▁does ▁not ▁become ▁activ ated ▁and ▁cannot ▁change ▁trans cription ▁patterns ▁to ▁in hib it ▁neur onal ▁out g row th . ▁ ▁Th era pe ut ic ▁In hib ition ▁ ▁It ▁is ▁reasonable ▁that ▁in hib ition ▁of ▁the ▁above ▁mechanism ▁could ▁aid ▁the ▁recovery ▁of ▁those ▁suffering ▁from ▁sp inal ▁cord ▁inj uries . ▁ ▁One ▁such ▁ther apy ▁is ▁currently ▁in ▁clin ical ▁tri als . ▁ ▁The ▁drug , ▁called ▁C eth rin , ▁is ▁produced ▁by ▁a ▁group ▁called ▁Al ser es . ▁ ▁C eth rin ▁is ▁a ▁RO CK ▁in hib itor ▁and ▁therefore ▁acts ▁in ▁the ▁above ▁path way ▁to ▁prevent ▁the ▁activation ▁of ▁RO CK ▁so ▁neur ite ▁out g row th ▁can ▁occur . ▁ ▁C eth rin ▁is ▁applied ▁as ▁a ▁paste ▁to ▁the ▁site ▁of ▁injury ▁during ▁decom pression ▁surg ery . ▁ ▁Reg ulation ▁of ▁Visual ▁Cor tex ▁Pl astic ity ▁The ▁N ogo - 6 6 ▁re ceptor ▁( N g R ) ▁limits ▁experience - dri ven ▁visual ▁cor tex ▁pl astic ity . ▁In ▁mut ant ▁m ice , ▁non - function al ▁Ng R ▁resulted ▁in ▁enh ancement ▁of ▁visual ▁cor tex ▁pl astic ity ▁after ▁the ▁critical ▁period ▁into ▁ad ul th ood , ▁such ▁that ▁adult ▁pl astic ity ▁in ▁the ▁mut ant ▁m ice ▁res emble d ▁normal ▁visual ▁pl astic ity ▁in ▁ju ven ile ▁m ice ▁bra ins . ▁This ▁function ▁of ▁Ng R ▁is ▁of ▁particular ▁interest ▁to ▁the