PMC5405888_01
MRI of the brain demonstrating the suspected cerebellar infarct (*): (a) sagittal; (b) transverse; and (c) dorsal T2-weighted images. Note the hyperintense wedge-shaped lesion visible in the right rostral cerebellum radiating dorsally towards the dorsal surface of the cerebellum and ventrally towards the fourth ventricle (*)
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On sagittal midline images, the lesion involved the rostral cerebellum (Figure 1).
(a) T2-weighted; (b) fluid-attenuated inversion recovery (FLAIR); and (c) T1-weighted transverse images of the brain at the level of the rostral cerebellum. Note the T2 and FLAIR hyperintensity compared with the grey matter and the T1 hypointense wedge-shaped lesion radiating ventrally towards the fourth ventricle (black arrow)
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On transverse images, the lesion was shown to be affecting the right cerebellum and had an upturned wedge-shaped appearance, radiating outwards dorsally and extending to the dorsal surface of the cerebellum and ventrally towards the fourth ventricle (Figure 2).
MRI of the brain: (a) midsagittal T2-weighted image; (b) T2-weighted transverse section at the level of the interthalamic adhesion; and (c) T2-weighted dorsal section at the level of the olfactory aperture. (a) Note the interthalamic adhesion that has lost its distinctly circular shape (white arrow), the corpus callosum that is significantly elevated (black arrow) and the overcrowding of the caudal-cranial fossa without cerebellar herniation (black arrow point). (b) Note the flattening of interthalamic adhesion (black arrow), the diminished suprasellar cistern secondary to the third ventricle expansion (+) and the narrowing of cerebral sulci, as well as the obliteration of the subarachnoid space around the dorsal convexity of the cerebral hemisphere. (c) Note the dilatation of the right olfactory recess (white arrow)
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A narrowing of the cerebral sulci and obliteration of the subarachnoid space around the dorsal convexity of the cerebral hemispheres was also evident (Figure 3). Overcrowding of the caudal cranial fossa without cerebellar herniation was also present (Figure 3).