anchor.diff:  				            			</li>
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                               		<img alt="H&E stained Rsph9" src="${baseUrl}/img/vignettes/Rsph9Slides.png" >H&E stained coronal sections of P7 mice revealed enlarged ventricles and blocked sinuses in the Rsph9tm1.1/tm1.1 mutant mice. 
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   				            	<div id="rsph9Viable" style="display: none">
  
positive.code:                         
                              <div class="section">
                              <c:set var="geneId" value="MGI:1922814"></c:set>
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                              	<h2 id="${geneId}"><t:formatAllele>Rsph9
  <tm1.1(KOMP)Vlcg></t:formatAllele></h2></h2>
                              	
                              	
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  				            		<p>
  				            		Radial spoke head protein 9 is a component of the radial spoke head in motile cilia and flagella.
  Rsph9 mutants showed partial pre-weaning lethality but viable to P7.
  Whole brain MRI and H&E staining of coronal sections of the P7 brain revealed severe hydrocephaly of the left and right lateral ventricles of the Rsph9 mutant.
  Coronal section through the nasal region showed that the sinuses of the Rsph9 mutants were filled with pus (asterisks).
  Both hydrocephaly and nasal blockage are phenotypes associated with Primary Ciliary Dyskinesia in humans.
  </p>
  				            		
  				            		<p>Phenotype data links</p>
  				            		<ul>
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negative.code: 							<h2 class="title" id="${geneId}"><t:formatAllele>Acvr2a<tm1.1(KOMP)Vlcg></t:formatAllele></h2>
  							
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                           			<p>
                           				Activin receptor IIA is a receptor for activins, which are members of the TGF-beta superfamily involved in diverse biological processes.<br>
  										Acvr2a mutants are subviable with most pups dying before postnatal day 7. Micro-CT analysis at E15.5 revealed variable penetrance of eye and craniofacial abnormalities. Eye phenotypes varied from normal (Embryo 1- (E1)), to underdeveloped (E2), to cyclopic (E3), to absent (E4). Craniofacial phenotypes varied from normal (E1) to narrow snout (E2), to an elongated  snout missing the mandible and tongue (E3, 4) and low set ears (E2, 3, 4).
  