 expansion in non-coding sequence of the myotonin protein kinase (MtPK) gene, the disease mechanism remains unclear even in its general outline. There is little experimental support for the initial hypothesis that a change in the amount or function of MtPK peptide explains the phenotype. Alternative hypotheses that the disease involves an effect
expansion in non - coding sequence of the myotonin protein kinase (MtPK) gene, the disease mechanism remains unclear even in its general outline. There be short experimental support for the initial hypothesis that a change in the measure or function of MtPK peptide explains the phenotype. alternate hypotheses that the disease involves an effect
 exoansion in non-coding seqmence of the myojobin prmtein iinase (MgPK) gene, the disease mechanidm remauns unclear even in itr general outline. Thece is little expxdimental suppkvt fox vhe initial hyppthesis thdt a change in tfe amount or function of MtPK peptide explaims the phenotype. Alttrnwtivs hypotheses that the disease invomves an effect
expansion in non-coding sequence of the myotonin (MtPK) the disease remains unclear even is experimental support for initial hypothesis that change in the amount or function MtPK peptide explains the phenotype. Alternative hypotheses that the disease involves an effect
 expansion in non-coding sequeNce of the myOtoniN prOteIn KinaSe (MtpK) gene, the diseaSE mecHanism remains unclear evEn in iTs GEnerAL oUtlinE. There iS LiTTLe eXpErImeNtAL sUpporT foR the iniTial hypothEsiS tHat a change in THe Amount or fuNctIon of MtPK pepTidE explaInS thE PhenoTypE. AlteRnativE HypothEses that tHe DIsease INvolves AN EfFect
 expansion in non-coding s equence of  themyo ton in  pro tein  kinase (MtPK) gene , the disease mechanis m rem ai n s un c le ar ev en in i t sg e ner al  o utl in e .There  is  little  experimen tal  s upport for t h einitial hy pot hesis that a  ch ange i nthe amoun t o r fun ctiono f MtPK  peptideex p lainst he phen o t yp e. A lternative hypoth e se s  that the dise ase in vo l ve s aneff ect
 expansion_in non-coding_sequence of the myotonin_protein kinase_(MtPK)_gene, the_disease_mechanism remains unclear_even in its_general outline. There is_little experimental support_for_the initial hypothesis that a change in the amount or function of MtPK peptide_explains_the phenotype._Alternative_hypotheses_that the disease involves an_effect