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The Federal Reserve's December announcement that it will begin decreasing the amount of securities it buys in its quantitative easing program this year points to a continued increase in long-term interest rates. But with economists expecting economic growth to be as good as last year's or better, the higher rates aren't expected to discourage companies from issuing debt.
At the end of 2013, the 10-year Treasury bond rate stood at 3.04 percent, up from 1.78 percent at the end of 2012. Milton Ezrati, senior economist and market strategist at Lord Abbett, an asset management company in Jersey City, N.J., expects long-term Treasuries to rise another 100 basis points this year.
The Fed is likely to be "less aggressive" about tapering its purchases given the weaker-than-expected December employment report, Ezrati said. "That means rates will probably do more of their rising later rather than earlier. But we do think rates are going to move up."
Short-term rates are still extremely low, though, with the Treasury three-month bill at 0.05 percent, and they're are likely to remain low given that the Fed isn't expected to raise the fed funds rate until 2015. "That's a very important distinction," Ezrati said. "The short end is the area where the economy is more sensitive."
Corporate finance teams have been making the most of lower rates while they can. According to a survey of 424 finance executives conducted by the Association for Financial Professionals (AFP), 55 percent said their company took at least one action in 2013 to take advantage of the low-interest-rate environment. Fifty-five percent of those who took action refinanced long-term debt, while 52 percent issued long-term debt, 36 percent renewed a revolving credit facility early, and 28 percent locked in rates with swaps or options.
"Everyone knew that at some point rates had to go up," said Kevin Roth, the AFP's director of research. "As a result, financial professionals began to lock in these low rates."
The AFP survey was conducted in early December, before the Fed announced its tapering, and two-thirds of the respondents said they expected little change in borrowing costs this year. Almost half (49 percent) said their company plans to take some action this year based on their expectations for interest rates.
Again the most popular plan, cited by 42 percent of those whose company plans to take some action, was to refinance long-term debt, while 37 percent said they will reduce their outstanding debt. And many cited plans to issue long-term debt for various purposes: 36 percent to finance an increase in capital expenditures, 18 percent to finance mergers and acquisitions (M&A), and 9 percent to purchase stock.
The talk of debt issuance comes on the heels of two years in which corporates increased their issuance of bonds. Last year, U.S. companies issued $1.07 trillion in new debt, up from $1.04 trillion in 2012, according to Standard & Poor's Corp.
Diane Vazza, director of global fixed-income research at Standard & Poor's, expects issuance to be flat to a little higher this year. "You're coming off a very high level," Vazza said. "Even flat is healthy issuance."
She estimated that two-thirds of the issuance over the last two years was refinancing and said some companies will still be refinancing this year, particularly speculative-grade companies. S&P data show U.S. companies still have significant amounts of debt maturing over the next few years: $1.4 trillion in 2015 and $1.2 trillion in both 2016 and 2017.
"We think what's going to take up the slack as we see less refinancing in 2014 is investment in business," Vazza said.
That's all against the background of a gradual increase in long-term rates. "Not a pop," Vazza noted. "That would choke issuance."
John Cryan, a partner at Fortuna Advisors, said that while finance theory suggests rising interest rates discourage businesses from investing and borrowing, companies consider the state of the economy as well as rates. "If interest rates are rising because of positive signs, and not because of increased risk and fear, you'll probably see higher investment than would be assumed using a pure quantitative model," Cryan said.
"I'm a bit optimistic on this," he added. "I think rising interest rates in the near term will increase investment, not decrease investment, even though it will cost slightly more."
Another aspect of rising rates is the possible effect on customers, Roth said: "Companies obviously need to be concerned about the demand for their products. Especially for companies that sell directly to consumers, they have to see how demand for their products will be affected if rates go higher."
Two sectors that are sensitive to interest rates—housing and autos—were "fairly resilient" last year, Roth said. "One of the things that will be interesting to see is whether demand for goods and services will continue to grow even as the cost of borrowing increases."


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Three of the four Florida juniors who declared for the NFL Draft made the right call, ESPN draft analyst Mel Kiper Jr. said Wednesday.
Three of the four Florida juniors who declared for the NFL Draft made the right call, ESPN draft analyst Mel Kiper Jr. said Wednesday. The one who didn't, he said, is outside linebacker Jelani Jenkins.
"Jenkins coming out. … I would have stayed another year and put together a full healthy year that shows what he's capable of," Kiper said on an ESPN draft teleconference Wednesday. "He could have been a second-round pick (if he'd done that this season). Now, it's the third or even fifth round (for Jenkins).
"With the injuries, I'm surprised he came out. (By coming back) he could have put together a great year and stayed at 100 percent and gone through the process. He could have upgraded over where he is. But if you're a third- or fourth-round pick and can play, you're going to be fine.
"He could have gone a little higher by going back (to UF for his senior season). He is the only (UF junior coming out) I would say I can find fault with."
Kiper said he has no problem with the other three Gators leaving early — defensive tackle Sharrif Floyd, safety Matt Elam and tight end Jordan Reed.
Kiper projects Floyd to go in the first round (to San Francisco with the 30th pick), Elam to go in the early to mid-second round and Reed to be taken possibly as high as the second round.
"I like Floyd. I like his athletic ability, and he can help a lot of teams," Kiper said. "He had a heck of a year. He's solidly in the first round. I have no problem with that decision.
"Elam could be the second safety off the board. I have no problem with that decision. Jordan Reed can catch the football and will have nice workouts. I have no issue with that."
Some other draft analysts are projecting Elam to go late in the first round.
"I thought about him as a borderline (first-round pick)," Kiper said. "I see more as going in the second round, probably more early in the second round."
Kiper said Reed, UF's leading receiver this past season, could go as high as the second round, but probably will hear his named called in the third round.
"I like him. He's one of those athletic tight ends that are in vogue in the league," Kiper said. "He can stretch the deep middle of the field.
"That tight end group is not all that strong. It's not a great year (for tight ends). The third tight end (slot) is wide open. There's a chance he'll be right there. He could be the third tight end taken or the fifth.
"Either way, he's in the third-round discussion, maybe the second if he runs a great 40."
At least three UF seniors — tailback Mike Gillislee, middle linebacker Jon Bostic and offensive tackle Xavier Nixon — also are projected to be taken in the draft. Other possibilities are safety Josh Evans and place-kicker Caleb Sturgis.
Contact Robbie Andreu at 352-374-5022 or email@example.com. Also check out Andreu's blog at Gatorsports.com.

subset: commoncrawl


text:
Ravenswood Manor Park
4626 N. Manor Ave. Chicago, Illinois 60625
(773) 478-3499
Fee: Free
Age Range: Any
The Horner Park Advisory Council is pleased to sponsor their 19th season producing free live entertainment under the pergola in Ravenswood Manor Park. We've got some great local talent lined up this year, including old friends and new finds.
Saturday, June 29, 5 pm: The Comedy of Errors, Spectralia Theaters Bard in the Parks


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- Are you an administrator at this school?
- Keep your school's information up-to-date.
- Are you a parent or student at this school?
- Encourage school administrators to update your school's profile.
Find out about this school's hours, enrollment, classes, sports, and more.
See a snapshot of student diversity at this school.
Learn more about this school's location and contact information, and explore other nearby schools.
St. John The Baptist
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4 out of 5 stars


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Galewood Park
5729 W. Bloomingdale Ave. Chicago, Illinois 60639
(312) 746-5089
Fee: Free
Age Range: Any
ParaNormanWhen a small town comes under siege by zombies, who can it call? Only misunderstood local boy Norman, who is able to speak with the dead. In addition to the zombies, he'll have to take on ghosts, witches and, worst of all, grown-ups, to save his town from a centuries-old curse. But this young ghoul whisperer may find his paranormal activities pushed to their otherworldly limits.
Rated PG | 92 minutes | 2012
Presented by Charter One, with additional support from RCN.


