PMC9895116_01
Timeline of hospital course with significant findings each day.
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F1
Since the creatinine and calcium initially improved with hydration and furosemide, it was assumed that the etiology of the AKI was a combination of prerenal (dehydration) and intrinsic (related to hypercalcemia) (see Figure 1).
Axial reformatted, lung window CT image through the lower chest demonstrating multiple noncalcified, well-demarcated pulmonary nodules throughout all lobes (black arrows).
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F2
Computed tomography (CT) revealed mediastinal lymphadenopathy that was granulomatous in nature and pulmonary nodules (see Figures 2, 3).
Coronal reformatted, soft-tissue window, CT image through the mediastinum with low attenuation, noncalcified, bihilar, and subcarinal adenopathy (white arrows).
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F3
Computed tomography (CT) revealed mediastinal lymphadenopathy that was granulomatous in nature and pulmonary nodules (see Figures 2, 3).
AP portable chest radiograph with diffuse but lower lobe predominant micronodular opacities and air bronchograms.
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F4
Radiology noted that, in hindsight, there may have been findings consistent with histoplasmosis, specifically diffuse micronodular and hazy pulmonary opacities with bilateral hilar fullness (see Figure 4).