text: Yo, check it out, we did a study
  To see if TAP gene polymorphism got some play
  In the game of systemic sclerosis, you know
  We analyzed TAP1 and TAP2, let's go
  
  We had 61 Korean patients with SSc, yeah
  And 100 healthy Koreans, we made it clear
  Using PCR-RFLP, we got the scoop
  On whether TAP genes had any loop
  
  We used HLA-DRB1 genotyping, just so you know
  To see the independent role of TAP genes, yo
  Patients were divided, let me break it down
  Anti-topoisomerase I antibody and clinical subsets in town
  
  TAP1 and TAP2 had their own sway
  TAP1*A/A genotype, anti-topo I-positive dcSSc's way
  TAP2*A1/C genotype, anti-topo I-positive lcSSc's vibe
  TAP2*Bky2 and *C alleles, anti-topo I-negative dcSSc's tribe
  
  And don't forget TAP2*B/E genotype, hold tight
  It's for anti-topo I-negative lcSSc, alright?
  Though TAP gene associations were kinda weak
  Some were independent of HLA-DR, let me speak
  
  TAP2*A1/C, TAP2*C, and TAP2*B/E, they shine
  Even without those risk HLA-DR alleles in line
  So TAP gene polymorphisms might play a role
  In the genetic susceptibility to SSc, that's the goal