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[Note: this is a regular feature of GeneaBloggers which highlights new genealogy and family history-related blogs as well as those recently discovered by members of GeneaBloggers. Use the Suggest A Blog! link in the menu bar to pass along information on new blogs.]
There are 9 newly-discovered genealogy and family-history related blogs that we've located this week for a total of 936 genealogy blogs on our list! Remember to try and help out these new blogs by:
– using the Follow feature if the blog is hosted on Blogger
– adding them to your blog reader
– adding a comment on their blog saying "hi" and "welcome"
Here are this week's new listings:
Ancestral Connections
Blog type: Genealogy vendor blog
Welcome to Ancestral Connections' new blog. This is the platform for us to begin a dialogue on genealogy. Feel free to comment, ask questions, and come back often to contribute. We look forward to conversing with you about our precious genealogy. Thank you!
Russ Bangerter is a professional genealogist and principal researcher for Ancestral Connections, Inc. Russ finds great joy in helping clients break down the "brick walls" that people often confront in researching their genealogy. Follow this link to the company website to learn more, http://www.ancestralconnect.com.
Betty's Genealogy Blog
Blog type: Individual family history
Greetings! This is the very first post on Betty's Genealogy Blog. My name is Betty, and I love to do research. This blog will be a place where I share new discoveries, little miracles, and "brick walls", as well as general discussions on history and genealogy, as I continue to search out all the branches of my family tree in the United States and beyond.
Clue Wagon
Blog type: Individual family history
After nearly 15 years in human resources, I left to stay home with my two small children. Then I started a blog for job seekers. After a year, of talking about resumes and interviews and crazy candidates, I finally worked up the courage to pursue my REAL dream: becoming a professional genealogist. Now, instead of talking about employment forms, I talk about census forms.
My blog is about my journey, my research, and occasionally about ice cream. It's fun. You should visit.
Dust To Dust GYRabbit
Blog type: Arizona genealogy, Cemetery blog, Graveyard Rabbit blog
I have become a blogging groupie. This is my second blog and it is dedicated to all my ancestors in all those forgotten cemeteries across our beautiful country. I will be showcasing the cemetery information I have about my ancestors but I also hope to use this blog as a repository for the numerous cemeteries located in the Yavapai county area of Arizona. We are not quite the high country but we are certainly not the desert. I have googled for information about cemeteries in this area of Arizona and have not found much. I hope I can help to supplement the resources for those searching for their pioneer families here in the mile high country of Arizona.
Finding Josephine
Blog type: African-American genealogy, Individual family history
I've been searching for information about my family since I was 12 and asked my grandfather why he looked so white unlike the rest of us Fords. His answer, that his grandfather was a plantation owner, his grandmother a slave on that plantation, ignited the spark and I've been trying to collect information ever since.
Blog type: Research blog
GenAnswers is a free genealogy question and answer community. Post your genealogy questions and share your knowledge with others by answering questions.
Gen Journey
Blog type: Individual family history
Well here it is. I guess you might call this a New Years Resolution project. Over the years I have saved several groups of family ephemera, another way of saying family junk to some. One of the things that has always struck me as rather odd is the idea of a family discarding this stuff. I suppose once all of the individuals who produced these letters, cards, and notes have died; those left behind may feel little attachment or regard for the documents left behind.
Perhaps if people realized that these documents have meaning even long after the individuals creating them are no longer with us, might be a little less likely to throw this tuff away. The documents our ancestors created say not only a lot about the individuals themselves, but also the times they lived in. These items speak to me and hopefully will speak to you too!
Reconnected Roots
Blog type: African-American genealogy
This is the African-American Genealogy Website Dedicated to Researching and the retelling of our Ancestor's story. You won't find much about the living here, as they are fully able to speak for themselves. However, there were many who came before us, who have lessons to offer. They did not have use of the internet to blog, or even have time to keep journals. But they did leave us bits and pieces of clues. The clues are the reason for this website. If we can piece together the story of our ancestors, we can gain a glimpse of how they lived, what burdens they carried and ultimately how they prevailed. The hope is with that knowledge, each of us will gain a sense of respect and pride for those that traveled here before us. The goal is that the retelling of their lives will empower us to make the most of ours.
Southwest Arkie
Blog type: Appalachian genealogy, Arkansas genealogy, Individual family history
Genealogy is my middle name. I've been bumping my head into brick walls since I was 18 years old and without exactly telling you my age, let's just say it's been over 30 years… When I am not stirring things up with own family tree, I stay busy taking care of several usgw county sites, I am also vice president of my local genealogy society, and I present programs to same gen society on topics ranging from "I know a little bit about this" to "I might can help you with that". Genealogy societies are a dying breed, but we do our best to hang on to what we have accomplished, and to push ourselves into the electronic age, and I am proud of that. I do lookups for anyone who asks, and I am also a volunteer for Random Acts of Gen Kindness and Findagrave. My own personal family research has been pushed to the back burner so many times that I can no longer recite names and dates and this makes me sad. This blog is going to be my attempt to slow down for a second and start "thinking" about it all. Hopefully if I see it in black and white, I might actually break down some of these brick walls.
© 2010, copyright Thomas MacEntee


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TOWSON, MD Feb. 4, 2013 – Towson dominated play inside with 48 points in the paint en route to an 81-68 victory over UNCW on Monday night, completing a sweep of the regular season men's basketball series at the Towson Center.
The Tigers won their second straight to square their record at 12-12 and 7-4 in the CAA. The Seahawks dropped both games on their two-swing to the mid-Atlantic region and fell to 8-14 and 3-7.
Towson, which defeated UNCW 79-74 back on Jan. 2 in Wilmington, shot 52.6 percent from the floor to hand the Seahawks a loss in their final appearance in the arena. The Tigers are moving into a new 5,200-seat facility next door in 2013-14.
Towson junior forward Jerelle Benimon led all players with 20 points and added 12 rebounds for his NCAA-leading 16th double-double of the season, Freshman playmaker Jerome Hairston added 18 points, junior guard Rafriel Guthrie chipped in 14 and junior guard Marcas Damas had 12.
Senior forward Keith Rendleman paced the Seahawks with 19 points and 12 rebounds for his 12th double-double of the season and 34th of his career. Redshirt freshman Craig Ponder collected 12 points and junior guard Chris Dixon and junior forward Shane Reybold contributed 11 points apiece to give UNCW four players in double figures for the first time this season.
"They're a very talented team," said third-year UNCW head coach Buzz Peterson. "I think they scored on seven of the last eight possessions in the first half and they hit a couple crazy shots against our zone. We gave up too many second and third chances and that hurt."
The Tigers, behind Benimon and Hairston, shot 57.1 percent in the first half to build a 42-32 lead at the break. Hairston scored 14 of his total in the first 20 minutes and Benimon went for 13 points early in the contest.
UNCW was still hanging around midway through the second half and trailed by just six points, 54-48, after Ponder dropped in a pair of free throws with 12:45 remaining.
But Towson scored the next 11 points behind Damas to pull away. Damas scored five straight points and two free throws by reserve forward Timajh Parker-Rivera extended the lead to 65-48 with 8:54 left.
Towson then enjoyed its largest lead, 79-55, when Hairston hit a floater in the lane before the Seahawks closed out the game by scoring 13 of the final 15 points.
After the Seahawks inched out to an 11-7 advantage on a jumper by Ponder, the Tigers reeled off 12 straight points for a 19-11 lead. Five different players scored and Mike Burwell's steal and layup at 9:47 gave the Tigers an eight-point cushion.
UNCW answered with a 12-0 run of its own. Dylan Sherwood started the spurt with a triple from the right wing and Reybold's twisting hook shot put the Seahawks ahead, 23-19, with 6:40 left in the opening period.
Towson closed out the first half strong, however, to take a 10-point lead into the lockerroom. After Cedrick Williams tipped in a miss to cut TU's lead to 35-32, the Tigers scored the final seven points of the half and Hairston's fadeway jumper with one second left in the first half gave the Tigers a working margin at halftime.
The Seahawks return home to open a two-game homestand Wednesday against Georgia State at 7 p.m. The Tigers entertain Delaware on Wednesday at 7 p.m.
GAME NOTES: UNCW still owns a 16-12 advantage in the series…The Seahawks finished 6-8 in 14 games at the Towson Center…Keith Rendleman made his eighth and final appearance against the Tigers…The Seahawks are 5-20 on the road under Buzz Peterson…UNCW used its 10th different starting lineup of the season…The Seahawks are 0-2 in February and 7-5 this month all-time against the Tigers…Neither team will be playing in the CAA Tournament due to APR issues…There were seven dunks in the contest, including three consecutive by UNCW to end the game…Reybold scored first for the Seahawks for the third straight game…The Seahawks had just nine turnovers…Towson outrebounded UNCW, 39-36…Marcus Graham had two points and three rebounds in the last three minutes of the contest…It was the third time this season the Seahawks had allowed more than 80 points.

subset: commoncrawl


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Staff at a Keighley school will embark on an exercise bike challenge in aid of a trainee PE teacher who was paralysed after a holiday in Greece.
Callum Hall, 20, was poisoned last year after he stepped on a sea urchin. The venom created an abscess on his spine, which burst and left him paralysed.
Staff and pupils at Holy Family Catholic School are now working together to try and raise money for Callum to aid his recovery and provide specialist equipment to help him walk again. Callum said he was hoping to compete in the 2016 Paralympics.
Jess Moran, head of year seven at Holy Family, said Callum lives in Yeadon but is known to staff at the Keighley school, who are part of the same church parish as Callum's family.
She added Holy Family headteacher Lawrence Bentley and deputy head Martin Hings had both taught Callum's siblings.
A Holy Family spokesman said: "Callum is in touch with the school and is grateful for any funds raised. If people would like to know more about his story, or want to sponsor him, they can visit his website at callumhalltrust.com or contact Jess Moran at Holy Family."
One of the fundraising activities to be undertaken by Mr Bentley and 17 other staff members will be a continuous exercise bike ride throughout the school day. This will start in assembly at 9am on March 15. Each member of staff will ride for 15 to 30 minutes on an exercise bike on the school stage, wearing T-shirts donated by MC Sports in Skipton.
Staff will collect sponsorship and will be supported by the students, especially at lunchtime when Mr Bentley and other senior managers will take to the bicycle stage.


