Category Archives: Medical Illness Environmental Home Testing



Powerline EMF Safe Distance Cell Phone RF Radiation Surveys




EMF EMI Surveys Consulting

EMF Testing / Magnetic Field Survey & Safe Distance Measurement Services








Magnetic / Electric / RF & EMF Shielding Consultations including proper grounding


Radon Gas Measurements & Surveys (Short / Long Term Testing)






Magnetized & Radiation Contaminated Metals, Stone and Imported Products




Hospital Infant Anti-Abduction Security Systems







OSHA Sound / Noise Level Surveys for Safety Audits




Nanotoxicology Safety Consulting



Commercial, industrial, occupational health and large scale residential applications.


Formaldehyde: Indoor Air Quality Pollutant and Testing


Formaldehyde Testing Dallas – Fort Worth










Formaldehyde Molecule

Formaldehyde Molecule





Testing Particulate Matter and Indoor Air Quality (IAQ)

Respirable Suspended Particles (RSP) are particles or fibers in the air that are small enough to be inhaled. Particles can exist in a number of states including solids, liquids, a combination of the two. Particulate matter that is either inherently organic life (such as viruses or bacteria) or are given off by biological life (such as mold spores, pieces of fungi, pollen, plant fibers, etc.) are what is known as bioaerosols.

There is another class of compounds which are chemicals released by biological organisms known as Microbial Volatile Organic Compounds or MVOCs. These are typically low molecular weight chemicals which have a high vapor pressure so they tend to remain in the air long enough to be detected and/or breathed in. Often they are responsible for odors that result from bacterial / toxic black mold growth or other primary/secondary metabolic byproducts of microorganisms.

In general, particles of 10 um (microns) or greater in diameter will tend to get caught in the mucous membranes of the nose before they have a chance to settle in the lungs. But particles between 2.5 um and 10 um (known as PM10 which is short for Particulate Matter 10 um) can migrate to tissues of the upper respiratory tract where they can cause health issues depending on the nature and the concentration of the contamination. Particles smaller than 2.5 um (known as PM2.5) can actually penetrate the alveoli, or small air sacs of the lungs and enter the blood stream.

The particle counter used by ScanTech measures both small (0.5 – 2.5 um) and large (2.5 – 10 um) particles to warn of potential hazards in the air you breathe.

Particle Size Penetration Respiratory System

The smaller the particles, the deeper they penetrate into the respiratory system




20,000+ Lung Cancer Deaths Annually in the United States






Radon Gas Risk Statistics

Radon Gas Risk Statistics




Radon Gas Inhaled Lung Damage Alpha Radiation

Radon Lung Damage Alpha Radiation









RADON FAQ PAGE for Dallas / Fort Worth & Surrounding Counties



Are these statements true and if so, how exactly do they work to cause malfunctions in cell replication? This is the science of etiology, or the causation mechanism of imbalance and disease including cancer.

Broken Chromosomes (at arrow points)

Broken Chromosomes (at arrow points)

First, we have to understand how cancer can arise. Since there are around 130 different types of cancer each with a different metabolic and chemical pathway, it is beyond the scope of this website to address them all in detail. But some underlying themes regarding all cancers are:

1) The abnormal growth and replication of a single damaged cancerous cell through several stages into a malignant tumor which (if untreated) may metastasize and spread throughout the body.

2) Cell damage leading to cancer comes from a variety and / or combination of factors including genetic predispositions and environmental factors.

3) Environmental factors account for most (90% +) of all cancers and include air pollution, ionizing radiation, (including radon gas) carcinogenic chemicals, bacteria, viruses, tobacco use, diet and a lack of exercise. (lifestyle) Your body and the cells within it are constantly repairing damage from these conditions. This in combination with genetic predispositions determine the “tipping point” at which a rogue cell escapes or overruns the body’s ability to halt a cancerous cell as illustrated below:

DNA Damage Mechanisms and Repair with regard to Cancer: An Overview

DNA Damage Mechanisms and Repair with regard to Cancer: An Overview

BTW, exogenous damage means “from outside” while endogenous damage comes from chemical processes generated by the body.

4) It can be impossible to tell exactly what caused a particular cancer, as most cancers have more than one possible instigating agent. But in some cases, certain causes are more likely than others.

Some Carcinogens and Linked Cancers

Some Carcinogens and Linked Cancers

A DNA adduct is a part of the DNA molecule which is covalently bonded with a carcinogenic molecule. Notice that there is more than one type of carcinogen that could be responsible for liver cancer, bladder cancer, and lung cancer. One carcinogen is found in Chinese herbs, while leukemia can also be induced by chemotherapy for other cancers. As this table illustrates, Western and Eastern medicine are not perfect.

