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Hi !
Could anyone explain in a simpler way of what is a transaction and how it is maintained in weblogic ???
What is a transaction ... (2 messages)
- Posted by: A JP
- Posted on: April 13 2001 21:17 EDT
Threaded Messages (2)
- What is a transaction ... by Durgaprasad Gannamani on April 15 2001 01:41 EDT
- What is a transaction ... by Tony Brookes on April 15 2001 02:00 EDT
What is a transaction ...[ Go to top ]
A transaction is a set of statements in which they should be executed all or none.
- Posted by: Durgaprasad Gannamani
- Posted on: April 15 2001 01:41 EDT
- in response to A JP
that means if transaction is completed means all statements r executed , if at all any problem occurs in the middle none of them should be executed.
weblogic maintains transactions in two ways depending on the "transaction-type" value that u have specified in
ejb-jar file.(CMT and BMT)
Container Managed Transaction - CMT
Bean Managed Transaction - BMT
there is a lot more to explain about this question. better u refer EJB secification or any book on EJB..
What is a transaction ...[ Go to top ]
Transactions are atomic units of work.
- Posted by: Tony Brookes
- Posted on: April 15 2001 02:00 EDT
- in response to A JP
For instance.
Debit my account by $100 and credit yours by $100.
Either both operations should occur, or neither. It would not be good if I was $100 down but it never made it to you.
They have other characteristics, like isolation, consistency, durability etc. (ACID is the acronym. If you search for this along with transaction at google then you'll find loads of information.)
Theoretically, you shouldn't care how WebLogic handles these. All you need to do is pull a UserTransaction from JNDI and then start using it.
Within J2EE you can leave transactionality to the server (container managed) or you can do it yourself (bean managed.)
With entity beans that last statement is false, you can't do BMT, it's part of the spec. It's late, I'm tired and I can't remember why! :-)
Hope that helps.


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wikiHow to Use Macros in Excel
Two Methods:Use Macros with Excel (Except for Excel 2008 for Mac)Use Macros with Excel 2008 for MacCommunity Q&A
Sometimes a spreadsheet has a process that has to be repeated with some regularity. You can set up a macro to repeat that process easily without requiring you to go through all the steps. Understand how to use macros in Excel to save time.
Use Macros with Excel (Except for Excel 2008 for Mac)
1Verify that your Excel preferences allow you to run macros. This setting is in the "Security" section, in most versions of Excel.
2Open the workbook that contains or will contain the macro.
3Verify that macros are enabled in the specific workbook, which will let the macro be run.
4Consider how you want to use the macro. Macros can be run from a menu, connected to a button in the workbook, set to run automatically upon opening a worksheet, or tied to a keyboard command.
5Record or write the macro at this time, to repeat the particular process that you want automated, if your workbook doesn't already have a macro installed that you want to use.
- To record the macro, find the "Record New Macro�" command. This can be in the "Macros" section of your "Tools" drop-down menu or ribbon, inside the macro window (again, usually found in "Tools"), or on a toolbar, depending on your version of Excel, your computer system, and your specific Excel setup.
6Open the macro menu, usually in the "Tools" section of Excel.
7Test-run the macro you want to use, making sure it works properly.
8Assign the macro to whatever button, key command, or worksheet auto-run you want to use with it.
9Run the macro as needed.
Use Macros with Excel 2008 for Mac
1Open the workbook where you need the macro.
2Open "Script Utility," found in the "AppleScript" folder, inside your global "Applications" folder. (The global "Applications" folder is the one in your computer, before you open your main user account folder.)
3Hit the "record" button and follow the process you want in the macro.
4Test that the recorded macro works as intended. (Not all actions are scriptable, meaning you cannot record them for an AppleScript.)
5Save the recorded AppleScript.
6Run the AppleScript as needed.
Community Q&A
How can I write macros that will change a spreadsheet as soon as I create it?wikiHow ContributorYou will first need a basic understanding of VBA. There are many tutorials on this.
- If you make your own macro, it's easiest to troubleshoot if you record each piece in small sections. Test each section independently, and you can re-record a section as necessary. Once it all works, compile the macro into a single program.
- If the macro is created on a computer system other than the one it is used on, the macro may require a bit of tweaking to run properly. Visual Basic for Applications (VBA, the programming language for Excel macros) differs a bit between the Windows and Mac versions of Excel.
- If you want the macros to be automatically enabled, some versions of Excel allow you to "Save As�" an XLSM file, a "macro-enabled workbook" and to set preferences for particular folders where macros will always be accepted.
- Macros can also damage Excel files, by accident or on purpose. Make sure to back up your spreadsheet before you start playing with a macro.
- Computer viruses can hide as macros on Excel workbooks. If a workbook has a macro without anyone giving an alert, verify that the macro was intentionally added by someone you trust.
Sources and Citations
Article Info
Categories: Microsoft Excel
In other languages:
Español: utilizar Macros en Excel, Português: Utilizar Macros no Excel, Italiano: Usare le Macro su Excel, Русский: использовать макросы в Excel, Deutsch: Makros verwenden in Microsoft Excel, Nederlands: Macro's gebruiken in Excel, Bahasa Indonesia: Menggunakan Makro di Microsoft Excel, ไทย: ใช้ Macro ใน Excel
Thanks to all authors for creating a page that has been read 544,819 times.


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Hey, wad up its $horty....I ordered this online for my weekly e*mail tip during pregnancy.......You have to read these (i'm putting more in the forums)........They are to help you feel good about yourself during your pregnancy! They really really have helped me, and I hope they help yall out too....Good luck mamis-2-b!!!! (remember I dint write them tho...If you really like them, theres a web site at the bottem of the page.......)
you absolutely do not have to feel uncomfortable and unattractive your entire 10 months! To help you out - here are some simple secrets that I discovered during my pregnancy to help me look and feel good...
Top 10 tips for looking hot during your pregnancy:
1. Don't give up on dressing up!
Being pregnant definitely does not mean that you have to give up on dressing up!
I know, when you don't feel good it's hard to get motivated to take that extra care - but believe me, you will feel much better when you do it. You don't have to go overboard, just put on black pants, a nice top, and some makeup and get out!
2. Buy in bigger sizes
buy regular clothes that are 1 or 2 sizes bigger than you would normally wear. This will help you to stay in style and feel good about the way you look until you're ready to fit into regular maternity clothes (depending on your body, i'd say around 4 months).
My favorite was a one-piece trendy sweat outfit - I bought it in a large, whereas pre-pregnancy I was a small! It was great because the waist was loose and slightly elastic, with a drawstring so I could adjust it and stretch it - I wore it up until I was 6 months!
3. Below the belly is in
low-rise is "in" - take advantage of it, it's the perfect fit for a pregnant tummy! I bought a pair of black low-waisted stretch pants that lasted me quite a while.
The best purchase I made, though, was a stretchy black, low-waisted skirt that lasted me my whole pregnancy (i also wore it a lot after I delivered!).
4. Flaunt that belly baby!
Buy fitted tops that show off your belly (don't hide in a tent!). Your pregnant belly is beautiful so show it off! If fitted isn't your style - that's ok - but trying to cover up in over-sized shirts will only make you look bigger! Really!
5. Quality is key
spend a little money on some key pregnancy pieces such as a white blouse, black pants, black skirt, jeans etc. Good quality products should be adjustable and fit you for most of your pregnancy.
6. Get help from the professionals
make sure you shop at a maternity store where the sales people are knowledgeable. The first pair of pants that I was "wrongly" advised to buy ended up fitting me for a whole two weeks!
7. Accessorize
wear a necklace, bracelet or nice shoes to "dress your outfit up". I loved my little sparkly necklace - it attracted everyone's attention away from those parts that I wasn't feeling so good about!
For those of us heading into winter a cute scarf, hat, or fun bag can also do the job!
8. Spoil your feet
swollen feet are one of the most common pregnancy symptoms. Besides a good footbath, choose flat slip-ons or zip up boots (if you're in winter!).
9. Stay cool
be sure to dress in layers - you don't want to get caught sweating buckets when your pregnancy thermostat kicks in (hello overheating!).
10. Stretch is best
flaunt your assets by wearing stretchy fabrics - and of course, showing some skin! You'll do even better if you dress for your body type and attract attention to your good parts, not your bad ones!
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A very useful & interesting site Thanks so much !!!! - Bobby
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POM Wonderful
by POM Wonderful
Product Type
450 ml (15.2 fl oz) glass
Nutritional Info
serving size 8 oz;
calories 150;
total fat 0g;
sat fat 0g;
cholest 0mg;
sodium 75mg;
total carb 37g;
fiber 0g;
sugars 31g;
protein 0g;
vitamin A 0%;
vitamin C 0%;
calcium 2%;
iron 4%
100% juice, pomegranate, orange, tangerine juices from concentrate and natural flavors
Review: Pomegranate Tangerine
Posted: Sep 25, 2003 at 12:00 AM (Last Updated: Oct 01, 2003 at 6:37 PM)POM Wonderful Tangerine use a nice mixture of citrus juices to create a product that has a taste that is slightly lighter on pomegranate flavor. This is probably a good choice for drinkers that are new to the flavor of pomegranate. The resulting taste is sweet, bitter, and fruity with a tart pomegranate finish. Overall, we like this product -- good taste, good packaging. The only potential dowside is that it needs to be in a refrigerated cooler, which may make getting it to retail difficult.


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They don't live here anymore, but Athens still likes to claim The B-52s as its own, and true to form, they'll be back in town Feb. 18 to play a benefit for the Georgia Theatre.
The show will take place at the Classic Center Theatre, and tickets go on sale at 10 a.m. Dec. 3.
