text:
Police Chief Joe Treviño said city residents who want fireworks this July 4 will have a chance to see the real thing without spending a dime.
Angel Care Ambulance Service is paying the cost of a first-class fireworks display at the Bee County Exposition Center for the third year in a row, said business owner Gabriel Aleman.
With the help of Bebe Adamez, Aleman is hoping to entice as many local residents as possible to come out and see thousands of dollars worth of aerial fireworks go off high in the sky.
Parking and entrance to the Expo Center grounds will all be free, Adamez said.
This year's celebration will not feature the daytime activities in the Lucas-McNeill Pavilions.
Turnout for the daytime vendors and events has been slim at best, Adamez said, mainly because of the summertime weather.
"It's just too hot," Aleman said.
But as the day begins to cool off at about 6:30 or 7 p.m., Adamez said the gates will open and fireworks fans will be able to park in the large lot north of the rodeo arenas.
That will give those attending a chance to park their pickups and set out their lawn chairs where they can best see the fireworks before the show starts at about 9 p.m.
Aleman said plans are to set off the fireworks closer to the center's old rodeo arena to give spectators parked on the grounds a better vantage point for viewing the display.
He said restrooms will be open in the new rodeo arena for those attending the fireworks event.
Angel Care is planning to have a refreshment stand to sell soft drinks and simple food, like sausage wraps.
"We'll have a tent set up or something," Aleman said. All proceeds will go toward Angel Care's Relay for Life donation next year.
Once again, the experts from the Beeville Volunteer Fire Department will be in charge of setting off the fireworks.
Adamez said security will be provided at the Expo Center grounds by the Bee County Sheriff's Office and Beeville Police Department.
"But that doesn't mean there won't be any officers patrolling in the city," Treviño said.
Officers will be out answering calls from neighbors who complain about fireworks going off near them.
The chief said it has been illegal to possess, set off, store or use fireworks inside the city since the City Council passed an ordinance against the fire threat in 1973.
"It's a Class C misdemeanor," the chief said. Although the fine is $200, violators also will be required to pay court costs.
"It's just not worth it," Treviño said.
Gary Kent is a reporter at the Bee-Picayune and can be reached at 358-2550, ext. 120, or at firstname.lastname@example.com.


text:
i appreciate that there is a boatload of new information of late that has distracted most everyone from the story that was paramount not too long ago, but for me, the long overdue third installment of the article about the disabled dumbs is the story i am waiting for with the keenest anticipation.
it seems to me to be the most important story of the year, since it indicates, if true, that the benevolent ets are taking a very active role in helping us through the shift, and goddess knows, we need it...
please, if possible, the third installment will be most appreciated, especially if you have something that is really credible, not just reports of unusual earthquakes and strange sounds. there doesn't seem to be much on the net about this story, which makes me think it couldn't have been true, because if there was really good evidence, the whole net would be buzzing with it by now, from many different sources, i would think.
but i would love to be proven wrong on that!

subset: commoncrawl


text:
Slow Cooker Rustic Italian Chicken:
Prep Time: 40 min
Total: 4 hrs 40 mins
Servings Total: 4 Servings
What you need:
4 boneless skinless chicken thighs
½ cup carrots, cut into ½-inch slices
¼ cup chopped red bell pepper
¼ cup sliced fresh mushrooms
1 clove garlic, finely chopped
1 teaspoon Italian seasoning
1/8 teaspoon salt
1/8 teaspoon pepper
1 ½ cups crushed tomatoes, undrained
1 ¼ cups uncooked penne pasta
Shredded Parmesan cheese, if desired
Chopped fresh Italian (flat-leaf) parsley, if desired
Make Slow Cooker Rustic Italian Chicken:
1. Spray 1 ½-quart slow cooker with cooking spray. Place chicken in slow cooker. Top with remaining ingredients except pasta, cheese and parsley.
2. Cover; cook on Low heat setting 4 to 5 hours. About 30 minutes before chicken is done, cook and drain pasta as directed on package.
3. Serve chicken with pasta. Garnish with cheese and parsley


