text: A.H. Cooper et al.·165 (2024) 2863–2876 
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  injury (Fig. S6A–C, http://links.lww.com/PAIN/C84), indicating 
  that any loss of neurons within specific neuronal subpopulations 
  was not biased towards soma size. Collectively, these data show 
  that unrepaired axonal damage to peripheral sensory neurons 
  induces a partial loss of Trpm81 and CGRP1 subpopulations, 
  but no major loss of myelinated afferents. 
  Based on our findings of preferential 
  
  developed transgenic recombinase driver lines, we have 
  shown that 
  loss is biased across molecularly defined 
  subpopulations. Nonpeptidergic nociceptive neurons are 
  particularly susceptible to loss, with almost all Mrgprd1 
  axotomized afferents lost following an unrepaired transection 
  injury (SNItrans) and roughly half lost following a model which 
  contrastingly allows for nerve regenerations (SNIcrush). 
  Finally, we have observed that the vulnerability of Mrgprd1 
  neurons extends to the in vitro setting and provide data to 
  support 
  loss is driven by a lack of 
  the hypothesis that 
  neurotrophic support following injury. 