PMC5664271_01
Axial CT angiogram showing bilateral cervical lymphadenopathy, the largest of which measures 4 x 3.4 x 2.5 cm (arrow). The posterior portion of the node demonstrates arterial phase enhancement while the anterior portion does not demonstrate it. This mixed density mass has the appearance of a necrotic or haemorrhagic lymph node.
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It did not demonstrate any vascular injury but revealed significant bilateral cervical lymphadenopathy, largest on the left side, level II measuring 4 x 3.4 x 2.5 cm, with mixed density suggestive of either a necrotic lymph node or, given the history of trauma and rapid swelling, haemorrhage into the lymph node (Figure 1).
Sagittal CT angiogram demonstrating extensive hypervascularity of lymph nodes in the left neck.
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Of note, the lymphadenopathy showed signs of hypervascularity which was more in keeping with an infectious aetiology (Figures 2 and 3).
Coronal CT angiogram of the neck demonstrating extensive hypervascularity of cervical lymph nodes bilaterally which are more pronounced on the left side.
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Of note, the lymphadenopathy showed signs of hypervascularity which was more in keeping with an infectious aetiology (Figures 2 and 3).
Ultrasound scan showing an enlarged left cervical lymph node with heterogeneous echogenicity.
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An ultrasound scan was performed to further characterise the lymphadenopathy, which demonstrated heterogeneous internal echoes with absent colour Doppler flow in the anterior aspect of the lesion (Figures 4 and 5).
Ultrasound scan of the lesion in question showing colour Doppler flow in the posterior aspect of the lesion only.
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An ultrasound scan was performed to further characterise the lymphadenopathy, which demonstrated heterogeneous internal echoes with absent colour Doppler flow in the anterior aspect of the lesion (Figures 4 and 5).
Ultrasound scan showing increased colour Doppler flow through enlarged left cervical lymph nodes.
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Ultrasonography also demonstrated generalised hypervascular cervical lymphadenopathy, again reflective of an infectious aetiology (Figure 6).