Tickets are $122 for a VIP meet and greet and seats in the orchestra pit; $56.50 for floor; $46.50 loge; and $36.50 for mezzanine balcony. A portion of the ticket sales will benefit efforts to rebuild the Georgia Theatre.
For more information, go to www.classiccenter.com.
© 2017. All Rights Reserved. | Contact Us


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Cheap Soho Cinders tickets
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The Show
Edgy, fun and deliciously naughty, Soho Cinders celebrates the capital's most colourful district and is set to get London's temperature soaring this summer. The potent mix of politics, sex-scandals and true love come together in this contemporary musical with an infectious score that you'll be humming long past the stroke of midnight!
The Story
When impoverished student Robbie becomes romantically involved with engaged Mayoral candidate James Prince, his lap-dancing step-sisters become the least of his problems! James and Robbie's worlds collide forcing them to fight for their own fairy-tale ending in this hilarious, satirical twist on the classic Cinderella story.
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Login Security
I just had a plugin database discrepenacy where I needed to get a new password cause well I lost it :). So I did the normal lost password stuff. And then I thought if anybody knew my email and my login name (or that matter any user) they could access my account as an admin. This is not good.
Is there a plugin out there that would require a secret word to be entered prior to retrieving a password?
Or am I just worrying over nothing?
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Data obtained from studies conducted in animal models and humans suggest that gender affects the metabolic response to fasting. However, differences in body composition between males and females confound the interpretation of these studies, because increased adiposity itself alters the metabolic response to short-term fasting. We evaluated whole body lipid and glucose kinetics during basal (14-h fast) and short-term fasting (22-h fast) conditions in six women and six men who were matched for adiposity (24 ± 2 and 23 ± 2% body wt as fat, respectively). Substrate kinetics were measured by infusing stable isotope labeled tracers of glucose ([2H2]glucose) and glycerol ([2H5]glycerol). Basal glycerol rate of appearance (Ra) in plasma, an indicator of whole body lipolytic rate, was greater in women than in men (2.1 ± 0.2 vs. 1.5 ± 0.1 μmol · kg body wt−1 · min−1; P < 0.05). However, the relative increase in glycerol Ra with continued fasting was blunted in women compared with men (40 ± 7 vs. 80 ± 4% increase; P < 0.05), resulting in similar lipolytic rates in both genders at 22 h (2.8 ± 0.2 and 2.6 ± 0.1 μmol · kg body wt−1 · min−1 for women and men, respectively). In contrast, glucose Ra was similar in men and women at 14 h (11 ± 0.6 vs. 12 ± 0.7 μmol · kg body wt−1 · min−1) and 22 h of fasting (9 ± 0.6 vs. 10 ± 0.6 μmol · kg body wt−1 · min−1). These data demonstrate the presence of sexual dimorphism in lipid, but not glucose, metabolism during both basal and short-term fasting conditions.
- fatty acids
- lipolysis
- glucose production
- stable isotopes
the adaptive response to fasting involves a carefully integrated series of metabolic alterations that are critical for survival. The initial events are characterized by an increase in the mobilization of adipose tissue triglycerides and a decrease in the production and oxidation of glucose. These processes provide fatty acids and ketone bodies as fuels to body tissues and reduce the need for muscle protein breakdown to release gluconeogenic amino acid precursors.
Data obtained from studies conducted in animals demonstrate that gender influences the metabolic responses to energy restriction (20, 21,46). Female rats use more fat and preserve lean body mass better than male rats during short-term fasting (20, 21) and during chronic undernutrition (20, 21, 46). The higher rate of protein catabolism in male than in female animals is responsible for the marked differences in survival observed between genders during low-energy feeding or total starvation. For example, 87% of female pigs, compared with only 22% of male pigs, survive after 1 yr of severe energy restriction (46).
Gender differences in the metabolic response to fasting have also been observed in humans. The increase in plasma fatty acid and ketone body concentrations (17, 34, 35) and the decrease in plasma glucose concentration (17, 34) that normally occur during short-term fasting (60–86 h) are greater in women than in men. Although these findings suggest that the mobilization of fatty acids from adipose tissue and the decline in hepatic glucose production are greater in women than in men during fasting, few studies have evaluated the physiological processes that are responsible for these differences. We are not aware of any studies that compared lipid kinetics in men and women during fasting, and we know of only two studies that have compared glucose kinetics (5, 6), with conflicting results. In one study (5), the decline in glucose rate of appearance (Ra) into plasma during short-term fasting (from 16 to 64 h of fasting) was greater in women than in men, whereas in the other study, the decline in glucose Ra (from 16 to 22 h of fasting) was similar in both genders (6). However, these studies are difficult to interpret because of the inherent differences in body composition between men and women. Females generally have more body fat than males (5, 20, 21), which can independently influence the metabolic response to fasting. Within the same gender, studies performed in humans (22, 23) and in rats (12, 19) have found that increased adiposity itself alters the metabolic response to short-term fasting.
The purpose of this study was to evaluate the effect of gender on lipid and glucose kinetics during basal and short-term fasting conditions. We measured whole body glycerol and glucose kinetics by using stable isotope labeled tracers at 14 and 22 h of fasting in men and women. Our men and women were matched on percent body fat to eliminate the potential influence of gender-related differences in body composition on substrate metabolism. We hypothesized that the increase in lipolytic activity and the decrease in glucose production that occur during early fasting would be greater in women than in men when both groups were matched on adiposity.
Six men (32 ± 4 yr old) and six premenopausal women (27 ± 2 yr old), who were matched on percent body fat, participated in this study (Table 1). All subjects were considered to be in good health after completing a comprehensive medical evaluation that included a history and physical examination, an electrocardiogram, and standard blood and urine tests. None of the subjects was taking regular medications or smoked tobacco. All subjects had a stable body weight for ≥2 mo and had been sedentary (regular exercise <1 h/wk) for ≥6 mo before the study. In female subjects, the study was performed during the first 2 wk of the follicular phase of their menstrual cycle. Each subject's fat mass (FM) and fat-free mass (FFM) were determined by dual-energy X-ray absorptiometry (Hologic QDR 1000/w, Waltham, MA). The truncal region was defined as the body segment between 1) lines passing through the humeral head and the apex of the axilla to separate the arms, 2) a line passing from the iliac crest to the perineum to separate the legs, and 3) a horizontal line passing just below the mandible to eliminate the head. Written informed consent was obtained from all subjects before their participation in the study, which was approved by the Human Studies Committee and the General Clinical Research Center (GCRC) Scientific Advisory Committee of Washington University School of Medicine in St. Louis, MO.
Experimental Protocol
The evening before the isotope infusion study, subjects were admitted to the GCRC at Washington University School of Medicine. At 1800, subjects consumed a standard meal containing 12 kcal/kg body wt (55% of total energy as carbohydrate, 30% as fat, and 15% as protein). At 2000, the subjects ingested a liquid formula (Ensure; Ross Laboratories, Columbus, OH), containing 250 kcal (40 g carbohydrates, 6.1 g fat, and 8.8 g protein) and then fasted until completion of the study the following day.
The following morning, an isotope infusion study was performed to measure whole body glycerol and glucose kinetics at 14 and 22 h of fasting. At 0530, catheters were inserted into a forearm vein for isotope infusion and into a radial artery for blood sampling. At 0630, a constant infusion of [6,6-2H2]glucose (priming dose: 18 μmol/kg; infusion rate: 0.22 μmol · kg body wt−1 · min−1; Cambridge Isotope Laboratories, Andover, MA) was started and maintained for 210 min. At 0830, a constant infusion of [1,1,2,3,3-2H5]glycerol (priming dose: 1.8 μmol/kg; infusion rate: 0.12 μmol · kg body wt−1 · min−1; Cambridge Isotope Laboratories) was started and maintained for 90 min. All isotopes were infused via calibrated syringe pumps (Harvard Apparatus, South Natick, MA). Blood samples were obtained before the isotope infusions were started to determine background glucose and glycerol tracer-to-tracee ratios (TTR) and every 10 min between 0930 and 1000 (for the 14 h of fasting) to determine plasma glucose and glycerol TTR and plasma substrate and hormone concentrations. At 1000, all isotope infusions were stopped, and catheters were kept patent by infusing 0.9% saline. At 1430, the isotope infusion protocol was repeated. Blood samples were obtained before restarting the isotope infusions to determine background glucose and glycerol TTR and every 10 min between 1730 and 1800 (for the 22 h of fasting) to determine plasma glucose and glycerol TTR and plasma substrate and hormone concentrations. Subjects remained in bed for the entire duration of the study.
Sample Collection
Blood samples were collected in 1) chilled tubes containing EDTA to determine plasma fatty acid, glycerol, and glucose concentrations and glycerol and glucose enrichments; 2) chilled tubes containing EDTA and Trasylol to measure insulin and glucagon concentrations; and 3) chilled tubes containing reduced glutathione and EGTA to determine plasma catecholamine concentrations. Blood samples were placed in ice, and plasma was separated by centrifugation within 30 min of collection. Plasma was stored at −70°C until final analyses were performed.
Sample Analyses
Plasma insulin (13) and glucagon (15) concentrations were measured by radioimmunoassay. Plasma catecholamine concentrations were determined by a single isotope derivative radioenzymatic method (43). Plasma glucose concentration was determined by using an automated glucose analyzer (Yellow Springs Instrument, Yellow Springs, OH). Plasma glycerol concentration was determined by gas chromatography-mass spectrometry (GC-MS) after [2-13C]glycerol was added to plasma as an internal standard. Plasma fatty acid concentrations were quantified by gas chromatography (Hewlett-Packard 5890-II, Palo Alto, CA) after adding heptadecanoic acid to plasma as an internal standard (32).
