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Julie Bowen: My kids flaked on me
Julie Bowen is "so proud" of her three children for raising money for the earthquake in Nepal.
The blonde actress is best known for playing larger-than-life mother of three Claire Dunphy in hit TV series Modern Family. She currently has a break from filming to spend time with her husband Scott and their offspring Oliver, John and Gustav, but a recent family outing didn't end up going to plan.
"I'm on hiatus with my real family and this weekend we decided to do some good. My children, I've got three kids... We did a lemonade stand to raise money for the victims of [the] Nepal [earthquake, which has killed thousands]. These are not this year's shorts I'm wearing right there," she joked to Jimmy Kimmel of her red hotpants, adding that her children hand squeezed around 160 lemons for the cause.
"We set up, we did it and I was so proud of my kids, they worked so hard. But after like, two hours, which is a long time in the life of a five-year-old, to go, 'Ughhh it's boring.' And I said, 'OK, but hang in there.' Dad had gone to get food... And they were supposed to come back. They left me alone in a canyon at the base of a very busy hiking trail and now I'm the one... I'm an adult."
She continued to amuse the audience by revealing how embarrassing it was for a celebrity, whose "salary you can Google", to be stood asking strangers for money.
On the topic of awkward incidents, Julie also recalled her last basketball game, watching the Los Angeles Lakers at the Staple Center. She and her friend arrived late but rather than waiting to get to their seats, a security guard urged them to cross the court during the game. The situation was made even more uncomfortable when another guard informed her that he had to stop five men from removing her from the venue following her move.
© Cover Media


text:
Sequoia's Robert Goldfarb and David Poppe are seeking a firm with as much earnings potential at a low price as Buffett's company 20 years ago, but they note the outcome is unlikely.
Robert Goldfarb and David Poppe, presidents of Ruane, Cunniff & Goldfarb and portfolio managers of Sequoia SEQUX, recently answered our questions on the virtues of a concentrated portfolio, the challenges that pharmaceutical companies will continue to face in light of government fiscal problems, and how proper long-term company analyses can lead to exceptional investments. They also stated that the universe of quality businesses has moved away from its all-time lows and discussed specific opportunities currently found in the retail sector.
1. You steered the portfolio away from a high concentration effort to a more diversified approach. What led you to this strategy change? Are you concerned that a more diversified approach could potentially dilute the portfolio?
We still believe strongly in the virtues of concentration. The best way to outperform an index of stocks is to own a concentrated portfolio of great businesses purchased at reasonable valuations. Our views on this subject haven't changed in 40 years. If you exclude Berkshire Hathaway BRK.A BRK.B, our concentration in our next five largest holdings is as high today as it has been through most of our history. But we came to view a 30% weighting in Berkshire Hathaway as inappropriate, largely because of Warren Buffett's acknowledgement that the law of large numbers would cause Berkshire's rate of earnings growth to slow materially in the future. We were fortunate to be able to reduce Berkshire to 12% in 2010 through two significant sales at reasonable prices when the company entered the Standard & Poor's 500 Index and the Russell 2000 Index, which forced index funds to buy the stock irrespective of price. Our current weighting in Berkshire is slightly more than 10%. Because Berkshire is selling at the same price at which we sold it nearly two years ago and Warren Buffett has significantly increased the company's intrinsic value through massive deployment of capital, we are extremely comfortable with this weighting and with Berkshire's prospects.
This has been a managed transition for Sequoia. In anticipation of one day owning less Berkshire, we hired a number of outstanding analysts during a period of several years. This new team has allowed us to analyze and purchase more businesses with the characteristics we have always liked. All the proceeds of our Berkshire sales were fully redeployed in new ideas. As long as these new businesses are as attractive as the ones we own and have the same price-value relationships, they should not dilute the performance of the portfolio. We would be nearly fully invested today had not we experienced a net inflow of more than $900 million in 2011, which we did not anticipate. We would love to find an earnings compounding machine as attractively priced as Berkshire was in 1990 and let it grow to a 30% weighting of Sequoia, but we view such a prospect as unlikely.
2. When you spoke to us during the summer, you mentioned that you didn't like the traditional Big Pharma companies because of the high reimbursement risk associated with them. Has this thesis remained the same? Why or why not?
The intensifying fiscal problems of governments in the United States and Europe have only reinforced our belief that pharmaceutical companies will face more challenging reimbursement issues in the years ahead.
3. Given today's environment, are there any areas that you're staying away from and plan on staying away from in the near term? Why? On the flip side, for what areas do you have the most enthusiasm?
We think of ourselves as item buyers. We are always trying to find the next great business, and we don't spend much time contemplating sector exposure. As long-term investors, we are not averse to looking hard and buying a terrific company selling at a reasonable price because its short-term outlook may cause others to stay away. The key is getting the long-term analysis right. A strong company in a disfavored sector can make for a great investment.
4. Do you think that quality is still selling at all-time lows?
We don't think quality is selling at all-time lows. We own a number of very high-quality businesses that sell at or close to all-time highs. It may be fair to say that some high-quality mega-caps, cyclicals, financials, and companies with heavy exposure to Europe have been marked down, but the universe of quality businesses is not uniformly cheap.
5. What opportunities are you finding in the retail sector?
We like the retailers we own but have a policy of not commenting on prospective investments. Of the retailers we own, TJX Companies TJX, Target TGT, Wal-Mart Stores WMT, and Costco Wholesale COST offer remarkable savings to consumers and seem well-positioned for the current economic times. O'Reilly Automotive ORLY and Advance Auto Parts AAP benefit from the aging of the U.S. auto fleet and the importance of inventory availability in automotive hard parts. They carry an essential, yet specialized offering that consumers tend to need in an emergency. If you want to drive the car, you must buy the part. Discounters and Internet retailers cannot readily compete in a business that requires immediate availability of massive numbers of slow-turning parts, which has the effect of protecting high margins for the few players that can earn a reasonable return on the inventory investment.
©2017 Morningstar Advisor. All right reserved.

subset: commoncrawl


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1872 Open Top® Army
"The graceful lines created by nineteenth century artisans have been captured fully in this late 20th century work of art in steel. The Cimarron Open Top is without a doubt the finest replica to come forth in the last decade." - "Texas Jack" Harvey -


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There's been no official word on new cast members joining "The Avengers: Age of Ultron," but series star Samuel L. Jackson's words do pack some punch!
According to the actor, who plays Nick Fury in the Marvel Universe, Elizabeth Olsen (the younger sister of Mary-Kate and Ashley Olsen) will be in the "Avengers" sequel.
"I don't know what she's doing, if she's on the inside or the outside," he told the Wall Street Journal, referring to if she'll side with Earth's Mightiest Heroes or someone else. "I haven't seen a script."
WATCH: Aaron Taylor-Johnson Discusses The Avengers: Age Of Ultron
The actress has long been rumored to play Scarlet Witch, with Aaron Taylor-Johnson rumored to play her brother Quicksilver.
Director Joss Whedon hinted at the siblings appearing in the next film back in April.
WATCH: Joss Whedon On The Avengers 2: How Does He Plan To Top The First Film?
"I've got these two characters [in the first draft of my script] – my favorite characters in the comic book, a brother-sister act," he said at the time. "They're in the movie, that's exciting."
According to Jackson, filming is slated to begin in March 2014.
-- Jesse Spero


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On Wednesday Homestead (Fla.) South Dade wide receiver C.J. Worton chose to sign with Florida over West Virginia. Though many considered Worton a lock for the Gators after picking up the offer and ...
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text:
What manufacturer makes washing machines with suds savers on them now?
By Jeltster from Canada
Add your voice! Click below to answer. ThriftyFun is powered by your wisdom!
Is your washer over 10 years old? Replace it with a new Energy Star qualified washer and you could save over $135 each year on your utility bills. That's like getting your High Efficiency (HE) detergent free year round.good luck.
Clothes washers undergo Energy Star evaluation at Whirlpool Corp.'s Clyde, Ohio, manufacturing plant. The test panel measures total energy consumed including water usage, recently added to the Energy Star specifications. Good luck.
As far as I know any machine that uses HE soap saves on soap. I have a Maytag that specifies using HE soap. HE means High Efficiency. You use less soap per load.
A friend who was rather inventive made his own suds saver form a small barrel. He is dead now so I can't get directions.
Add your voice! Click below to answer. ThriftyFun is powered by your wisdom!


text:
Hans Egede, who published a dictionary of Greenland Eskimo in 1739, says that the Eskimos themselves told him that they got the name from the Norsemen who once lived in Greenland. [Gordon, p.217-8]
cloud-cleaver, an imaginary sail jokingly assumed to be carried by Yankee ships. [W. Clark Russell, "Sailors' Word Book," 1883]


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As I mentioned, I saw women knitting all over China, especially in Jiangxi Province. And every child was wearing a hand-knit sweater. They were always knitting in the round with very long (about 18 inches) double-pointed needles.
Our guide in Jiangxi was surprised that I was a knitter. She said knitting was not popular among young people; it was mostly "Grandmas" who did it. In fact, her mother-in-law had already knit many things for the children she and her husband hadn't had yet! (Since she and her husband are both only children, the government allows them to have two children.)
This is a woman knitting at the yarn store in Nanchang. More about that in the next post. Let's just say I came home with some yarn!


