 heart morphometry, microscopic analysis of capillary density, fibrosis and apoptosis and determination of tissue ATP levels. Transgenic overexpression is a valuable tool to analyze the tissue function of PR-M. We believe that enhanced ATP production secondary to PR-M expression will diminish cardiac hypertrophy secondary to volume and pressure overload in the study models.
heart morphometry, microscopic analysis of capillary density, fibrosis and apoptosis and determination of tissue ATP degree. Transgenic overexpression is a valuable cock to analyze the tissue function of PR - M. We believe that enhanced ATP output secondary to PR - M expression will decrease cardiac hypertrophy secondary to volume and pressure overload in the survey models.
 hewrt morphometry, microscokic analysis of ccpillarb densify, fibroris and apoptosis and determmnatuon od tissue ATP levels. Trxnsgenic lverexprwssiib is a valnzble tool to zkalyzz vhe tissue funcjion of PR-M. Fe believe thad dnkanced ATP production secondary to PW-M exprrsdion will dimigish sardjac hypertrophy secondary to volums and pgessure overload on the study models.
heart morphometry, microscopic analysis of capillary density, apoptosis determination of ATP levels. Transgenic to the tissue function PR-M. We believe enhanced ATP production secondary to PR-M will diminish cardiac hypertrophy secondary to volume and pressure overload in the study
 heart morphometry, microscopIc analysis Of capIllAry DeNsitY, fibRosis and apoptoSIs anD determination of tissue aTP leVeLS. TraNSgEnic oVerexprESsION is A vAlUabLe TOoL to anAlyZe the tiSsue functiOn oF Pr-M. We believe tHAt Enhanced ATp prOduction secoNdaRy to PR-m eXprESsion WilL dimiNish caRDiac hyPertrophy SeCOndary TO volume AND pRessUre overload in the sTUdY Models.
 heart morphometry, micros copic anal ysisofcap il lary  den sity, fibrosis andapoptosis and determin ation  o f  tis s ue  ATPlevels. Tr a n sge ni cove re x pr essio n i s a val uable tool  to  a nalyze the t i ss ue functio n o f PR-M. We b eli eve th at  en h anced  AT P pro ductio n  secon dary to P R- M  expre s sion wi l l  d imin ish cardiac hyper t ro p hy secondary t o volu me an d pre ssu re overloa din th e  studym od e l s .
 heart_morphometry, microscopic_analysis of capillary density,_fibrosis and_apoptosis_and determination_of_tissue ATP levels._Transgenic overexpression is_a valuable tool to_analyze the tissue_function_of PR-M. We believe that enhanced ATP production secondary to PR-M expression will diminish_cardiac_hypertrophy secondary_to_volume_and pressure overload in the_study models.