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Buchanan County wins Governor's Food Bowl Challenge
GRUNDY, Va. - The Buchanan County Chamber of Commerce has again been named the small chamber membership division winner in the 2013 Governor's Food Bowl Challenge, winning the statewide event in its division for the second year in a row.
The Buchanan chamber collected the equivalent in food and funds of almost 19,000 pounds of food to benefit area food pantries on its way to win the 2013 title. Additionally, the chamber was named the small membership division per capita winner, collecting an average 170 pounds of food per chamber member.
More than 100 chambers across the state and more than 60 state government agencies participated in the five-week competition that began on Memorial Day and ended on Independence Day. The 2013 competition raised the equivalent of more than 400,000 pounds of food to benefit the more than 1 million individuals served by Virginia food banks. Through the challenge, donations could be in the form of canned goods, or funds. Each dollar raised was valued as four pounds of food according to the statewide competition rules.
"We were thrilled to learn that we won the challenge for 2013," said Buchanan County Chamber Executive Director Mary Belcher. "We felt good about our 2013 effort as we tallied our collection totals and determined that at 18,995 pounds, we had exceeded the 17,694 pounds we collected in 2012 to win the statewide challenge. What makes it special is that while we received the statewide recognition as a chamber, we did so for a project which in the end benefits our friends and neighbors here at home."
As part of the 2013 competition, the chamber again held an intra-chamber challenge which saw businesses in the chamber recognized for the efforts of their employees in collecting food and funds to benefit the drive. Six R Realty was recognized as the Buchanan small membership winner, collecting the equivalent of 1,333.32 pounds of food; West River Conveyors was the medium membership winner, collecting the equivalent of 2,118.39 pounds; CONSOL Energy was the large membership winner, collecting the equivalent of 6,987.80 pounds. TruPoint Bank was recognized with the community partnership award, collecting the equivalent of 3,477.43 pounds.
The chamber office also hosted four food drives including those held at Wal Mart, Garden Day, Save A Lot and Food City, collecting the equivalent of 2,726.62 pounds to benefit the 2013 drive.
Additional Buchanan chamber businesses participating in the 2013 drive included Sykes Enterprises, the Virginia Mountaineer, Southwest Virginia Professional Insurance, Appalachian Family Care and the Appalachian School of Law.
"We appreciate the efforts of all those who took the time to collect food and funds for this year's governor's food bowl challenge," said Cathy St. Clair, Buchanan County Chamber of Commerce president. "The win speaks to the generosity of our local businesses and their employees and to this community."
All of the 2012 winners in the statewide competition were repeat Governor's Bowl champions. Back-to-back winners in addition to the Buchanan chamber were Greater Augusta in the medium membership division, collecting the equivalent of 115,529 pounds and Greater Richmond in the large membership division, collecting the equivalent of 17,296 pounds. The Charlottesville Regional Chamber earned the per capita award for the large chamber division, collecting 17 pounds per person.
State agency winners included the (small) Office of the Governor, 9.096 pounds; Virginia Board of Accountancy, 337 pounds per person; (medium) Department of Motor Vehicles, 28,560 pounds; Department of Environmental Quality, 21 pounds per person; and (large) Department of Corrections, 19,698 pounds, 1.71 pounds per person.
The competition is organized regionally. Food collected in Southwest Virginia was donated to Feeding America Southwest Virginia, which operates a food bank in Abingdon, supplying local food pantries, as well as a mobile food pantry distribution program in Buchanan County.
"Earlier this summer I renewed my challenge to Virginia's business community and state agencies to compete in good fun for a worthwhile cause," said Gov. Bob McDonnell in announcing the 2013 challenge winners. "We could not do it without our partners at each Virginia Chamber of Commerce, the Federation of Food Banks and our teams of state employees."
Representatives of the winning chambers will be invited to attend a special event in Richmond in October to accept their 2013 food bowl challenge awards.
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Chapter 4101:2-7 Combustion air
[Comment: When a reference is made within this rule to a federal statutory provision, an industry consensus standard, or any other technical publication, the specific date and title of the publication as well as the name and address of the promulgating agency are listed in rule 4101:2-15-01 of the Administrative Code. The application of the referenced standards shall be limited and as prescribed in section 102.5 of rule 4101:1-1-01 of the Administrative Code.]
701.1 Scope. Solid fuel-burning appliances shall be provided with combustion air in accordance with the appliance manufacturer's installation instructions. Oil-fired appliances shall be provided with combustion air in accordance with NFPA 31. The methods of providing combustion air in this chapter do not apply to fireplaces, fireplace stoves and direct-vent appliances. The requirements for combustion and dilution air for gas-fired appliances shall be in accordance with the "International Fuel Gas Code".
Replaces: 4101:2-7-01
R.C. 119.032 review dates: 11/01/2016
Promulgated Under: 119.03
Statutory Authority: 3781.10(A)
Rule Amplifies: 3781.10 , 3781.11, 3791.04
Prior Effective Dates: 7/1/07

subset: commoncrawl


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http://www.automobilemag.com/reviews/suv/0704_2008_porsche_cayenne/Read Sam Smith's review of the new Cayenne. Do you think this minor refresh is enough to boost sales back up for Porsche?
I don't think a face lift will help the Cayenne much. The problem with the car isn't so much the looks as it is the badge. I'm not saying because it's a Porsche it's a bad car--quite the opposite actually. It's a car that can't live up to the Porsche name. To quote the writer:"But once the novelty wears off, boredom sets in."A Porsche shouldn't be boring. A Porsche should be a car that you can take to the track and do laps around your buddies with a big grin on your face. You can't do that with an SUV, even if it is a Porsche, so ultimately it will be a disappointing car no matter how much you change the styling.
Thats the thing... its an SUV not a car. Porsche is raising the bar for all SUV manufacturers with this rig. its making an SUV more fun to drive. if you want a true Porsche drive and feeling then dont get the Cayenne.


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State of the Union Preview
Tonight is the State of The Union Address. The White House says it'll build on the progressive themes President Obama laid out at his inauguration.
No question - money's the main issue tonight - especially 85 Billion in automatic cuts - now, just weeks away.
Tonight, instead of focusing on how budget cuts would affect the Pentagon, we're told President Obama will focus on the middle class .
Cuts to head start, research and thousands of government jobs. "His emphasis on the need to continue to create jobs or to continue to have our manufacturing sector expand -- that work isn't done," said White House Press Secretary Jay Carney.
In the audience tonight, the parents of a slain Chicago teenager who had just marched in the inaugural parade. Underscoring the President's push for gun control. "I'm happy that the murderer of Hadiya Pendleton is now off the street," said Cleopatra Cowley, Hadiya's Mother.
The President is also expected to address immigration reform. A move that may have more impact outside Washington. "The goal is-- is-- is to move public opinion because that's what moves Congress," said Former Senior Advisor to President Obama David Axelrod.
Lawmakers and former lawmakers on both sides say his clear focus must be the budget. "If we don't get it under control fairly soon, and have Congress and the president lead us in this area, then we will end up with a nation which is insolvent," said Former Senator Judd Gregg, (R) New Hampshire.
Republicans are already criticizing what happens after tonight's speech. President Obama hits the road - three cities in three days - to push his agenda. "Mr. President: If you're truly worried about this issue, why aren't you working with the Congress we elected to prevent it?" said Senate Minority Leader, Senator Mitch McConnell (R-KY).
Tracie Potts, NBC News.
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I do understand that this effect is bound to occur due to the nature of perspective projection. Having a rectangular projection plane will create this distortion and after seeing a helpful image on the topic I at least have a basic grasp as to why this distortion occurs. I believe I even saw the same effect on a real photo when I was searching for more information about it. The thing is that I don't have the slightest clue on how to lessen this distortion.
This is the code which renders my scene:
const int imageWidth = 600; const int imageHeight = 600; float aspectR = imageWidth/(float)imageHeight; float fieldOfView = 60.0f; // Field of view is 120 degrees, need half of it. Image testImage("image1.ppm", imageWidth, imageHeight); int main() { /* building scene here for now... */ std::cout << "Rendering scene..." << std::endl; renderScene(sceneObjects, 5); std::cout << "Scene rendered" << std::endl; return 0; } //Render the scene void renderScene(Object* objects[], int nrOfObjects) { //Create light and set light properties Light light1(glm::vec3(5.0f, 10.0f, 10.0f)); light1.setAmbient(glm::vec3(0.2f)); light1.setDiffuse(glm::vec3(1.0f)); //Create a ray with an origin and direction. Origin will act as the //CoP (Center of Projection) Ray r(glm::vec3(0.0f, 0.0f, 0.0f), glm::vec3(0.0f)); //Will hold ray (x,y)-direction in world space float dirX, dirY; //Will hold the intensity of reflected ambient and diffuse light glm::vec3 ambient, diffuse; //Loop through each pixel... for(int y=0; y<imageHeight; y++) { for(int x=0; x<imageWidth; x++) { //Normalized pixel coordinates, remapped to range between [-1, 1]. //Formula for dirY differs because we want to swap the y-axis so //that positive y-values of coordinates are on the upper half of //the image plane with respect to the center of the plane. dirX = (2.0f*(x+0.5f)/imageWidth)-1.0f; dirY = 1.0f-(2.0f*(y+0.5f)/imageHeight); //Account for aspect ratio and field of view dirX = dirX*aspectR*glm::tan(glm::radians(fieldOfView));; dirY = dirY*glm::tan(glm::radians(fieldOfView)); //Set the ray direction in world space. We can calculate the distance //from the camera to the image plane by using the FoV and half height //of the image plane, tan(fov/2) = a/d => d = a/tan(fov/2) //r.setDirection(glm::vec3(dirX, dirY, -1.0f)-r.origin()); r.setDirection(glm::vec3(dirX, dirY, -1.0f/glm::tan(glm::radians(fieldOfView)))-r.origin()); //Will hold object with closest intersection Object* currentObject = NULL; //Will hold solution of ray-object intersection float closestHit = std::numeric_limits<float>::infinity(), newHit = std::numeric_limits<float>::infinity(); //For each object... for(int i=0; i<nrOfObjects; i++) { //If ray intersect object... if(objects[i]->intersection(r, newHit)) { //If intersection is closer then previous intersection if(newHit<closestHit) { //Update closest intersection and corresponding object closestHit = newHit; currentObject = objects[i]; } } } //If an object has been intersected... if(currentObject != NULL) { //Get intersection point glm::vec3 intersectionPoint = r.origin()+closestHit*r.direction(); //Get light direction and normal glm::vec3 lightDirection = glm::normalize(light1.position()-intersectionPoint); glm::vec3 normal = glm::normalize(currentObject->normal(intersectionPoint)); //Factor affecting reflected diffuse light float LdotN = glm::clamp(glm::dot(lightDirection, normal), 0.0f, 1.0f); //Get diffuse and ambient color of object ambient = currentObject->diffuse()*light1.ambient(); diffuse = currentObject->diffuse()*LdotN*light1.diffuse(); //Final color value of pixel glm::vec3 RGB = ambient+diffuse; //Make sure color values are clamped between 0-255 to avoid artifacts RGB = glm::clamp(RGB*255.0f, 0.0f, 255.0f); //Set color value to pixel testImage.setPixel(x, y, RGB); } else { //No intersection, set black color to pixel. testImage.setPixel(x, y, glm::vec3(0.0f)); } } } //Write out the image testImage.writeImage(); }
I do want to mention that I have been playing around a bit with the parameters involved in my code. I tried putting the position of the ray/camera (CoP) further away from the image plane. As expected the scene zooms in as the field of view is supposed to become smaller. This did obviously not solve anything so I increased the field of view parameter (to neutralize the zoomed-in effect). Doing so removed the distortion (see second image) but this was just a temporal solution because I moved the blue sphere further out from the center of the scene which resulted in it being stretched again with the new settings (see third image).
This is the settings used for the three images:
Image 1: float fieldOfView = 60.0f Ray r(glm::vec3(0.0f, 0.0f, 0.0f), glm::vec3(0.0f)); Image 2: float fieldOfView = 85.0f Ray r(glm::vec3(0.0f, 0.0f, 20.0f), glm::vec3(0.0f)); Image 3: float fieldOfView = 85.0f Ray r(glm::vec3(0.0f, 0.0f, 20.0f), glm::vec3(0.0f));
Again I am totally lost in how to solve this problem. I don't even understand why the new settings I tried removed the distortion (temporarily). I realize some distortion will happen regardless but this seems extreme, furthermore the fact that I had to increase my fov to such a high value above gives me a bad feeling and makes me suspect my code might be at fault somewhere. Any help and explanation will be appreciated


