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It happened because it had to, the boundaries of propriety in professional hockey are well-defined and create certain inevitabilities.
So the consequences were thoroughly predictable when Nashville's Jordin Tootoo steamed into Buffalo goalie Ryan Miller on Saturday: several Sabres, including Miller, piled onto the little agitator like crows on fresh road kill.
Afterward Sabres left wing Matt Ellis, whose fists were in among the tangle of limbs flailing away at Tootoo, told the Buffalo News "Our response was good. Goose got in there, we all got in there. It was a five-man pack and we took care of business. Boston was a learning experience, and we took a few steps forward."
The Goose in question is burly centre Paul Gaustad, who engaged in a public bit of self-flagellation after l'affaire Bruins last month – wherein Boston's Milan Lucic also took liberties with Miller, concussing him with a borderline hit as he played the puck, and received no such pummelling for what is a mortal sin against the game's sacred unwritten code.
Gaustad pointed at himself after that ("I'm embarrassed") and said "there's no reason to be scared. We had to go after it, and we didn't."
As the weekend's events showed, the Sabres were determined to avoid having their honour sullied again; in that context, exacting immediate revenge is deemed progress.
Which cuts to the core of what modern hockey is above all else: a nightly referendum on personal responsibility, physical toughness and commitment.
Or what academics term an expression of 'hyper-masculinity'; a less politic description would be to call it a triumph of the meathead element.
In an era in which many within hockey and without are pushing for a sea change on several fronts – head shots chief among them – the clubby prevailing culture continues to be one of violence, rough justice and vigilantism.
Many influential hockey people and hordes of fans are okay with that. In fact, they have been for a very long time – there is a reason for Don Cherry's enduring popularity.
"I think there's something distinctive about hockey, it's not necessarily the same script of masculinity as in other sports ... this is also a game that was notoriously aggressive and violent from its beginnings," said Michael A. Robidoux, an ethnologist who teaches at the University of Ottawa and is a former major-junior player.
As the discussion over brain injuries in hockey rages on, there is anecdotal evidence that a shift is occurring at the grassroots level of the game.
Parents are grilling store clerks on the relative merits of various safety equipment, some doctors report an increase in the number of recreational players turning up in emergency rooms to be checked out for concussions.
South of the border there is actual data: a recent study by the Centers for Disease Control indicated the number of hospital visits for children suspected of having concussions shot up by 60 per cent in the past decade. Much of that is attributed to heightened awareness in football and hockey.
But where is the culture shift in the upper reaches of Canada's game, whose influence spreads downward?
Some brain-trauma specialists privately grumble that for all the attention drawn to concussions, there seems to be little meaningful change in pro hockey.
It's to wonder where the dominant culture comes from and why it continues to reign in the face of mounting scientific evidence of the dangers of many on-ice practices.
But social and behavioural change of any sort – whether you're talking about smoking, seat-belt use, drunk driving – is a painstaking process at the best of times.
"It can take a generation for norms, values, rules and institutions – a culture – to change. And sporting culture is generally more conservative," said Suzanne Laberge, a sports sociologist at the Université de Montreal. "Resistance to change is inevitable, but it doesn't make it impossible. It will require more injuries, more arguing, and more time."
The paradox is that within the sport there is a sense the game is changing perceptibly.
"I think we're definitely going in the right direction. It's been a lot better, there's a lot more awareness ... a lot of things have been going on in the last year or two," said Boston Bruins centre Patrice Bergeron, who missed 11 months of action because of concussion in 2007.
But where is the game headed, and at what speed? To understand hockey's cultural fabric today, it helps to look at the sport's roots.
While historians are far from unanimous on the birthplace of the sport, one thing they broadly agree on is that it started to become iconic in the early to mid-1800s, around the same time Canada came into being as a country.
"Old-time hockey is married to who and what Canadians think they are," observed Andrew Holman, a hockey expert and history professor at Bridgewater State University in Massachusetts. "Whenever there's been a crisis [in the game] it's been accompanied by a reactionary closing of ranks."
Or as Robidoux put it: "hockey is a part of Canada's national emancipation in a cultural sense."
To threaten the orthodoxy, then, is to attack that which the entrenched (mostly Canadian) hockey establishment sees as fundamental to its identity, and a defining element of the society that most venerates it.
Self-preservation is a powerful instinct.
In Holman's view, the current questions recall a similar period a century ago.
In the 1910s the angst was related to serious injury and deaths from stick-swinging incidents in senior and professional hockey, then in its infancy.
"There was a period of probably 10 years of media hand-wringing, public outcry and the authorities considering various options," Holman said. "The fix came when they opened up the game and allowed forward passing."
Among the reasons such a revolutionary idea came about, according to Holman: a parallel debate over football injuries, and aggressive top-down action on the part of league and team owners.
Former Montreal Canadiens great Ken Dryden, as thoughtful a chronicler of the game as there is, has been saying for years that the game needs another forward-pass moment.
Whether it can be conjured up is another question.
Robidoux pointed to recent, tentative shifts in the way players approach their safety as evidence that it is possible. But at bottom, he said, hockey culture amounts to "perpetual adolescence."
"It's a very insular culture, and it maintains a type of masculinity that isn't really relevant to other facets of life, like corporate environments," he said. "It's never-never land."
Robidoux, who spent a year cheek-by-jowl with minor-league players while researching his book Men at Play, held out several examples: players universally refer to teammates as boys, typically hire people to manage their finances, tend to socialize only among themselves, and have their luggage carried for them.
"How many other jobs can you think of where you're told when to have a nap?" he said.
Other hockey scholars, such as Holman, argue the roots of the current establishment culture – and the emergence of violence as a defining trait – lie in the democratization and later professionalization of the sport around the turn of the 20th century.
"That's really when winning by any means becomes really important," said Holman, who contends the game was a more genteel pursuit until the late 1800s.
In a recent e-mail, Dryden suggested the most interesting question to pose would be: imagine those people whose minds are hardest to change, and what it would take to sway them.
The answer is complicated, but in speaking to hockey people and academics who study the sport, it seems that a serious, career-threatening injury to the game's best player, Sidney Crosby, while a catalyst for self-examination, isn't enough.
As Laberge put it, social science and the breadth of human experience teach us that radical, revolutionary change is generally only brought about by the loss of life.
The current, intense focus on concussions dates back three or four years, and in that span there have been few fatalities, none in the NHL. They include Don Sanderson, a senior-league player who died after hitting his head on the ice during a fight.
Defenders of the orthodoxy mourn such tragedies like everyone else, all the while insisting hockey must continue to be a physical, violent game – that ridding it of donnybrooks and frontier justice would betray its traditions and make it a sport no longer worth watching.
There are also financial considerations at play: a healthy segment of hockey fandom loves fights and the sense of incipient danger that attends rough play.
And there are scores of people from owners to influential pundits and network television executives who consider radical change a threat to their livelihoods.
But as evidence mounts that head injuries can have long-lasting consequences, how long can the status quo prevail?
As in the 1910s, football is a little farther along the road to reform – the NFL has enforced a series of new measures over the objections of enraged traditionalists.
But hockey, as the experts point out, is an animal of a different colour.
"There is also one other thing that makes hockey different from the other major sports: there's no out-of-bounds," said Holman, who plays the game avidly "You can't really escape."Report Typo/Error
Follow @MrSeanGordonon Twitter:


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subset: commoncrawl


text: On Fri, Feb 19, 2010 at 01:13:19PM -0600, Sabuj Pattanayek alleged: > Hi, > > Is there a command to kill all active jobs? I'm going to use qstop and > qstart via cron to disable and enable running jobs on the queue during > certain periods but I want to also be able to kill all running jobs > after I run qstop. Is there a builtin command to do this either from > the torque or maui commands or do I just need to script this using the > output of qstat or showq -r ? qselect | xargs qdel -m "BOFH does not need usernames" -- Garrick Staples, GNU/Linux HPCC SysAdmin University of Southern California Life is Good! -------------- next part -------------- A non-text attachment was scrubbed... Name: not available Type: application/pgp-signature Size: 189 bytes Desc: not available Url : http://www.supercluster.org/pipermail/mauiusers/attachments/20100219/fa7e36e7/attachment.bin


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What do you wear to the Summer Olympics? Ask Duchess Kate.
The former Kate Middleton, now Catherine Duchess of Cambridge, is presenting a model of casual elegance in her appearances at the London Olympics, for which she and husband Prince William and brother-in-law Prince Harry are Olympic ambassadors.
She makes it look easy, in part because she's not afraid of repeating. Today she was jaunty in the same navy blazer she wore Monday, according to the HRHDuchessKate blog. She had on skinny jeans and a sailor-style navy-and-white striped top, her favorite high wedges, and she clutched an always-necessary umbrella.
She and William and Harry were in the stands with a thrilled crowd as royal cousin Zara Phillips took silver with the British equestrian team at the Greenwich Park stadium.
Tis is just the beginning: The young royals are expected to make more than two dozen appearances at various Olympic competitions over the next two weeks, besides attending more formal events in the evening as the duchess did Monday night.
No wonder Vanity Fair put her on its September cover under the headline "Kate the Great."
Sign up for Lifeline Live e-mail alerts


