 low affinity p75NTR) vis-vis ASM contractility. We hypothesize that TrkB is more important for [Ca2+]i regulation, while p75NTR regulates force. Using biochemistry, pharmacology, molecular biology, immunocytochemistry, fluorescence Ca2+ imaging, force measurement techniques,
low affinity p75NTR) vis - vis ASM contractility. We hypothesize that TrkB is more important for [ Ca2+]i regulation, while p75NTR determine power. Using biochemistry, pharmacology, molecular biology, immunocytochemistry, fluorescence Ca2 + imaging, violence measurement techniques,
 loa affinity p75NTR) vis-vis ARM contractiliti. Qe hypmthesiae that GrkB is more important for [Ce2+]i rwgulaupon, while p75NTR regulages force. Using buochtmistry, pharmacology, molegblar glologv, mmmunocytochemixtry, fluorascence Ca2+ imacivg, force measurement techniques,
low affinity p75NTR) vis-vis ASM contractility. We TrkB more important [Ca2+]i regulation, while pharmacology, biology, immunocytochemistry, fluorescence imaging, force measurement
 low affinity p75NTR) vis-vis ASM cOntractiliTy. We hYpoTheSiZe thAt TrKB is more importANt foR [Ca2+]i regulation, while p75NTr reguLaTEs foRCe. using BiochemIStRY, PhaRmAcOloGy, MOlEculaR biOlogy, imMunocytochEmiStRy, fluorescenCE CA2+ imaging, foRce Measurement tEchNiques,
 low affinity p75NTR) vis- vis ASM co ntrac til ity .We h ypot hesize that Tr k B is  more important for [C a2+]i  r e gula t io n, wh ile p75 N TR r egu la te s f or c e.  Usin g b iochemi stry, phar mac ol ogy, molecul a rbiology, i mmu nocytochemis try , fluo re sce n ce Ca 2+imagi ng, fo r ce mea surementte c hnique s ,
 low_affinity p75NTR)_vis-vis ASM contractility. We_hypothesize that_TrkB_is more_important_for [Ca2+]i regulation,_while p75NTR regulates_force. Using biochemistry, pharmacology,_molecular biology, immunocytochemistry,_fluorescence_Ca2+ imaging, force measurement techniques,