Plasma glycerol and glucose TTR were determined by GC-MS (MSD 5973 system with capillary column; Hewlett-Packard) as previously described (22, 23, 40). Briefly, plasma proteins were precipitated with ice-cold acetone, and hexane was used to extract plasma lipids. The aqueous phase, containing glycerol and glucose, was dried by speed-vac centrifugation (Savant Instruments, Farmingdale, NY). Heptafluorobutyric (HFB) anhydride was used to form an HFB derivative of glycerol and glucose, and ions were produced by EI ionization. Glycerol TTR were determined by selectively monitoring ions at mass-to-charge ratios (m/z) 253, 254, and 257, and glucose TTR were determined by selectively monitoring ions at m/z519 and 521.
A physiological and isotopic steady state was achieved at 14 and 22 h of fasting. Therefore, substrate (glycerol and glucose) rate of appearance (Ra) and rate of disappearance (Rd) from plasma were calculated by using Steele's equation for steady-state conditions (45). Glycerol Ra represents the rate of glycerol released into plasma from hormone-sensitive lipase hydrolysis of adipose tissue and intramuscular triglycerides and the rate of glycerol that is released into plasma during lipoprotein lipase hydrolysis of very low-density lipoprotein triglyceride. Glycerol released during lipolysis of intra-abdominal adipose tissue triglycerides that are cleared by the liver are not detected by systemic tracer infusion.
Statistical Analysis
A two-way analysis of variance (ANOVA), with gender and time as factors, was used to test for significant differences in substrate kinetics between men and women during fasting. SignificantF-ratios were followed by post hoc comparisons with the use of Student's t-test for multiple comparisons. Statistical analyses were performed by using JMP statistical software version 3.2 (SAS Institute, Carey, NC). A value of P ≤ 0.05 was considered to be statistically significant. All data are expressed as means ± SE.
Plasma Hormone Concentrations
Mean basal (14 h of fasting) plasma insulin concentration tended to be lower in women than in men, but the difference was not statistically significant (P = 0.09; Table2). The failure to demonstrate statistically significant differences in basal plasma insulin concentrations between groups may reflect a type II statistical error because of the small number of study subjects. With continued fasting, plasma insulin concentration decreased in both groups (P < 0.05), but the relative decline was greater in women (∼40% decrease) than in men (∼20% decrease) (P = 0.07), resulting in a significantly lower mean plasma insulin concentration in women than in men at 22 h of fasting (P < 0.05). Plasma glucagon concentrations were similar in both groups at 14 and 22 h of fasting. At 14 h of fasting, mean plasma epinephrine concentration was twice as high in women than in men (P < 0.05). Plasma epinephrine concentration increased significantly in men but not in women from 14 to 22 h of fasting, resulting in similar plasma epinephrine concentrations with continued fasting in men and women. Mean basal plasma norepinephrine concentration was similar in both groups and was not affected by continued fasting.
Plasma Substrate Concentrations
At 14 h of fasting, mean plasma fatty acid and glycerol concentrations were higher in women than in men; they increased with continued fasting in both groups (P < 0.05). Plasma glucose concentration was similar in both men and women at 14 h and 22 h of fasting (Table 2).
Substrate Kinetics
Basal conditions (14-h fast).
At 14 h of fasting, glycerol Ra, expressed per kilogram of body weight, per kilogram of FFM, or per kilogram of FM, was greater in women than in men (Table 3and Fig. 1). In contrast, mean basal glucose Ra (and Rd) was similar in men and women (Table 3 and Fig. 1).
Continued fasting (22-h fast).
Glycerol Ra increased with continued fasting in both groups. However, the relative increase in glycerol Rabetween 14 and 22 h of fasting was greater (P < 0.05) in men (80 ± 4%) than in women (40 ± 7%) (Fig.2). The absolute increase in glycerol Ra between 14 and 22 h, however, was similar in men (1.1 ± 0.2 μmol · kg body wt−1 · min−1) and women (0.8 ± 0.1 μmol · kg body wt−1 · min−1) (Fig. 2). In contrast to lipid kinetics, glucose Ra decreased (P< 0.05) with continued fasting in all subjects. The relative and absolute decreases in glucose Ra were similar in men and women (Fig. 2).
In this study, we examined the effect of gender, independent of adiposity, on whole body substrate kinetics during basal (14-h fast) and short-term fasting (22-h fast) conditions. Men and women were matched on percent body fat to eliminate the potential influence of gender differences in adiposity on substrate metabolism. Basal glycerol Ra was greater in women than in men, but the relative increase in glycerol Ra during brief fasting was greater in men than in women. In contrast, glucose kinetics (glucose Ra and Rd) were similar in men and women at both 14 and 22 h of fasting. These data demonstrate the presence of sexual dimorphism in lipid, but not glucose, metabolism. The mechanisms responsible for these gender effects are not known but may be related to differences in the major plasma hormones that regulate lipolysis. At 14 h of fasting, circulating insulin, an important inhibitor of basal lipolytic rate (25), tended to be lower in women than in men, whereas the increase in plasma epinephrine concentration between 14 and 22 h of fasting, an important stimulator of lipolysis during fasting (31, 48), was greater in men than in women.
Insulin and epinephrine are the key circulating hormones that regulate lipolysis of adipose tissue triglycerides. Insulin is the major regulator of basal lipolytic rates, whereas catecholamines are important for stimulating lipolysis during metabolic stress. For example, insulin withdrawal during pancreatic clamp conditions doubles basal whole body FFA Ra (25). In contrast, β-adrenergic blockade does not significantly alter basal lipolytic rates (1, 31) but prevents the normal increase in lipolysis that occurs during exercise (1, 14) or after burn injury (47). Therefore, it is possible that the lower plasma insulin concentrations present in our female compared with our male subjects contributed to the higher basal glycerol Ravalues in women. Although basal plasma epinephrine concentration was greater in our women than in men, it is unlikely to have influenced basal glycerol Ra, because the epinephrine concentrations in both groups were below the threshold needed to increase lipolysis (11).
Although several studies have evaluated lipolytic activity in men and women, the effect of gender itself on basal lipolytic rate remains unclear because of conflicting data between studies and differences in body composition between the male and female subjects. Earlier studies that used isotope tracer methods found that whole body fatty acid Ra, expressed per kilogram of FFM, was either similar (24) or greater (26, 29) in women than in men. The interpretation of data from these studies is confounded by differences in body composition and fat distribution between female and male subjects, which can independently influence lipolytic rate (22, 23). In the present study, basal glycerol release into plasma was greater in women than in men, who were matched on percent body fat. These results are particularly striking, because our men had greater amounts of abdominal fat than our women, and abdominal adiposity is associated with increased lipolytic rates (28).
The techniques that are available to measure whole body lipid kinetics have some limitations. The tracer infusion method that we used to evaluate glycerol Ra underestimates true whole body lipolytic rates. Systemically infused glycerol tracers are unable to detect glycerol released from visceral adipose tissue that is taken up by the liver and does not enter the systemic circulation. However, it is unlikely that this methodological limitation affected our conclusions because undetected glycerol release from visceral adipose tissue triglycerides probably accounts for <5% of whole body lipid flux (16). Therefore, the lower rate of basal glycerol kinetics observed in our male compared with our female subjects was due to the effect of gender itself rather than undetected lipolysis of visceral fat associated with gender-related differences fat distribution.
In contrast to lipid kinetics, basal glucose Ra was similar in men and women. Insulin is the major circulating hormone that regulates both basal rates of lipolysis (4, 33, 38) and glucose production (3, 42). In the present study, basal plasma insulin concentration tended to be lower in women than in men, which is consistent with previous studies that evaluated plasma insulin concentration in men and women (39, 49). The presence of similar glucose Ra values despite lower plasma insulin concentrations suggests that hepatic glucose production was more sensitive to insulin in women than in men. Although several studies have reported that insulin sensitivity with respect to glucose metabolism was similar in men and women (2, 10), other investigators have found that insulin sensitivity, assessed by an oral glucose tolerance test (7, 9) or a hyperinsulinemic euglycemic clamp (8, 39, 41, 49), was greater in women than in men after adjustment for body fat. Our results also suggest that the enhanced effect of insulin action on hepatic glucose production in women is not a result of insulin's indirect effect on glucose metabolism through alterations in lipolysis (36,37), because basal lipolytic rates and plasma fatty acid concentrations were greater in our women than in our men.
To our knowledge, this is the first study that has evaluated the effect of gender on lipid kinetics during short-term fasting. Several earlier studies have evaluated the effect of gender on plasma substrate concentrations during fasting. However, plasma substrate concentration represents the balance between substrate delivery into plasma and substrate tissue uptake but does not provide information regarding the dynamic metabolic events responsible for the observed concentrations. Moreover, the relationship between plasma substrate concentration and substrate metabolism can vary markedly depending on the physiological state (30). Therefore, direct assessment of substrate kinetics is necessary for understanding the metabolic adaptations that occur in response to fasting.
The results of the present study demonstrate that the relative increase in lipolysis that normally occurs during early starvation (between 14 and 22 h of fasting) was blunted in women compared with men. However, basal (14-h fast) whole body lipolytic rates were much greater in women than in men, and the absolute increase in the lipolytic rate was the same in both groups. Therefore, the attenuated increase in lipolysis during fasting in women did not compromise fatty acid availability as a fuel in these subjects. In fact, the relatively blunted lipolytic response in women may be beneficial by preventing excessive, and potentially harmful, increases in plasma fatty acid concentrations (44). In contrast, the decrease in glucose production during fasting was similar in both groups. It is not known whether the observations in substrate kinetics made during this brief period of fasting would persist with more prolonged starvation.
