PMC4100749_01
Images from patient 1. a. Brain CT (axial) the same day of onset, showing a hyperdense lesion in the left putamen (arrow); b. Brain CT(axial) 4 days after onset, revealing a slight high-density in the same area, but the density decreased compared with the initial CT (arrow); c. The FDG-PET scan (axial) 9 days after onset, revealing hyper-perfusion in the left side of the basal ganglion.
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Initial brain CT showed a hyperdense lesion in the left putamen (Figure 1a). Brain CT was performed again (4 days from onset), the high-density of the lesion decreased compared with the initial CT (Figure 1b). Regional cerebral glucose metabolism was significant increased on the left side of the basal ganglion (Figure 1c).
Images from patient 2. a. Brain CT (axial) the same day of onset, showing high density in the left putamen (arrow); b. Brain CT (axial) 24 days after onset, showing slight high-density in the same area (arrow), but the density decreased compared with the initial CT; c. MRI (axial) of the head 62 days after onset, demonstrating hyperintensity on T1-weighted images in the putamen (arrow); d. The FDG-PET scan (axial) 55 days after onset, revealing hypo-perfusion in the left side of the basal ganglion.
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Brain CT showed a hyperdense lesion in the left basal ganglion region (Figure 2a), which was misdiagnosed as hemorrhage at another hospital. CT scan (24 days from onset) showed slightly high attenuation of the left putamen, and the density was decreased compared with the initial CT scan (Figure 2b). MRI of the brain (62 days from onset) showed a strong T1 -weighted hyperintensity in the left lenticular nucleus, mainly in the putamen (Figure 2c). After written informed consent was obtained from patients, brain FDG-PET was performed 55 days after onset, regional cerebral glucose metabolism was significantly decreased on the left side of the basal ganglion (Figure 2d).