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I am 15 years old, turn 16 on friday. I was involved in a serious car accident in april 2009. I broke my cheek bones, broken jaw, broken nose, shattered eye socket and crushed back. I had facial surgery where i had metal plates put into my cheeks. Had lots of things put into my mouth to keep my jaw into place. Its been 3 months now since my surgery but the swelling and puffiness will not go down from my nose.
This is really affecting my life. I have tried lots of different things to get rid of the puffiness and swelling but nothing seems to work. I look into the mirror everyday and everyday i feel like the mirror is telling me something different. Can you please help me. I dont go out i stay in doors all day everyday. I havent been able to go to school and i am scared of going to college in september because of my nose.
Please help me get rid of puffiness and swelling to my nose.


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LAKELAND, FL (December 20, 2011) – Chabad Jewish Center invites members of the Polk County community at large to participate in the second annual public Menorah lighting in downtown Lakeland. The fun starts Tuesday at 5:00pm at Kryger Overlook Park, which is located at 108 S Massachusetts Ave, corner Main St in Lakeland.
Attendees will enjoy lively Chanukah music, chocolate Chanukah Gelt (coins), a Menorah kit and Dreidel to take home, and activities such as face painting, balloons, guess the Dreidels game, arts & crafts and a raffle. Hot, delicious homemade Latkes (potato pancakes) with applesauce and warm beverages will be served, compliments of Chabad. Hot dogs, Sufganiyot (Israeli-style jelly donuts), cotton candy and beverages will be available for sale.
At 6:00 p.m. the giant 9-foot Menorah will be kindled, overlooking beautiful Lake Mirror, followed by a few words from our honored guests, Lakeland Mayor Gow Fields and Polk County Sheriff Grady Judd.
This is a community-wide public event and admission is free.
For more information, sponsorship opportunities, or to volunteer to help at the event, please contact Chabad Jewish Center at (863) 510-5968.

subset: commoncrawl


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Nat Gas prices steady
The decline in Nat Gas futures prices yesterday were tempered a bit by the cold nearby temperatures, a major storm along the northeast and a short term forecast from NOAA that is more supportive for additional Nat Gas related heating demand than the forecasts from earlier in the week. That all said the spot Nat Gas futures prices is still solidly trading within the $3.20 to $3.50/mmbtu trading range that has been in play since early November of 2012.
Whether or not the current round of cold temperatures will strongly impact demand and thus prices it is still an uncertainty as to whether this is the beginning of a sustained cold spell or just another period that falls into the winter pattern experienced so far. The pattern has consisted of relatively short periods of colder than normal temperatures followed by periods of warmer than normal temperatures. Kind of a flip - flop winter pattern. So far this pattern has resulted in the overall winter heating season to date coming in colder than last year's very warm winter but still warmer than normal. The net result will likely be an end of the winter heating season inventory level about 15 to 20% above normal but below last year's record high end of season inventory level.
At the moment I still do not see the spot futures price breaking out of the existing trading range anytime soon. A few days ago I thought the short term momentum would have resulted in a test of the upper end of the trading range. After yesterday's bearish inventory report the probability of that happening has definitely decreased. So for the moment I would expect the market to remain range bound with the trading community focused primarily on short term trades around the parameters of the trading range. I do not expect any surge of long term trades entering the market.
Next week's inventory is likely to be more bullish than last year. Some of the early expectations I have seen are in a range of about 125 to 190 BCF net withdrawals. Last year for the same week the net withdrawal was 113 BCF while the five year average came in at 154 BCF. This report reflects a colder than normal period especially along the high demand east cost. As such I am leaning more toward a net withdrawal above the five year average. I will publish my estimate in Monday's newsletter.
In this week's EIA Weekly Nat Gas report residential and commercial natural gas consumption is almost exclusively used for heating, so colder weather brings increased consumption. Last year's record warm winter led to relatively low consumption in January and February 2012, the coldest months of the year. While this winter's heating season (November through March) began with relatively low natural gas use, particularly during the warm December 2012, consumption has increased with colder temperatures in 2013.
A return to cold temperatures drove prices back up in the Northeast. Prices at the Algonquin City gate trading location, which serves Boston, and the Transcontinental Pipeline's Zone 6 New York delivery point (Transco Zone 6 NY), which serves New York City, closed trading on Wednesday, January 30, at $7.42 per MMBtu and $3.99 per MMBtu, respectively. This represented a large decrease from their respective peaks last report week of $34.59 per MMBtu and $37.07 per MMBtu. However, the dip in prices at these two points proved to be temporary, as cold temperatures returned in the Northeast. By the close of trading on Friday, February 1, the Algonquin Citygate spot price was back above $30 per MMBtu and the Transco Zone 6 NY spot price was $13.28 (after reaching almost $14 per MMBtu at the close of trading the day prior). Although prices at these two market locations moderated somewhat by the end of the report week, they were well above their week-ago closing price, with Algonquin Citygate closing trading yesterday at $22.63 per MMBtu and Transco Zone 6 NY closing at $11.74 per MMBtu.
Looking at the individual or flat price situation for the two crude oils. The spot WTI contract has been in a short term downtrend since peaking at the end of January. That short term downtrend pattern currently remains in play. Brent has been in a medium term uptrend and is now hovering near a key technical resistance area of around $118/bbl. If this level is solidly breached the next stopping point is not until around the $121/bbl level.
On the refined product front the spot HO contract is still clearly in a medium term uptrend with the next level of resistance around the $3.26 to $3.27/gal level. From a fundamental perspective the nearby temperatures are below normal and the short term weather forecasts are more friendly than those from earlier in the week. The latest NOAA six to ten day and eight to fourteen day forecasts are both calling for a much larger area of the US expected to experience below normal temperatures for the February 12th through the 21th period. I believe that is what is currently helping to keep the HO sellers mostly on the sidelines. In addition the arb between Europe and the US East coast is not very supportive for movement of distillate cargoes in either direction.
RBOB gasoline lost some of its upside momentum as inventories have been building while demand has been relatively static. From a technical perspective the spot March RBOB contract seems to have peaked around the $3.07/gal level and looks poised to be entering into a short term downtrend. Thus for now the $3.07/gal remains resistance while technical support is not until around the $2.96/gal level.
At I discussed in detail this week the evolving geopolitical situation has certainly moved a bit closer to being in the forefront in the minds of the oil complex market participants and thus providing a layer of support for most of the commodities in the oil complex (WTI less so). In addition with relatively high correlations between oil, the direction of the global economy and the direction of equities any change in the directional pattern of the equity market will have a negative impact on oil irrespective of the oil fundamentals.
Equity markets have entered into a downside corrective pattern over the last week or so with the EMI Global Equity Index down by about 2% for the week so far and ahead of the last day of trading of the week in the US. If the downside correction continues to evolve it will limit any significant gains in oil prices from current levels and likely result in a push lower in oil prices.
Overall the macroeconomic data has been relatively supportive and suggestive that the global economy may have turned the corner and heading for a more accelerated pace of growth. In addition about two-thirds of corporate earnings beat to the upside... another sign that the economy is continuing to improve. Overnight the latest round of data out of China was supportive but could be a bit distorted... as it usually is... ahead of the major Lunar New Year holiday period coming up.
I am keeping my Nat Gas view and bias at neutral as the weather forecasts and nearby temperatures remain somewhat supportive. As I have been discussing for weeks the direction of Nat Gas prices are primarily dependent on the actual and forecasted weather pattern now that we are still in the heart of the winter heating season and currently those forecasts have turned a tad more supportive at the moment.
I am maintaining my view at neutral and keeping my bias at cautiously bullish even though the current fundamentals are still biased to the bearish side. However, the technicals and forward fundamentals are suggesting that the market could be setting up for a move to the upside now that the spot WTI contract has breached its upper resistance level.
Markets are mostly higher as shown in the following table.
Best regards,
Dominick A. Chirichella
Follow my intraday comments on Twitter @dacenergy
*Disclaimer: The information in the Market Commentaries was obtained from sources believed to be reliable, but we do not guarantee its accuracy. Neither the information nor any opinion expressed therein constitutes a solicitation of the purchase or sale of any futures or options contracts.
Copyright CME Group All rights reserved.