text:
Is Joe Blanton Really The $10 Million Man?
OK, actually $10.25 million. This is the amount that Blanton is apparently seeking for 2010. The Phillies have offered $7.5 million. If the two sides can't reach an agreement, the case will go to arbitration next month.
Is it just me, or does anyone else feel that Blanton and/or his agent are highly delusional in believing that he is worth over $10 million for the coming season?
Let's take a look at 2009, shall we? Blanton went 12-8, with a 4.05 ERA. He averaged 6.3 innings pitched in his 31 starts. In his career so far, Joe is 63-54, 4.21 ERA.
Not great, not horrible. What my husband would call "serviceable". Maybe not so bad as "mediocre", but somewhere between "OK" and "good". Since when is this kind of performance worth that kind of money?
This just in, the Phillies have signed Blanton to a three-year, $24 million contract extension. No specifics on the yearly breakdown, so it averages out to $8 million per year. Still not sure if I think this is what Blanton is worth, but at least it's closer to what the Phillies were offering, instead of what Blanton was asking.
Cliff Lee, on the other hand, would have been making $9 million this season, had the Phillies not traded him. Yeah, I'm still perturbed about that. Cliff's career numbers are 90-52, with a 3.97 ERA, and a Cy Young award to his name. And let's not forget his postseason performance!
So suffice it to say that I think Lee is the superior pitcher, yet we traded him and kept Blanton.
And now I know why.
Over the holidays, my brother-in-law revealed that my nephew has some pretty crappy karma when it comes to getting jerseys of popular Phillies and Eagles players. Quite often, said player ends up leaving the team, via trade or free agency. This happens more frequently than mere chance would seem to suggest.
So what did they get him this year? A Cliff Lee jersey!! And we all know what happened next.
*sigh* If only they'd gotten him a Blanton jersey instead.
(photos by me)
14 Comments
Hey, if you get him that Blanton jersey, perhaps he'll have to settle for the lower arb number–and then he can be traded! Hmmm!…;)
That Cliff Lee trade is still baffling to me, also. That had me scratching my head. I guess your FO couldn't stand prosperity (i.e, Lee and Halladay).
Your nephew and I both😦
So i'm gonna pass on t-shirts or anything that has to do with players' names unless I want the player to leave lol
If it makes you feel better we still have Gary Matthews Jr and his 20 something million dollar contract😛
With the way the economy is now, it is a surprise that the salaries are that high. I'm sure that the revenue from the season tickets is down which is an indication. I would have thought that Cliff Lee would demand more than Blanton. In fact, I would have been thrilled to see Lee in Dodger blue!
Greg – All I can say is that goodness this is NOT the same nephew that I bought the Halladay shirt for Christmas. Though it is his brother; I hope this bad karma isn't a genetic thing.😉
Mimi – Just have someone give you a Matthews jersey as a gift! (assuming you think they should unload his big contract) I'm always stumped by some of the big numbers some not-so-big players end up getting – they must have really good agents! – but the reality is they are now basically untradeable, no other team wants to pick up their contract. Aargh!
It's all your nephew's fault!!!!! LOL. I wouldn't be over losing Cliff Lee either, although having Halladay would certainly ease the pain. But can you imagine a rotation with both of them in it? Too amazing to contemplate.
Hmm, comments are coming up in a weird order again.
Cat – Though some teams' ticket sales may be down, I'm pretty sure the Phillies' are way up after the success of the past two years. I guess it's a small consolation that Lee went to an AL team, at least we won't have to face him this season.
Jane – That's the problem, I keep imagining what could have been. But yes, I'm very happy they got Halladay. Now if only Hamels can return to his 2008 form.
Is he an eight million dollar man, though? That really is the question.
Sounds about average performance for a D-Backs starter…and an average of a quality start? I'd take him on my team. Maybe not for 8 million dollars, though.
Congrats on #63!
See? that is why I do not get current player's jersey. I make exeptions with t-shirts though. I love my Jackie Robinson's jerseys.
Dodgers just signed Padilla. He behaved with us and pitched well so I like the signing
I'm gonna go ahead and get on the bandwagon of askin' "Why not keep Lee and Halladay and dump Blanton if need be?" Not sure Blanton is worth that much. Would have rather paid Lee that for a year.
Caroline – I still think $8 million is a bit high, but he's a solid middle-of-the-rotation guy, so I can live with it.
Emma – Yep, this is also why I have a generic jersey, with no name or number on the back. Also, it's too hard to pick a favorite – I support all my Phillies, and I can't afford to buy a jersey of each one! Good luck with Padilla this year, I hope he works out for the Dodgers.
Jeff – I think most Phillies fans would have rather paid Lee $9 million as opposed to Blanton's $8 million. I'm pretty sure they tried shopping Blanton before Lee, but there wasn't any interest out there (gee, what a surprise).
I know the whole jersey feeling. I got a Trot Nixon #7 jersey back in 2003. Luckly, it's a Boston home jersey, so there's no name on the back. Later, I got a Curt Schilling road Boston jersey. He retired a few years later. Then, I got a John Smoltz Boston road jersey, only for Boston to get rid of him a few months later. Why this happens to people, I have no clue. Now I'm stuck wearing the home #7 jersey to games because I'm afraid to get another jersey!!
It is tough buying a players jersey. I don't think i'll buy another one because of that reason. Nice post. (Lee would be a good example for your brother and me) http://clevelandbaseball.mlblogs.com/
Joe Blanton=$10 million dollars? I am quite confused as well. New post🙂
ive made the same jersey mistake as well. opted to go with an Ian Snell jersey over Zach Duke. Snell is now a Mariner and Duke has just signed an extension with the Bucs


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text:
Home List of Titles The SLUGGS survey: calcium triplet-based spectroscopic metallicities for over 900 globular clusters
Please use this identifier to cite or link to this item: http://hdl.handle.net/1959.3/236604
- Title
- The SLUGGS survey: calcium triplet-based spectroscopic metallicities for over 900 globular clusters
- Author(s)
- Usher, Christopher; Forbes, Duncan A.; Brodie, Jean P.; Foster, Caroline; Spitler, Lee R.; Arnold, Jacob A.; Romanowsky, Aaron J.; Strader, Jay; Pota, Vincenzo
- Abstract
- Although the colour distribution of globular clusters in massive galaxies is well known to be bimodal, the spectroscopic metallicity distribution has been measured in only a few galaxies. After redefining the calcium triplet index-metallicity relation, we use our relation to derive the metallicity of 903 globular clusters in 11 early-type galaxies. This is the largest sample of spectroscopic globular cluster metallicities yet assembled. We compare these metallicities with those derived from Lick indices finding good agreement. In six of the eight galaxies with sufficient numbers of high-quality spectra we find bimodality in the spectroscopic metallicity distribution. Our results imply that most massive early-type galaxies have bimodal metallicity as well as colour distributions. This bimodality suggests that most massive early-type galaxies experienced two periods of star formation.
- Publication type
- Journal article
- Research centre
- Swinburne University of Technology. Faculty of Information and Communication Technologies. Centre for Astrophysics and Supercomputing
- Source
- Monthly Notices of the Royal Astronomical Society, Vol. 426, no. 2 (Oct 2012), pp. 1475-1495
- Publication year
- 2012
- FOR Code(s)
- 0201 Astronomical and Space Sciences
- Keyword(s)
- Abundances; Galaxies; Globular clusters; Star clusters; Stellar content
- Publisher
- WIley-Blackwell
- 0035-8711
- Publisher URL
- Copyright
- Copyright © 2012 The Authors. Monthly Notices of the Royal Astronomical Society copyright © 2012 RAS.
- Peer reviewed


text:
Kerhonkson electrocuted in camper
On Wednesday, July 24, 2013, the New York State Police at Ellenville responded to an Ulster County 911 report of a subject that had been electrocuted in the Town of Rochester NY.
State Police responded to the area of Schwabbie Turnpike in Kerhonkson, NY and found JOHN A LASOSKI, age 53, unconscious. Members of the Kerhonkson/Accord Rescue Squad administered life saving measures and transported Lasoski to Ellenville Hospital where he succumbed to his injuries.
The investigation revealed that a large tree had fallen across the roadway and had taken down the power lines in its path. The fallen power lines had energized a camper that Lasoski was in. When he exited the camper, he touched a metal support, causing a ground, which then electrocuted him.
The State Police were assisted at the scene by Accord Fire Dept and Kerhonkson/Accord Rescue Squad.


