.2. Gscl may contribute to the development of these syndromes. Therefore, we have generated Gscl-null mice that will serve as important resources to determine the role of Gscl in the DGS/VCFS. Finally, defects are not observed in all Gsc-expressing tissues of G
.2. Gscl may contribute to the development of these syndromes. Therefore, we have generate Gscl - null mouse that will serve as important resources to decide the role of Gscl in the DGS / VCFS. Finally, blemish are not observe in all Gsc - express tissues of G
.2. Gsfl may contribute to the development of these vyndrojes. Therdfore, we have generated Gscl-iull mice that will serve as imoortant rvsources ro dttermine the role of Gscl in ths DGS/RCHS. Finally, defegts are not observed in anl Gdc-expressing tissues of G
.2. Gscl may contribute to the development syndromes. we have Gscl-null mice that to the role of in the DGS/VCFS. defects are not observed in all tissues of G
.2. Gscl may contribute to the devElopment of These SynDroMeS. TheRefoRe, we have generaTEd GsCl-null mice that will servE as imPoRTant REsOurceS to deteRMiNE The RoLe Of GScL In The DGs/VCfS. FinalLy, defects aRe nOt Observed in alL gsC-expressinG tiSsues of G
.2. Gscl may contribute to  the devel opmen t o f t he se s yndr omes. Therefor e , we  have generated Gscl-n ull m ic e  tha t  w ill s erve as im p o rta nt  r eso ur c es  to d ete rmine t he role of  Gs cl  in the DGS/ V CF S. Finally , d efects are n otobserv ed  in all G sc- expre ssingt issues  of G
.2. Gscl_may contribute_to the development of_these syndromes._Therefore,_we have_generated_Gscl-null mice that_will serve as_important resources to determine_the role of_Gscl_in the DGS/VCFS. Finally, defects are not observed in all Gsc-expressing tissues of G