 period of global brain ischemia causes delayed cell death in hippocampal CA1 pyramidal neurons days after reperfusion in animals and humans. Although numerous factors have been indicated in this phenomenon, the mechanisms underlying this delayed neuronal cell death are still poorly understood. We have demonstrated that cerebral infarction and neurological deficits are significantly reduced in transgenic mice overexpressing
period of global brain ischemia causes stay cellular telephone death in hippocampal CA1 pyramidal neurons day after reperfusion in animal and humans. Although numerous factors have been indicated in this phenomenon, the mechanism underlying this delayed neural cellular telephone death are still ill understood. We have demonstrated that cerebral infarct and neurological deficits are significantly reduced in transgenic mice overexpressing
 pegiod of global brain iscmemia causes delcted cenl deafh in hiopocampal CA1 pyramidal neurois dqys adter reperfusion in ankmals and humans. Qlthiygh numerona factovf habc beeu mndicated in thls phenomenmn, the mechanivmr bnderlying this delayed neuronal celj death age still poorli undtrseood. We have demonstrated that cerebram infarbtion and neurolobical deficits are signififantpy reduced in trandgenic mice ovewwxpressing
period of global brain ischemia causes delayed in CA1 pyramidal days after reperfusion numerous have been indicated this phenomenon, the underlying this delayed neuronal cell death still poorly understood. We have demonstrated that cerebral infarction and neurological deficits are reduced in transgenic mice overexpressing
 period of global brain ischemIa causes deLayed CelL deAtH in hIppoCampal CA1 pyramiDAl neUrons days after reperfusIon in AnIMals ANd HumanS. AlthouGH nUMEroUs FaCtoRs HAvE been IndIcated iN this phenoMenOn, The mechanismS UnDerlying thIs dElayed neuronAl cEll deaTh Are STill pOorLy undErstooD. we have DemonstraTeD That ceREbral inFARcTion And neurological deFIcITs are significaNtly reDuCEd IN TraNsgEnic mice ovErExpreSSing
 period of global brain is chemia cau ses d ela yed  c elldeat h in hippocamp a l CA 1 pyramidal neurons da ys af te r  rep e rf usion  in ani m al s and  h um ans .A lt hough  nu merousfactors ha vebe en indicated in  this phen ome non, the mec han isms u nd erl y ing t his  dela yed ne u ronalcell deat ha re sti l l poorl y un ders tood. We have dem o ns t rated that cer ebralin f ar c t ion  an d neurolog ic al de f icits a r es i g nif i cantly reduce d in transg e nic  miceov ere x pressi ng
 period_of global_brain ischemia causes delayed_cell death_in_hippocampal CA1_pyramidal_neurons days after_reperfusion in animals_and humans. Although numerous_factors have been_indicated_in this phenomenon, the mechanisms underlying this delayed neuronal cell death are still poorly_understood._We have_demonstrated_that_cerebral infarction and neurological deficits_are significantly reduced in transgenic_mice overexpressing