PMC9085360_01
Spine MRI showing an extensive multisegmental T2-weighted hyperintense signal between the median plane of C4 and the transition from C7 to D1 (arrows) with symmetrical anterior medullary involvement and restricted diffusion on DWI, but without spinal cord expansion or signs of spinal cord compression. There was a normal T2 vertebral artery flow void, with no apparent signs of acute vertebral artery dissection or intramural hematoma.
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T2-weighted sagittal MRI of the cervical spine disclosed a hyperintense lesion extending from C4 to D1 involving the anterior horns of the spinal cord, without signs of spinal cord compression and no apparent signs of acute vertebral artery dissection or intramural hematoma (Figure 1).
(a) TEE revealing a long-tunneled PFO. (b-d) TEE showing the final position of Cardia Ultrasept PFO 20 mm device, with no residual leak.
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TEE guidance, a Cardia Ultrasept PFO 20 mm device was successfully implanted, with no residual leak (Figures 2 and 3). The procedure was undertaken 8 months after spinal cord infarction, and a lower extremity venous Doppler was repeated prior to PFO closure in order to document resolution of deep venous thrombosis and ensure the safety of the procedure.
(a-c) Cardia Ultrasept PFO 20 mm device implantation.
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TEE guidance, a Cardia Ultrasept PFO 20 mm device was successfully implanted, with no residual leak (Figures 2 and 3). The procedure was undertaken 8 months after spinal cord infarction, and a lower extremity venous Doppler was repeated prior to PFO closure in order to document resolution of deep venous thrombosis and ensure the safety of the procedure.