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You're Different,You're Ok - Giclée Print
Limited edition digital giclée print of an original silkscreen print, layered and digitally altered.
Printed on Moab Entrada Cotton Rag 290 paper, signed and numbered.
25% of the purchase price of all giclée prints acquired directly from me, will go to support charities across the world.
I will be focusing on environment, asylum seeker support, equal rights, reproductive health, and children's health and education.
After purchase I will email you a confirmation of the charity receipt, please contact me if there is a charity you would like to donate to if you are purchasing a print from a country other than Australia.
A3 OPEN EDITION 30cm x 42cm with a 2cm border
A2 limited edition of 100 42cm x 60cm with a 2cm white border.
A1 limited edition of 15 60cm x 84cm with a 2cm white border.
Colours may differ due to computer monitor setting.
Prints are made to order and will be dispatched within 10 working days of confirmation of payment.
Packaging and shipping is included and posted via courier, or Australia Post - Registered service, with a signature for domestic and international.
Buyers are responsible for all import duties and taxes specific to their country of import from Australia.


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'Gaustad doesn't expect to be called up by Buffalo this season, unless a Sabre center gets hurt. He'll attend training camp next summer and hopefully be more effective there than he was in the last two years. If so, he could break camp with the Sabres — if they make it that far. The team is in bankruptcy court, and Buffalo businessman Mark Hamister, the major bidder, has pulled out. Rochester billionaire B. Thomas Golisano is next in line.'
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subset: commoncrawl


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Welcome to Experts at Laurier, a one-stop media resource featuring a broad database of experts who have agreed to make themselves available for media interviews. Contact them directly or call a media relations officer at 519-884-0710, ext. 3067 or 3070.
To search for a topic containing multiple words, please enclose the term in quotation marks, for example, "Canadian history"
Search Results
Your search returned the following Experts.
Click on the person's name for more information.
Associate Professor, Faculty of Social Work; Director Social Innovation Research Group; Director, Manulife Centre for Community Health Research
Areas of Expertise: University/community collaboration; Organizational change and diversity; Co-operatives and the social economy; Domestic violence; Aboriginal issues; Working with survivors of war and torture
Biography: Dr. Lafrenière teaches Master's and Ph.D. level courses in research methods, diversity, community development and social change. She is the founding director of the Social Innovation Research Group (S.I.R.G) which is a research and training incubator…
519-884-1970 ext.5237


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Introduction to SSL
Learn about Symantec SSL Certificates
Web servers and web browsers rely on the Secure Sockets Layer (SSL) protocol to help users protect their data during transfer by creating a uniquely encrypted channel for private communications over the public Internet. Each SSL Certificate consists of a key pair as well as verified identification information. When a web browser (or client) points to a secured website, the server shares the public key with the client to establish an encryption method and a unique session key. The client confirms that it recognizes and trusts the issuer of the SSL Certificate. This process is known as the "SSL handshake" and it begins a secure session that protects message privacy, message integrity, and server security.
Credentials for establishing identity are common: a driver's license, a passport, a company badge. SSL Certificates are credentials for the online world, uniquely issued to a specific domain and web server and authenticated by the SSL Certificate provider. When a browser connects to a server, the server sends the identification information to the browser.
To view a websites' credentials:
Trust of a credential depends on confidence in the credential issuer, because the issuer vouches for the credential's authenticity. Certification Authorities use a variety of authentication methods to verify information provided by organizations. Symantec, the leading Certification Authority, is well known and trusted by browser vendors because of our rigorous authentication methods and highly reliable infrastructure. Browsers extend that trust to SSL Certificates issued by Symantec.
Symantec SSL Certificates offer more services to protect your site and grow your online business. Our combination of SSL, vulnerability assessment and daily website malware scanning helps you provide site visitors with a safer online experience and extend server security beyond https to your public-facing web pages. The Norton Secured Seal and Symantec Seal-in-Search technology help assure your customers that your site is safe from search to browse to buy.
For businesses with a high profile brand, using Extended Validation (EV) SSL Certificates has proven to be an effective defense against phishing scams. For any online business, using SSL with EV may have a big impact on the bottom line. Online shoppers are more likely to enter their credit card and/or other confidential financial information into a website with the SSL EV green bar.
Concerns about identity theft and browser warnings erode consumer confidence, even on secured pages.
To regain their trust, site owners need an easy, reliable way to show customers that their transactions are secure and they are who they say they are. Certificate Authorities and Internet browser vendors have combined forces to establish the EV standard for SSL Certificates.
An EV SSL Certificate gives customers more confidence that they are interacting with a trusted website and that their information is secure. An EV SSL Certificate triggers high-security web browsers to display your organization's name in a green address bar and show the name of the Certificate Authority that issued it. The Certificate Authority uses an audited, rigorous authentication method and browsers control the display, making it difficult for phishers and counterfeiters to hijack your brand and your customers.
Symantec helped lead the development of Extended Validation and as of January 2012 has issued more EV SSL Certificates than any other Certificate Authority.* Our rigorous authentication practices set the standard for online identity assurance and are audited by KPMG. Continuous investment in research and infrastructure helps Symantec maintain the highest standard of practice in the industry and stay well ahead of evolving security risks.
* Includes Symantec subsidiaries, resellers, and affiliates.
Symantec is the premier provider of SSL Certificates. Try a free, instantly-issued Symantec test certificate to evaluate SSL encryption before you deploy it in a production environment.
|Symantec SSL Test Certificate
|How to try
|What visitors see
|If your website
|lies in a test environment to where visitors will not navigate
|If you want to
|What you get
|More information


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When I think of memorable "small" films by talented, independent female directors – movies like Gillian Armstrong's "High Tide," with Judy Davis and Colin Friels, Clare Peploe's "Rough Magic," with Russell Crowe and Bridget Fonda, and Toni Kalem's "A Slipping Down Life," with Lili Taylor and Guy Pearce – what sticks out is the way these filmmakers not only get the best from their female stars, but also find relaxed, seductive, enigmatic shadings in male stars often pushed toward melodrama by male directors.
Now writer-director Nicole Holofcener in "Enough Said" takes male and female icons – Julia Louis-Dreyfus, paragon of snappy urbanity and the well-turned line, and James Gandolfini, exemplar of the twisted humanity of hoodlums – and shapes them into surprising and ultimately enchanting human beings. With Gandolfini as an overweight archivist of old TV shows and Louis-Dreyfus as a masseuse who can sooth clients, not herself, "Enough Said" is a sharp, affectionate comedy-drama. It's witty without being glib, endearing yet not sappy. It deepens your own wells of humor while leaving a pang in your heart.
Of course, Gandolfini was staggeringly good in "The Sopranos," filling out a role that grew from neurotic mob boss to 21st century Everyman. During and after "The Sopranos," he was often cast as leaders like the military prison commander in "The Last Castle," the Mayor in "The Taking of Pelham 123" and Leon Panetta in "Zero Dark Thirty." It's natural that assertive parts would come his way, but he couldn't fulfill his talent in these underwritten roles.
He did better as a supporting actor, hitting a black-comic peak as a hit man unhinged by his id in last year's "Killing Them Softly" (no one was better at delivering the juicy George V. Higgins dialogue), and managing to be touching as a man dying of pancreatic cancer in the messed-up "Violet and Daisy." (Gandolfini died in June of a heart attack.) In "Enough Said," he gets to be expansive once again, in a different, gentler mode – and the movie expands right along with him.
Holofcener's plot is slight: Eva (Louis-Dreyfus) meets Albert (Gandolfini) and Marianne (Catherine Keener), a poet, at the same party. She becomes Albert's lover and Marianne's masseuse before she learns that they had been married. When Marianne complains about her clumsy, obese, under-achieving and altogether repellent ex-husband, Eva doesn't realize he's the same man she has found comforting, convivial and charming.
Once she does put one and one together, Eva, who's also gone through divorce, decides not to come clean with either Marianne or Albert. She likes the idea that Marianne could be her human "trip advisor." Her best friend, a psychotherapist named Sarah (Toni Collette), rebukes that idea – "Albert is not a hotel!" Eva responds, "If you could avoid staying at a bad one, wouldn't you?"
What makes "Enough Said" distinctive is that Holofcener doesn't treat this ploy as a gimmick – or not merely as a gimmick. It makes sense that Eva would yearn for decisiveness and clarity before she commits to another long-term relationship. The joke behind her character is that a masseuse who helps center a diverse crew of clients has a hard time getting "in the moment." Under Holofcener's guidance, Louis-Dreyfus creates a woman with tripwire reflexes that sometimes cause her to stumble.
When Eva asks Marianne what she does, Marianne answers, "I'm a poet." Eva responds, "And I'm a dreamer." It turns out that even Eva's ironies emerge from her instinct for ingratiation – the extra sparkle she brings to the most casual encounter comes from years of service as a masseuse and, yes, a mother. It means something when Eva, in bed with Albert, says, "I'm tired of being funny." Albert mutters, "Me, too." By then, Albert and the audience have come to cherish her odd, rapturous brand of sarcasm. She caps the pillow talk with, "But you're not funny."
Actually, he's a riot. Gandolfini proves remarkably nimble at Albert's put-ons and banter. He knows so much about TV, he says, because "I was raised like a veal – put in a dark room, and fed, and told not to move." The real font of his humor is his frankness. Eva says she likes his big mitts ("they're like paddles"); he tells her, "I like your ass." Eva may be the masseuse, but he's the one who's comfortable in his own skin. He's seductively loose, happy with his job, and apparently beyond caring that his ex-wife looks down on him.
Gandolfini's embedded confidence makes Albert unexpectedly sexy to Eva. He exudes generosity and warmth. He embodies the importance of developing wisdom of your own, without being hemmed in by anybody's pre- or post- conceptions. You can see why Eva might be irritated by the way he shakes his large bucket of popcorn to spread the butter, or mutters instead of whispering, but you still cringe when her annoyance shades into Marianne's disgust with his weight or how he removes onions from his guacamole. Gandolfini plays Albert as a softie, not a pushover. He seems to declare, like Mary J. Blige, "Just take me as I am/or have nothing at all."
Holofcener combines modern awareness and idiosyncratic humor with the common sense of classic American filmmakers. She shows us what her characters do for a living (Albert takes Eva through the TV section of the "American Library of Cultural History"), how they spend time with their friends (eating and ruefully complaining about spouses or ex-spouses), and how they relate to their kids.
Collette and Ben Falcone, as her husband, are terrific as a couple who've turned their marriage into a psychological dart game – the woman is no better at it for being a psychotherapist. Keener is both wispy and razor-edged as a woman more put-together than together. The movie boasts a trio of young dazzlers. Eve Hewson, as Albert's daughter, Tracey Fairaway as Eva's daughter and Tavi Gevinson as Eva's daughter's best friend layer in neediness, sensitivity, intelligence and pique. They elicit a range of motherly emotions from Louis-Dreyfus.
"Enough Said" is about knowing when there's enough in your life. This modest, marvelous movie makes you feel, as the saying goes, that enough is as good as a feast.
Contact the writer: firstname.lastname@example.com


