text: Matt Zimmerman wrote: > On Tue, Aug 02, 2005 at 08:15:22PM +0100, antgel wrote: > >>Matt Zimmerman wrote: >> >>>the issue is that they often don't apply to versions which are a few >>>months old. >> >>Not automatically, but perhaps if we had a dedicated team of a few people >>who can code, we could manually mould them to the version in stable? > > > Have you been following this discussion? That is exactly what we have been > killing ourselves doing for the past few years. It is a _losing battle_. I've been following a fair bit of the discussion, but it's hard to pull the facts out from the opinion.. I'm not belittling the Debian team efforts, and I'm sorry if I seemed like I was. If it is a losing battle, then it's one that we should try to equip ourselves[1] to win. If you are saying that we can't equip ourselves then fine, but it's a shame. We are on the same side here. Antony [1] This includes more manpower and liaising with Mozilla to see if they can help more than they are doing.


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MANCHESTER UNITED are monitoring the progress of hotshot Celtic striker Moussa Dembele.
A talent spotter from the Red Devils was deployed to Ross County on Wednesday to run the rule over the young Frenchman.
And the 20-year-old did not disappoint as he grabbed a late goal during the Hoops' 4-0 victory – despite starting on the bench.
Keep up to date with ALL the Manchester City news, gossip, transfers and goals on our club page plus fixtures, results and live match commentary
The former Fulham frontman has bagged 15 goals so far this season, including the winner in Sunday's Old Firm victory over bitter rivals Rangers.
United scout Stephen McCarthy was in attendance at the Global Energy Stadium to watch the rising star.
related stories
The youngster, who also scored twice against Manchester City in the Champions League, is one of the hottest properties in Europe following his explosive start to life at Parkhead.
He only joined the club last summer following his emergence through the academy and into the first-team at Fulham.
But it appears his stay in Glasgow will only be short-lived if his exploits in front of goal continue as his list of admirers grows by the day.

