PMC5420918_01
Axial T2 weighted (a, b) and FLAIR (c, d) MR imaging showing extensive white matter hyperintensity signal changes with a confluent pattern in periventricular and deep white matter of the cerebral hemispheres, without vascular distribution, more extensive on the right frontal region and involving all segments of the corpus callosum.
CRICC2017-1092537.001.jpg
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fig1
A brain Magnetic Resonance Imaging (MRI) was then performed and showed bilateral supra and infratentorial multifocal white matter lesions, with a pattern that did not follow vascular territorial distribution (Figures 1, 2, 3, and 4), involvement of the corpus callosum with restricted diffusion (Figures 1 and 3), and areas of necrosis (Figure 2).
Axial (a) and sagittal (b) T1 weighted MR images. Linear spontaneous hyperintensity of the cortex in the frontoparietal convexity, compatible with laminar necrosis.
CRICC2017-1092537.002.jpg
PMC5420918_01_CRICC2017-1092537.002.jpg
fig2
A brain Magnetic Resonance Imaging (MRI) was then performed and showed bilateral supra and infratentorial multifocal white matter lesions, with a pattern that did not follow vascular territorial distribution (Figures 1, 2, 3, and 4), involvement of the corpus callosum with restricted diffusion (Figures 1 and 3), and areas of necrosis (Figure 2).
MR diffusion weighted images. DWI (a) and ADC map (b) with significant restricted diffusion of the corpus callosum bilaterally in all the segments and in the right anterior frontal and parietal region.
CRICC2017-1092537.003.jpg
PMC5420918_01_CRICC2017-1092537.003.jpg
fig3
A brain Magnetic Resonance Imaging (MRI) was then performed and showed bilateral supra and infratentorial multifocal white matter lesions, with a pattern that did not follow vascular territorial distribution (Figures 1, 2, 3, and 4), involvement of the corpus callosum with restricted diffusion (Figures 1 and 3), and areas of necrosis (Figure 2).
Axial T2*. Punctate hypointensity foci of magnetic susceptibility diffusely in the central and subcortical cerebral white matter, without vascular distribution, corresponding to hemosiderin deposition.
CRICC2017-1092537.004.jpg
PMC5420918_01_CRICC2017-1092537.004.jpg
fig4
A brain Magnetic Resonance Imaging (MRI) was then performed and showed bilateral supra and infratentorial multifocal white matter lesions, with a pattern that did not follow vascular territorial distribution (Figures 1, 2, 3, and 4), involvement of the corpus callosum with restricted diffusion (Figures 1 and 3), and areas of necrosis (Figure 2).