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bibasilar atelectasis and mild cardiomegaly.
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<num>. normal chest radiograph. <num>. no evidence of free intraperitoneal air.
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no pneumonia.
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no acute findings in the chest. stable mild cardiomegaly.
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mild cardiomegaly. no focal consolidation to suggest pneumonia.
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no acute cardiopulmonary abnormality
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no acute cardiopulmonary process. no displaced rib fracture, however, if high clinical concern for rib fracture, dedicated rib series or ct is more sensitive.
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<num>. diffuse bilateral interstitial thickening, which in the setting of immunosuppression may represent an atypical bacterial, viral, or fungal infection. pcp infection may also have this appearance. although included in the differential, pulmonary edema or hemorrhage is less likely. <num>. right hilar opacity, which may represent a focus of pneumonia, however an underlying mass cannot be excluded.
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status post placement of a single-lead left-sided pacemaker with lead extending to the expected position of the right ventricle without evidence of pneumothorax. left basilar atelectasis/scarring.
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satisfactory placement of aicd leads with no evidence of pneumothorax.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17215130/s54394902/5b2ed827-ee959b9c-b22eea89-0ecb6ce7-44c612e2.jpg
moderate cardiomegaly. no definite pulmonary mass is identified. no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11376162/s54751613/cbcf123c-6530bae6-c06564da-486f85c6-d3515233.jpg
right picc terminates in the mid svc without definite pneumothorax seen. small bilateral pleural effusions. large rounded retrocardiac opacity most likely represents hiatal hernia with adjacent atelectasis.
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stable mild cardiomegaly with increased interstitial prominence consistent with mild pulmonary edema. trace bilateral pleural effusions. no focal consolidation.
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clear lungs. slight prominence of the main pulmonary artery may relate to a component of pulmonary hypertension.
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no acute cardiopulmonary abnormality.
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no intrathoracic metastases seen. refer to the rib x-ray report for more detailed rib evaluation.
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no acute cardiopulmonary process.
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findings consistent with fluid overload.
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no evidence of pneumonia.
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no acute cardiopulmonary process.
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small bilateral pleural effusions with prominent right basilar atelectasis. residual contrast seen in the neoesophagus as well as extending into the proximal bowel from barium study earlier today.
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no acute cardiopulmonary process. persistent cardiomegaly and pulmonary artery enlargement.
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no acute cardiopulmonary abnormality.
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low lung volumes. no acute cardiopulmonary process.
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worsening right middle lobe opacity and persistent bibasilar opacities most consistent with multifocal pneumonia, with a component of coexisting atelectasis in the right middle lobe.
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no acute intrathoracic process.
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no acute intrathoracic process.
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<num>. stable diffuse pulmonary opacities. <num>. endotracheal tube ends <num> cm from the carina. this should be advanced for more secure seating.
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unchanged position of chest tube and picc line. slight increase in right pleural effusion and bibasilar atelectasis.
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normal chest radiograph without evidence of pneumonia
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as above.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11296936/s51889997/c24739f8-085591fc-e14b869a-ddeceb89-ba6c3045.jpg
mild pulmonary vascular congestion interstitial edema without focal consolidation.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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standard positioning of the endotracheal and enteric tubes. low lung volumes with probable mild bibasilar atelectasis.
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no acute cardiopulmonary process. no pneumothorax seen. chronic appearing right lateral rib deformities.
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as above.
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no acute intrathoracic process.
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right basilar atelectasis.
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increasing small right apical pneumothorax with pleural catheter in place. small residual right pleural effusion and improving adjacent right lower lobe opacities, possibly representing reexpansion pulmonary edema in the setting of recent history of large volume thoracentesis.
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<num>. no evidence of acute cardiopulmonary disease or intrathoracic injury. <num>. compression deformities of t<num> and l<num>, the latter probably unchanged, the former new and age-indeterminate although not necessarily acute. <num>. moderate hiatal hernia.
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limited exam without definite acute cardiopulmonary process.
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limited examination, but no evidence of pneumonia.
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no acute cardiopulmonary abnormality. emphysema. enlarged right thyroid lobe.
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no acute cardiopulmonary process.
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stable appearance of lung status post right lower lobectomy. no evidence of acute cardiopulmonary process.
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repositioned picc, now terminating in the distal svc. otherwise no significant interval change when compared to the prior study.
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worsening bilateral moderate pleural effusions and bibasal opacities likely increasing atelectasis.
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<num>. persistent small to moderate size left pleural effusion with left basilar opacification, likely compressive atelectasis. <num>. low lung volumes. mild interstitial abnormality in the right lung base may reflect chronic changes and atelectasis.
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low lung volumes. right greater than left basilar opacities potentially due to atelectasis although infection cannot be excluded.
