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Why We Love Ed Whitlock
Sure, he holds just about every old-age running record in the books, including being the only person to run a sub-3 hour marathon in his 70s – he did it twice – and yesterday setting a new mark for 85-year-olds by completing the Toronto Marathon in 3:56:33.
But the reason Ed Whitlock is so great is because he's funny.
Here are a few quotes Whitlock uttered yesterday and today:
+ "My legs are shot today. I seemed to be OK yesterday after the marathon, walking around and that sort of thing, but rigor mortis has set in now."
+ (about his beat-up running shoes) "I wear them well past their due date."
+ (asked about the age of his singlet) "I don't know. (long pause.) Maybe 30 years? Older than my shoes."
+ " I ran too fast, really, at the beginning of the race. I swear I'll never do that and I always do it."
Here's Ed being interviewed at the finish by Canadian Running magazine.


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DoubleTree Club by Hilton Hotel Boston Bayside3
240 Mt. Vernon Street, Boston, 02125
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About the hotel
The DoubleTree Club by Hilton Hotel Boston Bayside is set near Boston's picturesque HarborWalk and offers all of the comforts of home, making us the preferred choice for Hotels in Boston near the Airport. Our hotel offers convenient access to I-93 and the Boston 'T' subway, making it easy to take a short 15-minute drive to Logan International (BOS) Airport or a quick T ride into downtown Boston. Complimentary shuttle service is also available to Logan Airport and downtown Boston.
Enjoy warm cookies on arrival. Relax in a guest room at DoubleTree Club by Hilton Hotel Boston. Spacious suites on the 6th floor furnish panoramic views of Boston Harbor, and pull-out sofa beds in the parlors offer ideal options for families. Stay connected in your guest room and the hotel lobby with WiFi access. Enjoy popular brands at the Marketplace Café and Take Away, where we offer Starbucks® coffee and premium deli meats on a menu of light bites, breakfast and burgers served all day. Our business center offers complimentary WiFi and printing. Host a briefing in an elegant boardroom. We're on the doorstep of UMass Boston, and corporations such as Gillette, Procter & Gamble and KPMG. Maintain any exercise program at this Boston hotel with our on-site fitness room, or use your guest key to gain complimentary access to the nearby UMass Boston Beacon Fitness Center. Stroll or jog along the scenic Harbor Walk and enjoy some of Boston's greenery and amazing ocean views.
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|Next book >>
Angel's Flight(1960)
A novel by Lou Cameron
The first time Ben Parker meets Johnny Angel, Ben is blowing bass with Daddy Halloway and the Hot Babies. Angel angles into the band by acing out the drummer. Parker's got a bad feeling about Angel, but has to hire him anyway. Johnny Angel, raised from the school of hard knocks, doesn't let anyone or any scruple get in his way. First, he steals Daddy's band, then he steals his daughter, Blanche. Then WWII happens - Parker serves, but Johnny cops a gay plea and gets out. When Parker gets back he hooks up with a talented but tubercular composer named Con Conners. And a sultry singer, Ginger. But Johnny Angel is right behind him, ready to do whatever it takes to climb to the top on someone else's talent, be it beg, borrow or steal... even kill. This is the ruthless story of the music business told from the inside, starting from the days of swing jazz to bebop, all the way to rock'n'roll.
Similar Books by other authors...
Used availability for Lou Cameron's Angel's Flight
See all available used copies of this book at Abebooks UK or Abebooks US
Paperback Editions
November 2016 : USA Mass Market Paperback
|Title: Angel's Flight (Black Gat Books)
Author(s): Lou Cameron
ISBN: 1-944520-18-X / 978-1-944520-18-2 (USA edition)
Publisher: Stark House Press
Availability: Amazon Amazon UK Amazon CA
Genre Pages


text:
How do i know when i'm in love? Does love for my gf keep growing? can it be more than 3 months without falling in love? any tips? thanks :)
Have an opinion?
Be the first girl to share an opinion
and earn 1 more Xper point!
Well... love is more of an idea, so it would very much depend on your definition of that idea.
Whenever you see her and your heart skips a beat. When all you think about is her. When she is the first and last person you think of. That is what love feels like. If you feel that then you're in love.
You cannot undo this action. The opinion owner is going to be notified and earn 7 XPER points.


text:
We're closing in on September 29, the date when Dave Parker's modern slasher flick The Hills Run Red bows on DVD, courtesy of Warner Home Video. To get you in the mood for some Babyface inspired carnage, we've gotten our hands on several clips including a "vintage" trailer!
A film fanatic's obsession with finding a complete print of a classic slasher movie leads him and two friends into the backwoods where the film was shot. They realize too late that filming never ended — the movie's star, the deformed, murderous BABY FACE, and his twisted family continue to film and kill unsuspecting victims. Now our heroes must survive the nightmarish onslaught or become part of the movie forever.
Look for the film on DVD from Warner Premiere soon! Pre-order below!
Got news? Click here to submit it!
Run red in the Dread Central forums?


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Youth Panel
The CREST youth panel
The British Science Association (BSA) believes that it is important to involve young people in decision making and give them a voice on the issues important to them.
Our CREST Youth Panel is a group of CREST Award holders aged 13-19 who meet twice a year at large science events across the UK to input into the CREST scheme's projects and those of our partners.
Members of the Youth Panel receive the following benefits during their time serving on the group:
- The title of CREST Youth Advisor
- A formal letter of thanks from the BSA's Chief Executive at the end of the year
- The opportunity to attend the British Science Festival
- The chance to attend influential CREST meetings and address stakeholders
- Membership of the CREST Alumni Network
- Membership of the BSA
- The opportunity to represent the BSA at events around the country
- The opportunity to participate in training sessions and develop new skills
- Support with travel costs to and from meetings (for them and an accompanying adult if under 18)
Students are also provided with an environment in which they can meet and speak freely about STEM experiences and opportunities and learn new ways to discuss and review thoughts, facilitated by British Science Association staff and influential partners.
How to join
To apply, you must be 13 – 18 years old, still at school or college and have completed a CREST Award.
If you are interested in applying, you should read our participant information, make sure you have permission from your parents/guardian or teacher, and fill in the nomination form.
Current CREST Youth Panel Members (2015/16)
Charlie Song
Aine Maree Duffy
Sorcha Grant
Naomi Genitoni-Smith
Abeera Qayyum
Robert Saunt
Phillippa Nightingale
Katja Stout
Scarlett Li-Williams
Anisha Mehta
Bethany Evans
Dilani Selanathan
Min Ju Lee
Jamie Kettle
Imogen White
Noel Rodrigo
Dilani Selanathan
George Kerr
Henry Kerr
Tara Crees
Aisling O'Rourke
Saheefa Ishaq
Gwenllian Roberts
Navyaa Mathur
Emily White-Musgrave
Sanna Masood
Klara Prela
Floriane Fidegnon-Edeh
Sally West
Shannon-Louise Taylor
Mythiri Sutharson
Kate Quillin
Lilian (Lily) Limmer
Lauren Parmenter
Samantha Farmer
Lauren May Banfield
Anabelle Simmonds
Sarmpari Uthayakumar
Mhairi McCann
Amelia Bryant
Sankha Gamage
Elle Laughran
Rianna Ghosal
Charlie Webster
Jade Lucas
Bethany Trott
Aarushi Wuppalapati
Florence Bradshaw
Alanna Wyncoll
Catrin Parry
Caija Addai
Apoorva Verma
Chris Conheeny
Tapton School
My Stem Club were assessed for their Bronze Award and had so much pride when they got it! The assessor was impressed at the skills they had learned in communication and facing challenges. The whole experience is so valuable!
CREST teacher
It's helped the self-esteem for some low achievers for whom it is their major achievement in school.


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DOVER-FOXCROFT | As many Maine high school baseball teams scramble to find enough pitching depth to face the new season which begins in less than two weeks, Foxcroft Academy coach Mark Chevalier has few such worries. All three of his team's top starters are back this spring, just as …
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Area Rugs Bedroom
Wednesday, February 15th, 2017
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Saturday, February 25th, 2017
Wednesday, March 8th, 2017


