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Ted Simons: Tonight's edition of "Arizona Horizon" looks at what has been described as the greatest generation, the men and women who served in World War II. That service and sacrifice is the focus of a new art exhibit, producer Shana Fischer and photographer Ed Kishel, give us a look at this moving tribute.
Shana Fischer: Each of these photographs has a story behind it. A story of courage, perseverance, and fortitude.
William Lancaster: I was a fighter pilot, in a P-38 lightning. And I was in the Army Air Corp.
Shana Fischer: Bill Lancaster was, was a teenager when he found himself in the middle of World War II in a plane that he had never flown before. This photo shows the cockpit of the plane. Hundreds of buttons and bill says that he had no idea what many of them were for.
William Lancaster: This airplane was, number one, it was multi-engine, two engines, and in all my flying, it was done in a single engine airplane. And, and I got in an outfit, but all they had was the twin engine airplanes, and I had to go out and, and earn to fly by myself because it does not have one fitted out for an instructor to be there to give me any instruction. So, you had to learn to fly the thing and survive it.
Shana Fischer: That lack of training proved to be deadly. Of the 90 men bill served with, 67 of them never came home. Even now, it's difficult for bill to remember his time in the war and those who did not return safely to their families.
William Lancaster: It's hard to see. I don't think anyone knows the bad side. I had, I had guys that went with me to combat, and they came over there and went down and the first time that they got in an airplane over there and tried to get up and fly, they killed themselves.
Del Ryland: Del Ryland was a farmer boy turned fighter pilot, he joined the ROTC, and soon found himself here in Arizona.
Del Ryland: I got my wings right here in Mesa, at Williams field. Went over to the military to Italy and flew there commissions and the war ended.
Shana Fischer: Bill and Del are two of the Veterans whose photographs are part of an installation at Belmont village, an assisted living facility in Scottsdale. The nearly two dozen portraits were taken by critically acclaimed photographer Thomas Sanders. What started a few years ago as a school assignment for Tom, has transformed into a way to preserve these stories before they are lost for good.
Thomas Sanders: My senior year of college, I had a homework assignment, to photograph a portrait of somebody, and living right around the corner, was this giant retirement community company. And I happened to go there asking the retirement community if there was an interesting, you know, person to photograph, and they said we have this World War II hero.
Shana Fischer: Tom's work caught the attention of Belmont's CEO Patricia will. She asked him to take part rats of Veterans in all of their facilities.
Patricia Will: As a reaction that we got, far surpassed anything that I could have imagined. Not just because there emerged a storyline from each of these extraordinary Veterans. But also, because as a result of hearing the stories, often for the first time, they began to engage with one another in very different ways. The experiences that they had often buried in their long-term memory. Merged and created a camaraderie among them.
Shana Fischer: For Muriel Pelham, it continues to this day. At 90 years old she volunteers with Veteran's groups. Stationed in Europe as a nurse, she spent several years there and came back and continued working in various hospitals. Muriel was one of 350,000 women who served during the war.
Muriel Pelham: I guess it means that, that I was, I was probably helping the women to, to crack the sailing because we had a hard time with a lot of things, a lot of opportunities to advance, and do other than routine care. So, I was glad that I was able to do that.
Shana Fischer: With 1,500 World War II Veterans dying each day in America, capturing these photos and the stories is crucial, but it's more than that.
Thomas Sanders: What is special about this project is that sometimes, the Veterans have never had the opportunity to share their stories, so with this project of photographing and interviewing the Veterans, the Veterans are able to be honored for their first time. And I think that that's, that's, you know, something very, very special for myself and for them. In a way, to give back through art by preserving history.
Shana Fischer: World War II and Korean war Veteran Carl Melin agrees. He's never really talked about his time in Japan. The exhibit is a way for his family and friends to learn more.
Carl Melin: I think that, that they can read the, the items, as to what they say, and what the different fellas thought about their activity. And about their period of time while they were serving. I don't want it to be lost in antiquity, so anything that can be done and said, pictured, proof is all to the benefit.
Shana Fischer: The Veterans are all very pleased with how their photos turned out. If not a bit surprised.
Carl Melin: Well, as everybody else, they wanted, they wondered who it was, the pictures, and the, they never -- they are never as flattering as you wish then to be, but they are factual and that's the way it is.
Muriel Pelham: While I was sort of surprised, frankly. I saw myself holding the picture and the clerk said I recognize more than the other because, because, you know, as you get older, your features change. Let's face it. And I am, in my 90s, so, I'm up there.
Del Ryland: I am thankful that I still have a head of hair. I guess that that's the only thing of appreciation, is I still have a little hair left.
Shana Fischer: For Tom, this project brings his family history into focus. His great uncle died in World War II, and his grandfather, a semi-famous photographer, snapped this shot of author Ernest Hemingway. It now sits in the Smithsonian.
Thomas Sanders: When I photograph the Veterans, I tend to use really dramatic lighting. Some of the Veterans have objects from World War II or other wars that they have hung onto, like their whole entire life, and so that helps to tell their story, so I have them hold that object up, and on top of that, I really try and not overly direct the Veterans too much. I have them hold the object, and I might have them move a bit in terms of posing, but I don't want too much of my own idea to be projected onto them. I just really am trying to capture their essence and soul.
Shana Fischer: Tom's portraits are much like the men and women themselves. Stark, unyielding, and stoic with hints of vulnerability. They tell an entire life story. One that will now last forever.
Thomas Sanders: The whole idea behind the project is making people more appreciative of the Veterans and soldiers, and through hopefully creating an interesting artistic portrait of the Veteran and reading their story, you know, I really hope that draws them in to help put their own lives in perspective.
Ted Simons: Tom's photos were gathered into an award-winning book titled "the last good war." If you would like to see the exhibit make an appointment by contacting Belmont Village in Scottsdale. That is it for now, I'm Ted Simons. And thank you very much for joining us. You have a great evening.