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Any SOLID arguments against evolution?
Discussion of everything related to the Theory of Evolution.
Moderator: BioTeam
Re: Any SOLID arguments against evolution?
Alex, I am aiming at anyone personally. I am not saying you or anyone is close-minded. I apologize to you if that is how you took it. I am merely stating what I assumed was self evident--that in general we all, including me, have presuppositions by which we form opinions, and beliefs. The most basic of these is that we exist, and then we can go from there. I am not escaping your request to present citations. I can do this, but it is much more time consuming. I have done this before in my small experience of this debate on other forums, and been accused of copying and posting, without really having any background knowledge.
Can I say that the opening post on this goes as follows:"Can anyone come up with any solid, completely unexplainable arguments that defy evolution with our current scientific understanding of the issue.
Note: Please do not run at me and bash me with your bibles."
Lets reverse this to see how this sounds. Can anyone come up with any solid...arguments that defy THE SCRIPTURES with our (that being knowledgeable experienced Christians) understanding on the issue? Please do not bash me with your science text books.
Would this question not be considered purely rhetorical from someone who was convinced of evolution? Yet you can hear the bias of someone is completely convinced that the scripture is trustworthy. I'm not saying that there is something wrong with this bias, but that it is self evident--no one has to explain it or make citations from pschycologists or philosophers to see it.
I asked questions in response to provoke thought, because I have seen that to simply say that this was all created by the word of God is to evoke the response that I suffer from blind faith. I want to show people that some Christians have actually thought this subject thoroughly. And that evolutionists, many who so confidently affirm that to believe anything other than on origins is ludicrous, have plenty of their own blind faith.
Alright, I see your point that one can read a degree of inherent bias into the original post. That said, I'm not so sure it was necessarily out of line. "The Bible says so" hardly counts as scientific evidence, especially when one could just as easily cite chapter and verse from the Qu'ran or Confuscious (misspelled?) as well as plenty of other religious scriptures.
My point is that the validity of any scientific theory is ultimately dependent on evidence. I am sorry if your efforts to present evidence have not met with success, but since you bring up the question of background knowledge, I must confess that I am curious as to the extent of your scientific education. Do you hold a science degree or are you simply an armchair enthusiast? If you're curious about my own credentials, I'm an undergrad student pursuing a double major in biology and chemistry. I plan to go to grad school and work on a doctorate.
Most of the questions you presented have nothing to do with biology. The big bang, for example, is a matter of astrophysics, just as the development of rock strata is a concern for geologists. I'm not saying that I wouldn't mind discussing those topics with you; I'm just skeptical if this is the right place to do so, since this is a biology forum. However, that's probably up to the mods to decide. Still, given that evolution is a biological phenomenon, it might be best to base your arguments on biological facts and theories.
I must admit I am a bit concerned by the way you seem to equate evolution with atheism, especially since many of the greatest Christian theologians (Augustine of Hippo comes to mind) have been strong supporters of evolutionary thought. Even then, evolution is at its heart a scientific concept, and thus should be kept independent from any religious affiliation. Maybe I'm reading too much into your words here, but that's just the impression I'm getting, and feel free to correct me if I've misinterpreted you.
I am also a bit concerned by this:
Again, I might be misunderstanding you, which is why I'm going to ask for a clarification before I go off half-cocked. Are you implying, or do you believe, that all "knowledgeable, experienced Christians" are going to believe and think exactly as you do?
Generally speaking, the more people talk about "being saved," the further away they actually are from true salvation.
#2 Total Post Count
Re: Any SOLID arguments against evolution?
My degree is in theology, but before that years of personal study of the Bible, so I have a good working knowledge of what is in it--a pretty good knowledge of prophecy also. I was a missionary for a number of years, but now do secular work. In science my level is laymen/student--but not according to what I can remember from high school--I am currently studying chemistry, but I also read much on the internet. I kind of take turns, so I don't get bored. One day I may study the geologic time scale, another day I may read my chemistry book. Another day I may study about the electron transport chain or another area of microbiology. Then I read alot of articles on the answers in genesis website. So yes you would call me a science enthusiast. I have 3 kids and a grandchild in the house at this point so I couldn't make a switch in careers at this point. I wish now I had majored in science. I could see myself being a high school science teacher (my wife is a 4th grade teacher).
Let me ask you a question, Alex. What do you with Adam and Eve and original sin since you are catholic. How do you blend evolution in with that?
Theology? Interesting; another favorite subject of mine. I'm sort of in the same boat as you are with being an enthusiast in a great many topics, and like you I don't always find the time to indulge my academic curiosities as much as I'd prefer. Although my excuse isn't a family; it's just the simple curse of being a college student! But despite my lack of spare time, I have a very strong interest in theology, and probably would've majored it if that hadn't have meant going to a private university, which I can't afford. Plus there's not much prospect for employment in theology unless I would go into the clergy, which I'm not exactly cut out for. I am currently reading a book called Unabridged Christianity which examines the beliefs of the early Christians and compares it to doctrines today; it's written by Fr. Mario Pamero, who's pastor of a Catholic church in Lafayette, Louisiana; about two hour's drive from where I am in the Houma-Thibodaux area. Anyway thanks for indulging my curiosity on that topic.
As for your question, I'd be more than willing to answer it, but as we've both agreed here isn't the place. Send me an e-mail and we can discuss it.
Generally speaking, the more people talk about "being saved," the further away they actually are from true salvation.
#2 Total Post Count
Re: Any SOLID arguments against evolution?
Hi Alex, sorry I did not have time to get back to you. i worked 73 hours this week--shooo!! I am getting a little read in my chemistry book about the basics of thermodynamics. i know generally about chemistry from the laymen articles I have read, but I want to get a strong foundation in my technical understanding of chemistry and microbiology. My motive being to be better equipped to speak in this debate.
I don't have alot of time but I did want to say on the issue on the emergence of life that I accept wholeheartedly the biblical account that we were created by God from "dust"--which is a outstanding metaphor for the elements. The fact that it is dust -- small particles of matter, rather than say a rock or by something other worldly say, speaks to all generations, especially us today.
Secondly, on the question of expecting all experienced Christians believing as I do. Technically no. Generally yes. Theologically, there is room for some speculation and interpretation on some doctrines, because we are not given the complete picture on some doctrines--eschatology (the study of end times) being one of them. There are essential doctrines for the salvation of the soul and then there are periphery doctrines in which we might in part err but will not disqualify us from a heavenly entrance.
For instance, no matter what denomination we are, we must be born again by water and spirit to be a Christian. Christ teaches this in the gospel John ch. 3. It is essential to be regenerated by God's spirit and become a new creature in Christ Jesus, or we are only religious. Of course, you know we do this by acknowledging the truths found in Romans 3:23, 6:23, Romans 10:9-11, Acts 2:38, Epeshians 2:8-10, along with Christ teaching to Nicodemis in John 3. These principles are essential for genuine scriptural faith no matter what our denominational or theological background.
Re: Any SOLID arguments against evolution?
Since you "research the debate" you might want to take a long look at www.talkorigins.org
Researching a debate means looking at both sides right? Not just stocking up on the best arguments for your side of it?
What did the parasitic Candiru fish say when it finally found a host? - - "Urethra!!"
Re: Any SOLID arguments against evolution?
Wait a minute; are you suggesting people should actually try to be open-minded? How dare you bring that kind of heresy in here!
Just kidding, of course!
Generally speaking, the more people talk about "being saved," the further away they actually are from true salvation.
#2 Total Post Count
Re: Any SOLID arguments against evolution?
http://gamahucherpress.yellowgum.com/bo ... ection.pdf
the Samuel Wilberforce T. H. Huxley debate
here is one slam dunk - noted over 160 years ago- and nothing has changed
As Dawkins notes the problem
Darwin said it made his view wrong
Goould says nothing has changed since Darwin
Even the arch evolutionist Dawkins states the Cambrian explosion is a major problem and gives support for the creationists
Gamila, you are presenting arguments from a website that claims that dinosaurs and humans coexisted, and even seems to suggest that some dinosaurs might still be alive in some parts of the world. Do I really have to explain how rediculous this is, and how unreliable it makes your source?
Generally speaking, the more people talk about "being saved," the further away they actually are from true salvation.
#2 Total Post Count
Re: Any SOLID arguments against evolution?
you just refuse to see what is written dont you- i have pointed this out in numerous post quoted your own experts
the myths of your science have just a to greater hold on you
go read up on the cambrian explosion
dawkin and gould have told you the problem with it
darwin has told you about it
and gould again
You just refuse to use logic, don't you?
Generally speaking, the more people talk about "being saved," the further away they actually are from true salvation.
#2 Total Post Count
"And we find many of them already in an advanced state of evolution, the very first time they appear. It is as though they were just planted there, without any evolutionary history."
This quote you just took out of context is from Richard Dawkins' "The Blind Watchmaker". And it seems to me that you haven't read the book...
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Thomas Lighting for every location in your home
We offer a brilliant range of lighting for indoor, outdoor, and bathroom locations, with fixtures specifically designed for each location.
For indoor lighting we offer Classic Traditional, Fresh Traditional, and Transitional styles, with an expansive selection of designs and finishes.
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6" Recessed Trim
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Joseph A. Weins, Janesville, WI (1958-2014)
January 11, 1958 - May 1, 2014
Joseph A. Weins, age 56, a lifelong Janesville resident, passed away Thursday afternoon, May 1, 2014, at St. Mary's Hospital, Madison. He was born January 11, 1958, in Janesville, the son of the late William & Agnes (Skelly) Weins. He attended school in Janesville, and was a graduate of Parker High School, Class of 1976. He attended Blackhawk Technical College, and received his Associate's Degree in Computer Science. Joe worked at Beloit Corp., and then worked at General Motors Corp. until he was permanently disabled in a traffic accident. On September 21, 1985, he married the former Nancy P. Terry at St. Mary's Catholic Church. Joe loved computers, and was very tech savvy. He loved the outdoors, and loved the Green Bay Packers and Milwaukee Brewers. Joe had been a Boat Driver with the Rock Aqua Jays. He enjoyed spending time with his family, and especially with his daughter and his grandchildren.
Joe is survived by his daughter, Lindsay (Phil) Wester, of Elkhorn, WI; his two beloved grandchildren; Marlee and Liam Wester, both of Elkhorn, WI; his two brothers, Brad (Mary) Weins, of Janesville, and Jim (Diane) Weins, of Milton; his two sisters, Peggy (Doug) Johnson, and Jean (Jeff) Ashenfelter, both of Janesville; his dear friend and the mother of his daughter, Nancy Terry Weins, of Janesville; his two aunts, Jean McCann and Kay Skelly; his two uncles, Lamar Kuhlman and Willis "Bill" Howland; and by many nieces, nephews, cousins, and friends. Besides his parents, he was preceded in death by his in-laws, Martin and Bunny Terry, and by several aunts, uncles and cousins,
Funeral services with a Mass of Christian Burial will be held at 12:30 p.m.. on Monday, May 5, 2014, at ST. MARY'S CATHOLIC CHURCH, 313 E. Wall St., with Fr. Stephen Umhoefer officiating. Burial will take place in Mt. Olivet Cemetery. Visitation will take place on Monday, from 11 a.m. until 12:30 p.m. at the CHURCH. In lieu of flowers, memorials would be appreciated to the Little Liam Fund (Joe's grandson) in his name.
Joe's family would like to extend their sincerest thanks to Katie Cossman and to the rest of Joe's in-home nursing staff
for the care and love shown to him!
Whitcomb-Lynch Funeral Home & Cremation Services
21 S. Austin Rd., (608)752-2444


