PMC7333059_01
EEG showing asymmetric background activity with the left frontotemporal intercritical discharges.
CRINM2020-6810237.001.jpg
PMC7333059_01_CRINM2020-6810237.001.jpg
fig1
Interictal electroencephalogram (EEG) showed left frontotemporal discharge persisting during sleep (Figure 1).
Serial MRIs in T2 and FLAIR sequences showing hyperintensity in the left frontoinsular region, lenticular, and caudate nuclei (a). Seven months after the first MRI, persistence of hyperintensity, left fronto-insular cortical and homolateral striatum atrophy, and dilatation of the ipsilateral ventricular system (b). After 3 years and 9 months from the first MRI, we noticed an increase of the left hemispheric atrophy (c).
CRINM2020-6810237.002.jpg
PMC7333059_01_CRINM2020-6810237.002.jpg
fig2
Brain magnetic resonance imaging (MRI) showed cortical and subcortical hyperintensity on T2-weighted (T2) and fluid-attenuated inversion recovery (FLAIR) images in the left frontoinsular region, homolateral lenticular, and caudate nuclei (Figure 2(a)). The brain MRI showed cortical hyperintensity on T2 and FLAIR images in left frontoinsular, left frontoinsular cortical atrophy with homolateral striatum atrophy, and dilatation of the ipsilateral ventricular system (Figure 2(b)). A control of the brain MRI was performed on September 2017 and showed an increase of the left hemispheric atrophy (Figure 2(c)).