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The Miami Heat and the Indiana Pacers play their sixth series game in Miami tonight for the Eastern Conference Championship in the NBA. Miami is ahead 3 to 2 in the series. They can close it out tonight. But they could have closed it out Wednesday night after having Indiana down 10+ points much of the game until late in the fourth quarter. Indiana won 93 to 90. That's despite the fact that Miami's four-time MVP superstar LeBron James, without question the greatest player in the game nowadays, had to sit the bench much of the evening due to early foul trouble. He only played 24 minutes and had seven points, his worst post-season game in his career. Why?
Lance Stephenson, that's why! Stephenson was guarding James and harassing him all night. Talking trash and getting under his skin. I think he got fined $10,000 for a flop. But he was all over LeBron. The best was when they were standing side-by-side. It may have been on the foul line, with both players bent over with their hands on their knees, waiting for play to resume. The cameraman then zoomed in on them standing together on screen in such a close-up shot that all you could see on the screen was their head's and upper bodies. It was such a great camera shot for what was about to transpire. Then, standing on the left side of James, Stephenson turned his face to LeBron's face, looking at him very close up. LeBron was staring straight ahead, seemingly aware but not paying any attention to his menace. Then Stephenson blew air in LeBron's ear. I have been an NBA fan almost all my adult life, and I've never seen that before. LeBron stayed dead still with his body and head and looked far right, thus in the opposite direction, as if to say, "did yall see that!" Then he went back to looking straight ahead. Then Stephenson did it again. LeBron started barely shaking his head trying to keep from smiling. To me, you could take that as blowing a kiss without the hand movement.
I thought it was so so funny, I howled laughing. But Indiana Pacers President Larry Bird, one of the top five superstars to ever play the game, must not have cracked a smile. Yet Bird is no slouch himself when it comes to trash talking on the court and talking humor. Houston Rockets coach Kevin McKale was Bird's teammate with the Boston Celtics, and he always said Larry Bird was one of the funniest guys he's ever known. And Larry Bird and Michael Jordan could go at that face-to-face pretty good talking trash at each other. One of the funniest things I've seen in the NBA was when Bird and McKale won one of their NBA championships and in the celebration ceremony right afterwards, their coach Red Auerbach was giving a speech in which he said the accolades should only go to the players. Larry Bird was standing right next to him and he blurted out, "Then you're overpaid!"
In interviews after the game, Bird said of Lance Stephenson's blowing in LeBron James' ear that it was "crossing the line" of what's fair game. That's his own player he's talking about. And Superstar Miami Heat player Dwayne Wade said of the incident, "whatever you get away with is fair game." As for LeBron, he said, "I'm just here to play basketball." Ha! That made it more funny to me. LeBron is trying to keep his head in the game, and Lance is trying to get him off track any way he can, whether it's with body blows or ear blows. I don't know if Mr. Larry Bird is right or not, but I thought it was hilarious.
Pacers teammates say they never know what in the world Lance Stephenson is going to do next with his antics on the court. He's built a reputation with his shenanigans. I'm wondering what it's going to be tonight. Can LeBron James over power everyone like he usually does, charging across the court like a steaming locomotive for a bunch of slam-dunks? Or will Lance Stephenson get on the track and blow some more of his own hot air to derail the charging locomotive?


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2012 40 Under 40 Honoree: Katie Davis
Big Brothers Big Sisters of the Ozarks
Wednesday, May 16, 2012 11:05 AM
: 28
: Bachelor's in journalism and strategic communication and master's in public administration, University of Missouri-Columbia
: I met my husband, Brent, at the University of Missouri in Columbia. We moved to Springfield after graduation and have been married for five years. We are expecting our first child, a girl, in July.
Recent travels
: Brent and I took a "babymoon" in January with another couple who were also expecting to an all-inclusive resort in Playa del Carmen, Mexico. … My favorite part was sitting in the plush cabanas by the pool, playing games and reading some great books.
Just for Fun: If you were throwing a party and could invite any two people, who would be your must-have guests?
Oprah and Mother Theresa. Both have been inspiring to me in regard to how they have given back to the community and helped the poorest of the poor.
When Katie Davis took the reins of Big Brothers Big Sisters of the Ozarks in late 2010 at age 27, she became one of the youngest CEOs among more than 300 Big Brothers Big Sisters agencies nationwide.
"The board of directors recognized my talent and leadership two years ago as they began grooming me for the position of CEO by promoting me to the assistant director position," Davis says.
The regional agency, which supports mentoring relationships between adults and children, serves 20 southwest Missouri counties with satellite offices in Branson and Joplin. Last year, the organization supported 1,012 matches between adults and children, and Davis says the organization achieved record annual revenues of $937,191.
A major test of skill blew her way on May 22, 2011, when the Joplin satellite office was in the direct line of the tornado that struck the city. Davis had led the agency in a move to go paperless just months before the tornado hit, and as a result, the Springfield staff was able to access Joplin's files immediately, contacting families to check on their safety. Three days after the storm, she had volunteers and staff organized to secure and clean up what was left of the office, and within a week, they had a storage pod filled with what was salvageable, and all the families were accounted for and safe.
Another career milestone was achieved through successful implementation of the 30 Men in 30 Days campaign to help serve more than 70 boys who were on a waiting list for Big Brothers.
BBBS of the Ozarks has experienced 85 percent growth during the past five years, Davis notes, allowing more children to be served by an expanded staff. In 2010, when she was still assistant director, Davis began working on a capital campaign for a 1,180-square-foot addition to accommodate growth.
"For 18 months, I worked with in-kind donors, board members, foundations and individual donors to secure just more than $150,000," Davis says. "Oct. 31, 2011, was my proudest professional accomplishment to date because we were able to break ground on our building addition with the cost of the addition fully funded."
Since moving into leadership with BBBS, Davis has worked to combat employee turnover, which she says is a reality many nonprofits face.
She initiated a staff retreat and advocated for additional benefits, including retirement and growth incentive plans.
"The growth incentive plan allows the staff to keep track of monthly revenue and expenses, while being rewarded for staying under budget in expenses and over budget in revenue," Davis says.
Click here for full coverage of the 2012 40 Under 40.
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Undertaking a home remodeling project is a big decision. As a homeowner, you weigh the costs versus the benefits of a potentially long and messy process. You worry about disrupting your family's routines while construction occurs. Fortunately, you can limit home remodeling difficulties when your team includes a construction manager. Let's review why this is important:
What does a construction manager do?
As the leader of your project, he/she verifies that all the work required to complete the job is carried out correctly and efficiently. Construction managers see the project through from start to finish. They schedule, coordinate, and hire the necessary workers for the job and make sure those workers have the appropriate skills and attitude. Your manager uses a variety of tools to confirm that your project runs smoothly, and, very importantly, stays on budget.
What happens after we hire a home remodel team?
First and foremost, a good construction manager will spend plenty of time with you to understand exactly what you want and can afford, resulting in an agreed-upon scope of work. Then, he/she will prepare the necessary documents, including plans and product specifications for new installations. The manager will make sure that any city or county construction permits required are obtained. Once the planning process is complete, remodeling can begin.
What tools does the construction manager use to track and manage my home remodeling project?
Some put together a construction manual that includes the contract, any change orders, project timeline, purchase orders and a detailed scope of work. This client-specific notebook helps the construction manager and his/her team to better assist and inform you throughout the project. The notebook typically includes all drawings and daily job tracking. It often includes budget spreadsheets so you are kept informed of ongoing costs.
But in this computer age, the tools of the trade for many construction managers utilize construction management software. Some are program-based; others are web-based or even use cloud technology. Some allow the construction team anywhere, anytime access from mobile platforms such as iPhones, iPads and Androids. Most include features such as project scheduling, project/construction management, customer and service management, project estimators, and budget tracking.
When you're ready for a home remodel, make sure your team includes a construction manager with the right tools of the trade to get your job done on time and on budget.


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Amazed at Arkansas Smart Core Diploma Requirements
Although Arkansas does not issue accredited diplomas to homeschooled students, I took a look at what is required to graduate from an Arkansas public school in the year 2010. Noticeably missing from the required 22 credits was a foreign language! Instead of requiring 6 career credits perhaps they should require a language. Just about every college I can think of wants to see at least two years of the same foreign language. Good thing homeschoolers have more options and flexibility than the state of Arkansas. This is one reason why we homeschool.


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Hotels in Grover Beach, California
Search & Compare Grover Beach Hotels
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Best hotels in Grover Beach
Grover Beach overviewGrover Beach is a relaxing place distinct for its beaches and oceanfront, and known for its spas and shops. You're sure to appreciate the ample beer and coffee options. Learn about local heritage at popular landmarks in the area like Mission San Luis Obispo de Tolosa. Be sure stop by Oceano Dunes State Vehicular Recreation Area and Pismo Beach Golf Course while you're here. A family-friendly area, Grover Beach also has something for everyone.
Where to stay in Grover BeachWhether you're planning to stay for a night or for the week, Grover Beach has accommodations to fit every need. There are 123 hotels in the Grover Beach area and you can shop as many as 26 deals for rooms on Hotels.com, some with discounts of up to 30%.
- • 54 2-star hotels with an average price of $53 per night
- • 50 3-star hotels with an average price of $70 per night
- • 18 4-star hotels with an average price of $105 per night
Here are the top hotels based on 35301 user reviews of Grover Beach hotels:
- • Holiday Inn Express Grover Beach
- • Seaview Inn
- • Pacific Inn
- • American Inn Grover Beach
How to get to Grover BeachFly in to San Luis Obispo, CA (SBP-San Luis Obispo County Regional), 8.3 mi (13.3 km) from downtown Grover Beach. Santa Maria, CA (SMX-Santa Maria Public) is the next closest major airport, 18 mi (29 km) away. Grover Beach Station is the closest train station.
What to see around Grover BeachFun activities include attractions such as:
- • Pacific Dunes Ranch Riding Stables
- • The Luffa Farm
- • Rancho Guadalupe Dunes Preserve
- • Reservoir Canyon Trail
- • Monarch Butterfly Grove
- • Central Coast Aquarium
- • Mustang Waterpark
- • San Luis Obispo Botanical Garden
- • Oceano Dunes
- • Pismo Lake Ecological Reserve
- • Pismo State Beach
- • North Beach Campground
- • Heritage House Museum and Gardens
- • Pismo Beach Golf Course
- • Pismo Beach Prime Outlets
- • Oceana Casino
- • The Great American Melodrama & Vaudeville
- • Pismo Beach Pier


