.normalize(); sceneRTT.addLight( directionalLight ); rtTexture1 = new THREE.RenderTarget( window.innerWidth, window.innerHeight, { min_filter: THREE.LinearFilter, mag_filter: THREE.LinearFilter } ); rtTexture2 = new THREE.RenderTarget( 256, 512, { min_filter: THREE.LinearFilter, mag_filter: THREE.LinearFilter } ); rtTexture3 = new THREE.RenderTarget( 512, 256, { min_filter: THREE.LinearFilter, mag_filter: THREE.LinearFilter } ); materialScreen = new THREE.MeshShaderMaterial( { uniforms: { tDiffuse: { type: "t", value: 0, texture: rtTexture1 }, opacity: { type: "f", value: 0.4 } }, vertex_shader: document.getElementById( 'vs-generic' ).textContent, fragment_shader: document.getElementById( 'fs-screen' ).textContent, blending: THREE.AdditiveBlending } ); var kernel = buildKernel( 4.0 ); blurx = new THREE.Vector2( 0.001953125, 0.0 ), blury = new THREE.Vector2( 0.0, 0.001953125 ); materialConvolution = new THREE.MeshShaderMaterial( { uniforms: { tDiffuse: { type: "t", value: 0, texture: rtTexture1 }, uImageIncrement: { type: "v2", value: blury }, cKernel: { type: "fv1", value: kernel } }, vertex_shader: document.getElementById( 'vs-convolution' ).textContent, fragment_shader: document.getElementById( 'fs-convolution' ).textContent } ); var plane = new Plane( window.innerWidth, window.innerHeight ); shader = ShaderTest["hatching"]; material1 = new THREE.MeshShaderMaterial( { uniforms: Uniforms.clone( shader.uniforms ), vertex_shader: shader.vertex_shader, fragment_shader: shader.fragment_shader } ); material2 = new THREE.MeshShaderMaterial( { uniforms: Uniforms.clone( shader.uniforms ), vertex_shader: shader.vertex_shader, fragment_shader: shader.fragment_shader } ); material1.uniforms.uDirLightPos.value = material2.uniforms.uDirLightPos.value = directionalLight.position; material2.uniforms.uDirLightColor.value = material2.uniforms.uDirLightColor.value = directionalLight.color; var lineColor1 = 0xff0000, lineColor2 = 0x0000ff; material1.uniforms.uBaseColor.value.setHex( 0x000000 ); material1.uniforms.uLineColor1.value.setHex( lineColor1 ); material1.uniforms.uLineColor2.value.setHex( lineColor1 ); material1.uniforms.uLineColor3.value.setHex( lineColor1 ); material1.uniforms.uLineColor4.value.setHex( 0xffff00 ); material2.uniforms.uBaseColor.value.setHex( 0x000000 ); material2.uniforms.uLineColor1.value.setHex( lineColor2 ); material2.uniforms.uLineColor2.value.setHex( lineColor2 ); material2.uniforms.uLineColor3.value.setHex( lineColor2 ); material2.uniforms.uLineColor4.value.setHex( 0x00ffff ); loader = new THREE.Loader( true ); document.body.appendChild( loader.statusDomElement ); loader.loadAscii( { model: "obj/leeperrysmith/LeePerrySmith.js", callback: function( geometry ) { createMesh( geometry, sceneRTT, 100 ) } } ); quadScreen = new THREE.Mesh( plane, materialConvolution ); quadScreen.position.z = -100; sceneScreen.addObject( quadScreen ); renderer = new THREE.WebGLRenderer(); renderer.setSize( window.innerWidth, window.innerHeight ); renderer.autoClear = false; container.appendChild( renderer.domElement ); stats = new Stats(); stats.domElement.style.position = 'absolute'; stats.domElement.style.top = '0px'; //container.appendChild( stats.domElement ); } function createMesh( geometry, scene, scale ) { var mesh1 = new THREE.Mesh( geometry, material1 ); mesh1.scale.x = mesh1.scale.y = mesh1.scale.z = scale; mesh1.position.x = -300; mesh1.position.y = -50; scene.addObject( mesh1 ); mesh = new THREE.Mesh( geometry, material2 ); mesh.scale.x = mesh.scale.y = mesh.scale.z = scale; mesh.rotation = mesh1.rotation; mesh.position.x = 300; mesh.position.y = -50; scene.addObject( mesh ); loader.statusDomElement.style.display = "none"; } /*****************************************************************************************/ // Convolution // - ported from o3d convolution shader sample // http://o3d.googlecode.com/svn/trunk/samples/convolution.html /*****************************************************************************************/ // We lop off the sqrt(2 * pi) * sigma term, since we're going to normalize anyway. function gauss( x, sigma ) { return Math.exp( - (x * x) / (2.0 * sigma * sigma) ); } function buildKernel( sigma ) { var kMaxKernelSize = 25; var kernelSize = 2 * Math.ceil( sigma * 3.0 ) + 1; if ( kernelSize > kMaxKernelSize ) kernelSize = kMaxKernelSize; var halfWidth = ( kernelSize - 1 ) * 0.5 var values = new Array( kernelSize ); var sum = 0.0; for( var i = 0; i < kernelSize; ++i ) { values[ i ] = gauss( i - halfWidth, sigma ); sum += values[ i ]; } // normalize the kernel for( var i = 0; i<kernelSize; ++i ) values[ i ] /= sum; return values; } var start = 0; function loop() { if ( ! start ) start = new Date().getTime(); var time = ( new Date().getTime() - start ) * 0.0004; if ( mesh ) { mesh.rotation.y = -time; } renderer.clear(); // Render scene into texture renderer.context.disable( renderer.context.DEPTH_TEST ); renderer.render( sceneBG, cameraOrtho, rtTexture1 ); renderer.context.enable( renderer.context.DEPTH_TEST ); renderer.render( sceneRTT, cameraPerspective, rtTexture1 ); // Render quad with blured scene into texture (convolution pass 1) quadScreen.materials = [ materialConvolution ]; materialConvolution.uniforms.tDiffuse.texture = rtTexture1; materialConvolution.uniforms.uImageIncrement.value = blurx; renderer.render( sceneScreen, cameraOrtho, rtTexture2 ); // Render quad with blured scene into texture (convolution pass 2) materialConvolution.uniforms.tDiffuse.texture = rtTexture2; materialConvolution.uniforms.uImageIncrement.value = blury; renderer.render( sceneScreen, cameraOrtho, rtTexture3 ); // Render original scene with superimposed blur to texture quadScreen.materials = [ materialScreen ]; materialScreen.uniforms.tDiffuse.texture = rtTexture3; materialScreen.uniforms.opacity.value = 1.5; renderer.render( sceneScreen, cameraOrtho, rtTexture1, false ); // Render to screen materialScreen.uniforms.tDiffuse.texture = rtTexture1; renderer.render( sceneScreen, cameraOrtho ); stats.update(); } function is_browser_compatible() { // WebGL support try { var test = new Float32Array(1); } catch(e) { return false; } // Web workers return !!window.Worker; } </script> </body> </html>
Low
[ 0.49048625792811806, 29, 30.125 ]
Us dating sites review kristiansund Thai massasje haugesund single date 778 Thai massasje haugesund single date Thelin Thai massasje fra Majorstua, Oslo. Juliette I've browsed a lot of other sport store templates, but this TemplateMonster's one has been the best choice of mine so far. Og var ganske så. Erotic, massage Posted by: First sex date on video.
Low
[ 0.44351464435146404, 26.5, 33.25 ]
Q: How to add a batch button in Joomla? hello all actually I'm developing a component for school in which i want to add a batch button for asking about fail & leave fail & reappear pass & promote, pass & leave and for this i want to add a batch button... I'm not so expertise in joomla development can anyone help me how to add a batch button i also try to copy batch code from articles view but not working. so i consider to learn it. HELP ME PLEASE A: Add this to your view.html.php to add the button to the toolbar: // Get the toolbar object instance $bar = JToolBar::getInstance('toolbar'); JHtml::_('bootstrap.modal', 'collapseModal'); $title = JText::_('JTOOLBAR_BATCH'); // Instantiate a new JLayoutFile instance and render the batch button $layout = new JLayoutFile('joomla.toolbar.batch'); $dhtml = $layout->render(array('title' => $title)); $bar->appendButton('Custom', $dhtml, 'batch'); and put this into your default.php: <div class="modal hide fade" id="collapseModal"> <div class="modal-header"> <button type="button" class="close" data-dismiss="modal">&#215;</button> <h3>My modal window</h3> </div> <div class="modal-body modal-batch"> </div> <div class="modal-footer"> </div> </div> to display an empty modal window with the heading "My modal window" when you click on the batch button. You can see how the modal window for the articles view is build at /administrator/components/com_content/views/articles/tmpl/default_batch.php (but you probably know that already)
High
[ 0.681379310344827, 30.875, 14.4375 ]
Q: Checked Checkbox not updated in UI I have a problem regarding checking checkbox by JS.When I checked the checkbox by JS it seems like in program that it's checked but in UI it's not updated . if anyone have solution for this <!DOCTYPE html> <html> <head> <title>Check</title> <meta name="viewport" content="width=device-width, initial-scale=1"> <link rel="stylesheet" href="http://code.jquery.com/mobile/1.1.0/jquery.mobile-1.1.0.min.css" /> <script src="http://code.jquery.com/jquery-1.7.1.min.js"></script> <script src="http://code.jquery.com/mobile/1.1.0/jquery.mobile-1.1.0.min.js"></script> <script> function getCheckedValue() { return ((document.getElementById("rock").checked)); } function setCheckedValue(toBeChecked){ document.getElementById(toBeChecked).checked="checked"; alert((document.getElementById(toBeChecked).checked)) alert($('#'+toBeChecked).attr('checked')) } </script> </head> <body> <div data-role="page"> <div data-role="header"> <h1>What kind/s of music do you listen to?</h1> </div><!-- /header --> <div data-role="content"> <input type="checkbox" class="music" name="music" id="rock" value="rock" > <label for="rock">Rock</label> <input type="button" value="Get" onclick="alert(getCheckedValue())"> <input type="button" value="Set" onclick="setCheckedValue('rock') "> </div> </div> </body> </html> A: Instead of using attr I would highly recommend you use prop. The preferred cross-browser-compatible way to determine if a checkbox is checked is to check for a "truthy" value on the element's property using one of the following: if ( elem.checked ) if ( $(elem).prop("checked") ) if ( $(elem).is(":checked") ) The .prop() method should be used to set disabled and checked instead of the .attr() method. The .val() method should be used for getting and setting value. $("input").prop("disabled", false); $("input").prop("checked", true); $("input").val("someValue"); A: I have worked after getting response from all of your efforts and the working code is below. <!DOCTYPE html> <html> <head> <title>Check</title> <meta name="viewport" content="width=device-width, initial-scale=1"> <link rel="stylesheet" href="http://code.jquery.com/mobile/1.1.0/jquery.mobile-1.1.0.min.css" /> <script src="http://code.jquery.com/jquery-1.7.1.min.js"></script> <script src="http://code.jquery.com/mobile/1.1.0/jquery.mobile-1.1.0.min.js"></script> <script> function getCheckedValue() { return (document.getElementById("rock").checked); //return $('input[name=music]').checked; } function setCheckedValue(checkboxName,toBeChecked){ $('input[name='+checkboxName+']').prop("checked", true).checkboxradio("refresh"); } </script> </head> <body> <div data-role="page"> <div data-role="header"> <h1>What kind/s of music do you listen to?</h1> </div><!-- /header --> <div data-role="content"> <input type="checkbox" class="music" name="music" id="rock" value="rock" > <label for="rock">Rock</label> <input type="button" value="Get" onclick="alert(getCheckedValue())"> <input type="button" value="Set" onclick="setCheckedValue('music','rock') "> </div> </div> </body> </html>
Low
[ 0.5079787234042551, 23.875, 23.125 ]
Articles During the last year, the Islamic State’s (ISIS) presence in the Philippines has fallen into the emerging narrative of the group’s defeat. Filipino forces ended ISIS’ months-long siege on the city of Marawi in October of 2017, just three months after the group’s loss of Mosul and roughly a week after its loss of Raqqah. From here, the comforting narrative sold itself: the mess of ISIS in the Philippines, like in other countries, was finally being cleaned up. But perhaps not. On Tuesday, July 31, ISIS’ East Asia Division announced that a militant "Abu Kathir al-Maghribi [the Moroccan]” carried out a suicide operation on a Philippine army post in Lamitan city on the island of Basilan. The operation, which ISIS reported to have killed 15 (news outlets have reported 11), shows that ISIS is anything but defeated in the region. Rather, it appears to be expanding its aims and capabilities there. This is ISIS’ First Ever Suicide Operation in the Philippines Before Tuesday, ISIS’ activity in the country before was limited to conventional armed clashes with Philippine forces in addition to projectile strikes and IED blasts. After being ousted from Marawi in October of 2017, ISIS continued to carry out such attacks in places like Sulu, where the group claimed nine attacks since January alone. Despite this activity, the group would still never perform a suicide operation—let alone a VBIED attack—in the country until this past Tuesday, which brings me to my next point. The Use of a VBIED Showcases a New Tactic Tuesday’s suicide operation took place when Filipino troops stopped a van with a lone driver who then detonated the explosives inside, confirming ISIS’ report of a VBIED. ...this first suicide operation, let alone one made via a VBIED, may very well have opened a Pandora’s box for similar attacks in the future. VBIEDs, an acronym for Vehicle-Born Improvised Explosive Devices, are staples of high-profile suicide operations in countries like Afghanistan, Iraq, and Syria, where ISIS maintains operational sites, materials, and weapons experts at hand. Constructing these weapons—which of course vary in degrees of sophistication (from armor-plated trucks in Iraq to the van used on Basilian)—require a baseline level of experience not before seen in the Philippines. With that, this first suicide operation, let alone one made via a VBIED, may very well have opened a Pandora’s box for similar attacks in the future. First Attack by Non-Regional Migrant Abu Kathir al-Maghribi is the first non-regional ISIS fighter to be attributed to an attack in the Philippines. Though fighters from neighboring countries like Indonesia and Malaysia have been identified as fighters with ISIS in the Philippines, the group’s promoted migrant membership there has been limited to Southeast Asia. Abu Kathir’s Moroccan nationality is all the more surprising considering the local-focus of ISIS’ migration calls to the Philippines during the past two years in its videos, magazines, and other media. For example, a June 21, 2016 official ISIS video dedicated to the Philippines features Malaysian fighter Abu ‘Awn al-Malizi. From Syria, he calls for fighters across East Asia unable to join the battles in the Philippines: To those who are unable to immigrate to the land of Sham [Syria]: Mobilize and join the mujahideen in the Philippines. If you have an excuse, then send your sons to the land of jihad in the Philippines. Likewise, an August 2017 video from ISIS’ al-Hayat Media Center features an Australian fighter, identified as “Abu Adam,” calling for Australians to take advantage of their country’s proximity to the Philippines, stating in English: As for those of you who can't make it to this battle, the Battle of al-Ahzab [Raqqah], in particular, to the muwahhidin [monotheists] in Australia, then you should go aid your brothers in the fight against the Crusader government of the Philippines. The Australian government has already pledged to help and assist the taghut [tyrant] there. So where is your pledge to Allah, O Muslim? Even those non-regionally focused calls to the Philippines have been open-ended at best. For instance, Issue 10 of ISIS’ Rumiyah magazine, released in June of 2017, features an interview with Abu ‘Abdillah al-Muhajir, whom the article identifies as “the amir [leader] of the soldiers of the Khilafah in East Asia.” In the interview, Abu ‘Abdillah is asked, “Do you continue to receive muhajirin [migrants] up until now? And is there a way open for those who wish to join you?” He answers generally: Yes – and all praise is due to Allah – we continue to receive muhajirin, and we welcome them. There are several safe paths and ways to achieve that… However, Tuesday’s attack—and ISIS’ subsequent glorification of him—sends a very new and very intentional signal to the group’s supporters: The Philippines is your battlefront, regardless of which corner of the Earth you’re from. We still don’t know how Abu Kathir reached the Philippines, or if he did so from his home country or battlefronts in Syria, Iraq, or elsewhere. However, reports out of the Philippines have described incoming militants from other battlegrounds. Ebrahim Murad, leader of the Philippines Moro Islamic Liberation Front (MILF), even claimed early this year: “Based on our own intelligence information, foreign fighters who were displaced from the Middle East continued to enter into our porous borders…” Such reports are not far-fetched; ISIS has very openly sought new territorial hubs after its losses in Iraq and Syria, and the Philippines, a country with a proven body of supporters, would be a fitting place for ISIS to invest resources. ISIS Central’s Newfound Coordination in Attacks Further suggesting the prospect that ISIS is increasing its investment in the Philippines is that the group claimed responsibility for Tuesday’s attack on the same day it happened. This timing is notable considering the group’s lack of communication with those on the ground in the Philippines. Since ISIS established a presence in the Philippines, the organization’s central command previously appeared to have very limited coordination and communication with its members there. During the peak of ISIS militants’ notorious siege on Marawi, the most up-to-date reports of activity came not from ISIS communiques, but rather messages from Filipino operatives on Telegram channels and chat groups. In late March, ISIS resumed operations in Jolo, Sulu. It was during this time that communications between fighters and ISIS’ central command improved, resulting in the group's first of six formal communiques issued on April 7, along with a dozen statements by ISIS’ ‘Amaq News Agency regarding clashes against Philippine forces. For Tuesday’s attack, ISIS claimed responsibility just hours after it took place via a formal communique, which was translated into several languages. The communique provided various details about the attack that were not published anywhere else, including the name of the attacker, stating: 15 Filipino Soldiers Are Killed in an Istishhadi Operation on the Island of Basilan With success granted by Allah, the istishhadi, Abu Kathir al-Maghribi (may Allah accept him) managed to reach a gathering of Crusader Filipino soldiers in the village of Bajanda near the city of Lamitan on the island of Basilan, where he detonated his explosive vehicle, killing 15 of them and injuring a number of others, and all praise is due to Allah. ISIS even provided a photograph of Abu Kathir just a few minutes later, suggesting that the group was prepared for his operation. Conclusion The factors surrounding Tuesday’s attack are telltale red flags that ISIS is looking to establish a more substantial base in the Philippines. ...the troubling facts cannot be changed: ISIS claimed its first suicide operation in the Philippines by a Moroccan man with unignorable speed, showing a direct line between the group and militants there. The Armed Forces of the Philippines has rejected various tenants of ISIS’ attack claim, casting doubt that the attacker was a foreigner. Regarding the attack itself, a military spokesman stated, “Our initial investigations show that it is not a suicide bombing as the driver stopped his van some 60 metres from the security checkpoint and asked people to help push the vehicle.” Of course, an attack like Tuesday’s deserves the upmost objective investigating and attention to details, and officials would be wise not to jump to conclusions or take a terrorist group’s claims at face-value. But the troubling facts cannot be changed: ISIS claimed its first suicide operation in the Philippines by a Moroccan man with unignorable speed, showing a direct line between the group and militants there. It's also critical to add that since ISIS was ousted from Marawi, it didn't stop exploiting the Philippines, but rather relocated its operations to Sulu, where its membership is comprised largely of former Abu Sayaf fighters. The situation is similar to those of Afghanistan, Yemen, Somalia, Mali, Nigeria, and other conflict zones, in that ISIS has been able to bring fighters from local factions under its so-called "caliphate" and establish firm footings. Thus, ISIS’s exploitation of the Philippines should not be seen as a solely local problem; it is one piece of a global operation to exploit conflict regions by first recruiting from the local groups. Looking forward, the Philippines government and neighboring countries must increase stability efforts and resolve longstanding tensions that feed into the recruitment narratives of groups like ISIS. Failure to do so will only play into the terrorist group’s hands.
Mid
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Arrested Development Netflix has greenlit a fifth season (and second on the digital platform) of sitcom “Arrested Development.” Series creator Mitchell Hurwitz and the entire series regular cast, including: Jason Bateman, Michael Cera, Jeffrey Tambor, Jessica Walter, Will Arnett, Tony Hale, Portia de Rossi, David Cross and Alia Shawkat will return in an unconfirmed number of new […]
Mid
[ 0.555831265508684, 28, 22.375 ]
It's new to me that vape shop will use bitcoin as their mode of payment. I have no idea which shop though. For sure many would love to buy vape using bitcoin because I notice that vape is trendy and easy to use not only that but also it is an environmental friendly. I guess there is no harmful content of it. It's just a favored fruits mix with some special ingredient More or less it will attract many customers because of bitcoin. Try to check Purse.io. There are shops there that sell Vape or electronic cigarettes and you can buy them with your bitcoins, including flavorings. If you live in the US, you can simply ask them to ship it to you directly. However, if you live outside the United States, you need to make a US address with USendHome and use that address to forward your order from whichever place you live. Try to check Purse.io. There are shops there that sell Vape or electronic cigarettes and you can buy them with your bitcoins, including flavorings. If you live in the US, you can simply ask them to ship it to you directly. However, if you live outside the United States, you need to make a US address with USendHome and use that address to forward your order from whichever place you live. i live on indonesia now,maybe i will check it,because i'm not like paypal or payoneer or seems method payment like that,i love bitcoin,if any store accept btc,i will buy at there Does it makes sense? Are you ready to pay 160 satoshis/byte or 25$+ for every transaction? If you need to buy something for sum <100$ better to find a vapestore who accept LTC or another coin with little fees.
Low
[ 0.5238095238095231, 35.75, 32.5 ]
We understand our reality, because of the way it's constructed inside of us. We use our conscious to follow the the grooves of our brain, and mistake reality for unfolding in the same way our brain/mind does. This is fallacy. Thus different blue prints, different reality competely.It's not hard to imagine possibilities other than causality.Really easy. Kid's can do it no problem. Not sure why adults have so much trouble with it. When using the imagination, which is the faculty of consciousness that manifests visible forms, this is true. But the imagination of visible causalities did not cause itself to appear. That cause is invisible, is unseen, therefore is unknown, therefore, cannot be analyzed by way of causalities. Which brings one to the realization that in the reality of man's awareness, there is no cause, nor is there an effect. Which brings one also to the realization that every word spoken or written that appears as a result of the erroneous belief in the reality of cause and effect is equal to every other word spoken or written that arises from the same erroneous belief. A = A. When using the imagination, which is the faculty of consciousness that manifests visible forms, this is true. But the imagination of visible causalities did not cause itself to appear. That cause is invisible, is unseen, therefore is unknown, therefore, cannot be analyzed by way of causalities. Which brings one to the realization that in the reality of man's awareness, there is no cause, nor is there an effect. Which brings one also to the realization that every word spoken or written that appears as a result of the erroneous belief in the reality of cause and effect is equal to every other word spoken or written that arises from the same erroneous belief. A = A. It's not hard to imagine possibilities other than causality.Really easy. Kid's can do it no problem. Not sure why adults have so much trouble with it. Kids also wet their beds, no problem. Those same kids have problems walking The world has a way of training magical thinking right out of a person. But do not despair, if you think grown-ups abandon it altogether, just notice how the lines outside a lottery ticket shop lengthen when the jackpot gets big. Magical thinking (dreams, prayers, believing wishing makes a thing happen) makes these people part with the relentless certainty of cause and effect. Worse still are the lottery veterans who now share this line with the newcomers on the bandwagon. They know from experience that they will not win. And yet here they are. It's not hard to imagine possibilities other than causality.Really easy. Kid's can do it no problem. Not sure why adults have so much trouble with it. Kids also wet their beds, no problem. Those same kids have problems walking The world has a way of training magical thinking right out of a person. But do not despair, if you think grown-ups abandon it altogether, just notice how the lines outside a lottery ticket shop lengthen when the jackpot gets big. Magical thinking (dreams, prayers, believing wishing makes a thing happen) makes these people part with the relentless certainty of cause and effect. Worse still are the lottery veterans who now share this line with the newcomers on the bandwagon. They know from experience that they will not win. And yet here they are. Imagination is an ability. People use it to form ideas of reality, in hopes of finding better ways of coping/understanding/affecting their own reality. Some people use it to wish circumstances into existance (such as winning the lotto), while others use it more reasonably. *shrugs shoulders* Well, you weren't talking about imagination in general. You were talking about imagining things happening acausally, and that kids do that easily wile adults do not. Maybe my example obscured my point. It is advantageous for the survival of the individual organism to learn that things occur in a causal fashion. "Imagining" anything else is detrimental. But often imagination plays a key role in understanding cause and effect. Here a child's imagination's lack of ability to see things causally can make him fail. For example, take a child learning to play a musical instrument. Adults may tell him it requires endless practice, but he can never have witnessed such a thing. Certainly he has not experienced it. All he knows is what playing the instrument competently sounds like, and that all of his hard practice has produced nothing like that. More often than not, such a child becomes frustrated and quits. I originally responded to your post because it sounded as if you were saying imagining things happen acausally - which children do easily and adults do not - is a good thing and that you were bemoaning its loss to the average grown-up. I think this is a bit simplistic. I think that children actually have a fantastic belief in cause and effect. They in fact yearn for it, as they perceive themselves as lacking that power that adults seem to have. They crave power, that is, they wish to control their environment. They wish the causes they furnish - their actions or desires, such as repetitive asking for something - to have the desired effect, which is getting what they have asked for. All people, big and small, want more power. This only happens if they learn to master cause and effect, which is a never-ending process. Since all things proceed causally, acausal thinking is detrimental and, if all goes well, gets beaten out of a child as he/she grows. At this point, the question is begged:Does Causality requires a prime mover?An original cause? And if so, doesn't that refute the idea of an infinite existence of light matter?That there must be a beginning to it? Or could the beginning of light matter, having spawned from dark matter, be sufficient to marry both ideas? That way, causality stays in place, and the original Cause was the need of dark matter to create an equal and opposite matter. Thus starting the chain of light matter, making causality plausible, while still leaving time out of the equation. Its late in the workday and i've had too much coffee, sorry for any incoherence in this post. Sazar wrote:At this point, the question is begged:Does Causality requires a prime mover?An original cause? Obviously not. An "original cause" would automatically become a process of causality in the first place; Any and every cause is the result of ripe circumstance, thus a conceived "original cause" was caused. The search for an "original cause" is an infinite wild goose chase. Sazar wrote:At this point, the question is begged:Does Causality requires a prime mover?An original cause? Obviously not. An "original cause" would automatically become a process of causality in the first place; Any and every cause is the result of ripe circumstance, thus a conceived "original cause" was caused. The search for an "original cause" is an infinite wild goose chase. This is one of the most irrational statement I have read anywhere. Causality requires vision. Whether that vision is visible or invisible to human intellectual awareness, that vision is preceded by thought - "first the blade, then the ear, then the full corn in the ear" -. Thought requires a thinker and it matters not what the thinker is labled, it is the thinker thinking the thought which is the prime mover, thus the causality manifested in consciousness. Consciousness projects the vision to the mind. The mind projects the vision to the imagination as an objectification of form. The imagination subjectifies these forms as time, space, distance and matter which become the realized thoughts of man. The search for an original cause cannot be found because the reality of existence is there is no logic for the existence of existence to begin with. Never give power to anything a person believes is their source of strength - jufa jufa wrote:The search for an original cause cannot be found because the reality of existence is there is no logic for the existence of existence to begin with. This is the only statement that makes any sort of sense. The rest of your post, which seems to try to explain some sort of origination of causality, contradicts this statement. Your problem of lack of understanding comes because to go beyond the mind, one must go through the mind. Have you went beyond the human mind? Nonetheless............................................... Because no one cannot find logic for existence does not negate the reality of your existence nor mine. This being a truth, and a present truth, action/reaction is witness of a cause without logic, yet a Cause which purpose thinking and doing. Never give power to anything a person believes is their source of strength - jufa jufa wrote:Because no one cannot find logic for existence does not negate the reality of your existence nor mine. This being a truth, and a present truth, action/reaction is witness of a cause without logic, yet a Cause which purpose thinking and doing. (I assume that was an accidental double negative in the first sentence) I get what you're saying Jufa, but the point is, no matter who or what "goes beyond the mind" or whatever mystical experience you're referring to, one will never find any "original cause," or a causeless cause. Ultimately speaking, of course. In the end, one can figure that the cause of Causality can only be Causality itself.. which isn't entirely accurate because it erroneously presumes that Causality is some sort of concrete "thing." Ah, but this is not true. Each and ever individual is the cause. In this realm they are the element matter of the universe. And when they discover this reality and the invisibility of themselves, they will have discovered that which they were seeking, they are. In this comprehension they will have no knowledge of existence because they will be existence. This is saying Cause is It own object of subjectiveness. This is why a human being will never find any "original cause," or a causeless cause. Never give power to anything a person believes is their source of strength - jufa jufa wrote:Ah, but this is not true. Each and ever individual is the cause. In this realm they are the element matter of the universe. And when they discover this reality and the invisibility of themselves, they will have discovered that which they were seeking, they are. In this comprehension they will have no knowledge of existence because they will be existence. This is saying Cause is It own object of subjectiveness. This is why a human being will never find any "original cause," or a causeless cause. and why, if a person is true to the invisible voice of the infinity of the cause of themself, that they will be moved to explore every ream of effect of this, their invisible, infinite cause. For now, as you say, this realm or effect or emanation of that of the element matter of the universe. Which means that before this movement into other realms of Awareness of one's cause of oneself can be experienced, it is necessary to be liberated from the realm of the element matter of the universe. Worlds within worlds within worlds of I Am That awareness await the discovery of the one who has come to see that the illusory bonds of birth and of death can be broken. Men talk of infinity, not realizing they are the infinity of themselves.
Low
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Thursday, 12 November 2015 Asylum Reviews: Rise of the Tomb Raider [Xbox One]. For over a week now, I've been paying Rise of the Tomb Raider every chance I get. It's very similar to the first in terms of gameplay and feel. And that's not a bad thing. If it ain't broke, don't fix it. As of this morning, I'm around halfway through the game. It is exciting and fun, and having been a huge fan of the 2013 reboot, it's very familiar, so getting used to controls was never a problem. I could dive right in with very little getting to grips with things. It's definitely a game I'm really enjoying, and I'm happy to see how the story pans out. Rise of the Tomb Raider sees Lara embark on a new adventure, to seek out what her father had been searching for before his death. In the icy setting of Siberia, we have her up against Trinity, battling against them as they race to find the "Divine Source", a extraordinary artifact that could be the answer to eternal life, which she has inadvertently led them straight to. We have new weapons and gear, the ability to change Lara's costume and you can also fulfill tasks for the local people that you will come across. The new gear, whilst easy to use and fantastic to play with, sometimes becomes slightly confusing as you try to rush through a tight spot and can't remember which of your expanded arsenal is correct. Is it the wire spool, which in effect turns your climbing axe into a grappling hook, or is it your bow to shoot a strong rope to swing across to the other side? Oh no wait, we used the wrong one and now we've plunged to our deaths. Aaand reload. The game does keep you gripped and make you want to journey on. Though there's not much in terms of "groundbreaking change" between this game and the last, it's definitely not something I'd fault it for. The formula works, so why change it? Obviously, at this point I'm yet to have 100% completed the game, even though I've been playing it a lot over the past week. I definitely like that it's longer than the previous one, as that was my one gripe that I had with it back in 2013. But I can honestly say that there's nothing big enough to really make me say anything bad about this game. The story is great (despite being quite a common theme), the gameplay is fun, the combat is fantastic - although melee is sometimes a tad off, in my opinion - the graphics are beautiful and I just loved it. The only thing that we did initially think was bad (until we realised it was a glitch) was the lack of aiming with your gun. From the beginning of a certain location in the game, and lasting for around 10 minutes or so (as we ran around this tiny spot confused on what to do), we had no reticule when aiming our gun (nor any HUD for our ammo in the top corner), and we lost the ability to pause, the ability to use survival instinct, and no info would come up when we picked up a collectible. This really frustrated us, as we hadn't yet used the gun before then, so weren't sure if this is just how it was, until maybe you unlocked the reticule via your upgrades at camp, but the lack of pause was infuriating, especially when you're wanting to check your progress. Luckily - or not so luckily, for Lara - Lara died when we couldn't aim properly to shoot a falling spiky block that was rushing towards us. And the checkpoint reloaded and woohoo, we finally had a reticule, thus realising that this was in fact a glitch and not some evil-doing of the development team. Allan started his own save and played past this part, not being affected by the glitch at all, so I'm glad to see it's not an outright bug affecting that portion of the game, but simply just something I'd randomly stumbled upon in my own playthrough. Overall, I'd definitely say that Rise of the Tomb Raider is worth purchasing, especially if you were a fan of the previous game. It's fun, it's interesting and it's definitely worth the time spent playing it. I just hope that continues on right until the end. It also looks outstanding and I'm really happy to have the game, as it's sure to be one that I play through to completion. I do think that releasing in the same week as Fallout 4 (same day for those of you in the US) however, was a big risk. I feel that all of the hype of Fallout 4 will have severely detracted from the hype surrounding this game, and thus the sales, and I hope that it doesn't hurt it too badly in the long run. So, are you picking up Rise of the Tomb Raider when it's released tomorrow? Or are you so lost in Fallout 4 that you won't be buying another game till New Year?
Mid
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1. Technical Field The present disclosure relates to an antenna device provided with a plurality of antenna elements and an electromagnetic band gap (EBG) structure. The present disclosure also relates to a wireless communication device and a radar device provided with such an antenna device. 2. Description of the Related Art The use of an EBG structure to ensure isolation between antenna elements in an antenna device that comprises a plurality of antenna elements and operates in an extremely high frequency (EHF) band is conventionally known (see Japanese Unexamined Patent Application Publication Nos. 2009-203147 and 2006-060537). An EBG structure exhibits high impedance at a prescribed frequency (antiresonance frequency). Consequently, an antenna device provided with an EBG structure is able to increase the isolation between antenna elements in this frequency.
