PMC5752986_01
Color fundus photographs of the right eye (A) at the time of presentation. On fluorescein angiography (B), nonperfusion of the cilioretinal artery was shown in the area of cilioretinal at the initial presentation. Optical coherence tomography (OCT) at baseline (C) showed the presence of intraretinal and subretinal fluid and hyperreflective inner retinal layers.
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The retinal veins were mildly dilated and tortuous and accompanied by adjacent retinal haemorrhages (Figure 1(A)). Fluorescein angiography demonstrated delayed filling and emptying of this artery (Figure 1(B)). A SD-OCT examination revealed retinal hyperreflectivity and thickening with loss of distinction of retinal layers (Figure 1(C)).
OCT angiography was performed 10 months later. Wedge-shaped area of capillary nonperfusion was revealed in both the superficial (A) and deep (B) retinal capillary plexus in area supplied by the cilioretinal artery and the choriocapillaris (C) was not affected.
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In OCT angiography, of the affected RE, there was attenuation of both the superficial capillary plexus and deep capillary plexus (Figure 2).
At follow-up, early phase of FA showing quite normal filling of the cilioretinal artery sparing (A). The retinal capillary network is well perfused on this magnification of the posterior pole. Spectral-domain OCT density map delineating areas of atrophy (blue) corresponding to cilioretinal arteriole occlusions (B). Spectral-domain OCT delineating areas of atrophy (arrows) corresponding to distribution of cilioretinal artery occlusion (C).
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The circulation of the retinal vessels had improved on FA (Figure 3(A)). SD-OCT density map clearly delineates the areas of atrophy corresponding to the distribution of the sclerotic arterioles (Figure 3(B)). Spectral-domain OCT scan demonstrated diffuse thinning of the inner nuclear layer, corresponding to the central zone of the cilioretinal artery occlusion (Figure 3(C)).