text: Yo, check it out, listen up, I got a tale to tell,
  'Bout a disorder that's common, it's called DM1, oh hell!
  Neuromuscular, messin' with the heart's conduction,
  But the reasons behind it? Still under construction.
  
  We had a theory, a hypothesis to explore,
  Said abnormalities in the sodium current, for sure,
  So we took these mice, transgenic in their genes,
  DMSXL they called 'em, DM1's mimicry machines.
  
  Injected 'em with flecainide, an antiarrhythmic drug,
  And damn, it caused some trouble, like a thunderous thud,
  Conduction abnormalities, tissular velocities went low,
  Strain rate droppin' too, DMSXL feelin' the blow.
  
  Now, the older they got, the worse it became,
  8 months in, the impact was stronger, no shame,
  Compared to the young ones, just 3 months old,
  DMSXL mice suffered more, their story unfold.
  
  Recorded ventricular action potentials, so precise,
  Using glass microelectrodes, givin' us the advice,
  Max upstroke velocity, it was lower in DMSXL's beat,
  Their hearts strugglin', feel the rhythm's heat.
  
  Whole-cell patch-clamp technique, we had to bring,
  Measuring INa inactivation, a faster fling,
  1.7 times quicker, in DMSXL it occurred,
  Affectin' the sodium current, that's what we inferred.
  
  Now, no Scn5a gene mutation, that we checked,
  No fibrosis, no channel protein abnormalities, what the heck?
  It's the sodium current, playin' a major role,
  In the electrical conduction block, takin' its toll.
  
  This discovery's a guide, a path for research to tread,
  Pharmaceutical and clinical, better therapy ahead,
  For the cardiac arrhythmias, DM1's wicked game,
  Let's unite, find a cure, and reclaim our rhythm's flame.