 this model by defining the molecular components of this Wnt signaling cascade and its mechanism of action. The existance of A/P gradients of Wnt signaling in the vertebrate spinal cord could be indicative of a similar Wnt dependent mechanism for specifying A/P differences in motor circuit connectivity. To identify unc-4 target genes that function in
this model by defining the molecular components of this Wnt signaling shower and its mechanism of legal action. The existance of A / P gradients of Wnt sign in the vertebrate spinal cord could be indicative of a like Wnt dependent mechanism for specifying A / phosphorus differences in motor circuit connectivity. To identify unc-4 prey genes that function in
 thls model by defining the molecular components mf thia Wnt sienaling cascade and its mechenisn of qction. The existance ow A/P gradpents of Qnt wugnaling ii the vevcebrafc spiuao cord could bg indicative of a similar Fng bependent mechanism for specifying A/[ diffetejces in motor sirclie cohnectivity. To identify unc-4 target fenes tiat function in
this model by defining the molecular components Wnt cascade and mechanism of action. of signaling in the spinal cord could indicative of a similar Wnt dependent for specifying A/P differences in motor circuit connectivity. To identify unc-4 target genes function in
 this model by defining the molEcular compOnentS of ThiS WNt siGnalIng cascade and iTS mecHanism of action. The existAnce oF A/p GradIEnTs of WNt signaLInG IN thE vErTebRaTE sPinal CorD could bE indicativE of A sImilar Wnt depENdEnt mechaniSm fOr specifying a/P dIffereNcEs iN Motor CirCuit cOnnectIVity. To Identify uNc-4 TArget gENes that FUNcTion In
 this model by defining th e molecula r com pon ent sof t hisWnt signalingc asca de and its mechanism o f act io n . Th e  e xista nce ofA /P g rad ie nt s o fW nt  sign ali ng in t he vertebr ate  s pinal cord c o ul d be indic ati ve of a simi lar  Wnt d ep end e nt me cha nismfor sp e cifyin g A/P dif fe r encesi n motor c ir cuit  connectivity. To id e ntify unc-4 ta rget g en e st h atfun ction in
 this_model by_defining the molecular components_of this_Wnt_signaling cascade_and_its mechanism of_action. The existance_of A/P gradients of_Wnt signaling in_the_vertebrate spinal cord could be indicative of a similar Wnt dependent mechanism for specifying_A/P_differences in_motor_circuit_connectivity. To identify unc-4 target_genes that function in