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subset: commoncrawl


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These authors contributed equally to this work.
This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution license (http://creativecommons.org/licenses/by/3.0/).
Development of the atherosclerotic plaque involves a complex interplay between a number of cell types and an extensive inter-cellular communication via cell bound as well as soluble mediators. The family of tribbles proteins has recently been identified as novel controllers of pro-inflammatory signal transduction. The objective of this study was to address the expression pattern of all three tribbles proteins in atherosclerotic plaques from a mouse model of atherosclerosis. Each tribbles were expressed in vascular smooth muscle cells, endothelial cells as well as in resident macrophages of mouse atherosclerotic plaques. The role of IL-1 mediated inflammatory events in controlling tribbles expression was also addressed by inducing experimental atherosclerosis in ApoE−/−IL1R1−/− (double knockout) mice. Immunohistochemical analysis of these mice showed a selective decrease in the percentage of trb-1 expressing macrophages, compared to the ApoE−/− cohort (14.7% ± 1.55
Atherosclerosis arises as a result of chronic inflammation of the artery wall, which occurs in response to a wide range of different stimuli, including fatty diets. The generation of atherosclerotic plaques involves a complex interplay between various vascular cell types and cytokine signalling networks. Although the molecular events that initiate the development of atherosclerotic lesions are still somewhat unclear, a large body of evidence supports a role for low-density lipoproteins (LDL), free radicals, infections [
In recent years, a family of negative regulators of MAPK and PI3K cascades, called tribbles (trb) have emerged. These are evolutionally conserved, kinase-like proteins; their catalytic domain is missing some of the conserved residues thought to be indispensable for kinase activity [
Human genetic evidence suggests that trb-1 may be a risk factor in the development of hyperlipidemia and cardiovascular disease [
However, whilst an important regulatory function for tribbles in vascular cell inflammation is supported by a number of studies and some expression data is available for individual tribbles homologues, no systematic study investigating the expression of the tribbles protein family has been performed in atherosclerosis.
In the study presented here, we have characterised the expression profile of all three mammalian tribbles in the ApoE−/− murine experimental model of atherosclerosis. We have also identified the potential regulation of tribbles expression by IL-1, a master cytokine involved in atherosclerotic plaque development,
We have previously reported a selective increase in trb-1 expression in human aortic vascular smooth muscle cells within human atherosclerotic lesions compared to non-diseased aorta [
The majority of trb-1 protein is detected as expected in the medial region (
Numerous macrophages and some large plaques are also routinely detected in the aortic roots of fat-fed ApoE−/− mice ([
Tribbles 1 (trb-1) expression in experimental mouse atherosclerosis.
Tribbles 1 expression in macrophages in experimental mouse atherosclerosis.
We have already shown that trb-2 is expressed in smooth muscle cells and endothelial cells cultured
In the stable mouse atherosclerotic lesions investigated here, trb-2 appeared to be weakly expressed (
Tribbles 2 (trb2) expression in experimental mouse atherosclerosis.
Tribbles 3 staining in experimental mouse atherosclerosis.
Since IL-1 is known to be critical in the development of atherosclerosis in mice [
However, in lesions from DN mice analysed for trb-1 positive staining, there were significantly less trb-1 positive macrophages/0.1mm2 of plaque than in ApoE−/− animals (14.7% ± 1.55, n = 4 compared with 26.3% ± 1.19 [ApoE−/−], n = 5) (
In order to investigate the biological significance of tribbles-1 expression levels in macrophages (as seen in the ApoE−/− aortic roots) and to determine if this was generally controlled by TIR receptor mediated inflammatory stimuli, first we established whether the Raw 264.7 macrophage cell line recapitulated our
Macrophage tribbles 1 expression is reduced in experimental atherosclerosis in mice lacking the ability to signal via the IL-1 receptor.
Next, the functional consequences of overexpressed trb-1 were tested in Raw 264.7 cells, transfected with a trb-1 expression construct. Transfected cells were then stimulated with LPS and IL-6 production examined by ELISA. Our results demonstrate that overexpressed trb-1 strongly inhibits the production of IL-6 in macrophages (
Trb-1 expression is induced by LPS and this protein controls IL-6 production in Raw 267.4 macrophages.
Molecular control of cellular function is achieved via the coordinated action of key intracellular signal processing systems. A number of these, including the MAPK signalling network and PI3K driven signal transduction pathways have been the subject of intense research in connection to cardiovascular disease and their potential as targets for therapeutic intervention [
The contribution of MAPK pathways has been elucidated recently. It has been shown that scavenger receptor (CD36) mediated signalling, triggered by oxLDL, is transmitted via src tyrosine kinases (lyn) and MAPK kinase kinase kinase (MAPKKK) proteins. Blocking lyn or JNK signalling via pharmacological inhibitors led to attenuated foam cell formation [
Cellular activation via the family of PI3K proteins has also been investigated in the context of atherosclerosis. Specifically, PI3Kγ and δ have been shown to control T cell migration, production of reactive oxygen species (ROS) in macrophages in response to proinflammatory stimuli, the secretion of proinflammatory cytokines, including IL-1 and IL-6 as well as activation of mast cells and platelet function [
The regulation of the MAPK and PI3K signalling pathways by tribbles family members is now well established [
To address these issues, we performed a systematic analysis of the expression pattern of all three mammalian tribbles in the ApoE−/− fat-fed model of atherosclerosis and show that these proteins are present in smooth muscle cells, endothelial cells and plaque resident macrophages.
IL-1 is widely appreciated as a major contributor of plaque development in atherosclerosis. In line with this, ApoE/IL-1R double knockout mice are protected from atherosclerosis, compared to ApoE−/− controls [
Our observations may provide a mechanistic explanation for the rapidly increasing genetic evidence that trb-1 is a significant risk factor in cardiovascular disease [
Tribbles-2 and -3 have also been detected in specific cells of the plaque. A recent study published by Deng
In addition to macrophages and smooth muscle cells, recent reports have suggested a role for tribbles in the control of adipocyte function [
ApoE−/− mice (stock no. 002052) and IL-1R1−/− (stock no. 3245) were obtained from the Jackson Laboratories; ApoE−/− IL-1R1−/− double deficient mice were generated by cross-breeding. All mice were on a C57BL/6 background and fed normal 'chow' (4.3% w/v fat, 0.02% w/v cholesterol) or 'Western' (18.5% w/v fat, 0.9% w/v cholesterol, 0.5% w/v cholate) diet. Animals were housed in a controlled environment with a 12 hour light/dark cycle at 22 °C. All experiments were performed in accordance with UK legislation under the 1986 Animals (Scientific Procedures) Act, and approval was granted by the University ethics review board. The investigation conforms with the Guide for the Care and Use of Laboratory Animals published by the US National Institutes of Health (NIH Publication No. 85-23, revised 1996).
Raw 264.7 cells (ATCC: TIB-71) were cultured in standard media and treated with of LPS, as specified in the appropriate figure legends.
Trb-1 protein was detected using a trb-1 antibody (Millipore) diluted 1:500 in 0.05% v/v Tween 20/5% v/v non-fat milk as previously described [
ELISA was performed to quantify the production of mouse IL-6 (R&D systems), as recommended by the manufacturer.
Paraffin-embedded sections of aortic sinus were stained immunohistochemically for smooth muscle actin (Dako, M0851, 1:150) to visualize vascular smooth muscle cells and Galectin-3 (R&D, AF1197, 1:50) to visualize macrophages. Trb antibodies used were: trb-1 (Millipore, 09-126) at 1:125, trb-2 (Sigma, HPA001305) at 1:200 and trb-3 (Millipore) at 1:200.
Standard immunohistochemical techniques were applied.
The number of different types of cells expressing trbs was assessed by morphometric measurement of histologically stained paraffin-embedded sections using a Lucia image analysis software system (Lucia G, Nikon, UK). The total and positively stained cell nuclei were counted within a fixed area. The values reported represent a mean from at least 5 sections for each animal.
In summary, this is the first systematic study of tribbles expression in experimental atheroma and the data suggest an important regulatory function for tribbles-1 in vascular cell pathophysiology. In addition, our data clearly demonstrate that inflammatory signals selectively control the expression of trb-1 in plaque macrophages, suggesting that this may be an important homeostatic control mechanism to modulate the inflammatory phenotype of these cells.
This work was supported by the Fondation Leducq and British Heart Foundation project grants PG/02/122 and PG/05/100.


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The main library is largely devoted to Church History with special emphasis on the recusancy period. It includes many standard works and is regularly updated with new publications.
In addition there is the historic Convent Library of antique books written in several different languages, dating from 1508 to 1850. Our reading room houses a number of Catholic learned journals and some theology. There is a further feminine collection of books by and about women, mainly in a religious context.
The archives will be of particular interest to those interested in the history of the Congregation of Jesus (previously named the Institute of Blessed Virgin Mary) or the annals of the Bar Convent and its school.
For the study of Mary Ward we rely mainly on transcripts, as the original papers are in Munich. The history of the Bar Convent itself is supported by its own documents, including letters, journals, registers and a special collection of account books dating from 1733, from which a vivid picture of school and convent life can be drawn.
Researchers and students should apply to the Archivist/Librarian in writing in the first instance, stating their area of study. A letter of recommendation is required from those who are new to the Bar Convent.


