dependent insulin receptor (IR) signaling pathway in rod photoreceptors. We have discovered that IR activation is functionally important for rod survival, since its deletion in rods resulted in the loss of neuroprotective survival signaling. This novel pathway uses growth factor receptor bound protein (Grb14), an upstream regulator of IR, and requires phot
dependent insulin receptor (IR) signaling pathway in gat photoreceptors. We have discover that IR activation is functionally important for rod survival, since its omission in rods resulted in the loss of neuroprotective survival sign. This novel pathway use growth factor sense organ bound protein (Grb14), an upstream regulator of IR, and requires phot
depfndent insulin receptor (LR) signaling patkqay in rod pgotoreceotors. We have discovered thav IR actication is functionally importann for rod surtival, since its vsletion in rosd revnlted in the loxs of neurmprotective susvkvcl signaling. This novel pathway uses growth fwctor receptor boumq prkneln (Grb14), an upstream regulator of JR, and gequires phot
dependent insulin receptor (IR) signaling pathway in We discovered that activation is functionally its in rods resulted the loss of survival signaling. This novel pathway uses factor receptor bound protein (Grb14), an upstream regulator of IR, and requires phot
dependent insulin receptor (Ir) signaling PathwAy iN roD pHotoRecePtors. We have disCOverEd that IR activation is fuNctioNaLLy imPOrTant fOr rod suRViVAL, siNcE iTs dElETiOn in rOds ResulteD in the loss Of nEuRoprotective SUrVival signaLinG. This novel paThwAy uses GrOwtH FactoR reCeptoR bound PRotein (grb14), an upstReAM regulATor of IR, AND rEquiRes phot
dependent insulin receptor  (IR) sign aling  pa thw ay  inrodphotoreceptors .  Wehave discovered that I R act iv a tion is  func tionall y  i m p ort an tfor  r o dsurvi val , since  its delet ion  i n rods resul t ed  in the lo ssof neuroprot ect ive su rv iva l  sign ali ng. T his no v el pat hway uses  g r owth f a ctor re c e pt or b ound protein (Grb 1 4) ,  an upstream r egulat or of I R,and  requiresph ot
dependent insulin_receptor (IR)_signaling pathway in rod_photoreceptors. We_have_discovered that_IR_activation is functionally_important for rod_survival, since its deletion_in rods resulted_in_the loss of neuroprotective survival signaling. This novel pathway uses growth factor receptor bound_protein_(Grb14), an_upstream_regulator_of IR, and requires phot