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Alzheimer's disease (AD) is a progressive, currently irreversible brain disorder. People with AD gradually suffer memory loss and a decline in thinking abilities, as well as major personality changes. These losses in cognitive function are accompanied by pathologic changes in the brain, including the buildup of insoluble protein deposits called amyloid plaques and the development of neurofibrillary tangles, which are abnormal collections of twisted protein threads found inside nerve cells. Such changes result in death of brain cells and breakdown of the connections between them. AD advances gradually but inexorably, from early, mild forgetfulness to a severe loss of mental function called dementia. Eventually, people with AD become dependent on others for every aspect of their care. The risk of developing AD increases exponentially with age, but it is not a part of normal aging.
AD is the most common cause of dementia among people age 65 and older and is a major public health issue for the United States because of its enormous impact on individuals, families, the health care system, and society as a whole. Scientists estimate that as many as 4 million people currently suffer with the disease, and annual costs associated with AD are estimated to exceed $100 billion.5,6 As the population ages, the numbers of people with AD and costs associated with increased prevalence could rise significantly.
The following section on Alzheimer's disease and related neuroscience describes recent advances in seven areas of AD research: early diagnosis, normal age-related cognitive change, the role of environmental factors in the development of AD, new animal models that may provide insights into the etiology of AD, preclinical studies of new preventive and therapeutic agents, clinical trials to test new therapies that may delay or prevent development of the disease, and studies related to easing caregivers' burdens.
Early Diagnosis of AD
Early diagnosis of AD benefits affected individuals and their families, clinicians, and researchers. For patients and their families, a definitive early diagnosis provides an opportunity to plan and to pursue options for treatment and care while the patient can still take an active role in decisionmaking. For clinicians, accurate early diagnosis facilitates the selection of appropriate treatments, particularly as new interventions are developed to stop or slow progression of symptoms. And for researchers, earlier and more accurate diagnosis may simplify clinical studies of new therapies and preventive measures by allowing early intervention, when cognitive loss is less severe and, consequently, the response is more easily measured. Research suggests that the earliest AD pathology may begin to develop in the brain 10 to 20 years before clinical symptoms yield a diagnosis. Scientists have made tremendous progress looking for ways to diagnose AD in its pre-symptomatic or pre-clinical stages, including reliable, valid, and easily attainable biological markers that can identify cases very early in the course of disease when treatment may be more effective. Eventually, combinations of specific strategies to image the brain, along with genetic, clinical, and neuropsychological assessments may become the key to identifying people at very high risk of developing AD.
Identification of Risk Factor Genes for Late-Onset Alzheimer's Disease. Until 2001, just four of the approximately 30,000 genes in the human genome were conclusively known to affect the development of AD pathology. Three of these genes are associated with early onset AD, and only one is associated with the more common form of the disease, late-onset AD. Recent genetic studies suggest that as many as four additional and as yet unidentified genes may also be risk factors for late-onset AD. Finding new risk factor genes will help identify pathways affecting the development or progression of AD and may eventually lead to better predictors of the disease even before it is clinically apparent.
Imaging Small Regions of the Brain in Humans and Genetically Modified Mice. Functional imaging, or the visualization of processes within the body in "real time," is potentially a useful tool for detecting changes in the brain that may suggest early AD, or for identifying markers that may indicate the extent of the disease. However, barriers to its optimal use remain. Traditional functional magnetic resonance imaging (fMRI), a common method for visualizing brain structures, allows imaging of structures a few millimeters in size, but no smaller. This resolution is insufficient for evaluating smaller structures, such as subregions of the hippocampus that are important to learning and memory. In addition, it requires that the person being imaged respond to specific instructions, an impossibility for many with cognitive impairments. A "new" method of fMRI has been developed that permits evaluation of these minute areas of the brain. In addition, this method, which is dependent on oxygen use in the brain during rest, does not require the person being studied to perform a mental task, making it easier to use among cognitively-impaired people. Although fMRI is currently available only in a research setting, these techniques could eventually be used to identify persons with loss of neurons in very specific brain regions—for example, in identification of persons at risk for developing AD.
Loss of Neurons in a Particular Brain Region is Associated with Onset of Cognitive Decline in Older Individuals. Participants in a recent study had detailed clinical evaluations within 12 months of death and were categorized as having no cognitive impairment, mild cognitive impairment (MCI—often a precursor of AD), or mild to moderate AD. At autopsy, people with MCI all had significant losses of neurons in the entorhinal cortex (EC) of the brain relative to those with no cognitive impairment, and these losses were as extensive as those in the patients with full-blown AD. In a second study, autopsies were done on people whose cognitive status had been assessed shortly before their death. This study found extensive loss of neurons in the EC in people with very mild AD. In contrast, those with no loss of neurons in the EC, but with plaques and tangles in their brains (the hallmarks of AD), showed no cognitive decline. These findings indicate that elderly people with MCI and very mild AD already have dramatic decreases in the number of neurons in a particular region of the EC, and that it is the onset of neuronal loss, rather than the development of plaques and tangles alone, that is associated with the onset of AD-related memory loss. This suggests that the development of interventions that will prevent, delay, or slow the degeneration of these critical neurons may be extremely beneficial to people at risk of AD.
Normal Age-Related Cognitive Change
While most people remain alert and mentally able as they age, some age-related changes in memory, learning, and attention are normal. Improved characterization of normal cognitive function and underlying brain changes throughout life will help us distinguish normal from abnormal age-related cognitive changes. A better understanding of what is "normal" and what is not may aid the early diagnosis of AD; it could also alleviate the anxiety of people who observe modest but perceptible changes in cognitive function in themselves or a loved one and fear that such changes are the harbingers of a decline into dementia.
Some People with Mild Cognitive Impairment (MCI) Progress to Alzheimer's Disease and Some Don't: How To Tell. Researchers evaluated people with MCI and, based on clinical findings, categorized their MCI as representing probable AD-related dementia, "incipient" AD-related dementia, or "uncertain" AD-related dementia. These volunteers were reassessed annually for up to 9.5 years, and at that time all the volunteers whose clinical findings had indicated probable AD-related dementia had developed the clinical symptoms of AD. However, many in the less severe groups had not. In another study, investigators categorized people as having normal cognition or having MCI or probable AD, both at entry into the study and at a subsequent clinical evaluation 2-4 years later. Each participant had an MRI scan at baseline and at follow up. The size of the hippocampus decreased in all groups, most rapidly in AD patients, less rapidly in those with MCI, and least in the control group. Within the control and MCI groups, those who experienced decline in cognitive function over time had a significantly greater decrease in hippocampal size than those who remained clinically stable. Previous studies have shown that baseline hippocampal volume can provide predictive information about which patients with MCI will decline to AD versus which will remain stable; together with this baseline information, serial measurements of hippocampal size through non-invasive MRIs may be a useful tool in the future for identifying people with MCI who will and won't progress to AD.
Early Life/Environmental Factors and AD
Environment May Protect Against Cognitive Decline and Alzheimer's Disease. Investigators hypothesized that recreational activities would be an excellent measure of mental activity, as these are less strongly influenced by economic and social factors than other risk factors for AD, such as the number of years of formal education. They recorded the extent to which 500 AD patients and age-matched healthy people had participated in recreational activities over their adult life. Patients with AD were found to have been much less active than healthy people of similar background in terms of both diversity and intensity of recreational activities engaged in during early and middle adulthood. These differences were not related to differing educational or income levels, age, or gender. People who were relatively inactive in midlife had a two and a half fold increased risk of developing AD. In a separate study, the relationship of social ties and support to patterns of cognitive aging over a 7.5 year period was examined in 1200 high functioning, community-dwelling adults aged 70-79. The results showed that greater baseline emotional support was a significant predictor of better-maintained cognitive function at the 7.5-year follow-up, controlling for known socio-demographic, behavioral, psychological, and health status predictors of cognitive aging.
Animal Models of Neurodegenerative Disease
Animal models that mimic human disease are central to research for many reasons. Animals and humans share many genetic and physiologic features, so experimental results obtained in animals can frequently (although not always) be extrapolated to humans. It is much easier to create specific genetic mutations and observe their effects in animals than to search for them in humans, and because the lifespan of most animals is relatively short, it is easier to observe the effects of those mutations over several generations. Recently, scientists have created several new models for research on neurodegenerative diseases, including AD.
The TAPP Mouse: The First Link Between Plaque and Tangle Formation. The neurofibrillary tangles (NFTs) that characterize AD are composed primarily of a form of the protein called tau. In addition to NFTs, another key feature of AD is the deposition of beta-amyloid (Aß) in insoluble amyloid plaques outside brain cells. The fragment is formed by clipping it out of the much larger amyloid precursor protein (APP). Although many scientists believe that excess production of Aß is a root cause of AD, it is still unclear how this causes pathology. In particular, scientists do not understand whether excess production of Aß leads to development of NFTs.
Now, a new mouse model may help us answer this question. A number of transgenic mouse models of AD have been developed by inserting human mutated APP genes into mice. Amyloid plaques, but not NFTs, form in these mice. A model for pathology of the tau gene has also been developed, but the NFTs in these mice do not usually form in areas of the brain that are vulnerable to AD. Scientists recently crossbred the tau mutant mice with the APP mutant mice to produce a new model, the TAPP mouse. The TAPP mice produce amyloid plaques; they also produce NFTs in regions of the brain that are vulnerable to AD, suggesting that APP or Aß peptide can influence the regional formation of NFTs. This is the first animal model in which the elusive connection between amyloid pathology and tangle formation can be investigated.
This improved animal model for AD may also be critical for success in developing therapies against NFT formation and the death of neurons in AD brain.