text:
Monday marks Square Root Day, with both the day of the month and the month being the square root of the last two digits of the year, or 4/4/16. The previous square root day fell on March 3, 2009 (3/3/09), with the next one coming on May 5, 2025 (5/5/25). As you let that sink in, check out some other intriguing dates that have been the source of fascination and speculation.
Pi Day -- This day falls on March 14 every year, thanks to the date being shortened to 3.14 or the first three digits of pi. The day was extra special in 2015, as adding the year stretched the symbolic celebration out a little longer to 3.1415. If you want to take it even further, 9:26 a.m. (or p.m. if you're not observing military time) on Saturday, March 14, 2015, marked 3.1415926, or the first eight digits of pi.
Odd Day - These days have consecutive odd integers, with last year's Nov. 13 (11/13/15) being the last Odd Day this century. Only six such dates occur each century.
Palindrome week - This marks a time when each date can be read symmetrically backward and forward (i.e. 5/10/15, 5/11/15, etc). The next such palindrome week will occur in June 2016 (6/10/16-6/19/16).
9/29/29 – Math lovers share an affinity for palindromic calendar dates, which can be read symmetrically backward and forward in their numerical date format. But Sept. 29, 1929 must have been a banner day for number crunchers as it's one of the few dates that have more than one palindromic form: 9/29, 9/29/29 and 9/29/1929.
1961 – Few can say they've lived in a perfect year, but for those around in 1961, congratulations. Those alive in 1961 did something no one else did in the last century, and that no one else will do for 4,000 years: read your year upside down or right side up. While there's no particular date that stands out as odd that year, the fact that this calendar anomaly won't occur again until the year 6009 may lend credence to the notion that the '60s were a time of new perspectives.
12/31/99 – The Prince song "1999" enjoyed new life as the world amped up massive celebrations in preparation for the new millennium. There was widespread fear that the world's data storage systems would fail, since many computers were programmed to handle only two-digit year dates. People thought the "Y2K bug" would shut down the world's computers and data storage systems. It didn't.
04/05/06 – Fans of odd calendar dates and other geeks were tickled by this date and time: 1:02:03 on 04/05/06.
06/06/06 – The Bible's Book of Revelation calls 666 the number of the beast, so more than a few eyebrows were raised as June 6, 2006, approached. In the days prior, the evangelical Raptureready.com website said the day had a "rapture index" of 156, suggesting that it was "fasten your seatbelts" time, The Associated Press reported. Others cashed in -- moviemakers released the remake of the horror film "The Omen" on the date.
07/07/07 – Couples flooded Las Vegas and overwhelmed wedding chapels on what Time magazine predicted might be the most popular wedding day ever.
08/08/08 – Opening ceremonies for the 2008 Summer Olympics in Beijing, China were held on Aug. 8 at 8 p.m. The number eight is considered auspicious in Chinese culture and traditions because its pronunciation sounds similar to the words for prosper, wealth and fortune in various regional dialects.
09/02/10 – A celebrity-studded gala event was held in Beverly Hills for what fans of the hit 1990s show "Beverly Hills, 90210" called International 90210 Day. Fans formed a Facebook page to lobby TBS for a show marathon and President Barack Obama for an officially recognized day. Both bids failed.
10/10/10 – "Binary 42 Day," or "Decimal Day," as some dubbed it, didn't have the negative connotations attached to it that other mathematically distinctive days seem to have. However, some embraced the uniqueness of this numerically divisible day to foster change in their environment through political action. A Global Work Party was held on this date by groups seeking climate solutions.
12/21/12 – This date was supposed to mark the end of a 5,126-year era in the Maya Long Count Calendar, but there are varying opinions on what the culmination of this date really means to humanity -- and to the ancient Mayans. While there have been a wave of books and movies depicting a coming apocalypse, some scholars say the Mayans foretold no such doomsday. However, astronomers did predict that for the first time in about 26,000 years, the sun would be aligned with the center of the Milky Way on that date.
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Scarlett Johansson through the years
Published On: Nov 21 2011 01:30:49 PM PST Updated On: May 05 2016 08:18:59 AM PDT
As Scarlett Johansson stars in "Lucy" take a look at images of the actress throughout her career.
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text:
Skip to comments.Formal Reprimand Issued To Flatulent Federal Worker
Posted on 12/21/2012 4:24:30 PM PST by rightwingintelligentsia
DECEMBER 21--A federal employee was formally reprimanded this month for excessive workplace flatulence, a sanction that was delivered to him in a five-page letter that actually included a log of representative dates and times when he was recorded releasing the awful and unpleasant odor in his Baltimore office.
In a December 10 letter accusing him of conduct unbecoming a federal officer, the Social Security Administration employee was informed that his uncontrollable flatulence had created an intolerable and hostile environment for coworkers, several of whom have lodged complaints with supervisors.
The worker, a 38-year-old Maryland resident, reportedly submitted evidence that he suffered from some medical conditions that, at times, caused him to be unable to work full days. But a SSA manager noted in the reprimand letter that, nothing that you have submitted has indicated that you would have uncontrollable flatulence. It is my belief that you can control this condition.
(Excerpt) Read more at thesmokinggun.com ...
And we wonder why people go on workplace shooting sprees.
Maybe he could join the Army and they could use his gift to gas out the enemies and cause them to surrender.
He forgot to say "Here, pull this." We humans never cease to amuse.
Federal Worker Takes "Farting Around" To A Whole New Level.
The pic at the link is of he and his "wife" with Pepe Le Pew? Looks like two guys.
"And just what is it you DO do?"
He probably eats menudo.
Otherwise, he was completely normal, married, kids and all.
A true stinking job environment!
It was in Baltimore. How could you notice the smell over the general stink of the City? Lots of mayors and local politicians in jail. Lots of crime. Lots of corruption.
That is the real "stink" of Baltimore, not one guy with bowel problems.
Did he carry around an aerosol to offer a courtesy stray before he fired? ;-)
The condition is probably "meteorism". Adolph Hitler had the same condition.
Off we go into the wild blue yonder,
Climbing high into the sun;
Here they come zooming to meet our thunder,
At 'em boys, Give 'er the gas! (Give 'er the gas now!)
Down we dive, spouting our flame from under,
Off with one heckuva roar!
We live in fame or go down in blue flames.
Hey! Nothing'll stop the U.S. Air Farts!
Says that he's lactose intolerant but will get gas-X to help out.
I have a child who is lactose intolerant and avoids anything that cause the incredibly foul gas that results.
A steady diet of Fiber One bars will do it
Maybe he should stop going to Taco Bell for lunch.
Sadly, I never worked for a company that would award such an employee performance. We did have a co-worker who was a little smell challenged. The boss allowed an expense account reimbursement for the one who bought all the soap and deodorant ...
I agree.
I wonder if his wife is female.
Also follow the 2 per person per visit limit!
This guy is no fool. A couple of more farts and he will be on Social Security disability and laughing every month when the check comes in.
