PMC5353457_01
(Panel a) Transthoracic echocardiographic four-chamber view showing the ovalar isoechoic mass originating from the right ventricular lateral wall and occupying half of the RV cavity. (Panel b) Cardiac magnetic resonance T1-weighted sequence with fat suppression short axis view shows homogeneous, intermediate-to-high signal intensity. (Panel c) Cardiac magnetic resonance T2-weighted sequence four-chamber view shows a diffused hyperintensity. (Panel d) Cardiac magnetic resonance T1-weighted sequence short axis view shows absence of late gadolinium enhancement
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F1
Echogenicity was the same as the myocardium, dimensions 35 x 28 cm [Figure 1 Panel a], and neither outflow tract obstruction nor interference with tricuspid valve dynamic was present. On CMR; the mass appeared homogeneous, with intermediate-to-high signal on T1-weighted [Figure 1 Panel b] and diffuse hyperintensity on T2-weighted images [Figure 1 Panel c]. No late gadolinium enhancement was present [Figure 1 Panel d]; first pass enhancement was late and heterogeneous [Video 2 and Figure 2 Panel a].
(Panel a) Cardiac magnetic resonance first pass perfusion imaging showing late and heterogeneous enhancement. (Panel b) Computed tomography modified four-chamber view shows no evidence of pericardial or extracardiac infiltration. (Panel c) On gross examination, the mass appeared russet-colored, homogeneous, and noncapsulated. (Panel d) Histologic specimen, hematoxylin and eosin, original magnification, x40: Multiple large, thin-walled, dilated vascular spaces (arrow) can be seen within the ventricular wall
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F2
No late gadolinium enhancement was present [Figure 1 Panel d]; first pass enhancement was late and heterogeneous [Video 2 and Figure 2 Panel a]. CT showed no extracardiac infiltration, the feeding artery was a branch of therightcoronary artery [Figure 2 Panel b]. No cellular atypia was present [Figure 2 Panel d].