anchor.diff:                              	<div class="twothird">
                            			<p>
                            				The Kldhc2 gene is located within a locus linked to an automsomal dominant disease that leads to fibro-fatty replacement of right ventricle myocardium leading to arrythmias (ARVD3 ; OMIM).<br>
  -                         				The gene is expressed in <a href="https://www.ebi.ac.uk/gxa/experiments/E-MTAB-3358?accessKey=&amp;serializedFilterFactors=DEVELOPMENTAL_STAGE:adult&amp;queryFactorType=ORGANISM_PART&amp;rootContext=&amp;heatmapMatrixSize=50&amp;displayLevels=false&amp;displayGeneDistribution=false&amp;geneQuery=KLHDC2&amp;exactMatch=true&amp;_exactMatch=on&amp;_queryFactorValues=1&amp;specific=true&amp;_specific=on&amp;cutoff=0.5">heart</a> (expression atlas link) and has been implicated in  <a href="http://www.ncbi.nlm.nih.gov/pubmed/16008511">endothelial differentation</a> and 
  +                         				The gene is expressed in <a href="https://www.ebi.ac.uk/gxa/experiments/E-MTAB-3358?accessKey=&amp;serializedFilterFactors=DEVELOPMENTAL_STAGE:adult&amp;queryFactorType=ORGANISM_PART&amp;rootContext=&amp;heatmapMatrixSize=50&amp;displayLevels=false&amp;displayGeneDistribution=false&amp;geneQuery=KLHDC2&amp;exactMatch=true&amp;_exactMatch=on&amp;_queryFactorValues=1&amp;specific=true&amp;_specific=on&amp;cutoff=0.5">heart</a> (expression atlas link) and has been implicated in  <a href="http://www.ncbi.nlm.nih.gov/pubmed/16008511">endothelial differentation</a> and
                            				<a href="http://www.ncbi.nlm.nih.gov/pubmed/16860314">myoblast differentation</a>. Heterozygote null mice have abnormal heart rhythms while the lethal ​embryos may have a heart defect.
                           			</p>
  -                        			
  -                        			<p>Phenotype data links</p>
  -                        			
  +
  +                        			<p>Phenotype data links</p>
  +
                               		<ul>
                               			<li>
                               				Viability:<a href="${baseUrl}/charts?accession=MGI:1916804&allele_accession_id=MGI:5548587&zygosity=homozygote&parameter_stable_id=IMPC_VIA_001_001&pipeline_stable_id=HRWL_001&phenotyping_center=MRC%20Harwell" target="_blank">Complete preweaning lethality</a>
  
positive.code: 				            			<li>
  				            				Adult and Embryo Phenotype Data: <a href="${baseUrl}/genes/${geneId}#section-associations">Table</a>
  				            			</li>
  				            			
  				            		</ul>
  								</div>
                              	<div class="half"><img alt="Tox3 MRI" src="${baseUrl}/img/vignettes/tox3MRI.png" >Caudal to rostral coronal sections of whole brain MRI with automated volume analysis revealed P7 Tox3tm1b/tm1b mutant mice had smaller (blue) and larger (red) tissues compared to the Tox3+/+ wildtype average.
  </div>
  				            		<div class="clear"></div>		
  				            		
  				            		
  				            	<div id="tox3Viable" style="display: none">
  				            		<h3>P3/P7 viability test Tox3</h3>
  				    
  				            		<img src="${baseUrl}/img/vignettes/Tox3Table.png">		
  	                           	</div>
  	                           	
  	                       <div id="tox3His" style="display: none">
  	                       		<img src="${baseUrl}/img/vignettes/Tox3Sections.png">
  	                       		<p>
  	                       			H&E stained coronal section through the brain of a Tox3+/+ wildtype embryo compared to a Tox3tm1b/tm1b mutant embryo indicated that the mutant embryo had a hypoplastic and dysplastic cerebellum (CE) with markedly reduced fissure formation. Higher magnification revealed that the transient external granular layer was absent in the Tox3tm1b/tm1b mutant  mice and the subjacent molecular layer was hypotrophic and irregular in thickness (arrow).
  	                       		</p>
  	                       </div>
  	                           
  	                           
  	                           
                              </div>
                          </div>
                          
                          
                          
                          
                          
                              <div class="section">
                              <c:set var="geneId" value="MGI:1922814"></c:set>
                              <h2 id="${geneId}"><t:formatAllele>Rsph9<tm1.1(KOMP)Vlcg></t:formatAllele></h2>
  
negative.code:                             <div id="1" class="inner">
                              <c:set var="geneId" value="MGI:1913761"></c:set>
                              	<%-- <a href="${baseUrl}/genes/${geneId}"> --%><h2 id="${geneId}"><t:formatAllele>Chtop<tm1a(EUCOMM)Wtsi></t:formatAllele></h2><!-- </a> -->
  
  
                              	<div  class="twothird">
  				            		<p>
  				            		Chtop has been shown to recruit the histone-methylating methylosome to genomic regions containing 5-Hydroxymethylcytosine, thus affecting gene expression.<br>
  
  
  				            		Chtop mutants showed complete preweaning lethality with no homozygous pups observed. High resolution episcopic microscopy (HREM) imaging at E14.5 revealed multiple phenotypes including edema, abnormal forebrain morphology and decreased number of vertebrae and ribs.<br>
  				            		</p>
  				            		<p>Phenotype data links</p>
  