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The Canadian Opera Company is delighted to be part of the ever-growing support of honeybees, Currently we host seven hives onthe roof of our opera house, the Four Seasons Centre for the Performing Arts. Follow the honeybees' progress on Parlando, with visits and posts from beekeeper Fred Davis!
NEW - Our bee blog has moved!
Visit Parlando for more updates about our honeybees.
We will be requeening all the hives this year which I think, together with what appears will be a long summer, should improve the longevity, health and disposition of the hives. I've noticed the queens have slowed brood production when we need it. A good beekeeper must consider and prepare for the next season. In other words, I have to wonder how well the hives will do late this summer if there aren't enough successors to the existing working bees. With a new queen comes lots of new brood and that means foragers as well. Plus, as bees get older, they can get less docile, but having lots of new bees in the mix tempers their attitude.
I caught a swarm and placed it in a box that is now on top of one of the hives at the COC. I'll check back next weekend to see if the existing hive accepted the queen. This is the fun and interesting stuff.
Honey supers are on each hive now. Keep your fingers crossed for a good harvest this year.
Posted by Gianna Wichelow / in Update from Fred / comments (0) / permalink
After a winter of being well wrapped up, the COC's honeybees are back outside on the roof of the Four Seasons Centre for the Performing Arts, and getting busy for another honey-making season.
Beekeeper Fred Davis tells us that the hives look healthy and active now that the warm weather is here. In other good news, the hives have been shifted slightly from last year's location, so they'll be even easier to see from the north bar on Ring Four and from the window of the north door to the roof. Fred thinks that, as they'll get more sun here, they'll be happier. Please take a minute when you're attending the opera this spring to check them out! Fred also plans to "re-Queen" three of the hives to strengthen them. Fascinating stuff!
Here at the COC, we believe we're the only performing arts building in North America that has taken this step to helping protect and repopulate this very important species. But we don't want to be the only one and urge other landmarks, especially in our city, to get on board and help the honeybees. They need us almost as much as we need them.
Posted by Gianna Wichelow / in Bee Update / comments (2) / permalink
I was very pleased to see that all our hives made it through the winter this year. I realize we had it easy this winter but, with the standard loss in Ontario being upwards of 30%, I was worried things might not have turned out as well as they have. Today, April 1, was the first chance I had to take a quick look inside the hives. Opening the hives early in the season and when it's cold can result in what is known as "chilled brood." Sounds ominous, and it can set back the hive quite a bit if those new babies get too cold. Essentially they can die from exposure. So I only opened the top of the hives and placed some pollen patties and sugar water in baggies inside. I hope the pollen patties (which are protein for the hive) and the sugar water (carbohydrates) give the bees enough energy over the next week or so when the temperatures drop again. I had a wet but satisfying day. I'll check in again with the bees next weekend.
Thanks for the good news, Fred, and the photograph of the well-wrapped hives.
Read the nutritional guidelines for the FSC Honeybees Honey!


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Upcoming information meeting on construction scheduled for 2015 on County NN
ELKHORN�— People using the Highway 12 and County NN interchange will have an easier time after two multimillion dollar projects scheduled for 2015, city and county officials said.
The Walworth County Public Works Department is holding a meeting Thursday to update the public on the County NN projects.
The Wisconsin Department of Transportation will be constructing new County NN bridges over Highway 12 and reconstructing the ramps and portions of the roads leading to and from the interchange. The highway would remain four-lanes with each bridge handling two lanes of traffic per direction, according to state project documents.
The bridges are "in very poor condition with areas of significant corrosion" and the height of the bridges is inadequate, resulting in some trucks hitting part of the bridge, according to state project documents.�
"We did have strong support for the bridge replacement," Kevin Brunner, Walworth County public works director, said of a September public information meeting. "It's a bridge that needs to be replaced, and County NN is in pretty poor condition as well."
The pavement on County NN east of Interstate 43 to Bray Road will be replaced. Besides routine maintenance, part of County NN has not had major work completed since being constructed in the 1960s, according to one project document. �
About 7,900 vehicles use the County NN and Highway 12 interchange each day, according one document.
The state's plans include accommodations for people crossing the highway on foot or bicycle.
A multiuse path will be added to the north side of County NN, starting east of Elkhorn High School and extending to the White River State Trail on the other side of Highway 12.
The city has agreed to work with the county and state in financing the extension of the multiuse trail into the city, City Administrator Sam Tapson said.
The city will extend the path from the east side of the high school to the western limit of the state's project that ends east of the I-43 overpass.
How much the extension will cost has not been determined because the city council has not decided if it will complete improvements on roads in the area at the same time, Tapson said.
The multiuse path will be 10 feet wide, Brunner said.
The state's project budget is about $10 million, Brunner said.
The Gazette was not able to reach representatives from the Wisconsin Department of Transportation to comment on the budget and project plans.�
The county's project will pick up where the state's ends. The $3.4 million project involving the reconstruction of County NN will start east of where the state's work will end near Bray Road. The county would complete work to within 650 feet east of Lakeland School, according to one project document.
"This is really an important connection because our county government campus is located on Highway NN, and you have to cross this bridge, and obviously NN is the entrance into the campus," Brunner said.
The county received a $270,000 federal grant from the Congestion Mitigation and Air Quality Program to offset part of the cost of the multiuse trail and sidewalk.
The construction will be completed in stages to maintain access between Elkhorn city limits, the government departments and the hospital.
"The county will have to work really closely with the state on the detouring as well as the signage to get people around and that's going to be key to this whole project," Brunner said.


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The Liquidity Management panel: Saif Ashraf of Google, Nicholas Ro of Toyota Financial Services and moderator Ben Campbell of Capital Advisors Group
AHA attendees
Diana L. Noteboom of ConocoPhillips
Cash Management winners: Diana L. Noteboom of ConocoPhillips, Christopher Salmon of Visa, Elizabeth Ecsy of Cunningham Lindsey Group, Kenny Tse of Google and moderator Michael Berkowitz of Citi Transaction Services
Sunnie Ho and Jayna Bundy of Microsoft, Dennis Tosh of Ford Motor, Aditya Agrawal of Google, Randall Finnessy of FLIR Systems and moderator Wolfgang Koester of FiREapps
AHA attendees
Kojo Ako-Asare of Google
Best Green Strategy winners: Michael Suriano of Honeywell International, Brian Brick of Cigna Global Health Benefits, Kojo Ako-Asare of Google and moderator Lee Horwitz of J.P. Morgan Treasury Services
Mickey Levy, chief economist at Bank of America Merrill Lynch, spoke at lunch.
Charles Wa of Google
Technology Excellence winners: Rex Queen of Milwaukee County, Charles Wa of Google, Anita Prasad of Microsoft, Pankaj Gudimella of Microsoft, Nishant Rohatgi of Toyota Financial Services and moderator Bob Stark of Kyriba
AHA attendees
Working Capital Management winners: Jim Scurlock of Microsoft, Jennifer Dale of Sprint Nextel, Theresa Karas of Stanley Black & Decker, Axel Martinez of Google and moderator Michael Fossaceca of Citi Transaction Services
Enterprise Risk Management panel: Laura Langone of Juniper Networks, Theresa Bourdon of Aon, Erika McBride of Paychex and moderator Marie Hollein of Financial Executives International
Brent Callinicos, treasurer of Google, which won the Overall Excellence Award.
Googlers with their AHA awards, along with Peter Frank, moderator of the Overall Excellence session (fifth from left)


