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Anna Wintour, Tom Florio Split Up the Vogue Pie
>> You've got to love Anna Wintour's excuse Rolodex — there's a new article in Forbes about how Vogue's publisher is using Model.Live to keep the magazine relevent — and Anna wasn't available for comment because she's "attending Fashion Weeks in Paris and Milan." Funny thing is, the article came out yesterday, and Milan Fashion Week doesn't start until Saturday . . . with Paris following after that.
But nevermind mincing details — let's look at the bigger picture. Vogue still makes its money from print ad pages, but this year's subscriptions are down — and Tom Florio, the aforementioned publisher, is worried about keeping Vogue relevent for the next generation. That's where Model.Live comes in — Florio created the show with his 16-year-old daughter in mind — without Anna Wintour's involvement: "I'm comfortable with it, because I feel that the principals of what we're doing are based on the same principals as the brand."
He says if Anna didn't like where the content was going, he would kill it, and a Conde Nast spokesperson said that Wintour understands Vogue.TV is run by the business department and stays informed about its programs. But there are a couple of concerns: Will partnering with brands like Express (which sponsors Model.Live) dilute the Vogue brand? And: If online video series produced by the business side become big moneymakers for Vogue, will Anna's sphere of influence at the company be diminished?
*image: source


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The Best Password Manager
Forgetting a password is annoying, but reusing the same credentials for all your online accounts is a potential disaster. The best password managers don't just help you keep up with your digital life; they also make it incredibly easy to protect your identity with Fort Knox-level passwords and regular security audits. After evaluating all the top software, our favorite was Dashlane, the Cadillac of password managers. We also recommend Enpass because its desktop app is completely free and fully functional for the first 20 logins and 1Password for its seamless integration with other apps.
To find the best password manager, we spent over 90 hours poring over the topic. We read a bunch of reviews and guides, evaluated 94 different password-saving services, and surveyed 219 people. There are plenty of popular password managers on the market that would absolutely love your business (including LastPass, RoboForm, and Sticky Password) but many of them don't jive with all the top operating systems (or the cloud).
We narrowed down the options to the ones with the best mobile apps, an extensive set of features, and a top-notch user experience on every device. Most importantly, though, we personally tested 15 different password managers in every form they come in (desktop apps, mobile apps, browser extensions, and any other clients available) for more than a week to find our top three picks.
The Three Best Password Managers
Freemium version
Cost for cloud sync
AES-256 encryption
Master password not stored in the cloud
Vault location options
Secure sharing
How We Found the Best Password Manager
We began our hunt for the best password manager by building an archive of every password manager-type software we could find (94 to be exact). Once the list was complete, our team spent a couple of days gathering a boatload of data for each one, including who the software was designed for, app ratings, feature lists, pricing, and security measures.
Next, we set out to determine which password-management features are the most important by reading the internet's most popular "top 10" lists; investigating security hacks; and dissecting reviews from CNET, PCMag, Lifehacker, and Tom's Guide. We also surveyed over 200 people about their online accounts and web browsing habits. From there, we translated our newfound knowledge into the following criteria, and whittled our selection of 94 apps down to the three best options available:
First, we removed any software options that aren't standard password managers.
We found 94 different applications during our massive Google search of all password managers ever, but many of them weren't a good fit for the average person's password collection. We eliminated open source software like Entropass that requires a certain level of technical knowledge and business-level software designed for teams.
Then, we cut software that didn't have an active changelog.
The landscape of cybersecurity is ever changing, and most developers publish changelogs to document how quickly and how often their software is updated. We removed any password managers with dusty, out-of-date changelogs, as that may be evidence that they haven't updated their software lately.
We removed software that didn't offer support for all (3) major desktop browsers, OS X, Windows 8, iOS, and Android.
A study about how we use electronic devices at home found that we switch among smartphones, tablets, and laptops an average of 21 times per hour and the average American owns four digital devices. So even if you're a brand or platform loyalist, chances are you're going to need a password manager that works across multiple devices and at least two operating systems. For that reason, we removed password managers that aren't compatible with all of the most popular operating systems.
We couldn't recommend password managers that lacked core features on desktop and mobile.
There are two cornerstone features of all password managers: automatic password capture and password generation. The first captures login credentials as you enter them and stores them in your vault. The latter allows you to generate extremely secure and complex passwords with a few customization options like the length and whether it's pronounceable or not. If both of those features weren't available on mobile and desktop, we chopped the manager from our list.
Finally, we removed password managers with an average mobile app user rating of less than 4.0.
We never experienced any real glitches while testing the password managers, but there were several apps (like Sticky Password and Keeper) that weren't entirely fun to use. We're talking about poor layouts, annoying push notifications, and invasive in-app popups.
We found that the app's user ratings were a great way to gauge how satisfied customers were with each password manager's overall performance. And our own experiences while testing these apps aligned with those ratings every time.
Password Managers Reviews and Testing
To evaluate the final 15 password managers, we purchased the apps and extensions for several different operating systems (OS X Yosemite, Windows 10, iOS 9.1, Android Lollipop 5.1.1) and installed them on a number of devices. Then we created a bunch of fake accounts (for sites from Facebook to Mint to Spotify) and spent a week doing all the things you do with a password manager: logging in and out of accounts, auditing passwords, updating passwords, and syncing credentials across devices. Some of our team members also imported their own personal stash of logins — some of which turned out to be pretty vulnerable.
After really digging in, the team unanimously hailed Dashlane as the best password manager money could buy. That doesn't mean you shouldn't consider other options, though. Here's a closer look at each of our finalists to help you decide which one's right for you and your wallet:
Best Overall
Dashlane is the Cadillac of password managers, complete with an impressive automatic password-changing tool and Security Dashboard.
Dashlane 5.0 is a digital wallet, automatic password updater, receipt tracker, and security auditor rolled into one well-designed package. So if you're looking for an option with the most bells and whistles possible, Dashlane is for you. If you're new to password managers, don't let that comprehensive list of features scare you; it's also the easiest one to use. Between the interactive setup process and a bunch of helpful push notifications, learning the ins and outs of the software is a fairly straightforward process.
The most impressive aspect of Dashlane is the automatic password changer. With literally one click, Dashlane logs you into the selected service, changes your password, and syncs your new credentials — there's even the option to change all of your "weak or reused" passwords in one fell swoop. Fresh and regularly updated passwords are the key to online safety, and Dashlane makes that process almost effortless.
Dashlane also excels when it comes to getting a feel for your overall safety. Its Security Dashboard menu serves as a hub that identifies your "Security Score," as well as passwords deemed as weak, reused, old, or compromised.
We did experience an iOS bug during testing that occasionally prevented us from logging in on an iPad Air and iPhone 6 Plus. But, four hours after submitting our support ticket, we received a response from customer service explaining the details of the bug and instructions on how to deal with it.
- It has an automatic password-changing tool that works with a huge selection (we counted 323) of popular sites, like Best Buy, Airbnb, and Basecamp.
- Its Security Dashboard goes far beyond the average security audit.
- Dashlane has a beautiful, flat interface that's easy to navigate and features a thorough quick-start guide.
- Your login credentials are stored on Dashlane's servers, not a third party's, so you don't need to link your account to another service.
- It's the only subscription-based password manager. If your subscription ends, your account reverts back to the freemium offering, which means you'll wave cloud sync and a few other niceties goodbye.
- We experienced an iOS bug during testing.
√ The master password (or a derivative) is the encryption cipher.
√ There's no stored record of your master password key or its derivatives (with the exception of the iOS Keychain when using Touch ID).
√ AES-256 Encryption
- Uses C++-written interactions to mitigate JavaScript-based attacks.
- User data cyphering and deciphering is performed using OpenSSL.
- Dashlane employs a DigiCert High Assurance CA-3 certificate for communications.
See the security whitepaper for a complete breakdown of the security architecture and Dashlane's release notes page for updates.
Best Free Password Manager
The desktop version of Enpass offers full functionality for zero dollars, and cloud sync on your smartphone is a $10 in-app purchase away.
Enpass is an utterly minimalistic software option that's best for someone who needs only the most basic features: a password auditor, password generator, and automatic password-capture tool. But what it does, it does well. We had no real issues throughout our entire week of testing. We did, however, observe a minor nuisance with the browser extension: When you click it to fill a form, it flashes in a way that looks, well, like it didn't do anything. The credentials are inputted just fine, but it took our team a few tries to realize it was actually working correctly.
Where Enpass truly shines is in its freemium plan. The desktop version is available for free and doesn't have any feature restrictions. The catch is that you're limited to 20 logins. Our survey results indicated that the average person frequents four different online accounts every day. So in that case, Enpass is the absolute best option you could get simply because it's free. Want to use your phone too? The app is $10. If you need more than 20 logins, though, and can't afford to pay, LastPass is your next-best option.
- The free desktop version is fully functional for the first 20 logins.
- It's a simple, clean manager.
- There are two versions of Enpass 5.0 for Mac due to an issue with the App Store — one version supports iCloud and the other doesn't.
√ The master password (or a derivative) is the encryption cipher.
√ There's no stored record of your master password key or its derivatives (with the exception of the iOS Keychain when using Touch ID).
√ AES-256 Encryption
Plus, Enpass doesn't host user data on its servers. All encryption and decryption operations are performed locally on your device.
See the security whitepaper for a complete breakdown of the Enpass security architecture.
Best Integration with Other Apps
1Password is a pricier, big league password manager that boasts built-in support for a ton of popular apps.
1Password is a sleek application that's gotten a lot of praise — it was even named an App Store Essential — and when it came to putting the rubber to the road, we couldn't disagree. One of our team members has been a 1Password user for years now, and our collective findings testing the app were no less impressive than what he experienced. Two-factor authentication, password auditor and generation tools, Apple Watch support, multiple vaults that allow you to create different buckets of credentials — it has all the essential features and a little bit more.
Using 1Password also has an interesting perk: AgileBits, its developer, has scored a ton of partnerships with other app makers (like eBay, Slack, Basecamp, and Swarm). That means 1Password is integrated into the login process, which takes the same autofill functionality that you experience on mobile browsers and adds it to apps.
For example, the mobile versions of Safari and Chrome allow you to enable automatic filling. Once it's enabled, you can click the share button while visiting a login page and you will be given the option to launch your password manager. By doing so, your credentials will be entered automatically. However, this streamlined process is not available when logging into apps — unless you're using 1Password. Clicking on the 1Password logo makes logging into a bevy of popular apps just as simple.
The alternative to this is manually switching to your password manager, finding the associated account, copying the credentials, switching back to your mobile browser, and then pasting the info into the box, which can obviously be a great pain.
- Many popular apps have built-in 1Password integration.
- Creating new logins from your phone is much easier with 1Password than any of the competition.
- A single license costs $50.
- It doesn't have a freemium version, and the free trial expires after 30 days.
√ The master password (or a derivative) is the encryption cipher.
√ There's no stored record of your master password key or its derivatives (with the exception of the iOS Keychain when using Touch ID).
√ AES-256 Encryption
Plus, data in transit is thrice encrypted: Once at rest, then by Transport Layer Security (TLS) and finally by 1Password's own transport encryption. All that encryption and decryption is performed locally on your device. Data is non-recoverable if you forget your master password, but recovery keys can be shared with team members.
See the security whitepaper for a complete breakdown of the security architecture.
Runners-Up for The Best
These two runners-up passed our initial cuts, but didn't stand out during hands-on testing:
SafeInCloud is the less-handsome twin brother to Enpass. Both have an almost identical line of core features. SafeInCloud's mobile app costs $5 less than Enpass, but the savings couldn't compensate for Enpass' slightly more navigable iOS and Android apps.
LastPass, a popular, long-standing password manager, also passed our initial cuts. However, it was recently hacked, and the encrypted master passwords of some of its users were stolen. Due to that fact (and its subpar design), we are not able to recommend LastPass over any of the competition.
Our Research
What You Need to Know About Password Managers
Password managers are one of the best (and least used) ways to protect your identity.
Only 7 percent of the people we surveyed use password managers. This information aligns well with a recent study performed by Google, which found that less than one-third of non-experts considered the statement "Use a password manager" to be effective or very effective advice. However, the consensus among experts who participated in Google's study (those who reported having five or more years of experience working in computer security) was that despite the overwhelming negative perception, using a password manager is better than not.
Here's why: Password managers make it incredibly easy to create strong, unique passwords for every account you have — and they're stored in a secure server under a layer of encryption. It takes a great deal of effort and know-how to do that manually, which is why most people wind up creating a weak password, storing it somewhere unsafe, and reusing it for a large percentage of their online accounts.
Your online habits and overall tech-savviness also heavily influence the security of your digital life, not just strong and unique passwords. As just one example, phishing scams are aplenty, and if you wander into a sketchy or unsecured site, you run the risk of falling prey to a variety of threats — even SSL-protected sites aren't impenetrable (XSS/SQL Injection, Cross site scripting, Active Mixed Content, etc.).
The bottom line is that if used incorrectly, password managers can create a single point of failure. But if used correctly — with 100 percent commitment — they are one of the best ways to prevent identity theft and to secure your online presence.
Password managers create a single point of failure, but they're still worth it.
Throughout our research, the most notable concern regarding password managers was that they're often considered a single point of failure. And technically, that's true. If a hacker managed to get ahold of your master password, your entire vault could be compromised. However, there are a couple of reasons why this scenario is highly unlikely.
The first is two-factor authentication. With this feature enabled, a second unique code (in addition to your master password) is required to log into your account on a new device. That means that a hacker would have to steal your master password and one of your devices in order to gain entry into your vault.
Secondly, your data is always encrypted. That means that even if your password manager's server is compromised, a hacker won't be able to make heads or tails of your data without the cipher (your master password). Cracking encrypted data takes a great deal of time and computational firepower — enough time for you to reset passwords for your most sensitive accounts.
There's no free way to sync your credentials on multiple devices.
Syncing your credentials across multiple devices is going to require a premium plan, regardless of which password manager you choose — they all charge for cloud sync, every last one of them. If you only want to use a password manager on your desktop or laptop, however, you might be able to get away with using a freemium option from LastPass or Enpass.
To give you an example of what life is like without cloud sync, imagine that you used the Enpass Chrome extension on your laptop to save your Yelp login credentials. Then, later that evening, you needed to login on your phone for the first time to leave a review at the new dive bar where you just dined. The Enpass app on your phone can't talk with the Enpass application on your laptop, and your password will not be there.
That leaves you with two options: Input the credentials from memory, or log into Enpass from the web and grab the credentials from there. And every time you change passwords (which should be often), you'll be required to manually update them on all your other devices. Cloud sync prevents such frustration.
The Best Password Managers: Summed Up
Take Action
- Count up your memorized passwords: Ask yourself how many online accounts you've devoted to memory. If it's more than three, you should probably consider using a password manager. If not simply for the convenience, use one to ensure that your identity is well-protected. When you're relying on your memory, you're passwords probably aren't as complex and secure as they could be.
- Give the password manager a try: Most password managers will give you a free spin. Start with a freemium version or sign up for a 30-day trial to get a taste of the super-secure, auto-filled good life.
- Audit your passwords: Once you've got your hands on a password manager, use the app's tools to see how secure you are, then upgrade any old, reused passwords. If you want to go the extra mile, take some time to create your own password update schedule and say goodbye to "Password123" forever.

