{'fraction_non_alphanumeric': 0.020380434782608696, 'fraction_numerical': 0.002717391304347826, 'mean_word_length': 5.299145299145299, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Sturge‐Weber syndrome without facial nevus: a case report and review of the literature

Sturge-Weber syndrome (SWS) is an example of a neuro-ectodermal disease characterized by facial port wine nevus affecting the area innervated by the first sensory branch of the trigeminal nerve, the retina of the eye and the ipsilateral leptomeninges. Seizures are the usual neurological manifestation in up to 80% of patients (I). The presence of facial nevus gives a diagnostic clue in a majority of children with SWS. In the absence of facial angioma, the diagnosis depends upon the radiological confirmation of the leptomeningeal angiometosis on MRA or conventional catheter angiography and intracranial calcifications demonstrable on CT scans.