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Master in Design Inc(1)6011 Reseda Blvd Suite #1, Tarzana, CA
Master in Design is a home remodeling company that specializes in kitchens, bathrooms, tiling and counter tops, and roofing. We know that in remodeling your home, we are helping you in a very important change in your life. Therefore, our sole focus is to work with you and be there for you throughout the whole process. From day one, we emphasis our personal attention to you to ensure that all of your options are 100% clear, that you are completely knowledgeable about the process, that all of your questions are answered, and that every potential bit of stress that may come from remodeling your home is relieved. We will be by your side until you are satisfied with every detail and every element of your remodeling job. Our creative eye and practical approach is also key when it comes to the design of your new kitchen, bathroom, roof, patio, or other room. Every job we tackle is wholly unique and fits the specifications of your taste and personality and the space that is being remodeled. At Master in Design, we emphasize your remodel as a design project that will refresh your home and your life. We are like interior designers with a hammer!Read MoreSee Full Profile
American EagleNo Reviews234 Galiceno Drive, San Jacinto, CA
Andy Rhee RemodelingNo Reviews2115 Arbutus St, Newport Beach, CA
Bibi ConstructionNo Reviews18750 Oxnard St, Tarzana, CA
Charlie's ConstructionNo Reviews4080 W 1st Ave # 183, Santa Ana, CA
Gale Force ConstructionNo Reviews16402 Gothard St Unit K , Huntington Beach, CA
CA Kitchen +Bath specializes in kitchen and bathroom renovations . Woodwork, painting, electrical, plumbing, and more. My services all come with the same promise of quality, dedication, and durability. We will take the time to understand your concerns and create a solution to meet your needs. Our extensive knowledge of kitchen and bathroom renovation will help you achieve exactly the space you are looking for.Read MoreSee Full Profile
HOME STARNo Reviews18032 Lemon Dr Unit C-629, Yorba Linda, CA
FULL SERVICE REMODELING COMPANY BASED IN ORANGE COUNTY CA WWW.HOMESTAROC.COMRead MoreSee Full Profile
Image Home Design, Inc.No Reviews20874 Calvin place , Woodlin Hills, CA
Image Home Design Inc. was founded in 2009 by Jacob Sherf in Woodland Hills, California and have since branched out to San Diego, California. From the office to the field, Image Home Design has a team of professionals and experts to provide the public with the best customer service and workmanship possible. Renovating thousands of homes since and providing home owners with top quality work to meet each clients expectations. We believe that customer service is the key to earning clientele for life, we focus on providing the best options of material, workmanship, warranties and guarantees. We are licensed, bonded, and insured to work on almost every residential property in Southern California.Read MoreSee Full Profile
Treeium IncNo Reviews5352 Laurel Canyon Blvd, Suite 200, Valley Village, CA
Treeium is a general contracting company with over 26 years of experience, which specializes in green home remodeling. We believe that change starts from the inside out, and that the home is a great place to start. For every need there is a green solution and this is why we offer remodeling services that help our customers create their dream homes in an efficient, sustainable, healthy way for both their families and the planet. Whether you have a small project on your hands, are looking to remodel your kitchen or bathroom, or need an entire home renovation, Treeium has the expertise and passion to help you do it in a way that's smart, efficient and sustainable, so your home will not only look good, but will also be healthier for you, the planet and your wallet. Thank you for trusting Treeium with your home improvement dreams!Read MoreSee Full Profile
Trusted Home Contractors IncNo Reviews18550 Burbank Blvd Suite #101, Tarzana, CA
Trusted Home Contractors has been providing excellence in the home remodeling field since 1975. Our services include Exterior Painting, Vinyl Siding, Windows,Sunroom, Patios and Patio Enclosures, Roof Replacement,. Our goal is to help you achieve your goal at a very competitive cost. Remodeling done affordable.Read MoreSee Full Profile
Alpha One Construction, Inc.18607 Ventura Boulevard, Unit 300, Tarzana, CA
American Pride, Inc.1201 East Chestnut Avenue, Suite C, Santa Ana, CA
Axel L. Espino Oliva1819 Monrovia Avenue, Unit 14, Costa Mesa, CA
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California Construction Co710 Kirkpatrick Drive, Hollister, CA
Douglas Charles Malo Construction1646 East Redbud Place, Pomona, CA
Green Construction27592 Ruisenor, Mission Viejo, CA
HFC25108 Marguerite Parkway, Unit A64, Mission Viejo, CA
HMR&R Services, Inc.31532 Railroad Canyon Road, Suite 102, Canyon Lake, CA


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It usually starts with a late night knock on the door. My 2-year-old boy, in his footie pajamas and tousled blonde hair, peeks at me with his eyes adjusting to the hall light.
It usually starts with a late night knock on the door.
My 2-year-old boy, in his footie pajamas and tousled blonde hair, peeks at me with his eyes adjusting to the hall light.
"I go potty," he says.
We let him try, of course, but unfortunately it's become a new tactic in a long list of methods to delay the inevitable: bedtime. Sometimes he wants more "cup," or sometimes he is frantic for his blue plastic wrench — it can't just be any ordinary plastic tool, but the very specific blue wrench that could be hidden anywhere around our house.
As of late, bedtime has become a dreaded ritual when our toddler refuses to sleep.
According to the National Sleep Institute, by the age of 2, most children have spent more time asleep than awake. Overall, a child will spend 40 percent of his or her childhood asleep, and that's important because the amount of sleep affects a child's mental and physical development.
But, trying to convince a 2-year-old who is refusing to go to sleep on schedule isn't so easy.
"The No. 1 thing for parents is to make sure there is a bedtime routine that starts an hour or more before going to sleep," said Tammy Morrow, a parent resource specialist with Alabama's Parent Assistance Line.
Things to do as part of that routine can include bath and a book, putting on quiet music, dimming the lights or turning on a night light to let the child know it's time for bed, Morrow said. It's also important to reduce vigorous activities before bedtime, she said, and give children a warning before the bedtime routine starts, so that they know when bedtime is near.
"Don't get into a tickling session or throw them up in the air before bedtime; no loud music or activities where they can't settle down," Morrow suggests. Local mom Ally Carson said consistency is key.
"We have always done the same thing: dinner, bath, books, bed," Carson said. "We lay them down, say good night, and then lights out. They have learned from literally four months on to put themselves to sleep."
Another mom, Haley Profitt, said she agrees.
"We always have the same routine and very rarely break it," she said. "Its always dinner, bath, snuggles, bed. Benjamin is 18 months and never fights bedtime. He lays right down and is asleep within minutes of laying down."
To help your child go to sleep, here are some bedtime tips from WebMD:
Make sleep a priority. Set regular bedtimes and wake-up times for the entire family.
Deal with sleep difficulties, including trouble falling asleep, waking up at nighttime, snoring and stalling going to bed. If your child seems overly tired and cranky during the day, tell your health care provider.
Work as a team. Come up with a bedtime strategy with your spouse or partner.
Routine is vital. Research has shown that a nightly bedtime routine can improve a child's sleep.
Make sure the bedroom is dark and quiet and the noise level in the house is low.

subset: commoncrawl


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Chav lad Mackenzie Cross from Hard Brit Lads has got an awesome body - slender, smooth, firm and athletic. This British bad boy starts stripping down, rubbing his package through his underwear, and then pulls out his nice long uncut cock and low hanging balls. And once he's naked, Mackenzie plays with his asshole, fingering himself while he jerks his hard dick!