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mild pulmonary vascular congestion with no overt pulmonary edema. no focal consolidation.
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<num>. no acute cardiothoracic process. <num>. right upper lobe opacity likely due to radiation changes or metastatic tumor spread, unchanged since <unk>.
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significant interval decrease in pleural effusion which is now very small.
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no clear signs of pneumonia.
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mild pulmonary edema. probable bibasilar atelectasis, but aspiration or infection cannot be excluded.
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low lung volumes without acute cardiopulmonary process. no overt pulmonary edema.
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no acute cardiothoracic process.
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stable appearance of the chest.
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left chest port-a-cath with its tip in the upper svc without evidence of complications.
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<num>. et tube in appropriate position terminating in the mid trachea. <num>. cardiomegaly and pulmonary vascular congestion without frank pulmonary edema.
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no infiltrates
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no acute cardiopulmonary process. no radiographic evidence of active tb.
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<num>. bilateral ill-defined opacities, most likely multifocal pneumonia in the appropriate clinical setting. <num>. small-to-moderate right pleural effusion.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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improved aeration of at least the right middle lobe. the right lower lobe still has substantial volume loss. mild pulmonary edema has slightly improved and remains mild. bilateral pleural effusions remain moderate.
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<num>. stable radiographic appearance of the chest, with no acute cardiopulmonary abnormality identified. <num>. overinflation of the lungs, probably related to copd in the setting of known emphysema on prior chest ct.
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a left-sided pacer remains in place with the leads unchanged in position and projecting over the expected locations of the right atrium and right ventricle, respectively. overall cardiac and mediastinal contours are stable. lungs appear well inflated without evidence of pulmonary edema, focal airspace consolidation or pneumothorax. no pleural effusions. degenerative changes in the thoracic spine with no acute bony abnormality identified.
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no acute cardiopulmonary process. no evidence of pneumothorax or consolidation.
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no acute abnormalities.
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bibasilar atelectasis, left greater than right. please refer to subsequent cta chest for further details.
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no acute cardiopulmonary process.
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<num>. enlarging moderate left pleural effusion. <num>. stable right calcified granuloma. <num>. stable mild cardiomegaly.
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no acute cardiopulmonary radiographic abnormality.
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tracheostomy tube remains in satisfactory position. right neck/superior mediastinal vascular stents and left bronchial stent are unchanged in position. there is residual but decreased right lateral chest wall subcutaneous emphysema. there are postoperative changes in the right hemithorax. interval appearance of faint right upper lobe opacity abutting the minor fissure concerning for aspiration. stable bibasilar opacities, which although could reflect atelectasis, aspiration should also be considered. no pulmonary edema. no pneumothorax.
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opacities suggesting pneumonia, particularly within the left lower lobe including retrocardiac opacification. within eight weeks, following treatment, follow-up radiographs are suggested to show resolution.
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ng tube in the stomach.
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no evidence of infection.
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cardiomegaly, hilar congestion with mild to moderate pulmonary edema.
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<num>. moderate central vascular engorgement without overt pulmonary edema. <num>. unchanged moderate cardiomegaly.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary abnormality.
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possible minimal pulmonary vascular congestion. otherwise, no acute intrathoracic process.
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bibasilar atelectasis.
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no acute intrathoracic abnormality.
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top normal to mildly enlarged cardiac silhouette without other acute cardiopulmonary process. trace/small pleural effusions may be present. irregularity of the inferior, anterior aspect of the lower thoracic vertebral body of indeterminate age; no priors are available for comparison. recommend correlation with history of trauma or infection to this site.
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possible trace bilateral pleural effusions. otherwise, no acute cardiopulmonary process.
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no apical pneumothorax is seen on today's exam. worsening right pleural effusion and right basilar atelectasis. worsening left retrocardiac basilar opacity.
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unremarkable chest radiographic examination.
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left lower lobe peribronchial opacification due to small pneumonia or recent substantial aspiration. probable copd. no evidence of cardiac decompensation.
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no focal consolidation. no multiple areas of scarring and/or atelectasis in both lungs.
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no acute cardiopulmonary process.
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<num>. perihilar edema with asymmetrical edema on the right versus coexisting pneumonia. follow up radiographs may be helpful in this regard. <num>. left lower lobe collapse with moderate left pleural effusion. <num>. small right pleural effusion.
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no acute cardiopulmonary process.
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endotracheal tube terminates <num> cm above the carina, in adequate position.
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persistence of increased interstitial markings may be due to an infectious etiology, likely viral, or chronic interstitial lung disease. follow-up in <unk> weeks for documentation of resolution is reccomended
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no evidence of acute cardiopulmonary process.