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After dozens of fresh windows installations last years, I reconed installing Windows 8 would be a walk in the park, but without an available USB stick, 2 failing external USB DVD-drives for the ISO I burned the installation isn't as smooth as expected. It didn't appear to me that i could simply use the internet installer supplied by microsoft. Downloading will take about half an hour on your average broadband connection, why did I bother downloading and burning the ISO, thats so 2011..
I downloaded the small installation file and about an hour later Windows 8 was installed and good to go. The first surprise is the boot time. The old Windows 7 Pro install took over a minute to boot, and now it boots within 20 seconds! I'm sure I'll scrape a few seconds off if I disable boot from USB/CD-rom in the bios.
Too bad my 3G modem isn't detected and installed automatically, it seems I have to do some driver-searching to sort this out.
Check here if you want a step-by-step guide to installing Windows 8 on your netbook from a USB memory stick.
Like with my Windows 7 i immediately started to tweak my Windows 8 installation to speed up my netbook. This post describes the steps I took to finetune my configuration.
The next time i'll be spending on getting used to the new start menu, and finding drivers for my beloved 3G card. Keep u posted!


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JV girls split 2 games
The Pequot Lakes junior varsity girls' basketball team split a pair of games last week – beating Perham 55-16 on Thursday, Jan. 3, and losing to Esko 49-41 on Saturday, Jan. 5.
The Patriots grabbed a 19-0 lead against the Yellowjackets, and led 32-11 at the half. Caitie Ryan led Pequot Lakes with 15 points while Kelsey Hart added 13, Laura Squires 11, Emily Buell seven, Alex Nelson six, Caitlyn Carkhuff two and Jessica Faacks one.
On Saturday, the Patriots fell behind Esko 23-11 at intermission. Pequot Lakes rallied in the second half to tie it 39-39 on a baskets by Hart and Ryan.
Ryan led the Patriots with 20 points while Morgan Lohmiller had nine, Hart six, Barb Miller four and Faacks two.


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It's been incredibly cold here for a couple of weeks now (it was 4 degrees this morning). This afternoon I went out to look at the hive and, peering through the mouse guard, was shocked to see two icicles hanging off the bottom-front of two of the frames (right near the entrance)! They were small, one about as big around as a pencil, the other even thinner.
I can still hear buzzing coming from inside the hive, and I see dead bees periodically, so they're still alive. Is this anything to worry about? Maybe increase ventilation?


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Cozy Private Room #2
Maximální počet hostů2
Počet místností1
Floor No.
- Vhodné pro rodiny s dětmi
- Internet/WiFi
- Klimatizace
- Parkování k dispozici
- Uzamykatelný pokoj
- Balkón/terasa
- Ručníky
Accommodation summary
|Rijecka, 23000 Zadar, Chorvatsko
|Typ nemovitosti
|Soukromý pokoj
|Ident. objektu
|Acceptance rate
|Response time
|A few hours
|Calendar updated
|More than half a year ago
There is a full size bed, a big closet, nightstands with a reading lamp, and free WI-FI access throughout the house. Towel is always provided and a 42" tower fan with ionizer is provided in the room.
There are 3 rooms in the house. My apt is located on the first floor. You'll have access to all the common areas including the kitchenette that consist of microwave and fridge, and bathroom that is shared with two other rooms. Absolutely no smoking inside the house but you may smoke at the patio.
The house is located in the heart of Zadar. You may find a lot of authentic sea food, Mediterranean, and Italian restaurants, bakeries etc. Pharmacies, Coffee shops, Supermarkets, are all located in walking distances. The old town is located roughly 20 mins by walk and nearest beach 15 mins by walk.
The house is close to the major bus stops. Bus takes you to old town, any beach or ferry port if you wish to visit any of the islands.
Parking space is provided and absolutely free.
**** The listing is only for the room NOT the whole apartment.
*****We do not accept reservations from guests with blank profiles and no photos of themselves. We appreciate your candor! :)
Domovní řád
|Cena za více než 2 ubytované(ých)
|16 € Cena za osobu/noc navíc
|Příjezd po
|Check-in before
|Odjezd před
|Stornovací podmínky
Sladana's place is very nice and clean. It's not too far away from the old town (about a fifteen to twenty minute walk) and it's very calm and peaceful out there, away from the tourist buzz. The bed is comfortable and everything was very clean. We can definitely recommend staying here.
Sladana is an amazing host and the apartament was perfect, me and my friend enjoy a very nice 5 days and if had time we d liked to stay more, the room was clean and nice with a cute balcony and the apricot of ever :) we could rich the center of Zadar in just 15 minutes walking, a really nice city :) so if you want a nice place i suggest you to go
The room was clean and comfortable. It is not far from the old city (20min by walking). It is like the description given by Sladana.
Ohodnoďte tomuto hostiteli i další ubytování
I had a wonderful time at Sladana's apartment. She is a very hospitable and polite person and gave me a lot of good tips for my time in Zadar. The apartment was very cozy and clean. Thank you Sladana!
We had a very nice stay at Sladanas Apartment. The rooms are very nice furnished and really clean. That made our stay in Zadar even better. We enjoyed the quiet balcony and had a lovely time. Thank you for having us!
Sladana is very gentle and hospitable person who introduced us to her hometown with all of details and worthy tips. We had a wonderful time at her place, calm and quite, surrounded by fruit trees in the garden. Hope to be able to visit Sladana place and Zadar soon. Thank you, Sladana, for these lovely 2 days!
A very nice place to stay in the center of zadar! Comfy and spacious, with airconditioning for the hot summerdays. Sladana is a very sweet host and gave us great tips for our stay. Thanks a lot!
Very good service from Saldana who had been amazing with us from day 1. She explained us what to visit in zadar and even gave us a map.
The house is very trendy and clean, we were amazed by the house.
The only thing is it was not possible to cook but apart from that, everything was great.
Sladana welcomed us friendly in her living room and proposed us some drinks. She gave us information about the sightseeing's in Zadar.
The interior of the house was modern, new, practical, hygienic and we could use a common permanent cleaned bathroom (with a bath). There was also a hair dryer. Furthermore, she told us that there was not a chance to take the bathroom towels to the fitness.
Although mentioned in our reservation that there would be a little kitchen and a fridge, there was only a microwave. But she gave us utensils and a plate. The room was clean, modern and big enough for two persons.
The internet connection was only working for 5 minutes.
We had a little conversation about the history of Zadar and the Balkan war, she was a friendly person.
- Ověřenou kvalitu ubytování
- Prověření každého hostitele
- Bezpečné platby prostřednictvím SSL šifrování


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Latest topics related to "David Overstreet"
By Jesse Pugh on February 22, 2011 at 12:57 pm
Time to continue our series on the history of the NFL Draft. Last week, we gave you the history of the 15th overall pick, which is currently held by the Dolphins. Today, we'll assess the drafting of running backs.
With countless mock drafts suggesting that the Fins will select Alabama RB Mark Ingram in the first round, I thought it would be interesting to test Troy Stradford's theory that running backs are a "dime a dozen."
In 2010, the NFL's leading rusher was Arian Foster of the Houston Texans, who ran for over 1,600 yards.
Read More
1 pages


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Discussion in 'Chevy Uplander & GM Minivan Forum' started by ChevyFan, Jan 12, 2007.
Are any of these vans still on the road?
I think i saw one last year in Antwerp :shock:
Someone should hold a minivan collision derby (what are they called???) ... you know, where cars crash into each other until only one is left running.
This is going to drive me nuts. I can't think of the term for the type of event where cars crash into each other on purpose. I can't even google for it, becuase I don't know what to google for!
Is it a destruction derby???
Hahah, found it. A DEMOLITION derby!
They should have a demolition derby for minivans!
In that case Dodge Caravans should be lit on fire, and used as obstacles in the arena.
I'd pay good money to see that.
I'm in! but hell hath no fury like a soccer mom. So careful with the mini van comments LOL.
If you're in a minivan, you're just bout always able to get into the carpool lanes...they'll say "Ahh, he's got 5 in there for sure" :biggrin:
Separate names with a comma.