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Prediction: Ryback
Hope: Barrett
Hope: Barrett
Efed stuff that doesn't matter
1 x IWA World Heavyweight Champion - Malcolm Adonis
Current WWE World Heavyweight Champion - Dean Ambrose
1 x EWNCW Tag Team Champion - Malcolm Adonis w/ Kyojin
Current No 1 Contender to the EWNCW Title - Arthur Lansdale
I lied, it matters.
"Tell me.. what is insane about wanting to create perfection? I'd be crazy not to." - Jarvis Cole
Hawow welcome to city wok take ur order pwease
shitt spent time smoking weed am nervous
pass the ball to pagey hes on his game tonight
"I'm so damn good when I'm finished there won't be a dry eye - or dry seat - sorry ladies - in the entire arena." #HEEL
Orton hears a who


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Dear Students,
In this topic you will learn about some major Careers in Biology (Animal Husbandry and Horticulture). This video lecture has been recorded by one of our female instructor Miss Maarij Rehman. She delivered her lecture in such a way that the students will understand all the topics easily and without any difficulty. I hope after watching her lectures, you will get a very good percentage in your exams without any tuition. These video lectures are now the property of our website (www.office.com.pk) and are also uploaded on our Youtube Channel. You can download these videos for free or learn online from our website. All the lectures of Chapter No. 1 (Introduction to Biology) are uploaded topic wise. In order to keep yourself updated from our latest news feed, don't forget to subscribe to our free Email Newsletters. Furthermore, I will also recommend you to join our Facebook study group and our Facebook Fan Page. I hope all of these will be beneficial for you all.


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A Study of the Effects of Amprenavir, a Protease Inhibitor, on Carbohydrate and Fat Metabolism in HIV-Infected Patients
This study has been completed.
Glaxo Wellcome
Information provided by:
NIH AIDS Clinical Trials Information Service
ClinicalTrials.gov Identifier:
First received: November 2, 1999
Last updated: June 23, 2005
Last verified: August 2000
- Full Text View
- Tabular View
- No Study Results Posted
- Disclaimer
- How to Read a Study Record
Detailed Description:
The purpose of this study is to evaluate the effects of a 3-drug anti-HIV regimen, amprenavir (APV) plus abacavir (ABC) plus lamivudine (3TC), on the way carbohydrates (sugars) and fats are metabolized (processed by the body) in HIV-infected patients who have received little or no previous anti-HIV drug therapy.
|HIV Infections
|Drug: Abacavir sulfate Drug: Amprenavir Drug: Lamivudine
|Phase 3
|Study Type:
|Study Design:
|Masking: None (Open Label)
Primary Purpose: Treatment
|Official Title:
|Effect of Amprenavir on Carbohydrate and Lipid Metabolism in Patients With HIV Infection
Resource links provided by NLM:
MedlinePlus related topics: HIV/AIDS
Drug Information available for: AmprenavirU.S. FDA Resources
Further study details as provided by NIH AIDS Clinical Trials Information Service:
|Study Start Date:
|April 1999
This single-site study uses an open-label, single-arm design to evaluate the effects of APV, a protease inhibitor (PI), plus concurrent nucleoside reverse transcriptase inhibitors (NRTIs) ABC and 3TC on carbohydrate and lipid metabolism in non-diabetic, HIV-1 infected patients initiating antiretroviral therapy. Treatment duration is 24 weeks, and patients serve as their own controls. The combination therapy is evaluated for safety, tolerability, efficacy, and effects on body composition. The proportion of patients who attain plasma HIV RNA below the lower limit of quantitation of the Roche Amplicor (less than 400 copies/ml) and Ultrasensitive (less than 50 copies/ml) is measured at 24 weeks after starting therapy with APV plus ABC plus 3TC.
|Ages Eligible for Study:
|18 Years and older (Adult, Senior)
|Sexes Eligible for Study:
|Accepts Healthy Volunteers:
Inclusion Criteria
Patients may be eligible for this study if they:
- Are HIV-positive.
- Are at least 18 years old.
- Have very little or no anti-HIV drug experience (see below for details) and have never taken ABC.
- Have a CD4 count greater than or equal to 200 cells/mm3 within 30 days prior to study entry.
- Have a viral load (level of HIV in the blood) greater than 500 copies/ml within 30 days prior to study entry.
- Agree to use effective methods of birth control during the study.
Exclusion Criteria
Patients will not be eligible for this study if they:
- Have taken both 3TC and stavudine (d4T).
- Have taken a protease inhibitor (PI) or nonnucleoside reverse transcriptase inhibitor (NNRTI) for more than 7 days.
- Have taken a PI within 60 days of study entry.
- Have a history of diabetes.
- Have a history of untreated gonad or thyroid disorder.
- Have a stomach or intestinal disorder which may affect the way the body absorbs the study drugs, or which may make them unable to take medications by mouth.
- Have received chemotherapy or radiation within 4 weeks prior to entry, or if they will need either of these during the study period.
- Have lost or gained a significant amount of weight (greater than 5%) within the past 2 months.
- Have a serious medical condition, such as heart disease.
- Are allergic to any of the study drugs.
- Have been treated for an infection or other medical illness within 14 days prior to study entry.
- Have had a fever (over 38.5 degrees Celsius) for any 7 days or chronic diarrhea (more than 3 liquid stools per day for 15 days) within 30 days prior to study entry.
- Have an AIDS-related cancer (other than Kaposi's sarcoma).
- Abuse alcohol or drugs.
- Have received certain medications.
- Are pregnant.
Contacts and Locations
Choosing to participate in a study is an important personal decision. Talk with your doctor and family members or friends about deciding to join a study. To learn more about this study, you or your doctor may contact the study research staff using the Contacts provided below. For general information, see Learn About Clinical Studies.
Please refer to this study by its ClinicalTrials.gov identifier: NCT00002245
Please refer to this study by its ClinicalTrials.gov identifier: NCT00002245
|United States, California
|LAC / USC Med Ctr / Infectious Diseases
|Los Angeles, California, United States, 90033
Sponsors and Collaborators
Glaxo Wellcome
|Study Chair:
|M Dube
More Information
Keywords provided by NIH AIDS Clinical Trials Information Service:
Additional relevant MeSH terms:
ClinicalTrials.gov processed this record on September 21, 2017
|ClinicalTrials.gov Identifier:
|NCT00002245 History of Changes
|Other Study ID Numbers:
|Study First Received:
|November 2, 1999
|Last Updated:
|June 23, 2005
Keywords provided by NIH AIDS Clinical Trials Information Service:
Drug Therapy, Combination
HIV Protease Inhibitors
Reverse Transcriptase Inhibitors
Anti-HIV Agents
Insulin Resistance
Additional relevant MeSH terms:
HIV Infections
Acquired Immunodeficiency Syndrome
Lentivirus Infections
Retroviridae Infections
RNA Virus Infections
Virus Diseases
Sexually Transmitted Diseases, Viral
Sexually Transmitted Diseases
Immunologic Deficiency Syndromes
Immune System Diseases
Slow Virus Diseases
Protease Inhibitors
HIV Protease Inhibitors
Reverse Transcriptase Inhibitors
Nucleic Acid Synthesis Inhibitors
Enzyme Inhibitors
Molecular Mechanisms of Pharmacological Action
Anti-Retroviral Agents
Antiviral Agents
Anti-Infective Agents
Anti-HIV Agents
Antibiotics, Antitubercular
Antitubercular Agents
Anti-Bacterial Agents
ClinicalTrials.gov processed this record on September 21, 2017


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NBA Houston Rockets Thermometer
NBA Houston Rockets Thermometer
- Made in the USA
- For indoor or outdoor use
- Durable plastic construction
- Built in hanger
- Measures the temperature in any climate.
Officially licensed thermometer. A great fan item to measure the temperature. All plastic construction with a built in hanger. Measures 12.75″ in diameter and comes ready to hang outdoors. Made in USA.
List Price: $ 19.99
Price: $ 19.99


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Proudly Gay, Proudly Made In Arlington Canvas Tote Bag
Product Color:
Product information
Why get a Canvas Tote Bag Proudly Gay, Proudly Made In Arlington? Are you one of those that can't leave home without 50 "essential items" that just won't fit into your pockets? Plenty of room for all your necessities and more. Or simply let it speak your mind! Due to its sturdy fabric it also makes a great shopping bag, which lasts and saves the environment.
This Proudly Gay, Proudly Made In Arlington product includes a full side and bottom gusset and extra long handles for easy carrying. Arlington Canvas Tote Bag made of sturdy 100% Cotton. Measurements: 15" x 18" x 6".
Sku: BL0BL0000010311616160032CRLIFU000