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The Cardinals have had a different reliever save each of their last four victories. On Friday night, the saver was an unlikely one.
Left-handed reliever Trever Miller, 38, hadn't retired any of the five hitters he had faced over his last five outings (three walks, two hits), but manager Tony La Russa called on him with Atlanta center fielder Nate McLouth representing the tying run and the Cardinals ahead by two runs in the 11th inning. The reason? McLouth was 1-for-13 with four strikeouts against Miller in his career.
On this occasion, Miller didn't exactly fool McLouth, but left fielder Matt Holliday ran down McLouth's drive at the warning track, and the Cardinals had a 5-3 win, their third in four games on their six-game trip.
Miller relieved right-hander Jason Motte, who also would have been the fourth different reliever to get a save in that span of four wins. Right-handers Mitchell Boggs, Eduardo Sanchez and Fernando Salas had recorded saves since Sunday.
Boggs and Sanchez
combined for three hitless innings Friday before Motte, who fanned two hitters,
and Miller took care of the 11th. The save was Miller's first since 2008, when
he was with
"Jason would be charging with fastballs, and McLouth has proved to us over the years he can turn on a fastball," La Russa said. "He did it to (Chris) Carpenter. Trever's got a couple more weapons."
As it turned out, Miller threw a fastball to McLouth, too, but the out was made.
The only downside for the Cardinals was that ace right-hander Carpenter will finish April winless. Carpenter worked seven innings, giving up three runs. In five of his six starts in April, Carpenter allowed three runs or fewer, but he still is 0-2.
2B Nick Punto didn't have any spring training at-bats and still is finding himself. But after going 0-for-5 to start Friday's game, Punto delivered a two-run, game-winning triple in the 11th inning. Punto has just five hits for the Cardinals, but four of them have come in 10 at-bats with men in scoring position. "He just plays with a calm," manager Tony La Russa said. "He's got a terrific approach to playing this game, and he's really fun to watch."
RHP Chris Carpenter hurt his right hand when batting in the top of the seventh inning but refused to come out of the game. On his second pitch in the bottom of the inning, Carpenter allowed a tiebreaking home run to CF Nate McLouth but escaped further damage later in the inning by getting a double-play ball with the bases loaded. "He got jammed and his fingers were ringing," manager Tony La Russa said. "He said he was starting to get his feeling back after about the second hitter."
Rookie 3B Daniel Descalso started two double plays and also delivered a game-tying sacrifice fly in the ninth inning. He was giving regular 3B David Freese a night off.
RHP Ryan Franklin, taken out of the closer's role after blowing four saves in five opportunities, proved ineffective in several subsequent short-relief outings. He will be the Cardinals' "long man" for a while. "Somebody's got to fill that role," manager Tony La Russa said.
RF Lance Berkman extended his hitting streak to nine games, going 1-for-4 with a walk. In that time, he is 19-for-35, raising his average to .402.
BY THE NUMBERS: 1 - Games the Cardinals have won out of the six starts made by RHP Chris Carpenter this year. The one win came Friday.
QUOTE TO NOTE: "I hit the ball off the end of the bat three times. It didn't feel real good."
- RHP Chris Carpenter, whose right hand stung for a while in the seventh inning, perhaps bothering him enough that he gave up a go-ahead homer to Braves CF Nate McLouth.
2B Skip Schumaker (strained right triceps) went on the 15-day disabled list retroactive to April 16.
OF Allen Craig (left groin strain) went on the 15-day disabled list retroactive to April 17. He began a rehab assignment with Class AA Springfield on April 29.
LHP Bryan Tallet (fractured right hand) went on 15-day disabled list April 13. He had screws inserted in his hand. He was scheduled to throw side sessions every other day in late April. Tallet, who is scheduled to have stitches removed from his hand May 1, hopes to return in early May.
RHP Bryan Augenstein (strained right groin) went on the 15-day disabled list April 13.
RHP Adam Wainwright (Tommy John surgery in February 2011) went on the 60-day disabled list March 25. He will miss the entire 2011 season.
Apr. 30 at BRAVES: Jake Westbrook (2-2, 7.40) vs.


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Ultimate Nerf Stryfe Mod - Mass Effect
This modification will allow you to control the motor speed of a Nerf Stryfe and thereby the impact force of the Nerf darts.
The Stryfe is my favourite Nerf gun, but like all electronic blasters in its stock form it is kind of weak. There are multiple instruction on the web on how to perform a voltage mod, but to change the speed you would always have to use different batteries. You might ask why would you want it to run on a lower speed? Well if you have ever heard how loud it is after the voltage mod the question answers itself.
As an added feature, the speed of the motors is indicated by nine LEDs on the side of the blaster.


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You're looking at the first officially licensed Jaws Bruce the Shark mask.
They didn't hold back with the design.
This mask is sculpted by Mikey Rottela, who made sure to capture all kinds of details from Bruce's final scene, including the tank and part of Quint's hand.
I feel like this would make a great cosplay if you had someone dressed as Brody armed with a gun and aiming while saying, "Smile you son of a bitch!".
Product Page ($79.99)


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Paula Tropiano, LPC, CCDP-Diplomate - Behavior Therapist & Addictions Specialist
Paula Tropiano, LPC, CCDP-Diplomate - Behavior Therapist & Addictions Specialist
|Professions: Counselor, Psychotherapist, Life Coach
|License Status: I'm a licensed professional.
|Primary Credential: Licensed Professional Counselor - PC003871
|Office 1
West Chester, Pennsylvania 19380 Click for Map Get Directions
|Office 2
Regency Office Park
Philadelphia, Pennsylvania 19118 Click for Map Get Directions
Billing and Insurance
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My approach is strength-based, collaborative and holistic. The foundation of our work together includes building your skills; increasing your awareness, sense of emotional stability, emotional intelligence, and resilience, along with helping you learn to advocate for yourself gracefully and effectively. These skills help reduce symptoms, disturbing emotions and difficulties and support the creation of a more manageable and fulfilling life. I am accustomed to collaborating with other healthcare professionals in the interest of my clients, including primary care physician, psychiatrist, and other specialists. I believe that my clients are resourceful individuals with insight and skill waiting to be tapped in the interest of improvement of their health and well-being.
Email or Call Paula Tropiano, LPC, CCDP-Diplomate - Behavior Therapist & Addictions Specialist at 1-800-651-8085 ext. 19846
More Info About My Practice
Out of network provider (check if your plan has out of network coverage) Contracted with some EAPs - ACESS and FIRST CALL
New Group: Increase Your Effectiveness and Self-Mastery
Much of the time, "What's next" in our lives involves integrating or reinventing ones' self, developing a new lifestyle, relationship changes, career decisions and more. Whether it is improving your health, preparing for a relationship, starting a new venture, this group is oriented towards achieving your goals. Email for eBrochure with objectives and details.
Specific Issue(s) I'm Skilled at Helping With
- Addiction - chronic relapse
- Mood disorders - overwhelming emotions
- Complex situations where there are multiple issues occurring simultaneously. For example; addiction, pain, depression, and occupational problems.
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What I Love about Being a Psychotherapist
Sometimes people ask me how I can listen to people's problems every day. I always tell them, I don't hear negatives as much as I hear about opportunity and possibilities!
There is always an opportunity for improvement. When clients can start to use their minds and strengths in more productive ways, things start to shift and change.
As quoted by Carl Jung - "Behind our wounds lie our greatest gifts".
It is an honor to work with people in the work of self development and to see the "real self emerge" from within.
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This paper is concerned with qualitative properties of the natural frequencies and principal modes of some basic vibrating systems: the taut, non-uniform string; slender non-uniform rods in longitudinal and torsional vibration; the slender beam in flexural vibration; the circular membrane. These systems may be modelled by lumped-mass systems, finite elements, or by differential equations. The paper summarizes results on the distinctness of natural frequencies, interlacing of frequencies corresponding to different end conditions, nodal patterns for modes, etc. It also briefly describes research on the reconstruction of such systems from natural frequency or principal mode data.
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23519 Brazos Town Crossing Rosenberg, TX 77471
Charming Charlie in Rosenberg, TX
Greetings, Rosenberg! Oh, how we "adorn" you!
Welcome to your local Charming Charlie jewelry store! Conveniently located at the northwest and southwest quadrants of Southwest Freeway and Thompson Road, our Rosenberg store in Brazos Town Center is your one-stop destination for all your accessorizing needs. This bustling venue is one of the area's most popular spots for shopping, dining and entertainment with plenty of stores and restaurants for locals to enjoy.
At Charming Charlie, we've made it easy to brighten up your wardrobe with our store's colorful arrangement of accessories. From classic crossbody handbags to on-trend layering necklaces, we have the designs you need in a wide range of hues to help you find your fabulous in a flash.
Call or stop by today!


