PMC10072962_01
Patient 1 CTG at admittance to the hospital.
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fig1
Cardiotocography (CTG) was interpreted as pathological (Figure 1).
PMC10072962_02
Patient 2 CTG at admittance to the hospital.
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fig2
CTG was pathological (Figure 2).
PMC10072962_03
Patient 3 CTG at admittance to the hospital.
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fig3
CTG upon admission was deemed preterminal (Figure 3), and an abdominal ultrasound showed no signs of fetal movements.
PMC10072962_04
Patient 4 CTG at admittance to the hospital.
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fig4
The following day the CTG was pathologic (Figure 4), and the baby was delivered by cesarean section.
Microscopic picture descripting trophoblast necrosis, chronic histiocytic intervillositis, and perivillous fibrin deposition. Placenta from Case 4.
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fig5
Placental pathology showed MPVFD, approximately 90% of the placenta was affected, and extensive histiocytic intervillositis was reported (Figures 5 and 6).
Placenta with sign of massive perivillous fibrin deposition. Placenta from Case 4.
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fig6
Placental pathology showed MPVFD, approximately 90% of the placenta was affected, and extensive histiocytic intervillositis was reported (Figures 5 and 6).
PMC10072962_05
Patient 5 CTG at admittance to the hospital.
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fig7
CTG upon admission was deemed pathological (Figure 7).