PMC7685367_01
CT images showed "bamboo-like changes" in the spine, and T12 showed a fresh three-column fracture (red arrow) (A) A pseudarthrosis with marked sclerosis at T12, and the upper part of the T12 vertebral body was damaged irregularly (red arrow) (B) The lesion involved the intervertebral space and the lower part of the T11 vertebral body, with pseudarthrosis and obvious hyperplasia (red arrow) (C) The scope of the lesions had extended, with severe thoracolumbar kyphosis and spinal canal stenosis (red arrow) (D) The position of the pedicle screw was well fixed, the kyphosis was obviously corrected, and the intervertebral bone graft was adequate (red arrow) (E) AL segmental bone grafts have good fusion (red arrow) (F).
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CT images showed "bamboo-like changes" in the spine, and the fresh three-column fracture of T12 was characterized by no marked displacement and reduction in vertebral body height (Figure 1A). CT images revealed pseudarthrosis with marked sclerosis at T12, and the upper part of the T12 vertebral body was irregularly damaged, with lesions identified as ALs (Figure 1B). CT showed that the lesion involved the intervertebral space and the lower part of the T11 vertebral body, with pseudarthrosis and obvious hyperplasia (Figure 1C). CT demonstrated that the scope of the lesions had extended, and there was severe thoracolumbar kyphosis and spinal canal stenosis (Figure 1D). Postoperative CT showed great positioning of pedicle screw fixation, obvious correction of kyphosis and adequate intervertebral bone grafting (Figure 1E). CT showed that there was no screw loosening or fracture, no loss of kyphosis correction, and good ALs segmental bone graft fusion (Figure 1F).
Histopathologic analysis showed fibrocartilage tissue calcification and degeneration, necrotic intervertebral disc tissue, fibrovascular hyperplasia, and focal aggregation of inflammatory cells (A and B).
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Histopathologic analysis showed degenerative fibrocartilage tissue calcification, necrotic intervertebral disc tissue, and fibrovascular hyperplasia, and focal accumulation of inflammatory cells (Figure 2).