Pesticide Creates a Rat Model of Parkinson's Disease. Parkinson's disease is a progressive neurodegenerative disorder characterized by selective death of neurons that make the neurotransmitter dopamine in a region of the brain called the substantia nigra. In an effort to develop a new model of Parkinson's disease, scientists exposed rats to rotenone, a common pesticide. Exposed rats showed pathological changes characteristic of Parkinson's disease, as well as motor behavior abnormalities, such as rigidity and decreased motor activity, that are frequently seen in Parkinson's disease patients. This new model of Parkinson's disease will be useful in designing and testing new therapeutic interventions, as well as further identifying environmental exposures that may be risk factors for developing the disease.
Preclinical Research
There are currently no effective, generally useful treatments for Alzheimer's disease; i.e., a treatment that works on large numbers of patients, that maintains its effectiveness for a long period, that works in both early and late stages of the disease, that improves functioning of patients in activities of daily living as well as on sensitive neuropsychological measurements, and that has no serious side effects. In addition, none of the treatments presently approved for AD alter the progressive underlying pathology of the disease. One way to treat the disease successfully may be to interfere with early pathological changes in the brain, including the development of amyloid deposits and the formation of neurofibrillary tangles. A number of promising approaches, many of them targeted at the reduction of amyloid plaques, are currently being developed and tested in various model systems. If these approaches prove safe and effective in animals, studies in humans could follow.
Promising Immune Treatment for AD. Using mice that were genetically engineered to produce Aß and develop AD-like pathology in the brain, investigators found that treatment with an antibody that recognizes Aß peptide results in clearance of Aß plaques from the brain. Based on these findings, the investigators hypothesize that treatment with antibodies may be a useful and important approach for the treatment and prevention of AD and other neurodegenerative diseases.
Phenserine Regulates Translation of $-Amyloid Peptide Message: A New Target for Alzheimer's Disease Drug Development. AD's tell-tale amyloid $-peptide (Ab) plaques are formed when a larger protein called amyloid precursor protein (APP) breaks down. Researchers are working to develop agents that reduce APP expression. In a recent study, investigators conducted laboratory tests of a drug called phenserine, originally developed to increase levels of the chemical messenger acetylcholine, which is depleted in the brains of people with AD. They discovered that phenserine inhibits APP formation in cells through a mechanism independent of acetylcholine. Current research is directed towards the design, synthesis, and development of agents that optimally and safely regulate APP and Aß levels with the aim of slowing or halting the molecular events that lead to AD.
Statins May Reduce the Risk of AD. Evidence increasingly suggests that high levels of cholesterol may have a role in the development of AD. Two recent studies found that the use of statins, the most common type of cholesterol-lowering drugs, may lower the risk of developing AD. In a third study, transgenic mice fed a high cholesterol diet had much higher levels of blood cholesterol and the mean number of amyloid deposits in their brains (a hallmark of AD) was 65 percent higher than those on the normal diet. Taken together, these results suggest that statins—or dietary interventions—may be effective treatments or preventives for AD.
Imaging Clearance of Plaques by Immunotherapy in Living Mice. Researchers have developed a powerful new imaging technique, multiphoton microscopy, that enables them to view changes in the brain caused by AD and subsequent changes induced by treatment. Multiphoton microscopy provides a resolving power 100 times greater than that of other noninvasive imaging techniques, and allows sufficient resolution to view very small structures and lesions in the brain such as plaques. In a recent experiment, antibodies specific for amyloid and labeled with a fluorescent dye were placed directly onto the surface of brains of anesthetized mice who had developed AD-like plaques. Using the new imaging technique, scientists noted reversal of existing amyloid-$ deposits in the brain within 3 days of treatment with the antibodies. These findings demonstrate the potential effectiveness of antibody-mediated passive immunization for the removal of plaques from the brain.
BACE1 is the Major Beta-Secretase for Generation of Amyloid-beta Peptides in Mouse Brain. A major focus of study has been the process by which the amyloid precursor protein (APP) is clipped apart by enzymes to release Ab fragments, which are then deposited in the brain as AD's characteristic plaques. A recently discovered enzyme that helps clip Ab out of the APP protein was given the name b-secretase. In order to identify the enzyme that is responsible for production of Aß in the brain, scientists developed a mouse model in which the gene for the BACE1 enzyme, a candidate for the active b-secretase, was selectively eliminated to see whether removing it would interfere with the clipping of APP to produce amyloid. Indeed, Aß peptides were no longer produced in brain cell cultures made from the "knockout" mice, suggesting that BACE1 is responsible for the cleavage of APP into Aß in the mouse brain. These findings will help in design of drugs to inhibit b-secretase activity, in hope of slowing plaque production. Furthermore, because the mice in which the BACE1 gene has been eliminated seem to develop normally, it may be possible to develop BACE1 inhibitors that interfere with Aß deposition without negative effects on other metabolic pathways in brain or other tissues.
Clinical Trials
Today, the few FDA-approved drug treatments for AD maintain cognitive function in AD patients in only a subset of patients and for only a limited time. However, an estimated 50 to 60 compounds are presently or will soon be tested in human AD clinical trials. These studies are sponsored by a number of sources, including the NIA, other NIH institutes, and the private sector, primarily pharmaceutical companies. Compounds now under scrutiny focus on three major areas of treatment: short-term maintenance of cognitive function; slowing the progress of the disease, delaying AD's onset, or preventing the disease altogether; and managing behavioral problems associated with AD.
Interest is currently focusing on compounds that directly target disease-related pathologies. A rapidly evolving research focus lies in prevention trials, and a number are underway to test the effectiveness of therapies in people without symptoms or who have only slight memory problems. Recruitment is now complete for the first NIH AD prevention trial, which will take place at more than 70 sites across the U. S. This trial compares the effects of vitamin E and donepezil (brand name Aricept) in preventing the development of AD in people diagnosed with mild cognitive impairment, a population at high risk for developing AD. Further examination of estrogen and studies of various classes of anti-inflammatory drugs and antioxidants are also ongoing, and as scientists test these currently available medications, the next generation of drugs is being developed, targeting specific abnormal cellular pathways uncovered by recent discoveries, including plaque and tangle formation and death of brain cells. Prevention trials are among the most costly of research projects, but, if successful, the payoff in terms of reduced disease and disability will be significant.
Caregiving of AD Patients
Most of the approximately 4 million Americans with Alzheimer's today are cared for outside the institutional setting by an adult child or in-law, a spouse, another relative, or a friend. Caregivers frequently experience significant emotional stress, physical strain, and financial burdens, yet they often do not receive adequate support. Several recent studies have explored the problems faced by caregivers of AD patients, as well as possible interventions to reduce their burdens.
Depression and Agitation in Alzheimer's Disease: Effects on Caregivers. Previous research has examined the factors contributing to stress and depression in caregivers of an AD family member. A recent study found that the greater the level of depression in the patient, the greater the level of depression in the caregiver. Wives of AD patients and caregivers in poor health themselves were at particular risk for depression. This study demonstrates that the well-being of the caregiver and care recipient are closely related. The findings also support interventions for caregivers early in the family member's illness.
Women Caring for a Family Member with Dementia Can Benefit from an Exercise or Nutrition Program. A one-year study involved 100 women age 49 to 82 years who were sedentary, free of cardiovascular disease, and caring for a relative with dementia. Participants received either a home-based, telephone-supervised moderate-intensity exercise training or nutrition education program. Exercise consisted of brisk walking for four 30-to 40-minute sessions per week. Compared with the nutrition education group, caregivers who exercised showed significant improvements in physical activity levels, stress-induced blood pressure reactions, and sleep quality. The nutrition group reported significant reductions in percentages of total calories from fats compared to exercisers. Both groups reported significant improvements in psychological distress, including depressive symptoms and self-rated stress level.
Selected Future Research Directions in AD and the Neuroscience of Aging
National Cell Repository Expansion. To facilitate the identification of AD risk factor genes, NIA is planning the expansion of its National Cell Repository. A national resource for research on AD, the Repository was created to collect DNA, cells, and information from families with multiple affected individuals with AD. Its activities include the production of a catalog of cell lines and DNA samples that are available for qualified scientists to study. Because most researchers rely on extended families, sibling pairs, or case-control studies to look for genes, the goal of the expanded Repository will be to develop cell lines and collect data from persons identified through the Alzheimer's Centers who fall within one of the three categories. While previous research has emphasized the identification of genes associated with the familial form of the disease, this expansion will allow us to more rapidly identify the underlying genetic mechanisms of the more common late-onset form of AD.
Alzheimer's Disease Cooperative Study. NIA's major AD clinical trials effort is the Alzheimer's Disease Cooperative Study (ADCS), a consortium of 83 medical research centers in the U. S. and Canada. Established in 1991, the ADCS conducts clinical trials on compounds in which large pharmaceutical companies are generally not interested, including drugs that were patented and marketed for another use but might be useful for treatment of AD, or novel compounds from investigators with inadequate resources for clinical trials. During its first decade, the ADCS established a research infrastructure to carry out clinical trials for promising new therapies for AD, developed new and more reliable ways to evaluate patients enrolled in these and other studies, and initiated a number of clinical trials. The latest 5-year award will allow that work to continue and will move AD treatment research in new directions, including the study of a cholesterol-lowering statin drug, an antioxidant, and a high-dose vitamin regimen. Studies of interventions to combat behavioral and other manifestations of AD, including agitation and sleep disturbances, are also ongoing. In addition, the ADCS will develop and refine evaluation tools and methodologies for AD prevention research.
Extending Caregiving Research to Clinical Practice. NIA will expand the research scope and objectives of its Resources for Enhancing Alzheimer's Caregiver Health (REACH) program to extend promising caregiver interventions to the clinical setting. The REACH program is a large-scale, coordinated study to examine the effectiveness of social, behavioral, environmental and technological interventions for reducing caregiving burdens of caring for persons with dementia. In the first phase of the program (1995-2001), investigators at six sites tested a series of interventions. Now, in Phase II, the outcomes of these exploratory interventions will be analyzed and used to develop intervention strategies for further evaluation across multiple sites. Of particular importance is the program's emphasis on evaluating these interventions in diverse racial and ethnic populations.
- Small, G et al. Diagnosis and treatment of Alzheimer disease and related disorders. JAMA 16: 1363-1371, 1997.
- Ernst, RL, et al. Cognitive function and the costs of Alzheimer's disease, Arch Neurol 54: 687-693, 1997.


