text: Yo, we did some comparisons, mRNA distribution is our mission,
  Checkin' out the sodium channel alpha subunits, no intermission,
  During development, we peeped the scene,
  Nabeta1.1 and Nabeta2.1, and the new kid on the block, beta3, so mean.
  
  In situ hybridisation studies, we brought the heat,
  Nav1.2, Nav1.3, Nav1.6, and beta3, embryonic stage they meet,
  Nabeta1.1 and Nabeta2.1 mRNA, nowhere to be found,
  Throughout this period, they were nowhere to be around.
  
  Nabeta1.1 and Nabeta2.1 came through after postnatal day 3,
  Stepped up their game, brain regions set free,
  Steadily increasing, reaching adulthood in most areas,
  Beta3 expression decreased differentially, but held strong in hippocampus and striatum, no hysteria.
  
  Emulsion-dipped slides, we saw a connection,
  Beta3 and Nav1.3 mRNA, a functional intersection,
  In the CNS, they co-localized, making moves,
  Functional interaction, sodium channel grooves.
  
  In Xenopus oocytes, we showed co-expression,
  Beta3 modifying Nav1.3, causing a sensation,
  Changing the equilibrium, fast and slow gating mode,
  Voltage dependence of activation and inactivation, beta3 took the road.
  
  To wrap it up, beta3 takes the crown,
  Dominant beta subunit during development, renowned,
  Modulating the kinetic properties of Nav1.3, no doubt,
  These findings unveil voltage-gated sodium channels' development route.