PMC4724073_01
Vimentin+, 20x
13256_2016_800_Fig4_HTML.jpg
PMC4724073_01_13256_2016_800_Fig4_HTML.jpg
Fig4
Immunohistochemistry showed strong immunoreactivity for vimentin (Fig. 4), smooth muscle actin (SMA) (Fig. 5) and desmin (Fig. 6), while p16 (Cyclin-dependent kinase inhibitor 2A) showed focal immunoreactivity (Fig. 7), estrogen (ER) and progesterone (PR) were positive (Figs. 8 and 9), Ki-67 (a monoclonal antibody) expressed a low proliferation index (Fig. 10), whereas p21 and p53 were negative.
Desmin+, 20x
13256_2016_800_Fig6_HTML.jpg
PMC4724073_01_13256_2016_800_Fig6_HTML.jpg
Fig6
Immunohistochemistry showed strong immunoreactivity for vimentin (Fig. 4), smooth muscle actin (SMA) (Fig. 5) and desmin (Fig. 6), while p16 (Cyclin-dependent kinase inhibitor 2A) showed focal immunoreactivity (Fig. 7), estrogen (ER) and progesterone (PR) were positive (Figs. 8 and 9), Ki-67 (a monoclonal antibody) expressed a low proliferation index (Fig. 10), whereas p21 and p53 were negative.
Estrogen+, 40x
13256_2016_800_Fig8_HTML.jpg
PMC4724073_01_13256_2016_800_Fig8_HTML.jpg
Fig8
Immunohistochemistry showed strong immunoreactivity for vimentin (Fig. 4), smooth muscle actin (SMA) (Fig. 5) and desmin (Fig. 6), while p16 (Cyclin-dependent kinase inhibitor 2A) showed focal immunoreactivity (Fig. 7), estrogen (ER) and progesterone (PR) were positive (Figs. 8 and 9), Ki-67 (a monoclonal antibody) expressed a low proliferation index (Fig. 10), whereas p21 and p53 were negative.
Progesterone+, 40x
13256_2016_800_Fig9_HTML.jpg
PMC4724073_01_13256_2016_800_Fig9_HTML.jpg
Fig9
Immunohistochemistry showed strong immunoreactivity for vimentin (Fig. 4), smooth muscle actin (SMA) (Fig. 5) and desmin (Fig. 6), while p16 (Cyclin-dependent kinase inhibitor 2A) showed focal immunoreactivity (Fig. 7), estrogen (ER) and progesterone (PR) were positive (Figs. 8 and 9), Ki-67 (a monoclonal antibody) expressed a low proliferation index (Fig. 10), whereas p21 and p53 were negative.