The mechanism(s) responsible for the gender differences in lipid metabolism in response to brief fasting is not known but could be related to differences in hormonal responses and adipose tissue sensitivity to hormone action. Catecholamines and insulin are the two major hormones that regulate lipid and glucose kinetics during fasting. Data from previous studies suggest that the increase in lipolysis during short-term fasting is related to an increase in plasma epinephrine concentration and increased adipose tissue sensitivity to epinephrine-mediated lipolysis (31, 48), in conjunction with a decrease in plasma insulin concentration and decreased adipose tissue sensitivity to the antilipolytic effect of insulin (27). In the present study, plasma epinephrine concentrations increased during continued fasting in men but not in women, which would enhance lipolytic activity in men. In contrast, the relative (but not absolute) decline in circulating insulin concentration was greater in women than in men, which would enhance lipolytic activity in women. We did not assess adipose tissue sensitivity to epinephrine and insulin, which would further elucidate the impact of changes in plasma hormone concentrations on lipolysis of adipose tissue triglycerides. The greater relative increase in lipolysis observed in men than in women suggests that an increase in circulating epinephrine and adipose tissue lipolytic sensitivity to catecholamines is very important for stimulating lipolysis during early fasting. Indeed, acute β-adrenergic blockade at 84 h of fasting in men decreases whole body lipolytic rates to basal (12-h fasting) values (31).
Our female subjects were studied during the follicular phase of their menstrual cycle. It is unlikely that studying women during the luteal phase of their cycle would have affected the results of our study. Although previous studies have demonstrated that menstrual cycle phase can affect substrate kinetics during extreme physiological conditions, such as high-intensity exercise (50), glucose and fatty acid kinetics are the same during the follicular and luteal phases of the menstrual cycle during basal conditions, low-intensity exercise, or short-term fasting (6, 18, 50).
To match men and women on percent body fat, we recruited female subjects who were lean [body mass index (BMI) = 21 kg/m2 and 24% body wt as fat] and male subjects who were slightly overweight (BMI = 26 kg/m2 and 23% body wt as fat), because women are generally fatter than men. However, these within-gender differences do not affect the interpretation of the results from our study, which was designed to evaluate the effect of gender, independent of adiposity, on substrate metabolism.
In summary, the results of the present study demonstrate the presence of gender differences in lipid but not glucose kinetics in men and women who were matched on percent body fat. Basal lipolytic rates (assessed by glycerol Ra) were greater in women than in men, whereas whole body glucose production and utilization were similar in both groups. During early fasting, the relative increase in whole body lipolytic rate was blunted in women compared with men, whereas the decline in the rate of glucose production was similar in both genders. These results demonstrate the need to study men and women separately when evaluating in vivo substrate kinetics in human subjects. Further studies are needed to determine the physiological and clinical importance of these gender differences in lipid kinetics.
We thank the nursing staff of the General Clinical Research Center for help in performing the studies, Guohong Zhao and Weqing Feng for technical assistance, and the study subjects for their participation.
This study was supported by National Institutes of Health Grants DK-37948, HD-01459–01, RR-00036 (General Clinical Research Center), RR-00954 (Mass Spectrometry Resource), and DK-56341 (Clinical Nutrition Research Unit).
Address for reprint requests and other correspondence: S. Klein, Washington Univ. School of Medicine, 660 S. Euclid Ave., Box 8031, St. Louis, MO 63110 (E-mail: sklein{at}im.wustl.edu).
The costs of publication of this article were defrayed in part by the payment of page charges. The article must therefore be hereby marked "advertisement" in accordance with 18 U.S.C. Section 1734 solely to indicate this fact.
- Copyright © 2001 the American Physiological Society


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I bought my first Jeep this week, and it's an XJ
Hi all. I'm new to the Jeep forums but I think I picked the best one to join. I have been around modded TJ's and XJ's for the last few years and this past week a deal on a lightly modded XJ came along that I just couldn't pass up.
Around here we use our jeeps almost exclusively off road, wheeling in the trails, swamps and woods of South Central Alaska. We wheel pretty much every week from end of April to the middle of October then the buggies get put away and the sleds come out.
Anyway, I bought this '01 XJ last week. It's set up with an 8" lift, Brand New 38.5X15 Boggers, Corporate 14 bolt with a spool in the rear. Hi-Pinion Dana 44 in front. Long Arm kit, OR-Fab bumper in rear, Warn 8000 winch in front, 4.10 gears, 4 new long travel rancho shocks.
The thing is in perfect condition It has a great running, coil on plug, fuel injected 4.0. The thing runs perfect and is well put together. I have already upgraded a few things like steering and suspension joints. I have a few more upgrades planned for it. High Steer kit, E-Locker for the front, Disc brake conversion for the rear, OR-Fab rocker guards with steps, POR 15 or smooth finish bed liner the entire inside of the cab front to back.
We have been having a blast with this thing for the last week.
I plan to be here quite a bit asking questions and getting opinions on the various improvements I want to make to this thing.
Question #1. Did I find the right place and is this where a guy with an XJ like mine would come for information and advice? :cheers2:
A few more pictures of my first few outings.
One of my good friends and long time Jeep enthusiast decided to take me up a pretty tricky trail up to a place called Baldy on the first day I owned the XJ. The trail up can get pretty greasy and very steep in the rocky sections. There are a few large mud holes but nothing over about 40" deep. The climb is about 1800' up to the top of the main hill.
Here we are at the top.
And back home after a fun day of challenging but fairly easy wheeling.
After 2 fun filled days of wheeling with friends my wife and I decided to take a little trip into the woods and do an overnight camp out. We are pretty accustomed to camping in the woods with either atv's and a tent or in the motorhome but this trip in the Jeep was really fun. There's nothing like staying warm and dry on your trip in to find a good spot to set up camp.
This little bog stopped us in our tracks. We were alone and the trees were a little far from the trail for any good winching. I tried for about an hour to get around this spot with no luck. I walked out into this peat bog until the mud was to the top of my waders and was only about 50' from the jeep. This is a nasty spot that only buggies with 46" or larger AG tires make it through and many times they don't make it. I pulled the winch out twice and decided I had had enough.
Welcome and congrats! Jeep looks good!
Hey thanks, yours is very cool too.
Be sure to check this out for some great deals on roof racks! http://www.discountramps.com/car-top...roof-racks.htm
Keep the pictures coming, we love them here!
Do you plan on stretching the Jeep any? It would look MEAN with those 38s! How is that 8" of lift treating you? The steering feel solid with that big tires or is the pump upgraded? The terrain you are in looks like that big of lift is ideal and low center of gravity isnt really needed... I may be wrong haha.
The springs are stock in the rear and very soft up front. There is no sway bar so it articulates really good.
No pump upgrade yet but my next mod is a high steer tie rod relocation. That will include a new panhard bar as well. Does the pump upgrade help eliminate the dreaded wabble when you hit off axis bumps at speed?
A few more wheelin pictures.
This is pretty much what our trails look like.
That thing is huge! Looks good man.
Welcome to the forum buddy. That XJ is awesome looking! I'd love to take a trip there but my 33"'s would never make it. But they suffice here in KY. A pump upgrade will help.
Awesome looking Jeep, more wheeling pics!
Alaska :drool: Those are some gnarly trails.
A few more pictures of the trails we wheel on around my house.
I urge you to read up on how to safely strap a vehicle to a trailer. By using ratchet straps in this manner, you are putting horizontal loads on components engineered for vertical loads. It's not safe at all!
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There is a region hack described for the Samsung C5500 that does not seem to work for US version players (https://www.videohelp.com/dvdhacks/samsung-bd-c5500/10794). Has anyone successfully applied this hack to a US region one player?
I tried to find a Brazilian firmware since it sounds like people were able to region free their other type Samsung players that way. However I downloaded the Brazilian, Australian, German, and English version of the software from Samsung and was surprised that the firmware filename was always the same. I did a byte comparison of the files and they are identical which makes me wonder if all players run on the same firmware. This cannot really be since the same player sold in different regions has different features. Unless the firmware for all regions is in the same file and depending on a local setting on the player it knows which part of the file needs to be updated.
Can someone post maybe a video tutorial on how they made the hack work on their player? The timing to enter the code seems to be of the essence but there are conflicting infos on how fast to type the code. I tried it at various speeds but could simply not make it work on a US player (even though a code seems to exist for those players).
Any help is appreciated.
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I have the same problem. I bought this BD player in US and I can't unlock, I already tried all codes and nothing.
Can anybody help us? I don't know, maybe if hack firmware, we could unlock this BD Player.
I have the same problem. I bought Samsung C5500 player in US and I can't unlock, I already tried all codes and nothing.
Can anybody help ?
Anyone able to change region on Samsung C5500 US version???
Guess this thread REALLY needs me to say this...
1) Samsung and Sony make the MOST DIFFICULT players in the world to unlock right now. If you want a region free player, you need to buy another brand.
2) A very small number of Samsung models have reported to be able to be unlocked. Most are sold in Australia. We got ONE report that one US model could be unlocked. ONE. That's it. Sorry but I don't remember the model number (because of reason #1 I care nothing about Samsung and Sony). The odds are it's not yours. Off the top of my head I would say that there's maybe 4-5 Samsung models in the entire world that can be unlocked. Sony doesn't even have that many.
3) Sometimes manufacturers make hardware changes during production or in other parts of the world. The hack instructions may be perfectly correct but only work on Australian players or maybe it did work on US models, but you had to buy one more than 1 year ago for it to work. It's not an exaggeration to point out that by the time you read about a hack, it may already be too late for it to work on YOUR model.
4) Based on threads like this one (we get posts all the time from Samsung and Sony owners wanting to unlock their players) the odds are that this is NOT your problem, but I will throw it out there anyway. In a few cases we've had hacks reported but the person who reported it left out some key piece of information and the hack won't work without it. For example they may say to input a number like 54321 but what you really have to do is input 5 enter 4 enter 3 enter 2 enter 1 enter for it to work or you may have to push an arrow key and they didn't tell you that.