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This article is an open-access article distributed under the terms and conditions of the Creative Commons Attribution license (http://creativecommons.org/licenses/by/3.0/).
Many times in the history of lead toxicology the view that "the problem" has been solved and is no longer a major health concern has prevailed, only to have further research demonstrate the prematurity of this judgment. In the last decade, an extraordinary amount of new research on lead has illustrated, all too clearly, that "the problem" has not disappeared, and that, in fact, it has dimensions never before considered. Recent risk assessments have concluded that research has yet to identify a threshold level below which lead can be considered "safe." Although children's intelligence has traditionally been considered to be the most sensitive endpoint, and used as the basis for risk assessment and standard setting, increased lead exposure has been associated with a wide variety of other morbidities both in children and adults, in some cases at biomarker levels comparable to those associated with IQ deficits in children. In adults, these endpoints include all-cause mortality and dysfunctions in the renal, cardiovascular, reproductive, central nervous systems. In children, IQ deficits are observed at blood lead levels well below 10 μg/dL, and the dose-effect relationship appears to be supra-linear. Other health endpoints associated with greater early-life lead exposure in children include ADHD, conduct disorder, aggression and delinquency, impaired dental health, and delayed sexual maturation. Studies employing neuroimaging modalities such as volumetric, diffusion tensor, and functional MRI are providing insights into the neural bases of the cognitive impairments associated with greater lead exposure.
Many times in the history of lead toxicology the view that "the problem" has been solved and is no longer a major health concern has prevailed, only to have further research demonstrate the prematurity of this judgment. In the last decade, an extraordinary amount of new research on lead has illustrated that "the problem" has clearly not disappeared, and that, furthermore, it has dimensions never before considered. This review broadly surveys the epidemiologic literature published in the last decade on the health effects of lead exposure, both in adults and children, focusing on the lowest observed adverse effect levels, the shapes of the pertinent dose-effect/dose-response relationships, and susceptible subgroups.
Recent prospective cohort studies conducted in cohorts drawn from the general population provide reasonably consistent evidence, in both men and women, that greater blood lead levels are associated with higher all-cause mortality and that deaths from cardiovascular diseases are largely responsible for the associations. This association is apparent in the range of blood lead levels below 10 μg/dL.
Early studies using data from the National Health and Nutrition Examination Survey (NHANES) suggested that a baseline blood lead level in the range of 20 to 29 μg/dL was significantly associated with all-cause mortality, using individuals with a baseline blood lead level below 10 as the reference group [
As population blood lead levels have fallen, it has become been possible to evaluate associations in the range below 10 μg/dL. Menke
In another analysis of NHANES III (N = 9,757), Schober
The associations between lead biomarkers and total and cause-specific mortality were also evaluated, over a follow-up interval of nine years, in another cohort, 868 men (mean age 67 years) enrolled in the United States Veterans Administration Normative Aging Study (NAS) [
In 2006, IARC [
Since the IARC evaluation, the results of additional studies published do not suggest a need to revise this conclusion. In a study using the data on 13,946 participants in NHANES III (1988–1994), Menke
Among 868 men participating in the United States Veterans Administration Normative Aging Study (NAS), Weisskopf
In a case-control study of primary smelter workers, arsenic exposure, but not lead exposure, was a risk factor for lung cancer in 141 cases and age-matched controls [
Some recent studies suggest associations that warrant future consideration. Alatise and Schrauzer [
Lead has long been known to be a renal toxicant. Some recent studies focused on patients with clinical kidney disease, with some evaluating whether greater lead exposure is associated with the loss of kidney function. A large study of incident chronic kidney disease (CKD) cases among lead workers in Sweden (926 cases, 998 controls) failed to find an increased risk of CKD or faster rate of decline in GFR over a 7–9 year follow-up interval [
Lin and colleagues reported a series of studies on patients with CKD, evaluating whether the rate of decline in kidney function over time differed depending on lead burden. Some studies were observational. Yu
A case-control study compared the blood and tibia lead levels of 55 African Americans with end-stage renal disease receiving chronic hemodialysis treatment with those of 53 age- and sex-matched controls [
Recently, studies have evaluated the association between increased lead exposure and subtle renal impairment in the general population. Using adult (≥20 years old) participants in NHANES III (N = 15,211), Muntner
In a sample of adults from Taiwan, China (n = 1,565), Lai
One study investigated the association between lead exposure and change over time in renal function. In the NAS cohort, Tsaih
Relatively little information is available on lead exposure and renal function in children. In a sample of 769 healthy 12–20 year olds in NHANES III, Fadrowski
The few data available on lead exposure and renal function in even younger children suggest that higher blood lead levels are associated with increased GFR (as estimated by serum creatinine or cystatin C levels), suggesting a paradoxical effect that, perhaps, reflects a hyperfiltration phenomenon [
Several factors have been reported to modify the association between blood lead level and kidney function, although the evidence is inconsistent. These include certain genetic polymorphisms, including ALAD, the vitamin D receptor and nitric oxide synthase [
Like renal impairment, hypertension has long been recognized as a consequence of occupational exposure to lead, raising the question of whether a similar, perhaps more modest, association is evident between lead burden and blood pressure, as well as other indicators of cardiovascular function, at the lower lead exposures experienced by the general population. Most reviews (e.g., [
Among studies conducted subsequent to those included in the Nawrot
The associations between lead and blood pressure might also differ depending on the exposure biomarker used. Park
The mechanisms suggested for the association between lead exposure and blood pressure include lead-related impairments in renal function, oxidative stress, effects on the rennin-angiotensin system and suppression of nitric oxide. Two studies have suggested another potential mechanism, a lead-related increase in homocysteine level. One study was in a random sample of 1,140 50- to 70-year-olds (the Baltimore Memory Study) [
As noted previously, deaths from cardiovascular disease are usually found to account for much of the association between lead and overall mortality [
Limited data are available on the association between lead exposure and blood pressure in younger age groups. Gerr
A series of studies in 9–11 year olds with blood lead levels closer to general population levels suggest that early lead exposure (blood lead level measured at a mean of 2.6 years: mean 4.0 μg/dL, range 1.5–13.0) mediates the association between lower family socioeconomic status and greater salivary cortisol response to acute stress. Additional studies of this cohort suggested that that early lead exposure produces cardiac dysregulation, expressed as altered patterns of sympathetic and parasympathetic activation in response to such stress [
Lead at high dose impairs reproductive outcomes, and a variety of endpoints pertaining to have been evaluated in relation to maternal and paternal exposures. A recent review concluded that fertility is reduced in couples during periods in which an occupationally-exposed male has a blood lead level >40 μg/dL or a blood lead level in the range of 25 μg/dL for several years [
High-dose lead exposure has long been recognized as a risk factor for eclampsia [
In a study conducted in Kosovo [
The evidence that increased paternal or maternal lead exposure is associated with the risk of a congenital malformation in offspring is inconsistent. In some studies reporting an association, exposure classification was based solely on job title rather than a lead biomarker, precluding estimation of the critical dose. An increased risk of neural tube defects has been reported in an ecologic study of women residing in an area with high lead levels in water [
Considerable evidence supports the hypothesis that adverse effects on fetal growth occur within the range of general population exposures. In Mexican women, women with a tibia lead level in the highest quintile at one month postpartum were 1.79 (95% CI 1.10–3.22) times more likely than infants in the other four quintiles to have a below average birth length [
The U.S. Centers for Disease Control and Prevention [
A recent meta-analysis evaluated the association between blood lead level and peripheral nerve conduction velocities, latencies and amplitudes in adults [
The association between blood lead level and postural sway, measured using the Neuromotor Test System (CATSYS), was evaluated in a cohort of 181 workers (121 lead-exposed, 60 controls) [
In a case-control study, the mean blood lead level of 100 patients with ET (3.3 μg/dL [SD 2.4]) was significantly higher than the mean blood lead level of 143 controls (2.6 μg/dL [SD 1.6]) [
Neither of these studies involved incident cases of ET, and the cross-sectional design used in each makes it uncertain whether the higher blood lead levels of the cases preceded or followed the diagnosis of ET. Even if elevated lead exposure preceded the diagnosis and the role of lead is causal, these studies are of limited use in identifying the critical dose. Because the mean age of participants was greater than 50 years in both studies, their blood lead levels might have been considerably higher in the past, when the damage leading to ET is likely to have occurred.
Past lead exposure has been associated with the risk of ALS in case-control studies [
Many of the studies evaluating the association between lead exposure and adult cognitive function have relied on bone lead level as the exposure biomarker. Beyond the lack of widespread capacity to measure bone lead levels, it is difficult to integrate the results of studies that used these two biomarkers. The correlation between bone lead level and a concurrent blood lead level is often poor because of the very different exposure averaging times that these two biomarkers capture. In a study of 50- to 70-year-olds with primarily environmental exposures to lead, the correlation between bone and blood lead levels was only 0.12 [
Similar findings have been reported in cohorts drawn from the general population. In the Baltimore Memory Study, a longitudinal study of urban adults of diverse ethnicity, Bandeen-Roche
van Wijngaarden
Most studies of lead and adult cognitive function have involved only males. Weuve
Several variables have been evaluated as effect modifiers of the association between lead exposure and adult cognition. Among pairs of workers matched in terms of lifetime weighted blood lead level, Bleeker
Recent studies in rodents and non-human primates suggest a mechanism by which developmental exposure to lead might be a risk factor for neurodegenerative disease in adulthood. Animals exposed to lead only in early life show elevations of beta-amyloid protein precursor (APP) mRNA, APP, and its amyloidogenic product, Abeta, in old age [
In NHANES 1999–2004, 1,987 20- to 39-year-olds were administered a DSM-IV-based Composite International Diagnostic Interview. Individuals with a current blood lead level in the highest quintile (>2.11 μg/dL; 13 with a level >10 μg/dL), compared with those in the lowest quintile (<0.7 μg/dL), had 2.3 (95% CI 1.1–4.8) times the risk of meeting diagnostic criteria for a major depressive disorder and 4.9 (95% CI 1.3–18.5) times the risk for meeting criteria for panic disorder, adjusting for sex, age, race/ethnicity, education and poverty to income ratio [
Several recent papers have reported the results of applying various brain imaging modalities to lead workers or, less commonly, to samples drawn from the general population. White matter appears to be particularly vulnerable to injury as a result of lead exposure. Stewart
Grey matter volume in the adult brain is also associated with past lead exposure, as higher tibia lead levels are associated with reduced total brain volume, total grey matter and volumes in several specific regions, including frontal, the cingulate gyrus and the insula [
Mounting evidence from the Cincinnati Prospective Lead Study suggests that blood lead levels in childhood predict brain structure and function in young adulthood. Cecil
Lead-related changes in brain function have also been found in this cohort of young adults. Using proton magnetic resonance spectroscopy to measure levels of brain metabolites
Changes in brain volume might mediate lead-associated changes observed in adults' cognitive function. Among former organolead workers, larger volumes in different brain regions were associated with better scores on tests of visuoconstruction, processing speed, visual memory, executive functioning and eye-hand coordination [
To achieve a more powerful analysis of the quantitative characteristics of the dose-response relationship between children's blood lead levels and their IQ scores, particularly at blood lead levels <10 μg/dL, the data from seven prospective cohort studies were pooled (N = 1,333) [
A similar, supra-linear, relationship has been reported in other, independent studies since the publication of this pooled analysis (e.g., [
Numerous other studies have reported adverse outcomes in children at blood lead levels <10 μg/dL. In a cross-sectional study of 534 6- to 10-year-old children, adjusting for age, race, socioeconomic status and caregiver IQ, children with blood lead levels of 5–10 μg/dL, compared to children with a level of 1–2 μg/dL, had a 5 point deficit in IQ, a 7.8 point deficit in reading, a 6.9 point deficit in mathematics, as well as deficits in spatial attention and executive functions [
In a cohort of 246 7.5-year-old African American children (mean blood lead level of 5.4 μg/dL, range 1–25 μg/dL), Chiodo
In a cohort of 261 8- to 11-year-old children, blood lead level (mean 1.7 μg/dL, range 0.42–4.91) was inversely associated with IQ score (coefficient −0.18, P = 0.003), adjusting for age, sex, maternal education, paternal education, income, maternal smoking during pregnancy, exposure to second-hand smoke after birth, birth weight, maternal age at birth and blood manganese level [
The potential import of the lead-associated reductions in IQ (and other cognitive functions) are suggested by studies conducted by Miranda
The potential long-term implications of early-life lead exposure are suggested by the results of a follow-up, at approximately age 30 years, of children who had been enrolled, at birth, in the Boston Prospective Study [
Older studies using blood or tooth lead levels as the primary exposure biomarker consistently identified dose-related increases in behavioral outcomes such as inattention, distractibility and hyperactivity [
In NHANES 1999–2002, parents reported whether they had ever been told by a health professional that their 6- to 16-year-old child met criteria for ADHD and whether the child was taking a medication for ADHD. Braun
In a study of 1,778 children in whom blood lead levels ranged from 0.1 to 10.1 μg/dL (geometric mean 1.8 μg/dL) and for whom parents completed on the Connors' scale for ADHD, the risk of ADHD, defined as a score exceeding the cut-off derived for children from the Republic of Korea, increased linearly with increasing blood lead level [
In a study of children 4–12 years old, 630 cases who met DSM-IV diagnostic criteria were matched to 630 controls on age, sex and socioeconomic status [
In a study conducted in Romania (N = 83, 8–12 years old), Nicolescu
In an early case series, Byers and Lord [
Studies using an ecological design have reported significant associations between air lead concentrations and homicide rate [
This limitation has been addressed, at least in part, in several recent cohort studies. Braun
In a cross-sectional study of 173 14- to 18-year-olds in Brazil, surface dental enamel lead level was associated, after adjusting for familial and sociodemographic confounders, with the risk of clinically significant elevation of parent-reported rule-breaking behaviour on the Child Behavior Check List (OR = 3.72, 95% CI 0.99–14.04) [
Three prospective studies of environmental lead exposure and criminal activities have been reported. In the Christchurch Health and Development Study, a birth cohort of 1,265 children, dentine lead level in a deciduous tooth, measured at 6–9 years of age, was related, in a dose-dependent manner, to the number of violent/property convictions and self-reported violent/property offenses committed between the ages of 14 and 21 [
Among 488 children in the Avon Longitudinal Study of Parents and Children (UK), higher blood lead level at 30 months of age was significantly associated with greater antisocial behaviors at age 7–8, as reported by an adult [
The strongest epidemiological evidence germane to the hypothesis that early life lead exposure increases the propensity to criminality has come from the Cincinnati Prospective Study, which has followed children from pregnancy into young adulthood. It had been reported previously that adolescents in this cohort with higher blood lead levels in early childhood had engaged in more delinquent acts, but these outcome data were based on adolescents' self-reports [
In a meta-analysis, Marcus
The biological plausibility of the hypothesis that elevated lead exposure is causally associated with aggression is supported by studies in experimental models, including cats [
Higher blood lead level has been associated with delayed sexual maturation in cross-sectional studies. Among 2,186 8–18 year old girls in NHANES II, breast and pubic hair development (Tanner staging) as well as age at menarche were significantly delayed in African-American and Mexican American girls with a blood lead level ≥3 μg/dL, compared with girls with a blood lead level of 1 μg/dL [
In a study of 138 10- to 17-year-old girls from the Akwesasne Mohawk Nation in the USA, Denham
Sexual maturation in boys (physician-assessed testicular volume and genitalia stage) was evaluated in 489 8- to 9-year-old boys in Chapaevsk, Russian Federation [
In adults, greater lead exposure has been associated with risk of tooth loss. In the NAS cohort, men in the highest tertile of tibia lead level were 3.0 (95% CI 1.6–5.8) times more likely than men in the lowest tertile to have more than nine missing teeth. Men in the highest tertile of patella lead level were 2.4 times as likely (95% CI 1.3–4.5) [
In children, lead exposure has been reported to be a risk factor for dental caries, but the evidence is mixed and lacks consistency across studies in terms of the patterns of associations. Among 24,901 participants ages 2 years and older in NHANES III (oldest age stratum was ≥12 years), a higher blood lead level was significantly associated with the number of delayed, filled, and missing surfaces in both deciduous and permanent teeth, adjusting for age, race, poverty-to-income ratio, cigarette exposure, sex, region, parent education, carbohydrate intake, dietary calcium intake and dental care [
In 543 6- to 10-year-old children enrolled in the New England Children's Amalgam Trial [
In analyses of 507 8- to 12-year-old children participating in another study of dental amalgam, Martin
Although IQ deficit in children is typically chosen as the critical endpoint for the purpose of risk assessment, recent studies, primarily conducted in developed countries, show that many aspects of health are impaired at exposure levels prevalent in the general (
Recent risk assessments have concluded that a "safe level" of lead exposure has not been identified. For example, in 2010, the FAO/WHO Joint Expert Committee on Food Additives (JECFA) argued that dose-response analyses do not provide any indication of a threshold for critical adverse effects (IQ loss in children, increased blood pressure in adults) and, as a result, withdrew its Provisional Tolerable Weekly Intake (PTWI) of 25 μg/kg body weight [
A disproportionate share of the morbidity associated with lead exposure is borne by developing countries. The World Health Organization noted that, in 2000, approximately 10% of children had a blood lead level of 20 μg/dL or higher, but that 99% of these children lived in developing countries and that lead exposure accounted for nearly 1% of the global burden of disease [


