PMC6401522_01
Axial images of brain MRI performed 7 days (A-C) and 22 days (D) after onset. MRI performed 7 days after onset revealed symmetrical and bilateral hyperintense lesions on diffusion-weighted imaging (DWI) (A), and T2-weighted imaging (B) throughout the entire corpus callosum (CC). Extracallosal lesions could also be seen (A,B). T2-weighted imaging had a considerable amount of distortion due to motion because the patient couldn't cooperate well (B). Gadolinium enhancement could be seen in part of splenium (C, arrow). 22 days after onset, follow-up MRI showed only mild remaining of the formerly impressively hyperintensity on D2-weighted imaging, and the formation of necrosis in the splenium (D).
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F1
TE, 15 ms) images were acquired after intravenous administration of 0.2 mL/kg body weight of gadopentetate dimeglumine at a rate of 2 mL/s. The MRI revealed symmetrical and bilateral hyperintense lesions throughout the entire CC, and in scattered parts of bilateral hemispheric white matter and cortex, visualized on diffusion-weighted imaging (DWI) (Figure 1A), T2-weighted (Figure 1B), and fluid attenuated inversion recovery sequence (FLAIR) (Figure 2A) imaging. Lesions that were enhanced by gadolinium could be seen in the splenium and some extracallosal regions (Figure 1C, 2B). Corresponded to the clinical improvement, follow-up head MRI performed 22 days after onset showed only mild remaining of the formerly impressively hyperintensity on T2-weighted imaging (Figure 1D). Intriguingly, necrosis without enhancement had occurred in the formerly gadolinium-enhanced lesion (Figure 1D, 2C).
Sagittal images of brain MRI performed 7 days (A,B) and 22 days (C) after onset. MRI performed 7 days after onset revealed hyperintense lesions on T2-weighted imaging throughout the entire CC and in some extracallosal regions (A). Lesions that were enhanced by gadolinium could be seen in the splenium (arrow) and some extracallosal regions (B). Follow-up head MRI performed 22 days after onset showed necrosis without enhancement had occurred in the formerly gadolinium-enhanced lesions (C).
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F2
TE, 15 ms) images were acquired after intravenous administration of 0.2 mL/kg body weight of gadopentetate dimeglumine at a rate of 2 mL/s. The MRI revealed symmetrical and bilateral hyperintense lesions throughout the entire CC, and in scattered parts of bilateral hemispheric white matter and cortex, visualized on diffusion-weighted imaging (DWI) (Figure 1A), T2-weighted (Figure 1B), and fluid attenuated inversion recovery sequence (FLAIR) (Figure 2A) imaging. Lesions that were enhanced by gadolinium could be seen in the splenium and some extracallosal regions (Figure 1C, 2B). Intriguingly, necrosis without enhancement had occurred in the formerly gadolinium-enhanced lesion (Figure 1D, 2C).