PMC4860977_01
Early (a) and late (b) phases of fluorescein angiography of the right eye demonstrated hypofluorescence because of media opacity, hyperfluorescence at the disk, vessel wall staining, and track-like transmission defects of RPE.
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F1
Fluorescein angiography (FA) revealed hypofluorescence due to blockage by vitreous opacities, disc leakage, vessel wall staining, and track-like transmission RPE defects all over the fundus [Figure 1].
Fundus photograph after initial treatment. Note the decreased vitritis, media haziness, and disk edema. A white linear subretinal lesion (nematode) is present in the inferonasal quadrant (white arrow) as well as a white intravitreal substance corresponding to the triamcinolone acetonide particles (black arrow).
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F2
The next day, the worm had moved to the inferonasal quadrant of the retina [Figure 2] and later changed its location from time to time [Figure 3].
Fundus photograph after worm mobilization.
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F3
The next day, the worm had moved to the inferonasal quadrant of the retina [Figure 2] and later changed its location from time to time [Figure 3].
Optical coherence tomography demonstrates the subretinal location of the worm presenting as two hyper-.reflective particles over the retinal pigment epithelium (white arrows).
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F4
A linear OCT scan along the nematode showed subretinal hyper-reflectivity which confirmed the subretinal location of the worm [Figure 4].
Fundus photograph immediately after application of laser photocoagulation to the nematode.
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F5
The patient was scheduled for direct laser photocoagulation (spot size, 200 microns; duration, 200 ms; power, 220 mW; number, 22) of the worm [Figure 5].