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Plus the winner well also get to see, Sleeping Beauty, a dazzling pantomime spectacular at The Waterfront Big Top @ Echo Arena Liverpool. The show stars, British Soap Awards 2012 'Villain of the Year', Andrew Lancel plus Jonathon Morris and for younger members of the family, Fifi Forget-Me-Not.
The prize is for the 2pm performance on 21st December.
Families are set for a magical festive treat this December when the Liverpool Waterfront is transformed into a captivating Christmas spectacular!
Running for over 3 weeks from Friday 14 December - alongside the pantomime Sleeping Beauty - the Liverpool Christmas Family Festival will host a wealth of family entertainment on the City's Waterfront like never seen before.
Situated on the Kings Dock site, adjacent to the BT Convention Centre and Echo Arena, the Liverpool Christmas Family Festival will transform the area into a space full of sparkling decorations and Christmas delights all conveniently located on one site. And the best news of all...visitors to the pantomime will receive free entrance to the Festival giving adding great value!
Traditional children's fun fair rides, festive entertainers, Santa's Grotto plus a host of Christmas Market stalls and a fully-licensed restaurant are just some of the fantastic attractions to be enjoyed. The Festival will create an enchanting and feel-good atmosphere with carol singers, traditional organ music and an abundance of twinkling lights to dazzle kids and families alike.
And it's not just the Family Festival that audiences can look forward to, as pantomime favourite Sleeping Beauty will run alongside the Festival giving visitors a fantastic entertainment experience and amazing value for money!
Produced by renowned show creator David Lee, Sleeping Beauty is one of the most well-loved pantomimes of all time and will captivate and entertain the audience as they follow the magic, pizazz and comedy escapades of this classic and glittering fairy tale.
The show is jam-packed with laughs, action, drama, surprises, live music & songs and plenty of audience participation, all performed by a fantastic star-studded cast and live band.
Andrew Lancel, Coronation Street's bad boy Frank Foster, stars as Carabose the Wicked Wizard and Master of the Underworld – a despicable character who casts a spell over anyone he dislikes especially those who forget to invite him to their party. Insanely jealous of everyone and obsessed with owning gold, he is mortified when the King and Queen overlook his invitation to the christening of their daughter, Princess Briar Rose. The Wicked Wizard angrily sweeps into the gathering – just before the last of the three Good Fairies is about to bestow her gift on the baby – and casts a spell of death upon the Princess. Only the payment of vast sums of gold each year by the King can prevent the Princess from falling to the fate of the Carabose's evil spell.
In addition to sensational costumes, the glittering set includes a rotating spinning tower castle plus a 15-foot smoke-breathing dragon. The special effects highlights include a digitally animated projection of a growing forest of thorns which envelops the castle where the inhabitants sleep for 100 years.
Other stars set to appear in the show include actor Jonathon Morris (best known for his role as Adrian Boswell in the sitcom Bread), children's TV favourite Fifi Forget Me Not plus Liverpool's open-audition winner Gemma Brodrick starring as Princess Beauty.
Sleeping Beauty will be Fifi's seventh pantomime appearance – playing the role of the Good Fairy she banishes evil and brings enchantment to the show which kids will love, with the unmistakable voice of Jane Horrocks really bringing the character to life on stage, whilst Radio City Breakfast Show DJ Rossie will be providing the voice-over narration to keep the plot rolling along.
To book tickets log on to www.ticketquarter.co.uk / www.echoarena.com or call the TicketQuarter Box Office on 0844 8000 410 or the Echo Arena Box Office on 0844 8000 400
Tickets available to personal callers at TicketQuarter Queen Square. Group Bookers can call the Echo Arena Group Booking Line on 0844 561 7672 PANTOMIME TICKET PRICES: **
* A Family Ticket consists of 4 seats – the group must include one adult over 18.
* Please note that there is an additional charge for some attractions in the Family Festival
Entrance to the Festival is free to pantomime ticket holders (on performance day only)
Festival only tickets = £5 per person & includes entrance into the market, Christmas Cracker Show & Santa's Play Area (Festival tickets are purchased on location on the day subject to availability)
Tickets / Tokens for Santa's Grotto & Funfair are available at the Festival
Waterfront Big Wheel Ride – only £3.50 per person for pantomime ticket holders
** All tickets are subject to a booking & handling fee
For your chance to win simply complete the form below. Closing date for entries 12 noon 30 November 2012.
See full story in the Leader


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Sign into our member site to view your information online.Forget your username or password? Not registered? Register here
benzocaine topical (generic name)
Dermoplast (brand name)
It is used to treat the itching, swelling, and pain of hemorrhoids
(BEN zoe kane)
Table of Contents
Top Learning Centers(Recursos en Español)
What is this medicine?BENZOCAINE (BEN zoe kane) causes loss of feeling in the skin. It is used to treat the itching, swelling, and pain of hemorrhoids.
What should I tell my health care provider before I take this medicine?They need to know if you have any of these conditions:
How should I use this medicine?This medicine is for external use only. Do not take by mouth. Follow the directions on the label or those given to you by your doctor or health care professional. Do not get this medicine in your eyes. If you do, rinse out with plenty of cool tap water. Do not put this medicine inside the rectum. Do not use this medicine more often than directed.
Talk to your pediatrician regarding the use of this medicine in children. While this medicine may be used on children as young as 12 years of age, precautions do apply.
What if I miss a dose?If you miss a dose, use it as soon as you can. If it is almost time for your next dose, use only that dose. Do not use double or extra doses.
What may interact with this medicine?
What should I watch for while using this medicine?Stop using this medicine if your symptoms do not get better in 7 days or if they get worse. Tell your doctor right away if you have any rectal bleeding.
What side effects may I notice from receiving this medicine?Side effects that you should report to your doctor or health care professional as soon as possible:
Side effects that usually do not require medical attention (report to your doctor or health care professional if they continue or are bothersome):
Where should I keep my medicine?Keep out of the reach of children.
Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F). Do not freeze. Throw away any unused medicine after the expiration date.
Remember, keep this and all other medicines out of the reach of children, never share your medicines with others, and use this medication only for the indication prescribed.


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Category: Kimberly Stewart baby name
This week for her Nameberry 9 report, Appellation Mountain's Abby Sandel looks at baby name regret and at new celeb and blogger babies.
Do you regret your child's name? If you could go back, would you choose something less common? More mainstream? Would you use that out-there option that others dismissed as too weird? Or maybe embrace something from your family tree that felt too old-fashioned to bestow on a newborn?
Every so often a study grandly announces that a percentage of parents – in the most recent article, it was a whopping 8%, rounded up to "a tenth" for the headline – wish they could get a do-over on their child's name. While plenty of parents report disappointment that their choice turned out to feel too ordinary, reports and comments tend to focus on the extreme cases: "Yes, I knew a couple who called their son Bullet and really wished they'd stuck with Bill."
But I can't help observe that parents who have picked out-of-the-box baby names seem more satisfied than those who gave it less thought. Rowan at Eponymia summed it up perfectly: "It hardly matters what the name turns out to be, but I believe naming someone is an honor, one that requires effort and thought."
Which brings us back to one of my new categories of favorite names – blogger kids. It comes as no surprise that writers put extra care into picking names for their children.
Ottilie Valentine – The deliciously frilly, but still edgy, name of Rowan's daughter. Her tale of spotting a related name on an athlete during the Summer Olympics, then thumbing through a short story collection and seeing it again is a great example of how, as she puts it, "the right name will find the right person."


text:
Tennessee Titans iPad Cleaning Cloth
This Tennessee Titans iPad microfiber cleaning cloth is great for keeping your iPad screen free of fingerprints, oil, dirt and smudges. The cloth is sized to perfectly fit the iPad screen so that it lays over it as added protection when the device is in a case. 7.5" x 9.5" cloth.
How long will my items take to ship?Most of the items we sell will ship within 1-2 business days from the date ordered. Every item on our website will tell you how long it takes to ship under the availability section when that item.
Once an item leaves the warehouse, Regular Ground shipments will be in transit for 2-5 business days & expedited shipments will be in transit for 1-3 business days.
You can click the "Estimated Delivery Dates" link next to the add to cart button on each product page to get an idea of when your merchandise will arrive to you.
How much does shipping cost?Our shipping charges are automatically calculated by our shopping cart after you enter your shipping information. Our shipping rates are based on the total cost of merchandise in your order:
|Order Amount
|Regular Ground Cost
|Orders Under $10
|Orders $10-$19.99
|Orders $20-49.99
|Orders $50-$74.99
|Orders $75-$99.99
|Orders $100-$149.99
|Orders $150-$199.99
|Orders $200-$299.99
|Orders $300-$399.99
|Orders $400-$499.99
|Orders $500-$599.99
|Orders $600-$699.99
|Orders $700-$799.99
|Orders $800-$899.99
|Orders $900-$999.99
|Orders $1,000 or More
*To obtain the shipping costs to Hawaii, Alaska, APO addresses & also for expedited shipping, please add your items to the shopping cart, enter your shipping address and use our shipping calculator for current rates.
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Israel Beat
Israel Beat
The Israel Beat blog is a place for poetry submission, concert announcemnets, upcoming shows and musings on Jewish music. The Israel Beat Jewish Music Podcast brings you live in-studio performances with up and coming Israeli musicians as well as interviews with the stars of the Jewish music world. Plus your music requests and the free CD give-away air live on the show. Past interviews have included Matisyahu, Avraham Fried, and Miri Ben-Ari. The Beat with Ben Bresky broadcasts live every Sunday from 6:00 p.m. to 7:00 p.m. Israel time on Arutz Sheva - Israel National Radio. For more info email email@example.com.
Israel Beat archives
Latest posts
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Becky Ringer T-shirts are an Idakoos specialtiy! Idakoos has many things to offer. Whether you want to get a Becky Ringer T-shirt for a special occasion, give a Becky T-Shirt as a present or need to represent with a funny Becky Ringer Tee during Holidays or Family and Friends get-togethers, Idakoos is sure to have the right Becky Ringer T-shirt for you! Even if it turns out you can't find the right Becky Ringer Shirt with the perfect design for your mother, father or children (or friends!); Idakoos offers all of its men's women's and children's Becky Ringer T-shirts as Custom Becky T-Shirts. Personalize any Becky Ringer T-shirt the way you want to ensure the perfect gift.
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The holiday season is a time for giving, and local law enforcement agencies are giving some people a second chance.
The Santa Clara County Sheriff's Office is working with the Campbell and Los Gatos police departments, among many others, during the month of December as part of Operation Second Chance. The program began on Dec. 1 and runs through Dec. 31.
Individuals with nonviolent misdemeanor criminal and traffic warrants can receive a citation with a new court date in lieu of being booked into county jail. Operation Second Chance is directed toward most misdemeanor warrants, no matter what the bail is, including warrants stamped "No Cite and Release" and "No Bail."
Individuals can go to any Santa Clara County law enforcement agency to receive their warrant citation, which will require a new "Promise to Appear" in court date.
Felony warrants and warrants involving violence do not qualify for the program. Law enforcement agencies are encouraging people with these types of warrants to take this opportunity to turn themselves in so they can take care of their matter in court.
"This program has been tremendously successful over the past seven years," said Sgt. Jose Cardoza of the sheriff's office in a press release. "More than 2,400 people have taken advantage of the program and avoided spending the holidays in jail by self-surrendering. They were issued citations with a new court date and released 'on the spot.' "
Individuals with outstanding misdemeanor warrants can go to their local police and sheriff's stations and surrender themselves on the outstanding warrants and be released instead of going to jail.
Local stations include:
Santa Clara County Sheriff's Office Headquarters, 55 W. Younger Ave., San Jose; 24 hours a day, seven days a week.
Campbell Police Department, 70 N. First St., Campbell: Monday, Tuesday, Thursday, Friday, 8 a.m. to 5 p.m., and Wednesday, 8 a.m. to 6 p.m.
Los Gatos Police Department, 110 E. Main St., Los Gatos: Monday through Friday from 9 a.m. to 4 p.m.
Contact the Sheriff's Office Records Department with any questions at 408.808.4717.