The odds are extraordinarily high that none of you will unlock your players. Sorry.
I have a BD-C5500, bought it in Portugal, and it was hacked and all good, but we had to get it repaired, and it was upgraded to the lastest firmware...now I can't get it set to region free again...any thoughts?
Unfortunately it's well known that upgrading firmware on any region free player may permanently make it unable to become region free again. Your only option is to downgrade the firmware to a version that was known to be able to be made region free. Do note that firmware downgrades are not without risk and it is possible to destroy your player by doing this so you are on your own here.
Still worth saying - if you want region free then DO NOT buy Sony or Samsung!
.okay I understand. I have the same issue the thing is that my disk is SUPPOSED to be region free as the amazon liars told me. It does not play though. i do not want region free i just want my blu ray to play a REGION Free or a Uk disk anyways
Similar Threads
Samsung BD-P1600 - Region Free Unlock codeBy kennyt in forum Blu-ray RippingReplies: 42Last Post: 11th Jul 2016, 11:55
Region free hack for Samsung C5500?By letshavefun3 in forum DVD & Blu-ray PlayersReplies: 1Last Post: 28th Feb 2011, 23:21
Samsung BD-C5500 - messed region changeBy GuillermoR in forum DVD & Blu-ray PlayersReplies: 6Last Post: 7th Aug 2010, 09:22
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[Linux] HOW-TO achieve perfect playback at 23.97 or 59.94 Hz with NVIDIA GPU hardware - Printable Version
+- Kodi Community Forum (https://forum.kodi.tv)
+-- Forum: Support (https://forum.kodi.tv/forumdisplay.php?fid=33)
+--- Forum: Tips, tricks, and step by step guides (https://forum.kodi.tv/forumdisplay.php?fid=110)
+--- Thread: [Linux] HOW-TO achieve perfect playback at 23.97 or 59.94 Hz with NVIDIA GPU hardware (/showthread.php?tid=70068)
- JumJum - 2010-04-03
Starstream Wrote:Did this on my XBMC Live system with Philips TV through Putty and worked fine.
- JumJum - 2010-04-03
TheShortOfIt Wrote:Hey guys... using this script too.. changing on 24hz and else works fine but I still have small judder in some movies... every few minutes... like a framerate drop... how can this be?
Without any info, that's like asking how long a string is!
Post your system info, your xorg.conf and a debug log in a new thread and I'm sure it can be figured out.
- seninha - 2010-04-04
I wonder if this will solve my problem.
On every movie I play SD or HD (DVD,ISO or MKV)when a shot goes from one side to the other I always notice that this doesn't go smoothly.
Hardware I use is a Asrock 330 ION.XBMC live is installed on my SSD drive.
Now I wonder if this is a software problem or as something to do with my Panasonic Plasma HD ready TV.
- ThEgOg - 2010-04-04
works great for me once i'd selected the right settings in xbmc.
using asrock 330 and a panasonic pf50, through a onkyo 606 amp.
many many thanks for this, must be easy if i can use it!!
- ThEgOg - 2010-04-04
seninha Wrote:I wonder if this will solve my problem.
what settings have you got selected in the "playback" options??
edit: think i've read your post wrong, have you used JumJum's script?
- seninha - 2010-04-06
No, because before I try this I want to know if my problem is Plasma+XBMC related.
Or as something to do with XBMC+hardware (Asrock 330 Ion)
- furii - 2010-04-06
seninha Wrote:No, because before I try this I want to know if my problem is Plasma+XBMC related.
without knowing your tv and the files you're trying to play that's impossible to say.
- KozmoNaut - 2010-04-07
Rather than creating a new thread, this thread seems to be the best place for the problem with my setup.
I'm using a 24" monitor ([email protected]) connected to a Geforce 7600 GS via dual-link DVI which works fine for just about everything, except I get persistent tearing in XBMC, both in the interface and during playback. I've tried mucking around with the nvidia vsync settings and the sync settings in XBMC to no avail.
I've checked all my settings and they seem to be correct according to this and other threads (and adjusted for a monitor rather than a TV), since both MPlayer and Kaffeine show no tearing at all when playing the same files that XBMC tears on (basically all files), but it's really noticable during the intro sequence of Deadwood with the flames and the horse riding across the screen. Major major tearing.
There is a slight inconsistency in how the refresh rate is shown, too:
Xrandr says 60hz
Nvidia-settings says 59.95hz
XBMC says 60hz, but if I pause a video and press 'o', it says 60.10fps:confused2:
Setting "DebugMode" to "on" in my xorg.conf tells me that [email protected] is what my monitor supports and what I should be getting, but no matter which combination of settings I try, I can't get rid of the tearing in XBMC.
Help? I are confoosed
- seninha - 2010-04-07
furii Wrote:without knowing your tv and the files you're trying to play that's impossible to say.
TV I have is a Panasonic TH-42PV70 HD-ready Plasma and files I try to play are DVD's (iso) , MKV and WMV.
That means that all my movies are giving this "problem"
- furii - 2010-04-07
seninha Wrote:TV I have is a Panasonic TH-42PV70 HD-ready Plasma and files I try to play are DVD's (iso) , MKV and WMV.
i would lean towards your tv being the problem. a quick google search shows several sites listing the resolution as 1024x768 which is not uncommon or surprising for a tv from ~2007. what is surprising is that they advertise it as 1080p when that resolution isn't even full 720p (1280x720). that said, it is also advertised as a multi-system tv which should properly display both NTSC, film and PAL files.
my suggestion is to run jumjum's script and see what kind of results you get. if it's not perfect your tv is most definitely causing your issues.
(the specs i got from third party websites here, here and here. i wasn't able to find it on panasonic's website)
- Esesen - 2010-04-07
I have similar HDReady Plasma TH-42PV8 & ONKYO 507 & ASROCK ION 330HT, try my xorg.conf
# nvidia-xconfig: X configuration file generated by nvidia-xconfig
I use FullHD resolution because i think that Panasonic scaler is better than Asrock scaler.
- seninha - 2010-04-07
Thanks, but can you also guide me how to use your lines
(I know how to upgrade Nvidia drivers using SSH, and thats all )
And , what do you mean by using FullHD? XBMC gives me a resolution of 720. And that 's also what I see on my screen whenever I change to HDMI 1 (thats where Asrock is attached to)
And last, what about this line:
Section "Monitor"
Identifier "Monitor0"
VendorName "ONK"
ModelName "ONK TX-SR507"
Should that give me any problem (i do not have an Onkyo)
- Esesen - 2010-04-07
i plug hdmi Cable to Onkyo & to Plasma only too with this configuration...
(sorry for my English)
- boba23 - 2010-04-08
man you should seriously update your guide.
The line in post #1, which sets the variables for the nvidia-settings tool, activates flipping there ...
With flipping activated, I DO GET servere stutter with h264 vdpau playback. As soon as I turn it off, that stuttering is gone.
I doubt that's only me experiencing that behaviour.
Also for me it makes no sense, that you would turn off flipping in xorg.conf, and then again activate it in nvidia-settings?
One small issue with mode switching here...
If XBMC crashes while playing a 23.97/24hz file, the next time XBMC is run it stays at that mode until the setting is changed manually.
Not a big deal, but it does make the GUI choppy.


text:
Note from Jennifer: At 20 something allergies, we mainly focus on healing allergies and other chronic illnesses with nutrition. This is one time I feel that a serious allergy issue must be addressed and am honored to have Dr. Kaayla Daniel share this guest post with us.
Please help spread the word by sharing on Facebook, Google +, Twitter, Pinterest, e-mail, and other social media channels.
Odwalla's Chocolate Protein Monster made the news last April when four consumers experienced severe allergic reactions. This set off a "nationwide allergy alert", a product recall and a lot of speculation about what might have caused the reactions.
Although all four of the victims were allergic to peanuts, and two were also allergic to tree nuts, the drink contained neither peanuts nor tree nuts. There was also no evidence of accidental or malicious cross contamination at the manufacturing plant.
No Peanuts?
With the possibility of contamination from peanuts unlikely, detectives are considering the potential for cross reactivity. Cross reactivity refers to a reaction that occurs when people allergic to one class of proteins react to another similar in structure.
A good example of cross reactivity is soy and peanuts, members of the grain-legume botanical family. In fact, scientists have known for years that people allergic to one are often allergic to the other.
Food safety experts say they are clueless as to what might have happened in the Odwalla case, but given that the Chocolate Protein Monster drink contained soy protein, and the victims all suffered from peanut allergies, the likely culprit is soy.
Soy Allergies on the Rise
Severe reactions to soy were once rare. Today they are increasingly common, and pose especially high risks to children already afflicted with peanut allergies.
In 1999, the journal Allergy reported that four children in Sweden died after eating a minuscule amount of soy "hidden" in hamburgers. The Swedish National Food Administration promptly warned parents and pediatricians of the soy-peanut link, and stated that children suffering from both peanut allergy and asthma are at very high risk.
Additional risk factors reported included other food allergies, a family history of peanut or soy allergies, a diagnosis of asthma, rhinitis or eczema, and/or a family history of those diseases.
The researchers found it took only a tiny, almost indiscernible, amount of soy to create a severe and even life-threatening reaction in susceptible individuals.
Even more surprising, they discovered severe allergic reactions could happen suddenly and unexpectedly to people with no known soy allergies. In fact, the reactions documented by the Swedes were very similar to the reactions to the Odwalla Chocolate Protein Monster drink.
Cross-Reactions May Cause Death
Tragically, the Swedish National Food Administration warning has not been publicized much in the U.S.
