anchor.diff:  								Gross morphology at E9.5 revealed that <t:formatAllele>Tmem100<tm1e.1/tm1e.1></t:formatAllele> mutant embryos have a large pericardial effusion with cardiac dysmorphology and enlargement (arrow).
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                               <c:set var="geneId" value="MGI:1337104"></c:set>
   							<h2 class="title" id="${geneId}"><t:formatAllele>Eya4<tm1b(KOMP)Wtsi></t:formatAllele></h2>
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positive.code:                             	<div class="twothird">
                           			<p>
                           				The Kldhc2 gene is located within a locus linked to an automsomal dominant disease that leads to fibro-fatty replacement of right ventricle myocardium leading to arrythmias (ARVD3 ; OMIM).<br>
                           				The gene is expressed in <a href="https://www.ebi.ac.uk/gxa/experiments/E-MTAB-3358?accessKey=&amp;serializedFilterFactors=DEVELOPMENTAL_STAGE:adult&amp;queryFactorType=ORGANISM_PART&amp;rootContext=&amp;heatmapMatrixSize=50&amp;displayLevels=false&amp;displayGeneDistribution=false&amp;geneQuery=KLHDC2&amp;exactMatch=true&amp;_exactMatch=on&amp;_queryFactorValues=1&amp;specific=true&amp;_specific=on&amp;cutoff=0.5">heart</a> (expression atlas link) and has been implicated in  <a href="http://www.ncbi.nlm.nih.gov/pubmed/16008511">endothelial differentation</a> and 
                           				<a href="http://www.ncbi.nlm.nih.gov/pubmed/16860314">myoblast differentation</a>. Heterozygote null mice have abnormal heart rhythms while the lethal ​embryos may have a heart defect.
                          			</p>
                          			
                          			<p>Phenotype data links</p>
                          			
                              		<ul>
                              			<li>
                              				Viability:<a href="${baseUrl}/charts?accession=MGI:1916804&allele_accession_id=MGI:5548587&zygosity=homozygote&parameter_stable_id=IMPC_VIA_001_001&pipeline_stable_id=HRWL_001&phenotyping_center=MRC%20Harwell" target="_blank">Complete preweaning lethality</a>
  
negative.code: 				            				 				<a href="${drupalBaseUrl}/phenoview/?gid=801-61-7&qeid=IMPC_GPO_006_001,IMPC_GPO_001_001,IMPC_GPO_002_001,IMPC_GPO_004_001,IMPC_GPO_003_001">E14.5</a>
  				            			</li>
                              			<li>
                              				Embryo Gross Morphology: <a href="${drupalBaseUrl}/phenoview/?gid=801-61-7&qeid=IMPC_GEM_049_001">E12.5</a>,
                              											<a href="${drupalBaseUrl}/phenoview/?gid=801-61-7&qeid=IMPC_GEO_050_001">E14.5-E15.5</a>,
                              											<a href="${drupalBaseUrl}/phenoview/?gid=801-61-7&qeid=IMPC_GEP_064_001">E18.5</a> images
                              			</li>
                              			<li>
                              				3-D Imaging: <a href="${drupalbaseUrl}/phenoview/?gid=801-61-7&qeid=IMPC_EMO_001_001">Images</a>
                              			</li>
                              			<li>
                              				Adult and Embryo Phenotype Data: <a href="${baseUrl}/genes/${geneId}#section-associations">Table</a>
                              			</li>
                              		</ul>
  
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                              		<img alt="E15.5 Cox7c embryo"src="${baseUrl}/img/vignettes/Cox7c.jpg">
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                          <div class="section">
                          <c:set var="geneId" value="MGI:1920939"></c:set>
  							<h2 class="title" id="${geneId}">
  								<t:formatAllele>Bloc1s2<tm1.1(KOMP)Mbp>
  								</t:formatAllele>
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                           				Biogenesis of lysosomal organelles complex 1, subunit 2 is a component of the
  										BLOC�1  complex,  which  functions  in  the  formation  of  lysosome�-related
  										organelles,  is  implicated  in  synapse  function,  and  is  associated  with  gamma-
  										tubulin  and  the  centrosome  [1].  Homozygous  mutants  show  complete
  										preweaning lethality, with embryonic lethality occurring around E15.5. Surviving
  										mutants  at  E15.5  show  edema,  hemorrhage,  and  abnormal  cardiovascular
  										development  (Fig  1). MicroCT  datasets  of  E15.5  embryos  also  reveal  lung
  										hypoplasia,  enlarged right  atrium,  and  compromised right  ventricle  of  the  heart
  										(Fig.1, arrow). Adult heterozygotes show abnormal immunophenotypes.
  									</p>
  									<p><i>[1] Falc�n-Perez et al., BLOC-1, a novel complex containing the pallidin and muted proteins involved in the biogenesis of melanosomes and platelet-dense granules, J Biol Chem 2002</i></p>
  
                          			<p>Phenotype data links</p>
  
                              		<ul>
                              			<li>
                              				Viability: <a href="${baseUrl}/charts?accession=${geneId}&allele_accession_id=MGI:5490495&zygosity=homozygote&parameter_stable_id=IMPC_VIA_001_001&pipeline_stable_id=JAX_001&phenotyping_center=JAX" target="_blank">Complete preweaning lethality</a>
  