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Today in Millbrae, State of California: 26/09/2017 21:09:41. Delivery price: 10.00$.
Range depends on the city and the date of delivery
Ordering flowers in our store to Millbrae, State of California, you should be sure that the bunch will contain only the freshest flowers in accordance with the presented pictures and customer preferences with delivery to Millbrae, State of California. When placing an order you should not only chose the available bunch from the catalogue, but chose the flowers for each bunch in accordance with your preferences and financial possibilities. Our flower delivery service ensures the perfect quality of executing your order.
The range of flowers and gifts is presented taking into account prices and specifications of this region. The delivery cost is not included in the cost of goods allowing take into account a delivery cost to Millbrae, State of California only once regardless the number of selected gifts for delivery. The time and cost of delivery depend upon the distance. To read more information you can here.
If you are not satisfied with the presented range of bunches and flowers and if the city is regional one you can place an individual order. We will quickly process your order and give the qualified answer as for the possibility of executing your order and its total cost with delivery to Millbrae, State of California.
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|«Thank you for efficient and timely delivery. Will buy again. Highly recommend! A+++ »
Alina, USA, San Gabriel, Email: ali...@yahoo.com
|«Hi all! Excuse, please, my terrible english. I want to say thanks to all YES team. My honey Lenochka in Riga was very suprised and happy becouse of nice roses. Thank you for it. Your service is excellent!»
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Send flowers to Millbrae. Candy `` Fazer `` delivery service in Millbrae. Send Bouquet of gerberas and chrysanthemums to Millbrae - USA


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World SeriesLogic puzzles require you to think. You will have to be logical in your reasoning.
In Major League Baseball, the World Series is a best of seven series. The first team to win four games is the champion. The Series is scheduled so that one team hosts the first two games and the last two games, while the other team hosts the middle three games. The World Series ends as soon as one team wins four games, so the Series could be completed in as few as four games.
In this fantasy Series, the New York Yankees played the Los Angeles Dodgers. Los Angeles hosted the first two games. The final scores of the games were:
Game 1: 4 to 3
Game 2: 6 to 5 (11 innings)
Game 3: 11 to 7
Game 4: 16 to 9
Game 5: 9 to 6 (10 innings)
Game 6: 2 to 1
Game 7: 13 to 8 (13 innings)
Which team won the World Series?
AnswerSeven games were played, which means that the teams split the first six games, each team winning three games. It does not matter who won each of the first six games, since neither team had yet to win four games. The winner of game 7 will have won four games, and therefore, the World Series.
The team that hosts the first two games also hosts the last two games. Since Los Angeles hosted the first two games, game 7 was in Los Angeles. In baseball, the home team bats last. Los Angeles batted last in game 7. Since game 7 went 13 innings, Los Angeles would have batted in the bottom of the inning. In this situation, the home team can never win by more than four runs. When the home team bats in the last inning, the game ends as soon as the winning run is scored. The only way the home team can win by more than one run is via a home run, in which case all runners on base at the time can score. A grand slam home run would only provide a margin of four runs. Therefore, the home team's margin of victory in an extra-inning game can never exceed four runs. The final score was 13 to 8, a margin of five runs. Los Angeles, being the home team, could not have won by that margin. The game was tied 8 to 8 going into the 13th inning. New York scored five runs in the top of the 13th inning.
Therefore, the New York Yankees won the World Series, four games to three.
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Posted 7 years ago
(26 items)
Lake Shore & Michigan Southern brass top, bellbottom railroad lantern made by the Buffalo Steam Gauge and Lantern Co., in Buffalo, NY, around 1880. This piece is an early no. 39 style offering from this manufacturer. This lantern is prominently marked LS&MS on the base and has early 1871 patent dates (Parmelee & Bonnell patents) stamped into the lid reflector. It also has a sliding globe adjuster with rotating collar on the chimney. The collar is stamped with an 1878 patent date, this patent assigned to C.T. Ham, then an employee and inventor with the BSG&L Co. This classic, double-guarded frame has early bail eyelets formed in the vertical guard wires for attaching the bail directly to the lantern frame, an improvement over the somewhat typical and problematic chimney bail attachment used in lanterns of this era. The clear, extended base globe is embossed LS&MS Ry in heavy relief. The fine vent hole filigree in the chimney makes this model particularly eye-appealing.


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- Description
- Tasting Notes
- What's Inside
- Delivery
- Occasions
Summer may be the season for weddings, but romance blossoms in the fall! Send your love this autumn season with this radiant bouquet of brilliantly colored flowers in a stunning, keepsake vase.
The dazzling blooms include fall favorites such as red roses, Asiatic lilies, alstroemeria, chrysanthemums, matsumoto asters, all delightfully arranged in a dramatic glass vase that comes complete with a satin ribbon.
Approximately 12 1/2" W x 17 1/2" H. To ensure the best arrangement, flower varieties and container may vary.Gift #16944
Fall in Love Bouquet is a hand delivered gift. These gifts are delivered by one of our local design centers located throughout the United States and Canada.
Typically, standard deliveries are made between 9:00 AM and 6:00 PM, with business deliveries before 5:00 PM. Hours may be extended during holidays. Please note in some locations we may need an additional 24 hours for the delivery of exotic flowers, wine and fruit baskets.
Same day delivery is available for most gifts when ordered before the cutoff time. The cutoff time for Fall in Love Bouquet is in the recipient's time zone.
Fall in Love Bouquet has been purchased most frequently by our customers for the following occasions:
Average Rating:(1 reviews) write a review
AngelineShe loved it!November 8, 2013
As far as I know the delivery was on time. It was a beautiful bouquet, she loves the flowers and loves the smell.
I must add, that my card wasn't signed and I was contacted twice (email and phone) to verify that, that was what I wanted. I liked that.
The vase wasn't the same as shown but, I'm not complaining.
I'll be ordering from GiftTree again.


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CATS Tickets
CATS is a musical composed by Andrew Lloyd Webber in 1978. It is based
on story of Old Possum's Book of Practical Cats by T.S. Eliot. It
has only been surpassed in highest number of performances by Webber's
other hit production Phantom Of The Opera. CATS is and has been one of the most-loved
musicals of all time, being translated into over 20 languages.
More about CATS.
CATS Information Continued
CATS was first shown in London's West End, at New London Theatre, on May 11, 1981. The show then made its debut on Broadway on October 7, 1982 at the Winter Garden Theatre.
The plot of CATS follows a particular English feline tribe, the Jellicles, who have come together for their annual Jellicle Ball at a junkyard to make the Jellicle Choice and announce the cat who can be reborn. Each cat "auditions" with a different song and/or dance number. Some minor complications involving the abduction of the Jellicle patriarch, Old Deuteronomy, occur before a cat is chosen.
The choosen cat turns out to be Grizabella, "the Glamour Cat", whose now-faded charms do not prevent her from singing "Memory", the show's best-known song. It has been recorded by over 150 artists, and is considered by some to be Webber's signature tune. The lyrics are based on Eliot's poems "Rhapsody on a Windy Night" and "Preludes".
Other cat characters dancing and singing their way into audiences' hearts in this musical include, Magical Mr. Mistoffoles, Rum Tum Tugger, Old Gumbie Cat, Macavity, Gus the theatre cat, Mungojerri and Rumpelteaser and Skimbleshanks the Railway cat. Dancers and singers, through the aid of some of theatre's most incredible make-up and costumes, are transformed from young, exuberant entertainers into sleek, skulking, playful cats.


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PITTSBURGH — Dick's Sporting Goods is partnering with a group of current professional football players who have had life experiences with breast cancer, as part of its campaign for National Breast Cancer Awareness month. Denver linebacker Von Miller, New England wide receiver Wes Welker and other gridiron stars will promote the "Sport Your Support" campaign and "Endless Ribbon" initiative via public relations and social media.
Throughout October, the retailer will feature a variety of pink items in stores and online from top brands including Nike, Reebok, Under Armour, Lady Hagen and Maxfli, and is encouraging consumers to join in showing their support for the cause.
To kick off the program, DICK'S Sporting Goods donated $250,000 to the National Breast Cancer Foundation (NBCF).
Consumers will also have the opportunity to "Sport Their Support" by participating in Dick's Sporting Goods' "Endless Ribbon" Facebook initiative. From now through Oct. 31, consumers can visit the Dick's Sporting Goods Facebook page, add a pink ribbon to their profile picture and dedicate it to a loved one or include it as an expression of hope. DICK'S will aggregate all participants' photos in its "Endless Ribbon" gallery, where the photos together will attempt to form the World's Longest Pink Ribbon.


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What I Love About Me: New York Fashion Week
This isn't exactly Fifty Shades territory: The mood is set in a glamorous 1930s movie star's satin-clad boudoir, not a dark dungeon. During Fashion Week, we saw models slink down the runway in sumptuous silks and lingerie-inspired looks at The Row, Donna Karen, and Narciso Rodriguez, to name a few. In the beauty world, this translates to bedroom eyes in the form of black-cat-tinged liner, roll-in-the-hay tousled hair (like at Versace, seen here), decadent perfumes, and shades of blush and bashful aplenty. Kind of makes you want to slip into something a little more comfortable, no? —Erin Flaherty
Fresh Sugar Petal Tinted Lip Treatment Sunscreen SPF 15, $23.
Skincare Myths, Busted


