PMC3961110_01
Series of images of the C2 metastasis. (A) Lateral X-ray shows a C2 destructive process, which was confirmed by (B and C) CT, (D) CT 3D and (E and F) MRI imaging. CT, computed tomography; 3D, three dimensional; MRI, magnetic resonance imaging.
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C2 vertebral body (Fig. 1A). Computed tomography (CT) scanning and CT three-dimensional (3D) reconstruction confirmed an erosive lesion of the left C2 vertebral body and the left transverse process with surrounding soft tissue mass (Fig. 1B-D). The brainstem and the cervical spinal cord were not compressed (Fig. 1E and F).
Bone scan showed diffuse increased uptake of the isotope at the level of the C2 vertebra, without other abnormality elsewhere in the skeleton.
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C2 vertebra, without other abnormalities elsewhere in the skeleton (Fig. 2). CT angiography identified that the left vertebral artery (VA) was topically encapsulated by the tumor and displaced, but did not show any pathological vascularization (Fig. 3).
CT angiography showed destruction of the C2 body and that the VA was displaced. CT, computed tomography; VA, vertebral artery.
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C2 vertebra, without other abnormalities elsewhere in the skeleton (Fig. 2). CT angiography identified that the left vertebral artery (VA) was topically encapsulated by the tumor and displaced, but did not show any pathological vascularization (Fig. 3).
Postoperative pathological examination confirmed moderately differentiated hepatocellular liver cancer (hematoxylin-eosin; magnification, x200).
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Philadelphia collar was applied to the patient and the severe neck pain disappeared. Pathological examination confirmed moderately differentiated hepatocellular liver cancer (Fig. 4).
Postoperative images at the six month follow up showed no signs of implant dislocation. (A) Lateral X-ray. (B and C) CT 3D. CT, computed tomography; 3D, three dimensional.
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The six-month follow-up X-ray (Fig. 5A) and CT 3D (Fig. 5B and C) showed no signs of implant dislocation and indicated persisting clinical success.