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The Standing Stone: a landslide-emplaced monolith on the North Branch Susquehanna River near Towanda, Pennsylvania
Inners, Jon D. (retired), Fleeger, Gary M., and Delano, Helen L., Pennsylvania Geological Survey, 3240 Schoolhouse Road, Middletown, PA, 17057
The Standing Stone is a gray sandstone monolith that stands erect on the southeast bank of the North Branch Susquehanna River 6 mi southeast of Towanda, Pennsylvania. First described by an officer of Sullivan's expedition against the Iroquois in 1779, the stone has given its name to a township on the opposite side of the river, and also marks the northwest boundary of historic French Azilum. It is situated at the base of a steep, northeast-southwest bluff toward the north end of the "incised-meander reach" of the North Branch on the glaciated, Allegheny Plateau. Bedrock is mapped as Upper Devonian Lock Haven Formation.
The stone is at least 25 ft high, 13.5 ft wide, and 4 ft across bedding. In the immediate vicinity of the Standing Stone are three other large rock slabs lying parallel to the river and leaning against the bank. The monolith is situated just offshore of a narrow Holocene alluvial terrace that occurs at the base of an extremely steep slope capped by a high sandstone cliff, the top of which lies 330 ft above the river.
The Standing Stone and the other rock slabs in its vicinity were emplaced by a large landslide involving collapse of a jointed section of cliff directly above. Weathering and downslope colluviation of a bed of soft, gray shale underlying the sandstone initiated the failure. The eroded toe of the slide along the river is about 350 ft wide and is now largely covered by the alluvial terrace. The slide material on the slope between the terrace and the cliff is composed of angular sandstone blocks in a shaly matrix. Many smaller landslides occur on the adjacent slope and on the surface of the slide itself. Based on radiocarbon dates on the "16-ft-terrace" from elsewhere on the upper North Branch, the large rock-fall event probably took place between 8000 and 10,000 yrs B.P.
Poster paper presented at the 38th Annual Meeting of the Northeastern Section Geological Society of America, Halifax, Nova Scotia, March 27, 2003.
Inners, Jon D., Fleeger, Gary M., and Delano, Helen L., 2003, The Standing Stone: a landslide emplaced monolith on the North Branch Susquehanna River near Towanda, PA: Geological Society of America Abstracts with Programs, v. 35, no. 3, p. 8.


