text: Figure 1. SNItrans induces death of small primary afferent neurons, accompanied by a reduction in volume, not cell density, of the dorsal root ganglion. (A) 
  Approach to differentially labelled intact afferents with tdTomato and damaged afferents with GFP after peripheral nerve injury using the AvilFlpO;Atf3CreERT2;RC:: 
  FLTG mouse line and schematic of experimental timeline. (B) Representative image of GFP, tdTomato, and NeuN expression in an L4 DRG, 2 weeks after SNItrans. 
  Scale bars 5 100 mm. (C and D) Stereological quantification of the total number of DRG neurons (C) or number of axotomized and intact neurons (D) in the L4 DRG 
  1, 2, 4, and 8 weeks after SNItrans or contralateral (contra) to injury. (C) One-way ANOVA with Tukey posttests; F4,10 5 37.98, P , 0.001. (D) Two-way RM ANOVA; 
  Timepoint 3 Color interaction F4,10 5 39.04, P , 0.001, n 5 3 mice; Tukey posttests (between injured groups): †P , 0.05 vs contra, ‡P , 0.05 vs 1-week. (E) 
  Volume of DRG-containing cells (ie, excluding white matter tracts) following SNItrans. One-way ANOVA with Tukey posttests; F4,10 5 21.25, P , 0.001, n 5 3. (F) 
  Neuronal density within the DRG following SNItrans. One-way ANOVA; F4,10 5 2.77, P 5 0.09, n 5 3. (G) Population distribution of uninjured and injured afferents by 
  cross-sectional area, 1 and 8 weeks post-SNItrans. Kolmogorov–Smirnov tests of cumulative distributions; Uninjured: D 5 0.08, P 5 0.18; Injured: D 5 0.32, P , 
  0.001; n 5 310 to 427 neurons from 3 mice.*P , 0.05,**P , 0.01,***P , 0.001 vs contra. ANOVA, analysis of variance; DRG, dorsal root ganglion; GFP, green 
  fluorescent protein. 