Mid
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Luxe Things: Luxetips Style! Forever 21 & Nordstrom Colorblocked Clutches Last year’s colorblocking trend is as strong as ever, but this time with accessories. If you are still not sure (and we hope that you are comfortable with it by now) how to or whether to attempt colorblocking, try adding some of these colorblock accessories to your wardrobe. The Forever 21XL Envelope Clutchis only $ 19.80. It looks like it costs much more. So what if you think you are too seasoned to shop at Forever 21? This is definitely one instance where it is okay. The colorblock clutch is a faux leather with polished frame and 29 inch chain shoulder strap. It closes with a magnetic snap and is has a fully lined polyester interior.Mixed metals are always fun and the price of this Top Choiceclutch is what is most exciting. At $ 13.90 the Top Choice Metallic Colorblock Envelope Clutch is not likely to remain at Nordstrom. This clutch has such versatility that you can wear it with jeans and a blazer or an evening dress. You can wear it as a neutral accessory or attempt to match it with one of the metallics in the purse. Like the Forever 21 XL, a chain strap is included. The removable strap is 21 inches long. I am literally restraining myself right now from putting this in my shopping cart (or perhaps it is more the fact that Hubby is sitting right next to me laying on my dominant arm- you know the arm that clicks “submit order.”)Until tomorrow,
Mid
[ 0.606280193236715, 31.375, 20.375 ]
Q: What Password do you Use When you're Installing Software on a Virtual Machine running 13.04? I am using Windows 8 and through virtualbox I am running 13.04 and it would be just great if I could actually use it for what I wanted it: installing and running GNU Octave. I can't seem to because when I attempt to install in the software centre I am missing one vital thing: a password. I downloaded it from here A: The password which you are looking for is adminuser as mentioned here
High
[ 0.67612293144208, 35.75, 17.125 ]
# frozen_string_literal: true require 'spec_helper' RSpec.describe Gitlab::Ci::Pipeline::Expression::Statement do subject do described_class.new(text, variables) end let(:variables) do { 'PRESENT_VARIABLE' => 'my variable', 'PATH_VARIABLE' => 'a/path/variable/value', 'FULL_PATH_VARIABLE' => '/a/full/path/variable/value', 'EMPTY_VARIABLE' => '' } end describe '.new' do context 'when variables are not provided' do it 'allows to properly initializes the statement' do statement = described_class.new('$PRESENT_VARIABLE') expect(statement.evaluate).to be_nil end end end describe '#evaluate' do using RSpec::Parameterized::TableSyntax where(:expression, :value) do '$PRESENT_VARIABLE == "my variable"' | true '"my variable" == $PRESENT_VARIABLE' | true '$PRESENT_VARIABLE == null' | false '$EMPTY_VARIABLE == null' | false '"" == $EMPTY_VARIABLE' | true '$EMPTY_VARIABLE' | '' '$UNDEFINED_VARIABLE == null' | true 'null == $UNDEFINED_VARIABLE' | true '$PRESENT_VARIABLE' | 'my variable' '$UNDEFINED_VARIABLE' | nil "$PRESENT_VARIABLE =~ /var.*e$/" | true '$PRESENT_VARIABLE =~ /va\r.*e$/' | false '$PRESENT_VARIABLE =~ /va\/r.*e$/' | false "$PRESENT_VARIABLE =~ /var.*e$/" | true "$PRESENT_VARIABLE =~ /^var.*/" | false "$EMPTY_VARIABLE =~ /var.*/" | false "$UNDEFINED_VARIABLE =~ /var.*/" | false "$PRESENT_VARIABLE =~ /VAR.*/i" | true '$PATH_VARIABLE =~ /path\/variable/' | true '$FULL_PATH_VARIABLE =~ /^\/a\/full\/path\/variable\/value$/' | true '$FULL_PATH_VARIABLE =~ /\\/path\\/variable\\/value$/' | true '$PRESENT_VARIABLE != "my variable"' | false '"my variable" != $PRESENT_VARIABLE' | false '$PRESENT_VARIABLE != null' | true '$EMPTY_VARIABLE != null' | true '"" != $EMPTY_VARIABLE' | false '$UNDEFINED_VARIABLE != null' | false 'null != $UNDEFINED_VARIABLE' | false "$PRESENT_VARIABLE !~ /var.*e$/" | false "$PRESENT_VARIABLE !~ /^var.*/" | true '$PRESENT_VARIABLE !~ /^v\ar.*/' | true '$PRESENT_VARIABLE !~ /^v\/ar.*/' | true "$EMPTY_VARIABLE !~ /var.*/" | true "$UNDEFINED_VARIABLE !~ /var.*/" | true "$PRESENT_VARIABLE !~ /VAR.*/i" | false '$PRESENT_VARIABLE && "string"' | 'string' '$PRESENT_VARIABLE && $PRESENT_VARIABLE' | 'my variable' '$PRESENT_VARIABLE && $EMPTY_VARIABLE' | '' '$PRESENT_VARIABLE && null' | nil '"string" && $PRESENT_VARIABLE' | 'my variable' '$EMPTY_VARIABLE && $PRESENT_VARIABLE' | 'my variable' 'null && $PRESENT_VARIABLE' | nil '$EMPTY_VARIABLE && "string"' | 'string' '$EMPTY_VARIABLE && $EMPTY_VARIABLE' | '' '"string" && $EMPTY_VARIABLE' | '' '"string" && null' | nil 'null && "string"' | nil '"string" && "string"' | 'string' 'null && null' | nil '$PRESENT_VARIABLE =~ /my var/ && $EMPTY_VARIABLE =~ /nope/' | false '$EMPTY_VARIABLE == "" && $PRESENT_VARIABLE' | 'my variable' '$EMPTY_VARIABLE == "" && $PRESENT_VARIABLE != "nope"' | true '$PRESENT_VARIABLE && $EMPTY_VARIABLE' | '' '$PRESENT_VARIABLE && $UNDEFINED_VARIABLE' | nil '$UNDEFINED_VARIABLE && $EMPTY_VARIABLE' | nil '$UNDEFINED_VARIABLE && $PRESENT_VARIABLE' | nil '$FULL_PATH_VARIABLE =~ /^\/a\/full\/path\/variable\/value$/ && $PATH_VARIABLE =~ /path\/variable/' | true '$FULL_PATH_VARIABLE =~ /^\/a\/bad\/path\/variable\/value$/ && $PATH_VARIABLE =~ /path\/variable/' | false '$FULL_PATH_VARIABLE =~ /^\/a\/full\/path\/variable\/value$/ && $PATH_VARIABLE =~ /bad\/path\/variable/' | false '$FULL_PATH_VARIABLE =~ /^\/a\/bad\/path\/variable\/value$/ && $PATH_VARIABLE =~ /bad\/path\/variable/' | false '$FULL_PATH_VARIABLE =~ /^\/a\/full\/path\/variable\/value$/ || $PATH_VARIABLE =~ /path\/variable/' | true '$FULL_PATH_VARIABLE =~ /^\/a\/bad\/path\/variable\/value$/ || $PATH_VARIABLE =~ /path\/variable/' | true '$FULL_PATH_VARIABLE =~ /^\/a\/full\/path\/variable\/value$/ || $PATH_VARIABLE =~ /bad\/path\/variable/' | true '$FULL_PATH_VARIABLE =~ /^\/a\/bad\/path\/variable\/value$/ || $PATH_VARIABLE =~ /bad\/path\/variable/' | false '$PRESENT_VARIABLE =~ /my var/ || $EMPTY_VARIABLE =~ /nope/' | true '$EMPTY_VARIABLE == "" || $PRESENT_VARIABLE' | true '$PRESENT_VARIABLE != "nope" || $EMPTY_VARIABLE == ""' | true '$PRESENT_VARIABLE && null || $EMPTY_VARIABLE == ""' | true '$PRESENT_VARIABLE || $UNDEFINED_VARIABLE' | 'my variable' '$UNDEFINED_VARIABLE || $PRESENT_VARIABLE' | 'my variable' '$UNDEFINED_VARIABLE == null || $PRESENT_VARIABLE' | true '$PRESENT_VARIABLE || $UNDEFINED_VARIABLE == null' | 'my variable' '($PRESENT_VARIABLE)' | 'my variable' '(($PRESENT_VARIABLE))' | 'my variable' '(($PRESENT_VARIABLE && null) || $EMPTY_VARIABLE == "")' | true '($PRESENT_VARIABLE) && (null || $EMPTY_VARIABLE == "")' | true '("string" || "test") == "string"' | true '(null || ("test" == "string"))' | false '("string" == ("test" && "string"))' | true '("string" == ("test" || "string"))' | false '("string" == "test" || "string")' | "string" '("string" == ("string" || (("1" == "1") && ("2" == "3"))))' | true end with_them do let(:text) { expression } it "evaluates to `#{params[:value].inspect}`" do expect(subject.evaluate).to eq(value) end end end describe '#truthful?' do using RSpec::Parameterized::TableSyntax where(:expression, :value) do '$PRESENT_VARIABLE == "my variable"' | true "$PRESENT_VARIABLE == 'no match'" | false '$UNDEFINED_VARIABLE == null' | true '$PRESENT_VARIABLE' | true '$UNDEFINED_VARIABLE' | false '$EMPTY_VARIABLE' | false '$INVALID = 1' | false "$PRESENT_VARIABLE =~ /var.*/" | true "$UNDEFINED_VARIABLE =~ /var.*/" | false "$PRESENT_VARIABLE !~ /var.*/" | false "$UNDEFINED_VARIABLE !~ /var.*/" | true end with_them do let(:text) { expression } it "returns `#{params[:value].inspect}`" do expect(subject.truthful?).to eq value end end context 'when evaluating expression raises an error' do let(:text) { '$PRESENT_VARIABLE' } it 'returns false' do allow(subject).to receive(:evaluate) .and_raise(described_class::StatementError) expect(subject.truthful?).to be_falsey end end end end
Mid
[ 0.541304347826086, 31.125, 26.375 ]
The entity Darkholy Darkholy is an evenly growing precise filling of a part of existence and all of the things which is included in it. Thereby it won't need anything to learn and developeDarkholy is also a fusion of the entity of pain and the entity of joy Last edited by LQ (2013-01-17 20:11:50) I see clearly now, the universe have the black dots, Thus I am on my way of inventing this remedy...
Low
[ 0.45991561181434604, 27.25, 32 ]
Q: understand java metric library units in jvisualVM Basically i am trying to understand the OneMinuteRate, RateUnit of the metric java library So i have hit the server login method with 51 request using jmeter and now i am trying to make sense of the data. 1 The COUNT gives the total number of times the method has been called. But what is the oneMinRate and rateUnit ? and what is event here ? EDIT Please also throw light on other attributes as well please A: The metrics library has some pretty good documentation on this. From your output, it appears that you are using a Timer in your code. From the docs, you can see that a Timer "is basically a histogram of the duration of a type of event and a meter of the rate of its occurrence." Note that those docs provide links for both the Meter and the histogram. From those docs we see that, "Meters measure the rate of the events in a few different ways. The mean rate is the average rate of events. It’s generally useful for trivia, but as it represents the total rate for your application’s entire lifetime (e.g., the total number of requests handled, divided by the number of seconds the process has been running), it doesn’t offer a sense of recency. Luckily, meters also record three different exponentially-weighted moving average rates: the 1-, 5-, and 15-minute moving averages." and, "Histogram metrics allow you to measure not just easy things like the min, mean, max, and standard deviation of values, but also quantiles like the median or 95th percentile." So, putting that all together and looking at what you've pasted we know: Your login code has been hit a total of 78 times In the last minute, 5 minutes, and 15 minutes that code has been hit 0 times/second (we know it's events/second because of the RateUnit) The median time between the timer being started and stopped (you'll have to look at the code to see where the timer gets stopped to see what's actually being measured) is 0.286543 milliseconds (we know the unit is milliseconds because of DurationUnit) The 99th percentile time (e.g. 99% of all calls took less time that this) was 10.449777 milliseconds etc. The only thing that's tricky and not terribly well defined in the above is bullet #2 about the rates. As stated in the docs, these are exponentially weighted moving averages so the 1 minute rate, for example, includes some information about the rate more than 1 minute ago. For the 1-minute rate, the weights in the average have been set so that what matters most is the data in the past minute. That's not terribly clear and the docs don't clarify. For the most part you can just think of these as the rate, in events/second, in the last 1 minute, 5 minutes, and 15 minutes. But if you really need to know the precise definition, you can find the weights that are being used in the source code.
Mid
[ 0.620087336244541, 35.5, 21.75 ]
life, love,art. heart failure and assorted ramblings Don't Talk Like That... I write to find out what my heart thinks....I write to expose and work through the ugly parts of this disease, open heart surgery that was less than successful and more recently an S-ICD (internal cardiac defibrillator) implanted in my chest that will hopefully restore my heart beat in case of sudden cardiac death. I am here to celebrate my life, to uncover my fears, to hold on to love, to learn how to live a full life with heart failure, to honor my creativity, and to explore all of my emotions out loud ...before anyone can say "Don't talk like that!" . Music is loading really slow lately...sorry... Wednesday, December 31, 2014 Don't We All? I have ugly questions, that they will not answer, don't we all? If more women die of heart failure than all cancers combined, how come we do not have more press and fundraising, do you have any idea how expensive this is? Where are they? Why do they tell you heart failure is not the death sentence it was years ago…..bull shit, it is exactly the same death sentence, they have just figured out how to make it last longer and collect more money. Does the medical industry ever understand what we go through, how sad, desperate, and frightened we are? Will they ever acknowledge me as a person with opinions, feelings, choices about my own life? I just want to live spectacularly and die well, I want my life to have meaning. Don't we all? The beach & flying my kite! i want to see legal gay marriage everywhere ROAD TRIP!!!Dave Matthews Band Tampa 2014 Take art classes-practice my craft develop more creative skills My very first self-portrait...does not look much like me, but it has all of the right words. Have a Sill String Fight! Get a tattoo tattooed my foot so I never never forget how strong I have to be! My friend thinks I should have "scared shitless" tattooed on the other foot! i want to write a book it is not a big book, but it is a published book! http://www.blurb.com/b/4868136-life-love-art-heart-failure-assorted-ramblings I FINALLY graduated from college with BA in Arts Administration! YAY......ME!!! December 2013 Swim in a Fountain! City of Casselberry... thank god, the camera battery died! Paint EVERYWHERE! Painted a baby grand piano with great art friends The Mondrian Piano! with embedded QR codes! Dave Matthews Band concert with the boys! and Grey Street was the encore...Woo-Hoo! Bucket List #7 DMB with both of my boys! Bucket List #36-C....... AND WE HAVE TICKETS 7-17-2013 we are going! Do nude nite And I have....2013-2014 & 2015 And I have....I have. It just gets better and more fun every year! Nude Nite 2016 Nude Nite 2015 with the "Girls" Both pieces sold...woo-hoo! So...I am not allowed to post these videos on Youtube...so you can access them directly on my One Drive...it will take you off of this site to watch....sorry Nude Nite 2015 Pirates and Picasso woo-hoo...it was awesome. click image for video have a dirty little secret! ooo-la-la! ......and that is all I am going to say about it! Bucket List...STILL WORKING ON IT i want to go skinny dipping at night i want to learn how to blow glass Doodle on my shoes.... The Cinderella Scenario i want to let balloon secrets fly i want to drive a convertable on a winding mountain road i want to NOT care what others think i wanto to kiss with pop-rocks! Dance on the table I want to ride in the front of the airplaine or a limosine just once! i want to change someone's life they say I do....but I need to change more! Paint Balloon darting i want to go to an old fashion drive-in movie! i want to see and catch fire flies! i want to die with NO regrets Just one amazing.... See this moon rise and my "Core Desire Feelings" for every situation...there is a suitable line from a song! Headphones on Heart of a Hero! Got myself a new "happy I am dancing my butt off song"! Woo-hoo! After 23 years...My last Artist's Way Group Summer 2017 Celebrate! 2016 Summer Artist Way Group Opening Reception 2016...Woo-Hoo! Beast Feast 2016 Me, Terry, Mary & Ernie....High school buds! Flagler Beach 2016 Yep....That's ME, too! Purple polka dots! Woo-hoo Dale Fox photography Checking it out! First Look after Open Heart Surgery 8/2015 A Different Voice..This is not about end of life, it is about LIFE! My Guys! at Darren's shipboard Wedding HOLY CRAP...I am one of the top 20 art mentors in the country! Professional Artist Magazine Such an incredible honor! Thank you! A Woman's Heart! A Finger, Two Dots, Then Me I finally did it...and with honors, too! Did not go to my college graduation, BA in Arts Administration, UMASS earlier this year, but my honor society chords arrived in the mail today....what a great surprise! They are not quite as impressive without the cap and gown, but very nice to have them anyway! Thank you UMASS! Sometimes.....I can pretend to be a responsible adult....but it rarely lasts very long! Fine Artist Creativity Facilitator Opening Exhibition Toast Yes that is champagne straight out of the bottle....ooops! caught me! Thank you 2015... Thank you 2014! My Dark Angel My sculpture with an ancient Sufi Poet If I were queen, I would.... wear my "kicking this shit happy heart crown" I made...all of the time! Flagler Beach Summer 2014 2015 AW Install 2014 Artist's Way Celebration of Creativity Hokey Pokey Newbie Initiation Artists Way 2013 "HOKEY POKEY" & "DECONSTRUCTION" Every new members of the Artist's Way Group dances the Hokey Pokey before their exhibit! Welcome to all the new phenomenal creaties I have had the honor of spending my summer with! 2014 Begins with "The Desire Map" Book Group What a great way to begin the year! 2013 a Phenomenal Year! 2013 Artist's Way Exhibition Leesburg Artist's Way- Umbrellas Orlando Sentinel Leesburg Artist Way-Umbrellas Beast Feast Leesburg Center for the Arts Fundraiser with my handsome Dad. Art & Music in the Park Pairings Leesburg Art Center Orlando Sentinel Cheryl Jones Evans Sanford Herald The Art Shows! Pairings 2012- Leesburg Leesburg Daily Commercial Leesburg Art Festival Poster Artist The Boys are Married!! BOTH of them! Jason & Steph Darren & Jill Search This Blog The Official F#ck it Bucket List! The Official F#ck it Bucket #1 The TOP #1...was just upstaged by a new and ultimate #1 "Fuck it" Doctors...Tests....Doctors....Tests...Doctors offices....hospitals....make them all go away!!! PLEASE #2 Whiners...ooooops, I may have just blown this one! #3 Waiting Rooms...refer to #1 #4 Bible Thumpers #5 My physical body that will not do what I want it to do! Damn! #6 Civic Ignorance and politics...ELECTIONS.....grrrrrr! #7 Unavoidable NAP attacks #8 S-ICD implanted right under bra elastic....not gonna kill me but...it is like having that "hockey puck" smashed into my ribs all day it feels like I am wearing a rock in my bra... yeee-ouch! #9 Learning how to forgive myself...I am really bad at it! How do we learn how to metabolize hurt, grief and pain so that it becomes a good thing in our lives????
Mid
[ 0.549327354260089, 30.625, 25.125 ]
Continuous measurement of enzymatic kinetics in droplet flow for point-of-care monitoring. Droplet microfluidics is ideally suited to continuous biochemical analysis, requiring low sample volumes and offering high temporal resolution. Many biochemical assays are based on enzymatic reactions, the kinetics of which can be obtained by probing droplets at multiple points over time. Here we present a miniaturised multi-detector flow cell to analyse enzyme kinetics in droplets, with an example application of continuous glucose measurement. Reaction rates and Michaelis-Menten kinetics can be quantified for each individual droplet and unknown glucose concentrations can be accurately determined (errors <5%). Droplets can be probed continuously giving short sample-to-result time (∼30 s) measurement. In contrast to previous reports of multipoint droplet measurement (all of which used bulky microscope-based setups) the flow cell presented here has a small footprint and uses low-powered, low-cost components, making it ideally suited for use in field-deployable devices.
High
[ 0.6778042959427201, 35.5, 16.875 ]
<?xml version='1.0' encoding='utf-8' ?> <indexdata> <key name="Viewing" > <topic name="Viewing the Revision History of a Content" url="viewing_the_revision_history_of_a_content.htm" /> <topic name="Viewing the Top Visitors Report" url="viewing_the_top_visitors_report.htm" /> <topic name="Viewing the Access Denied Pages/Datasets Report" url="viewing_the_access_denied_pages_datasets_report.htm" /> <topic name="Viewing the VRM Delay Analysis Report" url="viewing_the_vrm_delay_analysis_report.htm" /> <topic name="Viewing Agency List" url="viewing_agency_list.htm" /> <topic name="Viewing the VRM Source Metrics Report" url="viewing_the_vrm_source_metrics_report.htm" /> <topic name="Viewing the VRM Status Metrics Report" url="viewing_the_vrm_status_metrics_report.htm" /> <topic name="Viewing a Reply for a Feedback" url="viewing_a_reply_for_a_feedback_pmo.htm" /> <topic name="Viewing List of Themes" url="viewing_list_of_themes.htm" /> <topic name="Viewing the Reply for a Feedback" url="viewing_the_reply_for_a_feedback.htm" /> <topic name="Viewing the Top Referrer Report" url="viewing_the_top_referrer_report.htm" /> <topic name="Viewing a Reply for a Feedback - POC" url="viewing_a_reply_for_a_feedback_poc.htm" /> <topic name="Viewing the VRM Category Metrics Report" url="viewing_the_vrm_category_metrics_report.htm" /> <topic name="Viewing Menu Items in a Menu" url="viewing_menu_items_in_a_menu.htm" /> <topic name="Viewing the List of Feedback Received" url="viewing_the_list_of_feedback_received.htm" /> <topic name="Viewing the Top Search Phrases Report" url="viewing_the_top_search_phrases_report.htm" /> <topic name="Viewing Content List" url="viewing_content_list.htm" /> <topic name="Viewing Captcha Examples" url="viewing_captcha_examples.htm" /> <topic name="Viewing Blocks Content" url="viewing_blocks_content.htm" /> <topic name="Viewing Revision History" url="viewing_revision_history.htm" /> <topic name="Viewing the Assignee Wise VRM Statistics Report" url="viewing_the_assignee_wise_vrm_statistics_report.htm" /> <topic name="Viewing Blocks List" url="viewing_blocks_list.htm" /> <topic name="Viewing Reports" url="viewing_reports.htm" /> <topic name="Viewing the Top Pages Report" url="viewing_the_top_pages_report.htm" /> <topic name="Viewing the VRM Action Metrics Report" url="viewing_the_vrm_action_metrics_report.htm" /> <topic name="Viewing List of Mini Panels" url="viewing_list_of_mini_panels.htm" /> <topic name="Viewing the Page Not Found Error Report" url="viewing_the_page_not_found_error_report.htm" /> <topic name="Viewing the Content List" url="viewing_the_content_list.htm" /> <key name="Access Denied Pages" > <topic name="Viewing the Access Denied Pages/Datasets Report" url="viewing_the_access_denied_pages_datasets_report.htm" /> </key> <key name="Agency List" > <topic name="Viewing Agency List" url="viewing_agency_list.htm" /> </key> <key name="Assignee Wise VRM Statistics Report" > <topic name="Viewing the Assignee Wise VRM Statistics Report" url="viewing_the_assignee_wise_vrm_statistics_report.htm" /> </key> <key name="Blocks Content" > <topic name="Viewing Blocks Content" url="viewing_blocks_content.htm" /> </key> <key name="Blocks List" > <topic name="Viewing Blocks List" url="viewing_blocks_list.htm" /> </key> <key name="Captcha Examples" > <topic name="Viewing Captcha Examples" url="viewing_captcha_examples.htm" /> </key> <key name="Content List" > <topic name="Viewing Content List" url="viewing_content_list.htm" /> <topic name="Viewing the Content List" url="viewing_the_content_list.htm" /> </key> <key name="List" > <topic name="Viewing List of Themes" url="viewing_list_of_themes.htm" /> <topic name="Viewing the List of Feedback Received" url="viewing_the_list_of_feedback_received.htm" /> <topic name="Viewing List of Mini Panels" url="viewing_list_of_mini_panels.htm" /> </key> <key name="Menu Items" > <topic name="Viewing Menu Items in a Menu" url="viewing_menu_items_in_a_menu.htm" /> </key> <key name="Page Not Found Error Report" > <topic name="Viewing the Page Not Found Error Report" url="viewing_the_page_not_found_error_report.htm" /> </key> <key name="Reply" > <topic name="Viewing a Reply for a Feedback" url="viewing_a_reply_for_a_feedback_pmo.htm" /> <topic name="Viewing the Reply for a Feedback" url="viewing_the_reply_for_a_feedback.htm" /> <topic name="Viewing a Reply for a Feedback - POC" url="viewing_a_reply_for_a_feedback_poc.htm" /> </key> <key name="Reports" > <topic name="Viewing Reports" url="viewing_reports.htm" /> </key> <key name="Revision History" > <topic name="Viewing the Revision History of a Content" url="viewing_the_revision_history_of_a_content.htm" /> <topic name="Viewing Revision History" url="viewing_revision_history.htm" /> </key> <key name="Top Pages Report" > <topic name="Viewing the Top Pages Report" url="viewing_the_top_pages_report.htm" /> </key> <key name="Top Referrer Report" > <topic name="Viewing the Top Referrer Report" url="viewing_the_top_referrer_report.htm" /> </key> <key name="Top Search Phrases Report" > <topic name="Viewing the Top Search Phrases Report" url="viewing_the_top_search_phrases_report.htm" /> </key> <key name="Top Visitors Report" > <topic name="Viewing the Top Visitors Report" url="viewing_the_top_visitors_report.htm" /> </key> <key name="VRM Action Metrics Report" > <topic name="Viewing the VRM Action Metrics Report" url="viewing_the_vrm_action_metrics_report.htm" /> </key> <key name="VRM Category Metrics Report" > <topic name="Viewing the VRM Category Metrics Report" url="viewing_the_vrm_category_metrics_report.htm" /> </key> <key name="VRM Delay Analysis Report" > <topic name="Viewing the VRM Delay Analysis Report" url="viewing_the_vrm_delay_analysis_report.htm" /> </key> <key name="VRM Source Metrics Report" > <topic name="Viewing the VRM Source Metrics Report" url="viewing_the_vrm_source_metrics_report.htm" /> </key> <key name="VRM Status Metrics Report" > <topic name="Viewing the VRM Status Metrics Report" url="viewing_the_vrm_status_metrics_report.htm" /> </key> </key> <key name="Voting API" > <topic name="Configuring the Voting API" url="configuring_the_voting_api.htm" /> <key name="Configuring" > <topic name="Configuring the Voting API" url="configuring_the_voting_api.htm" /> </key> </key> <key name="VRM Action Metrics Report" > <topic name="Viewing the VRM Action Metrics Report" url="viewing_the_vrm_action_metrics_report.htm" /> <key name="Viewing" > <topic name="Viewing the VRM Action Metrics Report" url="viewing_the_vrm_action_metrics_report.htm" /> </key> </key> <key name="VRM Category Metrics Report" > <topic name="Viewing the VRM Category Metrics Report" url="viewing_the_vrm_category_metrics_report.htm" /> <key name="Viewing" > <topic name="Viewing the VRM Category Metrics Report" url="viewing_the_vrm_category_metrics_report.htm" /> </key> </key> <key name="VRM Delay Analysis Report" > <topic name="Viewing the VRM Delay Analysis Report" url="viewing_the_vrm_delay_analysis_report.htm" /> <key name="Viewing" > <topic name="Viewing the VRM Delay Analysis Report" url="viewing_the_vrm_delay_analysis_report.htm" /> </key> </key> <key name="VRM Reports" > <topic name="VRM Reports" url="vrm_reports.htm" /> </key> <key name="VRM Source Metrics Report" > <topic name="Viewing the VRM Source Metrics Report" url="viewing_the_vrm_source_metrics_report.htm" /> <key name="Viewing" > <topic name="Viewing the VRM Source Metrics Report" url="viewing_the_vrm_source_metrics_report.htm" /> </key> </key> <key name="VRM Status Metrics Report" > <topic name="Viewing the VRM Status Metrics Report" url="viewing_the_vrm_status_metrics_report.htm" /> <key name="Viewing" > <topic name="Viewing the VRM Status Metrics Report" url="viewing_the_vrm_status_metrics_report.htm" /> </key> </key> <key name="Weightage" > <topic name="Resetting the Weightage for Search Fields" url="resetting_the_weightage_for_search_fields.htm" /> <topic name="Specifying Weightage to the Search Fields" url="specifying_weightage_to_the_search_fields.htm" /> <key name="Resetting" > <topic name="Resetting the Weightage for Search Fields" url="resetting_the_weightage_for_search_fields.htm" /> </key> <key name="Specifying" > <topic name="Specifying Weightage to the Search Fields" url="specifying_weightage_to_the_search_fields.htm" /> </key> </key> <key name="Workflow" > <topic name="Sending Content Back in Workflow" url="sending_content_back_in_workflow.htm" /> </key> </indexdata>
Mid
[ 0.650359712230215, 28.25, 15.1875 ]
CALGARY -- The Stampeders have signed Calgary-born running back Matt Walter, the club announced Monday. Walter recently completed his fifth and final season with the University of Calgary Dinos, helping the team reach the Mitchell Bowl. Walter, who was drafted by the Stamps in the fifth round (34th overall) in the 2011 CFL Canadian Draft, is the U of C's all-time leading rusher with 4,143 career yards. "I was impressed with Matt's skill and competitiveness last year at training camp and look forward to his continued improvement in his attempt to become a Calgary Stampeder,'' said Calgary head coach and general manager John Hufnagel in a statement. "Matt showed during his university career that he can be adominant running back.'' Walter played for Canada at the 2011 world football championship in Austria, scoring six touchdowns in three games as Canada won silver.
High
[ 0.677494199535962, 36.5, 17.375 ]
//Copyright (c) 2018, Oracle and/or its affiliates. All rights reserved. //Licensed under the Universal Permissive License (UPL) Version 1.0 as shown at http://oss.oracle.com/licenses/upl. package eval import ( "encoding/json" "io/ioutil" "os" "testing" "github.com/oracle/speedle/pkg/cfg" "github.com/oracle/speedle/pkg/store" _ "github.com/oracle/speedle/pkg/store/etcd" _ "github.com/oracle/speedle/pkg/store/file" "github.com/oracle/speedle/api/pms" log "github.com/sirupsen/logrus" ) func WriteToTempFile(content []byte) (string, error) { tmpfile, err := ioutil.TempFile("", "authz-evaluator-") defer tmpfile.Close() if err != nil { return "", err } if _, err := tmpfile.Write(content); err != nil { return "", err } return tmpfile.Name(), nil } var testPS pms.PolicyStoreManager var configFile string var conf *cfg.Config func testMain(m *testing.M) int { var mConfig *cfg.Config var configFile string log.Infof("STORE_TYPE = %s\n", os.Getenv("STORE_TYPE")) if os.Getenv("STORE_TYPE") == "etcd" { log.Info("Start etcd!") configFile = "../cfg/config_etcd.json" defer os.RemoveAll("./speedle.etcd") } else { log.Info("Start file!") configFile = "../cfg/config_file.json" defer os.Remove("./ps.json") } var err error mConfig, err = cfg.ReadConfig(configFile) if err != nil { log.Fatal("Fail to read store config") os.Exit(1) } //Since we don't need watch function in the unit tests of evaluator, so disable it. mConfig.EnableWatch = false testPS, err = store.NewStore(mConfig.StoreConfig.StoreType, mConfig.StoreConfig.StoreProps) if err != nil { log.Fatal("Fail to NewStore") os.Exit(1) } conf = mConfig ret := m.Run() return ret } func preparePolicyDataInStore(data []byte, t *testing.T) error { var ps pms.PolicyStore err := json.Unmarshal(data, &ps) if err != nil { t.Fatal("Fail to prepare data", err) return err } err = testPS.WritePolicyStore(&ps) if err != nil { t.Fatal("Fail to prepare data:", err) return err } return nil } //This method can help to print service, policy or rolepolicy instance. When debugger cannot work, you can use it to //print data to help investigation. func printObject(p interface{}) { b, e := json.MarshalIndent(p, "", " ") if e != nil { log.Warning("Failed to marshal policy!") } log.Info(string(b)) } func TestMain(m *testing.M) { os.Exit(testMain(m)) }
Low
[ 0.518375241779497, 33.5, 31.125 ]
Chancellor Fariña calls BSA "inspiring" in recent speech April 17, 2014 Chancellor Fariña and Dr. McKeon In a recent speech held on April 17th at Cooper Union, Chancellor Fariña again mentioned Broome Street Academy, stating we were an "inspiring" school. Read the full article below and find the original release at Capital. Chancellor Carmen Fariña gave her most comprehensive feedback yet on this year's controversial state English Language Arts tests during a speech at Cooper Union, addressing the "opt-out" movement and saying she has been in frequent contact with the state education department about ways to improve the test. Delivering a variation on her 100 days speech from Saturday at a St. John's University event on Wednesday evening, Fariña told the group of education students that the Department of Education has convened a meeting of principals to give recommendations to the state about the tests. She acknowledged that the tests has spurred protest from "some parents with the support of some principals" and said the concerns were focused mostly on the 3rd grade tests. "We are in constant dialogue with the state education department," she said, adding that "for parents to buy in they must be part of the process." Fariña glanced at index cards as she spoke, and only briefly read directly from prepared remarks, as she begins to strike a balance from her preferred (but risky) method of speaking without remarks and the monotony, which she clearly disdains, and of reading from a speech written by someone else. She reiterated her support for the Common Core curriculum standards and repeated her key pillars of respect for teachers and collaboration between schools.She also gave some insight into her activity in recent weeks, mentioning a meeting with business leader Kathryn Wylde and the Partnership for NYC to help expand career and technical schools, a legacy of the Bloomberg administration. Fariña also mentioned a recent visit to the Broome Street Academy, a charter school, which focuses on helping homeless students, which she called one of her most "inspiring" school visits. She spoke in detail about her experience as an educator, recalling times she had comforted a crying teacher or held 21 parent meetings in a single year. She also briefly mentioned that she has created a new internal D.O.E. office for guidance counselors as of the last several weeks, and that she is working on a "dream initiative" program to help get students from Title I schools into the city's specialized high schools. When asked by one student what the greatest accomplishments were of her tenure as chancellor, Fariña said she was most proud of raising morale among teachers. When asked about her challenges, she said, "Making sure there are no more snow days."
High
[ 0.6616161616161611, 32.75, 16.75 ]
Q: (background) push notification, and built on phonegap build for android/iphone Is there a push notification plugin/approach for phonegap (android/iphone) where: the application can still be built using phonegap build. the push notification can be received even when the application is not running (in the foreground) I am trying HARD not do manage separate Android (Java) and iPhone (Xcode) projects. If not, what would be the easiest way to implement this for BOTH android & iphone? Urban Airship looks very close to doing this (not sure if can be built via phonegap build though). A: Currently what you describe here is not yet available with phonegap build. You need to use cordova (=phonegap) plugins for that and currently only one plugin is supported by phonegap build (see Supported plugins ). If you want something quickly you'll have to do with your own custom builds. Have a look at this repository of plugins. You'll see two Push plugins: PushNotification and UAPushNotification they seem to be both using Urban Airship integration. To reply to your second question, it does look like the push notifications are only visible once your application starts, not when it's stopped.
