PMC4375212_01
Head magnetic resonance imaging after onset. A: Diffusion-weighted image (DWI) showing a high-intensity area extending from the basal ganglia to the corona radiata of the left cerebrum and multiple small high-intensity areas at bilateral cerebral cortices. B: Fluid-attenuated inversion recovery showing several small high-intensity areas in the same locations detected on DWI (arrowheads). C: Magnetic resonance angiography demonstrating no steno-occlusive changes.
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Fig1
Diffusion-weighted imaging (DWI) showed a high-intensity area extending from the basal ganglia to the corona radiata of the left cerebrum and multiple small high-intensity areas at bilateral cerebral cortices (Figure 1A). Among these acute ischemic lesions, some showed hyper-intensity signals on fluid-attenuated inversion recovery (FLAIR) imaging (Figure 1B). Magnetic resonance angiography (MRA) demonstrated no steno-occlusive changes in cerebral arteries (Figure 1C).
Head computed tomography (CT) scans after thrombolysis. A: CT scan right after the neurological deterioration showing a subcortical hemorrhage in the left occipital lobe. B: Repeat CT scan the day after onset showing enlargement of the left occipital hemorrhage and two subcortical hemorrhages in the right frontal and right temporal lobes.
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Fig2
A CT scan immediately after the neurological deterioration showed a subcortical hemorrhage in the left occipital lobe (Figure 2A). A repeat CT scan the day after onset showed enlargement of the left occipital hemorrhage and two new subcortical hemorrhages in the right frontal and right temporal lobes (Figure 2B).