-fold. First, in parallel with Project by Griffin, we will continue to study the effect of survival time and of gene polymorphisms on the manifestation of specific pathological features in traumatic brain injury which mimic the pathology of Alzheimer's disease (AD) in the belief that this will offer valuable insights into the early molecular mechanisms underlying the
-fold. First, in parallel with Project by Griffin, we will continue to study the consequence of survival meter and of gene polymorphisms on the manifestation of specific pathological feature in traumatic brain injury which mimic the pathology of Alzheimer's disease (AD) in the impression that this will offer valuable insights into the early molecular mechanisms underlie the
-folf. First, in parallel with Project by Griydin, we will dontinue to study the effect of survmval time and of gene polymorphksms on tje manifwstauion of specific 'zthologleal fswturzs in traumatic nrain injurf which mimic dhd 'athology of Alzheimer's disease (AD) ig the brllef that this rill jffed valuable insights into the early molecunar mechanismx underlying the
-fold. First, in parallel with Project by will to study effect of survival on manifestation of specific features in traumatic injury which mimic the pathology of disease (AD) in the belief that this will offer valuable insights into the molecular mechanisms underlying the
-fold. First, in parallel with PrOject by GriFfin, wE wiLl cOnTinuE to sTudy the effect oF SurvIval time and of gene polymOrphiSmS On thE MaNifesTation oF SpECIfiC pAtHolOgICaL featUreS in trauMatic brain InjUrY which mimic tHE pAthology of alzHeimer's diseaSe (Ad) in the BeLieF That tHis Will oFfer vaLUable iNsights inTo THe earlY MoleculAR MeChanIsms underlying the
-fold. First, in parallelwith Proje ct by  Gr iff in , we  wil l continue tos tudy  the effect of surviva l tim ea nd o f  g ene p olymorp h is m s  on  t he  ma ni f es tatio n o f speci fic pathol ogi ca l features i n  t raumatic b rai n injury whi chmimicth e p a tholo gyof Al zheime r 's dis ease (AD)  i n  the b e lief th a t  t hiswill offer valuab l ei nsights into t he ear ly mo l e cul armechanisms  u nderl y ing the 
-fold. First,_in parallel_with Project by Griffin,_we will_continue_to study_the_effect of survival_time and of_gene polymorphisms on the_manifestation of specific_pathological_features in traumatic brain injury which mimic the pathology of Alzheimer's disease (AD) in_the_belief that_this_will_offer valuable insights into the_early molecular mechanisms underlying the