Low
[ 0.512396694214876, 31, 29.5 ]
Q: I can't enable or disable a user service: Failed to execute operation: No such file or directory I want to debug/test a program in eclipse that uses a Redis server so I decided to turn the server into a user service to have the privilege of running it. What bothers me is that I can start or stop the service but not enable/disable it. The error I get is: Failed to execute operation: No such file or directory Original /usr/lib/systemd/system: [Unit] Description=Advanced key-value store After=network.target [Service] User=arkos ExecStart=/usr/bin/redis-server /etc/arkos/arkos-redis.conf ExecStop=/usr/bin/redis-cli shutdown [Install] WantedBy=multi-user.target Edited and moved to /usr/lib/systemd/user: [Unit] Description=Advanced key-value store [Service] ExecStart=/usr/bin/redis-server /etc/arkos/arkos-redis.conf ExecStop=/usr/bin/redis-cli shutdown [Install] WantedBy=default.target Systemctl status: �� arkos-redis.service - Advanced key-value store Loaded: loaded (/usr/lib/systemd/user/arkos-redis.service; disabled; vendor preset: enabled) Active: active (running) since Tue 2015-08-25 09:19:25 UTC; 1min 55s ago Process: 644 ExecStop=/usr/bin/redis-cli shutdown (code=exited, status=1/FAILURE) Main PID: 736 (redis-server) CGroup: /user.slice/user-1000.slice/[email protected]/arkos-redis.service ������736 /usr/bin/redis-server *:0 Aug 25 09:19:25 arkos-vagrant redis-server[736]: | `-._`-._ _.-'_.-' | Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-._ `-._`-.__.-'_.-' _.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-._ `-.__.-' _.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-._ _.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-.__.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.471 # Server started, Redis version 3.0.3 Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.472 # WARNING overcommit_memory is set to 0! Background save may fail under low memory condition. To fix this issue add 'vm.overcommit_memory = 1' to /etc/sysctl.conf and then reboot or run the command 'sysctl vm.overcommit_memory=1' for this to take effect. Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.472 # WARNING you have Transparent Huge Pages (THP) support enabled in your kernel. This will create latency and memory usage issues with Redis. To fix this issue run the command 'echo never > /sys/kernel/mm/transparent_hugepage/enabled' as root, and add it to your /etc/rc.local in order to retain the setting after a reboot. Redis must be restarted after THP is disabled. Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.472 # WARNING: The TCP backlog setting of 511 cannot be enforced because /proc/sys/net/core/somaxconn is set to the lower value of 128. Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.472 * The server is now ready to accept connections at /tmp/arkos-redis.sock Journalctl: Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.470 # You requested maxclients of 10000 requiring at least 10032 max file descrip Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.470 # Redis can't set maximum open files to 10032 because of OS error: Operation Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.470 # Current maximum open files is 4096. maxclients has been reduced to 4064 to Aug 25 09:19:25 arkos-vagrant redis-server[736]: _._ Aug 25 09:19:25 arkos-vagrant redis-server[736]: _.-``__ ''-._ Aug 25 09:19:25 arkos-vagrant redis-server[736]: _.-`` `. `_. ''-._ Redis 3.0.3 (00000000/0) 64 bit Aug 25 09:19:25 arkos-vagrant redis-server[736]: .-`` .-```. ```\/ _.,_ ''-._ Aug 25 09:19:25 arkos-vagrant redis-server[736]: ( ' , .-` | `, ) Running in standalone mode Aug 25 09:19:25 arkos-vagrant redis-server[736]: |`-._`-...-` __...-.``-._|'` _.-'| Port: 0 Aug 25 09:19:25 arkos-vagrant redis-server[736]: | `-._ `._ / _.-' | PID: 736 Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-._ `-._ `-./ _.-' _.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: |`-._`-._ `-.__.-' _.-'_.-'| Aug 25 09:19:25 arkos-vagrant redis-server[736]: | `-._`-._ _.-'_.-' | http://redis.io Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-._ `-._`-.__.-'_.-' _.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: |`-._`-._ `-.__.-' _.-'_.-'| Aug 25 09:19:25 arkos-vagrant redis-server[736]: | `-._`-._ _.-'_.-' | Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-._ `-._`-.__.-'_.-' _.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-._ `-.__.-' _.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-._ _.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: `-.__.-' Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.471 # Server started, Redis version 3.0.3 Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.472 # WARNING overcommit_memory is set to 0! Background save may fail under low m Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.472 # WARNING you have Transparent Huge Pages (THP) support enabled in your kerne Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.472 # WARNING: The TCP backlog setting of 511 cannot be enforced because /proc/sy Aug 25 09:19:25 arkos-vagrant redis-server[736]: 736:M 25 Aug 09:19:25.472 * The server is now ready to accept connections at /tmp/arkos-redis.sock A: Symlink issue? I had a similar error message when using symbolic links. Apparently systemd doesn't follow symbolic links, the solution is simply to copy or move the file. User service? I believe that you need to add --user to the command line for units in user/: sudo systemctl --user enable arkos-redis.service A: In my case, I was placing my systemd unit files directly under /etc/systemd/system/... folders. Moved them to /lib/systemd/system instead and that resolved the issue with systemctl enable/disable commands.
Low
[ 0.47215496368038706, 24.375, 27.25 ]
BACKGROUND: Considering widely administration of methylphenidate and also its immunosuppressive effects on different organs, importance of related microscopic studies is obvious. OBJECTIVES: Determining histological effects of methylphenidate on adrenal glands and lymphatic organs in mice. METHODS: A total number of 30 adult male Balb/C mice were provided, weighed and divided into one control and two experimental groups. The control group received water by gavages once a day, for 40 days. The experimental groups were orally administered MPH hydrochloride (2mg/kg and 10mg/kg body weight,) respectively. Animals were anesthetized and blood samples were collected through cardiac puncture for analysis of blood cells. Spleen, thymus, lymph nodes and adrenal glands were removed and processed for microscopic studies through hematoxylin and eosin staining. Spleen samples were processed for plasma cell count and staining (label antibody CD138*). The data were analyzed using analysis of variance (ANOVA) and p Methylphenidate hydrochloride is one of the most frequently prescribed pediatric drugs for the treatment of attention deficit hyperactivity disorder. Besides its well-known addictive properties, amphetamine (AMPH) was found to influence the immune functions as a potent immunosuppressor. AMPH and its derivatives (eg. 3, 4- metylendodioxymethamphetamine and fenfluramine) cause a decrease in leukocyte and lymphocyte numbers in the peripheral blood (Freire-Garabal et al., 1991; Connor, 2004; Pacific et al 2001). It has been suggested that AMPH can act either directly on peripheral cells or indirectly by affecting neuroendocrine pathway. Acute and chronic AMPH administration also could cause a marked stimulation of the hypothalamic– pituitary – adrenal axis and sympathetic nervous system resulting in the elevation of glucocorticoids (cortisol) and catecholamines (epinephrine and norepinephrine) levels (Wrona et al., 2005; Seiden et al., 1993; Ruginsk et al., 2011; Zuloaga et al., 2015). Glucocorticoids and catecholamine are known to have strong immunomodulating properties (McEwen et al., 1997; Friedman and Irwin 1997). It has been shown that in vitro effects of glucocorticoids are immunosuppressive, however, it is becoming increasingly evident that the in vivo effects of glucocorticoids are frequently different from in vitro treatment or treatment with synthetic glucocorticoids such as dexamethasone (Fleshner et al., 2001). Cytogenetic effects have been observed in peripheral lymphocytes in children treated with MPH for 3 months raising questions about the genetic toxicity of this compound. MPH has been found negative in most genetox studies performed; however, no in vitro chromosome aberration data in human lymphocytes has been reported. A chromosomal aberration study in cultured human peripheral lymphocytes has shown that d, 1-methylphenidate (MPH, Ritalin) in concentrations up to 10 Mm neither induced structural nor numerical chromosome abnormalities (Suter et al., 2006). AMPH injection has produced differential effects on leukocyte populations in the peripheral blood. It has also induced a marked lymphopenia together with an increase in LGL- NK cell number, (lymphocyte subset) as well as an increase in granulocyte and monocyte numbers. The total number of WBC remained unaffected. In the spleen, AMPH- induced changes were more uniform – all the leukocyte populations were decreased. Parallel to LGL number increase, NKCC was enhanced in the peripheral blood after injection of AMPH (Schedlowski et al., 1993; Gagnon et al., 1992; Witt et al., 2008). Proliferation of human peripheral blood mononuclear leukocytes (PBML) is measured in the presence or absence of amphetamines, and has revealed that the abuse of amphetamines, especially the designer drugs, may adversely affect the activity of immunoregulatory cells and might lead to a compromised immune system in amphetamine abuser (Gagnon et al., 1992). Opioids and opioid agents are known to have profound immune suppressive effects. Results of the functional assays have shown that acute morphine administration inhibits peripheral blood lymphocyte activity and causes a definite decline in the peripheral blood lymphocyte counts in the rat (Flores et al., 1995; Liang et al., 2016). The aim of the present study was to determine overall effect of methylphenidate on immune system. The assessments were based on histological and histochemical evaluation in the mice lymphoid organs. In order to investigate the indirect pathway related to the effects of methylphenidate on the adrenal cortex, its histological structure was investigated histometrically. Materials and Methods Animals and experimental design: Thirty male adult Balb/C mice, at three months of age and weighing 25-30 g, were randomly divided into 3 groups of 10 animals each. The animals were housed individually in the cages located in a pathogen free, temperature and humidity-controlled colony room which was maintained under a 12 hours day–night illuminating cycle with free access to food and water. Prior to the experiment, the animals were prepared for manipulations during a 1 week acclimatization in the Experimental Medical Research and Application Center of the Faculty of Medicine, Islamic Azad University. The animals in the first group served as controls and received water without drug. Experimental groups received 2mg/kg and 10mg/kg body weight MPH respectively, as gavages for 40 days (Fazelipour et al., 2014). WBC and leukocyte subsets: At the end of the administration period, blood samples from the hearts of the animals were taken into heparinized (100 IU heparin/ml blood) tubes. From each sample, four blood films were prepared, air dried and stained with Gimsa. Total WBC counts were determined using the hematology analyzer. The percentages of lymphocytes, granulocytes, and monocytes were determined by counting 200 WBC with a microscope Gimsa staining. The number of each leukocyte subset was calculated as WBC number × percentage of individual leukocyte subset. Histological and Histochemical studies: The animals were sacrificed by cervical dislocation and spleens, thymuses, adrenal glands and lymph nodes were removed. Each sample was placed in 10% buffered formalin. Following the fixation, the tissue samples were processed using routine histological techniques. The spleen samples were fixed in alcoholic formalin and processed for plasma cell staining (label antibody CD 138*) (Dako-Denmark) and then splenic plasma cell counting. For immunohistochemistry procedure, paraffin blocks were sectioned at 3 microns and after several hydration steps by H2O2 and alcohol, usage of antibody including secondary antibody as administered was implemented. In this method the dilution rate of 1:50 has been used. Splenic plasma cells in unit area (1.44x104µm2 tissue area) were determined by counting in 10 randomly selected subcapsular white pulp regions using an ocular square micrometer and the results were expressed as cell count/ unit area (pc/UA). Histometrical measurements on the spleens, adrenal glands and thymuses were done with the aid of an ocular linear micrometer. For this purpose, 10 tissue semi- thin sections, 5µm, were taken from each animal and the values were expressed as Mean ± SD. Hormone analysis: Blood samples were collected from the hearts of all animals on day 40 after treatment and then serum specimens collected from these samples were frozen at -20 °C. After collection of all specimens, serum levels of cortisol were measured using ELFA (Enzyme Linked Fluorescent Assay) technique (Fardavard Company-France). Statistical analysis: The results are presented as mean±SD. One way analysis of variance (ANOVA) followed by the post-hoc Tukey test were used for the statistical analysis and a value of p<0.05 was considered significant. Thymus: Lymphoid tissue of the thymus is organized as a dense cellular cortex with lesser cellular medulla. The thickness of the capsules was increased significantly (p<0.05), though for the medulla it was decreased significantly (p<0.05). Hassal corpuscles were rarely seen in the thymic medulla (Table 1) (Fig. 3). Mesenteric lymphatic nodes: Mesenteric lymphatic nodes of the control group had larger cortical areas which were occupied by lymphoid follicles, paracortical zones formed by lymphatic cords and medullary areas containing large lymphatic sinuses. Some of the sinuses were with lymphocytes (Fig. 4). The thickness of the capsules of lymph nodes was significantly increased in treated groups (p<0.05) (Table1). Peripheral Blood: Percentage of the peripheral blood lymphocytes was declined significantly (p<0.05). Significant (p<0.05)) increases were also observed in neutrophil cell ratio of the methylphenidate treatment group (Table 2). Adrenal glands: Adrenal glands of the control animals showed typical morphology with a larger cortical area and a centrally located medulla region. Overall thickness of adrenal cortex was increased in the experimental group compared to those of the controls. Statistical analysis showed that methylphenidate with different doses could increase thickness of the glomerulosa and fasciculate layers of the adrenal cortex, and decrease the reticularis layer. On the other hand, the thickness of capsule and also the medullary layer were increased significantly in the experimental groups (10 mg/kg) compared to those of the control group (p<0.05). Significant changes in serum cortisol were observed in the MPH treated animals, however, no significant histopathological changes were seen in control specimens (p<0.05) (Table 3). Table 1. The histometric characteristics of lymphoid organs in different groups. Group 1 mice received 2 mg/kg water. Groups 2 and 3 were treated with 2 mg/kg and 10 mg/kg MPH. respectively. Organs/parameters Group 1 Group 2 Group 3 Spleen PC/UA 23.08±9.90a 9.67±4.43b 1.23±0.38c MC/UA 0.42±0.27a 0.63±0.22a 2.46±1.30b Thymus TCT(µm) 115.49±47.19a 281.39 ±77.70b 317.98±105.019c TM (µm) 21969.83±86a 16342.41b 18479.58c Lymph node CD (C/UA) 129,6±23.56a 363.41±86.18b 267.18±59.621c The values from seven animals are expressed as mean ± SD. Different superscript letters in the same rows indicate a significant difference, p<0.05. Methylphenidate hydrochloride is one of the most frequently prescribed pediatric drugs for the treatment of attention deficit hyperactivity disorders. From the results of the study it could be deduced that amphetamines as methylphenidate, can influence the peripheral blood and make significant changes in the number of leukocytes compared to the control group. Our study indicated that the number of lymphocytes, involved in immune functions, showed a significant decrease and the number of neutrophils was increased significantly compared to the control group, whereas the total percentage of leukocytes remained unaffected. In a study by Gagnon et al. the effect of amphetamines on proliferation of leukocytes has been shown. Also different studies show a variety of effects on leukocytes of the peripheral blood and similar results have been reported by other investigators. Many studies have revealed that amphetamines can affect proliferation of immune cells in other organs (Gagnon et al.,1992; Bredholt et al., 2013). It has been stated that methylphenidate structurally resembles cocaine, a narcotic, and it is expected to have a preventive effect on immune system (Rofael et al., 2003). Immunosuppression could be done by a variety of mechanisms which, besides the decrease of leukocytes, it could be through increasing catecholamines (Schedlowski et al.,1996). Our study indicated that methylphenidate could influence the adrenal gland inducing thickness of zona glumerolosa and zona fasciculata in the adrenal cortex. The significant increase in the thickness of zona fasciculata indicated increase in the corticosteroids and subsequently significant increase in the cortisol, and subsequent suppression of the immune system. Moreover, the thickness of medulla of the adrenal gland, responsible for synthesis and secretion of catecholamines showed a significant increase compared to the control group, which it was in turn another factor in suppression of the immune system in the present study. The serum cortisol level in experimental groups showed a significant increase which could be an indicator of suppression in the immune system due to its inhibitory effect on proliferation of lymphocytes. This was in agreement with another study that showed effects of cocaine, with a similar structure to methylphenidate, on the adrenal gland and increase in the cortisol that induced suppression of the immune system (Rofael et al., 2003). In the present study a significant decrease in the number of splenic plasma cells with a pattern of greater decrease in higher doses was seen. On the other hand, a significant increase in the number of megakaryocytes in spleen was observed. This increase showed that hematopoiesis could take place outside the bone marrow. From the results of this study, preventive effect of methylphenidate on immunity could be deduced by decrease in plasma cells and also decrease in lymphocytes in different lymphatic organs. Also, a study on the effect of morphine on the adrenal gland with similar findings to methylphenidate has been reported (Salback et al., 2001; Vinson and Brennan., 2013). Investigations have shown that the effects of different drugs, opioids and methylphenidate on the immune system could be induced through three mechanisms including: Direct effect of these substances on immune cells in peripheral blood, effect on hypothalamic pituitary adrenal axis which indirectly induces cortisol increase and 3 activating sympathetic nervous system which causes circulating levels of epinephrine from the adrenal medulla as well as norepinephrine from sympathetic nerve terminal leading to increase in the catecholamines (Wang et al., 2002). In other studies increase in the catecholamines due to usage of amphetamines has been observed. Methylphenidate could also affect immune cells in different organs by preventing their proliferation.16 Therefore, the present study indicated that methylphenidate influenced the spleen by significant decrease in plasma cells. Considering the spleen as the largest lymphatic organ and the only responsible organ for blood filtration, and so providing defense against blood antigens and microorganisms, reduction of plasma cells might represent an immunosuppression. On the other hand, significant increase in megakaryocytes in spleen, indicating a pathologic condition, implies inhibitory role of methylphenidate on immune system function. Also, increased catecholamine release has been associated with suppression of natural killer cell function and altered lymphocyte function (Schedlowski et al., 1993). An analysis of the effects of amphetamine on proenkephalin-derived peptides in brain areas and immune cells in rats showed that acute as well as a repeated amphetamine treatment decreased the concanavalin-A-induced lymphocyte proliferation, concomitantly with an increase of free metenkephalin in nucleus accumbens, prefrontal cortex, spleen, thymus and splenic macrophages (Assis et al., 2006). The possibility that the adrenergic system may stimulate the NK cells activity in the peripheral blood was confirmed by the studies of Schedlowski et al. (1993) and Benshop et al. (1997). Moreover, treatment with β-adrenoreceptor antagonist inhibits an increase in both circulating NK cells and NKCC elicited by stress or cathecolamine infusion (Glac et al., 2006). The present study showed that lymphoid tissue of the cortex in thymus was denser with fewer cellular portions than medulla. Besides, the thickness was increased in capsule while decreased in medullar region in the experimental groups due to effect of amphetamines on thymus. In lymph nodes also the thickness of capsule in experimental groups compared to the control group due to the effect of amphetamines was observed. In conclusion, it could be suggested that caution should be considered in prescription of amphetamines due to their effects on proliferation of immune cells in various organs, peripheral blood and also on the most defensive organ against blood pathogens i.e. spleen and possibly substitution with other medications.
High
[ 0.682719546742209, 30.125, 14 ]
Aged 90! We visited a female patient aged 90 years old. She is HIV-positive and she was taking care of her child that was HIV positive and she was not using gloves while she was bathing her. Now she hired a lady who is taking care of her, because she is living alone. On our arrival she was in bad condition and she was complaining about body pains, loss of appetite and swollen feet. She can’t walk. As care workers, we advised her to visit to the nearest clinic, but she refused. She told us that they would kill her.
Low
[ 0.47033898305084704, 27.75, 31.25 ]
Relationship between adenoid size and severity of obstructive sleep apnea in preschool children. To investigate the contributions of adenoid and tonsil sizes to obstructive sleep apnea syndrome (OSAS) in normal-weight children in two age categories: preschool and schoolchildren. Fifty-eight normal-weight (body mass index z-score<2) symptomatic children with OSAS (apnea-hypopnea index ≥ 2) were evaluated. The patients were divided into two age categories: preschool (age<6; n=33) and schoolchildren (age ≥ 6; n=25). Polysomnographic findings and adenoid and tonsil sizes were compared. The relative contributions of body mass index and adenoid and tonsil sizes were also investigated with a regression analysis. Adenoid grade and apnea index correlated significantly in preschool children (r=0.45, p<0.01). On regression analysis, adenoid grade was a significant predictor of the apnea index in preschool children. The influence of adenoid hypertrophy decreased from preschool to schoolchildren. Tonsil size had little influence on the apnea index in either group. Adenoid hypertrophy was a major contributor to OSAS in normal-weight preschool children. The upper airway morphology of younger children with OSAS differed from that of older children with OSAS.
High
[ 0.6650485436893201, 34.25, 17.25 ]
In the past two decades, there has been a great interest in the use of what are called nootropics, or “smart drugs”, to give their users an edge in the accomplishment of tasks requiring enhanced cognitive ability and a strong focus on the task at hand. No doubt you, too would like to augment your intelligence and ability to stay alert for an extended period of time! In the fast-paced, demanding world in which we live today, sometimes it just does not seem possible to keep up with the tasks expected of us every single day. For as long as people have had these demands, we have tried various supplements to stimulate ourselves to remain wakeful and alert to better accomplish those tasks. Caffeine is the classic example, used for centuries in coffee, tea, and other natural sources to provide an energy boost. Drugs such as cocaine have also been around for a long time, and in the twentieth century amphetamines became extremely popular to help people stay awake and alert, but also carried with them many detrimental side-effects. In the twenty-first century, Modafinil has become the drug of choice to maintain a state of wakefulness and alertness, without the side-effects that gave amphetamines a bad name. It works in a dramatically different way from those earlier drugs, as it produces monoamines which stimulate the hypothalamus to produce more of the hormones which control your sleep patterns. In other words, it’s a kinder, gentler way to keep awake and focused! While it was first developed to treat those suffering from narcolepsy, research has shown that it has many benefits for the treatment of diseases such as fibromyalgia, chronic fatigue syndrome, myotonic dystrophy, multiple sclerosis (MS), cerebral palsy, and Parkinson’s disease. It has even proven successful as a therapy for methamphetamine abuse and geriatric memory loss, and some people have used it successfully as part of a weight loss program, as it can help decrease caloric intake by as much as 38% a day. Most people know Modafinil as the drug of choice for students wanting to stay awake to write a paper or study for exams, or professionals who need to extend their working day well beyond the limits of their natural wakefulness and alertness, or members of the military whose tasks such as piloting a plane for long hours or overnight sentry duty cannot be safely accomplished without Modafinil’s help. Studies have shown that Modafinil, when used responsibly, is safe, effective, and has a beneficial impact on cognitive abilities. It is a huge step forward in the development of smart drugs, and is being used successfully by people all around the world. Is it the right nootropic for you? While today Modafinil is primarily known as a study aid and cognitive enhancer, it was originally developed to target a specific sleep disorder, narcolepsy, which causes its sufferers to drop off to sleep at any time, and when awake to be excessively drowsy. In the late 1970’s in France, neurophysiology professor Michel Jouvet at Lafon Laboratories developed a drug called Adrafinal to help those people stay awake during daytime. Lafon Laboratories later developed a different version which was metabolised more easily and was less likely to cause liver damage, called Modafinil. Before then, amphetamines had been the drug of choice for such conditions, and also for people who, while not suffering from narcolepsy or associated sleep disorders such as obstructive sleep apnoea or shift work sleep disorder, wanted or needed to stay awake, including students, long-distance drivers, and air force pilots. However, amphetamines have some serious side-effects that made it less desirable than Modafinil. They are extremely addictive, can cause anxiety, and interfere with falling asleep at night. Modafinil has none of those side-effects. It was approved as a treatment for narcolepsy in France in 1994, and in 1998 in the United States, with the FDA approving its use for sleep apnoea and shift work sleep disorder in 2003. The American pharmaceutical company Cephalon leased the rights to Modafinil from Lafon in 1993. When the original patent for Modafinil expired in 2007, Cephalon got it extended, but from 2012 on, it has been legal to produce generic versions of Modafinil. Military Use: Even before it was approved for use in France, the French Air Force was experimenting with its effectiveness in the Gulf War in 1991. By the time of the United States invasion of Iraq in 2003, it had become the drug of choice for fighter pilots. Many studies have concluded that it is an effective way to ensure wakefulness in military personnel. It is routinely given to fighter pilots, paratroopers, and naval personnel in the armed forces of countries including the United States, France, United Kingdom, India, and Canada. It is seen as safer than amphetamines, which were considered a contributing factor in the friendly-fire death incident in Afghanistan in 2004, when American fighter pilots who had taken amphetamines dropped a bomb on Canadian troops, killing four of them. Sports: A Canadian study found that Modafinil could increase endurance by 30% in athletes, and it became popular to use in the early 2000’s to improve performance. Athletes such as the American baseball player Barry Bonds and several American track and field champions, including 2003 100-metres world champion Kelli White were found to have used them. Modafinil was banned for use by the World Anti-Doping Agency as a performance-enhancing drug just before the 2004 Summer Olympics, and remains on the banned list today. However, many non-competitive athletes continue to use them to make it possible to extend training sessions beyond what would otherwise be possible: maybe they can help you! Recent scientific studies: In a 2015 study, Dr Ruairidh Battleday of the University of Oxford, and Dr Anna-Katharine Brem of Harvard Medical School, after reviewing twenty-four studies conducted from 1990–2014, concluded that the evidence clearly shows that Modafinil is a cognitive enhancer with negligible side-effects, and that it can be truly called the first real “smart drug”. In fact, they found that the benefits of using Modafinil increased along with the length and complexity of the tasks being performed. Provigil was the original brand of Modafinil released to the public, produced by Cephalon. It is available in Belgium, Ireland, Italy, South Korea, United Kingdom, United States, South Africa, Israel, and Finland. Modalert is manufactured by Sun Pharma in Mumbai, India, and is mostly sold to customers in the United Kingdom, United States, and Canada. It has a film coating for extended release over a longer period. Provake is the generic brand of Modafinil most commonly sold in India. It is manufactured by Ranbaxy Laboratories in Gurgaon, Haryana, India, a part of Solus. It was the first generic version of Modafinil. Modvigil is manufactured by Hab Pharma in India. It is sold in Australia and New Zealand. Modapro is another Indian brand of Modafinil, made by Cipla in Mumbai, India. Modiodal is manufactured by Hilltop Chemist in the United States. It is sold in France, Denmark, Greece, Cyprus, Mexico, Netherlands, Portugal, Sweden, Norway, Turkey, and Japan. Alertec is manufactured by Shire Pharma Canada, and is sold in Canada and Ecuador. Adrafinil, the precursor to Modafinil, is still available, and without a prescription in many countries. In larger doses, it can have the same effect as Modafinil, but the body has to first convert the Adrafinil to the Modafinil form. However, that conversion can cause damage to the liver, and so is not recommended for frequent use. When Modafinil is prescribed for narcolepsy, the dosage is 200-400 mg per day, divided into two dosages, taken first thing in the morning and then at noon. The dosage usually starts at 200 mg and is gradually increased to 400 mg until the physician is satisfied that the patient is reaping the full benefits of the medication. For obstructive sleep apnoea, the standard dosage is 200 mg a day, taken in the morning. Shift work sleep disorder sufferers are instructed to take their 200 mg an hour before they start their work shift. Using Modafinil as a brain enhancer When Modafinil is taken to enhance cognitive ability, it is recommended that you start with 100 mg in the morning, and then a further 100 mg, if necessary, at noon. Modafinil remains in the system for 15 hours, leaving you ready to sleep normally when your bedtime comes around. However, different people react differently to the drug, depending on weight and body chemistry, and the tasks facing them on any given day. Whereas a woman of 140 pounds juggling the normal stresses of working and caring for her family might find 50 mg at noon and an additional 25 mg at 5 pm sufficient for her needs, a student staying up all night writing an essay or studying for an exam might need more than 200 mg, spread out over 24 hours. Often 4 mg per kilogram of body weight is recommended as a daily dose. Studies indicate that anything more than 600 mg a day is not safe; a 1999 study found that dosages of 800 mg a day led to increased blood pressure and heart rate, and should be avoided. It is recommended to start with a low dose and work up until you achieve the desired results for you in alertness, increased cognitive function, and wakefulness. The practice of “stacking” or combining cognitive enhancement drugs and supplements to increase effectiveness is very popular, and there are many possible combinations suggested in chat rooms and forums on the Internet. Not all of them are a good idea. Caffeine has been used for centuries as a drug which helps alleviate sleepiness. Often taken as a cup of coffee or tea, it can also be ingested in capsule form. Caffeine and Modafinil can be an effective combination. Some evidence exists, in fact, that Modafinil helps clear caffeine more quickly from the body, since it stimulates the production of the liver enzyme CYP3A4, which metabolizes caffeine, along with other drugs. Many people suggest taking caffeine to augment the effects of Modafinil, but it needs to be used with caution. It is important to carefully monitor your intake of caffeine when using Modafinil. Too much caffeine in combination with Modafinil could lead to nervousness, insomnia, headaches, and dizziness. You should not exceed 100 mg from all sources in a day. Piperine or Bioprene is derived from the pepper vine plant, and can greatly increase the bioavailability of certain nutrients and drugs. This may include Modafinil. Some users have found success with combining 25 mg with a 200 mg dose of Modafinil. It would be best to start at a much lower rate of 5 or 10 mg and work your way up until you find a level that works for you. L-Theanine is derived from green tea, and can be used to temper the effects of Modafinil. It can ease the occasional feelings of anxiety or jitteriness that some people experience with Modafinil, and some swear by its ability to increase creative thought in conjunction with the focus obtained from Modafinil. 200 mg with the Modafinil, or taken later as needed, seems to be effective. The B-complex vitamins, especially B2, B6, and B12, can help maintain energy levels and promote mental acuity. They also contribute to dopamine production which augments the effect of the Modafinil. Besides, it’s always a good idea to take your vitamins! L-tyrosine is a non-essential amino acid which can have significant benefits when taken with Modafinil. It helps to regulate epinephrine and norepinephrine levels, which can lower stress levels, which can sometimes be elevated by Modafinil. Oxiracetam from the racetam group of nootropics, is often used as a study aid, as it boosts intelligence levels, memory, and energy levels. When combined with Modafinil, the two provide strong benefits. Another racetam that is often used is Aniracetam. It is important to include a choline supplement to keep up acetylcholine levels, as the racetams deplete this in the body, leading to severe headaches. There are many more combinations or stacks that are being experimented with to make the most out of the effects of Modafinil. It is important to approach any of these combinations with caution; start slow with low dosages until you know what impact the combinations will have on you. Binding agents also influence the efficacy of a drug. While they are non-medicinal ingredients, they are important to help deliver the drug effectively. While Cephalon uses lactose in its Provigil, most Indian manufacturers use cellulose, which fewer people are allergic to. Most users of Modafinil do not experience any negative side-effects, but some have been noted in a few users: anxiety loss of appetite back pain diarrhea headache elevated heart rate or palpitations nervousness difficulty sleeping stuffy or runny nose upset stomach and throwing up In some cases, simply reducing the dosage will eliminate the symptom. More serious symptoms of an adverse reaction include: agitation dizziness or fainting mood swings chronic sleepiness depression signs of liver problems (such as yellowing of the eyes or skin, abdominal pain, darkened urine) skin rash If you notice any of these symptoms, you should stop using Modafinil and consult with a doctor. The most serious reactions are: chest pain signs of severe allergic reaction, including the swelling of airways fever, blisters, and skin peeling off. This is called Steven-Johnson syndrome and is extremely serious. It can lead the Toxic Shock Syndrome. If you experience any of these symptoms, you should seek medical help immediately. Negative interaction with foods and other drugs Modafinil can interact negatively with some foods and other drugs, and so those should be avoided while using Modafinil. Grapefruit can have a serious effect on how Modafinil is removed by the system after ingestion. It can build up in the body and lead to serious problems. Anyone taking Modafinil should avoid eating grapefruit or drinking grapefruit juice. Alcohol should not be consumed while taking Modafinil, or in very strict moderation. It can have varying negative effects. Some users report that the combination intensifies the effect of the Modafinil, extending the period of wakefulness well into the desired sleep period. Others have experienced a much-decreased tolerance to alcohol, leading to much quicker inebriation. Most worrying is that some people have experienced black-out periods, where while they have been wide awake, walking and talking, they later have no memory of these periods of time. Overall, great care should be taken in the consumption of alcohol on days that you have taken Modafinil. Hormonal Birth Control Methods may not be as effective when taken in conjunction with Modafinil. Barrier methods such as diaphragms and condoms should be used as well for complete protection One of the reasons that you should always obtain medical advice before using Modafinil is so that a doctor and pharmacist can review the other medications you take so that they can determine whether it is safe for you to use Modafinil in conjunction with those medications. Who shouldn’t take it: Pre-existing health conditions Heart disease: There has been some evidence that people with a history of left ventricular hypertrophy, mitral valve prolapse or arrhythmia should avoid the use of Modafinil. Decreased Liver Function: Because Modafinil is metabolized in the liver, it could potentially cause some issues. While it puts less stress on the liver than Adrafinil, which has to be broken down in the liver to become Modafinil, it still could pose a risk for those suffering from liver disease, and should be avoided. Seizure disorder: Since Modafinil interacts with the central nervous system, it can cause seizures in those who have a pre-existing condition. If a patient who has suffered previously with seizures wishes to try Modafinil, it should be under close supervision, and use should be discontinued if there are any signs of Modafinil exacerbating the condition. Mental illness: Modafinil should not be used by someone with anxiety or agitation disorders. Pregnancy and Breast-feeding: No studies have been conducted regarding the safety of using Modafinil by pregnant or lactating mothers. Its use should be suspended for the duration of the pregnancy and breast-feeding. There are different laws on obtaining and using Modafinil, depending on where you live. In Mexico, for instance, no prescription is necessary. On the other hand, almost any use of Modafinil in Russia and China, even with a prescription, is illegal. In the United Kingdom and many other countries, it can be obtained legally with a prescription for conditions such as narcolepsy. It is not prescribed for cognitive enhancement use. In 2016, there have been some drastic changes to the British laws regarding Modafinil and other nootropic drugs. The new Psychoactive Substances Act has outlawed the production, supply, import, export and possibly even possession of any drug which “by stimulating or depressing the person’s central nervous system, [affects] the person’s mental functioning or emotional state.” However, since Modafinil is also classified as a “medicinal product”, it is still legal to import it. Under the new law it is still not legal to be using Modafinil without a prescription, and the penalties go as high as one year in prison for a first-time offense. So, it is a good idea to not carry it around on your person. This makes the use of a discreet and reliable source even more necessary in the United Kingdom today. When you have decided that you wish to use Modafinil for its cognitive enhancement properties, you should take the time to research your various options to obtain it as easily as possible, while at the same time ensuring that your retailer can guarantee that what you receive will be safe and effective. Not all brands are made alike. Without a prescription, you will have to look to mail-order sources from abroad to get your Modafinil. There are many retailers available, but you need to be confident that the source that you select will deliver to you the most reliable brand of Modafinil on the market in a discreet and timely manner. You need to be aware of which brand of Modafinil you are buying. If a retailer cannot confirm which factory has produced their stock, you should not purchase from that source. Modalert, manufactured by Sun Pharma in India, is the brand approved for use by NHS England, and has a UK office in London. Modafinil may be a safe drug, but it is still a drug, and you should take precautions to ensure that it is safe for you to use. It is therefore a good idea to have your medical history checked over by a medical doctor, and the order checked over by a pharmacist as well. A good supplier will do this for you. No doubt, if you are ordering a supply of Modafinil from abroad, you want it to be delivered as promptly as possible. Take this into account when you are selecting a supplier. The best suppliers will offer you a guaranteed delivery date, and also a refund or redelivery if your parcel gets stuck in customs. Price is an important consideration. You want a supplier who can offer you a price guarantee. This is an issue when your shipment goes through customs and you can suddenly be landed with an import duty bill. Finally, given the legal complexities surrounding the importation and use of Modafinil, it is of paramount importance to have a supplier who understands the laws regarding it. You want your shipment to be able to make it through Customs and be delivered to you quickly and discreetly. So, why order from brainsupplements.co.uk? Uses a reliable supplier, Sun Pharma, for Modalert, the same brand used by the NHS Guarantees your despatch within 24 hours of receiving an order Money back guarantee Free Delivery Ensures delivery in discreet packaging Would You Like To Know When We're Running A Promotional Offer? Signup now and stay notified of new products or offers. I will never give away, trade or sell your email address. You can unsubscribe at any time. Brain Supplements seeks to provide an informational channel for consumers by providing the benefits of 'smart' drug, as well as information about their proper usage, their synergy with other drugs and possible side effects. Brain Supplements also tracks the legal status of nootropic drugs in the United Kingdom and provides safe places for consumers to purchase online.