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Beogradska 41
Property Directions
Hostel City Break is located in Belgrade center in the Beogradska street (Belgrade Street), near one of the biggest squares- Slavia square. It is only 900 meters from the city center, Knez Mihailova street (Prince Michael Street) and Trg republike (Republic Square), the main square in the city and the largest shopping area in Belgrade. From the bus and railway stations the easiest way to get to City Break Hostel is to catch tram number 2, 3 or 9. The first stop is at Slavia square from which there is only few minutes walking up the Beogradska street to building number 41. The hostel is situated on the top floor.
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Huifeng wrote:How does one measure that, to ask of gain or loss?
~~ Huifeng
We really should be more mindful and respectful of Indra and the Four Mahārāja as they guard the cosmos against evil forces.
To drop them to the wayside is just terrible. An indication of disrespect to the gods.


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TOPLAPapp and GRID from Oliver Chesler on Vimeo.
Here's a video I make of two terrific iPhone Apps that make random digital sounds. TOPLAPapp (iTunes link) and Grid (iTunes link) are must haves for any noise nerds out there.
"TOPLAPapp is a sonic puzzle based around a virtual machine for sound synthesis. This machine only accepts a few valid instructions, and you control it by placing each command letter within a grid, along with setting some associated parameter sliders. The machine runs through the grid, following the instructions to create the output sounds, which are usually of a somewhat noisy character, hopefully interestingly so. The historical antecedents include instruction synthesis as pionneered at the Institute of Sonology in the 1970s, and the live coding movement, of modifying a running program as it acts." – Nick Collins
For more info: Nick Collins and Kin


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RAM Trucks presents the Invitation-Only Fund Raiser 'Road to the RAM® JAM'
"Road to the Ram® Jam," is an all-star country music series featuring five of today's best country artists leading up to an invitation-only 2012 Ram Jam concert event December 29th in Nashville, Tenn., where all five artists will perform.
• BNA recording artist Kellie Pickler launched the series. She is offering fans a chance to win a free download of her latest single "Tough" when they enter to win a trip for two to the Ram Jam concert. Fans can check out behind-the-scenes video footage of Kellie in her Ram 1500 Laramie Longhorn as she discusses her love for trucks, her new album 100 Proof, and how she continues to show support for our troops with help from the USO. Kellie has chosen the USO (USO.org) and their services that support wounded, ill and injured warriors as the charity to receive proceeds from the auction of her Longhorn truck in 2013.
• The remaining four participating artists will be announced throughout the year. All five artists will receive a one year lease on a 2012 Ram 1500 Laramie Longhorn truck. The trucks will then be auctioned off in early 2013 with all proceeds going to benefit a military-focused charity of each artist's choice.


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How to fix Monday Night Bore?
by, 11-12-2012 at 09:09 PM (9484 Views)
The Monday Night Bore
This is my first blog ladies and gentleman so bare with me on this one!
If there is one thing we all have in common its our frustrations with the current state of the WWE and its abysmal 'product'. As i sit here watching Raw i seem unable to enjoy it because even the smallest details frustrate and annoy me. I just want to walk backstage and tell Vince how to run the show, and here are the things i would say:
Introduction to Raw- From the moment Raw's broadcast begins i find myself annoyed with the theme song. The theme songs to Raw have gradually degraded but this one takes the biscuit. the WWE has always stayed true to its rock and metal routes, its what its always been about, the music goes with the destruction and chaos that SHOULD explode on a monday night. The current theme song does not only apply to the main stream status-quo but it can not be related to professional wrestling whatsoever. WHOOPS did i say wrestling??
That terrible Recap section at the start of Raw that makes it look like a episodic soap drama also needs to go.
The Ropes!! - This one is simple the ropes NEED to be RED end of.
The Set- Sometimes less is more, and in the case of the set this really rings true. HD screens as far as the eye can see. Strobe lights, lasers, flashes and bangs. The Raw set we all once loved now acquires more lights and props than the Las Vegas strip. A show that was once about feuds and pure professional wrestling is now about how expensive they can make the set look. It really takes away from the matches when the set makes you think someone has slipped you some LSD.
John Cena- What can i say. Give us a break and take some time off. We all need less super Cena for a while.
The Divas Division- The womens wrestling in WWE took a turn for the worse once Trish and Lita left and there are no signs of improvement. How can the audience relate and get into the Divas when they have no airtime. Even when they do its just thrown together into two minute matches that have no storyline or meaning.
Where are the Monsters? - Everyone loves a monster. Someone who would come down to the ring and when you saw them coming down the ramp you knew shit was about to go down. Ryback is the closest thing nowadays to a monster but its hard to feel his character when his dialogue solely consists of his requirements for food. And for anyone who has kane on there mind as they read this section, his reputation as a monster was destroyed a long time ago.
Time To Step Up- After seeing Randy Orton, John Cena and the Big Show in the main event picture for so many years i have started to know them in the ring more than they know themselves. The key to wrestling is suprising the audience and leaving them on the edge of there seats. All these guys cause for me is a YAWN. Time for Damien Sandow, Dolph Ziggler and Codey Rhodes to move up the card.
I feel like i have let some of my anger out now its in writing and thank you for reading. But i dont want to end this Blog on a negative note. So do i have anything positive to say? Yes I do.
Thank you to CM Punk for carrying the WWE over the past year. He and Daniel Bryan are the only reason i have a tiny bit of Faith left in the WWE. As long as true Wrestlers like this stay alive in a WWE ring we are safe.
Please comment and tell me what you think about the current state of the WWE, positive or negative i would love to hear your opinions!
Until next time!
Brad Roach


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Joe Dunne insists he has no intention of changing the way Colchester United play - despite their winless start to the season.
The U's boss has described their hugely impressive display at Notts County as the best passing performance he has seen from his side in his time as manager.
Colchester were dominant for large periods of the game at Meadow Lane and registered 24 shots on goal, in what was an excellent all-round team display.
However, the U's came away empty-handed and have managed only a point from their opening three matches in League One.
That has led to criticism from some fans but Dunne is sticking by his beliefs - and is confident Colchester's first win is just around the corner.
Dunne said: "We won't change the way we play - I will not change what I believe in.
"We won't change our philosophy and the players shouldn't change it, either.
"With more positive performances like this, the results will come.
"The way we play is exactly how we played at Notts County and in our previous games this season.
"It was the best passing performance I've had as a manager.
"My team played with belief and movement and my players looked so natural with their game.
"We've been consistent with our performances so far but the problem is that we haven't got the results.
"But we have to keep believing and stick together as a group, because if we do that then we'll win more games."
For more on this story and all of the latest Colchester United news, including Duguid's Diary, see Thursday's Daily Gazette.


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Cytokine Gene Polymorphisms in Gastric Diseases
Recruitment status was: Recruiting
- Full Text View
- Tabular View
- No Study Results Posted
- Disclaimer
- How to Read a Study Record
|Gastric Ulcer Duodenal Ulcer Gastric Cancer
|Procedure: IL-1, TNF-alpha, Il-10
|Study Type:
|Study Design:
|Allocation: Randomized
Intervention Model: Single Group Assignment
Masking: None (Open Label)
Primary Purpose: Diagnostic
|Official Title:
|Effects of Pro-Inflammatory and Anti-Inflammatory Cytokine Gene Polymorphism on the Development of Gastric Cancer and Peptic Ulcer in Japanese
|Study Start Date:
|January 2000
H. pylori infection has close associations with the development of peptic ulcer diseases as well as gastric cancer, gastric adenoma, and gastric MALT lymphoma. The association of the host genetics with the susceptibility to various gastroduodenal disorders has been intensively investigated in the pathogenesis of peptic ulcer and gastric cancer by H. pylori infection .
In chronic active gastritis induced by H. pylori infection, activated neutrophils and mononuclear cells produce several pro-inflammatory and anti-inflammatory cytokines. In fact, levels of pro-inflammatory and anti-inflammatory cytokines are elevated in gastric mucosa infected with H. pylori.
Cytokine polymorphisms are associated with various inflammatory and autoimmune diseases. Recently, cytokine polymorphisms are considered to play an important role in the pathogenesis of peptic ulcer and gastric cancer. However, the roles of the IL-10 polymorphisms on the pathogenesis of H. pylori-related gastric cancer and peptic ulcer have not been fully elucidated.It is unclear whether pro-inflammatory and anti-inflammatory cytokines polymorphisms were associated with pathogenesis of peptic ulcer and gastric cancer in Japan. Then, we intended to clarify the association between polymorphisms of IL-10 and the susceptibility to gastric cancer, gastric ulcer and duodenal ulcer in Japan, and to detect the individuals who have higher risks for gastrointestinal disease development.
|Ages Eligible for Study:
|Child, Adult, Senior
|Sexes Eligible for Study:
|Accepts Healthy Volunteers:
Inclusion Criteria:
Exclusion Criteria:
- patinets without H. pylori
Please refer to this study by its ClinicalTrials.gov identifier: NCT00197470
|Contact: Mitsushige Sugimoto
|Hamamatsu University School of Medicine
|Hamamatsu, Japan, 431-3192
|Contact: Mitsushige Sugimoto +81-53-435-2261 email@example.com
|Study Chair:
|MItsushige Sugimoto
|First department of medicine, Hamamatsu University School of medicine
|ClinicalTrials.gov Identifier:
|NCT00197470 History of Changes
|Other Study ID Numbers:
|Study First Received:
|September 12, 2005
|Last Updated:
|September 12, 2005
Additional relevant MeSH terms:
Stomach Neoplasms
Stomach Ulcer
Duodenal Ulcer
Pathologic Processes
Gastrointestinal Neoplasms
Digestive System Neoplasms
Neoplasms by Site
Digestive System Diseases
Gastrointestinal Diseases
Stomach Diseases
Peptic Ulcer
Duodenal Diseases
Intestinal Diseases
ClinicalTrials.gov processed this record on September 21, 2017