subset: commoncrawl


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Wednesday, May 23, 2007
Bocor lagi
I was a bit disappointed to see that all the fuss was about what I thought was a major floor cleaning exercise.
But then I heard some mutterings of "bocor lagi" from the cafeteria patrons. Things were beginning to look up. Finally I could contribute to the bocor discussion with a posting of my own. The press photographers, who were probably stationed at the courts to cover another newsworthy story, had a field day recording the incident from all angles.
Other people, maybe bloggers too, had the same idea and started snapping away on their camera phones. I made a note to myself not to let anybody else get the scoop on this and post this fast. Remember folks, you read about it first on Almost Anonymous.
My own efforts at investigative journalism uncovered the source of the minor flood. A pipe situated in a small "service room" burst. Apparently nobody realised it until some lawyers complained that their thousand-Ringgit Loake shoes were feeling a tad damp.
At least that kept the press occupied until they did some proper legal reporting. Moments later, they swarmed around Mr. M. Manoharan, a prominent lawyer, for some comments on perhaps the latest scandal on a Hindu person buried in a Muslim cemetery, corrupt municipal councilors or chicken smuggling. I couldn't actually hear him but from his fiery discourse I thought this must be something big.
I collared Mr. Mano himself in the lift and I asked, "What public interest case are you doing today, sir?" He said, "No lah. They just wanted to ask my opinion on this bocor thing." Hmmph. Any publicity is good publicity I suppose.
Whatever it is I'm sure members of the legal fraternity and court staff will enjoy the next few days eating in very very very hygienic conditions.
24 comments:
That was fast! I only saw the report about it on TV3 a short while ago and you've already posted it. With actual site pictures too. :)
TV3 actually showed one of JDCC's security guard having a war of words with a reporter. Kinda Ah Beng gangster-like. Did you catch that?
- Wed May 23, 03:07:00 PM GMT+8
- A Babe Of Very Little Brain said...
belum akil baligh, dah bocor.
- Wed May 23, 03:37:00 PM GMT+8
- Mr Incognito said...
blackcadillac73 >>> can't let TV3 get the scoop. i did see the guards having a few words with the photographers but I didn't recall any harsh words. maybe TV3 hyped it up to make the story more interesting.
Babe >>> *scratches head* ah i see. maybe people should walk around with rubber boots and hard hats over there
- Wed May 23, 04:30:00 PM GMT+8
- NinJaMoo said...
So how did you manage to be at the right place at the right time? Hmm?
- Wed May 23, 08:53:00 PM GMT+8
- Theta said...
Mr Incognito,
Are you a litigant as well? Or acting as an expert witness? Or attending a personal hearing that day?
There is always a story to hype up for the public, in this particular episode an actual leak, which should rightfully be the case, instead of minding another person's monthly leak! ;D
- Thu May 24, 05:04:00 AM GMT+8
- Diesel Electric said...
Bocor? Mana ada bocor? Batu Gajah tiap-tiap bulan ada bocor lah!
Your report, and the imbecilic response of certain MPs to previous bocor cases only shows that this country has a loooooooooong way to go.
Thank God for Britain! :)
- Thu May 24, 08:56:00 AM GMT+8
- Mr Incognito said...
NinJaMoo >>> i have a nose for this kind of thing. btw Utusan Malaysia has the same headlines today. so yesterday.
Theta >>> none of the above. sorry. try again. I don't think this will be the last we hear of leaks, real or metaphorical
Diesel Electric >>> somebody should photoshop a picture of those MPs with sanitary napkins plastered across their mouths
- Thu May 24, 09:16:00 AM GMT+8
Lawyers wear thousand ringgit Loake shoes? Owh, I've chosen the wrong profession! My sandal is only RM38.
- Thu May 24, 02:15:00 PM GMT+8
i am very envious.
- Thu May 24, 05:39:00 PM GMT+8
- izso said...
LOL! Ninjamoo sounded like it was you who burst the pipes for the story.. LOL! XD
Anyway, your pics are way more comprehensive than the newspapers. Hehe... good one dude. Or duddette. Whichever one you are.
- Thu May 24, 09:08:00 PM GMT+8
- Daily Nibbler said...
Thanks for the pics! Bocor, bocor, bocor,....
- Thu May 24, 10:09:00 PM GMT+8
- Mr Incognito said...
Dayang >>> maybe only one or two. the others probably wear dr. cardini
lil ms d >>> of the shoes?
izso >>> damn, i thought nobody would have guessed that i twisted the tap a bit too hard. dudette? MISTER Incognito lah
Daily Nibbler >>> u r welcome :) watch this space for more scoops!
- Thu May 24, 10:27:00 PM GMT+8
- UglyButAdorable said...
u might want to have a wind of change..hehh..hehhh...
- Thu May 24, 11:01:00 PM GMT+8
- akula said...
Woit, selfpimping journalism. Is it a new branch of masscomm that I am not aware of?;)
- Fri May 25, 01:00:00 PM GMT+8
- SP said...
So what's next hmmm? pipe bocor, MP bocor ... a nation of leaks of all sorts! Integrity bocor. National Unity bocor.. and the list goes on ...
- Fri May 25, 01:58:00 PM GMT+8
- Mr Incognito said...
UglyButAdorable >>> i'm quite happpy with the way things are, thank you
akula >>> this is blogland, bro. anything goes.
SP >>> add jamban bocor to your list (see today's Sun headlines)
- Fri May 25, 02:10:00 PM GMT+8
- pugly said...
How was YOUR thousand-ringgit Loake shoes? :-)
- Sat May 26, 10:50:00 PM GMT+8
- Blabarella said...
Loakes? Hmm, .. you may not be a litigant, but perchance you are a litigator?
- Sun May 27, 05:53:00 PM GMT+8
I went to Fitness First IOI Mall on Friday. Then tengah berlari-lari kat treadmill, suddenly, Whooooosh!!! Air mencurah2! Siling bocor!!! Gabra gila because cardio machines pun basah kena air, dahlah all the machines connected to power points. So, semua cepat2 lari masuk changing room. Seee.... issue bocor memang berleluasa...
- Mon May 28, 10:23:00 AM GMT+8
Mat scoop la you...should get the bachelor guy of the TV station to hire you lah since they are soo lambat! ;)
- Mon May 28, 11:03:00 AM GMT+8
- Mr Incognito said...
pugly >>> i binned them after I stepped into some mud last year
Blabarella >>> maybe u should be one since u r good at cross-examining.
Dayang >>> too bad not the other way round. bocor in changing room and everybody had to cepat2 lari keluar
miss golden tan >>> why don't u do some scoops and get his attention?
- Mon May 28, 11:13:00 AM GMT+8
- Blabarella said...
No affirmation but hmm, no denial either, heheh.
Me? Already in the profession lah (but never a litigator, nay). Well, WAS in the profession until I surrendered my practising certificate to became a trailing spouse as of last year. A much, much better career option, mind you!! :)
- Mon May 28, 03:10:00 PM GMT+8
- Mr Incognito said...
Blabarella >>> i'm much more interesting myterious. btw, a much better career option, indeed.
- Mon May 28, 04:45:00 PM GMT+8
- urpinkrockprincess said...
god..i dun get it..bocor? am i too slow? perhaps..huh~
- Thu Jun 14, 09:07:00 PM GMT+8