I used to know a guy who could clear out an entire movie theater with one blast. He claimed to have some disease he picked up in Viet Nam.
I saw a documentary just a while back, a Dr said of Hitler "He ate only vegetables, took opiates at night (they cause constipation) and Methamphetamine during the day..No wonder the man had wind!"
There was an episode of an old TV show I liked ("Dead Like Me") where an office worker had uncontrollable flatulence..it was a hoot! (or maybe a toot)
Two of them Fiber One bars for breakfast and I'm alone the rest of the day.
What constitutes "excessive workplace flatulence"?
"OK, Gene, here are the federal rules on farting. You can have three pants-splitters a day. You are only allowed one skunk-squeezer. And nothing that will leave a brown stripe in your underwear. Now, this does not include the state farting laws of Maryland, which, I'm told, really stink."
Or one of these;
High fiber diet, a little metphormin and sugar free cough drops. clear the room boys!
When are we going to see reprimends for Federal employees who are full of sh*t?
Looks like he's already done some workplace shooting.
Wonder if any of the incidents were like this;
I read a piece some time ago by someone who was present during his rants where he would be screaming with spittle flying and machine gun farting going on all at once. And of course, one wouldn't dare laugh.
Obviously he should either be the elevator operator OR restroom attendant.
He needs 'people exposure' to ferret out the weaklings and force them to retire, thereby saving the Govt money.
:Here I sit, broken hearted,
Paid to $hit and only farted:
Indian food will carve you a new @$$ hole!...
An in-law, while in England during WWII, was coming back to base after a night of leave, and was arrested for relieving himself on the side of the road. I wish we had a copy of the citation which read:
"Urinating on the King's Highway"
Work place flatulence?...Just fire back with a few bad Mexican burritos!...Don't get mad get even! Just start your own Air Biscuit Air Force!
When I was stationed on Guam in 1956 one of the Marine guards at the main entrance of the main Navy base was written up by an Officer's wife for farting on duty. The document said something like, "I observed Cpl..... lift his leg break wind and say aah."
>>> "I read a piece some time ago by someone who was present during his rants where he would be screaming with spittle flying and machine gun farting going on all at once. And of course, one wouldnt dare laugh."
Thank you for the visual. Oh, my achin' sides (from LAUGHING, dangit)!
(Quoting Dave Barry) "Bean burritos could help explain why most of Mexico is located outdoors."
Is this really serious? Not satire?
I can tell you Gas-X is not going to work. Maybe Gaviscon (I haven't tried, although I think it's more stomach-centered than intestines-centered). I have lots of chronic digestive disease, and occasionally I have gas inside so bad it's really painful. Gas-X relieves that, if you know what I mean.
Actually the SS complex is literally west of the edge of real Baltimore. Our family friend lives down the street from it, and not much further is my aunt. Although I must say Woodlawn generally is not a real good place; takes the color of the worst of Baltimore.
ping for MD
No wonder Hitler was so anti smoking. One match and the whole room could explode.
This whole story stinks.
Maryland "Freak State" PING!
My 1st "pro" job I had some pals down the hall in the "big office", with 4 people. The oldest of them (maybe 50s) was infamous as a flatulator. I didn't know this until I asked what the deal was with all of them having dust masks hanging from their lamps. That's when I found out they were symbolic of using protection against Tom and his frequent outbursts.
What is the workers name? Gaseous Clay? Fartholomew? Stinky Pete?
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INGREDIENTS:600 Tyson Fully Cooked Crazy Chipotle Drums, frozen, (approximately 10 cases) 8 fresh pineapples, whole, trimmed and cored, commercially prepared Pineapple Curry Dipping Sauce: ¾ cup curry powder, ground 2 15-oz. cans Coco Lopez Cream of Coconut, commercially prepared 2 ¾ gallons pineapple sweet & sour sauce, commercially prepared 1 quart lime juice, freshDIRECTIONS:Place chicken on foil-lined sheet pans. Heat in preheated conventional oven at 350°F for 35-45 minutes. Or heat ...
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Shooting victim was getaway driver
West Virginia State Police on Wednesday identified the man shot by a Parkersburg police officer after a purse-snatching at the Grand Central Mall Tuesday afternoon as 24-year-old city resident Steven Lewis Pfalzgraf.
Today, Parkersburg Police Chief Joe Martin plans to release the names of the officers involved in the incident.
"I just want to get them prepared mentally to deal with the effects of people knowing who they are and what happened to them," Martin said.
According to West Virginia State Police 1st Sgt. Michael Baylous, Pfalzgraf, last known address of 604 32nd St., Parkersburg, was driving a car into which John Ragalyi, no age listed, of New Matamoras, entered after allegedly stealing a purse from a woman in the mall food court shortly before 1 p.m.
The alleged theft was observed by two Parkersburg Police officers who were in the food court participating in an observation exercise for a surveillance class through the Homeland Security Office. Baylous said the officers identified themselves as police and ordered the suspect to stop. He did not, running to the parking lot between the food court entrance and J.C. Penney.
Ragalyi got into the car, in which his wife, Jenna, was also a passenger, Baylous said. One of the officers reached in and tried to remove the key from the ignition, which Martin said is not an uncommon tactic.
"That's a very good way of immobilizing a vehicle," he said.
Pfalzgraf then allegedly put the car in motion, dragging the officer along. After the officer was freed, investigators say the driver pointed the vehicle at the second officer and drove forward. At that point, Baylous has said, the car was a weapon, threatening the officer's life.
The second officer shot multiple times at the car, with at least one shot striking the driver. The car continued onto the outer road circling the mall lot. Eventually, it stopped in the Toys "R" Us parking lot.
The Ragalyis then reportedly fled. Pfalzgraf was found in the car and pronounced dead at the scene.
Martin said a witness' statement that a police car employed a pit maneuver- striking the suspect car to cause it to spin out of control- was inaccurate.
"That did not happen," he said.
Multiple area law enforcement agencies were participating in the Homeland Security training, but not the State Police, who were tasked with investigating the incident.
Martin said everything he's heard from the investigation so far leaves him confident his officers acted appropriately.
"Ultimately the goal of the police officer is to stop the threat or apprehend the person that's committed a crime," Martin said. "I'm glad they chose to take action. It's unfortunate it turned out the way it did."
The department will still conduct its own internal investigation of the matter.
The Ragalyis turned themselves in to Parkersburg Police Tuesday and were released pending a decision by Wood County Prosecutor Jason Wharton on whether felony or misdemeanor charges would be filed.
Neither Pfalzgraf nor the Ragalyis had prior criminal records in Wood County Magistrate Court; however, both the Ragalyis have faced charges in Washington County.
John Ragalyi has been in the Washington County Jail multiple times, Sheriff Larry Mincks said. In 2007 he pleaded no contest to a misdemeanor theft charge and was sentenced to 10 days in jail. In 2008, he was charged with drug trafficking but those counts were subsequently dismissed. He's also been taken to the jail four times on probation violations.
Jenna Ragalyi has a misdemeanor assault conviction on her record.


