-0.5 ppm). The hypotheses being tested are that respiratory epithelial cells produce adhesion molecules necessary to maintain normal epithelial function and that exposure to gaseous pollutants alter the synthesis of these products, adversely affecting tissue integrity. The second goal is to measure the expression of intercellular adhesion molecule-1 (ICAM-1) on
-0.5 ppm). The hypotheses being tested are that respiratory epithelial cells produce adhesiveness molecule necessary to maintain normal epithelial function and that exposure to gaseous pollutant alter the synthesis of these products, adversely affect tissue integrity. The second goal is to measure the saying of intercellular adhesion molecule-1 (ICAM-1) on
-0.5 ppl). The hypotheses being ttsted are that respiratocy epitgelial cdlls produce adhesion molecupew nectfsary to maintain vormal eppthelial duncuion and that exposure to gaseohd ponoutants alter jhe synthesiv of these proguztd, adversely affecting tissue integryty. The sfcond goal is jo mesfure nht expression of intercellular adhssion mmlecule-1 (ICAM-1) pn
-0.5 ppm). The hypotheses being tested are epithelial produce adhesion necessary to maintain exposure gaseous pollutants alter synthesis of these adversely affecting tissue integrity. The second is to measure the expression of intercellular adhesion molecule-1 (ICAM-1) on
-0.5 ppm). The hypotheses being testEd are that rEspirAtoRy ePiThelIal cElls produce adhESion Molecules necessary to maIntaiN nORmal EPiTheliAl functIOn AND thAt ExPosUrE To GaseoUs pOllutanTs alter the SynThEsis of these pROdUcts, adversEly Affecting tisSue IntegrItY. ThE SeconD goAl is tO measuRE the exPression oF iNTercelLUlar adhESIoN molEcule-1 (ICAM-1) on
-0.5 ppm). The hypothesesbeing test ed ar e t hat  r espi rato ry epithelialc ells  produce adhesion mole cules  n e cess a ry  to m aintain no r m alep it hel ia l  f uncti onand tha t exposure  to  g aseous pollu t an ts alter t hesynthesis of  th ese pr od uct s , adv ers ely a ffecti n g tiss ue integr it y . Thes econd g o a lis t o measure the exp r es s ion of interce llular  a d he s i onmol ecule-1 (I CA M-1)o n
-0.5 ppm)._The hypotheses_being tested are that_respiratory epithelial_cells_produce adhesion_molecules_necessary to maintain_normal epithelial function_and that exposure to_gaseous pollutants alter_the_synthesis of these products, adversely affecting tissue integrity. The second goal is to measure_the_expression of_intercellular_adhesion_molecule-1 (ICAM-1) on