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Case ascertainment in the Prenatal Determinants of Schizophrenia (PDS) study cohort. DIGS indicates Diagnostic Interview for Genetic Studies; SSDs, schizophrenia spectrum disorders.
Insel BJ, Schaefer CA, McKeague IW, Susser ES, Brown AS. Maternal Iron Deficiency and the Risk of Schizophrenia in Offspring. Arch Gen Psychiatry. 2008;65(10):1136–1144. doi:10.1001/archpsyc.65.10.1136
© 2017
Iron is essential for brain development and functioning. Emerging evidence suggests that iron deficiency in early life leads to long-lasting neural and behavioral deficits in infants and children. Adopting a life course perspective, we examined the effects of early iron deficiency on the risk of schizophrenia in adulthood.
To determine whether maternal iron deficiency, assessed by maternal hemoglobin concentration during pregnancy, increases the susceptibility to schizophrenia spectrum disorders (SSDs) among offspring.
Data were drawn from a population-based cohort born from 1959 through 1967 and followed up for development of SSD from 1981 through 1997.
Of 6872 offspring for whom maternal hemoglobin concentration was available, 57 had SSDs (0.8%) and 6815 did not (99.2%).
Main Outcome Measure
Prospectively assayed, the mean value of maternal hemoglobin concentration was the primary exposure. Hemoglobin concentration was analyzed as a continuous and a categorical variable.
A mean maternal hemoglobin concentration of 10.0 g/dL or less was associated with a nearly 4-fold statistically significant increased rate of SSDs (adjusted rate ratio, 3.73; 95% confidence interval, 1.41-9.81; P = .008) compared with a mean maternal hemoglobin concentration of 12.0 g/dL or higher, adjusting for maternal education and ethnicity. For every 1-g/dL increase in mean maternal hemoglobin concentration, a 27% decrease in the rate of SSDs was observed (95% confidence interval, 0.55-0.96; P = .02).
The findings suggest that maternal iron deficiency may be a risk factor for SSDs among offspring. Given that this hypothesis offers the potential for reducing the risk for SSDs, further investigation in independent samples is warranted.
Although the onset of overt schizophrenia typically manifests during adulthood, multidisciplinary evidence suggests that a considerable portion of its pathogenesis lies in early brain development, specifically the prenatal period. Pregnancy imposes substantial demands for oxygen and essential nutrients on the mother, the growing fetus, and the placenta. Failure of the maternal-placental unit to meet the demands of the fetus, particularly when occurring during critical periods of brain development,1 produces an adverse intrauterine milieu that can exert long-lasting, deleterious effects that extend into adulthood.
An extensive range of prenatal exposures has been linked to later development of schizophrenia among offspring. One potentially important exposure is maternal iron deficiency, which is a risk factor for several adverse birth outcomes2 and which can induce hypoxia during this period of high oxygen demand. Although iron deficiency during pregnancy has not been studied with regard to psychiatric outcomes among offspring, 2 of its correlates, fetal hypoxia3- 8 and nutritional deprivation,9,10 have been extensively examined in the schizophrenia literature and have been consistently implicated as risk factors for the disorder.
The primary direct consequence of maternal iron deficiency is anemia, indicated by low circulating hemoglobin levels, which impairs the oxygen-carrying capacity of the mother and reduces oxygen delivery to the developing fetus.11 Moreover, maternal iron deficiency might disrupt neurodevelopment through its effect on birth outcomes,2 several of which are putative risk factors for schizophrenia.12 Furthermore, iron is essential for a plethora of metabolic mechanisms associated with the development and maintenance of brain structures and functions relevant to schizophrenia,13 including myelination14,15 and dopaminergic neurotransmission.16 Therefore, failure to meet iron demands of the developing brain may have long-term consequences.
The literature on childhood development provides support for a plausible role of iron deficiency in the pathogenesis of schizophrenia. Motor, cognitive, and behavioral deficits are often observed in children who later develop schizophrenia17- 20 and comparable developmental abnormalities have been observed in iron-deficient infants and children,21,22 assessed via cord blood and maternal serum samples, suggesting that prenatal iron deficiency may set children on a similar developmental course, predisposing them to schizophrenia in adulthood.
Although these findings provide compelling grounds to hypothesize that maternal iron deficiency plays a role in the etiopathogenesis of schizophrenia, no study in the literature has explicitly tested this association. Iron is essential for synthesis of hemoglobin, the protein present within erythrocytes responsible for blood oxygenation. As an indicator of maternal iron status, prospectively acquired maternal hemoglobin concentration data were used to examine the hypothesis that maternal iron deficiency in pregnancy is a risk factor for schizophrenia among offspring. We also conducted exploratory analyses of the gestational timing of low maternal hemoglobin levels and the effect of sex of the offspring on schizophrenia risk. The data were drawn from the Prenatal Determinants of Schizophrenia (PDS) study,23 a prospective investigation of a large birth cohort that included direct bioassays of maternal hemoglobin levels and comprehensive measures of other prenatal and perinatal exposures.
As described in detail by Susser et al,23 the PDS study was derived from the birth cohort of the Child Health and Development Study (CHDS). Nearly all pregnant women receiving prenatal care from the Alameda County Kaiser Permanente Medical Care Plan (KPMCP) were recruited from 1959 through 1966.24,25 The 19 044 live-born offspring from this cohort were automatically enrolled in the KPMCP. The Kaiser membership was racially, educationally, and occupationally diverse and demographically similar to the population of the region at the time.
The PDS study cohort composed the subsample of 12 094 offspring born into the CHDS cohort and were members of the KPMCP at any time from January 1, 1981 (the year when medical records were computerized), through December 31, 1997, the last date for case ascertainment in the PDS study.23 Compared with the CHDS cohort, the PDS study sample had a slight underrepresentation of offspring of low-income and unmarried women and a modest overrepresentation of offspring of African American women but was otherwise comparable. Most individuals who left the KPMCP did so before age 10 years, before onset of the schizophrenic prodrome, thereby minimizing bias from loss to follow-up.
The outcome was schizophrenia and other schizophrenia spectrum disorders (SSDs), defined as schizophrenia, schizoaffective disorder, delusional disorder, psychotic disorder not otherwise specified, and schizotypal personality disorder, based on previous studies.26 Potential SSD cases were identified through computerized record linkages between CHDS and KPMCP identifiers (Figure). In the first stage of screening, the KPMCP inpatient registry identified subjects who were hospitalized with nonaffective or affective psychotic disorder diagnostic codes 295 through 299 from the International Classification of Diseases, Ninth Revision. Following review of their psychiatric and medical records by an experienced, board-certified research psychiatrist (A.S.B. and E.S.S.) for evidence of psychotic symptoms, 144 participants with potential SSDs were targeted for a full diagnostic assessment. Participants identified from outpatient and pharmacy registries who may have been treated for psychosis without hospitalization were also targeted for a full diagnostic evaluation. Overall, inpatient and outpatient registry screening procedures identified 183 participants for diagnostic interviews. Among the 170 survivors, 146 were contacted, and 107 completed the Diagnostic Interview for Genetic Studies (DIGS),27 which was conducted by clinically experienced interviewers. The DSM-IV diagnoses were assigned by consensus of 3 experienced research psychiatrists, based on the written DIGS narrative, medical records, and discussions with the interviewer. For the 76 potential subjects who were not interviewed, DSM-IV diagnoses were assigned by medical record review and confirmed by a research psychiatrist (A.S.B. or E.S.S.). A total of 71 SSD cases were ascertained, 44 by DIGS and 27 by medical record review.
All participants in the PDS study provided written informed consent. The study protocol was approved by the institutional review boards of the New York State Psychiatric Institute and the KPMCP.
The PDS analytic sample23 was defined to ensure independence of observations because the CHDS birth cohort included siblings, who represent nonindependent observations. This was achieved by randomly selecting only 1 sibling from each family. Because of the limited number of offspring diagnosed with SSD during follow-up, if a sibship included an affected sibling, only the affected sibling was chosen. If a sibship did not include an affected sibling, then 1 sibling was randomly chosen. The selection process produced a total of 7796 offspring, referred to as the PDS Cohort.23 In the present analysis, 5 offspring diagnosed as having SSDs were excluded (4 offspring diagnosed before the start of the PDS study and 1 who had an affected sibling in the PDS Cohort). Among the 66 participants with SSDs, maternal hemoglobin data were available for 57. The diagnoses of the 57 participants with SSDs were as follows: schizophrenia (n = 34), schizoaffective disorder (n = 13), delusional disorder (n = 1), schizotypal personality disorder (n = 4), and other schizophrenia spectrum psychosis (n = 5).
Hemoglobin concentration was chosen as a marker for iron deficiency because it is an excellent indicator of iron status and blood oxygenation.28,29 Extracted from well-documented prenatal medical records, maternal hemoglobin concentration data were available for 6872 of 7791 PDS Cohort offspring (88.2%). As the pregnancy of the mother advanced, the number of hemoglobin observations increased: 46% had hemoglobin assayed anytime during the first trimester, 78% during the second trimester, and 91% during the third trimester. Moreover, 59% of mothers of the offspring had 3 or more hemoglobin measurements, 31% had 2 measurements, and 10% had 1 measurement.
The mean value of maternal hemoglobin concentration during the entire pregnancy was chosen as the primary exposure because we did not have an a priori prediction that the effect of low maternal hemoglobin concentration on risk of SSDs was specific to any particular gestational period or trimester. This option maximized the number of participants in the analysis by enabling us to include all offspring with at least 1 maternal hemoglobin measurement. Nevertheless, because of the varying prenatal timing of the hemoglobin observations, we evaluated whether the temporal pattern might affect the results (see the "Data Analysis" subsection).
Hemoglobin concentration was first modeled as a continuous variable to assess SSD risk across the full spectrum of maternal hemoglobin values and to maximize statistical power. We also classified hemoglobin concentration into 3 main categories according to the World Health Organization's criteria28: hemoglobin level of more than 12.0 g/dL (reference), more than 10.0 but less than 12.0 g/dL (moderate), and 10.0 g/dL or less (low). (To convert hemoglobin to grams per liters multiply by 10.0.) Analyzing hemoglobin concentration as a categorical variable enabled us to evaluate whether the rate of SSD linearly increased with decreasing hemoglobin concentration or whether the rate was constant for hemoglobin concentration below a threshold level. Analogously, we modeled mean trimester-specific hemoglobin level as a continuous and a categorical variable to explore whether a critical gestational period of susceptibility existed.
In a supplementary analysis, we used the Maternal Intercurrent and Prenatal Conditions of the International Classification of Diseases, Ninth Revision, to identify women clinically diagnosed with anemic conditions during pregnancy: 2910 (iron deficiency), 2929 (other specified or pernicious), and 2930 (not otherwise specified). The 2 non–iron-deficient anemic categories were collapsed into a single category because of the limited number of women with these conditions and the lack of specific hypotheses concerning the "other specified" and "not otherwise specified" anemic conditions.
Cox proportional hazards regression30 was used to account for varying durations of follow-up while simultaneously adjusting for multiple covariates. For offspring diagnosed with SSDs, the onset date was approximated by the date of the first hospital admission or outpatient visit, whichever came first. Therefore, the length of the follow-up for offspring diagnosed with SSDs was quantified in days elapsed from birth until the onset date of the disorder. For the unaffected offspring, the follow-up length was quantified as days since birth until the KPMCP membership termination date or until the end of the PDS study, whichever came first.
Initially, we investigated the temporal pattern of hemoglobin level observations across pregnancy to validate our use of mean hemoglobin level as the exposure statistic. Linear regression was used to approximate such changes for offspring with 2 or more hemoglobin observations; intercepts and slopes were calculated and then added as SSD predictors in the Cox regression model. We also added a variable reflecting the timing of the first maternal hemoglobin observation. None of the time-related variables were statistically significant (full model: P = .49 for intercept; P = .27 for slope; P = .23 for timing of first hemoglobin measure), indicating that adjustment for temporal variation of hemoglobin observations would not affect the results.
Variables selected as a priori potential confounders were maternal ethnicity,31 maternal age, maternal education, maternal prepregnancy body mass index (calculated as weight in kilograms divided by height in meters squared),32 parity, maternal smoking during pregnancy, and maternal mental conditions, defined as psychosis, anxiety reactions, and drug/alcohol addiction. Potential confounders were considered for inclusion in the Cox regression model if the variables were statistically associated with hemoglobin and were also related to SSD at P <.10, using the χ2 statistic for categorical variables and the t test for continuous variables, and were retained in the final model if their inclusion produced at least a 10% change in the regression coefficient for hemoglobin concentration.
We also examined whether observed consequences of maternal anemia,2,12 including low birth weight (<2500 g), prematurity (<37 gestational weeks), and small-for-gestational-age status (defined as birth weight in the lowest 10th percentile according to gestational week), were potential mediators. The presence of mediation effects was tested by determining whether the addition of the variable to the Cox model attenuated the regression coefficient for hemoglobin concentration. We also examined whether hypoxia (defined in accord with prior research7,33,34 as preeclampsia, gestational hypertension, hypotension, or diabetes mellitus) was an independent risk factor for SSDs.
The PDS Cohort included only 1 sibling per family. Excluding subjects has the potential to create bias. To address this concern, we also analyzed the full PDS sample, including all siblings, which contained hemoglobin data for 9971 offspring, including 58 participants with SSDs. We used a frailty model (Weibull regression with shared γ frailty, as implemented in Stata statistical software35), that adjusts for possible dependence between siblings; the full PDS sample had insufficient data to fit a Cox regression model with shared γ frailty. For ease of comparison with previous PDS study analyses, we used the PDS Cohort as the primary sample and the sample in the frailty model for confirmation of the results. Each of the approaches has advantages and disadvantages: although the frailty model adjusts for dependence between siblings, a disadvantage is that its baseline hazard assumes a parametric form, which is more restrictive than the Cox model, which allows a nonparametric baseline hazard.