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<!DOCTYPE html> <html data-require="math graphie word-problems"> <head> <meta http-equiv="Content-Type" content="text/html; charset=UTF-8"/> <title>Finding the equation of a line</title> <script src="../khan-exercise.js"></script> <style type="text/css"> #answer_area input[type=text] { width: 30px; margin: 0px 0px 0px 5px; } </style> </head> <body> <div class="exercise"> <div class="vars"> <var id="SLOPE">randRange(-10, 10 )</var> <var id="YINTERCEPT">randRange(-10,10)</var> <var id="X1">randRange(-10,10)</var> <var id="Y1">SLOPE*X1+YINTERCEPT</var> <var id="X2">randRangeExclude(-10,10,[X1])</var> <var id="Y2">SLOPE*X2+YINTERCEPT</var> <var id="XL">randRange(1,4)</var> <var id="YL">SLOPE*XL+YINTERCEPT</var> <var id="XR">randRange(5,10)</var> <var id="YR">SLOPE*XR+YINTERCEPT</var> </div> <div class="problems"> <div id="unrestricted-domain" data-weight="2"> <div class="question"> <p> Consider a <em>linear function</em> <code>f(x)</code>. While you don't know the formula for <code>f(x)</code> you can nevertheless still evaluate it for given values of <code>x</code>. Find the slope-intercept form of <code>f</code>. </p> <p> <center><code>f(</code><input type='text' id='nameField' value = 'type x values here' size='3'/><code>)=</code> <span id='nameDisplay'>?</span></center> </p> <div class = "graphie"> (function(){ updateNameDisplay = function() { var input = parseFloat(this.value) $('#nameDisplay').text( SLOPE*input+YINTERCEPT ); }; var clearTextField = function(){ $('#nameField').val(""); }; $('#nameField').click(clearTextField); $('#nameField').keydown( updateNameDisplay ); $('#nameField').keyup( updateNameDisplay ); $('#nameField').keypress( updateNameDisplay ); })() </div> </div> <div class="solution" data-type="multiple"> <p> <code>f(x) = </code><span class="sol"><var>SLOPE</var></span> <code>\cdot x+</code> <span class="sol"><var>YINTERCEPT</var></span> </p> </div> <div class = "hints"> <p>The slope intercept form is <code>f(x) = slope \cdot x + y_{intercept}</code></p> <p>The <code>y</code>-intercept of a function is where the graph of <code>f</code> hits the <code>y</code>-axis, i.e. <code>y_{intercept} = f(0)</code></p> <p>so <code>y_{intercept}=<var>YINTERCEPT</var></code></p> <p>The slope of a line is the factor by which you multiply the change in abscissas to get the change in ordinates</p> <p><code>f(<var>X1</var>) = <var>Y1</var></code> and <code>f(<var>X2</var>) = <var>Y2</var></code></p> <p>so the change in abscissas from <code><var>X1</var></code> to <code><var>X2</var></code> is <code><var>X2-X1</var></code> and the corresponding change in ordinates is <code><var>Y2</var> - <var>Y1</var> = <var>Y2-Y1</var></code> </p> <p>Thus the slope of <code>f</code> is <code>\frac{<var>Y2-Y1</var>}{<var>X2-X1</var>} = <var>SLOPE</var></code></p> <p>Thus the equation of the line is <code>f(x)= <var>SLOPE</var> \cdot x + <var>YINTERCEPT</var></code></p> </div> </div> <div id="restricted-domain" data-weight="1"> <div class="question"> <p> Consider a <em>linear function</em> <code>f(x)</code>. While you don't know the formula for <code>f(x)</code> you can nevertheless still evaluate it for given values of <code>x</code>. The domain of this function has been restricted to the interval <code>[<var>XL</var>,<var>XR</var>]</code>. Find the slope-intercept form of <code>f</code>. </p> <p> <center><code>f(</code><input type='text' id='nameField' value = 'type x values here' size='3'/><code>)=</code> <span id='nameDisplay'>?</span></center> </p> <div class = "graphie"> (function(){ updateNameDisplay = function() { var input = parseFloat(this.value) if (input &gt;=XL &amp;&amp; input &lt;=XR){ $('#nameDisplay').text( SLOPE*input+YINTERCEPT ); } else{ $('#nameDisplay').text( "?" ); } }; var clearTextField = function(){ $('#nameField').val(""); }; $('#nameField').click(clearTextField); $('#nameField').keydown( updateNameDisplay ); $('#nameField').keyup( updateNameDisplay ); $('#nameField').keypress( updateNameDisplay ); })() </div> </div> <div class="solution" data-type="multiple"> <p> <code>f(x) = </code><span class="sol"><var>SLOPE</var></span> <code>\cdot x+</code> <span class="sol"><var>YINTERCEPT</var></span> </p> </div> <div class = "hints"> <p>The slope intercept form is <code>f(x) = slope \cdot x + y_{intercept}</code></p> <p>Since we cannot evaluate <code>f</code> at <code>0</code>, our general strategy will be to first find the slope, and then use the slope to work backwards from a point we know to the <code>y</code>-intercept.</p> <p>The slope of a line is the factor by which you multiply the change in abscissas to get the change in ordinates</p> <p><code>f(<var>XL</var>) = <var>YL</var></code> and <code>f(<var>XR</var>) = <var>YR</var></code></p> <p>so the change in abscissas from <code><var>XL</var></code> to <code><var>XR</var></code> is <code><var>XR-XL</var></code> and the corresponding change in ordinates is <code><var>YR</var> - <var>YL</var> = <var>YR-YL</var></code> </p> <p>Thus the slope of <code>f</code> is <code>\frac{<var>YR-YL</var>}{<var>XR-XL</var>} = <var>SLOPE</var></code></p> <p>Now since you have the slope, you should be able to figure out how much <code>f</code> changed from <code>x=0</code> to <code>x=<var>XL</var></code>. Combining this with the fact that <code>f(<var>XL</var>) = <var>YL</var></code> should let us figure out <code>f(0) = y_{intercept}</code> </p> <p><code>f</code> changed by <code>slope*(<var>XL</var>-0)=<var>SLOPE</var> \cdot <var>XL</var> = <var>SLOPE*XL</var></code> from <code>0</code> to <code><var>XL</var></code></p> <p>Thus <code>f(0) = <var>YL</var> - <var>SLOPE*XL</var> = <var>YINTERCEPT</var> </code></p> <p>Thus the equation of the line is <code>f(x)= <var>SLOPE</var> \cdot x + <var>YINTERCEPT</var></code>.</p> </div> </div> </div> </div> </body> </html>
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Comments (2) WOW! This is both beautiful and touching. Perhaps I should have passed by this LO without blowing it up and reading it. But I did. Today is January 19th...tomorrow is my 34th birthday. I should be happy...but my birthdays are bitter sweet. Tomorrow will mark the 18th anniversary of me learning I lost my first baby...I don't call it her (long story why I say "her") birthday because the remains were delivered later...and she died a bit before I began to "miscarry", exactly how long be for I'm not sure. I remember that birthday...it was supposed to be my "Sweet 16". It was anything but. First, I woke up bleeding that morning...spent 1/2 the morning at the ER, the other half of the day at an OB/GYN, the night on bed rest. At the OB's office, first they could find no heartbeat. Then, what the ultrasound showed still haunts me...there was part of a baby, only a skull with eye sockets, a nose indention, the form of closed lips......where did the rest of her go I wondered. I found out the answer to that question on January 24th. Gestational Trophoblastic Tumor...my oncologist told me, "Your baby saved your life. The cancer killed her before it began killing you. Had the baby not been forming you would not have the chance of survival that you do." They disected her "remains" and ran MANY tests. Only my OB and onco refered to the baby as "her", everyone else just called her "it". But, I named her anyways, my own coping mechanism..November Nicole...my precious angel. WOW! This is sooooo beautiful and sad at the same time. It touched my heart. I know the feeling. On March 1989 I lost a baby on my 13 week of pregnancy. He had an extra chromosome on the 13th pair. I got to know it was a boy so I named him Alan. I had another boy a year later, I have 3 kids but my youngest is not the third, he will always be the fourth. Alan is my third kid. It never goes away. I will always remember him sucking his thumb in the sonogram. Thanks for sharing this, you did a great job.
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National Taco Day is October 4. What better way to celebrate than to go to one of the restaurants below and order a giant margarita with a plate of tacos. Here are some deals my favorite restaurants are offering in honor of the soft or crispy taco – Los Gringos Locos Since today is such a special day, Gringos is serving three roasted pork belly tacos with caramelized onions, spices, rice and beans for $10. This pairs nicely with a $5 Habanero, Jicama, Prickly Pear, Pineapple or Mango margarita. 631 Foothill Blvd. La Canada, (818)790-2696. Casa Vega The Valley’s celebrity hot-spot is offering beef taquitos con guacamole or crispy four chicken flautas with creamy salso roja for 50% off their normal price on Friday, October 4 to celebrate National Taco Day. 13301 Ventura Blvd (818)788-4868. International flavors at Mondo Taco Taco Mondo One of my favorite taco shops located in a mini-mall offers braised short rib with a horseradish slaw, Moroccan lamb with a chimichurri sauce, grilled shrimp with chili sesame oil, soyrizo with peppers and mushrooms, and shredded pork topped with Dijon mushroom cream. Order one of the thirst-quenching frescas. I love the fresh watermelon or refreshing blueberry/ginger beverage. 1292 S La Brea Ave. (323) 939-0161. Petty Cash Hang out on Beverly during National Taco Day at one of the coolest taquerias in town. Chef Walter Manzke and his staff are offering one taco of your choice with a Modena on draft for $8 on Friday. DJ Stepdad – Davie Fernie will provide great tunes. 7360 Beverly Blvd. (323)933-5300. Goat Tacos Ebanos Crossing in downtown Los Angeles is a newer dining venue offering goat tacos that are braised in Mediterranean spices and garnished with yogurt slaw and rosemary oil. Pair them with a rum, mezcal, or tequila cocktail. 200 S. Hill St. (213)935-8829. Enjoy a good deal at the Americana at Brand during Happy Hour from 3 to 7 p.m. All drink specials and draft beers are $4. Sangria and well drinks are $5. Sip a martini, mojitos or margaritas for $6. Wines by the glass are $4 or order a bottle for $15. All go well with tacos. 750 Americana Way (818)551-1666
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self-care While doing a bit of research on the mind and negative thought patterns, I came across an inner critic quiz that helps you discover your inner critic patterns so you can create a plan for how to change them. Now I won’t list the 7 types of critics revealed in the quiz (you can discover them by taking the quiz via the link below), but I do want to start this post by defining ‘inner critic’. Self- Care Defined Everywhere you turn, someone is talking about self-care. It’s a super trendy hot topic these days. But what exactly is self-care? Why is self-care important? And how do you actually manage it and prioritize it? Over the next 4 (or maybe 6 posts), I’ll not only be sharing my perspective on this topic, but I’ll also be sharing what research and mental health experts are saying about how self-care protects and strengthens our overall health and well-being. Toxic Fishbowls Fish living in a goldfish bowl are not living their best life. Because most fish bowls lack a filtration system to keep their environment clean and free of toxins, very few goldfish survive for more than a few weeks. A bowl simply can’t provide a suitable living environment for any fish to live a healthy and long life.Likewise, an unfiltered or poorly filtered mind does not lead to living life more abundantly. When our thoughts go unchecked and unfiltered, we develop toxic mindsets that cause us to lead unhealthy, unproductive, and unhappy lives. But a new way of thinking will always precede a new way of living.
Mid
[ 0.633569739952718, 33.5, 19.375 ]
[Multidrug resistant effect of alternative splicing form of MAD2 gene-MAD2beta on human gastric cancer cell]. To study the effect of alternative splicing form -MAD2beta of mitotic arrest deficient protein 2 (MAD2) on the formation of multidrug resistance in human gastric adenocarcinoma cell SGC7901. RNA was extracted from a multidrug resistance cell line SGC7901/ADR. The full-length MAD2beta cDNA was obtained by RT-PCR and cloned into the pUCm-T vector, and then recombined into the eukaryotic expression vector pcDNA3.1 in forward direction. Subsequently, pcDNA3.1/MAD2beta vectors were then transfected into SGC7901 cells by lipofectamine. Sensitivity to drug was detected by MTT assay. Cell cycle alteration and intracellular fluorescence intensity were determined by FACS. A fragment of 0.53 Kb was obtained and confirmed by DNA sequencing which was a new alternative splicing form of MAD2 named as MAD2beta. pcDNA3.1/MAD2beta transfected SGC7901 cells (SGC7901/MAD2beta) were more resistant to ADR, VCR and MMC than the control cells (SGC7901/pcDNA3.1), and also ADR fluorescence intensity of SGC7901/MAD2beta cells was lower (P < 0.05) than that of SGC7901/pcDNA3.1 cells. MAD2beta could increase the multidrug resistance of SGC7901 cell line.
High
[ 0.716157205240174, 30.75, 12.1875 ]
Q: Generating unlimited electricity through magnets Well i had seen a toy, which i till date don't know was it battery powered or not? and also could it be used to actually create energy (I know that it is not possible but whats the problem?) Structure of a toy: Imagine a toy man having magnets at his feet, he is fixed to an axle and can just perform a motion in forward or backward direction, and also note that at the base of the man's feet (attached to that axle) is another magnet with same pole as that of the magnet of feet. What was happening: What was happening was that the man was continuously performing the motion and not stopping at all. The thing is that i saw it from a distance and so i could not know that wheter or not it used battery Questions Will it stop? Will it generate electricity forever if i put an turbine to at the axle 1. On Earth 2. In outer space 3. Under gravity + No Friction Well i know the simple answers in of most of them in form of yes/no but Why? Front View: A: With a generator of some sort extracting energy from the system, the system will stop in all three cases. By the principle of conservation of energy, you know that the total energy neither increases or decreases through any process. If the generator extracts energy from the system, the man will slow down and eventually stop. Intuitively, this is because of resistance in the generator. Even in an ideal world without friction, turning a magnet within a coil of wire (in order to generate an EMF in the coil) requires a constant force due to the retarding electromagnetic forces on the generator. (Consider science museum demonstrations in which you turn a wheel to light a light bulb. The wheel can be really tough to turn, and that's primarily because of electromagnetic force, not just friction.) Without a turbine, the question is marginally more interesting. In any environment with friction, the man will stop - again, since friction extracts energy from the system, it must slow down and eventually come to a halt. Only in the last case - the frictionless environment without a turbine - will the man perpetually remain in motion. Of course, this is a physically implausible situation, since any mechanical system will have at least some amount of friction.
Mid
[ 0.599542334096109, 32.75, 21.875 ]
1. Context {#sec76879} ========== Hepatitis C virus (HCV) is a global public health problem. About 170 million people, nearly 3% of the world's population, are chronically infected and at risk of developing liver diseases including cirrhosis and hepatocellular carcinoma. It has been estimated that three to four million are newly infected and 350000 deaths occur each year due to all HCV-related diseases. Liver dysfunction by HCV infection is one indication for liver transplantation. No vaccine or immune-globulins are available for prevention. Low effectiveness and high cost of the current antiviral drugs limit the treatments of HCV infection ([@A22915R1], [@A22915R2]). Nowadays the most common route of HCV transmission is through syringe sharing among intravenous drug users (IVDUs) with a prevalence rate of 31 to 89% in different geographical areas of the world. Risk of infection in some groups such as, patients on hemodialysis, hemophilia, infants of mothers with HCV infection, and multi-partner individuals is very high. Prevalence of HCV among patients on hemodialysis was 5.5% to 55.9% in different cities of Iran, although this number for patients with hemophilia was 15.65 to 76.7% ([@A22915R3]). The rate of HCV positivity among IVDUs was 52% in Tehran ([@A22915R4]). According to a recently conducted meta-analysis, the overall prevalence of HCV among blood donors in Iran's provinces and cities was 0.5% (95% CI: 0.4-0.6%). The highest prevalence of HCV was in Kashan (1.09%), and the lowest prevalence was in South Khorasan (0.03%). Hepatitis C virus prevalence had a decreasing trend over 11 years. The highest prevalence rate was 1.39% in 2005; then, the prevalence rate had a decreasing trend to 0.13% in 2007 and 0.03% in 2009 ([@A22915R5]). In Iran HCV prevalence in the general population is significantly lower when compared to the corresponding populations in surrounding countries such as Afghanistan (1.1%), Pakistan (4.7%) ([@A22915R6]), Turkey (1.0-2.1%), Kuwait (0.8%) and Iraq (7.1%) ([@A22915R7]). Hepatitis C virus is a small, enveloped virus that belongs to the genus Hepacivirus and the family Flaviviridae. The diameter of the virion is about 55-65 nm. The genome of HCV is a positive single-stranded RNA molecule of approximately 9.6 kb. It encodes a polyprotein from a single open reading frame flanked by two untranslated regions (UTR) at the 5\' and 3\' ends of the genome. This polyprotein precursor is cleaved by proteases giving rise to structural (Core, glycoproteins E1, E2) and nonstructural proteins (NS2, NS3, NS4A, NS4B, NS5A and NS5B). The HCV genome has a high rate of mutations therefore genetic heterogeneity of HCV is observed in patients ([@A22915R2], [@A22915R8]). Hepatitis C virus is classified into seven major genotypes (1, 2, 3, 4, 5, 6 and 7) and more than 67 subtypes (named a, b, c, etc.) ([@A22915R9]). Sequence differences range from 31% to 33% among genotypes and from 20% to 25% among subtypes. Distribution of HCV genotypes is variable. Genotypes 1 to 3 have a worldwide prevalence, whereas genotypes 4 to 6 are more geographically restricted. Genotype 4 is found mainly in North Africa and the Middle East, especially in Egypt. Genotype 5 is restricted to South Africa and genotype 6 is found in South East Asia ([@A22915R10]). The most dominant subtypes in south and north-west of Iran are 1a and 3a, respectively, while 1a predominates in all parts of Iran ([@A22915R11], [@A22915R12]). Hepatitis C virus genotyping is important for the management of infected individuals and for epidemiology purposes ([@A22915R10]). Genotypes indicate the route of acquisition, and affect the clinical outcome and response to treatment. For example response to interferon therapies in combination with ribavirin in patients infected with genotypes 1 and 4 is much lower than genotypes 2 and 3. Infection with genotype 1 may proceed rapidly to severe chronic disease, cirrhosis, and hepatocellular carcinoma. Studies have shown that subtype 3a is significantly associated with transmission through IVDUs in industrial countries ([@A22915R4]). Therefore, before any treatment, the genotype of the virus must be determined to estimate the dose, duration and type of antiviral drugs, and the virological monitoring method ([@A22915R10], [@A22915R12]). 2. Objectives {#sec77830} ============= There are studies on the prevalence of HCV genotypes in infected individuals from Iran, yet they have not provided information about the overall distribution of HCV genotypes. Knowledge of the prevalence and distribution of HCV genotypes in infected individuals is important for successful treatment, control strategies and researches in the future. The aim of this study was to estimate the overall prevalence and trend of HCV genotypes in Iranian patients. 3. Data Sources {#sec76883} =============== 3.1. Search Strategy and Selection Criteria {#sec76881} ------------------------------------------- The literature was searched to find scientific papers reporting the prevalence and distribution of HCV genotypes in Iran. Papers were identified through searching PubMed, Magiran, IranMedex, Scientific Information Databank (SID), and Google scholar databases. To search for as many related studies as possible, we used keywords including: "hepatitis C virus or HCV", "genotypes", "molecular epidemiology" and "Iran". We also manually searched relevant Persian journals. 4. Study Selection {#sec77831} ================== The inclusion criteria were: 1. full text articles in English or Persian language reporting on HCV genotypes among infected patients with Iranian nationality; 2. studies using HCV RNA as a detection index of infected subjects that had data indicating the prevalence of HCV genotypes using standard genotyping methods. The exclusion criteria were: 1. studies with unknown sample origins; 2. studies with overlapping subjects, time and place of sample collection; 3. studies that failed to present data clearly; 4. any contraries to the inclusion criteria. 5. Data Extraction {#sec77832} ================== All potentially relevant papers were reviewed independently by two investigators that assessed the eligibility of each article and the data were abstracted by the Excel data sheet. Irrelevant papers and papers that did not clearly report HCV genotypes were excluded. The following information were extracted from the papers: first author's name, study period (when the study was conducted), province or city of sampling, patients population, sample size, HCV genotypes and subtypes, genotype detection methods, gender, age, risk factors, etc.; although some papers did not contain all of these information. For studies with duration of more than one year, the middle period of the time frame was used for analysis of trend. In addition, for some studies in which the prevalence of genotypes was reported annually, prevalence results of each year was considered in trend analysis. Our analysis was in accordance with the items of systematic reviews and meta-analyses (PRISMA) guidelines for systematic review. 5.1. Statistical Analysis {#sec76882} ------------------------- We considered studies reporting HCV genotypes (or subtypes) in Iran and 95% confidence interval were calculated using random effects model and meta-analysis. The possibility of significant heterogeneity between studies was tested with the Q test (P \< 0.10 indicated statistically significant heterogeneity) and the I^2^ statistic. Freeman-Tukey's double arcsine transformation was used to stabilize variances. In addition, Forest plot was performed for study locations and years. Data manipulation and statistical analyzes were done using the Meta R statistical software package, version 2.13. In meta-analyses with sufficient number of published papers, a funnel plot was inspected visually to assess for publication bias. 6. Results {#sec76887} ========== 6.1. General Information of Samples {#sec76884} ----------------------------------- A total of 73 original articles were found by the electronic search that reported HCV genotypes and subtypes from provinces and cities of Iran ([Figure 1](#fig14820){ref-type="fig"}). After exclusion of ineligible papers, the final sample of the meta-analysis included 53 studies published between 1999 and 31 June 2014, including 44 studies in English, and 12 studies in Persian. Studies were done in 20 provinces or cities of Iran. In total, 22952 patients were meta-analyzed for pooled prevalence of HCV genotypes ([Table 1](#tbl19551){ref-type="table"}). The populations of studies were mainly chronic HCV infected patients including patients with hemophilia and thalassemia as well as those on hemodialysis, and multiply transfusion. In addition, a few studies included patients with a history of prison, dental visits, tattooing, unprotected sexual contact, kidney transplantation, intravenous drug use, and blood donation ([Table 1](#tbl19551){ref-type="table"}). Since the genotypes and subtypes in the patients\' populations were not reported separately, we were unable to analyze the prevalence of genotypes and subtypes according to population groups. Gender of patients was mostly male (45% to 100%) ([Table 1](#tbl19551){ref-type="table"}). As genotypes according to patients\' gender were not reported by all papers, we were unable to analyze the prevalence of genotypes according to gender. Due to data limitation, subtypes 2a and 2b were not analyzed. Data regarding risk factors were not complete in published papers thus their analysis was not possible. Different methods have been used for HCV genotyping in Iranian patients, including 5´-UTR restriction fragment length polymorphism (RFLP), DNA sequencing of NS5B or 5\'-UTR/core genes, high resolution melting analysis, nucleic acid hybridization, and type-specific PCR for core region. Due to space restriction, we couldn\'t show data of genotyping methods for each paper in [Table 1](#tbl19551){ref-type="table"}. ###### Distribution of Hepatitis C Virus Genotypes and Subtypes in Patients From Cities and Provinces of Iran ^[a](#fn16863){ref-type="table-fn"}^ Province or City Year of Study Sample Size Male Subtype 1a Subtype 1b Subtype 1a + Subtype 1b Genotype 2 Subtype 2a Subtype 2b Subtype 3a Subtype 3b Genotype 4 Genotype 5 Mixed Untypeable Reference ----------------------------------------------------------------------------------------------------- --------------------------------------------------------------- ------------- -------------- ------------------------------------------------- ------------ ------------------------------------------------- ------------ ------------ ------------ ------------- ------------ ------------ ------------ ----------- ------------ ---------------- **Tehran** Chronic HCV infected patients March 2003-December 2011 ^[b](#fn16864){ref-type="table-fn"}^ 11561 9645 (83.42) (44.90) (11.3) (0.70) (39.6) (0.20) (0.80) 292 (2.5) ([@A22915R12]) Injection drug users October 2008-June 2009 83 (97.80) 35 (42) 48 (58) ([@A22915R13]) Chronic HCV infected thalassemic, hemophilia patients September 2008-April 2010 152 109 (77.7) 92 (60.6) 12 (7.9) 42 (27.6) 2 (1.3) 4 (2.6) ([@A22915R14]) Occult HCV infected patients with chronic liver disease September 2007-March 2010 7 5 (71) 2 (29) 3 (43) 2 (29) ([@A22915R15]) HCV infected thalassemic, hemophilia patients September 2008-April 2009 133 100 (75.2) 85 (63.9) 8 (6.0) 35 (26.2) 2 (2.3) 3 (2.3) ([@A22915R16]) Blood donors and HCV infected patients December 2006 and January 2008 167 ND 87 (52.1) 14 (29.3) 55 (12) 11 (6.6) ([@A22915R17]) Patients with hemophilia 2009 367 343 (93.46) \(58\) (14.70) (0.80) (18.50) (1.10) (6.20) (0.50) ([@A22915R18]) Chronic hepatitis C patients 2002-2005 384 307 (79.9) 154 (40.1) 43 (11.2) 105 (27.3) 3 (0.8) 76 (19.8) ([@A22915R19]) HCV infected patients 2003-2005 2231 1853 (83.1) 886 (39.7) 271 (12.1) (1.3) (1.4) 613 (27.5) (0.2) (0.9) 33 (1.6) 401 (18) ([@A22915R20]) Chronic HCV infected IUDs December 2008-March 2009 36 35 \(25\) (16.70) (58.3) ([@A22915R4]) HCV infected individuals 1999 15 ND 7 (47) 3 (20) 4 (27) 1(6) ([@A22915R21]) Patients on hemodialysis 2004 66 36 (45.54) 19 (28.8) 12 (18.2) 2 (3) 20 (30.3) 2 (3) 11 (16.7) ([@A22915R22]) HCV infected patients 2003-2004 214 141 (65.88) 117 (52.88) 28 (14.01) 1 (2.1) 63 (27.57) 5 (3.44) ([@A22915R23]) HCV infected patients 2004 20 ND 11 (55) ^[c](#fn16865){ref-type="table-fn"}^ 4 (20) 5 (25) ([@A22915R24]) **Mashhad** HCV-infected patients, hemophilia October 2009-October 2010 382 299 (79) 147 (39.2) 41 (10.9) 4 (1.04) 9 (2.4) 9 (2.4) 150 (40) 13 (3.4) 3 (0.8) 4 (2.5) ([@A22915R25]) Hemodialysis, thalassemia, injection, blood donors 2003-2004 63 ND 19 (30.10) ^[c](#fn16865){ref-type="table-fn"}^ 10 (15.9) 33 (52.3) 1 (1.6) ([@A22915R26]) Chronic HCV infection 2000-2010 278 227 (82) (36.30) (12.60) (0.40) (49.60) (1.40) ([@A22915R11]) **Bushehr** Blood donors 2007-2008 60 55 (91.6) 23 (36.7) 3 (5) 1 (1.7) 2 (3.3) 22 (38.3) 1 (1.7) 1 (1.7) 1 (1.7) 6 (10) ([@A22915R27]) HCV infected patients 2007-2009 100 96 (96) 49 (49) 5 (5) 40 (40) 1 (1) 5 (5) ([@A22915R28]) Tehran and Rasht HCV infected patients 2009-2010 40 ND 19 (42) 7 (20) 14 (37.5%) ([@A22915R29]) **Guilan** Chronic hemodialysis patients January-May 2008 32 24 (75) 19 (59.4) 13 (40.6%) ([@A22915R30]) **Rasht (Guilan)** Thalassemia and chronic HCV infection December 1998-April 2006 30 17 (56.7) 24 (80) 6 (20) ([@A22915R31]) **Mazandaran and Guilan** Patients with thalassemia February and March 2010 28 15 (53) 9 (32.1) 1 (3.6) 18 (64.3) ([@A22915R32]) **Mazandaran** Thalassemia, hemophilia, hemodialysis, intravenous drug addicts 2009-2011 135 94 (69.6) 37 (27.40) 11 (8.20) 67 (51.10) 16 (11.9) ([@A22915R33]) **Qaemshahr (Mazandaran)** DIAs, hemodialysis, transfusion, surgery, kidney transplant 2007-2010 86 74 (86) 36 (41.8) ^[d](#fn16866){ref-type="table-fn"}^ 50 (58.2) ([@A22915R34]) **Sari and Ghaemshahr** Hemodialysis 2009-2010 113 77 (65.4) 68 (60.17) ^[d](#fn16866){ref-type="table-fn"}^ 44 (38.93) 1 (0.001) ([@A22915R35]) **Ahvaz** Patients with hemophilia February 2008-March 2009 42 39 (51.3) 26 (61.9) 11 (26.1) 5 (11.9) ([@A22915R36]) Blood donors 2007-2008 45 45 (100) 24 (53.3) 17 (37.7) 4 (8.8) ([@A22915R37]) Khuzestan province Hemodialysis patients March 2005-August 2006 17 9 (47.36) 7 (41.1) 4 (23.5) 6(35.2) ([@A22915R38]) HCV infected patients 2005-2007 280 188 (67.1) 130 (46.4) 45 (16.1) 6 (2.1) 99 (35.4) ([@A22915R38]) **Shahrekord** HCV infected patients 2009-2010 94 63 (98.4) 51 (54.26) 11 (11.71) 2 (2.12) 26 (27.66) 4 (4.25) ([@A22915R39]) Shiraz Chronic hepatitis: drug injection abusers, thalassemia, hemodialysis, sexual contacts, hemophiliacs 2010 634 550 (86.8) 137 (26.2) 55 (8.7) 4 (0.6) 15 (2.4) 259 (40.9) 8 (1.3) 156 (24.6) ([@A22915R40]) Thalassemia, history of prison, dental visits, hemophilia, hemodialysis and tattooing 2004-2005 238 152 (64) 103 (43.3) 39 (16.4) 96 (40.3) ([@A22915R41]) Patients with chronic hepatitis 2003-2006 118 26 (22) 11 (9.3) 48 (40.7) 14 (11.9) ([@A22915R42]) **Zahedan** HCV infected Patients July 2007-April 2009 52 39 (75) 28 (53.84) ^[c](#fn16865){ref-type="table-fn"}^ 2 (3.88) 12 (23.08 ) 7 (13.4) 3 (5.77) ([@A22915R43]) Thalassemia and chronic HCV infection August 2005-September 2007 20 13 (65) 3 (15) 5 (25) 8 (40) 4(20) ([@A22915R44]) **Yazd Province** IV drug addicts 2007 and 2010 71 68 (95.7) 25 (35) 46 (65) ([@A22915R45]) HCV infected patients March 2010-June 2013 191 152 (79.6) 74 (38.7) 13 (6.8) 3 (1.6) 96 (50.3) (2.60) ([@A22915R2]) **Isfahan Province** HCV infected Patients March 2007 and April 2009 97 95 (97.70) (29.50) (5.10) (2.00) (61.20) \(2\) ([@A22915R46]) **Golestan** Blood donors 2009-2010 91 70 (90.90) 15 (16.48) 15 (16.48) 1 (1.01) 2 (2.19) 12 (13.18) 19 (20.87) 6 (6.59) 7 (7.69) 14 (15.38) ([@A22915R47]) **Iran** General population patients 2004 -December 2007 206 83 (172) 53 (25.73) 36 (17.47) 4 (1.95) 96 (46.6) 11 (5.33) 6 (2.91) ([@A22915R48]) **Iran** ^[e](#fn16867){ref-type="table-fn"}^ HCV infected patients June 2002-May 2003 156 125 59 (37.8) 26 (16.7) 2 (1.28) 45 (28.8) 2 (1.28) 1 21 (13.46) ([@A22915R49]) **Different parts of Iran** ^**[f](#fn16868){ref-type="table-fn"}**^ HCV positive carriers 2000-2005 116 93 (80.17) (61.20) (13.80) \(25\) ([@A22915R50]) **Tehran, Tabriz, Guilan and Shiraz** Multiply transfused patients with thalassemia and chronic hepatitis C 2008 280 165 (59) 146 (52) 14 (5) 3 (1.1) 3 (1.1) 97 (34.5) 1 (0.4) 16 (3.4) 7 (2.5) ([@A22915R51]) **East Azerbaijan** HCV infected patients on dialysis 2006 55 49 (92.45) 42 (76.4) 3 (5.5) 1 (1.8) 3 (5.5%) 4 (10.9%) ([@A22915R52]) **West Azerbaijan** HCV infected patients 2006-2008 160 13 (8.12) ^[d](#fn16866){ref-type="table-fn"}^ 19 (11.87) 35 (21.87) 77 (48.12) 16 (10.0) ([@A22915R53]) **Tehran, Kerman, Kermanshah** HCV infected blood donors December 2006 and January 2008 103 101 (98.1) 53 (51.5) 4 (3.9) 39 (37.9 ) 7 (6.8) ([@A22915R54]) **Tehran, Sari, Tabriz, Oromieh, Kermanshah, Zahedan** ^**[g](#fn16869){ref-type="table-fn"}**^ HCV infected patients 1999-2003 125 ND 59 (47) 10 (8) 1 (0.8) 45 (36) 9 (7) ([@A22915R55]) **Markazi Province** Patients on hemodialysis March-July 2005 8 1 (12.5) 4 (50) 1 (12.5) 1 (12.5) 2 (25) ([@A22915R56]) **Markazi** Thalassemia and inherited bleeding disorders March-July 2004 23 31 (93.9) 9 (40.9) 2 (9.1) 1 (4.54) 4 (18.2) 6 (27) ([@A22915R57]) **Kerman** Patients with thalassemia, heroin abusers and tattoo 2004 -2006 85 57 (67.1) 33 (38.8) 7 (8.2) 25 (29.4) 20 (23.5) ([@A22915R58]) **Kermanshah** Patients, hemodialysis, thalassemia 2005-2006 122 108 (88.5) 35 (28.8) 30 (24.65) 21 (17.2) 22 (18) 4 (3.2 ) 10 (8.1) ([@A22915R59]) **Hormozgan** Chronic HCV infection March 2011-March 2012 509 478 (93.9) 316 (62.1) 117 (23) 76 (14.9) ([@A22915R3]) ^a^ Abbreviation: ND, non-determined; data are presented as No. or No. (%). ^b^In this study samples were from nine separate years (2003 to 2011) and genotypes distributions were presented separately for each year; meta-analysis was done for the period of the study. ^c^ Subtypes of genotype 1 were not determined, because of space limitation in table, we assumed them as 1a. ^d^ Subtypes of HCV genotype 1 had not been determined to subtypes 1a and 1b, had been reported the HCV genotype 1 as HCV genotype 1a/b (not meta-analyzed) ^e^In this study samples were from five regions of Iran and genotypes distributions were presented for each region. ^f^ In this study samples were from six region of Iran and genotypes distributions were presented for each region. ^g^ In this study samples were from six provinces of Iran and genotypes distributions were presented for each province: meta-analysis was done for provinces. ![Flow Chart of Literature Search for Original Articles in English and Persian Reporting HCV Genotypes and Subtypes From Provinces and Cities of Iran](hepatmon-14-12-22915-i001){#fig14820} 6.2. Prevalence of HCV Genotypes and Subtypes in Iran Provinces and Cities {#sec76885} -------------------------------------------------------------------------- All studies were divided into 20 groups according to the place of sampling. Some studies were reporting HCV genotypes from all of Iran without separating data according cities or provinces; we included them in a separate group named, Iran ([Table 1](#tbl19551){ref-type="table"}, [Figure 2](#fig14821){ref-type="fig"}). Estimated pooled prevalence of HCV genotypes in patients distributed in provinces and cities of Iran were as follows; Subtype 1a was the predominant genotype with a rate of 39% (34-44%, 95% CI); followed by subtype 3a, 32% (26-39%, 95% CI); subtype 1b, 13% (10-15%, 95% CI); genotype 4, 5.18% (3.27-7.5%, 95% CI); and genotype 2, 3.6% (1.6-8.3%, 95% CI), respectively. Untypeable HCV was 0.3% (95% CI: 0.07-0.16%). Remnants were mixed genotypes 1.96%, genotype 5, 0.056% and other subtypes ([Table 1](#tbl19551){ref-type="table"}, [Figure 1](#fig14820){ref-type="fig"}). Genotype 6 was reported only in one study with prevalence rate 1 (0.7%) with subtype 6a ([@A22915R60]). Estimated overall prevalence of HCV genotypes was variable in cities and provinces of Iran. For example, subtype 1a was maximum in East Azarbyjan, 76% (95% CI: 63-87%) and Guilan, 69% (95% CI: 56-80%) respectively; but minimum in West Azarbayjan, 8% (95% CI: 4-13%). Prevalence of subtype 3a was variable in cities and provinces of Iran, for instance, maximum in Guilan, 76% (95% CI 63-86%) and Isfahan, 61% (95% CI, 50-71%) yet, minimum in West Azarbyjan, 5% (95% CI, 1-15%). Prevalence of subtype 1b was 13% (95% CI: 10-15%) in Iran, while in Kermanshah, Zahedan and Hormozgan, it was 25% (95% CI: 17-33%), 25% (95% CI: 9-49%), and 23% (95% CI: 19-27%) respectively. Prevalence of genotype 2 was 0.7% (95% CI: 0.06-0.8%) in Tehran, while in Kermanshah it was 17.2% (95% CI: 11-25.1%) ([Figure 2](#fig14821){ref-type="fig"}). ![Forest Plot Showing Hepatitis C Virus Genotypes and Subtypes 1a, 1b, 2, 3a and 4 Prevalence Estimates in Patients of Iranian Cities and Provinces](hepatmon-14-12-22915-i002){#fig14821} 6.3. Time {#sec76886} --------- All studies were divided into 14 groups according to the year of study. The estimated annual prevalence rates of HCV genotypes over years are shown in [Figure 3](#fig14822){ref-type="fig"}. Funnel plots for detecting biases in published papers are presented in [Figures 4](#fig14823){ref-type="fig"} and [5](#fig14824){ref-type="fig"}. ![Forest Plot Showing Hepatitis C Virus Genotypes and Subtypes 1a, 1b, 2, 3a and 4, Prevalence Estimates in Iranian Patients According to Study Period](hepatmon-14-12-22915-i003){#fig14822} ![Funnel Plot Detecting Biases in the Identification and Selection of Studies for HCV Genotypes and Subtypes 1a, 1b, 2, 3a, and 4 in Iranian Cities and Provinces](hepatmon-14-12-22915-i004){#fig14823} ![Funnel Plot Detecting Biases in the Identification and Selection of Studies for HCV Genotypes and Subtypes 1a, 1b, 2, 3a, and 4 According to Study Periods](hepatmon-14-12-22915-i005){#fig14824} 7. Conclusions {#sec76888} ============== The frequency of HCV genotypes in different geographic regions is variable and changes over time, depending on the study population, the route of infection and virus mutation ([@A22915R3], [@A22915R11]). Considering countries around Iran, the predominant HCV subtype is 1b in Turkey, Russia, Moldova and Uzbekistan ([@A22915R10]), 4 in Saudi Arabia, Iraq, Qatar, Bahrain, Kuwait and Yemen ([@A22915R12]), and 6a in China ([@A22915R3]). The predominant subtype is 3a in Pakistan ([@A22915R6]) and India ([@A22915R33]). Despite the high number of visitors from and to Arab countries (Iraq, Qatar, Bahrain, Saudi Arabia, Kuwait) with predominant genotype 4, and visitors from north neighboring countries such as Tajikistan and Turkmenistan with the predominant subtype 1b; subtype 1a is dominant in Iran. It is probable that differences in the population, routes of transmission, and socioeconomic factors may explain this variation. The prevalence of genotype 4 in Iran is lower compared to the majority of Middle Eastern countries including the neighboring Arabic countries. In Iran genotype 4 is prevalent in patients on hemodialysis and this is related to communication with neighboring countries such as Iraq and Saudi Arabia during the pilgrims and Hajj ceremony. There are no reliable reports about HCV genotypes in Afghanistan, the eastern neighbor of Iran. During the past few years, due to the political situation of Afghanistan, a considerable number of immigrants came to Iran. These immigrants usually do many trips between Iran, Afghanistan and Pakistan ([@A22915R11]). Only one study has reported HCV genotype 5 (3.4%) in their study population ([@A22915R25]), which can be a result of genotyping mistake and should be confirmed by DNA sequencing. Genotype 6 was reported only in one study with a prevalence rate of 1/ 142 (0.7%) with subtype 6a in one patient who had referred to the Gastroenterology Department of Taleghani Hospital (Tehran, Iran) between June 2007 and June 2012 ([@A22915R60]). It seems that this patient was infected by HCV in a country other than Iran. In one study that was performed recently on 11561 chronic HCV infected patients in Iran, the results revealed that, 1a was common (44.9%) followed by 3a (39.6%) and 1b (11.3%) ([@A22915R12]). Distribution of HCV genotypes in our population is similar to the pattern on HCV in northern Europe, where genotypes 1, 2 and 3 are more frequent ([@A22915R25], [@A22915R33]). In the last decade, an increase in HCV genotype distribution has been reported in many countries, for example there has been an increase in the prevalence of the 3a, 1a, and 4 ([@A22915R12]). Documents have indicated that HCV infection has risen dramatically in the recent years in the Middle East region. This might be related to changes in the main route of transmission ([@A22915R61]). Subtype 1a and 1b are the dominant genotypes in older and 3a in young patients in Iran ([@A22915R25]). The high prevalence of subtype 3a in young patients suggests an increased number of IVDU as the main route of HCV transmission. In addition, recent studies have indicated that there is an increase in the frequency of 3a in the younger population of Iran, Germany, Serbia, Montenegro, and Slovenia; this is because the main route for transmission of HCV is IVDU, due to sharing of unsterilized needles and syringes. Therefore, young