▁and ▁in hib ited ▁by ▁acid ic ▁ph osph ol ip ids . ▁The ▁complex ▁then ▁serves ▁to ▁anchor ▁act in ▁fil aments ▁to ▁the ▁memb rane ▁and ▁thus , ▁helps ▁to ▁rein force ▁force ▁on ▁tal in ▁within ▁the ▁foc al ▁ad hes ions . ▁ ▁The ▁loss ▁of ▁vin cul in ▁impact s ▁a ▁variety ▁of ▁cell ▁functions ; ▁it ▁dis rupt s ▁the ▁formation ▁of ▁the ▁complex , ▁and ▁prevents ▁cell ▁ad hes ion ▁and ▁spread ing . ▁The ▁absence ▁of ▁the ▁protein ▁demonstr ates ▁a ▁decrease ▁in ▁spread ing ▁of ▁cells , ▁accompanied ▁by ▁reduced ▁stress ▁fi ber ▁formation , ▁formation ▁of ▁fewer ▁foc al ▁ad hes ions , ▁and ▁in hib ition ▁of ▁l am el lip odia ▁extension . ▁It ▁was ▁discovered ▁that ▁cells ▁that ▁are ▁def ic ient ▁in ▁vin cul in ▁have ▁growth ▁con es ▁that ▁advance ▁more ▁slowly , ▁as ▁well ▁as ▁fil op odia ▁and ▁l am elli po ida ▁that ▁were ▁less ▁stable ▁than ▁the ▁wild - type . ▁Based ▁on ▁research , ▁it ▁has ▁been ▁post ulated ▁that ▁the ▁lack ▁of ▁vin cul in ▁may ▁decrease ▁cell ▁ad hes ion ▁by ▁in hib iting ▁foc al ▁ad hes ion ▁assembly ▁and ▁prevent ing ▁act in ▁polym er ization . ▁On ▁the ▁other ▁hand , ▁over expression ▁of ▁vin cul in ▁may ▁restore ▁ad hes ion ▁and ▁spread ing ▁by ▁prom oting ▁rec ruit ment ▁of ▁c yt os ke let al ▁prote ins ▁to ▁the ▁foc al ▁ad hes ion ▁complex