PMC6000716_01
3D reconstruction of brain MRA (venous phase). (a) Perfil view in T2. (b) Left posterior oblique view in T1. (c) Perfil view in T1. (d) Superior view in T1
SNI-9-114-g001.jpg
PMC6000716_01_SNI-9-114-g001.jpg
F1
Brain CT, MRI, MRA [Figure 1] and CTA [Figure 2] demonstrated a venous malformation communicating with the superior sagittal sinus under the scalp, without arterial feeder or nidus.
CTA. (a) Right internal carotid artery in anteroposterior view, without arterial malformations. (b) Axial T1 with contrast, with the vascular lesion in posterior interhemispheric fissure
SNI-9-114-g002.jpg
PMC6000716_01_SNI-9-114-g002.jpg
F2
Brain CT, MRI, MRA [Figure 1] and CTA [Figure 2] demonstrated a venous malformation communicating with the superior sagittal sinus under the scalp, without arterial feeder or nidus.
Percutaneous embolization with Onyx  . Needle on the scalp and phlebography. (a) With subtraction. (b) Without subtraction. (c) Making a external compression around the lesion
SNI-9-114-g003.jpg
PMC6000716_01_SNI-9-114-g003.jpg
F3
Following this, an intracranial standard venous phase catheter cerebral DSA was performed, while the lesion was temporarily obliterated by external compression, followed by a percutaneous embolization in one single-session with Onyx   (ev3, USA) [Figure 3], a nonadhesive liquid embolic agent.
Final phlebography with exclusion of the vascular lesion. (a) Final view of the Onyx  without bone subtraction; (b) with subtraction and (c) inversion mode
SNI-9-114-g004.jpg
PMC6000716_01_SNI-9-114-g004.jpg
F4
The final phlebography [Figure 4] showed complete exclusion of the lesion.