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Odd food poisoning-like symptoms, strange tastes
Human Anatomy, Physiology, and Medicine. Anything human!
Moderator: BioTeam
Odd food poisoning-like symptoms, strange tastes
Second post (After this, I promise I'll stop cluttering the forum with newbie questions).
To give a brief background on why I'm posting, I'm trying to find a diagnosis for symptoms that are either of a paranoid non-existent hypochondriac illness in my head, or for a very real disease. I'm by no means a biologist so excuse my simple laymans terms. The doctors I've spoken to have no idea what I'm talking about.
Since about 1999, I've been getting some kind of problem, the doctors call it food poisoning, but I have a feeling it might be something else, I just don't know, because some of the symptoms baffle me, and they're worse than any bout of poisoning I've had, I feel like something is gravely wrong.
Luckily for me it is now a very rare thing (I used to have it once every few months as a child), the last time I had this illness was 2008.
One minute I'll be fine, then literally within minutes, my mouth entirely dries up, as if someone has sucked it dry with a vacuum cleaner, and then I get bloated. Whenever I burp, a foul rotten egg taste comes to my mouth (I can only describe the taste of it as a mixture of foul rotten eggs, burning toast/popcorn, and sulphur in one). It's so bad it literally makes me want to be sick. When I get this burp, I *know* I'm in trouble for the next 7 hours. It's followed immediately by the most painful, extreme case of the runs, abdominal pains (Feels like someone is boiling water in my intestines and stabbing it with a knife), and intense nausea. Luckily during my last bout in 2008, I was able to leave my campus and get to a medical centre where I was given a metoclopramide injection, I didn't throw up but I sure felt like it. The last time I had it, I remember counting that I vomited until my mouth was dry, and was gagging for about 6 hours nonstop at about an average rate of twice per minute, I could not sleep properly without half gagging and waking up).
Since then though, I've asked doctors and searched the web, and I just can not find the cause of this burp. For me, there's a difference between "food poisoning" where you get the runs, cramps, possible vomiting for a few days, but this illness is intense, and the symptoms are the worst I've ever had, it's enough to debilitate me. The burp worries me, it is almost as if my body is telling me something is gravely wrong.
I don't know what this is, but can anyone here shed some light on what it may be, how it can be (if at all) prevented, diagnosed, or treated? I could be wrong and perhaps it is just Norovirus or something, but what I find odd is that no-one I've spoken to who has suffered norovirus or food poisoning can seem to recall having foul belching. My only other thought is that it might be some kind of bacteria buildup from rotten food caused by gastroparesis or something, but it's just speculation. I'm trying not to worry about it (it really doesn't help now that I almost get a little shaky when I feel so much as bloated), but it would mean the world to me if I could find out what this blasted horrible illness is after a decade of searching. The only other information I have is that I vaguely remember the first time I got it, I had eaten a Banana Fritter from a truck stop at the young age of about 10 (my immune system was probably weak as anything), and within about 3 hours I was ill.
Last edited by blackmantis on Mon Mar 28, 2011 7:12 pm, edited 1 time in total.
Well, having had my share of food poisonings and stomach illnesses as a kid (and once or twice as an adult) I'd say that that doesn't sound like any normal food poisoning to me. I do not know why it would cause dry mouth first and then later such symptoms as you described.
I have a couple of wise old clinical microbiology profesors around here where I work, I could ask them about your problem next time I see one of them. If they haven't heard of anything like that, then I think it must be something else. And if it is something else, then I'd go for either some neurological problem (because of the dry mouth) or some secretory problem (like bile reflux from the intestines or something similar that causes irritation of the bowels/stomach and this burb-thing). Foul eggs usually contain sulphur compunds (that's why the smell), not sure if this bit of information is of any use.
Anyways, I try to be back in touch in few days.
Re: Odd food poisoning-like symptoms, strange tastes
Sorry, none so far. Though the person I thought might know something has been unavailable because he's been abroad a while. I'll try to remember to post here if I find anything!
Re: Odd food poisoning-like symptoms, strange tastes
Thank goodness that there is someone else out there that suffers from the same problem that I have. I'm not too keen of throwing up myself, and find that sometimes, I get those burps that you describe as well. Mine though just tastes like the burnt popcorn, but no one else that I know of in my town or family suffer from the same thing. I get the exact symptoms that you have (I'm actually experiencing the nausea and the stomach cramps right now) but what is strange to me is that I haven't eaten anything out of the ordinary. Though its not the most comfortable thing to have, and can get quite worrying, it's good for me to know that there is someone else out there that has the same thing. I'm asking around to my friends who have nurses as friends, to see if they have any answers, so if I find anything out, I'll reply ASAP because I would like to know as well.
Re: Odd food poisoning-like symptoms, strange tastes
Yes! Burnt popcorn is the same sort of "taste" I could describe it as. Other ways of describing it is rotten eggs, sulphur, burning eggs, it's a foul taste. There seems to be nothing known about what causes this as so many people often post things such as "Oh wow you have this too!? My doctor has no idea" etc etc, like a large population of people get it but it remains a mystery as to what causes it.
I've been doing a lot of studying, and even the symptom itself is hard to find. Had a good read through my Campbell Biology book (as I'm only starting out), but I have found a few things that can cause it (although I'm not looking to self-diagnose, but I have often referred these causes to my doctor as we're working on figuring it out):
* Gastroparesis (delayed emptying of the upper GI, food can sometimes rot and give that foul belch, although it is often only accompanied by bloating, and vomiting but not often diarrhea). It is unlikely gastroparesis is giving us both grief.
* Giardia Lamblia (This is my main suspicion at the moment. I have heard from various sources that Giardia can cause the foul belching, often with watery diarrhea and vomiting, however I've never had it last for more than about 6 hours, rather than the 2 days that the symptoms often go for with Giardia. The belch is perhaps considered insignificant in comparison to the other symptoms such as vomiting and diarrhea, that it is not essentially listed as a symptom, but I do get gas which is interesting, as I find I have to belch more often). I believe this is most likely what is causing our problems.
There is an interesting article here -> http://www.healthhype.com/sulphur-or-rotten-eggs-burps-belching-foul-smelling-gas.html which details sulfur belching and some common causes, however I still believe Giardia may be the culprit. It could be the taste of bile coming back up? Although the last time I had this bug, I did not vomit (probably due to my phobia willing me not to with every ounce of strength, and being shot up on metoclopramide hydrochloride), although I certainly felt nauseas.
Still, I'm always fascinated to hear if someone else has had this. There *must* be something or some pattern that we (the sufferers of this vile bug) are following to end up in such a miasma of symptoms. Is there anything you could perhaps tell me that might help? I live in Australia, where Sydney Water is commonly known to have Giardia in it, my mother details the scares of the 1990s in which most people caught it up to 6 times in a year, although most people I know who acquire the protozoan don't often report the foul belch.
I will update this thread with my findings, and I would appreciate any and all feedback from the biology community. If anyone can figure this one out, it's you guys. Not a single doctor I've talked to has a clue. Personally if I could come across an anti-emetic that was *extremely* effective against the vomiting caused by this bug, as well as something that could counter the nausea, I would not let it worry me.
hello there
well I cannot really imagine your symptoms, but considering burp...my Mom used to have that symptoms. Untill now, she still burps a lot (relatively compared to any in the family)
When she went to doctor, the said something wrong with her liver, some kind of enzymatic problem. Have you went on test for something like this? Now she take some digestive pill some other time.
Hope this help, I dont really know the taste of her burp
As for Giardia, if your local water contain such bacteria, there will be an outbreak in your area and you wont be the one having the symptoms, right?
Production Staff
PT. BioFarma
That's the strange thing though, we had a massive Giardia scare in Sydney in 1997 -> http://en.wikipedia.org/wiki/1998_Sydney_water_crisis
During this period (which I was getting sick in, I don't recall the date this illness commenced for me exactly), many many people I knew suffered from Giardiasis, including my mother, however I have asked her and many others who I knew were affected, not a single one of them reported symptoms matching my own. They often had watery diarrhea for 1-3 days, and often vomited. The vomiting was not so intense that they could not eat (as it would happen only a few times a day, they could still find a moment devoid of nausea to have some dry toast), however my symptoms thankfully never last beyond about 6 hours, although when I was younger.
Well the burp is just a side effect of this miasma of an illness or dysfunction. If it was *just* the burp, I would not be as worried (I've heard mint can suppress the hydrogen sulfide taste). Did your mother also suffer from profuse diarrhea and sometimes vomiting during her liver enzymatic dysfuntcion or was it just the foul belching?
What I aim to try and find out, is:
* What exactly causes this burp, and the taste of hydrogen sulfide? Is perhaps the liver malfunctioning and sending bile back to the stomach, or is the upper GI seizing up causing gastroparesis (which would cause the rotting food to be squeezed back up into the stomach and therefore cause the vomiting and foul taste)
* What illness causes the overall symptoms? Is this an illness caused by a bacteria, enteroviral or protozoan / parasitical infection? Many user accounts lead me to believe it may possibly be Giardia Lamblia, and I would rule out Helicobacter Plyori as I have been tested for it and the results came back negative. Or perhaps is it a gastrointestinal dysfunction? Is the upper GI seizing up somewhere? It's almost as if once the food has reached the upper intestinal tract, the body screams "get out right now!" as hard as it can, and suddenly it's extremely forceful in removing the food through both exits as fast as it can.
Interestingly enough, there is a 45 page long thread on it at Wrongdiagnosis. I have read the entire thread, and not a single person can have it diagnosed by a doctor!
45 pages right there, with not a single answer in it. This is a serious problem and it is going undiagnosed and unrecognized by doctors. I think some of the best gastroenterologists and pathologists need to look into this because it is truly distressing. Like myself, most people in the thread who have experienced it report developing an instant and extremely strong phobia towards the illness. (most likely due to the intensity, as no doctor seems to be able to understand that you can not eat or drink when you are vomiting every 2 minutes nonstop for 6-12 hours straight).
I think we have a dysfunction or bacterial/enteroviral/protozoan infection that has either not been discovered yet (as it only seems to affect select few people. Most people seem to never get these symptoms, however I have noted that usually the people who get it once experience recurring bouts of it throughout their life. I am the only person in my family to get this and I am unaware of any significant lifestyle changes I possess or have made that caused me to get this recurring bug). This is what worries me. It sounds like some kind of disease that manifests itself in the gut, and infects the person (so they expel most of it), and slowly over time, the bacteria builds up again, and causes another serious infection and the usual symptoms (Would this be a reasonable theory? Does H Plyori not perform a similar act of manifesting itself in the gut so it can reinfect?)
The only other cause I could think of, is perhaps some kind of small intestinal bacterial overgrowth? I've read that if the balance of bacteria in the small intestine is upset, the body reacts (often violently) in expelling the bacteria in order to cull the abnormally large numbers. This sounds like a valid and possible cause to me, although I don't quite understand the source of the foul hydrogen sulfide taste. Perhaps is this gas going through all of us every single day once food hits the upper intestine and beyond, and is broken down by sulfur reducing microbes until it reaches the anus, by which time it has lost most of the odor?
I am tempted to start a thread and detail my notes about it in the first post (as in edit/update it whenever I can). I will continue to post anything or any developments I find in this thread. I have noticed a lot of patterns about it.
* When I first started experiencing symptoms, my bouts of this strange illness/bodily dysfunction were spaced closely together. I would experience it about every 6 months. Interestingly enough, I never used to vomit much from it. I recall being in school one particular bout, and suffering from only the belching and lower GI symptoms, however I did not feel nausea from memory, just dehydrated, and was able to continue my studies, whereas as I've gotten older, the symptoms have become worse and more extreme each time.
Slowly over time I seem to be able to go longer and longer without acquiring it, my longest being from November 2003 to April 2008. I've read that many people seem to shake the problem for many decades, only to have it begin to occur again when they reach their 50s (in which it starts to occur much more often, perhaps as a result of an impaired immune system due to older age? Or perhaps a weaker gastrointestinal system?) The thought that I might acquire this problem almost every month when I'm in my 50s would definitely guarantee me taking a suicidal route to escape it, due to the helplessness. I feel that even if this problem had a diagnosis but no cure, it would make it a lot more clearer for me to understand, since it just seems to magically happen out of the blue with no pattern leading to it. I lose sleep at night running through notes, and information I've absorbed going through my head, repeating "there *must* be a cause for this", the body wouldn't just naturally make you belch hydrogen sulfide, vomit violently and experience fulminating dysentery. Something must be irritating the GI tract or it must be an immune system response.
Dear blackmantis,
I guess my mother did not have diarrrhea or vomit during those days.
Have you try on Gastroesophageal Reflux Disease? After reading your latest assumption, it came accross my mind.
Perhaps some others have post it in this thread. It a disease where youre stomach content is pushed upward to your esophagus.
Actually the main symptom is heartburn which i did not recall appear in your symptom list.
But it can cause burps, vomit, and nausea.
There are many causes for GERD, but it all reaches one point: weak lower esophageal sphincter (LES). This sphincter should retain your stomach content in your gastric. This sphincter can be weaken by some food or drink (acid, spicy, soda, coffee) and strenghten by others (mainly those contain hi protein).
But some people are just born with weak LES.
There are changes in lifestyle that you can do. Beside avoiding those food effecting your LES, sleeping position (maintain your head higher than the body), and clothing (use loose clothe) can do some help.
For diagnose, pH of your esophagus should be measured in certain time. It should be lower than normal esophageal pH. Sometimes it could have caused damage to your esophagus epithel.
Pharmacology handling include, sucralfate, PPI or some other gastritis regimen...
Non pharmacology handling can include some operations on implanting protein to strenghten your LES...
Have you asked your doctor bout this?
Production Staff
PT. BioFarma
Hi Adihutama,
Reflux does run in my family, however my father has never reported any of the symptoms I have encountered. He has been devoid of (any serious) illness since the 1970s. I would be tempted to say GERD, however I feel that the fulminating dysentery could not possibly be caused by it. The symptoms are just far too extreme. I have had my fair share of belching, acid reflux, and nausea from it, but never any extreme symptoms, although perhaps it could be a risk factor (Perhaps the excess amounts of stomach acid irritate the lining and cause gastritis?) However it still wouldn't explain passing over 20 litres of water an hour. To me it still only seems that the body picks something up, and as soon as it discovers it, it goes into absolute overdrive and its 100% main priority is to expel whatever the contents are to get rid of the food. It is much more forceful and violent than the general bouts of food poisoning I suffered as a child. I seem to also lose a lot of weight in these bouts. The last continual bout I had in 2008 resulted in me losing 13kg of body weight in under two weeks. It just seems too serious to be gerd, gastroparesis, or any of these typical illnesses, and I don't know anyone else with GERD who has had these symptoms.
I do recall the nausea being so strong it left me with obtundation. I recorded two videos on my cell phone of me talking during my last illness, and when I was well, I watched the videos and barely remembered it, similar to the feeling of being alcoholically intoxicated, (I couldn't focus, I slurred my speech) etc and it only seems to get worse as I get older. I'm only 22 at the moment, but when I used to get it as an 11 year old, I recall being able to suffer a bout during school time, I would make all my visits to the bathroom then return to class, completely unmoved by the whole thing, whereas the last two times it has grown in intensity, as if my body can no longer handle it. I have always got my iPod ready for the same occasion should I get this illness again, and I will record a log of all the details I can think of, in case there's one key symptom I'm overlooking due to the amalgamation of so many at once.
The problem is, I hear a different ostensible answer for the cause of this illness from every doctor and even from those who suffer it. One person will pipe up and say "Yes! I had this with serious diarrhea, vomiting and the belches for years, then I stopped eating food with red dye #40 in it and it went away!" and some will say "The cause is red meat and poultry, I stopped eating and I'm fine".
I haven't ruled this theory out, perhaps there was some kind of junk food I consumed as a child and didn't link to it, perhaps it was something completely innocent like a lollypop or candy (as I rarely eat candy or sweet products now), and I just happened to consume it in 2008 and it caused it, but it just seems so unlikely that one tiny foodstuff can cause such a violent reaction. The reaction is akin to that of mixing a gummy bear (sucrose) with molten potassium chlorate. Fulminating, instant, and forceful for six hours, and the symptoms are strong enough to get the message from your body to yourself, as if it to say "Never ever consume that again, something is seriously wrong.. I always remember feeling a sense of trepidation, dread, as if I naturally knew something was really wrong whenever I had this, even when I was young and did not fear it (which I learned to quickly, as most others do). Another theory by some is that they get these symptoms when stressed, or incredibly anxious, which could possibly make sense, as I did have the symptoms on my first day of college in 2008, but again, I just find it so hard to believe that something so stupid as anxiety could produce such an influx (or in this instance, outflux!) of horrible symptoms. When people tell me "oh yeah it's probably just a bug/migraine", I just simply cannot believe them.
Dear blackmantis,
well, since we are a biological creature, you have to open up your mind to every possibility.
I used to have skin exim (i am not sure whats the term in English) when my skin got ceratinized an it got so itchy. But the itch dont happen all the time. It only come when im in stress or under pressure.
Not to mention this neck muscle tension i had when the deadline comes. I can only nod or look up because turning my head left and right will cause some pain.
Doctor said the muscle tension is only a stressed related problem, and he only gave me some muscle relaxant and neurotropic vitamin.
And it works damn well, i also try to reduce my stress level, and it kinda work.
There must be some correlations between our anxiety, fear, stress and our biological function that can cause illness
Production Staff
PT. BioFarma
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text:
Nebraska Legislature Strikes a Blow Against ATM Litigation Abuse
The Nebraska state legislature has eliminated a state requirement that ATMs in that state notify consumers about transaction fees physically, on the machine itself, as well as on the screen before the transaction goes forward.
The Nebraska Credit Union League supported the legislation which comes after Congress passed similar federal legislation.
During a Jan. 22 public hearing before the Banking, Commerce, and Insurance Committee, Nebraska Credit Union League Director of Governmental Affairs Brandon Luetkenhaus testified, "Credit unions appreciate and support sound regulations that protect consumers and promote fair play in the market place. However, sometimes well-intended regulation can be unnecessary and burdensome and we believe that's the case with these physical disclosures."
Laws and regulations requiring the physical notices have been used to file lawsuits against credit unions and banks across the country.