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The Lion Roars – From Big Guns to Trophies, the White Tower's Basement Undergoes a Major Facelift - Friday, 30 March 2012
From big guns to banners and pikes to pistols – the storehouse and arsenal of the mighty Tower of London has been redisplayed and will be unveiled to the public on Saturday 31 March 2012.
The new-look basement galleries give a fascinating glimpse into the depths of the White Tower and it's even possible to see where the 11th century builders changed their minds about the historic building's shape when they were setting out the foundations for The Tower of London.
Storehouse is part of the Royal Armouries' project to transform the White Tower as a bold and visionary gift to the nation, in conjunction with Historic Royal Palaces (HRP).
The Royal Armouries – Britain's oldest museum – and HRP have already created three new galleries in the White Tower – Fit for A King; Treasures; and Power House.
The basement Storehouse project highlights include:
- Big Guns – for many centuries the Tower of London was home to the Board of Ordnance, providers of artillery to the British Army and Navy. The Tower housed not only to state-of-the-art guns but also antique pieces which were displayed to visitors. Today a selection of historic great guns and mortars has been re-presented to give visitors an up-close view of these triumphs of technology and decoration.
- Storehouse – for much of its approaching 1,000-year history, the Tower has been an arsenal packed with arms and armour of every kind. A selection of the pikes, swords, pistols and muskets which have filled the Tower's stores highlights the site's important role in supplying Britain's fighting forces in the past.
- Trophies – as well as showing the products of Britain's arms makers, the Tower has long been a showcase for trophies captured in foreign wars and curiosities brought back from service abroad. Exhibits come from as far afield as the Baltic and Burma, ranging from bells to door hinges and from a banner to breastplates.
- Continuity and Change – the White Tower has featured museum displays for some 100 years but many of the new basement gallery exhibits have been drawing tourists for much longer. Some have been featured in guidebooks to the Tower's curiosities since the 18th century.
Museum director Peter Armstrong said, "Basements are traditionally storehouses and can lack the glamour and appeal of main attractions but the re-display of these galleries brings the history alive, in a fascinating and highly visual way.
Working closely with Historic Royal Palaces, we have brought to life the Tower's role in ensuring that England's forces were armed effectively throughout the ages – an issue crucial to warfare, even today.
We also acknowledge the generous support of HISTORY™ who have helped make this new exhibition happen and created from our historic images a stunning presentation which explores the pivotal role of the White Tower at the heart of the site throughout centuries of change."
For more information and the latest news about the Royal Armouries:
Royal Armouries website
Royal Armouries newsletter
Follow us on Twitter
Find us on Facebook
Notes to editors
- Royal Armouries is the national museum of arms and armour and has sites in Leeds, HM Tower of London, Fort Nelson and Louisville, Kentucky. It is the first British national museum to open a permanent presence in another country
- Admission to the museum is free. However, there may be a small charge for some special events.
- Open all year daily, 10am-5pm. Closed 24-25 December
- Information Line: 0113 220 1999
- Website: www.royalarmouries.org
- The Royal Armouries Museum should not be confused with Royal Armouries International plc, the private sector corporate hospitality business.
Download the PDF press release.


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Bachelor Ben Flajnik is falling madly in love with cunning Courtney Robertson. But Courtney's only objective seems to be winning the game and not Ben's heart. Here are five reasons Ben seems to think that Courtney is the one for him.
4. Courtney pretends to be the outdoorsy girl that Ben is looking for — she caught a fish during the group date in episode four.
1. Courtney is using her sex appeal to attract him. A prime example of this is the skinny dipping scheme she pulls in the Jan. 30 episode.
3. Courtney tells Ben everything that he wants to hear, especially that she is falling for him. She even said she could see herself being his wife in the future.
5. Courtney pretends to care about Ben's feelings
2. Courtney pretends to like Ben's dog.
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To this latest ridiculous post all I can say is, "Are you serious?" Let's keep it simple. If a running back fumbles the ball and it's recovered for an 8 yard loss by the LT did that fumble hurt or help the team? If a player fumbles the ball without contact and had a clear path to the end zone, but the ball is instead recovered by a teammate short of the endzone did the fumble help or hurt the team?
The bigger issue was that Tiki Barber came into the league with ball security issues and when his touches increased his fumbles shot through the roof. Mostly, I'm guessing because the word got out that he was a fumbler, so where in the first 3 years he fumbled only occasionally (9 times)as an unknown RB, once his touches increased by year 4 every time he touched the ball defenders where trying to strip him due to his reputation as a fumbler.
To bring this full circle, Tiki Barber fumbled 45 times from 1999-2004, never did he receive an extended benching. Compare that to Wilson fumbling in his first game as rookie and then being benched for the majority of the season. It's a joke that TC would try to hold up this high standard for TOs as the reason.
As for Eli, I was definitely disappointed in his effort last year. Our most important player was our worst performing player, and it was totally unexpected.
As to Tiki, he'd already established himself as a game breaker that could make game changing plays by the time his fumbles increased as his load increased in 2000. His years of 2000-2003 were when he fumbled the most times, 9 (lost 3), 8 (lost 2), 9 (lost 6), and 9 (lost 6), respectively. Those were all under Fassel. Obviously, his coaching approach was different (and more forgiving) than is Coughlin's. Turns out that Coughlin's approach worked quite well for Tiki (and for subsequent RBs), despite his bellyaching.
On the other hand, Coughlin did not have Wilson "in the dog house" his rookie season as much as many would have us believe. He did have Wilson return kick offs the entire season (maybe 1 game was excluded, I don't remember). That skill requires the returner to catch and protect the ball before running, with all 11 guys running full tilt boogie at him and him alone. That, to me, showed Coughlin did have trust in him, in spite of his first play/fumble of the year at RB.
The "it's Coughlin and Gilbride's fault" for Wilson's unimpressive 1 and 1/3 seasons as RB is overwrought and overstated, in my view. The fumble argument concerning Wilson's rushing talents is a side issue, as Morehead, you, and a couple others have indicated. It truly is only in the minds of some fans as a reason for not giving him more touches than he's had so far, but there is no evidence that confirms that belief.
However, a player of his perceived talent, who was drafted in first round because of the perception, absolutely MUST make the most of his limited playing time early in his first year. Entering year 3, and he has yet to prove he should be the primary RB on this team.


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Everyone on this page wants a different player at #32.. a lot of teams are spending money on lLB's, Corners and WR's... Team needs will now be refocused...
Eagles - signing of Ryans might focus on them grabbing a S, OT or a rush DE.
Tampa - Jackson, Wright and Nicks.... LB is an area they may look at now. But, they may focus on OT or RB with such a high pick.
These are just a few teams that may focus on other areas.. Which makes me believe that there will be some players available at #32, that we didnt expect to be there.
Donta Hightower, Janoris Jenkins, Michael Floyd (cha-ching!)