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JERUSALEM (Aug. 5)
Dr. Nahum Goldmann, president of the World Zionist Organization, today warned Jewish youth throughout the world "not to submit to conformism" and urged the younger generation to "safeguard the uniqueness and separateness of Jewry."
Addressing some 400 delegates from 39 countries attending the second World Conference of Jewish youth, Dr. Goldmann said that many of the Jewish youth were abandoning Judaism in search of greater challenges in foreign fields. He said they could be attracted to challenges "on the home front" by a change of values and revification of Jewish life. He warned that the Diaspora community which failed to maintain contact with Israel could not continue to exist.
Rabbi Emanuel Rackman, of Far Rockaway, N.Y., assistant to the president of Yeshiva University and former president of the Rabbinical Council of America, told the delegates that the conference was being held during a crisis period for the Jewish people and its heritage, when "acculturation and assimilation are decimating the ranks and threatening the very survival of Diaspora communities."
On the other hand, he asserted, "many Israeli intellectuals and cultural forces are contributing towards a creeping alienation between Jews of Israel nationality and non-Israeli Jews, whose Jewishness is rooted principally in some form of religious identification of community or social interest."


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Protesters on 16-year-old boy's death: 'NYPD, KKK, how...
There are 34 comments on the The Raw Story story from Mar 12, 2013, titled Protesters on 16-year-old boy's death: 'NYPD, KKK, how.... In it, The Raw Story reports that:
A protest march in Brooklyn, New York, on Monday night turned into a confrontation between police and demonstrators amid reports of bottles being thrown at officers out of anger over the shooting death of a 16-year-old boy at the hands of New York Police detectives.
Join the discussion below, or Read more at The Raw Story.
#1 Mar 12, 2013
What the heck???
#2 Mar 13, 2013
This thug, gang member pointed a loaded gun at Police ...what do you think would happen?
#3 Mar 13, 2013
What are they talking about, KKK?
Since: Jul 10
#4 Mar 14, 2013
If I was the NYPD Commish, if after last night's 46 arrests the crowd doesn't get the message and they come back tonight looking for more, just open fire. Tell SWAT and the riot cops to lock and load, don't even bring cuffs, just extra ammo, and fill that street with blood until the gutter overflows. The good people of that neighborhood are being held hostage by a bunch of people who have no common sense and are only looking for an excuse to act like complete retards, which is pretty insulting to retards.
#5 Mar 14, 2013
There's no hope for these people. They are protesting the rightfull shooting of a thug gang member who was armed with a gun and pointed at the police....case closed .
Absolute savages who are doomed to continue to live and act the way they do with the help of their so called political " poverty pimp" elected officials .
#7 Mar 14, 2013
All those savages voted for Obozo.
#8 Mar 14, 2013
That's how Obama would see it! The SAVAGES behavior, silence.
#9 Mar 14, 2013
send in the drones....
#11 Mar 14, 2013
"The real issue in Brooklyn is cops have been harassing us for a long time," he said. "It needs to stop."
No, the real issue is that blacks commit the majority of shootings in NYC (and spare me the white boy psychos in Newtown and Aurora, etc.- none of that has anything to do w/crime in NYC). Flatbush has been a hole for years created by the very same black "community". No one has spoken out about why a 16 year old would have a gun on them. No one has spoken out why a 16 year old already has ONE felony, let a lone a few felonies, under his belt. No one has questioned what the hell this child's parents were doing that even allowed their son to have a criminal record.
In the past 20-30 years the majority of those who have killed cops in the line of duty, including black officers, have been black perps. Yet NO ONE speaks out about that either.
I am sick and tired of the same old story and the same old nonsense by the same group of people who can't see that THEY are the cause of the problem, not the police.
#12 Mar 14, 2013
Sounds like a good start,maybe next time your buddies will be smarter.It sure didn't work out for that GANG BANGER, pointing guns at people that will shoot back.
#13 Mar 14, 2013
The punk kid is dead ...that what happens when you point a gun at someone. The savages that are running wild in the street should be ashamed of themselves.
#14 Mar 16, 2013
I haven't heard a peep about this story on mainstream news networks. That's Good! I figured that liberal reporters would try to turn this into another Trayvon Martin incident, which it is far from.
#15 Mar 16, 2013
Good for the NYPD. I don't care what color skin you have. If you are stupid enough to point a loaded gun at the police, you dumb butt gets what's coming to ya. Thin the herd i say. But of course in the black "community" it is always someone else fault for the problem these savage apes have created for themselves. They are never responsible for their own mayhem they have created generation after generation. And they wonder why white people and middle/high income responsible hard working black people move out to the suburbs and enroll their children in private schools. Can you blame them?
#16 Mar 16, 2013
for the description, I CONCUR!
#17 Mar 16, 2013
Kkk?? Exquise me loool??? Wow these flatbush flatheads really get dramatic and blow things way out of proportion. Its always a huge race thing if a black person with a loaded gun gets mowed down. They turn everything into racial issue. Pretty sad that community is choosing to act like savages from the 1980s and causing further anguish to this kids family. It sucks that he had to die but why did he point a gun at officers? Anyone of any race does that and expect to die. Protest what?? Allow gang members to point guns at people? Ya thats great, your community looking real foolish right now. Put yourself in a cops shoes. Some young punk points a clear gun at you,,, now what its you or him??? Do you really care how old he is. That community should be protesting gang violence that puts cops on edge and gun control.
United States
#18 Mar 17, 2013
The "N" Word...
#21 Mar 17, 2013
"Obama: Leading us to failure"
Since: Jul 09
The United States of America
#22 Mar 17, 2013
Silly black people will be forever spinning theirs wheels never getting anywhere in life
United States
#23 Mar 17, 2013
Robb Barham
The Roman Empire
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[email protected] com
THEROMANEMPIRE.INFO [email protected] https://soundcloud.com/theromanempire
The Roman Empire
Leeds/Manchester, Britain (UK)
#24 Mar 18, 2013
Roman empire? You on that good crack huh? Get a grip homeslurpie, this about brooklyn.
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text: A few weeks ago I thought the Utes would finish the season winning four of their last five. The loss I thought would come against the Huskies, with the thinking the Utes have played well at home and could beat Arizona in Rice-Eccles Stadium. My feelings still haven't changed on that game and I'm still going with Washington tonight, but this game definitely looks a lot more winnable for the Utes than it did a few weeks ago. The Utes are doing a much better job with the offense since streamlining it and getting John White more involved. Washington's defense hasn't been that great against the run so if the Utes can get him going early it will bode well for an upset. Still, one has to wonder how much of Utah's last two wins were a product of poor opponents and how much was attributed to the Utes' improvement. Let's face it, Cal and Washington State were bad teams that had little fight in them. It will be a much better quality opponent the Huskies face today and it would be shocking to see them throw in the towel like Cal and Washington State did. We'll see, but I'm still thinking the Huskies get this one since it is an away game, but it could be close and a Utah win wouldn't be much of a surprise. Ok, keys to a Utah win.1. Get White going. The Utes need to make a statement in the first quarter and get some nice, sustained drives with White, just as they have in their recent games. Doing so will let quarterback Travis Wilson settle in and will let Utah's defense stay off the field. Perhaps most importantly it will take Washington's crowd out of the game some and that could be huge for the Utes. CenturyLink Field is loud, really, really loud. In fact, I think my ears are still ringing from watching a U2 show there a few years ago. This could be the loudest, most hostile crowd the Utes face this year. It would be good for the Utes to quiet them early.2. Get some turnovers. Washington quarterback Keith Price has thrown 10 interceptions and nine touchdowns this year, quite a difference from a year ago when he threw 33 touchdown passes. Some say his struggles relate more to the lack of talent around him than his play. But really it doesn't matter to the Utes, they need to take advantage of his mistakes and come away with interceptions. This would be a good time for Brian Blechen to get his first of the year.3. Smother Washington tight end Austin Seferian-Jenkins. He is Washington's big-play guy and will be the key player for the Utes to shut down. Utah defensive end Trevor Reilly has done well against tight ends this year and if he draws Seferian-Jenkins, keep an eye on that matchup. Reilly is having a great year and leads the Utes with 50 tackles and four pass breakups.4. Shut down Washington's run game. As posted previously, the Huskies are 3-1 when back Bishop Sankey hit the 100-yard mark with wins over Portland State, Stanford and Cal and a loss to Oregon. If the Utes can prevent him from making many big runs they'll go a long ways in getting an upset.5. Keep Price corralled. The Utes didn't finish the job like they should have against UCLA's Brett Hundley, another mobile quarterback similar to Price. But Joe Kruger showed a new level of play against Washington State now that he is healthy. Let's see if he can hunt down Price and get a few sacks. - Lya Wodraska


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Location: Mycotoxin Prevention and Applied Microbiology ResearchTitle: Biodiversity of important toxigenic fungi that threaten food safety Author
|Moretti, Antonio
|Mule, Giuseppina
|Proctor, Robert
|Susca, Antonia
|Logrieco, Antonia
Submitted to: Meeting Abstract
Publication Type: Abstract Only
Publication Acceptance Date: 9/7/2012
Publication Date: 9/7/2012
Citation: Moretti, A., Mule, G., Proctor, R., Susca, A., Logrieco, A.F. 2012. Biodiversity of important toxigenic fungi that threaten food safety. Meeting Abstract. Interpretive Summary:
Technical Abstract: Phenotypic and metabolic plasticity of toxigenic fungi that threaten food safety allows these microorganisms to colonize a broad range of agriculturally important crops and to adapt to a range of environmental conditions. In addition, trans-global transportation and trade of plant products significantly contribute to the spread of toxigenic fungi worldwide. The increase in studies on molecular biodiversity of toxigenic fungi at a global level, particularly studies addressing rapid detection systems, has also revealed the existence of intra- and inter-specific differences in mycotoxin biosynthetic pathways. For some of the most worrisome species of Aspergillus and Fusarium (sexual stage Gibberella), differences in mycotoxin biosynthetic pathways could be related to specific evolutionary adaptation of species and/or populations. Examples of these differences have been reported for: a) the fumonisin biosynthetic pathway in the black Aspergillus and G. fujikuroi species complexes; b) the trichothecene pathway in Fusarium; and c) the presence and absence of the aflatoxin biosynthetic pathway in two populations of A. flavus. A review of available information on the variability of the most relevant toxigenic species of Aspergillus and Fusarium with respect to mycotoxin profile, molecular diversity, phylogeny, and biosynthetic pathway will be provided in the presentation. We will emphasize the importance of increasing the knowledge of molecular biodiversity of toxigenic fungi to better understand host-pathogen and environment-fungus interactions and to prevent mycotoxin production at critical points of crop production from pre-harvest to storage.