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Yves Chabal
University of Texas, Dallas
"For the individual development and collaborative application of fundamental surface infrared spectroscopy and quantum chemical methods to silicon surface reactions important in microelectronics."Background:
Yves Jean Chabal, currently holds the Texas Instrument Distinguished Chair in Nanoelectronics at the University of Texas at Dallas, where he is a Professor and Chair of Materials Science and Engineering with a joint position in Physics. He obtained his B.A. in Physics from Princeton University in 1974 and his Ph.D. in Physics at Cornell University in 1980, under the guidance of A.J. Sievers. He then spent 22 years at Bell Laboratories, where he studied fundamental processes relevant to the microelectronics industry, such as semiconductor surface passivation by gas phase and wet chemical methods, wafer bonding, and molecular hydrogen in amorphous silicon, and developed with Krishnan Raghavachari a mechanistic understanding of hydrogen passivation and oxidation of silicon surfaces. He was then a faculty member at Rutgers University in the departments of Chemistry, Biomedical Engineering and Physics and the Director of the Laboratory for Surface Modification, where he developed in-situ methods to study Atomic Layer Deposition and chemical functionalization of silicon surfaces. He joined UT Dallas in 2008 and works on hydrogen storage, and nanoelectronics. He is a Fellow of the American Physical Society and the AVS, and a member of the ACS and MRS Societies. He received an IBM Faculty Award in 2004 and the Rutgers Board of Trustees Award for Excellence in Research in 2006. He is currently on the MRS Board of Directors and the Program Chair of the Surface Science Division of the AVS.
Selection Committee:
Ellen Williams, Manuel Cardona, Franz Himpsel, Jacquwline Krim, Barbara Garrison


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He's not your everyday rapper. He's so much more. Lonnie Rashid Lynn Jr. aka Common brings his uncommon rap style to Southern California this week performing at the Wiltern theatre on Thursday, Sept 6 and in Anaheim on Saturday, Sept 8 at the House of Blues.
On the heels of his hot-selling CD, "Finding Forever," which debuted No.1 on the Billboard top 200 charts and has maintained a strong top five position, the Chicago-born rapper is finally getting the love and respect that's long overdue. His single, "For the People" seems to be the song packing the most heat.
Common has launched a series of promotional appearances for what his friend and co producer Kanye West proclaims is "the best rap album of the year" – that's saying a lot considering Kanye's new CD streets next week.
Common explains the title "Finding Forever" represents his quest to find an eternal place in hip-hop and also his wishes to be an artist for the rest of his life. Best known for songs that focus on spirituality, poverty and other issues involving social awareness, he has clearly separated himself from the rest of the pack.
Success did not come quickly for him. It has taken 13 years and six CDs before he could ride the top of the music charts. 2007 appears to be the beginning of what will be a defining year for the soulful rapper who clearly stated months ago he craves success, but only to a certain point. "I'd be lying if I said I don't want to sell millions and become a household name, but I won't lose myself to do it," said Common.
Next up for the soft spoken rapper and star of those famous "Gap" commercials is another movie – this time he's co-starring with none other than Denzel Washington and Russell Crowe in "American Gangster," Ridley Scott's film about New York City cops who take on a Harlem drug kingpin.
If you missed him a few weeks ago, rapping and playing around lyrically with Stevie Wonder on KJLH's morning show, you don't want to miss him live in his only Los Angeles appearance this week. He's one uncommon rapper and he's worth the $88. Showtime set for 9pm at Wiltern, Thursday Sept 6 and at the House of Blues in Anaheim on Saturday, September 8 at 6pm.


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Topic: gem ox
Hi all I had my second treatment of gemox today. My first one went well. This time I felt some neuropathy in my hands and feet almost instantly while leaving the infusion room. Is it common for some to get hit that quickly? I would imagine it will get a little worse with each treatment but those who have been on it or have watched someone I am curious to hear your thoughts. It occurred almost immediately after I was done with I fusion. It was also sprinkling and while walking outside wow my hands feet and face started tingling and stinging. Hugs to all.


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'"Your initial reaction is that you're upset just because you lose," Sabres coach Lindy Ruff said. "You don't really lose anything by losing, but getting scored against with 3.9 seconds left makes you want to put your fist through the wall. You look at the home and home, and we got two out of four (points) against a team that's really been going good."'
You are here: Home / SabresBeat / Fluke goal is costly as Sabres lose in OT


