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Be the first to learn about new tours & upcoming events. Get inside info on destinations, preparing for your tour, training and nutrition - plus get in on special pricing on select tour dates! *Bicycle Adventures does not sell or give out your personal information.
Scotland – a land of myths, legends and majestic landscapes. Make your own history on this tour of the country's magical southwest. Ride through rolling farmland and deep forest. Stop to visit ancient castles and hear stories of epic battles. Sample the craft beers and delicacies of Dumfries and Galloway. Board a ferry to cross the Firth of Clyde to Scotland's 7th largest island, the Isle of Arran. Bicycle Arran's wilderness and discover the rich natural and cultural heritage of this amazing place. Recharge at the island's own distillery and brewery before setting off on an epic island-hopping final ride via the region's inter-island ferries. Cross to ride the Mull of Kintyre. Finish on the Isle of Bute. Board a private speedboat to cruise back for a final night of celebration on the Isle of Arran before heading back to Glasgow.
Note: Coming in early for the August trip? Book a couple of additional nights for the Edinburgh Festival!
Arrive into Glasgow. Ride to the birthplace of the bicycle, then on to Mabie Forest.
Meet in Glasgow. Drive for just over an hour to the magnificent Drumlanrig Castle. The 17th century "pink palace" is one of the country's finest examples of Renaissance architecture and makes a fitting place to commence your journey. Enjoy a virtually traffic-free shakedown ride on the estate roads. Head south through quiet and scenic countryside, passing Keir Mill where the modern day pedal cycle was invented in 1839 by local blacksmith Kirkpatrick Macmillian. Ride onward to Mabie Forest. Stay the next two nights at a historic country house hotel.
Explore the countryside of Dumfries and Galloway.
Largely undiscovered, Dumfries and Galloway is a cycling revelation, sporting over 700 km/450 miles of marked routes on quiet roads through beautiful scenery. Today: ride pure countryside where there are likely to be more sheep than cars! Visit the fantastically Scottish named Craigenputtock House, former residence of the famous author Thomas Carlyle. It has hosted many famous visitors over the years, including Ralph Waldo Emerson. Continue south, stopping by one of the best Farm Shops in Scotland for afternoon tea and cake. Continue onward to the quaint village of New Abbey, where a visit to the 13th century Sweetheart Abbey is a must before the short ride back to the Mabie House Hotel.
Distance: 48 miles. Elevation: 1,800 ft.
Ride the foodie's region of Castle Douglas. Then bike through Galloway Forest Park.
Today, cross the region from east to west. Bike through the 'food town' of Castle Douglas with its fantastic range of cafes, bakers, butchers and fishmongers, then turn north heading for Galloway Forest Park. Follow the shores of Loch Ken, then climb out of New Galloway into the heart of the Park. Watch for wild Red Deer and goats on this stretch of road leading to the final part of the day - a superb cruise on a gentle downhill gradient to the village of Newton Stewart.
Distance: 58 miles. Elevation: 3,100 ft
Cross the Firth of Clyde to ride the Isle of Arran.
After a relaxed Scottish breakfast, hop in the van for a shuttle north to reach the small ferry port of Adrossan. Board the boat and cross the Firth of Clyde to the fantastic Isle of Arran. Affectionately known as "Scotland in Miniature," the island is a perfect mix of coast, moorland and mountains. This afternoon: go for a short bike ride, relax at the hotel or kayak the coastline. Stay here for the next 3 nights.
Bicycle the Isle of Arran loop. Finish off with craft beer at the local brewery.
The complete loop of the Island offers 56 miles of fantastic riding. With plenty of hills it's a challenge, but the reward is one of the country's classic day rides. Head south from Brodick to tackle the loop clockwise, passing by Holy Isle with its Buddhist monastery. Rounding the southern end of the island, views to the Mull of Kintyre open up with Ireland just visible further in the distance. Heading up the west coast, the reward in the north is a visit to Lochranza Distillery. Enjoy a wee tonic of malt whisky before tackling the climb through Glen Sannox. On the other side you'll find one of the best descents anywhere on a brand-new road surface. Then it's a mostly flat cruise back to Brodick. Celebrate the day at the Isle of Arran Brewery with its fine range of craft beers.
Distance: 56 miles. Elevation: 4,300 ft.
Do the Scotland Island Hop route. Finish with a dram of the local whisky.
This last ride is a true adventure involving four short boat crossings, two peninsulas, two islands and sixty miles of amazing roads. Starting from the Glen Isle Hotel, bicycle north past Goat Fell, Arran's highest peak at 874 metres (2,866 ft). In the village of Lochranza - guarded by its ancient castle – climb aboard for a short ferry ride to the mainland and the Kintyre Peninsula. Ride to lunch in the scenic village of Tarbert. Back on the bikes, ride to the beautiful Kyles of Bute. Bicycling here is simply stunning with magnificent views across Scotland's west coast. Boat journey number three is a swift affair: just five minutes across the "Kyles" to the Isle of Rothesay. Here, welcome the flat island roads leading to a remote beach on the island's south end. Hop aboard a private speedboat and return in style to the Isle of Arran. Enjoy a final night celebration on the Island, toasted with a dram of the local whisky.
Distance: 60 miles. Elevation: 3,280 ft.
After a relaxing morning, head to Glasgow.
This morning, following a relaxed start, take the ferry boat back to the mainland. It's just a short hour's drive from the ferry dock to Glasgow. Choose to stay on an explore this vibrant city or depart from the International Airport.
Make your reservation now or (800) 443-6060.
As an optional extra, a person traveling solo on any inn-to-inn trip may reserve a room for his or her exclusive use. This will guarantee you a private room and private bath every night. Unlike other tour companies, if you're willing to share a room, there is no extra charge for coming solo, no matter when you sign up for the tour - even if you wind up with a private room by default.


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We've said it before, and we'll say it again: In a world of endless, needless reunions, there's one that always makes us happy: The Kids in the Hall. Dave Foley, Bruce McCulloch, Mark McKinney, Scott Thompson, and Kevin McDonald have teamed up for an eight-part murder mystery series called Death Comes to Town. As of now, the show, about a murder in a small town and the revealing trial that follows, is only set to air in January in Canada, where it's filming. Unacceptable. UPDATE! Especially now that we've established that the guys will all play multiple characters, men and women. "It is possible, though unconfirmed, that a special appearance or two by characters from the original series may be made," the release reads. "Original characters include the small town Mayor and his alcoholic wife, a germ gel sniffing town criminal, a pizza delivery woman with Alzheimer's, a 600lb ex-hockey star, and of course, Death himself."
I've been waiting for this moment since 2003, when the guys told me they were hoping to do a second film, tentatively titled Not Brain Candy 2. McDonald said he wanted to improv it out in rehearsals, McKinney wanted to fine-tune it on tour like the Marx Brothers, Thompson wanted to script it himself and have the others perform every line exactly as written. (McCulloch, whose last TV project was ABC's Carpoolers, conceived the idea for Death.) In August 2007, when the Kids released the 1988 pilot for their cult sketch show on DVD, Foley said they were still hoping to write a movie or do a limited run TV series, but scheduling was always an issue: "We're not really good at planning." After touring with new material last year (our behind-the-scenes gallery), they're finally in sync.
So here's to the Kids in the Hall. To comedy that still stands up. And, of course, to Reg. (Foley always said this is the sketch that best encapsulates their humor. Fitting that it involves a murder.) Who else is psyched for Death Comes to Town?


