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left basilar opacities, likely atelectasis, but pneumonia in the correct clinical setting should be considered.
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previously noted bibasilar airspace opacities have nearly resolved, with residual mild right basilar opacity, likely atelectasis, though infection is not completely excluded. previously noted bilateral effusions have also resolved.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11286562/s55286023/905d4947-bc78ee25-4ceae0eb-fdff558d-e5fe9b97.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10903037/s54813929/47d7c4a1-6b813eed-405c8ab9-3b435527-999f78df.jpg
numerous known pulmonary nodules better seen on prior ct chest. no pneumonia or acute cardiopulmonary process.
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<num>. no evidence of amiodarone toxicity. <num>. possible left ventricular aneurysm or worsening dilatation. no evidence of cardiac decompensation (pulmonary edema, vascular congestion, or pleural effusions).
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left lower lobe pneumonia.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18015004/s59622916/02274a21-924828c9-19bb256a-38804736-106b97ca.jpg
no focal consolidation to suggest pneumonia. mild basilar atelectasis.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14787496/s51830383/b495e81c-7f6a094b-cd96137b-337feceb-cfb047ea.jpg
increased interstitial markings suggestive of interstitial edema.
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mild pulmonary vascular congestion and edema. lower lung subtle opacities may reflect atelectasis though difficult to exclude an early pneumonia.
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no acute cardiopulmonary process.loss of height of the l<num> vertebral body is again seen, query slightly progressed compared to the prior study.
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no acute findings in the chest.
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right base opacity and blunting of the right costophrenic angle may be due to combination of consolidation and pleural effusion. consolidation may be due to infection and/or aspiration.
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no acute cardiopulmonary process. hyperexpanded lungs and flattening of the diaphragm, suggestive of copd.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15129946/s55583770/2c26db1a-936cd9ab-91fc485e-555485bb-434b2cc5.jpg
mild pulmonary vascular congestion. no displaced rib fracture seen, although if there is continued clinical concern, a dedicated rib series may be obtained.
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no radiographic evidence of an acute cardiopulmonary process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19538400/s59410325/67c7ce9e-30f960e8-994060dc-6a610f0e-feb9f29c.jpg
interval improvement in pulmonary edema and left pleural effusion. mildly improved right mid lung, basilar opacity.
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low lung volumes. possible small bilateral pleural effusions.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14458255/s54345105/f4906d1a-9def4508-0beadfcb-9af7599a-63f5a8ff.jpg
no acute intrathoracic process.
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no acute intrathoracic process.
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unremarkable chest radiograph. no acute cardiopulmonary process.
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the cardiac silhouette remains enlarged. bibasilar predominant opacities seen on the prior study persists, but appear improved in the interval. right upper lobe pulmonary nodule seen on ct from <unk> was better assessed on ct. mediastinal contours are stable. no pleural effusion or pneumothorax.
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<num>. small left pleural effusion. <num>. leftward deviation of the trachea with prominence of the right superior mediastinal contour could be due to a thyroid goiter, but not grossly changed in the interval. clinical correlation is recommended.
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no evidence of acute cardiopulmonary disease.
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no acute cardiopulmonary abnormality.
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endotracheal tube <num> cm above the carina.
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increased bilateral mid to lower lung opacities are likely worsening pulmonary edema. superimposed right lower lung consolidation cannot be ruled out.
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probable rll pneumonia. new borderline cardiac decompensation.
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no acute intrathoracic process.
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dual lead left-sided pacer remains in place. the lungs appear clear without evidence of focal airspace consolidation, pulmonary edema or pneumothorax. subcutaneous emphysema in the anterior chest wall in the region of the pacemaker best seen on the lateral projection. improved aeration at the right base. trace bilateral pleural effusions.
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no pneumothorax.
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cardiomegaly with mild pulmonary edema. patchy consolidation in the left midlung on the frontal view and over the lung bases on the lateral view likely also localizing to the left which could represent superimposed infection.
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findings most consistent with consolidative pneumonia in the left lower lobe. followup radiographs are recommended to show resolution after treatment in approximately eight weeks.
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<num>. minimal left basal atelectasis. <num>. small bilateral pleural effusions, decreased in size compared to <unk>. <num>. no evidence of aspiration.
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normal chest radiograph.
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hyperinflation without focal consolidation.
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cardiac device lead tips in the right atrium and right ventricle are not significantly changed since prior.
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bronchial inflammation but no evidence of pneumonia.
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<num>. moderate right pleural effusion, extending into the horizontal fissure. <num>. mild pulmonary vascular congestion.
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coarse interstitial marking with basilar predominance, likely reflective of chronic lung disease without evidence of an acute intrathoracic abnormality.
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mild progression of peribronchial infiltration. superimposed infection is also possible.
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persistent opacity within the left lung base, likely reflecting a combination of small pleural effusion and atelectasis, though infection is not excluded.
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no change.
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vague left lower lung opacity, possibly pneumonia; short-term follow-up radiographs may be useful to reassess if clinically indicated.
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mild pulmonary edema, improved since prior imaging.
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no acute cardiopulmonary process. no pneumothorax identified.
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no acute cardiopulmonary process.