text:
Current Issues in the Diagnosis and Management of Wilms' Tumor
Current Issues in the Diagnosis and Management of Wilms' Tumor
Dr. Paulino has written an excellent review of our present knowledge of Wilms' tumor. Not everyone would agree, however, that ultrasound has replaced the intravenous pyelogram (IVP). The National Wilms' Tumor Study Group (NWTSG) recommends IVP together with real-time ultrasonography as the preoperative imaging studies for the abdomen.[1] The former is used to establish the presence of a functioning opposite kidney and of any congenital abnormality. The latter identifies the presence and status of inferior vena cava thrombi.
As neither CT nor MRI can determine, with certainty, the status of the opposite kidney, the NWTSG still calls for its mobilization at laparotomy, as Dr. Paulino notes. He appropriately points out that those who advocate eliminating exploration of the opposite kidney[2,3] base their recommendation on data from a very small numbers of patients, compared with the NWTSG data. The NWTSG, therefore, continues to recommend mobilization of the opposite kidney based on the accuracy and lack of morbidity of this procedure.
National Wilms' Tumor Study 4
As the results of the third National Wilms' Tumor Study (NWTS 3) were very good (see the author's Table 2), NWTS 4 (1985 to 1994) was designed to attempt to simplify and shorten treatment for those considered at low risk, while intensifying treatment for those at high risk. Although, in NWTS 3, the addition of cyclophosphamide (Cytoxan, Neosar) to vincristine, dactinomycin (actinomycin D [Cosmegan]), and doxorubicin achieved better results in patients with stages I to IV anaplastic Wilms' tumor, the numbers were uneven due to weighing of the randomization (21 patients receiving three drugs and 12 patients receiving four drugs), and it was felt appropriate to continue the randomization into NWTS 4.
For the remainder of the patients (excluding those with rhabdoid tumor of the kidney, who are not included in NWTS 4), the trial aimed to answer two questions: (1) Is pulse-intensive therapy (ie, a single dose of dactinomycin and doxorubicin for patients with stage III or IV, favorable histology and clear cell sarcoma) more effective than standard therapy (5 days of dactinomycin and 3 days of doxorubicin)? (2) What is the optimal duration of therapy (6 vs 15 months)? This issue of therapy duration was tested in all patients except those with stage I, favorable histology and anaplastic tumors.
A simplified version of the schema of NWTS 4 is shown in Figure 1. The results, which have been partly published in abstract form, suggest that 2-year survival rates are equivalent with the pulse-intensive and standard regimens,[4] and that toxicity (particularly hematologic) is less severe for most patients on the pulse-intensive arm.[5]
A recent analysis of NWTS 4 has also shown no demonstrable differences between the 6- and 15-month courses of therapy.[unpublished data, NWTS 4]
Patients with anaplastic tumors (stages II-IV) derived a benefit from the addition of cyclophosphamide to dactinomycin, vincristine, and doxorubicin if the tumor histology showed a diffuse pattern. No benefit could be discerned for focal anaplasia, however.[6]
National Wilms' Tumor Study 5
In NWTS 5, which began enrollment in 1995, there are no randomizations. The outline is shown in Table 1, and the radiotherapy regimens are detailed in Table 2. A major objective of the protocol is to determine the prognostic significance of the loss of heterozygosity of chromosomes 16q and 1p. This stems from an analysis of 232 patients from NWTS 3 and 4 showing that overall and relapse-free survival at 2 years was significantly worse for those with the 16q abnormality and was marginally so for those with the 1p abnormality.[7]
Also, as Dr. Paulino discusses, there is evidence that stage I patients under 2 years of age with tumors weighing less than 550 g may not need further therapy. In NWTS 1, 2, and 3, there were no differences in disease-free or overall survival in these patients regardless of the therapy employed.[8] In NWTS 5, therefore, these patients will be treated by nephrectomy only.
1. D'Angio GJ, Rosenberg H, Sharples K, et al: Position paper: Imaging methods for primary renal tumors of childhood: Costs versus benefits. Med Pediatr Oncol 21:205-212, 1993.
2. Koo AS, Koyle MA, Hurwitz RS, et al: The necessity of contralateral surgical exploration in Wilms' tumor with modern non-invasive imaging technique: A reassessment. J Urol 144:416-417, 1990.
3. Goleta Dy A, Shaw PJ, Stevens MM: Re: The necessity of contralateral surgical exploration in Wilms' tumor with modern non-invasive imaging technique: A reassessment (letter). J Urol 174:171, 1992.
4. Green DM, Breslow N, Beckwith JB, et al: A comparison between single dose and divided dose administration of dactinomycin and doxorubicin: a report from the National Wilms' Tumor Study Group (abstract). Proc Am Soc Clin Oncol 15:457, 1996.
5. Green DM, Breslow, NE, Evans I, et al: Effect of does intensity of chemotherapy on the hematological toxicity of the treatment of Wilms tumor. A report from the National Wilms Tumor Study. Am J Pediatr Hematol Oncol 16:207-212, 1994.
6. Green DM, Beckwith JB, Breslow NE, et al: Treatment of children with stages II to IV anaplastic Wilms' tumor: A report from the National Wilms' Tumor Study Group. J Clin Oncol 12:2126-2131, 1994.
7. Grundy PE, Tezerow PE, Breslow NE, et al: Loss of heterozygosity for chromosomes 16q and 1p in Wilms' Tumors predicts an adverse outcome. Cancer 54:2331-2333, 1994.
8. Green DM, Breslow NE, Beckwith JB, et al: Treatment outcomes in patients less than 2 years of age with small, stage 1, favorable-histology Wilms' tumor: A report from the National Wilms' Tumor Study. J Clin Oncol 11:91-95, 1993.


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By Mary Buckley
March 12, 2012 | 1 comment
The third time might be the charm for the redevelopment of several properties in the Mequon Town Center.
Blair Williams, president of WiRED Properties, consulted with the Plan Commission tonight on a concept for the redevelopment of two houses owned by Shaffer Development at 11215 and 11229 N. Cedarburg Road and a vacant city-owned lot between the northernmost house and City Hall.
Beginning in 2007, Cindy Shaffer brought plans for the site to the commission but the economic downturn effectively stopped planning for the site.
The latest plan for the site would be a two-phased development with the first phase including the two houses and city land. The American Legion Post, 6006 W. Mequon Road, would be redeveloped in a second phase. That building is considered a historic building and would require permission from the Landmarks Commission before it could be razed.
Williams told the commission that the first phase could stand on its own. Under discussion is a mixed used, three and half story building that would face Cedarburg Road. Williams said between 8,000 to 11,000 square feet of first floor of the building would be dedicated to retail or office use, with 20 to 25 apartments on the upper floors.
Surface retail parking would be accessed from an entrance on Cedarburg Road, which would require a median cut on the street to allow access for northbound vehicles.
The underground residential parking would be accessed through the City Hall parking lot off Cedarburg Road.
Williams said Cindy Shaffer, owner of Shaffer Development, is discussing the Legion property with its owner. Currently, the proposal for that site calls for a commercial building of about 14,000 square feet. The discussions include having the Legion post as a tenant in the new space.
The project would require the city land, access from Cedarburg Road and tax incremental finance district assistance. The project would likely be eligible for TIF incentive plan assistance. Williams said it would help bridge an anticipated gap in financing.
WiRED Properties has redeveloped two major areas on North Oakland Avenue in Shorewood. The Cornerstone property, which has retail on the first floor and apartments on the upper floors, was completed more than a year ago. A similar development, The Ravenna, is under construction across the street.
Williams said he believes there is "significant untapped demand for new multi family construction and a desire for new retail" in Mequon. Although he did not name any businesses, he said he has had conversations with a bar/restaurant owner and the owner of a café. Both are interested in the retail space available in the first phase of the project.
The Plan Commission indicated support for the concept and will see architectural and site plans at a later date.
Before the plan would come back to the commission, Director of Community Development Kim Tollefson said the finalized concept would go to the Economic Development Board for a discussion about the sale of the city-owned property and the TIF incentive program.
Tollefson said the plan "advances the spirit and intent of the Town Center" and has staff support.


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"I had a great visit," Lacy said. "The pre-game atmosphere on the sidelines, the crowd in the second half and seeing the facilities were the things that really stood out most for me. The pre-game was great. When me and Christian (Morris) walked out of the tunnel, the fans were going crazy. It was cool watching the linemen warming up and they have a great crowd for sure.
"They started off slow but in the second half, they got rolling and it was the loudest I've ever heard any stadium, ever. That crowd was crazy in the second half and it was a really fun game atmosphere to be at. The city was cool, the people were great and overall, I think Nebraska is definitely a place where I could fit in at well."
So the obvious question is, how strong is Lacy's commitment to UCLA after the visit?
"I'm a Bruin man, I'm solid," Lacy said. "Nebraska was great, it's a great school but I'm a Bruin. I'll be going to UCLA unless something drastic changes and I can't even think of what that would be. I loved it at UCLA and there's a reason why I committed there."


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Father: Harold Leon Morrison b: 17 JUL 1925 in Menlo, Chattooga County, Georgia
Mother: Alice Esma Roberts b: 26 DEC 1925 in Jamestown, Cherokee County, Alabama
Marriage 1 Living Acker
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text:
Scuffles are predicted as members of the opposition Orange Democratic Movement plan to sit on the government benches in protest at Kibaki's disputed re-election.
"It's going to be very acrimonious. We should definitely expect some drama," XN Iraki, a political analyst, told Reuters.
Both Kibaki and the opposition leader, Raila Odinga, are due to be sworn in as legislators during the session.
Despite international pressure for the two to meet for talks, today will mark the first time they have been in the same room since Odinga accused Kibaki of rigging his re-election.
More than 600 people have been killed and hundreds of thousands displaced since the disputed poll.
Security was tight before today's session, with soldiers deployed around the parliament building and some roads in the area blocked off by riot police.
Neither party has the two-thirds majority needed to elect the speaker. Odinga's party has 99 seats in the 222-seat legislature, compared with Kibaki's 43.
The ODM hopes to elect its candidate, Kenneth Marende. Both sides have been courting legislators from minority parties in the hope of drumming up support. Independent Kenyan analysts have warned that both parties may be trying to bribe parliament members to secure votes.
Salim Lone, an opposition spokesman, said, "We are going to go to parliament and sit on the government side and that will make clear who won the election."
The former UN secretary general Kofi Annan is expected in Nairobi tonight, after mediation by the United States and the African Union last week failed to bring Kibaki and Odinga together for talks.
In an effort to step up pressure on Kibaki, Odinga called for three days of protests across the country to start tomorrow, in defiance of a government ban on demonstrations.
Plans for a similar protests earlier this month were postponed after demonstrators were dispersed using water cannons, teargas and gun shots.
Speaking in Geneva, Annan appealed for calm before his departure and alluded to the proposed protests.
"Pending this (mediation), no party should create facts on the ground or engage in acts that complicate the search for a negotiated solution," he said. "The purpose of our mission is to help the Kenyan people find a peaceful and just solution to the current crisis."
Annan faces a tough task. Yesterday, the public works minister, John Michuki, a member of Kibaki's inner circle, said Annan had not been invited.
"We won the elections. We do not see the point for anyone coming to mediate power-sharing. We have not invited Kofi Annan or any other eminent personality to come and mediate," he said.


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OCZ Technology has launched its RevoDrive series PCI-Express SSDs, which deliver speeds over 500MB/s reads and random small file writes up to 80,000 IOPS.
The OCZ RevoDrive will be initially available in 120GB and 240GB capacities, with MSRPs of US$389.99 and US$699.99, respectively.
- Wireless broadband developments in Southeast Asia markets
As of 2013, the 10 ASEAN nations had a total of over 700 million mobile subscriptions, with the CAGR from 2003-2013 reaching 24%. This Digitimes Research Special Report analyzes the various mobile broadband markets in ASEAN and looks at the respective trends in 4G LTE development for those markets.
- 2015 global tablet demand forecast
This Digitimes Research Special Report provides a 2015 forecast for the global tablet market and analyzes the strategies of key market players such as Google, Apple, Intel, and Microsoft.
- 2015 China smartphone panel trend forecast
This Digitimes Research Special Report analyzes the strategies of key China-based major panel makers BOE, Tianma and IVO for attacking the different market segments through technology and pricing, and their relationship to local vendors Huawei, Lenovo, ZTE, Xiaomi and Coolpad.