Indeed, the Soyfoods Association of North America – and even many allergy support groups – recommend soy nut butter and soy nuts for children allergic to peanuts and tree nuts.
As a result, few people have heard of the deadly soy/peanut connection, and numerous adverse reactions have been reported.
Tragically, there have also been several deaths.
Six years ago, 13-year old Emily Van Der Meulen died on April 13, 2006. Emily had a severe peanut allergy and assiduously avoided peanuts, but did not know she should also have avoided soy. She died after eating a meal that was apparently peanut free but contained a tiny amount of soy. Just as in the Swedish study, she had not previously reacted to soy.
On February 22, the Weston A.. Price Foundation learned the sad news of the death of a nine year old girl who was allergic to peanuts but died after drinking soy milk:
"My cousin's daughter just passed yesterday after having soy milk for the first time. She was known to have peanut allergies and asthma. My cousin watched what she ate but he was not aware of the relationship between the peanut allergy and soy. She had an asthma attack and did not respond to treatment."
Hundreds of other mystery deaths may also have been caused by the soy-peanut connection. Indeed, it's a question that needs to be asked whenever we hear someone with peanut allergies dies suddenly after eating a hamburger, a burrito or some other food that did not contain peanut ingredients.
The obvious question is, why so many reactions, and why now?
Genetically-Modified (GM) Soy
The main reason appears to be the increased number of allergenic proteins found in genetically modified (GMO) soy.
As reported by Robyn O'Brien on the AllergyKids website: "According to previously undisclosed research and the Peanut Genome Initiative, it appears that in the genetic engineering of soy, a soy allergen was created that is 41 percent identical to a known peanut allergen, ara h 3.
This new allergen, now found in soy, is recognized by 44 percent of peanut allergic individuals. Recent studies out of the University of London conducted by Gideon Lack support this undisclosed research and highlight the role that conventional soy (and soy formula) play in the development of the peanut allergy. . . In the United States, 90 percent of soy now contains these new proteins, chemicals and allergens."
Why hasn't this news gotten out?
Why do so many allergy support groups neglect to issue warnings?
Given the fact that soy ingredients are in more than 60 percent of processed or packaged foods and nearly 100 percent of fast foods, this is simply irresponsible.
Not surprisingly, the reason appears to be the usual principle of profits over people. According to Robyn O'Brien, "Leading pediatric allergists and researchers have been funded by the agrichemical corporation responsible for engineering these proteins, chemicals and toxins into soy."
Sadly, it's been months since the Odwalla case made the six o'clock news. As yet the culprit hasn't been identified, and the investigative spotlight has not yet shone on soy. Right now, the best we can do is hope, pray and join the Weston A. Price Foundation, AllergyKids and other truth telling organizations and make a concerted grassroots effort to share this information with as many people as possible.
Dr. Kaayla Daniel is Vice President of the Weston A. Price Foundation and author of The Whole Soy Story: The Dark Side of America's Favorite Health Food.
For more from Dr. Kaayla, the Naughty Nutritionist™, visit her website where she outrageously and humorously debunks nutritional myths. You can follow her blog at www.drkaayladaniel.com.
©copyright 2012 Kaayla T. Daniel
* * * * *


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CBT sub's huge swollen balls electrostim
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Super Monkey Ball 3D (Nintendo 3DS)
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Super Monkey Ball as you've never experienced before! Join AiAi and friends as you tilt and roll your way in to the world of 3D! Lose yourself in each vibrant world as you twist, turn and aim to collect every banana in the fastest time possible. Everyone's favorite monkeys are jumping out of the screen with excitement. Are you ready to join the fun!?
Let yourself be completely immersed in the world of Super Monkey Ball like
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There was an alarming amount of hand-wringing on Friday about the Patriots screwing over a guy who apparently can't decide whether he wants to play this year. Brian Waters, who was contracted to play for 1.4 million in 2012, hasn't shown up. Not for offseason training, not for minicamp, not for training camp, the preseason or - at the very least - the first regular season game. But the report by the Boston Herald that the Patriots want to shave Waters' salary down to 950,000 brought people out of the woodwork to decry this as another example of the Patriots squeezing a player.
Which is odd, because last week Aaron Hernandez got a fat new contract and people were hurtling over the barricades to salute the Patriots' generosity. But back to Waters. There's a lot of backstory to the situation that's unknown. For instance, a sourcetold me by text Friday night that "what was reported today is not true." Since the reports - or at least the details - have been foggy, I asked whether every report, including the Boston Globe saying it learned "months ago" that Waters agreed to a deal that would cut his salary but pay him bonuses for every time he was on the 46-man active gameday roster, was untrue as well. The source said he didn't know everything that's been reported. I've had multiple sources tell me the notion of a pay cut is inaccurate. But,perhaps contrary towidely held beliefs, solid sportswriters don't just say, "Maybe I'll make up a story about the Patriots requesting Waters take a pay cut" and go with it. The information was passed on in seemingly vague terms to Jeff Howe who reported it for the Herald. Is the "pay cut" Howe reported part of the deal Waters agreed to where he'd be paid for being on the 46? Seems plausible. Frankly, I'd never heard of that agreement nor was it reported locally although the Globe says it knew about it "for months." My take is that the team's been exceptionally accommodating to a player who has shown no inclination to return and has cold feet about leaving home. I also heard rumblings that, at some point in the offseason, Waters was offered much more to return than the 1.4M but he didn't budge. But there's been nothing concrete enough around that to build a story around. As it's been for much of this odd impasse. Additional reasons for Waters' not wanting to leave Texas have been quietly alluded to.As a result,I think it's wise to tread lightly on questioning his commitment to football too strongly because of that. Nobody's indicated if it's a family illness or some other dynamic but a wide berth has been given.
But the fact he's past the point where he was expected to come in - clearly - means the Patriots are at peace with moving on. They've gone through six weeks of work without him. Might the offensive line be best if Dan Connolly was at center and Waters was at right guard? Definitely, presuming Waters has kept himself in football shape. But Connolly is no slouch and he's shown that in subbing for Logan Mankins in 2010 and locking down the center spot after Dan Koppen went down last year.
The offensive line was nobody's idea of a dominant group in the preseason. But the interior wasn't a glaring problem, it was the edges - most troublingly the right edge when Marcus Cannon was over at right tackle. So where are we? There's a 35-year-old guard who can play for the Patriots if he chooses. The Patriots have moved on but they've left the light on for him. It comes down to this: the Patriots have addressed the position. They don't have aneed for Brian Waters. And he needs them even less.


text: +--------------------------------------------------------------------+ Midlands Graduate School in the Foundations of Computing Science MGS 2007 16-20 April 2007 University of Nottingham, UK The Midlands Graduate School (MGS) in the Foundations of Computing Science was established in 1999, and organises an annual Spring School on the mathematical foundations of computing. It is primarily aimed at PhD students, but everyone is welcome to attend, including computer scientists, mathematicians, and participants from outside of the UK. PROGRAMME The programme will consist of ten short courses given over a period of five days. The speakers are as follows: Roy Crole Operational Semantics Neil Ghani Category Theory Paul Levy Typed Lambda Calculus Thorsten Altenkirch Dependently Typed Programming Roland Backhouse Algorithmic Problem Solving Martin Escardo Domain Theory and Denotational Semantics Achim Jung Stone Duality Henrik Nilsson Advanced Functional Programming Colin Stirling Concurrency Theory Steve Vickers Topology and Logic LOCATION The school will be held on the Jubilee Campus of the University of Nottingham, which has won many awards for its design and environmental features. Nottingham is centrally located in the UK, and is easily reachable by road, rail and air (Nottingham East Midlands Airport.) REGISTRATION The registration fee will be around 350 pounds, including accommodation. We also hope to have a number of free places for PhD students. To register your interest in MGS 2007, and be informed when registration is open (the number of places will be limited), please contact the organiser, Henrik Nilsson <nhn at cs.nott.ac.uk>. FURTHER DETAILS Google search - MGS 2007 Web page - http://www.cs.nott.ac.uk/~nhn/MGS2007/ Xmas seminars - http://www.dcs.shef.ac.uk/~georg/mgs_xmas06.html +--------------------------------------------------------------------+ This message has been checked for viruses but the contents of an attachment may still contain software viruses, which could damage your computer system: you are advised to perform your own checks. Email communications with the University of Nottingham may be monitored as permitted by UK legislation.


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Sonic ReducerKelis Food · Various Artists Dio: This Is Your Life · Thee Oh Sees Drop
Rooster Roundabout: This week's music highlights
Yup, watching Belle and Sebastian cover Journey's "Don't Stop Believin'" for a group of kids at Children's Healthcare of Atlanta might be the cutest thing in music this week. You can check that out below, but you might need to have a tissue handy.
A few weeks ago, I posted a track from Buzz Osborne's upcoming solo acoustic album This Machine Kills Artists. And now King Buzzo has posted another snippet from that forthcoming record, which, despite being acoustic, is pretty heavy and dark. But that's a good thing. You can hear his new song ("Drunken Baby") by heading over to A.V. Club.
For those of you that think Lana Del Rey is all that and a bag of stale potato chips, the moment you've waited for is finally here. The singer-songwriter has come out with the first track from her upcoming sophomore LP Ultraviolence. Some blogs have already touted the tune as the "song of the summer." Getting ahead of yourselves much? But I'll let y'all be the judge of that. You can listen to "West Coast" below.
I guess it's only appropriate that I mention tomorrow is Record Store Day. Not only is it a way to promote shopping at your local record stores, it opens your horizons to new and exciting music emerging from the depths of post-Top 40-whatever. Meh ... political correctness aside, I'm not much of a fan of Record Store Day. Most record stores don't carry the releases the peak my interests, and sometimes it makes me wonder whether it's a trick to get you into the stores in the first place. Regardless, you'll probably see me at Mecca tomorrow. But you can read Pitchfork's top Record Store Day Releases by clicking here.