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I'm not sure why Lamborghini bothered hauling a bunch of media down to Miami, Florida last weekend to help promote its new Avantador Roadster. Apparently, the 700 horsepower open-roof version of its $450,000-plus flagship Aventador V12 supercar is sold out until mid-2014. If you're reading this, and you're one of the lucky (and obviously wealthy) few who have already put down a deposit for a Roadster and are waiting patiently in line to get your hands on one, it must be comforting to know that your cars are being "broken-in" for you by some of my colleagues…
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Analysis of policy issues related to Infrastructure and Human Development in Bangladesh using the MAMS MDG version. The analysis is carried out by Yeasmin Sayeed and Jörgen Levin, School of Economics Örebro University, as part of Yeasmin's Licenciate dissertation in Economics at Örebro University under the supervision of Dr. Jörgen Levin. It is expected that the dissertation will be completed in 2013.
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Last updated: 2011-07-15


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The face of Honolulu is changing. New developments are coming up and old businesses are getting a facelift and a name change.
Real estate experts say it's a positive move, but warn we need to remember the needs of the future.
If a possible sale goes through, condominiums will be built behind the historic Honolulu Advertiser building.
Kamehameha Schools and developer Peter Savio will renovate and sell units at the Pagoda Terrace as fee simple condominiums for low-to-middle income working families.
Residential buildings are coming up or being proposed all around Honolulu and real estate analyst Stephany Sofos says it's a good thing.
"The kupuna need care, students need housing and lower and middle income homes are desperately needed in Hawaii," said Sofos.
Sofos says taking care of the immediate housing needs of today is important and positive.
But, she cautions -- while it is very good for today, all the development could cause problems in the future.
"We're going to build all of this nice housing have all of these people in the core of Honolulu, but our sewer system hasn't been upgraded, our roads haven't been upgraded, our electricity hasn't been upgraded," said Sofos.
On the commercial side, Sofos is pleased to see the old buildings being given new life.
Petco and Walgreens signs are up at the old Beretania Street Safeway.
Medical buildings across from McKinley High School are slated to become Longs Drugs, while the old Longs Drugs store in Moiliili is being turned into Ross.
"They're re-energizing previous product and that saves them a lot of money and that is regenerating the neighborhood, which is a great thing," said Sofos.
Sofos says some of the developers may have been holding on to money, waiting for the right moment to act. Others are finding money available now through financing.


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I can't see the poll from my phone but I'd vote for Iman if he's up there. Every great team needs one to two lockdown defenders. I don't expect him to be the same player he once was in January, but I would hope he could provide some sort of a defensive spark.
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CCSS Progression Activity
Intended Audience:Teachers, Administrators, Professional Development Coordinators
This activity allows participants to gain an understanding of the integral role each grade level plays in preparing students for college and career readiness. This activity illustrates the progression of standards throughout the grade levels and highlights College and Career Readiness as the goal of the Common Core Standards. The use of backwards design when developing the standards is emphasized.
Suggested Use for these Documents:
By highlighting a single standard and noting the differences that occur from grade to grade, professional development leaders may use these printable charts to lead an activity showing the progression of the Common Core Standards from kindergarten through 12th grade.
Materials Necessary:
- Progression Activity Cards Literature OR
- Progression Activity Cards Writing
- Progression Activity Powerpoint
Send questions and comments to:
Resource Links:
Return to Common Core Professional Learning Series Home Page
This work is licensed under a Creative Commons Attribution-ShareAlike 3.0 Unported License


