 of this proposal is to test two hypotheses for the molecular mechanism of resistance to tumor development in mice carrying the Gtrosa26 insertion. One is that the resistance is a function of the high levels of expression of the beta-galactosidase-neomycin resistance fusion protein that is encoded by the insertion. The second hypothesis
of this proposal is to test two hypotheses for the molecular mechanism of resistance to tumor growth in mouse carrying the Gtrosa26 insertion. One is that the resistance is a routine of the high levels of construction of the beta - galactosidase - neomycin resistor fusion protein that is encode by the interpolation. The second hypothesis
 of this proposal is to tesu two hypotheses yir the molechlar mecfanism of resistance to tumoc decelopnent in mice carrying ghe Gtrosw26 insertuon. Ibe is that the reslftandc is c hunction of the high levens of expressimn oy the beta-galactosidase-neomycin resiftance gudion protein trat ps enckded by the insertion. The second hgpothesps
of this proposal is to test two the mechanism of to tumor development insertion. is that the is a function the high levels of expression of beta-galactosidase-neomycin resistance fusion protein that is encoded by the insertion. The second hypothesis
 of this proposal is to test two Hypotheses For thE moLecUlAr meChanIsm of resistancE To tuMor development in mice caRryinG tHE GtrOSa26 InserTion. One IS tHAT thE rEsIstAnCE iS a funCtiOn of the High levels Of eXpRession of the BEtA-galactosiDasE-neomycin resIstAnce fuSiOn pROtein ThaT is enCoded bY The insErtion. The SeCOnd hypOThesis
 of this proposal is to te st two hyp othes esfor  t he m olec ular mechanism of r esistance to tumor dev elopm en t  inm ic e car rying t h eG t ros a2 6ins er t io n. On e i s thatthe resist anc eis a functio n  o f the high  le vels of expr ess ion of  t heb eta-g ala ctosi dase-n e omycin  resistan ce fusion protein t ha t is  encoded by the i n se r tion. The seco nd hyp ot h es i s 
 of_this proposal_is to test two_hypotheses for_the_molecular mechanism_of_resistance to tumor_development in mice_carrying the Gtrosa26 insertion._One is that_the_resistance is a function of the high levels of expression of the beta-galactosidase-neomycin resistance_fusion_protein that_is_encoded_by the insertion. The second_hypothesis