male individuals are more susceptible to acquire HCV infection ([@A22915R12]). Considering forest plots in [Figure 3](#fig14822){ref-type="fig"}, our results showed that trend of hepatitis C virus genotypes 1a and 1b was fluctuating from 1999 to 2013, with a sinus shape, reaching maximum levels in 2012 and minimum levels in 2013; the reasons of these fluctuations are unknown. The overall trend of genotypes 2 and 4 was decreasing yet there was an increase for genotype 2 in 2012 and genotype 4 in 2011. The overall trend of subtype 3a decreased from 2001 to 2003 yet increased from 2004 to 2011, while it suddenly decreased in 2012 followed by a sharp increase to maximum levels in 2013. The reasons of these fluctuations is unknown. It must be noted that changes in the route of transmission and easy travelling in the recent years are the probable cause. Further researches are needed to find definitive answers. Mixed HCV infection with more than one genotype is possible. Numerous reports suggest that HCV infection with one genotype does not prevent becoming infected with other HCV genotypes. Multiple exposures to HCV may occur from time to time. Mixed infections may lead to severe disease, poor response to antiviral therapy or relapse after treatment. Studies have reported that mixed infection with two genotypes of HCV occurs in approximately 1% of HCV infected patients, and 1.6 to 31% of patients with multi-transfused hemophilia. In one study multiple HCV subtypes (1a and 3a) were detected in 2.5% of the patients ([@A22915R12]). In the present study the prevalence of mixed HCV infection was 1.96%, which is compatible with previous reports. A funnel plot is helpful for detecting biases around the line of identity in the meta-analysis. In our study the bias assessment plot was symmetric for subtypes 1a, 1b, 3a and genotype 4, yet asymmetric for genotype 2 in papers reporting HCV genotypes in Iranian cities ([Figure 4](#fig14823){ref-type="fig"}). Research insufficiency may be the reason of bias for genotype 2. Also, according to study periods, bias assessment plots were symmetric for genotypes or subtypes 1a, 1b, 2, 4 and 3a ([Figure 5](#fig14824){ref-type="fig"}). Forest plots of the present study indicated heterogeneity in the published papers ([Figures 2](#fig14821){ref-type="fig"} and [3](#fig14822){ref-type="fig"}). On the other hand, forest plots showed that, as the sample size increases confidence intervals of estimated prevalence become narrower. Thus, large sample sizes increase the precision of the study. In addition, samples collected from some parts of Iran were not adequate for obtaining firm conclusions. A proportion of HCV in Iranian patients was reported untypeable. Presence of untypeable HCV samples in some studies indicates the inability of genotyping methods to discriminate HCV genotypes. To be able to identify untypeable HCV genotypes, sequencing and phylogenetic analysis of viral genome is needed for characterization. Thus, the future goal of researches should be to sequence genomes of untypeable samples. However, in some papers of the present study genotypes of HCV were detected by sequencing. The transmission of HCV occurs mainly through infected blood, blood products, and contaminated devices, such as syringes and needles. Today, intravenous drug use is an important route of HCV transmission ([@A22915R4], [@A22915R61]). Precise data about the pattern of HCV genotypes and continuous monitoring of the genetic diversity of virus isolates, especially among high-risk individuals (hemophilia, thalassemia, hemodialysis, multiply transfused patients and intravenous drug use) is essential for understanding HCV epidemiology ([@A22915R33]). The evaluation of HCV risk factors among patient populations is important. Most studies did not report on HCV genotypes in patient populations completely. Because of this defect in data, we were unable analyze the prevalence of HCV genotypes according to patient's population. Male gender was reported as an independent predictive factor for HCV infection in previous studies ([@A22915R61]). The findings of a recent study showed that there was no association between having a variety of HCV genotypes and gender, level of education, risk factors, job, income, HIV infection, HBV infection, IV drug abuse and presence of underlying diseases ([@A22915R3]). In our study most of the HCV infected patients were male ([Table 1](#tbl19551){ref-type="table"}). However, most studies have not reported prevalence of HCV genotypes according to gender and above-mentioned factors separately. Thus, we were unable analyze the prevalence of HCV genotypes according to gender and risk factors. A range of studies on the prevalence of HCV genotypes in Iranian patients have been conducted. A meta-analysis of these prevalence data would be important for public health policy making and planning of clinical services addressing the needs of these infected people. The advantage of this meta-analysis is that it merges a large sample size and long study period, which help draw a reliable estimate of HCV genotypes distribution in Iran. Our study had a number of limitations that should be mentioned. First, different HCV genotyping methods were used by various studies and this may impact the obtained results of this meta-analysis. Second, although study populations were all Iranian patients, yet some populations were special patient groups and including them in a meta-analysis is somewhat challenging. Furthermore, data limitations of some studies did not allow their analysis. In conclusion, our study showed that the most frequent subtypes of HCV in Iran were 1a, 3a and 1b, respectively. This frequency differs in cities and provinces of Iran and neighboring countries. Also, this meta-analysis reported on the overall estimate for distribution of HCV genotypes in Iran. Therefore, due to the possibility of changing dominant viral genotypes in communities based on migrations and viral genome mutations, it is important to determine distribution of HCV genotypes in different geographical areas and its trend with time for epidemiological and patient management purposes. We would like to thank the Kurdistan University of Medical Sciences for their supports. **Funding/Support:**The submitted manuscript was a meta-analysis and the Kurdistan University of Medical Sciences funded the project.
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Q: Elegant way to add "selected" into a dropdrown form using PHP I have a form that looks like this: $guests = 2; // Just for testing $form = 'Guests<br /><select name="guests"> <option value="0">0</option> <option value="1">1</option> <option value="2">2</option> <option value="3">3</option> <option value="4">4</option> <option value="5">5</option> <option value="6">6</option> <option value="7">7</option> <option value="8">8</option> <option value="9">9</option> </select>'; But the list of guests can be up to 100, so I probably shouldn't do a check for which value is selected manually. What would be a good way for me to make the selected value become the selected value when the user sees this form? Thanks! A: What I normally do with dynamic select menus is use a foreach loop over an array, and just check each item. Something like: $dropdownName = "guests"; //Name of dropdown $defaultValue = "0"; //value to select if there isn't one already set $items = array('name'=>'value', 'anotherName'=>'anothervalue'); //items, name=>value echo '<select name="'.$dropdownName.'">'; //Start Select $selectedItem = (isset($_POST[$dropdownName])?$_POST[$dropdownName]:$defaultValue); //If a value is set, use it, otherwise use the default foreach ($items as $name=>$value) //build select { echo '<option value="'.$value.'"'.(($selectedItem == $value)?' selected="selected"':'').'>'.$name.'</option>'; } echo '</select>'; A: $guests = 2; // Just for testing $form = 'Guests<br /><select name="guests">'; for($i = 0; $i <= 100; $i++) $form .= "<option value='$i'{$guest == $i ? ' selected=\'selected\'' : ''}>$i</option>"; $form .= '</select>'; EDIT: $i < 101 was pretty lame...
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Parental Alienation and the Bystander Effect This is a true story. A parent recently made a call to the local Family Maintenance Enforcement Program, which had indicated by letter that should he have any concerns about ongoing child support issues, he could call the agency to discuss matters. His "concern" had in fact escalated over a period of thirteen years of forced estrangement from his child to a profound fear for the health and well-being of his son, now in his early twenties. Despite the invitation to call the agency, the curt reply to his desperate entreaties to the program officer was, first, that parental alienation was not an issue of professional concern to the agency as "the jury is still out on whether parental alienation even exists"; and second, that there was absolutely nothing the agency could do for him. The call was then abruptly ended by the program officer. Tragically, this lack of response is routinely reported by parents alienated from their children, who seek the help of legal, child welfare and mental health professionals, and anyone who will listen to them, in a desperate attempt to find someone to intervene in this serious abuse of their children. As they muster the courage to break through shame and speak about their fears, anxiety, and profound grief, they continue to be subjected to a mean-spirited cultural response, where their woundedness is often ignored or, worse, mocked and ridiculed. In the rare instances where parents are listened to, there is rarely any offer of support in regard to the alienation. These responses are illustrations of the "bystander effect," which is the typical response not only of lay people but also, alarmingly, of child and family professionals, to reports of parental alienation. In such an atmosphere alienated parents feel cut off and further alienated, isolated and alone, and their children remain at risk. The bystander effect is an attitude of indifference and apathy, a simple refusal to get involved or offer assistance to another in need. Most alienated parents are thus justifiably afraid of disclosing the alienation, trauma and abuse suffered by their children and themselves. They are repeatedly subjected to the bystander effect, particularly by professional helpers. The "professional bystander" effect, where the lack of action of others discourages a professional service provider from intervening in an emergency situation, applies to the phenomenon of parental alienation; and the main features of the bystander effect, including ambiguity, a reticence to act, lack of empathy, perceiving abuse as "normal" human behavior, fear of becoming a target, and diffusion of responsibility, are all present in regard to parental alienation situations in which professionals become involved. Ambivalence and ambiguity exist among professionals despite what the research says about parental alienation; often, rather than immersing and educating themselves in the research, professionals monitor the reactions of other service providers to determine if it is necessary to intervene. If it is determined that others are not reacting to the situation, bystanders will interpret the situation as not an emergency and will not intervene, an example of pluralistic ignorance. Few people want to be the first to take action in ambiguous situations, particularly if they lack empathy in regard to the suffering of those affected; and they are slow to help a victim because they believe someone else will take responsibility. This is where the denial of parental alienation among some in the mental health field is most harmful. The bystander phenomenon is particularly tragic and alarming because parental alienation is one of the most serious, yet largely unrecognized, forms of psychological abuse toward children, and affects a much larger number of North Americans than previously assumed. Alienating parents’ behavior constitutes psychological abuse when they manipulate and influence children to participate in depriving themselves of love, nurturance, and involvement with their other parent. Denial of and indifference to this form of abuse of children is reminiscent of society’s denial in the early twentieth century of the prevalence of physical and sexual abuse of children (Warshak, 2015). Parental alienation is also a form of psychological domestic violence, as the suffering of targeted parents is deep and unending, and represents a complex trauma of profound magnitude (Kruk, 2011). According to Bernet (2010), there is not only a large body of research validating the existence and harms of parental alienation, with over 500 articles on the subject, but also the published testimonies of thousands of adults who attest to having suffered through it as children, and other parents who are currently traumatized, watching helplessly as their relationships with their children are being destroyed. Harman & Biringen (in press) sampled a representative poll of adults in the United States, and found a startling rate of 13.4% of parents reporting that they have been alienated from one or more of their children by the other parent, with half of those reporting the alienation as severe. This percentage represents approximately 10.5 million parents in the US alone who are facing what they perceive to be parental alienation. The sheer magnitude of parental alienation indicates that this is a major social problem and a social justice issue for children and families. This takes the issue out of the realm of disinterested reportage and into the realm of action. And this is where the problem lies: professional inaction in the face of abundant evidence of the large-scale and serious harms of parental alienation to children and parents. The real issue is the indifference of legal and mental health professionals who seem to be unaware of the existing research, deny the existence of parental alienation, and contribute to the harms suffered by children and parents through their inaction. Professional incompetence in both the assessment and treatment of parental alienation is a serious problem, as many mental health practitioners lack knowledge and competence in working with this population (Lorandos et al, 2013; Baker & Sauber, 2012). This is not an acceptable state of affairs. Mental health professionals need to be held accountable, for both their denial and their inaction. It is the responsibility of professional service providers to support parents in the fulfillment of their parental responsibilities to their children's needs. The disregard of children's primary need for the love and care of both of their parents after parental separation requires a proactive approach on the part of professionally knowledgeable and competent mental health professionals working with this population. In regard to parental alienation, the system is the problem; that is, the roots of alienation lie primarily in the adversarial nature of legal determination of parenting after divorce. Parents are set up to fight in an effort to win primary residence or custody of their children, and the system rewards those skilled in adversarial combat. Parents often win their case by disparaging the other parent as a parent, in effect engaging in alienating behavior, and alienating behavior is thereby encouraged. Once they obtain a court order, residential parents are placed in a position to exercise their revenge with impunity, confident that the non-resident parents have little or no rights. The saying, “power corrupts and absolute power corrupts absolutely” holds true in primary residence awards. Thus the removal of a fit and loving parent as a primary caregiver from the life of a child, I would argue, is in itself a form of parental alienation, as children are robbed of their parent’s routine care and nurture, as well as that of their extended family. Shared parenting, on the other hand, reduces the risk and incidence of parental alienation, because children continue to maintain meaningful routine relationships with both of their parents, and are thus less susceptible to the toxic influence of an alienating parent. At the same time, with shared parenting parents are not threatened by the potential loss of their relationship with their children, and a parent is less likely to denigrate the other parent in an effort to bolster their own sense of parental identity and obtain a primary residence order. Thus it behooves service providers to advocate for shared parenting in the interests of the children and families with whom they work. Source: Shutterstock In the words of Dietrich Boenhoffer, "Silence in the face of evil is itself evil: Not to speak is to speak. Not to act is to act." Each of us has great potential power to act to help those in need, and to influence others to act. Above all else, in regard to parental alienation as a largely unrecognized form of child abuse and domestic violence, the message to professionals and non-professional lay people alike should be, “Don't be a bystander.” It takes moral courage to act, and in the case of parental alienation, action is urgently needed. Baker, A. & Sauber, R. (2012). Working with Alienated Children and Families: A Clinical Guidebook. New York: Routledge. Your opinion should not be published as anything other than that: it's just your opinion. That would becfine, of course, except that your opinion is presented as if it is based on evidrnce and it's not. It's just a regurgitation of s bunch of other opinions. You really should have made that clear at the outset -of the article. Pseudoscience at its worst - you should move to the US - we love pseudoscience here. Oh - and to the owners of Psychology Today - I just canceled my subscription. Enough with the pseudoscience already. Yuck. I could not disagree more with your comments about shared parenting:...Children are less susceptible to the toxic influence of an alienating parent. At the same time, with shared parenting parents are not threatened by the potential loss of their relationship with their children, and a parent is less likely to denigrate the other parent in an effort to bolster their own sense of parental identity...." A relentless, many years long assault on the target parent by the toxic, narcissistic, alienating parent ABSOLUTELY has a PROFOUND negative effect on children and a PROFOUND negative effect on childrens' relationships with the target parent. In addition, a shared parenting arrangement has ZERO influence on the likelihood of a toxic, narcissistic, alienating parent to denigrate the target parent. Toxic, narcissistic, alienating parents are 100% likely to continue denigrating the target parent whether the court awards shared parenting, or not. Toxic, narcissistic, alienating parents are very skilled at instilling profound fear in both target parents and children. After sufficient abuse, it is not uncommon for children to decide that it is much safer to go along with and spend more time with the toxic, narcissistic, alienating parent. Thanks, Susan, for your comment. I fully agree with you in regard to the relentless denigrating behavior of alienating parents, which in many cases will persist unabated, regardless of the child's residential arrangement. However, the influence and impact of the alienating parent on the child will be mitigated when the child is living in a shared as opposed to a sole residential arrangement. When the non-alienating parent is able to influence the child on an equal footing (ie within a shared parenting arrangement), their positive influence will go a long way to counterbalance the poison of the alienating parent's continued attempts to undermine the child's relationship with the other parent. Children desperately need that positive influence, as opposed to being in the exclusive care of a toxic alienating parent, which is the worst possible outcome. I agree that shared parenting is a great start, but in my opinion, it is not the end. When there is shared custody, there are many anecdotal reports of children simply staying in their room at the targeted parents home and declining to engage. And if I remember correctly, in "Adult Children of Parental Alienation", Amy Baker reported that 6% of parental alienation cases happened in an "intact family". I would bet you never stopped a scared Snd confused 7 year old girl off with a father who leaves her with girlfriends and basically ignored her while blaming her for all of his problems. I really resent psychologists like you - when kids like mine grow up with psychological problems because of court ordered situations like this you know what you will say? You'll say "I'm sorry". You should be sorry - too bad that means absolutely nothing to kids. I believe that the NPD/BPD parent is NOT to be trusted and they are not interested in shared parenting anyway. It's an all or nothing situation for them and is all about control. One must not be naive to think that whilst they are visiting their child they will not be manipulating them. They do not know how to stop. Maybe when the children are much older this might be a safe thing to do when they are old enough to recognise the alienator's dysfunction. I think it is far to dangerous for shared parenting in these kind of severe alienation cases. Hi Susan - I want to thank you for writing a great comment. You articulated the hell that my life has been with a narcissistic ex. I am also happy to write that I am sick to death of reading these ridiculous accounts by these pseudo scientists who refer to nebulous "research studies" that allegedly establish that 50/50 parenting is the best thing since sliced bread. Every time I read some long involved explanation of how "co-parenting" after divorce should be, the overwhelming feeling I always get is this: if I had had my child with a person who is this reasonable after divorce I would never have had to leave this person and go through a nightmare divorce and custody battle in the first place. And another thing - ad a physician I actually can speak with authority and say this - there is straight up no such thing as "parental alienation syndrome". The person who came up with this theory committed suicide shortly after it was discovered that his research findings were completely fabricated (much like the person who presented research "establishing" a link between vaccinations and autism" in the Lancet. Christine, so if your ex is a narcissist, I can see why co-parenting is basically impossible. Some narcissists engage in "pathogenic parenting". Does this sound like what they are doing: https://sites.google.com/site/centralohiopa/summary-of-pathogenic-parenting Many narcissists are able to effectively obtain full custody by completely ignoring court orders and other means. I believe when a normal range parent ends up with no custody, then shared parenting sounds good to them. However, if the narcissist engages in DSM V995.51, child psychological abuse, then I would see it as a child protection issue, probably a temporary one. Few recognize V995.51 when they see it. Hi Howie, that was really nice if you to respond in a calm way to me. My comments to you were aggressive and I'm sorry about that. Whenever someone writes/speaks to me in a kind way about this subject it make me feel good and bad at the same time. Good in the sense that instead of saying - you are crazy, you are the problem, you should try harder- you say yes, these kinds of extreme situations exist. Unfortunately the legal system is more dangerous than dealing with the narcissist. My experience was that my ex's psychological profile clearly pointed out my ex's psych issues but the judge didn't care. I still am not over test and have done everything in my power to keep my ex happy do God doesn't take me back to court. I just want my child to get to 18 as intact as possible so she has a chance at a happy life. I never thought this would be the thing that I would be aiming for in life. It's just so terrible. Thanks for your nice comment though. The alienation does not end when children turn 18 years-old. They become "adults" who speak for themselves and legal strategies shift from family court to civil/criminal court. Custody issues become harassment charges and restraining orders based on gross false allegations being filed by these "adults." So it's not possible to alienate a child from a parent? If the narcissist does this to a parent through the children what do u call it ? At one time the diagnosis you do agree with was once probably called a pseudoscience ? Funny if its not real or doesn't exist how come it's a criminal act in Mexico and Brazil ? Hi Christine, THANK GOD someone has written this!!! THIS (PAS) theory was thrown out long ago by Numerous PhD's in the Academe. That this fruitless, ongoing subject is still being discussed now - Has one asking themselves 'What Social Engineering motives' are really behind this ongoing push of a defeated theory. The studies of the ill effects of PDs on children, and there safety is the element that needs more attention throughout family court worldwide. Maybe if the Australia family court had taken notice of the dangers, instead of have the ambulances at the bottom of the cliffs waiting, we as a country wouldn't seen any more fathers throwing their children of bridges to their death. Thank you for addressing the need for legal and mental health professionals to take the lead on acknowledging and acting upon evidence of parental alienation. However, more than these strangers who encounter our children are needed to prevent, stop, and desist alienating parents from this insidious form of spousal and child abuse. "The sheer magnitude of parental alienation indicates that this is a major social problem . . . " This social problem includes family, friends, neighbors, teachers, and those whom the children know and trust to help them deal with the trauma of parental erasure. The high-functioning, narcissistic, borderline, alienating parent inflicts psychological torture (gaslighting, mobbing, social bullying) on the targeted parent as the children watch in horror. This experience determines the child's fate . . . to succumb to the alienator's programming demands or suffer the same fate as their beloved parent. The silence of those trusted adults in these children's lives are equally culpable for the life-altering trauma that these children experience. Make no mistake. The derailment of parental alienation efforts requires acknowledgement and action by EVERY person in the life of a child suffering from effects of alienating programming and brainwashing. Children, youth, young adults, and even adults deserve the validation of these alienating experiences and responsible action. “The only thing necessary for the triumph of evil is that good men should do nothing.” Dr. Kruk, on behalf of myself and all the other alienated parents, your focus and attention to this very unfortunate situation of Parental Alienation are very much appreciated. That said, I don't think your article goes far enough in its critique of the shortcomings in your profession, and the mental health profession's culpability in this epidemic. To me, as a layperson, it is shameful that the current APA position on Parental Alienation is that it "does not have a position", on whether or not this problem even exits. Given the large and growing problem that PAS creates for families and society, for the APA and other professionally trained therapists to actively try to ignore the problem is (in my personal opinion) less a matter of passive bystanding, and more an issue of willful blindness. While there are precious few mental health professionals such as yourself (and only a few dozen nationally) who are involved in understanding and bringing this epidemic to light, child and family professionals practicing in areas they don't have expertise, tend to make a bad situation significantly worse. Many in the mental health community have never been trained or are not overseen in this area, which has been one of the reasons that this problem has been allowed to grow and become more out of control over these last few years (the legal profession is equally culpable, but at its root PAS is a mental health issue). This is not an attack on you, but intended to argue that the abject failure of an entire industry of supposed medical professionals, is in many cases not innocent indifference. Any improvement will have to come from within the mental health vocation, and PAS victims will be left to suffer. What a great post - you nailed it Neal. This is what all of us target parents are trying to do. We are joining groups on the internet and writing letters to the APA to try and raise MH awareness about this terrible phenomenon and put an end to it's denial and trivialisation. The only way is to get recognition from the MH professionals. Friends, I just joined CAPRA as one of the lead plaintiffs in an upcoming landmark federal class action lawsuit against all 50 States and the Federal Government, because I qualify as: (1) a biological parent whose child custody was unconstitutionally removed without due process; and, (2) I have been directly impacted by that during the last four years, i.e., within the statute of limitations. This class action is on behalf of *both* types of "family court" -- for wrongful victims of divorce-and-similar-with-kids *and* for wrongful victims of child protection services -- and includes suing on behalf of ANY parent affected by either "family court" type. It's totally free to join, and the federal court relief being demanded includes the full restoral of my child custody rights under law, plus a nice share of the large amount in civil damages expected. Although no amount of money could ever replace the lost time, WE MUST FIGHT BACK! It just requires entering someone's referral Code to join, so PLEASE use mine - 80233BM406 - in the CAPRA registration form, located on the bottom of the homepage at http://parentalrightsclassaction.com Plus, check out their power-packed Legal Tips page for all parents fighting family court. REFERRAL CODE 80233BM406 What on earth Susan? As an attorney I have never seen a case like what you are referring to. Also, as an attorney well versed in family law (the 8th circle of hell as far as I am concerned) it's always the parents like you who are victimized by everyone - the judge, the system, your ex, your child- you should post your psychological test you had to submit to during your custody proceeding. I would get my life that a diagnosis of narcissism is lurking throughout the entire result report. I'm on to people like you but lmthank God you exist - I just bought the most beautiful second vacation home in Lake Tahoe because of narcissists like you - thanks! But I feel really sorry for your kids. Poor you - I'm sure none of it your fault. What your article fails to mention is the people that use the term parental alienation as a justification for their initial bad, thoughtless,immoral actions. When a divorce happens due to infidelity and the children are blindsided immediately and forced to accept the OW or OM into their already crushed life, resentment is already there for the parent who is perpetrating this, they are alienating themselves from their children by their rash choices. It is disgusting when the parent who is stable is accused of Parental alienation for justification purposes. The disconnect and faulty argument in this "Anonymous" posting is that "infidelity" happens between a husband and wife and children of any age should not be involved. Period! An alienating parent will usually use infidelity or other marital issues as reasons to encourage their children to reject a loving parent. Those adults who are unable to separate their marital issues from family issues are the ones who put their children at risk for psychological abuse. Parental alienation is the process by which psychologically-impaired parents put their own selfish needs before their children's. They develop a campaign against their children's other parent and drag the children in the middle as a weapons.That is parental alienation, not the "rash choices" made in the context of the marriage. First of all the "selfishness " begins when infidelity happens, why don't we all take a big step back and look at why we are speaking about co parenting, our society is one where marriage, vows, the union of two lives and family are nothing but disposable, everything nowadays can just be replaced with very little care or thought and God forbid there be consequences. If marriage ends and there are no children involved its unfortunate but as soon as children are brought into this world, and mind you they did not ask to be born, we CHOSE to get married and we CHOSE to have children, selfishness needs to be replaced with selflessness. So our goal now is to raise our children in a secure home, teaching them to be kind, moral people, teaching them to hold to their word and commitments, teaching them not to steal, cheat or lie. Now every marriage has its issues, it wouldn't be marriage without ups and downs. Infidelity destroys lives and yes infidelity is between a husband and wife but it in turn destroys a family unit, period! Every case is different but I'm speaking on behalf of the women and men that get left abruptly and the the OW or OM is introduced immediately, thats the definition of selfish. My children and I were blindsided, the same children that were raised to be good, kind, honest and moral. So when this happens and mind you my kids were pre teen, teens, they knew in their hearts that their fathers actions were not appropriate and they refused to accept or and conform to it. THEY chose to not be part of his life, I never told them or coerst them to feel the way they felt. I never made a campaign against him, didnt use them as weapons, instead got them into counseling and told them to follow their hearts. Children have feelings about this and for the parents out there that are alienating, I dont condone it but stop using parental alienation as an excuse to justify bad ,rash choices. Fyi, my boys do not have a relationship with their father and it is because they choose that, they are happy, secure, moral, honest young men. I have an idea, lets all remember what marriage and parenting is, a commitment. Stop turning parents who are not alienating into the villans and take a look at the voices of the children involed, every case is different! I agree wholeheartedly that both professional/institutional forces and individual apathy combine to reinforce the status quo. Two well known quotations on the “Bystander Effect” come to mind: First they came for the Socialists, and I did not speak out— Because I was not a Socialist. Then they came for the Trade Unionists, and I did not speak out— Because I was not a Trade Unionist. Then they came for the Jews, and I did not speak out— Because I was not a Jew. Then they came for me—and there was no one left to speak for me. - Pastor Martin Niemöller “First they ignore you, then they ridicule you, then they fight you, and then you win.” - Mahatma Gandhi The good news is that Parental Alienation has progressed to the “then they fight you” phase using Gandhi’s characterization. Although Parental Alienation was ultimately not included as a specifically named disorder in the last revision of DSM, the intense debate leading up to the decision makes it clear it’s on the radar of mental health professionals... and remains very much on the reform agenda. Likewise, shared parenting as one solution approach to Parental Alienation has recently been embraced by the Social Science community with academic naysayers changing sides or, in some instances, girding themselves for an ideological last stand in a forlorn attempt to save professional face. The battle is far from won, but the tide is definitely turning. Moral courage is required now more than ever for the last big push. Thanks for a great article. Wow - George? Are you my ex? Why is everyone always against you? Gandhi??? Ghandi knows you're a self obsessed person. For you to even put yourself on the same level as the truly oppressed shows your real character. Poor you - noone understands you do they? Don't kid yourself - they do. You just don't like what they see. Thank you for shining some light on this very serious social and mental health issues. Unless the general public have an understanding of what alienation is, they will never take action to help out those in need. Much appreciated. In my experience, there has been very little support from mental health professionals in addressing my antisocial ex. The court awarded him 50% custody after an exhaustive case. He routinely breaks the parenting agreement, denies access during holidays, disparages, 'smears' me to authority figures, and excludes me from my children's lives during his seven consecutive parenting days (denies calls, etc.) It is terribly expensive to take him to court every time he breaks the agreement. Additionally, my children have seen two therapists who've stood by doing nothing to stop his behavior. He's presents himself an intelligent professional, but he is manipulative, calculated, and arrogant in behavior. His agenda is to cause pain in my life and the children seem to be his possessions. I've experienced the "bystander effect". How loud can I cry for help? No one seems to care about verbal and emotional abuse. I am told to fight him in court but I do not have the financial resources, nor are they available to help with legal fees. Ohio Legal Aide will not assist those with a parenting agreement already established. My children and I continue to be traumatized. Sadly, I look forward to their 18th birthdays because I will finally be free from their father. Agreed, because most therapists aid the alienator and therefore harm the child, and because it is the job of the American Psychological Association to fix that, I am joining a demonstration on Aug 4th at the headquarters of the American Psychological Association: Here are two event descriptions for the same event: https://www.facebook.com/events/904259996338780/ https://www.facebook.com/events/1901841500042511/ in 1988 my husband left me for another woman when my children were 4 and 14 months. I was devastated but FOR THE SAKE OF THE GIRLS he had regular visitation, even when he brought his girlfriend with him from the very first time! He used his visitation to entice the children to want to live with him. There were 3 custody battles which I won, without question as the better, more responsible parent. When the girls were 13 and 10 he began yet another custody battle. By this time, I was out of money, my mother refused to support me financially and emotionally and told me, "Let him have them! Leave them alone and they'll come home wagging their tails behind them!" I knew I would never see them again!!!!! My ex instructed my children to run away from home at every opportunity and to tell their teachers that I had been hitting them! I asked for the court's intervention, but all they did was give him MORE visitation. More visitation to create havoc in my life! After having the girls for the entire summer they refused to come home! I HAD NO CHOICE BUT TO ALLOW MY GIRLS TO GO LIVE WITH HIM AND HIS WIFE! My first open school night in my daughter's new school, I read in my 10 year old's note book a composition she wrote, "The Best Thing I Ever Did" "The best thing I ever did was leave my mother and go to live with my father...." I left the school in tears! I never got visitation as even though the court ordered it, my ex said, "They don't want to go, I can't force them!" And the judge never said, "FORCE THEM!" However, the judge did tell me I had to pay CHILD SUPPORT. I paid, $88,000.00 to him when I was single and struggling and he did NOT need the money! Today I am almost 62 years old. My baby is now almost 30 years old and hasn't talked to me since she was 10! My older girl is 32 and has two children of her own. We had a short reconciliation but now she is keeping her children from me as her father once kept her from me. I have PTSD from which I have never recovered. I do not trust anyone. I have no close friends. And if I died, no one would care, except my pets. Seriously. Thank you for this. I stumbled upon this term today and realized that my NPD father did this to me as a child and quite literally broke me as a person, even to this day. This is yet another threat I will unravel.
