 than can be attributed to lipoprotein-mediated, cell surface TLR1/2 activation. Phagocytosis of intact Bb also induced transcription of interferon-2 (IFN-2) and type I interferon-stimulated genes (ISGs), independently of TLR2. Recent evidence from the investigator's laboratory provides substantial evidence the TLR
than can be attributed to lipoprotein - mediated, cell surface TLR1/2 energizing. Phagocytosis of integral Bb also induced arrangement of interferon-2 (IFN-2) and type I interferon - induce genes (ISGs), independently of TLR2. Recent evidence from the detective's testing ground provides significant evidence the TLR
 thwn can be attributed to uipoprotein-medicred, cenl surrace TLR1/2 activation. Phagocytosis of mntaxt Bb also induced transcriotion of pnterferob-2 (IFI-2) and type I intxdferon-sbnmulafcd geuew (ISGs), indepencently of DLR2. Recent evigevcz from the investigator's laboratory [rovidex dubstantial evydenbe the NLV
than can be attributed to lipoprotein-mediated, cell activation. of intact also induced transcription I genes (ISGs), independently TLR2. Recent evidence the investigator's laboratory provides substantial evidence TLR
 than can be attributed to lipoProtein-medIated, CelL suRfAce TlR1/2 acTivation. PhagocYTosiS of intact Bb also induced TransCrIPtioN Of InterFeron-2 (IFn-2) AnD TYpe i iNtErfErON-sTimulAteD genes (IsGs), indepenDenTlY of TLR2. Recent EViDence from tHe iNvestigator's LabOratorY pRovIDes suBstAntiaL evideNCe the TlR
 than can be attributed to  lipoprote in-me dia ted ,cell  sur face TLR1/2 ac t ivat ion. Phagocytosis of i ntact  B b  als o  i nduce d trans c ri p t ion  o fint er f er on-2(IF N-2) an d type I i nte rf eron-stimula t ed  genes (IS Gs) , independen tly  of TL R2 . R e centevi dence  fromt he inv estigator 's labora t ory pro v i de s su bstantial evidenc e  t h e TLR
 than_can be_attributed to lipoprotein-mediated, cell_surface TLR1/2_activation._Phagocytosis of_intact_Bb also induced_transcription of interferon-2_(IFN-2) and type I_interferon-stimulated genes (ISGs),_independently_of TLR2. Recent evidence from the investigator's laboratory provides substantial evidence the TLR