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Classically known for its indispensible role in embryonic development, the Notch signalling pathway is gaining recognition for its regulation of adult tissue homoeostasis and aberrant activation in disease pathogenesis. The pathway has been implicated in cancer initiation and development, as well as early stages of cancer progression by regulating conserved cellular programs such as the epithelial-to-mesenchymal transition. We recently extended the role of Notch signalling to late stages of tumour progression by elucidating a stroma-dependent mechanism for the pathway in osteolytic bone metastasis. Of clinical significance, disrupting the Notch pathway and associated molecular mediators of Notch-dependent bone metastasis may provide novel therapeutic strategies to combat aggressive bone metastatic disease.
The Notch pathway regulates cell fate decisions during embryonic development by facilitating short-range signalling between neighbouring cells that are in physical contact. Initially discovered for yielding a 'notched' wing phenotype in Drosophila due to a partial loss of function, the pathway has since been implicated in specifying the development of several different tissues and organisms (Artavanis-Tsakonas et al, 1999). In a context-dependent manner, Notch signalling coordinates a wide range of fundamental processes and cellular programs including proliferation, apoptosis, migration, growth, and differentiation. The pathway has only recently been associated with the maintenance of adult tissue and pathogenesis of cancer.
The four mammalian Notch receptors are single-pass type-1 transmembrane proteins that are expressed on the cell surface (Logeat et al, 1998). The extracellular domain contains epidermal growth factor (EGF)-like repeats that are responsible for ligand binding and a negative regulatory region that prevents receptor activation in the absence of ligands. The intracellular domain of Notch receptors contain three common regions: (1) a subtransmembrane region that associates with transcriptional components in the nucleus (Honjo, 1996), (2) six tandem ankyrin repeats that are necessary for transcriptional activity, and (3) a PEST sequence that regulates stability and protein turnover (Greenwald, 1994; Bray, 2006). The receptors are activated via interactions with Notch ligands, which are also type-I transmembrane proteins with multiple EGF-like repeats in their extracellular domain (D'Souza et al, 2008). The five mammalian Notch ligands are separated into two subgroups: Delta-like (Dll1, Dll3, and Dll4) or Serrate-like (Jagged1 and Jagged2), based on the structural similarity with their Drosophila homologues (Bray, 2006). Ultimately, the diverse functions affected by the Notch pathway are dependent on the signalling interaction between neighbouring cells (Sprinzak et al, 2010).
The Notch pathway is activated when a signal-sending cell expressing a membrane-bound ligand physically interacts with a signal-receiving cell expressing a Notch receptor (Gray et al, 1999). Upon ligand binding, the Notch receptor is cleaved twice, first by an extracellular matrix metalloprotease (Brou et al, 2000) and then by the transmembrane protease complex γ-secretase, releasing the Notch intracellular domain (Schroeter et al, 1998; Mumm et al, 2000; Six et al, 2003) (Figure 1A). After dissociating from the cell membrane, the Notch intracellular domain translocates to the nucleus where it interacts with the DNA-binding protein CSL (Rbp-Jκ in mice; CBF1 in humans) to affect transcriptional responses (Jarriault et al, 1995; Wu et al, 2000). In a recent example, genomic analysis using Chip–chip arrays demonstrated that the promoter region of Notch1-regulated genes were bound by CSL, which helped reveal several new CSL-dependent targets of Notch signalling (Hamidi et al, 2011). The most prominent targets of the Notch pathway include a set of basic helix–loop–helix factors of the hairy and enhancer of split (Hes) and Hes-related repressor protein (Hey) families (Iso et al, 2003; Kopan and Ilagan, 2009). These transcription factors execute Notch signalling functions, including maintenance of stem cells, specification of cell fate, differentiation, proliferation, and apoptosis (Radtke and Raj, 2003). Although many of the essential pathway components have been identified, we are still uncovering new regulators of Notch signalling using fundamental developmental and molecular biology (Wang et al, 2008; Karaczyn et al, 2010; Sethi et al, 2010; Dalton et al, 2011).
An oncogenic role for Notch was first demonstrated in T-cell acute lymphoblastic leukaemia (T-ALL) with the identification of a chromosomal translocation that resulted in the expression of a truncated, constitutively active form of the Notch1 protein in T-cells (Ellisen et al, 1991). Experimental work using transgenic mouse models revealed that constitutive activity of the Notch pathway in hematopoietic progenitors drives the specification of an immature T-cell lineage, which then evolves into a highly aggressive monoclonal T-cell leukaemia with the cooperation of additional mutations (Pear et al, 1996; Radtke et al, 1999). Further clinical evaluation revealed activating mutations of Notch1 in more than 50% of patient cases (Weng et al, 2004). The characterisation of aberrant Notch signalling in T-cell leukaemia provided a foundation for researchers to explore the pathway's importance in other cancers (Table 1).
The Notch pathway has been implicated in the tumourigenesis of several solid tumour malignancies including, but not limited to, non-small cell lung adenocarcinoma (Dang et al, 2000), melanoma (Bedogni et al, 2008), ovarian carcinoma (Park et al, 2006), medulloblastoma (Hallahan et al, 2004), and Kaposi's sarcoma (Curry et al, 2005). The pathway has also been shown to contribute to the pathogenesis of breast cancer. Constitutive activation of the Notch pathway in mammary tissue leads to the development of breast cancer in distinct mouse models. These studies employed the mouse mammary tumour virus (MMTV) system, which led to the identification of the Notch1 and Notch4 gene loci as frequent viral insertion sites that consequently resulted in the development of breast adenocarcinoma (Gallahan and Callahan, 1987). Similar to the mechanism in T-ALL, the MMTV insertion resulted in the constitutive expression of an active truncated form of the Notch receptor, revealing an oncogenic role for Notch signalling in mammary tissue (Jhappan et al, 1992; Dievart et al, 1999). Several studies have since shown that constitutive activation of the pathway confers tumourigenic properties to mammary epithelial cells (Raafat et al, 2004; Zang et al, 2007). However, unlike T-ALL, it is ambiguous as to how the pathway is aberrantly activated in human solid tumour malignancy, as underlying genomic mutations are rarely found in patient samples. Considering the lack of evidence showing genetic mutations in Notch, there should be greater emphasis on uncovering the source of Notch signalling activation in solid tumours. One avenue of exploration is characterising the potential heterogeneity of ligand and receptor expression within the primary tumour. Along this line of reasoning, a possible mechanism would involve a subset of primary tumour cells expressing Notch ligands, which have been shown to be aberrantly induced by local microenvironment paracine signals (Zeng et al, 2005), may activate the Notch pathway in a different subset of primary tumour, or signal-receiving, cells. Such a scenario would parallel Notch signalling events that take place during development when executing cell fate decisions (e.g. lateral specification) (Heitzler and Simpson, 1991; Greenwald, 1998; Sassoli et al, 2011).
Beyond its role in tumourigenesis, Notch signalling has also been associated with cancer progression, particularly through its regulation of the epithelial-to-mesenchymal transition (EMT). Epithelial-to-mesenchymal transition is a conserved cellular programme that confers mesenchymal features to epithelial cells during development and is postulated to provide epithelial tumour cells with migratory properties, facilitating early steps in metastasis such as invasion. The Notch pathway regulates EMT with the contribution of several players and processes, such as TGFβ signalling (Timmerman et al, 2004; Zavadil et al, 2004), β-catenin activity (Balint et al, 2005), the transcription factor Slug (Leong et al, 2007), and most recently hypoxia (Bedogni et al, 2008; Sahlgren et al, 2008). Notch signalling has also shown to directly regulate mediators of invasion, such as matrix metalloproteinase-9 and vascular endothelial growth factor (Wang et al, 2006). Interestingly, the Notch ligand Jagged1 is also associated with cancer progression; analysis of clinical samples revealed that it is highly expressed in metastatic prostate cancer compared with localised disease (Santagata et al, 2004) and overexpressed in breast cancer patients with poor prognosis (Reedijk et al, 2005). Despite these associations, until recently, the functional mechanism of Notch signalling in breast cancer metastasis was poorly defined.