text:
The Fiber Disease
Human Anatomy, Physiology, and Medicine. Anything human!
Moderator: BioTeam
Simple, it;s all about the fusion power and they don't give a damn about us or those magnetic fields they are busting thru.....this is there way to test the nuclear weapons but in a safer way. We have radiation poisoning from the solar/plasma cells......the wind turbines too.
Why do you think they ignore the magnetic fields? Simple, b/c they are evil, greedy A-holes. The end.
Check this out.....It was written this summer and its on insects and rashes but what caught my eye is where it said A) if one has symetrical lesions (on both sides of body) it is a sure bet it's from an insect? What kind of stupid comment is that? Or maybe it's me that's stupid, but I have had insect bites for over 40 years that were NOT symetrical but that ended last year....now they are all of a sudden symetrical?
Public release date: 28-Jun-2006
Johns Hopkins Medical Institutions
Hopkins researchers develop new quick tool to sort out insect bites in children
Guidelines should save money, stop unncessary testing
Children afflicted with insect-bite rashes are often misdiagnosed or referred for extensive and costly tests, but a new, easy-to-remember set of guidelines developed at the Johns Hopkins Children's Center should help.
Called SCRATCH, the letters form a memorable acronym for symmetry, cluster, Rover, age, target/time, confused, household). It is a guide to the symptoms and features that help pediatricians and others to recognize the source of a rash.
Insect-bite skin rashes mimic the symptoms of a variety of conditions, ranging from fungal infections, scabies, allergies and environmental contacts, to HIV-associated dermatoses. Reactions to a bite are often delayed, making it difficult to trace exposure.
"SCRATCH could spare many children and their parents from going through invasive-not to mention expensive-procedures if pediatricians recognize the problem early on," says Raquel Hernandez, M.D., a third-year resident at the Children's Center and lead author of the article, published in the July online edition of Pediatrics.
Hernandez and co-author Bernard Cohen, M.D., head of dermatology at the Children's Center, developed SCRATCH by examining a month's worth of patient records from visits to the Children's Center dermatology clinic. They found that the majority of children who were eventually diagnosed with an insect-bite rash had undergone extensive lab tests and skin biopsies before they were referred to Hopkins.
The most common misdiagnosis was scabies, a skin infection caused by a parasite that produces red, itchy lesions. Many of the children were treated repeatedly for scabies.
"These guidelines are really intended to make pediatricians consider insect-bite hypersensitivity as a diagnosis and think twice before referring a child for a skin biopsy or another invasive procedure," Cohen says.
Using the tool is straightforward, Cohen adds. If the rash fits the SCRATCH criteria, it's likely bug-borne.
S for Symmetry
Erruptions are usually symmetric and appear on exposed parts of the body, such as face, neck, arms, legs. Younger children may have rashes on their scalps. Diaper areas, palms and soles are not affected. The trunk is rarely affected. By contrast, scabies causes rashes on palms, soles and between toes and fingers.
C for Clusters
Lesions appear in "meal clusters," described as breakfast, lunch and dinner. The linear or triangular clusters are typical of bedbug bites, but also appear in bites caused by fleas.
R for Rover Not Required
Presence of pets in the household is not a criterion for diagnosis because a bite might occur outside of the home.
A for Age Specific
The condition is most prevalent in children between the ages of 2 and 10.
T for Target Lesions and Time
Target-shaped lesions - named so for their resemblance to the bull's eye on a target -- are typical of insect-bite hypersensitivity. Time indicates the chronic/recurrent nature of the eruptions. Many patients may have delayed reactions and may not experience flareups until months or years after the intial exposure. Most children develop full immunity by age 10 and no longer have recurrent rashes.
C for Confusion
Parents often express confusion and disbelief at the suggestion that there might be fleas or bedbugs in their homes. "One of the primary criteria is that if the parents don't believe me, I am probably right," Cohen says.
H for Household with Single Family Member Affected
Unlike conditions that have similar symptoms, such as scabies and atopic dermatitis, insect-bite rashes often appear in a single member in a family.
"Common sense might tell us that fleas and mosquitoes would affect other members of the family, but we must keep in mind that these rashes develop in children who have hypersensitivity that others do not have," Hernandez said.
and yes....once again, I ran across another strange HIV article, only that there is no write up this is so new....but here is the title:
[HIV-associated skin diseases. 1: Follow-up and epidemiology of HIV infection, pathogen-induced HIV-associated dermatoses]
http://www.eurekalert.org/pub_releases/ ... 062806.php
You mentioned the vagina....well, I dunno for sure but I have been saying strep is going to play a role as much or really MORE than Staph does with our illness...Remember, I said Strep, not Strip Sarah!! LOL Then, I found this:
[Vaginal colonization of group B Streptococcus: a study in 267 cases of factory women]
http://www.ncbi.nlm.nih.gov/entrez/quer ... t=Abstract
and favorite quote of the day that I heard???
Ultimately, an understanding of these principles that underlie natural photosynthesis should point the way to the development of efficient, robust artificial systems for solar energy storage.
hey great point ..
years ago had circular saw accident.. like nipped the tip of my diget.... i had some stiches on the middle finger of the left hand ...
.. oddly another circular scar is forming on adjacent finger....
so mundane. but odd enough for headfirst reasearch
Welcome to Department of Science and Technology, Govt. of India ::
AF - 514 Phase I Selections from the 06.1 Solicitation
Why don't we have AIDS?
Thanks for the link Bartz.
http://www.ncbi.nlm.nih.gov/entrez/quer ... med_docsum
"Treatment of cutaneous protothecosis with a variety of topical and systemic antibacterial, antifungal, and antiprotozoal agents has met with variable success (47, 49). Surgical excision is recommended for small localized lesions. Amphotericin B and the azoles (itraconazole, fluconazole, and ketoconazole) appear to be the most effective medical treatment (11, 49, 67, 71, 93). Cure of olecranon bursitis generally requires bursectomy (47, 49). Repeated drainage has failed; however, drainage coupled with local instillation of amphotericin B has been curative (20, 76). Treatment of disseminated protothecosis is best accomplished with amphotericin B, although the optimal dose and duration of therapy are uncertain (49, 93)."
It seems odd that no one with the fibers has AIDS when everything I read that could be connected to what I suffer from says things like this, "Disseminated protothecosis is rare and almost always occurs in individuals with severe immunocompromise from cancer treatment, a prior solid organ transplant, or AIDS (36, 43, 48, 54, 93, 104)" ~ scroll half way down in link provided
How did your test results turn out if I may ask?
seems we all could have a case of chronic
"hyperMEMtotic Man/hardware-in-the-looptosis"
Low-Cost Day/Night Imaging Sensors for Micro/Mini-Uninhabited Aerial Vehicles (UAVs)
Title: Hyperspectral Identification for Collaborative Tracking
Abstract: Many problems exist, particularly in defense and security scenarios, in which long-term tracking of objects is important. Long-term tracking generally requires the use of collected features in order to uniquely identify the object of interest. As conditions under which the features are collected changes, so do the features. While different sensor phenomenologies have been developed to collect feature measurements (i.e., measurements that depend on target attributes such as shape or color), this effort is focused on using dynamically collected features from hyperspectral sensors under changing operating conditions to extend track lifetime. Changing operating conditions preclude the use of a priori feature databases; thus, a feature database that is built "on-the-fly" is required to reliably track vehicles over longer periods. Furthermore, the feature database must be dynamically managed since, as conditions that affect the collected features change, the previously collected features may no longer be used to realiably identify the vehicle. Toyon Research proposes to analyze and statistically model hyperspectral feature data, and develop a feature database management algorithm to accommodate changing operating conditions. Moreover, Toyon will analyze the utility of using hyperspectral signatures and the database management algorithm to extend track life.
Digital Receiver Geolocation Technology Simulation
Abstract: Man/hardware-in-the-loop laboratory simulation is the most cost-effective methodology for evolving/maturing advanced receiver geolocation technologies because the battlefield can be brought to the laboratory through multi-spectral synthetic battlespace simulation. Current laboratory laboratory RF threat environment simulators do not provide the required fidelity to accurately simulate the parameters needed to develop ultra-precise direction finding and geolocation capabilities. DRA proposes to solve this challenging technology limitation by developing an Advanced RF Geolocation Simulation Testbed (ARGST) for rapid prototyping of advanced RF receiver processor geolocation. The ARGST flexible architecture will enable the development of advanced geolocation technologies for single and multiple aircraft within a controlled laboratory environment enabling repeatable test and step-by-step evaluation/debugging capabilities. ARGST will provide simulator technology to develop advanced geolocation capabilities for receiver/processor technologies within new military concepts such as Advanced Threat Alert (ATA) Advanced Technology Demonstration, and Lightweight Modular Support Jammer (LMSJ) for application to military aircraft such as F-35, F-15, F-16, F-117, B-2, and C-130. During Phase II, DRA will develop a prototype capability using the Sensors Directorate's Integrated Demonstrations and Applications Laboratory (IDAL) as a testbed to demonstrate key performance characteristics. The Phase II effort will provide a building-block capability for rapid evolution of advanced RF receiver/processor technology.
I found this to be interesting. It seems to state some things that Tam was saying, but what do I know????
http://www.canbiotech.com/CommonData/Ne ... /032.4.pdf
Any opinions on products like this for potential topical "spot" treatment for this condition?
CIMEX BIOTECH — CryoPen®
Cimex contracted Nucleus to create a medical animation for training physicians how to use their product, the CryoPen®, a medical device that freezes tissue without the use of cryogenic liquids or gases.
Nucleus created a 2D medical animation showing physicians how the CryoPen® works to treat skin lesions. Using a cross-sectional view of a skin lesion, the animation demonstrates the CryoPen® freezing skin layers, identifying the frost line, kill zone, transition zone and recovery zone of the frozen lesion.
http://www.nucleusinc.com/medical-devic ... ne=cryopen
An alternative may be purchased in drug stores. (Dr. Scholl's wart remover)
During my years of pre-awareness, I had this type of procedure done by a dermatologist. It worked like a charm on that occasion and it never came back. I wonder what reaction the fibers have to this type of chemical process?
Mr. Barz,
Your links prove very very much to the point.
http://www.canbiotech.com/CommonData/Ne ... /032.4.pdf
Link cyanobacteria and bionanotechnology
"Alginates are produced by brown algae and the two bacterial genera Pseudomonas and Azotobacter"
Chapter Abstracts: Microbial Bionanotechnology: Biological Self ...
The exploitation of nature in the field nanotechnology is just beginning, ... rich substance found in most cyanobacteria and in some heterotrophic bacteria. ...
http://www.horizonpress.com/hsp/abs/absnano.html - 20k - Cached - Similar pages
Link biofilm production/ quorum sensing
Current Endosymbiont. Photosynthetic Cyanobacteria in cytoplasm of flagellated protozoan Cyanophora ... Correctly use the term quorum sensing in your answer. ...
microvet.arizona.edu/Courses/ MIC438/decker/Biofilms/Biofilms.html - 21k - Cached - Similar pages
(paste in google)
About: "biofilms in relation to microbial resistance":
"The study of biofilm communities benefits from the efforts of investigators from many different disciplines, including environmental and clinical biologists, surface chemists, engineers, and mathematical modelers, who bring their unique questions, perspectives, and technologies to bear on this phenomenon. Unfortunately, it's difficult to keep abreast of the scientific literature in one's own field, let alone others"
http://www.pubmedcentral.gov/articleren ... tid=438604
Last edited by tamtam on Sun Aug 27, 2006 9:52 pm, edited 2 times in total.
Okay Mr. TamTam, but HOW did I become infected. Was this stuff released as a pesticide? Is it sold to the general public? I need to know. I will take action, but I can't just go and say "Hey, look, I have some fibers in my skin and you did it." They will lock me up so fast. My children and I are suffering as are many, many people, as you know. We need to know Who and Why for starters. You help is much appreciated.
To answer your question Mr. Barz,
This type highly modified micro organism is most associated with target of a proteome research center.
Proteome research centers are synonymous with biodefense.
Best is that people unite and create a legal base for action.
Last edited by tamtam on Sun Aug 27, 2006 10:29 pm, edited 1 time in total.
We present the nucleotide sequence of a 5248 bp-long region of the mitochondrial (mt) genome of the dermatophyte Trichophyton rubrum. This region which represents about 1/4 of the total mt genome of this species reveals a compact organization of genes including: the glutaminyl tRNA, the methionyl tRNA, the cytochrome oxidase subunit I gene, the arginyl tRNA, the mitochondrial version of the ATPase subunit 9 gene, the cytochrome oxidase subunit II gene and a part of the NADH dehydrogenase ND4L and ND5 gene "complex". The main features of the part of mt DNA sequenced is the non-interrupted COXI gene and the presence in the mitochondrial version of the ATPase 9 gene of a small group IA intron. The extensive amino-acid sequence similarity with the equivalent gene in Aspergillus nidulans and Neuropora crassa indicates that this gene codes for a dicyclohexylcarbodiimide binding protein. The conserved arrangement of this portion of the mt genome and the presence of tRNAs between the protein-coding genes are compatible with a large polycistronic transcript processed by the excision of tRNAs, or similar secondary structures, as proposed for other fungal or mammalian mt DNAS.gene name description
AT5G19540.1 expressed protein
AT5G19530.1 spermine/spermidine synthase family protein, similar to SP|P09158 Spermidine synthase (EC 188.8.131.52) (Putrescine aminopropyltransferase) {Escherichia coli}; contains Pfam profile PF01564: Spermine/spermidine synthase
AT5G19520.1 mechanosensitive ion channel domain-containing protein / MS ion channel domain-containing protein, contains Pfam profile PF00924: Mechanosensitive ion channel
AT5G19580.1 glyoxal oxidase-related, contains similarity to glyoxal oxidase precursor (Phanerochaete chrysosporium) gi|1050302|gb|AAA87594
AT5G19570.1 expressed protein
AT5G19550.1 aspartate aminotransferase, cytoplasmic isozyme 1 / transaminase A (ASP2), identical to SP|P46645 Aspartate aminotransferase, cytoplasmic isozyme 1 (EC 220.127.116.11) (Transaminase A) {Arabidopsis thaliana}
AT5G19560.1 Encodes a member of KPP-like gene family, homolog of KPP (kinase partner protein) gene in tomato. Also a member of the RopGEF (guanine nucleotide exchange factor) family, containing the novel PRONE domain (plant-specific Rop nucleotide exchanger), which is exclusively active towards members of the Rop subfamily.
AT5G19510.1 elongation factor 1B alpha-subunit 2 (eEF1Balpha2), identical to elongation factor 1B alpha-subunit (Arabidopsis thaliana) GI:6686821 (AT5G19500.1) tryptophan/tyrosine permease family protein, contains Pfam profile PF03222: Tryptophan/tyrosine permease family (AT5G19490.1) repressor protein-related, similar to repressor protein (Oryza sativa) GI:18481624 () similar to eIF4-gamma/eIF5/eIF2-epsilon domain-containing protein [Arabidopsis thaliana] (TAIR:At3g02270.1); similar to PREDICTED: similar to Translation initiation factor eIF-2B gamma subunit (eIF-2B GDP-GTP exchange factor) [Gallus gallus] (GB:XP_422427.1); contains InterPro domain Nucleotidyl transferase (InterPro:IPR005835); contains InterPro domain Bacterial transferase hexapeptide repeat (InterPro:IPR001451)
catalyzes the transfer of a formyl group from
formylmethanofuran to tetrahydromethanopterin
"Function Code:9.10 - Metabolism of Cofactors and
Vitamins, Porphyrin and chlorophyll metabolism"
Function Code: - Cell Processes, Transport of
carbohydrates organic acids alcohols and lipids"
PRODUCT=methyl coenzyme M reductase system, component A2
homolog"sensory transduction histidine kinase
Carbohydrate Metabolism,
Pyruvate and acetyl-CoA metabolism phosphoenolpyruvate synthase homolog
"Function Code:10.09 - Metabolism of Macromolecules,
DNA replication--modification--repair--and recombination "product="DNA helicase II, pyruvate dehydrogenase / acetolactate synthase"
"Function Code:12.04 - Cell Processes, Transport of anions"
/product="sulfate transport system ATP-binding"
"Function Code:"sulfate transport system permease protein"Energy Metabolism, Oxidative
PRODUCT="H+-transporting ATPase"
"Function Code:10.02 - Metabolism of Macromolecules,Transcription--mRNA synthesis and modification (includes
PRODUCT="DNA helicase related "
FUNCTION: Metabolism of Macromolecules Aminoacyl tRNA synthetases and tRNA modification
PRODUCT="ATP-dependent RNA helicase, eIF-4A family"
"Function Code:10.11 - Metabolism of Macromolecules,
DNA degradation--restriction/modification"
PRODUCT="thermonuclease precursor"
"Function Code:10.11 - Metabolism of Macromolecules,
DNA degradation--restriction/modification"
"product="modification methyltransferase,
"Metabolism of Macromolecules,
DNA degradation--restriction/modification"
product="endonuclease III homolog"
"Function Code:10.11 - Metabolism of Macromolecules,
DNA degradation--restriction/modification "
product="5-methylcytosine-specific restriction enzyme
McrB related protein"
"catalyzes the reduction of
methenyltetrahydromethanopterin to
product="H(2)-dependent methylenetetrahydromethanopterinn dehydrogenase"
"Function Code:12.05 - Cell Processes, Transport of cations"
product="potassium channel related protein"
"Function Code:10.09 - Metabolism of Macromolecules,
DNA replication--modification--repair--and recombination "
product-"DNA helicase II"
"Function Code:9.10 - Metabolism of Cofactors and
Vitamins, Porphyrin and chlorophyll metabolism"
product="cobalamin biosynthesis protein N"
"Function Code:11.04 - Cell envelope, Murein
sacculus and peptidoglycan "
product="UDP-N-acetylmuramyl tripeptide synthetase
related protein"
"this stereospecific enzymes reducesthe S isomer of
methionine sulfoxidewhile MsrB reduces the R form;provides protection against oxidative stress"
product="methionine sulfoxide reductase A"
"Function Code:2.08 - Energy Metabolism, Electron
transport ; similar to, pir:LN:JC4919, p()=2.7E-67,
product="2-oxoacid:ferredoxin oxidoreductase, alpha
"Catalyzes the two-electron reduction of the C2 and
C3(1) diene system of 15,16-dihydrobiliverdin"
product="ferrodoxin oxidoreductase beta subunit"
"Function Code:13.07 - Other, Unclassified "
product="intracellular protein transport protein"
"Function Code:10.09 - Metabolism of Macromolecules,
DNA replication--modification--repair--and recombination"
/product="Rad32 related protein"
"Function Code:12.01 - Cell Processes, Transport of
amino acids--peptides and amines "
/product="cationic amino acid transporter related protein"
"Function Code:12.12 - Cell Processes, Broad regulatory functions "
/product="sensory transduction regulatory protein"
"Function Code:10.09 - Metabolism of Macromolecules,
DNA replication--modification--repair--and recombination"
/product="DNA-dependent DNA polymerase family X"
"Function Code:10.09 - Metabolism of Macromolecules,
DNA replication--modification--repair--and recombination ;
/product="DNA helicase II related protein"
"Function Code:14.01 - Unknown, Conserved protein"
/product="PcrB-like protein"
"Function Code:10.04 - Metabolism of Macromolecules,
Ribosomal proteins"
/product="ribosomal protein L40"
"Function Code:9.10 - Metabolism of Cofactors and
Vitamins, Porphyrin and chlorophyll metabolism"
/product="magnesium chelatase subunit"
"Function Code:9.10 - Metabolism of Cofactors and
Vitamins, Porphyrin and chlorophyll metabolism"
/product="magnesium chelatase subunit ChlI"
Function Code:3.04 - Lipid Metabolism, Sterol
biosynthesis "
/product="3-hydroxy-3-methylglutaryl CoA reductase"
"Catalyzes the only substrate-level phosphorylation
in the TCA cycle"
/product="succinyl-CoA synthetase alpha subunit"
"Function Code:5.14 - L-Amino Acid Metabolism,
Phenylalanine--tyrosine and tryptophan biosynthesis ;
/product="3-dehydroquinate dehydratase"
"Function Code:12.06 - Cell Processes, Chaperones"
/product="heat shock protein X"
"Function Code:2.06 - Energy Metabolism, Nitrogen
metabolism "
/product="nitrogenase reductase related protein"
"Function Code:11.02 - Cell envelope, Surface
polysaccharides and lipopolysaccharides "
/product="polysaccharide biosynthesis protein"
"catalyzes the reversible formation of fructose
1,6-bisphosphate from glycerone phosphate and
D-glyceraldehyde 3-phosphate"
/product="fructose-bisphosphate aldolase"
"catalyzes the formation of 3-dehydroquinate from
3-deoxy-aribino-heptulonate 7-phosphate"
/product="3-dehydroquinate synthase"
"Function Code:10.07 - Metabolism of Macromolecules,
Aminoacyl tRNA synthetases and tRNA modification "
/product="tRNA nucleotidyltransferase"
"DNA primase is the polymerase that synthesizes
small RNA primers for the Okazaki fragments on both
template strands at replication forks during chromosomal
DNA synthesis"
product="DNA primase small subunit"
"DNA primase is the polymerase that synthesizes
small RNA primers for the Okazaki fragments on both
template strands at replication forks during chromosomal
DNA synthesis"
/product="DNA primase large subunit"
"Function Code:10.07 - Metabolism of Macromolecules,
Aminoacyl tRNA synthetases and tRNA modification"
/product="methionyl-tRNA synthetase"
"Function Code:11.02 - Cell envelope, Surface
polysaccharides and lipopolysaccharides "
/product="N-acetylglucosamine-1-phosphate transferase"
Function Code:14.01 - Unknown, Conserved protein"
/product="conserved protein (contains ferredoxin domain)"
Function Code:9.10 - Metabolism of Cofactors and
Vitamins, Porphyrin and chlorophyll metabolism "
/product="precorrin-3 methylase"
"Function Code:13.08 - Other, Adhesion "
/product="adhesion protein"
"Function Code:12.02 - Cell Processes, Transport of
carbohydrates organic acids alcohols and lipids"
/product="ABC transporter"
Function Code:12.05 - Cell Processes, Transport of
/product="manganese transport system membrane protein"
"Function Code:1.03 - Carbohydrate Metabolism,
Pentose phosphate cycle "
/product="ribose 5-phosphate isomerase"
"catalyzes the reduction ofglycerol-1-phosphate to
/product="glycerol-1-phosphate dehydrogenase"
"catalyzes the formation of prolyl-tRNA(Pro) from
proline and tRNA(Pro)"
"prolyl-tRNA synthetase"
"Function Code:10.02 - Metabolism of Macromolecules,
Transcription--mRNA synthesis and modification "
/product="transcriptional regulator""catalyzes the formation of N6-(1,2,-dicarboxyethyl)-AMP from L-aspartate, inosine
monophosphate and GTP in AMP biosynthesis"
/product="adenylosuccinate synthetase"
"Function Code:10.11 - Metabolism of Macromolecules,
DNA degradation--restriction/modification "
/product="O6-methylguanidine-DNA methyltransferase"
"Function Code:12.12 - Cell Processes, Broad
regulatory functions "
/product="sensory transduction histidine kinase"
Amino acid transport in taxonomically diverse cyanobacteria and
identification of two genes encoding elements of a neutral amino acid
permease putatively involved in recapture of leaked hydrophobic amino
Mitochondrial DNA sequence analysis of the cytochrome oxidase subunit I and II genes, the ATPase9 gene, the NADH dehydrogenase ND4L and ND5 gene complex, and the glutaminyl, methionyl and arginyl tRNA genes from Trichophyton rubrum.
http://www.kazusa.or.jp/cyano/Synechocy ... BC16A10BA3
CyanoBase/Synechocystis: Gene Category List
de Bievre C, Dujon B.
http://www.ncbi.nlm.nih.gov/Taxonomy/Br ... e=1&unlock
http://www.ncbi.nlm.nih.gov/COG/old/pal ... 811COG0811
http://www.ncbi.nlm.nih.gov/Structure/c ... ?uid=31153
Here is more interesting information:
http://pubs.acs.org/subscribe/journals/ ... roblems%22
Tam Tam,
You make it sound so easy. First of all you need more convincing evidence that this is what we are infected with. Where should we go to get tested and analyzed for this modified bio crap? Solid proof. Yes you have a video, and yes you could be expert witness I assume. However, what doctor or scientist will even risk his career or even give up their time to evaluate us? Is Wymore on your side? Does he believe in your findings? Who do you recommend we get to help us. I am just one person. I am struggling to even get any kind of diagnosis from a physician. I am a lost sole. Just what the government wants me to be. May I personally ask why you have not sought litigation? I have many more unanswered questions. PLEASE HELP.
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- Admissions
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- About
An annual conference that brings together students, alumni, faculty, staff, stakeholders and industry experts to continue the exchange of substantive public administration principles and best practices.
Designed to further thought and discussion on matters relevant to leadership in the public sector, the Key Executive Leadership Speaker Series provides an opportunity for DPAP students and Key Executive Leadership Alumni to hear from senior government leaders in an interactive and dynamic environment.
This special networking forum creates an environment where federal executives can explore and discuss solutions to common challenges that can impede the public policy implementation process and hinder the success of a strong workforce.


