Semaglutide is a GLP-1 receptor agonist that regulates appetite and glucose metabolism through several mechanisms. It works by slowing the absorption of food in the gut, reducing the feeling of hunger, and promoting feelings of fullness or satiety.

When we eat, certain nutrients such as carbohydrates are broken down into glucose and other substances in our digestive system. These then enter our bloodstream, causing an increase in insulin levels to regulate blood sugar levels. In response to this, GLP-1 (glucagon-like peptide-1) is released from the intestinal cells. GLP-1 acts on the pancreas to produce insulin and also signals to the brain to reduce the sensation of hunger.

Semaglutide mimics the effects of naturally occurring GLP-1 by binding to its receptors on the cell surface, stimulating the release of insulin and suppressing glucagon secretion. This helps lower blood sugar levels after meals while simultaneously increasing feelings of satiety, leading to reduced hunger and food intake.

Overall, semaglutide's ability to reduce hunger stems from its action as a GLP-1 analogue, which modifies glucose metabolism and appetite signaling pathways in the body, resulting in increased feelings of fullness and decreased feelings of hunger.
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