text: Yo, check it out,
  I'm 'bout to drop some knowledge, no doubt.
  Basic fibroblast growth factor, FGF2,
  It's a mitogen, yo, for smooth muscle cells, true.
  After balloon catheter injury, it's necessary,
  For their proliferation, it's quite legendary.
  
  But hold up, listen close, here's the deal,
  Intimal smooth muscle cells, they got a different feel.
  FGF2 ain't needed for their growth,
  They weakly respond, that's the truth, yo.
  
  Now let's dive deep, study the scene,
  ERK signaling and cell cycle proteins, mean.
  FGF2, it activates ERKs 1 and 2,
  Cyclin D, cyclin E, CDKs 2 and 4, they come through.
  Expressed in intimal smooth muscle cells, it's clear,
  After FGF2 infusion, they appear.
  
  But wait, hold up, don't get too excited,
  Phosphorylation of Rb ain't been ignited.
  CDK 2 activation? Nah, not here,
  Cyclin A expression? It don't appear.
  
  Elevated levels of inhibitors, they prevail,
  p15(INK4b) and p27(Kip1), they don't fail.
  Compared to medial smooth muscle cells, they be high,
  FGF2 stimulation can't make 'em say goodbye.
  
  So what does it mean, what's the conclusion?
  Despite the activators and their inclusion,
  The inhibitors hold 'em back, like a brake,
  In FGF2-stimulated cells, the cycle's at stake.
  
  Yo, that's what the study suggests,
  In the world of smooth muscle cells, it invests.
  ERKs and activators can't override,
  The inhibitors' power, they won't subside.