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proper interval positioning of an ng tube.
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<num>. no evidence of pneumonia. chronic biapical scarring and pleural thickening is stable. <num>. mild cardiomegaly is stable.
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mild interstitial abnormality with mild bronchial wall inflammation. this may reflect early edema. no focal consolidation convincing for pneumonia is identified.
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scattered opacities with a lungs are concerning for worsening metastatic disease. please correlate clinically.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16099802/s58015061/027af72e-837dc230-10ded8e2-e84cf5fe-4da44960.jpg
mild to moderate pulmonary edema.
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no acute cardiopulmonary process.
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<num>. increased area of opacity in the right upper lobe and left perihilar region, concerning for pneumonia. <num>. moderate right pleural effusion with compressive atelectasis. findings were communicated with dr.<unk> by dr.<unk> <unk> telephone at <time>pm on <unk>.
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small-to-moderate bilateral pleural effusions with overlying atelectasis along with interstitial edema consistent with fluid overload.
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normal chest radiograph.
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bilateral pleural effusions, slightly increasing and suggestive of chf. left-sided retrocardiac atelectasis persists and possibility of infective course is likely. no other interval changes are seen.
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no acute cardiopulmonary process.
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stable chest findings. no radiographic evidence of increasing pulmonary congestion or acute infiltrates during the last five-month examination interval.
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no acute cardiopulmonary process.
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cardiomegaly. the cardiac silhouette has also likely increased since prior which raises possibility of superimposed pericardial effusion. decreased degree of pulmonary edema compared to prior.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no evidence of acute intrathoracic process.
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unremarkable chest radiographic examination.
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decreased moderate bilateral pleural effusions following pigtail catheter drainage. no definite pneumothorax. unchanged bilateral lower lobe collapse.
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no evidence of acute disease. no free air identified. normal bony structures.
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<num>. no acute intrathoracic process. <num>. copd.
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no definite evidence of acute cardiopulmonary process.
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no signs of free air.
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low lung volumes. no evidence of pneumonia.
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right hemidiaphragm elevation with opacification posteriorly suggesting extensive adjacent lung atelectasis, though cannot exclude developing infectious process. possible right pleural effusion as well. if findings do not resolve on subsequent radiography, evaluation with chest ct could be considered, preferably with intravenous contrast if possible.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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top-normal to mildly enlarged cardiac silhouette. no pulmonary edema.
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congestion and mild edema. limited exam.
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hyperinflated lungs with coarsened lung markings compatible with known emphysema. unfolded thoracic aorta.
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no acute cardiopulmonary process.
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endotracheal tube low in position, coursing into the right mainstem bronchus. subsequent chest radiograph demonstrates withdrawal above the level of the carina. enteric tube courses below the level of the diaphragm, inferior aspect data the field of view. the lung volumes. increased perihilar opacities concerning for pulmonary edema and/or aspiration given clinical scenario.
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similar prominence of central pulmonary vessels without frank congestion. possibly a trace new pleural effusion on the right.
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mild right basilar atelectasis.
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no acute cardiopulmonary abnormality. no subdiaphragmatic free air.
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dobbhoff tube should be pushed down at least <num>-<num> cm. monitoring devices are unchanged in position. persists minimal bibasilar atelectasis.
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normal chest radiographs.
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no acute cardiopulmonary abnormality.
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unchanged small left effusion with improved right trace effusion and pulmonary edema
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no pulmonary infiltrates or other acute cardiopulmonary process.
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unchanged moderate to severe cardiomegaly with slightly improved mild pulmonary edema.
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mild bibasilar atelectasis. no evidence of acute cardiopulmonary process.
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<num>. right mainstem bronchus intubation. retraction by <num>-<num> cm is recommended. <num>. advancement of the og tube by at least <num>-<num> cm would result in more optimal positioning. <num>. mild left basal atelectasis
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the tip of the nasogastric tube extends into the stomach.
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no acute cardiopulmonary abnormality.
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endotracheal tube terminates <num> cm above the level of the carina. nasogastric tube is high in position, terminating in the distal esophagus ; recommend advancement so that it is well within the stomach.
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no acute cardiopulmonary process.
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<num>. linear plate like atelectasis in the left lower lobe. <num>. heterogeneous opacity in the right lower lobe is most consistent atelectasis however superimposed infection cannot be excluded. of note ct is more sensitive in detection of atypical infection in immunocompromised patients.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process. of note, conventional radiographs are not sensitive for evaluation of traumatic injury. repeat imaging with a marker at the site of patient's pain and lateral views may be helpful.
