 layers, indicative of a defect in cardiomyocyte maturation. Interestingly, recent work by our group and others clearly indicate that FAK is activated in cultured cardiomyocytes by a variety of hypertrophic stimuli including, phenylephrine (PE), endothelin I (ET-1), angiotensin II (AII), and hyp
layers, indicative of a defect in cardiomyocyte maturation. Interestingly, recent workplace by our group and others intelligibly indicate that FAK is activated in cultured cardiomyocytes by a diverseness of hypertrophic stimuli including, phenylephrine (PE), endothelin I (ET-1), angiotensin II (AII), and hyp
 lajers, indicative of a deftct in cardiomyocitw matucation. Jnterestkngly, recent work by our gronp abd otyers clearly indicate ghat FAK ps activared mn cultured cardmkmyocytcf by w vaxixty of hypertrokhic stimuli including, phetyue'hrine (PE), endothelin I (ET-1), angiotensig II (AIO), wnd hyp
layers, indicative of a defect in cardiomyocyte recent by our and others clearly in cardiomyocytes by a of hypertrophic stimuli phenylephrine (PE), endothelin I (ET-1), angiotensin (AII), and hyp
 layers, indicative of a defect In cardiomyOcyte MatUraTiOn. InTereStingly, recent wORk by Our group and others clearLy indIcATe thAT FaK is aCtivateD In CULtuReD cArdIoMYoCytes By a Variety Of hypertroPhiC sTimuli includINg, PhenylephrIne (pE), endothelin i (ET-1), AngiotEnSin ii (AII), aNd hYp
 layers, indicative of a d efect in c ardio myo cyt ematu rati on. Interestin g ly,recent work by our gro up an do ther s  c learl y indic a te t hat  F AK  is  a c ti vated  in  cultur ed cardiom yoc yt es by a vari e ty  of hypert rop hic stimuliinc luding ,phe n yleph rin e (PE ), end o thelin  I (ET-1) ,a ngiote n sin II( A II ), a nd hyp
 layers,_indicative of_a defect in cardiomyocyte_maturation. Interestingly,_recent_work by_our_group and others_clearly indicate that_FAK is activated in_cultured cardiomyocytes by_a_variety of hypertrophic stimuli including, phenylephrine (PE), endothelin I (ET-1), angiotensin II (AII), and_hyp