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Spring in the Pocono Mountains
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Surrounded by a long list of high-energy excursions that include everything from snowmobiling in the winter to biking and paddle-boating in the summer, the all-inclusive, couples-only Paradise Stream Resort boasts individually appointed rooms that are each named for the area's most well-known attractions. Some rooms, like the Champagne Tower Suite, the Garden of Eden Apple Suite, and the Lakeside Villa share log-burning fireplaces, private whirlpool tubs, king-sized beds, and personal refrigerators. Spend the morning by treating yourself to an in-room spa treatment or playing a round of tennis, and then have a romantic afternoon drifting along the water in a rowboat. Later, choose a nearby hiking trail in the Pocono Mountains, and return to the resort for a swim in the pool and a drink at the poolside bar.
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Fay tweeted the other day that Janish was closer than HRod at coming back and that neither was 'too close' or something like that. I would expect a few weeks at best before seeing him come back.
In the same way that a baseball season never really begins, it never really ends either. - Lonnie Wheeler, "Bleachers, A Summer in Wrigley Field"
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The Cincinnati Reds have promoted left-handed pitcher Donnie Joseph to Triple-A Louisville. Joseph, 24, owns a .89 ERA with a 4-2 record in 26 games out of the Blue Wahoo's bullpen. He also had a 46/8 K/BB ratio.
"Since I've been with the Reds in 1989, we've never had a farm system this loaded," Bowden said. "If we were the New York Yankees and had unlimited dollars, we could have traded for Colon, (Jeff) Weaver, Rolen, (Cliff) Floyd, (Kenny) Rogers and Finley and gotten them all -- and still held onto our top five prospects. That's an amazing statement."
Former Mudcat Brad Boxberger has been promoted to San Diego. He joins two other Mudcats there, Cameron Maybin and Yonder Alonzo.
Batsfan, Yas was up for a few days but was sent down earlier this week.
Brandon Dailey was sent to Billings. He has struggled lately, and the Mustangs were looking desperate for a second baseman anyway. James Ewing was sent down from Bakersfield to replace him. Speaking of Dayton, that's where Bill Bray has started his rehab assignment. Jason Christian, the Cincinnati native, was finally activated from the Blaze's disabled list. He hadn't played since early April. Following Donnie Joseph's promotion to Louisville, Mark Serrano was activated in Pensacola. I forgot to mention that Kanekoa Texeira was put on the disabled list (shoulder) in Louisville when Feierabend was officially assigned there.
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This is just a little more info on the two Dutch players the Reds recently signed.
The Cincinnati Reds have inked two more Dutch players. Already in late May 19-year-old outfielder Aldrich Guzman got a minor league contract following his performance at the MLB European Academy tryout in Haarlem on April 27. He has played for the Euro Stars and the Tridents in the past. We already had received a tip about Guzman's signing after his name popped up in Baseball America's transaction list, but we initially couldn't confirm the signing.
The second new Cincinnati Red is 18-year-old right-handed Ibrahim Redan. He is currently playing for the AamigoO ADO Lakers in the Dutch Hoofdklasse. In eight games (seven starts) he has an ERA of 5.92 with 20 strikeouts in 38 innings. Guzman and Redan are expected to make their professional debuts in the Arizona Summer League for the AZL Reds. The 2012 season in this Rookie league starts on June 20.
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Can anyone tell me on average how much money a person can make on each hive that is doin well with making the honey in the southern states? As in arkansas and Louisiana?
Can anyone tell me on average how much money a person can make on each hive that is doin well with making the honey in the southern states? As in arkansas and Louisiana?
"You can make a million dollars keeping bees.
All ya gotta do is start out with three million." -TFBM
Solomon Parker is our resident AK specialist.
But, this is an answer almost impossible to supply. Too many variables. How good a business person are you? Are you treating your beekeeping like a business, or like a hobby? If the latter, the enjoyment factor outweighs the investment return factor.
A friend of mine w/ a cpl thousand colonies figures he spends around $175.00 per colony each year. I haven't asked him what he earns from each colony.
Mark Berninghausen
Last year here in S.E. Texas, and due to the record drought, most saw no honey at all and many experienced the loss of many hives. This year is different and most are harvesting a lot of honey. Each year is different depending mostly on the weather.
The value of your product will vary greatly depending on where you are, what's your competition, quality, and what's your attitude. And are you good at marketing. The one key that I recommend, is that if at possible, cut out the middle man. If you don't have the time to run your own sales point, see if you can sell it on consignment at local stores. That will at least support your profit margin a little more than just selling it to the store so they can mark it up 100%. After all, one can go to Walmart and get bulk honey for $2-3/lb. But that's because a packer somewhere bought it by the barrel for probably less than $1/bbl, from a commercial beekeeper that just wanted to get rid of it. If you have surplus, don't just dump it to a packer at a low cost, unless you absolutely have to. It doesn't go bad, so if you have space, just store it up until you find a more lucrative outlet. As Michael mentioned above, last summer was bad down here, and honey was hard to come by. With the drought, the cost of hay for livestock shot through the roof, and people were importing from as far away as Iowa. Not sure that honey jumped that much, but it's just a good example of how climate is also a factor.
But IMHO, the quality of store honey doesn't hold a flame to the quality of my own, fresh out of the back yard. I harvested out 3 lbs a couple weeks ago, cut it as chunk comb, and sold it for about $6/lb. I sold all four jars before noon, simply by bringing them to work and telling a few friends. I also set out a jar of incidental honey that I had collected when fixing comb problems. It was cloudy with pollen, but the taste was not compromised. I meant to set it out as bait, for samples, but also by noon, someone took the "free" on the lid a little to literal, and swiped the whole jar! Whoever it was didn't even bother to say "thanks"
Be proud of your product, and don't waste a drop. Use everything you can from the hive, and you should see good value, as long as the margins and the market is there to fill. I'd also recommend focusing on niche markets. Again, you can go buy liquid honey almost anywhere. But have you ever seen chunk comb on the store shelf? Not I. What's funny to me, is cutting chunk comb is actually less work than extracting (again, IMO), yet I can demand a higher price as it is a hard to come by commodity, and people absolutely love it! In AR and LA you might find the market already saturated with "local" honey, and that is certainly where the biggest gains are. For once I am lucky to live in the suburbs, because very few people keep bees in the city, even with the recent trend in urban beekeeping. The upside to this is higher demand, as well as a more diverse customer base. Plus in the suburbs, so many people have flower beds and flowering trees, my bees basically work from home!
The best thing I believe one can do from a marketing standpoint, is to first establish good will, even if it mean losing a little at first, like my jar of "free" honey. It got attention, and I just left a note beside it to call me for more information.
Then again I didn't even mention apitherapy, package bees, or queen rearing. I think all that will fall under a niche market heading. I'd like to try it all some day. Vertically integrate using what is best for you. I don't know how one guy could spend $175/hive in a year, unless he is feeding a lot, and counting in tangibles like fuel into the cost of a hive. Seems he must be putting in a lot of new packages or something. I spent $135 to get one package and a clipped/marked queen. Another $60 in the hive, and $50 worth of a cheap suit, smoker, and hive tool, and I'm in business. If my splits take, I'll be up to 4 hives in a couple weeks. From an asset standpoint, each established colony could sell for $200-250. A nuc (as they are now) should sell for around $150. So if I sold three as nucs, I'm already over double my original investment. I don't intend to do that, but perhaps you can see the dollar signs.
I don't mean to make it sound like it's too good to be true, because indeed it is...for now. If your serious about trying to make money as a beekeeper, you can't do it because you think you will be cashing in on honey money. Chase those niche markets and you should see better margins. Or again, vertically integrate.
Alright, I've probably put everyone to sleep, I'm out!
Last edited by Tom Brueggen; 06-28-2012 at 06:03 AM. Reason: typos
After 20 months I'm over a 20 hives and growing. See my videos! http://www.youtube.com/channel/UC8fVrmUsyYlRuASdX6UQk1g
What would be normal? Can I ask what your ballpark figure is? I have to wonder now if you dumped your bees in the fall and bought new in the spring would your costs be this high?$175.00 per colony each year.
Brian Cardinal
Zone 5a, Practicing non-intervention beekeeping
I don't believe any beekeeper really wants to just dump bees in the fall and buy more in the spring. But the majors that focus on pollination for example, may not have the time or resources to overwinter 1000's of hives. It's easier from a business standpoint to just set them in the outyard and pray. The time/resources they would have to invest in feeding and maintaining the hives through the winter would outweigh the cost of just replacing a package in the spring.
Just to clarify, I'm not lashing out at pollination service providers. Again, it's just another niche market, and from a business standpoint, you have to do what makes money. After all, I paid $135 for one package of bees, because I just bought one. The majors buy 100's or even thousands of packages at once, earning a substantial discount from buying in bulk. Some package supplier out there cashes in early, as it is easier to do that than expend the effort of trying to sell one package at time to some hobbyist like me. Now a good pollination services company would buy out that package supplier, to lower their costs that much more, and raise profits.
My goal in beekeeping is to experience every facet of it as quickly as possible, and then decide which key items are best for me to pursue. So let's say, I'll focus on raising bees and manufacturing hives, so then I can sell established colonies. The honey I would sell just as extra cash. If your goal is to make all your money through honey, then you better find a way to really cut the cost of your bees. If you want to sell bees you better find a way to get cheap feed and queens.
In the end, bees are just another form of livestock, just like raising cattle or hogs. People just don't realize it, and think that bees are something special.
I cannot speak for ballpark figures, as I have not been in it long enough to even estimate. I have spoken with a friend about his friend that does commercial pollination services. This guy runs 3-5000 hives. If each hive gets $100 for pollination, do the math, that's $500,000 per pollination job, assuming he uses all hives. If they all survive, and can be moved, say from CA after the bloom up to the NE for blueberries and cherries, he might pick up another $100/hive there. But there are massive costs incurred in between on transport and maintenance. In the end, in a good year, he may pocket $150-200K, which is nothing to snub your nose at. But that takes a lot of work, and even more risk to run a program that big. In a bad year he stands to lose just as much.
I've been keeping a detailed balance sheet of my paltry operation. Part is for proof to my wife that it is not always a losing game. The other part is for records. If I do find it to be a losing game, then I'll know that I need to change my strategy or abandon the chase. The people that lose everything tend to keep poor records and run a pretty bad operation. I'd venture to say that the ones that lose big, are the same ones that buy in too big, expecting to make a quick buck without truly understanding the bees or the market. Start small, and when you find what works, ramp up quick. And don't always look to cash in early. It takes six years to create a new breed of orchid flower, but when it sells, it can be game changer! Patience will prevail.
After 20 months I'm over a 20 hives and growing. See my videos! http://www.youtube.com/channel/UC8fVrmUsyYlRuASdX6UQk1g
You can make money off of your hobby bee hives in the south if you try - after you stop spending a lot of money on equipment.
Last year I overwintered 10 hives and harvested an average of a bit more than 15 quarts each from 7 of them. My feed cost was ababout $20 per hive, medication was almost nothing. That was one year - my first honey year, not every year. You can do the math.
It wasn't a lot, but it paid for my equipment up to that point including a small maxant extractor. This year my honey is looking a little better, and I have only bought some frames - so profit should be better. I might make a dollar an hour.
Also my entire extended family gets to enjoy homegrown honey all year.
Here,in a bad year,$0.00.In the best of all possible years,when everything goes just right,
around $300.00.That would be with the sale of some honey and a nuc or two.I freely admit that I have not had that second scenario happen to all hives in a single year.I am happy with something in the 100-200 range.Mine is a backyard operation and I don't do pollination.
Everyone's operation is different.
Hey Tom,
Bring me a gallon of your "free" honey and I will be a nice guy and tell you who ate it!
I understand that commercial beeks are in it for the money, however, I'm in it for the enjoyment. I walk over every morning, just to watch the girls fly in and out. I love watching them. I'm diabetic,so don't really care about the honey, however, I ama candle maker, and do care about the beeswax. To each his own.
Guys, the OP simply asked for an average profit per hive that can be reasonably expected when things are going well in AR and LA.
I am curious, too.
(6th year, 15 hives, Zone 5b, 5500')
The most I have ever made off of a single hive in a single season was about $500.00 That was after expenses. But that was using the hives in nuc production and selling 8 nucs from each hive.
With the longer season you should be able to do better than that.
$500 net from one hive!? Wow!
(6th year, 15 hives, Zone 5b, 5500')
Here in Northwest Arkansas, there are a few beekeepers but no commercials. There is only one commercial operation that I know of and that is down in the delta. So it can be done.
This year, I harvested about $1000 worth of honey and sold three queens and three nucs. If I don't watch out, I'm going to start breaking even. This is my ninth year and this is the first year that I'm coming close to breaking even. Part of that is I'm in the first year where I have as many hives as I want. I'll lose some this winter to even the numbers out and set me up with drawn comb and then I'll be right where I want to be.
You want to make money? Have as many hives as you want, no more, no less, and make sure it's a sustainable population. That's all I have for you. Exactly how much is wild eyed speculation at best.
If you ask around people will tell you that there is more money in selling bees than honey but the OP specifically asked about honey. If two dollars is the going rate for bulk honey than a hive that produces 100 pounds brings in $200 gross. Even with all the variables I can't see the average being too far away from the $200 mark.
Brian Cardinal
Zone 5a, Practicing non-intervention beekeeping
Here in NY, I know, not AR or LA, I had an average per colony production of about 30 lbs. So, selling that honey at about $3.00/lb that's $90.00 per colony. I also averaged $38.80/colony for pollination, across all of the colonies I had. Even though I didn't use all of my colonies in pollination. But, that's how one averages things, isn't it? I also sold some nucs. Around 100 nucs @$80.00 each. So, on average, what would that be from 500 colonies. $16.00?
$90.00 plus $38.80 plus $16.00 equals $144.00 income per colony. Somehow that doesn't look right. I must have missed something. If it takes $175.00 to support a hive, then I've lost $31.00/colony. Which I don't think I did. Something is wrong w/ my figures.
Better luck to Onesuperdog.
Last edited by sqkcrk; 06-28-2012 at 04:01 PM.
Mark Berninghausen
Actually I would disagree. What you have to do is do all aspects of honey production and sales, nuc production and sales and pollination service and do them well. You have to work as hard as your bees.
Mark Berninghausen