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Ecclestone says Bahrain race is on
By Alan Baldwin
SHANGHAI (Reuters) - Next week's Bahrain Grand Prix will go ahead unless local officials decide otherwise, Formula One supremo Bernie Ecclestone said on Thursday.
"The race is on the calendar, it's scheduled. The only people that can do anything about it is the national sporting authority in the country that can ask for it to be withdrawn from the calendar," he told Reuters at the Chinese Grand Prix.
"Unless it gets withdrawn by the national sporting authority in the country, then we'll be there."
Local organizers, who had to cancel last year's race at Sakhir due to civil unrest, have been adamant that this year's event is safe to go ahead despite continuing sectarian divisions and street violence in the kingdom as well as threats targeting the grand prix.
They have denounced "scaremongering" and "huge misconceptions" about the situation.
John Yates, the former assistant commissioner of the Metropolitan police who has been hired to oversee reform of Bahrain's police has also said he felt safer in the Gulf kingdom than he often did in London.
Ecclestone, 81, played down reports that he would be discussing the situation with teams and governing International Automobile Federation (FIA) in Shanghai amid pressure on the sport to cancel from rights activists. Continued...


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Church commended for immunization clinic
Posted by : February 16, 2013| On :
Monitor Staff Writer
KAUFMAN–Kaufman County Commissioners Court commended an effort started by a Sunday School class in Forney, with the help of several partners that has been successful in immunizing 3,250 children.
A dream of Inez Williams of the First Methodist Church in Forney took shape after partnering with The Caring for Children Foundation of Texas and Blue Cross Blue Shield.
She and the Seekers and Boosters Sunday School Classes at the church committed financing and set aside a place for a once-a-month shot clinic, Kaufman County, judge Bruce Wood said.
"Their efforts have resulted in over 3,250 children receiving more than 8,370 immunizations, resulting in saving families approximately $670,000.
"Therefore, I, Bruce Wood, Kaufman County Judge, commend the continued dedication of The Caring for Children Foundation of Texas, the Seekers class of First United Methodist Church of Forney, and others involved for their ongoing service to insure the children of Kaufman County are receiving the needed immunizations for a healthier life."
Lynda Shirley, Care Van Program manager, said the church's efforts to advertise the clinic on a bill board on Highway 80, and through other avenues has been very helpful.
"They also volunteer the night of the clinic, helping families fill out the paperwork and contribute financially to the program," she said.
Blue Cross and Blue Shield of Texas partners with the Caring For Children Foundation of Texas by paying all the administrative costs of their Care Van Program, which includes Shirley's salary, maintaining the van and supplying it with immunizations.
The clinic is held 5-7 p.m. the second Tuesday of the month at the church. "We'd like to see more children taking advantage of the program," Shirley said. But this depends on the school nurse referring families to the free clinic.
Children who are uninsured or underinsured are the ones to benefit from the program, she said.
The First United Methodist Church in Forney has been holding the clinic for nearly eight years now.
Blue Cross and Blue Shield of Texas spokeswoman Michelle Riddell expressed her appreciation for those committed to the community and its children.
"I am so impressed and moved by what they've been able to do and Blue Cross and Blue Shield of Texas is proud to be a partner in their vision," she said.
Pastor Chris Allen of the church referred to the Matthew 25:40. "Jesus says that when we do kind things for those in need we are serving him. First Methodist Forney has a tradition of reaching out in Christ's love. I can only say that I am very blessed to be serving a church whose outreaching love is so close to the heart of Jesus."
In other action, commissioners:
•approved interlocal agreements with cities in Precinct 4, including Mabank, and ratified a plan to split the cost of road material between the county and city to maintain CR 4006 in a twice yearly basis.
• approved the purchase of a Boom Mower for Precinct 3 for $32,862 and a used Caterpillar 307C Excavator for Precinct 1 for about $59,000.
• paid bills totaling $980,652.64.


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Originally Published by: Fox News Insider
A writer at Forbes Magazine has identified a phenomenon called the "death spiral states." Sounds intimidating, right? Fox Business Network's Melissa Francis broke down what you need to know on America Live today.
The category includes 11 states where private sector workers are outnumbered by people who are dependent on the government. That number would include state workers, and people who are receiving welfare or pension. For example, in California the ratio of "takers versus makers" is bigger than one. If you own a software company that employs 100 people, you're supporting 139 other people in the state who are on the "takers" list.
Credit worthiness was the second factor worked into this equation. To calculate that, Forbes looked into large debts, uncompetitive business climate, weak home prices and unemployment trends for each state.
So what's the takeaway? If you live in one of the states on the list, there are things you can do such as rent instead of buy a home.
New Mexico ranked at the bottom of the list, with 153 takers for 100 workers. For the full "death spiral" list, check out the clip below!
To Continue Reading and to view the video, please go to: http://foxnewsinsider.com/2012/11/28/are-you-living-in-a-death-spiral-state-and-if-so-what-should-you-do-about-it/
[amazon_enhanced asin="0470182393″ /]


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Greater Noida, Feb 18 (PTI) Two officers of the YamunaExpressway Industrial Development Authority were allegedlykidnapped by farmers near here this evening.
Deputy Chief Executive Officer R K Singh and DeputyCollector S P Singh had gone to Dankore area for negotiationswith farmers over compensation for land when they werekidnapped, police said.
The District Magistrate, SSP and other senior officialswere present at the site of negotiations, where the farmersgot provoked and became violent, the police said.
Police had to resort to cane charge in order to controlthe agitated farmers.
In the melee, farmers kidnapped the two officers and tookthem to an unknown destination, police said.
A police team was searching for the officials till latenight but was not able to trace the kidnapped officers. PTICORR


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Bike Parking III
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- Width43 cm
- Length138 cm
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Bike Parking III is a necessary small architecture piece, that should appear in populous spaces. The product belongs to the Outdoor Furniture line, where we offer many other interesting elements for exteriors and urban environment.


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Mayor Rob Ford paid tribute to Doug Holyday, "as solid as they come," by bestowing a ceremonial Key to the City upon the veteran municipal politician, who has moved to Queen's Park.
"As my deputy mayor, Doug stood beside me through thick and thin. Very, very few people would do that," Mr. Ford said Tuesday in an emotional ceremony at City Hall.
Mr. Holyday also alluded to the controversies and allegations that have gripped the mayor's tenure, noting: "Rob, it certainly has not been dull."
A former mayor of Etobicoke and city councillor since amalgamation, Mr. Holyday won the provincial by-election for Etobicoke-Lakeshore earlier this month, giving the Progressive Conservatives a sought-after toehold in Toronto. The mayor has picked Scarborough Councillor Norm Kelly as his new deputy.
The crack-cocaine saga also continued to dog Mr. Ford Tuesday; although the topic did not arise directly after Mr. Holyday's key ceremony, journalists gathered later by the mayor's office to ask about a member of Mr. Ford's entourage, following reports that police are investigating attempts by his associates to obtain a video allegedly showing the mayor smoking from a crack pipe. Mr. Ford, who has previously denied the crack allegations, said simply: "It's done. Have a good night."
Mr. Holyday's key ceremony was attended by nine city councillors, bureaucrats, Mr. Holyday's son, Stephen, daughter-in-law, Margaret, and grandchildren, Andrea, Jennifer and Thomas. Mr. Ford, who called Mr. Holyday an inspiration and mentor, touted the new MPP's "level-headed approach" and "voice of reason."
"I can't thank you enough for your friendship, your guidance and support for my 13 years in politics and before that as a friend of the family," Mr. Ford said.
A humbled Mr. Holyday, in turn, credited the work of city staff, in particular during tense labour negotiations.
To councillors, he said: "We didn't always see eye to eye, as you know, but I never really had a bad word with anyone here that ever lasted." He also recalled early-morning phone calls by reporters inquiring about something that had happened overnight regarding the mayor — sometimes before Mr. Holyday had even heard about it.
"It's been exciting and I wish you all the best as you go forward," he said to Mr. Ford. "I think you'll do well because you're doing the right thing for taxpayers and the citizens of Toronto, and I think that's the most important thing. That's what people care about."
National Post


