PMC6424709_01
(A) shows presumed ocular toxoplasmosis-related retinitis inferotemporal to the macula on fundus examination. (B) shows SD-OCT with accomulation of subretinal fluid under the chorioretinitis lesion. (C) shows fundus fluorescine angiography with early hypofluoresence at the chorioretinal lesion site. (D) shows fundus fluorescine angiography with late leakage at the site of chorioretinal lesion.
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Fluorescein angiography showed early hypofluorescence of the lesion and involved vessels with progressive hyperfluorescence due to leakage of the dye in addition to leakage from the involved blood vessels indicating vasculitis (Fig. 1).
(A) shows scarring of the chorioretinal lesion upon resolution with the formation of vitreomacular traction. (B) shows SD-OCT of the macula with superficial traction involving the fovea. (C) shows SD-OCT with vitreoretinal traction at the site of scar.
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SD-OCT showed Vitreoretinal traction involving the superficial layers of the macula (Fig. 2).
(A) shows scarring at the area of previous chorioretinal lesion with peripheral suspecious area of chorioretinitis. (B) shows fundus fluorescine angiography with early hyperfluorescence peripheral to the scar. (C) shows fundus fluorescine angiography with late leakage peripheral to the scar indicating an active satellite lesion.
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Patient was being closely followed up to detect any progression of traction. 2 months after complete resolution, the patient has developed a chorioretinitis lesion on the border of the previous scar which and on fundus fluorescine angiography it has shown a characteristic early hypofluoresence with late leakage (Fig. 3) indicating a toxoplasmosis satellite chorioretinal lesion for which another cycle of spiramycin has started for 45 days.