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Shots fired during attempted robbery near Ball State campus
MUNCIE, Ind.
Ball State officials alerted students after a reported robbery attempt near campus Tuesday night.
Police say shots were fired during the incident at University Avenue and Reserve Street around 8:45 p.m. Investigators say the attempted robbery happened during an arranged meeting to sell personal items. Both the victim and suspect pulled out weapons, with the victim shooting a handgun into the air.
No injuries were reported in the incident.
The Muncie Star Press reported university officials issued a text message alert to students around 9:30 p.m.
The incident occurred less than a mile from the Ball State campus. The suspect fled the scene in a direction away from the campus, but had not yet been apprehended, the newspaper reported.


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JACKSONVILLE, Fla. - With college football officially kicking off next week, fans packed the Jacksonville Sports Council's inaugural Speaker Series luncheon Thursday to hear ESPN analyst Kirk Herbstreit's take on the upcoming season.
Herbstreit said he likes Florida State's chances to repeat as national champion, particularly with the return of Heisman-trophy winner Jameis Winston at quarterback.
"I think they have probably the best talent in the country," Herbstreit said. "I'm anxious to see them play, I really am, because when you win a title, I think everybody wants to watch and they want to scrutinize what you're doing, especially Jameis for winning the Heisman."
He said the Seminoles will have to avoid a potential letdown, however, if coach Jimbo Fisher hopes to prove that his team has staying power.
The Gators, on the other hand, are out to prove something else: that last season was just a blip. Herbstreit said he thinks Florida can reverse last year's woes that led to a 4-8 season, including an embarrassing loss to Georgia Southern.
The addition of offensive coordinator Kurt Roper will help in that effort, Herbstreit said.
"They've got to stay healthy. Last year, they had to deal with more injuries than anybody in the country," he said. "Things seemed to just spiral out of control from there. When your own fans are turning on you, it tells you how bad it is, so they need to get off to a really good start to re-establish some confidence and belief, and I think they will."
Florida opens with games against Idaho, Eastern Michigan and Kentucky.
While Herbstreit thinks the Gators can improve from last year, Georgia is his pick to win the SEC East.
Quarterback Hutson Mason and running back Todd Gurley lead a solid group of offensive players, and former Florida State defensive coordinator Jeremy Pruitt comes over to take the reins of a talented defense.
"They start with some tough games: Clemson and South Carolina, but I think they're up to it," Herbstreit said. "I like the Dawgs."
Copyright 2014 by News4Jax.com. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.


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I'm running postgers 7.1.3 on a origin 2000 running Irix 6.5.13 and oid2name hangs up. It's because on line 68 "while ((c = getopt(argc, argv, "H:p:U:P:d:t:o:xh?")) != EOF)" we're trying to compare a char (unsigned int on IRIX) with EOF(-1 here). it's fixed declaring int c instead of char c. hope it helps
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Rimbey is a town in central Alberta, Canada. It is located at the junction of Highways 20 and 53 in the Blindman River valley area, approximately northwest of Red Deer and southwest of Edmonton.
Provincially, Rimbey is part of the Rimbey-Rocky Mountain House-Sundre electoral district and federally in the Wetaskiwin riding.
Officially made a community in 1902, the first name given to the settlement at the turn of the century was Kansas Ridge as many of the settlers came from Kansas. Among them were the three Rimbey brothers (Sam, Ben, and Jim) for whom the town was officially named after in 1904. The Rimbeys moved to Canada from Scott County, Illinois, having moved to Illinois in the 1830s from Maryland. They were originally from Pennsylvania.
In 1919 the Lacombe and Blindman Valley Electric Railway (later part of the Canadian Pacific Railway) reached Rimbey, and there was much enthusiasm for the "new town" by the tracks (now Highway 20). Two grain companies built elevators the following year and Rimbey's population swelled to 319 by 1921.
The Second World War brought abrupt changes to Rimbey, as young men and sometimes their families left the village. When war was over some returned and others did not. Many new faces came to Rimbey and the population surged to 634 by 1946.
Research Tips


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Whats your favorite pistol
My top 3 are glock 22c with laser and night sights along with suppresor
My second is 5-7 pistol which the only reson is because its going right threw armour no matter what
My 3rd i would like to get it is a glock 18c with a laser suppresor and extended mag
But the only way i could get this is im class 3 or buy it illegally
I only have two pistols.
Number one is my CZ75 Pre B in 9mm
Number two ist my Sig Sauer P226 Classic in 9mm
GunBoss, we have an "Introductions" area. Would you stop by and say "hello"?
My top three are the Colt Single Action Army Artillery model in .45 Colt, WWII Vintage Colt 1911, and either the Ruger SR9c or the XDm 9mm.
The Dracos are fairly cool, but I find them not practical. I would like to own one someday though ;).
I have two favorites. A Springer 4" barrel sitting on an Officer's Frame and my Colt Gold Cup Trophy:
I don't have any shooting pistols but I do have some self defense revolvers and auto loaders and my favorites are a XD40SC and LCP-CT BUG. The wife's favorite is her LCR-CT .38.
my favorite, any of the 1911's, Colt, Remington, Wilson, Springfield, Dan Wesson, ect. ect. in any caliber 45acp, 38 super or 9mm.
My Medusa M47... :cool:
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Welcome, Im sorry you are dealing with all of this
I was on the lupus spectrum for 3+ yrs and managed it with diet and exercise before things got bad last November and I was formerly DXed in Feb of this year. I am 31 and have a 12 year old son that I raised for 8 yrs as a single mother before marrying the most amazing supportive man in the world. Its hard to explain to them what you are dealing with and my son definitely has a reaction to my declining health. We are currently in therapy for some coping skills because we are so bonded. The Spoon Theory is what I gave everyone at my office who was curious becuase "i dont look sick" just fat and tired
Best of luck and be patient with yourself!


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Why is yahoo taking my pictures off of my blogs when they are marked mature content in the settings?
today yahoo took 3 pictures off of my blogs stating they did not meet yahoo guidelines. my blogs are marked mature content. i thought when marked mature content that meant pictures as well as text. how do you contact them and place a complaint?
Why not try for a little class instead? Would it kill you?
How can i find pictures for young, mature,and old rivers?
Pls gimme the picures (labeled)pls.!!
Pictures are in the link below!!!!
Young rivers usually occur in highland or mountainous regions and have narrow, rocky channels with many boulders. The water may form waterfalls or foam and gurgle as it rushes over the rocks. Young rivers have few tributaries.
Mature rivers are those that have reached flat land as their tributaries pour more water into them. They often become flooded during periods of heavy rain or snowmelt. Their beds tend to be muddy rather than rocky because of the sediment (particles of sand or soil) carried into them by swift-flowing streams, and the valleys through which they flow are usually fairly broad. Few waterfalls occur on mature rivers, but they may have many bends or loops as they curl across the land looking for the lowest level to follow.
A river is old near its mouth, where layers of sediment build up over time. Here, the land is wide and flat, and the water travels more smoothly than in young and mature rivers.
What are some sexy ideas for revealing pictures to send to my boyfriend? Mature answers please?
Before anyone yells at me to not do it....to bad. I want too. Im in love with my boyfriend with him and we have been dating for a very long time.
Like what to wear that might be in my closet
How to pose?
Hop should i wear my hair?
what do you mean revealing pictures and what do i wear in my closet...doesnt make sense, typical woman
Are illustrated books with mature story lines popular?
I'm currently writing a mature horror story which involves death etc but the book is to illustrated in my own style with pictures.
Is there such thing has a mature picture book? And are they popular? Because if not I'm going to have to make a guyren's book which means I'll have to change my story.
BTW it is not a graphic novel.
The Hobbit had illustrations, though I do think originally it was intended to be a guyren's book (in my library it's found in the adult SciFi section). There is also a book by Kenneth Oppel called Darkwing, a prequel to his Silverwing series intended for YA, which is illustrated -- and beautifully. It's one of my favorite books. The storyline is not horror, but there are some really interested and fairly creepy scenarios which lead to some disturbing illustrations.
And no, I don't think they're popular -- or if they even ever were. But I like them. Neil Gaiman has a few illustrated books that are what I would call guyren's horror, like Coraline. First time I read it, my face was asking, "Wtf?!" But he is a great author and has great illustrators, and it all works together well.
It shouldn't matter what's popular and what's not, however. You do what you like, and what makes you happy, and what makes you feel as though your goals in creating this story and making it true and real have been accomplished. I have been told numerous times to illustrate my stories, and I think in a way it is very helpful and makes the story much more alive, depending on your writing style. Narnia was also illustrated. ;)
What can I do to my hair/makeup to look more mature. (pictures)?
I'm 13 but everyone says I look like an 11 or 12 year old (i'm almost 5 foot, so i'm kinda petite)
Well I wanna look older! Plus I like this guy...and, well, you know...:P
Another question, and I'm not asking this to be lame and get compliments and stuff, I just wanna honestly know if you think I'm even somewhat pretty. I know that shouldn't matter and stuff but I wanna know.
so here are some recent pictures...
in the fifth one down I have darker highlights (it was about a year ago and my hair is sooo blonde naturally) should I get them again?
Try to look like Jessica, she's a hottie.
You'll be a cutie when you grow up, no need to rush it.
Pictures of easy to manage, mature looking above the shoulder cuts?
No pixie cuts or short short bobs
Im 15 I look 13 so I need somthing that looks my age or more
PICTURES PICTURES PLEASe
I always bring in pictures of farah fath when i get my hair done.. ac4.yt-thm-a03.yimg.com/image/098… , ac4.yt-thm-a01.yimg.com/image/207… ,
Selling Pictures (fetishes)Mature only needed for answers!?
Is there any place that lets you set up a page for free to sell pictures? They won't be nude but I noticed there were alot of people out there that have fetishes that aren't the normal boobs, butt, etc. I would love to hit that market and sell pics of feet, legs,etc. Any ideas on how and where I could get started.
I would hit up craigslist...there are lots of people out there who would be willing to buy them from you not like film companies or anything but just like everyday joe's...that way you can name ur own price and only sell to who you want to. Just post sumthing up there and you can include pics if you want you should get plenty of responses.
HELP!! How can I look cuter and more mature? (Pictures)?
I am 16(and a half!). I think that I generally look younger than I really am. I don't think I am ugly; I am average/ kinda pretty, but nothing special. I have never had a boyfriend or been kissed, which is pretty embarrassing. And btw, you might mention I need to lose weight, but I am already working on that! haha Thank you so much for answering! Any advice is appreciated!
here is me:
hmm I think you do look 16...So no worries bout that, and looking cuter isn't a big deal for you. You got your attractive blue eyes ;) as soon as you take off your braces, or maybe even with those braces...you'll have ur bf ;)
How long does it take for a rottweiler to fully mature? (pictures of cute doggies!!)?
as in growing
and what are some good foods to feed them? any other suggestions on stuff to do? i got a how to train your rottweiler book and i've looked up more stuff on the internet but more suggestions won't hurt
here are some pics, i doubt any of you care but i don't believe in false advertisement
First off your rott is beautiful!!! I love rottweilers and they truly are fantastic dogs!
Rottweilers mature very slowly! They are considered puppies until they are 2yrs. Growth wise they do not finish filling out until between 3-5 yrs. As for food get a high grade food with alot of protein in it.
Extra tips
- please make sure to socialize your puppy well. even though they are great dogs they tend to be more protective so if you socialize them really well you lower the risk of any aggressive tendencies when they get older.
- also they do need exercise, but i don't suggest biking or jogging a long time with them until 2 or 3 yrs. they just aren't mature physically enough to handle it and it can cause alot of hip problems in the long run.
© adultspankingtgp.com, mature pictures