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To understand finance professionals' feelings about financial risk management, as well as their thoughts on the economy overall, TD Bank polled CFOs, controllers, treasurers, and directors of finance from across the United States. Respondents come from both midmarket companies, defined by TD Bank as being under $250 million in annual revenue, and "corporates," defined as organizations with revenues above $250 million.
The survey revealed that these executives' confidence in the U.S. economy is increasing—and their confidence in their own organization is growing even faster. The largest proportion (41 percent) said they are more confident about the U.S. economic outlook over the next 12 months than they were over the previous 12 months. Thirty-one percent said they're less confident today, and 29 percent said their confidence level hasn't changed in the past year.
But when asked how they feel about the prospects for their organization over the next 12 months, vs. the previous year, a full 60 percent said they're more confident, and nearly half of those (26 percent of all respondents) said they're much more confident today. In contrast, only 11 percent of respondents are less confident about the next year than the past year, and only 3 percent are much less confident. Fred Graziano, head of regional commercial banking and executive vice president with TD Bank, says these numbers are up from the bank's 2012 survey: "The difference from a year ago is that companies are feeling a little bit better about themselves and how they manage their business."
Indeed, many of the organizations represented in the survey have made substantial changes to their risk management policies in the past several years. Half have gained more visibility into their cash position since the start of the financial crisis, and nearly as many have begun evaluating suppliers' risk management practices and terminating business relationships that carry too much risk (see Figure 1, below). More than a third have improved their risk reporting practices, and more than one in five have implemented an enterprise risk management (ERM) program since 2008.
However, most organizations did not respond to the financial risks brought to light in the financial crisis by making significant management or technology changes. Only 11 percent have deployed new risk management software, and fewer than 5 percent have established a chief risk officer (CRO) position.
"Companies have made internal changes, such as adding people and improving reporting," Graziano says, "and then externally they've gone out and done a better job of evaluating their vendors and suppliers. By adding more resources to risk management, they've positioned themselves better, both internally and externally."
Although they've gained important insights into their financial risks, most companies' willingness to take on financial risks hasn't changed much in the past year. When asked how they would rate their organization's current financial risk appetite compared with a year earlier, the majority (56 percent) said it hasn't changed. Nearly a quarter (24 percent) are somewhat more willing to take financial risks this year than they were last year, and 3 percent are much more willing to take risks. However, 10 percent are somewhat less willing—and another 7 percent much less willing—to take financial risks today.
Respondents made clear the reasons why they're reluctant to take on new risks: They feel a good deal of uncertainty in the external environment, both in terms of regulations that may be coming in the future and in terms of the economy and their competitiveness in their marketplace (see Figure 2, below). "In 2008 and 2009, the big concern among CFOs was their ability to secure credit," Graziano says. "This year, that concern is near the bottom of the list. Instead, CFOs today are thinking about things like understanding financial risk, regulatory risk, and cash positions."
Indeed, when asked to identify the risk that concerns them most, more than a third of survey respondents chose "regulatory changes/uncertainty." Nearly 20 percent cited economic uncertainty, and 15 percent cited competitive pressures. Fewer than 1 percent said that securing credit is their primary concern, with 2 percent saying they're most worried about the rising cost of credit.
Perhaps because external uncertainties are their number-one concern, more than half of the surveyed finance executives said that their ability to forecast future financial risk is an ongoing challenge. Many also struggle to control their current financial risks, in large part because they feel they don't have the right data to manage those risks.
Corporate policies are creating some challenges, as well. Twenty percent of respondents said they're operating without a clear financial risk management strategy. And similar proportions of CFOs grapple with their company's conservative risk appetite (16 percent) as with their organization's aggressive risk appetite (12 percent).
"When you start taking on credit, and you want to invest in your company—whether it's people, buildings, equipment, or other fixed assets—you want to have the ability to forecast, and you want to feel comfortable that there's some stability, before you make that long-term investment," Graziano says. "I think what this survey emphasizes is that companies are forecasting a little better, but there's still a lot of uncertainty about the future. CFOs would like to face less uncertainty, but they're finding ways to manage those external risks."


