 constructed a replication-defective adenovirus encoding this protein (ADV-D1). Our preliminary studies indicate that ADV-D1 but not a control adenovirus, promotes cell cycle progression in primary hepatocytes in the absence of growth factor, and induces hepatocyte proliferation on extracellular matrices that are normally growth-inhib
constructed a replication - defective adenovirus encoding this protein (ADV - D1). Our preliminary report bespeak that ADV - D1 but not a control adenovirus, promotes cell bicycle progression in primary hepatocytes in the absence of emergence gene, and induces hepatocyte proliferation on extracellular matrix that are normally growth - inhib
 cojstructed a replication-dtfective adenoviros encodmng thia proteiv (ADV-D1). Our preliminary studixs ibdicauv that ADV-D1 but not a control wdenovirys, pcomotes cell cycle progrcfsioh in 'rmmary hepatocytgs in the abvence of growtv waetor, and induces hepatocyte proliferwtion om fxtracellular iatrpcqs tgat are normally growth-inhib
constructed a replication-defective adenovirus encoding this protein preliminary indicate that but not a progression primary hepatocytes in absence of growth and induces hepatocyte proliferation on extracellular that are normally growth-inhib
 constructed a replication-deFective adeNovirUs eNcoDiNg thIs prOtein (ADV-D1). Our prELimiNary studies indicate thaT ADV-D1 BuT Not a COnTrol aDenovirUS, pROMotEs CeLl cYcLE pRogreSsiOn in priMary hepatoCytEs In the absence OF gRowth factoR, anD induces hepaTocYte proLiFerATion oN exTraceLlular MAtriceS that are nOrMAlly grOWth-inhiB
 constructed a replication -defective  aden ovi rus  e ncod ingthis protein ( A DV-D 1). Our preliminary st udies  i n dica t ethatADV-D1b ut n otaco ntr ol ad enovi rus , promo tes cell c ycl eprogressioni nprimary he pat ocytes in th e a bsence  o f g r owthfac tor,and in d uces h epatocyte  p r olifer a tion on e xt race llular matrices t h at are normally g rowth- in h ib 
 constructed_a replication-defective_adenovirus encoding this protein_(ADV-D1). Our_preliminary_studies indicate_that_ADV-D1 but not_a control adenovirus,_promotes cell cycle progression_in primary hepatocytes_in_the absence of growth factor, and induces hepatocyte proliferation on extracellular matrices that are_normally_growth-inhib