PMC3072479_01
The first MRI (a-c) showed cortical-subcortical patchy hyperintense areas on T2 FLAIR-weighted images (a) localized in the parietal-occipital lobes with asymmetric representation for left predominance. No signal alteration was seen in DWI images (b) in presence of high signal on ADC maps indicating vasogenic edema (c). MRI exam after 3 months demonstrated complete resolution of the hyperintense areas on FLAIR-weighted (d) images and ADC maps (e). The MR-angiography of intracranic circle, performed with the 3D-TOF technique (f, g), showed the presence of left hyperplastic AChA (white arrows, tridimensional images f) supplying partially the distribution of the ipsilateral posterior cerebral artery (PCA) (arrows, tridimensional images f). In g the same anatomical abnormalities is represented by MIP reconstruction
10194_2010_284_Fig1_HTML.jpg
PMC3072479_02_10194_2010_284_Fig1_HTML.jpg
Fig1
PMC3072479_02
The first MRI (a-c) showed cortical-subcortical patchy hyperintense areas on T2 FLAIR-weighted images (a) localized in the parietal-occipital lobes with asymmetric representation for left predominance. No signal alteration was seen in DWI images (b) in presence of high signal on ADC maps indicating vasogenic edema (c). MRI exam after 3 months demonstrated complete resolution of the hyperintense areas on FLAIR-weighted (d) images and ADC maps (e). The MR-angiography of intracranic circle, performed with the 3D-TOF technique (f, g), showed the presence of left hyperplastic AChA (white arrows, tridimensional images f) supplying partially the distribution of the ipsilateral posterior cerebral artery (PCA) (arrows, tridimensional images f). In g the same anatomical abnormalities is represented by MIP reconstruction
10194_2010_284_Fig1_HTML.jpg
PMC3072479_02_10194_2010_284_Fig1_HTML.jpg
Fig1