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You are here:
Related Information
- What Is Epidermolysis Bullosa? (fast facts, easy-to-read)
- ¿Qué es la epidermólisis ampollosa? (Esenciales: hojas informativas de fácil lectura)
- Order a NIAMS publication to be mailed
Health Information
Epidermolysis Bullosa
November 2016
Questions and Answers about Epidermolysis Bullosa
This publication describes epidermolysis bullosa (EB) and its symptoms and contains information about diagnosis and treatment, as well as research efforts on epidermolysis bullosa. It also discusses issues such as skin care and quality of life for people with epidermolysis bullosa. If you have questions after reading this publication, you may wish to discuss them with your doctor or a dermatologist (a specialist in treating skin conditions).
- What is epidermolysis bullosa?
- Who gets epidermolysis bullosa?
- What causes epidermolysis bullosa?
- How is epidermolysis bullosa diagnosed?
- What are the symptoms of epidermolysis bullosa?
- How is epidermolysis bullosa treated?
- What is the value of genetic counseling?
- What research is being conducted on epidermolysis bullosa?
- Where can people find more information about epidermolysis bullosa?
What is epidermolysis bullosa?
Epidermolysis bullosa is a group of blistering skin conditions. The skin is so fragile in people with epidermolysis bullosa that even minor rubbing may cause blistering. At times, the person with epidermolysis bullosa may not be aware of rubbing or injuring the skin even though blisters develop. In severe epidermolysis bullosa, blisters are not confined to the outer skin. They may develop inside the body, in such places as the linings of the mouth, esophagus, stomach, intestines, upper airway, bladder, and genitals.
The skin has an outer layer called the epidermis and an underlying layer called the dermis. The place where the two layers meet is called the basement membrane zone (see illustration Skin Structure). The main forms of epidermolysis bullosa are epidermolysis bullosa simplex (EBS), junctional epidermolysis bullosa (JEB), and dystrophic epidermolysis bullosa (DEB). Epidermolysis bullosa simplex occurs in the outer layer of skin; junctional epidermolysis bullosa and dystrophic epidermolysis bullosa occur in the basement membrane zone. These major types of epidermolysis bullosa, which will be described throughout this text, also have many subtypes.
Who gets epidermolysis bullosa?
Epidermolysis bullosa occurs in all racial and ethnic groups and affects males and females equally. The disease is not always evident at birth. Milder cases of epidermolysis bullosa may become apparent when a child crawls, walks, or runs, or when a young adult engages in vigorous physical activity.
What causes epidermolysis bullosa?
Most people with epidermolysis bullosa have inherited the condition through faulty genes they receive from one or both parents. Genes are located in the bodys cells and determine inherited traits passed from parent to child. They also govern every body function, such as the formation of proteins in the skin. Several genes are known to underlie the different forms of epidermolysis bullosa. Genes are located on chromosomes, which are structures in each cells nucleus.
In an autosomal dominant form of epidermolysis bullosa, the disease gene is inherited from only one parent who has the disease, and there is a 50-percent chance (1 in 2) with each pregnancy that a baby will have epidermolysis bullosa. In the autosomal recessive form, the disease gene is inherited from both parents.
Neither parent has to show signs of the disease; they simply need to "carry" the gene, and there is a 25-percent chance (1 in 4) with each pregnancy that a baby will have epidermolysis bullosa. Epidermolysis bullosa can also be acquired through a mutation (abnormal change) in a gene that occurred during the formation of the egg or sperm reproductive cell in a parent. Neither the sex of the child nor the order of birth determines which child or how many children will develop epidermolysis bullosa in a family that has the faulty gene.
Although epidermolysis bullosa simplex can occur when there is no evidence of the disease in the parents, it is usually inherited as an autosomal dominant disease. In epidermolysis bullosa simplex, the faulty genes are those that provide instructions for producing keratin, a fibrous protein in the top layer of skin. As a result, the skin splits in the epidermis, producing a blister.
In junctional epidermolysis bullosa, there is a defect in the genes inherited from both parents (autosomal recessive) that normally promote the formation of anchoring filaments (thread-like fibers) or hemidesmosomes (hem-ee-DES-mo-soms) (complex structures composed of many proteins). These structures anchor the epidermis to the underlying basement membrane. The defect leads to tissue separation and blistering in the upper part of the basement membrane.
There are both dominant and recessive forms of dystrophic epidermolysis bullosa. In this condition, the filaments that anchor the epidermis to the underlying dermis are either absent or do not function. This is caused by defects in the gene for type VII collagen, a fibrous protein that is the main component of the anchoring filaments.
Epidermolysis bullosa acquisita (EBA) is a rare autoimmune disorder in which the body attacks its own anchoring fibrils with antibodies, the special proteins that help fight and destroy foreign substances that invade the body. In a few cases, it has occurred following drug therapy for another condition; in most cases, the cause is unknown.
How is epidermolysis bullosa diagnosed?
Dermatologists can identify where the skin is separating to form blisters and what kind of epidermolysis bullosa a person has by doing a skin biopsy (taking a small sample of skin that is examined under a microscope). One diagnostic test involves use of a microscope and reflected light to see if proteins needed for forming connecting fibrils, filaments, or hemidesmosomes are missing or reduced in number. Another test involves use of a high-power electron microscope, which can greatly magnify tissue images, to identify structural defects in the skin.
Diagnostic techniques make it possible to identify defective genes in epidermolysis bullosa patients and their family members. Prenatal diagnosis can now be accomplished by amniocentesis (removing and examining a small amount of amniotic fluid surrounding the fetus in the womb of a pregnant woman) or sampling the chorionic villus (part of the outer membrane surrounding the fetus) as early as the 10th week of pregnancy.
What are the symptoms of epidermolysis bullosa?
The major sign of all forms of epidermolysis bullosa is fragile skin that blisters, which can lead to serious complications. For example, blistering areas may become infected, and blisters in the mouth or parts of the gastrointestinal tract may interfere with proper nutrition.
Following is a summary of some of the characteristic signs of various forms of epidermolysis bullosa.
Epidermolysis Bullosa Simplex
A generalized form of epidermolysis bullosa simplex usually begins with blistering that is evident at birth or shortly afterward. In a localized, mild form called Weber-Cockayne, blisters rarely extend beyond the feet and hands. In some subtypes of epidermolysis bullosa simplex, the blisters occur over widespread areas of the body. Other signs may include thickened skin on the palms of the hands and soles of the feet; rough, thickened, or absent fingernails or toenails; and blistering of the soft tissues inside the mouth. Less common signs include growth retardation; blisters in the esophagus; anemia (a reduction in the red blood cells that carry oxygen to all parts of the body); scarring of the skin; and milia, which are small white skin cysts.
Junctional Epidermolysis Bullosa
This disease is usually severe. In the most serious forms, large, ulcerated blisters on the face, trunk, and legs can be life-threatening because of complicated infections and loss of body fluid that leads to severe dehydration. Survival is also threatened by blisters that affect the esophagus, upper airway, stomach, intestines, and urogenital system. Other signs found in both severe and mild forms of junctional epidermolysis bullosa include rough and thickened or absent fingernails and toenails; a thin appearance to the skin (called atrophic scarring); blisters on the scalp or loss of hair with scarring (scarring alopecia); malnutrition and anemia; growth retardation; involvement of soft tissue inside the mouth and nose; and poorly formed tooth enamel.
Dystrophic Epidermolysis Bullosa
The dominant and recessive inherited forms of dystrophic epidermolysis bullosa have slightly different symptoms. In some dominant and mild recessive forms, blisters may appear only on the hands, feet, elbows, and knees; nails usually are shaped differently; milia may appear on the skin of the trunk and limbs; and there may be involvement of the soft tissues, especially the esophagus. The more severe recessive form is characterized by blisters over large body surfaces, loss of nails or rough or thick nails, atrophic scarring, milia, itching, anemia, and growth retardation. Severe forms of recessive dystrophic epidermolysis bullosa also may lead to severe eye inflammation with erosion of the cornea (clear covering over the front of the eye), early loss of teeth because of tooth decay, and blistering and scarring inside the mouth and gastrointestinal tract. In most people with this form of epidermolysis bullosa, some or all the fingers or toes may fuse (pseudosyndactyly). Also, individuals with recessive dystrophic epidermolysis bullosa have a high risk of developing a form of skin cancer called squamous cell carcinoma. It primarily occurs on the hands and feet. The cancer may begin as early as the teenage years. It tends to grow and spread faster in people with epidermolysis bullosa than in those without the disease.
How Is epidermolysis bullosa treated?
People with mild forms of epidermolysis bullosa may not require extensive treatment. However, they should attempt to keep blisters from forming and prevent infection when blisters occur. Individuals with moderate and severe forms may have many complications and require psychological support along with attention to the care and protection of the skin and soft tissues. Patients, parents, or other care providers should not feel that they must tackle all the complicated aspects of epidermolysis bullosa care alone. There are doctors, nurses, social workers, clergy members, psychologists, dietitians, and patient and parent support groups that can assist with care and provide information and emotional support.
Preventing Blisters
In many forms of epidermolysis bullosa, blisters will form with the slightest pressure or friction. This may make parents hesitant to pick up and cuddle young babies. However, a baby needs to feel a gentle human touch and affection. Parents can learn the best ways to handle babies to minimize the chances of blisters.
A number of things can be done to protect the skin from injury. These include:
- Avoiding overheating by keeping rooms at an even temperature.
- Applying lubricants to the skin to reduce friction and keep the skin moist.
- Using simple, soft clothing that requires minimal handling when dressing a child.
- Using sheepskin on car seats and other hard surfaces.
- Wearing mittens at bedtime to help prevent scratching.
Caring for Blistered Skin
When blisters appear, the objectives of care are to reduce pain or discomfort, prevent excessive loss of body fluid, promote healing, and prevent infection.
The doctor may prescribe a mild analgesic to prevent discomfort during changes of dressings (bandages). Dressings that are sticking to the skin may be removed by soaking them off in warm water. Although daily cleansing may include a bath with mild soaps, it may be more comfortable to bathe in stages where small areas are cleaned at a time.
Blisters can become quite large and create a large wound when they break. Therefore, a medical professional will likely provide instructions on how to safely break a blister in its early stages while still leaving the top skin intact to cover the underlying reddened area. After opening and draining, the doctor may suggest that an antibiotic ointment be applied to the area of the blister before covering it with a sterile, nonsticking bandage. To prevent irritation of the skin from tape, a bandage can be secured with a strip of gauze that is tied around it. In milder cases of epidermolysis bullosa or where areas are difficult to keep covered, the doctor may recommend leaving a punctured blister open to the air.
A moderately moist environment promotes healing, but heavy drainage from blister areas may further irritate the skin, and an absorbent or foam dressing may be needed. There are also contact layer dressings where a mesh layer through which drainage can pass is placed on the wound and is topped by an outer absorbent layer. The doctor or other health care professional may recommend gauze or bandages that are soaked with petroleum jelly, glycerin, or lubricating substances, or may suggest more extensive wound care bandages or products.
Treating Infection
The chances of skin infection can be reduced by good nutrition, which builds the body's defenses and promotes healing, and by careful skin care with clean hands and use of sterile materials. For added protection, the doctor may recommend antibiotic ointments and soaks.
Even in the presence of good care, it is possible for infection to develop. Signs of infection are redness and heat around an open area of skin, pus or a yellow drainage, excessive crusting on the wound surface, a red line or streak under the skin that spreads away from the blistered area, a wound that does not heal, and/or fever or chills. The doctor may prescribe a specific soaking solution, an antibiotic ointment, or an oral antibiotic to reduce the growth of bacteria. Wounds that are not healing may be treated by a special wound covering or biologically developed skin.
Treating Nutritional Problems
Blisters that form in the mouth and esophagus in some people with epidermolysis bullosa are likely to cause difficulty in chewing and swallowing food and drinks. If breast or bottle feeding results in blisters, infants may be fed using a preemie nipple (a soft nipple with large holes), a cleft palate nipple, an eyedropper, or a syringe. When the baby is old enough to take in food, adding extra liquid to pureed (finely mashed) food makes it easier to swallow. Soups, milk drinks, mashed potatoes, custards, and puddings can be given to young children. However, food should never be served too hot.
Dietitians are important members of the health care team that assists people with epidermolysis bullosa. They can work with family members and older patients to find recipes and prepare food that is nutritious and easy to consume. For example, they can identify high-caloric and protein-fortified foods and beverages that help replace protein lost in the fluid from draining blisters. They can suggest vitamin and mineral nutritional supplements that may be needed, and show how to mix these into the food and drinks of young children. Dietitians can also recommend adjustments in the diet to prevent gastrointestinal problems, such as constipation, diarrhea, or painful elimination.
Surgical Treatment
Surgical treatment may be necessary in some forms of epidermolysis bullosa. Individuals with the severe forms of autosomal recessive dystrophic epidermolysis bullosa whose esophagus has been narrowed by scarring may require dilation of their esophagus for food to travel from the mouth to the stomach. Other individuals who are not getting proper nutrition may need a feeding tube that permits delivery of food directly to the stomach. Also, patients whose fingers or toes are fused together may require surgery to release them.
What is the value of genetic counseling?
Epidermolysis bullosa is a difficult, sometimes painful, and often disfiguring disease. Most adults with signs of epidermolysis bullosa or who know they carry the gene would like to spare future generations, including their own potential offspring, from this condition. With the knowledge of specific gene mutations that cause epidermolysis bullosa, it is now possible to determine the specific gene mutation in the family and then to conduct prenatal tests on pregnant women with a fetus at risk of epidermolysis bullosa to determine the status of the fetus. A genetic counselor can test for gene mutations, provide information on the likelihood of passing the gene for epidermolysis bullosa to children, and provide advice on future childbearing. Genetic counseling can be a crucial step in helping families make decisions about their family planning.
What research is being conducted on epidermolysis bullosa?
At one time, research on epidermolysis bullosa was limited to describing the disease and understanding what happens in the layers of skin. Today, research focuses on finding gene mutations and their effect on the tissues, copying genes, reproducing gene mutations for research to correct them, inserting healthy genes to replace missing or mutated genes, and screening those who may have a gene mutation causing epidermolysis bullosa.
Scientists are attempting to transplant stem cells into the skin of mice affected by epidermolysis bullosa to see if such therapy can restore the basement membrane and reduce the fragility of the skin. And the possibility of using the stem cells extracted from a person affected by epidermolysis bullosa and transplanting them back into the host is being explored, perhaps setting the stage for future adult stem cell therapies.
Applying newer diagnostic techniques, investigators are beginning to link specific gene defects with the protein problems they produce. Now that the epidermolysis bullosa-causing gene mutations can be identified, doctors can identify ova (eggs) that do not contain an abnormal gene. These ova can be selected for in vitro fertilization, thus improving the chances of having healthy children in families with the epidermolysis bullosa gene.
Research on skin biology continues; researchers are looking at how skin cells form and which molecules regulate gene expression. Understanding how healthy skin develops will help researchers discover if and when development goes wrong and causes diseases.
More information on research is available from the following websites:
- National Institutes of Health (NIH) Clinical Research Trials and You was designed to help people learn more about clinical trials, why they matter, and how to participate. Visitors to the website will find information about the basics of participating in a clinical trial, first-hand stories from clinical trial volunteers, explanations from researchers, and links on how to search for a trial or enroll in a research-matching program.
- ClinicalTrials.gov offers up-to-date information for locating federally and privately supported clinical trials for a wide range of diseases and conditions.
- NIH RePORTER is an electronic tool that allows users to search a repository of both intramural and extramural NIH-funded research projects from the past 25 years and access publications (since 1985) and patents resulting from NIH funding.
- PubMed is a free service of the U.S. National Library of Medicine that lets you search millions of journal citations and abstracts in the fields of medicine, nursing, dentistry, veterinary medicine, the health care system, and preclinical sciences.
Where can people find more information about epidermolysis bullosa?
National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)
Information Clearinghouse
National Institutes of Health
1 AMS Circle
Bethesda, MD 20892-3675
Phone: 301-495-4484
Toll free: 877-22-NIAMS (877-226-4267)
Fax: 301-718-6366
Email: firstname.lastname@example.com
Website: https://www.niams.nih.gov
If you need more information about available resources in your language or another language, please visit our website or contact the NIAMS Information Clearinghouse at firstname.lastname@example.com.
Other Resources
Dystrophic Epidermolysis Bullosa Research Association of America, Inc.
Website: http://www.debra.org
National Society of Genetic Counselors
Website: http://www.nsgc.org
The NIAMS gratefully acknowledges the assistance of the following individuals in the preparation and review of the original version of this publication: Jo-David Fine, M.D., M.P.H., National Epidermolysis Bullosa Registry, Lexington, KY; Martin I. Hassner, DebRA, New York, NY; Alan N. Moshell, M.D., NIAMS/NIH; Amy S. Paller, M.D., Children's Memorial Hospital, Chicago, IL; Juoni J. Uitto, M.D., Ph.D., Thomas Jefferson University, Philadelphia, PA; and David T. Woodley, M.D., Los Angeles County/University of Southern California Medical Center, Los Angeles, CA.
The mission of the National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), a part of the U.S. Department of Health and Human Services' National Institutes of Health (NIH), is to support research into the causes, treatment, and prevention of arthritis and musculoskeletal and skin diseases; the training of basic and clinical scientists to carry out this research; and the dissemination of information on research progress in these diseases. The NIAMS Information Clearinghouse is a public service sponsored by the NIAMS that provides health information and information sources. Additional information can be found on the NIAMS website at https://www.niams.nih.gov.
For your information
This publication contains information about medications used to treat the health condition discussed here. When this publication was developed, we included the most up-to-date (accurate) information available. Occasionally, new information on medication is released.
For updates and for any questions about any medications you are taking, please contact
U.S. Food and Drug Administration
Toll free: 888-INFO-FDA (888-463-6332)
Website: https://www.fda.gov
For additional information on specific medications, visit Drugs@FDA at https://www.accessdata.fda.gov/scripts/cder/daf/. Drugs@FDA is a searchable catalog of FDA-approved drug products.
For updates and questions about statistics, please contact
Centers for Disease Control and Prevention, National Center for Health Statistics
Toll free: 800-232-4636
Website: https://www.cdc.gov/nchs
This publication is not copyrighted. Readers are encouraged to duplicate and distribute as many copies as needed.
Additional copies of this publication are available from:
National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)
Information Clearinghouse
National Institutes of Health
1 AMS Circle
Bethesda, MD 20892-3675
Phone: 301-495-4484
Toll free: 877-22-NIAMS (877-226-4267)
Fax: 301-718-6366
Email: email@example.com
Website: https://www.niams.nih.gov
NIH Publication No. 16–7038


