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et tube terminates appropriately above the carina. there is a non-displaced left <num>th rib fracture. left lower lobe consolidation is concerning for pneumonia.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18001923/s52887706/7c6d7014-d218b5e5-4c2a229f-7ccc5771-6fbc19dc.jpg
mild pulmonary vascular congestion. no focal consolidations concerning for pneumonia identified.
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no focal consolidation concerning for pneumonia.
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normal chest radiograph. no pneumonia.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10157506/s56637894/f631ed1d-c047c292-459853f2-6aaab768-cfaa30b3.jpg
basilar atelectasis, without convincing signs of pneumonia.
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no acute findings in the chest.
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endotracheal tube terminates <num> cm above the carina, in appropriate position. enteric tube has been withdrawn slightly, terminating at the gastroesophageal junction. recommend advancement so that it is well within the stomach. interval placement of right ij central venous catheter terminates in the mid to lower svc without evidence of pneumothorax.
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no acute cardiopulmonary abnormality.
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suboptimal patient positioning limits assessment however there appears to be an increase in the degree of pulmonary edema when compared to the prior study. the tip of the right-sided picc cannot be evaluated but is likely in the right atrium.
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no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13412043/s56584202/c5faa34f-dc60c44e-9b73be76-7411f12f-d9bc41c5.jpg
no pneumonia.
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no evidence of acute cardiopulmonary disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11327070/s50001886/55d370ca-bdd36532-d321a013-50ad1c19-867da3cf.jpg
small bilateral pleural effusions. no focal consolidation or pneumothorax.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16562665/s54027444/943fbfce-ddfef92d-e56b1d78-53136a25-e057fc0d.jpg
moderate to large right pneumothorax with leftward mediastinal shift.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15528228/s53404768/fd50af19-a74e5bcb-8ec60e65-21d70a8d-00ea5add.jpg
no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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bilateral pleural effusions, right greater than left. new consolidation projects over the right lower lung zone worrisome for infection. prominent central pulmonary vasculature persists without overt pulmonary edema.
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no acute cardiopulmonary abnormality.
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bilateral pleural effusions with likely loculated component along the right major fissure. pulmonary vascular congestion. cardiomegaly.
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opacities in the lower lungs suggesting pneumonia.
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near-complete opacification of the right hemithorax with an area of aerated lung seen projecting over the right upper hemithorax. shift of the mediastinum to the left suggests pleural effusion. underlying consolidation is difficult to exclude.
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no acute cardiopulmonary process.
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increased right basilar atelectasis, with a small right pleural effusion.
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no evidence of acute cardiopulmonary process.
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small bilateral pleural effusions and cardiomegaly.
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unchanged small right apical pneumothorax.
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vague left infrahilar density, possibly associated with lower airway inflammation or infection, or potentially early or mild bronchopneumonia, but not of definite significance.
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endotracheal tube somewhat low in position, terminating <num> cm above the level of the carina, recommend withdrawal by approximately <num>-<num> cm for more optimal positioning. enteric tube courses below the level of the diaphragm into the left abdomen, distal aspect not included on the image. right greater than left perihilar and infrahilar opacities may be due to asymmetric pulmonary edema however, underlying aspiration and/or infection may be present. the above findings were discussed with dr. <unk> at <time> p.m. on <unk> by dr. <unk> via telephone. splaying of the carina which can be seen with left atrial enlargement.
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no active cardiopulmonary disease.
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no acute cardiopulmonary process.
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normal chest radiograph, without focal consolidation concerning for pneumonia.
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<num>. left lower lung atelectasis, less likely. <num>. increased intravascular volume, no pulmonary edema.
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no evidence of acute cardiopulmonary disease.
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interval improvement in left pleural effusion and lung volumes.
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normal chest radiographs.
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slightly low lung volumes with mild bibasilar atelectasis.
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no left pneumothorax is convincingly demonstrated.
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enteric tube coils within the expected location of the cervical esophagus. tip is not visualized.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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low lung volumes with bibasilar atelectasis. elevation of the right hemidiaphragm is of unknown chronicity. comparison with previous radiographs is recommended.
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emphysema. mild bibasilar atelectasis.
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peribronchial opacities, likely in the left lower lobe are concerning for infection.
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<num>. enlargement of the main pulmonary artery suggesting pulmonary hypertension. <num>. markedly enlarged cardiac silhouette, pleural effusions, and prominent vascular structures suggest fluid overload due to cardiac decompensation.