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Professional Global Leader | Trusted Advisor
- Ambidextrous leader, who can leverage not only logic, financial acumen & attention to detail but also judgement, intuition & imagination, to drive change and innovation - PMI-certified Project Management Professional (PMP) - Critical Chain Project Management (Concerto) Practitioner - Almost a decade of consulting experience with Accenture, Infosys for clients such as BHP Billiton, National Australia Bank, Telstra, ANZ Bank, Australian Government, Wesfarmers, Commonwealth Bank of Australia - Can be defined using 4 Cs: Committed professional with Commercial mindset who leverages strong Client relationship skills to drive Change - End-to-end program management & delivery experience handling $15-25m transformation programs having teams of size ~10-100 - Capital Markets Industry Certifications from Chartered Institute for Securities & Investment, New York Institute of Finance, NSE & BSE - Trading & Portfolio Management experience with ICICI Securities - MBA (Finance) & Computer EngineerMore
- Leadership role (~45+ indirect reports) reporting to Executive General Manager (who reported to CEO) - I led the delivery arm of the business unit (EPMO) responsible for delivering change across all clients
- Profit & Loss (P&L) accountability for $25m+ portfolio
- Managed the master portfolio spread across all clients with the mandate to enhance customer satisfaction, revenue generation and profitability ;
- Leading a team of 20+ project / program managers & 25+ business analysts, was responsible for providing strategic direction and leveraging skills / expertise from all business units to ensure on-time, on-budget & quality delivery of a portfolio of ~50+ projects / programs
- Responsible for strategic client engagement, pipeline prioritisation / conversion / management, revenue generation and practice development
- Mandated with capability development for delivering change, screened 150+ applicants & conducted 50+ interviews to ramp-up the team from ~15 to 45+ ;
- Helped realise cost savings of $250k+ in less than 100 days of joining
- Increased staff engagement & morale as demonstrated by staff engagement surveys & balanced scorecard metrics
- Improved on-time / on-budget metrics to 90%+ and enhanced quality as well as customer satisfaction levels ;
- Leveraged Accenture Delivery Methods / Tools and various client project methodologies / frameworks for managing / executing large transformation programs (team size ~ 75; value ~$10-20m; executed from sites across multiple geographies) as well as for creating & implementing various program management / financial management models / dashboards to track / control programs and for steering committee reporting
- Helped BHP Billiton at various stages of its SAP transformation journey performing roles like Project Controller and Program Management Office (PMO) Lead across various sites in multiple continents. In Melbourne, program managed end-to-end implementation of multiple projects (~10) running in parallel each having various streams of work and respective leads reporting into me and another senior manager. Was responsible for managing the complex P&L / budgets for each project stream maintaining the critical balance between Accenture profitability, program risks / quality and client satisfaction. Role involved planning, workplan management, status reporting, financial tracking / reporting / control, risk / issue management, stakeholder management, benefits realization, cross-project interdependency management, deliverables management and various other day-to-day program management processes as well performing account management activities like resourcing, billing, relationship development and up-selling / cross-selling. Managed all commercials and vendor relations in cases leading to significant cost savings ( $100k) to the program and thus directly benefitting the client bottom-line
- As Program Lead, helped Wesfarmers in kicking-off a $17 million project from scratch and establishing planning, workplan management, status reporting, financial tracking / reporting / control, risk / issue management, and day-to-day project management & control. Managed the program team consisting of client permanent employees, client contractors and vendor. One of the key responsibilities was vendor management and the associated "time & material (T&M)" design phase within aggressive timelines & budget. At the time of rolling-off was successful in helping position the client to realise ~15% cost savings (AUD 100k+) for the design phase without compromising on quality and timelines (infact there was a potential of finishing 2 weeks earlier than planned). The client also had very peculiar internal finance processes and I was successful in not only self-learning them on-the-go but also kept the financial forecasts variance under the client threshold every month! Lastly, trained a senior project manager who was recruited for the role and seamlessly handed over the program to him within the defined transition period
- In Brisbane was responsible for setting-up Program Management Office from scratch for $15 million+ project of BHP Billiton having team size of ~50+. Role involved establishing planning, workplan management, status reporting, financial tracking / reporting / control, risk / issue management, deliverables management, developing program vision and various other day-to-day project management processes. Responsibilities included project management and control using Concerto (based on Critical Chain Project Management methodology) as well performing account management activities like resourcing and billing
- In Singapore and Perth, planned and estimated a strategic change (M&A) program (current as well as the next phase) using critical chain project management methodology. Involved in resourcing and mobilising the teams in Perth as well as Singapore. Responsibilities include project management and control using Concerto, financials management (including billing), status reporting, risk / issue management and stakeholder management. Managed financials / billing for multiple Iron Ore / Metallurgical Coal projects. Provided guidance / advice to other Project Controllers regarding planning based on critical chain project management using Concerto
- Helped National Australia Bank (NAB) develop a Direct (online) Trading Platform as well as a Private Wealth Management offering, primarily targeted at high net worth individuals. As Project Manager for Bank work stream, role involved coordinating amongst the various NAB / MLC teams, risk / issue management, stakeholder management, project monitoring / control to ensure timely delivery, within budget, of Online Trading / Private Wealth Management Solution
- Helped Commonwealth Bank of Australia (CBA) finalise requirements related to Wholesale / Retail Banking products as part of their Core Banking Modernisation project ;
- Conceptualized, promoted and incorporated this company offering financial and technological solutions
- Intermediary and investment consultant for retail, high net worth individuals and corporations helping them leverage the booming Indian economy by profitably participating in Indian Equity and Commodities Markets
- Analyst offering customised solutions based on the risk profile of each investor by planning a personalised asset allocation program and effectively utilising various available cash and derivative products including futures / options and related strategies like spreads, straddles, strangles, strategies based on Greeks etc.
- Developed financial models for option pricing, derivatives pay-off analysis, wealth management etc.
- Actively involved in foreign exchange trading as well as dealing in currency futures
- Currently focusing on developing strategies to leverage Contracts for Differences for maximizing profits
- Designed and implemented various end-to-end software solutions catering to the needs of financial services sector, primarily, mutual funds, investment banks, portfolio managers and stock brokers ;
- Intermediary and advisor to clients investing in Equity, Futures & Options and Derivatives Markets
- Role involved equities / derivatives / commodities sales, trading and portfolio management ;
- Functional Consultant for Eagle implementations and other asset management / portfolio accounting solutions such as Calypso
- Developed industrialised capabilities for implementing Calypso across investment management firms ;
- Biggest assignment involved implementation of a financial messaging solution for UBS Investment Bank, one of the largest financial institutions in London, United Kingdom
- Played an active role in the Project Management, Requirements Gathering, Analysis, Testing and Implementation stages of the project lifecycle
- Instrumental in setting up quality processes and tools for the entire program which can be leveraged across the complete software development life cycle
- Also acted as Offshore Test Manager and Offshore Project Manager for the same project
- Involved in pre-sales activities to global clients across verticals including Financial Services, Utilities, FMCG
- Spearheaded a research on financial networks including SWIFT, Euroclear, FEDNET, CHAPS, BACS etc. and thus contributed towards designing a generic financial messaging solution ;
- A Channel Finance project directly under the guidance of Head of Channel Finance, India
- The project aimed at identifying companies for channel finance opportunities and estimating the market size involved financial analysis of 7000 odd companies spanning 100 odd industries
- A comprehensive database giving the required financial details was created, which the bank would use to target the companies during the roll out of the product ;
- A Customer Relationship Management (CRM) project for Wholesale Banking
- The project aimed to identify an ideal model of CRM for wholesale banking & describe its implementation issues, develop an action plan to implement the model of CRM identified and study the existing systems (Corporate Banking, Phone Banking and Internet Banking) and give guidelines for achieving the CRM goals using the currently captured data
- Deliverables included a CRM model for financial services, Competitive Analysis Report, Gap Analysis Report and an action plan for ICICI Bank including a People-Process-Technology framework, Data Warehousing details and other implementation issues of an Enterprise-Wide solution
Highlights: POSITIONS HELD & PARTICIPATION ,More
Education and Qualifications
|MBA (Finance)
|Jamnalal Bajaj Institute of Management Studies
|Bachelor of Engineering - Computer
|Bachelor Degree
|Sardar Patel College of Engineering
|Higher School Certificate
|Senior Secondary Education (Yr12)
|Mithibai College
|Secondary School Certificate
|Junior Secondary Education (Yr10)
|St. Francis D'Assisi High School


text:
ScienceDaily (Nov. 16, 2009) — The constellation of Orion is a hotbed of massive star formation, most prominently in the Great Nebula that sits in Orion's sword. The glowing gas of the Nebula is powered by a group of young massive stars, but behind it is a cluster of younger stars and clumps of gas. Still gathering together under gravity's pull, these gas clumps will eventually ignite into stars.
The way that massive stars form remains mysterious, in part, because massive stars are rare and tend to spend their youth enshrouded by dust and gas hiding them from view.
"We know how these stars die, but not how they are born," said Lincoln Greenhill, a principal investigator of the study and part of a team comprising scientists from the Harvard-Smithsonian Center for Astrophysics (CfA) and the National Radio Astronomy Observatory (NRAO).
Unlike Hubble and other visible-light telescopes, radio telescopes can penetrate dusty veils around stars. The research astronomers studied a massive young protostar called Source I (pronounced "eye") at radio wavelengths, using the National Science Foundation's Very Long Baseline Array (VLBA) as a powerful "zoom lens."
The VLBA yielded even sharper images than the famous Hubble photos of "proplyds," or protoplanetary disks in Orion. The team observed Source I at monthly intervals over two years and then assembled the individual images into a time-lapse movie.
The VLBA detected thousands of silicon monoxide gas clouds called masers -- naturally occurring laser-like beacons often associated with star formation. Some masers were as close to the protostar as Jupiter is to our Sun, which is also a record. Many of the masers existed long enough for their motions to be tracked across the sky and along our line of sight, yielding their 3-d motions through space.
"In astronomy, it's rare to see changes over the course of a human lifetime. With this new movie, we can see changes over just a few months as gas clumps swarm around this young protostar," added Smithsonian astronomer and co-author Ciriaco Goddi.
The resulting movie reveals signs of a rotating accretion disk, where gas is swirling closer and closer to the protostar at the center. It also shows material flowing outward perpendicular to the disk in two large V's -- actually the edges of cone-shaped streams of gas. Such outflows foster star formation by carrying angular momentum away from the system.
Intriguingly, the outflow streams appear to curve as they leave the disk. "The bending path of these masers provides key evidence that magnetic fields may be influencing gas motions very close to the protostar," pointed out Claire Chandler of NRAO, a co-principal investigator of the study.
Source of image of the Great Nebula in Orion:http://www.freewebs.com/jezstar/imagesfromspace.htm
Credit & Copyright: Jon Christensen