text:
Dressed in a shimmery white leotard and a furry white vest, Cyrus went on to the stage to accept the best video award for her song "Wrecking ball".
"I don't think I'm going to be able to fit that in my purse so I'll put it right here for now," the 20-year-old said, before placing the award near her feet.
"I couldn't fit this award in my bag, but I did find this," she said, before pulling out the supposed joint.
She then proceeded to light it and take a long drag.
Cyrus performed on her track "We can't stop" twerking in a silver skin-tight dress at the event.


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1960 Fashion Timeline 1950- 1800-2000 1980- 1990 1800-1810
a high waistline and skirt
with petticoats or hoops
underneath 1830-1840
Wool, linen, cotton and silk were now the most usual material to make clothes. Sleeves became enormous and necklines became very wide. 1820-1830
the sleeves and shoulders got
bigger with wings. Patterns and
decoration and colour were now
used much more making the
dresses nicer 1840-1850
Woman started wearing bloomers.
The best clothing you can have
now are skirts with lower waist and tight bodices 1920-1930 1890-1900
sleeves were huge and
dresses were only big
in the back bit until the next year 1810-1820
Soft colours returned, sleeves got fuller at the shoulder, firmer fabrics were used to make the clothes 1850-1860
Off shoulder v-necked tops,
flowers and bows on skirts 1860-1870
hoops were replaced by a new undergarment that did change the shape of the long dress. 1870-1880
a very small hoop was worn
to keep the mass of fabric away
from the feet. Fabrics were light
in colour and weight. 1880-1890
Accessories such as gloves
hats etc. were the main part
of fashion. 1900-1910 1910-1920 1930-1940 1940-1950 1960-1970 1970-1980 1990-2000 the sports wear changed a lot especially the swimming and cycling wear the first world war starts women go into uniform as they join the services to support
the war effort Hollywood movie stars influenced the people
with there beautiful styles that
were mad out of the best
fabric in that century the second world war starts shortage of money leads to
shortages of fabric
and clothing Mary quant is
often credited
a lot with inventing
the miniskirt
which people started
which people started wearing
from that time until now fashion is
by the youthful
styles worn in
"swinging" London trends skinny legged jeans and also glam rock becomes the latest Shoulder pads were used for both men and women, 1990 -2000 fashion changed
an awful lot because in the olden days it was fancy
clothes but now it is the pop age and not only
the pop age people also started wearing expensive clothes and fabrics shops set trends
for women with
the level of the
dresses and skirts
up to the knees for
the first time
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Fashion Timeline 1800-2000
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Edda hoor Veraon 5 March 2013
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Industry News
Bodycentral Physical Therapy Wins Silver Stevie Award in 2012 Stevie Awards for Women in Business
Bodycentral Physical Therapy received an award for their community involvement program of the year on November 9th at the 9th annual Stevie Awards for Women in Business held at an event in New York City.
Winners Announced at Event in New York City
TUCSON, ARIZONA – November 14, 2012 – BODYCENTRAL PHYSICAL THERAPY has been named the winner of a SILVER Stevie® Award in the COMMUNITY INVOLVEMENT PROGRAM OF THE YEAR category in the 9th annual Stevie Awards for Women in Business.
The Stevie Awards for Women in Business are the world's top honors for female entrepreneurs, executives, and the organizations they run. All individuals and organizations worldwide are eligible to submit nominations – public and private, for-profit and non-profit, large and small. The 2012 awards received entries from 17 nations and territories. Nicknamed the Stevies for the Greek word for "crowned,"
More than 1,200 nominations from organizations of all sizes and in virtually every industry were submitted this year for consideration in a wide range of categories, including Entrepreneur of the Year, Executive of the Year, Company of the Year, Mentor of the Year, Women Helping Women, and Communications Campaign of the Year, among others. BODYCENTRAL PHYSICAL THERAPY won in the community involvement program of the year category for companies and organizations.
"I'm extremely proud of the extra time and effort our team takes to provide community outreach programs to Tucson and the surrounding areas. It is an honor to receive an international award," said Jennifer Allen, co-founder of Bodycentral Physical Therapy. "Tonya (Bunner) and I believe we have a responsibility to share our knowledge and skills with the people of our community. We are here to improve the overall health and wellness of the people in southern Arizona."
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JOSE ESTEBAN LAZO MORALES, 39 years of age, married, a stock-raiser and
resident of San Pedro de Lóvago, in the Department of Chontales, was arrested at home
members of the State Security on November 13, l983 at approximately 11:30 p.m. and taken
to the base of operations of that agency, which operates in the city of Juigalpa, the chief town
of the Department of Chontales, and that on November 18 of that same year he was returned to
his home as a corpse.
The corpse of Lazo Morales was brought to his home by four members of the
Security in a coffin that was screwed shut and they ordered that it should not be opened and then
burried it in the early hours of the morning. Despite that warning, the family opened the coffin
and found that the corpse had the following injuries: fracture of the nose, bruises on the
eyebrows, blood on the head and in the ears, the lower part of the abdomen was purple, there
were fractures of the ribs and two rather deep, crescent-shaped wounds, four inches long on the
The family was also handed a medical-legal certificate issued by Dr. Luis N. Gadea
the medical examiner of the Department of Chontales, which stated that the prisoner Lazo
Morales had died of a heart attack. That certificate shows some peculiar features including the
fact that that physician states that when Lazo Morales arrived as a prisoner he was extremely
nervous and that the fractures and wounds were the result of falls, due to fainting fits he had
suffered while he was imprisoned, i.e., that that medical examiner gave opinions that normally
a pathologist who only examines the corpse could not give, unless he is giving the version of
the authorities responsible for the arrest.
On February 27 of this year (l984) the case was brought to the attention of the
Complaint Department of the Ministry of the Interior and a request was made for an immediate
investigation so as to determine the responsibilities in the case, but so far we have received no
news that the investigation being requested is being made.
Mr. Jose Esteban Lazo was detained for counterrevolutionary activities linked to
Nicaraguan Democratic Front in San Pedro de Lovago, Chontales. The trial against him could
not continue due to his death, brought on by cardiac arrest.
According to Doctor Luis N. Gadea, the then forensic physician of the Department
Chontales Mr. Jose Esteban Lazo Morales did in fact die of cardiac arrest, and stated that
according to his professional undertaking, after having concluded the legal medical exam, he
did not find any signs of torture.
For his part, the Minister of Interior also roundly denied the claimant's allegations
The facts reported in the petition whose pertinent parts have been transmitted to
Government of the State in reference shall be presumed to be true if, during the maximum
period set by the Commission under the provisions of Article 34, paragraph 5, the Government
has not provided the pertinent information, as long as other evidence does not lead to a different
In view whereof:


text:
Pedigree of the family affected with RDS Arg195Leu–related central areolar choroidal dystrophy. Solid symbols indicate clinically affected individuals; open symbols, clinically normal individuals; and slashed symbols, deceased individuals. Individuals of generations III and IV underwent clinical examination and were analyzed for the peripherin/RDS gene. Dots refer to individuals identified as carriers of the Arg195Leu mutation.
Age–visual acuity relationship in 9 affected family members. The visual acuity is expressed in decimal notation (20/20 = 1.0; 20/200 = 0.1). Each symbol represents the visual acuity of an affected eye at a specific age. Solid circles represent patients who had been examined only once, whereas the longitudinal change in visual acuity is illustrated by the lines joining open circles, open squares, and crosses for 3 individuals.
Fundus images of individual III-5, aged 45 years, showing a ring-shaped configuration of retinal pigment epithelial clumping at 3° to 4° eccentricity to the foveal center. The fovea itself reveals tiny, hard drusenlike deposits. Visual acuity is 1.0 OU. Color perception is inconspicuous.
Autofluorescence images of individual IV-3, aged 14 years (A), and individual III-5, aged 45 years (B), depict central oval areas of diffusely increased autofluorescence up to 11.0° eccentricity. This area is not assessable by conventional fundus imaging. Speckled autofluorescence structures form a ringlike configuration in correspondence to funduscopic pigment clumps. Note the tiny dark spots of incipient retinal pigment epithelial atrophy next to high autofluorescence clumping (white arrows).
For amplitude and latency analysis, the multifocal electroretinogram (mfERG) traces were averaged across 5 rings that contained hexagonal stimulus fields of identical eccentricity.12 Amplitude and latency of each ring average were evaluated by fitting template mfERG traces17 as derived from our database of 50 healthy individuals. The mfERG of individual III-5 (right eye) exhibits latencies above normal limits as indicated by the gray background (dark blue ±1 SD, light blue ±2 SDs) in rings 1 through 3. In the central ring, the latency delay is accompanied by a preserved amplitude, indicating a beginning dysfunction in the foveal part of the retina.
Age-dependent variability in pattern of fundus autofluorescence: left eyes of 7 individuals affected by central areolar choroidal dystrophy stages 2 through 4. Individuals IV-7 (A), IV-6 (B) (stage 2 disease), and IV-4 (C) (stage 3 disease) show an abnormal speckled pattern of autofluorescence that reveals enlargement with age and encloses a single patch of geographic atrophy in individual IV-4 (C). There was a range of autofluorescence variability within individuals disclosing central areolar choroidal dystrophy stage 4: III-3 (D), III-2 (E), III-1 (F), and III-6 (G). The characteristic speckled pattern extends beyond the vessel arcades and the optic disc with more advanced age. Sharply demarcated dark areas indicate loss of lipofuscin-loaded retinal pigment epithelial cells. Note that the size of geographic atrophy is not necessarily linked to age.
Keilhauer CN, Meigen T, Weber BHF. Clinical Findings in a Multigeneration Family With Autosomal Dominant Central Areolar Choroidal Dystrophy Associated With an Arg195Leu Mutation in the Peripherin/RDS Gene. Arch Ophthalmol. 2006;124(7):1020-1027. doi:10.1001/archopht.124.7.1020
© 2017
JANEY L.WIGGS,MD, PhDAuthor Affiliations: Department of Ophthalmology, University of Würzburg, Würzburg, Germany (Drs Keilhauer and Meigen), and Institute of Human Genetics, University of Regensburg, Regensburg, Germany (Dr Weber).
To characterize clinical findings associated with a mutation in codon 195 (Arg195Leu) of the peripherin/RDS gene in a large multigeneration family of European decent.
Sixteen members from 2 generations underwent ophthalmologic examination, including best-corrected visual acuity, examination of the anterior segments, and inspection of the ocular fundus after pharmacologic mydriasis. All affected family members underwent Farnsworth Panel-D15 color testing. Five selected family members with early stages of the disease underwent multifocal electroretinography. Full-field electroretinography was performed in 2 family members with more advanced fundus changes. Finally, former patients' records and fundus images were analyzed to determine the course of the disease in affected individuals.
Nine family members in 2 generations were diagnosed as having autosomal dominant central areolar choroidal dystrophy. The family demonstrated an age-dependent increase of central granular fundus abnormalities with progressive development of geographic atrophy. Interindividual phenotypic variability was apparent and ranged from predominantly drusenlike depositions to single perifoveal pigment clumps. Age of onset of visual disturbances varied between 27 and 48 years. All individuals who manifested signs of disease were found to carry an Arg195Leu mutation in the peripherin/RDS gene.
Age of onset, progression of the disease, and characteristic fundus abnormalities share similarities to previous reports on families with central areolar choroidal dystrophy associated with peripherin/RDS gene mutations in codons 172, 142, and 195, respectively. However, striking variability in individual phenotypic findings and age of onset in our family suggests that additional factors that modify the defined peripherin/RDS gene mutation Arg195Leu likely influence the severity of the disease.
Clinical Relevance
Caution should be advised in predicting the clinical course and severity of the disease based solely on a specific mutation in the peripherin/RDS gene.
A number of mutations in the peripherin/RDS gene that affect codons 142, 172, and 195 (Arg172Trp, Arg172Gln, Arg142Trp, and Arg195Leu) are known to cause progressive central areolar choroidal dystrophy (CACD).1- 10 Early stages of the disease are characterized by a fine, at times barely discernible, mottling of the parafoveal pigment epithelium. Later, progressive macular atrophy leads to a significant decline in the central visual acuity (VA) in most cases.
Several reports3,5,7,10 on clinical findings focus on differences in age of onset of visual complaints with respect to distinct mutations in the peripherin/RDS gene. So far, a single family with CACD from Japan has been shown to carry an Arg195Leu mutation; 3 affected members of this family were described to develop visual symptoms between the fourth to fifth decades of life.10 Herein, we report the clinical findings of a large family from Bavaria, Germany, with 9 affected members in 2 generations cosegregating an Arg195Leu mutation in the peripherin/RDS gene.
Sixteen members from 2 generations underwent ophthalmologic examination, including best-corrected VA, examination of the anterior segments, and inspection of the ocular fundus after pharmacologic mydriasis. Digital fundus (Funduskamera FF 450 plus; Zeiss, Jena, Germany) and confocal autofluorescence images (HRA 1; Heidelberg Engineering, Heidelberg, Germany) were obtained in all individuals. Autofluorescence images were recorded according to a standard protocol described previously.11 All affected family members underwent Farnsworth Panel-D15 color testing. Five selected family members with early stages of the disease underwent multifocal electroretinography (mfERG). This method identifies localized dysfunction of the retina (eg, maculopathies) where the full-field electroretinogram is only mildly affected.12,13 The mfERG recording procedures adhered to the corresponding International Society for Clinical Electrophysiology of Vision guidelines.14 The mfERGs were recorded binocularly with dilated pupils and DTL (Dawson-Trick-Litzkow) electrodes using a VERIS system (version 4.16; Electro-Diagnostic Imaging, Inc, San Mateo, Calif). Full-field electroretinography was performed in 2 family members with more advanced fundus changes. The method was performed in concordance with the International Society for Clinical Electrophysiology of Vision standard protocol.15 Finally, former patients' records and fundus images were analyzed to determine the course of the disease in affected individuals.
Mutational analysis of the 3 coding exons of the peripherin/RDS gene, including at least 20 base pairs of intervening sequences representing the splice consensus motifs, was performed with the following primer combinations: exon 1: rds1c.f (5′-CCG GAC TAC ACT TGG CAA G-3′)/rds1b.r (5′-ATA GCT CTG ACC CCA GGA CTG-3′); exon 2: rds2a.f (5′-AAG CCC ATC TCC AGC TGT CTG-3′)/rds2b.r (5′-CTT ACC CTC TAC CCC CAG CTG-3′); and exon 3: ex3F (5′-CCA GCG ATT CTC CCA GAT T-3′)/ex3R (5′-GAG TTG GAT GAG GGG GAG AT-3′). Polymerase chain reaction fragments were directly sequenced with the Big-Dye Terminator Cycle Sequencing Ready Reaction Kit (Perkin Elmer, Weiterstadt, Germany) and analyzed on an ABI 310 automated sequencer (Applied Biosystems, Weiterstadt, Germany).
The pattern of disease inheritance in 4 generations was consistent with an autosomal dominant trait of CACD in this family (Figure 1). After obtaining informed consent, 9 siblings (III-1 through III-3, III-5, III-6, and III-8 through III-11) (aged 43-61 years) were examined clinically. Five of them revealed signs of the disease. In addition, 7 children (IV-1 through IV-7) aged 14 to 36 years of 2 affected siblings (III-5 and III-6) underwent close ophthalmologic examination. Affected family members reported progressive decrease in central VA, with the onset ranging from age 27 to 48 years. Three individuals (III-5, IV-3, and IV-6) aged 14 to 45 years were not aware of their disease until examination. The age-VA relationship is plotted in Figure 2. None of the affected individuals was aware of problems with night vision or side vision.
Four clinical stages were defined according to the classification established by Piguet et al4 (Table). Consistent with stage 1, two affected family members with perifoveal pigment mottling and tiny foveal, hard drusenlike deposits; preserved central VA (1.0-1.25); and normal color perception were identified (individuals IV-3 and III-5, aged 14 and 45 years). Perifoveal pigment clumps (Figure 3) revealed high autofluorescence properties indicative of abnormal accumulation of retinal pigment epithelium (RPE) lipofuscin.16 Adjacent spots of low autofluorescence are suggestive of incipient RPE atrophy (arrows in Figure 4). The images depicted central oval-shaped areas of diffusely increased autofluorescence up to 11.0° eccentricity. This area was not assessable by conventional fundus imaging. The mfERG17 (Figure 5) showed latencies out of normal limits in rings 1 through 3, indicating photoreceptor dysfunction up to approximately 9.6° eccentricity. Reduction of amplitudes, indicating photoreceptor degeneration, was found in rings 2 and 3.
Two affected family members (IV-6 and IV-7), aged 32 and 30 years, respectively, were classified as having stage 2 disease defined by scattered pigment clumping of the central posterior pole without the presence of focal patches of geographic atrophy (GA). The VA was slightly reduced to 0.8 OU.
Farnsworth Panel-D15 color testing identified specific color disturbances that affected the blue axis. Funduscopy revealed diffuse depigmentation of the central posterior pole, including tiny clumps of pigment and yellow, hard drusenlike deposits. Family member IV-7 had been examined at 25 years of age with VAs of 1.0 OU. During the past 5 years a change from a coarse-grained pigmentation of the central fundus to a more dusty aspect was observed. Autofluorescence images exhibited a speckled central oval-shaped area (Figure 6A and B) reaching eccentricities up to 14° (IV-6). Individual tiny spots of high autofluorescence intensities corresponded to larger pigment clumps in the color image. The mfERG disclosed delayed latencies and reduced amplitudes in all rings of both family members.
Their elder sister (IV-4, aged 34 years, stage 3 disease) experienced her first visual symptoms by age 27 years. Six years later she was found to have a severe decrease in central acuity to 0.05 OU. Results from a saturated Farnsworth Panel-D15 color test showed unspecific dyschromatopsia with no preference of axis in both eyes. Autofluorescence images depicted characteristic speckling within the central oval area similar to the pattern observed in her younger siblings. In addition, the left eye revealed an isolated patch of sharply demarcated chorioretinal atrophy (Figure 6C). The mfERG revealed latencies outside normal limits in all rings whereas amplitudes were extinguished in rings 1 through 4 and markedly reduced in ring 5. Full-field electroretinography displayed a normal scotopic response whereas photopic responses were near the lower limit of normal.
The elder 4 siblings of generation 3 (III-1 through III-3 and III-6, aged 52-61 years) presented with markedly reduced central acuity (range, counting fingers to 0.1) and advanced GA consistent with stage 4. The eldest sibling (III-6, aged 61 years) was first seen at the age of 37 years when he experienced a significant loss in VA to 0.05 OD and 0.1 OS. He had not been aware of visual symptoms before that time. His eldest sister (III-1) underwent examination at the age of 43 years with a bilateral VA of 0.4. She reported gradual visual deterioration during the past 15 years, down to 0.05 in the right and counting fingers in her left eye at the age of 57 years. The 2 youngest sisters (aged 52 and 54 years) realized severe decline in VA (defined as ≤0.1) by the age of 48 years (III-2) and 32 years (III-3), respectively. Farnsworth Panel-D15 color test findings showed unspecific dyschromatopsia with no preference of axis in all family members with stage 4 disease. Fundus examination depicted multiple areas of sharply demarcated RPE atrophy surrounded by typical tiny scattered or confluent spots of hyperpigmentation and small, hard drusenlike depositions. Family members III-2 and III-3 displayed an overall yellowish fundus, including myriads of tiny, hard drusenlike deposits, partly forming confluent plaques. Older family members showed fundus changes to extend beyond the vessel arcades and the nasal side of the optic disc. The pattern of the macular lesion, shape, and size of the GA depicted striking symmetry between both eyes except for individuals III-1 and III-2, who disclosed more advanced alterations in their left eyes. Peripheral retina was inconspicuous in all affected family members. Former fundus images of individual III-6, aged 37 years, and of individual III-1, aged 43 years, confirmed progression of the disease from a pattern of central RPE mottling to advanced GA. Autofluorescence images disclosed the characteristic speckled pattern extending beyond the vessel arcades and the optic nerve head by the sixth decade of life. Sharply demarcated dark areas of RPE atrophy preferentially involved the papillomacular area in individual III-3 (Figure 6D-G). Full-field electroretinography revealed markedly reduced photopic responses, whereas scotopic responses reached the border of normal limits (individual III-6, aged 61 years). Peripheral visual fields were unremarkable.
Sequence analysis of the 3 coding exons in the peripherin/RDS gene revealed a heterozygous point mutation (nucleotide 584 G>T) in codon 195, resulting in an arginine to leucine change at this position in all 9 affected family members analyzed. Conversely, 7 unaffected relatives in generations III and IV did not harbor the Arg195Leu mutation. Thus, the molecular findings show strict cosegregation of the Arg195Leu mutation with the clinical phenotype of CACD in the family.
The clinical features associated with the Arg195Leu mutation in the peripherin/RDS gene in our family can be categorized as a progressive macular dystrophy with relative preservation of peripheral retinal function. There is a marked similarity to patients carrying the codon 172 (Arg172Trp) and codon 142 (Arg142Trp) mutation with regard to the characteristic RPE mottling within a central oval macular configuration and the progression toward GA with age. The appearance of drusenlike deposits as observed in individuals III-2 and III-3 in this family has also been noted in some members of the Dutch families with the Arg142Trp mutation8 and in patients with the Arg172Trp mutation originating from the Zermatt area of Switzerland.4
Consistency in the age of onset and progression of the disease has been reported in families segregating the Arg172Trp mutation in the peripherin/RDS gene. For example, 11 individuals of 11 ancestrally related British families were found to exhibit first visual symptoms by the age of 30 years, and a severe decrease in central acuity (≤20/200) was reported by the late 40s and early 50s in these families.7 Similarly, 24 affected members of the Swiss family with the same mutation had initial visual symptoms in their 30s (stage 2; VA, 0.8-1.0; mean ± SD age, 33 ± 3 years) and decreases in VA below 0.1 by their 40s to 50s.4 Comparably, severe visual impairment (VA ≤20/200) in 7 members of a Swedish family segregating the Arg172Trp mutation was noted in their fourth to fifth decades of life. Affected members of the Swedish family were found to develop a degeneration of more peripheral parts of the retina with advancing age, suggesting additional involvement of rods late in the disease.6 Age of onset of first visual symptoms was also reported in 30 affected family members of 7 Dutch families that harbored the Arg142Trp mutation. In contrast to individuals affected by the Arg172Trp mutation, onset of visual symptoms in the Dutch families occurred later in life; affected family members experienced visual deterioration in their mid 40s,18 and disabling decrease of vision was noted by their 60s to 70s.5,8,18
Finally, 3 Japanese family members segregating the Arg195Leu mutation were reported to manifest first visual disturbances between the fourth and fifth decades of life. The time of severe visual loss was not assessable in this relatively small family. The authors suggested that the clinical course of CACD due to the Arg195Leu mutation may be less severe than the one associated with Arg172Trp.10
In contrast to the previous reports, our family revealed a striking variability both in the age of onset of visual impairment and in the age at which there was severe visual loss. Onset of first visual symptoms occurred between the ages of 27 and 48 years; one affected member did not complain about visual problems at the age of 45 years. Five family members experienced a severe decrease in central VA to 0.1 or less between the ages of 33 and 52 years. In contrast to the Swedish family, function of rods remained preserved up to the age of 61 years.
The peripherin/RDS gene appears to play an essential role in the assembly, orientation, and structural stability of outer segment discs and to account for an increased turnover of instable membranous segments.19 The encoded glycoprotein has been localized to the rim region of the disc membranes of both rods and cones. The functional basis of why some mutations of this gene account for predominantly rod and others for mainly cone dysfunction has been widely discussed. Interactions with other gene products, as previously shown, for example, with ROM1 and peripherin/RDS mutations in retinitis pigmentosa, have been proposed as a possible explanation.20 However, no such associations have so far been reported to occur in peripherin/RDS-related CACD. Recently, a transgenic mouse model with cone-dominated macular dystrophy carrying the Arg172Trp mutation in the peripherin/RDS gene has been reported. The authors speculated that the preferential damaging effect of this mutation on cones may be accounted for by differences in the cone outer segment structure, which may render cones more susceptible to conformational changes than rods.21 On the other hand, this hypothesis does not explain the relatively late involvement of foveal cones in the course of the disorder in contrast to the early alteration of perifoveal cone photoreceptors. Outer segments of perifoveal cones may be more susceptible to alterations in stabilizing peripherin/RDS molecules than foveal cones. Blue cones may play a role, since they are mostly absent from the foveal center, are most numerous at 0.35° to 0.5° from the foveal center,22 and are markedly different from red and green cones in their anatomical structure and cellular function.22- 24 The ring of high blue cone density corresponds to the perifoveal region, where the disease is initially observed in our family. Two of 4 young family members had blue cone deficit. The involvement of blue cones, however, is not supported by the facts that they are relatively sparse (compared with red and green cones) and that the disease spreads into neighboring areas that affect other types of photoreceptors. Finally, we cannot rule out the presence of thus far unknown protective factors for foveal photoreceptors or segment stability factors at the site of the fovea, which could account for the preservation of central acuity up to the age of 45 years, such as in family member III5.
In summary, we observed a striking variability in age of onset and macular phenotype in our relatively large family with an Arg195Leu mutation in the peripherin/RDS gene. Also, a single mutation in this gene has been reportedly noted to cause various phenotypic features, including retinitis pigmentosa, fundus flavimaculatus, and pattern dystrophy in affected members of a single family.25 Thus, a wide phenotypic range associated with an identical mutation in the peripherin/RDS gene is not uncommon. Further investigations may identify whether additional genetic factors may exert an influence on individual phenotype expression and/or whether environmental influences are responsible for the wide range of pathologic changes.
Correspondence: Claudia N. Keilhauer, MD, Department of Ophthalmology, University Hospital Würzburg, Josef-Schneider-Str 11, D-97080 Würzburg, Germany (firstname.lastname@example.com).
Submitted for Publication: July 1, 2005; final revision received November 7, 2005; accepted November 14, 2005.
Funding/Support: This study was supported by grant 01KS9603 (IZKF Würzburg) from the Bundesministerium für Bildung und Forschung.
Financial Disclosure: None reported.
Acknowledgment: We thank the family members for their participation.