Corroborating previously published data, we found that elevated expression of Jagged1 was associated with an increased incidence of breast cancer relapse. These findings were extended by interrogating a separate clinical data set with more diverse outcome measures such as organ-specific metastasis, which showed that elevated Jagged1-expression levels was associated with bone metastasis. Functional studies in mice demonstrated that Jagged1-expressing breast cancer cells promote bone metastasis by activating the Notch signalling pathway in the bone microenvironment (Sethi et al, 2011) (Figure 1B). These findings suggest a new paradigm for Notch signalling in breast cancer progression, defining a requirement for the pathway in the bone stroma as opposed to tumour cells during the formation of bone metastasis. Interestingly, a role for the Notch pathway in the tumour-associated stroma has been shown to facilitate other cancer-promoting functions, such as angiogenesis. In head and neck squamous cell carcinoma, cancer-mediated activation of the pathway in endothelial cells supported tumour neovascularisation and angiogenesis (Zeng et al, 2005). Similarly, Delta-mediated Notch signalling was shown to facilitate tumour angiogenesis by maintaining the sprouting integrity of the developing tumour vasculature (Noguera-Troise et al, 2006).
In the bone microenvironment, tumour-derived Jagged1 engaged the Notch pathway in two distinct cell types: osteoblasts and osteoclasts. Jagged1-mediated activation of the pathway in osteoblasts conferred a growth advantage to bone metastatic tumour cells. Mechanistic studies revealed that the proliferative gain was dependent on osteoblast-secreted IL-6, which was transcriptionally regulated by the Notch pathway and its downstream target Hey1 (Sethi et al, 2011). Independent studies have shown that IL-6 is associated with a poor prognosis in breast cancer (Zhang and Adachi, 1999; Salgado et al, 2003) and is capable of supporting tumour growth in stromal-dependent mechanisms (Sasser et al, 2007; Studebaker et al, 2008; Ara et al, 2009). In neuroblastoma (Ara et al, 2009) and multiple myeloma (Mitsiades et al, 2006), IL-6 derived from stromal cells was shown to be an important mediator between cancer cells and the bone microenvironment by supporting tumour survival and affecting osteoclast differentiation, respectively. Of note, activation of other developmental pathways in osteoblasts has also been shown to promote bone metastasis (Sethi and Kang, 2011). Paracrine Sonic Hedgehog signalling by prostate cancer cells was shown to promote bone metastasis by inducing osteoblast differentiation through a Gli1-dependent mechanism (Zunich et al, 2009).
Jagged1-expressing tumour cells demonstrated a severe osteolytic phenotype in mice, suggesting a potential effect of tumour-derived Jagged1 on osteoclastogenesis (Sethi et al, 2011). Coculture experiments between Jagged1-expressing tumour cells and primary bone marrow cells demonstrated a strong induction of osteoclastogenesis. Often, tumour cells affect osteoclast maturation indirectly through osteoblast-dependent regulation of RANKL and OPG signalling (Lu et al, 2009); however, our results supported a distinct mechanism as Jagged1-expressing tumour cells directly associated with pre-osteoclasts to promote their maturation. These findings were supported by the direct application of recombinant Jagged1 protein to pre-osteoclasts, which demonstrated a strong upregulation of osteoclast maturation (Sethi et al, 2011). Transcriptional profiling of osteoclast-differentiation markers more definitively confirmed these results. Integrating these findings, it appears that tumour-derived Jagged1 directly engages Notch pathway in pre-osteoclasts, promoting their differentiation into mature, multinucleated osteoclasts, a mechanism that can help explain the severe osteolytic phenotype observed in Jagged1-mediated bone metastasis.
When metastatic cancer cells spread to the bone, the influence of the microenvironment and resultant adaptations undertaken by tumour cells alter them in ways that render them resistant to cell-autonomous therapies that would otherwise effectively treat their corresponding primary tumour (Emmenegger and Kerbel, 2010; Karlou et al, 2010). Experimental mouse models have also shown the therapeutic inadequacies of targeted agents in treating metastatic lesions (Man et al, 2002; Francia et al, 2009). These observations collectively support the rational for targeting the microenvironment of the metastatic lesion, in conjunction with the tumour cells directly, to better control disease that has spread to distant organs. The molecular mechanisms underlying Jagged1-mediated bone metastasis suggested that targeting the Notch pathway in the bone microenvironment might prove as an effective strategy in the treatment of bone metastasis. Currently, γ-secretase inhibitors (GSI) are small molecules that can potently disrupt the Notch pathway; they are also gaining popularity as anticancer agents based on preclinical studies (Shih Ie and Wang, 2007). Combining these pieces of information, we decided to test whether administration of GSI could interrupt communication between Jagged1-expressing tumour cells and the bone microenvironment in an attempt to treat breast cancer bone metastasis. GSI therapy reversed the bone metastasis-promoting functions of Jagged1 by disrupting the Notch pathway in bone stromal cells. Gene-expression analysis of bone metastases demonstrated that GSI-treated mice displayed a downregulation of Notch target genes in the tumour-associated stromal compartment, including a decrease in IL-6 levels. Overall, the results revealed a stromal-dependent mechanism for Notch signalling in supporting tumour outgrowth in the bone and suggest that targeting the pathway in the tumour-associated stroma may improve treatment for breast cancer bone metastasis (Sethi et al, 2011).
Importantly, other mediators and regulators contributed to Notch signalling in bone metastasis and therefore represent potential targets for therapeutic intervention. Jagged1 was shown to be the prime mediator of Notch-dependent bone metastasis and would be an ideal target for monoclonal antibody therapy as it is a cell-surface protein. Monoclonal antibodies targeting other Notch pathway ligands, such as Delta-like 4, have been shown to disrupt angiogenesis and tumour growth, demonstrating the potential therapeutic advantage of targeting Notch ligands (Noguera-Troise et al, 2006; Ridgway et al, 2006). The cytokine IL-6 is released by osteoblasts in response to Jagged1-mediated bone metastasis and a potential therapeutic target. Tocilizumab is a humanised monoclonal antibody against the interleukin-6 receptor (IL-6R) that has been approved to be used as an immunosuppressive drug in the treatment of rheumatoid arthritis. Additional inhibitors of IL-6 and its downstream Jak-Stat pathways are also under active clinical development.
Of pathological significance, the TGF-β pathway was shown to regulate Jagged1 expression during breast cancer bone metastasis. TGF-β is sequestered in the bone matrix and often times released in response to bone degradation, a process that is largely at play during osteolytic bone metastasis (Korpal et al, 2009). TGF-β was established as a critical regulator of Jagged1-mediated bone metastasis. Enforced expression of Jagged1 in SMAD4-knockdown breast cancer cells, which are severely impaired in their ability to form productive osteolytic bone metastases due to the defective reception of microenvironment TGF-β cues (Kang et al, 2003, 2005), restored the bone metastasis-promoting functions of these malignant cells. The TGF-β pathway has also been targeted by therapeutic agents currently being tested in clinical trials (Korpal and Kang, 2010) and would be a candidate pathway to disrupt in combination with Notch signalling in the treatment of breast cancer bone metastasis.
As we appreciate the therapeutic potential of molecular mediators that support communication between tumour cells and the bone microenvironment in the generation of bone metastasis, it will be critical to test the efficacy of these agents in the appropriate patient population. With respect to Notch signalling in bone metastasis, there is preliminary data that link Jagged1 and IL-6 expression to the basal-like subtype of breast cancer (Sansone et al, 2007; Sethi et al, 2011, unpublished observation). It is therefore imperative to design robust clinical trials, evaluating these agents in select patient groups that are most likely to benefit from the particular therapeutic intervention. These agents should be administered either alone or in combination with other inhibitors targeting the cross-talk between tumour and bone stromal cells in the treatment of bone metastasis.
PubMed Central Canada is a service of the Canadian Institutes of Health Research (CIHR) working in partnership with the National Research Council's national science library in cooperation with the National Center for Biotechnology Information at the U.S. National Library of Medicine(NCBI/NLM). It includes content provided to the PubMed Central International archive by participating publishers.