Maternal hemoglobin concentration ranged from 6.90 to 15.20 g/dL (mean [SD], 11.47 [0.98] g/dL). Women with low gestational hemoglobin concentrations (≤10 g/dL) were significantly younger, more likely to be of a race/ethnicity other than white, less educated, and had more prior live births and a higher prepregnancy body mass index compared with women who had adequate hemoglobin concentrations (Table 1). Maternal mental conditions were not significantly related to maternal hemoglobin concentration (P = .35); moreover, the prevalence of mental disorders was lower among mothers who had low hemoglobin concentrations compared with mothers who had adequate levels of hemoglobin. Although maternal ethnicity was the only variable to fulfill the confounder criteria, for comparability with studies that controlled for socioeconomic status and to limit finite sample bias,36 we also added maternal education to the final Cox regression model.
We first examined hemoglobin concentration modeled as a continuous variable in relation to SSDs. The mean maternal hemoglobin concentration was significantly higher among the 6815 unaffected offspring (mean [SD], 11.47 [0.97] g/dL) compared with the 57 offspring diagnosed with SSDs (11.12 [1.09] g/dL) (P = .006; t test for equality of means). Increasing concentration of hemoglobin was associated with a lower rate of SSDs (rate ratio [RR], 0.66; 95% confidence interval [CI], 0.51-0.85; P = .001). Adjusting for maternal ethnicity and education, we observed that for every 1-g/dL increase in mean maternal hemoglobin level, the rate of SSDs decreased by 27% (adjusted RR, 0.73; 95% CI, 0.55-0.96; P = .02). The frailty model produced similar estimates (RR, 0.67; 95% CI, 0.51-0.87; P = .004) (adjusted RR, 0.75; 95% CI, 0.56-1.00; P = .049).
Using discrete categories of hemoglobin concentration (Table 2), we observed that as hemoglobin level decreased, the SSD rate increased. A maternal hemoglobin level of 10 g/dL or less was associated with a 5-fold increase in the SSD rate (RR, 5.17; 95% CI, 2.05-13.04; P < .001); the effect declined slightly following adjustment for maternal ethnicity and education (adjusted RR, 3.73; 95% CI, 1.41-9.81; P = .008). Moreover, an almost 8-fold increased rate for SSDs was observed for hemoglobin levels less than 9.0 g/dL, although its corresponding 95% CI was wide because only 2 offspring with SSDs had mothers with hemoglobin levels less than 9.0 g/dL (adjusted RR, 7.82; 95% CI, 1.64-37.27; P = .01). Similar estimates were produced by the frailty model; a hemoglobin level of less than 10 g/dL was associated with a greater than 5-fold increased rate of SSDs (RR, 5.37; 95% CI, 1.77-16.34; P = .003) (adjusted RR, 3.59; 95% CI, 1.20-10.78; P = .02). In summary, a significant dose-dependent effect of maternal hemoglobin concentration on the rate of SSDs was observed.
To complement the hemoglobin findings, we examined the effect of clinically diagnosed maternal anemia during pregnancy on the SSD rate. Of 57 mothers with a clinical diagnosis of iron deficiency, 3 offspring were diagnosed with SSDs. In contrast, only 1 offspring among the 252 mothers identified with non–iron-deficient anemia developed SSDs. Adjusting for maternal ethnicity and education, iron-deficient anemia conferred an almost 5-fold increased rate of SSDs (adjusted RR, 4.78; 95% CI, 1.48-15.40; P = .009) compared with offspring whose mothers were not diagnosed with clinical anemia. No elevated rate of SSDs was observed for non–iron-deficient anemia (adjusted RR, 0.40; 95% CI, 0.06-2.89; P = .36).
To investigate the causal process by which maternal hemoglobin levels affected the SSD rate among offspring, we considered whether prematurity, low birth weight, and small-for-gestational-age status mediated the association between low maternal hemoglobin level and SSDs. Offspring whose mothers had low hemoglobin levels had significantly higher rates of prematurity (P < .001) and marginally higher rates of small-for-gestational-age status, but not of low birth weight compared with the offspring whose mothers had adequate hemoglobin levels. When each of these posited mediators was entered individually into the Cox model, there was no alteration in the hemoglobin coefficient, thereby providing no evidence of mediation. We also examined the effect of hypoxia, assessed as described in the "Data Analysis" subsection of the "Methods" section. We found that although the prevalence of hypoxia increased as hemoglobin decreased (P = .053), hypoxia was not associated with SSDs and did not alter the regression coefficient for hemoglobin concentration.
Mean maternal hemoglobin concentration decreased from the first to the second trimester (Table 3). During the third trimester, the mean hemoglobin concentration of mothers of offspring without SSDs slightly increased, whereas the mean hemoglobin concentration of mothers of offspring with SSDs continued to decline. Using trimester-specific hemoglobin level classified as a continuous variable, we found an inverse association between increasing concentration of third-trimester hemoglobin and SSDs (adjusted RR, 0.74; 95% CI, 0.57-0.95; P = .02). No association was apparent between SSDs and either first-trimester (adjusted RR, 1.38; 95% CI, 0.89-2.1; P = .15) or second trimester hemoglobin concentration (0.88; 0.66-1.17; P = .31). When hemoglobin was analyzed as a categorical variable, a 3-fold, statistically significant increased rate of SSDs with a maternal hemoglobin concentration of 10.0 g/dL or less was observed during the second and third trimester, but we detected no evidence of a first-trimester effect.
We stratified the data by sex of the offspring to explore the hypothesis that sex might modify the effect of low hemoglobin level on the rate of SSDs. Among women, 6 of 20 participants with SSDs had mothers with hemoglobin levels of 10.0 g/dL or less. The female adjusted RR for a hemoglobin level of 10.0 g/dL or less was 6.70 (95% CI, 1.54-29.03; P = .01). Among men, a weaker effect for a maternal hemoglobin level of 10.0 g/dL or less was observed (adjusted RR, 2.06; 95% CI, 0.49-8.64; P = .32), reflecting the occurrence of maternal hemoglobin levels of 10.0 g/dL or less in only 3 of 37 men with SSDs.
Intrauterine exposure to low maternal hemoglobin levels was associated with a nearly 4-fold statistically significant increased rate of SSDs among offspring. A dose-response effect was observed; the SSD rate increased as maternal hemoglobin concentration decreased. Adjusting for potential confounders, including measures of parental socioeconomic status, did not appreciably alter the magnitude or significance of the hemoglobin effect. The validity of the finding is enhanced by 4 factors: (1) the offspring were drawn from a continuously monitored birth cohort; (2) maternal hemoglobin data were prospectively acquired; (3) a significant association between the clinical diagnoses of maternal iron deficiency anemia and SSDs was demonstrated, indicating internal consistency; and (4) two statistical approaches (frailty and Cox model) produced similar results.
This study is the first, to our knowledge, to specifically investigate the association between maternal hemoglobin levels and SSDs among offspring. A previous publication that examined many prenatal and obstetric events reported an odds ratio of 1.80 (95% CI, 0.8-3.8) for maternal anemia (defined as a hemoglobin level of <10.5 g/dL in the second trimester).37 Our results are compatible with past findings demonstrating that intrauterine exposure to famine in the Netherlands from 1944 through 1945 and in China from 1959 through 1961 were each associated with an increased risk of schizophrenia among offspring, although iron would have been only 1 of many nutrients that were deficient.9,10
We considered whether genetic loading for schizophrenia in mothers could nullify the association because women with schizophrenia often have suboptimal health behaviors, including nutritional intake.38 Conceivably, low hemoglobin levels resulting from poor iron intake might be a marker for mental illness. Therefore, the increased rate of SSDs among offspring might partly result from maternal genetic transmission of susceptibility for the disorder and not from low hemoglobin levels. Although we found that maternal mental conditions were marginally associated with SSDs (adjusted RR, 1.84; 95% CI, 0.90-3.74; P = .09), this effect was independent of low hemoglobin levels, as evidenced by no appreciable change in the hemoglobin coefficient following adjustment for this variable. We also explored whether low hemoglobin level was a surrogate for inadequate antenatal care. The addition of timing of initiation of prenatal care, the number and timing of the visits, and the number of hemoglobin assessments to the Cox model had no effect on the magnitude or the statistical significance of the hemoglobin estimate (results available from the corresponding author). Therefore, we found no evidence that either maternal mental illness or level of prenatal care confounded the association between low maternal hemoglobin level and SSDs.
Because SSD is an etiologically heterogeneous syndrome, several mechanisms associated with maternal iron deficiency might be involved in its pathogenesis. We posit 2 main classes of causal mechanisms. The first class involves the direct effect of iron on brain development. Iron is necessary for a plethora of metabolic processes involved in the development of brain structures and functions associated with SSDs.39 Iron is a coenzyme of dopamine synthesis; its deficiency can alter dopamine receptor density and activity,16 which has long been implicated in the pathophysiological mechanisms of schizophrenia. Iron is also an essential cofactor for lipid and cholesterol synthesis, integral to normal brain development; brains of persons with schizophrenia have profound myelin and oligodendrocyte abnormalities.15 Moreover, inadequate energy metabolism can ensue during times of iron deficiency, especially in rapidly developing brain structures.39 Support for these mechanisms is provided by preclinical studies, which found that myelin deficits persisted despite restoration of iron status at weaning40,41 and that perinatal iron deficiency altered the timing and the expression of critical genes relevant to hippocampal development.42 The second class comprises indirect pathways involving pregnancy and obstetric complications. Iron deficiency, the most common cause of maternal anemia, is a risk factor for poor pregnancy outcomes,2,12 including low birth weight, prematurity, and small-for-gestational-age status. However, our data provided no evidence of mediation by these 3 adverse outcomes of maternal anemia, thereby not substantiating the indirect mechanisms. In conclusion, although we cannot disregard the possibility that low maternal hemoglobin levels may merely be a marker for an independent schizophrenia risk factor, our results, in conjunction with strong biological plausibility for this exposure in the origins of neurodevelopmental perturbations, favor the contribution of low maternal hemoglobin level to the pathogenesis of schizophrenia.
The effect of maternal hemoglobin level on SSDs varied by trimester: second- and third-trimester exposure to low hemoglobin levels, but not exposure during the first trimester, were each associated with an increased rate of SSDs. This may reflect physiological fluctuations of maternal and fetal iron demands throughout pregnancy; iron requirements in the first trimester are less than those before pregnancy and rapidly increase in the second and third trimesters to support the expansion of maternal and fetal blood supply and fetal development. The increased demand for iron as pregnancy advances suggests that the fetus may be more susceptible to the effects of maternal iron deficiency during later gestation rather than earlier. Although the Dutch famine study found that the increased schizophrenia risk from nutritional deficiency was limited to first-trimester exposure,9 these pregnant women experienced protein-calorie malnutrition, micronutrient deficiencies, and the psychological stress of war and/or famine, unlike the relatively healthy pregnant women in the CHDS. The trimester-specific results, however, must be viewed with caution because statistical power for the first-trimester analysis was limited by a small sample size of 19 cases of SSDs.
Because sex differences in age of onset, symptom severity, and clinical course have been well documented in the schizophrenia literature,43 we explored whether the sex of the offspring modified the effect of low maternal hemoglobin levels on the rate of SSDs. We found some modest suggestion of a sex difference, such that a maternal hemoglobin level of 10.0 g/dL or less had a more deleterious effect on female than male offspring. Preclinical studies of iron deficiency provide support for this finding; brain iron and regional dopamine deficits persisted in female offspring but not in their male counterparts, despite restoration of iron status at weaning.40,41 Moreover, preclinical and human studies revealed sex differences in the turnover rates44 and gene expression of oligodendrocytes,44,45 the predominant iron-containing cells in the brain, as well as in erythropoietic activity.46 The role of chance cannot be excluded because the numbers of iron-deficient male and female offspring diagnosed with SSDs were relatively small.
One limitation of this study is the modest number of offspring diagnosed with SSDs. This produced relatively broad CIs and limited our ability to comprehensively evaluate the effects of gestational timing and sex of the offspring on the hemoglobin-SSD association.
We also considered the potential bias introduced by the exclusion of 11.8% of the offspring whose mothers lacked hemoglobin data. Mothers with missing data were more likely to be white, to have more prior live births, to have experienced hypoxic complications, and to have smoked during pregnancy and were less likely to attend college; their offspring were less likely to have been premature and small for their gestational age. Thus, no clear pattern emerged, suggesting that the hemoglobin data were missing approximately at random. Most important, the SSD rate was not correlated with missing hemoglobin data (P = .65) and the insertion of a missing-value indicator into the Cox regression model did not alter the effect size and statistical significance for maternal hemoglobin level (adjusted RR, 1.40; 95% CI, 0.68-2.91; P = .36).
A further limitation concerns the use of maternal hemoglobin levels as a marker for fetal iron deficiency. Although cord blood would have provided a more direct indicator of iron deficiency and fetal hypoxia, we did not have cord serum available. However, previous studies have shown a strong association between maternal and cord serum iron levels at delivery,47,48 indicating that supply of this nutrient to the fetus is reduced in mothers with iron deficiency.49 Moreover, infants born to iron-deficient women were at risk for iron deficiency during the first year of life, although the infants exhibited normal iron endowment at birth.50,51
To our knowledge, this is the first study to report a significant association between low maternal hemoglobin levels and SSDs among offspring. A postulated etiologic role of maternal iron deficiency is consistent with a large body of research, suggesting that perinatal iron deficiency has long-lasting neural and behavioral effects. A multitude of genetic and environmental factors govern brain development, many of which are involved in pathways that affect the delivery of oxygen and nutrients to the fetus. Maternal iron deficiency, assessed via maternal hemoglobin concentration, may disrupt these essential pathways and other iron-dependent processes involving dopaminergic neurotransmission, myelination, and energy metabolism. Disturbances of these pathways during critical periods of fetal development might heighten the susceptibility to schizophrenia in adulthood. Therefore, further investigation in independent samples is warranted.
Correspondence: Alan S. Brown, MD, MPH, New York State Psychiatric Institute, 1051 Riverside Dr, Unit 23, New York, NY 10032 (email@example.com).
Submitted for Publication: October 31, 2007; final revision received April 18, 2008; accepted May 9, 2008.
Author Contributions: Drs Insel, Susser, and Brown had full access to all the data in the study and Drs Schaefer and McKeague had partial access to the data in the study. All authors take full responsibility for the integrity of the data and the accuracy of the data analysis.
Financial Disclosure: None reported.
Funding/Support: This study was supported by grants 1R01MH 60249 and 1K02MH65422 (Dr Brown) from the National Institute of Mental Health, the Frontier Fund of Columbia University, and grants N01HD13334 and N01HD63258 from The Eunice Kennedy Shriver Institute of Child Health and Human Development.
Previous Presentations: This study was presented in part at the Annual Meeting of the Society of Biological Psychiatry; May 17, 2007; San Diego, California.
Additional Contributions: Barbara A. Cohn, PhD, Michaeline Bresnahan, PhD, and Justin Penner, MA, contributed to the study.


