anchor.diff:                              	<div class="half">
                            			<p>
                            				Rab34 is a member of the RAS oncogene family, which are small GTPases
  -										involved in intracellular vesicle transport. Rab34 is known to be Golgi-bound, involved lysosomal positioning. Rab34 is a potential target of Gli1 and a possible 
  -										component of hedgehog signaling. The Rab34 knockout is the first reported null allele for this gene, resulting in complete preweaning lethality.  Phenotypes 
  -										include patterning defects, such as polydactyly and facial clefting, as well as abnormal eye development and severe lung hypoplasia (Fig  1,  lu =  lung).. The mutants are 
  +										involved in intracellular vesicle transport. Rab34 is known to be Golgi-bound, involved in lysosomal positioning. 
  +										Rab34 is a potential target of Gli1 and a possible 
  +										component of hedgehog signaling. The Rab34 knockout is the first reported null allele for this gene, 
  +										resulting in complete preweaning lethality.  Phenotypes 
  +										include patterning defects, such as polydactyly and facial clefting, as well as abnormal eye 
  +										development and severe lung hypoplasia (Fig  1,  lu = lung). The mutants are 
   										subviable at E18.5, lethality presumably occurring perinatally.
   									</p>
   									                        			
  
positive.code:                             	<div class="half">
                           			<p>
                           				Biogenesis of lysosomal organelles complex 1, subunit 2 is a component of the
  										BLOC1  complex,  which  functions  in  the  formation  of  lysosomerelated 
  										organelles,  is  implicated  in  synapse  function,  and  is  associated  with  gamma-
  										tubulin  and  the  centrosome  [1].  Homozygous  mutants  show  complete 
  										preweaning lethality, with embryonic lethality occurring around E15.5. Surviving 
  
negative.code: 										Homozygous mutants show complete preweaning lethality, with embryonic lethality occurring after 										
  										E18.5. Hemorrhaging is seen in surviving E18.5 mutants, as is severe edema and small embryo size 										
  										(Fig 1, left). Among other defects, microCT analysis 	reveals brain defects, lung hypoplasia and absent										
  										renal pelvis in the kidney (Fig 1, middle, right). Phenotypes of heterozygotes include abnormal body composition										
  										and abnormal blood chemistry.
  									</p>
                          			
  