ular ▁mechan isms , ▁including ▁production ▁of ▁ant im icro b ial ▁pe pt ides ▁( AMP s ), ▁re active ▁o xygen ▁species ▁( RO S ) ▁or ▁activation ▁of ▁pro phen ol ▁ox id ase ▁cas cade . ▁Cell ular ▁part ▁of ▁insect ▁inn ate ▁imm unity ▁are ▁hem ocy tes , ▁which ▁can ▁eliminate ▁path og ens ▁by ▁nod ulation , ▁enc aps ulation ▁or ▁ph ag oc yt osis . ▁The ▁inn ate ▁response ▁during ▁imm une ▁prim ing ▁diff ers ▁based ▁on ▁the ▁experimental ▁setup , ▁but ▁generally ▁it ▁involves ▁enh ancement ▁of ▁hum oral ▁inn ate ▁imm une ▁mechan isms ▁and ▁increased ▁levels ▁of ▁hem ocy tes . ▁There ▁are ▁two ▁hypoth et ical ▁scenarios ▁of ▁imm une ▁induction , ▁on ▁which ▁imm une ▁prim ing ▁mechanism ▁could ▁be ▁based . ▁The ▁first ▁mechanism ▁is ▁indu cement ▁of ▁long - last ing ▁def ences , ▁like ▁circul ating ▁imm une ▁mole cules , ▁by ▁the ▁prim ing ▁ant ig ens ▁in ▁the ▁host ▁body , ▁which ▁remain ▁until ▁the ▁secondary ▁encounter . ▁Second ▁mechanism ▁describes ▁a ▁drop ▁after ▁the ▁initial ▁prim ing ▁response , ▁but ▁stronger ▁defence ▁upon ▁secondary ▁challenge . ▁The ▁most ▁probable ▁scenario ▁is ▁the ▁combination ▁of ▁these ▁two ▁mechan isms . ▁ ▁Trans - gener ational ▁imm une ▁prim ing ▁ ▁Trans - gener ational ▁imm une ▁prim ing ▁( T G IP ) ▁describes ▁the ▁transfer ▁of ▁parent al ▁imm un ological ▁experience ▁to ▁its ▁pro gen y , ▁which ▁may ▁help ▁the