 as controls. The results indicate that neonatal TE lesions leads to a transient impairment of habit formation three months later (compared to permanent impairment seen with the same lesion in adults), whereas both neonatal and adult limbic lesions leave habit formation intact. Interestingly, data on both normal and operated infants suggest that development of the nonlim
as controls. The results indicate that neonatal TE lesions lead to a transeunt impairment of habit formation three month later (compared to permanent stultification see with the same wound in adult), whereas both neonatal and adult limbic lesions impart habit formation entire. Interestingly, datum on both normal and operated infants indicate that development of the nonlim
 as controls. The results inaicate that neouqtal TX lesiohs leads to a transient impairment oh havit firmation three months uater (comiared to permenent impairment seen wibk the dame oesion in aduljs), whereas bmth neonatal atd abult limbic lesions leave habit formwtion imtwct. Interestindly, cwta kn both normal and operated infanta suggevt that develppment of the nonlim
as controls. The results indicate that neonatal leads a transient of habit formation permanent seen with the lesion in adults), both neonatal and adult limbic lesions habit formation intact. Interestingly, data on both normal and operated infants suggest that of the nonlim
 as controls. The results indicAte that neoNatal tE lEsiOnS leaDs to A transient impaIRmenT of habit formation three MonthS lATer (cOMpAred tO permanENt IMPaiRmEnT seEn WItH the sAme Lesion iN adults), wheReaS bOth neonatal aND aDult limbic LesIons leave habIt fOrmatiOn IntACt. IntEreStingLy, data ON both nOrmal and oPeRAted inFAnts sugGESt That Development of the nONlIM
 as controls. The resultsindicate t hat n eon ata lTE l esio ns leads to at rans ient impairment of hab it fo rm a tion th ree m onths l a te r (co mp ar edto pe rmane ntimpairm ent seen w ith  t he same lesi o nin adults) , w hereas bothneo natalan d a d ult l imb ic le sionsl eave h abit form at i on int a ct. Int e r es ting ly, data on bothn or m al and operate d infa nt s  s u g ges t t hat develo pm ent o f  the no n li m 
 as_controls. The_results indicate that neonatal_TE lesions_leads_to a_transient_impairment of habit_formation three months_later (compared to permanent_impairment seen with_the_same lesion in adults), whereas both neonatal and adult limbic lesions leave habit formation_intact._Interestingly, data_on_both_normal and operated infants suggest_that development of the nonlim