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March 1, 2011. Bodh Gaya
While requesting that the press respect His Holiness' privacy during his retreat and puja program in Bodh Gaya, Mingyur Rinpoche addressed a gathering of local press members. Mingyur Rinpoche is a world-renowned Buddhist teacher and author of the bestselling book, The Joy of Living.
Speaking to reporters yesterday, Mingyur Rinpoche said, "We are very grateful that His Holiness's name has been cleared." Rinpoche thanked the Indian government and Tibetan government-in-exile and was also thankful for the blessings from His Holiness the Dalai Lama and for the support from the Himalayan people, Indian people and people all around the world. "We appreciate everyone's support," he said.
"I feel very sad and upset about the allegation that he is a Chinese spy," Rinpoche commented. "This allegation has hurt the disciples of His Holiness. He is an enlightened being, but for us it's been very hurtful and upsetting."
Dr. AK Sinha, who is in charge of northeast India for the Coordinating Committee of the All India Buddhist Organization, also addressed the press. "I am a Buddhist and His Holiness the Karmapa is my respected teacher," Dr. Sinha said. "One of India's best values is bringing harmony to society and His Holiness the Karmapa is also aimed for that."
Following his retreat in Bodh Gaya, His Holiness will travel to Varanasi, to celebrate the Tibetan lunar new year. He will also participate in an associated puja cycle and confer four days of Dharma teachings from March 9 through 12.
For press enquiries, please contact:
Deki Chungyalpa
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(91) 8894502910


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As far as expectations go, Greater Western Sydney coach Leon Cameron's have already been exceeded this AFL season.
But the Giants' emphatic defeat of bogey team Adelaide and a new top-four placing now takes the cake.
The Giants prevailed 16.12 (108) to 12.12 (84) over the Crows in a thrilling encounter at Spotless Stadium on Saturday, as their relentless midfield held off the usual impact of livewires Taylor Walker (four goals) and Eddie Betts (three goals).
After their upset win against Hawthorn two weeks ago, the home side finally turned the tables on the South Australian outfit that has thrashed them by an average of 87 points in the five meetings to date.
It makes it three on the trot for the Giants – the club's biggest winning streak in its short history – who continue their blazing run towards an inaugural AFL finals campaign.
Cameron said the league's newest team had so far outdone his predictions for 2015.
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"When you're sitting at eight rounds and you're six and two, yeah you take it any day of the week.
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And compete they did.
In-form forward Cam McCarthy (four goals) kicked off the onslaught as the Giants scored five goals in the first 14 minutes to bounce to a 34-point lead.
The Crows looked shell-shocked as their rudderless defence fell short against the flair of Toby Greene, Tom Scully and the sparky Dylan Shiel (34 disposals), who kicked one major and ran off on the odd breakaway.
Adelaide finally managed a shot on goal in the 16th minute; a free-kick which Walker struck perfectly.
But Tomas Bugg responded, notching his first major of the season to help the Giants to their highest first-term score of 40.
Adelaide steadied the ship, though their depth was sorely tested in the absence of Matthew Jaensch (knee) and halfback Brodie Smith (concussion) among others.
Their stocks took a further hit when Rory Laird was stretchered off with a concussion in the third quarter after a bad head clash with teammate Jake Lever, while Luke Brown also took a worrying hit and Sam Kerridge went off with an ankle injury.
And even a promising rally in the final period couldn't produce the right result for the visiting side.
"We showed something in the last quarter, but GWS may have put the cue in the rack then," Adelaide coach Phil Walsh said.
"At least it was something for the fans who hadn't turned off the telly back in Adelaide."
Greene also kicked three goals while Jeremy Cameron continued his solid form up front, slotting two to take his tally for the last three games to 14.
But the 22-year-old was also put on report during the first term for forceful front-on contact with Kyle Cheney.


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This property has been removed by the agent. It may be let or temporarily removed from the market
4 bedroom semi-detached house to rentForest Road, Loughborough, LE11
£1,340 pcm| £309 pw fees apply
Property Description
Letting information:
|Added on Rightmove:
|12 September 2016 (376 days ago)
Key features
- EPC D Rating
- Other Fees Will Apply
- Managed By Your Move
- Well Presented
- Accommodating Landlord
- Modern Kitchen And Bathroom
- Three Large Double Bedrooms
Full description
**CALLING STUDENTS 2017!!** Did you know that organised students get the best houses? If you are looking for a 3/4 bedroom house for next year then look no further! This immaculately presented house offers 3 double bedrooms and a smaller single, modern bathroom with shower over bath, modern kitchen with gas hob, living area and bike storage. Lovely garden to front and rear and on major bus routes. CALL NOW - 01509 231731
More information from this agent
To view this media, please visit the on-line version of this page at www.rightmove.co.uk/property-to-rent/property-56064358.html
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Property reference 335528568142_L. The information displayed about this property comprises a property advertisement. Rightmove.co.uk makes no warranty as to the accuracy or completeness of the advertisement or any linked or associated information, and Rightmove has no control over the content. This property advertisement does not constitute property particulars. The information is provided and maintained by Your Move , Loughborough.
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Some words from the builder DJpaulii Built on Teweran – https://www.youtube.com/user/Teweran Make sure to check out my facebook-page! Feel free to make a cinematic of this! Infos Texture Pack You can use any Texture-Pack you want! Download Feel free to download this project, BUT be carefully with the rights (more Infos in the next spoiler below)! Rights You are not allowed …Read More »
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Here are a few fishing tips that will surely help you catch more fish on your next fishing trip.
Fishing Tip # 1: You Gotta Hunt For The Fish
This may seem pretty obvious fishing tactic, but you need to know where fish are… and many people don't. You can search for fish with your bare eyes… if you are fishing for bass, look for downed trees and stumps or logs in the water. If you are fishing for northern pike, look for weed beds. Panfish in many lakes in Wisconsin seem to be almost anywhere you drop a line, which makes "hunting" for them fairly easy, but try to spot a downed tree, stumps, or even shallow bays. For walleyes, you're going to want to look for drop-offs or other abnormal looking structures on the bottom of the lake or river. Now, unless you are on a super crystal clear body of water, it's going to be quite difficult find structures under water with your bare eyes… and that, my friend, means that if you really want to find the fish, you've gotta make an investment in a good fish finder.
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Fishing Tip # 3: Knot Tying
A very simple trick to tying knots is to wet the line before you tighten your knot either with water or your saliva. Adding this lubrication will help prevent any stress or damage to your line when you give it that final tighten. It will also make your knot much tighter. If you want to make sure your knot is really tight, add a drop of superglue after you've tightened it.
Fishing Tip # 4: Use Lighter Line
Many people think that if you are fishing for bigger fish, you've got to use heavy strong line. Not true. Use a lighter line. For instance, if you are bass fishing, you shouldn't have to use line any heavier than 4 pound… just make sure you have your drag set right and you'll never break your line… you'll also catch more fish.
Fishing Tip # 5: Use Fish Attractant
Out of all my fishing tips, this is one of the most controversial. Some say it works and some say doesn't. I am a big believer of this stuff! I used to think it was a waste, but I cannot tell you how many times I've gone fishing with my dad (who swears by this stuff) and he catches way more fish than I do fishing the same bait and lures. The only difference was that he was using fish attractant. What are good attractants to use? Well, they are all very similar… I've used this fish attractant and had some great results… so, just pick one and try it out… my guess is that you'll be swearing by this stuff to once you use it.
Fishing Tip #6: The Evening Secret
Many fishermen have reported excellent results when fishing in the late evening, night, or early morning hours. Additionally, some use The Evening Secret for even better results. It is well known that most species of fish feed in the evening, so if you haven't tried fishing during these magical hours, you are really missing out…
Fishing Tip #7: Hire a Guide or Book a Fishing Charter
If you are fishing for a particular species of fish or are fishing an area that you don't know very well, consider hiring a fishing guide or booking a fishing charter. This could provide you with invaluable information, saving you a lot of wasted fishing time!
Fishing Tip #8: Many Fishing Lures are Made to Attract Fisherman
Be careful when buying fishing lures… many lures are made to attract the fisherman more than the fish. Big bright fancy coloring doesn't necessarily attract the fish… just do your homework, especially when your paying close to or over 10 bucks a lure.
Hopefully, these fishing tips will help you out. Try them out next time you are fishing. Remember to check back often, as we will be adding more fishing tips as time goes on.


