olar concentrations cause complete inhibition. The model for control suggests that charged tRNA becomes associated with EF-1, which is specific for aminoacyl-tRNAs and is present in sufficiently high concentrations in cells to sequester the charged forms from an inhibitory role. Support for this model include: (1) the rapid onset of inhibition
olar concentrations cause complete inhibition. The exemplar for restraint suggests that charged tRNA becomes associated with EF-1, which is specific for aminoacyl - transfer rna and is present in sufficiently high concentrations in cells to impound the charged forms from an inhibitory function. Support for this model admit: (1) the rapid onset of inhibition
olag concentrations cause cumplete inhibitnin. The model for congrol suggests that charged tCNA vecomtf associated with DF-1, which ps specifuc fie aminoacyl-tRNAs akb is lvesenc mn sufficiently high concantrations in weuld to sequester the charged forms frjm an imhlbitory role. Sopporu fjr tgps model include: (1) the rapid onsst of iihibition
olar concentrations cause complete inhibition. The model suggests charged tRNA associated with EF-1, and present in sufficiently concentrations in cells sequester the charged forms from an role. Support for this model include: (1) the rapid onset of inhibition
olar concentrations cause coMplete inhiBitioN. ThE moDeL for ContRol suggests thaT CharGed tRNA becomes associatEd witH Ef-1, WhicH Is SpeciFic for aMInOACyl-TRnAS anD iS PrEsent In sUfficieNtly high coNceNtRations in celLS tO sequester The Charged forms FroM an inhIbItoRY role. supPort fOr this MOdel inClude: (1) the rApID onset OF inhibiTIOn
olar concentrations causecomplete i nhibi tio n.Th e mo delfor control su g gest s that charged tRNA be comes  a s soci a te d wit h EF-1, wh i c h i ssp eci fi c  f or am ino acyl-tR NAs and is  pr es ent in suffi c ie ntly highcon centrationsincellsto  se q ueste r t he ch argedf orms f rom an in hi b itoryr ole. Su p p or t fo r this model incl u de :  (1) the rapid  onset  o f  i n h ibi tio n
olar concentrations_cause complete_inhibition. The model for_control suggests_that_charged tRNA_becomes_associated with EF-1,_which is specific_for aminoacyl-tRNAs and is_present in sufficiently_high_concentrations in cells to sequester the charged forms from an inhibitory role. Support for_this_model include:_(1)_the_rapid onset of inhibition