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Losing weight for many people can be a real struggle. There are many reasons why this may be the case, but why does it sometimes seem so confusing and at times disheartening?
Is it all down to genetics? Why do some people seem to stay overweight no matter what they seem to try? Why do some lose weight only for it to all come back again and often even more than before?
Some conflicting information:
There are many theories banded about regarding the best way to lose weight and undoubtedly sometimes these work for some people. But there rarely seems to be universal success.
There are food temptations everywhere and it can seem like a constant battle to try to make headway. Some of the conflicting advice and information includes these opposites:
- Don't eat meat - Only eat meat.
- Don't eat fruit and veg - Just eat fruit and veg.
- Eat a big breakfast - Skip your breakfast.
- Eat lots of little meals - Eat just one big meal.
- Don't eat carbohydrates - Just eat carbohydrates.
- Eat a high protein diet - Eat a low protein diet...
No wonder people get confused.
The result of this confusion is that it can lead to a lack of action or being unsure which direction to take.
It's time to get back to basics
So is there any truth out there about weight loss? Is there anything that is fundamental?
The overall basic equation is something that in its purest form is hard to argue with. Here it is:
"Over time, if you eat more than your body needs, some will get stored as fat."
This can be reworded into a kind of weight loss equation like this:
On the face of it this seems to make good sense. In simple terms, if you eat more than you need, then over time your body weight will increase.
On the other side of the coin, if you eat a bit less than your body requires, then you will lose weight.
This is a very simplistic way of looking at it, but it does help to give us a firm foundation on which to build our understanding of how to achieve our ideal weight.
There are actually quite a few aspects to this simple equation above. Each of the three things leading to YOUR BODY WEIGHT can be controlled by you.
This includes the food types you eat, how much you eat, the amount of exercise you get and whether your body is properly using what it consumes and also whether your body eliminates waste properly. Numerous articles can be written about each part, and this is what we intend to publish in the coming months.
Don't forget that it can take time
One thing that many get frustrated by, is their speed of progress, or rather the lack of it.
Gaining excess weight can often take months, years or even longer. Often it just creeps up on a person over a long period of time.
Research suggests that people often lose patience with a diet or program if they don't see almost instantaneous results and herein lies a danger:
Trying to lose weight too quickly can backfire and you end up worse than before.
Steady progress is the best way
Rapid weight loss is not very healthy, and in many cases consists of mainly water loss and also some serious muscle mass loss. This can leave the body lacking in energy and you end up craving more food than before.
A person can't maintain this kind of starvation routine for very long and the body rebels, almost as a self defence mechanism, and you are left needing to eat even more food than before, just to get back to "normal".
It is vitally important that you maintain patience when trying to lose fat. It doesn't happen quickly unfortunately. This is how the human body has evolved over many thousands of years. It loses weight and gains weight fairly slowly over many weeks, months or years. So stick with it.
Just knowing it can take a while to lose weight can be a blessing or a curse. It may make some people decide not to bother or give up, but for others just knowing it can take a while, helps avoid some of the disappointment that can happen from time to time.
Is there a way to make things easier?
Yes there are things you can do to help make this journey easier. These include things you can physically do to better organise your day and the foods you eat and also mental things and attitude changes you can make.
We will be issuing further articles in due course about all these things which can help speed up your progress towards your goals. In the meantime here is some product and program news which may help:
Some further information which may assist you
The next product to be launched will be the new carbohydrate formula called GoodCarbs. It is a low glycemic food supplement and can be used by anyone including those people on a low carbohydrate diet.
There will also be a series of special reports available free to our newsletter subscribers which will deal will the real facts about carbs and help to cut through the clutter of lies and misinformation about carbohydrates.
It is possible that you can achieve a healthy body and at a good weight for you, no matter what that weight may be.
As part of our regular newsletter there will be examples of successful natural treatments to help you slim and achieve your ideal weight.
|Live demonstration: Our new weight
loss program will be released shortly. Here is a little demonstration.
Click here now.
| See:
daily comments
daily diet tips
before photo's
It is possible to achieve your ideal weight!
We have researched the latest ideas on weight-loss and we are currently producing our reports. It is expected that the first part of it will be available shortly.
Further parts will then be released regularly via our regular organic and health newsletter.
If you would like to register for this free newsletter please go to the free newsletter subscriber page - click here.
Become inspired and motivated
We often hear stories from people who had previously struggled. They told us that they thought they wouldn't be able to do it. They often said it was just their body type and that it is just the way it is.
They often said things like they were "big boned" or that it is all down to genetics...
We don't dispute that genes can play a part. Sure they can.
This applies to muscle growth for athletes and for excess weight in others. BUT genetics are a minor part of and reason for being overweight.
It is important to realise that it is possible to get weight under control and get a slimmer body. There are hundreds and possibly thousands of amazing before and after stories on youtube of people who have made dramatic changes.
You can go there and see for your self. Use these stories and body transformations to inspire you and to help you get motivated.
Building a positive attitude and developing your willpower is important and this will help you overcome any obstacles which my get in your way.
Make the decision to do something and create your own targets and goals. You Can Do It :-)


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Karachi: Organisers of the Sharjah Limited Overs tournament have refused to reschedule the four-nation event from April to March this year, infuriating Pakistan.
"We requested the Sharjah organisers to reschedule the tournament from April to late March which could have allowed us to rearrange our cancelled home series against New Zealand but they refused," Pakistan Cricket Board (PCB) chairman Lieutenant General Tauqir Zia told reporters. The PCB had wanted the Cricket Benefit Fund Series (CBFS) - the organisers of two annual tournaments in Sharjah - to move the event involving Pakistan, New Zealand, Sri Lanka and possibly South Africa from April 9 to 19 to the last week of March. That would have enabled Pakistan to host New Zealand in April and May. Pakistan's cricket schedule was knocked out of line after the September 11 attacks on the United States and the retaliatory US-led action against Afghanistan. New Zealand subsequently called off its September tour to Pakistan citing security reasons, causing huge financial losses to the PCB. The West Indies has also said it is reluctant to play a series in Pakistan later in January given the current tensions between Pakistan and India and has asked for it to be shifted to a neutral venue. Zia said such a move would involve substantial losses for the PCB. "We would like to play West Indies on our grounds because playing on neutral venues would mean a major fall in revenues," he said. "Former West Indian captain Clive Lloyd is in Pakistan and we are sure he would give a feedback on security to the West Indies Board." Lloyd arrived in Pakistan on Thursday on a short coaching assignment with Pakistan's youngsters preparing for this month's Junior World Cup in New Zealand. Pakistan, which is currently on a two-Test tour of Bangladesh, played only six Test matches last year, only one of which was played at home.
Pak hopes Windies tour will go ahead
Pak aims for clean sweep against Bangla
England in India