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This review is from: G.SKILL TridentX Series 16GB (4 x 4GB) 240-Pin DDR3 SDRAM DDR3 2666 (PC3 21300) Desktop Memory Model F3-2666C11Q-16GTXD
Pros: Fast! My setup is an ASUS P8Z77-V-Deuxe MB, i7-3770K @ 4.6GHz(P95 stable), Corsair H100 cooler, a CoolerMaster 1200W PS and two GTX 680's in SLI. Except for one experience(see below), G.Skill memory has always worked per spec the first time!
Cons: Cost, No stable OC @ 2800 and above yet
Other Thoughts: I had originally bought the 2600 version of this memory and could not get a stable XMP OC above 1921 MHz. I talked to Tim(very helpful) at G.SKill and tried everything he suggested(i.e. Vccsa should be @ 1.25v), but still no luck. RMA'd the part back to NewEgg for a refund and decided to buy the 2666MHz set. 2nd time was a charm, and NewEgg really treated me very well during the whole process!!READ FULL REVIEW
This review is from: G.SKILL Ripjaws X Series 16GB (4 x 4GB) 240-Pin DDR3 SDRAM DDR3 1866 (PC3 14900) Desktop Memory Model F3-14900CL9Q-16GBXL
Pros: None
Cons: None
Other Thoughts: This build was my first build in almost four years when I built two machines with QX9650 Extreme CPU's. This build was much easier and faster. I spent a lot of time four years ago getting 2 GB Corsair DDR3 1800MHz (XMP) memory to work (stable). The G.SKILL 1866MHz (XMP) worked right out of the box with zero problems!!READ FULL REVIEW
This review is from: Corsair P256 2.5" 256GB SATA II MLC Internal Solid State Drive (SSD) CMFSSD-256GBG2D
Pros: I own three of these P256's. I have two in a RAID 0 cofiguration in my ASUS P5E3 Desktop(Win7 64 bit WEI score=7.9). I have another one in my new ASUS G73JH-A1 Laptop(Win7 64 bit WEI score=7.4).
Cons: Price and Babysitting -- you have to ocasionally do a secure erase with G-Parted to get them back to top notch performance. Trim commands work with single drives and Win7, but don't work yet in RAID(multiple drives) cofigurations. I will be very happy when the RAID controllers allow the Trim command to be passed through to the SSD Drives.
Other Thoughts: They really shine in my ASUS G73JH Laptop!!!READ FULL REVIEW


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Richard L. Kastenhuber, 64, of 2422 Slater Road, died at 3:30 p.m. Wednesday, March 20, at Salem Com
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World Of Warcraft: Outland & Draenor Video Comparison
World Of Warcraft's next expansion pack – Warlords Of Draenor – is set in the past, prior to the orc invasion of Azeroth.
That means it's also prior to Draenor's destruction, which we now know as Outland. Though many of the areas revealed so far aren't parts of present day Outland, there are some similarities.
With the beta underway, we decided to take a look at Shadowmoon Valley to find out how the two differ in Outland and Draenor.
We've created a video comparison, taking a look key landmarks in Outland to find their equivalent in Draenor. While it's completely different, Blizzard has made sure the 'older' version in Draenor retains some of the more recognisable landscape features.
Take a look in our World Of Warcraft Outland and Draenor video comparison:


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FRENCH-L ArchivesArchiver > FRENCH > 2004-03 > 1080492595
From: "Judith J. French" <>
Subject: [FRENCH-L] John French of Woodbridge, NJ vs. Aaron
Date: Sun, 28 Mar 2004 11:49:55 -0500
Hi French Family
I was asked on the list about the relation of Aaron Sr. & Jr. with my David
Sr. & Jr. I will try to answer as best I know so far. I want to put things
out there so more families can be searching. This is long and I hope it
prints out.
I have an original copy of the little red book sometimes referred to as the
Aaron Book of 1910 compiled by Charles Newton French, handed down to me from
my father and I assume came from Elston Marsh French (1868-1931) my great
grand uncle, his father being Phineas Mundy French of N. Plainfield, N.J..
Phineas had two wives and many children also. My family line is from the
first wife, Elston from the second wife. My father and his siblings
inherited Elston's estate. I have seen how corrupted the Aaron book has
become regarding John of Woodbridge and sons John & Richard have become.
The first five pages of the book clearly states that it is a compilation of
many New England French families not related to each other but he listed
them. He clearly states that it is unknown who the father of Aaron Sr. 1739
is. Here is the Preface of the first page of Charles Newton French's book
of 1910, I will type now:
"This small volume contains a portion of the genealogical data which the
writer has collected, at odd times, during several years past. The greater
portion of that which refers to the French families of New England, and a
few others, has been gleaned from books; it is published herein because the
compiler believes that some will be interested in such sketches of early
settlers from whom have descended so many persons of the name French who now
reside in the United States. The genealogy of descendants of Aaron French,
with a few exceptions, has been compiled from unpublished records, public
and private, from epitaphs and traditions. During the Revolutionary war,
Aaron lived in a part of New Jersey which suffered much at the hands of the
British. Many church, town and other records were then lost which, if
preserved, would have been useful in the preparation of this work. That
Mary Clark was the name of the first wife of Aaron French, is traditional,
but it is so generally believed that the compiler accepts it as true; she
probably died December sixteenth, 1795. Other Clarks who have intermarried
with Frenches, belong to several distinct families. The compiler takes this
opportunity to express his appreciation of the services rendered to Rev.
Alfred Connet, editor of Genealogy of the Connet Family, Mrs. Mary Ann
(French) Clark and Mrs. Sarah (French) Ripley, who furnished copies of Bible
records of the three sons of Aaron French, and other data; and all others
who have aided in this compilation. Charles Newton French. Chicago, August
8th, 1910."
He then lists the following families of various places in small paragraphs
with their family starting with William French (1603) of Ma., Edward French
(1590) of Ma., Thomas French who was at Ma. in 1638, who went to Conn., in
1643-50., John French of Dorchester, MA removed to Braintree, Ma (1612),
Philip French of New York City, speaker of General Assembly whose son Philip
bought land in New Brunswick, New Jersey, with one of his daughters Susannah
married Wm Livingston, Gov. of New Jersey 1776-90., JOHN FRENCH lived at
Woodbridge, NJ as early as 1670., Thomas French, who established the Quaker
colony in West New Jersey, b. 1639 in England and died 1699 in N.J., He
came to America in 1680, and last mentioned is Francis French, who lived at
New Gretna, New Jersey was born probably before 1740 in Monmouth Co. NJ, he
had on surviving son Thomas who married Hannah Johnson.
Next I will copy what his paragraph says about Aaron:
"Aaron French, (1) b. Sep. 8, 1739, lived at or near New Providence, N.J.,
as early as 1764, and in Essex Co. until shortly before 1790, when he moved
to Pa.; he d. Aug. 31, 1805. It is said that he had two or more brothers,
on of whom removed to N. Y., and another "went south". The compiler
believes that the latter was Francis of New Gretna, in the south of N. J.;
and that Joseph of Raritan Landing, Francis of New Gretna, and Aaron (1) of
New Providence, were sons of Joseph (Thomas, Jr., Thomas), of Shrewsbury;
however, Aaron (1) may have been s. or gr. s. of John (John) of Middlesex
Co., whose desc., unlike those of Richard, the other s. of John of
Woodbridge, have not been traced. The genealogy of descendents of Aaron
French (1) follows, in this volume."
What he is saying, is John Jr. is the one that work has not been done on
until recently. Richard is established and so is John Sr. So, if you read
all of the pages it is all "compiler believes" so it still open. John of
Woodbridge, bricklayer and his sons John and Richard are separate entities
and came from England when skilled men were hired and imported from England
on settlements. Woodbridge was settled by men from Massachusetts, not
Quakers, and listed documented landowners along with John. In another email
I will type each small paragraph in order. I have seen some paragraphs on
some websites but not complete. I will also copy more info from some of my
family books. I am hoping other French members might recognize something
and we can learn more.
Supposedly Charles Newton French would be my 4th cousin 3 times removed but
I am not sure about it yet. The DNA is only as good as a documented
contributor. My old handwritten family tree, had my John listed as the John
French of 1612 of Braintree, Mass. As far as I know, that John line lived
and died in MA. The dates do not match anyway. My original family is East
New Jersey. The current DNA result for John 1612 shows no relation either.
The possible closeness to my distant cousins on the 3 marker difference
might be through John Jr., Richard's brother, whose son William's children
and relatives had names of Noah, Aaron, Moses, Joseph, Nathanial, Lafford,
etc. It seems Richard went one direction and John Jr. went another. My side
of the family names seems to have remained the same like John, Richard,
Robert, David, Isaac, James, Charles, William, Edwin, Sarah, Mary, Susannah,
Margaret, Ann, Phoebe, Elizabeth, etc. Later first and middle names came
from the wives side of the families, like Badgley, Willis, Mundy, Wilcox and
Noe. These families were established along with mine of the Passaic Valley
with the David Sr. & Jr. I do hope this year to at least put the family on
line. There is still work being done by others and wish not to do that yet.
I have given info to some people on their websites when I found the need to
correct errors on intermediate family.
So far the DNA of my first cousin, son of my father's oldest brother has not
had an exact match yet with any of the Frenchs being tested so far. It
shows 4 markers off the current Thomas Quaker line of West Jersey which
means perhaps 1000 years ago somewhere in Europe and then 3 markers off from
the other Frenchs which still puts it back in Europe. There is only one
Aaron DNA 12 marker test listed showing and not 25 markers and shows as 3
markers off in just 12 from my family, yet it appears there are a lot of
marriages between all of the New Jersey families. I am still open about
possibilities but from what I know so far John of Woodbridge was in East
Jersey and owned land 10 years before Quaker settlement in N.J.
I had in the beginning the wrong info on a family tree that a member did
years ago out of misinformation in many corrupted books. The first part of
the tree is correct but the bottom part was wrong. I started adding up the
history and it did not jive. Then another member posted my family before I
knew them and tied it to the Thomas line. So as you venture around
websites, you will find my family adopted into three or more lines. I am
fortunate to have the family bible of 1861 and another relative has the one
from 1855. I have photos also of David Jr. and Margaret Noe and above them.
I believe my John corrupted into a few Thomas books that are in circulation
and is an assumption. There are Richard's, John's and David's in that line
but I think things have gotten mixed up including sticking my David line
into it with wrong siblings and wives.
My family connection, I believe, is through John of Woodbridge, his son
Richard, his son Robert Sr. which I mentioned in an earlier email who is the
father of my David Sr. listed on the DNA. There is current work being done
on this line. We need to find more DNA participants of the New Jersey
Frenchs. Robert Sr. had two wives and many children. My line is from the
first wife. My original family remained in New Jersey but is seems second
wives moved the families to New York. Three related Frenchs of these
families were found in New York but have refused to test with DNA even
though it would be paid for. It is a shame. So many lines are daughtered
out too.
Aaron: Military Service: American Revolution, Honor DAR Patriot
Index-Centennial Edition.
Reference in book by Charles Newton French: Aaron French lived at New
Providence, New Jersey as early as 1764 and moved to Washington Co.
Pennsylvania between 1787-88 where he acquired a large tract of land lying
four miles southwest of Amity and partly within the present county of
This is how the actual paragraph reads on John of Woodbridge:
"JOHN FRENCH (ffrench) lived at Woodbridge, N. J., as early as 1670; owned
lands there and on Staten Island; d. 1713, at Woodbridge. His will names his
w., Susannah, dau., ANN (w. of James Leith), and sons, JOHN and RICHARD.
John (John) lived in Middlesex Co. as late as 1721; Richard (John) settled
near Springfield, Essex (now Union) Co.; Elston Marsh French, Plainfield, N.
J., and others of that vicinity, are desc. of Richard. "
(The full original unpublished will states son John and son Richard)
The Aaron book states the other theory that Aaron descends from John Jr.
brother of Richard. John French of Woodbridge is AKA John the bricklayer.
Lived and owned land 10 years before the Quakers came to West Jersey. My
family is East Jersey. I would like to see the documentation that backs up
the connection between John, brick mason in Woodbridge in 1670's and Thomas
from Nether Heyford in Burlington in 1680.
Another theory is Noah known as the express rider descends from John Jr.
That is being worked on by another member, whom I believe is correct.
Taken from book entitled French Family History, American Genealogical
Research Institute, Heritage Press, Inc. Washington, D.C. 1977 listed in
Patriots and Veterans page 75, Noah French born 1754, died 14 November 1843:
private in a New Jersey Unit; married (1st) to Mary Rolph and (2nd) to
Joanna Ralph Campbell.
Posted on Ancestry.com website, Sep 6, 2003 Entry 1629 by Jan-Marie McCabe:
"Noah French enlisted in the Fall of 1775 and served at various times
amounting to two years as a Private and Express Rider under Captains Peter
Layton, Stephen Day, David Bates, and Jeremiah Dunn. He enlisted again on
June 12, 1788 as an Ensign under Captain Dunn and Colonel Nathan Luse, and
again on October 13, 1789 as a Captain under Captain Dunn and Colonel Luse.
Noah's final enlistment was on June 5, 1793 as a Captain in the Sussex
County (New Jersey) Militia. During the Revolutionary War, Noah engaged in
the Battles of Middlebrook, Newark, and Springfield (New Jersey). [Source:
Noah French Military and Pension files, National Archives, Washington, D.C.]
Lived at Morristown, NJ during Revolution. Enlisted and served two years,
first as a private and express rider of Light Horse in Captain Dunn's
company, New Jersey Lin, under Colonel Ludlum. After the war he moved to
Mendham, NJ, and thence to Bridgeport [sic], VT. About 1802 he moved to
Essex Co, NY, and it was while there that he made application for and
received his pension. Filed with his application for pension is a leaf torn
from his family Bible, which records his two marriages and the children of
each marriage. [Source: Colonial & Revolutionary Lineage of America, Volume
3, page 211] "
ELSTON MARSH FRENCH, Plainfield, N. J. (9321). Son of Phineas Munday and
Sarah J. (Lees) (2nd wife) French; grandson of David and Margaret (Noe)
French, Jr.; great-grandson of David French, private New Jersey Militia;
great-grandson of Lewis Noe, private Second New Jersey Militia, pensioned.
(Taken from Alberta Lisk)
"Records of the New Providence Presbyterian Church on Turkey Creek, N.J.
show that Aaron and 3 of his children were baptized there between 1764 &
1775. Church's seating list include: Abraham Clark, David Clark, David
French and Aaron French. About 1787 or 88, moved to Washington Co. PA where
he acquired 400 Acres of land lying 4 mi. SW of Amity and partly within the
present Greene Co. PA. He named it "The New Republic".
Taken from book entitled French Family History, American Genealogical
Research Institute, Heritage Press, Inc. Washington, D.C. 1977 listed in
Patriots and Veterans page 70, David French born 6 February 1747, died 8
October 1838; private in a New Jersey Unit; married to Sarah Wilcox.
Head Stone, United Methodist Cemetery, 1441 W. Springfield Ave, New
Providence, NJ says:
David French, Pvt. Somerset Co., NJ, Militia, Rev. War, Feb 6, 1745, Oct 8,
1838 (Sr)
Head Stone, United Methodist Cemetery, 1441 W. Springfield Ave, New
Providence, NJ says:
David French, died July 24, 1879, aged 93 years, 5 months, 24 days. (Jr)
-----Original Message-----
From: Doris Smith [mailto:]
Sent: Tuesday, March 23, 2004 2:11 PM
Subject: Re: [FRENCH-L] David French NJ
Judith...what is the connection between this David
French and Aaron French 1739-1805 married to Mary
Clark? Aaron was a member of the New Providence
Presbyterian Church.
Is your David French buried in New Providence, NJ in
the cemetery across from the Methodist church?
Doris Smith
--- "Judith J. French" <>
> http://www.warrennj.org/wths/index.htm
> On the opposite corner from Moore and Genung was the
> property of David
> French. (SR)French, who was born in 1747 and served
> in the Somerset Brigade,
> came from Connecticut Farms and married Sara Wilcox,
> daughter of Peter
> Wilcox, on May 30, 1776. The Frenchs had ten
> children and their son David
> became a prominent member of the Union Village
> church.
> By 1844 Union Village had four classes led by David
> French, (JR) Martin
> Ruckman, William Clark, and Lines Tucker and
> seventy-four members. The
> church did not continue to grow, however, and in
> 1852 had only fifty-four
> members and three classes led by David French,
> Daniel Hand and Daniel W.
> Day.
> Judith J. French
This thread:
|[FRENCH-L] John French of Woodbridge, NJ vs. Aaron by "Judith J. French" <>


