PMC6679893_01
(a) RE, inflammatory cells in vitreous humor, epiretinal membrane, preserved foveal architecture with parafoveal retinal thickening (mild macular edema), without cysts or serous detachment (b) LE, inflammatory cells in vitreous humor, epiretinal membrane without traction, preserved foveal architecture without cysts or serous detachment.
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fig1
B-scan Macular Optical Coherence Tomography (OCT) from the RE revealed inflammatory cells in vitreous humor, epiretinal membrane, and preserved foveal architecture, with parafoveal retinal thickening (mild macular edema), without cysts or serous detachment (Figure 1(a)). In the LE, inflammatory cells in vitreous humor, epiretinal membrane without traction, and preserved foveal architecture without cysts or serous detachment were revealed (Figure 1(b)).
(a) RE, blockage of choroidal fluorescence secondary to vitreous opacities, pronounced ischemic areas in inferotemporal arcade, collateral vessel formation, and leakage near the optic nerve head and along the retinal veins. (b,c) LE, blockage of choroidal fluorescence secondary to vitreous opacities, leakage in foveal area along the retinal vessels, and staining of nasal retinal veins.
CRIOPM2019-8239205.002.jpg
PMC6679893_01_CRIOPM2019-8239205.002.jpg
fig2
Fluorescein angiography showed on the RE blockage of choroidal fluorescence secondary to vitreous opacities, pronounced ischemic areas in inferotemporal arcade, collateral vessel formation, and leakage near the optic nerve head and along the retinal veins (periphlebitis) (Figure 2(a)). In the LE, blockage of choroidal fluorescence secondary to vitreous opacities, leakage in foveal area (macular edema (ME)) and along the retinal vessels, and staining of nasal retinal veins (periphlebitis) were shown (Figures 2(b) and 2(c)).