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text:
In responding to O'Brien's inquiries, Paul could have simply apologized to the Huntsman campaign, taken responsibility for the tweet and moved on to talking points about the Fed and Iran. But he instead pulled a Ron Paul, denying, blustering, parrying. After O'Brien read aloud the offending tweet to him, the candidate replied:
Well, I didn't quite understand even what you just read but, uh, obviously I didn't send it, so I don't even understand. I'm sorry I didn't catch the whole message about Jon Huntsman. I haven't talked about Jon Huntsman in a long time, so I don't know what's going on there.
The "snitty" message, O'Brien stressed, was sent out "under your name, under your Twitter handle." More Paul stonewalling ensued, this time with a dismissal of the issue's "importance." "It just seems to be irrelevant to me," he snarked.
Not. O'Brien was right to push hard on this question. Here's a campaign that thrives on Twitter and it's sending playground-level messages to another candidate over its medium of choice. That's news.
Also news is that Paul, again, shows little concern or control over scribblings transmitted under his name. Too bad Paul didn't seize this opportunity to highlight the progress he's made on this front. Decades ago, such scribblings, in Ron Paul's newsletters, demeaned entire classes of people, chiefly minorities and disadvantaged groups. In this case, the scribblings merely tweak a sole privileged white man who has failed to catch fire with Iowa voters. Hey, that's an improvement, Paul could have argued.


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text:
" Brand: King British / Food Type: Fish Food "
* Prices may differ from that shown
As mentioned in a previous review I feed a combination of flake and pellet food to my fish, this is my current pellet food choice.
Originally I bought my small tub two years ago when I first started keeping a few guppies in a small tank, but despite the staff at Pets at Home recommending it I soon found that the pellets were too big for the small fish and probably contributed to them dying quickly (of course living in a student flat and finding that the water smelt of alcohol would have done nothing to help the last few fish I had). But as the packaging does say on it that 'very small fish should be fed King British Flake Food' so the mistake was partically mine.
Later as my fish tanks grew larger in capacity and the fish in them have grown also I have found that the pellet food makes a good alternative to simple flakes. The pellets tend to sink when they are sprinkled on the water, so are more suited to mid water or bottom feeders, this sinking also means that they are caught in the currents in the aquarium meaning the fish can feed more naturally chasing their food. Something my larger pair of Angelfish and Tiger Barbs seem to relish, ignoring the flake food and chasing the pellets until they are all eaten or settled in the gravel (from where they'll eat them later). Whereas my smaller pair of Angels dont particularly seem to enjoy the pellets, in that aquarium its the Emperor Tetras which seem to enjoy them more but in both tanks the smaller fish like the Neon Tetras and smallest Emperors tend to ignore the pellets simply because they are too large for them to eat.
The packaging is a fairly typical tub, made of blue plastic and sporting fairly typical labels with the name of the product and feeding recommendations.
This product claims to be
For healthy active and colourful fish
Exceptional Nutrition
High Protein semi floating formula.
That the pellets are semi floating seems odd how can something semi float? As any scuba diver knows things float (are bouyant) or sink (have negative bouyancy) until something is added to make them neutrally bouyant, none of these qualities apply to these pellets but they do sink slowly thus allowing the fish to chase them, so I can forgive the slightly odd claim.
It also states that the food contains Immuno boosters, comprising of seaweed extracts.
Opening the tub there is a slighty fishy smell but it isnt a strong one, the pellets themselves are very small with a reddish brown colouring.
The feeding instructions are add two or three pellets several times a day, sufficient to to last a few minutes per feed. Now to me this seems a very small amount, so I feed a small pinch of pellets at a time, probably more than 2 or 3 per fish (since I presume this is what is meant) and then add more only when all the flakes fed at the same time have been taken from the surface. But I am careful that I dont over feed, my fish are greedy little so and so's and especially this weekend there were friends looking after the dog who told me they had fed the fish because they looked hungry when I checked in the tank the turquoise gravel was liberally speckled with pellets which the fish were ignoring (yet still doing the 'wiggly feed us' dance). Over feeding is bad for the water quality so I would rather feed more regularly in small amounts.
If I remember rightly I paid about £3 for a 45g tub, which is currently about half full. The tubs have a recommended use by date which for mine is the end of this month, as they will last around 2 years from when you purchase them, as I only started using the pellets again a couple of months ago I would say that they are good value as long as you are going to use them all. I have not seen other size tubs avaliable.
Overall I would say that if you want to feed pellets these seem a good option but at the end of the month I'll be throwing this tub away and quite possibly trying another brand it depends what I see in the shop when I go to replace them (other than the gorgeous 300 ltr tank I want but have nowhere to keep), if another brand seems less appreciated by my babies then I will happily switch back to King British pellets.
King British make a full range of foods from pellets and flakes for tropicals, to marines, and coldwater (goldfish) as well as algae wafers and freeze dried bloodworms, I have only seen the range sold at Pets at Home or B.A.S in Bolton but Im sure it can be found at other specialist shops.
High protein semi floating formula


text: Thought I'd pay the Adox site a visit after quite a while, and was surprised to see Atomal F advertised. I have used Atomal FF since I started B&W photography and absolutely loved the results I got on all film types. One lucky APUGer grabbed the last batch I had (I'm not using it quite so fast since moving to BKK), and I was resigned to never seeing it again. Is this the same as Atomal FF? (I did note only the one F in its name.) Anybody tested it and able to report their findings/results? If it's as good as the original, I'd be happy to part with some $$ to snap some up.


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A Guide to the Precursor Award Season: Keep Your Emotions in Check
by Joey Magidson
November 7, 2013
Ladies and gentleman, we're now entering a critical and (at least for me) fascinating section of the awards race which is known as the precursor season. Between now and when Oscar night roles around in about five months, nearly every awards body, critics group, and guild will chime in with their best and worst in 2013 cinema. Some unexpected films will rise in the Oscar race, while some will surprisingly tumble. Mostly though, things will begin to crystalize, so I felt that it was prudent to explain what's about to go down and how to properly filter it. That way you don't get too high or too low depending on how the new season goes.
So far, we've only had the Gotham Awards announce their nominees. They're pretty small time in the grand scheme of things, but they work as an example of why you need to keep an even keel about this. Yes, 12 Years a Slave led with three nominations and will likely pop up just about everywhere going forward, but nominations were also given out to films like Afternoon Delight, Concussion, and Upstream Color. Does anyone actually think that they'll come even close to sniffing Oscar gold? No? Good, let's move on then.
As the season progresses, you'll see more independent minded precursors like Gotham continue to cite smaller flicks, while others will go the more Hollywood/mainstream route. That doesn't even include bigger fish like the Golden Globes (which will get their own post later this year) since they have their own, sometimes bizarre, way of doing things that sometimes involves silly things like bribery. Basically, there are different types of precursors, and they're not at all created equal.
One early one that actually does impact things a bit is the National Board of Review, which will come at the beginning of December. For better or worse, they act as tastemakers to some degree, and while plenty of times they take a liking to something that goes nowhere, they also can make a contender. One perfect example is how Woody Harrelson was more or less a fringe contender for Best Supporting Actor with The Messenger in 2009, but once he won NBR, he was basically on a straight line towards an Oscar nomination. It can happen just as simply as that.
The early part of the season basically sets the stage, while at the end of the season the guilds really just lock contenders in to one degree or another. This makes for an awkward almost secondary calm before the storm, as the race obviously evolves a bit with each precursor, but only a select few actually wind up being real sea changes in the season. What you notice more in the early part of the game are trends emerging.
For example, a lot of people are simply expecting 12 Years a Slave to clean up all season long. If that's what happens consistently, that'll be a trend. The same goes for which of the many contenders for Best Actor wind up getting snubbed. We might see the precursors mix and match the names all for the rest of the year, but we also might see the same five or so emerge consistently, giving us an idea of who's got momentum, and that's one of the key elements in the Oscar race.
You rarely see contenders slip into the Oscars without momentum. Certain nominees may not be juggernauts, but it's never their first mention. Precursor nominations and wins get them noticed by the folks who actually are members of the Academy and vote. For a sports analogy, the precursors are almost like the minor leagues and Oscar voters are like scouts right now. Their binoculars and radar guns are trained on the films and performances they think can make it in the majors. If they succeed down here, they might get the call up to the big leagues.
The other thing to keep in mind is that one precursor on its own means nothing. If you're rooting hard for a particular film or performance to get an Oscar nod and it misses at a precursor, it doesn't mean the Academy will ignore it and not bestow a nom on it. At the same time, if a long shot shows up once or twice on the precursor circuit, it's not suddenly a top tier contender. In short, keep your emotions in check.
If you're like me, you follow every single one of these things and keep track in order to try to make sense of it all, but I wouldn't expect most of you to be like that. Basically, the guide to the precursor season is simply to keep some manner of perspective about things. When big events go down, I'll let you know about them, trust me, but don't get too up or too down about any one award. It's a marathon, not a sprint. Thoughts?
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CORNISH-L ArchivesArchiver > CORNISH > 2012-01 > 1326152484
From: Joy Langdon <>
Subject: Re: [CORNISH] Mother's age at Birth
Date: Mon, 9 Jan 2012 23:41:24 +0000 (GMT)
References: <D0BD2A091D2541668896F679FB18776E@user2f690b1bcf>
In-Reply-To: <D0BD2A091D2541668896F679FB18776E@user2f690b1bcf>
I suspect you have identified the marriage of James STEER aged 59, widower and Jane DALE aged 49, widow, St Just in Penwith 19 Aug 1845 as the marriage of Emily's parents. The censuses on http://www.freecen.org.uk/cgi/search.pl reveal that this isn't the case.
In 1851 this couple are living at Princess Street, St Just with Elizabeth Jane DALE , daughter in law (a term which was used for stepdaughter at that time) aged 14 and a visitor, Jane CASLEY aged 14.
In 1851 Emily, aged 3, is living at Dock Lane, Madron with her mother Jane aged 41, Mariner's wife, and siblings Jane aged 20, Elizabeth aged 14, Mary Ann aged 12, James Thomas aged 9 and Louisa aged 6.
Just to confuse things, there are several baptisms which match the names, ages and birthplaces of Emily's siblings but the mother is Jennifer (i.e. Louisa STEER 28 Sep 1845 St Just in Penwith, parents James and Jennifer, occ sailor and Mary Ann STEER 06 Dec 1838 Falmouth parents James and Jenifer)). Jane was used as a variant of Jennifer. There is a marriage of James STEAR and Jennifer REYNOLDS 7th Jan 1830 St Just in Penwith.
Obtaining the birth certificate would confirm Emily's mother's maiden name.
From: Jill Penrose <>
Sent: Monday, 9 January 2012, 21:05
Subject: [CORNISH] Mother's age at Birth
My ancestor, Emily Parker Steer's birth was registered December qtr 1848 at Penzance.
St Just church register says she was baptised 3 June 1848 parents James and Jane Steer.
In 1872 her marriage record says she was aged 24 therefore confirms born 1848.
If this is correct her mother was aged 50 and father 61 when Jane gave birth to Emily which would be very old by todays ages.
My question is would the birth certificate give the true parents? I am wondering if Emily is actually the daughter of James' older daughter by his first marriage.
Any suggestions?
Regards from
Jill in New Zealand
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This thread:
|Re: [CORNISH] Mother's age at Birth by Joy Langdon <>