text: Starting 2 hives this spring. Just beginning bee keeping but my husband has been a long time bee keeper. He lost all his hives many, many years ago when Varroa first hit, then tried to get back into bees a few years ago with 4 nucs but lost them as well. So he didn't really want to try bees again, but I miss having them and miss the wonderful honey so thought I would give it a go myself. Hope all goes well. I'm really looking forward to the experience!!


text: How much land is needed in all directions from a breeding yard to control the drone pool for mating? If I have a mating yard at point A; and I have drone mother yards 1 to 2 miles awhile, north, south, east, and west of the mating yard, do I control enough territory to increase my chances of the queens mating with my target drone population. Are the drone mother colonies to close or too far from the mating yard? Drone mother yards each contain about 16 colonies. I now control such an area. There are no commercial yards within 10 miles, and no known hobby beekeepers in this area. I am trying to control the area to retain VSH charicteristics in my operation. Input?


text: Taken from April's post.11) This is a one block per swapper swap. You will have to make 35 blocks per large group (36 swappers in each group) or 15 blocks per small group (16 swappers in each group) (If you wish to do two groups please be aware this means you will have to mail 70 F8s and make 70 blocks or any combination of the possible group size). Please be aware of this before you commit yourself if you do not think you would be comfortable making 35 blocks in 4 months. (70 for 2 groups)


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Check out the newest episode of 3OH!3's "Making the Album" series. Check back for more episodes every week leading up to the June 29th release of Streets of Gold.
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-3OH!3 WebCrew