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While our clients are hard at work on the field, LMM is hard at work off the field trying to maximize its clients' visibility.
As all professional athletes know, marketing and endorsement opportunities can be very lucrative and exciting.
LMM is connected at the highest level to companies that utilize star athletes and celebrities to endorse their products or services.
In implementing an individualized marketing plan for each client, LMM meets with each client in an effort to determine his individual marketing goals.
LMM takes into consideration such things as a player's position, the city in which he is selected to play and the image both he and his team projects nationally and in its local market. An individualized marketing plan tailored for the client is then implemented.
• Clothing Endorsements
• Shoe Endorsements
• Cell Phone Endorsements
• Television Commercials
• Television Appearances
• Radio Commercials
• Radio Appearances
• Auto Dealership Trade
• Autograph Signings
• Memorabilia Signings


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As many readers will know I am passionate about getting good people into parliament, and admire good politicians even if I oppose them, like that cunning bastard Russel Norman.
Something that has troubled me for years is how many good candidates run for National but never make it into politics, missing out at selection or becoming disillusioned after taking one for the team and getting nothing back from the team so giving up entirely. Too many good people are lost to National by the callous indifference the party hierarchy shows them.
Last January David Farrar suggested that a few of us set up a training program for good people who want to run for National, and with the help of others I have done this. As an extension of this program I am in the process of creating a web site giving an honest opinion on how to win a National selection, how to build a career as a politician and how not to end up having your career wrecked, by National apparatchiks who are too cowardly to stand themselves, by getting you to run in a red seat.
As I have no official role in the party I can be open and honest with answers to questions, and answer based on what is best for the individual, not the small group of people in power now, who don't care what happens to National in ten or twenty years.
I'll post some FAQs later today, but I expect there will be more so please email them in.
THANK YOU for being a subscriber. Because of you Whaleoil is going from strength to strength. It is a little known fact that Whaleoil subscribers are better in bed, good looking and highly intelligent. Sometimes all at once! Please Click Here Now to subscribe to an ad-free Whaleoil.


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come rave with us in the block9 field
RA x Choose Love 20.07.17
facebook event page ticketsContinue reading →
facebook event page ticketsContinue reading →
For anyone that didn't attend, missed the live broadcast, or just want to see/ hear it again – here's the YouTube archive of Boilerroom's first ever full Drum & Bass takeover, plus the tracklist for my set.
Despite technicals, it was an honour to play alongside some of our scene's pioneers and heroes. Enjoy the selection x
The Next Chapter 3.11 (April 2017) – Tracklist Mako – Held You in My Arms Subsequence – Shunyata Wagz – Darkstar Unnamed – Untitled DJ Addiction & Villem – Someone Somebody Commix – Kosmos 2251 Calibre – Better Than Me *Calibre Interview (featuring various tracks from The Deep and Grow LPs)* Calibre – Blind for […]Continue reading →
The Next Chapter 3.10 (March 2017) – Tracklist Calibre – Up in Smoke Calibre – Complain Calibre – No-One Gets You Klute – Angel Makers Klute – Lose My Way Klute – Soul Boy Future Cut – Hideous Kinky Survey – Multiple Identities Seba – Bon Voyage Mark System – Dissolve Andy Pain – NZT48 […]Continue reading →
Wednesday 3rd May 2017 sees Drum & Bass fully take over Boiler Room, for the first time ever. Hear (watch) me playing some old skool Jungle/D&B selections, alongside pioneer heavyweights Fabio, Grooverider, Doc Scott, Randall, plus a very special guest whose name will be revealed in good time. If you're in London and want to […]Continue reading →
It gives me great pleasure to present to you another in-depth interview with the legend himself, Calibre. I caught up with him earlier this month (April) at the launch party for his latest album, The Deep. We talked about a lot of different things; music, dreams, politics, travelling, the current state of the world… extremely […]Continue reading →
Today, 25th April 2017, was the 18th anniversary of Valerie Kemi Olusanya's – aka DJ Kemistry – passing. To mark the occasion, Storm released a bunch of photos and recordings from the 10th anniversary tribute event which took place at Cargo, Shoreditch in 2009. It was a heavily emotional and uplifting night full of incredible […]Continue reading →
Darcus Howe 26.02.43–02.04.17.
We lost another hero. Pioneering Black Panther activist, lawyer, journalist, broadcaster, WARRIOR for the liberation of African and African-Caribbean people in the UK. If you're not familiar with his work, please read up on him. A giant that won't (can't) be forgotten.
May he rest in eternal power and peace x


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Handmade Candles - Simple Elegance
Handmade with 12 oz creamy paraffin wax, this white candle is poured into a 3" x 5" mason jar. It is strongly scented with Jasmine, an alluring and sensual floral that is delicate, sweet, and flowery.
Aruba Coconut Scented Candle
$ 13.00
Aruba Coconut Scented Mason Jar Candle
$ 16.00
Bamboo & White Grapefruit Scented Mason Candle
Cedar Scented Mason Jar Candle