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when me and my boyfriend first started dating, he was always making out with me, and wanting sex every night. it's been three months that we've been together, and now he doesn't like kissing me in public, and some nights he doesn't even want sex! I don't know what's wrong. is it something with me? advice please!
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This sounds bad, but maybe he is getting ready to break up? Or maybe you are doing something he doesn't like, and its his way of "getting back" at you. Like his own little revenge. Whatever the case may be, don't feel like YOU are the one doing anything wrong, even if he says its your fault. Unless you like, cheated on him or something, there is really no excuse for him to be such a jerk.00|00|0
no, there's nothing wrong with you. but with him. he may have someone else.00|00|0
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Few cohort studies have focused on risk factors for end-stage renal disease (ESRD). This investigation evaluated the prognostic value of several potential novel risk factors for ESRD after considering established risk factors.
We studied 177 570 individuals from a large integrated health care delivery system in northern California who volunteered for health checkups between June 1, 1964, and August 31, 1973. Initiation of ESRD treatment was ascertained using US Renal Data System registry data through December 31, 2000.
A total of 842 cases of ESRD were observed during 5 275 957 person-years of follow-up. This comprehensive evaluation confirmed the importance of established risk factors, including the following: male sex, older age, proteinuria, diabetes mellitus, lower educational attainment, and African American race, as well as higher blood pressure, body mass index, and serum creatinine level. The 2 most potent risk factors were proteinuria and excess weight. For proteinuria, the adjusted hazard ratios (HRs) were 7.90 (95% confidence interval [CI], 5.35–11.67) for 3 to 4+ on urine dipstick, 3.59 (2.82–4.57) for 1 to 2+ on urine dipstick, and 2.37 (1.79–3.14) for trace vs negative on urine dipstick. For excess weight, the HRs were 4.39 (95% CI, 3.38–5.70) for class 2 to class 3 obesity, 3.11 (2.51–3.84) for class 1 obesity, and 1.65 (1.39–1.97) for overweight vs normal weight. Furthermore, several independent novel risk factors for ESRD were identified, including lower hemoglobin level (1.33 [1.08–1.63] for lowest vs highest quartile), higher serum uric acid level (2.14 [1.65–2.77] for highest vs lowest quartile), self-reported history of nocturia (1.36 [1.17–1.58]), and family history of kidney disease (HR, 1.40 [95% CI, 1.02–1.90]).
We confirmed the importance of established ESRD risk factors in this large cohort with broad sex and racial/ethnic representation. Lower hemoglobin level, higher serum uric acid level, self-reported history of nocturia, and family history of kidney disease are independent risk factors for ESRD.
Patients with end-stage renal disease (ESRD) experience high rates of morbidity and mortality. Incidence and prevalence counts of ESRD in the United States are expected to increase by 44% and 85%, respectively, from 2000 to 2015.1 Reducing the incidence of ESRD is widely recognized as a major public health goal. However, few longitudinal cohort studies have been conducted focusing on identifying risk factors for ESRD.
Prior studies2,3 have identified the following independent risk factors for ESRD: older age, proteinuria, diabetes mellitus (DM), elevated blood pressure, African American race, and elevated serum creatinine level (or decreased estimated glomerular filtration rate). More recent studies4,5 have established overweight and obesity as independent risk factors.
In addition to these established risk factors, many potential "novel" risk factors have been suggested, ranging from cigarette smoking to history of urolithiasis. Many of these novel risk factors have not been rigorously examined in the context of a longitudinal cohort study design. Compared with cross-sectional study or case-control study designs, the advantages of a cohort study include reducing recall and ascertainment biases and establishing temporal sequence, which is important for determining possible causality. The goal of this study was to evaluate the prognostic value of several potential novel risk factors for ESRD after considering established risk factors. Simultaneous assessment is important because some of the proposed novel risk factors (eg, high serum uric acid level or low hemoglobin level) may be highly correlated with established risk factors (eg, baseline kidney function).
Identification of novel risk factors that predict ESRD independent of traditional risk factors may help improve risk stratification and facilitate more focused preventive efforts among higher-risk subgroups. It may also uncover new potential therapeutic targets to combat the rising tide of ESRD.
The study population is the large well-characterized cohort of Kaiser Permanente of Northern California members who participated in the Multiphasic Health Testing Services Program in Oakland and San Francisco.4 Kaiser Permanente of Northern California is a large integrated health care delivery system that cares for more than one-third of the insured population in the greater San Francisco Bay area. The Multiphasic Health Checkup was a voluntary health assessment offered at initial and yearly open enrollment periods. Details of the study population have been described previously.4,6 In the present analysis, we focused on the subset of subjects who were examined between June 1, 1964, and August 31, 1973 (N=177 570). We chose this period because subjects examined during this time underwent much more detailed assessment of comorbidities and laboratory testing than subjects examined in later visits.4,6 Institutional review boards at the collaborating institutions approved the study. Because the study was a secondary analysis of existing data, the need for obtaining informed consent was waived.
As described previously,4,6 we identified cases of ESRD treated with maintenance dialysis or renal transplantation by matching our cohort against the US Renal Data System ESRD treatment registry. We a priori chose ESRD from any etiology as the outcome of interest rather than specific causes of ESRD because of uncertainty about the accuracy of ascribed causes. For example, it is clear that hypertensive nephrosclerosis is often diagnosed without solid evidence that this is the underlying cause.7 Many patients with DM and chronic kidney disease may not have underlying diabetic glomerulosclerosis.8 Deaths were ascertained using the California Automated Mortality Linkage System.4,6 Occurrence of ESRD and death were assessed through December 31, 2000.
The Multiphasic Health Checkup database contains a broad array of potential risk factors measured using standard protocols previously described.4,6 All potential novel risk factors considered had less than 10% missing data, with the following 3 exceptions: self-reported history of kidney disease, self-reported history of kidney surgery, and family history of kidney disease. For these 3 dichotomous variables, missing values were coded as absent, which we believe would not introduce substantial bias given the rarity of these particular exposures. Novel risk factors were further divided into the following 4 groups: clinical, family history, occupational exposure, and behavioral (Table 1).
We designed our analytic approach a priori to minimize the likelihood of false-positive associations because of the many novel risk factors examined. We screened each novel risk factor in a univariate Cox proportional hazards model to determine if it was associated with risk of ESRD. We used quartile cutoffs for continuous variables to avoid assumptions regarding the nature of the associations. Only risk factors that had univariate associations of P<.05 were further considered in subsequent multivariate models. There were no prespecified searches for interactions.
We then constructed 5 separate multivariate Cox proportional hazards models. The first model included only the following established risk factors: sex; DM; level of educational attainment; African American, Asian, or other race; overweight and obesity (body mass index); elevated blood pressure or serum creatinine level; older age (age at the Multiphasic Health Checkup); and urine dipstick levels of protein, glucose, and hemoglobin. The year when the examination took place was also included given our previous findings from this cohort.6 Next, we examined the prognostic value of each of the 4 groups of potential novel risk factors already described. Therefore, the second model included all the established risk factors and the novel clinical risk factors. The third model included the established risk factors plus the novel family history variables. The fourth model included the established risk factors plus the novel occupational exposures. The fifth model included the established risk factors plus the novel behavioral risk factors. Using a likelihood ratio statistic, we compared the predictive value of the first model with that of each of the second, third, fourth, and fifth models. Novel risk factors from the second to the fifth models were further considered only if the likelihood ratio statistic indicated that the entire category of novel risk factors contributed additional prognostic information over and above the established risk factors.
The final multivariate model included established risk factors and individual predictors chosen from the 4 novel risk factor groups that contributed additional information and had an individual multivariate association with ESRD. The cutoff value was P<.05.
Among 177 570 subjects followed up for a total of 5 275 957 person-years, there were 842 cases of treated ESRD identified. Among ESRD cases, the mean (SD) duration between the baseline examination and the development of ESRD was 24.5 (6.8) years (median, 25.7 years; interquartile range, 20.2–29.7 years). The baseline characteristics of subjects are given in Table 2.
As summarized in Table 3, potential novel risk factors that were associated on univariate analysis with the development of ESRD included the following: history of stroke, gout, kidney disease, kidney or bladder stones, nocturia, and kidney surgery; left ventricular hypertrophy on electrocardiogram; higher serum cholesterol, lower hemoglobin, and higher uric acid levels; family history of kidney disease; selected occupational exposures (to lead or other metal fumes; asbestos, cement or grain dust; ammonia, chlorine, ozone or nitrous gas; chemicals, cleaning fluid or solvents; engine exhaust fumes [>2 h/d]; extreme heat; insect or plant spray; silica, sandblasting, grinding or rock dust); current smoking; and lack of consumption of alcohol (compared with consumption of 1–2 alcoholic drinks a day).
Based on differences in the likelihood ratio statistic between the base model that included only established risk factors, independent information was contributed by the novel clinical risk factors (P<.001) and by family history of kidney disease (P=.02). Therefore, occupational novel risk factors (P=.14) and behavioral novel risk factors (P=.11) were not considered further. Of note, the same conclusion about types of risk factors to include in our final model was reached when history of coronary artery disease was forced into the novel clinical risk factors model9,10 (P=.17) or when pack-years of smoking were used in alternate analysis of behavioral novel risk factors (P=.08).
Among the novel clinical risk factors, only hemoglobin level, history of nocturia, and serum uric acid level demonstrated P<.05 in multivariate analyses; therefore these, along with family history of kidney disease, were included in the final model. The results of the final multivariate model are shown in the Figure. Persons in the highest quartile of serum uric acid level had a more than 2-fold higher adjusted risk of developing ESRD compared with those in the lowest quartile (adjusted hazard ratio [HR], 2.14; 95% confidence interval [CI], 1.65–2.77). Independent associations with ESRD were also observed for lower hemoglobin level (HR, 1.33; 95% CI, 1.08–1.63 for lowest vs highest quartile), self-reported history of nocturia (1.36; 1.17–1.58), and family history of kidney disease (1.40; 1.02–1.90).
The importance of established risk factors was confirmed in this comprehensive evaluation (Figure). These included proteinuria (HR, 7.90 [95% CI, 5.35–11.67] for 3 to 4+ on urine dipstick, 3.59 [2.82–4.57] for 1 to 2+, and 2.37 [1.79–3.14] for trace vs negative on urine dip-stick); excess weight (HR, 4.39 [95% CI, 3.38–5.70] for class 2 and 3 obesity, 3.11 [2.51–3.84] for class 1 obesity, and 1.65 [1.39–1.97] for overweight vs normal weight); elevated serum creatinine (HR, 4.25 [95% CI, 2.49–7.25] for a level of >2.00 mg/dL, 3.16 [2.11–4.73] for a level of 1.60–2.00 mg/dL, and 1.24 [1.05–1.45] for a level of 1.10–1.59 mg/dL vs a level of <1.10 mg/dL); African American race (HR, 3.02 [95% CI, 2.58–3.54] vs white race); higher blood pressure (HR, 2.94 [95% CI, 2.21–3.92] for stage 2 hypertension, 2.33 [1.78–3.05] for stage 1 hypertension, and 1.72 [1.32–2.24] for prehypertension vs normal); DM (HR, 2.53 [95% CI, 1.97–3.25] presence vs absence); older age (HR, 0.55 [95% CI, 0.37–0.83] for age >60 years, 1.51 [1.17–1.96] for age 51–60 years, 2.23 [1.79–2.79] for age 41–50 years, and 1.91 [1.52–2.38] for age 31–40 years vs age ≤30 years); lower educational attainment (HR, 1.55 [95% CI, 1.21–2.00] for no college and 1.45 [1.11–1.90] for some college vs college graduate or higher); and male sex (HR, 1.22 [95% CI, 1.02–1.45]).
Results were unchanged in sensitivity analyses when we added a linear term for serum creatinine level to the model to minimize residual confounding within categories of serum creatinine level. Results were also unchanged when estimated glomerular filtration rate was used instead of serum creatinine level to measure renal function.
This study contributes to our epidemiologic knowledge of ESRD by identifying, through a rigorous screening process, 4 risk factors for ESRD that seem to be independent of previously established prognostic factors. These include lower hemoglobin level, elevated serum uric acid level, self-reported history of nocturia, and family history of kidney disease.
Lower hemoglobin level has been found to be a risk factor in some11 but not other12 prior studies. Among patients with type 2 DM and overt nephropathy, Mohanram et al11 noted that a hemoglobin reading less than 13.8 g/dL (to convert hemoglobin to grams per liter, multiply by 10.0) was associated with increasing risk of ESRD even after adjusting for concurrent kidney function. The pathophysiologic mechanism underlying the association between lower hemoglobin level and risk of ESRD is unclear. Tissue hypoxia seems unlikely with such modest declines in hemoglobin level, but it is possible that more severe anemia developed in the years following the baseline examination. It is also possible that this observation may be explained by residual confounding; for example, inflammation can lower hemoglobin level, or variations in hemoglobin level may reflect differences in underlying glomerular filtration rate not captured by serum creatinine concentration level. Nevertheless, markers of disease may still be useful clinically for providing prognostic information.
Although the concept of "gouty nephropathy" was discredited in the past, there has been resurgent interest in the pathophysiologic role of uric acid in cardiac and renal disease via detrimental actions such as stimulation of vascular smooth muscle cell proliferation and induction of endothelial dysfunction. Animal studies have shown that hyperuricemia accelerates renal disease progression in the remnant kidney model via a mechanism linked to high systemic blood pressure and cyclooxygenase 2–mediated thromboxane-induced vascular disease.13 Iseki et al14 reported that men and women with hyperuricemia in a screened Japanese cohort were more likely to develop ESRD. However, after adjusting for blood pressure and serum creatinine level in that study, hyperuricemia was no longer an independent predictor for men but remained so for women (for serum uric acid level ≥6.0 mg/dL [to convert serum uric acid level to micromoles per liter, multiply by 59.485]). Our results extend this novel observation to a larger, more diverse cohort and suggest that greater consideration should be given to randomized controlled trials to examine whether lowering the serum uric acid level would be an effective renoprotective strategy.15
It is interesting that nocturia (defined herein as self-report of "always having to interrupt sleep to urinate") emerged as an independent risk factor for ESRD because it is a widely held clinical belief that nocturnal polyuria is an early sign of chronic kidney disease due to decreased urinary concentrating ability,16 although some data suggest that increased salt, not water excretion, is more important.16 Therefore, nocturia may reflect subtle early renal disease not captured by serum creatinine level or urine dipstick analysis. We also cannot exclude the possibility that nocturia reflects undiagnosed DM. An alternative hypothesis is that nocturia reflects a high volume of ingested fluid that is detrimental (especially among patients with existing kidney disease), as high urine volume increases intratubular volume and pressure and these stretch forces induce fibrosis.17 Practically speaking, our data lend no support to the notion that a high volume of water intake should be recommended in clinical practice as being beneficial to kidney function.18
Our finding regarding family history of kidney disease (defined herein as self-report of kidney disease in a first-degree relative) is in agreement with previous studies that demonstrated the clustering of kidney disease within families19 and the heritability of traits such as glomerular filtration rate and albuminuria.20 These data further underscore the need to study the role of genetic influences on kidney disease beyond monogenic disorders such as polycystic kidney disease or Alport syndrome. These results also support the public health strategy of using family history of kidney disease to identify persons at increased risk to undergo screening for kidney disease.21
Our findings have several important implications. Our study confirmed the importance of established ESRD risk factors in this large diverse cohort with broad sex and racial/ethnic representation. As summarized in the Figure, independent risk for ESRD includes the following: male sex,22 older age,12 higher blood pressure,10,23 higher body mass index,4 DM (and more glycosuria),24 higher serum creatinine level,25 proteinuria26 (but not hematuria27), African American race,28 and lower educational attainment (and presumed lower socioeconomic status).29
The nonstepwise association in risk of ESRD with age (after adjusting for renal function and other comorbid conditions) is likely due to competing causes of mortality among older participants. The absence of a statistically significant association with smoking, alcohol intake, hematuria on dipstick analysis, and serum cholesterol concentration is consistent with prior literature showing that these are at best only weakly associated with the development of ESRD.2,3
The Figure shows the relative importance of these risk factors. The critical importance of proteinuria is highlighted by the fact that, on average, even trace proteinuria on urine dipstick is associated with a more than 2-fold increase in risk of ESRD. The strong association between excess weight and risk of ESRD is most concerning given the epidemic of overweight and obesity in the United States and other countries. It is notable that such a strong association was seen even after adjusting for concurrently determined blood pressure, proteinuria level, and DM status. Being African American (or belonging to other racial minority groups) remained a pronounced risk factor for ESRD even after controlling for extensive baseline explanatory variables. Consistent with what we previously concluded using a different analytic approach,30 the association between elevated blood pressure and ESRD does not seem to be due to confounding by clinically evident baseline kidney disease. We agree with prior commentators12,31 who stressed that data such as these would allow better identification of patients with heightened risk of ESRD for early screening to implement timely preventive interventions.
Our study also contributes toward the goal envisioned by some researchers to develop a summary equation to predict the likelihood of developing ESRD. For example, Taal and Brenner32 proposed the use of a "renal risk score" similar to the Framingham coronary heart disease risk score.
Another area in which these data could be applicable is in the selection of potential kidney donors. Quantitative estimates about risk of ESRD for living donors would seem essential to defensible donor selection practices. This need for reliable data regarding long-term risk is particularly urgent given the current trend toward accepting live kidney donors with "isolated medical abnormalities."33 Our findings could be used in conjunction with data regarding changes in blood pressure, urinary protein level, and glomerular filtration rate after kidney donation to provide more accurate informed consent of kidney donors.34,35 Of course, corroborating studies should be conducted among kidney donors.
Finally, the identification of novel modifiable risk factors such as hyperuricemia opens the door to potentially new interventions beyond blood pressure control and blockade of the renin-angiotensin-aldosterone system to reduce the risk of ESRD.
The strengths of this study include the long duration of follow-up, the unbiased ascertainment of ESRD, and the large size of the study cohort, which was diverse in terms of age, sex, and race/ethnicity. We were able to count actual treated ESRD cases and did not rely on surrogate outcomes such as slope of glomerular filtration rate loss. It cannot be assumed that the risk factors and the strengths of associations are identical, as most patients who experience progressive loss of kidney function do not develop ESRD but rather die of competing causes such as cardiovascular disease.36 Our study also differs from published studies37,38 that included treated ESRD along with "deaths attributed to kidney disease" as a composite outcome because patients who died and were not offered or declined renal replacement therapy are likely to be a special subset of subjects, and risk factors for these 2 outcomes may be different.
Our database not only captured the established risk factors for ESRD but also included a large array of potential novel risk factors. We systematically evaluated all of the potential novel risk factors and adopted a conservative analytic strategy to reduce chance findings. This study contributes to the literature by identifying lower hemoglobin level, elevated serum uric acid level, self-reported history of nocturia, and family history of kidney disease as independent novel risk factors for ESRD. We were also able to compare the relative strengths of associations of established and novel risk factors for ESRD in a comprehensive analysis within the same population. The availability of serum creatinine level and dip-stick urinalysis findings at baseline in all subjects represents an advancement over several prior studies.23,24,28,39
Limitations of this study include the fact that exposures were only assessed once. Misclassification (eg, of blood pressure category) would generally tend to bias our estimates toward the null, so the actual underlying associations may be stronger than those observed herein. In the intervening years between baseline assessment and development of ESRD, some subjects may have developed incident disease (eg, DM). Treatment of kidney disease has also changed over time, although we believe that this would not likely affect the relative associations seen between, for example, high serum uric acid level and risk of ESRD. Although many patient characteristics were assessed as potential predictors of ESRD, we were unable to assess variables such as illicit drug use, use of analgesic medications, or circulating inflammatory markers. Because our study was conducted among insured members of a northern California integrated health care delivery system, our results may not be completely generalizable to other populations.
In summary, this study contributes new information to our understanding of risk factors for ESRD. By studying women and men of different races/ethnicities, we quantified the magnitude of association of established risk factors with ESRD in a large and diverse cohort. We further established that lower hemoglobin level, elevated serum uric acid level, self-reported history of nocturia, and family history of kidney disease are independent novel risk factors for ESRD.
Funding/Support: This study was funded by grants HL71074 and DK61520 from the National Institutes of Health.
Financial Disclosure: None reported.
Role of the Sponsor: The National Institutes of Health had no role in the design and conduct of the study; collection, management, analysis, and interpretation of the data; and preparation, review, or approval of the manuscript.
Disclaimer: Some of the data reported herein were supplied by the US Renal Data System. The interpretation and reporting of these data are the responsibility of the authors and in no way should be seen as an official policy or interpretation of the US government.
Author Contributions: All authors had full access to all the data in the study and take responsibility for the integrity of the data and the accuracy of the data analysis. Study concept and design: Hsu, Iribarren, and Go. Acquisition of data: Iribarren, Darbinian, and Go. Analysis and interpretation of data: Hsu, Iribarren, McCulloch, Darbinian, and Go. Drafting of the manuscript: Hsu, Iribarren, and Go. Critical revision of the manuscript for important intellectual content: Hsu, Iribarren, McCulloch, Darbinian, and Go. Statistical analysis: Hsu and McCulloch. Obtained funding: Hsu and Iribarren. Administrative, technical, and material support: Iribarren, Darbinian, and Go. Study supervision: Iribarren and Go.
PubMed Central Canada is a service of the Canadian Institutes of Health Research (CIHR) working in partnership with the National Research Council's national science library in cooperation with the National Center for Biotechnology Information at the U.S. National Library of Medicine(NCBI/NLM). It includes content provided to the PubMed Central International archive by participating publishers.


