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ChayanM
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MIMIC-Impression-Only-Data-Train-Test

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/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15228243/s54303800/6789480e-53d455e8-57f21359-43c5edc4-42cdba70.jpg
peribronchial abnormality left lower lobe, not clearly pneumonia. followup advised.
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no acute intrathoracic process, specifically no pneumothorax seen.
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no acute intrathoracic abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17728787/s53490577/2faccc24-d017c540-d0c47080-261dd620-6906efde.jpg
bibasilar streaky opacities likely reflective of atelectasis.
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no acute cardiopulmonary abnormality.
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<num>. bilateral small pleural effusions with associated bibasilar subsegmental atelectasis. <num>. no focal consolidation or pneumothorax. mildly hyperinflated lungs.
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normal chest radiograph.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14709711/s52839884/df76cc7a-07f5467a-013e3dfc-8ddec0cf-f9569228.jpg
patchy opacities in the left middle lung field without obscuring the left heart border suggest left lower lobe consolidation worrisome for infection and/or aspiration.
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no acute cardiopulmonary process. partially imaged, there may be an ovoid calcification projecting over the soft tissue lateral to the right humeral head, could relate to calcific tendinosis, not well assessed on this study.
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no evidence of neoplasm or copd.
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<num>. essential complete resolution in moderate central pulmonary edema since <unk> with residual trace fluid in the major fissure. . <num>. no focal pneumonia.
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trace bilateral pleural effusions with mild left basilar atelectasis. no focal consolidation.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11490051/s56775631/247d594e-9ac7249a-b7ee93c6-98f25392-9f8aa1e5.jpg
new, severe pulmonary edema. small bilateral pleural effusions.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13174516/s56006203/25e40922-3c18aa04-8e01dc65-cd380294-adb657b5.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13056000/s52320417/e34a815f-5e94f0d3-e9db75d6-cce2014a-f2194c38.jpg
no evidence of acute cardiopulmonary disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17898039/s58958670/41b99b08-1b9ec92a-def8774b-2b370eb3-57edb2a1.jpg
patchy opacities within the lung bases with mild peribronchial cuffing is concerning for infection in the correct clinical setting.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14195178/s52957683/366decd2-c61f8001-4067d055-06f281a9-fca175a5.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17311139/s53863531/47525562-6abff6e0-6af8ea17-84123ff8-64f8c5f9.jpg
interval decrease of left pleural effusion status post thoracentesis. no pneumothorax.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13119476/s57467669/9ffa3eff-c30d6758-b905985c-7e130eee-bd2bb02a.jpg
<num>. no pneumonia appreciated. <num>. worsening pulmonary venous congestion with new right pleural effusion and persistent left pleural effusion. <num>. there is worse cardiomegaly and/ or pericardial effusion.
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<num>. mild pulmonary edema and possible small bilateral pleural effusions. <num>. focal basilar opacity is likely atelectasis; however, infection cannot be excluded.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14213416/s50180084/e178dbf0-f7b91c53-f674467d-9d87ede0-a5cc621e.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12727378/s56850080/d4d14dea-baa5cfaa-935e3484-725c94d7-309f48b2.jpg
no evidence of acute cardiopulmonary disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19244673/s50750864/1dff8a98-c67892d4-cdd4338f-43721ec8-8b37f35e.jpg
enteric tube terminates in the stomach. low lung volumes with bibasilar linear atelectasis, unchanged.
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cardiomegaly with mild interstitial edema.