The World Health Organization lists both ionizing radiation (such as UV or Ultraviolet Light) as being a carcinogen and NON-IONIZING radiation (which would include EMF, RF, Cell Phones, Smart Meters, Microwaves, etc.) as being a POSSIBLE carcinogen though the mechanism through which that could happen is not known or proven.

As an aside, the difference between ionizing radiation and non-ionizing radiation is that ionizing radiation is a photon/electromagnetic wave with enough energy to knock electrons off of an atom (typically in the outer valence shell) so that a charged particle called an “ion” is created. The line between the two types is typically found in wavelengths of radiation shorter than 400 nm (nanometers) or in frequency terms around 700 THz. (Terahertz) At the frequency ranges of ionizing radiation or higher, it is common to express the photon or electromagnetic wave in terms of energy, which at the aforementioned boundary is slightly over 3 eV. (electron Volts)

Ionization of an Atom by Displacement of an Electron from a Radiation Event


So back to the original question – could EMF cause damage to a cell in such a way that cancer can arise? To answer that, we have to look at actual DNA damage in more detail which will be featured in the next post.

DNA Damage in the Cell and Mechanisms of Cancer: A Quick Overview Part II

DNA can be damaged in many different ways through a variety of mechanisms, many of them from exogenous (external) causes, and some from endogenous (internal) factors. On average, this damage tallies out to 10,000 to over 1,000,000 molecular lesions per cell per day in the human body.

Fortunately, most of this damage is either automatically repaired on the DNA itself or the damage can set off a global response to prevent reproduction of the mutated/damaged cell. But occasionally the body’s systems do not catch the error in time (or the damage is repaired incorrectly) and the cell is allowed to multiply into a tumor – either benign or malignant cancer.

The forms of DNA damage can be classified by agent and the effect on the DNA strand itself.

Exogenous (External)

1) UV radiation from the sun which is technically a form of EMF, but it is electromagnetic radiation that is at a high enough frequency / energy level that it is crosses over into the ionizing radiation regime. UV-A radiation can lead to the formation of free radicals which cause indirect damage. UV-B radiation can crosslink adjacent thymine and cytosine bases and create thymine or pyrimidine dimers, which is known as direct damage.

2) Other forms of high energy radiation (X-Ray, Alpha, Beta, Gamma)

3) Viruses (also known as oncoviruses) are most common but certain bacteria and parasites can be responsible

4) Chemical carcinogens including from natural sources (such as plant toxins or asbestos) known as mutagens. Particularly aromatic compounds that can insert themselves between the planar nitrogenous bases of the DNA helix. (also known as intercalating agents)

5) Hydrolysis (cleaving of bonds by water)

6) Thermal disruption

7) Some studies show or suggest evidence of damage from certain Electromagnetic Fields (EMFs) including both low frequency power lines and higher frequency RF (Radio Frequencies) including those in the microwave range which are used in cell phone / tower communication.

Endogenous (Internal)

1) Attack by Reactive Oxygen Species (ROS) or Reactive Nitrogen Species (RNS) generated in the course of normal bodily functions which can form hydrogen peroxide and ammonia respectively. (both are toxic) The presence of these compounds come from oxidative deamination which form oxoacids in the liver.

Single Strand Breaks – a break in one of the two deoxyribose-phosphate backbone / strands in a DNA molecule

Double Strand Breaks – breaks in both of the two deoxyribose-phosphate backbone / strands in a DNA molecule

Examples of DNA Damage

Examples of DNA Damage

Point Mutation – A mutation that alters a single nitrogenous base. It can be either the substitution of one base for another, or the deletion / addition of a single base or small number of bases.

Base Substitution Mutation – The substitution of one base pair for another in DNA. Because of the degenerate nature of the genetic code, base substitution may or may not alter the amino acid encoded. If the new codon from the base substitution still results in the same intended amino acid, then the mutation is considered “silent”. But if it results in a different amino acid being produced, then it is termed a “missense mutation”  as the sense (bio-genetic intelligibility) of the codon produced after transcription of the mutant gene is altered. There are two categories: Transitions and Transversions. A transistion does not change the type of bases in the base pair as in a pyrimidine vs. a purine, but a transversion does. A variety of human genetic disorders (such as sickle cell anemia) are caused by base substitutions.

Nonsense Mutations – A special category of base substitution arises when a base is converted in such a way that the transcribed codon is converted to a stop codon. This stop codon results in premature termination of the translation and leads to a truncated (incomplete) protein.