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1333 Broadway, Suite 510 Oakland, CA 94612510-251-9470
Virtually all working Californians are "employees" for purposes of workers' compensation. Exemptions are few. People in business for themselves and some unpaid volunteers may not be covered. Maritime workers and federal employees are protected by federal workers' compensation laws.
Any injury caused by the job is covered -- everything from first-aid type injuries to serious accidents. Job-related illnesses are covered too. (For example, common colds and flu aren't covered, but if you catch tuberculosis while working at a TB hospital, that's covered.) The key is whether the injury or illness is caused by your job
Coverage begins the first minute you're on the job and continues while you are working. You don't have to work a certain amount of time or earn a certain amount before you're protected. Coverage is automatic and immediate.
Report the injury to your employer or supervisor immediately and complete a simple claim form if more than first aid is required. The claim form will ask what, where, when and how it happened. Your employer or supervisor will see that you get medical help right away and file the necessary reports.
Prompt reporting is the key. Benefits are automatic but nothing can happen until your employer knows about the injury Insure your right to benefits by reporting every injury, no matter how slight. Even a cut finger can lead to disability if an infection develops
California law guarantees three kinds of workers' compensation benefits: Reasonable and necessary medical care to cure and relieve effects of the injury. Not just doctor bills, but also medicines, hospital costs, fees for lab tests, X-rays, crutches, even travel expenses for required medical treatment. State law requires pre-authorization of non-emergency medical care, and there are limits on some types of treatment, so stay in touch with your claims administrator to make sure you get appropriate care and your bills will be paid.
Tax-free payments to help replace lost wages. Most injuries only keep you from working temporarily, and "temporary disability" payments will usually continue until the doctor says you're able to return to work or that your condition is permanent and stationary.
Additional cash payments will be made after you're able to work if there is a permanent disability - for example, the loss of a finger on an eye - or if you can't return to work at all. If the injury results in death, benefits will be paid to your financial dependents.
Supplemental Job Displacement Benefits in the form of a non-transferable voucher to use for education-related retraining or skill enhancement at a state-approved school or other resources that can help you find a new job. If 60 days after receiving a doctor's report that your condition is permanent and stationary and that you have a permanent disability, your employer has not offered you regular, modified or alternative work, the claims administrator has 20 days to offer you a voucher. The maximum voucher is $6,000 but there are limits on what you can receive for some items. If you qualify, you'll receive information on the types of expenses that are covered, the limits, documentation requirements, and deadlines for using the benefit.
Temporary disability (TD) benefits are generally two-thirds of your wages subject to minimums and maximums set by the State Legislature. For 2017 injuries, the minimum is $175.88 per week, and the maximum is $1,172.57 per week.
At the beginning of each year, the minimum and maximum weekly rates that will apply for injuries that occur during that year are subject to adjustments based on increases in the State Average Weekly Wage, so the amount of your payment will depend on the rate in effect on your date of injury, as well as your pre-injury earnings. A claims adjuster will send you a letter explaining how much you will receive.
Temporary disability isn't paid for the first three days you're unable to work, unless you're hospitalized as an in-patient or are off work more than 14 days. After that you should receive a check every two weeks until the doctor says you can go back to work. State law allows temporary disability payments for a single injury to continue for no more than 104 compensable weeks within five years from the date of injury; or for a few very serious types of injuries such as amputations, severe burns or chronic lung disease, the state allows payments for up to 240 weeks within five years of the date of injury. If the maximum temporary disability period expires before you can return to work, or if temporary disability is delayed or denied, you may be able to get State Disability benefits through the California Employment Development Department (EDD), but you need to apply in advance, so contact EDD at 1-800-480-3287 (http://www.edd.ca.gov/) for forms and information.
If the injury is very serious and you won't be able to work a year or more, you may be eligible for additional benefits from Social Security. Contact your local Social Security office or ask your employer or claims administrator for information.
Permanent disability benefits may be payable after you recover to the fullest extent possible. At that point, the doctor who treated you will evaluate the permanent effects of your injury. You and your employer may agree to rely on the treating doctor's report to establish your permanent disability payment. If you don't agree on the treating doctor's report, you may contact the claims adjuster, an information and assistance officer at the Division of Workers' Compensation, or your attorney if you are represented. They can explain your rights and the process for resolving disputes, which varies depending on a number of factors. Your level of permanent disability will be based on the doctor's report (including the doctor's opinion about how much of your permanent disability was directly caused by your work), as well as factors such as your age, pre-injury occupation, type of injury and date of injury. The weekly benefit is subject to minimums and maximums set by the state, which vary by date of injury and your level of permanent disability. If you have a permanent disability, the calculation of the benefit will be fully explained in a letter.
Death benefit payments to financial dependents are set by state law according to the number of dependents and the date of injury. Payments are made at the same rate as temporary disability payments, but no payments will be less than $224/week. Workers' compensation also pays a burial allowance of up to $10,000.
Start by contacting your employer or the claims administrator who handles your company's workers' compensation claims (the name and phone number should be posted at your worksite and should also be available from your employer). Many times problems and questions can be resolved with a simple phone call. If you still need assistance, you may call or write for free help from an Information and Assistance Officer employed by the State. Some problems may need to be resolved by the Workers' Compensation Appeals Board, which is a court of law. You can represent yourself or hire an attorney to represent you. If you hire an attorney, you will not pay the attorney directly, but the attorney fees will be paid out of your permanent disability, and are generally 12 to 15 percent of your award. Also, if you hire an attorney, other people involved in your case, including your claims adjuster, may no longer be able to speak directly to you about important matters, but will be required to communicate through your lawyer, and state Information & Assistance offices may no longer be able to provide free assistance or advice to you. In addition, if you choose to stop having an attorney represent you, or you want to change lawyers, your original lawyer can still claim a portion of your benefits. As long as you don't have a lawyer, Information and Assistance Officers can review your situation, answer your questions and intervene on your behalf. The Division of Workers' Compensation has 24 local Information and Assistance Offices around the state. Check the following list for the address and phone number of the office nearest you.
Information and Assistance Offices, California Division of Workers' Compensation
Anaheim1065 N. PacificCenter Drive, Anaheim, CA 92806714-414-1801
Bakersfield1800 30th Street, Room 100, Bakersfield, CA 93301-1929661-395-2514
Eureka100 "H" Street, Room 202, Eureka, CA 95501-0481707-441-5723
Fresno2550 Mariposa Mall, Room 2035, Fresno, CA 93721-2219559-445-5355
Goleta6755 Hollister Avenue, Room 100, Goleta, CA 93117-5551805-968-4158
Long Beach300 Oceangate Street, Suite 200, Long Beach, CA 90802-4304562-590-5240
Los Angeles320 W. 4th Street, 9th Floor, Los Angeles, CA 90013-1954213-576-7389
Marina del Rey 4720 Lincoln Blvd., 2nd Floors, Marina del Rey, CA 90292-6902310-482-3820
Oakland1515 Clay Street, 6th Floor, Oakland, CA 94612-1519510-622-2861
Oxnard1901 N, Rice Ave., Ste. 200, Oxnard, CA 93030-7912805-485-3528
Pomona732 Corporate Center Drive, Pomona, CA 91768-2653909-623-8568
Redding250 Hemsted Drive, Second Floor, Suite B, Redding, CA 96002530-225-2047
Riverside3737 Main Street, Suite 300, Riverside, CA 92501-3337951-782-4347
Sacramento160 Promenade Circle, Suite 300, Sacramento, CA 95834-2962916-928-3158
Salinas1880 North Main Street, Suite 100, Salinas, CA 93906-2037831-443-3058
San Bernardino464 W. Fourth Street, Suite 239, San Bernardino, CA 92401-1411909-383-4522
San Diego7575 Metropolitan Drive, Suite 202, San Diego, CA 92108-4424619-767-2082
San Francisco455 Golden Gate Avenue, 2nd Floor, San Francisco, CA 94102-7104415-703-5020
San Jose100 Paseo de San Antonio, Room 241, San Jose, CA 95113-1402408-277-1292
San Luis Obispo4740 Allene Way, Suite 100, San Luis Obispo, CA 93401-8736805-596-4159
Santa Ana605 W Santa Ana Blvd., Bldg 28, Room 451, Santa Ana, CA 92701-4070714-558-4597
Santa Barbara* Satellite office 411 E Canon Perdido Street, Suite 2, Santa Barbara, CA 93101805-884-1988
Santa Rosa50 "D" Street, Suite 420, Santa Rosa, CA 95404-4771707-576-2452
Stockton31 East Channel Street, Room 344, Stockton, CA 95202-2314209-948-7980
Van Nuys6150 Van Nuys Blvd., Room 105, Van Nuys, CA 91401-3370818-901-5367
Workers' Compensation Fraud Is A FelonyAny person who makes or causes to be made any knowingly false or fraudulent material statement or material representation for the purpose of obtaining or denying workers' compensation benefits or payments is guilty of a felony and may be punished by imprisonment in county jail for one year, or in state prison for up to five years, and/or fined up to $150,000 or double the value of the fraud (whichever is greater) and ordered to pay restitution for any medical evaluation or treatment. (IC § 1871.4).