text:
Crisp back in lineup; Cespedes serves as DH
NEW YORK -- Coco Crisp returned the A's lineup for Thursday's series finale after missing Wednesday's 7-4 victory over the Yankees with ongoing neck soreness. Crisp was back in the leadoff spot and playing in center field.
Crisp, who sustained a neck strain about a month ago, is more likely to receive more days off in the upcoming weeks to help it heal.
"I don't know when he's going to be fully healthy," manager Bob Melvin said. "There probably will be some days here where he'll be on and days when he'll be off to try to get him healthier."
Yoenis Cespedes was not in the field for Thursday's game as he served as the designated hitter. Brandon Moss took Cespedes' usual place in left field. Melvin made the decision to have Cespedes get a little bit of a break to help with the soreness in his right shoulder.
Melvin said Cespedes doesn't feel discomfort when he swings the bat, but more when he has to make a throw in the outfield.
"He is having a little bit of those shoulder problems still," Melvin said. "We're trying to take as much pressure off as we can. This is as much of a day off without getting a full day off as I can give him today."
Pitcher Eric O'Flaherty is getting closer to making his return to the A's. O'Flaherty is scheduled to pitch an inning for Triple-A Stockton on Friday. Melvin said O'Flaherty should be done with simulated bullpens as he has been recovering from Tommy John surgery in May 2013.
"He's going to be pitching regular games now," Melvin said.
It is still unclear how many games O'Flaherty will pitch in the Minors. Melvin said he still hopes O'Flaherty will return to the A's in early July.
Nate Taylor is a contributor to MLB.com. This story was not subject to the approval of Major League Baseball or its clubs.