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Art Print
Milwaukee Transit System Mapby Carticulate Maps
Collect your choice of gallery quality Giclée, or fine art prints custom trimmed by hand in a variety of sizes with a white border for framing.
This map of a theoretical rapid transit system is a take on what local and regional rail could look like in Milwaukee, WI. It's design is inspired by multiple rail maps from systems across the US and more than just a basic theoretical system looks to embed layers of history and political challenges that are hidden in transit maps across the nation. And above all, it's fun to have a transit map for your city.


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Google: Download a Million Books for Free
Google yesterday announced that the company has released more than one million out of copyright, public domain books in the EPUB format. Google Books titles are already available in PDF form but the addition of EPUB means it'll be easier for users to read downloaded Google Books content on various different devices that support the format.
The announcement comes at a time when Google Books is facing investigations from both the Department of Justice and the European Union with regard to a deal made with the Authors Guild last October. A June report in the NYT says the inquiry is a result of complaints made by concerned critics who say the settlement would give Google an exclusive license to profit from millions of books.
The Authors Guild took legal action against the search giant after Google struck deals with major university libraries to scan and copy millions of books in their collections. While many of these were older books in the public domain, millions of others were still under copyright protection and the Guild said Google's scanning was "a plain and brazen violation of copyright law." The Mountain View company maintained that the digitizing of these books represented a "fair use" of the material, however the two agreed to settle in a $125 million agreement. Google's $125 million was to go toward legal fees, establishing Book Rights Registry and at least 45 million for authors and publishers whose in-copyright books and other copyrighted texts had been scanned without permission.
Legal issues aside, anyone plan on downloading some of the EPUB content? Sony's latest ereader supports the format, which will likely make it an attractive option to those shopping around for a device to read all these Google Books on. Let us know what you think in the comments below!
ssalimIf they are still under copyright protection, why did the major university library agreed with Google on scanning their collections?
Oh sure, don't help kids gain knowledge and education by reading library's public books online and at the same time saving gas going to the library and/or time going there.
Stupid lawyers.
doomtombInformational books should just be public domain anyway. Authors and book publishers are still doing things the old-fashioned way. All this information is accessible via the internet anyways. Like my college forcing me to buy new editions of textbooks all the time just so the bookstore and the publisher can make more money. $150-200 for some books in just absurd. Save a tree, download the .pdf.
ssalimIf they are still under copyright protection, why did the major university library agreed with Google on scanning their collections?
Oh sure, don't help kids gain knowledge and education by reading library's public books online and at the same time saving gas going to the library and/or time going there.
Stupid lawyers.
AnonymousNevermind that they're out of print, although I suppose that all of these authors think that THEIR intellectual property is "special", and that they should be "compensated", despite the book being out of print and having an expired copyright(that's like 20 or 50 years?). 99.999% of all books out there are complete garbage anyways...
doomtombInformational books should just be public domain anyway. Authors and book publishers are still doing things the old-fashioned way. All this information is accessible via the internet anyways. Like my college forcing me to buy new editions of textbooks all the time just so the bookstore and the publisher can make more money. $150-200 for some books in just absurd. Save a tree, download the .pdf.
Very true. The other way is just as annoying as colleges all try to take advantage of textbooks. One professor which I took class from, always use out-of-date and out-of-print books. Since students can't get the book anywhere, they have to resolve the problem themselves. It would be much simpler to have those book of public domain available for download.
Don't be a hypocrite! You aren't going to college so you can give your work away for free.
Sure, you could get most information on the internet, but how do you know it is accurate. When writing essays, you need to be able to site a reliable source. Unfortunately, Wikipedia doesn't count. It takes a long time to write a text book, whose information also has to be sourced. Also, most text books aren't published, creating additional cost for the author. How would you like to write a text book for free? You may not agree with the price or the medium or the book (paper or digital), but that is a different story.
azxcvbnm321Yes everything should be free including your labor. People should just work for free, I mean they don't have anything else to do anyway other than sit around. Might as well be scrubbing my toilet while chatting with their friends on Bluetooth right?
And because one person decides to give something away, that means everything should be free too. I had a friend who didn't want this almost new couch anymore a couple of years ago and I took it. Now that this couch is broken down, I was thinking of going to the furniture store and telling them that they should give me those couches they have for free. I mean I could find a couch on the street somewhere, they're all free anyway so why is this greedy store trying to sell me something that I got one time for free?
Dude, I also remember that McDonalds had this free promotion a while ago for a small cup of their new iced coffee. Well it's pretty hot today so I went in there and asked for more free coffee, but they looked at me and said there wasn't anymore free coffee! Can you imagine that? It should be free, water costs almost nothing and neither do beans. Hell, I can get free coffee from my moms at anytime, coffee should be free.
Which reminds me that I'm moving soon and the last time I moved, my friends all helped me for free. I've never had to pay anyone to help me move before, it was all free. I think movers should work for free, it's not like people have anything better to do. I can always find friends to help me move for free so I think I should be able to ask anyone off the street to help me for free too. I mean why are people wanting to be paid when I got this for free in the past?
Humm, somehow I'm in college, but I don't understand why everything isn't for free. Maybe once I graduate from college, I'll understand why people want to be paid for work, even though in the past I've been able to gain from their work for nothing. You know, I have this friend who says that maybe those people who gave away their work were just being very very generous people and that I should be grateful, but I'm the one in college and I say that I'm entitled to whatever it is I want. I want it so I should have it for free. It's the people who want to be paid for work that are greedy, it's not that the people who give away stuff for free are generous.
I'm in college and I'm outraged that I have to pay for anything. These professors, all they do is talk during class but they expect me to pay for hearing them talk! Well I've heard people talk before for free, and I always talk during class for free too, for some reason others don't like that and don't appreciate my free talking, but I do it anyway. Yet this professor is so greedy that he wants me to pay to hear him talk. Talk should be free. I'm in college and I'm educated so I'm more right--I'm not more stupider than you. The people who make fun of the educational system and the falling standards don't know what they are talking about. I'm in college and I'm smart, I'm not stupider than nobody. I'm more better than you. Everything should be free.
on the one hand "just scanning them" begs the question "for what?" also, copyright notices on published material often have something about not reproducing, storing or transmitting too. i'd guess scanning counts as storing and that's partly why the Googlers paid the fine.
BlessedmanI must agree on some points that yes these authors spend a lot of time getting this information together for textbooks, maybe if our governments really cared about the future of this planet they would pony up the dollars and give these books away so everyone can learn from them. Education is one of the most important things in the world and yet if you don't have the money you don't get one... Now that truly is ignorant. I agree with google's ideology of "Easier to ask for forgiveness then permission". Plus everyone keeps crying about the authors, what about the copyright lawyers? If people didn't violate these laws those poor guys would go hungry, we need more violators! Everyone start scanning their textbooks and emailing the PDF's to everyone they know and we will make it like a chain letter. Until it all becomes futile to try and stop the movement!