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Classic styling gets a hip update with the look of exotic skins in the Dayton oxford from Stacy Adams. It's crafted from patent and ostrich-embossed leathers in a design that makes the wingtip look ultra-modern and effortlessly luxurious.
Stacy Adams Men's Dayton Ostrich Oxford
Dress Lace-Ups item created by admin, Jul 13, 2011
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>Why do you like Free Software and Ubuntu?
My Answer:
I consider software important. Really really important. Why?
Most people don't really understand just how much power software has over our current society. Let's pretend all the software in the world was really just one single person. What could that person do?
- Change everyone's bank account balances (and change local records to match so noone notices!)
- Choose many elected officials
- Isolate individuals socially, make their emails go unanswered, etc (or worse make seem to send emails creating arguments)
- In some places, arrest people
- Ruin a credit score
- Change the news
- Change history
- Nuke the world (or just provide bad information to leaders and let them do it)
Yes, quite skynetish, eh? Luckily software isn't just one individual, nor was it written by one person. But the power is still held by all of the software in the world (or really the people who created it).
I believe that an essential part of democracy is allowing those that are interested to have a hand in controlling the systems that have the power. How?
The (A)GPL ensure perpetually that the end user of the software will have the clear ability to take control of the software on their devices. The AGPL could also be used to force transparency, another democratic necessity, of the methods used in voting, banking, etc.
A world with just Free Software (let's just say all AGPL for simplicity) should be substantially closer to perfect competition. Why?
- It lowers the barrier to entry, you can just pick up the source code to Gmail and make a new product. (Which Gmail can then copy from you).
- More sellers. The more sellers, the more responsive a supplier needs to be to you cause you can switch.
- Similar Products. They are mostly compatible with their specific value adds that will be likely incorporated into the next version of a competitor.
Perhaps this sounds a lot like Linux distros to you? 🙂
Well, Why Ubuntu?
We aren't there yet
Ubuntu makes some of the necessary compromises (proprietary drivers, etc) that give many more people, more of the freedom and control then they would have had without it. Right now, I feel Ubuntu has the best chance of getting us closer.
People need help to get involved with politics; communities exist to help concerned citizens to help change their governments. The Ubuntu community is here to help new users and contributors get involved helping to shape their software. Which is critical, because even if you have the freedom or right, doesn't mean you have the knowledge to use it. Receptive communities, like Ubuntu's, help get the knowledge to you.
Where are you going with this?
I was actually trying to answer the question, Why do I hate Microsoft? (I mean, come on, I don't want Mono included on the default Ubuntu install, so I clearly hate Microsoft or Novell, or somebody)
I don't hate Microsoft. I don't consider myself anti Microsoft or anti Mono. I have actually set people up to use both Windows and Mono applications before, gasp! And I actually like Novell. (I'm also a MCSA)
I consider myself pro Free Software and pro community created and governed languages (as well as content, and much more).
We have a better way to create software, a more democratic control structure over what we do, and most importantly, we give users great software and a path (through the community) to help them be in control of their software and have a say in its future.
Related Posts, somewhat referenced:
SciAm Column on "Rational Atheism"; The Dangers of Being "Anti" Rather Than "Pro"
Doctor Mo previously had a post called "Why do you like Microsoft"


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- This event has passed.
Eureka Springs Into Music – Classical Music
March 15, 2016 @ 7:00 pm - 9:00 pm$20.00
Event Navigation
Special performance March 15th, featuring Sara Sant'Ambrogio and Sara Perkins plus members of the Arkansas Philharmonic Youth Orchestra. Adult tickets are $20, under 17 is free.
Sara Sant'Ambrogio doesn't believe there should be artificial barriers between different types of music–or audiences. It's all music and it's all about communicating the human condition and emotions through music.
Two years ago, Sara and her Eroica Trio performed for a standing room only crowd in Eureka Springs and fell in love with the area. It was then she decided Eureka Springs would be the perfect locale for an annual music festival.
The week-long program will include several youth outreach elements including mini concerts, presentations and workshops at area schools. There will be a youth "competition" where students will receive instruction from Sara, and then will be selected to perform with the bee and other professional musicians at one of the concerts.
"Eureka Springs into Music" is a project of Sara Sant'Ambrogio, the City of Eureka Springs, and the Eureka Springs Arts Council.


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Verbatim - Black - remanufactured - toner cartridge ( equivalent to: HP CE505X ) - for HP LaserJet P2054, P2055, P2056, P2057Item#: 13705850 | Model#: 99230 |
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The municipality of La Pêche has approved a 3KM skating path that will run through the Gatineau Hills.
Local businessman Dave Mayer plans to maintain the trail all winter with an old firetruck for flooding and an ice surfacing machine to keep it smooth.
Businessman gets approval to build a 'Canal skateway without the canal' through forest in #Gatineau Hills. https://t.co/w4czc7pUUT #ottnews pic.twitter.com/NNeHkHlMgX— Ottawa Citizen (@OttawaCitizen) October 28, 2016


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Zyxel ICARD GOLD 1YR AV+IDP TURBO CARD (TURBOCARDGOLD1YR)Item#: 9298880 | Model#: TURBOCARDGOLD1YR
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Great work from our neighboring agency Story.
Seems so strange to see such bright modern imagery on a Whisky Society bottle.
They are very limited edition go get a move on.
The Far Flung Flavours are three new bottlings which have been chosen as they represent some of the most exciting and unusual taste experiences available from the Society. Each eye-popping limited-edition illustration is representative of the weirdly wonderful tastes and scents of the curious single malt within."
Our journey takes us to a special trio belonging to a tribe never before seen in these parts. Their clothing is unusual yet alluring; their tastes and aromas speak a different language. These are the Far Flung Flavours – guaranteed to surprise and delight all that have the pleasure of crossing their path. As with any epic expedition places are extremely limited, with only enough set supplies for just thirty members.