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no signs of pneumoperitoneum. no acute findings.
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right upper lobe consolidation compatible with pneumonia in the proper clinical setting. repeat after treatment is suggested to document resolution.
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multiple bilateral pulmonary nodules consistent with patient's known pulmonary amyloid disease as seen previously. there is no new opacity suggestive of a focal infection.
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increased aeration in the left upper lung and resolution of mediastinal shift status post bronchoscopy. mild fluid overload, with moderate right and small left pleural effusions.
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new left lung near complete collapse with increased ipsilateral mediastinal shift. finding may be due to migration of mucus plugging after referenced brochoscopy.
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normal chest radiograph.
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fissural fluid, likely accounts for subtle opacity at the right lung base. otherwise, unremarkable.
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no evidence of pneumonia.
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new left pleural effusion and bibasilar atelectasis. ct scan could be considered for further evaluation of new pleural effusion etiology.
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possible trace pleural effusions versus pleural thickening accounting for blunted cp angles bilaterally. no signs of pneumonia.
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as above.
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improved right ventilation with persistent pleural effusion.
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bibasilar atelectasis without definite focal consolidation.
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findings suggesting pneumonia in the left lower lobe. follow-up radiographs are recommended to document resolution within <unk> weeks.
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hyperinflated, but clear lungs.
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right jugular catheter positioning with tip ending in atriocaval junction. no sign of acute cardiopulmonary process.
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interval improvement of the left lung collapse.
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mild bibasilar atelectasis
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no acute intrathoracic process.
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<num>. endotracheal tube terminating <num> cm above the level of the carina, slightly low in position and recommend withdrawal by <num> to <num> cm. this finding and recommendation discussed with dr. <unk> on <unk> via telephone at <time>pm. <num>. nasogastric tube in appropriate position. <num>. subtle right upper lobe opacity, nonspecific, could be due to aspiration or infection.
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unchanged exam. bibasilar atelectasis.
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cardiomegaly with pulmonary edema and small bilateral effusions.
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no acute cardiopulmonary process. no pneumothorax.
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no acute intrathoracic process.
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no acute intrathoracic abnormality.
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no appreciable change in moderate right pleural effusion and right basilar subsegmental atelectasis.
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no acute cardiopulmonary abnormality.
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increased central peribronchial markings raising possibility of inflammation without evidence of focal consolidation.
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no acute cardiopulmonary process.
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left picc in the low svc/cavoatrial junction
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<num>. moderate right pleural effusion with consolidation which may represent atelectasis and/or infection. <num>. small left pleural effusion with lll consolidation.
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no substantial interval change from the prior radiograph. continued moderate size left and small right pleural effusions with bibasilar opacities, likely atelectasis, but infection cannot be completely excluded. diffuse pulmonary and pleural nodules compatible with metastases as well as lymphangitic carcinomatosis as seen previously.
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focal opacity at the left lung base laterally, potentially atelectasis, although infection cannot be entirely excluded. clinical correlation is suggested.
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improvement in pulmonary edema.
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endotracheal tube tip approximately <num> cm above the carina.
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<num>. asymmetric opacity within the right lung apex. apical lordotic views are recommended to assess whether this reflects a true pulmonary lesion. <num>. no acute cardiopulmonary abnormality otherwise detected.
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no acute cardiopulmonary abnormality.
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no pneumonia, edema or effusion.
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right upper and lower lung field patchy opacities may reflect areas of infection, but appear improved compared to the previous radiograph. moderate left pleural effusion appears mildly increased from prior, with continued left basilar compressive atelectasis.
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<num>. enteric tube is coiled within the stomach and terminates above the level of the ge junction and should be repositioned before use. <num>. bibasilar opacities, right greater than left are likely representative of atelectasis.
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<num>. enteric catheter ends within the superior aspect of the gastric fundus. <num>. low lung volumes, with minimal bibasilar atelectasis.
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no acute cardiopulmonary abnormality.
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no pneumonia, edema or effusion.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no evidence of pneumothorax.
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no acute cardiopulmonary process.
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moderate left and small right pleural effusions. background pulmonary edema, similar to prior.
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patchy and linear right basilar opacity probably represents atelectasis and less likely a focus of aspiration or early pneumonia. followup radiograph may be helpful in this regard.