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Hello, I guess I'll call these issues I've been having "attacks". The first one was about a year ago now. I was asleep (it ONLY happens when I'm asleep in the middle of the night) and suddenly I had painful chest pain that woke me up. I felt like I couldn't breathe. Tried to walk to the bathroom because nauseous and dizzy kept "blacking out" and falling. The paramedics were called. They hooked me to a EKG machine and told me to stand up, when I stood up my blood pressure dropped to 38/60. I began to feel better (and I didn't have insurance) so I decided against them driving me to the hopsital. It has happened 3 times since then however the first time was the only severe time. But I find myself waking up in the middle of the night with chest pain feeling like a weight is on my chest and I can't breathe. I'm 23 years old. I've never been told I have a heart murmor (but my mom has one if that matters). I've never had any other issues and like I said this just started a year ago. I'm not overweight or underweight and I excersie regularly and eat decently amd always have. I have never gained or lost a significant amount of weight and have never had issues with my heart (until now I guess). What could it be? Thanks!READ MORE
Chest pain randomly only when sleeping & dizziness. What could it be? Help!
Hi KMC1990,
I encourage you to contact a doctor for an evaluation and to determine the cause of these symptoms/episodes.I realize you still may not have insurance, but contact a doctor's office to learn your payment options and cost when scheduling an appointment.
All the best,
Lisa Nelson RD
FROM OUR COMMUNITYYou should know Answers to your question are meant to provide general health information but should not replace medical advice you receive from a doctor. No answers should be viewed as a diagnosis or recommended treatment for a condition. Content posted by community members does not necessarily reflect the views of Remedy Health Media, which also reserves the right to remove material deemed inappropriate.DISPLAY TEXTLINK URL
Most PopularLiving WithWhat to Eat With Atrial FibrillationLiving WithCelebrities with Atrial Fibrillation


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Forums: Index > The Wizengamot archive > Slug club members
In the 6th film, there are several photos of former Slug Club membwes. Were others shown that weren´t in the book? I only know about Regulus. Lucius and Wendy Slinkhard.--Rodolphus 12:42, September 18, 2009 (UTC)
- Barnabas Cuffe was present in the movie, too. I heard about Wendy Slinkhard, but I do not recall seeing her on the film. -- Seth Cooper owl post! 10:14, September 21, 2009 (UTC)
Gwenog Jones is also seen. Were there any members that were not memtioned in the book at all?--Rodolphus 17:16, September 23, 2009 (UTC)
As the DVD has been released, we can get closer looks and I repeat the question.--Rodolphus 18:01, November 20, 2009 (UTC)
- The DVD has only been released so far in Australia, Brazil, Israel, the Philippines, and most of Europe.[1] The UK will have to wait until December 7th for the DVD, and Canada and the US until the 8th, but there's a gallery of screenshots available at the Leaky Cauldron. ★ Starstuff (Owl me!) 06:48, November 21, 2009 (UTC)


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Hand and Foot Jams for Crack Climbs
Jamming isnt something you learned by climbing trees as a child. Instead of grabbing normal holds, you wedge body parts into cracks. Itll take some practice, but once you learn the techniques, cracks become your roads to success on all kinds of rock. And when the crac...
Jeff Achey at Climbing Magazine
6 Crucial Wide-Crack Techniques
I was barely halfway through a 90-foot route when I used the last of my breath to wheeze Take! Blood from my skinned elbows leaked through my shirt, and sweat dripped into my eyes when I realized I simply couldnt climb anymore. The route was the classic 5.9 offwidth...
Matt Kuehl at Climbing Magazine


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Stanley Cooper
Stanley Cooper (August 4th,1926 / New York City)
» Click to list general statistics » OR, select a date :
Fountain Of Youth
Ponce de Leon looked high and low
In Florida, of all places
He searched for it so long ago
But could find no Fountain traces
read more >>
- Hits: Number of visitors who read the poem on the given date.
- Posts: Number of visitors who posted the poem to a friend (via e-mail) on the given date.
- Dates that have "0" hits may not be shown on the list.
- Dates, poems and poets that have "0" hits may not be shown on the list. Statistics are generated daily.


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Where Am I Eating? by Kelsey Timmerman is the 2016 book choice. From September 1 to October 31, people throughout Tippecanoe and White Counties are reading, meeting, and discussing this one book. New York Times bestselling author Timmerman will be here for three programs September 1!
Explore Timmerman's stories from a four-continent farming adventure. Read about Colombian farmers growing coffee, modern-day slaves on an Ivory Coast cocoa farm, and Chinese apple farmers.
America increasingly relies on imported food. Find out how this impacts us and other people around the world. Join us in programs for all ages.
Revisiting the Family Farm
Author Fred Whitford
Wednesday, September 21
7:00 p.m., Downtown
Book Sale & Signing
See never-before-published photographs of Indiana farms. Ask questions. The author of Enriching the Hoosier Family Farm, Fred Whitford, is also a Purdue Clinical Engagement Professor.
Ice Cream Cone Day Stories
Thursday, September 22
6:30 p.m., Klondike & Wyandotte
It's National Ice Cream Cone Day! Families enjoy "Ice Cream Specialties" and learn about frozen sweets. A craft and treats are included.
Hunger & Food Insecurity
Tuesday, September 27
7:00 p.m., Downtown
For one in six Tippecanoe households hunger is a reality. Indiana's Emergency Food Resource Network (IEFRN) experts discuss the issues and local resources.
National Coffee Day: Baristas & Connoisseurs
Thursday, September 29
6:30 p.m., Klondike
Learn about Fair Trade practices, standards, and producers. Chat with Greyhouse Coffee representatives. Participate in a tasting.
Candy Crush
Saturday, October 1
3:00 p.m., Downtown
Hear sweet stories. Analyze the ingredients and geography in popular candy bars. Join in experiments, games, and tastings. Grades K-6.
Discussion: Where Am I Eating?
Thursday, October 6
12:00 p.m., Downtown
Bring a lunch or snack and talk about the book with committee member and Reference Librarian Sherri McGlothlin.
Family Farmers' Market & Stories
Monday, October 10
6:30 p.m., Klondike
Hear special Farmers' Market stories. Enjoy a take-home a craft to cut, glue, and paint.
Food For Thought
Author David Hoppe
Monday, October 10
7:00 p.m., Downtown
Discover passionate Hoosiers who are creating our state's contemporary food scene and learn about this writer's discoveries while researching his book, Food For Thought: An Indiana Harvest.
Discussion: Where Am I Eating?
Tuesday, October 11
7:00 p.m., Wyandotte
Chat about the book with committee member and Wyandotte Branch Manager Leanne York.
Eating Local: Jodee Ellett
Purdue Extension
Wednesday, October 19
7:00 p.m., Downtown
Purdue Extension Local Foods Coordinator Jodee Ellett discusses how our food shopping choices help create a strong, local food system for Greater Lafayette.
One Great Read committee members are required to host at least one activity per year. Anyone interested in joining the committee or hosting an event may call 765-429-0113.
All library programs are sponsored by Friends Associations.
Peggy Fields, Barnes and Noble Booksellers Community Relations Manager, 449-4330,


