▁Similarly , ▁gli al ▁scar ▁formation ▁was ▁significantly ▁reduced ▁in ▁the ▁N - ac et yl gl uc os am ine ▁ 6 - O - s ulf otr ans fer ase - 1 ▁m ice , ▁and ▁as ▁a ▁result , ▁n erve ▁reg ener ation ▁was ▁less ▁in hib ited . ▁ ▁Other ▁in hib itory ▁factors ▁Prote ins ▁of ▁o lig od end rit ic ▁or ▁gli al ▁de br is ▁origin ▁that ▁influence ▁neur or eg ener ation : ▁NO GO ▁– The ▁protein ▁family ▁N ogo , ▁particularly ▁N ogo - A , ▁has ▁been ▁identified ▁as ▁an ▁in hib itor ▁of ▁rem y el ination ▁in ▁the ▁C NS , ▁especially ▁in ▁auto imm une ▁medi ated ▁dem y el ination , ▁such ▁as ▁found ▁in ▁experimental ▁auto imm une ▁en ce phal omy el itis ▁( E AE ), ▁and ▁multiple ▁s cl eros is ▁( MS ). ▁N ogo ▁A ▁functions ▁via ▁either ▁its ▁am ino - N ogo ▁termin us ▁through ▁an ▁unknown ▁re ceptor , ▁or ▁by ▁its ▁N ogo - 6 6 ▁termin us ▁through ▁Ng R 1 , ▁p 7 5 , ▁T RO Y ▁or ▁L ING O 1 . ▁An tagon ising ▁this ▁in hib itor ▁results ▁in ▁improved ▁rem y el ination , ▁as ▁it ▁is ▁involved ▁in ▁the ▁R ho A ▁path way . ▁N I - 3 5 ▁a ▁non - per miss ive ▁growth ▁factor ▁from ▁my elin . ▁M AG ▁– My elin -