text:
To look younger
Amazing - Marina Del Ray, CA
To look younger
Carla was recommended by a friend to me and I'm thrilled with the way she does my botox and fillers. Super professional. Can't wait to go back!
5 out of 5 stars
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text:
β-Adrenergic receptor (AR) signaling is a regulator of skeletal muscle protein synthesis and mitochondrial biogenesis in mice. We hypothesized that β-AR blockade blunts postexercise skeletal muscle mitochondrial protein synthesis rates in adult humans. Six healthy men (mean ± SD: 26 ± 6 yr old, 39.9 ± 4.9 ml·kg−1·min−1 peak O2 uptake, 26.7 ± 2.0 kg/m2 body mass index) performed 1 h of stationary cycle ergometer exercise (60% peak O2 uptake) during 1) β-AR blockade (intravenous propranolol) and 2) administration of saline (control). Skeletal muscle mitochondrial, myofibrillar, and sarcoplasmic protein synthesis rates were assessed using [2H5]phenylalanine incorporation into skeletal muscle proteins after exercise. The mRNA content of signals for mitochondrial biogenesis was determined using real-time PCR. β-AR blockade decreased mitochondrial (from 0.217 ± 0.076 to 0.135 ± 0.031%/h, P < 0.05), but not myofibrillar or sarcoplasmic, protein synthesis rates. Peroxisome proliferator-activated receptor-γ coactivator-1α mRNA was increased ∼2.5-fold (P < 0.05) at 5 h compared with 1 h postexercise but was not influenced by β-AR blockade. We conclude that decreased β-AR signaling during cycling can blunt the postexercise increase in mitochondrial protein synthesis rates without affecting mRNA content.
- mitochondrial biogenesis
- propranolol
- stable isotope tracer
- aerobic exercise
decreased mitochondrial content and function have been reported with aging and may contribute to chronic disease (30) and muscle wasting (37, 49). Because of their potential role in the pathogenesis of disease, mitochondria are a target of lifestyle and pharmacological therapies. For example, aerobic exercise stimulates mitochondrial proliferation (15) and likely contributes to the health benefits of exercise training (22). Conversely, certain drug classes cause mitochondrial dysfunction (9) and may contribute to skeletal muscle myopathies (10). Given the high prevalence of chronic disease and the widespread use of drug therapies and lifestyle recommendations for treatment of chronic diseases, it is necessary to understand how concurrent exercise and drug therapy affect mitochondrial biogenesis.
Recent evidence suggests that mitochondrial biogenesis following aerobic exercise is partly mediated through β-adrenergic receptor (AR) signaling (25). In mice, selective β2-AR stimulation under resting conditions increased the mRNA content of a regulator of mitochondrial biogenesis, peroxisome proliferator-activated receptor-γ coactivator-1α (PGC-1α) (24). Additionally, mice that were treated with the nonselective β-AR antagonist propranolol had a blunted increase of PGC-1α mRNA following aerobic exercise (25). Such findings are consistent with human aerobic training studies reporting blunted adaptations of select mitochondrial enzymes and maximal aerobic capacity during nonselective β-AR antagonist treatment (1, 46, 52). β-AR antagonists (β-blockers) are used to treat cardiovascular diseases and are commonly prescribed in conjunction with exercise (39). It is possible that β-blockers may impair mitochondrial adaptations and limit the beneficial effects of aerobic exercise (35). Such a negative relationship between β-blockers and exercise adaptations implies that drug prescription may be counterproductive to exercise recommendations.
Miura et al. (25) demonstrated that mitochondrial biogenesis may be regulated by β-AR signaling; however, the assessment was limited to changes in mRNA content for proteins that regulate mitochondrial biogenesis and was performed in mice. Changes in mRNA content may not lead to changes in mitochondrial protein content (28); therefore, it is necessary to use additional determinants, such as protein synthesis rates, to determine mitochondrial biogenesis. Another recent report using mice found that β2-AR agonist treatment increased mixed skeletal muscle and mitochondrial protein synthesis rates after 7 days of treatment (20). However, it is possible that there are interspecies differences in β-AR signaling between mice and humans.
Initially, to examine whether the findings of Miura et al. (25) were applicable to humans, we performed a 1-h infusion of the nonselective β-AR agonist isoproterenol (36). Contrary to the results reported by Miura et al., we did not increase mitochondrial biogenesis, as measured by mRNA signaling and incorporation of a stable isotope into mitochondrial proteins or whole body and skeletal muscle protein synthesis (36). Our dose of isoproterenol increased heart rate and blood pressure and was previously used to increase metabolic rate above resting values (5). The report of Koopman et al. (20) prompted us to examine further whether β-AR and changes in protein synthesis were unique to mice.
Our previous study was a gain-of-function design to evaluate if β-AR stimulation could induce mitochondrial biogenesis at rest (36). Here we conduct the loss-of-function experiment in which β-AR signaling is diminished during exercise. We hypothesized that blocking β1- and β2-ARs prior to and during a 1-h bout of cycling would decrease mitochondrial protein synthesis and mRNA content of genes related to mitochondrial biogenesis.
Ethics approval.
The Institutional Review Board of Colorado State University approved the protocol. Each volunteer was informed of the potential risks, and written consent was obtained prior to enrollment in the study, which followed the guidelines set forth by the Declaration of Helsinki.
Study overview.
Six healthy adult men were studied during 2 or 3 days separated by ≥3 wk. First, a subset of three subjects was used to confirm the efficacy of our β-AR blockade protocol (see below). All subjects then performed 1 h of stationary cycle ergometer exercise [60% of unmedicated peak O2 uptake (V̇o2peak)] during 1) β-AR blockade (intravenous propranolol) and 2) administration of saline (control). The propranolol trial was always performed before the saline trial to make any adjustments in workload necessary for the subject to complete 1 h of exercise. An identical workload was repeated on the saline control day. After exercise on both days, the subjects rested for 5 h during infusion of a stable isotope and subsequent skeletal muscle sampling to determine protein synthesis rates and mRNA content of skeletal muscle.
Subject characteristics.
The subjects (Table 1) were healthy, on the basis of a medical history questionnaire, and free of any cardiac abnormalities, as determined by an exercising 12-lead electrocardiogram. The three subjects (Table 1) who volunteered for verification of β-AR blockade were a subset of the entire group. Exclusion criteria included a resting heart rate <40 beats/min because of the risk of hypotension during β-AR blockade. V̇o2peak was determined during incremental stationary cycle ergometer exercise (Velotron, RacerMate, Seattle, WA) via indirect calorimetry (ParvoMedics, Sandy, UT). Body composition was determined using dual-energy X-ray absorptiometry (Lunar Discovery W, GE Medical Systems, Madison, WI).
Verification of β-AR blockade.
The efficacy of our β-AR blockade procedure was verified as previously described (26). Briefly, subjects arrived at the laboratory after an overnight fast and 24 h of abstention from vigorous exercise. Subjects were instrumented for the measurement of beat-by-beat heart rate (3-lead electrocardiogram) and blood pressure. An intravenous catheter was inserted into an antecubital vein, through which the nonselective β-AR agonist isoproterenol was administered in a continuous and incremental fashion. Dosing rates were increased every 5 min (9, 12, 15, 18, 21, and 24 ng·kg fat-free mass−1·min−1) until heart rate was increased 25 beats/min above resting levels. Next, the nonselective β-AR antagonist propranolol was intravenously infused, first as a priming dose (0.25 mg/kg at 1 mg/min) and then as a maintenance dose (0.006 mg·kg−1·min−1) throughout the 1 h of cycling at 60% V̇o2peak. After exercise, the infusion was stopped and the subject rested in bed. At 1 h postexercise, the isoproterenol dose that previously raised heart rate to 25 beats/min above the resting level was repeated to determine if heart rate and blood pressure increased. β-AR blockade was demonstrated by the absence of change in heart rate or blood pressure during β-AR stimulation with isoproterenol.
Study protocol.
The study protocol is shown in Fig. 1. Subjects refrained from physical activity on the day before each trial, and an evening snack was provided for consumption on the night before each trial. The subjects reported to the laboratory following an overnight fast, and intravenous catheters were inserted into a dorsal hand vein for arterialized blood sampling and an antecubital vein for infusions. Beat-by-beat electrocardiograms were continuously recorded throughout the study day, and blood pressure was monitored with an automated cuff. On the first trial, propranolol was administered as a priming dose (0.25 mg/kg at 1 mg/min) and then as a maintenance dose (0.006 mg·kg−1·min−1) using a precision pump (Harvard Apparatus, Holliston, MA) following previously utilized protocols (2–4, 26). Immediately after the priming dose, the subjects began cycling for 1 h at 60% V̇o2peak during infusion of the maintenance dose. Expired air was collected every 15 min to confirm steady-state O2 consumption (V̇o2), as determined by indirect calorimetry. On day 2 of the trial, the subjects received an isovolumetric dose of saline, and the same absolute workload was performed. Immediately after exercise on both days, the subjects consumed a liquid meal (57% carbohydrate, 28% fat, 15% protein; Ross Laboratories, Abbott Park, IL) to replace the calories burned during exercise, as determined by average V̇o2 and respiratory exchange ratio. The beverage also contained sufficient protein (>20 g) to maximally stimulate protein synthesis following exercise (27). A primed continuous infusion of [2H5]phenylalanine (2 μmol/kg prime, 0.05 μmol·kg−1·min−1 continuous infusion) was performed for 5 h following exercise. Skeletal muscle samples (∼100–150 mg) were collected with a Bergstrom needle under local anesthesia (1% lidocaine) at 1 and 5 h following exercise. Intermittent arterialized-venous blood samples were collected from a heated dorsal hand vein. [2H5]phenylalanine (Cambridge Isotope, Cambridge, MA) was prepared in sterile isotonic saline by a pharmacy (Medical Center of the Rockies, Loveland, CO) and tested for pyrogenicity. As an additional precaution against infection, all solutions were passed through a 0.2-μm filter before being infused intravenously.
Muscle protein synthesis.
Skeletal muscle protein synthesis rates were determined for subsarcolemmal mitochondrial, myofibrillar, and sarcoplasmic enriched fractions, as previously described (36). We used differential centrifugation to separate protein fractions that are not pure fractions but predominantly contain the proteins of interest. Briefly, ∼70 mg of muscle tissue were homogenized and centrifuged at 800 g to pellet a crude myofibrillar fraction. The supernatant was centrifuged at 9,000 g to separate the sarcoplasmic fraction (supernatant) and a mitochondrial fraction (pellet). The mitochondrial pellet was washed using a series of centrifugations, and the final mitochondrial, myofibrillar, and sarcoplasmic pellets were washed with 500 μl of 100% ethanol, centrifuged (1,000 g, 30 s, 4°C), and rinsed with water (repeated twice). Protein pellets were solubilized in 1 N NaOH (50°C, 15 min) and hydrolyzed into free amino acids (6 M HCl, 120°C, 24 h). Free amino acids were added to cation exchange columns and then derivatized to their t-butyldimethylsilyl derivatives, as previously described (12).
Samples were analyzed using gas chromatography-mass spectrometry (GC-MS; 7890A GC with 5975C MS, Agilent Technologies, Santa Clara, CA) in electron impact mode with selected ion monitoring at mass-to-charge ratios (m/z) of 234 (m + 0), 237 (m + 3), and 239 (m + 5), with m + 0 representing the parent ion. Samples were injected in duplicate, and the m + 0 peak area was kept within standardized limits to minimize differences in abundances due to concentration differences. Abundances were adjusted to standard curves that were derivatized from a single set of diluted standards and analyzed before and after each GC-MS analysis. Internal controls were included within each batch of samples to ensure consistency between GC-MS analyses. Tracer enrichments of muscle samples were determined as tracer-to-tracee ratio from a standard curve using the ratio of m + 5 to m + 3 (12). Plasma enrichments were determined from the ratio of m + 5 to m + 0. The fractional synthesis rate (FSR) was determined using the standard precursor product relationship: FSR = ΔEm/Ep, where ΔEm is change in enrichment of muscle proteins and EP is enrichment of the precursor. We adjusted for non-steady-state plasma tracer enrichments by calculating the precursor enrichment as the integral of the plasma phenylalanine enrichment over time (54). We used circulating plasma [2H5]phenylalanine as the precursor, as reported by others (8, 17, 18, 43), which may underestimate the true synthesis rates (see Ref. 44 for review). The low natural abundance of the infused tracer and washout period between separate trials allowed the assumption that background tracer enrichment is zero (45).
Real-time PCR.
Real-time PCR was used to determine changes in mRNA content for PGC-1α and downstream targets (Table 2). Total RNA was extracted from ∼10 mg of skeletal muscle using standard TRIzol methods (Invitrogen, Carlsbad, CA) and reverse-transcribed to cDNA, as previously described (36). Approximately 10 ng of cDNA were amplified using 20-μl reactions with a manufactured master mix (Thermo Fisher, Rockford, IL) in triplicate in clear 96-well plates using TaqMan probes and a real-time PCR system (model 7300, Applied Biosystems, Carlsbad, CA). PCR conditions were as follows: hot start (2 min at 50°C, 15 min at 95°C) followed by 40 cycles of denaturing and annealing (15 s at 95°C, 1 min at 60°C). The relative quantity of each gene target was normalized to a reference gene (β2-microglobulin), and fold changes were determined using the cycle threshold (2−ΔΔCt) method (31).