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Nayo Thomas
Coordinator of Operations & Special Events
Nayo serves as Coordinator of Operations and Special Events with the Global Learning Network. Prior to joining the America Achieves team, she worked for the United States Air Force at Buckley Air Force Base and at the Department of Veterans Affairs in Denver, Colorado. She then continued her work for nonprofits, working for the National Association of Black Cardiologists and the Henry Stimson Center for Nuclear Nonproliferation and Disarmament in Washington, DC. Nayo attended Howard University in Washington DC, where she double majored in Political Science and Business. During college, Nayo fell in love with education after she interned with Jumpstart and Head Start as a team leader. Interacting with parents and teachers, as well as teaching in the classroom with the prekindergarten and preschool age students, helped her revisit the importance of public education. In her spare time, Nayo loves skiing and dearly misses the mountains of her home state of Colorado.
What draws you to the work of America Achieves?
I love the passion that everyone has about the future of public education at America Achieves. I believe working at America Achieves will allow me the chance to grow and evolve and gain a better understanding of education reform.
What do we most need to focus on to improve the lives of America's youth?
To improve the lives of America's youth, we must focus on education and family engagement. If youth do not have a good support system that encourages them to do more and tells them that anything is possible, then it will be harder for youth to be successful in the future.


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Uptown Market is the premier merchant marketplace located in The Porters Neck area and was established in 2004. 12,000 square feet of vintage furnishings, coastal cottage, industrial and primitive furniture makes this a one stop shop destination. There is an endless array of gifts perfect for any occasion. There is also a diverse variety of clothing jewelry and accessories. We also have artisan jewelry and custom made furniture. Farm tables and headboards made out of shutters and doors are one of our specialties.
What makes Uptown Market special?
We have a high turnover of furniture not people. We value our customers and we strive for 100 percent customer satisfaction. We have an in house delivery option for smaller pieces of furniture and an outside company for larger jobs.


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About Us
Olive Fee Fee Auto Care is a family business that has been on the corner of Olive Blvd. and Fee Fee Road for over 35 years. We are a full service auto repair shop with a stable work force covering two generations. Our primary goal is to provide customers with personal contact with their automotive technichian pluss honest and excellent automotive repair service. We are an ASE Certified Shop.


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Bhubaneswar: The Indian cricket team arrived here Monday, ahead of their one-day match against New Zealand to be played at the Barabati Stadium in Cuttack on Thursday.
The team drove to the Mayfair Lagoon Hotel where they would be staying till Friday.
New Zealand are expected to arrive here on Wednesday and will be putting up at the same hotel. The teams will have to drive to Cuttack, about 25 km from here, for match practice.
A large crowd of cricket enthusiasts were present at the airport to welcome the cricketers.
Meanwhile, there was a big rush for tickets, the sale of which started today in Cuttack for the first day-night match to be held in the city.
Many had arrived near the stadium to buy tickets before dawn and police had to resort to a mild lathicharge to restore order. A large number of young boys and girls were seen waiting in the queue braving a persistent drizzle.


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08/09 Beckham #32 Ac Milan Home Soccer Jersey Combo
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WelcomeAlthough I am a new Seller on Ebay, my aim is to keep 100% positive feedback. Please trust my company will offer you the best service and quality products. 0 8/ 09 Beckham #32 Ac Milan Home Combo Players Beckham No.32 Club Ac Milan Italy Size Chest Length M 40 -42" 28"/72 CM XL 44 -46" 30"/77 CM
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※Normally it will take 4-7 Business Days for shipping & delivery. However,since this is an international delivery, shipping time will be highly depended on the courier company and local customs/duties.