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This recipe is simple enough to serve as an everyday meal, and elegant enough to serve when entertaining guests. The herbs and sherry give it a "fancy" touch sure to please, and the biscuits give it a "down home" appeal.
Yield: 6 servings.
· 1½ pounds chicken breasts, skinless, boneless, cut into 1-inch pieces
· 1 Tablespoon cooking oil
· 12 oz. fresh Florida mushrooms, sliced (4½ cups)
· 2 small Florida onions, shopped
· 2 cloves garlic, minced
· 1 cup reduced-fat sour cream
· 2 Tablespoons all-purpose flour
· 1 teaspoon seasoned salt
· 1/2 teaspoon pepper
· 1 cup low-fat Florida milk
· 1/2 cup chicken broth
· 1/4 teaspoon marjoram
· 1/4 teaspoon basil
· 1/4 teaspoon oregano
· 2 Tablespoons dry sherry
· 1 recipe herbed biscuits (see recipe below)
· green onions, sliced for garnish
Herbed Biscuits
· 1¾ cups packaged dry biscuit mix
· 3 Florida green onions, thinly sliced
· 1/4 teaspoon oregano
· 1/4 teaspoon marjoram
· 1/4 teaspoon basil
· 1/2 cup low-fat Florida milk
Herbed Chicken
Step 1: In a skillet, cook chicken in hot oil over medium-high heat for 4 to 5 minutes or until no longer pink. Remove chicken and set aside. Add mushrooms, onion, and garlic to skillet. Cook, uncovered, for 4 to 5 minutes or until liquid is evaporated. Set aside.
Step 2: In a small bowl, stir together sour cream, flour, seasoned salt and pepper. Add sour cream mixture, milk and broth to mushroom mixture. Cook and stir until thickened and bubbly. Add marjoram, basil and oregano and lemon juice and cook for an additional minute. Add chicken and sherry; heat through. Serve over biscuits and sprinkle with green onions.
Herbed Biscuits
Stir together all ingredients just until dough clings together.
On a lightly floured surface, knead dough 10 to 12 strokes. Roll to a 1/2 inch thickness. Using a 2-inch biscuit cutter or a biscuit cutter of your choice, cut dough into 12 biscuits. Re-roll and cut the trimmings as necessary.
Arrange biscuits on an ungreased baking sheet. Bake in a 425 degree oven for about 12 minutes or until golden.
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Fri 26th Aug 2011 12:00pmGet Joggly's Byron Bay Surf Report
Week of average left overs as the swell faded. Mayb a break this week. kind of hoping for that.
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GE Profile large capacity washer and dryer. Dark gray. Excellent condition. Dryer has sensor from washing machine to auto set drying time
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Material Matters
A clear vision and a touch of alchemy are at play in this artfully modern classic that overlooks the Santa Cruz mountains and Silicon Valley. The design is intentionally hard to date, an at once timeless and contemporary study in materials and form that is both medieval and modern, well-tailored and glamorous. The minimalist palette of architectural materials—Minnesota limestone, plaster, bronzed steel and glass—crosses the threshold from outdoors to in and provides a clean-lined yet warm backdrop for the noteworthy furnishings and art. On the exterior, the lacy grid of glass and steel windows reveals the substantial yet airy feel of the architecture. A floating plane of steel on the roof caps the two story façade of Minnesota limestone. Inside, the design for the master bathtub started with a single block of white onyx, which was carved into an intricately faceted piece of working sculpture.