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RailRiders Smack 16 Hits and Blank IronPigs, 7-0
SWB Scores Three in the First and One in the Second to Set Tone Early
By Andrew Kappes / Scranton/Wilkes-Barre RailRiders | June 15, 2013 6:05 PM ET
Allentown, Pa. - The Scranton/Wilkes-Barre RailRiders (Triple-A/New York Yankees) smacked 16 hits and shutout the Lehigh Valley IronPigs (Philadelphia Phillies) on Saturday night, 7-0. Zoilo Almonte accounted for four of those hits, and Walter Ibarra doubled twice to drive in two runs.
Scranton/Wilkes-Barre gave the RailRider pitching staff all they would need in the first inning. Addison Maruszak led off the game, and was hit by a pitch from IronPig starter Raul Valdes. J.R. Murphy followed with a base hit, his first at the Triple-A level. Almonte then singled to center to load the bases. That setup Dan Johnson, who singled to left-center field to drive home Maruszak and Murphy. Almonte moved to third on the play, and scored when Josh Bell hit a sacrifice fly to right field.
Maruszak came around to score again in the second. He hit a one-out double to left field, and was plated when Murphy singled to right-center field. The RailRiders led 4-0 after an inning and a half.
RailRider starter David Huff was dominant, and took a perfect game into the fifth inning. Cody Overbeck broke it up with a single up the middle. Still being stretched back into a starting role, Huff went four and one-third innings, and allowed just the two hits, and struck out two batters.
The RailRiders added insurance in the eighth by scoring two runs. Cody Grice led off with a walk, stole second, and then scored on the first of Ibarra's doubles. Ibarra proceeded to steal third base, and score on a sacrifice fly to right field by Maruszak.
The scoring was capped in the ninth, when Bobby Wilson was hit by a pitch and came around to score when Ibarra doubled for the second time.
Chase Whitley (1-0) worked scoreless frames in the sixth and seventh to earn his first win of the season. Mark Montgomery, Sam Demel, and Matt Daley also contributed to the shutout.
Valdes (1-2) allowed four runs over six innings and was handed the loss.
Scranton/Wilkes-Barre took their first game in the pursuit of the IronRail trophy on Saturday night, and will look to even the series on Sunday afternoon. Ivan Nova (2-0) has won both of his starts for SWB, and will get the nod on Sunday. Lehigh Valley will counter with Tom Cochran (2-2, 5.08). First pitch is scheduled for 1:35 p.m. and pregame coverage will commence at 1:05 p.m. on NEPA Sports Radio - The GAME, Northeast PA's largest sports radio network: 100.7 FM, 1340 WYCK-AM, 1400 WICK-AM, 1440 WCDL-AM and 106.7 FM.
- SWB RailRiders - All Aboard! -
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(CNSNews.com) – The Bureau of Labor Statistics (BLS) reported Friday that America's unemployment rate is the highest since 1983, but while most of the job losses have been in the private sector, public sector jobs – including jobs in government, education and health services – continued to increase.
Experts conclude that the number of government jobs will continue to grow in the coming years.
The nation's unemployment rate is now at 8.1 percent. In February, 651,000 additional jobs were lost, and the number of unemployed nationwide is now 12.5 million. A little over 5 million people have lost their jobs over the last 12 months.
In the private sector, construction, manufacturing, and business services all lost more than 100,000 jobs in February, and the service-providing sector lost more than 375,000 jobs, according to the BLS.
Yet public sector jobs grew in February, as they had in previous months. The government added 9,000 jobs last month, and the education and health service fields added 26,000 positions.
Back in January, the government sector added 31,000 jobs, while the education and health service fields grew by 43,000, according to the BLS.
These latest data follow a trend that has continued for more than a year. According to the BLS, the private sector lost an estimated 3.65 million jobs in 2008, but government jobs increased by nearly 150,000 in the same time period.
John Palatiello, president of the public affairs consulting firm John Palatiello & Associates, said that President Obama's plans to continue to expand government while the private sector struggles is a strange way to go about strengthening the economy.
"[This trend] creates a very interesting and unfortunate irony: You have rising unemployment in the private sector and you have rising employment in the government sector," he said. "It seems to me that, if you really want to stimulate the economy, you should do the opposite."
"You should be trying to get more and more things out of the government and into the private sector because that has a much more stimulative effect," said Palatiello. "You should put the brakes on some forms of government spending and increase the number of people who are on payrolls, paying taxes and employed in private sector jobs."
The Obama administration has indicated that many more federal jobs are on the way. Last week, the president unveiled a $3.6 trillion budget that included a variety of projects in education, health care and energy independence.
Experts have suggested that to accomplish the goals set out in the budget, hundreds of thousands of workers will need to be added to the government's payroll. Independent estimates set the number of new workers needed at 100,000, while the conservative Heritage Foundation said the number could be as high as 450,000.
Bill Beach, director of the center for data analysis at the Heritage Foundation, said the group analyzed the effects of the recently passed American Recovery and Reinvestment Act based on the details available.
After applying the details to a large scale U.S. economic model, the center determined that there will be a rapid acceleration in public sector employment starting this year and continuing through 2012.
This is partly because a large amount of the spending as detailed in the Act goes toward public sector projects – infrastructure, bridges, highways, schools, government buildings, health care and similar work, according to Beach's analysis.
Beach said he expected 450,000 jobs to be added to the public sector in that time, the largest boost coming with an increase of 130,000 government workers in 2012.
The projects in the stimulus package – in the private and public sectors – are expected to boost the country's gross domestic product in the short term. However, Beach warned that most of the boost provided by pouring money into federal programs will be temporary.
"I use the word 'temporary' because the growth rates fall back to less than potential and less than optimal levels as soon as the temporary effects are over," he said. "You're getting the typical reaction in the economy of government spending, which is temporary, and the moment the construction project is over or [the government project ends], it goes away."
Beach also suggested that funding for government projects is likely to be included in federal budgets for years to come.
"The programs for people – the income-transfer programs, the welfare programs – those will continue on from budget to budget," said Beach. "That's what history tells us will happen.
"You have to have a rare moment like the Welfare Reform Act of 1996 to actually cause government to shrink back. So we're going to assume that history is the right guide and that, once you start these things, they stay in the budget until you physically take them out," Beach added.
Palatiello also echoed the probability that the trend of government spending is unlikely to stop anytime soon.
"Ronald Regan once said that the closest thing to eternal life that we have here on Earth is a federal program," he said. "They never end."


text: RT Video/DVD DB /z-wcorg/ DS http://worldcat.org ID 654864806 LA English audio, with English cloced-captioning; optional version of feature with French menu, titles, credits, and subtitles. T1 Reel Injun A1 Diamond, Neil, Bainbridge, Catherine., Hayes, Jeremiah., Fon, Christina., Ludwick, Linda., Eastwood, Clint,, Jarmusch, Jim,, Means, Russell,, Greene, Graham,, Beach, Adam,, Trudell, John., Hill, Charlie,, Rezolution Pictures Inc., National Film Board of Canada., CBC Newsworld., Telefilm Canada., Rogers Group of Funds., Telepool GmbH., Bayerischer Rundfunk., Association relative à la télévision européenne., AVRO (Firm), Mongrel Media., PB Distributed in Canada by Mongrel Media PP [Canada] YR 2010 AB Hollywood has an impressive track record, one that spans more than 4,000 films, of blatantly misrepresenting Native people and their cultures. Featuring interviews with filmmakers and activists such as Clint Eastwood, Jim Jarmusch and Russell Means, Reel Injun delves into the fascinating history of the Hollywood Indian with razor-sharp insight and humor, tracing its checkered cinematic evolution from the silent film era to today.