text: I Am a Being of Light and Dark Me Too Personal Stories, Advice, and Support 17 People Report Group Sort popular recent I'm Darker Than Lighter If anything, I'm probably more closely related to being Vegeta than anyone else. I can be pretty proud and I'm definitely stubborn as hell. Not to mention that I hold grudges.Regardless, I know which side I'm on. deleted 26-30 0 Mar 2, 2012 More Stories


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Thursday, Oct. 17
• The Rotary Club of Dove Mountain meets. Details: 7 a.m.; The Highlands Club House; 4949 W. Heritage Club Blvd.; email@example.com.
• The Rotary Club of Oro Valley meets. Details: 7 a.m.; Oro Valley Country Club, 300 W. Greenock Drive; email@example.com.
• The Rotary Club of SaddleBrooke meets. Details: 11:30 a.m.; SaddleBrooke Country Club, 64485 E. SaddleBrooke Blvd.; $15; firstname.lastname@example.com.
Friday, Oct. 18
• Arizona Small Business Development Centers holds the 4th Annual Veterans Conference with resources for veterans. Details: 9 a.m.-3 p.m.; Tucson Police Department, Westside Service Center, 1310 W. Miracle Mile; firstname.lastname@example.com or 620-1241.
• The Joel D. Valdez Library holds a Proposal Writing Basics Workshop. Details: 9:15 a.m.; 101 N. Stone Ave.; free but registration required; 791-4010.
Saturday, Oct. 19
• The Marana Chamber of Commerce holds a Grand Opening and Ribbon Cutting at AZ Air Time. Details: 11 a.m.-2 p.m.; 3931 W. Costco Drive; 682-4314.
• SCORE offers the Financial Projections Workshop. Details: 1-4 p.m.; SCORE Office; 1400 W. Speedway Blvd.; $40; 505-3636.
Tuesday, Oct. 22
• The Rotary Club of Marana meets. Details: 7 a.m.; Nana's Kitchen, 8225 N. Courtney Page Way; 395-2508 or www.maranarotary.org.
• The Marana Chamber of Commerce holds its Fourth Tuesday Mixer. Details: 5:30-7:30 p.m.; Chopstix, 8225 N. Courtney Page Way; free; 682-4314.
Wednesday, Oct. 23
• The Marana Chamber of Commerce holds a Community and Regional Update. Details: 7:30-9 a.m.; Marana Operations Center Community Room, 5100 W. Ina Road; free; 682-4314.
• The Kiwanis Club of Oro Valley meets. Details: 6:45 a.m.; Fairfield Inn and Suites, 10150 N. Oracle Road.
• The Oro Valley Optimist Club meets. Details: 11:30 a.m.; El Conquistador Country Club, 10555 N. La Cañada Drive; www.orovalleyoptimistclub.org.
• The American Business Women's Association (ABWA) El Sol Chapter meets. Details: 5 p.m.; Viscount Suites Hotel, 4855 E. Broadway Blvd.; $20; www.abwa-elsol.com.
Thursday, Oct. 24
• The Greater Oro Valley Chamber of Commerce holds its Monthly Mixer. Details: 5-7 p.m.; The Forum, 2500 N. Rosemont Blvd.; $10, $5 members; 297-2191.
Saturday, Oct. 26
• The Tucson Hispanic Chamber of Commerce holds Noche de Exitos Gala and Bi-National Business Awards. Details: 6 p.m.; Casino del Sol, 5655 W. Valencia Road; $99; 620-0005.


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TAM: We would like to introduce a new blogger to the TAM Family, Carl Zimmer. Zimmer is a blogger and writer for both The New York Times and Discover Magazine, which also hosts his blog, The Loom. Zimmer writes on a wide range of scientific topics, including tattooing. He is the author of 12 books, the most recent of which is Science Ink: Tattoos of the Science Obsessed. Time for everyone to get our "geek" on… Read More »
Daily Archives: May 30, 2012
Tattoo Expo Napoli 2012 Preview
The 9th edition of Tattoo Expo Napoli is arriving, the big summer event in Naples (Italy) dedicated to the world of body art, culture and much more. The Expo, organized by the Cultural Association Napolindelebile and Micromutazioni in the huge spaces of Mostra d'Oltremare in Naples, this time will host 170 international tattoo artists, international suppliers in the business and a complex series of events scheduled during the three days (29, 30 June and 1st of July) like: art expositions, workshops, live performances (including suspensions, bondage, live music),the airbrush show and many contests… just to mention some of the attractions… Read More »


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Spotlight on Pro Bono
There is an impressive and growing array of pro bono work opportunities available to today's bar. For example, many attorneys prefer counseling and transactional work to litigation. At the Pro Bono Luncheon at the Section's Atlanta conference in November, Rachel Spears, Executive Director of The Pro Bono Partnership of Atlanta described how her organization helps these attorneys find pro bono work that is similar to what they do every day. The Pro Bono Partnership of Atlanta represents nonprofit organizations exclusively. "Like any employer," explains Spears, "nonprofits regularly have employment-related legal issues; and when they do, they often come to us for legal counsel."
"Our volunteer attorneys assist nonprofits by, for example, drafting employee handbooks, advising on employment decisions, responding to EEOC notices and Georgia Department of Labor inquiries, classifying workers as contractors or employees and counseling on overtime policies. About 20% of our work is employment-related; but we also assist nonprofits with contracts, corporate governance, intellectual property, real estate, tax, technology and other legal issues."
In order to be eligible for Pro Bono Partnership services, nonprofits must have 501(c)(3) tax-exempt status and serve low-income or disadvantaged individuals in the greater Atlanta area. Typical clients include afterschool programs for at-risk youth, transitional housing providers, health clinics for the uninsured and advocacy organizations for the disabled. If the Pro Bono Partnership of Atlanta did not exist, most of these organizations would have to navigate their legal issues without assistance from an attorney.
In addition to providing direct legal advice to clients when a legal issue arises, the Pro Bono Partnership strives to be proactive by educating all nonprofits about potential legal pitfalls before they happen. They provide information on topics such as the proper use of independent contractors, the Fair Labor Standards Act, social media policies and best practices in hiring and firing through newsletter articles, monthly webcasts and free in-person workshops. Many of these resources are available on their Website.
Spears explained that the Pro Bono Partnership has made a difference. "Since we opened in 2005, we have advised about 550 nonprofit clients on over 3000 legal matters with assistance from over 1500 attorney volunteers. Many of our volunteer attorneys are in-house at Atlanta-area corporations. In 2011 alone, we provided about $2.3 million worth of free legal services to nonprofits."
Because employment issues are so common among Pro Bono Partnership clients, they are always looking for employment attorneys to volunteer. If you are interested in volunteering with Pro Bono Partnership of Atlanta, please email firstname.lastname@example.com. There are programs similar to the Pro Bono Partnership in most major cities around the country, including New York City, Chicago and San Francisco. For a list of such programs, go to their Helpful Links page. If there is no such program in your area, you can contact a nonprofit directly and ask if they can use pro bono assistance. There is a good chance they will say yes.
This article was prepared on behalf of the Section's Pro Bono Work Committee by William Bush, who represents employees at the Legal Aid Society of Middle Tennessee office in Cookeville, Tennessee.
Opening Page
Comments from the Chair
Special Feature: How a Natural Disaster Impacts Leave Taken under the Family and Medical Leave Act
Tech Corner: Whose Social Media Account Is it? The Employee's or the Employer's?
Flash Co-Chairs:Jeremy J Glenn, Meckler Bulger et al | Elana Hollo, National Labor Relations Board | Katherine Huibonhoa, Paul Hastings LLP | Amy F. Shulman, Broach & Stulberg LLP
American Bar Association Section of Labor and Employment Law321 N Clark | Chicago, IL 60654 | (312) 988-5813