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Hormonal Contraceptives Pose Low Risk in Women With Diabetes »
"The risk of thromboembolic events from most hormonal contraceptives for women with diabetes is elevated but still relatively low, new research finds.
The results, from a large US claims database, were published online November 29 in "...
Read the Hormonal Contraceptives Pose Low Risk in Women With Diabetes article »
Cigarette smoking increases the risk of serious cardiovascular side effects from oral contraceptive use. This risk increases with age and with heavy smoking (15 or more cigarettes per day) and is quite marked in women over 35 years of age. Women who use oral contraceptives should be strongly advised not to smoke.
The use of oral contraceptives is associated with increased risks of several serious conditions including myocardial infarction, thromboembolism, stroke, hepatic neoplasia and gallbladder disease, although the risk of serious morbidity and mortality is very small in healthy women without underlying risk factors. The risk of morbidity and mortality increases significantly in the presence of other underlying risk factors such as hypertension, hyperlipidemias, obesity, and diabetes.
Practitioners prescribing oral contraceptives should be familiar with the following information relating to these risks.
The information contained in this package insert is principally based on studies carried out in patients who used oral contraceptives with higher formulations of estrogens and progestogens than those in common use today. The effect of long-term use of the oral contraceptives with lower formulations of both estrogens and progestogens remains to be determined.
Throughout this labeling, epidemiological studies reported are of two types: retrospective or case control studies and prospective or cohort studies. Case control studies provide a measure of the relative risk of a disease, namely, a ratio of the incidence of a disease among oral contraceptive users to that among nonusers. The relative risk does not provide information on the actual clinical occurrence of a disease. Cohort studies provide a measure of attributable risk, which is the difference in the incidence of disease between oral contraceptive users and nonusers. The attributable risk does provide information about the actual occurrence of a disease in the population (adapted from refs. 2 and 3 with the author's permission). For further information, the reader is referred to a text on epidemiological methods.
Thromboembolic Disorders And Other Vascular Problems
Myocardial Infarction
An increased risk of myocardial infarction has been associated with oral contraceptive use. This risk is primarily in smokers or women with other underlying risk factors for coronary artery disease such as hypertension, hypercholesterolemia, morbid obesity, and diabetes. The relative risk of heart attack for current oral contraceptive users has been estimated to be two to six (4-10). The risk is very low under the age of 30.
Smoking in combination with oral contraceptive use has been shown to contribute substantially to the incidence of myocardial infarctions in women in their mid-thirties or older with smoking accounting for the majority of excess cases (11). Mortality rates associated with circulatory disease have been shown to increase substantially in smokers, especially in those 35 years of age and older among women who use oral contraceptives.
Table II. Circulatory Disease Mortality rates per 100,000 women years by age, smoking status, and oral contraceptive use
Adapted from P.M. Layde and V. Beral, ref. #12.
Oral contraceptives may compound the effects of well-known risk factors, such as hypertension, diabetes, hyperlipidemias, age and obesity (13). In particular, some progestogens are known to decrease HDL cholesterol and cause glucose intolerance, while estrogens may create a state of hyperinsulinism (14-18). Oral contraceptives have been shown to increase blood pressure among users (see section 9 in WARNINGS). Similar effects on risk factors have been associated with an increased risk of heart disease. Oral contraceptives must be used with caution in women with cardiovascular disease risk factors.
An increased risk of thromboembolic and thrombotic disease associated with the use of oral contraceptives is well established. Case control studies have found the relative risk of users compared to nonusers to be 3 for the first episode of superficial venous thrombosis, 4 to 11 for deep vein thrombosis or pulmonary embolism, and 1.5 to 6 for women with predisposing conditions for venous thromboembolic disease (2,3,19-24). Cohort studies have shown the relative risk to be somewhat lower, about 3 for new cases and about 4.5 for new cases requiring hospitalization (25). The risk of thromboembolic disease associated with oral contraceptives is not related to length of use and disappears after pill use is stopped (2).
A two- to four-fold increase in relative risk of postoperative thromboembolic complications has been reported with the use of oral contraceptives (9). The relative risk of venous thrombosis in women who have predisposing conditions is twice that of women without such medical conditions (26). If feasible, oral contraceptives should be discontinued at least four weeks prior to and for two weeks after elective surgery of a type associated with an increase in risk of thromboembolism and during and following prolonged immobilization. Since the immediate postpartum period is also associated with an increased risk of thromboembolism, oral contraceptives should be started no earlier than four weeks after delivery in women who elect not to breast feed or four weeks after a second trimester abortion.
Cerebrovascular disease
Oral contraceptives have been shown to increase both the relative and attributable risks of cere- brovascular events (thrombotic and hemorrhagic strokes), although, in general, the risk is greatest among older ( > 35 years), hypertensive women who also smoke. Hypertension was found to be a risk factor for both users and nonusers, for both types of strokes, and smoking interacted to increase the risk of stroke (27-29).
In a large study, the relative risk of thrombotic strokes has been shown to range from 3 for normotensive users to 14 for users with severe hypertension (30). The relative risk of hemorrhagic stroke is reported to be 1.2 for nonsmokers who used oral contraceptives, 2.6 for smokers who did not use oral contraceptives, 7.6 for smokers who used oral contraceptives, 1.8 for normotensive users and 25.7 for users with severe hypertension (30). The attributable risk is also greater in older women (3).
Dose-related risk of vascular disease with oral contraceptives
A positive association has been observed between the amount of estrogen and progestogen in oral contraceptives and the risk of vascular disease (31-33). A decline in serum high density lipoproteins (HDL) has been reported with many progestational agents (14-16). A decline in serum high density lipoproteins has been associated with an increased incidence of ischemic heart disease. Because estrogens increase HDL cholesterol, the net effect of an oral contraceptive depends on a balance achieved between doses of estrogen and progestogen and the activity of progestogen used in the contraceptive. The activity and amount of both hormones should be considered in the choice of an oral contraceptive.
Minimizing exposure to estrogen and progestogen is in keeping with good principles of therapeutics. For any particular estrogen/progestogen combination, the dosage regimen prescribed should be one which contains the least amount of estrogen and progestogen that is compatible with a low failure rate and the needs of the individual patient. New acceptors of oral contraceptives agents should be started on preparations containing 0.035 mg or less of estrogen.
Persistence of risk of vascular disease
There are two studies which have shown persistence of risk of vascular disease for everusers of oral contraceptives. In a study in the United States, the risk of developing myocardial infarction after discontinuing oral contraceptives persists for at least 9 years for women 40-49 years who had used oral contraceptives for five or more years, but this increased risk was not demonstrated in other age groups (8). In another study in Great Britain, the risk of developing cerebrovascular disease persisted for at least 6 years after discontinuation of oral contraceptives, although excess risk was very small (34). However, both studies were performed with oral contraceptive formulations containing 50 micrograms or higher of estrogens.
Estimates Of Mortality From Contraceptive Use
One study gathered data from a variety of sources which have estimated the mortality rate associated with different methods of contraception at different ages (Table III). These estimates include the combined risk of death associated with contraceptive methods plus the risk attributable to pregnancy in the event of method failure. Each method of contraception has its specific benefits and risks. The study concluded that with the exception of oral contraceptive users 35 and older who smoke, and 40 or older who do not smoke, mortality associated with all methods of birth control is low and below that associated with childbirth. The observation of an increase in risk of mortality with age for oral contraceptive users is based on data gathered in the 1970's (35). Current clinical recommendation involves the use of lower estrogen dose formulations and a careful consideration of risk factors. In 1989, the Fertility and Maternal Health Drugs Advisory Committee was asked to review the use of oral contraceptives in women 40 years of age and over. The Committee concluded that although cardiovascular disease risks may be increased with oral contraceptive use after age 40 in healthy non-smoking women (even with the newer low-dose formulations), there are also greater potential health risks associated with pregnancy in older women and with the alternative surgical and medical procedures which may be necessary if such women do not have access to effective and acceptable means of contraception. The Committee recommended that the benefits of low-dose oral contraceptive use by healthy nonsmoking women over 40 may outweigh the possible risks.
Of course, older women, as all women who take oral contraceptives, should take an oral contraceptive which contains the least amount of estrogen and progestogen that is compatible with a low failure rate and individual patient needs.
Table III. Annual Number of Birth-related or Method-related
deaths associated with control of fertility per 100,000 nonsterile women, by
fer- tility control method according to age
| Method of control
and outcome
|No fertility control methods*
|Oral contraceptives nonsmoker**
|Oral contraceptives smoker**
|Diaphragm/ spermicide*
|Periodic abstinence*
| * Deaths are birth-related
** Deaths are method-related
Adapted from H.W. Ory, ref. #35.
Carcinoma Of The Reproductive
ORGANS AND BREASTS Numerous epidemiological studies have been performed on the incidence of breast, endometrial, ovarian, and cervical cancer in women using oral contraceptives. While there are conflicting reports, most studies suggest that use of oral contraceptives is not associated with an overall increase in the risk of developing breast cancer. A meta-analysis of 54 studies reports that women who are currently using combined oral contraceptives or have used them in the past 10 years are at slightly increased risk of having breast cancer diagnosed although the additional cancers tend to be localized to the breast. There is no evidence of an increased risk of having breast cancer diagnosed 10 or more years after cessation of use.
Some studies suggest that oral contraceptive use has been associated with an increase in the risk of cervical intraepithelial neoplasia in some populations of women (45-48). However, there continues to be controversy about the extent to which such findings may be due to differences in sexual behavior and other factors.
Hepatic Neoplasia
Benign hepatic adenomas are associated with oral contraceptive use, although the incidence of benign tumors is rare in the United States. Indirect calculations have estimated the attributable risk to be in the range of 3.3 cases/100,000 for users, a risk that increases after four or more years of use especially with oral contraceptives of higher dose (49). Rupture of benign, hepatic adenomas may cause death through intraabdominal hemorrhage (50, 51).
Studies have shown an increased risk of developing hepatocellular carcinoma in oral contraceptive users. However, these cancers are rare in the U.S., and the attributable risk (the excess incidence) of liver cancers in oral contraceptive users approaches less than one per million users.
Ocular Lesions
There have been clinical case reports of retinal thrombosis associated with the use of oral con- traceptives. Oral contraceptives should be dis- continued if there is unexplained partial or complete loss of vision; onset of proptosis or diplopia; papilledema; or retinal vascular lesions. Appropriate diagnostic and therapeutic measures should be undertaken immediately.
Oral Contraceptive Use Before Or During Early Pregnancy
Extensive epidemiological studies have revealed no increased risk of birth defects in women who have used oral contraceptives prior to pregnancy (56,57). The majority of recent studies also do not indicate a teratogenic effect, particularly in so far as cardiac anomalies and limb reduction defects are concerned (55,56,58,59), when taken inadvertently during early pregnancy.
The administration of oral contraceptives to induce withdrawal bleeding should not be used as a test for pregnancy. Oral contraceptives should not be used during pregnancy to treat threatened or habitual abortion. It is recommended that for any patient who has missed two consecutive periods, pregnancy should be ruled out before continuing oral contraceptive use. If the patient has not adhered to the prescribed schedule, the possibility of pregnancy should be considered at the time of the first missed period. Oral contraceptive use should be discontinued until pregnancy is ruled out.
Gallbladder Disease
Earlier studies have reported an increased lifetime relative risk of gallbladder surgery in users of oral contraceptives and estrogens (60,61). More recent studies, however, have shown that the relative risk of developing gallbladder disease among oral contraceptive users may be minimal (62-64). The recent findings of minimal risk may be related to the use of oral contraceptive formulations containing lower hormonal doses of estrogens and progestogens.
Carbohydrate And Lipid Metabolic Effects
Oral contraceptives have been shown to cause a decrease in glucose tolerance in a significant percentage of users (17). This effect has been shown to be directly related to estrogen dose (65). Progestogens increase insulin secretion and create insulin resistance, this effect varying with different progestational agents (17,66). However, in the non-diabetic woman, oral contraceptives appear to have no effect on fasting blood glucose (67). Because of these demonstrated effects, prediabetic and diabetic women in particular should be carefully monitored while taking oral contraceptives.
A small proportion of women will have persistent hypertriglyceridemia while on the pill. As dis- cussed earlier (see WARNINGS), changes in serum triglycerides and lipoprotein levels have been reported in oral contraceptive users.
Elevated Blood Pressure
An increase in blood pressure has been reported in women taking oral contraceptives (68) and this increase is more likely in older oral contraceptive users (69) and with extended duration of use (61). Data from the Royal College of General Practitioners (12) and subsequent randomized trials have shown that the incidence of hypertension increases with increasing progestational activity. Women with a history of hypertension or hypertension-related diseases, or renal disease (70) should be encouraged to use another method of contraception. If women elect to use oral contraceptives, they should be monitored closely and if significant elevation of blood pressure occurs, oral contraceptives should be discontinued. For most women, elevated blood pressure will return to normal after stopping oral contraceptives, and there is no difference in the occurrence of hypertension between former and never users (68-71).
The onset or exacerbation of migraine or development of headache with a new pattern which is recurrent, persistent, or severe requires discontinuation of oral contraceptives and evaluation of the cause.
Bleeding Irregularities
Breakthrough bleeding and spotting are sometimes encountered in patients on oral contraceptives, especially during the first three months of use. Non-hormonal causes should be considered and adequate diagnostic measures taken to rule out malignancy or pregnancy in the event of breakthrough bleeding, as in the case of any abnormal vaginal bleeding. If pathology has been excluded, time or a change to another formulation may solve the problem. In the event of amenorrhea, pregnancy should be ruled out.