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Rationale: Biopsychosocial models of asthma have been proposed in the literature, but few empirical tests of social factors at various levels of influence have been conducted.
Objectives: To test associations of neighborhood, peer, and family factors with asthma outcomes in youth, and to determine the pathways through which these social factors operate.
Methods: Observational study of youths with asthma (n = 78).
Measurements and Main Results: Youths completed questionnaires about neighborhood problems, peer support, and family support. Biological (IgE, eosinophil count, production of IL-4) and behavioral (youth smoking, exposure to smoke, adherence to medications) pathways were measured. Asthma symptoms and pulmonary function were assessed in the laboratory and at home for 2 weeks. Lower levels of family support were associated with greater symptoms (β coefficients: −0.26 to −0.33, P < 0.05) and poorer pulmonary function (β: 0.30, P < 0.05) via biological pathways (Z statistics from 1.19 to 1.51, P < 0.05). Higher levels of neighborhood problems were associated with greater symptoms (β coefficients: 0.27–0.33, P < 0.05) via behavioral pathways related to smoking (Z statistics = 1.40, P < 0.05). Peer support was not associated with symptoms or pulmonary function.
Conclusions: This study indicates that family factors may affect youths' asthma via physiologic changes, whereas community factors may help shape the health behaviors of youths with asthma.
Biopsychosocial models of asthma have been proposed in the literature, but few empirical tests of social factors at various levels of influence have been conducted.
This study indicates that family factors may affect youths' asthma via physiologic changes, whereas community factors may help shape the health behaviors of youths with asthma.
The social environment has long been viewed as an important determinant of asthma outcomes in youth. Consistent with biopsychosocial approaches to conceptualizing the onset and course of asthma (1, 2), research indicates that factors such as exposure to violence (3), abrasive family relationships (4, 5), and psychological stress (6–8) all relate to adverse outcomes in youths with asthma.
However, most previous studies have focused on social factors at a single level (e.g., the family). At the broadest level of social influence, the neighborhood, factors such as greater violence in the neighborhood and lower socioeconomic status of neighborhoods have been associated with greater asthma morbidity and heightened allergic inflammatory responses (9–11). At a more circumscribed level of social influence, one's peers, high stress in combination with low levels of social support increases risk for asthma exacerbations (12). At the most proximal level of social influence, the family, parenting difficulties and parental stress have been associated with markers of inflammation and high risk for onset of childhood asthma (5, 6, 13).
By contrast, fewer studies have provided a more complete picture of the relative contributions of neighborhood, peer, and family factors, or asked whether they influence asthma through similar versus different mechanisms. Understanding these contributions has important implications for interventions. For example, associations between neighborhood factors and asthma may suggest the need for interventions aimed at making community-wide changes. In contrast, associations between family factors and asthma would suggest specific interventions targeted at the dynamics among family members. In addition, there is a need for research that situates individuals within the larger social context in which they live. The present study addresses this gap by investigating social connections at the family and peer level. The goals of the present study were as follows: (1) to examine how social factors at the neighborhood, peer, and family levels relate to morbidity in a sample of youths with asthma; and (2) to identify mechanisms linking social factors and disease outcomes by exploring both biological and behavioral pathways. We hypothesized that greater neighborhood problems, a lack of peer support, and a lack of family support would all relate to greater asthma morbidity, and that immune and behavioral variables would comprise pathways linking social factors to asthma (social factors → behavioral/biological → asthma clinical outcomes).
Seventy-eight youths who were physician-diagnosed with asthma (70% allergic asthma) were recruited from Vancouver, BC, Canada, from asthma clinics, newspaper advertisements, and school and community center postings. The youths ranged in age from 9 to 18 years, were fluent in English, free of acute respiratory illness at the time of their visit (by parent and youth report), had not had a prednisone course for at least 2 weeks, and had no chronic illnesses other than asthma. The protocol was approved by the University of British Columbia Research Ethics Board.
Please refer to the online supplement for additional information.
The Social Support Scale for Children assesses the degree to which youths lack a parent who understands, values, or cares about them (14).
The Social Support Scale for Children also assesses the degree to which youths do not have at least one close friend who they can talk to and who understands them (14).
The Chicago Youth Development Study (CYDS) Community and Neighborhood measure assesses the degree to which youths perceive problems in their neighborhood, such as crime, gangs, drugs, and graffiti (15).
Youths were asked the number of days over the past 6 months that they had smoked cigarettes. Youths were also asked the number of days over the past 6 months that they had been exposed to smoke. These variables were log-transformed in analyses because of their nonnormal distribution.
Families brought their youths' medications to the research center, and each drug's name and dose was recorded from the label. Adherence was determined with respect to daily prescribed medications, and defined as the number of days youths had taken an inhaled corticosteroid during the past 14 days.
A complete blood count with five-part differential (Bayer Advia 70 hematology system; Diamond Diagnostics, Holiston, MA) was performed to obtain eosinophil counts. Total serum IgE was measured using an automated fluorescence immunoassay (Pharmacia CAP system; Phadia, Inc. Portage, MI), and was log-transformed because of its nonnormal distribution. Production of the Th2 cytokine IL-4 by peripheral blood mononuclear cells stimulated for 48 hours with phorbol myristate acetate (25 ng/ml) and ionomycin (1 μg/ml) was measured as described previously and as described in the online supplement (7).
Pulmonary function was evaluated through spirometry according to American Thoracic Society guidelines (16). FEV1 was derived and calculated as a percentage of predicted values, based on age, sex, ethnicity, and height (17). Measures were taken at least 4 hours after the last use of a short-acting bronchodilator, and at least 24 hours after the use of a long-acting bronchodilator, following the protocol of a multisite clinical asthma trial (18). Youths also monitored PEF at home using an electronic monitor (Quadromed, Hoechberg, Germany). Three peak flow readings were taken upon awakening each day for 2 weeks. Morning PEF% was calculated as a percentage of each youth's laboratory best result, and averaged across the 2 weeks. Youths who did not complete at least 10 to 14 days of measurements were excluded from peak flow analyses.
In the lab, youths were interviewed about symptoms during the previous 2 weeks. The frequency of daytime, nighttime, and exertional symptoms were probed as per the National Asthma Education and Prevention Program, Expert Panel Report 2, (NAEPP/EPR2) guidelines (19). After the lab visit, youths kept a diary of the intensity of their symptoms every day for 2 weeks. Diary symptoms were averaged across the 2-week home monitoring period.
Variables that could provide alternative explanations for the above relationships were included as covariates in statistical analyses. This included demographic characteristics (age, sex, ethnicity), and asthma severity, determined from the NAEPP/EPR2 guidelines based on the higher of symptom frequency and medication use, paralleling the approach of previous researchers (20).
Three sets of statistical analyses were conducted. In the first set, we tested associations of social factors with both asthma clinical outcomes and with proposed pathways (behavioral and biological variables). To do this, we conducted multiple regression analyses in which the outcome variable (asthma symptoms, pulmonary function) was regressed onto covariates (age, sex, ethnicity, asthma severity) in step 1 and then onto the predictor variable (social factor) in step 2. The same procedure was then followed using behavioral and biological mechanisms as the outcome variable. Standardized regression coefficients (β) are presented, as well as percentage of variance in the outcome variable that can be accounted for by the predictor variable (ΔR2, an indicator of effect size). β coefficients indicate the number of standard deviations the dependent variable changes for each standard deviation increase in the independent variable. For example, a 0.5 coefficient between neighborhood problems and symptoms would indicate that one standard deviation increase in neighborhood problems is associated with half a standard deviation increase in symptoms. Conversely, a −0.5 coefficient between neighborhood problems and pulmonary function would indicate that a one standard deviation increase in neighborhood problems is associated with half a standard deviation decrease in pulmonary function.
The second set of analyses consisted of statistical mediational tests. We note that this study is cross-sectional and observational, and hence we cannot draw causal inferences about whether social factors influence behaviors/biology and in turn clinical outcomes. Nonetheless, statistical mediation tests provide a method whereby one can test whether data are consistent with our proposed model of social factors → behavioral/biological → clinical outcomes. To do this, we applied the Sobel test with the distributional properties recommended by MacKinnon and colleagues (21). This statistic tests the significance of an indirect pathway using a products of coefficient test that gives a Z statistic. Z scores greater than 0.97 indicate a statistically significant pathway. Although we propose an indirect pathway through behaviors/biology, it could be the case that social factors are associated with clinical outcomes for reasons that have nothing to do with behaviors or biology (e.g., environmental exposures, health care characteristics). If this were the case, we would observe nonsignificant Z statistics. Hence, what the Sobel test does is to evaluate whether each specific indirect pathway we have proposed (e.g., family support → inflammatory processes → clinical outcomes) is statistically significant. Another way that one can think of these analyses is as testing whether the overlap between social factors and clinical outcomes is also shared with behaviors or biology.
The Sobel test assesses the significance of the indirect pathway, αβ, by dividing the indirect pathway effect by its standard error [αβ/square root of (α2 × σβ2 + β2 × σα2)], where α represents the relation between the independent variable (e.g., family support) and the proposed mediator (e.g., inflammatory processes), β represents the relation between the mediator and the dependent variable (e.g., clinical outcomes) adjusted for the independent variable, and σ represents standard error. MacKinnon and colleagues have argued that the traditional Sobel test has low power because the αβ product is often not normally distributed, and based on extensive simulation studies, they recommend a critical value of 0.97 for the 0.05 significance level.
For statistical mediation analyses, we created composite scores of behavioral, biological, and clinical variables to reduce the number of statistical analyses and minimize the probability of type I error. This was done after conducting factor analyses to ensure that this approach was empirically justified. For behaviors, we created a composite smoking variable by standardizing and summing responses to youth smoking and exposure to smoke. For biological variables, we standardized each variable (because of the vastly different ranges) and then summed values (IgE, eosinophil count, IL-4). For clinical variables, we created a composite symptom score by averaging responses to each of the three symptom questions asked during the lab visit as well as symptoms reported on daily diary cards at home. We also created a composite pulmonary function variable by averaging values for FEV1% and home PEF%. However, because some youths were missing peak flow data (n = 26), we also tested mediation using just the FEV1% variable (hence with the full sample). Similar patterns emerged with both approaches, and are reported for the full sample below.
The third set of analyses tested whether family, peer, or neighborhood factors were more strongly related to asthma outcomes by conducting simultaneous regression analyses in which covariates of age, sex, ethnicity, and asthma severity were entered in the first step, and then the three social factors (neighborhood, peer, family) were entered simultaneously in the second step to determine which social factor had the strongest independent relationship with asthma clinical outcomes.
Descriptive information about the sample is presented in Table 1. See Figure 1 for a depiction of some of the findings below.
After controlling for age, sex, ethnicity, and severity, multiple regression analyses revealed that lower levels of family support were associated with marginally lower laboratory FEV1% (β = 0.20, ΔR2 = 3.9%, P = 0.098), and significantly lower at-home morning PEF % (β = 0.30, ΔR2 = 8.0%, P = 0.046). In addition, lower levels of family support were associated with marginally greater daytime symptoms during the past 2 weeks (β = −0.19, ΔR2 = 3.5%, P = 0.09), greater nighttime symptoms during the past 2 weeks (β = −0.26, ΔR2 = 6.7%, P = 0.027), and greater exertional symptoms during the past 2 weeks (β = −0.33, ΔR2 = 10.2%, P = 0.005).
In contrast, friend support was not associated with any pulmonary function or symptom variables (all P > 0.2).