Heart rate, blood pressure, and V̇o2 were compared between trials using two-way (trial × time) ANOVA with repeated measures. Skeletal muscle FSR was compared using a one-way ANOVA. Changes in mRNA content were determined using two-way (trial × time) ANOVA with repeated measures on the ΔCt values (53) and are expressed as fold increase compared with the 1-h biopsy on the saline control day. Multiple comparisons were performed using Bonferroni's correction. Statistical significance was set at P = 0.05. Power analysis revealed that a sample size of six with standard deviation of 0.06, α at 0.05, and β ∼0.83 is able to detect a difference of 75% between treatments.
Verification of β-AR blockade.
β-AR stimulation with isoproterenol increased heart rate and blood pressure above resting values. Repeating the β-AR stimulation following β-AR blockade (propranolol infusion) and 1 h of cycling had no effect on heart rate or blood pressure, indicating the efficacy of our β-AR blockade procedure (Fig. 2).
Response during exercise.
Compared with the control condition (saline administration), exercising heart rate and blood pressure were decreased during β-AR blockade (Fig. 3, A and B). Absolute workload was identical between conditions, resulting in a V̇o2 that was not different between trials (P > 0.05; Fig. 3C). Respiratory exchange ratio was not different between trials (data not shown).
Plasma enrichment of [2H5]phenylalanine was not different between saline and propranolol trials (Fig. 4A). Skeletal muscle enrichments are reported in Table 3. Skeletal muscle FSR (Fig. 4B) of subsarcolemmal mitochondria was decreased with β-AR blockade (0.217 ± 0.076 and 0.135 ± 0.031%/h with saline and propranolol, respectively, P < 0.05). The FSR was not different between trials for myofibrillar (0.154 ± 0.061 and 0.102 ± 0.038%/h with saline and propranolol, respectively, P > 0.05) or sarcoplasmic (0.122 ± 0.034 and 0.0898 ± 0.025%/h with saline and propranolol, respectively, P > 0.05) proteins. The 95% confidence intervals for the difference with propranolol were −0.15 to −0.01%/h for mitochondrial proteins, −0.13 to 0.02%/h for myofibrillar proteins, and −0.10 to 0.04%/h for sarcoplasmic proteins.
Real-time PCR.
Real-time PCR revealed that PGC-1α mRNA was increased at 5 h compared with 1 h after cycling, with no differences between β-AR blockade and the control condition (P < 0.01 for time; Fig. 5A). Downstream mRNA signals for mitochondrial biogenesis were not different between time points or between conditions (Fig. 5, B–F).
Propranolol was infused during a 1-h bout of cycling to determine if nonselective β-AR blockade could alter skeletal muscle protein synthesis and mitochondrial biogenesis. Postexercise mitochondrial protein synthesis was greater during the saline than the propranolol trial. PGC-1α mRNA was increased at 5 h compared with 1 h postexercise but was not different between trials. Other downstream mRNA signals for mitochondrial biogenesis were not different between 1 and 5 h postexercise or between trials. These data imply that β-AR signaling is an important physiological determinant of postexercise mitochondrial biogenesis in adult humans.
Verification of β-AR blockade.
Nonselective β-AR blockade was confirmed in three subjects. The ability of isoproterenol to stimulate heart rate and blood pressure was determined before and after cycling with propranolol. The lack of cardiovascular stimulation at 1 h after cycling with propranolol indicates nonselective β-AR blockade during the acute period following cycling, which is when skeletal muscle protein synthesis rates and signals for mitochondrial biogenesis are elevated (14, 33, 51). Our biopsies were collected at 1 and 5 h after propranolol infusion, which is during the 3- to 6-h half-life of propranolol (34). We did not determine if there was complete blockade of all β-ARs on skeletal muscle. However, the demonstration of nonselective β-AR blockade combined with the half-life of propranolol indicates that β-AR signaling was decreased during our measurements of skeletal muscle protein synthesis and mRNA. As expected, the magnitude of increase in heart rate and blood pressure was decreased during cycling with β-AR blockade, suggesting that β-AR signaling was decreased in all subjects. Previous work has shown increased catecholamine signaling during exercise at 60% V̇o2peak (19). Thus our cycling workload was sufficient to increase β-AR signaling, and propranolol was able to blunt β-AR signaling.
Our study was designed as an integrative approach to assess mitochondrial biogenesis during recovery from aerobic exercise. PGC-1α and downstream mRNA content are increased by 2 h and have remained elevated for 8 h following exercise (32, 33). Additionally, skeletal muscle protein synthesis is increased in the hours following exercise and can remain elevated for 72 h (23, 51). We are confident that the timing of muscle sampling was adequate to capture any changes in mRNA content for mitochondrial biogenesis or changes in protein synthesis rates. We assessed mitochondrial biogenesis as changes in mRNA transcript content of select mitochondrial signaling proteins and mitochondrial protein synthesis. Previous studies of β-AR signaling and mitochondrial biogenesis have reported changes in mRNA content of mitochondrial signaling proteins (24, 25); however, such measurements do not take into account posttranscription or translational regulation. Our measurement of mitochondrial protein synthesis represents the cumulative regulation of signals for mitochondrial biogenesis.
The synthesis rate of skeletal muscle subsarcolemmal mitochondrial proteins was blunted in the several hours following cycling with β-AR blockade. Our results provide short-term evidence that nonselective β-AR blockade during cycling can impair mitochondrial adaptations and support long-term studies that showed decreased mitochondrial enzyme activity during training with β-blocker drugs (1). A blunted response of mitochondrial protein synthesis to individual training sessions may lead to decreased adaptations over the long term.
The regulation of β-AR signaling on mitochondrial protein synthesis appears to be mediated through β2-ARs. Previous reports showed that selective β2-AR agonists, but not β1-AR or α-AR agonists, increased PGC-1α mRNA at rest and selective β2-AR blockers abolished the postexercise increase of PGC-1α mRNA (25). Additionally, the blunted increase of mitochondrial enzymes during aerobic training was reported during treatment with nonselective β-AR blockers (e.g., atenolol or propranolol), but not selective β1-AR blockers (e.g., metoprolol or atenolol) (1, 16, 46). Others have reported no difference in V̇o2peak adaptations between selective and nonselective β-blocker treatments (42, 52). It is possible that different degrees of β-AR blockade due to various drug doses or binding preferences within selective or nonselective classes can explain variable training adaptations. It is important to note that studies with a greater focus on specific mitochondrial responses (e.g., mitochondrial enzyme activity) appear able to detect impairments with nonselective β-blockers that are not detected with whole body measures (e.g., V̇o2peak). Our approach was to measure acute changes in protein synthesis that may explain long-term differences in mitochondrial adaptations with nonselective β-AR blockade.
Myofibrillar protein synthesis was not different between the propranolol and saline trials. While β-AR antagonists are used to treat cardiovascular disease, β-AR agonists have been used for the treatment of muscle loss, such as with aging or disease (see Ref. 21 for review). In particular, selective β2-AR agonists stimulate skeletal muscle protein gains over several weeks in aging rats (41). We have shown that infusion of isoproterenol (a nonselective β-AR agonist) did not acutely stimulate skeletal muscle protein synthesis in humans (36). Recent data in mice showed that chronic treatment with a selective β2-AR agonist increased skeletal muscle protein synthesis of myofibrillar, mitochondrial, and sarcoplasmic fractions at 7 days, but not acutely or at 28 days of treatment (20). It is possible that our acute treatment with propranolol would result in significant decreases in myofibrillar and sarcoplasmic proteins if carried out for an extended period of time.
The primary outcome of our current study was mitochondrial protein synthesis, for which we demonstrate a difference in FSR. However, we were potentially underpowered to detect differences in the secondary outcomes of myofibrillar (−33%) and sarcoplasmic (−26%) FSR. The 95% confidence intervals for the difference between treatments are weighted toward a decrease in protein synthesis with propranolol treatment. It is possible that including additional subjects would reveal decreased protein synthesis in myofibrillar and sarcoplasmic fractions. Such a global regulation of protein synthesis rates is consistent with the increase in protein synthesis following selective β2-AR agonist treatment shown by Koopman et al. (20) and could be mediated through β-adrenergic regulation of the mammalian target of rapamycin pathway (11).
Our values of skeletal muscle FSR were determined following exercise in the fed state. The fractional synthesis of skeletal muscle mitochondria is higher than that of myofibrillar proteins at rest (36, 38) or following exercise (13) and is increased with intravenous amino acid infusion (6). Our measured mitochondrial protein synthesis rates are slightly higher than those reported by others because of the experimental conditions (i.e., postexercise and feeding) and use of a zero-background labeling to calculate rates (45). The high rate of mitochondrial protein synthesis reported here and by others indicates a high rate of mitochondrial protein turnover that can be stimulated by exercise (51) and nutrition (6).
We included postexercise nutritional supplementation to avoid a negative energy balance following exercise and to increase external validity. Because the subjects reported to the laboratory after an overnight fast and given the length of the protocol, the subjects would have been fasted for ∼18 h. It is known that negative energy balance decreases nitrogen balance (48). Furthermore, a prolonged fast and exercise would likely result in AMP-activated protein kinase activation and, therefore, PGC-1α activation. Even though our study did not include an exercise + fasting group, our results indicate that β-blockade can impair mitochondrial protein synthesis in the fed state following exercise.
Exercise V̇o2.
The 1-h cycling workload was adjusted to maintain 60% V̇o2peak during the first trial (propranolol), and the same absolute workload was repeated for the second trial (saline). The respiratory exchange ratios were not different between trials, indicating that substrate utilization was similar during the trials. Others have reported similar indirect calorimetry results during cycling exercise following propranolol treatment (50). Postexercise signals for mitochondrial biogenesis can be influenced by dietary habits that alter glycogen content (32) but are not related to circulating glucose or free fatty acid availability (40). Thus the potential influence of β-AR signaling on mitochondrial biogenesis is likely to be within skeletal muscle and not due to changes in circulating substrate metabolism during exercise.
Contrary to our hypothesis and previous studies with mice (25), mRNA content of markers of mitochondrial biogenesis was not different between exercise with propranolol and with saline. PGC-1α mRNA was increased in both trials at 5 h compared with 1 h postexercise, which is similar to previous reports of human exercise trials (33). To minimize subject burden, we did not collect a muscle sample before exercise and cannot determine the change in mRNA content from resting values. However, our biopsies at 1 and 5 h after exercise provide two times points to compare between propranolol and saline trials and are within the time frame that PGC-1α and downstream mRNA are elevated (32).
Exercise-induced changes of PGC-1α mRNA content are dependent on exercise intensity (47) and appear to be regulated by the relative intensity, rather than the absolute intensity (29). Despite an acute blunting of heart rate response and increased perceived effort with propranolol, our exercise bouts were performed at the same relative intensities. Support for the same relative exercise intensity was that subjects were tested within 4 wk with no changes in exercise patterns in the intervening period, and equal respiratory exchange ratio, which scales to relative exercise intensity (7), between trials.
Perspectives and Significance
We previously reported that acute nonselective β-AR stimulation during rest did not increase mitochondrial protein synthesis or mRNA markers of mitochondrial biogenesis. Our current investigation indicates that β-AR signaling can modulate exercise-induced mitochondrial protein synthesis. The postexercise recovery period is a critical time for skeletal muscle remodeling, and decreasing sympathetic signals to skeletal muscle may attenuate adaptations. Further work should consider if chronic intake of β-AR blockers would decrease mitochondrial protein synthesis and contribute to impaired mitochondrial adaptations during exercise training.
We conclude that nonselective β-AR blockade can blunt the postexercise increase in mitochondrial protein synthesis rates. Nonselective β-AR blockade did not alter the mRNA content for signals of mitochondrial biogenesis, suggesting that the impaired protein synthesis response is independent of changes to mRNA transcripts. Our results indicate that nonselective β-AR antagonists can impair mitochondrial adaptations to acute aerobic exercise and may lead to decreased training adaptations.
Funding was provided in part by the Gatorade Sport Science Institute (to M. M. Robinson). B. F. Miller is supported by National Institute on Aging Grant 1-K01-AG-031829-01A1.
No conflicts of interest, financial or otherwise, are declared by the authors.
We thank the subjects for their enthusiasm to complete the exercise protocols. We also thank Gary Luckasen and Dennis Larson for medical oversight and Joshua Drake for study assistance.
- Copyright © 2011 the American Physiological Society


