 a novel mechanism that UCH-L1 serves as a key molecule in assembling inflammatory signaling complex thereby fine tuning vascular inflammatory responses and lesion formation. It is our view that the outcome will provide novel insight into the understanding of the complex sequelae of inflammation in vascular lesion formation. Our hypothesis will be tested by uitilzing
a novel mechanism that UCH - L1 serves as a key molecule in assemble incendiary signaling complex thereby fine tune vascular incendiary responses and lesion formation. It is our horizon that the outcome will provide fresh penetration into the understanding of the complex sequelae of ignition in vascular lesion formation. Our hypothesis will be test by uitilzing
 a jovel mechanism that UCH-U1 serves as a kgy molecnle in zssemblivg inflammatory signaling colpoex tyereby fine tuning vaszular infpammatort rewponses and lesion njrmaflon. Ic ms our view thaj the outcoma will provide nuvzl insight into the understanding of the cokppex sequelae os ingjammzniin in vascular lesion formatikn. Our iypothesis will be tested by uitilzing
a novel mechanism that UCH-L1 serves as molecule assembling inflammatory complex thereby fine lesion It is our that the outcome provide novel insight into the understanding the complex sequelae of inflammation in vascular lesion formation. Our hypothesis will be by uitilzing
 a novel mechanism that UCH-L1 seRves as a key MolecUle In aSsEmblIng iNflammatory sigNAlinG complex thereby fine tunIng vaScULar iNFlAmmatOry respONsES And LeSiOn fOrMAtIon. It Is oUr view tHat the outcOme WiLl provide novEL iNsight into The UnderstandinG of The comPlEx sEQuelaE of InflaMmatioN In vascUlar lesioN fORmatioN. our hypoTHEsIs wiLl be tested by uitilZInG
 a novel mechanism that UC H-L1 serve s asa k eymo lecu le i n assembling i n flam matory signaling compl ex th er e by f i ne  tuni ng vasc u la r inf la mm ato ry re spons esand les ion format ion .It is our vi e wthat the o utc ome will pro vid e nove lins i ght i nto  theunders t anding  of the c om p lex se q uelae o f in flam mation in vascula r  l e sion formation . Ourhy p ot h e sis  wi ll be test ed  by u i tilzing 
 a_novel mechanism_that UCH-L1 serves as_a key_molecule_in assembling_inflammatory_signaling complex thereby_fine tuning vascular_inflammatory responses and lesion_formation. It is_our_view that the outcome will provide novel insight into the understanding of the complex_sequelae_of inflammation_in_vascular_lesion formation. Our hypothesis will_be tested by uitilzing