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This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution license (
In this study we found that licochalcone E (LicE), a recently isolated retrochalcone from
Inflammation performs an important role as the first line of host defense mechanisms against infection of pathogenic microorganisms and functions to clear injurious materials (reviewed in [
Macrophages are the primary pro-inflammatory cells and the activation of macrophages plays a vital role in the initiation and propagation of inflammatory responses by production of cytokines including interleukin (IL)-6, IL-1β and tumor necrosis factor (TNF)-α, nitric oxide (NO), prostaglandin E2 (PGE2) and other inflammatory mediators (reviewed in [
The main signal transduction molecules in macrophages that mediate inflammatory responses include proteins in the nuclear factor-kappaB (NF-κB) and mitogen-activated protein kinase (MAPK) families (reviewed in [
Identification of bioactive natural compounds with anti-inflammatory properties and investigation of their anti-inflammatory mechanisms can be an effective strategy to reduce the incidence and severity of inflammation, thereby reducing the risk of a wide variety of diseases related to inflammation. Licorice has been used not only in foods such as sweeteners but also as medicine for thousands of years. In Oriental traditional medicine, licorice has been used in the treatment of a wide variety of diseases ranging from peptic ulcers to tuberculosis [
Licochalcone E (LicE,
To determine whether LicE exerts anti-inflammatory properties in animals, LicE or vehicle was topically applied to ears of ICR mice one hour prior to TPA treatment. The degree of inflammation was estimated by measuring the weight of ear tissue obtained by ear punch. TPA treatment increased the ear weight and LicE treatment dose-dependently reduced the TPA-induced increase in ear weight (
In order to explore the detailed mechanisms by which LicE exerts anti-inflammatory properties we next examined whether LicE inhibits LPS-stimulated inflammatory responses in RAW 264.7 cells. LicE (2.5–7.5 μmol/L) dose-dependently inhibited LPS-induced secretion of NO and also drastically inhibited the secretion of PGE2 (
We next determined whether LicE could inhibit LPS-stimulated activation of NF-κB signaling in RAW 264.7 cells. LPS-stimulated phosphorylation of IKK-αβ was significantly decreased by LicE treatment (
Because the activation of AP-1 leads to gene expression of inflammatory mediators, Western blot analyses for c-Jun and various upstream signaling proteins were conducted. In RAW 264.7 cells LicE treatment inhibited LPS-induced phosphorylation of AKT and p38 MAPK (
Licorice root is one of the most frequently prescribed plant products in Oriental medicine and has been shown to exert anti-inflammatory properties [
In the present study, topical administration of LicE (0.5–2 mg) suppressed TPA-induced ear edema in mice. Currently, it is not possible to relate these doses to a human treatment. Cho and colleagues reported that the topical administration of LicE (20 μg–1 mg) reduced oxazolone-induced chronic allergic contact dermatitis [
In the present study we noted that the ability of LicE to inhibit TPA-induced skin inflammation is comparable to that of dexamethasone that is a synthetic glucocorticoid with potent anti-inflammatory activities. However, when dexamethasone is administered orally or parenterally more than a few days, a wide variety of adverse effects have been reported to occur [
Macrophages actively participate in inflammatory responses by releasing a sequence of pro-inflammatory cytokines (IL-6, TNF-α and IL-1) and mediators (NO and PGE2) that recruit additional immune cells to the sites of inflammation. Thus, suppression of the release of these molecules is a good strategy to inhibit inflammation-related diseases [
In the present study, we have also demonstrated that LicE inhibited LPS-induced iNOS and COX-2 mRNA expression (
The NF-κB family of transcription factors performs a vital role in coordinating the expression of a broad variety of pro-inflammatory genes including iNOS, COX-2, IL-1, IL-6 and TNF [
AP-1 is also a key transcription factor regulating the expression of iNOS and COX-2 which are central mediators of inflammation [
Phosphoinositol 3-kinase/AKT and MAPKs including JNK, ERK1/2 and p38 MAPK have been well characterized as potential signaling pathways regulating NF-κB activation [
Antibodies against iNOS and COX-2 were purchased from BD Transduction Laboratories (Palo Alto, CA, USA). Antibodies against inhibitor of κB kinase (IKK)β, NF-κB p65, lamin B and α-tubulin were purchased from Santa Cruz Biotechnology (Santa Cruz, CA, USA). Antibodies against P-IKKαβ (Ser-176/180), IKKα, IκBα, P-IκBα (Ser-32), AKT, P-AKT (Ser-473), stress-activated protein kinase/c-Jun-
All animal experimental protocols were approved by the Animal Care and Use Committee of Hallym University (Hallym2011-04). Four-week-old female ICR mice (Koatech, Seoul, Korea) were acclimatized to laboratory conditions for 1 week by providing laboratory chow and tap water
The ear samples were fixed in 40 g/L of paraformaldehyde for hematoxylin and eosin (H&E) staining and IHC. IHC analysis were performed as previously described [
RAW 264.7 macrophages (American Type Culture Collection, Manassas, VA, USA) were maintained in Dulbecco's Modified Eagle Medium (DMEM) containing 100 mL/L fetal bovine serum (FBS), 100,000 U/L of penicillin and 100 mg/L of streptomycin. The effects of LicE (0–7.5 μmol/L) on cell viability were determined in the presence of 1 mg/L of lipopolysaccharide (LPS) by the 3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyltetrazolium bromide (MTT) assay as described previously [
RAW 264.7 cells were plated in 100 mm-diameter dishes at 1 × 106 cells per dish with DMEM containing 100 mL/L FBS, serum deprived in DMEM containing 10 mL/L FBS and treated with various concentrations (0–7.5 μmol/L) of LicE in the absence or presence of 1 mg/L LPS. Cell lysates and nuclear extracts were prepared and Western blot analyses were conducted as previously described [
Total RNA was isolated using RNeasy Plus Mini Kit (Qiagen, Valencia, CA, USA) and cDNA was synthesized as described previously [
RAW 264.7 cells were co-transfected with pGL-miNOS-1588 [
Nuclear extract was prepared and EMSA was conducted as previously described [
Data are expressed as mean ± SEM and were analyzed by ANOVA. Differences between treatment groups were analyzed by Duncan's multiple range test, using SAS for Windows, version 9.1 (SAS Institute). A
In summary, we have demonstrated that LicE isolated from licorice exhibits potent anti-inflammatory properties in mouse skin and murine macrophages. LicE inhibited LPS-induced iNOS and COX-2 mRNA expression as well as their promoter activities. LicE also inhibited the secretion of the pro-inflammatory cytokines IL-1β, IL-6 and TNF-α and their corresponding mRNAs. Furthermore, LicE inhibited NF-κB and AP-1 signaling and phosphorylation of AKT, SAPK/JNK, p38 MAPK and ERK1/2. Inhibition of AKT and MAPK activation by LicE may contribute to the inhibition of AP-1 and NF-κB signaling, which, in turn, leads to decreased expression of inflammatory cytokines and inducible enzymes.
This research was supported by Hallym University Specialization Fund (HRF-S-11) and the National Research Foundation of Korea grants funded by the Korea government (MEST) (No. 2012-0004903, 2010-0029233, 2012-0000642). The authors thank Jong-Gab Jun and Jae-Ho Jeon for the generous gift of LicE.
The authors declare that they have no conflict of interest.
Chemical structure of licochalcone E (LicE).
Licochalcone E (LicE) inhibits TPA-induced ear edema formation in mice. LicE (0, 0.5, 1, or 2 mg) was topically applied to the mouse ear 1 h prior to the topical application of 5 nmoles of TPA (4 h). (
Licochalcone E (LicE) suppresses the production of NO and PGE2 as well as the expression of iNOS and COX-2 in LPS-stimulated RAW 264.7 cells. Serum-deprived RAW 264.7 cells were treated with various concentrations (0–7.5 μmol/L) of LicE in the absence or presence of 1 mg/L LPS for 24 h. (
Licochalcone E (LicE) suppresses LPS-induced mRNA expression of IL-6, IL-1β, and TNF-α in RAW 264.7 cells. Serum-deprived RAW 264.7 cells were treated with various concentrations (0–7.5 μmol/L) of LicE in the absence or presence of 1 mg/L LPS for 24 h. (
Licochalcone E (LicE) suppresses LPS-induced NF-κB signaling in RAW 264.7 cells. Serum-deprived cells were treated with various concentrations (0–7.5 μmol/L) of LicE for 40 min. LPS was then added and incubated for an additional 10 min (for the determination of P-IKKαβ, IKKα, IKKβ and P-IκBα) or 20 min (for the determination of IκBα and p65). (
Licochalcone E (LicE) suppresses LPS-induced AKT, MAPK and AP-1 signaling in RAW 264.7 cells. (
Licochalcone E (LicE) inhibits TPA-induced phosphorylation of SAPK/JNK, c-Jun and ERK1/2 in mouse skin. LicE (0, 0.5, 1, or 2 mg) was topically applied to the mouse ear for 1 h prior to the topical application of 5 nmoles of TPA (4 h). Ear sections were stained with an antibody raised against P-SAPK/JNK, P-c-Jun or P-ERK1/2 and counterstained with hematoxylin. (


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LeBron James won Sports Illustrated's "Sportsman of the Year" and he may be only getting better.
MIAMI -- On Saturday after beating the Brooklyn Nets and watching his high school football team, St. Vincent-St. Mary's, win the Ohio state championship in football, LeBron James walked out of the Heat locker room grinning with both hands in the air.
"I'm all ringed up," James shouted as he exited the room.
And on Monday, we learned that his long line of accolades will get even longer.
Sports Illustrated named James the 2012 "Sportsman of the Year" after winning his first NBA championship, his first Finals MVP, his third most valuable player award and an Olympic gold medal in London.
Hard to top that. According to Sports Illustrated, no one did.
"It's very humbling," James said, after Monday's Heat practice, about winning the award. "It was a long journey."
Just last season, James played in front of crowds with posters that read "RINGLESS" in capital letters. But now, he has his own NBA championship ring and a "Sportsman of the Year" award to boot.
A wave of public negativity followed James after he left the Cleveland Cavaliers organization for Miami in the summer of 2010, but James said he grew on and off the court before winning the 2012 title.
"Going through what I went through my first year here [in Miami], making the transition from Cleveland and ultimately winning a championship, it makes it sweeter," he said.
James has received not one, but two championship rings this past season. What many may not know is that James also received a ring from the Kentucky Wildcats after they won the 2012 NCAA tournament championship. Though James never attended college after going straight to the pros after high school, the 27-year-old has close ties to Kentucky coach John Calipari and his team.
It's just the second time in history that a player won the basketball triple crown: an MVP, an NBA title and an Olympic gold medal. Michael Jordan achieved that bundle of accolades in 1992 and like James, he won a Finals MVP, as well.
Despite all the recognition, the 27-year-old doesn't consider himself a finished product.
"It was a great year, an unbelievable year and I will never forget it," James said. "But time waits for no one and I understand that."
A post-championship letdown hasn't taken effect for James. Though his scoring numbers are down mostly due to a dip in free throws, James is averaging a career-high 8.9 rebounds and shooting a career-high 53.3 percent from the floor. James is also registering a league-leading 28.2 player efficiency rating (PER) so far this season.
James' on-court game continues to grow. For much of his career, the three biggest knocks on James' game were said to be the lack of a post game, a reliable jump shot and a knack for coming up big in crunch time. But the numbers show an enormous leap in all three categories.
This season, James is using post-up plays almost twice as often as he did two seasons ago, according to SynergySports. He is also shooting a career-high 43.8 percent from downtown and posting the highest PER in the league in clutch situations (when the game is within five points in the final five minutes of the game) among qualified players.
There's not much left for James to accomplish on the basketball court aside from winning more titles. James won a championship at a younger age than Jordan, who was 28 when he first grasped the Larry O'Brien trophy. James needs to win five more titles to tie Jordan's mark.
Ahead of Wednesday's game against the Washington Wizards, James took a moment to reflect on how far he's come over the years and how he's dealt with the pressure.
"It's a challenge when everything you do or say can be used against you," James said. "The thing that's helped me is that I've been watched and followed since I was 16 years old. They praise you and you make one mistake and they bring you down. They praise you again and then bring you down again so I've had a lot of hardships, but it all makes it sweeter in the end."
Statistics from NBA.com were used in this story.