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iTunes subscription problem
View Single Post
Oct 11, '11, 9:32 pm
Regular Member
Join Date: April 23, 2010
Posts: 1,507
Religion: Catholic
Re: iTunes subscription problem
Sometimes there is a need to shout so that one can be heard.
This is a problem very much resolvable by the Catholic Answers Live staff, and Apple has little to do with it. All the CAL staff has to do is redirect the subscription's URL to the new URL from the old one. Just a few lines of code to change the feed - that falls on the CAL staff, not Apple. I have seen this done before by some podcast producers, and there are easy tutorials available online, even some from Apple.
This is basic 101 podcasting - CAL should be on top of this.
View Public Profile
Find all posts by Windfish


text:
BaitMasters is a new linkshell specifically for Fisherman of all levels. If your interested in fishing or are currently fishing and would like to join a fishing based linkshell as well as a social shell that is not formal then we are the place for you! There are only 2 basic rules, you must fish and you must show everyone respect just like you would anywhere else!
If this interests you then send me a tell on either of my guys:
or send Kherkus the Co-Leader a tell as well for a pearl.
/Welcome and I will see you in game!
Good Day Everyone


text:
Tim McCarver gave me my one and likely only mention during a national telecast of a World Series game. He credited me for a line in the Chicago Tribune during the 1987 Minnesota-St. Louis series on ABC.
I wrote that the teflon roof of the ugly Metrodome "looks like your grandmother's old jello mold."
"I remember that line," said McCarver 25 years later when I reminded him of it.
Whether he did or not, it was quite a thrill for a young reporter to get some exposure on national TV.
Fortunately for McCarver, he has had much better material to work with through the years. It's been quite a run for the former St. Louis and Philadelphia catcher, who decided to give broadcasting a try in 1980.
The pinnacle comes next week when McCarver will be honored in Cooperstown. He is the 2012 recipient of the Ford C. Frick Award presented by The National Baseball Hall of Fame and Museum for excellence in baseball broadcasting. McCarver only is the second primary television analyst to win the Frick Award, joining Tony Kubek, who received the honor in 2009.
The honor is long overdue. His numbers during a 32-year broadcast career are almost Gretzky-like. Tuesday, he will work his 21st All-Star Game. The next closest are Joe Buck and Curt Gowdy with 14. In October, he will be on the call for his 23rd World Series.
McCarver had a notable 16-year stint working games for the Mets. He has the distinction of being the only MLB analyst to have worked for all four major broadcast networks. Since 1996, he and Joe Buck have been a team at Fox.
Now 70, McCarver remains trim and enthusiastic about his job. Yet with one more year remaining on his contract, he knows he might have a decision to make about his broadcast future after the 2013 season.
I met with McCarver on a Friday morning while he was in town to call a Cubs-Boston game at Wrigley Field. Here's the first part of our Q/A.
How do you feel about getting this award?
If somebody told me back in 1980 that I would have a 32-year career, and that I'd be receiving this honor, I'd say no way. For three years, I couldn't even break into the Phillies broadcast booth. I was just hoping to make it, much less be mentioned as a Ford Frick winner. Believe me, when I started out, this award wasn't even close to being on the radar.
How do you think you'll feel being up on the stage in Cooperstown?
I've only been to Cooperstown once when Steve Carlton was inducted. I suppose it's a very personal summation of your professional life. It makes me proud of what I've accomplished. That's what makes this award so fulfilling.
How have you viewed your role as an analyst?
I had no training to be a broadcaster. My training came from being behind the plate. When you come to think about it, that's a good way to be trained.
You see the choreography of the game from behind the plate. Without realizing it, you're storing up all this information.
You're looking at all the positions on the field. You see what the shortstop is doing. You see the second baseman cheating in for a doubleplay. So it all gives you an advantage.
Your timing was good. The baseball broadcast in the 80s evolved into putting more emphasis on analysis.
My job was different than the great voices of the game. My job was to explain the how and why. Whenever I've gotten into trouble, it's because I've gotten away from explaining the how and why.
People watching on TV can see how something happened during a game. Fortunately, whether they realized it or not, they wanted to know the how and why it happened. I was in a position to explain the game as I saw it, and I saw it differently than a lot of people.
Early on, you had a reputation for being extremely candid, perhaps more so than what was the norm back then. How did players react to you?
Remember, I had played with a lot of the guys. One night, I did a Phillies game and Mike Schmidt hit a ball off the top of the wall. He always hustled, but he watched the ball and got a double. I said, 'Schmidt should be on third base.' Then I said, 'Often, hitters are like artists. They step back and admire their work. They don't run as hard. It's understandable why he's on second, but he really should be on third.'
Mike and I are close friends. The next day, he was acting cool towards me. Common sense says you should deal with it right away. I said, 'Schmidty, is everything OK?' He said, 'No, it's not. Don't ever on the air say I didn't hustle.' That's what his father told him I said.
I said, 'I didn't say that.' I explained to him what I said and we were fine.
In New York, I guess I got this reputation (for being overly candid). Listen, I played with a lot of guys who were very direct and honest. Bob Gibson, Bill White, Curt Flood. They said what they felt. I learned it from them. I always approached playing the game in a candid way. I guess it carried over into broadcasting.
Some players may be upset with me from time to time, but overall, nobody can question my fairness. I have no regrets in the way I approached things back then and the way I approach things today.
You have your own critics. Some people say you talk too much and overanalyze.
Did I talk too much (when he first started)? Absolutely. I talked too much because of my enthusiasm for the game. That was applicable back in 1985, but then it followed me into the 90s. (By then), it wasn't true. I learned. Of course, I did. You're always trying to improve yourself. You're talking about your business. You're talking about the way you do your job.
How do you feel about the critics?
Whenever you hear the term human nature, it's always for something negative. Nobody will ever say, 'He's a great guy, but that's human nature.' What is it about we humans that we tend to use that term negatively?
I try not to get caught up in it. I don't read the blogs. I've got a job to do. I don't pay attention to the negative stuff.
Tuesday: McCarver reflects on his broadcast partners and talks about how much longer he wants to work.


text:
Abstract: Rural non-agricultural employment (RNAE) is being
increasingly emphasized as a potential pathway out of rural poverty for
people who are unable to secure their income in agriculture. Although
average earnings in the rural non-agricultural sector are higher than in
agriculture, it is unclear whether income prospects are systematically
better in non-agricultural activities than in agriculture. This paper tests
for existence of earnings differentials between agricultural and rural
non-agricultural employment, while controlling for worker and household
characteristics. A theoretical farm household model is proposed that
predicts that there will be no sectoral earnings differential for unskilled
labor, whereas skilled labor will be better off in the non-agricultural
sector. Based on Peruvian household data, the empirical findings do not
support the notion that unskilled workers would earn a higher income by
switching from agriculture to RNAE. Instead it tends to be the relatively
well educated who might benefit from higher returns to education in RNAE
than in agriculture, consistent with the predictions of the theoretical
Keywords: non-agricultural employment; income diversification; wage differentials; Peru; Latin America; (follow links to similar papers)
JEL-Codes: J24; J31; J43; O12; R23; (follow links to similar papers)
21 pages, February 29, 2008, Revised February 21, 2009
Before downloading any of the electronic versions below
you should read our statement on
Download GhostScript
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Full text versions of the paper:
Programing by
Design by Joachim Ekebom


text:
|From: Robby Findler (robby at eecs.northwestern.edu)
Date: Sat Aug 13 17:26:34 EDT 2011
Well, someone has figured out how to defeat the recaptcha thingy and I'm now getting about (on average, ish) two spam bug reports per hour for the last several days (and a slower, but still annoying rate for several weeks before that). Any advice? (poking around the web suggests that captchas are basically useless nowadays anyways). Robby
|Posted on the dev mailing list.


text:
Recent Posts
Fancy A Date?
Developer- and Publisher-specific Articles: n-Space
Skylanders Swap Force set to release this fall for 3DS
Skylanders will be venturing out into handhelds again this fall with the launch of Skylanders Swap Force for 3DS. The n-Space-developed entry will feature a new story, new levels, and three new starter-pack characters (Free Ranger, Rattle Shake, Volcanic, and Eruptor). The story follows the attack on Boomtown, the hometown of Flynn, and the kidnap of Cali by a new villain, Count Monkeybone. Players must save the town and Cali from this new menace.
Skylanders Giants event taking place Feb. 5 at flagship Toys "R" Us
Skylander fans will be able to snag the upcoming character Ninjini before its official launch, and a few discounted figures as well, on February 5 at an event at Toys "R" Us Times Square. The festivities are in honor of the annual International Toy Fair, and in celebration, the first to line up will receive a free Ninjini figure. Those who show up later will still be able to participate in giveaways and a day-long sale, which includes $25 off a Giants Starter Pack and a 'buy 2, get 1 free' for figures. Characters will be on hand to greet attendees, and playable demos will be setup at kiosks.
Skylanders franchise surpasses $500 million in sales
It appears that the Skylanders craze continues unabated as Activision has revealed that the franchise has surpassed $500 million in retail sales. The figure includes all games, accessory packs, and figures sold in the U.S., and according to Activision, this milestone has been reached "faster than any other kids' gaming property." The latest title in the series, Skylanders Giants, has earned more than $195 million in the U.S. and was the best selling kids' game of last year.
Skylanders Giants invading retail this Sunday
Gamers of all ages have made Toys for Bob's Skylanders a hit, and for those who've been waiting for a new adventure, the wait is almost over. Activision has announced that the savior of many a parent's sanity will return this Sunday with Skylanders Giants. The sequel will once again pit portal masters against the nefarious and stubbornly resilient Portal Master Kaos. Joining the original crew, who carry over all stats and customizations, are 16 new characters, including eight double-sized Giant Skylanders. The new Wow Pow system adds a new upgrade path for new and old Skylander alike—looks like it's time to break out Trigger Happy.
Heroes of Ruin demo brings RPG action to Nintendo eShop
A new demo has launched on the Nintendo eShop for Square Enix's upcoming 3DS action-RPG Heroes of Ruin. The trial allows players to create a character from two of the game's four classes, the sword-wielding Vindicator and firearm-toting Gunslinger, and explore the Elder Forest. The forest hosts several side quests and a boss battle against the spirit known as Michadele, all which can be played through solo or with friends via local and global multiplayer.
Heroes of Ruin to set off on perilous journey this June
Square Enix has announced that their upcoming 3DS action role-playing game Heroes of Ruin will be hitting store shelves on June 26. Built from the ground up for the 3DS, Heroes of Ruin will support online multiplayer, with drop-in/drop-out play, StreetPass for accessing the Traders Network, and real-time chatting via the built-in microphone.


