PMC3037983_01
Fluorescein angiogram 24.0 seg, 3:53 and 8:32. Fluorescein angiography shows delayed arterial and venous filling, large areas of ischemia in the superior quadrants, retinal hemorrhages, periphlebitis, macular edema, upper trunk venous occlusion and lower trunk venous stasis associated with occlusion of the superior temporal arterial branch.
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F1
Fluorescein angiography revealed delayed arterial and venous filling, large areas of ischemia in the superior quadrants, retinal hemorrhages, periphlebitis, macular edema, upper trunk venous occlusion and lower trunk venous stasis associated with occlusion of the superior temporal arterial branch (fig. 1).
OCT exhibited macular thickening (626 mum) secondary to diffuse edema and presence of microcysts.
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F2
Optical coherence tomography (OCT) showed macular thickening (626 mum) secondary to diffuse edema and presence of microcysts (fig. 2).
OCT 2 months after the suspension of INF demonstrated decreased macular retinal thickness (322 mum) and partial resolution of edema.
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F3
She presented with visual improvement in the left eye (20/60) and decreased macular retinal thickness (322 mum) (fig. 3).
Control 4-month angiographies revealed large areas without capillary perfusion in the upper quadrants, neovascularization, and diffuse macular edema.
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F4
Large areas without capillary perfusion in the upper quadrants, neovascularization and diffuse macular edema were found in the control angiographies (fig. 4).
After 3 years of follow-up, fundoscopy revealed optic disc pallor, superior retinal atrophy, and macular hole.
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F5
After 3 years of follow-up, best-corrected visual acuity in the left eye was 20/400, the anterior segment exhibited no changes, and fundoscopy showed optic disc pallor, superior retinal atrophy, and macular hole (fig. 5).