Hmmm... trippy sound effects, a slick guitar riff quietly sneaking around the bush and lush vocals … The Black Keys' new tune (the title track from their forthcoming Turn Blue) isn't half bad. I just may be tricked into hearing more. Could this be a change of heart or a momentary lapse of judgment? I don't know, but it's one good ditty. I'm impressed, Keys. The tune should tide fans over till May 13, when the album hits stores. Preach on!
I'll admit that the only reason I decided to write about this follow-up item is because Kelley Deal is involved. Sure, everyone knows about her sister Kim (Pixies, Breeders, Amps, etc.), but Kelley Deal is a good songwriter in her own right. Check out The Kelley Deal 6000 if you haven't yet. Now Deal is featured on a track by Motel Beds ("Tropics in The Sand"). It's supposed to be on a split 7-inch that'll be out on Record Store Day, but you can hear the track over at Stereogum if you don't plan on participating. Cause why would you do that anyways?
The only stuff I've heard from The Pains of Being Pure at Heart were tracks on a compilation album I got for free at Austin City Limits. And I don't even remember the names of the songs; that's how out of touch I am. But they've shared a new track via SoundCloud, and if I can describe it in one word: cute. It doesn't hit hard like Bass Drum of Death, but it's not overtly twee like She & Him. It holds a nice place in the middle. Let's call it cute without the "e." Wait, that spells "cut." That doesn't work. Just listen to the damn thing.
Well this just seems to be a good week for streaming albums in full ahead of their release dates. You can head over to NPR to listen to Kelis' new record Food. And you can head over to The New York Times to listen to Eels' The Cautionary Tales of Mark Oliver Everett. And then head over to Consequence of Sound to listen to Nas' Illmatic XX reissue. Thank your kind publications for letting you get a sneak peak before you go purchase these albums … assuming you will actually purchase them.
If you ever wanted to see Josh Homme of Queens of the Stone Age play a disappointing gay man … here's your chance.
The diva herself is back to claim her spot on the music charts. Mariah Carey came out with a new single this week, and the accompanying video finds Carey embracing the tropical outdoors as she sings this longing-for-love ditty. The song is titled "You're Mine (Eternal)," and it's pretty much up to par with the stuff she showcased on The Emancipation of Mimi (see "We Belong Together" and "Shake It Off"), which isn't a bad thing. It just means Ms. Carey knows her niche, and she's stickin' to it. Watch the video below.
'Member how I mentioned Thee Oh Sees were going on temporary hiatus … and how frontman John Dwyer was coming out with a solo electronic LP under the name Damaged Bug … and how I expressed regret that such garage-psych weirdos are hard to find—at least good ones. Well now Thee Oh Sees are coming out with a new record titled Drop, which is scheduled to "drop" April 29. While this is good news … great news, in fact … a part of me is still wondering whether the band is back together or just giving fans one last hurrah before they leave us lonely. Head to Stereogum to view the cover art and tracklist, and listen to the album's opening track below.
My first recollection of Dolly Parton was seeing her musical cameo in the film adaptation of "The Beverly Hillbillies." And I remember as a kid being shocked at the revelation that she was the original singer of "I Will Always Love You." No offense, Dolly, but Whitney nailed it. Regardless, I've always had this fondness and appreciation for Parton, and old roommates used to have "Jolene" blasting because the Austin hipsters caught on to old country ... for about a week. Back to the topic at hand, Parton has announced a new album (Blue Smoke) that's set to come out May 13, and the singer iterates that, "On this CD I think there are all of the colors of my life in all the areas of music that you've allowed me to dabble in through the years." For more info, surf on over to Billboard.
Bright Eyes emerged within my circle of friends during junior year of high school, and most alternative, hip kids caught on to Conor Oberst's project pretty quick, trading copies of Digital Ash in a Digital Urn, among others. I really only fell into the deep trance of music love when I heard I'm Wide Awake, It's Morning. (See "Landlocked Blues"). But Oberst is fairly prolific and a great songwriter, so it's only appropriate that he's still putting out material. He's announced a new album (Upside Down Mountain) and has debuted a track off the record. It's titled "Hundreds of Ways," and you can listen to it below.
It looks like Kelis is officially back! I mentioned a while back that Kelis was coming out with a new record (titled Food), which was produced by TV on the Radio's Dave Sitek. And now Kelis has released a track from the album. Judging from these sounds, this woman has come a long way from "Milkshake." Not like that was a bad song or anything, but you get the idea. Listen to "Rumble" below.
The lineup for Austin Psych Fest has surfaced, and just from glancing at it, I can authoritatively predict this year's fest is going to be friggin' sweet. It's okay, Burqueños; Austin's only about a 12-hour drive away … and that's only if you drive like old people screw. But just to give you a minor glimpse as to the reputable names gracing them stages with their presence(s), the performing groups include The Brian Jonestown Massacre (who are great live!), The Dandy Warhols, Black Lips, The War on Drugs, Pure X, The Horrors and much, much more. You can check out the full lineup over at Stereogum. The festival itself happens from May 2 to 4 at Carson Creek Ranch.
I'd never listened to Metronomy before watching their video for the track "Love Letters," off their upcoming record of the same name. And I'll admit that the only reason I even watched the video was because of its director: Michel Gondry. An old roommate had a DVD compilation of Michel Gondry-directed videos from names like The White Stripes, Beck and Bjork, and it's nice to see his aesthetics are consistent and constantly evolving at the same time. As for Metronomy, I probably won't give them more than the time I've already invested in writing this, but you can view the video below.
Brody Dalle: That's a name I haven't come across in some time. Granted, when I was a big fan of The Distillers, it was Brody Armstrong, but that's neither here nor there. Needless to say, Ms. Dalle has been working on some new material for an upcoming album. Music blogs don't seem to know the name of the album or when it comes out, but Brody—we're on a first-name basis—has come out with a new track (titled "Meet The Foetus / Oh The Joy"). That's a mouthful, and you can stream the track below.
I've always been a fan of music biopics. From What's Love Got to Do with It to La Bamba to The Temptations, I'm all about them. And I'm especially excited about the forthcoming Tupac biopic because it's going to be directed by John Singleton (of Boyz n the Hood fame). No word yet on who is playing the man himself or when the film will hit theaters, but this is exciting news nonetheless.
Just in time for spring, The Walkmen's Walter Martin delivers a track that's so twee you might hate it so much that you'll end up loving it forever, snuggling with it under a blanket and singing along to it till you hate it once again. But right now, I love it. And it features Yeah Yeah Yeahs' Karen O, so it's got that added oomph factor. But don't take my word for it. The track (titled "Sing to Me") comes off Martin's upcoming solo LP We're All Young Together, which hits stores and online markets on May 13. That's plenty of time for you to love it, then hate it and then love it again.
I'm sure a lot of people were sad after LCD Soundsystem decided to call it quits, especially after releasing the stellar This Is Happening in 2010. But all good things … Anyways, front man James Murphy recently told Rolling Stone that he's working on the live album for their final show at Madison Square Garden (the subject of the rock doc Shut Up and Play the Hits). You can read more of interview here.
Spotify wins out again (as if taking over the world's streaming sinuses weren't enough). Led Zeppelin's entire catalogue will now be available for free listening on the service. So if you're one of those who's too lazy to get out to a record store (or even purchase the physical albums online), then your wish has come true. So head over to your Spotify page, and climb that "stairway to heaven."
Don't you love it when innovative and interesting musicians become such restless artists that they keep releasing innovative and interesting music over and over? Well, this is from judging one song. But St. Vincent, aka Annie Clark, has announced a self-titled album that's set to hit music markets on Feb. 25. Clark has made the track "Birth in Reverse" available online. You can hear that below.
Don't you just love love songs? No? What's wrong with you? Having already spilled my guts by ruminating over the "myth of the love song," I've just come to accept that it's a notion that never dies. Maybe that's why artists like Beck, Fiona Apple, Blake Mills, Jim James and more have taken to covering famous love songs for the Sweetheart 2014 compilation. You can hear Jim James' cover of Bob Marley's "Turn Your Lights Down Low" below, and head over to Pitchfork to get a full tracklist.
I feel as if Black Lips are one of those bands that you can only really appreciate if you were into them from the beginning. Granted, I've only heard their album Good Bad Not Evil (which was a good album), but after seeing them live at Emo's in Austin, Texas, the underwhelming concert wasn't enough to make me a faithful listener. Oh well … now the band has announced a new record (Underneath the Rainbow), and it was partially produced by the overrated Black Keys' drummer Patrick Carney. Let's just say I probably won't get to this one.
The bossy lady herself, Kelis, has announced that she's coming out with a new album. The album, supposedly titled Food, is being produced by Dave Sitek (of TV on the Radio) and is slated to come out on April 28. Kelis' last album took listeners on a techno-club journey, whereas her previous efforts had a more pop-friendly rap/R&B vibe to them, so who knows what this new record holds? Either way, I'm intrigued.
Mark March 25 on your calendars. It will be a golden day indeed. Because that's the day that a "lost" Johnny Cash album (titled Out Among the Stars) is scheduled to be released. According to Pitchfork, the album was recorded in 1981, and John Carter Cash (Cash's son, duh) took the helm in restoring the recordings, which contain duets with June Carter Cash and Waylon Jennings. This is good news, folks. Good news.
You mean to tell me that kid from Home Alone is in a "pizza-themed Velvet Underground cover band"? Seriously? You're not joking? Come on … And there's a demo??? And a video of their first concert???
