How is saccadic movement managed in the brain? I haven't been able to find any decent articles on this specific topic. So I have three (hopefully quick) questions:


What part of the brain is actively controlling saccadic movement?
If this part were to be removed, does saccadic movement cease to exist and cause the person to go blind from not being able to look around?
Additionally, is there a disorder which eliminates saccadic movement in some way?
The superior colliculus can be regarded as the center of saccadic eye movement. It contains a topographically organized representation of the visual space in its upper layers, and saccade-related activity in its deeper layers (Van Gisbergen et al., 1987).
Ablation of the superior colliculus unilaterally eliminates saccades contralateral to the lesion, and hence bilateral ablation will likely eliminate all saccades (Schiller et al., 1987). However, the superior colliculi do not mediate conscious vision, but mediate eye movements. Blindness results after ablation of the primary visual cortex, and obviously after removal of the retinae, or the optic nerves. Also note that "not being able to look around" is not dependent only on saccadic eye movement, because head-scanning is still possible. 
A number of conditions may affect saccades. For example, stroke in the brain stem affecting the superior colliculi or cranial nerves may slow saccades and lesions in the brain stem may abolish them completely (Ramat et al., 2007). 


References
- Ramat et al., Brain (2007; 130: 10–35
- Schiller et al., J Neurophysiol (1987); 57(4): 1033-49
- Van Gisbergen et al., Neurosci (1987),  21(2): 541-55