text:
Automotive safety: An ISO 26262 perspective
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Safety has been a key aspect in the automotive industry even from its earliest stages, but the importance with which it is regarded has become far greater in recent times. Currently the biggest compound annual growth rate (CAGR) in automotive electronics revenue can be attributed to safety applications. Increasingly car manufacturers are making safety a key selling point with which to differentiate themselves from their competition. But with a growing amount of electronics content making up a car's bill of materials, there is now a necessity to switch from the long-established best practices approach to well-defined universal guidelines. As a result, industry protagonists have joined forces to develop a standard with far-reaching implications.
The word "safety" is subject to various different interpretations. However, when applied to modern automobile design it can generally be categorized using the following structure:
1. Passive safety: Assuming that an accident is effectively inevitable, the aim of passive safety mechanisms is to minimize the severity of that accident. The passive safety elements found within a vehicle include seatbelts, crumple zones, etc.
2. Active safety: The systems that are concerned with active safety (based on the knowledge of the current state of the vehicle) will aim to avoid accidents altogether in addition to the minimization of its effects if an accident occurs. Seatbelt pre-tensioning, airbag deployment, predictive emergency braking, anti-lock braking systems and traction control are all examples of this.
3. Functional safety: This focuses on ensuring that all of the electrical and electronic systems (such as power supplies, sensors, communication networks, actuators, etc), including (but not limited to) all active safety related systems, function correctly. Functional safety is dealt with by the ISO-26262 standard (published in November 2011).
It is important to state from the beginning that functional safety does not mean that there is no risk of a malfunction taking place -- instead, functional safety implies the absence of unacceptable risk due to hazards caused by malfunctioning behavior of electrical and electronic systems.


