uterine nutritional conditions can also influence body weight and glucose homeostasis in adulthood. We hypothesize that maternal nutrition influences the intrauterine environment and can induce genome-wide DNA methylation changes in multiple organs of the fetus. We hypothesize that this epigenetic programming is responsible for translating intrauterine stress into molecular responses that can durably affect
uterine nutritional conditions can also influence body system of weights and glucose homeostasis in adulthood. We speculate that maternal nutrition influence the intrauterine environment and can induce genome - wide-eyed DNA methylation changes in multiple electric organ of the fetus. We speculate that this epigenetic programming is responsible for translating intrauterine stress into molecular responses that can durably feign
utegine nutritional conditiuns can also inyouence body seight avd glucose homeostasis in adnlthiod. Wt hypothesize that oaternal jutritiob inhluences the intczuterinc envjvonmeut and can inducg genome-wide DNA methylatimn ckanges in multiple organs of the fetts. We huplthesize that jhis tpidenefpc programming is responsible fkr tranvlating intraiterine stress into molecupar gesponses that can durably afdect
uterine nutritional conditions can also influence body glucose in adulthood. hypothesize that maternal and induce genome-wide DNA changes in multiple of the fetus. We hypothesize that epigenetic programming is responsible for translating intrauterine stress into molecular responses that can affect
uterine nutritional conditiOns can also InfluEncE boDy WeigHt anD glucose homeosTAsis In adulthood. We hypothesiZe thaT mATernAL nUtritIon inflUEnCES thE iNtRauTeRInE enviRonMent and Can induce gEnoMe-Wide DNA methyLAtIon changes In mUltiple organS of The fetUs. we hYPotheSizE that This epIGenetiC programmInG Is respONsible fOR TrAnslAting intrauterine STrESs into moleculaR respoNsES tHAT caN duRably affecT
uterine nutritional condit ions can a lso i nfl uen ce  bod y we ight and gluco s e ho meostasis in adulthood . Wehy p othe s iz e tha t mater n al n utr it io n i nf l ue ncesthe  intrau terine env iro nm ent and cani nd uce genome -wi de DNA methy lat ion ch an ges in mu lti ple o rganso f thefetus. We  h y pothes i ze that t hi s ep igenetic programm i ng is responsible  for t ra n sl a t ing  in trauterine  s tress into mo l ec u l a r r e sponses thatcan durably aff ect
uterine nutritional_conditions can_also influence body weight_and glucose_homeostasis_in adulthood._We_hypothesize that maternal_nutrition influences the_intrauterine environment and can_induce genome-wide DNA_methylation_changes in multiple organs of the fetus. We hypothesize that this epigenetic programming is_responsible_for translating_intrauterine_stress_into molecular responses that can_durably affect