PMC3946458_01
Defect on first pass perfusion at the site of myocardial thinning.
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PMC3946458_01_gr1.jpg
fig1
There was myocardial involvement in the basal anterior, anteroseptal and inferoseptal; mid cavity anterolateral, anterior, antero septal, inferoseptal, and inferior; apical septal, anterior, inferior and lateral wall and apex of the LV with segments of wall thinning (wall thickness <5 mm), severe hypokinesia to akinesia in part and apical dyskinesia with perfusion defect (Fig. 1) and post contrast hyperenhancement extending from the subendocardial to the epicardial surface, varying in transmurality from <25% to >75% wall thickness and near transmural infarction consistent with non viable myocardium (Fig. 2, Fig. 3).
Delayed post contrast enhanced imaging in the 4 chamber view demonstrating basal, mid cavity and apical septal subendocardial scar formation with near transmural involvement in part.
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PMC3946458_01_gr3.jpg
fig3
There was myocardial involvement in the basal anterior, anteroseptal and inferoseptal; mid cavity anterolateral, anterior, antero septal, inferoseptal, and inferior; apical septal, anterior, inferior and lateral wall and apex of the LV with segments of wall thinning (wall thickness <5 mm), severe hypokinesia to akinesia in part and apical dyskinesia with perfusion defect (Fig. 1) and post contrast hyperenhancement extending from the subendocardial to the epicardial surface, varying in transmurality from <25% to >75% wall thickness and near transmural infarction consistent with non viable myocardium (Fig. 2, Fig. 3).