Some women may encounter post-pill amenorrhea or oligomenorrhea, especially when such a condition was preexistent.
Ectopic Pregnancy
Ectopic as well as intrauterine pregnancy may occur in contraceptive failures. However, in progestogen-only oral contraceptive failures, the ratio of ectopic to intrauterine pregnancies is higher than in women who are not receiving oral contraceptives, since the drugs are more effective in preventing intrauterine than ectopic pregnancies.
Physical Examination And Follow-Up
It is good medical practice for all women to have annual history and physical examinations, including women using oral contraceptives. The physical examination, however, may be deferred until after initiation of oral contraceptives if requested by the woman and judged appropriate by the clinician. The physical examination should include special reference to blood pressure, breasts, abdomen, and pelvic organs, including cervical cytology, and relevant laboratory tests. In case of undiagnosed, persistent, or recurrent abnormal vaginal bleeding, appropriate measures should be conducted to rule out malignancy. Women with a strong family history of breast cancer or who have breast nodules should be monitored with particular care.
Lipid Disorders
Women who are being treated for hyperlipidemias should be followed closely if they elect to use oral contraceptives. Some progestogens may elevate LDL levels and may render the control of hyperlipidemias more difficult.
Liver Function
If jaundice develops in any woman receiving such drugs, the medication should be discontinued. Steroid hormones may be poorly metabolized in patients with impaired liver function.
Fluid Retention
Oral contraceptives may cause some degree of fluid retention. They should be prescribed with caution, and only with careful monitoring, in patients with conditions which might be aggravated by fluid retention.
Emotional Disorders
Women with a history of depression should be carefully observed and the drug discontinued if depression recurs to a serious degree.
Contact Lenses
Contact lens wearers who develop visual changes or changes in lens tolerance should be assessed by an ophthalmologist.
See WARNINGS section.
Pregnancy Category X. See CONTRAINDICATIONS and WARNINGS sections.
Nursing Mothers
Small amounts of oral contraceptive steroids have been identified in the milk of nursing mothers and a few adverse effects on the child have been reported, including jaundice and breast enlargement. In addition, combination oral contraceptives given in the postpartum period may interfere with lactation by decreasing the quantity and quality of breast milk. If possible, the nursing mother should be advised not to use combination oral contraceptives but to use other forms of contraception until she has completely weaned her child.
Sexually Transmitted Diseases
Patients should be counseled that this product does not protect against HIV infection (AIDS) and other sexually transmitted diseases.
Pediatric Use
Safety and efficacy of NECON (norethindrone and ethinyl estradiol tablets ) Tablets has been established in women of reproductive age. Safety and efficacy are expected to be the same for postpubertal adolescents under the age of 16 and for users 16 years and older. Use of this product before menarche is not indicated.
Information For Patient
See Patient Labeling.
1. Source: Trussell J. Contraceptive Efficacy Table from Hatcher RA, Trussell J, Stewart F, Cates W, Stewart GK, Kowal D, Guest F, In Con- traceptive Technology: Seventeenth Revised Edition. New York, NY: Irvington Publishers, 1998. 2. Stadel BV, Oral contraceptives and car- diovascular disease. (Pt. 1). N Engl J Med 1981; 305:612-618. 3. Stadel BV, Oral contraceptives and cardiovascular disease. (Pt. 2). N Engl J Med 1981; 305:672-677. 4. Adam SA, Thorogood M. Oral contraception and myocardial infarction revisited: the effects of new preparations and prescribing patterns. Br J Obstet Gynaecol 1981; 88:838-845. 5. Mann JI, Inman WH. Oral contraceptives and death from myocardial infarction. Br Med J 1975; 2(5965): 245-248. 6. Mann JI, Vessey MP, Thorogood M, Doll R. Myocardial infarction in young women with special reference to oral contraceptive practice. Br Med J 1975; 2(5956): 241-245. 7. Royal College of General Practitioners' Oral Contraception Study: Further analyses of mortality in oral contraceptive users. Lancet 1981; 1:541-546. 8. Stone D, Shapiro S, Kaufman DW, Rosenberg L, Miettinen OS, Stolley PD. Risk of myocardial infarction in relation to current and discontinued use of oral contracep- tives. N Engl J Med 1981;305:420-424. 9. Vessey MP. Female hormones and vascular disease-an epidemiological overview. Br J Fam Plann 1980; 6(Supplement): 1-12. 10. Russell-Briefel RG, Ezzati TM, Fulwood R, Perlman JA, Murphy RS. Cardiovascular risk status and oral contracep- tive use, United States 1976-1980. Prevent Med 1986; 15:352-362. 11. Goldbaum GM, Kendrick JS, Hogelin GC, Gentry EM. The relative impact of smoking and oral contraceptive use on women in the United States. JAMA 1987; 258:1339- 1342. 12. Layde PM, Beral V Further analyses of mortality in oral contraceptive users; Royal College of General Practitioners' Oral Contraception Study. (Table 5) Lancet 1981; 1:541-546. 13. Knopp RH. Arteriosclerosis risk: the roles of oral contraceptives and postmenopausal estrogens. J Reprod Med 1986; 31(9) (Supplement) :913- 921. 14. Krauss RM, Roy S, Mishell DR, Casagrande J, Pike MC. Effects of two low-dose oral contraceptives on serum lipids and lipoproteins: Differential changes in high-density lipoproteins subclasses. Am J Obstet 1983; 145;446-452. 15. Wahl P, Walden C, Knopp R, Hoover J, Wallace R, Heiss G, Rifkind B. Effect of estrogen/progestin potency on lipid/lipopro- tein cholesterol. N Engl J Med 1983; 308:862- 867. 16. Wynn V, Niththyananthan R. The effect of progestin in combined oral contraceptives on serum lipids with special reference to high density lipoproteins. Am J Obstet Gynecol 1982; 142: 766-771. 17. Wynn V, Godsland I. Effects of oral contraceptives on carbohydrate metabo- lism. J Reprod Med 1986; 31 (9) (Supplement) :892-897. 18. LaRosa JC. Atherosclerotic risk factors in cardiovascular disease. J Reprod Med 1986; 31 (9) (Supplement): 906-912. 19. Inman WH, Vessey MP. Investigation of death from pul- monary, coronary, and cerebral thrombosis and embolism in women of child-bearing age. Br Med J 1968; 2(5599): 193-199. 20. Maguire MG, Tonascia J, Sartwell PE, Stolley PD, Tockman MS. Increased risk of thrombosis due to oral con- traceptives: a further report. Am J Epidemiol 1979; 110(2):188-195. 21. Petitti DB, Wingerd J, Pellegrin F, Ramacharan S. Risk of vascular disease in women: smoking, oral contraceptives, noncontraceptive estrogens, and other factors. JAMA 1979; 242: 1150-1154. 22. Vessey MP, Doll R. Investigation of relation between use of oral contraceptives and thromboembolic disease. Br Med J 1968;2(5599) :199-205. 23. Vessey MP, Doll R. Investigation of relation between use of oral contraceptives and thromboembolic disease. A further report. Br Med J 1969; 2 (5658): 651-657. 24. Porter JB, Hunter JR, Danielson DA, Jick H, Stergachis A. Oral contraceptives and non-fatal vascular disease-recent experience. Obstet Gyneco 1982; 59 (3):299-302. 25. Vessey M, Doll R, Peto R, Johnson B, Wiggins P. A long- term follow-up study of women using different methods of contraception: an interim report. J Biosocial Sci 1976; 8: 375-427. 26. Royal College of General Practitioners: Oral Contraceptives, venous thrombosis, and varicose veins. J Royal Coll Gen Pract 1978;28: 393-399. 27. Collaborative Group for the Study of Stroke in Young Women: Oral contraception and increased risk of cerebral ischemia or thrombosis. N Engl J Med 1973; 288: 871-878. 28. Petitti DB, Wingerd J. Use of oral contraceptives, cigarette smoking, and risk of subarachnoid hemorrhage. Lancet 1978; 2: 234-236. 29. Inman WH. Oral contra- ceptives and fatal subarachnoid hemorrhage. Br Med J 1979; 2 (6203): 1468-1470. 30. Collaborative Group for the Study of Stroke in Young Women: Oral Contraceptives and stroke in young women: associated risk factors. JAMA 1975; 231: 718- 722. 31. Inman WH, Vessey MP, West- erholm B, Engelund A. Thromboembolic disease and the steroidal content of oral contraceptives. A report to the Committee on Safety of Drugs. Br Med J 1970; 2: 203-209. 32. Meade TW, Green- berg G, Thompson SG. Progestogens and cardiovascular reactions associated with oral con- traceptives and a comparison of the safety of 50- and 35-mcg oestrogen preparations. Br Med J 1980; 280 (6224): 1157-1161. 33. Kay CR. Progestogens and arterial disease-evidence from the Royal College of General Practitioners' Study. Am J Obstet Gynecol 1982; 142: 762-765. 34. Royal College of General Practitioners: Incidence of arterial disease among oral contraceptive users. J Royal Coll Gen Pract 1983; 33: 75- 82. 35. Ory HW. Mortality associated with fertility and fertility control: 1983. Family Planning Perspectives 1983; 15: 50-56. 36. The Cancer and Steroid Hormone Study of the Centers for Disease Control and the National Institute of Child Health and Human Development: Oral contraceptive use and the risk of breast cancer. N Engl J Med 1986; 315: 405-411. 37. Pike MC, Henderson BE, Krailo MD, Duke A, Roy S. Breast cancer in young women and use of oral contraceptives: possible modifying effect of formula- tion and age at use. Lancet 1983; 2: 926-929. 38. Paul C, Skegg DG, Spears GFS, Kaldor JM. Oral contraceptives and breast cancer: A national study. Br Med J 1986; 293: 723-725. 39. Miller DR, Rosenberg L, Kaufman DW, Schottenfeld D, Stolley PD, Shapiro S. Breast cancer risk in relation to early oral contraceptive use. Obstet Gynecol 1986; 68: 863-868. 40. Olson H, Olson KL, Moller TR, Ranstam J, Holm P. Oral contraceptive use and breast cancer in young women in Sweden (letter). Lancet 1985; 2: 748-749. 41. McPherson K, Vessey M, Neil A, Doll R, Jones L, Roberts M. Early contraceptive use and breast cancer: Results of another casecontrol study. Br J Cancer 1987; 56: 653-660. 42. Huggins GR, Zucker PF Oral contraceptives and neoplasia; 1987 update. Fertil Steril 1987; 47: 733-761. 43. McPherson K, Drife JO. The pill and breast cancer: why the uncertainty? Br Med J 1986; 293: 709-710. 44. Shapiro S. Oral contraceptives-time to take stock. N Engl J Med 1987; 315: 450-451. 45. Ory H, Naib Z, Conger SB, Hatcher RA, Tyler CW. Contraceptive choice and prevalence of cervical dysplasia and carcinoma in situ. Am J Obstet Gynecol 1976; 124: 573-577. 46. Vessey MP, Lawless M, McPherson K, Yeates D. Neoplasia of the cervix uteri and contraception: a possible adverse effect of the pill. Lancet 1983; 2: 930. 47. Brinton LA, Huggins GR, Lehman HF, Malli K, Savitz DA, Trapido E, Rosenthal J, Hoover R. Long term use of oral contraceptives and risk of invasive cervical cancer. Int J Cancer 1986; 38: 339-344. 48. WHO Collaborative Study of Neoplasia and Steroid Contraceptives: Invasive cervical cancer and combined oral contraceptives. Br Med J 1985; 290: 961-965. 49. Rooks JB, Ory HW, Ishak KG, Strauss LT, Greenspan JR, Hill AP, Tyler CW. Epidemiology of hepatocellular adenoma; the role of oral contraceptive use. JAMA 1979; 242: 644-648. 50. Bein NN, Goldsmith HS. Recurrent massive hemorrhage from benign hepatic tumors secondary to oral contraceptives. Br J Surg 1977; 64: 433-435. 51. Klatskin G. Hepatic tumors: possible relationship to use of oral contraceptives. Gastroenterology 1977; 73: 386- 394. 52. Henderson BE, Preston- Martin S, Edmondson HA, Peters RL, Pike MC. Hepatocellular carcinoma and oral contraceptives. Br J Cancer 1983; 48: 437-440. 53. Neuberger J, Forman D, Doll R, Williams R. Oral contraceptives and hepatocellular carcinoma. Br Med J 1986; 292: 1355-1357. 54. Forman D, Vincent TJ, Doll R. Cancer of the liver and oral contraceptives. Br Med J 1986; 292: 1357-1361. 55. Harlap S, Eldor J. Births following oral contraceptive failures. Obstet Gynecol 1980; 55: 447-452. 56. Savolainen E, Saksela E, Saxen L. Teratogenic hazards of oral contraceptives analyzed in a national malformation register. Am J Obstet Gynecol 1981; 140: 521-524. 57. Janerich DT, Piper JM, Glebatis DM. Oral contraceptives and birth defects. Am J Epidemiol 1980; 112: 73-79. 58. Ferencz C, Matanoski GM, Wilson PD, Rubin JD, Neill CA, Gutberlet R. Maternal hormone therapy and congenital heart disease. Teratology 1980; 21: 225-239. 59. Rothman KJ, Fyler DC, Goldblatt A, Kreidberg MB. Exogenous hormones and other drug exposures of children with congenital heart disease. Am J Epidemiol 1979; 109: 433-439. 60. Boston Collaborative Drug Surveillance Program: Oral contraceptives and venous thromboembolic disease, surgically confirmed gallbladder disease, and breast tumors. Lancet 1973; 1:1399-1404. 61. Royal College of General Practitioners: Oral contraceptives and health. New York, Pittman 1974. 62. Layde PM, Vessey MP, Yeates D. Risk of gallbladder disease: a cohort study of young women attending family planning clinics. J Epidemiol Community Health 1982; 36: 274-278. 63. Rome Group for Epidemiology and Prevention of Cholelithiasis (GREPCO): Preva- lence of gallstone disease in an Italian adult female population. Am J Epidemiol 1984; 119: 796-805. 64. Storm BL, Tamragouri RT, Morse ML, Lazar EL, West SL, Stolley PD, Jones JK. Oral contraceptives and other risk factors for gallbladder disease. Clin Pharmacol Ther 1986; 39: 335- 341. 65. Wynn V, Adams PW, Godsland IF, Melrose J, Niththyananthan R, Oakley NW, Seedj A. Comparison of effects of different combined oral contraceptive formulations on carbohydrate and lipid metabolism. Lancet 1979; 1: 1045- 1049. 66. Wynn V Effect of progesterone and progestins on carbohydrate metabolism. In: Progesterone and Progestin. Bardin CW, Milgrom E, Mauvis-Jarvis P. eds. New York, Raven Press 1983; pp. 395-410. 67. Perlman JA, Roussell-Briefel RG, Ezzati TM, Lieberknecht G. Oral glucose tolerance and the potency of oral contraceptive progestogens. J Chronic Dis 1985; 38: 857-864. 68. Royal College of General Prac- titioners' Oral Contraception Study: Effect on hypertension and benign breast disease of progestogen component in combined oral con- traceptives. Lancet 1977; 1: 624. 69. Fisch IR, Frank J. Oral contraceptives and blood pressure. JAMA 1977; 237: 2499-2503. 70. Laragh AJ. Oral contraceptive induced hypertension - nine years later. Am J Obstet Gynecol 1976; 126: 141-147. 71. Ramcharan S, Peritz E, Pellegrin FA, Williams WT Incidence of hypertension in the Walnut Creek Contraceptive Drug Study cohort: In: Pharmacology of steroid contraceptive drugs. Garattini S, Berendes HW. eds. New York, Raven Press 1977; pp. 277-288, (Monographs of the Mario Negri Institute for Pharmacological Research Milan.) 79. Schlesselman J, Stadel BV, Murrary P, Lai S. Breast cancer in relation to early use of oral contraceptives. JAMA 1988; 259: 1828-1833. 80. Hennekens CH, Speizer FE, Lipnick RJ, Rosner B, Bain C, Belanger C, Stampfer MJ, Willett W, Peto R. A case-control study of oral contraceptive use and breast cancer. JNCI 1984; 72: 39-42. 81. La Vecchia C, Decarli A, Fasoli M, Franceschi S, Gentile A, Negri E, Parazzini F, Tognoni G. Oral contraceptives and cancers of the breast and of the female genital tract. Interim results from a case-control study. Br J Cancer 1986; 54: 311- 317. 82. Meirik O, Lund E, Adami H, Bergstrom R, Christoffersen T, Bergsjo P. Oral contraceptive use and breast cancer in young women. A Joint National Casecontrol study in Sweden and Norway. Lancet 1986; II: 650-654. 83. Kay CR, Hannaford PC. Breast cancer and the pill - A further report from the Royal College of General Practitioners' oral contraception study. Br J Cancer 1988; 58: 675-680. 84. Stadel BV, Lai S, Schlesselman JJ, Murray P. Oral contraceptives and premenopausal breast cancer in nulliparous women. Contraception 1988; 38: 287-299. 85. Miller DR, Rosenberg L, Kaufman DW, Stolley P, Warshauer ME, Shapiro S. Breast cancer before age 45 and oral contraceptive use: New Findings. Am J Epidemiol 1989; 129: 269-280. 86. The UK National Case-Control Study Group, Oral contraceptive use and breast cancer risk in young women. Lancet 1989; 1: 973-982. 87. Sch- lesselman JJ. Cancer of the breast and reproductive tract in relation to use of oral contraceptives. Contraception 1989; 40: 1-38. 88. Vessey MP, McPherson K, Villard-Mackintosh L, Yeates D. Oral contraceptives and breast cancer: latest findings in a large cohort study. Br J Cancer 1989; 59: 613-617. 89. Jick SS, Walker AM, Stergachis A, Jick H. Oral contraceptives and breast cancer. Br J Cancer 1989; 59: 618-621.This monograph has been modified to include the generic and brand name in many instances.
Last reviewed on RxList: 3/24/2008
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A sham of a cameo. They happen most frequently in movie remakes, so the remake can pretend to have a connection to the original.
Did you see the post-credits scenes with Dirk Benedict and Dwight Schultz in the A-Team movie? What a shameo! They both said they regretted doing it afterwards.
by Marvelyn August 19, 2012