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• Glitter and Glue , Kelly Corrigan: The memoir by the author of The Middle Place offers a heartfelt examination of motherhood from all sides.
• Other notable releases: fiction — Somerset, Leila Meacham; The Wily O'Reilly: Irish Country Stories, Patrick Taylor; nonfiction — The Triple Package: How Three Unlikely Traits Explain the Rise and Fall of Cultural Groups in America, Amy Chua and Jed Rubenfeld
• Dallas Buyers Club (R): In the inspiring real-life drama, a Texas cowboy (Matthew McConaughey) with HIV seeks alternative treatments and unites a band of outcasts in a struggle for life and dignity.
• Other notable releases: Escape Plan, Justice League: War
• Fable: Anniversary (Xbox 360): Vivid graphics enliven the role-playing classic, which includes 1,000 achievement points to challenge veterans and newcomers.
• Other notable releases: Bravely Default (Nintendo 3DS); The Lego Movie (Nintendo 3DS and Wii U, PlayStation 3 and 4, PlayStation Vita, Xbox One, Xbox 360); The Sims 3: Roaring Heights (personal computer)
• After the Disco , Broken Bells: The side project of indie-rock songwriter James Mercer (the Shins) and alt-hip-hop producer Brian "Danger Mouse" Burton returns with a second effort.
• Other notable releases: The Satanist, Behemoth; From His Head to His Heart to His Hands: An Audio/Visual Scrapbook, Michael Bloomfield; Love Marriage & Divorce, Toni Braxton & Babyface; Kin, Pat Metheny Unity Group; The Royal Sessions, Paul Rodgers; Hydra, Within Temptation
— From staff reports


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April 17, 2013 —
PORT JERVIS, NY — UpFront Exhibition Space will hold its sixth annual spring artists reception and exhibition on April 20 from 6 to 9 p.m. At the reception, UpFront will present live music by Walt Edwards along with complimentary food and refreshments.
UpFront currently has 40 artists of all mediums included in this exhibit including nine new artists
The public is invited to attend this event at no charge. This show will run through Saturday, May 31. UpFront Exhibition Space is located at 31 Jersey Ave. Gallery hours are Thursday to Sunday 12 noon to 5 p.m. Call 845/856-2727.


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Sorry but I don't think IT visit's these boards... and if he does, he prob wont answer
I like this team, but we are not setting ourselves up for the future. Isiah amazingly brings in talent, but each year it is getting harder to make bigt deals. With each trade he has brought in an overpaid player with a contract no other team will touch. This is a temporary fix to a growing problem. Our payroll is spread out to far into the bench. Guys like Jerome James and Malik Rose who won't see regular playing time are costing our team 15million this season! They won't even see the floor very often! And if Rose does see the floor it comes at the expense of Chandler, who could be using that time to develope. I respect Rose. He is a good player, just not for the money and he's to far down on depth chart to make an impact anyway. Rose would only play garbage time, the time most teams allow for rookies and works in progress. So not only is he expensive, he is clogging up the development of our young players. I admitt he is a good influence on young players and in the looker room, but that is a coaches job, not a players to influence and mold young players. Besides how much influence does he have when he doesn't even see the floor, how much do young players respect a guy who rides the pine? Jerome James, everyone should realize he has absolutely no value at all. He is not a mentor, role model or talent for anyone to admire or respect. Buying him out would be expensive, but worth it. If we had bought him out and signed Nichols, we could play and develope Nichols in garbage time, the only time James would ever see the floor, and it would have cost the Knick only about 1.6million more than letting Nichols go. (800k slaray 800k luxary tax. What we pay James now and what we would pay him if we bought him out is basically the same, so the only additional cost would be Nichols salary and luxary tax...about 1.6million and we'd have a future star or a valuable role player or a bust, but it would only have cost 1.6 extra and wouldn't really effect the cap. And now all we have is jerome james an obvious bust, who still gets paid. Heck we could have pretended he is still on the roster if it would make Isiah feel better, he will play just as much.
Last edited by jzero29; Nov 06, 2007 at 14:59. Reason: spelling
Let's...Go...Lottery! Let's...Go...Lottery!!! Say it with me Knicks fans!
Sorry but I don't think IT visit's these boards... and if he does, he prob wont answer
haha your very funny. I know Isiah doesn't read these forums, I am venting. Because I don't understand what he's doing, I am hoping other knicks fans read this and input about what they think or believe or want to vent.
Let's...Go...Lottery! Let's...Go...Lottery!!! Say it with me Knicks fans!
i didnt read the whole thing but i read enough, once i read we could use chandler to develop this year, man forget developing right now, its all about wins, and its too bad chandler not playing but wins first
You hit it right on the head. The orginzation, media, and fans are all tired of losing. Everybody needs to see a winning record atleast to give us the illusion were moving in the right direction.
I don't know if anybody listens to Stephen A Smiths radio show but he mentions atleast once a day that in 3 years Lebron is going to make a choice between Brooklyn or the Knicks. We'll still have money problems but Marbury, Rose, and James would all be off the books. And we'd have potential trade pieces like Lee,Nate,Mardy, chandler, and any other draft picks we select in the upcoming years.
Let's...Go...Lottery! Let's...Go...Lottery!!! Say it with me Knicks fans!
yp stop talking about IT dont u got something better to do he got to wins already and he did good in the preseaon so when he dont get the wins then u could if no shut up
Did U See What He Did In The End Of The Game He Now Doing His Job Is Not That I Like How He Do It But He Is Geting Better So Dont Talk All Sa **** Ya Been Talking O Yeah We Should Bring Patrick In The Staff Some Where
da 0.2 seconds thing that shir was funny made everbody come back i know the denver coach was mad and yo i dont know about u but you gotta remember if he dont make the playoof he is gettong fired just like joe torre **** in da end of it pat we be the new coach but he if he does good he stays so let see is he does a good job and stop talking about him that much cuase is kinda hard because his good teams in our division like clevend boston and miami so chill
So let me get this straight, I don't understand what your saying your against? Why you object to developing Rookies and why I don't understand why ROse and James are on the roster instead of Nichols? your saying in your first post, that rookies shouldn't play at all and wins should come first and forget developing. I agree with wins part, but you can't forget about developing players, lee and balkman would never have played, they would never have gotten chance to display talent if they didn't get to come off the bench and play, in blowouts and other times. That was a different team then and they were losing a lot. So now that we are starting to win, I don't think we should play around with chandler during crucial times. I think he should come off the bench only after the game has been decided(aka garbage time) This gives Chandler time on the floor to develope and play against NBA players. Much like what the Nets did with A.Wright.
In your second post you say, that you can't develope players in garbage time either. Are you saying chandler should not step on the floor all year? If you don't want to play him when the game is on the line and you don't want to play him durring a blow out(knicks winning or losing) when do you play him? You know you don't play starters in blow outs for risk of injury, so you'd rather play Jerome James or Malik Rose then Chandler? Further more I was bitching about even having a guy like malik rose and Jerome james(even if healthy) who wouldn't/shouldn't see the floor except in blowouts or several injurys. Why not have Demetris Nichols instead? If you've notice, Isiah has been playing 8 guys, small rotation, much like many other NBA team. Other Nba teams only let rookies play minimal minutes, these minutes(comming mostly in garbage time). Having Jerome James take up or Malik rose is a waste. GUys like Chandler and Nichols should be getting those minutes. Yes I know nichols is gone, it is part of why I don't get what Isiahs doing, why have two players that cost 5.8 and 7.1 mill, take up a spot on the bench and who are not productive memebers, why not have a rookie on the bench, and when you would put jerome james in the game or malik rose, put in those rookies. Because If you've been watching, Rose and James aren't playing, he will at some point, but I don't think it will be during a crucial part of a game, unless their is an injury or several players have foul trouble and it won't be for long. Why not put a rookie in for the small amount of minutes to DEVELOPE them. DO you think those times are so important that you should have James and Rose in? Like it will make a difference having them? Why not keep young talented players and let washed up vets go play with their millions.
This has always been a problem with Isiahs knicks, bench is to deep and to expensive. Do you really need a guy who makes 7.1mill this year(Rose) and plays only 12.8mpg(will play evenless this year) or a guy who earns 5.8million a year (Jerome James)and plays only 6mpg (Won't play at all this year)
These guys are wastes on the roster, they are expensive and won't even play except for garbage time or 2-3 minutes during a regular game, where I'd rather have a rookie playing.
DO i think Rose is a better NBA player than Chandler, Yes i do, but the amount of important time rose will play this year is minimal and in that little time do i think Chandler or a guy like Nichols would be that big of a drop off to affect this team? No. So why not Go cheap and have the rookies instead of the expensive players. We could of have another diamondd in the rough (balkman) in Chandler. Again, because you don't seem to understand, I'm not saying start chandler or even have him come of the bench before other guys that arre currently playing. I'm saying why even have Rose and CHandler on the team. Buy them out, maybe they'll take a buyout for less than full value of contract, saving the knicks money that they would have paid them for doing nothing but riding pine. Then they could have kept some of the rookies that they let go and maybe next year or the year after that they start to be productive.
Let's...Go...Lottery! Let's...Go...Lottery!!! Say it with me Knicks fans!