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Playboy of the Western World: Saturday, January 28 at 2pm
The NOCCA Institute
Saturday, January 28, 2012 from 2:00 PM to 4:00 PM (CST)
Event Details
NOTE: Pre-sales for this show have ended. Tickets can still be purchased at the door.
Box office opens one hour prior to curtain.
NOCCA Drama Students Offer Irish Comic Classic
The Playboy of the Western World
Nims Black Box Theatre
January 25-28 at 7 PM
January 28 at 2 PM
New Orleans Center for Creative Arts (NOCCA) invites the public to enjoy the student production of John Millington Synge's Irish comic masterpiece, The Playboy of the Western World at NOCCA's Nims Black Box Theatre, (2800 Chartres Street, New Orleans). Performances run January 25-28 at 7 PM, with an additional 2 PM matinee on Saturday, January 28.
Students from across the region will be featured in the production, including: Daisa Bell, Madeline Daste, Luke Diamond, Rexton Laird, Asia Nelson, Jelani Pitcher, Daisy Rosato, Holly Settoon, Darnell Thomas, Austin Thompson, Ricky Watson Jr., Devin Williams, and Dean Wray.
About The Playboy of the Western World
Directed by NOCCA Theatre Arts faculty member Janet Shea, this sensual Irish comic classic, drunk with poetry and ironic laughter, takes a hilarious look at murder, mayhem, deceit and first love. Riots greeted the show's Dublin premiere in 1907, but the play still strikes a chord today, in our society of instant fame and notorious celebrity, because of Synge's indictment of humankind for the folly and fickleness of the love of hero.
When & Where
2800 Chartres Street
New Orleans,
Saturday, January 28, 2012 from 2:00 PM to 4:00 PM (CST)
Add to my calendar
The NOCCA Institute
Founded in 1973, the New Orleans Center for Creative Arts is Louisiana's arts conservatory for high school students. NOCCA offers intensive instruction in Classical Music, Creative Writing, Culinary Arts, Dance, Drama, Jazz, Media Arts, Musical Theatre, Theatre Design, Vocal Music, Visual Arts, and academics. Students come from across Louisiana, attending via full-day, afternoon, and after-school sessions. Admission to NOCCA is by audition, and there is no tuition.
The NOCCA Institute is NOCCA's nonprofit partner, providing supplemental funding for NOCCA and advocacy for its world-class program. Some of the Institute's more notable endeavors include: a comprehensive Financial Aid Program that funds students' supplies and fees associated with important summer training programs across the country; an Artists-in-Residence Program that brings more than 100 professional visiting artists into NOCCA's classrooms each year; the capital campaign for NOCCA's current home and expansion projects along the Press Street corridor, including Press Street Station, 5 Press Gallery, and Press Street Gardens; and concert, gallery, and literary events for the community. The Institute also oversees rentals of the NOCCA campus, making it available to arts organizations, individuals, corporations, and other groups.
Unless otherwise noted, we're unable to issue refunds. If an event takes place over multiple days, however, we'll do our best to honor such requests by switching tickets to a different time.