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Better DVD playback - nVidia or ATI?
Subject: Graphics Cards | February 1, 2006 - 12:13 PM | Jeremy Hellstrom
Extremetech compares the quality of DVD playback from ATI and nVidia using the filters in PowerDVD as the base for their experiment. If you are setting up a media PC or just starting to watch more DVD's via your PC you might be interested in the results. Mind you, since they give away standalone DVD players with TicTac's and just about anything else you buy now, maybe you still watch them without the PC at all.
"Graphics cards are all about 3D, right? Read most graphics card reviews, anywhere on the 'net or
in print, and you'll see they focus almost entirely on 3D performance and features. We understand
this because that's why we would buy these expensive devices in the first place. Windows 2D
performance is essentially a solved problem (at least until Windows Vista), so what else is there
for the graphics card to do?
One word: video. Video on the PC is tremendously popular. Peer-to-peer networks share large video
files while web-based video (including the ever-popular movie trailers) expand rapidly thanks in
large part to broadband connections. Now with the PC's emerging role as a media device for the
living room, video becomes even more important."
Here are some more Graphics Card articles from around the web:
src="http://www.pcper.com/site_gfx/icon-video-small.gif" alt="Click Here to go to Video Cards" border="0">
Graphics Cards
src="http://www.pcper.com/site_gfx/icon-video-small.gif" alt="Click Here to go to Video Cards"
- Nvidia ships their worst-kept secret: The GeForce 7800 GS @ NGOHQ
TARGET="_blank">Walton Chaintech Announced the Newest GeForce 7 Series Graphics Cards @
- nVidia Quadro
FX4500 @ LostCircuits
Dual @ [H]ard|OCP
VF700-Cu LED VGA Cooler @ ThinkComputers.org
All-In-Wonder X1900 @ HotHardware.Com
All-In-Wonder X1900 @ Motherboards.org
Card Series Part II @ Legit Reviews
RX1800XT-VT2D512E 512MB-Version RX1800XT Videocard @ PCstats.com
TARGET="_blank">PowerColor X800GTO16 @ Overclockers Online
All-In-Wonder X1900 graphics card @ Techreport
TARGET="_blank">Visiontek Radeon X1900XTX @ GDHardware.com
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The Highland Lakes Legacy Fund, a family of funds at Austin Community Foundation, was created a decade ago to efficiently build on the Highland Lakes communities' abundant strengths. With strong community-based support and leadership, the Legacy Fund connects local donors with local causes, invests assets for tomorrow and fosters philanthropy.
The Legacy Fund has grown to include 85 funds totaling more than $4 million and has provided grants to nonprofits serving the community and scholarships to students pursuing their educational goals.
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Sandra Del Kehoe
Sandra Del Kehoe, 67, of Bristow, Va., died Sunday, Oct. 20, 2013, at Capital Care Hospice in Arlington, Va.
A Mass of Christian burial is at 11 a.m. Tuesday at St. Leo The Great Catholic Church in Fairfax, Va.
Family will receive friends following Mass during a reception at St. Leos.
In lieu of flowers, donations may be made to the Virginia Knights of Columbus charities.
Mrs. Kehoe was born Sept. 13, 1946, the daughter of Dell and Dorothy Robinson. She married Donald R. Kehoe July 1, 1967.
She was a Fairfield resident up until her marriage. The couple moved to St. Louis. In 1977, they moved to the Washington, D.C. area. She was employed as a contracts manager for several defense contractors. They moved to Cheshire, Conn., in 2000 then moved back to the Washington, D.C., area for retirement in 2011.
Survivors include her husband, Donald R. Kehoe; two children, Stephanie Kehoe and Steve Kehoe and spouse Wendy; four grandchildren; one sister, Rosemary Trehey of Hot Springs, Ark.; and one brother, John Robinson of Fairfield.


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LOS ANGELES, Jan. 29 (UPI) -- Hollywood actress Ashley Judd and race car driver Dario Franchitti announced Tuesday they are ending their marriage after more than 11 years.
"We have mutually decided to end our marriage. We'll always be family and continue to cherish our relationship based on the special love, integrity, and respect we have always enjoyed," the estranged couple said in an exclusive joint statement to People.com.
No reason was given for the split.