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no opacity convincing for pneumonia.
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findings suggesting mild vascular congestion or fluid overload, without definite evidence for injury.
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normal chest radiographs.
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no acute cardiopulmonary process.
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mild pulmonary vascular congestion and trace bilateral pleural effusions. probable mild bibasilar atelectasis.
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top normal heart size without signs of edema or pneumonia.
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right picc ends in the upper svc. no pneumothorax.
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no acute cardiopulmonary process.
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mild interstitial edema and persistent bilateral effusions. rounded opacity projecting over the right lung apex, potentially summation of shadows however nonurgent repeat pa suggested when patient is amenable to exclude underlying parenchymal lesion.
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no acute intrathoracic process
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near-complete resolution of previously noted diffuse parenchymal opacities. no new focal consolidation to suggest pneumonia.
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normal chest radiographs.
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no evidence of injury.
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low lung volumes with scattered atelectasis. please refer to concurrently performed ct abdomen pelvis regarding findings of pulmonary emboli.
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no acute intrathoracic process.
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increasing opacity at the right lung base may reflect pneumonia in the proper clinical context. relatively unchanged appearance of the left lower lobe and retrocardiac opacity.
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no acute cardiopulmonary abnormality.
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no definite acute cardiopulmonary process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15931189/s51870573/16ccf106-5fd7c9a3-57beb44e-dab5225e-544a8a6d.jpg
no acute cardiopulmonary process.
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as above.
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<num>. no evidence of pneumonia. <num>. conventional chest radiography is not sensitive for detection of rib fractures.
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<num>. et tube should be pulled back <num>-<num> cm. <num>. right ij should be pulled back <num>-<num> cm. <num>. left retrocardiac opacification, which likely represents atelectasis.
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no acute cardiopulmonary abnormality.
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no acute process.
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no significant interval change when compared the prior study.
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no acute cardiopulmonary abnormality.
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consecutive radiographs initially show worsening of the known left pneumothorax and near complete atelectasis of the left lung with subsequent re-expansion of the left lung on the most recent radiograph. the left pneumothorax has resolved, and there is now left mid lung subsegmental atelectasis and re-expansion pulmonary edema. the patient has been intubated with the et tube terminating at the level of the clavicles. the tiny right apical pneumothorax has resolved.
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mild interstitial edema. grossly stable right perihilar mass, remains concerning for malignancy.
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no change.
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given the patient's immunocompromised status, the above findings are most consistent with pcp <unk>. findings were discussed with dr. <unk> at <unk>:<num> on <unk>.
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no evidence of acute cardiopulmonary disease.
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dobbhoff tube is still in a relatively high position with the tip just beyond the gastroesophageal junction and could be advanced for more optimal placement.
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tip of the left picc projects over the low svc.
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minimal bibasilar opacities likely represent atelectasis, but infection cannot be excluded in the appropriate clinical setting.
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<num>. hyperinflation, likely due to emphysema. <num>. diffuse interstitial abnormality is likely due to a fibrotic interstitial lung disease. <num>. no evidence of pneumonia.
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mild pulmonary vascular congestion without frank edema.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19181182/s56431557/611e9202-d1e0da6a-bd02a297-29a9e5d8-773f878f.jpg
mild bibasilar patchy opacities likely reflective of atelectasis.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14274761/s59129181/0808a350-e3357e06-0d0022b3-843a5116-7dd573c8.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15909503/s56632657/ade05395-7e7b39fb-eb9db243-82857529-d67b7484.jpg
no acute cardiopulmonary process.
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increasing right pleural effusion and pulmonary edema, exaggerated by low lung volumes status post extubation.
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right lower lobe pneumonia. interval removal of the endotracheal tube and enteric tube.
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no acute findings in the chest.
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no focal consolidations concerning for pneumonia identified.
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no acute cardiopulmonary process. no evidence of a large hiatal hernia.
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no acute cardiopulmonary process.
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predominantly interstitial lung pattern which raises concern for atypical pneumonia versus fluid overload. multiple compression deformities of the thoracic spine of unknown chronicity.
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no acute cardiopulmonary process. if high clinical concern, dedicated rib series could be considered.
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no acute findings in the chest. please refer to subsequent cta chest for further details.
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no acute intrathoracic process.
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no pneumonia.
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retrocardiac opacity which is most compatible with a hiatal hernia.