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The Morning Sun - Saturday, January 01, 2011
Ruth Ann Mannaioni Wingebach
ARMA — Ruth Ann Wingebach age 87 of Arma died at 11:15 AM on December 30 at Girard Medical Center after a short illness.
Ruth was born October 29, 1923 in Breezy Hill, Kansas. She was the daughter of William and Armida Ponsi Mannaioni.
She has lived in the same home for 83 years. She graduated from Arma High School in 1941.
She married Bernard C. Wingebach on February 4, 1947 in Our Lady of Lourdes Catholic Church in Pittsburg, KS, who preceded her in death in 1993.
She was a homemaker and also worked as a clerk for Woolsworth in Pittsburg, KS, and then later in life as a clerk for Crowell's Drug Store, Pittsburg, KS.
She was a member of St. Joseph Catholic Church in Arma, and served as treasurer for St. Ann's Altar Society from 1996-present, she was also a 61-year member of the American Legion Auxiliary Post 182 and served as President of the American Legion Auxiliary from 1998-2000. She was also a member of the Eagles Auxiliary, and a member of 8140 Auxiliary in Pittsburg.
Survivors include one daughter Janice Roller (Husband Tony) of Cape Coral, FL, one son Bernie Wingebach (wife Judy) of Arma, four grandchildren, one great-granddaughter and one step granddaughter, with 4 step great grandchildren.
She is preceded in death by her parents, one sister Edith Felan, and five brothers Morris Mannaioni, Richard Mannaioni, Thomas Mannaioni, James Mannaioni, and Lebro Mannaioni.
Funeral Mass will be Monday, January 3 at 9:30 AM at St. Joseph Catholic Church in Arma, with Father Lawrence Carney as celebrant.
Burial will be in the Pittsburg Garden of Memories.
The family will have a rosary at 6:30 PM on Sunday, January 2 with visitation following from 7-8 PM at the Bedene Funeral Home.
Friends may visit her at the Bedene Funeral Home any time after 10 AM on January 2.
Ruth requested donations be given to the St. Ann's Altar Society or the American Legion Auxiliary Post 182, and donations also may be left at the Bedene Funeral home, 517 E. Washington, Box 621, Arma, Kansas, 66712.
Wingebach, Ruth Ann Mannaioni 63571325
b. Oct. 29, 1923 d. Dec. 30, 2010
Garden of Memories Cemete...
Crawford County
Kansas, USA
Sherry Kaye Richmond Prettyman
JOPLIN, Mo. — Sherry Kaye Prettyman went home to be with The Lord December 30, at 6:55 A.M. after a long illness.
Kaye was born August 17, 1937 in Hannon, Missouri to Emmett and Sadie (Talbott) Richmond.
She was the youngest of eight siblings. Kaye lost both her parents when she was twelve years old. She was raised from age twelve in Liberal, MO by her sister and brother-in-law, Roma and Dale Charlton.
She married Clemith Prettyman on August 31, 1957, in Miami, OK. They had two children, Tamara Lynn and Kenny Dean. They moved to Joplin in 1961.
In 1970 Kaye completed her bachelor's of science degree in education at Missouri Southern State College. She then went on to complete her master's of science degree at Pittsburg State University. She taught elementary school in Joplin, at Washington Elementary and Irving Elementary, for twenty-seven years until retiring in 1997.
Kaye was a longtime member of Southside Bible Baptist Church in Joplin where she helped teach Bible school and keep the church records.
Kaye loved scrapbooking and playing slot machines. She was an avid collector of children's dishes and Hallmark Christmas ornaments. For many years she enjoyed having coffee with friends each morning at McDonald's.
Kaye was a loving mother and grandmother, always putting family first. Her home was the family headquarters for holiday and family gatherings.
Kaye is survived by her husband, Clem Prettyman, of Joplin, daughter Tammy Meadows and son-in-law Dale of Joplin, son Ken Prettyman of Gardner, KS, grandchildren Sarah Brown and husband Barry, of Neosho, Chelsea Prettyman of Joplin, Ethen Prettyman, of Olathe, KS, Kathy Desourdy, of Neosho, Shawn Meadows, of Joplin, Whitney Barker, of Olathe, KS, great-grandchildren David Desourdy and Nina Brown, niece Beverly Long and husband Ron, of Liberal, MO, sisters Helen Marlow and husband Howard, of Kansas City, MO, Alice Overstreet and husband Charles, of Carthage, MO, brother Lloyd "Buck" Richmond and wife Vernice of Sheridan, Wyoming, and many nieces, nephews, and loving friends.
Funeral services will be at 1 P.M. Tuesday, January 4, 2011 at the Southside Baptist Church at 2302 Grand Street, Joplin, Missouri with the Rev. Freddie Wright officiating. Ken Prettyman will give the eulogy.
Burial will be at the Rosebank Cemetery at Mulberry, Kansas.
The family will receive friends from 6 to 7:30 P.M. Monday at the Bedene Funeral Home at Arma, Kansas.
The family suggests memorials to the Southside Baptist Church, 2302 Grand Street, Joplin, Missouri. Donations may be sent to the church or Bedene Funeral Home, 517 E. Washington, Box 621, Arma, Kansas 66712.
Prettyman, Sherry Kaye Richmond 63571015
b. Aug. 17, 1937 d. Dec. 30, 2010
Rosebank Cemetery
Crawford County
Kansas, USA
Lois May Thurman Maddox
GIRARD — Lois May Maddox, 85 of Girard, KS passed away December 30, 2010 at the Girard Medical Center following a long illness.
She was born November 16, 1925 in Kansas City, KS the daughter of Roy Albert and Edith Dale (LaMunyon) Thurman.
On June 3, 1950 she was united in marriage to Jake Maddox, Jr. in Iola, KS; he preceded her in death on February 5, 1994.
Lois was a homemaker and also worked a short time for an airplane manufacturing company in Wichita. Church membership was held in the Christian Church, she was also a member of AARP.
Survivors include a daughter Linda Hudson of Girard, three brothers Fern Thurman of Arkansas City, KS, Louis Ray Thurman of Lenexa, KS, Jim Thurman of Wichita, KS, two grandchildren Larry Hudson, Jr. of Australia and Dee Dee Hudson of Basehor, KS along with three great grandchildren Jake, Tristan and Kaylyn.
She was preceded in death by her parents, husband, six brothers Everett, Earl, Floyd, Lloyd, Melvin and Jim Thurman, three sisters Helen Marie(infant), Darlene Wheeler and Thelma Rhodes.
Graveside services will be 11 am, Tuesday, January 4, 2011 at the Pleasant View Cemetery with Rev. Dennis Johnson officiating.
Family will receive friends at the Bath-Naylor Funeral Home in Weir beginning at 10 am Tuesday until the procession leaves for the cemetery.
Condolences may be left at www.bathnaylor.com.
Memorials may be made to Southern Care Hospice and/or The American Cancer Society; these may be left at or mailed to the funeral home. Services by Bath-Naylor Funeral Home 104 W. Main, Weir, KS 66781
Maddox, Lois May Thurman 63579373
b. 1925 d. Dec. 30, 2010
Maddox, Jake, Jr 26904200
b. Oct. 7, 1921 d. Feb. 5, 1994
New Pleasantview Cemetery
Cherokee County
Kansas, USA
Thurman, Roy Albert 58441673
b. Jul. 29, 1891 d. Jan. 9, 1950
Thurman, Edith Dale 58441665
b. Dec. 20, 1894 d. May 2, 1993
Latham Cemetery
Butler County
Kansas, USA


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Do you enjoy making rhythm based videos? Well VidRhythm for iPhone makes it so easy that even a baby could make a song. VidRhythm uses sounds you create via vocal or by whatever object you can tap or play notes on. There are a set amount of genre styles to choose from and a set of themes to go along with the song. Depending on the song you choose will determine what action happens next. You may be asked to sing a note, a word, tap on something or make the sound of a drum.
When this process is complete VidRhythm will take all of the samples and mix them into one song. To give you an idea of how a song will end up I've included a video below that I created as an example. So if your looking for the next app to waste countless hours with check out VidRhythm on the App store.


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Sony is demonstrating two slim TVs that incorporate OLED (organic light-emitting diode) technology at CES 2007.
The 11- and 27-inch TVs are about 3mm and less than 10mm thin, respectively, (when the thinnest part of the body is measured). The 27-inch model features a full high-definition (HD) panel while the 11-inch TV is designed with a wide-SVGA panel.
- Wireless broadband developments in Southeast Asia markets
As of 2013, the 10 ASEAN nations had a total of over 700 million mobile subscriptions, with the CAGR from 2003-2013 reaching 24%. This Digitimes Research Special Report analyzes the various mobile broadband markets in ASEAN and looks at the respective trends in 4G LTE development for those markets.
- 2015 global tablet demand forecast
This Digitimes Research Special Report provides a 2015 forecast for the global tablet market and analyzes the strategies of key market players such as Google, Apple, Intel, and Microsoft.
- 2015 China smartphone panel trend forecast
This Digitimes Research Special Report analyzes the strategies of key China-based major panel makers BOE, Tianma and IVO for attacking the different market segments through technology and pricing, and their relationship to local vendors Huawei, Lenovo, ZTE, Xiaomi and Coolpad.


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Answer: Each person will be issued a "user code" to access their particular print job that is sent to a network copier. The print job will not print until the user code is entered.
How do we copy incidentals, such as:
Progress reports
Parent newsletter
Parent notes
Re-teaching material
Intervention material
DCA analysis sheet
Bulletin board materials
Concept board materials
Alabama History workbook materials (maps)
Substitute material
Kronos reports
AR forms
Weekly Reader skills
EQT study guides
Study guides for all subjects
ARMT/SAT materials
Fluency practice
Answer: All of the above material will be carefully looked at by Xerox and the end user
to determine whether it should be printed at the school level or Print Shop
3. I have to print patron labels, barcode labels and
Accelerated Reader book level labels. Will I be able to print labels?
Answer: Yes
4. Are inkjet printers going to be removed from the classrooms and office areas?
Answer: Inkjet printers will remain in their current location.
However, the Document Solution Contract/Xerox will not be fully implemented until
after January, 2011, at which time specific printing will be discussed with all
end users to determine the best method of printing.
5. Will the Central Office fund the maintenance/support for ink Jet printers?
Answer: After the Document Solution Contract has been fully implemented,
the Central Office will no longer provide funding for the maintenance/support
for ink jet printers at both the Central Office and Schools. The decision is
based on how costly it is to print on an ink jet printer as opposed to printing
on a laserJet or Copier.
6. There are times when I use my inkjet printer to print posters for classroom
use. I splice together many sheets to creat large posters. Will this be handled
with the Xerox contract?
Answer: Yes. The print job that is described above can be submitted to the
Central Print Shop.
7. Will laserjet printers be allowed to stay connected to the network in the classrooms?
Answer: As of now, all print devices will remain connected. Once the program is fully implemented, it
will be determined at that time which print device remains connected to the network.
Per the contract, the school system will purchase paper based on a formula determined by the number of copies, copiers, printers, etc.