Movie soundtracks: Those fuckers can be a hefty bore or a good time … depending on how you look at it. David O. Russell's new film American Hustle is getting the soundtrack treatment that will be available in stores on Dec. 24. But the reason this soundtrack is extra special is because it has a new track by Jeff Lynne … yes, the front man, songwriter, composer, singer, the list goes on and on, of E.L.O. The track is titled "Stream of Stars," and you can give it a listen below.
Wow … I must have been half-asleep if I didn't even know The Sounds had released a new album (titled Weekend) in October. Oops. But now they've premiered a video for their track "Hurt the Ones I Love." This is my first introduction to their newer stuff, which seems to follow more closely to their work on Crossing the Rubicon. But I've always been more partial to the dance-pop-punk craze that dominated Living in America. You can view their new video below.
I could get better with rap. OK, I need to get better with rap. While my musical palate is rudimentary at best when it comes to rap and hip-hop, I always appreciate a good beat, some slick rhymes and good production. And that's what's happening on Busta Rhymes & Q-Tip's track, "Butch & Sundance," off their mixtape The Abstract & The Dragon, which became available this week for streaming and downloading. You can hear the track below.


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Sun Oct 14, 2012, 05:29 PM
alp227 (29,895 posts)
Austerity Protests Are Rude Awakening in Portugal
Source: NYT
Portugal has long been regarded a role model in the grinding euro zone crisis. In return for an international bailout, its government cut services and raised taxes while its citizens patiently endured with little of the popular outcry seen elsewhere in southern Europe.
That is, until now.
Suddenly, the Portuguese, too, have joined the swelling ranks of Europes discontented, following Greece and Spain, after the government tried to take another step up the austerity path last month. For many here, it was one step too far, driving tens of thousands into the streets in the largest protest of Portugals crisis.
As Pedro Passos Coelho, Portugals center-right prime minister, prepares to announce a new budget on Monday filled with still more steep tax increases and public sector job cuts he faces the kind of popular backlash that was, until recently, absent from the political and social landscape here.
Read more: http://www.nytimes.com/2012/10/15/world/europe/portugal-readies-budget-with-job-cuts-and-tax-increases.html?pagewanted=all
16 replies, 3791 views
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Response to alp227 (Original post)
Sun Oct 14, 2012, 05:42 PM
bloomington-lib (946 posts)
1. Is austerity working anywhere? Why do people accept that this strategy works without evidence?
Even with evidence that people's lives actually get worse. And that is what this is all supposed to be about...improving peoples lives.
Response to bloomington-lib (Reply #1)
Sun Oct 14, 2012, 05:55 PM
HankyDub (246 posts)
3. The moneyed interests like austerity
So that's why you don't need evidence that it works. When it fails, they have a brilliant solution...more austerity.
Response to bloomington-lib (Reply #1)
Sun Oct 14, 2012, 06:00 PM
sabrina 1 (62,325 posts)
4. No, it has never worked anywhere, well not for the people. It works fine for corrupt
Bankers as it forces the people to pay their gambling debts and helps to keep their big bonuses coming.
It also achieves a long held goal of the Ruling Class, to destroy Social Programs because they cannot stand to see the people getting anything with money they believe would be better used by investing it on Wall Street.
People are starting to die now in Europe as a result of these draconian and criminal policies. Elderly people in Britain whose benefits have been cut, are unable to make it eg. In Greece some people are committing suicide and there are huge fears that many vulnerable people will die from lack of necessities during the coming months.
Austerity = Crime by the wealthy perpetrated against the working class.
Response to sabrina 1 (Reply #4)
Sun Oct 14, 2012, 06:55 PM
cstanleytech (15,062 posts)
5. Well how do they keep the social programs and pay for them then?
After all the article does say they have been raising taxes yet they appear to be still having problems so why? Taxes not raised high enough? Taxes not being raised for the wealthy? What I am trying to ask I guess is what exactly is the main issue, the one thats causing the majority of the problems.
Response to cstanleytech (Reply #5)
Sun Oct 14, 2012, 07:24 PM
sabrina 1 (62,325 posts)
6. What caused the problems was the Global Financial Meltdown caused by the corruption
that every day we are seeing more revelations about. The people had nothing to do with that, they did not profit from it and they did not give their governments permission to have their pensions and their mortgages used as gambling chips in the Wall Street Casino. And had they known the corrupt practices that were being used risking their homes and pensions, they would have been worried long before the extent of the corruption was revealed.
See Iceland, it is a microcosm of what happened everywhere else. The difference there was they arrested their corrupt bankers and government officials and then installed a new government. They are the only country affected by the Global Meltdown to be on the rebound because they refused to accept the austerity programs being imposed on the rest of Europe.
There was plenty of money to bail out the corrupt banks but not enough to provide life saving Health Care and benefits to those who most needed it? Sorry, but there never should have been bailouts. There is never enough to feed this greed while taking from the people.
Banks should have been nationalized, crooked bankers and politicians who aided and abetted them, should have been arrested and any ill-gotten profits they made confiscated.
There is no reason why Social Programs should be sacrificed so that crooked bankers can be bailed out only to continue their corrupt practices.
What was needed was a program to provide jobs, when people have jobs they pay taxes. Instead all the money was spent on bailing out the criminals and unemployment rose to alarming proportions in countries like Spain and Greece and elsewhere.
These policies were never going to work. They will eventually have to be discarded, as things just keep getting worse. It is pure insanity to continue doing over and over again what failed so spectacularly already.
Health Care is a right in Europe as it should be and should be untouchable as it involves LIVES. But those running things right now do not care about the lives of ordinary people and sooner or later unless someone steps in, the unrest will grow to where they can no longer control it. It wouldn't be the first time in history where Governments put so much unfair pressure on populations that they rose up and took matters into their own hands.
Response to sabrina 1 (Reply #6)
Sun Oct 14, 2012, 11:17 PM
cstanleytech (15,062 posts)
9. Should have? Perhaps, considering how many judges and politicians are in the back pocket
of those same banks then and even now almost all over the world though I dont think its likely.
Response to sabrina 1 (Reply #6)
Mon Oct 15, 2012, 10:54 AM
hunter (27,259 posts)
12. That's exactly it -- corruption.
Austerity won't solve the problem. The corrupt bankers and their government minions must be removed from their offices.
Response to sabrina 1 (Reply #6)
Mon Oct 15, 2012, 11:58 AM
dipsydoodle (42,239 posts)
14. Refused to accept the austerity programs being imposed on the rest of Europe ?
Iceland isn't in the Euro.
Response to dipsydoodle (Reply #14)
Mon Oct 15, 2012, 12:44 PM
sabrina 1 (62,325 posts)
16. What has got to do with these programs being imposed on populations?
WE are not in the Euro either, yet up to the point where OWS took to the streets, OUR Government was talking the same Austerity language, 'share the sacrifice', 'cut social programs' 'the deficit has to be cut so we attack Social Programs'. The Simpson/Bowles Deficit Commission may as well have been run by Brussels. The burden for the gambling debts of the Wall Street Bankers was being put on the people, taking away their benefits etc, just as it has been in Europe. OWS and an election looming at least made the Dems drop that discussion, for NOW. But many believe we will start hearing it again after the election regardless of who wins. These are Global policies.
Argentina wasn't in the Euro either.
The same gang are running all of this, whether you are in the Euro or not. They have their puppets all over the world.
Greece and Ireland and Portuga, Spain, et al all should have done what Iceland did, and so should WE in the US. We now know that the US Treasury helped bail out foreign banks. Why? They did not tell the American people they were using their money to bail out these Global institutions.
Being in the Euro has zero to do with these policies, which every country in or out of it, should resist strenuously. They are a disaster and were when implemented in third world countries, who were also not in the Euro.
Response to cstanleytech (Reply #5)
Sun Oct 14, 2012, 08:11 PM
PSPS (6,929 posts)
8. "Raising taxes" has become shorthand for lazy journalism. What it means is not taxing the wealthy.
Response to cstanleytech (Reply #5)
Mon Oct 15, 2012, 10:43 AM
vinny9698 (1,016 posts)
11. They raise taxes on the middle class
Then the middle class has less disposable income, so they buy less, causing business to lose sales. People get laid off, so less people are working, less taxes are collected.
Response to vinny9698 (Reply #11)
Mon Oct 15, 2012, 12:26 PM
cstanleytech (15,062 posts)
15. Odd, that sounds like Romneys plan to save the american economy. nt
Response to alp227 (Original post)
Sun Oct 14, 2012, 05:44 PM
xchrom (108,903 posts)
2. Du rec. Nt
Response to alp227 (Original post)
Sun Oct 14, 2012, 07:28 PM
lib2DaBone (8,124 posts)
7. Portugal, Spain, Greece, Italy, United States.....
"Austerity" Def: Austerity is the code word that means that the 1% of the wealthiest people in the world will attempt to place the burden of their bad decisions on the backs of the working poor.
Response to lib2DaBone (Reply #7)
Mon Oct 15, 2012, 11:03 AM
kelliekat44 (7,759 posts)
13. Brilliant!!! So true.
After the world-wide 2% have shafted the 98% by stealing, scamming, cheating, lying, drugging, and refusing to pay decent wages for honest hard work, using as war fodder, stealing land and otherwise ripping the off the 98% the 98% need to rise up and fight back with whatever weapons they have and many are doing it by striking, protesting, and voting. More of the 98% will get the message and refuse the homage paid in the past. Reckless abandonment of the world is what the 2% are guilty of and after centuries the people are beginning to rise up.
Response to alp227 (Original post)
Sun Oct 14, 2012, 11:39 PM
2ndAmForComputers (3,527 posts)
10. The lesson here is:
They should have started protesting earlier.