PMC4772632_01
a Fundus photography before any treatment. The fundus is nearly invisible because of the opacification of the vitreous cavity. b Optical coherence tomography shows choroidal folds and serious retinal fluid near the posterior pole. c Fundus photography after 6 months of treatment. The fundus is nearly clear. d Optical coherence tomography shows the disappearance of choroidal folds and serious retinal fluid near the posterior pole.
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F1
Fundus examination was difficult because of opacification (fig. 1a). Fluorescein angiography showed leakage corresponding to serious retinal detachment areas (fig. 1b). Ocular inflammation could be stabilized by oral prednisolone (fig. 1c, d).
Clinical time course. Administration of Ca blockers was ceased because the patient's systolic blood pressure was much too low; after the attack, it increased significantly and was too high. PSL = Prednisolone.
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F2
In addition, her systolic blood pressure was too low (approximately 85-100 mm Hg with no symptoms), so we ceased the administration of Ca blockers for her hypertension (fig. 2).
MRI shows hyperintense imaging on T2. Fluid-attenuated inversion recovery sequences. a Axial views show hyperintensity on both posterior lobes on the day of the attack. b Axial views show that dot hyperintensity spots remain on both posterior lobes 6 months after the day of the attack.
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F3
The MRI showed a high signal intensity in both posterior lobes, consistent with PRES (fig. 3a). The MRI findings with axial views showed that hyperintense spots remained on both posterior lobes 6 months after the day of the attack (fig. 3b).