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no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16814111/s52146228/05c1b870-9034352e-2662071b-640ad4cb-75b56615.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19353810/s56166938/4e979588-e211e2ea-cfcf5455-42305342-65065469.jpg
right basilar region of consolidation compatible with pneumonia in the proper clinical setting. recommend repeat after treatment to document resolution.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10291098/s55749918/68764a61-445eec37-c42a4e23-61ee67fa-87b6b5b6.jpg
retrocardiac opacity, consistent with continued atelectasis in the left lower lobe, has slightly worsened. there also ill-defined opacity along the right minor fissure and right lower lobe could represent atelectasis or new consolidation/pneumonia in appropriate clinical setting.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19020115/s59735127/d8aab91c-6a560dc8-32fa32a0-65abf0af-b18f6df4.jpg
no change. no acute disease
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16217957/s50929174/cfe25277-ebef3d4b-5a8c72cf-92a65edc-1ee6883e.jpg
increased size of left pleural effusion which is now large with left basilar opacity, likely atelectasis, but infection cannot be excluded. patchy right basilar opacity may reflect atelectasis. tiny right pleural effusion.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16420422/s59629255/c42277d5-9ba23b18-c38f270b-533f74af-5e6faf5f.jpg
low lung volumes with bibasilar atelectasis. otherwise, no acute pulmonary process identified.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12844682/s51953283/c12ff616-146074fb-54e97e32-a96053ea-623aa6d9.jpg
there is a left subclavian line terminating near the brachiocephalic vein. mild decrease in the right lower lobe subsegmental atelectasis, otherwise no significant interval change.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12032671/s58462432/29198956-a51cd65a-bf5f5baf-dd87a134-7699daf4.jpg
left subclavian picc line tip is again located within the azygos vein
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endotracheal tube positioned appropriately. nasogastric tube tip in the distal esophagus for which advancement is recommended. bibasilar atelectasis noted.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11003787/s58843965/0d9bf88f-316791b9-f0389401-b048e705-e482e947.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11124859/s54021091/0fb7c776-128e837a-6365f7b7-bc95d3d2-a3dc53c0.jpg
no significant change in gas component in the left hemithorax, indicating the apparent increase in air-fluid levels in the left hemithorax on radiographs on <unk> was secondary to patient positioning rather than a bronchopleural fistula.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19890079/s57390073/e6f74c2b-cbfe057b-07347f73-dd00fde7-a0889cfb.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14479229/s56658010/035eaa54-c3f1a070-55308799-db7199ad-9d42e4c0.jpg
stable chest radiograph. no pneumonia or pleural effusions.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13085066/s54639696/33ef1aee-964b1f29-75140082-0de968bc-a5e3cfd9.jpg
no evidence of acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12327475/s51236851/b26f99dd-eedff985-6dad309b-c1aec3cf-609eb93b.jpg
small left pleural effusion, likely secondary to hepatic hydrothorax. no focal consolidations concerning for pneumonia identified.
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<num>. right internal jugular central venous catheter in expected position with no evidence of pneumothorax. <num>. stable small right pleural effusion
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left chest port-a-cath with its tip in the upper svc without evidence of complications.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18386137/s59470850/79f670b8-40eb55f1-e0538f76-2700ddd6-f9fde8d7.jpg
normal chest radiograph.
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as above.
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no acute cardiopulmonary process.
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stable appearance of the chest without evidence for acute cardiopulmonary abnormalities. please note that ascending aortic diameter cannot be accurately measured on this exam.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10194479/s56681875/0fb53d09-03bf0d60-34476eb2-dc5b494c-2f1d8b2c.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19014032/s59445163/1f2375ac-62c78cd4-851ae990-82807053-257ed706.jpg
bibasilar atelectasis.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18703638/s53546960/2f0dc94e-20daa7c8-a8c86d6b-52e999a8-40cee2ad.jpg
no acute cardiopulmonary abnormality.
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small bilateral pleural effusions with bibasilar atelectasis.
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new prominence of the cardiomediastinal silhouette and central vessels suggests mild cardiac decompensation. no pleural effusion. no focal opacification concerning for pneumonia.
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no acute cardiopulmonary process.
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enteric tube terminating below the diaphragm, extending off the inferior edge of the image.
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right hemithorax opacification consistent with effusion, atelectasis and right upper lobe mass as seen on ct chest from earlier today.
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there are no signs of acute cardiopulmonary process.
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interval enlargement of left-sided pleural effusion which may be partially loculated laterally. underlying left base atelelectasis, infection also possible. slight progression of right basilar parenchymal opacity is compatible with pneumonia in the proper clinical setting.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11414493/s57862890/16fb30d3-210049ce-eb294344-243ae5cc-71e8e222.jpg
<num>. left lower lobe airspace opacity, consistent with pneumonia. <num>. thoracic scoliosis.
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improving left lower lobe opacity with a substantial residual convex opacity, however. differential considerations include round atelectasis or loculated pleural effusion. a mass is also not possible to exclude at this site, however. depending on clinical circumstances, continued radiographic surveillance could be considered to show resolution or alternatively the opacity could be evaluated with chest ct, preferably with intravenous contrast if possible, when clinically appropriate.
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no evidence of acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10025630/s59510909/efce28d3-2472e2d6-8b2617b2-4986c96d-a7b0c5a9.jpg
new right lower lobe opacity is at minimum atelectasis. however given the clinical history, superimposed pneumonia is not excluded.
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no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12656203/s56751175/62ca943f-f92b2b32-1acbc7ac-5d7ff103-abbcad1f.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14895434/s59501106/f51659f7-b99fc38b-95874c3f-ad25f4dc-1b72e3bc.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13192445/s50003359/ae344f7c-2ffad83d-4eb9da67-7e677ff5-705d685b.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14884845/s59382236/12ed227c-dfec5a88-a05ebdbd-19d51938-6fd840fa.jpg
increased interstitial markings including at the subpleural regions, similar to possibly slightly progressed as compared to the prior study. no definite new focal consolidation.
