PMC3858884_01
Left femoral nerve. Compound muscle action potential showing a marked amplitude reduction (0.7 mV). Study 2 (9 months).
CRIM.ORTHOPEDICS2013-368290.001.jpg
PMC3858884_01_CRIM.ORTHOPEDICS2013-368290.001.jpg
fig1
In the absence of clinical improvement, it was decided to perform a second study of control, nine months after the first one, which showed a worsening of the parameters evaluated, with greater loss of nerve fibers in the left femoral nerve conduction and profuse presence of pathological spontaneous activity, expression of active axonal damage, or an underlying progression (Figures 1 and 2).
Left vastus medialis. Pathological spontaneous activity (fibrillation potentials, fasciculations). Study 2 (9 months).
CRIM.ORTHOPEDICS2013-368290.002.jpg
PMC3858884_01_CRIM.ORTHOPEDICS2013-368290.002.jpg
fig2
In the absence of clinical improvement, it was decided to perform a second study of control, nine months after the first one, which showed a worsening of the parameters evaluated, with greater loss of nerve fibers in the left femoral nerve conduction and profuse presence of pathological spontaneous activity, expression of active axonal damage, or an underlying progression (Figures 1 and 2).
Vastus medialis. Pattern at full effort reveals significant loss of motor units. Study 3 (18 months).
CRIM.ORTHOPEDICS2013-368290.003.jpg
PMC3858884_01_CRIM.ORTHOPEDICS2013-368290.003.jpg
fig3
Finally, eighteen months after the surgery, without clinical changes, a new EMG study was practiced, which showed a nearly complete disappearance of pathological spontaneous activity and stabilization in neurogenic pattern (Figures 3, 4 and 5).
Vastus medialis. Motor unit action potentials highly polyphasic, low voltage, and short duration. Study 3 (18 months).
CRIM.ORTHOPEDICS2013-368290.004.jpg
PMC3858884_01_CRIM.ORTHOPEDICS2013-368290.004.jpg
fig4
Finally, eighteen months after the surgery, without clinical changes, a new EMG study was practiced, which showed a nearly complete disappearance of pathological spontaneous activity and stabilization in neurogenic pattern (Figures 3, 4 and 5).
Muscle atrophy after 18 months.
CRIM.ORTHOPEDICS2013-368290.005.jpg
PMC3858884_01_CRIM.ORTHOPEDICS2013-368290.005.jpg
fig5
Finally, eighteen months after the surgery, without clinical changes, a new EMG study was practiced, which showed a nearly complete disappearance of pathological spontaneous activity and stabilization in neurogenic pattern (Figures 3, 4 and 5).