PMC3258084_01
Anterior segment of the right eye at first visit. Note the rough surface and opacity of the cornea due to multiple penetrating injuries.
opth-5-1767f1.jpg
PMC3258084_01_opth-5-1767f1.jpg
f1-opth-5-1767
Slit lamp examination revealed severe corneal opacity and edema, and a rough surface of the cornea (Figure 1) without iridocyclitis, hyphema, fibrin exudates, or any infectious signs in the anterior chamber.
Dense vitreous opacity was removed with a wide-angle viewing system through the clearer inferior-temporal portion of the cornea.
opth-5-1767f2.jpg
PMC3258084_01_opth-5-1767f2.jpg
f2-opth-5-1767
Removal of the lens remnant was performed with a vitreous cutter under endoscopic view, followed by removal of the dense vitreous opacity under the wide-angle viewing system through the clearer inferior-temporal portion of the cornea (Figure 2).
Vitreous incarcerations into the scleral perforation wounds were observed and were excised under the endoscopic view.
opth-5-1767f3.jpg
PMC3258084_01_opth-5-1767f3.jpg
f3-opth-5-1767
Moreover, vitreous incarcerations into the scleral perforation wounds were observed and excised under endoscopic view (Figure 3).
Photograph of anterior segment of the right eye 2 months after keratoplasty.
opth-5-1767f4.jpg
PMC3258084_01_opth-5-1767f4.jpg
f4-opth-5-1767
Five months after vitrectomy, penetrating keratoplasty was performed (Figure 4) in another hospital, which allowed us to make ophthalmoscopic and optical coherence tomography (OCT) observations, but the visual acuity in the right eye stayed at 0.02.
Optical coherence tomography image of the right eye 8 months after injury.
opth-5-1767f5.jpg
PMC3258084_01_opth-5-1767f5.jpg
f5-opth-5-1767
OCT revealed thinning of the retina posterior pole (Figure 5), which might be the cause of the remaining poor vision.