After controlling for age, sex, ethnicity, and severity, greater neighborhood problems were associated with greater daytime symptoms during the past 2 weeks (β = 0.33, ΔR2 = 9.8%, P = 0.004), as well as with greater symptoms reported during the 2-week home monitoring period (β = 0.27, ΔR2 = 6.2%, P = 0.029). Neighborhood problems were not associated with pulmonary function variables.
Table 2 contains standardized coefficients for analyses reported below.
After controlling for age, sex, ethnicity, and severity, multiple regression analyses revealed that lower levels of family support were associated with biological measures in a direction detrimental to asthma, including higher levels of total IgE (β = −0.27, ΔR2 = 6.8%, P = 0.043), higher eosinophil counts (β = −0.31, ΔR2 = 9.1%, P = 0.009), and greater production of IL-4 (β = −0.44, ΔR2 = 18.6%, P = 0.0004). In contrast, family support was not associated with any behavioral measures, including youth smoking, exposure to smoke, or adherence to medications.
Friend support was not associated with any biological variables or with youth smoking or exposure to smoke (all P > 0.15). There was an unexpected negative association of high friend support with low adherence to medications (β = −0.40, ΔR2 = 12.8%, P = 0.012).
After controlling for age, sex, ethnicity, and severity, greater neighborhood problems were associated with detrimental behaviors, including greater amounts of youth smoking (β = 0.44, ΔR2 = 17.0%, P = 0.0001), greater exposure to secondhand smoke (β = 0.42, ΔR2 = 14.9%, P = 0.001), and less adherence to asthma medications (β = −0.36, ΔR2= 8.7%, P = 0.039). In contrast, neighborhood problems were not associated with biological variables.
We next conducted statistical mediation tests, with Z > 0.97 indicating a statistically significant pathway. For family support, we tested biological and behavioral pathways to both symptoms and pulmonary function. We found that biological processes constituted a significant pathway linking family support to asthma symptoms (Z = 1.51), consistent with the idea that a lack of family support may result in increased allergic inflammation, which could then lead to an exacerbation of symptoms (see Figure 2). In contrast, the behavioral pathways of smoking (Z = 0.33) and adherence to medications (Z = 0.58) did not form significant pathways linking family support and asthma symptoms.
Second, we tested pathways between lack of family support and reduced pulmonary function. Biological processes also formed a significant pathway linking these variables (Z = 1.19), whereas the behaviors of smoking (Z = 0.33) and adherence to medications (Z = 0.62) did not. These data suggest that a lack of family support may increase inflammatory processes, and in turn reduce pulmonary function.
We then tested pathways between neighborhood problems and asthma symptoms. The behavioral measure of smoking formed a significant pathway linking neighborhood problems with asthma symptoms (Z = 1.40), whereas the behavioral measure of adherence to medications (Z = 0.17) did not. In addition, biological processes did not form a significant pathway linking neighborhood problems with asthma symptoms (Z = 0.51). Given that neighborhood problems were not associated with pulmonary function, we did not test pathways between these two variables. We also did not test pathways related to peer support, given that peer support was not associated with pulmonary function or asthma symptoms. See Figure 2 for a summary of pathways.
Last, we tested whether family, peer, or neighborhood factors were more strongly related to asthma outcomes by conducting simultaneous regression analyses. With respect to pulmonary function, a lack of family support (β = 0.29, ΔR2 = 6.0%, P = 0.036) but not peer support (β =0.04, ΔR2 = 0.1%, P = 0.75) or neighborhood problems (β = −0.14, ΔR2 = 1.6%, P = 0.27) was associated with reduced pulmonary function. This suggests that, when compared against one other, family factors emerge as having a stronger relationship with pulmonary function than neighborhood or peer factors.
With respect to asthma symptoms, neighborhood problems were associated with greater symptoms (β = 0.26, ΔR2 = 6.0%, P = 0.023), whereas there was only a marginal association for lack of family support (β = −0.23, ΔR2 = 3.8%, P = 0.067). Peer support was not associated with symptoms (β = 0.08, ΔR2 = 0.4%, P = 0.55). This suggests that, when the three are compared against one other, neighborhood factors have the strongest relationship with asthma symptoms, with family factors also contributing, but more modestly, to symptoms.
The present study tested the association of the social environment at multiple levels (neighborhood, peer, family) with asthma outcomes in youth. Although the study was cross-sectional and hence definitive conclusions cannot be drawn about directionality, evidence was consistent with the hypothesis that a lack of family support was related to increased asthma symptoms and poorer pulmonary function via allergic inflammation. In addition, evidence was consistent with the hypothesis that greater neighborhood problems were related to greater asthma symptoms via smoking behaviors. Peer support was unrelated to asthma outcomes. These findings suggest the possibility that, among youths with asthma, the family and the neighborhood play a more important role than peer support.
With respect to the family, our findings are consistent with previous observational studies documenting that dysfunctional family interactions predicted children with persistent atopic symptoms at age 3 (22). The experience of low levels of support from family may activate hormonal and inflammatory processes that contribute to asthma (23, 24). Our findings suggest the intriguing possibility that family factors may affect youths' asthma through direct biological mechanisms, such as allergic inflammation, rather than through medication adherence and other health behaviors, such as smoking.
Neighborhood factors were related to asthma outcomes through behavioral rather than biological pathways. This may be because neighborhoods set up norms for what types of behaviors are acceptable and because people have a tendency to copy the behaviors of those around them (25). For example, neighborhoods with higher rates of smoking would both expose youths to greater amounts of smoke and create social norms about the acceptability of smoking. Other researchers have suggested that community-level interventions could help change behavior at the individual level by providing informational networks that supplement gaps in individual patient knowledge (26). Such interventions, including providing community-wide asthma education and sponsoring health fairs, represent a recent trend with some promising initial results (27).
Unlike family and neighborhood factors, peer factors were not associated with asthma outcomes. It may be the case that social support networks are important for asthma among adults (28) but less so among youth. In particular, the influence of peers increases during adolescence, and thus the lack of peer findings in the present study may have been due to the broad age range of youths (9–18) in this sample.
The present study also provides evidence that is consistent with biopsychosocial models of asthma. For example, low levels of family support were associated with greater production of IL-4, higher levels of IgE, and more eosinophils. IL-4 induces B-cell production of IgE, which in turn activates cells to release histamines and leukotrienes, leading to airway constriction and edema (29). Eosinophils release inflammatory mediators, such as leukotrienes that contribute to airway injury and inflammation, and are associated with symptoms and severity level of asthma (30, 31). The present study also went beyond previous mechanistic research by including clinical outcomes and showing that low levels of family support were associated with heightened inflammatory markers, which in turn were associated with greater asthma symptoms and poorer pulmonary function. However, we acknowledge that this is merely one explanation for how social factors relate to asthma morbidity, and that there may be numerous other factors that also play into this relationship.
In simultaneous regression analyses, family factors were associated with pulmonary function more strongly than were peer or neighborhood variables. Given that measures of pulmonary function reflect airway obstruction, this suggests that family-level characteristics play an important role in youths' degree of airway obstruction, perhaps through effects on inflammation. In contrast, neighborhood factors were associated with asthma symptoms more strongly than were peer or family variables. To the extent that asthma symptoms reflect perceived disease burden, this suggests that neighborhood-level characteristics are important for how youths experience their asthma, perhaps in part due to environmental triggers such as smoke exposure. Overall, these patterns indicate that different components of asthma may be shaped by different social environment factors. As such, the most comprehensive picture of the determinants of asthma morbidity may come from situating an individual patient within the larger social context in which she/he exists.
In the present study, adherence to medications was not a significant pathway linking social factors to symptoms or pulmonary function. It could be that medication adherence is related to social variables other than family support or neighborhood problems, such as families' knowledge about asthma or the quality of physician–patient relationships. Alternatively, it is possible that some families inaccurately reported medication adherence, and that measurement error obscured relationships between adherence and asthma outcomes. Although adherence to medications is clearly an important determinant of asthma morbidity in youth (32), this study raises the possibility that links between adherence and symptoms may in part reflect the role of other health behaviors and biological processes.
Strengths of the present study include the assessment of social environmental factors at the neighborhood, peer, and family levels. In addition, the assessment of biological and behavioral pathways allowed us to develop models of how the larger social environment may get "under the skin" of an individual youth to influence disease.
Limitations of the present study include the sample size and the cross-sectional observational design. The cross-sectional nature of this study means that we cannot know for certain the directionality of effects. For example, although our proposed model is that neighborhood and family factors have effects on behaviors and inflammatory processes, which in turn affect morbidity, it is also possible that youths who experience more symptoms have more difficult family relationships, or that symptoms result in changes to inflammatory profiles, which can regulate central nervous system function in ways that affect social behaviors. To more clearly assess directionality, longitudinal studies are needed that repeatedly assess these factors over time. In addition, another limitation was the self-report nature of behavioral variables such as medication adherence and smoking. Self-reports may be biased because of a desire to present oneself in a positive light, or because of difficulties with recall about behaviors over extended periods of time, all of which may have clouded associations with these variables. Future studies should use other approaches, such as electronic monitoring of medications. Medication regimens also differed across children, potentially complicating conclusions about links between the social environment and immune and clinical outcomes; however, asthma severity, which takes medication regimen into account, was controlled for in the present study. Finally, it should be noted that youths were recruited into this study on the basis of physician diagnosis of asthma and that physician diagnosis is not always accurate.
Future research is needed that places this study in a larger context by investigating the relative contributions of social versus other factors, such as genetics, allergen exposure, and viral infections in asthma, given that social factors in the present study accounted for only a modest percentage of the variance in clinical outcomes. Effect sizes in the present study were small, likely because asthma is a complex, multifactorial disease. Nonetheless, ascertaining the relative contribution of specific social environmental factors is important for gaining a more comprehensive understanding of childhood asthma. Future research is also needed to understand how relationships of social factors with asthma and the associated mechanisms change with age, pubertal status, and developmental milestones.
In sum, the present study provided evidence consistent with the hypothesis that poor family support is associated with decreased pulmonary function and greater asthma symptoms through biological mechanisms involving inflammation. In contrast, evidence suggests that neighborhood problems are associated with greater asthma symptoms via behavioral pathways related to smoking. Poor family relations may foster psychological experiences with direct physiologic consequences, whereas problematic neighborhoods may operate by providing role models for maladaptive health behaviors. Overall, these findings suggest that the larger social environment is linked to asthma, and that there are multiple pathways through which family and neighborhood factors may come to affect functioning in children with asthma.
Supported by National Institutes of Health grant HL073975 and the Canadian Institutes of Health Research.
This article has an online supplement, which is accessible from this issue's table of contents at www.atsjournals.org
Originally Published in Press as DOI: 10.1164/rccm.200610-1473OC on June 7, 2007
Conflict of Interest Statement: None of the authors has a financial relationship with a commercial entity that has an interest in the subject of this manuscript.
PubMed Central Canada is a service of the Canadian Institutes of Health Research (CIHR) working in partnership with the National Research Council's national science library in cooperation with the National Center for Biotechnology Information at the U.S. National Library of Medicine(NCBI/NLM). It includes content provided to the PubMed Central International archive by participating publishers.


