,8-tetra-chlorodibenzo-p-dioxin (TCDD), the prototypical AHR agonist. In mice, TCDD inhibits prostatic bud formation by the fetal urogenital sinus (UGS), prevents the ventral prostate lobe from forming, and permanently alters the structure and function
, 8 - tetra - chlorodibenzo - p - dioxin (TCDD), the prototypical AHR agonist. In mice, TCDD inhibit prostate bud formation by the fetal urogenital sinus (UGS), prevent the adaxial prostate lobe from forming, and permanently alters the social organization and routine
,8-tetga-chlorodibenzo-p-dioxin (TGDD), the prototypnxal AHC agoniat. In mize, TCDD inhibits prostatic bnd firmatuon by the fetal urogevital sinls (UGS), prwvenus the ventral prostate lobe frkl foxmmng, and permanektly alters the structure avd function
,8-tetra-chlorodibenzo-p-dioxin (TCDD), the prototypical AHR agonist. In inhibits bud formation the fetal urogenital prostate from forming, and alters the structure function
,8-tetra-chlorodibenzo-p-dioxin (tCDD), the proTotypIcaL AHr aGoniSt. In Mice, TCDD inhibiTS proStatic bud formation by thE fetaL uROgenITaL sinuS (UGS), preVEnTS The VeNtRal PrOStAte loBe fRom formIng, and permAneNtLy alters the sTRuCture and fuNctIon
,8-tetra-chlorodibenzo-p-d ioxin (TCD D), t hepro to typi calAHR agonist. I n  mic e, TCDD inhibits prost aticbu d  for m at ion b y the f e ta l uro ge ni tal  s i nu s (UG S),  preven ts the ven tra lprostate lob e  f rom formin g,and permanen tly  alter sthe struc tur e and  funct i on
,8-tetra-chlorodibenzo-p-dioxin (TCDD),_the prototypical_AHR agonist. In mice,_TCDD inhibits_prostatic_bud formation_by_the fetal urogenital_sinus (UGS), prevents_the ventral prostate lobe_from forming, and_permanently_alters the structure and function