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Results 1 to 4 of 4
02-23-09, 13:02 #1Registered User
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- Feb 2009
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Unanswered: Error 3071 due to =Avg([RAW_SCORE]) in Report footers
This expression is typed incorrectly, or it is too complex to be evaluated. For example, a numeric expression may contain too many complicated elements. Try simplifying the expression by assigning parts of the expression to variables. (Error 3071)
The query runs fine ... but the report gives this error. I have determined it is the average of RAW_SCORE that is causing the problem. The report runs fine if I eliminate the Avg function or if I limit the number or records it is reporting on by separating the departments.
The query is as follows:
SELECT [CSR_LN]+", "+[CSR_FN] AS [CSR Name], USERS.first_name AS CSR_FN, USERS.last_name AS CSR_LN, [SUPV_LN]+", "+[SUPV_FN] AS SUPERVISOR, USERS_1.first_name AS SUPV_FN, USERS_1.last_name AS SUPV_LN, USERS_2.last_name AS EVALUATOR, EVALSCORE.[YES] AS YES_SCORE, EVALSCORE.[NO] AS NO_SCORE, EVALSCORE.NA AS NA_SCORE, USERS.active_flag AS ACTIVE, IIf([EVALUATOR_ID] Between 692 And 695,2,IIf([EVALUATOR_ID]=755,2,1)) AS EVAL_type, EVALSCORE.department_id, ([YES_SCORE]/([YES_SCORE]+[NO_SCORE])) AS RAW_SCORE, EVALSCORE.evaluation_date FROM ((EVALSCORE INNER JOIN USERS AS USERS_2 ON EVALSCORE.evaluator_id = USERS_2.user_id) INNER JOIN USERS ON EVALSCORE.csr_id = USERS.user_id) INNER JOIN USERS AS USERS_1 ON EVALSCORE.team_leader_id = USERS_1.user_id WHERE (((EVALSCORE.department_id)=1 Or (EVALSCORE.department_id)=4 Or (EVALSCORE.department_id)=6 Or (EVALSCORE.department_id)=10) AND ((EVALSCORE.evaluation_date) Between [Start Date?] And [End Date?])) ORDER BY EVALSCORE.evaluation_date;
Thank you in advance for your assistance, as our IT department does not support Access reporting.
02-23-09, 13:26 #2Registered User
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Try this perhaps: Check for null values on within your data:
=AVG(RAW_SCORE) might be trying to calculate the average of a subset which includes erroneous or null data;
RAW_SCORE is calculated from other fields:
So check that none of these underlying fields are null.Windows Server 2003-8 / Terminal Services / SQL 2000 / Access 2003 / Office 2003-7 / Exchange 2003-7 / Blackberry Enterprise Server / AutoCAD / Lambert And Butler / Red Bull
02-23-09, 13:29 #3Registered User
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- Feb 2009
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Error 3071 due to =Avg([RAW_SCORE]) in Report footers
Some of this data will be "0". For instance if all criterion are met ... then the no field will be 0 ... or vice versa.
02-23-09, 14:59 #4Registered User
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I have been trying to work around the whole Average thing ... but it seems no matter how I do the math ... whether counting the # of records ... adding the [Raw_Score] field and dividing ... it results in the error. Is there an average function that will not freak out when there are zeroes?


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HARROGATE (WATE) – A Claiborne County man faces charges after a suspicious package was found at the post office in Harrogate.
Shawn David Bailey, 26, is charged with aggravated criminal trespassing and having a hoax explosive device. Federal charges are pending.
The package was found at around 8:15 a.m. Friday by an employee at the Harrogate postal facility, according to Wendy Boles with the U.S. Postal Inspections. She said a hazmat team responded out of an abundance of caution. Knox County Bomb squad also responded.
Claiborne County deputies say electrical wires could be seen coming out of the package. The facility reopened after no explosive materials were found.
Deputies were able to locate Bailey at his home thanks to evidence found at the scene. A motive has not yet been determined.


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We are uniquely capable of advising you based on a 360-degree view of your assets and finances. Not just your investments, but all your assets: properties, any businesses, and even the "human capital" waiting to be developed within your family , as you and your loved ones transition from one phase of life to another. Look for planning-related articles in Navigator, our digital quarterly newsletter.
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Tips on how to protect against online theft and fraud, plus all the ways LNWM guards your financial privacy and security, by LNWM's Director of Technology Bill Frizzell.
One of the biggest reasons people choose to work with LNWM is because we help our clients navigate the big changes in life, be it a significant transition or an overwhelming event, such as the loss of a life partner, which can leave even the most capable people a bit shell-shocked.
Conventional wisdom on retirement savings can lead you down a path of limited options and potentially higher taxes. LNWM Client Advisor Susan Talton discusses how to make deliberate decisions during the three major phases of retirement, resulting in more control and fewer surprises.
Tax reform is likely in 2017. What will this mean for you, your family, your business? A Q&A with Kristi Mathisen, LNWM's Managing Director of Tax and Financial Planning, provides insight into what's being considered and what is likely in the next two years.
"Buy! Sell! Now! With property prices at all-time highs, that's what a lot of people are thinking. But does the excitement come from real opportunity or a false sense of urgency? Key things to consider when buying or selling in a revved up market, from LNWM tax advisor Kristi Mathisen and client advisor Bridget Burgess.
What financial choices, by decade, add up to a significant lifetime advantage? Here's what we think is essential, from LNWM Tax Expert Kristi Mathisen and LNWM Client Advisor Sam Craig, based on decades of experience working with multiple generations within families.
As most Americans prepared for the holidays, Congress got busy, too. Toward the end of 2015, major changes to Social Security, Medicare, and some key tax breaks were approved.
At today's low interest rates, a loan to a family member can make great financial sense. Just as important, it can foster trust, accountability and responsibility. Interested? Then be sure to read Kristi Mathisen's advice about success factors for family loans, as well as avoiding the pitfalls, including gift taxes.
When your goals change or new opportunities arise, we're here to help you make decisions that take into account the full impact on your life and on your finances.
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At Laird Norton Wealth Management, many of our clients are involved in the community and we often advise them on philanthropic planning and the best ways to give. In the aftermath of the market meltdown, one of the most frequent questions that we answer is "how do you retain your ability to be philanthropic and not feel that you are burning your financial candle at both ends?"
Economic downturns are difficult for philanthropies and donors alike. In uncertain times, the thoughtful deployment of your resources is more important than ever. By following our four simple principles, you can continue to have a positive impact on the community causes you love even while you're paying more attention to your pocketbook.
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Kenyan Drake to Host A-Day Autograph Signing to Benefit the Autism Society of Alabama
Alabama fans have a chance to meet former Crimson Tide running back Kenyan Drake this A-Day in Tuscaloosa.
Alabama fans have a chance to meet former Crimson Tide running back Kenyan Drake this A-Day in Tuscaloosa.
Even on a mediocre day from the quarterbacks, at least two Alabama wide receivers stood out above the rest.
Robert Foster and ArDarius Stewart shared the Dixie Howell Memorial Most Valuable Player of the A-Day Game Award after the pair combined for 243 yards and two touchdowns on 14 receptions Saturday at Bryant-Denny Stadium...
A-Day is upon us, which means Tuscaloosa's population will swell by at least 75,000--which could make for some traffic delays this weekend. Are you ready?
The University of Alabama's open spring scrimmage happens Saturday, and fans have an opportunity to purchase extra accommodations to upgrade their A-Day fun.
Alabama football announced that the A-Day Spring Football Game will be played on Saturday, April 18 at Bryant-Denny Stadium.
The University of Alabama announced that its annual A-Day game will be played on April 19 at 2 PM CT.
Marking the end of Spring practices, the A-Day game will be played inside Bryant-Denny Stadium and will be free to the public once again...
Take a look at our huge photo gallery from the 2013 A-Day Game. The White team defeated the Crimson team 17-14 on a 7-yard touchdown run by TJ Yeldon, who was also awarded player of the game...


