:B signaling could sensitize PC cells to erlotinib and gemcitabine induced killing, and (b) whether the inhibition of tumor progression could correlate with the down regulation of FoxM1/Notch-1/NF-:B signaling in animal tumor tissues. The results of our research will aid in designing
: B signaling could sensitize PC cells to erlotinib and gemcitabine induce killing, and (b) whether the prohibition of tumor progression could correlate with the down rule of FoxM1 / Notch-1 / NF-:B signaling in animal tumor tissue. The results of our research will help in designing
:B slgnaling could sensitize PC cells to erlotinib and gsmcitabive induced killing, and (b) whevher the unhibition of tumor prugression could cirreoqte with tis down vzgulaflon oy HoxM1/Notch-1/NF-:B sinnaling in dnimal tumor thsruzs. The results of our research will wid in cedigning
:B signaling could sensitize PC cells to gemcitabine killing, and whether the inhibition with down regulation of signaling in animal tissues. The results of our research aid in designing
:B signaling could sensitize Pc cells to erLotinIb aNd gEmCitaBine Induced killing, ANd (b) wHether the inhibition of tUmor pRoGRessIOn Could CorrelaTE wITH thE dOwN reGuLAtIon of foxm1/Notch-1/Nf-:B signalinG in AnImal tumor tisSUeS. The resultS of Our research wIll Aid in dEsIgnINg
:B signaling could sensiti ze PC cell s toerl oti ni b an d ge mcitabine indu c ed k illing, and (b) whethe r the  i n hibi t io n oftumor p r og r e ssi on  c oul dc or relat e w ith the  down regu lat io n of FoxM1/N o tc h-1/NF-:Bsig naling in an ima l tumo rtis s ues.The  resu lts of our re search wi ll aid in designi n g 
:B signaling_could sensitize_PC cells to erlotinib_and gemcitabine_induced_killing, and_(b)_whether the inhibition_of tumor progression_could correlate with the_down regulation of_FoxM1/Notch-1/NF-:B_signaling in animal tumor tissues. The results of our research will aid in designing