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This property has been removed by the agent. It may be let or temporarily removed from the market
Property Description
Letting information:
|Not Specified
|Added on Rightmove:
|22 February 2012 (1948 days ago)
Full description
SHORT LET. A superb one bedroomed apartment, which offers contemporary accommodation including an open-plan reception room/kitchen in a convenient location close to Wood Green Underground Station (Piccadilly Line) and Alexandra Palace Station (National Rail). The property is presented in excellent condition and also comprises a bedroom and a smart bathroom. Crompton Court is situated off Mayes Road, which is well served by nearby amenities around Wood Green, while Crouch End, Alexandra Park and Muswell Hill are within easy reach.
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|Sat 13 Feb 2016, 8:00pm–1:00am
We are very excited to have New Zealand's very own Elvis impersonator, George Brooking, coming to the Tikokino Country Hotel on Saturday the 13th of February.
His show starts at 8 pm and he'll be singing all the great Elvis songs we know and love so don't miss this opportunity to enjoy a great evening of entertainment.
George is a man of many talents and will also DJ the night away after the show playing all of your favourite songs.
Don't forget to mark this night in your diary or on your calendar so you don't miss out!
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Bradenton Beach, FL, USA < change city >
Bands (29)
Coordinators (23)
Caterers (22)
Florists (22)
Wedding Day Beauty (19)
DJs (17)
Cakes (16)
Officiants (14)
Limos (12)
Decorations (9)
Invitations (9)
Rentals (9)
Videographers (8)
Wedding Fashion (5)
Ceremony Musicians (4)
Ceremony Sites (92)
Reception Sites (71)
See All Vendors >
THE BEACH HOUSE RESTAURANT < this is my business >
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|Ceremony Sites, Restaurants, Reception Sites, Attractions/Entertainment
200 Gulf Dr N
Bradenton Beach, FL 34217
1 rating
9 descriptions
25 weddings
Add Your Review >
Kevin & Karen's Wedding
"Dinner with Shar & Family"
Anna Maria Wedding In June
"Beach Bar Option!"
Sarasota Wedding In November
"7 p.m. Nov. 4"
Longboat Key Wedding In June
"This is a Jacob's and HT Family favorite. If you are not opposed to great food, good service and tropical drinks with that extra shot on top...while being served at a table directly on the beach, then this place is worth the drive!"
Jeff and Lynn's Wedding
"The BeachHouse restaurant is located in Bradenton Beach at the west end of Cortez Road. It is the perfect place for family dining with plenty of room for all the kids to play on the beach while you enjoy a tropical drink and great food. Enjoy your meal inside the dining room or on the waterfront deck. There is even covered Gulf front dining as well, which makes it perfect rain or shine."
Siesta Key Wedding In July
"Mom and Dad's favorite vacation resturant."
Kelly and Andrew 's Wedding
"Tuesday night dinner"
Patricia and Yusuke's Wedding
"We did our ceremony on the beach right behind the restaurant and held the cocktail hour and reception in an outdoor pavillion at the restaurant. Nicole and Shawn at the BeachHouse did an excellent job, from answering our millions of questions about the venue to arranging for a vegan option on the menu. We got lots of compliments on the food from our guests; I only wish I'd had more time to enjoy my meal--the potatoes and portabello mushroom were so flavorful! The restaurant clearly has plenty of experience doing weddings for out-of-towners, which made for stress-free planning for us."
Carolyn and Edward's Wedding
"Dinner Thursday night at 7PM."
Bradenton Wedding In April
Venice Wedding In April
Anna Maria Island Wedding In July
Sarasota Wedding In November
Nikki and Billy's Wedding map
Tammy and Steve's Wedding
Anna Maria Island Wedding In April
Anna Maria Island Wedding In April
Anna Maria Island Wedding In January
Melissa and Stephen's Wedding
Longboat Key Wedding In July
Longboat Key Wedding In March
Anna Maria Wedding In December
Amanda and Jason's Wedding
Amanda and Jason's Wedding
Lido Beach Wedding In November
Bao and Lan's Wedding
Kristin and Vinny's Wedding
The Beach House Restaurant
200 Gulf Dr N
Bradenton Beach, FL 34217


text:
The Mod Dress
Kate Spade Necklace
Jewelry counts, too. These resin-and-metal beads are literally blocks of color.
To buy: $102, piperlime.com.
Forever 21 Dress
A little neon goes a long way. Case in point, this mod, 60s-inspired polyester shift.
To buy: $28, forever21.com for info.
Aldo Wedges
Get inches without the blisters, thanks to these stable suede platform wedges.
To buy: $90, aldoshoes.com.