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Lukas stared out from the hanger bay of the Terminus. It was a long way down to the surface of Noveria and he had to admit he was a little bit nervous about what he was preparing to do, not that he let his team know that, after all what would there reaction be if they knew even he was a bit worried about this?
Pushing the fear from his mind he brought his suits HELLJUMPER systems online and ran a diagnostic check just be safe. as he looked around he could see the rest of his team doing the same thing.
Wilhelm informed him that they were approximately 5 minutes out from the drop zone. 5 minutes to prepare for whatever Cerberus had to throw at him. Checking over his Saber rifle he paused to see what everyone else was carrying to battle. He could Johan wielding his stupidly over-sized Striker Assault Rifle, next to him stood Frau Delia who was carrying a custom built pistol he couldn't quite identify. At the other end of the cargo bay stood Jerik and Denk, a


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|13 Dec 2014
KB3011970 Query
After running an update check today I was prompted to install KB3011970, even though it's already installed.
I couldn't find any other threads about this and don't get why it needs downloading/installing again (no reboot was required).
My update history now lists KB3011970 twice, one for the 9th and one with today's date. Can someone tell me what's going on with this patch?
Many thanks for your help.
|My System Specs
|13 Dec 2014
The original update was pulled and has itself been updated after problems with the original - they may have released the new version already.
|My System Specs
|13 Dec 2014
Hi NoelDP, thanks for getting back so quickly.
Thanks for letting me know about this - I'd read the update had been pulled but thought it was usual for re-released updates to have different numbers to the originals.
|My System Specs
|13 Dec 2014
Only if they've had to make significant changes to it - the KB number itself is set at the beginning of the project, and can go through a number of iterations in release (most of which aren't noticed by the end-user).
|My System Specs
|13 Dec 2014
Windows 7 Professional SP1 - x64 [Non-UEFI Boot]
Check your installed updates in Program & Features and you will find the MS Silverlight v5.1.31211.0 is the newest.
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Paulson Says Business Is Over-Regulated
Tuesday, November 21, 2006
Treasury Secretary Henry M. Paulson Jr. yesterday criticized the nation's "ever-expanding rulebook" and its burdensome legal system for constraining the economy but rejected wholesale revisions to a corporate accountability law under attack from business groups.
Paulson, a former chairman of the investment bank Goldman Sachs, called for striking a regulatory balance as he delivered his first major policy address on the subject since joining the government in July. "Excessive regulation slows innovation, imposes needless costs on investors, and stifles competitiveness and job creation," he said in a speech to the Economic Club of New York.
But under questioning, Paulson stood behind the controversial Sarbanes-Oxley law, passed in 2002 after financial scandals rocked the stock market and devastated investor confidence. Policymakers do not need to reopen the law, Paulson said. Instead, he expressed confidence in the work of securities regulators and accounting industry overseers, who are racing to make audit rules more flexible for small businesses that have complained about the regulations' cost. The eagerly awaited proposed revisions are to be unveiled within weeks, Christopher Cox, chairman of the Securities and Exchange Commission, has said.
"I don't think there is a single principle in [Sarbanes-Oxley] that is ill-founded," Paulson said after the speech.
Paulson's remarks come as trade groups such as the U.S. Chamber of Commerce exhort lawmakers and regulators to retool corporate rules, which they contend are leading fewer companies to list on U.S. exchanges. A chamber-affiliated coalition led by William M. Daley, who was an official during Bill Clinton's presidency, and A.B. Culvahouse, counsel to former president Reagan, issued data yesterday that they said reflected a decade-long decline in market share of corporate listings in the United States.
Some consumer advocates warn that tighter rules make for a convenient scapegoat. Barbara Roper of the Consumer Federation of America pointed to other factors, including steep investment banking fees and changes in the global economy, for the dip in U.S. public offerings.
Business groups insist the changes would combat what they say is an overly onerous application of the law. "No one is talking about a wholesale rollback," said Robert S. Nichols, president of the Financial Services Forum, a coalition of large banks for which Paulson once served as chairman. "That would be a very serious mistake in our view. It's a matter of discussing the implementation and execution."
In his speech, Paulson sought to take the debate beyond recent corporate reforms and into broader terrain. He singled out class-action lawsuits as an Achilles' heel in the legal system, which he contended have "gone beyond protection" of investors and now hamper business competition.
Statistics compiled by NERA Economic Consulting suggest that the number of class-action lawsuits filed against corporations and executives has plummeted in the past two years, especially since the indictment of the law firm Milberg Weiss Bershad & Schulman, which had been a leader in filing plaintiff cases before being charged with improperly paying people to serve as clients.
"Investors already are getting pennies on the dollar for massive losses they suffered in recent scandals," said Roper, of the consumer group. "I don't think you can sell that message after the experience we've been through."
Separately, Paulson called for more "constructive" dialogue among prosecutors and corporate executives, citing possible changes to the Justice Department's guidelines on charging companies with crimes. The 2002 prosecution of accounting firm Arthur Andersen for charges of obstructing justice helped push it out of business, an issue that Paulson said has reduced choice and competition in the auditing industry.
An unusual coalition including the American Civil Liberties Union and the U.S. Chamber of Commerce testified before Congress this year in support of relaxing parts of the Justice guidelines, known as the Thompson memo, named after former deputy attorney general Larry D. Thompson.
Paulson said he would host a conference early next year to explore problematic rules, accounting and legal issues and to lay a foundation for possible new initiatives within the executive branch and Congress. Last week, Sen. Christopher J. Dodd (D-Conn.), who is set to lead the Senate Banking Committee when Congress returns in January, expressed skepticism that the Sarbanes-Oxley law had hampered U.S. competitiveness.
But the wider topic appears to have drawn some bipartisan support. Sen. Charles E. Schumer (D-N.Y.), whose constituents include many Wall Street executives, yesterday referred to "a growing consensus . . . that we need to do a complete and comprehensive review of America's and New York's worldwide competitive position in financial services."