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Duck women lose to Illinois, fall to 0-8
CHAMPAIGN, Ill. -- The University of Oregon women's basketball team lost 80-62 to Illinois on Wednesday night at Assembly Hall in Champaign. The Illini improved to 6-4 overall with the win, while UO dropped to 0-8 on the year.
Jillian Alleyne registered game-highs with 24 points and 16 rebounds for the Ducks. Alleyne, a freshman from Fontana, Calif., made 11-of-15 from the field in the loss. Jordan Loera scored 11 points for UO, the only other Duck to tally double-figures for points.
The Ducks' defense limited Illinois to 34.7 percent (25-of-72) on field goal shooting and 38.1 percent (8-of-21) from behind the 3-point arc. However, Oregon made just 32.9 percent of its field goal attempts (25-of-76), while hitting only 6-of-29 on 3-pointers (20.7 percent).
The Illini had a large advantage at the free throw line (22-of-35, 62.9 percent), shooting 26 more free throws than UO (6-of-9, 66.7 percent).
Illinois had five players score in double-figures, led by Karisma Penn who posted a double-double with 17 points and 12 rebounds.
Oregon once again got off to a good start with the Ducks up 5-0 after an inside basket from Alleyne and a 3-pointer from Loera, both field goals assisted by Danielle Love. Oregon held a six-point lead, 9-3, at the 17:12 mark in the first half.
But the Illini came out of the first media timeout and took control of the game, heating up from behind the 3-point arc to take a 23-13 advantage with eight minutes in the half.
Illinois, which shot 33.3 percent on field goals (12-of-36) in the first half, took a 36-20 advantage into the halftime locker room. Alleyne had 10 points and 11 rebounds to lead the Ducks over the first 20 minutes. UO was just 9-of-40 from the field and limited to 2-of-16 from behind the 3-point arc.
The teams played early part of the second half even, with UO cutting the lead to nine points, 50-41, with 11:51 in the game. But Illinois regained momentum of the contest, stretching the lead to 18 points, 62-44, with eight minutes on the clock. The Illini then took a 76-56 advantage with 3:01 in the game, their largest lead of the game.
Oregon (0-8) will return home to Eugene for a game this Sunday against Fresno State. Tip-time is set for 1 p.m. (PT) at Matthew Knight Arena.


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I thought this might be a good place to condense all the rumors, speculation, and new mock drafts that will come out today so we can try and ease the load on PD and not crash. Obviously, it would make sense for confirmed deals to get their own thread, but this way rumors can be kept ino one place instead of taking over the board as it seems we will have quite a few today with all the uncertainty around this draft.
I'll start it up with Chard Ford's mock draft 7.0
He has us taking Lawson and he ranks his certainty at 60%.


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SANTA CLARA -- The Arizona Cardinals' slumping offense look like a pass rusher's dream. Their quarterbacks -- yes, plural -- have been sacked 29 times in the past four games.
Talk about a golden opportunity for the 49ers to increase their sack total in Monday night's NFC West encounter in Arizona.
"It means we have a good chance of getting to the quarterback," said Aldon Smith, whose 51/2 sacks account for half of the 49ers' total.
That quarterback is expected to be John Skelton, who hurt his ankle in the season opener and didn't return until two weeks ago, once Kevin Kolb sustained rib and chest injuries.
The Cardinals' offensive line is a patchwork unit because of injuries, and they've had an open-door policy when it comes to pass rushers the past month.
They overcame eight sacks in a Sept. 30 win over the Miami Dolphins that extended their undefeated start to 4-0. Then came a three-game losing streak, and along with it were nine sacks against the St. Louis Rams, five against the Buffalo Bills and seven against the Minnesota Vikings.
"That's good then," 49ers linebacker Ahmad Brooks said. "It puts more pressure on their offensive line from stopping us, and when that's the case, we smell blood."
Defensive coordinator Vic Fangio has maintained for weeks there's nothing wrong with the 49ers' pass rush and that sack totals are misleading. They've tallied 11 sacks in seven games, which is tied for the league's eighth fewest.
Backing up Fangio's claim: the 49ers are allowing the league's fewest yards per game (272.3) and fewest passing yards (173.4), along with the second-fewest points (14.3; the Chicago Bears are first at 13.0 points per game).
The Cardinals' defense counters with the league's fourth-best averages in terms of scoring (16.9) and passing yards (191.3). They haven't allowed more than 21 points in any game, and that's offset their offense's deficiencies after a 4-0 start that caught many off guard.
"I was surprised," Brooks said, "because I was like, 'Dang, we've got competition.'?"
The 49ers needed only to look back to their last meeting with the Cardinals to confirm that statement. Unable to hold a 19-7 lead, the 49ers tallied only two sacks in a 21-19 loss at Arizona on Dec. 11.
It's the Cardinals who are fearing mistakes heading into this rematch, especially coach Ken Whisenhunt.
Asked if the Cardinals offensive line has shown any signs of progress, Whisenhunt somehow stayed composed to offer a realistic review.
"It's hard when you give up that many sacks," Whisenhunt said. "We play better for a little while and then have breakdowns."
The Cardinals tried addressing their line in free agency by signing former 49ers lineman Adam Snyder, who's lined up at right guard and is questionable for Monday's game because of a leg injury.
With offensive tackles Levi Brown and Jeremy Bridges on injured reserve, the Cardinals are on pace to give up 80 sacks this season. Only the 1986 Philadelphia Eagles allowed more sacks (104) since the league began recognizing that stat in 1982.
Offensive tackles D'Anthony Batiste and Bobby Massie have yielded 22 of the Cardinals' 35 sacks, according to ProFootballFocus.com.
The 49ers' defense helped spark the Cardinals' shuffling of quarterbacks last season. Kolb got knocked out early of their Dec. 11 game with a concussion, caused by Brooks' inadvertent knee to the back of Kolb's head as Justin Smith pulled down the Cardinals' higher-priced quarterback. Skelton entered and pulled off a surprising comeback, starting a trend.
Aldon Smith doesn't care whom he's sacking as long as it's contributing to the defense's dominance.
"Selfishly we'd all like more sacks," Smith said. "Teams are a little more prepared, and quarterbacks are getting rid of the ball faster. But we're still getting pressure and doing OK."
For more on the 49ers, see Cam Inman's Hot Read blog at blogs.mercurynews.com/49ers. Follow him on Twitter at twitter.com/CamInman.