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no significant interval change. no focal consolidation to suggest pneumonia.
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no evidence of acute cardiovascular or pulmonary abnormalities on standard pa and lateral chest view. previously existing left lower lobe atelectasis and suspicious pleural effusion is not present anymore.
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new increased opacity with silhouetting of the lateral border of the descending aorta, suggestive of a consolidation in the retrocardiac region, which in the appropriate setting may represent an acute pneumonia.
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low lung volumes. mild pulmonary vascular congestion. no large pleural effusion. no focal consolidation.
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<num>. endotracheal tube appropriately positioned. orogastric tube may be advanced for more optimal positioning. <num>. stable findings in the chest as compared with recent ct.
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no evidence of acute process.
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no acute cardiopulmonary process.
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<num>. no acute cardiac or pulmonary process. <num>. no definite rib fracture. if there is persistent concern for a rib fracture, further evaluation could be performed with a dedicated rib series including an appropriately placed radiopaque skin marker.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13443154/s52742559/99931013-6aac9583-b2675696-0006452d-02dae84d.jpg
no radiographic evidence of apical mass.
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resolved multifocal pneumonia.
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moderate cardiomegaly, though normal pulmonary vasculature. no consolidation.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12431768/s58957737/e168f502-ca9d1e5e-4588c329-542d7b81-21cbd9db.jpg
no focal consolidation. mild increased interstitial markings bilaterally may relate to mild edema and appear decreased compared to the prior study; however, atypical infection is not entirely excluded in the appropriate clinical setting.
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dobbhoff tube below the diaphragm and likely post-pyloric.
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no acute intrathoracic abnormality.
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no acute cardiopulmonary process.
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stable appearance of the chest with no acute process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16277559/s55430972/88c1abb2-54c2e811-bbcde8da-b78f2cec-905b6699.jpg
hazy right basilar opacity partially obscuring the right costophrenic angle suggesting a small effusion. otherwise, no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10029411/s55015852/8722b33e-306810c3-0e85ae3c-905f1644-d47dc3df.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14108013/s57322226/ddd99626-294204e6-cd63026e-2d5f2f0f-d736cb14.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17757767/s55386220/1b10b77c-797112a9-faf47d0e-500fdd11-1a3bc8f2.jpg
no acute cardiopulmonary process. no evidence of free air beneath the diaphragms.
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no evidence of pneumonia.
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no evidence of acute disease.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14182166/s55927361/f81c9343-f873623e-a969c9a4-03668f8f-11fd6f1e.jpg
no evidence of acute cardiopulmonary disease. no significant change.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13503962/s58195642/069852a0-8016a9aa-cfb719c1-e9ef311f-702e4ff8.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16743170/s54422873/fb574991-d37822d7-1ac68e0e-c2769c9d-ff61e5e7.jpg
no evidence for infectious process.
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minimal left basilar atelectasis.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19268639/s56565604/eba5928b-676968c2-9e162058-b5d58dc6-45e69a63.jpg
increased perihilar opacity extending peripherally concerning for increasing mass with or without intraparenchymal bleed. right pleural effusion.
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atelectatic changes at the right lung base.
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no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10578743/s56787420/4d91e29c-18b162d8-a629b2a3-6741ea0b-b8cc2ff3.jpg
improving multifocal pneumonia. small, residual bilateral pleural effusions.
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no focal consolidations concerning for pneumonia.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13058695/s57804341/803af010-d57e23ae-c74cd2e5-c0071fdd-4ab73c22.jpg
no acute cardiopulmonary process. stable moderate cardiomegaly.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11125653/s53199299/6b1c62aa-25c9ed00-e5d5ea66-b225d08b-140dd639.jpg
normal chest x-ray.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13122104/s55893094/564bce62-923db0b7-a7a1671d-1e0c9ef9-8e1611c2.jpg
cardiomegaly with mild pulmonary edema and small right pleural effusion.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12962644/s59393953/9f48346e-db4a260d-02246763-15d0cfa3-66c70478.jpg
new mild pulmonary interstitial edema.
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no acute cardiopulmonary process.
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normal chest radiographs.
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no signs of pneumonia.
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suggest followup conventional problem lateral chest radiographs in <num> months to follow this sub <num> mm right upper lobe nodule, of indeterminate chronicity.
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<num>. stable chest with mildly increased heart size. <num>. chronic fibrotic changes and calcified pleural plaques compatible with asbestosis exposure. heavy calcified pleural plaques limits evaluation for small lesions or abnormalities. <num>. bibasilar atelectasis and fibrotic changes.
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<num>. left pectoral pacemaker seen with transvenous leads in the right atrium and right ventricle. no pneumothorax. <num>. mild hyperinflation of the lungs.
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no evidence of pneumonia.
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<num> cm left lower lobe mass-like opacity, concerning for primary lung malignancy arising in a setting of chronic interstitial lung disease. round pneumonia is considered less likely. further evaluation with chest ct is recommended. recommendation(s): further evaluation with chest ct is recommended.
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no acute cardiopulmonary process.
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normal chest radiograph.
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mild bibasilar opacities appear slightly increased compared to <unk>