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In this article, we will show you, how to create a custom toolbar on the windows taskbar. Your personal toolbar can make your work much faster, as you don't need to search for files and folders on your computer. You can add them to your personal toolbar and you will have fast access to this files.
This tutorial is made for Windows 10, but the same procedure you can use for previous versions of Windows.
- Click your right mouse button on the Windows taskbar and select Toolbars > New toolbar
- Now find and select the folder in your computer to use as toolbar
- Now your new toolbar will be displayed on the right side of windows taskbar.
- You can add to this folder files and folders for which you want to have quick access. You can add there also shortcuts of files and folders.


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Candles and Carols
By Mark Hayes
To play the media you will need to update yourFlash plugin.
SAB choir, piano; optional orchestra
A service of hope, peace, joy, and love. Composed by Mark Hayes. Choral Cantatas. Advent, Christmas. 48 pages. Published by Lorenz Publishing Company (LX.65-1997L).
Item Number: LX.65-1997L
"Candles and Carols" is perfect for the small- or medium-sized choir for Advent and Christmas. The considerable talents of Pamela Martin and Mark Hayes have created an intimate, participatory experience for both choir and congregation, which celebrates the traditional Advent themes of hope, peace, joy, and love by observing the tradition of lighting the Advent candles. This work may be performed in individual movements throughout the weeks of Advent and Christmas, or in its entirety for any Advent Sunday, for Christmas Eve/Day, or for special seasonal celebrations. As each candle is lit and each narration spoken, beautiful settings of familiar carols--each having a newly written verse by Pamela Martin--may be sung by the choir, the congregation or any combination of the two. Choral part-writing is very accessible, and optional vocal solos are featured on some carols. The work may be performed with a full orchestra, a smaller ensemble, or a fully orchestrated Accompaniment CD.
About Digital Downloads
Digital Downloads are downloadable sheet music files that can be viewed directly on your computer, tablet or mobile device. Once you download your digital sheet music, you can view and print it at home, school, or anywhere you want to make music, and you don't have to be connected to the internet. Just purchase, download and play!
PLEASE NOTE: Your Digital Download will have a watermark at the bottom of each page that will include your name, purchase date and number of copies purchased. You are only authorized to print the number of copies that you have purchased. You may not digitally distribute or print more copies than purchased for use (i.e., you may not print or digitally distribute individual copies to friends or students).


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Aloevera Gel used in this conditioner is a skin Soother, Skin & Hair Rehydrator, Detoxifier and healer. It helps repair & remove the damaging effects of dryness, pollution, grime and acts as a hair conditioner. Regular use enhances the effect. Daily use.


