PMC4450339_01
Brain primary tumor.
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Seven years later he was diagnosed with local recurrence and underwent two craniotomies to achieve radical resection (Figure 1).
Liver metastases, segment VI.
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Five years after the last intervention, he was admitted to hospital due to diverticulitis and an incidental finding of a small lesion in the body of T10 vertebra was demonstrated on CT scans, as well as multiple, atypical, hypervascular lesions in both liver lobes, the larger being 8 cm in diameter, initially considered hemangiomas (Figures 2 and 3).
Liver metastasis, segment VIII.
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fig3
Five years after the last intervention, he was admitted to hospital due to diverticulitis and an incidental finding of a small lesion in the body of T10 vertebra was demonstrated on CT scans, as well as multiple, atypical, hypervascular lesions in both liver lobes, the larger being 8 cm in diameter, initially considered hemangiomas (Figures 2 and 3).
H&E stain, 200x.
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Histopathology revealed two tumors (8.7 cm and 3.8 cm) consisting of relatively bland mesenchymal cells, with no unique characteristics, packed around an elaborate network of vessels (Figures 4 and 5), confirming the hypervascularity and nonepithelial origin of the neoplasms, which exhibited mild nuclear pleomorphism, low mitotic rate (<5 mitoses/10 HPF), and few areas of necrosis.
H&E stain, 400x.
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fig5
Histopathology revealed two tumors (8.7 cm and 3.8 cm) consisting of relatively bland mesenchymal cells, with no unique characteristics, packed around an elaborate network of vessels (Figures 4 and 5), confirming the hypervascularity and nonepithelial origin of the neoplasms, which exhibited mild nuclear pleomorphism, low mitotic rate (<5 mitoses/10 HPF), and few areas of necrosis.
CD34 stain, 40x.
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Multiple hypervascular hepatic lesions.
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Two of the liver lesions were hypervascular, although the caudate lobe metastasis was only slightly vascular with a capillary network. Transarterial embolization was successful, due to hyperselective arterial catheterization (Figures 7 and 8).
Disappearance of hepatic lesions after transarterial embolization.
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Two of the liver lesions were hypervascular, although the caudate lobe metastasis was only slightly vascular with a capillary network. Transarterial embolization was successful, due to hyperselective arterial catheterization (Figures 7 and 8).