 lines transfected with either V1a or V1b receptors showed that the pathways involved in transactivation of the MAPK pathway by VP are cell specific and independent of the V1 receptor subtype. This data suggest that VP released into the pituitary portal circulation during stress activates V1b receptor transcription through transactivation of the EGF receptor
lines transfected with either V1a or V1b receptors showed that the pathways imply in transactivation of the MAPK nerve pathway by VP are cell specific and independent of the V1 sense organ subtype. This data hint that VP released into the pituitary portal circulation during tension activate V1b receptor arrangement through transactivation of the EGF receptor
 lijes transfected with eitmer V1a or V1b recgprors siowed tgat the oathways involved in transacvivarion if the MAPK pathway by VP are cvll specidic end independent of the V1 recepflr sbbvype. This data xuggest thdt VP released ivtl the pituitary portal circulation quring xtgess activates V1b geseptkg uranscription through transactivafion of the EGF receltor
lines transfected with either V1a or V1b that pathways involved transactivation of the cell and independent of V1 receptor subtype. data suggest that VP released into pituitary portal circulation during stress activates V1b receptor transcription through transactivation of the receptor
 lines transfected with eitheR V1a or V1b recEptorS shOweD tHat tHe paThways involved IN traNsactivation of the MAPK pAthwaY bY vP arE CeLl speCific anD InDEPenDeNt Of tHe v1 ReCeptoR suBtype. ThIs data suggEst ThAt VP released INtO the pituitAry Portal circulAtiOn duriNg StrESs actIvaTes V1b ReceptOR transCription tHrOUgh traNSactivaTIOn Of thE EGF receptor
 lines transfected with ei ther V1a o r V1b  re cep to rs s howe d that the pat h ways  involved in transacti vatio no f th e  M APK p athwayb yV P  ar ece llsp e ci fic a ndindepen dent of th e V 1receptor sub t yp e. This da tasuggest that  VP  relea se d i n to th e p ituit ary po r tal ci rculation  d u ring s t ress ac t i va tesV1b receptor tran s cr i ption throughtransa ct i va t i onofthe EGF re ce ptor
 lines_transfected with_either V1a or V1b_receptors showed_that_the pathways_involved_in transactivation of_the MAPK pathway_by VP are cell_specific and independent_of_the V1 receptor subtype. This data suggest that VP released into the pituitary portal_circulation_during stress_activates_V1b_receptor transcription through transactivation of_the EGF receptor