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no evidence of acute cardiopulmonary disease.
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unchanged mild hyperexpansion. no evidence of pneumonia.
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<num>. severe emphysema and pleural plaques. <num>. focal consolidation in the right upper lobe which most likely represents pneumonia.
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no acute cardiopulmonary abnormality. left picc tip at the low svc, in unchanged position.
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<num>. improved mild cardiomegaly. no pneumothorax. <num>. lobulated soft tissue obscuring the descending thoracic aorta and paraspinal line is likely hiatal hernia. recommend attention on followup.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality. mild elevation of the left hemidiaphragm.
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no evidence of acute disease.
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small bilateral pleural effusions with overlying atelectasis. previously seen pulmonary edema is somewhat improved in the interval. streaky left base opacity may be due to combination of pleural effusion and atelectasis, although underlying consolidation is not excluded.
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no acute cardiopulmonary process.
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no acute intrathoracic process. stable elevated right hemidiaphragm.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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copd. retrocardiac density may represent a moderate to large hiatal hernia, not appreciated on the prior examination.
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top-normal to mildly enlarged cardiac silhouette. otherwise, no acute cardiopulmonary process.
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left lower lobe and right mid lung opacities have completely resolved.
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mild pulmonary edema with small to moderate size bilateral pleural effusions and bibasilar atelectasis.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
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mild interstitial edema.
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no significant interval change when compared to the prior study.
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the endotracheal tube could be advanced for more optimal positioning. no acute cardiopulmonary process.
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right upper lobe collapse secondary to right hilar mass.
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nondisplaced fracture through the posterior fourth rib. no pneumothorax.
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no acute cardiopulmonary abnormality.
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<num>. unchanged linear retrocardiac opacity, likely minimal atelectasis. <num>. stable mild cardiomegaly.
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no acute intrathoracic process.
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mild pulmonary edema. unchanged enlargement of both hila, compatible with hilar lymphadenopathy and enlarged pulmonary arteries.
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<num>. bibasilar and subtle right middle lobe opacities, suggestive of atelectasis although superimposed pneumonia is difficult to exclude. <num>. stable, mild cardiomegaly.
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no acute intrathoracic process.
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linear left basilar atelectasis. no evidence of pneumonia.
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no acute intrathoracic process.
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mild cardiomegaly with hilar congestion.
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very vague medial upper lung opacities, which may be due to scarring from prior pneumocystis infection, including small cystic areas versus air bronchograms. active infection is difficult to exclude however. however, small but focal retrocardiac opacity with air thickening may be due to pneumonia or lower airways infection or inflammation in the left lower lobe.
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massive cardiomegaly. interstitial edema and small right effusion. focal opacity projecting over the right third rib potentially due to edema but followup after treatment is suggested to document resolution.
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cardiomegaly with hilar congestion, small bilateral effusions and probable pneumonia in the right mid to lower lung.
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no acute intrathoracic abnormalities identified.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process; specifically, no evidence of pneumonia. the results were discussed with <unk> <unk> at <time> pm on <unk> via telephone by dr. <unk> <unk> minutes after the findings were discovered.
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increasing right effusion, increasing right pneumothorax. no evidence of tension. numerous right rib fractures.
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no focal pneumonia. atelectasis and low lung volumes.
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no acute cardiopulmonary process. findings were discussed with <unk> via telephone after image acquisition at <num> p.m. on <unk>.
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limited due to rotation with possible mild edema.
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no evidence of pneumonia.
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<num>. left lower lobe collapse. moderate layering left pleural effusion exacerbated by left pulmonary edema worse than on <unk>. <num>. improved right pulmonary edema and basilar atelectasis.
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unchanged exam. stable retrocardiac opacification, most likely atelectasis though pneumonia not definitively excluded. findings suggestive of pulmonary arterial hypertension.
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persistent mild to moderate pulmonary edema and enlarged cardiac silhouette.
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no acute cardiopulmonary process. healed left rib fractures. mild cardiomegaly.
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<num>. chronic severe restrictive pulmonary fibrosis. <num>. no evidence of pneumonia, cardiac decompensation or other acute abnormality.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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single lead icd in situ. mri compatibility will be assessed by the body mri section.
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no acute cardiopulmonary process.
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no significant interval change.
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little change.
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cardiomegaly without superimposed acute cardiopulmonary process.
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no significant interval change.
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no evidence of pneumonia.
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unchanged small right apical pneumothorax.
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no acute cardiac or pulmonary process.