High
[ 0.6825595984943531, 34, 15.8125 ]
[Primary ileal adenocarcinoma, showing squamous differentiation and ossification]. A 55-year-old man underwent an ileocecal resection because of an intestinal obstruction. The resected specimen contained a sessile tumor near the ileal end. This tumor was 9 x 7 cm in size, with a nodular surface of brown tint. Microscopically, it was a well-differentiated adenocarcinoma, the tumor tissue extending into the muscularis propria. The malignant tissue was associated partially with adenomatous areas and, in malignant nests, with foci of lamellar and intracellular cornification. Further, there was focal ossification in the fibrous stroma. Stains for argyrophil and argentaffin cells revealed strong positive reactions, and immunohistochemical studies were positive for calcitonin and CEA. These findings suggest the multi-potentiality of a small intestinal carcinoma.
Mid
[ 0.6476683937823831, 31.25, 17 ]
Q: Android, How to optionally include stuff based on API level I have an app that opens various sub activies. Whenever the mobile is on API level 11+, I would like to show a button to open an activity that opens a calendar. However, when e.g. designing the activity XML, I get error: View requires API level 11 (current min is 8): activity_calendar.xml Is there any way to markup xml/code files that they should only be avilable/compile-for API level 11... And then write code that based API-level includes the option to show/open the calanedar activity requiring aPI level 11? This is a general question although in my case, it is caused by wanting to use CalendarView. (I know there are some Apache version 2.0 license calendar alternatives which I am currently considering.) A: For code: if (Build.VERSION.SDK_INT > Build.VERSION_CODES.GINGERBREAD) { // do stuff for newer than Gingerbread } For XML /res/layout-v8 Change the versions as you need.
Mid
[ 0.62566844919786, 29.25, 17.5 ]
Bielefeld University of Applied Sciences The Bielefeld University of Applied Sciences (Fachhochschule Bielefeld) is the second largest education institution in Bielefeld. It divides itself into five faculties (Fachbereiche): Divisions Faculty 1: Design (Gestaltung) The Faculty of Design is located in a separate building close to the Oetker Concert Hall as well as a public park called Bürger Park (park of the commons). Three different branches of design and arts are available to the carefully selected students: "Photography & Media", "Graphic and Communication Design" and "Fashion Design". There are accepted about 25-30 freshmen in each branch per year. The school is rated among the top five of Germany's schools offering a focused education in photography, honoured by the state-funded "Special Research Centre on Photography & Media". The school's photography students are also entitled to compete - among the students of other famous European institutions like the Royal College of Arts London and the École Nationale Supérieure des Arts Décoratifs Paris - in the annual Leica Photography Award. Faculty 2: Campus Minden (Architecture, Civil Engineering, Engineering and Computer Science) The Faculty is located in the town of Minden. Faculty 3: Engineering and Mathematics Faculty 4: Social Sciences Faculty 5: Business and Health External links Category:Universities and colleges in North Rhine-Westphalia Category:Universities of Applied Sciences in Germany Category:Bielefeld
High
[ 0.696148359486447, 30.5, 13.3125 ]
Davis went 8-5 a year ago in his first season at FIU, guiding the Panthers to their first bowl game since 2011 and leading them to their first winning record in Conference USA play since joining the league five years ago. There’s some clear momentum going into 2018, though there’s also some significant holes to fill — quarterback being among them. “We’ve got some huge goals for 2018,” Davis said. First, though, they’ve got to make a huge decision at quarterback. The graduation of Alex McGough means someone else takes the reins of the FIU offense, with returnee Christian Alexander and Bowling Green graduate transfer James Morgan vying for the spot. Someone will obviously have to start in the opener against Indiana on Sept. 1, but Davis has floated the possibility that both may play and the race may last through the season’s first couple weeks. “You can’t have a great football team unless you have a really good starting quarterback,” Davis said. “Those two guys, they are competing every single day.” Morgan appeared in 19 games for Bowling Green over the last two years, startiang 13 of them and completing 279 of 538 passes for 3,342 yards and 25 touchdowns. Alexander completed 20 of 34 passes for the Panthers in 2016, and hasn’t thrown a pass in a game since. “Competition helps everyone show the best of their abilities,” Alexander said. “So may the best man win.” The good news is that whichever one wins the quarterback job, he’ll be protected by a veteran offensive line. The bad news is that FIU will have at least 13 players in new starting spots, seven of those on the defensive side. But Davis said the offseason training program FIU put in place is going to help make a lot of transitions go smoothly. “Lot of years of coaching, and I don’t know that I’ve seen a football team work any harder in preparation for the start of the season than this group of guys,” Davis said. “We’re blessed.” Here’s some of what to know about FIU going into 2018: HELLO AGAIN For the first time since 2007, FIU will take the field against Miami — its South Florida neighbor. The teams’ game in 2006 was marred by a brawl that saw 31 players suspended. They played the next year as well, without the fireworks. The Sept. 22 game will be emotionally charged, given how many players on both sides grew up playing with and against each other in the Miami area. And remember, Davis is a former Miami head coach who essentially put together the Hurricane team that won the 2001 national title under Larry Coker. THE SCHEDULE FIU won’t be racking up too many frequent-flier miles this season. The Panthers play seven games at home, an eighth “at” Miami and only leave the state of Florida four times. But there’s not a lot of time to ease into things, with Indiana in Week 1 and the Conference USA opener at Old Dominion the following week. DISCIPLINE FIU wasn’t the most disciplined team a year ago, averaging 6.5 penalties per game. It’ll be a point of emphasis in 2018 since this team figures to have less of a margin for error than the experienced club from last season. RED ZONE If FIU is going to repeat or improve on last year’s eight-win mark, continued excellence in the red zone will be critical. FIU led the nation in red zone efficiency last season. The Panthers were 40 for 41 on their trips inside the opposing 20, with 30 touchdowns and 10 field goals. MORE CANES Davis’ staff has three people who are very familar to the Miami football scene — given their ties to the Hurricanes. Defensive line coach Kenny Holmes and running backs coach Tim Harris Jr. are both Miami graduates, and wide receivers coach Aubrey Hill is a former Miami assistant.
High
[ 0.6666666666666661, 34.75, 17.375 ]
Q: How to put arrows in Owl Carrousel? Is there any way to put navigation arrows in owl carousel by js or html+css? It is working correct, but I think it's no so good you have to drag the image to navigate, therefore a arrow would fix this problem for me. I found some answers through the internet but none of them works for me, when I modify something on the js, the carrousel stops working. <div class="owl-carousel owl-theme" > <div class="item itemmb"> <img class="img-fluid img-card" src="Imagens/dent.jpg"> <h4 class="h4 text-center my-3"> Test </h4> <h4 class="p text-center my-3 sb-m saiba-mais"> Test </h4> </div> <div class="item"> <img class="img-fluid img-card" src="Imagens/ped.jpg"> <h4 class="h4 text-center my-3"> Test </h4> <h4 class="h4 text-center my-3 sb-m saiba-mais"> Test </h4> </div> <div class="item"> <img class="img-fluid img-card" src="Imagens/ped.jpg"> <h4 class="h4 text-center my-3"> Test </h4> <h4 class="h4 text-center my-3 sb-m saiba-mais"> Test </h4> </div> <script type="text/javascript"> $('.owl-carousel').owlCarousel({ loop:true, navigation: true, margin:20, nav:true, responsive: { 0:{ items:1 }, 600:{ items:3 }, 1000:{ items:5 } } }) </script> A: It's because of the responsive initialization.If you don't have enough items there won't be any arrow or dots dispayed $('.owl-carousel').owlCarousel({ loop: true, margin: 20, nav: true, responsive: { 0: { items: 1 }, 600: { items: 1 }, 1000: { items: 1 } } }) <link href="https://cdnjs.cloudflare.com/ajax/libs/OwlCarousel2/2.3.4/assets/owl.carousel.css" rel="stylesheet" /> <script src="https://code.jquery.com/jquery-3.3.1.min.js"></script> <script src="https://cdnjs.cloudflare.com/ajax/libs/OwlCarousel2/2.3.4/owl.carousel.min.js"></script> <div class="owl-carousel owl-theme"> <div class=" itemmb"> <img class="img-fluid img-card" src="http://www.windmillsonline.co.uk/wp-content/uploads/2013/03/img-banner-thefoundation.jpg"> <h4 class="h4 text-center my-3"> Test </h4> <h4 class="p text-center my-3 sb-m saiba-mais"> Test </h4> </div> <div class=""> <img class="img-fluid img-card" src="http://www.windmillsonline.co.uk/wp-content/uploads/2013/03/img-banner-thefoundation.jpg"> <h4 class="h4 text-center my-3"> Test </h4> <h4 class="h4 text-center my-3 sb-m saiba-mais"> Test </h4> </div> <div class=""> <img class="img-fluid img-card" src="http://www.windmillsonline.co.uk/wp-content/uploads/2013/03/img-banner-thefoundation.jpg"> <h4 class="h4 text-center my-3"> Test </h4> <h4 class="h4 text-center my-3 sb-m saiba-mais"> Test </h4> </div> </div>
Low
[ 0.505681818181818, 22.25, 21.75 ]
Q: Is it possible to access a UI element from another thread if the thread doesn't modify that element? Let's assume that the code running in the thread that instantiated the form/control/element (usually the main thread) does not modify/access that element at the same time, is it possible to: get the Text property of a TextBox. enumerate a ListView. subscribe to a Form's Closing event. (Knowing that the hook will be called from the thread that instantiated that form) I have tried all 3 and the program doesn't seem to complain about it. I had always thought that you had to invoke any call that wants to even remotely touch anything UI related (read or write). I understand very clearly why I need to use the IsInvokeRequired/Invoke pattern when modifying an element, but I cannot see why accessing properties/events would cause any problems. A: It's definitely possible, however, it could lead to unexpected behaviour. Also, other thread-related bugs also need to be taken into consideration e.g. race conditions/deadlocks see Managed Threading Best Practises. I would always stick to accessing the UI on the UI thread to be on the safe side. A: And what are you doing to ensure that the UI thread isn't modifying the control while you're reading from it? The whole reason for marshaling to the UI thread is so that you don't need to worry about that case. Enumerating the listbox in particular is going to be the easiest to break as it will take the longest time (this creating the largest window for a race condition). You should be marshaling to the UI thread for all 3 of those things.
Mid
[ 0.585987261146496, 34.5, 24.375 ]
Get the latest Swans news sent straight to your inbox Sign up Thank you for subscribing We have more newsletters Show me See our privacy notice Invalid Email Swansea City are reportedly targeting Wales international Hal Robson-Kanu - but want to haggle down Reading's £4m valuation. Winger turned striker Robson-Kanu, 26, has just one year left on his contract at the Madejski Stadium and is in hot demand having played a significant part in helping Wales top qualifying Group B for Euro 2016 and surge into the top 10 of FIFA's rankings. The former Arsenal FC trainee has 23 caps and two goals for his country and played in the Royals team that lost out to Arsenal in last season's FA Cup semi-final. More:Live Transfer News and Gossip Mirror Football say the Swans have tabled at least two bids already but Reading are holding out for £4m. If Robson-Kanu does not get a move this summer, he is likely to let his contract expire and move on a free next summer. More:Wales 10th - What a farce! More:Download our Fantasy Football app
Low
[ 0.5011337868480721, 27.625, 27.5 ]
The day before, Ryan was spotted filming scenes with a female co-star for his new movie Selfless, which is about an extremely wealthy elderly man dying from cancer who undergoes a radical medical procedure that transfers his consciousness to the body of a healthy young man. He starts to uncover the mystery of the body’s origins and the secret organization that will kill to keep its secrets.
Low
[ 0.50592885375494, 32, 31.25 ]
‘Nigeria’s total dependence on oil suicidal’ Imagine waking up one day to discover that Nigeria’s oil wells have dried up or that all her customers now produce oil or that fossil fuel is no longer to be used in order to save our planet! What does Nigeria’s future look like when this happens? How can Nigeria transition from fossil fuel to clean energy? These and more were the questions the Green Deal Nigeria (GDN) team, an initiative of the Heinrich Boll Foundation, sought to answer at a recent Press conference in Lagos to intimate the Press on what GDN is all about. The event featured three presenters – Bloggers Japheth Omojuwa, Mercy Banku Abang-Asu and Azeenarh Mohammed who spoke on different aspects of the economy and the way forward. Japheth Omojuwa who spoke on Nigeria’s oil sector decried a situation where Nigeria spends whatever accrues from oil instead of using it as a seed to grow the economy. “We should use whatever we get from oil as a form of seed to develop our agriculture, renewable energy, etc.” Oil sector losses: He said that in 2009, Nigeria got $25b from oil instead of about $86b, because “we sold crude worth about $86b but we were only able to access $25b because of certain factors which include a defective tax process, middlemen, swop etc. Said Omojuwa; “The Federal Inland Revenue Service (FIRS) does not have the capacity to tax these multinational companies. They go to them to ask them what their production is, and based on that, they are taxed. “NNPC is supposed to make as much as $5b – $7b from sale of crude in Nigeria, but it loses money instead. Algeria’s Sonatrac made about $2.5b while NNPC lost about $2.5b. Thirdly, we deal with marketers in the sale of crude which means that some of the money they are making, we as a people should have been making. “There is also the swop issue. Conventionally, it is not bad if what we swop is very low quality crude for refined products like some countries do. But we swop the best crude in the world for refined products so we are losing money. There is also corruption and inefficiency in the process. Again, we don’t have the platforms. For over 50 years of oil production, we are unable to supply the major technical know-how and the platforms so they are left for the multinationals.” When the oil wells dry up: Omojuwa maintained that even if Nigeria’s oil wells don’t dry up, certain realities will occur. “Right now, China, our potential biggest customer, has abundance of shale gas. US, our biggest customer, is decreasing its import and it has been shown that US will surpass Saudi Arabia in oil production by 2020. If other countries have options and oil is being discovered in other parts of Africa, it will no longer be as politically powerful as it is today. There is also the issue of climate change. The argument for the planet to be responsible for its climate has been increasing for obvious reasons. Last year, Nigeria experienced one of its worst flooding in over 50 years and this year, there are expectations that it will be more, so it means that as a planet, whether we like it or not, our need to survive means we have to look for alternatives.” PIB and gas flaring: He said that gas flaring is costing Nigeria about $2.5b “so if we are able to tap it, we will be getting as much as $2.5b.” He regretted that the current PIB is not taking care of this problem. The Director, Nigeria & West Africa Office of Heinrich Boell Foundation, Christine K harped on the need for the gas issue to be resolved or else “the National Gas Company which has no interest in regulating the sector, will be run like the NNPC.” Omojuwa advised that market forces should be allowed to determine what happens in the oil sector like in telecom sector, noting that the only thing the telecom sector did wrong was the the non-production of phones in Nigeria. “We propose that there should be a federal energy commission to take care of all our energy needs which will be linked with the Ministry of Environment because everything that happens in this industry affects the environment so you have one holistic approach to energy development.” Agriculture/Climate change: Mercy Abang-Asu dwelt on Agriculture and Climate change and what must be done to enable Nigeria feed her estimated 255 million population by 2030 despite ravages of climate change. “Farmers and fishermen have experienced challenges they have never faced before. With 60% of Nigerians in the agric sector, this is a threat, yet most of them still rely on yesterday’s knowledge. She said Nigeria has about 40 million hectares of uncultivated arable land and all she needs is to provide the farmers with better information through research, training and improved seeds. “A situation where we have challenges of climate change, having only 5% of farmers having access to improved seeds is a threat,” she stated. Renewable Energy: Azeenarh Mohammed on her part, focused on renewables. “According to scientists, if the seven billion people in the world today all live the life of the global north, we will have the carbon footprint of 18 billion people so the challenge is to find a responsible way of living without destroying the planet.” She wondered why despite Nigeria’s abundant fossil and renewable energy options, majority are still suffering from energy poverty which undermines their aspirations and lowers their quality of life. “Instead of flaring our gas, we can turn it into electricity. Unfortunately, as at today, nobody knows who owns the associated gas that comes with oil. The new PIB is also silent on that. “Figures from the Energy Commission (EC) shows that it costs about N200m to build a small hydro dam for a community that has a stream and a local government can afford to do that and give the people electricity 24/7 so why don’t they do it? Some of these local governments receive as much as N300m monthly,” she said.
Mid
[ 0.648648648648648, 33, 17.875 ]
The invention relates to multi-computer systems. More particularly, the invention relates to methods and equipment for recovery processing in the event of a computer failure. Multi-computer systems employ a plurality of computers. A single computer runs a single kernel, whereas a multi-computer runs multiple kernels. As so defined, a single computer may include one or many processors. The constituent computers in a multi-computer may be in distinct physical units (e.g., chassis or circuit boards) or individual processors together in the same physical unit or some combination of both. One example of a multi-computer system is one in which redundant computers are present. The use of redundant devices in computer systems is advantageous in critical applications because the redundant devices increase system availability, survivability, and robustness. In a multi-computer systems one or more computers may be redundant computers able to take over processing workload abandoned by a primary computer that has failed. A redundant computer may be connected to peripheral storage devices (e.g., disk drives) through separate hardware paths than used to connect a primary computer to the peripheral storage devices. A redundant computer may be inactive unless and until the primary computer fails. Alternatively, multiple active computers in the same scalable computer system may be redundant with respect to each other in the sense that one of the computers can takeover execution of an application program (also called simply xe2x80x9capplicationxe2x80x9d) originally executed by computer that subsequently fails. A computer stores data to peripheral storage by issuing one or more xe2x80x9cwrite requestsxe2x80x9d (sometimes simply referred to as a xe2x80x9cwritesxe2x80x9d) to a peripheral storage device. Typically write operations are asynchronous, i.e., the computer issues a write request and is notified of its completion some time later. A busy computer may have multiple writes outstanding at the same time. Once issued, a write request may be delayed, perhaps indefinitely. Sources of indefinite delay include failure of communications channels between the computer and the storage device, and failures within the storage device itself. Normally, a computer xe2x80x9ctimes outxe2x80x9d such writes and reissues them if necessary. Failing receipt of a write completion acknowledgment, the requesting computer typically records the failed state of the peripheral storage device. When an operating computer fails, it may have pending writes that have been issued but neither completed nor timed-out. When a pending write requests from a failed computer is effectuated (i.e., actually written on a storage device), the phenomena is referred to herein as a xe2x80x9cghost write.xe2x80x9d That is, a ghost write is effectuated on behalf of a dead computer. A ghost write may seriously interfere with recovery processing whereby a redundant computer takes over for the failed computer. When an operating computer fails, a recovery routine is executed to transfer application programs (also called, more simply, xe2x80x9capplicationsxe2x80x9d) to a redundant computer. An application is typically terminated and then restarted on the redundant computer. Recovery of an application following unexpected termination usually involves reading peripheral storage utilized by the application and analyzing the data to determine the state of the application program at the time of the failure. If necessary, the state of the peripheral storage devices is altered so as to eliminate partially completed writes. The objective of the alteration is to back up in time to a known good, consistent state, because partially completed writes place the storage devices contents into an inconsistent state. A ghost writes can interfere with the recovery processing by either (1) corrupting the data read from the peripheral storage or (2) overwriting corrections made for the purpose of restoring consistency. In one respect, the invention is a method for preventing ghost writes in a multi-computer system. A first computer in the multi-computer system issues one or more write requests to a storage device, each write request normally being effectuated after being pending for a time. The method generally comprises the steps of detecting a condition indicative of a failure associated with the first computer and preventing the effectuation of write requests issued by the first computer to the storage device and pending at the time of the detected condition. In one embodiment, the condition indicative of a failure associated with the first computer comprises at least one of the group consisting of a reduction in the number of known operating computers in the multi-computer system, and a state change of a volume group associated with the computer on the storage device from inaccessible to accessible and a reduction in the number of known operating computers in the multi-computer system. In another embodiment, the failure is a communication failure associated with one or more write requests to a storage device connected along a plurality of redundant communications channels. In yet another embodiment, the preventing step comprises at least one action from the group consisting of issuing a fibre channel target reset on a fiber channel connected to the storage device, issuing a fibre channel remote logout on a fiber channel connected to the storage device, issuing a fibre channel remote logout and a requiring logout from multiple fibre channels on a fiber channel connected to the storage device, and issuing a bus device reset on a bus connected to the storage device. In other respects, the invention is computer software embedded on a computer readable medium. The computer software comprises instructions for implementing the methods just summarized. In yet another respect, the invention is an apparatus. The apparatus comprises a first computer, a storage device, at least one adapter connected between the first computer and the storage device, and an application executing on the first computer. The adapter comprises a protocol capable of selectively eliminating write requests issued to the storage device before effectuation of the selected write requests. The application comprises an activation procedure that executes upon startup of the application. The activation procedure is connected to the adapter and commands the adapter to eliminate uneffectuated write requests. In one embodiment, the activation procedure executes following at least one event from the group consisting of a reduction in the number of known operating computers in the multi-computer system, and a state change of a volume group associated with the computer on the storage device from inaccessible to accessible and a reduction in the number of known operating computers in the multi-computer system. In another embodiment, the activation procedure performs at least one action from the group consisting of issuing a fibre channel target reset on a fiber channel connected to the storage device, issuing a fibre channel remote logout on a fiber channel connected to the storage device, issuing a fibre channel remote logout and a requiring logout from multiple fibre channels on a fiber channel connected to the storage device, and issuing a bus device reset on a bus connected to the storage device. In comparison to the prior art, certain embodiments of the present invention are capable of achieving certain advantages, including the ability to recover more satisfactorily from a computer failure when there is the potential for ghost writes. Those skilled in the art will appreciate these and other advantages and benefits of various embodiments of the invention upon reading the following detailed description of a preferred embodiment with reference to the drawings.
Mid
[ 0.620192307692307, 32.25, 19.75 ]
Q: Reading file on system startup from registry [C++] I want my program to read a config file which is right next to the "exe file. That's working properly, but when the program starts with system from run folder in registry (HKEY_CURRENT_USER), it simply can't read the file. The exe file and the config file are in one folder in "Program Files(x86)". Here is a snippet of a code starting on program startup: TCHAR szPath[MAX_PATH]; GetCurrentDirectory(MAX_PATH, szPath); char filepath[MAX_PATH]; sprintf(filepath, "%s%s", szPath, "\\data.cfg"); if(!(file = fopen(filepath, "rt"))) { MessageBox(hwnd, "Could not load config file", "ERROR", MB_OK); } else { fgets(ReadLine, 20, file); sscanf(ReadLine, "#KEYSTROKE %s", &ReadChar); fgets(ReadLine2, 20, file); sscanf(ReadLine2, "#STARTUP %s", &ReadChar2); fgets(ReadLine3, 20, file); sscanf(ReadLine3, "#CTRL %s", &ReadChar3); fclose(file); } Any help would be appreciated. Thanks in advance. A: You should use GetModuleFileName() and extract folder info from there.
Mid
[ 0.569047619047619, 29.875, 22.625 ]
WELCOME TO MY YOGA WEBSITE. I hope to make people understand that anyone can practice yoga. It can be done from a chair. You don't have to be able to make your body into a pretzel. You don't have to be model thin. You don't have to make a lot of time for it. All you have to do is become aware of your breath, slow down from your daily life and your practice begins. Namaste. Pages Thursday, April 14, 2011 I am teaching today at The Yoga Source on Wiles Rd in Coral Springs. I have to take some pictures of the studio and put them up here. It is such a wonderful peaceful place not only to teach but also to take a class. I feel as if I am able to breathe deeper just by walking in the door. I love my 4:30 class. It is a weird time and it's not the largest class that they have at the Yoga Source but the students that come are really into it. Most of them are people who are just off work and they need to come down off the wall a little. I always make sure there is time for pranayama (breath control) even if it is just a few minutes. Pranayama helps us come in contact with our bodies and become aware of the changes in the body from moment to moment. It also allows us to slow down and really breathe deeply. I can't even think of starting a class without pranayama. I usually start on the back but maybe I'll start in seated today. It's always good to shake things up a little. I am going to do some balance postures today. I have been neglectful with balance lately. It is not my strong suit so I tend to not do those postures but I need to be more mindful about incorporating balance postures in my classes. I'm thinking today I will do tree pose and dancers pose. I may even bring in standing stick. I'll see. I always end my classes with by rubbing lavender essential oils in my hands and walk to each student and place my hands over their faces so that that can smell that wonderful relaxing small. I do that in my home classes as well. It always helps the students to get in the mode of letting go. Tuesday, April 5, 2011 Yesterday at about 5:20 pm my cell phone rang with an unfamiliar number. I picked it up and it was Collette from the Yoga Source. She told me she had recently been in a car accident and wasn't feeling well. She then asked me if I would sub her 6:00 class. I wildly thought what kind of classes she taught when she told me that my students would be "upstairs". Upstairs is the hot yoga room. Then I realized that she was one of the hot yoga teachers. She sounded so desperate that I decided to do it. Yep, I taught my first hot yoga flow class and it was quite the unusual experience. I introduced myself and one of the students asked me who I was and where was Collette? Sandee who is also a hot yoga flow teacher came to me before class and told me that this person might very well get up and leave and not to be upset. He was a difficult student who wanted what he wanted. I told him my name and that I was subbing this class. He said "I don't know you. Where is Sandee?" I told him that Sandee was downstairs teaching a class right now and maybe he wanted to go there. He agreed and left. It really didn't bother me even a little bit. That happened to me at Juliana's and I've already realized that if someone leaves your class it's their problem not yours. I was kind of glad that I was able to get him to go. I started the class in seated so pranayama was short. I was already sweating my butt of after pranayama. We did some seated postures and then I brought them into downward dog. Sun salutations and then everything you could think of from there. Knees chest chin, cobra, warrior, arm sweeping just flowing from one move to another. I just kept the class moving. About 50 minutes into the class I started having trouble. I found it difficult to breathe. I decided to slow it down and bring them into some postures to hold. We did some forward bending and then after a vinyasa I brought them down to the belly. We did locust, flying locust, cobra, bow and anything else I could think of to stay on the belly. I brought them down to the back at about 7:10 and did some knees to chest and basic stuff. Then relaxation complete with lavander essential oils. All of the students thanked me and said that my class was exactly what they needed. That was nice to hear. I had made it through my first hot yoga flow class and I was a complete success. I am pretty damn proud of myself. :)
Mid
[ 0.6083743842364531, 30.875, 19.875 ]
Chicago (AFP) – The chief executive of embattled United Airlines unequivocally apologized Tuesday for an incident in which a passenger was dragged off a plane, and promised a thorough review of the airline’s practices. The apology came after a torrent of criticism of the carrier’s action on a flight Sunday and its initial explanation of it. In images now seen around the world, a passenger was forcefully removed and bloodied in the process — the entire event captured on video by passengers and posted on social media. The 69-year-old passenger had refused to be “bumped” off the overbooked flight — an airline practice that has come under increased scrutiny since the incident. “I continue to be disturbed by what happened on this flight and I deeply apologize to the customer forcibly removed and to all the customers aboard,” CEO Oscar Munoz said. “I want you to know that we take full responsibility and we will work to make it right.” The comments were in stark contrast to the company’s initial response, in which it seemed to at least partially blame the passenger, inflaming worldwide outrage. US media published an email Munoz sent earlier to employees, in which he said the passenger “defied” authorities and “compounded” the incident. “Our employees followed established procedures for dealing with situations like this,” the CEO wrote. – ‘No compassion or concern’ – Andy Holdsworth, a crisis management specialist at the British PR firm Bell Pottinger, said United’s initial response focused on the wrong thing. “Whilst the passenger’s behavior was not good, United have shown no compassion or concern for the man,” he said. Munoz said Tuesday that the company will conduct a “thorough review” of its procedures, including “how we handle oversold situations” and how the airline partners with airport authorities and law enforcement. He promised to release the results of the review by April 30. But the public relations damage was done, with calls for boycotts, the US Department of Transportation promising a review of the airline’s actions, and even the White House weighing in. “Clearly, when you watch the video, it is troubling to see how that was handled,” White House Press Secretary Sean Spicer said. The furor battered United’s stock Tuesday, sending it down 2.9 percent in afternoon trading and closing down 1.1 percent. It was the second time in about two weeks the airline found itself in the middle of a firestorm. In late March, two teenage girls were prevented from boarding a flight in Denver because they wore leggings. The airline defended its action at the time by saying the girls were flying on passes that required them to abide by a dress code in return for free or discounted travel. “They will need to be careful that these small incidents all start to add up and only remind us of the last incident as well as the current one,” Holdsworth said. – ‘You don’t really have any rights’ – The passenger on the overbooked Sunday flight from Chicago to Louisville, Kentucky was one of four involuntarily bumped off in order to make room for United crew that needed to be repositioned. The incident shined a new light on the practice of overbooking and bumping passengers off flights, which airlines increasingly rely upon to avoid losing money on empty seats when some passengers do not show up for scheduled flights. If they were to stop overbooking, “the only way of trying to compensate for that over the long term would be to raise fares on everyone else,” said industry analyst Robert Mann. Instead, airlines sell more tickets than there are seats on a plane, and are generally able to properly forecast demand to avoid major disruptions in getting passengers to their destinations, Mann said. But, sometimes, they miscalculate and there are more passengers than a flight can handle. In those instances, airlines offer travel vouchers and cash compensation to entice passengers to voluntarily give up their seats for later flights. When enticing does not work, airlines have wide latitude under the law. “If you’re still in the terminal waiting to board, you can be told you can’t board, even if you have a reservation,” Mann said. “And once you’re on board, you are subject to being deplaned based on the order of the crew. So you don’t really have any rights.” Last year, 434,000 passengers volunteered to be bumped off flights, while another 40,000 were bumped involuntarily and compensated.
Mid
[ 0.5650224215246631, 31.5, 24.25 ]
tag:blogger.com,1999:blog-7941261488380355005.post2962002046537190025..comments2015-03-30T10:25:51.448-07:00Comments on Raising Matt Cain: Pat the BatM.C. O'Connorhttp://www.blogger.com/profile/[email protected]:blogger.com,1999:blog-7941261488380355005.post-64922410283964155252010-08-01T17:52:23.821-07:002010-08-01T17:52:23.821-07:00Matt MUST beat these guys! How can he ever really...Matt MUST beat these guys! How can he ever really be a great Giant if he never beats them!! Come on 18 starts is not exactly a small sample size. I know we can all make excuses about run support and &quot;luck&quot; but I don&#39;t want to hear it. Even if he has to throw a shutout AND hit a homer, Cain MUST beat these assholes!JC Parsonshttp://www.blogger.com/profile/[email protected]:blogger.com,1999:blog-7941261488380355005.post-8072666336903882882010-08-01T15:43:36.780-07:002010-08-01T15:43:36.780-07:00Yeah, Freddy&#39;s been soft swingin&#39; and miss...Yeah, Freddy&#39;s been soft swingin&#39; and missin&#39; for a while now. <br /><br />I guess that the Padres felt that they really needed a hitter so they got a little creative. Another hitter would have been swell, however, I&#39;m guessing most of the other teams see us as the pitchers store. I wouldn&#39;t want to lose Dirty or Madbum for a hitter. <br /><br />If our starters keep pitching good, if our bullpen can be a little more stable, and our offense can provide enough runs, I&#39;ll feel fine. <br /><br />The Yankees had a pretty bad bench so I guess adding Kearns and Berkman fixed that up for them. Kerry Wood they figured could do as good a job as Park if not better.<br /><br />I must say though, if our pitching holds up and our guys keep playing good, we won&#39;t need the additional hitters. Pitching wins it in the long run, especially strikeout pitchers. Timmeh and Dirty are K machines, as well as most of the bullpen. Walks I feel are over exaggerated. It&#39;s only worth one bag. The Giants give up the 2nd fewest hits in baseball. I believe pitchers have a bigger role in hits than people think. Hits win games, walks not as much. <br /><br />Last year, the Phillies could have won. Had they played Lee a game earlier, they would have had more momentum. They should have had more faith in J.A. Happ. I feel that he would have done better than Blanton. Poor faith and management in the pitching department led to the Phillies losing pretty easily without Cliff Lee. Good pitching silences good hitting and if we can bring some of our good pitching into the postseason, we have a real [email protected]:blogger.com,1999:blog-7941261488380355005.post-90981220140770066862010-08-01T14:39:08.302-07:002010-08-01T14:39:08.302-07:00I&#39;m a little surprised the Padres were able to...I&#39;m a little surprised the Padres were able to get a good player so easily, and it makes me wonder why the Giants could not. Ludwick would have been a perfect fit, and could have been added to the team for less than the two relievers cost us. I know we need the bullpen help, but another hitter would have been nice. We just have to hope that The Big Three--Posey, Huff, Torres--don&#39;t slow down at all and that Sandoval can find his 2009 form.<br /><br />I realize the Giants are a good team and playing well, but take a look at the Yankees. They are the team with the BEST RECORD IN BASEBALL and they SCORE THE MOST RUNS and they added TWO hitters (Berkman and Kearns)!! How is it that they feel they need MORE offense and the Giants feel that they DON&#39;T need more offense? Explain, please. <br /><br />Also, I&#39;ve had enough of FSanchez batting 2nd. That&#39;s ridiculous--there&#39;s nothing about his bat that justifies putting him up so high in the lineup. We&#39;d be better off with Sandoval, Burrell, or Uribe in the top four.M.C. O'Connorhttp://www.blogger.com/profile/[email protected]:blogger.com,1999:blog-7941261488380355005.post-32710276108860255232010-08-01T14:26:09.719-07:002010-08-01T14:26:09.719-07:00It&#39;s time to shut the door on these assholes. ...It&#39;s time to shut the door on these assholes. I still can&#39;t believe half of the trades that they made. The scary thing is, I could see Sabean making those moves if the Dodgers didn&#39;t beat him to the punch. Ted Lilly is having a down year according to FIP (and even his record could tell you that), Scotty&#39;s been a bit lucky but isn&#39;t the best fielder, Theriot has a cool name but he will probably make them worse. Octavio will probably be about average for them. I know I said this yesterday but it just makes me feel good every time I think about it. <br /><br />Go [email protected]:blogger.com,1999:blog-7941261488380355005.post-53964325748939854882010-08-01T13:20:09.473-07:002010-08-01T13:20:09.473-07:00I agree it&#39;s time for Matty to beat these guys...I agree it&#39;s time for Matty to beat these guys.Greg Wurzhttp://www.blogger.com/profile/[email protected]
Mid
[ 0.566929133858267, 36, 27.5 ]
Acute respiratory distress syndrome update. Respiratory failure and eventual ventilator dependence are hallmarks of the acute respiratory distress syndrome (ARDS). ARDS carries a significant mortality in all patient populations and the treatment strategies are largely supportive in nature. Despite years of clinical experience, ARDS continues to pose a substantial threat and no definitive therapy has yet to be described. We will present an overview of management options.