Frameshift Defects – The addition or deletion of a single base has much more profound consquences than does the substitution of one base for another. Frameshift mutations alter the reading frame in the mRNA (messenger RNA) downstream of the mutation, which in turn will mean the majority of the protein will be altered. Frameshifts can also cause the same effect as Nonsense Mutations as 3 in 64 codons are stop codons which means there is a relatively high probability the protein coding will be prematurely stopped at the wrong point.

Triplet Repeat Expansion Mutations

DNA Damage & Repair Sample Table

DNA Damage & Repair Sample Table

DNA Damage Responses

DNA Damage Responses

Cancers, Diseases and Other Physical Illnesses Believed to Be Linked to EMF/RF Radiation

This is a list of the various conditions whose risk factors are believed to be correlated to EMFs (electromagnetic fields) from power lines, cell towers, cell phones, smart meters, microwaves and other electrical/electronic devices. This is not a confirmation or affirmation that any of these are necessarily true or to suggest hard safety level limits; but it is part of the continuing blog and educational journey of determining the true causation and effect relation.

Where applicable, I have put abbreviations and/or descriptions of these conditions as they traditionally appear in research literature for convenient reference to coming articles on the epidemiology of possibly EMF related diseases and conditions. Also the risk factors they are allegedly linked to are listed as well.


In the developed world, about 50% of all adult men and roughly 33% of all adult women develop cancer during their lifetime.

Cancer is generally described as an unwanted / unregulated growth of cells with malignancies having a potential to metastasize and invade other organ systems and tissues. Cancer types are organized by organ and cell type they originate from and by the morphological characteristics of the cancer cells themselves.

Carcinogenesis is a multi-stage process which may include genetic (alteration of DNA composition and structure) and epigenetic (alteration in gene expression without DNA alteration) changes. It is suggested that external stimulus such as certain types and levels of EMF/RF may contribute to the multi-stage cancer process by influencing epigenetic changes resulting in effects such as abnormal cell proliferation and differentiation, apoptosis or modified adaptive responses.


Leukemogenic factors are agents which are or believed to be related to promoting the development of leukemia.

Childhood Leukemia (CL) – Power lines  (rates are 4.5 per 100,000 developed world and 2.7 per 100,000 in developing countries) CL is a family of biologically heterogeneous neoplasms and represents the most common cancer in children with 30-50 new cases per million children developing worldwide annually.

subtypes include:

Acute Lymphoblastic Leukemia (ALL) – Represents 80% of all cases of CL in white populations aged 0-14 years but has a peak incidence between 2-5 years. Most common chromosomal aberrations in ALL are hyperdiploidy (35%) and the presence of the TEL-AML1 gene fusion. (25%)

Acute Myeloid Leukemia (AML) – Represents 15 % of all cases and incidence rises with increasing age and has few causative agents, but include benzene, alkylator, topoisomerase II inhibitors and ionizing radiation (which includes radon gas in up to 13% – 25% of all cases estimated worldwide) which is of a wavelength far beyond that of power lines.

In general from birth, the incidence of leukemia rises to a peak of around 3 years of age, then declining briefly before steadily rising throughout life. This is unlike most cancers as it has a peak incidence early in life and a short latency.

Acute Nonlymphocytic Leukemia (AnLL) – In adults, benzene exposure is causally related. ( > 40 ppm cumulative exposure = >120 ppb annually over a 76 year lifetime)

Acute Lymphocytic Leukemia – Limited evidence of benzene related exposure causality.

Multiple Myeloma – Limited evidence of benzene related exposure causality.

Non-Hodgkin Lymphoma – Limited evidence of benzene related exposure causality.

Chronic Lymphocytic Leukemia (CLL)

Chronic Myeloid Leukemia (CML)



Glioma  (a malignant form of brain cancer) – Allegedly linked to cell phones as it has a short latency and is fast growing.

Glioblastomas (most common form of brain cancer)


Meningioma (a diverse set of tumors in the meninges of the brain; usually benign) – Allegedly linked to cell phones though the Interphone study shows reverse causality.

Acoustic Neuroma or Vestibular Schwannomas (benign intracranial tumor, but can be dangerous) – Allegedly linked to cell phones



Breast Cancer

Lymphatic Cancers

Hematopoietic Cancers

Pituitary Gland Tumors

Uveal Melanoma (cancer of the eye)



Amyotrophic Lateral Sclerosis (ALS) / Motor Neuron Disease

Alzheimer’s Disease

Parkinson’s Disease


Myeloproliferative Disorders (MPDs)

Lymphoproliferative Disorders (LPDs)

Idiopathic Environmental Intolerance (IEI-EMF)









2) Which leads to the next issue:









6) It is a politically charged subject.



















Correlation is not causation.

