res ia ), ▁over g row th ▁of ▁the ▁um bil ical ▁st ump , ▁and ▁ab normal ities ▁of ▁the ▁gen ital ia ▁and ▁an us . ▁The ▁medical ▁comp lications ▁associated ▁with ▁this ▁condition ▁are ▁often ▁severe ▁and ▁may ▁well ▁be ▁life - th re aten ing ▁in ▁inf ancy ▁or ▁early ▁child hood . ▁ ▁Gen et ics ▁Several ▁mut ations ▁in ▁the ▁F G FR 2 ▁gene ▁( a ▁gene ▁coding ▁for ▁a ▁protein ▁called ▁fib ro blast ▁growth ▁factor ▁re ceptor ▁ 2 , ▁which ▁is ▁involved ▁in ▁important ▁signal ing ▁path ways ) ▁are ▁known ▁to ▁cause ▁Be are – Ste vens on ▁cut is ▁g yr ata ▁synd rome ; ▁however , ▁not ▁all ▁patients ▁with ▁the ▁condition ▁have ▁a ▁mut ation ▁in ▁their ▁F G FR 2 ▁gene . ▁Any ▁alternative ▁underlying ▁causes ▁are ▁currently ▁un ident ified . ▁The ▁synd rome ▁follows ▁an ▁aut os om al ▁dominant ▁pattern , ▁meaning ▁that ▁if ▁one ▁of ▁the ▁two ▁available ▁gen es ▁car ries ▁a ▁mut ation ▁the ▁synd rome ▁will ▁result . ▁Currently , ▁no ▁famil ial ▁histor ies ▁are ▁known ▁( in ▁other ▁words , ▁there ▁are ▁no ▁reports ▁of ▁cases ▁in ▁which ▁a ▁parent ▁carrying ▁a ▁mut ation ▁in ▁their ▁F G FR 2 ▁gene ▁then ▁propag ated ▁said ▁mut ation ▁to ▁his ▁or ▁her ▁child ). ▁ ▁Di agn osis ▁ ▁Tre at ment ▁ ▁Inc idence ▁Be are – Ste vens on ▁cut is ▁g yr ata ▁synd rome ▁is ▁so