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You know how I'm always on about how Hourglass doesn't really cater to dark skin beyond a certain point, so the brand was permanently, sorta off my radar? Well, Hourglass came through. Clearly someone over at head office has been listening to not only the criticism of Hourglass itself, but to the uproar from brown girls and allies in the beauty community about the absolute lack of representation and colour choices available in everything from foundation to blush and beyond. I mean there's always been grumbling, but in the last few months black girls were like "um…you can't fug wit me? Cool, I can't fug wit you either". When I say they came through, they did so like the Roman army, they conquered, put down stakes and made the scene their own. 26 shades people. 26. Their new Vanish Seamless Finish Foundation comes in 26 shades of awesome that range from the very lightest to the very darkest, and undertones for all! Got peachy undertones (raises hand)? Got golden undertones (raises hand again cause weird), got red, olive or neutral undertones? Not to worry, they got you. And for once, brown girls aren't saddled with just neutral or red undertones in their foundation, the undertone process is very democratic, and we finally have more than two shades to choose from. From a mainstream brand, this is kinda huge ya'll.
I was so damn spoiled for choice that as per usual I got confused as hell, so lemme tell you about it. What had happened was…I contacted Hourglass and they made recommendations. I took one look at the shade they recommended and said bye. After messing about the site for a week I finally settled on the shade Amber, but for whatever reason I couldn't check out on the Hourglass site. SpaceNK saved the day with an Indulge Incentive so I purchased there, because I was feeling kindly disposed to Hourglass I went ahead an picked up the Ambient lighting palette. One, because I still wanted to try Radiant Light, and two I was peripherally interested in Incandescent Light as well. I was also looking to get some Lilah B. Cosmetics and they sent along a sample card of all the blushes that I'd wanted to try so score.
I'm sure you guys have seen pictures of the foundation all over social media so I don't have to describe it to you. But I really really want to cause it's stinking pretty. The triangular tube (can that be a thing? Triangular and tubular?) is sleek and heavy. It has a twist up mechanism and is the standard Hourglass brown.
So listen, I got the foundation and what was the first thing that I did? I panicked when I saw the shade. I was like oh my god that's too light! So it went back to SpaceNK. Since I was ordering some stuff from Barney's I ordered the shade Warm Almond which is two shades darker than Amber, and also another Ambient Lighting blush in Radiant Magenta which we'll get to later. Here's the thing though? Amber was the perfect shade, I realised that because Warm Almond is perfect. Right now. In the summer. In the winter, it's not going to be a good fit since I can get at least three shades darker in the summer time. So I'm likely going to reorder the Amber shade. Both of these have a warm/golden undertone, I didn't swatch Amber or even touch it since I immediately knew that I was sending it back, but Warm Almond blended in seamlessly, another clue that I should have kept Amber since my arm is always darker than my face by at least a shade. Swatches below.
The first time I wore this I had some…problems. Not knowing that the finish was satiny was a huge part of it. I used the Radiant Light all over and wound up looking quite shiny for most of the day. The funny thing was, I wasn't oily in the least. It didn't break down or separate and I spent a good hour outside in ninety degree weather. The coverage goes to full in a flash, you've been warned if that's not your thing, and the finish? It looks like skin, your skin, just better. The triangular shape of the stick allows it to be easily used for under eye concealing which it does remarkably well without creasing. The second time that I wore it I used my Joe Fresh translucent finishing powder to much better results. This foundation is thick and pigment packed, cake face is super easy. I feel like it has a bit of a learning curve too, and I was concerned with the cost to amount ration until I realised just how little you need to get complete coverage that will hide most of your imperfections. With colour correcting, you won't even need concealer.
Speaking of colour correcting, I also picked up one of the new Ambient Light Correcting primers. Now, as far as I can tell, these don't really colour correct all that much or you, at all? There's Mood Light, a purple/pink infused primer much like the Mood Light powder. Dim Light a neutral beige which I stayed away from because even though it's the darkest of the three, it's also neutral, no thank you ma'am. Finally there's Luminous Light a champagne pearl and the one that I got.
It comes in a pretty unassuming squeeze tube, knowing Hourglass I was expecting fancy, but nope. I've always loved Hourglass primers. I absolutely love love love the Veil primer. That stuff kidnaps your foundation and holds it hostage on your face till you decide to remove it. What I didn't love? On a brown chick like me, things could get white real fast and I wouldn't know it till it had dried down. I found this primer to be exactly like the Veil primer, except that the champagne colouring eliminated the white cast, and it does leave not a luminous finish, but more of a pearlescent one that doesn't interfere with your foundation, but in turn imparts luminosity. There are no pictures of it blended out below because it literally disappears and the pearlescent attributes can't be seen on camera.
About that blush I got…Radiant Magenta. Look, when you find an Ambient Lighting Blush that works for you, it's like the skies open up and angels start singing right? But the thing is, unless you go into a store and pick one up, examine it before you walk out you really don't know what you'll get, because of that damn fancy marbling. You could like I did with Mood Exposure, luck out. Or you could strike out on two damn blushes like I've done. When will I learn? I was just so excited I didn't really think about it…and here's what I got for my troubles.
This blush is more Radiant Lighting powder than magenta colour. Which I wouldn't mind so much…nope, no. I still would have minded even if I hadn't gotten the palette. So I sent it back, which lemme tell you was an ordeal. I've never purchased Cosmetics from Barneys.com before, and…um…this will be the last time that I do so. I've purchased from Barney's before, all sorts of things. I used to date a dude that had a Barneys addiction and he worked right across the street so we frequented the place a lot. I've never had any issue returning anything, be it cosmetics, clothing what have you to the store. But when I called to try and return this, Doris (who realllllly needs to be retrained in customer service…and friendliness), made me feel like I had stolen something and was trying to scam her into taking it back. So yeah, if I'm not in the city and can take it back, Barneys.com just became a huge no for me, I'll stick to Nordstrom, SpaceNK and Bergdorf's for my high end cosmetics.
And I think that I may finally have found the Ambient Lighting powders that work well for me. Radiant Light was definitely a good choice, but I also found that I liked Dim Light even better because it's quite muted and chill, while Incandescent light makes a wonderful highlighter. Who knew.
Final Thoughts.
The foundation is going to wind up HG most likely. It lasts a good ten hours and I only had to blot once, which was pretty hilarious since all I had to do was tap the spot with my hand and boom, gone. It hasn't broken me out in the week that I've worn it, and although it is way more coverage that I normally wear, not only can I not feel it on my skin, but it looks natural and just like skin. The primer has already achieved HG status, just on the strength of it being exactly like the Veil primer without whiting me out. Lack of colour correcting properties be damned.


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A study in the May 1 edition of the journal Clinical Infectious Disease shows that generic antiretroviral drugs distributed in four developing countries meet United States Pharmacopeia standards, undermining the reasoning offered for the Bush administration's reluctance to buy the drugs. USP, a nongovernmental agency made up of representatives from the pharmaceutical industry, consumer organizations, and the federal government, as well as pharmacy, medical, and other health care professions, works closely with the Food and Drug Administration to ensure the quality of medicines for human and veterinary use.
Bush administration officials oppose using funds from the five-year, $15 billion international AIDS initiative for generic antiretroviral drugs because they say the drugs have not been evaluated to ensure that they meet U.S. safety and efficacy standards. The World Health Organization has already approved the use of the drugs.
The new study, conducted by researchers at the National Institutes of Health and the University of Alabama, evaluated six different types of anti-HIV drugs using the Uniformity of Dosage Units test. All of the drugs, which were obtained from doctors in Lithuania, South Africa, Jamaica, and Zambia, fell within the USP-approved efficacy range when stored according to manufacturer specifications. Researchers next plan to study the drugs for bioequivalency, which tests how much of the drugs are absorbed into the bloodstream.


