text: Yo, check it out, listen to what I gotta say,
  Immune responses to gut microbiota, we gotta play it safe,
  Avoid unnecessary inflammation, keep it cool at the mucosal surface,
  In this study, we analyzed Toll-interacting protein, no need to be nervous.
  
  Tollip, the inhibitor of TLRs and IL-1 cytokine signaling,
  We studied its expression in intestinal epithelial cells, no need for guessing,
  S-IECs and L-IECs, murine small and large,
  Comparable mRNA expression, that's a charge.
  
  But hold up, Tollip protein was only found in L-IECs,
  S-IECs didn't have it, no release,
  Even in germ-free mice, with no bacterial colonization,
  TOLLIP expression in L-IECs showed no relation.
  
  Now let's dive deep, explore the post-transcriptional repression,
  3´-UTR-mediated translational regulation, a new session,
  The region +1876/+2398, that's where it's at,
  Repressing Tollip expression, that's a fact.
  
  MiR-31, the culprit behind this repression,
  Its target sequence in the region, a clear confession,
  Inhibiting miR-31 brought back the translational repression,
  A key player in Tollip's expression.
  
  And here's the kicker, miR-31 was higher in S-IECs,
  Compared to L-IECs, that's what we see,
  It represses Tollip mRNA translation in S-IECs,
  That's how the story goes, no need for tweaks.
  
  To sum it up, Tollip translation is held back in S-IECs,
  Thanks to miR-31, it's a complex masterpiece,
  But in L-IECs, the protein's on the rise,
  Maintaining intestinal homeostasis, that's the prize.