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<num>. probable mid right lung pneumonia. <num>. symmetrically obscured inferolateral heart borders are likely related to a large pericardial fat pad.
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clear lungs. large amount of pneumoperitoneum. the patient is reportedly status post recent cholecystectomy ; amount of air appears larger than would be expected for cholecystectomy <num> days prior, unclear whether findings may relate to post surgical change, bowel perforation not excluded.
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interval resolution of previously seen pulmonary edema. no acute cardiopulmonary process.
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<num>. right lower lobe pneumonia. <num>. mild interstitial edema with stable cardiomegaly.
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<num>. mild interstitial abnormality which could be seen with mild vascular congestion, although other etiologies such as airway inflammation could be considered. <num>. abnormal right upper mediastinal contour; although this appearance is frequently seen with benign tortuosity of the great vessels, investigation with chest ct is recommended when clinically appropriate, unless more remote prior radiographs are available to show long-term stability of this appearance. this finding and recommendation for chest ct was documented in the clinical er discharge summary.
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bilateral pleural effusions with associated volume loss in the lower lungs. an underlying infectious infiltrate can't be excluded. the appearance is slightly worse compared to the study from the prior day.
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resolved right basilar airspace opacity which may have been due to atelectasis. no other relevant change.
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coarsened interstitial marking suggesting chronic underlying interstitial process. more confluent opacities at the lung bases, left greater than right. while these could be chronic in nature, superimposed infection would be possible
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feeding tube tip in the esophagus
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no acute cardiopulmonary process.
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<num>. small bilateral pleural effusions have improved since <unk>. <num>. mild bibasilar atelectasis has improved since <unk>, less likely pneumonia.
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worsening bibasilar opacities, which most likely represent atelectasis. no definite evidence of pneumonia.
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<num>. no radiographic evidence for acute cardiopulmonary process. <num>. new left <unk>-<unk> opacity. recommened dedicated chest ct for further evaluation. findings were entered into the radiology dashboard by dr. <unk> at <time>pm on <unk>, <num> minutes after discovery.
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no evidence of acute cardiopulmonary disease.
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bibasilar atelectasis, superimposed infection cannot be excluded. multiple fractures of the bilateral shoulders are likely chronic but incompletely evaluated on this study.
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no rib fracture identified.
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increased streaky opacities at both bases are most suggestive of increased bibasilar atelectasis. the possibility of early infectious infiltrates is considered less likely, but cannot be entirely excluded. mild upper zone redistribution again seen. doubt overt chf.
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no signs of pneumonia.
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moderate cardiomegaly with stable left lung volume loss. stable retrocardiac opacity is compatible with atelectasis but may represent pneumonia in the correct clinical setting.
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no acute cardiopulmonary abnormality.
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<num>. mild pulmonary edema and minimally increased pleural effusion on the right. <num>. persisent atelectasis in the right lower lobe.
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stable severe cardiomegaly. substantial improvement in pulmonary edema and pleural effusions with only small residual right pleural effusion. findings suggest pulmonary arterial hypertension.
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<num>. left lower lobe pneumonia. <num>. mild cardiomegaly. impression point #<num> was discussed with dr. <unk> by dr. <unk> at <time> p.m. via telephone on the day of the study, immediately after discovery of the finding.
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no evidence of acute cardiopulmonary process.
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lower lobe bronchial inflammation could be due to aspiration. overall stable appearance of the chest, similar to examination performed <unk>.
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right ij catheter ends in the mid svc.
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interval retraction of endotracheal tube from a right mainstem bronchus with improvement in left lung opacification.
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slight interval improvement in cardiac enlargement and chf
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moderate cardiomegaly without acute cardiopulmonary process.
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slight interval improvement in chf findings. otherwise, i doubt significant interval change.
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no radiographic evidence of pneumonia.
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no significant interval change.
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<num>. the ett projects of the level of the clavicular heads, and a repeat film with better positioning would be helpful to assess the true ett position of clinically indicated. <num>. low lung volumes, with unchanged pulmonary edema and increasing moderate bilateral pleural effusions.
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nasogastric tube terminating in the stomach.
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no acute cardiopulmonary process.
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<num>. newly placed right picc terminates in the upper svc. <num>. overall stable appearance of bilateral pleural effusions and atelectasis.
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no acute cardiopulmonary process.
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moderate cardiomegaly with mild chronic pulmonary vascular congestion and mild atelectasis in the lung bases. no focal consolidation.