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Some highlights worth noting from the Dole Food Co. 10-K report on fiscal year ending Jan. 1, 2011. I found interesting some specific details about the market share for Dole's banana and salad business.
Some notable details from the Dole report:
At January 1, 2011, we had approximately 37,600 full-time permanent employees and 36,700 full-time seasonal or temporary employees, worldwide. Dole is the world's largest producer and marketer of high-quality fresh fruit and fresh vegetables.
On October 28, 2009, Dole completed a $446 million initial public offering of its common stock and received net proceeds of $415 million.
For the fiscal year ended January 1, 2011, Dole generated revenues of approximately $6.9 billion and operating income of approximately $194 million. At January 1, 2011 we had total assets of $4.3 billion.
The DOLE brand was introduced in 1933 and is one of the most recognized brands for fresh and packaged produce in the United States, as evidenced by Dole's 68% unaided consumer brand awareness — more than twice that of Dole's nearest competitor, according to a major global research company (Millward Brown).
The global market for fresh fruit and vegetables is approximately $675 billion, with just over $100 billion in the U.S.
Market Share Leader. Our most significant products hold the number 1 or number 2 positions in their respective markets. We maintain number 1 market share positions in North American bananas, North American iceberg lettuce, celery, cauliflower, and packaged fruit products, including our FRUIT BOWLS, FRUIT BOWLS in Gel, Fruit Parfaits and fruit in plastic jars.
Valuable Asset Base. We are an asset rich company, which provides significant competitive advantages to our operations and value to our investors. In addition to the DOLE trademark, we have an impressive base of tangible assets. We own 117,000 acres of farms and other land holdings, including 25,400 acres of farmland in Oahu, Hawaii and approximately 2,600 acres of peach orchards in California. We have the largest dedicated refrigerated containerized fleet in the world, which, at year end, included 13,800 refrigerated containers, 11 owned and 17 chartered vessels. We intend to return six of the chartered vessels during 2011. We own and operate over 60 ripening and distribution centers in Europe and Asia. We own and operate over one million square feet of state-of-the-art vegetable processing facilities. Additionally, our packaged food business processes its product lines in over 1.9 million square feet of owned manufacturing facilities.
Diversity of Sourcing Locations. We currently source our fresh fruits and vegetables from over 20 countries and distribute products in more than 90 countries.
We will continue to shift our product mix toward value-added food categories while maintaining and building on our key market leadership positions in commodity fruits and vegetables. For example, we have successfully increased our percentage of revenue from value-added products in our fresh vegetables and packaged foods businesses, where our packaged salad lines and FRUIT BOWL and other non-canned products now account for approximately 53% and 55% of those businesses' respective revenues. Value-added food categories are growing at a faster rate than traditional commodity businesses and typically generate stronger margins. We plan to continue to address the growing demand for convenient and innovative products by investing in our higher margin, value-added food businesses.
Our fresh fruit business segment has four primary operating divisions: bananas, European ripening and distribution, fresh pineapples and Dole Chile. We believe that we are the industry leader in growing, sourcing, shipping and distributing consistently high-quality fresh fruit. The fresh fruit business segment represented approximately 68% of 2010 consolidated revenues.
We are one of the world's largest producers of bananas, growing and selling approximately 153 million boxes of bananas in 2010. We sell most of our bananas under the DOLE brand. We primarily sell bananas to customers in North America, Europe and Asia. We are the number 1 brand of bananas in both the U.S. (an approximate 34% market share) and Japan (an approximate 32% market share) and the number 2 provider in Europe (an approximate 9% market share). In Latin America, we source our bananas primarily in Honduras, Costa Rica, Ecuador, Colombia, Guatemala and Peru, growing on approximately 32,000 acres of company-owned farms and approximately 71,500 acres of independent producers' farms. We ship our Latin American bananas to North America and Europe in our refrigerated and containerized shipping fleet. In Asia, we source our bananas primarily in the Philippines. Bananas accounted for approximately 40% of our fresh fruit business segment revenues in 2010.
Consistent with our strategy to focus on value-added products, we have continued to expand our focus on higher margin, niche bananas. While the traditional "green" bananas still comprise the majority of our banana sales, we have successfully introduced niche bananas (e.g., organic). We have also improved the profitability of our banana business by focusing on profitable customer relationships and markets.
While bananas are sold year round, there is a seasonal aspect to the banana business. Banana prices and volumes are typically higher in the first and second calendar quarters before the increased competition from summer fruits.
Approximately 90% of our total retail volume in North America is sold under contract. The contracts are typically one year in duration and help to insulate us from fluctuations in the banana spot market. Our principal competitors in the international banana business are Chiquita Brands International, Inc. and Fresh Del Monte Produce, Inc.
Fresh Pineapples
We are the number 2 global marketer of fresh pineapples, growing and selling more than 32 million boxes in 2010. We source our pineapples primarily from Dole-operated farms and independent growers in Latin America, Hawaii, the Philippines and Thailand. We produce and sell several different varieties, including the sweet yellow pineapple. We introduced the sweet yellow pineapple in 1999, and now market a substantial portion of this fruit under the DOLE TROPICAL GOLD ® label. Varieties of pineapple other than the sweet pineapples are also used in our packaged products. Our primary competitor in fresh pineapples is Fresh Del Monte Produce Inc. Pineapples accounted for approximately 8% of our fresh fruit business segment's revenues in 2010.
Dole Chile
We began our Chilean operations in 1982 and we are the largest exporter of Chilean fruit. We export grapes, apples, pears, stone fruit (e.g., peaches and plums) and kiwifruit from approximately 600 primarily leased acres and 12,400 contracted acres. The weather and geographic features of Chile are similar to those of the Western United States, with opposite seasons. Accordingly, Chile's harvest is counter-seasonal to that in the northern hemisphere, offsetting the seasonality in our other non-tropical fresh fruit. We primarily export Chilean fruit to North America, Latin America and Europe. Our Dole Chile business division accounted for approximately 5% of our fresh fruit business segment's revenues in 2010.
Fresh Vegetables
Our fresh vegetables business segment produces and markets fresh-packed and value-added vegetables. We source fresh vegetables from Dole-owned, leased and contracted farms. Our value-added products are produced in state-of-the-art processing facilities in Yuma, Arizona, Soledad, California, Springfield, Ohio and Bessemer City, North Carolina. Under arrangements with independent growers, we purchase fresh produce at the time of harvest and are generally responsible for harvesting, packing and shipping the product to our central cooling and distribution facilities. In 2010, value-added products accounted for 53% of our revenues for this segment. The fresh vegetables business segment accounted for approximately 15% of 2010 consolidated revenues.
Fresh-packed Vegetables
We source, harvest, cool, distribute and market more than 20 different types of fresh and fresh-cut vegetables, including iceberg lettuce, red and green leaf lettuce, romaine lettuce, butter lettuce, celery, cauliflower, broccoli, carrots, brussels sprouts, green onions, asparagus, snow peas, artichokes and radishes as well as fresh strawberries and raspberries. Products are grown by independent farmers under seasonal contracts, with harvesting primarily provided by us. Many of our fresh-packed vegetables are packaged in the field reducing handling and increasing product quality. We sell our fresh-packed vegetables products primarily in North America, Asia and, to a lesser extent, Western Europe. Based on our estimates, we are the largest supplier of iceberg lettuce and celery, and the third largest producer of cauliflower and strawberries in the U.S. Our primary competitors in this category include: Tanimura & Antle, Duda Farm Fresh Foods, Ocean Mist Farms, the Nunes Company, Inc. and Driscoll Strawberry Associates, Inc.
Our value-added vegetable products include packaged salads and packaged fresh-cut vegetables. Our U.S. unit market share of the packaged salads category for DOLE branded product, as reported by IRI, was approximately 26% for the 2010 fiscal year. New product development continues to drive growth in this area. Packaged salads go through a three-step process: (i) vegetables are grown for us by farmers under seasonal contracts, (ii) vegetables and other ingredients are delivered to our plants where they are washed three times in chilled, chlorinated water, and packaged under strict cold-chain and HACCP (Hazard Analysis and Critical Control Points) standards, and (iii) salads are shipped to retailers' warehouses for delivery to stores. Our primary competitors in packaged salads include Chiquita Brands International, Inc. (which markets Fresh Express), Ready Pac Produce, Inc. and Taylor Fresh Foods, Inc.
Our top 10 customers in 2010 accounted for approximately 31% of total revenues. No one customer accounted for more than 7% of total 2010 revenues. Our customer base is highly diversified, both geographically and in terms of product mix. Each of our segments' largest customers accounted for no more than approximately 20% of that segment's revenues. Our largest customers are leading global and regional mass merchandisers and supermarkets in North America, Europe and Asia.
Dole is undertaking strong measures to improve food safety. We spearheaded the industry-wide Leafy Greens Marketing Agreements in California and in Arizona. We developed and adopted enhanced Good Agricultural Practices, which include raw material testing in the fields, expanded buffer zones and increased water testing. We also use radio-frequency identification (RFID) tags to track leafy greens as they move from fields to trucks and through processing.
Dole's annual report: Market share and other tidbits
Some highlights worth noting from the Dole Food Co. 10-K report on fiscal year ending Jan. 1, 2011. I found interesting some specific details about the market share for Dole's banana and salad business.


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Woman charged over death of Ponthir couple
9:59am Thursday 3rd July 2014
9:59am Thursday 3rd July 2014
A WOMAN from Caerwent appeared at magistrates court yesterday charged with causing the death of an elderly couple through dangerous driving.
Taylor McDonnell, 20, of Carrow Road, appeared before Cwmbran Magistrates Court charged with two counts of causing death by dangerous driving.
It is alleged McDonell caused the death of Joyce and Denis Drew on November 13 while driving a Toyota Aygo on Caerleon Road in Ponthir.
The case was sent to crown court by magistrates and is scheduled to appear at Newport Crown Court on July 17.
McDonnell was released on unconditional bail.
Mrs Drew, 86, died shortly after the incident and Mr Drew, also 86, had been in hospital but died on November 19.
The Argus previously reported how Mr Drew, a former headteacher who also completed national service with the RAF, was born in Abersycan and had met his wife on a blind date while in the West Midlands.
The couple married on March 31, 1959.
Mr Drew had teaching jobs in Abersychan and Griffithstown and was headteacher at Usk CV, Llanyrafon and Hollybush Primary schools before he retired.
Mrs Drew worked in administration and at a travel agency before working as a registrar of births, deaths and marriages in Pontypool.
She was also a stalwart member of Ponthir WI, a driver for WRVS meals on wheels and raised money for St David's Foundation Hospice Care.
Last year, speaking to The South Wales Argus, the couple's daughters Sarah England and Helen Adam, said that they felt "privileged to have had them as parents" and were "very proud of them".
Further tributes were also made to the couple from friends.
Glyn Parry, 86, of Sebastopol, had known Mr Drew for more than 80 years.
He said: "They were a devoted and loving couple. He was a very good headteacher and teacher."
Reverend Kevin Lake, vicar of Caerleon and Llanfrechfa, said: "They will be truly missed but we have wonderful and cherished memories of them."
© Copyright 2001-2017 Newsquest Media Group


