It has also been shown that both prokaryotic and eukaryotic cells are capable of chemotactic memory. In prokaryotes, this mechanism involves the methylation of receptors called methyl-accepting chemotaxis proteins (MCPs). This results in their desensitization and allows prokaryotes to "remember" and adapt to a chemical gradient. In contrast, chemotactic memory in eukaryotes can be explained by the Local Excitation Global Inhibition (LEGI) model. LEGI involves the balance between a fast excitation and delayed inhibition which controls downstream signaling such as Ras activation and PIP3 production.
It has also been shown that both prokaryotic and eukaryotic cells are capable of chemotactic memory. In prokaryotes, this mechanism involves the methylation of receptors called methyl-accepting chemotaxis proteins (MCPs). This results in their desensitization and allows prokaryotes to "remember" and adapt to a chemical gradient. In contrast, chemotactic memory in eukaryotes can be explained by the Local Excitation Global Inhibition (LEGI) model. LEGI involves the balance between a fast excitation and delayed inhibition which controls downstream signaling such as Ras activation and PIP3 production