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Master of Illusions on NatGeo Program
When Arthur Shapiro appears in the April 29 episode of National Geographic Channel's Brain Games, expect to see some mind-blowing revelations about illusions.
After all, when it comes to entries for the annual Best Illusion of the Year Contest, sponsored by the Neural Correlate Society (a nonprofit organization that promotes scientific research and education in the neural correlates of cognition), nobody else comes close.
This year again, Shapiro is in the running with the finalists—after 11 top-10 placements in past years, including two first-place illusions, one second-place illusion, and two third-place illusions. He'll present his new illusion May 13 at the Neural Correlate Society's competition in Naples, Florida.
No wonder NatGeo flew the AU psychology professor out to Circus Circus in Las Vegas to film him discussing the workings of the brain. "What the brain is always trying to do is figure out how things have changed," Shapiro says in the interview. "And motion is an exquisite way of figuring [that] out."
You can see some of Shapiro's illusions here, but the important point to know is that the illusions are created not to dazzle viewers but to illustrate a point about visual science.
After returning from a sabbatical at the University of Cambridge in 2001, Shapiro began working on visual science problems whose solution "expressed themselves in terms of an illusion, something that was compelling to look at." Coincidentally, this was about the time that the Best Illusion of the Year contest began.
By participating in the contest, Shapiro, who has been publishing research on vision science since 1992 and is currently editing The Oxford Compendium of Visual Illusions, could suddenly potentially reach a much greater number of people than the small coterie of experts that academic specialists tend to attract.
The Neural Correlate Society contest, where attendees vote for the best illusion, typically draws 700 or more people, and the best illusions website has millions of visitors. Brain Games will give his research further exposure. The launch of the National Geographic Channel series, on April 22, had 1.5 million viewers, the highest-rated series beginning in the channel's history.
"When you do basic vision science, you take one dimension, you manipulate it and you see how things change. You figure out mathematical models that can fit your data; you test specific hypotheses. It's really good, it's really important, I love doing these types of experiments," he says with a smile. "But when you do this type of research, you may get the respect of 30 fellow researchers. When I express my work in terms of visual illusions, I am able to convey similar ideas about vision and the brain, but thousands of people are interested."
The Best Illusion of the Year contest allows Shapiro and others to pose as compelling illusions problems such as how the visual system organizes objects and the importance of color.
"Suddenly you have a way of approaching vision science that appeals to a larger audience and can pull people into the field and convince them why these questions are worth studying and why you don't have easy answers to them."
What does Shapiro think makes a good illusion?
"Everyone's going to have a different take on that. If you're an artist, a good illusion may be one with aesthetic appeal. From my point of view, a good illusion is one that is instructive about the visual system, says something fundamental about how our brain is processing the world, and is compelling enough that there's no obvious answer."
Just a few weeks before his presentation, Shapiro's understandably a little cagey about this year's top-10 finalist illusion, which he collaborated on with AU education student Alex Rose-Henig. The illusion is called "Tusi or not Tusi," a reference to the thirteenth-century Persian scholar Nasir al-Din al-Tusi. (You can check out a quick biography here, and an animation of a Tusi-couple, which relates to Shapiro's new illusion, here.)
"The brain is really extraordinary. Illusions allow you to ponder how the brain works." Shapiro adds with a laugh, "It's mind-boggling."
The third show in the 12-part Brain Games series, "Motion Commotion," can be seen at 9 p.m. Monday, April 29, on the National Geographic Channel.