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Associate Justice
|This article needs additional citations for verification. (September 2008) (Learn how and when to remove this template message)
Associate Justice or Associate Judge is the title for a member of a judicial panel who is not the Chief Justice in some jurisdictions. The title "Associate Justice" is used for members of the Supreme Court of the United States and some state supreme courts, and for some other courts in Commonwealth countries, as well as for members of the Supreme Court of the Federated States of Micronesia, a former United States Trust Territory.[1] In other common law jurisdictions, the equivalent position is called "Puisne Justice".
In the United States, judicial panels are non-hierarchical, so an Associate Judge has the same responsibilities with respect to cases as the Chief Judge.
An Associate Judge usually has fewer or different administrative responsibilities than the Chief. On the Supreme Court of the United States, the most junior Associate Justice (currently Justice Neil Gorsuch), has the task of answering the door when the Justices are in private conference.[citation needed]
There are eight (seven between February 13, 2016, and April 10, 2017. On April 10, 2017, Justice Neil Gorsuch filled the vacancy after Antonin Scalia died.) Associate Justices on the United States Supreme Court, and three Associate Justices on the Supreme Court of the Federated States of Micronesia.[2]
In New Zealand and New South Wales, Associate Judges of the High Court of New Zealand and Supreme Court of New South Wales respectively supervise preliminary processes in most civil proceedings. Associate Judges have jurisdiction to deal with such matters as: summary judgment applications, company liquidations, bankruptcy proceedings, and some other types of civil proceedings.[3] In the New Zealand legal system, Associate Judges were formerly known as Masters.
See also[edit]
- ^ "The Federated States of Micronesia Mourns the loss of one of its Founding Fathers: Chief Justice Andon Amaraich", Government of the F.S. Micronesia, January 28, 2010
- ^ "THE SUPREME COURT OF THE FEDERATED STATES OF MICRONESIA", official Legal Information Service
- ^ "Archived copy". Archived from the original on 2006-10-03. Retrieved 2006-09-01.
|This law-related article is a stub. You can help Wikipedia by expanding it.


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Parents with students starting Kindergarten in August 2017 are invited to register their child during our annual Kindergarten Round Up event on February 7 from 4:30 p.m. to 6:30 p.m. You will also have the opportunity to meet the teachers and visit kindergarten classrooms. Children registered on this date will receive a free gift.
Registration will take place at the following Laveen schools: Cheatham, Desert Meadows, Laveen, M.C. Cash, Paseo Pointe, Rogers Ranch, and Trailside Point. Kindergartners enrolled at Paseo Pointe must be part of the Dual Language Immersion program, a 50/50 model where students spend half their day in a classroom where teaching and learning is conducted in Spanish and the other half in English.
Students may be enrolled in any of our schools regardless of where they live, space permitting. Families living outside of a particular school's transportation zone will be responsible for providing transportation.
Please bring the following items to register your child.
* Proof of Residence (examples include: utility bill, rental agreement, mortgage statement, etc.)
* Child's Immunization Record
* Child's Original Birth Certificate (copies not accepted)
* Parent/Guardian's Photo ID
Children must be 5 years old on or before August 31, 2016 to register. Children turning 5 after August 31, but by December 31, may be evaluated for early entrance at a later date.
Laveen offers free Full Day Kindergarten at all schools.
View the flyer for additional details.


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Systems theory in anthropology is an interdisciplinary, non-representative, non-referential, and non-Cartesian approach that brings together natural and social sciences to understand society in its complexity. The basic idea of a system theoryin social science is to solve the classical problem of duality; mind-body, subject-object, form-content, signifier-signified, and structure-agency. System theory, therefore, suggests that instead of creating closed categories into binaries (subject-object); the system should stay open so as to allow free flow of process and interactions. In this way the binaries are dissolved.
Source: Systems theory in anthropology – Wikipedia


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Words containing dorfbeue
Found 1 words containing dorfbeue. Browse our Scrabble Word Finder, Words With Friends cheat dictionary, and WordHub word solver to find words that contain dorfbeue. Or use our Unscramble word solver to find your best possible play! Related: Words that start with dorfbeue
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