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Well I guess this took longer than anyone expected, but I guess BEA Systems have held off long enough for Oracle to purchase them for a decent price ( over 19$ a share ) or over inflated price.
There are some more details on the news wire (1) (2) and from Oracle.
Unless BEA Systems have oil rights or an endless supply of gold bullion stored at their head quarters, it is really unclear to me why Oracle would want to pay so much for the company. They do have a decent user base, but this is dwindling and there is so much technology overlap with what Oracle sell today.
For Oracle, I am not sure what company comes next... Larry, how about SAP..


text:
News & Events
Irish Citizens Decide Seminar
A half-day seminar, 'Irish Citizens Decide: A Review of the Irish Convention on the Constitution' will review the work, operation and outcomes of Ireland's Convention on the Constitution, which met from late 2012 to early 2014. The seminar will take place at Newman House on Friday, 13th November 2015. Read More
News & Events
IBIS Annual Conference 2015
The IBIS Annual Conference for 2015, 'Women in Leadership North and South', took place at the European Parliament Office, Molesworth Street, Dublin on Monday, 6th July. Minister for Justice and Equality, Frances Fitzgerald, TD, gave the keynote address. Read More
News & Events
The Flags Protest and its Aftermath
An IBIS lecture was held on Tuesday, 14th April on the topic 'The Flags Protest and its Aftermath: Crisis Looming or Crisis Resolved?' The speaker was Dr James Wilson. Read More
Institute for British-Irish Studies (IBIS),
School of Politics & International Relations,
University College Dublin, Belfield, Dublin 4
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Email: email@example.com
Latest from the IBIS Blog: 'Cohesion, Sharing and Integration' policy backward step for community relations. VIEW BLOG
15 Years On
Reflecting on the successes and failures of the 15 years since the Belfast/Good Friday Agreement
IBIS is part of a group which has come together to discuss how we might use 2013 to reflect on the successes and failures of the 15 years since the Belfast/Good Friday Agreement. This conversation will take place largely online, leading to a culminating event sometime in autumn 2013. To find out more about the initiative click here
IBIS Research Project
Addressing cultural legacies of conflict: Towards an inter-cultural and inter-dimensional dialogue on women, peace and security
Dr Melanie Hoewer is Principal Investigator for this research project, which began in October 2012 and looks at lessons learned from 1325 processes, in particular from the Irish cross-learning model and at the impact of international organisations in promoting UNSCR 1325, e.g. through the Irish OSCE Chairmanship.
For a full description of the project, click here


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The J.O. Combs Unified School District is excited to open our doors to the community, non-profit groups and businesses of all sizes. You can rent a field, gymnasium, classroom, multi-purpose room, or cafeteria. Our District has seven schools with a variety of facilities to meet your organization's needs. We look forward to working with you.
Submit all facility rental applications to the individual school site where you are requesting rental.
Our Schools


text:
One dead as Australian heatwave breaks records
Melbourne is sweating amid temperatures that have halted tennis matches, sparked bushfires and melted at least one Lindt chocolate in minutes.
A Melbourne man demonstrated the extreme temperatures by videoing chocolate melting on the pavement in under three minutes.
Melbourne is on course to sweat through a fourth day above 41 degrees today, the first time the city has recorded such prolonged heat in records dating back to the mid-1850s.
At the Australian Open tennis tournament, 970 spectators were treated for heat-related illnesses.
The city copped 43.9 degrees on Thursday, with heat so extreme the Australian Open tennis was halted for some matches for several hours.
More delays may happen today with the bureau tipping a high of 44 degrees for the city.
A new report on Australian cities has estimated that Melbourne had the highest annual average number of heat-related deaths, with about 200 a year. By comparison, the state's (Victoria) 2013 road toll was 242.
The number of heat-related deaths in Melbourne is expected to more than double by 2030.
During 2009's January heatwave, there were 374 more deaths across Victoria than would be expected for the week when comparing data over five years. The greatest number of deaths occurred in those aged 75 or older.
Adelaide's 44.2 degrees yesterday was the fourth in a row above 42 and a fifth is likely today. To the city's south-east, Mount Gambier broke its record maximum on Thursday.
The 44.1-degree reading beat the previous record by half a degree, said Blair Trewin, senior climatologist with the bureau.
Canberra, Melbourne and Adelaide each topped 40 degrees for a second day in a row for the first time in more than 70 years of records for all three cities, said Brett Dutschke, senior meteorologist at Weatherzone.
Both Adelaide's and Melbourne's maximums for today hinge on the timing of a cool change, with its arrival potentially adding or reducing the maximums by a couple of degrees, Dr Trewin said.
Residents in some Grampians and northern Grampians towns east of Melbourne have been told it is too late to leave their homes as an out-of-control blaze burns in a westerly direction.
Police confirmed a person has been killed in the Grampians bushfire.
The body was found at Roses Gap, 44km south-east of Horsham in the northern Grampians.
"The death is being treated as occuring as a result of the fires," Victoria Police said.
The Country Fire Authority issued an emergency warning just after 6am today for the towns of Wartook, Brimpaen, Heathvale and Laharum, urging all residents in the area to take shelter inside immediately and not leave their homes.
Winds are pushing the blaze in a westerly direction towards Wartook. It crossed the Northern Grampians Road between Heathvale and Wartook just after 6am on Friday. The Wartook Valley, between Flagstaff Track and Roses Gap, are affected by the blaze.
The towns of Glenisla, Cherrypool and Glenisla Crossing have also been warned about a 11,300 hectare fire heading in a south-westerly direction from Pohlners Road.
The CFA has advised communities to "watch and act" according to conditions.
Further north, fire authorities in New South Wales are gearing up for a tough few days, with the huge heatwave searing most of south-eastern Australia showing no real sign of abating until next week.
"We're still talking about Saturday" as the day of most risk, a spokesman for the Rural Fire Service said. "There are plenty of resources ready to go."
Total fire bans are in place for four regions in the state's south-east, with fire-danger ratings very high or high for most of NSW today.
No real relief was expected around most of Sydney until Monday.
The city's eastern suburbs and north shore will be areas of refuge from the heat, with maximums in the downtown and Terrey Hills regions reaching 29 degrees, the Bureau of Meteorology said.
Both areas can expect 31 on Saturday before a late cool change arrives.
The change is likely to be a modest one, with the city still expecting 27 on Sunday before Monday's maximum comes in at about 24.
There is little chance of significant rain at this point from the cooler conditions, said senior bureau forecaster, Peter Gajewski.
Before then, towns such as Penrith and Richmond can expect 39 degrees on Friday and similar conditions on Saturday.
Saturday will potentially be the most dangerous for fire, as stronger winds are forecast along with the cool change.
Sydney Morning Herald


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2012 NISSAN Versa SV Sedan 4D
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2012 NISSAN Versa SV Sedan 4D
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text:
Irma Evacuation Part 1
5 days ago
Congrats to you. I'm told by reliable sources this summer will not be as hot as last summer. I got that from the Channel 9 weatherman.Best of luck with this one.
3:1? Makes it almost even.... (sorry, I couldn't resist!)
I am really delighted for you. And I hope your AC has an Industrial High setting. If not, you can come and stay with us for a while....
I totally thought I commented on this! CONGRATULATIONS! I'm so so glad G will have a sister...she can tell her all about Maws :)


