PMC6637666_01
Magnetic resonance imaging (MRI) shows hypersignal intense lesions in the subcortical white matter in the bilateral and asymmetric parietal lobe in both fluid-attenuated inversion recovery (FLAIR) sequence (a and b) and apparent diffusion coefficient (ADC; (c)); however, these lesions were not recognized in diffusion-weighted imaging (DWI; (d)). T2*-weighted gradient echo imaging reveals some subcortical microbleeds in the right parietal lobe (e). Postgadolinium T1-weighted images show no enhancement (f).
CRINM2019-5308208.001.jpg
PMC6637666_01_CRINM2019-5308208.001.jpg
fig1
Precisely, subcortical hyperintensity was noted in the bilateral and asymmetric parietal lobe on T2-weighted and fluid-attenuated inversion recovery (FLAIR) images and apparent diffusion coefficient (ADC); however, these lesions were not recognized in diffusion-weighted imaging (DWI), indicating vasogenic edema (Figure 1).
A follow-up brain magnetic resonance imaging (MRI) performed after 4 months shows a reduction of hyperintensity on fluid-attenuated inversion recovery (FLAIR; (a)) and apparent diffusion coefficient (ADC; (b)) in the right parietal lobe. The reduced subcortical microbleeds in the right parietal lobe are noted (c).
CRINM2019-5308208.002.jpg
PMC6637666_01_CRINM2019-5308208.002.jpg
fig2
After 4 months, brain MRI revealed a reduction in hyperintensity on FLAIR images and reduced subcortical microbleeds in the right parietal lobe (Figure 2).