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Nail salon businesses seem to be flourishing and most small communities have a few to choose from. Make your salon stand apart from the rest by using a dynamic and memorable business logo that will attract clients again and again. Allow it to reflect some aspect of the business itself or get creative and relate it somehow to your family or business name. Brainstorm, then talk to your printing company to incorporate bold bright colors for the greatest impact.
The Bottle
The nail polish bottle is instantly recognizable among manicure enthusiasts, so take advantage of that fact by using it as your salon logo. Have an artist make a modern-styled line drawing of a polish bottle and then situate it at an angle to add more interest and movement to your design. Print the line drawing in color to make it more eye-catching.
A broader look at the nail salon logo will incorporate a fingerprint along with a slogan like "Make Your Mark with June's Nails." Use a colored fingerprint design to create your own spin on the classic fingerprint logo. Use a raised print on your business cards and printed materials to add that tactile quality to the card your clients will never forget.
Polish Swathe
Nothing is more luxurious than a pearly pool of nail polish, and a nail salon logo design can incorporate this trade product in its logo. Use a thick swathe of nail polish as if just brushed on paper from a salon bottle as your logo and make a color statement by using a bold pearlized color that can be reproduced on all your business cards, flyers and price lists. Check with your advertising printer first to see what color options you have, and then create your logo.
Claw Marks
Although you wouldn't want to encourage a client to use her nails as a weapon, animal claw marks as a business logo for your nail salon would make your salon stand out and imply that your techs can create "wild" designs and fashion nails that leave their mark. Imagine claw marks that run down a few inches and have a ragged interior edge to make the graphic more interesting. Incorporate the "wild" logo into your store slogan.
Nail files, emery boards, clippers and cuticle scissors are all tools-of-the-trade for a nail salon, so consider these shapes for your nail salon logo. Use a front, close-up view of nail clippers in metallic ink for an interesting salon logo or a combination of tools in silhouette for a more dynamic logo. Be sure to use color in your logo to attract more attention on your business cards and printed materials.