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Charles Johnson of Endicott - Part 2
a $10 bill, one $2 and one $1 bill, along with some currency. Tbe bills I saved by folding very tightly and placing them between my teeth next to my cheeks . When it was my turn to be searched I was leisurely eating Johnnie cake. I afterward had occasion to be thankful that I had saved this money.
Our three weeks being up, we were taken to Petersburgh where we lived under an old tobacco factory for a period of two weeks. At this camp everything was taken from us, therefore I was unable to keep a diary, which I would like to have done.
The next move was from Petersburgh to Salisbury, North Carolina. This prison or stockade was built on a site which consisted of about seven or eight acres. The stockade was built of timber about one foot thick by sixteen feet long. It was built about four feet under ground and twelve feet above, and was continually guarded by rebels, who were looking down upon us all the time. For the first few weeks of our stay here, we had but very little shelter. And the nights by this time, were becoming very cold and rainy most of the time. We would sleep in piles, about a hundred in a pile. Some would sleep under for a while, then those on top of the pile would root down so as to take their turn at getting warm. This went on for some time, until they took mercy and gave us a few of their condemned tents. Although these were not very good, and far from enough, still we considered them better than nothing. We made the best of them by sleeping with our feet to the center of the tent and our heads to the outside, in this way we were enabled to get a larger number in the tents. Those of us who were less fortunate dug holes underground, where we spent the winter. It was not long, however, before we had plenty of room in our tents, our comrades were dying off by the hundreds.
When ration time came we were put into divisions, about 1,200 to a division. These twelve divisions, divided into companies of 100 men and a sergeant. The sergeant was detailed to see about drawing our rations. My company was quite aways from the cook house and in that way many times distance served a purpose and other times it did not.
Each day the rebel lieutenant would come through to count us for that day's rations. He would start in at one end of the line and after he had counted us a great many would slip around and be counted again; in this way we would receive two rations They were aware of this, but somehow or other failed to catch the guilty parties.
The sergeant had two men detailed to help him distribute the rations, which were passed out once a day. Usually this was only a very small piece of Johnny cake and sometimes soup, made of peas or beans, the soup being carried to us in a barrel with holes through each side where a stick was placed through and in this way carried by two men.
They would deal out to the men near the cook house first, and work on down the line. In this instance distance did us more harm than good. Many times the rations would run out before the lines had received their rations, so we would have to go without that day and many others. Some would go for three days without a thing to eat, but I always managed to buy something with the money I had saved. With the money I could always find something.
When we first went into camp there was no way of getting water, except from a creek about 15 rods away. Two men guarded, were allowed to take a barrel and get water from the creek. I always tried to get out on that detail, because I could buy hoe cake from the peddlars for a confederate dollar. When we got back to our camp, I would sell and double the value. This I would save, so I could buy again.
There was one time, though, I bought a meat pie, which was almost my undoing. I always thought it was made of horse meat. I surely was never able to finish that pie, the piece I ate made me deathly sick.
Inside this stockade in which we were was an old tobacco factory about three stories high. Around this were log cabins where darkies lived. This old tobacco factory was used as a hospital, and here I was taken. It was not long before I became acquainted with the man who had charge of the place. Through him I used to receive a little extra to eat. He would give me small chunks of something I thought was opium. This I would make into pills.
The water and food of this hospital caused practically all who ever reached there to die of chronic diarrhea.
After we had been in the prison for some time some of our men dug a hole about 16 feet deep. This would allow the surface water to run in and we got our water in this way. To get the water l took a regular army cup and put a bail in it. The bail being made from a strip of leather cut from an old boot leg. Each morning, early, before the water was riled, l would go and draw water. The fevered patients would ask for water, and if l could get a chew of tobacco for a drink I would exchange the tobacco for rations as I never used the tobacco. The guards were old union farmers and when no one was around I would get tobacco from them, too.
When our men died during the night the other men in the tent would take anything which they could wear from the dead men. They did this because they were nearly naked themselves and suffering from cold and exposure. The dead were taken to the deadhouse, being thrown any way into the wagon. A squad of men were taken out to a rise of ground where they dug a trench and put the dead bodies in three deep and covered them. . When we got out in the spring, the place looked like a plowed field. I shall never forget the ghastly sight. of men's hands and heads dragging over the wheels of the wagon as they were being conveyed from the deadhouse to their graves.
Source: E.- J. WORKERS' REVIEW (August 20, 1919), page 21. STORY OF A CIVIL WAR VETERAN
By A. B.
Submitted by Debbie Barnes
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text:
Competitiveness – A Comparison of China and Mexico
Latin American countries have lost competitiveness in world markets in comparison to China over the last two decades. The main purpose of this study is to examine the causes of this development. To this end an augmented Ricardian model is estimated using panel data. The explanatory variables considered are productivity, unit labor costs, unit values, trade costs, price levels (in PPP), and real exchange rates in relative terms. Due to data restrictions, China's relative exports (to the US, Argentina, Japan, Korea, UK, Germany, and Spain) will be compared to Mexico's exports for a number of sectors over a period of eleven years. Panel and pooled estimation techniques (SUR-estimation, panel Feasible Generalized Least Squares (panel/pooled FGLS)) will be utilized to better control for country-specific effects (differences between American, Argentinian, Japanese, Korean, German, British, and Spanish markets), cross-section specific (sector-specific) effects, and correlation over time.
If you experience problems downloading a file, check if you have the proper application to view it first. In case of further problems read the IDEAS help page. Note that these files are not on the IDEAS site. Please be patient as the files may be large.
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References listed on IDEAS
Please report citation or reference errors to , or , if you are the registered author of the cited work, log in to your RePEc Author Service profile, click on "citations" and make appropriate adjustments.:
- Ehsan U. Choudhri & Lawrence L. Schembri, 2002. "Productivity performance and international competitiveness: an old test reconsidered," Canadian Journal of Economics, Canadian Economics Association, vol. 35(2), pages 341-362, May.
- Alan V. Deardorff, 2014.
"Local comparative advantage: Trade costs and the pattern of trade,"
International Journal of Economic Theory,
The International Society for Economic Theory, vol. 10(1), pages 9-35, 03.
- Alan V Deardorff, 2004. "Local Comparative Advantage: Trade Costs and the Pattern of Trade," Working Papers 500, Research Seminar in International Economics, University of Michigan.
- Alejandro Cuñat & Marco Maffezzoli, 2007. "Can Comparative Advantage Explain the Growth of us Trade?," Economic Journal, Royal Economic Society, vol. 117(520), pages 583-602, 04.
- Alejandro Cunat & Marco Maffezzoli, 2003. "Can Comparative Advantage Explain the Growth of US Trade?," Working Papers 241, IGIER (Innocenzo Gasparini Institute for Economic Research), Bocconi University.
- Alejandro Cuñat & Marco Maffezzoli, 2005. "Can Comparative Advantage Explain the Growth of US Trade?," CEP Discussion Papers dp0669, Centre for Economic Performance, LSE.
- Alejandro Cunat & Marco Maffezzoli, 2005. "Can comparative advantage explain the growth of US trade?," LSE Research Online Documents on Economics 19919, London School of Economics and Political Science, LSE Library.
- Cuñat, Alejandro & Maffezzoli, Marco, 2005. "Can Comparative Advantage Explain the Growth of US Trade?," CEPR Discussion Papers 5348, C.E.P.R. Discussion Papers.
- Lücke, Matthias & Rothert, Jacek, 2006. "Central Asia's comparative advantage in international trade," Kiel Economic Policy Papers 6, Kiel Institute for the World Economy (IfW).
- Busse, Matthias, 2003. "Tariffs, Transport Costs and the WTO Doha Round: The Case of Developing Countries," Estey Centre Journal of International Law and Trade Policy, Estey Centre for Law and Economics in International Trade, vol. 4(1). Full references (including those not matched with items on IDEAS)
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Jessica Lange explains how to go crazy without losing your mind
Typically when a lauded actress holes up in the Hamptons with 20 cats and a fat suit, questions abound over her "transformation" for such a bizarre role. But Jessica Lange - who's already walked a mile in the paper slippers of Blanche DuBois and Frances Farmer - is far from typical.
"The hardest thing for me, as an actor, is to play a part that is on the straight and narrow. Just, you know - normal. I hate to say it, but it's true," says Lange, who definitely managed to avoid that problem when she added Edith "Big Edie" Bouvier Beale to her resume with Grey Gardens.
It could be the actress's extensive experience with slightly off-kilter ladies, but Lange is particularly forgiving of eccentricity. "I never thought of Francis as crazy. I never thought Blanche DuBois was crazy," she says. "They're survivors. They're trying to figure out how you get from A, to B, to C, and along the way, there are all these forces that are coming into play. With Big Edie, I always found her incredibly intelligent, deeply sensitive, with an extraordinary sense of humor."
Lange also rejects the idea that Big Edie sequestered her daughter at home, stunting the girl's growth into a well-adjusted young woman. "They were complicit in this life together," she says. "One thing I did appreciate about the film is that we were not trying to explain it in hard and fast terms. Because I don't think you can. There were such levels of complexity and this strange symbiosis. Really what you've got is a love story."


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All of the stories about the 7,643-yard "monster" that is Torrey Pines got me to thinking: What is the toughest golf course in the world? Is it Oakmont? Carnoustie? Will it be Torrey Pines after the completion of the United States Open? I need to do more research (read: play more courses) to get a larger sample, but I do have a couple of candidates right off the bat.
Six years ago, I took 123 unsightly strokes around Bethpage Black in advance of the 2002 United States Open. (What do you want from me? My last name is Hack.) Then, in 2006, I was (un)lucky enough to play Winged Foot before that year's Open, a round so brutal that I stopped keeping score. When I look back at both experiences, though, I actually enjoyed the Black, even as I failed to record a single par. Winged Foot left me near tears. That was no fun. (Hey, Phil, I was such an idiot, too). Winged Foot is the toughest course in my little world.
What is the toughest course in yours? Tell us in the space below. Looking for more candidates? See Golf Magazine's lists of the Top 100 Courses in the U.S. and the Top 100 in the World.


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June 25 2011
Cantlay still leads as third round starts
By Brian Wacker, PGATOUR.COM
CROMWELL, Conn. -- The third round is under way at TPC River Highlands, where players are being sent off both tees in threesomes. The final group of the day -- Patrick Cantlay, Johnson Wagner and Nick Watney -- will tee off at 1:45 p.m. ET.
For Cantlay, the 19-year-old amateur who just finished his freshman year at UCLA, it will obviously be the first time he's carried a lead into the weekend on the PGA TOUR in what is just his second start out here.
He turned plenty of heads with his second-round 60 -- a course record -- and now will try to build on that. Given the soft conditions, he'll need to.
"He knows how to look at a golf hole and what an architect is trying to do," Cantlay's college coach, Derek Freeman said. "It's amazing because I've never coached a kid that has that ability at that age."
Freeman, who is here this week, talked to Cantlay Friday night after the round and told him to enjoy what was a special day and to keep things simple.
Cantlay obliged, having a low key dinner last night before calling it a night.


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October 10, 2001, 06:30 PM
I am looking for a full auto machine pistol, or any good deal on a assault type weapon. Right now I am looking for a m-11 9mm or .45 acp mac-10.
thanks chris:cool:
thanks chris:cool:
View Full Version : Want a class 3 weapon in Ga.