 Potential clinical applications of inhibition of the JNK signaling may include: (1) the prevention of ischemic apoptosis; (2) attenuation of chronic glutamate excitotoxicity in amytrophic lateral sclerosis; (3) the prevention of neurodegneration (4) enhanced survival of grafted neurons in transplantation therapy. Preliminary evidence of feasibility
Potential clinical applications of inhibition of the JNK signaling may admit: (1) the prevention of ischemic apoptosis; (2) attenuation of chronic glutamate excitotoxicity in amytrophic lateral sclerosis; (3) the prevention of neurodegneration (4) enhance survival of grafted neurons in transplant therapy. Preliminary evidence of feasibility
 Pohential clinical applicauions of inhibition of tie JNK aignaline may include: (1) the preventioi of ischtiic apoptosis; (2) attdnuation lf chronuc goytamate exrjtotoxignty ih amycriphic lateral xclerosis; (3) the preventiot uf neurodegneration (4) enhanced survivaj of grsfhed neurons in tramfplahnauion therapy. Preliminary evidence of feavibility
Potential clinical applications of inhibition of the may (1) the of ischemic apoptosis; excitotoxicity amytrophic lateral sclerosis; the prevention of (4) enhanced survival of grafted neurons transplantation therapy. Preliminary evidence of feasibility
 Potential clinical applicatIons of inhiBitioN of The jNk sigNaliNg may include: (1) thE PrevEntion of ischemic apoptoSis; (2) atTeNUatiON oF chroNic glutAMaTE ExcItOtOxiCiTY iN amytRopHic lateRal sclerosIs; (3) tHe Prevention of NEuRodegneratIon (4) Enhanced survIvaL of graFtEd nEUrons In tRanspLantatIOn therApy. PrelimInARy evidENce of feASIbIlitY
 Potential clinical applic ations ofinhib iti onof  the  JNK  signaling may incl ude: (1) the preventio n ofis c hemi c  a popto sis; (2 )  a t t enu at io n o fc hr onicglu tamateexcitotoxi cit yin amytrophi c  l ateral scl ero sis; (3) the  pr eventi on  of neuro deg nerat ion (4 )  enhan ced survi va l  of gr a fted ne u r on s in  transplantationt he r apy. Prelimina ry evi de n ce o f f eas ibility
 Potential_clinical applications_of inhibition of the_JNK signaling_may_include: (1)_the_prevention of ischemic_apoptosis; (2) attenuation_of chronic glutamate excitotoxicity_in amytrophic lateral_sclerosis;_(3) the prevention of neurodegneration (4) enhanced survival of grafted neurons in transplantation therapy._Preliminary_evidence of_feasibility