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This print started out as a baseball card more than twenty years ago. Every aspect of it is designed to preserve the original look and feel of the card, from the printing pattern to the slightly worn edges. In fact, this print started by finding the actual card in our vault and then painstakingly re-mastering it with a team of designers. The authenticity extends to manufacturing; each print is made on high-quality, cotton fine art paper that measures 1/64 inches in thickness. Framed prints feature 2-inch flat wood or metallic frames with high-end, glare-resistant glass. And all prints are officially licensed by MLB and the National Baseball Hall of Fame.
STORYMike Schmidt was already bound for Cooperstown. He had hit 500 home runs and been named to more than a decade's worth of NL All-Star teams. Schmidt was coming off his last season ever hitting more than 30 home runs and receiving MVP votes when this card was originally released. As a print, you can get a close-up view of the fight that Schmitty still had in him toward the end of his career.
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I guess I could always call my OB, but I'm not sure if my symptoms warrant a visit.
2 days ago my one side of my right breast was very hard. I had this before in my left breast and I was told by my OB to nurse like crazy and it should go away. Following the same advice, I nursed on the hard right breast and it eventually cleared up. This time however, I felt intense nipple pain during nursing but it somewhat goes away after the first 5 mins. The pain is only at the nipple. I examined my nipple and notice that there was a bit of discoloration near one of the ducts. It's a bit white but it's very small. I instantly thought of thrush, but my nipple doesn't have the characteristics of thrush except for this white bit, and it's quite small, about the size of a pin head. Also, LO doesn't have any symptoms of thrush and nurses quite happily. His latch is also good.
I also noticed a dip in my supply with this breast as well. I was only able to pump half the amount I used to (I checked the pump and membranes, all checked out fine).
Does this sound like thrush?


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On Monday, September 30, the Health IT Policy Committee's Privacy and Security Tiger Team held a Virtual Hearing on Accounting of Disclosures, which is a listing meant to show patients all disclosures of their personal information that are made by a HIPAA-covered entity. On behalf of PPR, Dr. Peel provided written testimony that details the importance of implementing robust AODs, as well as recommendations for quick implementation using existing health IT and meaningful use requirements.
Read the full comments here.