text:
Singapore to Sydney
January 4-February 10
By Kimberly Garza
Ethan Rohloff/Destination New South Wales
Seven nights aboard SeaDream Yacht Club's SeaDream II
Round trip from Singapore
Your round-the-world journey begins with a weeklong SeaDream cruise of Southeast Asian nations Singapore, Malaysia and Thailand. Climb aboard the SeaDream II, built to accommodate just 112 passengers on intimate, luxurious jaunts. Here, you can indulge in a session with a Thai massage therapist, dine in elegance in two restaurants or catch some sun and waves at the marina. Extending from the ship's stern, the large, retractable marina water-sports hub features glass-bottom kayaks, stand-up paddleboards, snorkeling gear and more. Count on more luxe touches in the staterooms, such as Belgian linens, Turkish cotton bathrobes and marble bathrooms.
The ocean adventure starts in Singapore, a tiny island (about 274 square miles) with big personality -- the cityscape claims everything from modern high-rises to verdant gardens, Victorian houses to cosmopolitan centers. Hit up high-end shops and boutiques on the 1.3-mile Orchard Road, a popular tourist destination, or pop into an eatery. Thanks to Singapore's rich multicultural heritage -- a blend of Chinese, Malay, Indian and more -- a plethora of cuisines is available.
With Singapore under your belt, you'll head next to Pulau Pangkor, Malaysia. Here, a variety of excursions is at your choosing; opt for a guided tour of a local rubber plantation and a sea turtle sanctuary, or perhaps spend the day teeing off at the Damai Laut Golf & Country Club. After that, it's three days of cruising Thailand, stopping to explore the sugar-white sand and coral reefs of Ko Adang, the resort hub Phuket and the rugged, mountainous island Ko Tarutao, where animals like flying lemurs and monitor lizards frolic. Circle back to Malaysia for two full days; this time around, admire the golden beaches and stunning bridge of Penang -- perhaps stopping at the Snake Temple -- and the colonial melting pot of Malacca before returning to Singapore.
***Fly to Cairns, Australia***
"Port Douglas & the Great Barrier Reef" with Trafalgar
Round trip from Cairns
The trip: Get to know our planet's largest reef system on a four-day jaunt along the coast of Australia with Trafalgar.
The gist: Start in the laid-back seaside village Port Douglas, which offers such sights as the pristine Four Mile Beach, shops, galleries and eateries. The next day, you'll head out to the legendary Great Barrier Reef, one of the world's most diverse ecosystems. Gather at Agincourt Reef, at the edge of Australia's continental shelf; this coral ribbon teems with parrot fish, sea turtles, dolphins, eels, anemones and much more. Grab your equipment and view the reef and its inhabitants up close while diving or snorkeling, or keep dry and watch it all from within a Quicksilver semisubmersible craft (skimming about 3 feet below the surface). Observe a fish feeding from the Quicksilver's underwater viewing platform before heading back to shore. On land, you'll visit the Tjapukai Aboriginal Cultural Park to learn about Australia's indigenous people. Navigate from the lowlands to high above the tree canopy aboard the Skyrail Rainforest Cableway; in Kuranda, a rail journey lets you admire the region's scenery.
Points of interest: Port Douglas boasts two UNESCO World Heritage-recognized gems: the reef itself and the nearby Daintree and Cape Tribulation rain forest.
Don't miss: In Kuranda -- known as "the village in the rain forest" -- the Kuranda Heritage Markets have drawn visitors for more than 20 years. The markets are open daily, with local tradespeople selling their wares from traditional stalls. Handmade souvenirs include clothing, paintings, jewelry and more.
Did you know? The Great Barrier Reef stretches more than 1,400 miles. It's the only living thing that can be spotted from outer space.
"From the Outback to the Glaciers" with Collette Vacations
Cairns to Sydney
The vast scope of what Australia and New Zealand have to offer is staggering, but three weeks may be enough time to take in the highlights on a tour of the neighboring nations. Begin in Australia at Cairns, known as the gateway to the Great Barrier Reef and the state's scenic northern stretch. T-shirts and shorts are typical attire in this easygoing city, fringed with white-sand beaches and close to the world's oldest surviving tropical rain forest. You'll explore Cairns at your leisure for two days before heading to the Great Barrier Reef via high-speed catamaran. There, choose your own adventure: Take in the sights from a glass-bottom boat, stroll on easy walking trails or opt for diving or snorkeling equipment for more undersea exploration.
Then it's on to the Outback. You'll fly to Uluru (the Aboriginal name for Ayers Rock) and the nearby sandstone domes of Kata Tjuta (the Olgas). These stunning landscapes were formed by erosion and upheaval and are sacred to Aboriginal culture. Witness the sunset's color-changing effect on Uluru over a Champagne toast. A drive through the Red Centre takes you to Alice Springs, where you'll spend a full day learning about the Aborigines and their lifestyle, mingling with members of the Warlpiri tribe. From there, it's back to the city with a trip to Melbourne and a viewing of the historic Fitzroy Gardens for one day before zipping off to New Zealand.
Start your tour of the island nation in Christchurch, a popular destination steeped in English culture. Wind your way past the spectacular Southern Alps, with snowcapped peaks and waterfalls en route to the Franz Josef Glacier to spot New Zealand's famous mountainous masses of ice. A Collette motor coach journey gives you a great view of the landscape before you arrive in Queenstown for two full days. Return briefly to Christchurch (where you'll sightsee at spots like a working sheep farm and the city's elegant park and garden district) before heading back to Sydney to wrap up the trip.
"Sydney" with Pacific Delight Tours
Round trip from Sydney
The trip: An expansive five-day tour of the capital of New South Wales, one of Australia's iconic hubs.
What's in store: Cruise past famous sights like the Harbour Bridge and the Opera House. You'll also swing by world-renowned Bondi Beach, where the rest of the day is at your leisure. Day 3, hop aboard a luxury day cruiser at King Street Wharf to admire the skyline from the water. Guides will provide insightful commentary, and a buffet meal is provided. Then head for the hills at the Greater Blue Mountains World Heritage Site; at Featherdale Wildlife Park you can pet and feed locals like koalas, kangaroos, wallabies and emus. A walk introduces you to the region's diverse terrain, from blue-tinged forests to a tumbling waterfall. Round out the day with a trip to the Waradah Aboriginal Centre to watch a corroboree (Aboriginal dance ceremony).
The digs: Accommodations are provided at the Grace Hotel, a heritage-listed landmark in the heart of Sydney. Built in the 1920s, the Grace has been restored to its former self, displaying Art Deco architecture and a neo-Gothic exterior, with a heated indoor pool, rooftop gym and recreation center and an intimate restaurant.
Snapshot: Your city stay includes a visit to Mrs. Macquarie's Chair, a seat carved from a rock ledge on the eastern fringes of the Royal Botanic Gardens. Elizabeth Macquarie was the wife of Lachlan Macquarie, governor of New South Wales in the early 1800s; the seat was carved for her as she was known to frequent the spot for its panoramic views of the harbor. Today, the chair is considered one of the best vantage points in the city.
The information in this story was accurate at the time it was published in January/February 2014. Please visit Vacations To Go or call (800) 338-4962 for current rates and details.


text:
Thanks indeed for the information! Hope to see those docs for modders soon.
Enjoy your well deserved Xmas holidays!
Thanks really much Scott for your posts...really appreciated
Personally i'd like to see an improved version of Brianza, Northamptonshire and..why not, some other good new real tracks (la sarthe? Nordschleife? ).
I see lots of great things in your words (specially about FFB) so..can't wait to try it.
Happy holidays from Italy!
Very good to hear that at some point in time we can look forward to new things for Dirt. I think dynamic grips alone would be a major factor in realism if they could be adjusted in a very broad range to accommodate what we do on dirt ovals. Looking forward to the release and digging into the new engines!
Remember guys, it doesn't really matter when they release anything, as long as it's ready and correct. That's how modders do it and it seems to work out pretty good for them, so we really shouldn't expect anything else from ISI.
Do you believe me? You don't have to of course. I don't "promise" anything. I know what the situation has been, is and will be--I know what my tasks are for the next year overall. I also know that VERY soon now people will finally be racing online with rF2... Believe it or not
Edit: By the way, I truly understand the frustration. We have been slaving on this thing all through 2011 to get it ready. When we realized it just wasn't going to be possible we were just as frustrated. We're all pretty tired and spent, and would have loved this to have gone out months ago. Personally, not only have I not spent as much time my family as I would have liked, but in my spare time I'm also working on my own game, code and art (totally un-related to racing--it's a space sim), which is recreational and therapeutic for me. I had to set that aside for over a year because I've had no spare time--it's all been going to rF2. Fortunately, I love what I do and truly believe in what rF2 is and will become in time...
Besides, it happens in game development and there's not much you can really do about it except keep going until it's ready. We don't normally really talk about things like that because that's part of this career--it's what we signed up for. Besides, usually it's hard for the people waiting to play the game/sim to grasp that. They were told a date and they expect a product...
I know because I've been on the other end, too--waiting seemingly forever for a product to come out. I remember back in the early 90's first hearing about a space-sim called "X-Wing" and waiting over a year for that to release--drove me nuts
We've been there on both sides--we're right there with you...
"There is no spoon..."
If it's ready, let us have it lol
I don't mind ISI absence for one week, trust me if i have the beta the time will fly by, and the week vacation wont be noticed.
And again it can be hard to understand that from the fans' perspective. You guys just want it, and I don't blame you. But we still have to do it properly, which means proper testing for a week or so--it's just the way it is
Edit: By the way, I'm glad you wouldn't mind, Shum. Fact is we don't feel right about doing it. You guys have waited all this time--we don't want to put out something that crashes if we can help it, or disappear on you all after it goes out, even if it's for a week. It would just be wrong on our part to do that...
"There is no spoon..."
Is the third historic track ready to be released with the beta? I'm not sure from re-reading the OP if you meant its done or not, yet.
e: on the subject of Toban, i ran a couple of races there today, great track - cant wait for the new/old version
Yeah, Toban was always my fav. original rF1 track--can't wait to get back to this and finish it up...
"There is no spoon..."
sorry, Scott. Excurse me please. I respect your team very much. It was my emotional and unconscious comment... your sim-racing products are the best in the world. I played f1 challenge at first, and later rfactor. I found that there are no better sims than yours. So I looked forward to appear your new creation... And it will be so boring to wait still one year.. I really hoped to be beta-tester this year... It's terrible if release will be delayed for months again..
How long do you think we will have to wait from the start of open beta to final release?
Last time i uploaded (touched rF) a video on youtube was september 2009. I can wait 2 more weeks
And i understand how it wouldnt be welcome to have a beta who crashes after all this wait ^^
Now i rather wait with some new footage & screenies, just saying .
"There is no spoon..."
WOW, 6 months? You gotta be kidding me? That's dissapointing as ****!
"There is no spoon..."
how about racing againist AI in beta-stage? is there some progress in offline racing? I already wrote that offline racing was my problem in rfactor. I liked f1challenge AI more than rfactor's AI and I hoped AI in rF2 will be better and offline racing wil be more interesting..for example - racing with different classes when faster AI are intelligent, carefull and don't push player's car
6 months?
In this case, I'm really curious how Tim could tell us, that we will be able to play final release in 2011?


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ESSEX skipper Ryan ten Doeschate admitted his team was gutted to be knocked out of a second quarter final in a month by Warwickshire.
A superb all-round performance from Rikki Clarke saw Warwickshire to the One-Day Cup win at Chelmsford, to add to the Twenty 20 Blast victory they secured on their way to winning that title.
Clarke, who hit an explosive 78 not out in the Twenty20 Blast Trophy success last weekend, he struck a more sedate 68 and captured three for 40 to book his team a place in the semi-final.
A disappointed ten Doeschate said: "It was a big disappointment to fall at the quarter-final stage again but full marks to Clarke. He played really well and got the ball to move about at the start of the innings and reaped reward form that ability."
Oliver Hannon-Dalby also played a significant part in the visitors' win with four for 44 including the wicket of Jesse Ryder who had revived Essex hopes with a belligerent 90 from 95 balls that included three sixes and seven fours.
Jesse Ryder gave Essex some hope
He had the New Zealander caught at backward point by Varun Chopra with 83 still needed, and that departure led to a rapid decline in the innings as the final three wickets in the space of two overs.
But it was Clarke who stole the limelight after Chopra (36) and Will Porterfield (27) both fell to off-spinner Tom Westley with the total on 62 and 67 respectively.
Clarke emphasised his class to enable Warwickshire to seize the initiative. In reaching 50 from only 71 balls, he hit only two boundaries including a six but his ability to coax the ball into the gaps ensured that the scoreboard kept ticking over smoothly.
Graham Napier celebrates the wicket of Jeetan Patel
Jonathan Trott was equally adept in finding the gaps on a sluggish pitch and he made 61, with six fours before left-arm seamer Reece Topley had him caught behind.
It was Topley who also removed Clarke, this time with the help of Graham Napier at deep mid-off but if Essex thought they had seen the last of Clarke, they were sadly mistaken.
Given the new ball, he trapped Westley lbw for five before yorking Greg Smith for 10 as the home side slumped to 38 for three in the ninth over, Mark Pettini having been caught behind off Hannon-Dalby for six.
Ryder arrived to give the innings impetus but he was unable to find a partner at the other end to provide the tempo needed to mount a serious challenge.
James Foster and ten Doeschate did their best with 19 runs each but needed 39 and 25 balls respectively to gather those runs.