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Onset of Type 2 diabetes occurs when the pancreatic β-cell fails to adapt to the increased insulin demand caused by insulin resistance. Morphological and therapeutic intervention studies have uncovered an inflammatory process in islets of patients with Type 2 diabetes characterized by the presence of cytokines, immune cells, β-cell apoptosis, amyloid deposits, and fibrosis. This insulitis is due to a pathological activation of the innate immune system by metabolic stress and governed by IL-1 signaling. We propose that this insulitis contributes to the decrease in β-cell mass and the impaired insulin secretion observed in patients with Type 2 diabetes.
The concept of islet inflammation and its role in the pathogenesis of Type 2 diabetes is discussed.
Major progress has been achieved in our understanding of the pathogenesis of Type 2 diabetes (3, 13, 34, 52). Over-nutrition and inactivity promote insulin resistance. To cope with this increased demand of insulin secretion, the pancreatic islet needs to enhance its secretory activity. In most individuals, such an adaptation does occur during early stages of increased metabolic stress (characterized by over-nutrition). Although successful adaptation of the β-cell permits maintenance of normal metabolism throughout life in most subjects, this adaptation eventually fails in some individuals, depending on the genetically determined ability of the β-cell to adapt and the severity of the resistance to insulin. The reasons for this failure to maintain sufficient insulin secretion are a combined decrease in β-cell mass and defective insulin secretion. Several mechanisms have been proposed to explain this failure, including ER stress, oxidative stress, amyloid deposition, lipotoxicity, and glucotoxicity (28, 33, 51, 54, 65). Interestingly, all these factors may induce an inflammatory response, whereas some may be the result of the inflammation (16, 17, 19, 31). Initially, an inflammatory response is probably deployed to promote β-cell repair and regeneration. However, as it becomes chronic, the situation gets complicated by, e.g., the activation of auto-inflammatory processes that may then become deleterious. Interestingly, similar auto-inflammatory processes occur in insulin-sensitive tissues. Therefore, Type 2 diabetes can be considered an auto-inflammatory disease. Consequently, it can be treated by agents blocking this vicious cycle such as IL-1 antagonists, or more downstream by NF-κB modulation via salsalate or similar compounds (15, 59).
In this article, we will review the evidence for insulitis in Type 2 diabetes, the mechanisms inducing this inflammatory process, its physiological and pathological role, and the therapeutic consequences.
Evidence for an Islet Inflammatory Process in β-Cell Failure During Type 2 Diabetes
In 1967 it was first proposed that Type 2 diabetes is not solely due to insulin resistance but also to a failure of the insulin producing β-cell to secrete an adequate amount of insulin (5). Based on numerous investigations, it is now evident that impaired insulin secretion is not only an important etiological factor in the pathogenesis of the disease but also the driving force dictating the dynamics of the disease. Indeed, although insulin resistance is present in all stages from pre-diabetes to overt diabetes, it remains constant in a single individual as long as his or her body weight remains unchanged. In contrast, the onset of diabetes and its progression is largely determined by the progressive failure of the pancreatic islet. At a pre-diabetic stage, insulin production will increase to adapt to the enhanced demand. When this adaptation fails, diabetes occurs. This failure then continues to progress, and affected individuals will require increasing anti-diabetic treatment, until eventually all Type 2 diabetics require exogenous insulin to control their glycemia. In an attempt to understand the underlying cause of this progressive failure, several mechanisms have been described. It appears that in all instances they constitute various components of an inflammatory process. The first evidence for an inflammatory process in the pancreatic islet arose from the observation that hyperglycemia induces β-cell apoptosis (14). By unravelling the underlying mechanism, it turned out that high glucose concentrations induce the Fas receptor, which in turn is upregulated via glucose-induced IL-1β production (41, 43, 44). Similarly, recent evidence shows that fatty acids also promote an inflammatory response (Refs. 1, 21; and see below). More downstream, ER stress and oxidative stress also appear linked to inflammatory events. In support of insulitis in Type 2 diabetes, elevated numbers of immune cells have been detected in islets of patients with Type 2 diabetes in conjunction with increased levels of cytokines and chemokines (2, 21, 53). Of note, every animal model of Type 2 diabetes investigated to date displays islet immune cell infiltration (19, 21). Furthermore, a strong argument for the occurrence of an inflammatory process in islets is the well described fibrosis observed in tissue sections of patients with Type 2 diabetes, characterized by amyloid deposits. Indeed, fibrosis is a hallmark of the end stage of a chronic inflammatory process. Although the concept of insulitis is recent in Type 2 diabetes, it is well established in Type 1 diabetes and was considered as pathognomonic. Although the precise aetiology of the insulitis in both diabetes types remains to be fully understood, differences certainly exist; for example, Type 1 diabetes is a more autoimmune-mediated process. However, a common final effector pathway seems to be activated in both types of diabetes (17). Keeping in mind that metabolic stress is a more recent threat than viruses or other possible causes of Type 1 diabetes, it is not surprising that islets respond with similar mechanisms developed over their long period of evolution. Of interest is the predominant role of IL-1β, which is upregulated in islets of patients with both Type 1 and Type 2 diabetes (2, 43, 45). This master cytokine regulates numerous cytokines and also chemokines (Refs. 12, 20; and see below). Thereby, it contributes to the recruitment of immune cells implementing a broad inflammatory response. Of note is that IL-1β will also induce itself in β-cells, engendering a vicious cycle (Ref. 2; and see below). Thus insulitis may be considered as an integral component of the pathology observed in Type 2 diabetes, and its dependence on IL-1 supports the concept of an auto-inflammatory nature of the disease.
Regulation of IL-1β Expression by Nutrients in Human Islets
Elevation of circulating nutrients such as glucose and free fatty acids (FFAs) induce an inflammatory process within numerous tissues in the body (32, 68). The first demonstration that a high glucose concentration indeed induces IL-1β release was obtained with human islets (43), with similar observations following in rat islets (24), retinal cells (70), and human monocytes (8). Moderately elevated glucose concentrations (11 mM) were sufficient to induce transcriptional activation of IL-1β expression in human islets (2).
More recently, we also found that long-chain FFAs induce several cyto- and chemokines in human and rodent islets (1). Oleate, palmitate and stearate, which are the most abundant FFAs in human nutrition, and hence in the circulation, stimulate IL-1β expression when added individually or as mixtures. Similarly, FFAs induced the IL-1-dependent cyto- and chemokines IL-6 and IL-8 in human islets and CXCL1 (also known as chemokine KC) in mouse islets. These stimulatory effects of FFAs on proinflammatory mediators are not restricted to islet cells but were also observed in numerous other cell types such as muscle (57), macrophage, and adipocyte cell lines (47, 58, 60), and in coronary artery endothelial cells (62). The combination of FFAs with elevated glucose concentrations further increases IL-1β expression (1) and the release of various cyto- and chemokines (21).
But how do these distinct and "nonspecific" nutrients lead to elevated expression of IL-1β and of IL-1-regulated cyto- and chemokines in islets? Some insight was obtained when we examined specific agonists of receptors of the innate immune system, specifically the IL-1/Toll-like receptor (TLR) family (1). Their stimulation typically leads to activation of pro-inflammatory processes in response to microbial pathogens and cell stressors (49). In human islets, specific TLR2 and TLR4 agonists induce IL-1β; however, the strongest inducer of IL-1β expression is IL-1β itself, which increases its own expression by an auto-stimulatory mechanism. IL-1β auto-stimulation was particularly pronounced in purified β-cells compared with whole islets, which consist of different cell types (2). Furthermore, it was NF-κB dependent and could be interrupted by blocking the IL-1 receptor with the specific IL-1 receptor antagonist (IL-1Ra) by neutralizing the receptor ligand or by deleting the intracellular receptor docking protein required for signal transduction (Myd88). Using these tools, we could demonstrate that both elevated glucose concentrations and FFAs increase IL-1β expression by promoting IL-1β auto-stimulation via IL-1 receptor activation. Thus IL-1β auto-stimulation via its receptor plays a pivotal role in the upregulation of intra-islet IL-1β expression provoked by nutrients. Noteworthy, we observed the highest levels of IL-1 receptor I expression in mouse islets and in an insulin-secreting β-cell line compared with 22 other rodent tissues (1). These data suggest that the pancreatic β-cell may be exquisitely sensitive to changes in local IL-1β concentration due to its high number of IL-1 receptors (1, 55).
Although these findings are compatible with a role for the IL-1 system as an amplifier of pro-inflammatory stimuli in islets, it leaves us with the question of how glucose or FFAs trigger and maintain this process. This is currently an open question, particularly for glucose, whereas for FFAs several mechanisms were postulated. It has been hypothesized that FFAs stimulate pro-inflammatory factors via direct activation of the lipid-sensing TLR2 and TLR4 in various cell types (39). Results with islets from TLR KO mice indeed show that FFA-induced pro-inflammatory factors are partly TLR dependent. Human islets express functional TLR2 and 4 (1, 64), and their activation results in increased expression of IL-1β and various cyto- and chemokines. Again, the TLR2/4 response is further amplified by IL-1β auto-stimulation. Thus the IL-1 system in islets not only amplifies the response to circulating nutrients but also to specific TLR agonists. Intriguingly, it was observed that obese subjects and rodents display elevated levels of circulating gut flora-derived endotoxins that could potentially trigger TLRs (4, 7), or, alternatively, inflammatory factors derived from other organs could stimulate TLRs in islets. To this end, it was recently shown that serum amyloid A binds to TLR2 (6), which is prominently expressed on purified human and rodent β-cells (J. Ehses, unpublished data, and Ref. 1). Alternative to TLR stimulation, FFA metabolites such as sphingosines have also been implicated in the induction of a pro-inflammatory response (29).
Overall, we conclude that elevated concentrations of various nutrients such as glucose and FFAs may activate the IL-1β system in pancreatic islets. Probably depending on duration of the exposure and the composition and concentrations of the ambient nutrients, sufficient amounts of IL-1β may be triggered to overcome the protective effect of the naturally occurring IL-1Ra (9), thus activating the potent IL-1 receptor. This auto-stimulation engenders a vicious cycle precipitating a broad inflammatory process, as detailed in the next section (see also Figure 1⇓).
IL-1 is a Master Regulator of Tissue and Islet Inflammation in Type 2 Diabetes
It has been known for a long time that obesity and Type 2 diabetes are associated with chronic activation of the innate immune system (50). Indeed, this activation appears to be systemic, given the increased levels of circulating cytokines and chemokines correlating with the disease and their potential in predicting Type 2 diabetes onset (30, 61). Local tissue inflammation is increasingly recognized in the pathology of both obesity-associated insulin resistance and β-cell failure in human Type 2 diabetes (16, 68). In all cases, this inflammation is characterized by increased tissue-infiltrating macrophages (21, 66). Indeed, two independent studies have now shown increased islet-associated CD68+ cells in Type 2 diabetic patients (21, 53). Furthermore, data from laser-captured β-cells from patients with Type 2 diabetes have indicated increased expression of IL-1β mRNA and chemokines, potentially responsible for this immune cell infiltration (2, 46). However, the contribution of IL-1 in the regulation of this islet inflammatory response in vivo was unknown until recently.
A number of animal models of Type 2 diabetes have now been characterized to various degrees regarding the presence of an islet inflammatory signature, including the GK rat, the high-fat diet (HFD)-fed mouse, the db/db mouse, the Psammomys Obesus gerbil, the fatty Zucker rat, and the Cohen rat (19, 21, 40, 67). In a number of these models, increased islet IL-1β expression and increased numbers of islet macrophages are an underlying feature. We recently extensively characterized the islet inflammatory process in the GK rat, showing increased islet expression of IL-1β and other pro-inflammatory cytokines (IL-6, TNFα), increased expression of a number of chemokines (CXCL1/KC, MCP-1, MIP-1α), together with immune cell infiltration in the islet (20). Since an important property of IL-1β in auto-inflammatory diseases is to increase the local expression of chemokines and adhesion molecules (12), we investigated the role of IL-1 in tissue inflammation in the GK rat, with a focus on the pancreatic islet. Elevated IL-1β mRNA levels were found in 2-mo-old GK rat islets and liver, with no increases in skeletal muscle or adipose tissue. To assess the biological impact of this finding, we blocked IL-1β activity with IL-1Ra in vitro and in vivo. IL-1Ra was able to suppress GK islet IL-6 and chemokine secretion (CXLC1/KC, MCP-1, and MIP-1α) in vitro, indicating that IL-1 activity is partially responsible for driving GK islet cytokine and chemokine expression. Treatment of GK rats with IL-1Ra, administered via either mini-osmotic pumps or subcutaneous injections, reduced fed hyperglycemia over the 4 wk of treatment. This was due to both peripheral and islet anti-inflammatory effects impacting on both insulin sensitivity and β-cell function in a dose-dependent manner. Indeed, analysis of liver and islet inflammation in high-dose IL-1Ra-treated animals (100 mg/kg day IL-1Ra) indicated reductions in inflammatory markers in both tissues. Specifically in the islet, IL-1Ra reduced islet cytokine and chemokine expression, with concomitant decreases in islet macrophage infiltration. Supportive of this reduction in immune cell infiltration, further work has indicated that markers of endothelial cell activation, such as adhesion molecule expression (Vcam1), were also suppressed by IL-1Ra treatment (36). Finally, reduction in islet inflammation correlated with improvements in proinsulin to insulin processing in IL-1Ra-treated animals. Since this IL-1Ra intervention study in the GK rat had minimal effects on β-cell mass, while improving β-cell insulin processing and hyperglycemia, we propose that IL-1β drives tissue inflammation, which then impacts on both insulin sensitivity and β-cell functional mass in Type 2 diabetes. However, this does not exclude some additional direct cytotoxic effects.
Interestingly, IL-1Ra treatment of the GK rat also reduced islet IL-1β expression itself, supporting that the auto-stimulatory role of IL-1β in the islet (as described above) plays an important role in the islet inflammatory process in vivo.
In summary, both our in vitro data on human and rodent islets and our in vivo studies on the GK rat support a central role for IL-1 in driving the expression of tissue proinflammatory cytokines and chemokines that subsequently attract immune cells to islets. This inflammatory process leads to impairment of β-cell function. These data support the concept that Type 2 diabetes is an IL-1-driven auto-inflammatory disease.
IL-6 Regulation of the Pancreatic α-Cell
Pancreatic islet pathology in Type 2 diabetes is characterised not only by reduced β-cell function and mass but also by increased proportion of α-cells relative to β-cells, together with relative hyperglucagonemia due to α-cell dysfunction (10, 63, 71). Having recently found elevated IL-6 levels in pancreatic islets of models of Type 2 diabetes (20, 21), we re-explored its role in the regulation of α- and β-cells (22).
We confirmed the previously published detrimental effects of IL-6 on insulin secretion in both human and rodent islets in vitro (35). Beyond this, however, we found that the glucagon-producing α-cell is a primary target of IL-6. Islet α-cells express the IL-6 receptor and functionally respond to IL-6 kinetically prior to β-cells. IL-6 was found to increase glucagon secretion under fasting conditions (low glycemia) in isolated islets and when systemically administered in vivo. Furthermore, IL-6 promoted α-cell proliferation and prevented α-cell apoptosis due to metabolic stress while exacerbating β-cell apoptosis. Thus, in contrast to the negative effects on the β-cell, IL-6 acts as a growth and survival factor and promotes glucagon secretion in the α-cell.
Further studies in vivo supported this hypothesis. For this, we studied the role of IL-6 in the setting of obesity and insulin resistance by feeding wild-type and IL-6 KO mice HFD. Although HFD increased systemic IL-6 levels in wild-type animals, this did not occur in IL-6 KO animals. Most striking, IL-6 KO mice did not show increased α-cell mass under these conditions, demonstrating that IL-6 signaling is necessary for the stimulating effect of HFD on α-cell mass. Consistent with these α-cell-specific findings, IL-6 KO animals displayed reduced fasting glucagon levels relative to wild-type animals on HFD. However, in contrast to the expected improvement in glucose tolerance in these animals, IL-6 KO mice on HFD displayed fed hyperglycemia, impaired glucose tolerance, and impaired glucose-stimulated insulin secretion compared with diet-matched wild-type animals. This effect has previously been published using IL-6 KO animals from the same source that we used (11). Since insulin sensitivity was similar among genotypes, the phenotype can be attributed to impaired β-cell function and suggests a role for the α-cell in maintaining β-cell function in vivo. Indeed, recent reports support this notion. Thus α-cell expression of prohormone convertase-1/3 (allowing the α-cell to produce GLP-1) or overexpression of the glucagon receptor on β-cells promotes improved insulin secretion (25, 69), thus supporting the hypothesis that the pancreatic α-cell can regulate β-cell function in vivo in a paracrine manner. That α-cell mass expansion is an important feature of the pathology of Type 2 diabetes was shown in Type 2 diabetic baboons, in which increased α-cell proliferation and mass together with hyperglucagonemia was recently described as part of the islet remodeling process (26).
Thus IL-6 regulation of the pancreatic α-cell represents a novel endocrine loop in the regulation of glucose homeostasis. Although it appears that this mechanism may exist to compensate for impaired β-cell function in the setting of obesity and/or Type 2 diabetes, it cannot be ignored that elevated IL-6 levels may also drive the relative hyperglucagonemia of diabetic patients. How this pathway contributes to the IL-6-mediated effects during physiology [e.g., during exercise, where systemic IL-6 levels are elevated (23)] or during the pathophysiology of obesity and diabetes in humans remains to be examined.
Role of Insulitis in Obesity and Type 2 Diabetes: From Islet Adaptation to Failure
Inflammation is not in itself a disease but a manifestation of a disease. Initially, it has beneficial effects such as preventing spread of infection or promoting regeneration. However, if prolonged or excessive, it may exacerbate disease by tissue destruction. It is likely that, in the case of islet inflammation in Type 2 diabetes, similar phenomena occur. Indeed, the endocrine pancreas has a remarkable capacity to adapt to conditions of increased insulin demand as encountered in obesity and pregnancy by increasing its functional mass. This may be triggered by limited hyperglycemic events that would provoke β-cell production of very low concentrations of IL-1β followed by Fas upregulation (41, 42). At low concentrations of IL-1β and in the presence of FLIP, Fas engagement would lead to β-cell proliferation and enhanced function via NF-κB and PDX1 (56). Indeed, NF-κB has been reported to have beneficial effects on both insulin secretion and β-cell proliferation (27, 48). If this response becomes excessive due to prolonged or repetitive exposure to nutrients, this initially adaptive process may become deleterious. IL-1β will then decrease FLIP and Fas engagement and switch to promote deleterious effects. In addition, IL-1β initiates the vicious cycle of inducing itself and promoting an enhanced release of chemokines, which leads to recruitment of macrophages (2). Possibly, these macrophages then produce a high amount of IL-1β (67) and other cytotoxic factors, which then impair the function of the β-cell. It is important to note that these mechanisms may be responsible for both decreased β-cell mass and impaired function. Depending on duration and magnitude of the effect as well as the individual regenerative capacity, the functional impairment may predominate over the more definitive decrease in β-cell mass. Interestingly, such an auto-inflammatory process occurs also in the insulin-sensitive tissues and in end organs such as kidney, eye, and the vasculature. Therefore, tissue inflammation may not only underlie β-cell failure and insulin resistance but also participate directly in the complications of diabetes, such as nephropathy, retinopathy, and cardiovascular disease.
Clinical Validation and Therapeutic Consequences
Based on the above-described predominant role of IL-1β, we initiated clinical trials of IL-1 antagonism in Type 2 diabetes. In a proof-of-concept study, the naturally occurring antagonist of IL-1β, IL-1Ra, was tested in a placebo-controlled study of 70 patients (38). At 13 wk, glycated hemoglobin was significantly improved due to enhanced β-cell secretory function. Remarkably, the improvement promoted by IL-1 blockade lasted for at least 39 wk following treatment withdrawal (37), thus reflecting the disease-modifying potential of this therapy. Novel therapeutic approaches designed to modulate IL-1 are under development with agents lasting a month or longer (18). Rather than just palliating hyperglycemia, IL-1 antagonism may represent a novel treatment principle directed against the pathogenesis that underlies diabetes mellitus, whereby the progressive decline in functional β-cell mass could be prevented or even reversed. Due to the auto-inflammatory nature of the whole metabolic syndrome and based on preclinical studies, it is expected that IL-1 antagonism will also enhance insulin sensitivity and prevent complications like blindness, cardiovascular events, and nephropathy. Numerous ongoing clinical studies are based on this assumption.
- Copyright © 2009 the American Physiological Society


