PMC9858885_01
CTPA of the chest showing no signs of pulmonary embolism, patchy ground glass opacities within the lingula and left lower lobe, mild dependent changes with subsegmental atelectasis within the bilateral lower lobes, and mildly enlarged main pulmonary artery (black arrows). CTPA: computed tomography pulmonary angiogram
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FIG1
An elevated D-dimer prompted chest computed tomography pulmonary angiogram (CTPA) (Figure 1), which revealed no signs of pulmonary embolism, patchy ground glass opacities within the lingula and left lower lobe, mild dependent changes with subsegmental atelectasis within the bilateral lower lobes, and mildly enlarged main pulmonary artery, suggestive of underlying pulmonary artery hypertension, with an upright X-ray showing signs of cardiomegaly (Figure 2).
Upright X-ray showing mild cardiomegaly (black arrows). X-ray: X-radiation
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FIG2
An elevated D-dimer prompted chest computed tomography pulmonary angiogram (CTPA) (Figure 1), which revealed no signs of pulmonary embolism, patchy ground glass opacities within the lingula and left lower lobe, mild dependent changes with subsegmental atelectasis within the bilateral lower lobes, and mildly enlarged main pulmonary artery, suggestive of underlying pulmonary artery hypertension, with an upright X-ray showing signs of cardiomegaly (Figure 2).
ECG on admission depicting tachycardia and ventricular hypertrophy in leads 4-6. ECG: electrocardiography
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FIG3
Electrocardiography (ECG) revealed sinus tachycardia and left ventricular hypertrophy (LVH) (Figure 3).
2D Doppler echocardiogram showing an EF of 36%. 2D: two dimensional, EF: ejection fraction
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VID1
Nuclear medicine myocardial perfusion single-photon emission computed tomography (SPECT) at rest and stress indicated no evidence of a fixed defect or stress-induced ischemia (Figure 4).