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رستوران پرشين هاوس در ملبورن.
هرهفته كله پاچه وحليم روزهاي شنبه ويكشنبه صبحها جهت صرف صبحانه با نان سنگك وپاركينگ اختصاصي
Persian House Restaurant in Melbourne
Delicious Persian & Afghan Cuisines.
Persian House is strategically located at 247B Belmore Road, Balwyn North VIC. We serve the most authentic and the most delicious Afghan Cuisine in the region. Treat your family and friends with meals that they will never forget from our best sellers and house specials. Our lunch menus include Seekh Kebab Meal, Lamb Korma Meal, Chicken or Cauliflower Curry and Kofta Korma. Try our bestsellers like Korma Sabze, Shirazi Salad, Koobideh Sandwich, Zereshk Polo, Baghali Polo and Bamejan Shekampor.
The official website of Persian House provides our customers with an easy and secure way to order from our takeaway menu online. Safely pay via credit card or cash. It is free to use and after each order you will receive an SMS confirmation with your approximate delivery or pick up time. Enjoy your food!
Address: 247B Belmore Rd, Balwyn North, VIC, 3104, Australia
Tel: 0450317050
Visited 1299 times, 4 Visits today


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The forum is moderated for new users, approved one of your posts and responded to it here: http://forum.ipxe.org/showthread.php?tid=8244
On Thu, Dec 1, 2016 at 4:49 PM, Lukas Zapletal <lu...@zapletalovi.com> wrote: > Hello, > > our project (www.theforeman.org) provides functionality to generate > bootdisk for individual hosts. These are either iPXE or SYSLINUX based. The > iPXE version does load the kernel/initramdisk directly via HTTP, the > SYSLINUX has embedded linux kernel. > > Now, we had no UEFI support, until now. I am able to implement SYSLINUX > bootdisk type (we call it Full host image) with Grub2 and that was pretty > simple. > > I would like to add ipxe.efi into the image and be able to boot it, which > is also doable either directly, or via chainbooting from Grub2. But the > problem is, with SYSLINUX we've been able to provide the script via > initramdisk (it's call embedding in iPXE terms I believe): > > default ipxe > label ipxe > kernel /ipxe > initrd /script > > This worked fine, but I am unable to do the same with ipxe.efi. > > set default=0 > set timeout=1 > menuentry "Chainload iPXE - this does not work" { > linuxefi /ipxe.efi > initrdefi /script > } > menuentry "Chainload iPXE - this loads iPXE but no script" { > search --no-floppy --set=root -f /ipxe.efi > chainloader /ipxe.efi + 1 > } > > Is there a way to embed a script? What I want to avoid is building iPXE > from scratch (with embedded script), I don't want to make our application > complex and compiler and tools are not usually available on production > installations. > > If there is no way of doing that from Grub2, I am fine booting iPXE > directly, that's just more work when generating the ISO (hybrid) image for > me, but feasible too. > > Tried to post into the user forum, but it did not work (form was submitted > but no post appeared). Later on, I saw a PostgreSQL error page, so sending > to the dev list if you don't mind. Thanks for help! > > -- > S pozdravem / Best regards > Lukas Zapletal > > _______________________________________________ > ipxe-devel mailing list > firstname.lastname@example.com > https://lists.ipxe.org/mailman/listinfo.cgi/ipxe-devel > >
_______________________________________________ ipxe-devel mailing list firstname.lastname@example.com https://lists.ipxe.org/mailman/listinfo.cgi/ipxe-devel


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October 25, 2013 Total Views: 4707 Today: 1
Hotel Apartamentos La Lonja Valencia, Apartamentos La Lonja is located in on a pedestrian street in Valencia's Old Town, 350 metres from Valencia Cathedral. It offers air-conditioned apartments with a balcony and a washing machine. The La Lonja apartments feature free Wi-Fi and a seating area with a TV and DVD player. The kitchen includes a microwave and a coffee maker. The apartments are just 50 metres from Valencia's Lonja de la Seda, the medieval market which is now a UNESCO World Heritage Site. You can drive to the spectacular City of Arts and Sciences in 10 minutes. Valencia's bullring and Estació del Nord Station are under 15 minutes' walk from the apartments. Valencia Airport is a 17-minute drive away. (Hotel Apartamentos La Lonja Valencia hotels booking).