High
[ 0.693150684931506, 31.625, 14 ]
I think to use DB2/CLI from the i to access a MS Sequel Server, you must have a DRDA acces provider installed. This is notoriouly difficult to do, as there do not seem to many products that can provide this support. Generally, the standard alternative approach is to use a Java based JDBC driver (type 3?) to connect to the remote server. Scott Klement has published several articles covering this. can anyone help me with the connection parameters to access the MSSQL data base using SQLCLI? i have been searching all day and the only example i found for SQLCLI was for attaching to another AS400 data base. in the example i have been trying to work with, i have the following connect statement: rc = SQLConnect(hdbc : szDSN : cbDSN : szUID : cbUID : szAuthStr : cbAuthStr); cbDSN = SQL_NTS i have changed szDSN to the name of the data base (which i am able to access via ms access odbc driver) but i do not know how to tell it the location (ip address) of the server that the MSSQL data base resides on. This mailing list archive is Copyright 1997-2015 by MIDRANGE dot COM and David Gibbs as a compilation work. Use of the archive is restricted to research of a business or technical nature. Any other uses are prohibited. Full details are available here. If you have questions about this, please contact
Low
[ 0.5053763440860211, 35.25, 34.5 ]
Q: Array indexing triggers repeated evaluation of the element? I am writing a program to generate first k numbers of the form , where a and b are non-negative integers. The algorithm I am using exploits the fact that every number in this series is generated from adding either 1 or sqrt(2) to a previous number in the series, with the series starting at 0. We'll collect the numbers in the series in an array where each element is expected to be evaluated lazily on demand. The first number in the series is 0 so we'll initialize the first element of the array with 0. We'll maintain two pointers i and j both starting at index 0 (first number in the series). The next number in the series is min(A[i]+1, A[j]+sqrt(2)). If the next number in the series comes from pointer i then pointer i will be incremented (as we never want to add 1 to A[i] again) and similarly if the next number in the series comes from pointer j then j will be incremented to point to next index i.e j+1. Both i and j will be incremented if A[i] + 1 = A[j] + sqrt(2). I think the snippet below captures the above algorithm, however, upon running the code in ghci, it looks like function f is being called repeatedly with parameters 1 0 0 and the program never finishes. I don't understand how this call is being made although it seems like the call is being triggered by v A.! i. import qualified Data.Array as A import Debug.Trace compute :: Int -> Int -> Double compute x y = (fromIntegral x) + (fromIntegral y) * (sqrt 2) firstK :: Int -> [(Int,Int)] firstK k = A.elems v where v = A.listArray (0,k-1) $ (0,0):f 1 0 0 f c i j | c == k = [] | otherwise = traceShow (c,i,j) $ let (ix,iy) = v A.! i (jx,jy) = v A.! j iv = compute (ix+1) iy jv = compute jx (jy+1) in if iv < jv then (ix+1,iy):f (c+1) (i+1) j else if iv > jv then (jx,jy+1):f (c+1) i (j+1) else (ix+1,iy):f (c+1) (i+1) (j+1) Output - λ> firstK 50 (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) (1,0,0) ... ... A: chi's answer discusses in detail the exact problem with your code, and the fix that would let you keep your implementation idea but get the answer you're hoping for. But I think it's also interesting to see how a native Haskell speaker would approach the same algorithm: I'm going to just use native lists, and instead of a pointer into an array I'll use the list's pointers themselves to track where we are. It looks like this: vs :: [(Int,Int)] vs = (0,0) : merge [(a+1, b) | (a, b) <- vs] [(a, b+1) | (a, b) <- vs] where compute (a,b) = fromIntegral a + fromIntegral b * sqrt 2 merge xs@(xh:xt) ys@(yh:yt) = case compare (compute xh) (compute yh) of LT -> xh:merge xt ys EQ -> xh:merge xt yt GT -> yh:merge xs yt In the recursive call to merge, using xt (instead of xs) is akin to incrementing your i pointer; using yt (instead of ys) is akin to incrementing your j pointer. One nice thing about vs is that you don't have to declare ahead of time how many pairs you want -- you just get a lazy infinite list of them (up to rounding issues with using Double for compute of course!). The code is also significantly shorter. Here's an example run in ghci: > take 10 vs [(0,0),(1,0),(0,1),(2,0),(1,1),(0,2),(3,0),(2,1),(1,2),(4,0)] A: The issue is that arrayList forces all the spine of the list too early, before the array is actually built. That makes the recursion loop forever. We can, however, fix that using a helper function as follows. -- A spine-lazy version of arrayList arrayListLazy :: (Int, Int) -> [a] -> A.Array Int a arrayListLazy (lo,hi) xs = A.array (lo,hi) $ go lo xs where go i _ | i > hi = [] go i ~(e:ys) = (i, e) : go (succ i) ys firstK :: Int -> [(Int,Int)] firstK k = A.elems v where v = arrayListLazy (0,k-1) $ (0,0):f 1 0 0 f c i j | c == k = [] | otherwise = traceShow (c,i,j) $ let (ix,iy) = v A.! i (jx,jy) = v A.! j iv = compute (ix+1) iy jv = compute jx (jy+1) in if iv < jv then (ix+1,iy):f (c+1) (i+1) j else if iv > jv then (jx,jy+1):f (c+1) i (j+1) else (ix+1,iy):f (c+1) (i+1) (j+1) Small test: > firstK 10 [(0,0),(1,0),(0,1),(2,0),(1,1),(0,2),(3,0),(2,1),(1,2),(4,0)]
Low
[ 0.512087912087912, 29.125, 27.75 ]
This invention relates to a system for inspecting the surface of a moving aluminum surface. More particularly, the invention relates to a system for automatically detecting flaws in the surface of a moving object and storing a visual image of the flaw for inspection thereof. Strobed illumination or electronic or mechanical shuttering are used to arrest surface motion to provide an undistorted view of surface characteristics.
High
[ 0.6666666666666661, 33.75, 16.875 ]
New mutations in the ataxia telangiectasia gene. We report the detection of four new mutations in the ataxia telangiectasia gene (ATM). Reverse-transcribed RNA extracted from cultured cells was analysed for mutations by polymerase chain reaction amplifications and restriction endonuclease fingerprinting. Three deletions and a base substitution are described. The deletions reported here would result in severe disruptions of the ATM gene product by leading either to a protein truncation (a 4-bp deletion) or the loss of stretches of 53 and 58 amino acids (a 159-bp deletion and a 174-bp deletion, respectively); whereas the base substitution would lead to an amino acid change from a highly conserved glycine to an arginine residue.
High
[ 0.6599496221662461, 32.75, 16.875 ]
INTRODUCTION ============ The facial nerve is the motor nerve of facial expressions and loss of its function leads some aesthetic and emotional problems. Nerve has a long and complicated course from the brainstem to the facial musculature. As well as most of the otological and otoneurological procedures, surgeries of the parotid gland and the face are conducted around the facial nerve. So that, it is vulnerable to iatrogenic injuries. According to the severity of traumatic insult, affection of function may differ from slight disfigurement to total loss of function \[[@b1-ceo-2016-00997]\]. Insulin-like growth factor 1 (IGF-1) is a naturally produced peptide which is shown to be synthesized within the brain and peripheral nerves. It is also found to be a trophic factor for various tissues. IGF-1 receptors were shown in neural tissues of reptiles, amphibian, and mammals such as rats, rabbits, and chicken. IGF-1 stimulates the development and growth of neurons and glial cells \[[@b2-ceo-2016-00997]-[@b4-ceo-2016-00997]\]. Moreover, IGF-1 was shown to promote neurite outgrowth and neuronal survival. The expression of IGF-1 in the nervous system was investgated by immunohistochemical techniques in rats. Following crush injury of a motor nerve, accumulation of IGF-1 immunoreactive material within the damaged area was shown invitro by Nachemson et al. \[[@b5-ceo-2016-00997]\]. Regeneration of the peripheral nerves after injury seems to be enhanced by the release of trophic factors. These factors stimulate the sprouting of axons as well increased survival of neurons. IGF-1 is one of these factors. Among these molecules, IGF-1 seems to be promising. Schwann cells of the uninjured motor nerves express a little amount of IGF-1. However, after a traumatic injury, IGF-1 concentrations increase, and it is excreted into the extracellular space. It exerts an insulin-like and trophic effect not only on the neural tissues but also on muscle and connective tissues. It is found to accelerate myelination and axonal regrowth *in vivo* \[[@b5-ceo-2016-00997]\]. The aim of this study is to investigate the efficacy of locally administered IGF-1 on the recovery of facial nerve functions after a crush injury. MATERIALS AND METHODS ===================== This study was conducted in Animal Research Laboratory of Dokuz Eylul University, between 24 June and 29 August 2013. Twenty adult New Zealand rabbits weighed 1,550--2,200 gr were recruited for the study. All animals were kept in the same environmental condition throughout the study with free access to water and food. Standard rabbit foods were provided for animals. The approval of Animal Ethical Committee of the University was obtained before the study (No. 22.05.2013/58/2013). Animals were assigned into three groups. Right facial nerves of all animals were dissected and clamped for 30 seconds to make a crush injury. The first group consisted of the rabbits which have crush injury alone (n=7). The second group had crush injury plus locally applied 3 mg of porcine skin gel soaked with 1 mL saline (n=7), and the third group was the study group with crush injury plus locally applied 3 mg porcine skin gel soaked with 1 mL (100 μg) of IGF-1 (mecasermin; Increlex, Ipsen Biopharmaceuticals, Inc., Basking Ridge, NJ, US) (n=7). On the 10th and 42nd days, the animals were anesthetized with intramuscular ketamine hydrochloride (35 mg/kg) and xylazine hydrochloride (5 mg/kg) before the electrophysiologic examinations. So that, the electrophysiological study of facial nerves could be performed on both sides of the faces of the animals. As electrophysiological changes do not appear on electromyography (EMG) until 10 days, we decided to make the first examination on the 10th day. To see the regenerative changes on EMG, we performed the second test on the 42nd day. All animals were sacrificed after the electrophysiologic examinations were completed. Both right and left extratemporal facial nerves were cut and histopathologically investigated by light microscopy. Surgical procedure ------------------ A right vertical preauricular incision was done. After dissecting soft tissues, parotid gland was mobilized to anterior, and the main trunk of the facial nerve was found after emerging from temporal bone and was gently dissected. By using a vascular clamp (Dietrich bulldog clamp, CODMAN, Codman & Shurtleff inc., Paramount, US), 150 gr of engaging force was applied to the nerve trunk for 30 seconds. A silk thread was loosely tied around the nerve body at both proximal and distal sites of the injury ([Fig. 1](#f1-ceo-2016-00997){ref-type="fig"}). For the first group, the wound was closed without doing anything further. For the second group, saline solution, and for the third group, IGF-1 solution were locally applied as described above ([Fig. 2](#f2-ceo-2016-00997){ref-type="fig"}) and then the wound was closed. Electrophysiological procedure ------------------------------ All electrophysiological recordings were completed under intramuscular ketamine hydrochloride (5 mg/kg) anesthesia. The right and the left facial nerves were separately tested for each animal. Surface electrodes were placed onto nasal muscle, nasal tip (reference electrode) and auricula (ground electrode). Facial nerves were supramaximally stimulated at the mastoid apex, and motor responses of nasal muscles were recorded. Amplitudes and latencies of compound muscle action potentials of the facial nerves on both the crushed (right) and noncrushed sides were compared. Test results were compared between groups and within groups. Follow-up --------- All animals were kept at same conditions, and they had free access to water and food. They were visually inspected twice weekly for the movement of vibrissae to ensure their survival. One of the animals was lost. Sacrification of the animals and harvesting the facial nerves ------------------------------------------------------------- Six weeks after crush injury of facial nerves, all animals were anesthetized with the same protocol. Crushed right facial nerves were dissected and harvested between the silk ties by using previous incisions. A similar neck incision was done on the left side of each animal and left facial nerve was dissected. Afterward, left facial nerve sample of the same length also harvested. All cuts on facial nerves were achieved with a number 15 surgical blade. Nerve samples were laid down on parafilm blocks. All neural tissues were stained with either hematoxylin eosin or special gomori trichrome and examined under light microscopy. In histopathological examination, axonal order, structure of myelin sheath, the status of Schwann cells, existence of angiogenesis, thickness of the epineurium and extensiveness of collagen were noted. The thickness of epineurium was recorded as the micrometer. The number of vacuoles was counted per 100 times high power field. The degree of Schwann cell proliferation was recorded as low, medium or high and collagenization as none, low, medium or high. Statistical analysis -------------------- All the data was analyzed by using SPSS ver. 15.0 (SPSS Inc., Chicago, IL, USA). For the comparison of the electrophysiologic findings on the 10th and 42nd days, Kruskal-Wallis test was used. The Mann-Whitney *U*-test was used for the evaluation of intergroup differences. Wilcoxon test was used in the evaluation of intragroup time and side variables. The median values were given. The statistical significance was accepted to be *P*\<0.05. Histopathological findings were analyzed by chi-square test. Axonal order, degree of demyelination, the number of Schwann cells, appearance angiogenesis, the thickness of the epineurium and existence of collagen parameters were estimated during the histopathological investigation. RESULTS ======= Electrophysiologic findings --------------------------- The amplitudes of facial nerve responses at the crushed side were significantly lower in the second group on day ten compared with the third group (*P*\<0.05). Motor latencies of the three groups were not different at 10th and 42nd days ([Table 1](#t1-ceo-2016-00997){ref-type="table"}). Histopathological findings -------------------------- All neural tissues stained with either hematoxylin eosin or special gomori trichrome were investigated by light microscopy and axonal order, structure of myelin sheath, status of Schwann cells, existence of angiogenesis, thickness of the epineurium and presence of collagen were noted. The thickness of epineurium was recorded as the micrometer. The number of vacuoles were counted per 100 times high power field. The degree of Schwann cell proliferation was recorded as low, medium or high, and collagenization as none, low, medium or high. In all groups, noncrushed left facial nerve specimens were evaluated first. Axons were in order. Myelination, Schwann cell proliferation, collagenization and the thickness of the epineurium were all within normal limits. Upon evaluation of crushed right facial nerves of groups 1 and 2, we reached the findings below: the orders of the axons were irregular. Myelination was diminished, and Schwann cell proliferation was more evident. Epineurium was found to be thinned, and collagenization was widespread. On the contrary, in study group (locally IGF applied group) axonal order was less disturbed. Both myelination and Schwann cell proliferation were close to normal. Epineurium was also normal, and the invasion of collagen was in a lesser degree. Locally applied IGF-1 was found to reduce the damage of the facial nerve after crush injury. In the third group, vacuolization was less evident than the second group as well as the myelination and the collagenization ([Fig. 3](#f3-ceo-2016-00997){ref-type="fig"}) (*P*\<0.001). In comparison to the first and the second groups, there were not any statistically significant differences in these parameters (*P*\>0.05) ([Table 2](#t2-ceo-2016-00997){ref-type="table"}). DISCUSSION ========== Facial paralysis is known to cause severe problems in patient's appearance, with severe impact on individual's life together with psychological and social problems. Crush injuries of facial nerve can occur due to external traumas or iatrogenic causes. Like many otological and otoneurological procedures, parotid surgery and maxillofacial interventions also carry the risk of injuring facial nerve. When the injury is slight, the only pathologic mechanism is the blockage of axoplasmic flow which leads a temporary facial paralysis. With the return of axoplasmic flow, this paralysis can recover without any sequela. However, in more severe injuries, endoneurium can be damaged, and even some axons can be permanently disrupted. In this case, recovery of facial functions should not be proper and disturbing symptoms such as synkinesis, mass movements or crocodile-tear syndrome can be seen. These symptoms are attributed to misdirection of regenerated axons within damaged neural tubules. Recovery is related with severity of traumatic insult as well as the severity of the symptoms. When the compression-induced ischemia is more prominent, nerve healing is expected to be more rapid and recovery, thus less problematic. If the mechanical disruption of the axons are in excess, healing should take longer time and only with sequela \[[@b1-ceo-2016-00997]\]. Some authors prefer rats in order to study the injury of peripheral nerves \[[@b6-ceo-2016-00997],[@b7-ceo-2016-00997]\]. However, atypical posttraumatic axonal reactions can be seen in rats. So that, we chose rabbits to investigate the crush injury of facial nerve. Anatomy of the facial nerve in rabbit resembles human anatomy. In contrast with rats, a marked chromolysis and complete remission of paralysis were shown within a few weeks after crush injury \[[@b8-ceo-2016-00997]\]. One of the most important aspects of follow-up is to predict the prognosis. This is necessary not only for counseling the patient, but also for planning future interventions in totally paralytic and progressive cases. For this purpose, some qualitative and quantitative methods as well electrophysiologic and morphometric analyses were described. In this study, we selected motor responses of facial nerves as it is an objective electrophysiologic test in order to guess the prognosis. Electrophysiologic responses were used in peripheral nerve injury models of various animals with varying success \[[@b9-ceo-2016-00997],[@b10-ceo-2016-00997]\]. After any traumatic insult, prevention of severe edema formation and inhibition of inflammatory reactions are expected to shorten the recovery time, together with better functional results. For this aim, many medications have been tried such as corticosteroids, adrenocorticotrophic hormone, vasodilators, vitamins, and antiviral agents. There are plenty of studies in which various molecules have been tried for treatment of nerve injury. Topdag et al. \[[@b11-ceo-2016-00997]\] claimed that a tumor necrosis factor (TNF)-α antagonist (etanercept) was effective in nerve crush injury model. Jang et al. \[[@b12-ceo-2016-00997]\] found that topical dexamethasone enhanced the regeneration. Regeneration of peripheral nerves after crush injury is a very complex issue and various factors may affect the recovery. Some medications, neural stem cells and electrical stimulation have been all tried to enhance the recovery. Among many trials, growth factors such as brain derived neural growth factor, ganglion derived neural growth factor, and fibroblast growth factor (FGF) were found beneficial in some *in vivo* studies; though they have not been studied in clinical practice up to date \[[@b11-ceo-2016-00997]-[@b14-ceo-2016-00997]\]. IGF-1 and IGF-2 receptors were found in neural tissues of aves, reptiles, amphibia, and mammals. IGF-1 stimulates the development and growth of neurons and glial cells. Its serum level is controlled by growth hormon. Moreover, IGF-1 was shown to promote neurite outgrowth and neuronal survival. However, after an injury IGF-1 reactive material is overexpressed. There are some *in vitro* and *in vivo* studies, which indicate that IGF-1 promotes nerve regeneration \[[@b4-ceo-2016-00997],[@b5-ceo-2016-00997],[@b15-ceo-2016-00997]\]. Fex Svenningsen and Kanje \[[@b16-ceo-2016-00997]\] showed that IGF-1 increased Schwann cell proliferation surrounding the myelinated nerve fibers in peripheral nerve cultures. These authors have reported that IGF-1 is expressed in human neural stem cells and is potentially a neurotrophic and neuroprotective substance. So that IGF-1 may stimulate neurite outgrowth and provide neuroprotection against neurotoxic agents \[[@b17-ceo-2016-00997]\]. Thanos et al. \[[@b18-ceo-2016-00997]\] found that local application of 50 µgr/mL IGF-1 on crossed facial nerve grafts led to improvement in orbicularis oris functions. They also proved that myelination, axonal regeneration, and diameters of the axons were improved. Emel et al. \[[@b19-ceo-2016-00997]\] investigated the effects of local IGF-1 application on crush injury of sciatic nerve in rats. They pointed out that sciatic function index and sensory function had recovered. The diameters of axons and the thickness of myelin sheaths had significantly increased. In a review of Werner and LeRoith \[[@b20-ceo-2016-00997]\], IGF-1 is referred as a neuropeptide, which has some biological activities such as enhancing neuronal survival, neurogenesis, angiogenesis, excitatory, and inhibitory neurotransmission. It was said to be locally produced in central nervous system in addition to the portion coming from peripheral circulation via blood-brain barrier. Tiangco et al. \[[@b21-ceo-2016-00997]\] found that in a model of end to side nerve anastomosis, continuous application of IGF-1 with an osmotic pump enhances the rate of axonal regeneration and functional recovery of paralyzed muscles. D'ercole et al. \[[@b22-ceo-2016-00997]\] claim that in mutant hide mice IGF-1 has a central role in the growth and development of the central nervous system. It was found to stimulate myelination as well as oligodendrocyte development and neuronal survival. On the contrary, Yuan et al. \[[@b23-ceo-2016-00997]\] found IGF-1 ineffective in the prevention of death of motor neurons secondary to avulsion or axonal transection in newborn rats. Ozdinler and Macklis \[[@b24-ceo-2016-00997]\] studied corticospinal motor neurons *in vitro* and found that IGF-1 improves axonal out growth. Kimpinski and Mearow \[[@b25-ceo-2016-00997]\] have worked on sensorial nerves and showed that IGF-1 causes neuritic growth similar to nerve growth factor (NGF). When applied on cell bodies, IGF enhances distal neurite growth within the compartment which contains NGF. Skouras and Angelov \[[@b1-ceo-2016-00997]\] claimed that combination of manual mechanical stimulation with the local IGF-1 application was an effective method in recovery after facial nerve anastomosis. Kiryakova et al. \[[@b26-ceo-2016-00997]\] also reached similar conclusions in a study conducted on rats. They concluded that IGF-1 application led to the increase of terminal Schwann cells in the neuromuscular junction. Our histopathologic findings are similar with many of these results and point out that IGF-1 application is beneficial on hastening the recovery after crush injury of the facial nerve. Since continuous release of IGF-1 is required for the nerve regeneration, the way of application is an important issue. Some authors had to use osmotic pumps to deliver various growth factors to the site of injury. However, it is rather a difficult way for animal experiments and also in clinical applications. Compliance is not very high, and also it is relatively expensive. Another method of continuous controlled release of molecules may be to use hydrogels. These biodegradable gels make a complex with drugs. While polymers decrease in size, these drugs are continuously released. Gnavi et al. \[[@b27-ceo-2016-00997]\] used hydrogel for vascular endothelial growth factor and found that regeneration of neural tissues was enhanced. Matsumine et al. \[[@b14-ceo-2016-00997]\] used gelatin hydrogel and micropipettes to deliver basic fibroblast growth factor and found this method to be effective. Lee et al. \[[@b28-ceo-2016-00997]\] used IGF-1 soaked gelatin sponges onto the round window in a guinea pig model of acoustic trauma which was severe enough to prolong auditory brain response (ABR) values. These findings were histopathologically supported with enhanced recovery of outer hairy cells. Nakagawa et al. \[[@b13-ceo-2016-00997]\] found that majority of sudden deafness patients who do not respond to corticosteroids benefited from the local application of IGF-1. A piece of IGF-1 soaked gelatin hydrogel had been inserted in the round window niche in this group of patients. Komobuchi et al. \[[@b29-ceo-2016-00997]\] studied the effects of basic fibroblast growth (b-FGF) factor on intratemporal facial nerve injury model in guinea pigs. They histopathologically and electrophysiologically proved that application of b-FGF alone, without any supporting material, does not have any beneficial effect on recovery; however, its application in a hydrogel base led to better recovery within 6 weeks. Our method of application gelatin-based hydrogel to deliver to the site of injury for a given period is similar to the study cited above. The prediction of prognosis after crush injury of the facial nerve is important in the treatment planning. Inspection is a straightforward and reliable method; but also needs documentation. Different electrophysiologic and histomorphometric analysis have been developed to evaluate the functional recovery and neuronal regeneration. Electroneurographic (ENG) and EMG recordings are most widely used techniques \[[@b9-ceo-2016-00997],[@b10-ceo-2016-00997],[@b30-ceo-2016-00997]\]. So we used electroneurographic recordings. Volk et al. \[[@b10-ceo-2016-00997]\] investigated the value of different electrophysiologic parameters in 259 patients for the determination of the prognosis. They found that apart from initiation of treatment and House Brackman (HB) grading, electrophysiologic investigtions and stapedius reflex tests are highly important indicators in the assessment of recovery time in facial paralysis. When the facial nerve is damaged either demyelination or axonal degeneration can occur. The crush injury model we made causes axonal damage, so the decrease in amplitudes of motor responses must be the leading electrophysiologic feature. The prolongation of motor latencies indicates demyelination. So that, the latencies were expected to be quite normal. In our study, we found remarkable amplitude attenuation in the group which was applied only saline. In the IGF-1 group, no attenuation of the amplitudes did occur. This result points to the capability of IGF to improve the recovery of facial nerve injury. The histopathologic results supported the electrophysiologic data. The orders of the axons were irregular, myelination was diminished, and Schwann cell proliferation was more evident in the non-IGF groups. On the contrary in the study group (locally IGF applied group) axonal order was less disturbed, as well as myelination. Schwann cell proliferation was close to normal. Epineurium was normal, and collagenization was in a lesser degree. These findings reveal that IGF-1 is beneficial in the recovery of facial nerve after injury. This animal study may contribute to the clinical studies and IGF-1 may be developed as a candidate drug in the future. In conclusion, local application of IGF-1 in a slow releasing gel was found to be efficacious in the recovery of facial nerve crush injury in rabbits. Since this molecule is a natural peptide and considered to be beneficial in clinical use, it should be worthy of being tried in clinical drug studies on facial nerve injury cases. HIGHLIGHTS ========== ▪ Local application of insulin-like growth factor 1 (IGF-1) was found efficacious in the recovery of the facial nerve crush injury in rabbits. ▪ Electrophysiologic studies can be used in animal model of facial injury. ▪ IGF-1 may be applied for treatment of facial paralysis. No potential conflict of interest relevant to this article was reported. ![Surgical procedure is seen as a silk thread was loosely tied around the nerve proximal and distal site to make a crush the injury of the rabit's facial nerve.](ceo-2016-00997f1){#f1-ceo-2016-00997} ![Insulin-like growth factor 1 solution was locally applied with porcine skin gel was applied to the third group (study group).](ceo-2016-00997f2){#f2-ceo-2016-00997} ![(A) Left nerve without trauma, normal appearence. (B) Group 2 (salin applied group) right nerve with crushed injury. The order of axons degenerated, myelinization is reduced. Schwan cell proliferation is increased (medium grade). Epineurium is thinner, kollagenization is increased. (C) Group 3 (insulin-like growth factor applied group) right nerve with crushed injury, reduce damage can be seen. The order of axons has become normal. Myelinization has improved. Schwann cell proliferation and kollagenization have been low grade. The epineurium has become normal (trichrome staining, ×100).](ceo-2016-00997f3){#f3-ceo-2016-00997} ###### Electrophysiological findings of the facial nerves in all groups Group Motor latencies (ms) Amplitudes (µV) ------- ---------------------- ----------------- ----------- ----------- 1st 1.40±0.15 1.64±0.45 1.61±1.32 1.95±0.62 2nd 1.42±0.11 1.47±0.20 1.87±0.95 2.37±0.89 3rd 1.44±0.16 1.40±0.23 2.93±1.48 2.99±1.38 ###### The summary of histopathological findings of all groups Group No. of animal Epineurium thickness (µm) No. of vacuoles Schwann cell-proliferation Axons ------- --------------- --------------------------- ----------------- ---------------------------- ----------- 1st 6 40.83 (38--45) 1.00 (0--3) Low Regular 2nd 7 29.50 (27--32) 28.17 (16--44) High Irregular 3rd 7 54.86 (52--57) 5.86 (2--11) High Regular Values are presented as number or mean (range).
Mid
[ 0.6347607052896721, 31.5, 18.125 ]
Secretary of State Rex Tillerson will meet Tuesday with Cuba's top diplomat, U.S. officials said, as the United States is seeking answers about mysterious "attacks" on its diplomats in Havana. Tillerson's meeting with Cuban Foreign Minister Bruno Rodriguez is believed to be the highest-level diplomatic contact between the two countries since the start of President Donald Trump's administration in January. It comes as the delicate rapprochement between the longtime foes, started under President Barack Obama, is being jeopardized by mounting alarm over the diplomats' illnesses. The meeting at the State Department will follow a speech Rodriguez delivered at the United Nations last week in which he harshly criticized Trump and his administration's policy toward the communist island. In a bid to show it's a good neighbor, Cuba has also offered to send doctors and humanitarian aid to Puerto Rico to help the U.S. respond to Hurricane Maria. Get Breaking News Delivered to Your Inbox For months after U.S. diplomats started falling ill in Havana, the U.S. and Cuba sought to prevent the issue from becoming an overriding irritant in the relationship. Neither country disclosed publicly that the incidents were occurring, even after Washington in May expelled two Cuban diplomats to protest Havana's failure to protect Americans on its soil. Yet while the U.S. has avoided blaming Cuba directly for the incidents, the growing public outrage has forced both countries to adopt a tougher tone. Tillerson has said that closing the newly re-opened U.S. Embassy in Havana is under consideration, and several U.S. lawmakers have called on the Trump administration to expel all Cuban diplomats from Washington. Embassy workers have complained of nausea, headaches, exhaustion and trouble concentrating and remembering words, and some have been diagnosed with mild traumatic brain injury and permanent hearing loss, according to the American Foreign Service Association and medical records examined by CBS News. CBS News reported earlier that the top official in charge of security at the embassy was among those injured. CBS News is not naming the Regional Security Officer, who was scheduled to undergo medical treatment, according to a source. When contacted by CBS News, the individual declined to comment. Meanwhile, the latest development illustrates how far-reaching the attacks have been, affecting one of the most senior leaders of the U.S. embassy that only reopened in 2015. CBS News Radio broke the story last month. Cuba has denied any knowledge or involvement. In his U.N. speech last week, Rodriguez said early results from Cuba's own investigation have "found no evidence whatsoever that could confirm the causes or the origin" of the incidents, implicitly casting doubt on the U.S. version of accounts. "It would be unfortunate if a matter of this nature is politicized," said Rodriguez, who has represented Cuba on the world stage for years. Last week, U.S. diplomats held another high-level meeting with Cuba amid concerns about the incidents. That meeting at the State Department included Cuban official Josefina Vidal, who has been the public face of Cuba's diplomatic opening with the U.S., and other Cuban officials. Of the 21 medically confirmed U.S. individuals affected — diplomats and their families — some have permanent hearing loss or concussions, while others suffered nausea, headaches and ear-ringing. Some are struggling with concentration or common word recall, The Associated Press has reported. The U.S. has said the tally of Americans affected could grow as more cases are potentially detected. The State Department has emphasized that the U.S. still doesn't know what has occurred.
Mid
[ 0.5783664459161141, 32.75, 23.875 ]
Atypical AV junctional reentrant tachycardia following AV nodal modification. The authors present a patient who initially underwent anterior approach atrioventricular (AV) nodal modification for treatment of typical AV junctional reentrant tachycardia (AVJRT) and subsequently developed clinical episodes of a previously undocumented type of supraventricular tachycardia. Findings during electrophysiologic studies suggest that this tachycardia is due to both anterograde and retrograde conduction in a slow AV nodal pathway. A "slow pathway" potential was identified and dissociated from the local atrial and ventricular depolarizations. Posterior approach AV nodal modification was successfully used to ablate this tachycardia. These findings suggest that atypical AVJRT occurring after AV nodal modification may be "slow-slow" AVJRT.
High
[ 0.6706056129985231, 28.375, 13.9375 ]
In conventional optical lithographic practice a thin film of a photoresist material is applied to a substrate. The photoresist film is exposed to a desired pattern of radiation and the exposed photoresist is developed to reproduce the desired pattern. The pattern can be reproduced onto the surface of the substrate by an etching process. However, in this approach resolution is limited by the wavelength of the exposure radiation and as the feature size becomes smaller the equipment becomes increasingly expensive and complex. Imprint lithography (IL) has gained considerable attention as a cost effective and technically feasible method for fabricating nanometer-dimension structures. Due to its low cost relative to conventional optical lithography tools and next generation lithographies (NGL), and because of the apparent ability to produce devices with critical dimensions as small as 10 nm, IL can play an important role in the integrated circuit and hard disk drive industries as well as telecommunications, specifically in the production of surface acoustic wave devices. For imprint lithography techniques generally, a relief pattern in a template is used, in conjunction with polymeric materials to create a desired pattern on a substrate. The process, illustrated generally in FIGS. 1a–1d, involves coating the surface of a substrate 110, that can be silicon or quartz, with a thin film of a deformable polymer material 115 (FIG. 1a). Subsequently, the film is imprinted with a pattern of trenches by applying pressure to a patterned template 120 (FIG. 1b). Application of pressure can be mechanical or by fluid pressure such as disclosed in U.S. Pat. No. 6,482,742 to Chou. The imprinted film can be cured by the application of heat, illumination, pressure or combinations thereof. Following the curing step (FIG. 1c), a reactive ion etch (RIE) process can be used to remove unwanted material from the trenches imprinted on the surface of the polymer film and transfer the pattern into an under lying structure that can be the substrate or other functional thin films (FIG. 1d) (cf. Voisin, U.S. Patent Application Publication US 2003/0205657, Nov. 6, 2003). The imprint lithography process is discussed in detail in U.S. Pat. No. 5,772,905 to Chou. For integrated circuit manufacturing or other high resolution applications, the template should have a precise pattern that pressing transfers, inherently with minimal demagnification, to the surface of the integrated circuit device. Numerous variations of the basic IL method outlined above have been developed. Discussions of these methods can be found in Bailey et al., J. Vac. Sci. Tech. B 18(6), 3572–3577 (2000), Colburn et al., Proc. SPIE, 3676, 379–389 (1999), Tan et al., J. Vac. Sci. Tech., B16(6), 3926–3928, (1998), and Sreenivasan et al., Proc SPIE, 4688, 903–909 (2002). One particular IL component critical to its cost effectiveness is the template or mask. According to a cost-of-ownership study by Sreenivisan (ibid.), the IL mask is estimated to cost about $40,000. This mask cost assumes volume production and an inspection and repair infrastructure that is not currently available. Actual mask cost in prototyping volumes will likely approach $100,000, particularly since the mask is a “1×” technology. That is, the feature sizes that are printed are equal to their corresponding features on the mask. This is in contrast to the reduction technology practiced in most commercial optical photolithography technologies that typically employ 4× or 5× reduction. As with all masks, as the feature size of IL masks decreases, the time required to pattern or “write” a mask increases. Consequently, not only does the mask cost increase but also the turnaround time to fabricate a mask increases. Thus, for small-volume manufacturing and prototyping, mask costs can become prohibitively expensive, i.e., the mask amortization cost schedule far exceeds the technology generation duration.