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|10 Dec 2009
Win 7 Bootloader - multiple drives
I have searched around on ths forums but can't find an answer.
My work laptop that has pointsec (disk encryption) on it, which is the first thing that loads.
If I was to install Win7 onto a external usb HD, would it overwrite the bootloader on the internal laptop HD?
Should I go into the Bios and try to disable the internal drive (not sure i can get to the bios)?
|My System Specs
|10 Dec 2009
Windows 7 Ulti. x64
Welcome to SevenForums schander
Not sure if you can do that, I would love to see the documentation if I am wrong. Good luck to you in finding your answer. Hope that you enjoy your time on 7 and SF.
|My System Specs
|10 Dec 2009
I have searched around on ths forums but can't find an answer.
My work laptop that has pointsec (disk encryption) on it, which is the first thing that loads.
If I was to install Win7 onto a external usb HD, would it overwrite the bootloader on the internal laptop HD?
Should I go into the Bios and try to disable the internal drive (not sure i can get to the bios)?
The only tutorial I've seen for doing this is for XP and it is extremely complicated.
The questions you ask need to be answered by your IT dept. at work, or whoever maintains that internal encryption. Any attempt to disconnect in BIOS setup could brick the laptop.
|My System Specs
|10 Dec 2009
Thanks for your replies.
I will try and talk with work IT, but there worse than useless..... and in India....
|My System Specs
|Thread Tools
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Till now i have only used a single drive setup on my PC. I am planning to buy a new disk drive for my HD camcorder videos and for other backup. I have read some forums about the RAID setup for data security. I have no idea what RAID is and i do not want to loose any data form either of my drives. ...
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I recently decided to try out Windows 7 (loving it by the way) and have decided I would like to stick with it. However, I have run into a problem with having XP on one drive and Windows 7 on another. I have two drives, which for the sake of simplicity I'll call Drive1 and Drive2. I have XP...
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Tag Archives: timetable
Chippenham Festival timetable released!
Posted on 24th May 2017 by sword
We've been asked by a surprising number of people where we'll be appearing at Chippenham and when. Always ones to court any kind of adoration, we take great pleasure in publishing this timetable of what we'll be up to this bank holiday … Continue reading →
Posted in Chippenham Folk Festival, Performance, Timetable | Tagged Chippenham, Northgate Rapper, performance, schedule, timetable | Leave a comment


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THE ALMIGHTY have officially cancelled their December U.K. tour.The following is the group's statement on the matter: "With great sadness, we announce the UK tour in December has been cancelled. With slow ticket sales for the shows scheduled for THE ALMIGHTY in December and after careful consideration, [THE ALMIGHTY frontman] Ricky [Warwick] has made the decision to cancel the shows and to accept a tour of America with DEF LEPPARD where news of his solo album release is being well received. "As most of you are aware, the band have not been active in 2002 and have no management or record deal. This has been a very difficult decision to make and the band would like to thank their fans who had bought tickets and apologize for the inconvenience caused. Your support has been incredible and is appreciated by the band."
THE ALMIGHTY Cancel U.K. Tour
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Each spring, Trillion participates in an annual day of service where our studio's team volunteers our time to the community in and around NJ. This year, Trillion participated in the city of Summit's Annual Earth Day Clean Up. As a local business in the city of Summit, we were really happy for the opportunity to clean up our little corner of earth.
The Trillion work "crew" was assigned a small triangle of Summit in the wooded area at the intersection of Glenside and Morris Avenues — just minutes from our design studio. Donning work gloves and our Trillion T-shirts, our crew spent the morning cleaning up the area. We picked up 10 large bags of recyclables and an astonishing 9 large bags worth of trash. We also collected a range of surprising finds that included hubcaps, road signs, a car tire, old gears and bolts, an ice tea cooler and Christmas decorations.
Trillion also designed and produced reusable "Earth Day Clean Up in Progress" signs that we donated to the town for future Summit Earth Day Clean Up events. We are proud to be a Summit Green Star company; a program that recognizes local businesses and community organizations for their sustainable practices.
As a creative agency that designs a lot of not-for-profit work, our studio's goal is always to be good local and global citizens who care about our community and the environment. Our studio has developed a niche working with environmental conservation organizations whose mission is to protect and conserve the environment such as the NY-NJ Trail Conference, New York League of Conservation Voters and the New Mexico Wildlife Federation, and we love the fact that we were able to participate in a local environmental effort in the town we work in.
Our sincerest thank you goes out to our partners and friends at The Resourceful Business, another amazing Summit business, and Summit resident and fellow art director, Rich Montminy for donating their time and joining our crew. We also want to thank the city of Summit for allowing us to organize a Summit business Earth Day clean up day before the main event on Saturday.
If you are interested in learning how design studios can participate in Earth Day, click here. See more photos from our clean up event on our Facebook page.
Join us!
If you are interested in participating in Trillion's next day of service or have an event that is looking for corporate volunteers, we can't wait to hear from you.


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MTN Project Fame West Africa season 7 finalist and Wahala Free Music signed Afro pop Star Emekus has dropped his first single for the year.
He calls his first piece of work for the year "Pray".
Pray is a song that talks about all the stress he passed through before making the cut as an artiste.
In his own words "the road to success is full of contours".
In this one he buttresses the need for prayers in our lives and our journey to success.
After dropping several hit songs last year and working with different producers like LeriQ and Tee-Y Mix last year, the "Marry You" crooner is one of the artistes to watch out for this year.
This one was produced by Wizzy Y.
Talks of an album this year is not way off point as a result of the immense talent he carries under his small dreadlocks.
Download, enjoy and share your thoughts.
Download : Emekus – Pray


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My Nonprofit Reviews
Cheryl Vermey
Review for Maternal and Child Health Consortium of Chester County, West Chester, PA, USA
I have been committed to the empowerment of women and children for my whole professional career. When I relocated to Chester County I learned of MCHC and the fabulous work they do in the community to serve this population. Now that I am on the Board, I can say that I have rarely encountered such a dedicated group of professional staff, volunteers and board members who really care about the mission of the organization. MCHC makes a real and demonstrated difference in the quality of life and health of those we serve.
More Feedback
I've personally experienced the results of this organization in...
the dedication of the staff and volunteers. The board regularly receives reports on the outcomes of the services, which are excellant. I have done site visits and seen first hand the difference MCHC makes.
If I had to make changes to this organization, I would...
Encourage more members of tthe community to become involved!