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The extraordinary location of the hotel on the shoreline creates a special atmosphere in all of its 197 rooms. The combination of comfort, accessibility and peace is conducive to productive work and the true hallmark of the hotel. On the top floor, conference guests are welcomed into the bright and modern conference rooms. The hotel caters for groups with excellent personal service. The conference facilities are also appropriate for smaller trade fairs, workshops and exhibitions. All facilities have modern equipment and wireless Internet access. Further facilities available to guests in the air-conditioned hotel include a lobby with 24-hour reception and check-out service, a hotel safe, currency exchange facilities and a bar, pub and restaurant. Guests may also make use of the room and laundry services and a garage.
The hotel is located next to the central station for buses, trains, taxis and ferries right in the centre of Helsingborg. There is a beach just 500 m from the business hotel, while Frederiksdal Museum and Garden is 2 km away and Sofiero Castle is 4 km away. The city is Malmo is approximately 65 km from the conference hotel, Angelholm Airport is roughly 35 km away and Sturup Airport is around 60 km away.
Several rooms have a beautiful view of the sea and all of them have Internet access. Each room features an en suite bathroom with shower, bathtub and hairdryer, satellite TV, a radio, safe, minibar, ironing set and individually regulated air conditioning and heating.
Guests of the hotel are always welcome to exercise in the gym, or relax on the sun terrace or in the sauna. Spa treatments are also available. The nearby beach is sandy. The nearest golf course is approximately 6 km from the hotel.
A breakfast buffet is served each morning and � la carte options are available for lunch and dinner.
American Express, MasterCard and VISA are accepted as methods of payment.
If heading south on the E4/E6/E20, take the exit for Helsingborg N and the city centre. Before the central station turn right onto Kungstorget. If heading north on the E6/E20, take the exit for Helsingborg and the city. After the central station turn left onto Kungstorget.
Room Facilities:
Satellite cable TV
Internet access
Iron and board
Individual airconditioned
Individual heating
Wheelchair accessible
Disabled bathroom