text:
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There are outstanding changes not yet made by the legislation.gov.uk editorial team to Landlord and Tenant Act 1987. Any changes that have already been made by the team appear in the content and are referenced with annotations.
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This section lists the commencement orders yet to be applied to the whole Act. These effects are included in this view as they may be (but won't necessarily be) relevant to the specific provision that you are viewing. Where applicable the commencement orders are listed under two headings, firstly those that bring some part of the Act you are viewing into force and secondly, those that bring into force legislation that affects some part of the legislation you are viewing. If you are viewing a prospective version or there is a prospective version available there may be commencement orders listed here that are relevant to the provision you are viewing.
Commencement Orders bringing legislation that affects this Act into force:
(1)The requisite majority of qualifying tenants of the constituent flats may serve a notice on the purchaser requiring him—
(a)to give particulars of the terms on which the original disposal was made (including the deposit and consideration required) and the date on which it was made, and
(b)where the disposal consisted of entering into a contract, to provide a copy of the contract.
(2)The notice must specify the name and address of the person to whom (on behalf of the tenants) the particulars are to be given, or the copy of the contract provided.
(3)Any notice under this section must be served before the end of the period of four months beginning with the date by which—
(a)notices under section 3A of the M1Landlord and Tenant Act 1985 (duty of new landlord to inform tenants of rights) relating to the original disposal, or
(b)where that section does not apply, documents of any other description—
(i)indicating that the original disposal has taken place, and
(ii)alerting the tenants to the existence of their rights under this Part and the time within which any such rights must be exercised,
have been served on the requisite majority of qualifying tenants of the constituent flats.
(4)A person served with a notice under this section shall comply with it within the period of one month beginning with the date on which it is served on him.]
Annotations are used to give authority for changes and other effects on the legislation you are viewing and to convey editorial information. They appear at the foot of the relevant provision or under the associated heading. Annotations are categorised by annotation type, such as F-notes for textual amendments and I-notes for commencement information (a full list can be found in the Editorial Practice Guide). Each annotation is identified by a sequential reference number. For F-notes, M-notes and X-notes, the number also appears in bold superscript at the relevant location in the text. All annotations contain links to the affecting legislation.
Amendments (Textual)
F1Ss. 11, 11A, 12A-12D, 13, 14 and crossheading substituted for ss. 11-15 (1.10.1996) by 1996 c. 52, s. 92(1), Sch. 6 Pt. II; S.I. 1996/2212, art. 2(2) (with Sch.)
Marginal Citations
M11985 c 70.
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text:
The final debate of the 2012 presidential election will begin in just a few minutes. Unless Mitt Romney brings it up, I doubt that there will be any questions about any of the Islamic radicals that have been frequent White House visitors since President Obama took office. Heck, it's like he's rolled out the red carpet for them.
A year-long investigation by the Investigative Project on Terrorism (IPT) has found that scores of known radical Islamists made hundreds of visits to the Obama White House, meeting with top administration officials.
Court documents and other records have identified many of these visitors as belonging to groups serving as fronts for the Muslim Brotherhood, Hamas and other Islamic militant organizations.
The IPT made the discovery combing through millions of White House visitor log entries. IPT compared the visitors' names with lists of known radical Islamists. Among the visitors were officials representing groups which have:
- Been designated by the Department of Justice as unindicted co-conspirators in terrorist trials; Extolled Islamic terrorist groups including Hamas and Hizballah;
- Obstructed terrorist investigations by instructing their followers not to cooperate with law enforcement;
- Promoted the incendiary conspiratorial allegation that the United States is engaged in a "war against Islam"— a leading tool in recruiting Muslims to carry out acts of terror;
- Repeatedly claimed that many of the Islamic terrorists convicted since 9-11 were framed by the U.S government as part of an anti-Muslim profiling campaign.
Read the whole thing, and remember this on election day.


text:
Hi there,
Soon I will be starting a new project that will be a mens styled magazine. I was thinking about making a front page myself to pull wordpress content in. But at the same time after reading about wordpress, I can do anything with wordpress really anyway... So would a full wordpress site be the most functional and efficient means of creating my website?
There's no reason why you couldn't get it to do whatever you might want it to do.
Whether or not it's the best choice, depends on any number of factors. But overall, if you're going to be managing it, I would pick the system that you find the easiest to use, and that allows you the most flexibility to do the things you need it to do.
This means auditioning them and trying them out yourself .... get a little firsthand evaluation.
Thanks for the advice, I agree I should go with what I'm most comfortable with, thanks.


text:
Penn Hills Summer Playground Program offers outdoor opportunities for kids
What is it?
Parents looking for a little peace and quiet around the house once school gets out can enroll their children in the Penn Hills Parks and Recreation Summer Playground Program. Children will have a chance to get outdoors and participate in summer activities at several of Penn Hills' parks.
When is it?
The program will run from 9 a.m. to 2 p.m. Monday through Thursday, and from 9 a.m. to 1 p.m. on Fridays.
What's available?
Parks and recreation director John Scaglione said children will have access to a variety of games, sports, arts and crafts. "Penn Hills Park offers the opportunity for nature hikes, and Universal Park is pretty wide open and great for sports," Scaglione said. Special events, including a visit from a Penn Hills K-9 officer and the program's annual mud decathlon, will take place throughout the summer.
Who's in charge?
Scaglione said children will be under the supervision of instructors who are all college-aged or older, who help lead the day's activities. "We don't call it a daycare or program it that way. It's more of a classic summer playground program," Scaglione said. The program is geared toward children ages 6 to 12.
Where do I sign up?
Interested parents can sign up at the parks and recreation building, located at 6600 Leechburg Road. The fee is $50 for the first child, and $40 for each additional child. For more information, call 412-798-2147.
Patrick Varine is an editor for Trib Total Media. He can be reached at 412-320-7845 or email@example.com.


text:
Marker Cluster in Google Maps indicates multiple nearby locations marked up in an aggregated form in a map. You can create and display marker clusters on your Google map by activating the WP Google Map Pro plugin for WordPress. The Map below is marking multiple locations using draggable markers over the same Google map.
This google map wordpress plugin is available on codecanyon to get it.
Customize Product according to your requirements?
We will be glad to discuss any of your needs related to our wordpress plugins . if any of functionality you'd like to see in products, you can reach us by via email firstname.lastname@example.com.


text:
This week we're getting Replicator Wednesday hosted at the SF Techshop in SoMA.Ā

If you're in the area, come by and bring your RepRaps*, Makerbots, Printrbots, Cubifys, Solidoodles, Shapercubes, Mosaics, BfB's, Ultimakers, Stratasi, Z-Corpsy, and yes of course bring your Type A Machine Series1's!

If I left anythingy off that list, than bring it in and show it to me (Miloh) for a photo-op and a chance to win a prize!ĀIf you don't got a 'bot, but you do gotta bot (ie. want to be 3d printing), then just bring your bad self. We will show you how to slice and dice and fabricate objects for 3D printing. We'll be there from 6PM to 10PM in the meeting room on the Mezzanine floor, and the first 2 pizza pies are on me.

San Francisco's Techshop is at [926 Howard St] and you DO NOT need to be a member to join us. Just put down the spool and the slic3r and come join us. When you get there, tell the friendly staff that you are with the World Domination 3D printing Society.ĀParking is free after 6PM in nearby areas of SoMa. ĀYou really ought to take BART or MUNI though, because nothing beats taking a extra 2 seats with your Mendel Max. No one's going to fault you taking some extra space for your big bad 80/20 'ellbot. Especially if it talks or sings. If it talks or sings I give you TWO prizes.

One more time: Come to Replicator Wednesday at SF Techshop, July 11th, 2012. Ā6PM to 10PM*Ā

Hope to see you there!