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I think I checked all the right boxes, but I do not get an email when someone is reply a post wich I am subscribed to.
I do not know what else I can try but subscribe to a topic and fill in my e mail adress in the user controll box.
can anyone help?


text:
Product Details
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Images used may be generic. Actual product may vary.
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Vehicle Applications
|2003 - 2004


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text:
'); var yuipath = 'clientscript/yui'; var yuicombopath = ''; var remoteyui = false; } else // Load Rest of YUI remotely (where possible) { var yuipath = 'http://yui.yahooapis.com/2.9.0/build'; var yuicombopath = 'http://yui.yahooapis.com/combo'; var remoteyui = true; if (!yuicombopath) { document.write('
Tonight, all flags must burn, in place of steeples.
Autonomy must return into the hands of the people.
Well said. And with that, this seems like a good place for this:
And this is just my general overall feeling about this thread...
However, THIS could be a good reason to actually trade Hibbert...
And... scene.
Thank you for your time.
the foster thtat played two seasons ago vs the bulls in the postseaoson was lights out. or the foster that pulled down like 21 rebounds in a detroit pistons postseason game.. half of which were offensive rebournds. Foster was is in his own right a BAMF as well. defenswively he pissed players off and was always solid on putbacks and made great passes.
point is. even the Foster from two seasons ago vs the bulls was probably in many respects better than Smits or Hibbert. Fosters back would not allow him to play the same minutes. otherwise on a team like this one where scoring is not requird from the center, Foster would probably average 15 rebounds a game and 7 pts. would probably lead lg in offensive rebounds. unselfish and smart.
smits was not durable, and only gave you offesne. he was right for those teams because offense was needed.
Foster was a helluva lot more than a carreer backup. he did everything but score. and thats not a bad thning to have on certain teams where offense is alreadythere.. JO, Artest, Miller, Jackson, Tinsley... Foster did the dirty work and he did it very damn well.
i disagree with dealing Hibbert. I do not think he is as overpaid as some on here believe. the "LITERALLY" stealing money from pacers is an extermist OP.
for conversations sake. what is that everyone on here believes Hibbert should be paid? if the contract could be reworked and negotiated right now what would you expect to see Hibbert paid
The lifelong backup who started 79 out of 82 games for an ECF 61-win team and who started 345 out of 764 career games. Oh, Lord that's hilarious.
55% of the time... it's right every time.
If I had both the time and skill... I'd love to edit that gif. Put a sign on the shopping cart, "Trade Market." Replace the kid tossing the cereal box with the kid tossing Roy's head with a concerned look on his face, cut to the Dikembe swat, showing a now smiling/happy Hibbert floating way...
Yeah, that is one year..... How about the others? How about being hurt all of the time? Foster was a run-of-the-mill post player for almost all of his career.... ...
That doesn't make sense...
This thread is still happening?
This needs to be done...
Jeff Foster (Career Avg)-
764 345 20.6 1.9 3.9 .497 0.0 0.1 .130 1.0 1.7 .615 2.8 4.1 6.9 0.9 0.7 0.4 0.8 2.5 4.9
Mel Daniels (Career Avg)-
639 35.2 7.3 15.6 .468 0.0 0.1 .088 3.8 5.8 .657 4.5 10.4 14.9 1.8 0.6 1.5 3.0 3.6 18.4
Last edited by 5_7_Clash; 02-10-2013 at 01:50 PM.
Yeah but team chemistry was better with Foster
I loved me some Jeff Foster, but lets get real here. Hans is already a better overall player than Jeff. To say otherwise is willful ignorance of the facts.
G GS MP FG FGA FG% 3P 3PA 3P% FT FTA FT% ORB DRB TRB AST STL BLK TOV PF PTS Foster 764 345 21 2 3.9 0.5 0 0.1 0.13 1 1.7 0.62 2.8 4.1 6.9 0.9 0.7 0.4 0.8 3 4.9 Hans 216 30 20 3 7.2 0.43 0 0 0 3 3.8 0.77 1.9 2.8 4.6 0.5 0.6 0.2 0.9 2 9.1
You cannot be serious. Tyler is somewhat fortunate to be 2nd string. We would get killed with him starting.
Lance is finally home. Whether he becomes our starting PG or he's 6th man, he's getting big minutes and he's here to stay. #llortontnia
And we would get killed even worse with Foster starting.
Sorry you lost me, claiming tyler being better than Foster is insane, go over that stat row, and you will see WHY Jeff was at least twice the player hansboroough is. Oh wait, I get it, you only look at the points avg, I seeeeee, lol well that explains ALL. In fact the entire thread is explained in one go, shall I lock it now ?
So Long And Thanks For All The Fish.
If you've done 6 impossible things today?
Then why not have Breakfast at Milliways!
Season Age Tm Lg Pos G GS MP FG FGA FG% 3P 3PA 3P% FT FTA FT% ORB DRB TRB AST STL BLK TOV PF PTS foster 2002-03 26 IND NBA C 77 2 10 1 2.3 0.36 0 0 0 0 0.8 0.54 1.5 2.1 3.6 0.7 0.4 0.3 0.4 1 2.1 hans 2012-13 27 IND NBA PF 51 0 16 2 4.6 0.43 0 0 0 3 3.5 0.73 1.6 2.5 4.1 0.4 0.3 0.3 0.8 2 6.4
So in each players' fourth year as a pro, one player has seen more floor time, got to the stripe more and converted, gotten more rebounds and averaged 3x more points. And the other one is Jeff Foster.
And I also agree that the sooner this thread is taken out back and shot, the better the internet in general will be.
what am i looking at here that proves Tyler > Foster? The 4ppg more at a lower shooting percentage? You can't be serious. The only thing Tyler does better than Jeff and will only do better than Jeff is score. Foster dominated on the glass, especially offensively, was a superior defender and a better passer.
I ablsolutely do not disagree. I meant in my time as Pacers fan which is back Vern Fleming, Greg Drieling, Person so forth. Cannot disagree with you when his # hangs from the rafters.
[QUOTE]HA. im a big Hans supporter and he is no where near the player Fiesty Foster was in his prime. Foster was 6'10 easy and could gaurd most teams Centers. Even did a decent job against Shaq.
Foster better passer, rebounder, defender, leader, and played just as hard. Foster was a better version of Dennis Rodman. Foster dominated games with his offensive rebounding.
The only reason he no longer started for this team was due to back problems. If i had a healthy Foster in his prime or HIbbs right now i would take Foster. if Hibbert hits his prime with some offensive game then i would reconsider. I would take Foster over Smits as well for the long haul, other than a 3 year career run under Bird Smits had durability issues.
Hans better than Foster right now. C'mon Man.


text:
Jay, rest assured, the Pacers will not get Tyson Chandler. The best the Pacers can hope for is AL Harrington for the 7th pick, and the Pacers will have to take maybe Scottie Pippen back so the salaries match.
After thinking about it and reading what they are saying out of Chicago, there is no way the Bulls would have ever traded Tyson and the 7th pick for AL, dispite the excitement last night we should all accept the fact that won't happen.
I do think however if Luke Jackson is available at the 7th pick the Bulls will draft him and trade him to the Pacers for AL, I think that will happen.
Whether that is a good trade or not, I don't have the slightest idea, for two reasons.
1) I don't think the Pacers will stop there, more moves will be made.
2) I have never seen Jackson play so I have no idea. But I will say if the Pacers make this move and Jackson is not the Pacers starting shooting guard by his second season then it was a huge mistake. He also should be able to play 15 minutes per game next season, if he is not ready or good enough to do those two things, then Bird just made his first mistake.
Because make no mistake about it, this is Bird's trade, yes I know DW has a say so, but I highly doubt Walsh makes this trade if Bird were still in Naples, Florida


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For a low-carb meal, this would be great with Mary's Perfect Salad or Roasted Broccoli with Garlic.
Baked Chicken Stuffed with Pesto and Cheese
(Makes two servings, but recipe can easily be doubled or tripled; recipe created by Kalyn in 2005 and adapted again in 2011.)
2 large boneless, skinless chicken breasts
2 T Basil Pesto with Lemon or your favorite commercial pesto
2 T low-fat sour cream
2 T grated mozzarella cheese
(I used heaping tablespoons of pesto, sour cream and cheese)
2 eggs, beaten (or if they're large eggs one will probably be enough)
3 T finely grated Parmesan cheese
3 T almond flour (or almond meal, but almond flour is better for this because it's more finely ground)
black pepper to taste
Click Here for Printer Friendly Recipe (original recipe from 2005)
Find More Recipes Like This One on Pinterest:
Nutritional Information?
Easy Recipe for Baked Pesto Chicken from Kalyn's Kitchen
Fettucine with Chicken, Cherry Tomatoes, and Basil Pesto from Mountain Mama Cooks
Foil-Baked Salmon with Basil Pesto and Tomatoes from Kalyn's Kitchen


text: Yeah, I was taken back a bit by that too. But regardless of how we, the outsiders, view how the mods have treated others, we are not privy to the conversations they have had back and forth with the specific users whose NOC threads get insta-locked. Nor should we be privy to these conversations; as Bmac pointed out, that shit should be between user and mod... Not the general public.


text:
Malcolm wrote:gregkavarnos wrote:So you are saying that true compassion only arises via pointing out? Is that why everybody here has devoted their lives to voluntarily assisting lepers in Bangladesh?
What I said was that unlimited compassion arises from recognizing your real nature. I also said that compassion was innate and everyone has it.
Yes our real nature is open compassion!
http://www.mindful.org/in-body-and-mind ... he-kindest