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No one designer is the same, and only you can create and maintain the personal conditions that make your unique talents shine. At Designer Fund's Women in Design: Voice and Risk event, I spoke to Dropbox's Anisha Jain, Pinterest's Tiffani Jones Brown, and independent designer Jessica Hische about how to carve out a professional niche and do your best work.
Here's what we learned from the conversation.
What's your design superpower?
Anisha Jain, design leader at Dropbox, loves to ask her interviewees, "What's your design superpower?"
Your design superpower is the special talent you bring to your team and project, and Jain believes everyone has one (at least). As you gain experience as a designer, your strengths and weaknesses will reveal themselves. Once you have this clarity, you will best be able to contribute meaningfully, know where you have room to grow, and work with people who have complementary skills.
The key to finding and using your superpower is a mix of self-awareness, soliciting feedback from others, and communicating your limitations. Here are two tips to try.
Tip 1: Log a day in the life.
Carry around a notebook and pen for a few days to write down every little activity you do. Next to each activity, log how you feel immediately after doing it. Are you energized? Optimistic? Anxious? Sleepy?
Over time, you'll notice patterns about the rewarding and successful activities. Do more of those, and limit activities that drain you—or plan ahead for them. If big team meetings or administrative work leaves you frazzled, schedule them later in the day so you can tackle creative projects in the morning and early afternoon while your mind is fresh—or set aside one or two days of the week when you'll do draining work (more on this later). You can also share responsibilities with coworkers who complement your style. For example, extroverts and introverts can take turns attending meetings and step in for one another as needed.
Tip 2: Carve out your areas of incompetence.
"This is one of the best pieces of advice I've ever received," Jain said. It's common to spend a lot of time figuring out what you're good at and promoting those abilities, while neglecting to communicate to others where your abilities fall short. It's okay to admit what doesn't fit you. In fact, it's incredibly helpful, because it lets you ruthlessly cut distracting projects on your plate so you can focus your time and contribute more meaningfully to the group. And when you need to do something that's not a core skill, admitting a shortcoming puts you in a position to learn from someone else.
Create the daily conditions to be your best self
Everyone needs a certain cocktail of conditions to be creative and productive. No one will set this up for you: It's your professional responsibility to create situations that let your talents come alive each day.
Tip 1: Separate shallow and deep creative work.
Jessica Hische, an independent lettering artist, noted that she has two working modes: one, a hyper-organized, administrative, figure-stuff-out mode, and another where she's creative, intuitive, and spontaneous. Organizational tasks are very important for her business, but they also give her a sense of accomplishment that masks the fact that she isn't making progress on other, deeper work.
"I could sit there and answer 50 emails in six hours and think, 'I accomplished so much today. Answered 50 emails,' when really, I got nothing done," said Hische.
Hische now groups shallow work together on her calendar and leaves long, separate stretches of time for deep work. For example, she established "Administrative Mondays"—one day a week where she answers most of her email, follows up on projects, and does any logistical organization. And during the week, she sometimes ignores her inbox after noon to leave the afternoons for creative work.
Tip 2: Fight for what you need.
Tiffani Jones Brown, creative director at Pinterest, pointed out that it's up to you to create a schedule that supports your needs as an individual, especially when you're within a company or a larger system that doesn't hand you what you need automatically.
Carve out space in your schedule for health, wellness, and creativity. Work with your manager to take time off as appropriate, especially during real emergencies, so that your work situation is sustainable and you can continue to contribute your best work.
Another everyday example is to take a second look at the meetings on your calendar. If you're an introvert or feel drained by tense interpersonal interactions, you might find that meetings deplete your "battery" quickly. Try to minimize them or schedule them strategically so they don't drain you right before you do important creative work.
Finding this balance is a perpetual struggle that only gets trickier as designers step into leadership roles. As the head of Pinterest's creative organization, Jones Brown describes herself as an introvert who needs mental space and time to be creative. However, another core part of her executive role is to meet with many people across the company. So she imposed a six-meeting-per-day limit so her creative side has room to breathe. (She admits that for her, six meetings per day may still be too many.)
Manage negative inner voices
We all have a chorus of inner voices. Some sound like mentors who cheer us on as we try new things. Others, like the inner critic, hold us back from taking risks. A little dose of awareness can help you chase wild projects, learn new skills, and reach for promotions despite the presence of negative voices.
Tip 1: Practice mental reframes when you notice inner negativity.
Let's say a new project is taking you three times longer than you expected, and you're at the end of your rope in the confidence department. You think, "I'm so slow," "I'm the worst designer on this team," and "I'm always going to feel this bad in my job."
Try not to buy into these negative voices immediately. Notice them as signs that you're in the middle of a learning experience and not that you're a bad designer now and forever. Practice some mental reframes: "I'm learning now, so it's normal to struggle and feel confused and take longer than expected." If you fail, try thinking, "That was an opportunity where I learned something."
Another example of a negative internal voice is the one that whispers, "You don't belong here." That's impostor syndrome: You worry that you're a phony and people will discover you sooner or later. We all have room to grow, but the difference here is that these fears aren't based on your actual design skills and instead reflect a mental story about your identity and belonging.
Try to relabel these feelings as impostor syndrome, and put them into perspective with the broader context: Impostor syndrome is very common, especially among high achievers and among women or minorities who step into leadership roles (where new executive peers may not look or speak like you).
Tip 2: Feeling off? Take care of your body.
Negative voices are stronger when our bodies are suffering. If you're feeling hopeless or down on yourself at work, don't immediately question your competence as a professional. Instead, go through a mental checklist of the necessities that help your body function at its best—this might include food, sleep, caffeine, vitamin D, etc. Are you missing any of them today?
You can also keep a "secret calendar" to track days when you feel anxious or depressed, then look for patterns over time. Bodily awareness can remind you that your negative voices get louder and softer based on physiological factors, so it's no cause to panic. Take a breath, take care of your body, and soon you'll be ready to tackle the world again.
[Photos: Melanie Riccardi, unless otherwise noted]
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Dr. Peter G. Williams
Baltimore, Maryland - United States
No approved endorsements
About Dr. Peter G. Williams
Biomedical researcher with extensive experience in directing projects that apply biotechnology to medical assays and research projects to create cost effective solutions in the biotechnology field. Areas of expertise include identifying and managing new product opportunities, driving the design and development of medical assays, establishing clinical research goals and building collaborations with other laboratories. Demonstrated ability in identifying market issues, practical application of technology, managing budgets and achieving organizational goals.
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Arthur McBride - Paul Brady 1977 MP3
Definitive version of Paul Brady's classic. I had to reduce it to under 100 MB to fit it all onto one file. Well worth it though.....
Paul Brady sings Nothing But The Same Old Story MP3
Paul Brady, Irish singer-songwriter sings his song Nothing But The Same Old Story accompanied by Donal Lunny.
Paul Brady - The Island MP3
They say the skies of Lebanon are burning Those mighty cedars bleeding in the heat They're showing pictures on the television Women and children dying in ...
The Lakes of Pontchartrain - Paul Brady 1977 MP3
Paul singing this classic with Andy.
Paul Brady Live At The Embankment 1976 MP3
Mary and the Soldier - Paul Brady 1976 MP3
Paul sings Mary and the Soldier accompanied by Donal and Andy at the Embankment, Blessington Rd, Tallaght, County Dublin (as it was THEN!)
The Homes of Donegal - Paul Brady, written by Seán McBride MP3
This song was written by Seán McBride in about 1955. Seán was a native of Cruit island which is in the Rosses area of Donegal (West Donegal). He was a ...
Homes of Donegal - Paul Brady, Glen Hansard, Mundy, Moya Brennan & Mairéad Ní Mhaonaigh MP3
The Homes of Donegal performed by an array of Irish musical talent lead by Paul Brady, including Moya Brennan (Clannad), Glen Hansard (The Frames), ...
Paul Brady - The Long Goodbye MP3
A video shot in the back of a cab and on a tiny stage in Arlene's Grocery, a funky club on Stanton Street down in the village, with the legend that is Paul Brady.
Paul Brady sings Crazy Dreams on 'Later With Jules' 1996 MP3
Paul Brady sings Crazy Dreams with Liam Genockey, John Illsley and Kenny Craddock on 'Later with Jules', BBC TV 1996.
Tiernan McBride's film of Paul Brady's 'Arthur McBride'.mpg MP3
Long gathering dust on a shelf this is Tiernan McBride's 1977 film of Paul Brady's song 'Arthur McBride'. A wonderful document of a wonderful song.
The Streets of Derry - Cara Dillon with Paul Brady MP3
Cara Dillon - vocals Paul Brady - vocals Sam Lakeman - piano Catriona MacKay - harp Aly Bain - fiddle Ronan Browne - whistle Todd Parks - bass.
Paul Brady's "The Island" MP3
Written 20 years ago, the lyrics of Paul Brady's "The Island" ring even more true today. In this song with additional commentary, Brady expresses the devastation ...
Paul Brady - Steel Claw (Live) MP3
Paul Brady's new album Dancer In The Fire is available from Propermusic.com: http://bit.ly/Jzuroy & Amazon: http://amzn.to/I9irXR Filmed for the ...
Andy Irvine & Paul Brady - Arthur Macbride (1976) MP3
fae the andy irvine & paul brady album (1976)
Paul Brady - Nobody Knows MP3
The Best Of Irish Music.
Lakes Of Pontchartrain - Paul Brady MP3
From BBC's Folk Hibernia.
WoodSongs 723: Mary Black and Paul Brady MP3
Broadcast from the Convention Center in Dublin, Ireland, this broadcast spotlights the legacy artists of Ireland. Mary Black has been a dominant presence in Irish ...
Nothing But The Same Old Story - Paul Brady & Donal Lunny MP3
From the series Bringing it All Back Home Paul Brady & Donal Lunny perform Nothing But The Same Old Story.
Paul Brady on Mountain Stage MP3
Beloved Irish singer-songwriter Paul Brady sits down with host Larry Groce "Backstage at Mountain Stage." You can hear this episode beginning August 3rd.
Paddy's Green Shamrock Shore - Paul Brady MP3
Paul Brady in 1977 with Andy Irvine.
Mary Black & Paul Brady - I will Be There MP3
The Best Of Irish Music.
Sons of Anarchy - Gimme Shelter - Paul Brady & The Forest Rangers MP3
Son of Anarchy - fan video Music : Gimme Shelter - Paul Brady & The Forest Rangers.
Paul Brady - Nothing But The Same Old Story MP3
Paul Brady :: Doyles Pub :: Dublin MP3
www.ArbutusYarns.com :: Paul Brady plays live at The Ruby Sessions in Doyles Pub in Dublin, December 2010. Filmed by Myles O'Reilly and Stephen ...
Paul Brady - Lakes of Pontchartrain (New Recording) MP3
A new recording of an old favorite song of mine. From Paul Brady's Nobody Knows Album, Available at iTunes.
Paul Brady - Crazy Dreams MP3
Irish artist Paul Brady - Crazy Dreams From The 1979 Album , "Hard Station"
Paul Brady : 'The World Is What You Make It' MP3
an uplifting, magnificent song by the former member of influential Irish folk band Planxty.
Autumn Gold: Andy Irvine And Paul Brady MP3
Autumn Gold Words & Music: Andy Irvine My mood is clothed in Autumn's shades My Summer sun begins to fade Fading away with the waning year Changing ...


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Bang for the Buck: Cost-Effective Control of Invasive Species with Different Life Histories
Strategies for controlling invasive species can be aimed at any or all of the stages in the life cycle. In this paper we show how to combine biological data on population dynamics with simple economic data on control cost options to determine the least costly set of strategies that will halt an invasion. We then apply our methods to oyster drills (Ocinebrellus inornatus), an economically important aquaculture pest that has been accidentally introduced worldwide. If the costs of intervention were the same across life stages, extermination of adults would be an inefficient way to control species with the population dynamics characteristics of invaders. In the oyster drill case, however, efficient control targets adults because they are much easier to find.
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|Date of creation:
|08 Apr 2004
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| Web page: http://www.rff.org
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- Boyd, James & Wainger, Lisa, 2003. "Measuring Ecosystem Service Benefits: The Use of Landscape Analysis to Evaluate Environmental Trades and Compensation," Discussion Papers dp-02-63, Resources For the Future.
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Complementary Cures: More Than 530 "Blended Prescriptions" for Healing Dozens of Common Conditions--Better and Faster (Prevention's Best)
Complementary Cures combines the best of several healing techniques and brings you "blended prescriptions" for most of your common health problems, from acne to ulcers. Using the wisdom of professional M.D.s, Acupuncturists, nutrutionists, Eastern medicine experts, herbalists, and massage therapists, this indispensable volume brings you healing wisdom from all over the world, healping you cure your particular ailment as quickly, effectively and safely as possible.
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