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mildly increased opacity in the right cardiophrenic angle. although atelectasis can have this appearance, in the right clinical setting this finding could reflect early pneumonia.
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no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13760045/s57607062/79af20d4-20561318-b666b152-1b5a9e2d-14064fc4.jpg
right midlung opacity consistent with pneumonia in the proper clinical setting. recommend repeat after treatment to document resolution.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19099630/s56895602/7c398e4b-7fe5a2dc-b6fb81ac-a41b3561-3878dc13.jpg
no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16707579/s56844649/c0693110-00325653-91275581-03db72e3-e85baaee.jpg
<num>. new right-sided ij terminates in the low svc. <num>. slight interval increase in bibasilar consolidations concerning for pneumonia. <num>. mild pulmonary edema. these findings were discussed with dr. <unk> by dr. <unk> at <num> a.m. on the day of the exam by phone.
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no significant interval change. no focal pneumonia or pneumothorax.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19944016/s55366047/b36f775c-58e16aa6-86e7f4b9-a1a46757-d0291b49.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10169411/s54119922/37281ac6-58c66b54-d28b6e3c-9bd8d70a-eed8e86a.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11354018/s50966247/549e301a-6098f43b-47f79e28-aa83fade-6c2064f6.jpg
no acute cardiopulmonary abnormality.
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<num>. minimal opacification at the right lung base, likely atelectasis. no definite evidence of pneumonia. <num>. hyperinflation and coarse interstitial markings, likely due to interstitial lung disease.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13349207/s57751678/ce1bcc97-5ecfa218-1cea210c-01271d8f-c7675e9f.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19759491/s52749045/c8fbdee0-da83ffe5-649d918a-6bb64062-4f454c6b.jpg
persistent mild edema and left lower lobe atelectasis vs fluid in the fissure. unchanged from <unk>. bronchial obstruction cannot be excluded.
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minimal blunting of the left costophrenic angle could be due to atelectasis or a very trace pleural effusion. no focal consolidation seen.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16094645/s53612477/008ca058-40701f65-b5c7082f-490f9deb-80ef0278.jpg
background hyperinflation. no focal consolidation detected.
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<num>. no acute cardiopulmonary process. <num>. asymmetrical elevation of the left hemidiaphragm is new from <unk>.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12095092/s56674065/84a8e392-17ca2138-fdc96039-6e91c078-f492dcef.jpg
probable scarring seen in the right upper lung. more subtle regions of opacity in the lungs bilaterally could represent a superimposed infectious process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12165147/s52177225/9f98bbb4-21751808-7003e6e7-fea590ae-fd203544.jpg
increasing right cardiophrenic angle opacity; in the appropriate clinical setting may represent pneumonia. findings also suggesting mild vascular congestion.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14798613/s58074738/988977cc-d7cd2532-f71a4eef-8b7caa3b-8dd2ba68.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10723086/s54380189/38aa35fe-a5b42b89-8152bd93-c757db87-201e4df1.jpg
findings suggesting pulmonary vascular congestion but improved.
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<num>. diffuse sclerosis of the imaged bones compatible with diffuse metastatic disease from prostate cancer. <num>. within the limitations of this study, no definite parenchymal consolidation is identified. no pleural effusions or pneumothorax.
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mild pulmonary edema .
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no significant interval change since <unk>. persistent moderate to large left-sided partially loculated pleural effusion.
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mild cardiomegaly.
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low lung volumes with bibasilar airspace opacities, likely atelectasis, with small bilateral pleural effusions, similar compared to prior. no pulmonary edema.
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bilateral pleural effusions with adjacent consolidation new from prior exam raises concern for fluid overload. correlate with renal function.
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increased hazy opacification of the right hemithorax likely reflects a layering pleural effusion. grossly unchanged opacities at the right lung base may reflect atelectasis and/or pneumonia.
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no evidence of acute cardiopulmonary process.
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moderate left pneumothorax without significant rightward mediastinal shift.
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no acute cardiopulmonary abnormality. no radiopaque foreign bodies are identified along the aerodigestive tract.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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<num>. diffuse bilateral airspace opacities are nonspecific, however may represent severe pulmonary edema, diffuse alveolar hemorrhage, ards, and/or multifocal infection. <num>. new at least moderate cardiomegaly since prior exams from <unk> raises concern for pericardial effusion. no radiographic evidence of tamponade.
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low lung volumes with mild pulmonary vascular congestion and probable bibasilar atelectasis.