-Ronald Miloh Alexander

*family members of the reprap Darwin Mendel and Huxley are cataloged and disqualified from sweepstakes. Ā3rd cousins and evil twins will be considered on a case by case basis.

**we are very generous with our time since waiting on 3D prints has already STOLEN OUR YOUTH. Ā;-)

ps ĀReplicator Wednesday will return to Noisebridge on starting July 18th!


text:
Let's not overlook gun use in suicides
Isn't it great that the NRA began its annual convention on the same day the CDC released the latest suicide statistics?
Suicide rates in persons ages 35-64 increased 28 percent from 1999 to 2010 and it is now a more common cause of death than motor-vehicle accidents. The most common method of committing suicide is by firearm, accounting for 10,393 deaths in 2010, which is 48 percent of all suicides that year. In addition, there are accidental gun deaths.
Guns don't kill people. People kill themselves with guns.
Timothy C. Fagan, M.D.
Physician, Tucson
Chuck George deserves support, good wishes
Re: the May 3 article "KOLD's George says he's depressed."
I'm sure I'm not alone in wishing Chuck George success in his battle with depression. Judging from his on-air persona, this was probably one of the last diagnoses I would have thought of. He is so well-liked in our community, I hope he will continue with his treatment and stay on the job. This is nothing to be ashamed of, and he is not alone!
Jan Foiles
Retired, Tucson
Sen. Melvin falsifies school money numbers
State Sen. Al Melvin was on Bill Buckmaster's local news radio show recently where Melvin once again implied the state funds public education at $9,000 per pupil. This despite AZ Fact Check proving it false during his 2012 campaign and the Joint Legislative Budget Committee's report that state-only funding per student has been less than $5,000 every year since FY 2004. In fact, Arizona leads the nation in cuts to per-pupil funding since 2008 - almost 22 percent.
These facts are important because one of Melvin's key issues is "universal school choice where every child in the state has $9,000, which is roughly what we are spending now." Forget that a state appeals court ruled vouchers for private schools unconstitutional in 2009.
With over 1 million students in the state, the total bill is over $9 billion, more than the state's entire budget for 2013. This isn't a bold new idea, it is fantasy.
Linda Thomas
Colonel, USAF (ret.), Oracle School Board member,
The opinions expressed in this letter are Thomas' and not those of the Oracle School Board.
No, Obama gets vote as worst president
Re: the May 1 letter to the editor "No need to wait 50 yrs. to know Bush was worst."
Bush is the worst president? I believe Obama is the worst president ever. I'm disappointed that you would publish a letter with such strong opinions, and that is why I'm giving mine.
Audrey Turner
Retired banker, Tucson
Destroy a gun? An abomination!
Re: the April 30 article "Bill signed preventing Tucson from destroying 'buyback' guns."
Do Arizonans really idolize their guns? The thousands of lives destroyed by guns? Just the price we pay for the Second Amendment. But destroy guns? Oh, the horror!
Betty Feinberg
Bottom line in debate on guns is freedom
Re: the May 5 letter to the editor "Flake didn't 'stand up' for this person."
I'm sorry the writer just doesn't get it. While it's true background checks for gun purchasers would do nothing to control the anger and violence in our country, the lady asks why the strong objection to any of the gun legislation? Freedom, lady. Most Americans live here for our precious freedom. Got it?
Arthur H. Phillips
Retired, SaddleBrooke
Perseverance, grit abound at Basis, UHS
Re: the May 5 column "Let's keep Basis, UHS in perspective."
The high ranking of Basis and UHS by varied publications is a measure of their student's efforts: to do well in school, understand the material taught, attain high grades, and perhaps even get a scholarship. UHS, a public school, ranks highly in several scholastic areas, not just AP scores.
With two sons who have done well at UHS and on several AP tests, I have seen firsthand their many, many hours of study; a better measure of work ethic, perseverance and grit I do not know.
A.P. Lorenzen
Retired, Tucson
We may know truth on Benghazi - or not
Re: the May 7 article "Diplomat: Special ops held from Benghazi attacks."
Now maybe, just maybe, the truth will at last come out on how this administration really let us down by denying fighter assistance during the Benghazi attacks.
But fear not, you liberals! Hillary Clinton will get another case of the mysterious vapors. She'll fall down again and won't remember very well. Then the rest of the Cabinet will circle the wagons, hold hands and blame it on the past administration.
See? Not to worry.
Steve Shero
Retired, Tucson


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I've learned that Zodiak USA CEO Grant Mansfield will leave the company in June to return to his native U.K. Mansfield served as Managing Director and Group Director Factual, Entertainment and Comedy for RDF TV in the U.K. before he was named CEO of RDF USA in January 2010. Nine months later, the company was renamed Zodiak USA following Zodiak Entertainment's acquisition of RDF Media Group. Word is that Mansfield, who uprooted his family to move to Los Angeles, had been looking to return to London. Search is under way for a new CEO to replace him. Under Mansfield, Zodiak USA launched Celebrity Wife Swap and Secret Millionaire on ABC, both of which he executive produced. Additionally, the company created a franchise with truTV's flagship series Hardcore Pawn, which has spawned a spinoff, Hardcore Pawn: Fort Bragg. Coming up new reality series Steve-o's Killer Karaoke on TruTV and second seasons of Being Human on Syfy and Secret Millionaire.
Zodiak USA CEO Grant Mansfield To Exit
5 Comments
Uh oh. Looks like trouble in paradise.
Tony Yates deserves the job (if he wants it) – he's a class act, one of the brightest producers / executives out there and one of the good guys.
A confused entity right off the bat when a hair brained Belgian was at the center of things and tried to manage the merging of French, Swedish, English and Italian culture. Bad mix, mediocre talent and most important a lack of vision for what a company could and should do in order to make money in the global business of television and media. Too bad, missed opportunity and although hair brained, the Belgian was a nice guy.
Someone has to say it … and everyone is thinking this. Every show mentioned in this article was set up while Chris Coelen ran RDF. There is not a single new show on this list. That explains a lot. They've not sold anything worth while in 2 years.
LOL…so true!!


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BMW ends X3 (E83) production in Austria and begins X3 (F25) production in Spartanburg
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Tar Heel Faithful
Location: WNC
Join Date: Oct 2005
Posts: 14,125
Mein Auto: 2006 325i; 2015 Yamaha R3
View My Garage
Originally Posted by
What's wrong with your Dad's X5?
Airbag warning light (restraint system) tripped twice within a month, just about a month after delivery. Annoying wind whistle from the passenger side, not sure what causes it but it's audible everywhere in the cabin. Couple rattles in the cabin, not sure where they're coming from either (me thinks something in the door panels?). The cruise control stalk is a bit loose I think, because it clicks loudly every time I tap it (as opposed to my mom's 325i cruise stalk, which works silently).
__________________________________________________ ____________________________________________
There's other things that aren't really faults or defects, but just a matter of design and engineering. The interior dash panels don't look very well-fitted or line up particularly evenly. The suspension is way too soft; the car bobs forward and backward every time one taps the brakes or accelerates lightly due to the massive weight shift. It just doesn't work on single-lane roads and the steering is more numb than ever. The power delivery isn't very linear (I will attribute this to a lower-than-minimum recommended cetane level in US diesel fuel).
The Nevada leather feels like vinyl especially on the door panels. The standard seats are uncomfortable no matter how I adjust them. The stereo is woeful and although I know it's the base system and don't expect a lot, I can't get the equaliser to adjust the way I want- the highs are too sharp and piercing, yet the overall sound is muddy. The instrument cluster centre display is too dim, and I wish the green indicator lights weren't at the very sides of the cluster.
When I worked at a dealership 2 years ago, part of my job was to drive all the cars up to the front and photograph them. Out of all the models I drove, the X5 was my least favourite. It simply felt big, cheap, and uninspired. Complete opposite of the 5er or 7er. But I don't often live at home and don't really drive his car much, so it's not like I really mind too much. (Until I go home and he insists that I drive it to the store)
Ironically, I'm the only one that can consistently park the huge thing correctly, lol.
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text:
High-school basketball is pure and uncomplicated, free from pro ball's outrageous contracts or college coaches' locker-room readings of Nietzsche. Cheer on the innocence of youth with this GrouponLive deal to see the first session of the Chicago Elite Classic at the UIC Pavilion. For $13, you get one general-admission G-Pass (valid for 100- or 200-level seating) on Saturday, December 1, starting at 9 a.m. (up to a $27.01 value, including all fees). Doors open at 8:30 a.m. Because the ticket is a G-Pass, Groupon customers can use it to enter the venue directly; they will not need to redeem their Groupon at will call. The first session includes the following games:
- 9 a.m. – Wendell Phillips vs. De La Salle
- 11 a.m. – University High School vs. St. Vincent–St. Mary
- 12:45 p.m. – St. Joseph vs. Mater Dei
- 2:30 p.m. – Proviso East vs. Lone Peak
The Chicago Elite Classic brings together some of high-school basketball's brightest talent, splitting invites between top Illinois programs and schools from around the country. An intracity contest kicks off the weekend, as Chicago's Wendell Phillips takes on De La Salle, whose star shooting guard, Alvin Ellis—recently committed to Minnesota—looks to continue to improve his game after a bitter loss in 2012's regional title match, in which he scored a career-high 42 points. After running suicides down Interstate 90 all the way from Akron, Ohio, 2016 prospect Vincent King leads St. Vincent–St. Mary—LeBron James's alma mater—against U-High, whose own star forward, Keita Bates-Diop, Jr., dominates in the paint as one of the top juniors in the nation.
Later, the squad from Maywood's Proviso East hits the hardwood to take on Lone Peak, whose dominant program has captured four Utah state titles in the last six years. Proviso East's roster includes returning guards Sterling Brown and Paris Lee, whose on-court heroics garnered the team an undefeated 2011–12 record and their first postseason berth in 20 years. Led by legendary coach Gary McKnight—whose lifetime 926–83 record makes him the winningest coach in California history—Santa Ana's Mater Dei brings the heat against St. Joseph and 6'6" sophomore guard Paul Turner, whose imposing height eases dunks and complicates the team's annual submarine retreat.
Due to security restrictions, G-Passes must be printed out and presented in person at the event. They cannot be redeemed through Groupon's mobile app.