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Dr Stuart Roy
|Org Unit
|Australian Centre for Plant Functional Genomics (ACPFG)
|+61 8 8313 0549
2 20
Plant Genomics Centre
Biography/ Background
Positions held:
2016 - Executive Management Group Australian Centre for Plant Functional Genomic
2015 - Chair ACPFG Postgraduate Committee
2013 - Management Group Australian Centre for Plant Functional Genomics
2013 - 2014 Chair of the Australian Centre for Plant Functional Genomics' Resource Management Group
2009 - 2013 - Member of the Australian Centre for Plant Functional Genomics' Science Advisory Group (Chair in 2013)
2005 - Salt Focus Group Leader Australian Centre for Plant Functional Genomics and the University of Adelaide, Australia
2004 - Postdoctoral Research Fellow Australian Centre for Plant Functional Genomics and the University of Adelaide, Australia.
Project: Identification of novel salt tolerance genes in plants
2001-2004 Broodbank Research Fellow Department of Plant Sciences, University of Cambridge, UK,
Project: Development of single cell molecular biology techniques
1998-2001 Ph.D. (Supervisor: Prof Roger Leigh) Department of Plant Sciences, University of Cambridge, UK & Gonville and Caius College, Cambridge, UK,
Project: Single cell measurements of vacuolar hydrolase activities in senescing leaves of barley
1994-1998 BSc (hons) in Plant and Environmental Biology, First Class, Awarded Margaret Laing Bell prize. The University of St Andrews, UK
Dissertation: Light repair of UV-B induced 6-4 photoproducts in wheat (Supervisor: Dr Alyson Tobin)
Funding recieved:
Broodbank research fellowship, University of Cambridge
GRDC 2010-2013, 'Development of high salinity tolerant winter cereals germplasm' (with M Tester, G McDonald, J Eglinton, D Mather, K Chalmers, M Quinn, R Munns)
USAID 2013-2017, "Abiotic stress tolerant bio-engindeered cereals' (with A Jacobs, P Langridge, Sergiy Lopato et al.)
GRDC 2013-2016, "Development of salinity tolerant wheat and barley' (with M Gilliham, B. Berger, R. James, T. Colmer, E. Barrett-Lennard)
Research Interests
Loss of crop yield due to high concentrations of Na+ and Cl- on agricultural land is a significant problem for Australian farmers. An estimated 4.6 M ha of Australian farmland is affected to some degree by saline soils - currently 1 in 2 Western Australian farms and 1 in 5 South Australian farms are affected by salinity. By 2050 it is expected that around 14 M ha of Australian agricultural land will be affected by dryland salinity, therefore it is imperative that we identify genes and cellular processes which will increase the salinity tolerance of our crop plants.
Crops undergoing salt stress experience an immediate reduction in growth and tillering (shoot independent ionic stress) and an increase in premature senescence due to the accumulation of toxic ions (shoot depedent ionic stress).
Our group has a number of projects all aimed at obtaining a better understanding of the processes involved in both the shoot independent and the shoot dependent ionic stress. We are using both forward and reverse genetics to identify mechanisms for improving the salinity tolerance of crops.
Quantitative trait locus (QTL) mapping for novel genes involved in Na+ and Cl- accumulation in cereals
Identification of genes involved in the tolerance to the shoot independent ionic stress
Development of molecular markers to novel salinity tolerance traits
Understanding the mechanisms controlling the plant's reponse to salinity stress
Salt stress physiology
PhD and Honours projects available:
See the "File" section below for the range of PhD and Honours projects available in the ACPFG Salt Reseach Group
Characterisation of a novel salt stress signalling pathway in Arabidopsis
Aims and Objectives
Techniques to be used
Identification of the plant cell Na+ sensing receptor
Aims and Objectives
Techniques to be used
Characterisation of a salt tolerance gene from barley
Aims and Objectives
Techniques to be used
See the "File" section below for the range of PhD and Honours projects available in the ACPFG Salt Reseach Group
Li., B., Qiu, J., Jayakannan, M., Xu, B., Li, Y., Mayo, G.M., Tester, M., Gilliham, M., Roy, S.J. (2017) AtNPF2.5 Modulates Chloride (Cl-) Efflux from Roots of Arabidopsis thaliana, Frontiers in Plant Science 10.3389/fpls.2016.02013
Gilliham, M., Able, J.A., Roy, S.J. (2016) Translating knowledge in abiotic stress tolerance to breeding programs. The Plant Journal DOI:10.1111/tpj.13456
Schilling, R.K., Tester, M., Marschner, P., Plett, D.C., Roy, S.J. (2016) AVP1: one protein, many roles. Trends in Plant Science http://dx.doi.org/10.1016/j.tplants.2016.11.012
Qiu, J., Henderson, S.W., Tester, M. Roy, S.J., Gilliham, M. (2016) SLAH1, a homologue of the slow type anion channel SLAC1, modulates shoot Cl- accumulation and salt tolerance in Arabidopsis thaliana. Journal of Experimental Botany (in press)
Li, B., Byrt, C., Qiu, J., Baumann, U., Hrmova, M., Evrard, A., Johnson, A.A.T., Birnbaum, K.D., Mayo, G., Jha, D., Henderson, S.W., Tester, M., Gilliham, M. Roy, S.J. (2016) Identification of a stelar-localised transport protein that facilitates root-to-shoot transfer of chloride in Arabidopsis. Plant Physiology 170: 1014-1029.
Amarasinghe, S., Watson-Haigh N.S., Gilliham, M., Roy S., Baumann, U. (2016) The evolutionary origin of CIPK16: A gene involved in enhanced salt tolerance, Molecular Phylogenetics and Evolution DOI:10.1016/j.ympev.2016.03.031
Roy, S.J., Collins N.C., Munns R (2016) Abiotic stress genes and mechniams in wheat. In Encyclopedia of Food Grains, Elsevier
Takahashi F, Tilbrook, J., Trittermann, C., Berger, B., Roy, S.J., Seki, M., Shinozaki, K., Tester, M. (2015) Comparison of leaf sheath transcriptome profiles with physiological traits of bread wheat cultivars under salinity stress. PLoS One 10: e0133322
Adem, G.D., Roy, S.J., Plett, D.C., Zhou, M., Bowman, J.P., Shabala, S. (2015) Expressing AtNHX1 in barley (Hordeum vulgare L.) does not improve plant performance under saline conditions. Plant Growth Regulation DOI 10.1007/s10725-015-0063-9
Thoday-Kennedy, E.L., Jacobs, A.K., Roy, S.J. (2015) The role of the CBL-CIPK calcium signalling network in regulating ion transport in response to abiotic stress. Plant Growth Regulation 76: 3-12
Schmoeckel, S.M., Garcia, A.F., Berger, B. Tester, M., Webb, A.A.R., Roy, S.J. (2015) Different NaCl-induced calcium signatures in Arabidopsis thaliana genotypes Col-0 and C24. PLoS One. DOI: 10.1371/journal.pone.0117564
Hairmansis, A., Berger B., Tester M., Roy S.J. (2014) Image-based phenotyping for non-destructive screening of different salinity tolerance traits in rice. Rice 7:16
Adem, G.D., Roy, S.J., Zhou, M., Bowman, J.P., Shabala (2014) Evaluating contribution of ionic, osmotic and oxidative stress components towards salinity tolerance in barley. BMC Plant Biology. 14: 113
Roy, S.J., Negrão, S. and Tester, M. (2014) Salt resistant crop plants. Current Opinion in Biotechnology 26: 115-124.
Schilling R., Marschner, P., Shavrukov, Y., Berger, B., Tester, M., Roy, S.J.#, Plett, D. (2014) Expression of the Arabidopsis vacuolar H+-pyrophosphatase gene (AVP1) improves the shoot biomass of transgenic barley and increases grain yield in a saline field. Plant Biotechnology Journal 12: 378-386
Tilbrook, J. and Roy, S.J. (2013) Salinity Tolerance In Plant abiotic stress, 2nd edition, M.A. Jenks and P.M. Hasegawa eds, Wiley-Blackwell Publishing, New York, USA
Roy, S.J., Huang, W., Wang, X.J., Evrard, A., Schmoeckel, S.M., Zafar, Z.U. Tester, M. (2013) A novel protein kinase involved in Na+ exclusion revealed from positional cloning, Plant, Cell and Environment 36: 553-568.
Shavrukov, Y., Bovil, J., Afzal,I., Hayes, J., Roy, S.J., Tester, M., Collins, N.C. (2013) HVP10 encoding V-PPase is a prime candidate for the barley HvNAX3 sodium exclusion gene - evidence from fine mapping and expression analysis, Planta 4: 1111-1122
Roy, S.J., Conn, S.J., Mayo, G.M., Athman, A. and Gilliham (2012) Transcriptomics on small samples. In Plant salt tolerance, methods and protocols; Methods in Molecular Biology 913: 335-350 T.A. Cuin and S. Shabala, eds Humana Press, Springer, New York, USA
Roy, S.J. and Tester, M. (2012) Approaches for increasing salinity tolerance of crops, in Encyclopedia of sustainable science and Technology, R.A. Meyers, ed, Springer, New York, USA
Roy, S.J., Tester, M. and Gaxiola, R. A., Flowers, T. J., (2012) Plants of Saline Environments, in McGraw-Hill Encyclopaedia of Science and Technology, McGraw-Hill, New York, USA
Roy, S.J., Tucker, E. & Tester, M. (2011) Genetic analysis of abiotic stress tolerance in crop plants. Current Opinion in Plant Biology 14: 1-8
Golzarian, M.R., Frick, R.A., Rajendran, K., Berger, B., Roy, S.J., Tester, M., Lun, D.S. (2011) Accurate inference of cereal plant biomass using information extracted from high-throughput plant images, Plant Methods 7:2
Plett, D.C., Safwat, G., Møller, I.S., Gilliham, M., Roy, S.J., Shirley, N., Jacobs, A., Johnson, A.A.J. and Tester, M. Improved salinity tolerance of rice through cell type-specific expression of AtHKT1;1, PLoS One. 5: e12571
Jha, D., Shirley, N., Tester, M. & Roy, S.J. (2010) Variation in salinity tolerance and shoot sodium accumulation in Arabidopsis ecotypes linked to differences in the natural expression levels of transporters involved in sodium transport. Plant, Cell & Environment 33: 793-804
Møller, I.S., Gilliham, M., Jha, D., Mayo, G.M., Roy, S.J., Coates, J.C., Haseloff, J. and Tester, M. (2009) Shoot Na+ exclusion and increased salinity tolerance engineered by cell type-specific alteration of Na+ transport in Arabidopsis. The Plant Cell 21:2163-2178
Rajendran, K., Tester, M. & Roy, S.J. (2009) Quantifying the three main components of salinity tolerance in cereals. Plant, Cell & Environment 32, 237-249
Roy, S.J., Gilliham, M., Berger, B., Essah, P.A., Cheffings, C., Miller, A.J., Widdowson, L., Davenport, R.J., Liu, L.-H., Skynner, M.J., Davies, J.M., Richardson, P., Leigh, R.A. and Tester, M. (2008) Investigating glutamate receptor-like gene co-expression in Arabidopsis thaliana. Plant, Cell & Environment 31: 861-871
Schnurbusch, T., Huang, C., Collins, N.C., Sutton, T., John, U., Roy, S., Paltridge, N., Tester, M., Langridge P. & Fincher, G.B. (2007) GM wheat and barley II. Prospects for enhanced productivity and quality. Agricultural Science 21: 4-10.
Wagstaff C., Rogers H., Buchanan-Wollaston, V., Bramke, I., Stead, A., Roy, S., Leigh R. (2006) Expression profiling from single cells of senescing plant tissues. Comparative Biochemistry and Physiology A - Molecular & Integrative Physiology 143:S120-120
Roy, S.J., Cuin, T.A. and Leigh, R.A. (2003) Nanolitre-scale assays to determine the activities of enzymes in individual plant cells, Plant J. 34: 555-564.
Roy, S.J. & Tester M. Salinity tolerance in plants, WO2010/025513.
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Hopes for a salt-tolerant barley lift after first test
GRDC scholar anchors barley trial
Australia and Saudi Arabia work on grain crops
- 2014 Honours projects - Honours_Project-salt2014.pdf [161.2K] (application/pdf)
- 2014 PhD projects - Salt_Group_PhD_Portfolio2014.pdf [1.0MB] (application/pdf)
To link to this page, please use the following URL: http://www.adelaide.edu.au/directory/stuart.roy


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Shares of Splunk (SPLK) surged almost 7% in Tuesday morning trading after an investment firm gave the big data software developer a vote of confidence.
Northland Securities raised its rating for Splunk stock from "market perform" to "outperform." Northland has set a price target of $66 for Splunk shares, thought that is down substantially from an earlier price target of $95 for SPLK shares, InterCoolerOnline notes.
In November, Splunk shares topped $74 after the company reported higher-than-expected sales and added more customers.
On Friday, Splunk stock closed at $45.99 per share.
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