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We encourage you to visit our website regularly to keep current on the exciting activities, news and accomplishments of the Bloomfield schools.
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Town Square
East Meadow Mess: School Traffic Standing Still
Original post made by JLS Mom, South of Midtown, on Nov 26, 2012
East Meadow is socked in today because the traffic light at Waverly is out. If you can avoid East Meadow do it! Bike, Walk or drop off somewhere else. I hope it's fixed for Tuesday.


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Dr. Shaik Jeelani, Ph.D., P.E., serves as the Vice President for Research and Sponsored Programs at Tuskegee University. He joined Tuskegee University as an Assistant Professor of Mechanical Engineering in 1974. He served in the College of Engineering from 1977 to 1996 as a faculty member, Assistant Dean, Associate Dean and Interim Dean.
As a Professor and administrator in the College of Engineering, Jeelani developed robust recruitment and retention programs that enabled the college maintain high enrolment and produce graduates of highest caliber. He initiated a research program in Materials Science and Engineering, which resulted in the establishment of the Materials Research Laboratory (MRL). The MRL was later transformed to the Center for Advance Materials (T-CAM), which still functions as the largest multidisciplinary research, education and outreach center at Tuskegee University.Jeelani raised more than $50 million for research at T-CAM during last 30 years.
Jeelani has authored/co-authored more than 700 publications including more than 300 papers in refereed journals and more than 400 technical presentations at national and international conferences. He has also co-authored several book chapters, in the field of Materials science and Engineering, with his colleagues.
In 1996, Jeelani was appointed as Tuskegee University's first Vice President for Research and Sponsored Programs with a charge to facilitate the development and implementation of sponsored research and other programs that receive external support. He also has the responsibility of establishing and implementing policies governing the conduct and accountability for all research and other sponsored activities, and ensuring that all sponsored research projects comply with the local and federal guidelines.During Jeelani's tenure as the Vice President, both the annual funding for research and sponsored programs and the number of faculty submitting proposals to federal agencies and foundations have more than doubled.
Jeelani spearheaded the development and the implementation of Tuskegee University's first Ph.D. program in Materials Science and Engineering (MSE), which was started in 1998. Tuskegee University is now recognized as the largest producer of African American PhDs in MSE.
In 2011 the Board of Trustees of Tuskegee University approved the establishment of the Department of MSE in the College of Engineering. Jeelani serves as the founding chair of this department, which houses both the Ph.D. and the newly approved Master's programs in MSE.
Jeelani holds membership in several professional organizations and serves on several national boards. Recently, the American Society for Mechanical Engineers (ASME) elected him as a fellow, which is the highest honor the ASME bestows on its selected members.
Jeelani has won numerous awards including Tuskegee Teacher of the Year, Tuskegee Faculty Achievement, The UNCF Professor of the Year and SECME's Global Messenger Awards. In 2011 President Barack Obama presented him the Presidential Award for Mentoring.
Jeelani received the B.E. degree from Osmania University, India, the M.S. and the Ph.D. degrees in Mechanical Engineering from North Carolina State University in 1971 and 1974, respectively. He is a registered Professional Engineer in the State of Alabama.
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Iam so sorry to hear that. I was really hoping she would skate well. I thought she had a good chance for a gold this year at Europeans. Her short program is so beautiful this year.
Why is everyone saying she had a shot at Euros? Is Kostner not going to Euros?
Last year korpi won silver and kostner took the whole fall off and korpi did well and made the gpf
It is too bad she is missing Europeans. It would have been great to see her go up against Kostner and the Russians. I really dont think she would have won, I think that would have still been Kostner, but who knows, Carolina can be inconsistent sometimes too and is probably still building her season at this point.


text: If you've ever been to Birchwood Lodge in Sister Bay during the Christmas holidays, you'll recall that a huge, fresh, evergreen tree stands in the lobby next to the fireplace. In past holiday seasons we have always purchased a tree that fits the size and scale of the lobby. This year another beautiful tree stands… Continue Reading


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The Internet is a global system of interconnected computer networks that use the standard Internet Protocol Suite (TCP/IP) to serve billions of users worldwide. A highway is a public road, especially a major road connecting two or more destinations. Any interconnected set of highways can be variously referred to as a "highway system", a "highway network", or a "highway transportation system".
I got that in the internet highway!! (from scrubs)
by allnewtocho May 15, 2011


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I got around to cleaning out my nesting blocks and was surprised at 90% losses this year. Actually I wasn't too surprised as I did a few things differently and the neighbors pivoting irrigation system drenched my nesting blocks for half the summer. 90% losses is staggering but lessons were learned and certain things won't happen again Anyway heres 2 pics of some leafcutter cocoons that I removed and then cleaned. The yellow ones were made with the petals of bindweed on the outside and haygrazer (corn-like plant) on the inside. The yellow is always a striking contrast to the greens and browns that are usually present in cocoons and it reminds me spring/summer isn't too far away.
Cocoons that were removed:
Cleaned and separated cocoons: