, ▁and ▁the ▁amplit udes ▁increase ▁again . ▁In ▁LE MS , ▁this ▁remains ▁ins u fficient ▁to ▁reach ▁a ▁level ▁sufficient ▁for ▁transmission ▁of ▁an ▁imp ulse ▁from ▁n erve ▁to ▁mus cle ; ▁all ▁can ▁be ▁attributed ▁to ▁ins u fficient ▁calci um ▁in ▁the ▁n erve ▁terminal . ▁A ▁similar ▁pattern ▁is ▁witness ed ▁in ▁my as then ia ▁gra vis . ▁In ▁LE MS , ▁in ▁response ▁to ▁exer cis ing ▁the ▁mus cle , ▁the ▁C MAP ▁amplitude ▁increases ▁greatly ▁( over ▁ 2 0 0 %, ▁often ▁much ▁more ). ▁This ▁also ▁occurs ▁on ▁the ▁administration ▁of ▁a ▁rapid ▁burst ▁of ▁elect rical ▁stim uli ▁( 2 0 ▁imp uls es ▁per ▁second ▁for ▁ 1 0 ▁seconds ). ▁This ▁is ▁attributed ▁to ▁the ▁infl ux ▁of ▁calci um ▁in ▁response ▁to ▁these ▁stim uli . ▁On ▁single - fi ber ▁exam ination , ▁features ▁may ▁include ▁increased ▁j itter ▁( seen ▁in ▁other ▁dise ases ▁of ▁neur om us cular ▁transmission ) ▁and ▁blocking . ▁ ▁Blood ▁tests ▁may ▁be ▁performed ▁to ▁exclude ▁other ▁causes ▁of ▁mus cle ▁disease ▁( e lev ated ▁cre at ine ▁kin ase ▁may ▁indicate ▁a ▁my os itis , ▁and ▁ab normal ▁thy roid ▁function ▁tests ▁may ▁indicate ▁thy ro to xic ▁my op ath y ). ▁Ant ib od ies ▁against ▁voltage - g ated ▁calci um ▁channels ▁can ▁be ▁identified ▁in ▁ 8 5 % ▁of ▁people ▁with ▁EM G - conf ir med ▁LE MS . ▁Once