PMC9616236_01
Neuroradiological findings. a) Diffusion-weighted imaging reveals the high intensity legions in the corticomedullary junction of the bilateral hemispheres. b) Fluid-attenuated inversion recovery imaging reveals diffuse high-intensity signal of white matter. c) Arterial spin labeling at the onset shows hypoperfusion in the left hemisphere (arrow). d) Magnetic resonance angiography reveals no significant intracranial arterial occlusive lesions. e) Arterial spin labeling and f) N-isopropyl-p-[123I] iodoamphetamine single-photon emission computed tomography at six days after the admission reveals no apparent hemodynamic changes.
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Diffusion-weighted imaging revealed linear hyperintensity lesions in the white matter adjacent to the cerebral cortex (Fig. 1a), and those were isointense on apparent diffusion coefficient. Fluid-attenuated inversion recovery imaging revealed diffuse bilateral white matter hyperintensity (Fig. 1b). In addition, hypoperfusion of the left temporal lobe was identified on arterial spin labeling (ASL) (Fig. 1c). Magnetic resonance angiography revealed no occlusive lesion in major intracranial cerebral artery (Fig. 1d). N-isopropyl-p-[123I] iodoamphetamine (123I -IMP) single-photon emission computed tomography (SPECT) conducted at six days from admission revealed no evidence of perfusion abnormality (Fig. 1f). No significant temporal changes were seen in major intracranial cerebral artery between initial and the follow-up MRAs.