ðɪs pɚɹˈɪfɚɹəl ɑːɹtˈɪɹiəl vˌæsəkənstɹˈɪkʃən ænd hˌaɪpɚtˈɛnʃən ˈæktᵻvˌeɪts ðə bˈæɹoːɹsˌɛptɚz, ɹɪzˈʌltɪŋ ɪn ɐ pˌæɹəsˌɪmpəθˈɛɾɪk sˈɜːdʒ ɚɹˈɪdʒᵻnˌeɪɾɪŋ ɪnðə sˈɛntɹəl nˈɜːvəs sˈɪstəm wˌɪtʃ ɪnhˈɪbɪts ðə sˌɪmpəθˈɛɾɪk aʊtflˈoʊ; haʊˈɛvɚ, ðə pˌæɹəsˌɪmpəθˈɛɾɪk sˈɪɡnəl ɪz ʌnˈeɪbəl tə tɹænsmˈɪt bᵻlˌoʊ ðə lˈɛvəl ʌvðə spˈaɪnəl kˈoːɹd lˈiːʒən.
This peripheral arterial vasoconstriction and hypertension activates the baroreceptors, resulting in a parasympathetic surge originating in the central nervous system which inhibits the sympathetic outflow; however, the parasympathetic signal is unable to transmit below the level of the spinal cord lesion.