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I usually don't post every vid to come from activists in NH, cuz that's like 10+ vids a week, but when it's really bad, why not self-plug? Plus, this is just across the border, so it's Vermont staties being authoritarian thugs this time.
But don't take my word for it, see for yourself.
So yeah, this made me sick. Would anyone actually care to defend such tyrannical action?


text:
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text:
|From: Matthew Flatt (mflatt at cs.utah.edu)
Date: Wed May 12 12:17:49 EDT 2004
At Wed, 12 May 2004 17:06:58 +0400, Alex Ott wrote: > What do you think about C++ aproach to encoding and locale conversion for > ports and strings? If I understand C++'s approach to ports/streams correctly, the encoding between strings and bytes is a (mutable) property of the stream object. In MzScheme 299, functions like `read-char' and `write-char' assume a UTF-8 decoding of the stream. To get a different kind of decoding, you wrap the port to convert between UTF-8 and the other encoding. (In the near future, `(lib "port.ss")' will provide a port->port wrapper.) For obvious reasons, I would be opposed to a mutable property on ports to specify the encoding. I did consider making a port's encoding a property that is specified when the port is created, but that's too early in many cases --- exactly the mess of having distinguish text and binary streams. To put it another way, MzScheme's strategy is more like Java's, where a basic stream works in terms of bytes, and conversions are implemented by wrappers on basic streams. Technically, MzScheme's protocol can introduce wasted work (e.g., an encode followed by an immediate decode), but the advantage is that there's only one kind of port. We don't have to add `current-byte-input-port', `open-input-byte-port', etc. Matthew
|Posted on the users mailing list.


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In the last couple years, you've seen mass-produced, 100 percent electric cars take to the streets in the Northwest. In the same vein, now come the first battery powered buses. And we're not talking about trolley buses that get juice from overhead wires, as correspondent Tom Banse explains.
Wenatchee-based Link Transit is the first out the gate in our region. It plans to replace five diesel-powered trolley buses with battery electric models. A California company called EBus has delivered three so far. Link Transit general manager Richard DeRock says right now the battery buses are just running a few hours in the mornings while bugs get worked out. But DeRock says dramatic fuel cost savings are already apparent in each electric bus.
DeRock: "We can operate (one) for a month for about $90 worth of electricity. That compares to about $1400 worth of diesel fuel that we were using previously. So, there is almost a 14 to 1 cost savings on the energy."
Next in line in the region to deploy an electric bus is probably Ben Franklin Transit, serving Washington's Tri-Cities. They're using federal grant money to refit a regular city bus with an electric motor and batteries. Finally, the Seattle area's Metro Transit is shopping for battery electric buses. But a spokeswoman says the agency hasn't found a model that is "viable" for its needs yet. I'm Tom Banse reporting.
Copyright 2012 Northwest Public Radio


text:
Watermelon Vest by Maureen Mason-Jamieson
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Watermelon Vest
Pattern is no longer available at original source and is no longer available online at other sources. No Internet Archives exist.


text:
Pianist Jamal returns to his heart in Pittsburgh
Pianist Ahmad Jamal says he always is happy to return home to the "phenomenon" of Pittsburgh.
He admits to constantly marveling over the great cast of characters this city has produced — from writer Gertrude Stein to pianist Erroll Garner — and believes the area's work ethic has a great deal to do with it.
"Pittsburgh people know the philosophical truism that if you are spending your time trying to please everybody, you are a damn fool," he says. "They are a great group of risk-takers. Who plays bass like Ray Brown or sings like Billy Eckstine or plays sax like Stanley Turrentine?"
That sort of appreciation, he says, makes him proud to be part of the group of jazz legends from Pittsburgh. He also is glad to perform in the cozy settings of the Manchester Craftsmen's Guild on the North Side, where he will be Sept. 27 and 28.
The setting seems far removed from recent appearances at the Marciac Festival in France or concerts in Johannesburg, South Africa, or New York City. But the "thought of coming to Pittsburgh always speaks to me," he says.
He is here for a special occasion, but says the function is not the important element. It is the trip home that is, he says.
Jamal will perform a concert Sept. 27 and then be the featured guest the next evening at the MCG Jazz Legends Party, the annual fundraising gala. At that event, MCG Jazz will add members to its list of local jazz legends, those who have made Pittsburgh the heart of their musical lives.
Added this year will be singers Michele Bensen and Etta Cox, trombonist Al Dowe, and saxophonists Kenny Blake and Jim Guerra.
Jamal will appear with bassist Reginald Veal, drummer Herlin Riley and percussionist Manolo Badrena.
Jamal, 83, started touring right after graduating from Westinghouse High School, and began recording in 1951. As he shaped his personal style, he began to carve a niche in jazz built around an assertive style that can be as aggressive as it is restrained.
That work makes his playing immediately identifiable and has earned him such honors as being named a jazz master by the National Endowment for the Arts and being inducted into the French government's Order of Arts and Letters.
His work and appreciation has allowed him to reach the stage where he can control his performance schedule. He says he tends to pass on work in clubs, preferring concert hall settings.
"I won't say I never play clubs," he says. " 'Never' is a strong word."
His current album, "Saturday Morning," was released earlier this month and is made up of seven Jamal originals and three others. It is led by the 10-minute title cut, which is a look at that special time of the week.
"When I started playing it, the title just suggested what the song was about," he says. "Relaxing. Enjoying the day. Doing grocery shopping. All my compositions suggest to me what they are going to be called."
Bob Karlovits is a staff writer for Trib Total Media. He can be reached at firstname.lastname@example.com or 412-320-7852.


text:
LaMarcus Aldridge scored 22 points, Brandon Roy added 19 and the Portland Trail Blazers beat the Denver Nuggets 101-92 on Tuesday night.
Joel Przybilla had 19 rebounds and 10 points, going 4-of-4 on free throws in the final 2:40 to help Portland (18-11) tie the Nuggets atop the Northwest Division.
Aldridge put an exclamation point on the victory with a thunderous rebound and dunk with 20 seconds remaining in the second game of a home-and-home series.
Linas Kleiza had 20 points, including five 3-pointers, for the Nuggets, who lost three in a row before beating Portland 97-89 on Monday night in Denver. Nene had 17 points and 13 rebounds, and Chauncey Billups added 17 points on 4-of-12 shooting.
A sore elbow kept Denver's leading scorer, Carmelo Anthony, out for the second consecutive night.
Roy averaged more than 36 points during a five-game span before being held to eight points and 3-of-11 shooting Monday. His offensive woes continued through the first three quarters Tuesday night as he made just three of his first nine shots. But the All-Star had 12 points in the fourth quarter while Portland rallied.
Roy also finished with six assists. Greg Oden started for the Blazers but picked up five fouls in 10 minutes and didn't score.
The Blazers led by 12 in the second quarter but Denver whittled the lead after Portland had turnovers on three consecutive possessions. J.R. Smith hit a pull-up jumper for Denver to start a 7-0 run and the Nuggets trailed 47-42 going into halftime.
Personal fouls and bad passes cost the Blazers the lead in the third quarter. Nene stepped in front of Travis Outlaw's telegraphed inbounds pass and had an easy dunk on the other end to tie it at 58.
Kleiza hit back-to-back 3-pointers to give Denver a 64-63 edge, but the Blazers regained the lead 78-76 on an alley-oop from Rudy Fernandez to Roy with 8:11 to go in the fourth.
Denver and Portland play twice more this season – during the first week of March and the last week of the season.
Notes:@ Portland is 10-2 at home this season. ... Anthony is expected to miss Friday's game against Philadelphia, too.


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Fish list
Discussion in 'Beginner Discussions' started by Alias, 26 Apr 2011.
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