PMC3268171_01
(A) Hyperfluorescence was observed at the margin of the old lesion and in the new lesion on early phase fluorescein angiography. (B) The new lesion showed hyperfluorescence in the late-phase.
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F1
Fundus examination showed a whitish-yellow inflammatory lesion near an atrophic, pigmented retinochoroidal scar located in the superotemporal quadrant (Fig. 1A). Focal posterior hyaloid thickening and detachment were observed in the new lesion (Fig. 1B).
(A) Fundus photography revealed a whitish-yellow inflammatory lesion near an atrophic, pigmented retinochoroidal scar located in the superotemporal quadrant. (B) Spectral domain optical coherence tomography showed increased reflectivity from the inner retinal layer, retinal thickening and choroidal shadowing. Focal posterior hyaloid thickening and detachment were also observed (arrow). (C) Hyperreflectivity, thinning of the neurosensory retina and a disorganized retinal pigment epithelium layer were observed.
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F2
Hyperfluorescence on early-phase FAG was observed at the margin of the old pigmented lesion and in the new lesion (Fig. 2A). The new lesion also showed hyperfluorescence in the late phase (Fig. 2B). SD-OCT showed hyperreflectivity, thinning of the neurosensory retina and a disorganized retinal pigment epithelium (RPE) layer (Fig. 2C). Serum IgM was negative but an elevated serum level of IgG (75 IU/mL; reference value, <4 IU/mL) to toxoplasma was observed.
(A) The lesion resolved and had a scar-like appearance. (B) Spectral domain optical coherence tomography showed thinning of the retina, loss of normal striations, and a disorganized retinal pigment epithelium layer in addition to posterior hyaloid detachment (arrow).
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F3
Six weeks later, the lesion had resolved (Fig. 3A) and the patient's right-eye visual acuity had improved to 20 / 20. SD-OCT performed after treatment showed thinning of the retina, a disorganized RPE layer and posterior hyaloid detachment (Fig. 3B).