High
[ 0.669473684210526, 39.75, 19.625 ]
Autodesk Maya 2018 Win Title: Autodesk Maya 2018 Win Info: Scene ISO release added Autodesk Inc., a world leader in 3D design software for entertainment, natural resources, manufacturing, engineering, construction, and civil infrastructure, announced the release of Maya 2018, is an industry leading 3D animation software application that enables video professionals who work with animated film, television programs, visual effects, and video games to create highly professional three-dimensional (3D) cinematic animations. Prior to two-dimensional (2D) and 3D animation software, manual hand animation tools such as drawing paper and pencils, erasers, paints and brushes, light tables, and transparencies only offered a subset of what can now be done with programs such as Maya. Maya 1.0 was originally developed and released in 1998 by Alias Wavefront and seven years later in 2005, Autodesk, Inc. acquired Maya and renamed it to “Autodesk Maya”. Since its original release, Maya has become widely used in the film industry to create graphics for Academy Award winning films such as Rango and Hugo. Maya is also becoming more widely used in the video game industry to create visual effects for games such as Call of Duty and Halo. Maya includes MEL, short for Maya Embedded Language, and Python scripting, which both allow you to take advantage of its open architecture by programming complicated or repetitive commands. These programmed commands help to save valuable time and also offer a method of sharing them with others who might find them useful. In the film and television industry, Maya is the de facto standard for 3D visual effects, computer graphics, and character animation. Individuals who work in or are currently pursuing careers in the 3D animation, character modeling, visual effects, and other animation fields will discover many benefits of using Maya. The scene assembly and accelerated modeling workflows built into Maya not only maximize productivity but also help to streamline your design experience, putting creativity back at your fingertips while increasing efficiency. Complex animation tasks that are nearly impossible to construct by hand are easily created using Maya’s robust tools. The grease pencil, automatic joint centering, camera sequencer, and weight distribution tools are just a few of the features that Maya offers to help animators focus more on the workflow and express more creatively. With Maya’s enhanced Viewport 2.0 and DX11Shader (DirectX 11) rendering engine, blurred reflections and shader effects, as well as translucency, substance textures, and many other features can be worked with directly within the Maya viewport. 3D models and visual effects are rendered in real-time, allowing artists to work in an environment that nearly matches final output. Large and complex worlds are easily created and managed with Maya’s Open Data platform, which handles production assets as discrete smart data elements and enables artists to quickly test different representations for the best result. Maya offers many other tools and features that can enhance productivity such as Volume Attributes, Paint Effects Surface, Clip Matching, URI Support, File Path Handling, PySide Pythod Qt Binding, Inline Help, and much more. Seems like another disaster for old beloved Maya. Why Autodesk bought Maya???😥😥 Waiting for complete change log Seems it’s the time to switch to another package like Houdini😞😞😞 O renderman!! It’s cause of you I’m still using Maya I wouldn’t leave Maya for all the money in the world, Autodesk improved almost every area of the program in the last 5 years, the modelling , the uv mapping, the animation and so on, just disappointed if this is indeed all that’s been added, maybe this version is incomplete or a beta. A shame if they didn’t add the division layers for sculpting and all the other things they showed in the video bellow in the beginning, was really waiting for this. https://www.youtube.com/watch?v=X7WbSIRtpgU
Mid
[ 0.5919811320754711, 31.375, 21.625 ]
The associations between current recommendation for physical activity and cardiovascular risks associated with obesity. To examine associations between current recommended physical activity levels and body mass index (BMI) with some cardiovascular disease (CVD) risk factors (total cholesterol, high-density lipoprotein cholesterol (HDL-C), non-HDL-cholesterol (non-HDL-C), C-reactive protein (CRP), fibrinogen, and blood pressure), general health score (GHQ12) and predicted coronary heart disease (CHD) risk. Further analysis of the cross-sectional Scottish Health Survey 1998 data. Five thousand four hundred and sixty adults 16-74 years of age. After controlling for some confounding factors, obesity was significantly associated with higher odds ratio (OR) for elevated cholesterol, CRP, systolic blood pressure, non-HDL-C and lower HDL-C (P<0.001), and with greater predicted CHD risk compared to BMI <25 kg/m(2). Regular self-reported physical activity was associated with smaller OR of lower HDL-C and higher CRP, and average predicted 10-year CHD risk in obese subjects, but did not eliminate the higher risk of the measured CVD risk factors in this group. The OR of these two risk factors were still high 4.39 and 2.67, respectively, when compared with those who were inactive with BMI <25 kg/m(2) (P<0.001). Those who reported being physically active had better GHQ scores in all BMI categories (P<0.001). Reporting achievement of recommended physical activity levels may reduce some CVD risk factors, predicted CHD risk and improve psychosocial health, but may not eliminate the extra risk imposed by overweight/obesity. Therefore, increasing physical activity and reducing body weight should be considered to tackle CVD risk factors.
Mid
[ 0.648910411622276, 33.5, 18.125 ]
module Griddler class Engine < ::Rails::Engine initializer 'griddler.routes', after: 'action_dispatch.prepare_dispatcher' do |app| ActionDispatch::Routing::Mapper.send :include, Griddler::RouteExtensions end end end
Low
[ 0.475524475524475, 25.5, 28.125 ]
Q: APK: native libraries name issues related to version number (SO.X.Y) Issue Built APK looks wrong and prevents my application from loading its native library because it cannot find a dependency when calling dlopen(): dlopen failed: library "libboost_filesystem.so.1.68.0" not found. jniLibs content My jniLibs directory, for the target platform, contains the following files: libboost_filesystem.so.1.68.0 is the "real" shared object. libboost_filesystem.so is a symlink to libboost_filesystem.so.1.68.0 APK Content After building, the APK contains a libboost_filesystem.so which now is the binary object (not a symlink). It seems like Android build system followed the symlink, grabbed content of the "pointed-to" file, but used the name of the symlink instead. I have tried to remove the symlink from the jniLibs folder, but doing that it seems that the xxx.so.VERSION files are then ignored. Question How can I embed my "full name" shared object into the jniLibs without the Android build system messing with it ? A: No you can't. You should avoid versioning the so file. See https://stackoverflow.com/a/45058227/192373 for instructions. It's also quite natural that Android does not support this technique, because your native libraries belong to your APK and no version conflict can occur. Consider linking boost filesystem statically to avoid extra lookup.
Mid
[ 0.5525114155251141, 30.25, 24.5 ]
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"Now,if you ask one more time,your head is mine,but for now,sure,",I said,looking at her.She nodded, but raised her laserbeam almost at me.I got up,and tased her head."Lucky she's alive,and lucky I made it,",I thought,the thought of what I just said running through my mind.Soon,shots fired,and we started running.None of us were shot down,only 3 wounded,but we're good from here.Next minute I know,there's a slaying wizard above me.Dark thunderstorm clouds fill up the surface. "Don't worry,Princess,I'm coming home,",I'm thinking as I shoot him down with all I got.Soon,2 more arrive,and as if I can't get any closer to evil at this point,I can't use my tranquilizer anymore. It's every dog for its dog now.I have no choice but to use my mist spray for this kind of situation. The cold mist I use from my flask saber bottle helps,and they soon fall asleep suddenly. It can't be magic,could it?The question hangs over my head like a raincloud,waiting to attack. Soon,I'm running through the darkness,trying to get away from the Glass Shard Nightmares,the nightmares I had as a kid up until now.It sickens me to see these nightmares follow me around my whole life,but tonight's the night to get through all of that. It won't be soon until long after I finally escape and make it to the Dark Side.Mickey Tindlehttp://www.booksie.com/mickey_tindleLove letter number 4 <>http://www.booksie.com/gay_and_lesbian/miscellaneous/sukihanauchi/love-letter-number-4-<>2014-12-04T02:17:57-01:00<>Sukihanauchihttp://www.booksie.com/sukihanauchiLove letter number 3<>http://www.booksie.com/gay_and_lesbian/miscellaneous/sukihanauchi/love-letter-number-3<>2014-12-04T02:05:58-01:00<>Sukihanauchihttp://www.booksie.com/sukihanauchiLove letter number 2http://www.booksie.com/gay_and_lesbian/miscellaneous/sukihanauchi/love-letter-number-22014-12-04T02:02:39-01:00<>Sukihanauchihttp://www.booksie.com/sukihanauchiLove letter number 1http://www.booksie.com/gay_and_lesbian/miscellaneous/sukihanauchi/love-letter-number-12014-12-04T01:57:13-01:00Love letter. Changing names in it so everyone is comfortable.Sukihanauchihttp://www.booksie.com/sukihanauchiTo love and be loved <3http://www.booksie.com/gay_and_lesbian/miscellaneous/skeptic_windego/to-love-and-be-loved-<32014-09-17T01:48:22-01:00I'm not too sure what to call this, nor how to describe it. I'll leave it up to you, the reader, to decide how to describe this.Skeptic Windegohttp://www.booksie.com/skeptic_windegoswinging under the starshttp://www.booksie.com/gay_and_lesbian/miscellaneous/charl2890/swinging-under-the-stars2014-07-07T19:37:55-01:00Just a short song almost poem, just my vent for my undying love for a girlcharl2890http://www.booksie.com/charl2890Instant http://www.booksie.com/gay_and_lesbian/miscellaneous/koala/instant2014-04-28T09:09:37-01:00I just...dont really know what I'm going to do, and it really really hurts sometimes, like I really wish that I could cry though of course that will just make my head hurt, or maybe just stop thinking about you so then I would be a better friend Koalahttp://www.booksie.com/koalaThe storyhttp://www.booksie.com/gay_and_lesbian/miscellaneous/girlwonder12/the-story2014-01-27T06:47:25-01:00raw material from a story i am writing, about how i overcame myself and coming out. Only a preview of my story but it needs to be told and maybe it can save people livesgirlwonder12http://www.booksie.com/girlwonder12One Of Those Dreamshttp://www.booksie.com/gay_and_lesbian/miscellaneous/reece_oak/one-of-those-dreams2014-01-13T20:21:14-01:00I don't know if I've mentioned, but I'm a lesbian. I have a crush just like anyone who's straight. I write about her a lot. Sometimes to her, but it ends up being too deep to read to her. Maybe you will like it.Reece Oakhttp://www.booksie.com/reece_oak
Low
[ 0.5115740740740741, 27.625, 26.375 ]
Bras WOMEN'SBras Read More +Read Less -+- From everyday options to designs with added wow-factor, shop our current range of bras and get ready to refresh your underwear drawer with some truly fabulous picks. After plus size options which offer the perfect fit for your unique curves? Shop Triumph for soft t-shirt bras ideal for day-to-day wear or browse large bras from Curvy Kate in gorgeous prints, luxe textured fabrics and sexy plunge cuts. Those after new lingerie for an upcoming date night should check out Figleaves bras in silhouette-shaping designs and silky materials, while Glamorise are your go-to brand for bras which offer up supreme support and comfort without compromising on style.
Low
[ 0.519362186788154, 28.5, 26.375 ]
Three agendas for changing the public stigma of mental illness. Antistigma programs may be guided by 3 differing agendas: services (promote treatment engagement), rights (help people achieve rightful goals), and self-worth (facilitate self-worth and efficacy). This study examined the construct validity of this perspective by examining the factor structure of importance ratings of the 3 agendas. The study examined how importance might be viewed differently by the population as a whole versus a subsample of people who reported previous experience with mental health services and hence could be directly harmed by stigma. 373 individuals recruited using Mechanical Turk completed importance ratings for each of the 3 agendas. Measures of public stigma were completed to examine concurrent validity of importance ratings. Those who reported taking medications for a psychiatric disorder were divided into a separate group and completed a measure of self-stigma. Outcomes seemed to confirm the factor structure of the 3 agendas model thereby offering partial support for the framework. Group analyses showed the services agenda was viewed as more important than rights or self-worth. People with mental health experience viewed the services agenda as more important than the other 2. However, dividing the mental health group into low and high self-stigma revealed that those with low self-stigma rated the rights agenda as more important. Conclusions and Implication for Practice: Participants with lower self-stigma identify the harm brought by stigma and thus endorse rights and self-worth more than those with higher self-stigma. Implications of these findings are discussed to assist to prioritize agendas for public health campaigns. (PsycINFO Database Record
High
[ 0.7075812274368231, 36.75, 15.1875 ]
Many conventional regulators include a valve body and a control assembly for regulating fluid flow through the valve body. The control assembly generally comprises a control element such as a valve plug, for example, coupled to a diaphragm or other pressure sensing device for automatically moving the control element in response to pressure changes at the outlet of the valve body. Additionally, some conventional regulators include a spring that biases the control element into a predetermined position in the valve body, e.g., an open position or a closed position. So configured, during operation, the spring naturally biases the control element into its predetermined position and changes in the outlet pressure change the position of the control element to enable or disable fluid flow through the valve body, as desired. Fluid flowing through the valve body can generate vibrations in the system. High and low frequency resonance caused by these vibrations can hamper the operational integrity of the regulators. One solution for reducing resonance is to include a spring clip surrounding a portion of the load spring to dampen vibrations. One conventional design of a spring clip 1 includes a generally U-shaped member such as that depicted in FIGS. 1 and 2. The spring clip 1 depicted in FIGS. 1 and 2 includes a body plate 2 and a pair of opposing arm plates 3 extending outwardly therefrom. When assembled into the regulator, the spring clip 1 is positioned into a cylindrical cup shaped member 4, as depicted in FIG. 2, and then a bottom portion of a spring 5 is disposed positioned between the opposing arm plates 3 and body plate 2. This assembly process can be tedious and time-consuming as the spring clip must be manually manipulated to spread the arm plates 3 apart.
Mid
[ 0.5654885654885651, 34, 26.125 ]
“This is a place,” says the 20-year-old Celtics rookie guard and top draft choice, “that you don’t want to come back to.” LANCASTER, Texas — Marcus Smart exits the silver Ford Mustang, surveys the apartment complex, and shakes his head. In the overcast morning light, with thunderstorms stretched gray across the summer sky, he tries to remember the last time he returned to this Dallas suburb and retraced his childhood steps. But he cannot remember. It has been that long. Eight years ago, Marcus prayed: “Just please get me out of this.” He prayed as he sprinted through the complex’s parking lot while a Bloods gang member gave chase, pulled a gun, and started firing. He prayed as he zig-zagged between cars while seven bullets whizzed by. He prayed as he ran harder and faster than he ever will, knowing that this could be it. “I promise,” he prayed, “I’ll do better.” Today, Marcus calls that moment the lowest point in a life racked by tragedy, with so many family members and friends dying from everything but old age — a toll so steep it does not seem real. One relative dropped a loaded shotgun during a children’s game of cowboys and Indians in this town many years ago. It discharged straight into the heart of another relative, who was 5. Another relative was found shot to death one morning in a front yard in Tyler, Texas. He was 16. The Meadows, across the street from Marcus Smart’s boyhood home, was a setting for gang warfare. Kim Leeson/Boston Globe A train struck one of Marcus’s ex-Amateur Athletic Union teammates four years ago in rural Oklahoma. He was 17. Then there was Todd Westbrook, Marcus’s oldest half-brother, who died at 33 from cancer. The deaths came in rapid order. “Oh, God,” says Marcus’s mother, Camellia, as she fights back tears. “At one point, it was like ‘pop, pop, pop, pop, pop.’ ” When Marcus was 3, his grandmother — Camellia’s mother — died. He was close to her and attended the funeral, but after that, he told Camellia, “If I don’t have to go, I don’t want to.” “I used to hate seeing someone up there and it was someone you know, you care about,” he says now, “and there’s nothing you can do to help them.” Marcus has a guaranteed multimillion-dollar contract with the Boston Celtics, who selected him sixth overall in June’s National Basketball Association draft. He has a multiyear apparel deal with Adidas, also worth more than $1 million. He has his health. And with Celtics star point guard Rajon Rondo out 6 to 8 weeks because of a broken left hand, Smart figures to play a more prominent role, perhaps starting through the first month of the regular season. But what he has seen makes him more grateful than most. “I just wake up and I thank God every day,” he says, “because I easily could’ve been in jail or six feet under.” . . . Marcus stands on a sidewalk in front of a single-story, multifamily brick home on the 1500 block of North Bluegrove Road. A large magnolia tree shades the front yard. “This is it,” he says. There is a metal gate on the door; that was not there when his family moved here from DeSoto just after Todd died, when Marcus was 9. Across the street is a notorious trio of apartment complexes — on the left and right called The Pinks; in the middle, the Meadows. Marcus recalls the area, known as the “1500 block,” as a place where Crips and Bloods gangs waged war, where drugs were rampant, where police sirens howled, where the Fourth of July was an excuse to fire off more gunshots than normal because outsiders mistook them for fireworks. “At the time when I was here,” Marcus says, “if you heard ‘Lancaster, 1500 block, Meadows,’ everybody said, ‘Oh, you live by the Meadows. We won’t come over there. We’ll talk to you later.’ ” A jogger in Lancaster, Texas, passed the North Bluegrove Road boyhood home of Celtics rookie Marcus Smart. Kim Leeson/Boston Globe/Globe Freelance . . . Born three weeks prematurely at just 6 pounds, Marcus spent nearly a month in the hospital with a feeding tube as doctors ran tests to make sure he was OK. He would be fine; more than fine. When Camellia took him back for his two-month checkup, he dwarfed other children his age. By age 1, she called him huge. “Ever since then, he’s been big,” she says. But for as big and old as Marcus always appeared for his age, there came one harrowing night when his family asked him to become a man. He was 9 years old. His aunt sat Marcus and a cousin down on a couch and tried to talk, but could only cry. Finally, she started to say “Todd . . . ” “And I just knew what she was about to say next,” Marcus says. He burst into tears. “Todd is dying,” the aunt told Marcus. “The doctor said he’s brain dead. His heart is still beating, but he’s not there anymore, and they’re ready to take him off life support.” Marcus bolted out of the house, shattering a glass door, screaming in the yard. Neighbors looked on, concerned. His cousin tackled him. “This can’t be happening!” Marcus wailed. Todd, who had become a father figure and mentor to his siblings as their mother and father worked nights, teaching his brothers how to dress (always clean, ironed clothes), speak (yes sir, no sir), shave, shake a man’s hand, and more. Todd, who would take Marcus swimming, to the gym, to get haircuts, doughnuts, ice cream, or just horse around. And when chemotherapy sapped Todd’s energy, Marcus and a brother would go into Todd’s room to watch the Discovery and History channels and shoot balled-up socks into a hoop-shaped wire hanger hooked on the door. Todd, who was 15 when doctors found a tumor behind his eye following an all-district sophomore basketball season, but who went through treatment, returned as a senior, and led his team to the state semifinals. Todd, who checked himself out of the hospital one night, played in a high school game with his left eye swollen shut, scored 30 points, and was called “The comeback kid” by newspapers. “This can’t be happening!” Marcus wailed. The cancer spread to Todd’s lungs and stomach. Marcus had learned there was no cure. Then in late 2003, Marcus was asked what he wanted for Christmas, and he said he only wanted one more Christmas with Todd. He got his wish, and Todd gave him a gold chain with his initial “M” to hang around his neck. Early the following year, after one family visit, Todd gave Camellia a long hug, called her the best mom in the world, and said he loved her. She said she loved him, too, thinking he would fight another day, as he had for 18 years. Marcus Smart was one of several NBA players who volunteered their time for the Seeds of Peace camp in Otisfield, Maine, last month. Barry Chin/Globe Staff Marcus saw family in the hospital waiting room, everyone in tears. He tore down the hallway into Todd’s room and Camellia grabbed him. “She’s crying and I’m crying and I just see Todd’s body there, lifeless,” Marcus says. “It’s like a mannequin.” He touched Todd’s feet. “Why is he so cold?” Marcus asked his mom. “It’s because he’s not there anymore,” she said. He climbed on top of Todd’s chest and started shaking him, shouting “Wake up!” Remembering it all now, sitting in an apartment near downtown Dallas on a recent afternoon, Marcus pauses. Tears stream down his cheeks. “I’m sorry,” he says, wiping his face. After a deep breath, he resumes his story. “My mom grabbed me and she’s holding me and she said, ‘I’m sorry, there’s nothing we can do for him now.’ ” He kissed Todd on the forehead. Then, as he exited the room, Marcus’s other brothers, Michael, nearly twice his age, and Jeff, even older, stopped him. “The family is all on you now,” they told him. “It’s time to grow up. We had our chances, and we blew it. Now it’s your turn. You’re the last one. You’re all mom has left.” Marcus’s first love was football — he played strong safety and wide receiver — but when Todd died, Marcus switched to basketball. “What happened with him kind of intensified the game for me even more,” he says. “Basketball became my love.” He is often told he shares an uncanny resemblance to Todd on the court, with his aggressive style, but Marcus says he does not understand how that is possible. He says he never had a chance to study Todd’s play; a house fire destroyed Todd’s game footage. But Marcus honors Todd with his all-out effort, a trait that coaches have praised for years. “I’ve always played hard,” Marcus says. “That’s how I was raised. But now I’m playing more with a purpose. Now I’m playing not just for myself, I’m playing for my brother, for my family. I’m going to go out there and give it all I have, because he gave it all he had for 18 years, and he lost his battle. I don’t want to lose my battle.” . . . Marcus, back in Lancaster, is wearing a lime green shirt, black shorts, and sneakers. No one has recognized him — yet. “It feels weird being here,” he says. “It really does.” He is standing on the 1500 block. “I got kicked out of these apartments plenty of times,” he says. He recalls hitting a mailman with a rock, then the mailman calling police and describing what Marcus was wearing. Marcus stripped down to his boxers, raced down to the street, turned the corner, and hid in his house. “I didn’t come out for about three days,” he says. Marcus liked throwing rocks — at people, cars, windows, anything. It was fun, a competition among friends to see who could hit the most difficult targets from the longest distances. “It just turned into something where you’re constantly doing it and you get addicted to it — the adrenaline rush,” he says. Michael had warned him not to throw stones. He said that one day he would hit the wrong person. . . . Camellia Smart held her son Marcus’s Big 12 league award at her house in Flower Mounds, Texas, in January. Marcus Smart starred for two seasons at Oklahoma State. Stephen Pingry/Tulsa World Not long after Todd’s death, Camellia noticed a hole in one of Marcus’s sneakers, which she found odd. Todd had taught him to keep up appearances, but with Todd gone, Marcus said, “It just wasn’t as important anymore.” His new stance worried Camellia, who says, “That’s when the changes started.” There is no manual for how to channel grief, and Marcus channeled his into anger, engaging in several fights per week at school. “The smallest thing would set me off — just the way you look at me would set me off because I was so angry,” he says. He took anger management classes. They taught him exercises to calm down — take a deep breath, close your eyes, count to 10. “I thought it was all stupid,” he says. “What is counting to 10 going to do for me? I’m still going to be angry when I’m done counting.” His anger boiled over in a school fight that ended with Marcus slamming a student’s head face-first into the concrete, again and again, before a teacher finally pulled him off. “They said I almost killed him,” Marcus says. For that, he was kicked out of school and sent to the Texas Alternative Education Program, which he described as prison, with paddlings for punishment and mandatory uniforms. After serving 30 days, he got out and returned to public school, but said students looked at him like a criminal. “So I put a wall up,” he says. “I kind of established this mean personality, this person that you don’t want to talk to, that you don’t want to mess with.” Another way he lashed out was by throwing rocks, and late one spring evening, with a friend on a second-floor balcony in The Pinks complex, Marcus hit a man in a hooded sweat shirt from about 15 yards away, knocking him off his bike. Marcus and his friend shared high-fives and laughs, and then looked away. When they looked back, they saw only the bike. Soon, they heard heavy footsteps running up the stairs toward them. Marcus and his friend jumped over the balcony, sprinting to the adjacent complex. The man followed and pulled his gun. Drenched with sweat and full of fear, Marcus and his friend ducked into a nearby apartment. “I thought my life was over,” Marcus says. But when the door opened and the man saw who Marcus was — the brother of Michael, a fellow Bloods gang member — he walked away. The next day, on a playground, Marcus was playing basketball with Michael when the same man approached. Marcus dropped the ball and started backpedaling. Michael was not sure why. “Your little brother was throwing rocks at me last night,” the man told Michael. “He almost got hurt.” . . . Marcus once found a crack cocaine rock in Michael’s room. He did not know what it was, and he nearly tasted it. “But something told me not to,” Marcus says. Michael had given up basketball and fallen into a gang life filled with drugs, guns, and violence. Marcus saw his friends buy drugs from Michael, and he saw Michael accumulate goods that matched what his friends had reported stolen. “How do you tell your friend, ‘Oh, I think my brother stole that?’ ” Marcus asks. “You don’t, because that’s your brother.” Though just 10 at the time, Marcus tried to convince his brother to change his ways. And Michael, resorting to threats if necessary, kept the gang leaders from recruiting Marcus to the life. But Michael did not turn his own life around until after he nearly died from a cocaine overdose. Eventually, Camellia was determined to leave Lancaster, and they moved to Flower Mound, a suburb west of Dallas. To demonstrate how different it is, Michael points to an open door in their home there on a recent afternoon. “You couldn’t leave the door open in Lancaster,” he says. “If you do, you’re crazy. They’ll come in and steal your stuff while you’re probably sitting on your couch.” In Flower Mound, Marcus reunited with a best friend, Phil Forte. They played basketball together in high school and then at Oklahoma State. Marcus nearly turned professional after winning the Big 12’s Player of the Year honors as a freshman, but he returned for one more year. Then in February of his sophomore year, just before tip-off at Texas Tech, Marcus was told that his mother had been rushed to the hospital and that she did not want him to know. “I was scared,” he says. “It was heavy on my mind and my heart.” Camellia, whose blood pressure was acting up, watched from her hospital bed as Marcus lost his temper on court, engaging in an altercation with a fan late in the game — an altercation that began with the fan allegedly calling him a “trashy [racial expletive],” Michael says. Said an Eastern Conference scout: “If the racist comment was true, I know for certain, me, I’m not confrontational but I would’ve punched the guy.” No matter the provocation, Marcus says there is no excuse for his actions, which resulted in national headlines, a three-game suspension, and revealed a side of Marcus that he had worked so hard to keep in control — his temper. “Off the court, I’m a totally different person,” he says. “I’ve heard people be like, ‘Oh, so sweet, like a big ole teddy bear.’ But I guess I still have that look on my face in a game. I guess I still have a vibe where it’s intimidating.” His fire should fit in fine in Boston. After all, from Red Auerbach punching out an opposing owner to Larry Bird mixing it up with Dr. J, to Rajon Rondo bumping a ref, fire is in the Celtics’ DNA. “I guess that’s why they have all those banners up there,” Marcus says. . . . Marcus has done better, as he promised God on that night when he ran and prayed for his life, a night that marked both his lowest point and a turning point. “When you experience things like that at a young age, it does change you,” he says. “It humbles you in a way that you would never imagine.” But more than anything, he has been humbled by the loss of a father figure, a brother, Todd. “I could’ve easily let the passing of my brother control how I live my life,” he says. “But I was determined not to let him die in vain and to make a negative into a positive. I thank God every day because now I’m here, not only living out my dream but his dream, too.” On the night he was drafted, Marcus wore a bespoke blue suit jacket with several images stitched into the lining — an Oklahoma State logo, a map of Texas, an “M” for Marcus, and a copy of the tattoo that graces his arms, an RIP for Todd. . . . What does Marcus Smart hope to accomplish in the NBA? “Just to have a long career,” he says. “That’s it.” Barry Chin/Globe Staff/Globe Staff Before he climbs into the Mustang, leaves Lancaster, and heads to a workout, Marcus pulls up to a single-story, three-bedroom brick home on Princeton Drive where his family lived before leaving Lancaster for good. He was in sixth or seventh grade then. He and his brother still got in trouble over at the 1500 block, which was a walk or a bike ride away. As he stands in the road, a man exits the house, short and upbeat with a country accent. They make small talk, and the man asks Marcus what he hopes to achieve in the NBA. “Just to have a long career,” he says. “That’s it.” It is a simple answer, but there are layers there. He is playing for something he values in a way few others ever will, something that is fleeting, something that ran out too soon for those he loved. He is playing for time, as much as possible, for him, for them. Baxter Holmes can be reached at [email protected]. Follow him on Twitter @BaxterHolmes.
Mid
[ 0.6157303370786511, 34.25, 21.375 ]
Acute haemorrhagic keratoconjunctivitis following laser in situ keratomileusis. We report two cases of acute haemorrhagic keratoconjunctivitis which occurred following laser in situ keratomileusis (LASIK) during an ongoing epidemic. Both cases underwent preoperative investigation and surgery on the same day. The possible sources of contamination include the paramedical staff, the contact instruments used for performing preoperative investigation, surgeon, nurse, surgical instruments and eye drops. However, the flap was intact with no haze or regression and at 1 year follow up, the visual acuity was maintained at 6/6 in both the patients. We recommend greater caution while performing contact investigations and strict surgical asepsis during LASIK surgery, routinely as well as during epidemics of conjunctivitis.
High
[ 0.661698956780923, 27.75, 14.1875 ]
The popularity of social network services is ever increasing. For example, users may leverage a social network service to find and interact with other users, such as friends, business contacts, and so on. This interaction may be performed in a variety of ways, such as through messaging, photo sharing, posting videos, and so on. However, the ever increasing popularity of the social network services has also resulted in an ever increasing number and variety of social network services. Consequently, it has become more and more difficult for the users to locate and interact with each other across these different social network services, which may be frustrating to the users and foil the social interaction supported by the social network services.
Low
[ 0.5275423728813561, 31.125, 27.875 ]
Crest Complete Multi-Care Toothpaste - reviews I always buy Crest toothpaste, and recently I have run across the Cinnamon multi-benefit plus whitening toothpaste. I have to say, I really like this product. I am not a huge fan of mint, so to find a toothpaste in cinnamon was a huge plus. It is a bold and spicy clean that foams just like other … Read more I admittedly am very particular when it comes to dental care products for our family, and Crest is our preferred brand of toothpaste. It is difficult to find a toothpaste that can take care of all aspects of oral care such as cavity protection, tartar control, and whitening. Read more When it comes to toothpaste I buy whatever catches my eye on the shelf first. I will admit I am not very picky about which toothpaste I buy. However I must say I have always been pleased with the Crest toothpaste line and this toothpaste is no exception. So what do I like about this one? Read more
Low
[ 0.53469387755102, 32.75, 28.5 ]
White Men For You In Atkinson, Date Who You Want Don't Give Up. Discover Sexy Single White Men Today. Try It! Meeting the one has never been easier than when you do it online with DateWhoYouWant. We have profiles from a wide range of single White men in Atkinson. Create a free account at DateWhoYouWant today and browse the profiles of members from all over the world. All of the members at DateWhoYouWant have free choice of who they'd like to interact with before they choose whether or not to meet in person. Dating shouldn't be a chore. DateWhoYouWant makes it a breeze to find single White men in Atkinson - you just need to sign up with us to see if there is anyone on there for you! When you choose to opt into a free DateWhoYouWant account, you are given the freedom to create a profile for yourself, browse other member's profiles and make a decision whether to upgrade. If you decide you like our services, upgrading is so easy. I am an individual who knows what he wants in life and from a relationship, I am 20 but I find myself out of place with people my own age due to the fact that they choose to act like idiots, especiall... more Intelligent guy seeks black female for romance.Hi, I consider myself an intelligent, open-minded, and optimistic person. I love good conversation and would appreciate a woman who can converse easily and freely. I particularly like to converse with... more Laughing, smiling, living, cherishing each dayFun-loving and open to the innumerable experiences to be had in our vast and wonderful world! Love exploring, trying new things, going new places, meeting new people, and immersing in different cultur... more Ever dance with the Devil in the pale moonlight?Always looking for an excuse to go out and enjoy the outdoors. If not then you can find me reading, writing or sketching away the day. A very big dog lover ( Great Danes are the love of my life. ) ... more I’ve been compared visually to Liam Neeson, but I am a hard working person who has had to reinvent himself a couple of times, but I think that it makes me a better person because of it. I enjoy Boston even though I live in the burbs. I love to swim... more smile, it looks good on youMedical professional in the Air Force, love winter and all the sports it has to offer. I am not christian or religious at all, i am an agnostic atheist...that seems to matter to some... no i'm not on ... more
Low
[ 0.42794759825327505, 24.5, 32.75 ]
Epigallocatechin gallate inhibits phorbol ester-induced activation of NF-kappa B and CREB in mouse skin: role of p38 MAPK. The modulation of intracellular signaling network involved in an inappropriate expression of cyclooxygenase-2 (COX-2) is a pragmatic approach for chemoprevention with a wide variety of dietary phytochemicals. Epigallocatechin gallate (EGCG), a major green tea polyphenol, is one of the most extensively investigated chemopreventive agents. Our previous study revealed that EGCG inhibited expression of COX-2 and activation of mitogen-activated protein kinases (MAPKs) in mouse skin stimulated with a prototype tumor promotor 12-O-tetradecanoylphorbol-13-acetate (TPA). This study was aimed at identifying transcription factors as molecular targets of EGCG in downregulating COX-2 expression. We found that EGCG inhibited TPA-induced DNA binding of NF-kappaB and CREB in mouse skin in vivo. EGCG also suppressed TPA-induced phosphorylation and subsequent degradation of IkappaBalpha, and prevented nuclear translocation of p65. We also examined whether extracellular signal-regulated protein kinase (ERK) and p38 MAPK, which are known to regulate activation of NF-kappaB, can also modulate CREB DNA binding. Pretreatment with U0126 and SB203580, pharmacological inhibitors of ERK and p38 MAPK, respectively, showed that SB203580, but not U0126, attenuated TPA-induced CREB DNA binding in mouse skin. Taken together, EGCG inhibited TPA-induced DNA binding of NF-kappaB and CREB by blocking activation of p38 MAPK, which may provide a molecular basis of COX-2 inhibition by EGCG in mouse skin in vivo.
High
[ 0.7111756168359941, 30.625, 12.4375 ]