PMC5548266_01
MRI of case 1.. Notes: On the day of admission, abnormal signal intensity in the splenium of corpus callosum with iso-signal intensity on T1-weighted images, mild hypersignal intensity on T2-weighted images and without contrast enhancement of splenial lesions, and hypersignal intensity on FLAIR and DWI images were observed. After 21 days of treatment, second cranial MRI indicated significant absorption of splenial lesions compared with the first scan and obviously decreased area of enhancement of the cerebral pia mater.. Abbreviations: FLAIR, fluid-attenuated inversion recovery; DWI, diffusion-weighted imaging; MRI, magnetic resonance imaging.
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f1-ndt-13-2085
DWI showed high signal intensity of the corpus callosum lesions without enhancement and the leptomeninges showed slight enhancement (Figure 2, row 1).
PMC5548266_02
MRI of case 1.. Notes: On the day of admission, abnormal signal intensity in the splenium of corpus callosum with iso-signal intensity on T1-weighted images, mild hypersignal intensity on T2-weighted images and without contrast enhancement of splenial lesions, and hypersignal intensity on FLAIR and DWI images were observed. After 21 days of treatment, second cranial MRI indicated significant absorption of splenial lesions compared with the first scan and obviously decreased area of enhancement of the cerebral pia mater.. Abbreviations: FLAIR, fluid-attenuated inversion recovery; DWI, diffusion-weighted imaging; MRI, magnetic resonance imaging.
ndt-13-2085Fig1.jpg
PMC5548266_02_ndt-13-2085Fig1.jpg
f1-ndt-13-2085
DWI showed high signal intensity of the corpus callosum lesions without enhancement and the leptomeninges showed slight enhancement (Figure 2, row 1).
Magnetic resonance imaging of case 2.. Notes: On the day of admission, abnormal signal intensity in the splenium of corpus callosum with iso-signal intensity on T1-weighted images, mild hypersignal intensity on T2-weighted images, mild enhancement of the meninges and lack of contrast enhancement of splenial lesions, and hypersignal intensity on DWI and FLAIR images were observed. On day 26, splenium of bilateral corpus callosum lesions in the splenium of corpus callosum almost disappeared; however, abnormal signal intensity was observed in bilateral periventricular white matter. The abnormal enhancement of meninges decreased. On day 35, abnormal signal intensity in the bilateral periventricular white matter almost disappeared.. Abbreviations: FLAIR, fluid-attenuated inversion recovery; DWI, diffusion-weighted imaging.
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DWI showed high signal intensity of the corpus callosum lesions without enhancement and the leptomeninges showed slight enhancement (Figure 2, row 1). The MRI on day 26 showed that the splenium corpus callosum lesions had almost disappeared, but showed symmetric interspersed lesions in bilateral periventricular white matter (Figure 2, row 2). T2-weighted and DWI images; most of the abnormal signal in bilateral periventricular white matter disappeared, while a small lesion in the white matter in right basal ganglia did not diminish (Figure 2, row 3). The patient was discharged on day 42 because of clinical recovery.
MRI of case 3.. Notes: At 7 days after admission, cranial MRI showed abnormal signal intensity in the bilateral thalamus, para-lateral ventricle white matter, and splenium of corpus callosum; iso-signal intensity on T1-weighted images; mild hypersignal intensity on T2-weighted images; significant contrast enhancement of the cerebral pia mater and tentorium of right cerebellum; hypersignal intensity in bilateral periventricular white matter and splenium of corpus callosum on DWI and FLAIR images. On day 30, cranial MRI showed that lesions in the splenium of corpus callosum almost disappeared. Abnormal signal intensity in bilateral periventricle white matter reduced. Contrast enhancement of the cerebral pia mater significantly decreased. The enhancement of the tentorium of right cerebellum mildly decreased.. Abbreviations: FLAIR, fluid-attenuated inversion recovery; DWI, diffusion-weighted imaging; MRI, magnetic resonance imaging.
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SCC and white matter periventricle (Figure 3). Significant enhancements of the cerebral pia mater and right tentorium of the cerebellum were observed, indicating possible meningitis. SCC and white matter periventricle (Figure 3). Significant enhancements 106/L, monocyte content was 90%, total proteins was 95 mg/dL, and chloride was 117 mmol/L. The enhancement of the tentorium of right cerebellum mildly decreased (Figure 3).
PMC5548266_03
MRI of case 3.. Notes: At 7 days after admission, cranial MRI showed abnormal signal intensity in the bilateral thalamus, para-lateral ventricle white matter, and splenium of corpus callosum; iso-signal intensity on T1-weighted images; mild hypersignal intensity on T2-weighted images; significant contrast enhancement of the cerebral pia mater and tentorium of right cerebellum; hypersignal intensity in bilateral periventricular white matter and splenium of corpus callosum on DWI and FLAIR images. On day 30, cranial MRI showed that lesions in the splenium of corpus callosum almost disappeared. Abnormal signal intensity in bilateral periventricle white matter reduced. Contrast enhancement of the cerebral pia mater significantly decreased. The enhancement of the tentorium of right cerebellum mildly decreased.. Abbreviations: FLAIR, fluid-attenuated inversion recovery; DWI, diffusion-weighted imaging; MRI, magnetic resonance imaging.
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PMC5548266_03_ndt-13-2085Fig3.jpg
f3-ndt-13-2085
SCC and white matter periventricle (Figure 3). Significant enhancements of the cerebral pia mater and right tentorium of the cerebellum were observed, indicating possible meningitis. SCC and white matter periventricle (Figure 3). Significant enhancements 106/L, monocyte content was 90%, total proteins was 95 mg/dL, and chloride was 117 mmol/L. The enhancement of the tentorium of right cerebellum mildly decreased (Figure 3).