PMC6476132_01
This is a 3-dimensional reconstruction of the CT aortogram, with the arrow indicating the saccular component of the aneurysm.
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fig1
A computed tomography (CT) aortogram revealed an infrarenal aneurysm with signs of dissection and retroperitoneal hematoma formation, indicative of leakage (see Figures 1-3).
Multiplanar images demonstrating the infrarenal abdominal aorta. The dissection is seen arising 3.3 cm distal to the take-off of the left renal artery and extending to the iliac bifurcation, with an involved length of 7 cm.
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fig2
A computed tomography (CT) aortogram revealed an infrarenal aneurysm with signs of dissection and retroperitoneal hematoma formation, indicative of leakage (see Figures 1-3).
This is an axial view at the level of L4, demonstrating the saccular aneurysm at its maximal caliber of 5.1 x 4.8 cm (W x AP). An intimal flap is seen along the right lateral aspect of this dilatation. The saccular component of the aneurysm arises from the false lumen and is oriented anterolaterally to the right. No definite perivascular contrast extravasation is appreciated indicating that the point of rupture has likely been walled off. A heterogenous, predominantly hypodense fluid collection exhibiting minimal peripheral enhancement is appreciated at the right retroperitoneum, probably subacute hematoma (arrow).
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fig3
A computed tomography (CT) aortogram revealed an infrarenal aneurysm with signs of dissection and retroperitoneal hematoma formation, indicative of leakage (see Figures 1-3).
This is a section of the vascular wall adjacent to the main thrombus. Neutrophilic infiltrates (thin arrow) can be appreciated within the layers of the wall. At the intimal side, there is fibrin deposition (thick arrow) indicating an endothelial state favorable towards coagulation.
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fig4
Micrographs of the aortic wall biopsy showed fibrin deposition necrosis and calcification with peripheral viable cellular infiltrates consisting of neutrophils and foamy macrophages consistent with an atherosclerotic process (see Figures 4-6).
This is a section taken from the main thrombus showing heavy deposition of eosinophilic fibrin material (*) with entrapped erythrocytes and inflammatory cells ( ).
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fig5
Micrographs of the aortic wall biopsy showed fibrin deposition necrosis and calcification with peripheral viable cellular infiltrates consisting of neutrophils and foamy macrophages consistent with an atherosclerotic process (see Figures 4-6).
At the periphery of the thrombus with viable cellular material, clusters of foamy macrophages or foam cells can be seen.
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fig6
Micrographs of the aortic wall biopsy showed fibrin deposition necrosis and calcification with peripheral viable cellular infiltrates consisting of neutrophils and foamy macrophages consistent with an atherosclerotic process (see Figures 4-6).