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UnitedHealthcare and Sesame Workshop
UnitedHealthcare shares Sesame Street's passion for encouraging kids to be healthy and stay healthy.
Together we have partnered to develop Food for Thought: Eating Well on a Budget, a bilingual, multimedia educational outreach program aimed at helping low-income families make food choices that are affordable, nutritional, and set the foundation for lifelong healthy habits. Program outreach includes bilingual (English and Spanish) Food for Thought kits with an original DVD starring the Sesame Street Muppets, including the Super Foods, and a documentary showing families with children using a variety of strategies for maintaining lifelong healthy habits despite limited financial resources. Food for Thought kits also include a parent/caregiver guide, child-friendly recipes, and a children's storybook, and offer tools and resources to help families make choices that that lead to healthy growth.
UnitedHealthcare, a long-time supporter of nutritional, exercise, and obesity-avoidance programs, supports a wide range of programs and services across the country that promote nutrition and healthful eating.
UnitedHealthcare also supports Sesame Workshop's Lead Away and A Is for Asthma initiatives to increase awareness on lead poisoning prevention and to help families proactively handle children's asthma.
Sesame Workshop and UnitedHealthcare's We have the Moves, launching in 2013, provides educational and fun materials for families to help them incorporate physical activity into their daily routines.
UnitedHealthcare is part of UnitedHealth Group, a diversified health and well-being company, which provides services to one out of every eight children in the United States. Dedicated to helping people live healthier lives and making health care work better, UnitedHealth Group businesses serve more than 75 million people.


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Artist description:
Flash Draw Created Feb 20, 2009 470 x 338px Unless noted Copyright 2009 Tammy158.
We encourage you to submit a comment and let the artist know what you think of this drawing. Please log in or join to post a comment.
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Moonfairy Mar 20, 2009
great draw!! so cute too. 5/5
Pucca_luvs_garu Feb 22, 2009
Spongebob is so awesome! C: 5/5
sweetlove15 Feb 21, 2009
wooop! spongebob!! 5/5
MahaRizvi98 Feb 21, 2009
this is my favourite
MahaRizvi98 Feb 21, 2009
hahaaha thats cute
shobu Feb 20, 2009
cute i'm watching it right now.
Page 1


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Fact: I stated that each NEC monitor in this series sold in Europe is individually certified. They are not individually certified in North America.
"While it is true that LUT based display profiles can be an advantage over simple matrix profiles on certain types of displays that have very non-linear additive color properties (i.e. they do not behave Grassmanns law of additive color well), the IPS panels used on the PA series and internal 3D matrix correction make the displays additive color response very linear. This can be represented very accurately by a simple matrix."
"At the end of the day, SpectraView does not support LUT profiles." "you could always try profiling the display using a 3rd party package that does support LUT profiles and see if you get any improvement."
He did. And he did.
Exactly as Mark said over and over again in his posts before he quit this thread, the whole argument turns on whether the display is truly linear. When tested, this wide-gamut display is not perfectly linear. So the matrices approximate the true curves, which takes us back to the previous post. LUT wins.
The fact remains that NEC sells the same monitor in Europe with far more sophisticated software than SpectraView, feature by feature.
So for whatever reason, NEC gives European users the capability of building LUT-based, CIECAM02-capable profiles, in addition to simple matrix-based profiles, and they don't offer that to North American users.
You still haven't offered your explanation for why that is. Instead, you sound like a NEC shill, defending them 'till death do you part, regardless of the evidence. Maybe it's time for full disclosure, because you sound anything but objective. Sorta like when you're talking about... awe, forget it.


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The Onion Field (Paperback)
Usually Ships in 1-5 Days
This is the frighteningly true story of two young cops and two young robbers whose separate destinies fatally cross one March night in a bizarre execution in a deserted Los Angeles field.
About the Author
Joseph Wambaugh is the hard-hitting best-selling writer who conveys the passionate immediacy of a special world. A master storyteller...authenticity oozes from his books.
Praise For…
"A complex story of tragic proportions... more ambitious than In Cold Blood and equally compelling!"—The New York Times