text:
Step aside, Barack Obama.
After Friday, the most powerful man in Washington, D.C., isn't the President of the United States. Instead, it's the newly-minted defensive tackle of the Washington Redskins.
That's the practical fallout of the decision to give Albert Haynesworth a seven-year, $100 million contract with $41 million guaranteed.
As one league source observed, "He runs the coach, he runs the team, he runs everything."
Why? Because, like a top-five draft pick whose obscene contract insulates him as a practical matter from any real consequence for acting like a fool, Haynesworth can do and say whatever he wants in 2009 (and likely beyond) without consequence.
Apart from the team's limited options when it comes to taking action against Haynesworth, the Redskins now have their own T.O. — a guy whom ownership will choose to enable rather than discipline since any antisocial behavior in which he engages, and for which he is punished by the team, will prompt a chorus of "I told you so."
And that's where it gets interesting. As one source with intimate knowledge of Albert's ways observed, "You don't know what Albert is gonna do and you can't tell him what to do."
Hey, Terrell, do you have any extra popcorn?
Step aside, Barack Obama.
AliBabba says: Mar 2, 2009 8:13 AM
Florio, WTH is this. This is hardly news or rumor. This is just some ridiculous conjecture. What has Haynesworth done in his tenure in DC or the NFL for that matter to warrant TO comparisons. Furthermore, why don't you just call the deal the 4yr $48M deal it is and not the agent fluffing $100M deal it's not.
oHIoTitan says: Mar 2, 2009 8:31 AM
Enjoy ur headache 'Skins!! He might make u 3 plays a game, but will give u more headaches than u realize. Over/under on fights with teammates in camp- 3/1
brianforster says: Mar 2, 2009 8:38 AM
This signing benefits every single player on our defense.
Our ends should see sack totals increased due to collapsed pocket.
Our cornerbacks should get more picks due to increased pass rush.
Our linebackers should be freed up to make more plays on the ball.
Remind me again, who is gonna fight the man that is going to make them all look better?
Pro Football America says: Mar 2, 2009 8:41 AM
You're better than this, Florio.
smiley says: Mar 2, 2009 8:45 AM
First of all….this isn't Dallas. This is D.C. and Big Al WILL act accordingly, $100M or not. Greg Blache won't put up with any player trying to run his defense. Hail!
truetruth says: Mar 2, 2009 8:49 AM
Please……..Redskin fans are dreaming. This just goes to show the powers that be in DC have no clue how to build a franchise……I feel sorry for all the Redskin fans out there………
This signing has doomed the 'Skins to a few years of mediocre performance.
The signing by iteslf isn't bad, for specific teams. The Redskins NOT being one of them. Redskins have many holes to fill and this is just another example of bad allocation of funds……sad!
shaunypoo says: Mar 2, 2009 8:51 AM
caveat emptor
apeck says: Mar 2, 2009 8:56 AM
This is stupid. Stick to reporting facts.
We_Miss_You_Dan says: Mar 2, 2009 9:01 AM
@AliBabba: You're absolutely right! It is an insult (to TO) to compare him to TO because obviously TO can control his anger. I have never seen TO stomp a man's face with cleats, but I have seen Haynesworthless. And even better: He had the nerve to argue with his head coach about leaving the field! I would have loved to have been a fly buzzing around that conversation!
Fisher: Get off the effin field moron!
Worthless: But I din't do nuffin! He axed if my mama was available. So I represented.
What a loser. I wonder who he's gunna curb stomp in DC?
TheDPR says: Mar 2, 2009 9:06 AM
And that's where it gets interesting. As one source with intimate knowledge of Albert's ways observed, "You don't know what Albert is gonna do and you can't tell him what to do."
The 'Skins apologists commenting are cracking me up.
We DO know one thing about Albert. Whatever it is he decides to do he's going to be doing it at at least 90mph and probably on the beltway.
truncalep says: Mar 2, 2009 9:07 AM
Your all negative all the time Flufio
truncalep says: Mar 2, 2009 9:08 AM
All the jealous voices, its hilarious!
josereyes0 says: Mar 2, 2009 9:10 AM
Money ≠ power. Most importantly what the hell were the Redskins thinking signing a player to $41MM in guaranteed money who has never eve played a 16 game season?
DeeDubb says: Mar 2, 2009 9:27 AM
I'm sorry I thought this was a FACT driven site rather than one of OPINION. What is the point of this ridiculous editorial piece, seriously? Florio, I've heard you in interviews and read enough of your stuff to believe that you are more professional and rationale than this. If you have a hard-on for ripping Dan Snyder and the Skins, so be it but try not to let effect your work.
As somebody else said, you're better than this Florio.
brandon2443 says: Mar 2, 2009 9:30 AM
Hey he stomped a cowboy why not bring him to D.C.?
shaunypoo says: Mar 2, 2009 9:40 AM
"I'm sorry I thought this was a FACT driven site rather than one of OPINION."
It's a blog, he can say whatever he wants because this is not a news outlet, it is a blog. Just because he includes facts in most of his posts, doesn't make it a legitimate news outlet, it is a blog. In case you missed it, it is a blog. It is for entertainment purposed only.
bizurk says: Mar 2, 2009 9:52 AM
so, ummmmm, what's the story?
The internet equivalent of writing on bathroom walls.
racer-x says: Mar 2, 2009 9:55 AM
If snyder wasn't foaming at the mouth to 'reward' albert for that incident, his agent should have been playing that card. snyder hates the cowboys more than winning games.
TO is a poor analogy. Leaving all the behavior comparisons aside, Dallas insulated itself with a contract that was easily voided after any year he misbehaved. They were never on the hook with a cost prohibitive contract. TO also delivered every year. TO also played every game. I doubt Haynesworth will be able to make any of those claims, but he won't have to.
smiley says: Mar 2, 2009 10:19 AM
I love all the hatin'!! We still signed the best DT in the league. Hail!!
Vox Veritas says: Mar 2, 2009 10:27 AM
"What has Haynesworth done in his tenure in DC or the NFL for that matter to warrant TO comparisons."
Well let's see… he skull-stomped a guy on national TV. Then when he got flagged for that he went after the refs and his coach had to get between them. He also kicked Justin Hartwig in the face. And he went after Will Ofenheusle with a pipe. Of course Hartwig and Ofenheusle were his teammates at the time. The only things that have kept Haynesworth out of prison are the local Tennessee hillbilly cops and courts that sat by and watched him commit assault with a deadly weapon and did nothing, while pretending that the lack of a formal complaint from the victim precludes their ability to make an arrest and put him on trial. You're right, there's no comparison.
"This signing benefits every single player on our defense. "
Sure, until he attacks one of them. And assuming that he doesn't go on cruise control. He probably will, because not only is he mentally challenged, he's physically lazy and injury-prone.
"First of all….this isn't Dallas."
You got that right. It's the worst team in the NFCE… the worst in spite of Snyder's open wallet strategy of going after every player with a recognizable name to the tune of $600 million plus worth of contracts since he bought the team. It's the Deadskins, who haven't put together back-to-back winning seasons since Gus Frerotte was a snot-nosed kid in Tulsa, Oklahoma. Holy shit, the Cowboys have won three Super Bowls since the last time the Deadskins had back to back winning seasons!
baltiblingbling says: Mar 2, 2009 10:34 AM
The worst part about this is I have to drive on the beltway which will increase in accidents by 50% due to fat albert weaving in and out of trafic at 130 mph trying to get to practice. And smiley…you may have signed the best DT in the league but having 1 great player doesn't often equate to success especially when you overpaid. Hail!!..my ballz
Tuck You says: Mar 2, 2009 10:38 AM
I keep hearing that this guy will make the players around him better. OK, well the rest of their D-Line is trash so who freakin cares?
NYG= Best D-Line in the NFL. Period.
MTechnik says: Mar 2, 2009 10:57 AM
Awww, wow, listen to the whining from Dallas fans. A team that can't drop TO because of the cap hit, and haven't won a playoff game in over 10 years. Sorry your owner is too poor after his stadium to fix up his coaching staff. And on top of that, getting into a pissing match with a Dallas legend, Dan Reeves.
You Dallas boys are sad that Gurode's head will get bashed in twice a year.
Go troll elsewhere.
4G63 says: Mar 2, 2009 11:15 AM
You Skins fans need to get this shirt ASAP:
Vox Veritas says: Mar 2, 2009 11:18 AM
"You Dallas boys are sad that Gurode's head will get bashed in twice a year."
Too bad Haynesworth can only do it after a whistle. I guess you missed Gurode serving up a tall stack of pancakes to Haynesworth play after play after play last time they met. But yeah after the whistle, Haynesworth is a real terror to anybody's that's laying on the ground and defenseless. I hope he's got strong knees because I don't think he's gonna like it when 360 pound Bigg Davis dives at them while Gurode is kicking his overrated ass up high.
AliBabba says: Mar 2, 2009 11:20 AM
To back up that this is only a 4 year $48M deal, from Peter King:
The breakdown of the cap numbers for this deal:
2009: 6.7 million
2010: 25.3 million (uncapped year)
2011: 5.7-6.7 million
2012: 7.9 million
$7-8M in cap space for the possible capped years doesn't represent much risk considering the upside a player like Haynesworth brings. Think what you want but the Skins put little besides Snyder's wallet on the line for this deal…. its a very good move
Vox Veritas says: Mar 2, 2009 11:41 AM
"Think what you want but the Skins put little besides Snyder's wallet on the line for this deal…. its a very good move"
Sure, if he plays up to a contract that pays $12 million per year, with almost all of it guaranteed to be paid whether he actually shows up or not. And Dan obviously ain't made of money, he was laying workers off a couple of weeks ago.
smiley says: Mar 2, 2009 12:27 PM
Haaaa…we love when y'all hate us! Keep hatin'! Now sing with me…Hail to the Redskins! C'mon, you know the words, you've heard it enough.
Mangy66 says: Mar 2, 2009 1:15 PM
Wow!!! There's a lot of Skins haters out there. Just when I thought I'd seen all of them…more creep out of the woodwork.
Heck…if we could win some Championships, we'd be the Lakers of the NFL. We've got a ton of you haters.
At least Skins fans know (too bad the championships were so long ago) what it's like to be a Lakers/Yankees fan.
Kind of a Regal feeling…being put on a pedestal (no matter the kind of pedestal) by you haters, as we stare down on you fools (even if it's just of the offseason). Oh…and I love it when an Eagles fan is one of the haters. What has your team done? At least I can give a Cowboys fan an inkling of respect because they have a more recent Lombardi Trophy than we do.
We'll take the beating when the team sucks…Ahhh…the trials and tribulations of a REAL fan (and not a HATER).
baltiblingbling says: Mar 2, 2009 1:39 PM
Wow!!! Mangy66 has got to be white trash….HATER HATER HATERS omg SHUT UP!!!! Danie boy had to make this signing because the once great(yea i'll admit it) franchise that was the redskins is sliding into a shit hole. He has destroyed a prolific NFL franchise. No one cares about them anymore they are an afterthough in their own devision. NY won the superbowl a year ago the eagles made it to the league championship game this year and sorry dallas will always be the most talked about team in that devision…what have the redskins done? NOTHING, NO ONE IS HATING THEY ARE JUST SAYING YOU ARE AN AVG TEAM who overpaid for a player who will never in his life play 16 games in a year!!!! Why do you think you are the yankees of football your not even close. No one cares about you! danny had to make this signing to get his face on ESPN and make sure he continues to fill that crap heap of a stadium up.
AlwaysSonny says: Mar 2, 2009 1:45 PM
Bwahahah…dude just compared the skins to the yankees and the lakers…excuse me….BWAHAHAHAHAHHA…Tearing down that statement is too easy…moving on, there is no such thing as a Redskin hater. To win, is to have haters. Y'all don't win shit. Ever. But go ahead and keep talking about those ancient times when you did, in fact, have a winning team. As if success in the 1980's translates into haters in 2009. Ah, the month of March, best time to be a skins fan…that or the 80's, whichever.
Mangy66 says: Mar 2, 2009 2:49 PM
Baltimoron: White trash…eh? That's what people who live in a suburb of DC like to think. Keep it "Bling Blingin" in Bama-More. You just summed yourself up in the first sentence.
Sonny: Learn to read. Learn to comprehend. Everything you tried to "tear down" my argument with…I already mentioned in the "argument". You really are THAT DUMB…aren't you? You go stand in the dumb kids line with the Baltimoron. No soup for you!!!
ten-a-key tyrants says: Mar 2, 2009 8:18 PM
This article is lame but I will say this, if Albert Haynesworth plays like he says he will for the skins…they can win the NFC East. If Campbell develops, Portis peaks at the right time, and you throw in a suffocating defense…look out. Haynesworth with the skins is interesting. That is an insane amount of money for an NFL player, let alone a DT.


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What's your current daily driver + pics
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Originally Posted by
does it happen to have the 3.5L motor?
me and my gf have a slightly older one with the 3.5 and at light cruise and heavy acceleration it pings very very bad.
ive tried everything going from cheap gas to mid/high grade , new plugs and wires , checked all vacum lines, no error codes/check engine light.
hell i even tried to seafoam it once, it got better but that only lasted about 3 days
Yes it has the 3.5 G-code High Output motor. If you have no vacuum leaks (have you checked your brake booster? mine leaks a little and I can hear it whistle on the pedal side inside the passenger compartment - it's on my to-do list but haven't gotten to it yet!) and your ignition is in good working order, the fact that the seafoam helped a little is a good indication you have a severe carbon problem. I would recommend taking it to a shop that carries BG products, and have them do the BG Induction Cleaning Service with the K2902 service kit. The kit comes with two cans of carbon buster, that they put in a pressure vessel and blow through your intake while the engine is running .. cleans the throttle body, back sides of the intake valves, and the combustion chambers. The kit also includes a can off 44K fuel system cleaner that gets dumped in the tank to continue the cleaning while you drive. We charge $139.95 for the service, and in my experience it is worth every penny.
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Many have heard about Niall Horan's crush on Katy Perry. There have been tweets, photos, and many flirting encounters between the two. Tonight at the 2013 NRJ Music Awards, as Katy Perry got up out of her seat to accept an award, she made sure to hug Niall Horan and let out a fake little cry.
Niall's reaction to Katy Perry mentioning 1D in her speech aww pic.twitter.com/BUXs89ovJQ
— Saisha Tang (@Saisha_Tang) December 14, 2013
What's even more priceless is the One Direction boys' reactions to the hug. Perry even mentioned the One Direction boys in her acceptance speech.
PHOTOS: 2013 NRJ Music Awards – Red Carpet Celebrity Arrivals
It's the 2013 NRJ Music Awards. Celebrities like Will.i.am, Katy Perry, and One Direction are in the house. Check out the red carpet pics.Click here to read more
For more One Direction News:
WATCH: One Direction Performance & Interview on Japan Music Station
One Direction took the stage in Japan to perform "Best Song Ever" in addition to a little interview with Music Station. Check out both videos here.Click here to read more
Barbara Palvin, Niall Horan's New Girlfriend? 5 Fast Facts You Need to Know
Barbara Palvin is rumored to be dating Niall Horan as the two have been spotted on dates. Check out all the info on the alleged couple and all about Palvin.Click here to read more