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If you choose to study a Higher Education course at College, the fees can be considerably cheaper than university study and there are a number of loans available to help fund the cost of your course.
• For full-time courses (September 2017 entry) fees range from £6150 for Foundation Degree to £9250 for Bachelor Degrees
• For part-time courses (September 2017 entry) fees range from £3960 to £4500 for 120 credits per year
Course fees can be found on individual course factsheets HERE.
Please note: these are subject to change from Teesside University.
If you are a new student enrolling on a designated Higher Education programme and studying at least 30 credits a year, you can apply for a loan to cover the full cost of fees each year.
This means that your course will be free at the point of entry and you only make repayments if you are earning over £21,000 a year.
Your loan repayments will start in the April four years after you begin your course, even if you are still studying, or in the April after you leave your course, whichever comes first.
Repayments are based on what you earn, not on the amount you borrow.
If you are earning less than £21,000 a year at this point you will not begin repaying your loans. If you are earning over £21,000 a year your repayment will be calculated as 9% of any income you have over £21,000. After 30 years, any outstanding balance is written off.
Click HERE to view The Student Loan Companies top tips on staying safe online when applying for loans.
Funding from your employer Employers may be able to pay part or all of your fees upfront. You get to study for a qualification that will improve your ability to do your job and advance your career prospects, and your employer benefits from a better qualified, more productive and motivated employee.
If your company doesn't have any sponsored programmes, but you still think further study would be beneficial to you both consider making a proposal in support of your request.
The first person to talk to will be your Line Manager but you should also be prepared to make a case to other senior staff or the Personnel Manager, who may make decisions on training expenditure.
Teesside University offers a wide range of financial support for Higher Education students who may need additional help funding their studies.
• Access To Learning Fund
• Disabled Student Allowance
• Elite Athlete Bursary Scheme
Click HERE for further information about financial support at Teesside University.
Part-Time study
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Thursday, March 26, 2015
SEBI Amends Delisting, Takeovers and Buyback Regulations
1 comment:
REACTION (instant, primarily common sense based)
To one's mind, the point of intriguing poser of most concern is in regard to the declared effective date of 24th March 2015. That is an odd date; which is not in sync say with the normally adopted, or to-be-most- ideally preferred one, being the beginning date of 1st April, of a financial year, for fiscal matters; such as say, for tax laws.
Further, the said effective date is the same as that of the Gazette Notification (s); as such, despite the several drastic changes, of a highly complex nature hence requiring time to get a true grip, those are expected to be/have been complied with as on and from that date. To be precise, SEBI permits no time at all, much less a reasonable time for even the amendments to be read and understood, not to speak of compliance in letter and spirit.
The subject notified amendments, prima facie of a substantial /substantive nature, are seen to be in respect of those extant regulations, the earliest of them (i.e. the last mentioned one) dating as far back as to 1998. Further, that is purported to have been made in exercise of vested powers and in pursuance of clause (f) of sub-section (2) of section 68 of the Companies Act, 2013.As has been the common and widely aired genuine complaint in industry and legal circles, the new company law itself is riddled with several areas on which the enactment is incomplete or there is either no clarity or is potent with possible controversies and legal disputes.
In short, in one's independent perspective, even without going into the merits or otherwise of the subject amendments,- apart from the difficulties likely to be faced in the matter of compliance by companies on one side, so also of implementation, monitoring and enforcement by the authorities on the other,- the wisdom in prescribing 24th March 2015 as the effective date, is highly questionable.
For an appreciation in proper light of the viewpoint projected above, recommended to study a similar intriguing situation brought about because of an amendment of tax law with an odd effective date. Reference is to the insertion of section 234 D of the IT Act, as critically analysed in (2008) 173 TAXMAN 80.< It may be added, the enactment came to be amended vide the Explanation 2 later inserted later; but, as perceived, not wholly to serve the expected purpose as canvassed in the cited article.
In the ultimate analysis, in profound public interest, to be precise for better, if not ideal, administration, what should be of the most concern, with natural justice in focus, crying for devoted thinking is this: - Is there no hope at all for any improvement in the obtaining state of affairs, in the matter of legislation, or making rules by regulatory authorities or of the kind?!
- March 27, 2015 at 8:39 AM


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IBM Rational Transformation Workbench v3.1 Getting Started (GC23-8532-00)
Getting Started introduces Rational Transformation Workbench. This guide provides an overview of the workbench tools and discusses basic concepts. It describes how to install the product and how to use common workbench features.
Click below to view this publication
Other language versions may be available from the Publications website.
Document information
More support for:
IBM Asset Transformation Workbench
Reference #: GC23-8532-00
Modified date: 21 November 2007
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timepiece and its cryptic display is unquestionably ugly -- even the "stainless steel" band reeks of cereal box quality. But there's just something about those flashy lights that stirs the soul of nerds everywhere, making it seem quite the bargain at $132.85. It's okay, we won't tell mommy.
[Via BoingBoing]


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Category Archives: Motivational
The Key To Abundant Life
Living Life at a Level 10
In John 10:10 Jesus said, "I have come that you might have life and that you might have it more abundantly!" This type of abundant life is something I call "Level 10" living. On October 10th, 2010, I was reading John 10:10—there it was—a divine synergy! I saw "Level 10 Living". Jesus said something vital: […]
It isn't too late
3 Thoughts on God's Redemptive Power
Do you ever feel like you've messed up beyond repair? You know those things that weigh on your heart, the shadows of past regrets, failures and pain? Romans 8:28 says that "God causes all things to work together for our good." All things—that includes the hurtful decisions and the unfortunate events that are a part […]
Discerning the Spirit of Envy
3 Observations on Jealousy
I was talking to some young ministers recently who have become popular on the national landscape. They shared that they sense jealousy and criticism from their peers in response to what they say. I've noticed something kind of interesting. When we are being critical in ministry, or pick on other people, it's often not because […]
How To Keep From Sabotaging Your Own Transition
3 Questions To Ask During Transition
One of the challenges of transition is understanding how to exit one phase or stage of life and enter the next one. Jesus was tested in the wilderness while going into His ministry and tested even more in Gethsemane when transitioning out. You are the most vulnerable during strategic times of transition. There are 3 […]
The Five Lies of the Battle You Face
Warfare is inside your mind, more than in your circumstance. Don't believe the lie. Reading some inspiring words from Seth Godin got me thinking today about the new 'Information Economy'. Don't believe the lie. David slew the Giant after picking five smooth stones. Use them to slay the THE 5 LIES. The FIRST LIE… is […]
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God is Calling, Follow The Flow!
Your Most Neglected Relationship May Surprise You
Emotional Intelligence author Daniel Goleman, once used the term "feeling tone" to describe the ultimate measure of communication. Communication involves a sender and a receiver. If you are sending accurate content but the feeling is off, your message is compromised by the feeling you mixed into it. I have often sent the wrong message when […]


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Parachutes are for Pansies is a challenge in Mr. Torgue's Campaign of Carnage. It takes place in the Forge itself, the southern part of the wider forger area, and is worth 4 badass points.
This challenge involves a simple jump from a high location, however to reach the lofty height Vault Hunters must first negotiate a number of catwalks populated with hostile bikers. Many of these adversaries are long range units with assault rifles and the occasional shotgun, and the ranks are rounded out with some buzzard air support and a few psychos.
The drop location is directly above the entrance ramp to the Forge. A low wall between two bandit turrets blocks the most direct jump, but jumping up onto this before leaping out towards the entrance gate ensures challenge completion.
- This jump provides a suitable finale to the mission Kickstart My Heart as jumping down from the top of the Forge is the fastest way back to the gate.