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Transfer Students
If you are transferring in credits from another college, you should not schedule a testing appointment until you receive official notification from the Office of Admissions with your transfer evaluation. You may be exempt from all or part of the placement testing.
Request from your institution an official transcript be sent to:
Bristol Community College
Office of Admissions
777 Elsbree Street
Fall River, MA 02720


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Iraqi Prime Minister Nuri al-Maliki said on Monday that an Iraqi intelligence forces had located and killed al Qaeda's leader in Iraq, Abu Ayyub al-Masri. According to Reuters, Maliki said the forces also killed Abu Omar al-Baghdadi, the leader of al Qaeda's local affiliate, the Islamic State of Iraq, in an operation backed by U.S. forces.
Maliki said Masri, also known as Abu Hamza al-Muhajir and thought to be an Egyptian, and Baghdadi died in Thar-Thar, a rural area 80 km west of Baghdad. The operation took place over the last couple of days.
"The attack was carried out by ground forces, which surrounded the house, and also through the use of missiles," Maliki told a news conference. "U.S. forces also participated." He said the house was destroyed and the two bodies were found in a hole in the ground inside in which they had been hiding.
[1] http://www.albawaba.com


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I Left My Heart In Zalec Women T-Shirt
Product Color:
Product information
The Women T-Shirt I Left My Heart In Zalec is our old-school favorite. Perfect for use in sports where you are bound to sweat and need wide range of movement. Short sleeves to let cool air in on those hot and sunny days. Also great for every day use and to show everyone your love for I Left My Heart In Zalec.
This product is made of the finest combed jersey which feels soothing against your skin. Shoulder-to-shoulder taping guarantees long life and a good fit. Zalec Women T-Shirt is available in 17 different color options. Heather Grey: 80% Cotton/20% Polyester. Remaining Colors: 100% Cotton.
Sku: TC2BL0000039481313133665RE0000000


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For those of us who are a size 12 and up, finding style tools can be a challenge. For those of us who are a 12 or greater for sizing and have a lack of fashion sense, this is an obstacle. I was recently introduced to Sonsi's Style Your Way-Outfitting Tool and it's very, very fun. (I can see it also getting very, very expensive, very very quickly, because you just keep finding adorable this to go with that and oh if I got those shoes and this bag I could wear this top to XYZ and to 123…)
Finally! There is a tool that makes outfitting your life easy. Introducing our premiere Style Your Way – Outfitting Tool: Create and save a personal online wardrobe, share your individually styled outfits with friends, and tap into the other's looks in the Sonsi community for inspiration. Consider it your new destination for personal style inspiration. With access to thousands of styles, it's truly fashion at your fingertips. Whether you are out and about, playing with the kids, or relaxing at home, this unique outfitting experience makes it easy to plan, coordinate, and accessorize your outfits before bringing them home.
Simply by starting a free account with Sonsi and creating an outfit, then sharing it with a friend (don't worry, they just send items, not sizes chosen- although I was able to choose items without needing to give sizesz), you will be entered to win a $200 Gift Card to Sonsi! I just did it, although I am not eligible to win because of this post- but I wanted to see just how simple it was to enter- and it really is. Plus, then you can show off your creation- this week (July 18-21) the theme for outfits to style is "Casual Friday" and here's my take on it:
Fun and also comfortable (which is how I like to be). I think I could even be brave enough to wear this, especially by simply printing out or ordering these items right from home at once!!
(I would like to know if the shoes pictured with the capris would look good or ridiculous on my wide feet- because I think they are super cute!)
Nearly 60 percent of American women are size 12 and up, and are no longer content to be the silent majority. Sonsi's new upgrade sets the industry bar for personalized online customer service and social engagement to a new high. Sonsi's new Style Your Way outfitting tool takes shopping online for clothes to a new interactive level by letting plus-size women create a personal online wardrobe, share their individually styled outfits with friends and other women for feedback, and tap into each others' inspiration. Once they find an outfit they love, women can purchase their styled outfits directly from the tool.
Sonsi®, the award-winning online shopping and social destination designed exclusively for plus size women, is injecting the dog days of summer with a much-needed dose of retail therapy. Sonsi has launched the Style Your Way™ Summer Challenge, inviting women to put their fashion styling talents to the test. Starting today, full figured women coast to coast will have an opportunity to use Sonsi's new, innovative outfitting tool to create head-to-toe summer ensembles for the chance to win a $200 gift card to put towards an outfit of their choice.
Celebrity stylist Susan Moses, who has worked with such full figured A-listers as Mo'Nique, Kathy Bates, Emme, Wynonna Judd, Chaka Khan, Angie Stone, Gabourey Sidibe and Jill Scott, will select one winning outfit per week for four weekly summer style challenge categories: Casual Friday, Girls' Night Out, Day at the Beach, and Summer into Fall. Participants can select from thousands of fashion must-haves from among Sonsi's more than 380 apparel and accessories brands.
Citing Sonsi's Style Your Way outfitting tool as a breakthrough that puts the glamorous world of fashion styling at every curvy woman's fingertips, Sonsi General Manager and Publisher Jill Hutchison is certain the new tool will help full figured women discover just how much fun social shopping can be. "We're challenging plus size women to discover their inner stylist this summer," says Hutchison, "and with this season's lively colors, flattering silhouettes and fun accessories, we know they're going to have a blast! But, the best part is, they can create as many outfits as they want for each of the style categories, giving their chances of winning a major boost."
Beginning July 18th and running through August 12th, women can submit their Style Your Way Summer Challenge entries until Thursday at midnight for each of the four consecutive weeks. The weekly winner will be announced each Friday (July 22, July 29, August 5 and August 12) and each weekly winning outfit will be featured on Sonsi. Visit the www.Sonsi.com/SYWChallenge for official rules
Sonsi's new Style Your Way tool not only takes the guesswork out of finding the perfect outfit, it also makes it possible for traditionally underserved plus-size women to unleash their inner fashionistas. "As a professional stylist, nothing is more fun for me than finding the perfect outfits for my clients," says Moses. "Now, fashionistas everywhere will have an entree into my exciting world of fashion styling, thanks to Sonsi's Style Your Way Summer Challenge."
Go ahead and Style Your Way- and feel free to share with me, I'm always up for new inspirations!! Share them to email@example.com.
This post was made possible by Mom Spark Media. Thoughts are my own.
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text: Georgia-Pacific Professional has launched an updated environmental section of its Web site that outlines the company's sustainability initiatives. The site, http://www.gppro.com/sustainability, highlights the company's Green by Design platform, a designation for products that are environmentally and socially responsible as well as economically sound. The refreshed site makes it easy to learn facts about the sustainable


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"Could GMOs end up doing devastating damage to our species in the years to come? Genetically engineering food crops such as corn, potatoes, tomatoes, wheat, and others are not being adequately tested to insure they will not harm the humans and animals that consume them. Scientists believe specific dangers of consuming GMOs include organ failure, cancer, infertility, and other illnesses in lab animals. The scientists who speak out against such dangers are reporting severe persecution." Airborne cross pollination is also occurring and non-GMO crops are being contaminated.
Read more at http://wakeup-world.com/2011/11/10/monsantos-crimes-against-humanity/ .


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I have a new Frame by Frame video out today, directed and edited by Curt Bright, on the 1945 films Les Dames du Bois de Boulogne by Robert Bresson.
I have blogged about this film before; as I wrote then, "one of Robert Bresson's most incandescent works, this early film also marks the teaming of two of France's most personal and idiosyncratic artists: Robert Bresson and Jean Cocteau. Cocteau (whose 1949 film Orpheus [Orphée] mesmerized post-World War II audiences), in addition to his numerous other accomplishments, wrote the dialogue for Les Dames du Bois de Boulogne, loosely based on Denis Diderot's short story Jacques le Fataliste et Son Maître. Elina Labourdette plays Agnès, a young woman who has been forced into a life of prostitution in wartime Vichy, France, in order to support herself and her ailing mother (Lucienne Bogaert).
At the same time, Hélène (the serpentine Maria Casarés) is breaking up with her longtime lover, Jean (Paul Bernard), and, feeling jilted by him, concocts an elaborate plot for revenge. Contacting Agnès and her mother, Hélène offers to take over their debts, move them out of the brothel they call home, and set them up in a sleek, modern apartment, with no strings attached. We discover too late Hélène's true motives; she is doing all of this so that Jean will 'accidentally' meet Agnès, fall in love with her, marry her, and then become the subject of public ridicule because of Agnès's past. All of this goes off with clockwork precision, but Jean, when confronted with the monstrousness of Hélène's treachery, shakes off his bourgeois prudishness, embraces Agnès despite her fall from grace, and the film ends on a note of hope and Bressonian redemption. This film never fails to stun me with its sheer, vibrant beauty and psychological insight; I return to it again and again, and it never disappoints."
I wrote an essay on the film in Senses of Cinema 46; you can read it here.


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I'm going to say this before it starts. If you are somehow happy about this, and feel like this is "justice." Then you're life is pitiful and you are likely using your dislike for Apple (or any other company unreasonably) as a cover up for how your life never turned out the way you wanted it to.
Lol I agree. So funny. Karma can be a down right horrid b*tch!
BTW Apple will win this case.
Based on the fact that Foxconn is a company in China, they will throw their weight around. So will all the companies that makes parts for iDevices, they will join in this fight if it gets that far.
Also the fact that all the previous released pictures of these parts showed that Apple came out with it first, it is documented by the date of the pictures. Basically all those "spy pictures" will save Apple from this lawsuit.
If Apple does end up getting sued, the whole world if going to look at the Chinese courts as a joke. No one in their right mind would believe this.
Besides, after looking at the chinese design, I'm pretty sure Apple could sue them. The buttons, camera placement, even the whole friggen outer body of this "GooPhone" is almost a replica of the iPhone 4 model. Only differences I see is the extended body of the device and the android icon on the back. I'm almost positive that Apple's lawyers could wipe these guys off the face of the earth.
How is karma a b**** in this situation ? Samsung was found to have blatantly copied parts of Apples intellectual process. Apple was award well over 1 billion in damages, and can potentially block Samsung products from entering the market. Samsung was at fault and Apple defended itself.
If you made an invention and someone copied it, you would sue right ?
Also learn something about patents and patent laws before commenting. This type of behavior would never work in an American court, and will not work in the Chinese court either
I hope they lose it's time they ate some crow instead of bullying everyone.
Ell then isn't apple gonna sue them for their goopphone looking like an iPhone 4s?