Blood pressure that is abnormally high.
A rapid heartrate that exceeds the range of the normal resting heartrate for age.
Primary trimethylaminuria is characterized by a fishy odor resembling that of rotten or decaying fish that results from excess excretion of trimethylamine in the urine, breath, sweat, and reproductive fluids. No physical symptoms are associated with trimethylaminuria. Affected individuals appear normal and healthy; however, the unpleasant odor often results in social and psychological problems. Symptoms are usually present from birth and may worsen during puberty. In females, symptoms are more severe just before and during menstruation, after taking oral contraceptives, and around the time of menopause.
An increased susceptibility to pneumonia as manifested by a history of recurrent episodes of pneumonia.
A decrease in the number of neutrophils in the peripheral blood.
Clinical utility gene card for: trimethylaminuria.
Archaebiotics: proposed therapeutic use of archaea to prevent trimethylaminuria and cardiovascular disease.
Relationships between flavin-containing mono-oxygenase 3 (FMO3) genotype and trimethylaminuria phenotype in a Japanese population.
Bonitos with low content of malodorous trimethylamine as palliative care for self-reported Japanese trimethylaminuria subjects.
Genetic polymorphism of the flavin-containing monooxygenase 3 (FMO3) associated with trimethylaminuria (fish odor syndrome): observations from Japanese patients.
Primary trimethylaminuria (fish odor syndrome) and hypothyroidism in an adolescent.
Fish odor syndrome (trimethylaminuria) supporting the possible FMO3 down expression in childhood: a case report.
Epilepsy and trimethylaminuria: A new case report and literature review.
Diagnosis and management of trimethylaminuria (FMO3 deficiency) in children.
Clinical utility gene card for: Trimethylaminuria - update 2014.
A novel mutation in the flavin-containing monooxygenase 3 gene (FMO3) of a Norwegian family causes trimethylaminuria.
A novel deletion in the flavin-containing monooxygenase gene (FMO3) in a Greek patient with trimethylaminuria.
Trimethylaminuria (fish odor syndrome): genotype characterization among Portuguese patients.
FMO3 allelic variants in Sicilian and Sardinian populations: trimethylaminuria and absence of fish-like body odor.
Trimethylaminuria (fish-odor syndrome): a case report.