PMC3515947_01
(a) Sagital, (b) axial, and (c) coronal noncontrast images show sellar/suprasellar mass
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F1
T1 isointense with moderate contrast enhancement [Figures 1-4]. The diagnoses considered based on imaging included pituitary macroadenoma, primary sinus abnormality, plasmocytoma, metastasis, lymphoma, or chordoma.
Coronal noncontrast magnetic resonance imaging (MRI) shows a homogeneous soft tissue abnormality occupying the sella and sphenoid sinus
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F2
T1 isointense with moderate contrast enhancement [Figures 1-4]. The diagnoses considered based on imaging included pituitary macroadenoma, primary sinus abnormality, plasmocytoma, metastasis, lymphoma, or chordoma.
Coronal contrast magnetic resonance imaging (MRI) shows homogeneous enhancing soft tissue abnormality occupying the sella and sphenoid sinus
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F3
T1 isointense with moderate contrast enhancement [Figures 1-4]. The diagnoses considered based on imaging included pituitary macroadenoma, primary sinus abnormality, plasmocytoma, metastasis, lymphoma, or chordoma.
Sagittal magnetic resonance imaging (MRI) with contrast; large homogeneous mass occupying the sella turcica, sphenoid sinus, and prepontine cistern; the infundibulum is minimally deviated to the right and normal pituitary appears to be elevated and is seen underneath the optic chiasm
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F4
T1 isointense with moderate contrast enhancement [Figures 1-4]. The diagnoses considered based on imaging included pituitary macroadenoma, primary sinus abnormality, plasmocytoma, metastasis, lymphoma, or chordoma.
Coronal (a) and sagittal (b) magnetic resonance imaging (MRI) noncontrast showing transsphenoid resection of the majority of the large sellar/suprasellar mass
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F5
The MRI showed gross total resection [Figures 5 and 6].
Sagittal contrast enhanced magnetic resonance imaging (MRI) shows resection of the majority of the sellar/suprasellar mass with thickening and nodular enhancement along the clivus. (a) Recurrence of the mass. After repeat endoscopic resection of the lesion (b)
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F6
The MRI showed gross total resection [Figures 5 and 6]. Although the patient was asymptomatic, we performed a routine surveillance MRI six months after surgery, given the rarity of the lesion, which showed a recurrence of the mass [Figure 6a]. We performed repeat endoscopic resection of the lesion [Figure 6b], and follow-up imaging six months after surgery shows no further recurrence.
Histological features of giant cell reparative granuloma; a: Multinucleated giant cells dispersed within fibroblastic matrix, hematoxylin and eosin (H and E)-stained section; original magnification x100; b and C Osteoclast-like giant cells , H and E-stained section and smear; original magnification x200; bar =200 um (panels B and C)
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F7
The final histopathological diagnosis was consistent with GCRG [Figure 7].