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WASHINGTON (HedgeWorld.com)--In response to persistent reports of market timing and late trading at mutual fund companies, U.S. House and Senate subcommittees are planning separate hearings Nov. 3 and 4.
The Senate's Financial Management subcommittee will hold a hearing Nov. 3 at 10:30 a.m. EST. Among the witnesses called to testify are New York State Attorney General Eliot Spitzer; Stephen M. Cutler, director of the Division of Enforcement for the U.S. Securities and Exchange Commission; and Rep. Richard H. Baker, R-La., chairman of the House Capital Markets subcommittee.
Mr. Baker and his House subcommittee will conduct their own hearing Nov. 4 at 10 a.m. Included on that list of witnesses are Mr. Spitzer, Mr. Cutler and William Galvin, Secretary of the Commonwealth of Massachusetts.
Both hearings are meant to delve more deeply into mutual fund trading practices to determine whether further industry regulation is needed to protect investors.
In a statement announcing the House hearing, Rep. Baker said that since July, when the House Financial Services Committee sent to the full House a bill designed to improve transparency of mutual fund fees and cost structures and increase corporate governance, "a number of extremely disturbing revelations" about mutual fund trading practices have come to light.
"Subcommittee members have a serious responsibility to get the facts about these particular incidents of late trading and market timing," Rep. Baker said, and to then ask "whether our proposed legislative reforms for mutual funds go far enough."
The Senate subcommittee's hearing announcement notes that the purpose of the hearing is to "explore the extent and impact of alleged trading abuses ... and consider regulatory reforms necessary to mitigate such practices in the future."
Other witnesses who have been called to the Senate subcommittee's hearing include Mary L. Schapiro, vice chairman and president of regulatory policy and oversight for the National Association of Securities Dealers, Washington; John C. Bogle, founder and former chief executive officer of The Vanguard Group, Malvern, Pa.; and Paul G. Haaga Jr., chairman of the Investment Company Institute, Washington.
The House subcommittee's full witness list is expected to be released Friday or Monday, subcommittee spokeswoman Brookly McLaughlin said.
Mr. Spitzer is in demand as a witness largely because he and his office are credited with first peeking behind the curtain around mutual fund trading practices. In September, Mr. Spitzer announced that an investigation of the mutual fund industry by his office had uncovered "widespread illegal trading schemes" that potentially cost mutual fund investors billions of dollars a year.
At the same time, Mr. Spitzer said he had reached a US$40 million settlement with hedge fund Canary Capital Partners LLC, Secaucus, N.J., in connection with late trading and market timing of mutual fund shares Previous HedgeWorld Story.
Since then, the mutual fund industry has seen a parade of allegations, resignations and litigation related to late trading and market timing of mutual fund shares by large investors, including hedge funds, and in some cases mutual fund portfolio managers themselves.
Lawsuits filed against Putnam Investments, Boston, by the SEC, Massachusetts regulators and fund investors claim that some of Putnam's own mutual fund portfolio managers used inside information to time personal trades of Putnam mutual fund shares--which ordinary fund investors could not do and which the fund's prospectus said was an improper practice. Putnam has fired the managers and, in a statement, said it had done nothing fraudulent and was cooperating with federal and state regulators.
Meanwhile several large banks that offer mutual funds are dealing with fallout from Canary Capital's mutual fund trading practices. Mr. Spitzer alleged that Canary Capital had special trading relationships with Bank of America Corp., Charlotte, N.C.; Janus Capital Group Inc., Denver; Bank One Corp., Chicago; and Strong Financial Corp., Menomonee Falls, Wis.
On Oct. 15, Mark A. Beeson, president of Bank One Group's One Group mutual fund division, resigned. He had been named in Mr. Spitzer's complaint against Canary Previous HedgeWorld Story. In mid-September, Mr. Spitzer and the SEC filed state criminal and federal civil charges against Theodore Sihpol III, who was responsible for executing trades at Bank of America's Nations Funds. The charges alleged Mr. Sihpol helped Canary conduct after-hours trades of Nations Funds mutual fund shares Previous HedgeWorld Story.
Mr. Spitzer also has subpoenaed roughly a dozen additional hedge funds, as well as mutual fund companies Invesco Funds Group Inc., Denver, and The Vanguard Group, in connection with his probe Previous HedgeWorld Story. Additionally, the SEC officials have asked brokerage houses to provide the commission with documentation about the trades by their largest customers who trade mutual fund shares Previous HedgeWorld Story.


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ACParadise Network: American Cosplay Paradise American Cosplay Experience American Cosplay Snapshots
Cosplayer sievkatronshea > Costume of Hikaru Shidou (Magic Knight Rayearth)
- Most Recent Photo
- Series
- Magic Knight Rayearth
- Character
- Hikaru Shidou
- Special Variation:
- Celestial Artbook
- Year Completed:
- 2008
- Construction Difficulty:
- Difficult
- Awards
- Judge's Award AZ '08 (group), Best in Show NekoCon '08 (pair), Honorable Mention Craftsmanship Katsucon '09
- Costume worn at:
- Animazement 2009
- Animazement 2008
- Anime Expo 2009
- Katsucon 2009
- NekoCon 2008
About this Costume
- Construction Details:
This is like...a costume and a half. This was my first attempt at armor making. All of the armor is made from DAS airdry clay and all of the gems are resin-cast.
This is also one of the hardest wigs I've made - mostly because Hikaru's little 80s rattail is absolutely ridiculous looking and would look absolutely horrible IRL. So. I compromised with a thicker and UBER LONG braid made from loose extensions that were wefted and sewn into the wig. The rest of the wig is a chopped up femme fatale from Amphigory. :D
Compared to the wig and the armor, the actual sewing part of this costume was really easy. I'm always adding new stuff to this costume, so it keeps changing a little everytime I wear it.
Wig Details
- Wig Brand
- Amphigory
- Wig Name
- Femme Fatale
- Styling Time
- 4 hours
- Styling Notes
- This wig started out as a femme fatale and then I chopped it to bits. The braid was two really long packs of loose extensions from Dr. Locks, which I made into wefts and then sewed into the base of the wig. Braiding was more difficult than anticipated because of the length of the extensions.
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I don't know if anyone can help me but I am trying to set up my Denon CD players so they can be started by remote control.
The "D3" function instructions in the manual are clear as mud. Does anyone know what the settings are so I can use the front panel controls plus a remote start switch?
A picture of the digital readout with the correct settings would be awesome.
Thanks for any help you can provide.


text: Exit this survey REGISTRATION - CoffeeTime Webinar: 4/09/2013 To register for the Coffee Time Consultation Webinar, please provide the following information: * 1. First and Last name: * 2. Credentials: Select all that apply MD DO NP PA RN Other (please specify) * 3. Practice Name: * 4. Check one: Pediatric Practice Family Practice * 5. County where practice is located: Baker Benton Clackamas Clatsop Columbia Coos Crook Curry Deschutes Douglas Gilliam Grant Harney Hood River Jackson Jefferson Josephine Klamath Lake Lane Lincoln Linn Malheur Marion Morrow Multnomah Polk Sherman Tillamook Umatilla Union Wallowa Wasco Washington Wheeler Yamhill * 6. Number of years in practice: * 7. Email addresses will be used to provide login and dial-in information, the webinar link, and any applicable handouts. Please provide your email address: * 8. Would you like to be notified about upcoming Coffee Time Consultation Webinars or other educational opportunities for primary care providers? Yes No Next


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Customers Who Bought This Product Also Bought
A Sci-Fi Shocker
The Person Next To You Could Be One Of Them.
Fresh veggies on Aisle 1. Fresh kills throughout the store. Something terrible is happening at Hastings Market. Something bloody. Something deadly. Something inhuman. And caught up in the grisly horror inside the market are shoppers turned hostages, unearthly alien terrors... and a dedicated commando band of raiders on a search-and-destroy mission to stop the intruders dead cold no matter where they hide.
From Raw Feed, creators of the Rest Stop and Otis shockers, comes the sci-fi/horror tale Alien Raiders, a nerve-shredding battle for human survival against aliens that invade our world by invading our bodies. Be alert. Beware. Be afraid. The creatures' next host could be your neighbor. Your kid brother. No - it's you!
- Length
- Rating NR
- Released:
- Added Nov 25 2008
- Production Year 2008
- Empire SKU 1440509
- UPC Code 883929013852
- Studio Warner Bros.
- Packaging Keep
- Number of Discs 1
- Disc SS-DL
- Subtitles English, French and Spanish
- Hidden Terror: The Making of Alien Raiders
- Blood, Sweat and Fears: The Special Effects of Alien Raiders
- Tape #9: Sterling Explains Alien: Alien Parasite Physiology and Meeting Ritter
- Tape #12: Spookie's Job: The Alien Buster Explains His Talent
- Whitney Cam: Blog Posts After the Buck Lake Incident
- Raw Feed Movies Trailer Gallery
- Audio
English Dolby Digital 5.1
Spanish Dolby Digital Stereo
- Video
Widescreen 2.35:1 Color (Anamorphic)
- Daniel Myrick
- John Shiban
- Tony Krantz


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It is a rivalry.
When the Warriors and Clippers face off at Oracle Arena on Thursday, there will be playoff intensity — on the floor and in the stands. There will be trash talk, a certain amount of chippiness, a palpable sense of enmity.
Unless you're Mark Jackson.
"Forgive me," the Warriors coach said Wednesday. "I was raised on Magic and Bird. Lakers and Celtics. Yankees and Red Sox. Real rivalries. So let's pump the brakes a little bit."
True, Warriors-Clippers hasn't reached that level. But that doesn't mean it isn't a rivalry.
This is a rivalry, and a good one. Even without Chris Paul — the Clippers' star guard and chief rabble-rouser is injured — the game Thursday night will feel like a rivalry.
That Jackson won't call it a rivalry — and he's not alone among Warriors and Clippers — could be another indicator of the dislike these teams share. Neither is willing to admit it cares that much about the other.
Warriors-Clippers hasn't yet produced a game that grandfathers will tell future generations. But the matchup has all the markings of a growing rivalry: Good teams. Popular players. A common goal. Geographic proximity. And, most important, drama that seems to build on itself at every meeting.
It started in November of 2012 when the Warriors won in L.A. and celebrated a little too much for the Clippers' liking. Paul said the Warriors acted as if "they won the Finals."
The next time the teams met, in Oakland, the Warriors won again. Stephen Curry hit a big 3-pointer and celebrated by galloping downcourt. The only thing that angered the Clippers more was the Golden State bench openly laughing when Blake Griffin took a shot that hit the side of the backboard.
The teams completed their season series in January — the Warriors winning three of four — but the ensuing 10-month hiatus hardly served as a cooling-off period. On the second night of the 2013-2014 season, the Warriors traveled to L.A., and the bad blood was in evidence before the teams even took the floor. A pregame Bible study, usually a joint venture, was broken in two at the Clippers' request. Once on the floor, the teams got in a scuffle, Andrew Bogut and DeAndre Jordan engaging in a shoving match.
There was another scuffle when the teams met again, on Christmas Day in Oakland — two scuffles in fact. Draymond Green was ejected after an incident with Griffin. Griffin was ejected after an incident with Bogut. Then Bogut and Paul punctuated the Warriors' win with more pushing and shoving.
The NBA nation was buzzing about Clippers-Warriors. And now, we eagerly anticipate Round 3 on Thursday night in Oakland.
Waiting to see if the teams get together before the game and sing "Kumbaya." Waiting to see how the Warriors bench reacts when something goes well. Waiting to see what happens when Bogut or Matt Barnes applies a hard foul.
Even Jackson had to admit the entertainment value of the rivalry.
"It's not a rivalry," he said.
Well, the bad blood between the teams.
"We don't have blood."
Then the intense competition.
"OK. Thank you for the remix," Jackson said with a smile. "It makes it exciting. We embrace it. They embrace it. … It's good for basketball. Certainly it's good for both teams."
Because it's a rivalry.
Contact Marcus Thompson at firstname.lastname@example.com. Follow him on Twitter at twitter.com/ThompsonScribe.
Thursday's game
L.A. Clippers (33-15) at Warriors (27-19), 7:30 p.m. TNT
Clippers have increased
their lead over the Warriors. PAGE 4


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