text: Kennel and pet boarding facilities are available to help dog and cat owners care for their furry friends while they are away. Daycare is common for short term care, while boarding is for a longer period of time. Whether you are heading out on vacation or need the pet watched for a while, there are many boarding houses as well as hotels and training camps for canines and cats. Many of these resorts offer cage-free kennels, but all boarding facilities are different. Sitting for your pets takes quite a bit of work. At such resorts they often groom, walk, train and exercise your pet. Necessities should always be provided for, such food and water, as well as transportation – though puppy day care and other pet sitters may not offer taxi service to travel with your animal. You may require more information before allowing your dog to reside in someone's house or kennel.


text:
Xtra Points: Don't pin Packer loss on officials
San Francisco 49ers head coach Jim Harbaugh overflows with bravado.
He paces the sideline during games with his heart on his sleeve, often looking for an official to berate. Every postgame handshake with an opposing coach turns into a showdown, sometimes ending with Harbaugh's counterpart chasing after him for a fight—though it should be said that Detroit's Jim Schwartz has his own brand of crazy.
Pertinent to what happened Sunday, another part of that bravado is Harbaugh's propensity to make unorthodox decisions most NFL coaches wouldn't even consider.
Let's get to the critical point in Sunday's game. You might remember Clay Matthews pouncing on Colin Kaepernick after the 49er quarterback already stepped out of bounds short of the first-down marker on 3rd and 6. The penalty for the late hit was flagged, which would've resulted in an automatic first down, but offensive lineman Joe Staley grabbed Matthews, voiced his displeasure with the hit and got flagged himself for unsportsmanlike conduct.
It was announced that the fouls offset and that no yardage penalty would be assessed either way. The teams replayed third down, and the 49ers scored on a touchdown pass from Kaepernick to the unstoppable Anquan Boldin.
Except, as everyone knows now, it shouldn't have been third down again. Since the fouls were after the conclusion of the original third-down play, the play should've counted and the next down should've been 4th and 2.
Conventional wisdom dictates a field goal try in such a situation, but as we know, there's nothing conventional about the way Harbaugh coaches a football game.
Who can say that Harbaugh, encouraged by the roaring crowd that just watched its team get into it with the opponents, wouldn't have thought, "You know what, we can pick this up!" And who's to say the 49ers wouldn't have gotten that first down and eventually scored that touchdown anyway?
Before Packer fans get to blaming the officiating, I would suggest looking first at the 400 yards passing Kaepernick put up and then at the two turnovers Green Bay committed, especially Eddie Lacy's fumble that led to another 49er touchdown.
Yes, it's unfortunate this group of referees made this mistake, but there were plenty of things the Packers could've done before and after that point that could've helped them win the game.
Alas, on to Washington.


text:
Austin Town Hall Park
5610 W. Lake St. Chicago, Illinois 60644
(773) 287-7658
Fee: $5
Age Range: All ages
Austin Town Hall will host Theatre in the Parks. Come and enjoy the talents of the performers in this 3 day event. Performances are from 6p.m.-8p.m. each night on February 21,22, and 23. Fee $5 per viewing. All ages welcome.


text:
Death notices for Sept. 30, 2013
Larry M. Brotzman, 64, Janesville, died Friday at St. Mary's Janesville Hospital. Arrangements are pending at Whitcomb-Lynch Funeral Home & Cremation Services, Janesville.
Amy E. Harriman, 42, Beloit, died Friday at Mercy Hospital and Trauma Center, Janesville. Services will be at 4 p.m. Wednesday at Rosman, Uehling, Kinzer Funeral Home, Beloit. Visitation will be from 2 p.m. until the time of services Wednesday at the church.
Margaret Hannah Howe, 82, Beloit, died Saturday at University Hospital, Madison. Services will be at 6 p.m. Wednesday at Daley Murphy Wisch & Associates Funeral Home and Crematorium, Beloit. Visitation will be from 3 p.m. until the time of services Wednesday at the funeral home.
Esther Marie Krueger, 92, Watertown, died Friday at Marquardt Manor, Watertown. Services will be at 11 a.m. Tuesday at the chapel at Derrick Funeral Home, Lake Geneva. Visitation will be from 4 to 8 p.m. Monday at the funeral home.
Dawn Renee Norton, 42, Walworth, died Sunday at Aurora Lakeland Medical Center, Elkhorn. Services will be at 7 p.m. Thursday at Haase-Lockwood & Associates Funeral Home, Elkhorn. Visitation will be from 5 p.m. until time of services Thursday at the funeral home.
Rena Teubert, 94, Edgerton, died Saturday at Skaalen Nursing & Rehab, Stoughton. Services will be at 11 a.m. Thursday at St. John Lutheran Church, Edgerton. Visitation will be from 10 a.m. until the time of services Thursday at the church. Albrecht Funeral Homes & Cremation Services, Edgerton, is assisting the family.