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You Are Here: Home » News From NINDS » Proceedings »
DMRF/NINDS Dystonia Workshop - "From Gene to Function in Dystonia"
Chairs: Xandra Breakefield, Laurie Ozelius, Phyllis Hanson, William Dauer
January 19 - 21, 2001
Phoenix, AZ
Overview: Xandra Breakefield
The discovery in 1997, of the TORA1 (DYT1) gene, responsible for early onset torsion dystonia, has catapulted dystonia from the realm of neurology and human genetics into protein structure/biochemistry, cell biology and animal models. Unusual aspects of the disease include: the childhood window of susceptibility during a period of motor learning; low penetrance of a dominant disease gene; and apparent lack of frank neurodegeneration. These suggest a novel mode for neuronal dysfunction with important implications in basic neuroscience. A large number of genes (up to 14) underlie various forms of the hereditary dystonias and indicate a broad and dynamic field of players subserving the control of fine movement within the basal ganglia. Biochemists are intrigued as the encoded protein, torsinA, is one of the few members of the AAA+ chaperone family of proteins involved in a human disease and apparently the first to reside in the endoplasmic reticulum (ER). In fact, the torsin gene family first appears in evolution with the emergence of multicellular organisms. The mutation (GAG deletion) responsible for most cases of early onset torsion dystonia is predicted to disrupt ring formation necessary for protein function. The tendency for the mutant protein to form large, whorled, ball-like inclusions when expressed at high levels in cells and the apparent involvement of one of the nematode homologues in nuclear rotation suggest a role for the torsins in membrane dynamics critical to neuronal development and function.
Several themes were raised throughout the meeting, which may provide clues for torsin function. First, many convergent lines point to a relationship between dystonia and Parkinson's disease (PD). This includes the fact that many forms of dystonia present with parkinsonian features and, conversely, that early onset PD frequently presents initially as dystonia. Lewy bodies found in the brains of PD patients contain both a-synuclein and torsin. Dopaminergic neurons in the substantia nigra have a critical role in both diseases and express high levels of torsin A and a-synuclein, which has been implicated in the etiology of PD. Homozygous mice with the A53T alpha synuclein mutation manifest with dystonic-like movements. A second theme was that defects in mitochondrial function due to mutations in both mitochondrial and nuclear genes can cause dystonia, as attested by Leber's optic atrophy and deafness-dystonia. Interestingly, the mammalian protein with highest homology to the torsins, SKD3, is a mitochondrial protein that increases in response to oxidative stress. Third, signs point to a number of neuronal types which may contribute to the manifestation of dystonia, including expression of torsin in select neurons in the basal ganglia, substantia nigra, cerebellum and hippocampus. Levels of torsin are upregulated in striatal interneurons during a window of development associated with high synaptogenesis, and torsin is found in the adult primate brain at symmetric synapses in the basal ganglia. Understanding how torsin functions in neuronal dynamics will be critical to resolving the disease etiology and possibly in understanding aspects of motor development and function. Torsin may affect membrane trafficking involved in neuronal polarity and integrity needed for synaptic communication.
Genetics - Laurie Ozelius
It was clear from this meeting that the discovery of the gene for early onset dystonia has led to an explosion of work on the TOR1A protein and its role in disease and underscores the need to find other genes responsible for the various forms of dystonia.
With regard to TOR1A, (Laurie Ozelius, Albert Einstein University) a new 18 bp deletion mutation was described that removes 6 amino acids from the C-terminus of the protein including a putative phosphorylation site. In addition, studies aimed at determining genetic factors that might influence the penetrance of early onset dystonia were discussed. Using the simplest model of inheritance (autosomal dominant with a high gene frequency), six chromosomal regions of positivity (multiple positive markers) have been identified on chromosomes 2, 5, 7 (two regions) and 8 (two regions). One of the regions on chromosome 7 and another on chromosome 8 overlap with regions that have been implicated in myoclonus dystonia and adolescent onset dystonia (DYT6), respectively. If genes influencing penetrance can be identified, this may lead to new treatments aimed at delaying or preventing the manifestation of the mutant TOR1A gene in carriers.
Two forms of dystonia, Leber's optic atrophy (Doug Wallace, Emory) and deafness-dystonia (Craig Blackstone, MGH) are caused by mutations in the mitochondrial and nuclear genes, respectively, which encode proteins critical to mitochondrial function. In Leber's, a heterozygous state of a particular mutation in the mitochondrial genome causes blindness, while a homozygous state leads to childhood-onset, generalized dystonia,with varying phenotypes mediated by degrees of blockage in oxidative phosphorylation. The deafness-dystonia gene (nuclear) encodes a protein needed for generation and division of mitochondria within the cell. Overexpression of a mutated form of an interacting gene leads to whorled, membrane inclusions, as seen with mutant torsinA, and long tubular mitochondria.
Several other forms of dystonia are associated with parkinsonism, dopa-responsive dystonia (DRD) is phenotypically similar to early onset dystonia; however some individuals become parkinsonian later in life. This form of dystonia shows a dramatic therapeutic response to small doses of L-dopa which resolves both the dystonic and parkinsonian, symptoms without the side effects usually associated with PD. Heterozygous mutations in GTP cyclohydrolase 1, the rate limiting enzyme in the formation of the co-factor tetrahydrobiopterin, or homozygous mutations in tyrosine hydroxylase, the rate limiting enzyme in the production of dopamine from tyrosine, both cause DRD. Treatment with L-dopa bypasses both of these blocks to the pathway, thus allowing the patients to make adequate levels of dopamine. Rapid onset dystonia parkinsonism is a rare form of dystonia characterized by acute onset usually in hours to days of either dystonia, parkinsonism or both syndromes. Patients with this disorder have only very slight benefit from L-dopa, although levels of the dopamine metabolite, homovanillic acid, is decreased in at least some of the patients.
The gene for this disorder has been linked to chromosome 19 but has yet to be identified. Finally, the X-linked form of dystonia known as Lubag (Andrew Singleton, Mayo Jacksonville) is found only in the Capiz province of the Philippines due to a founder mutation. This form of dystonia usually begins focally but rapidly progresses to generalized dystonia with about half the cases also having parkinsonism. This is the only form of hereditary dystonia known to have degeneration in the brain, including regions of the basalganglia. The identification of the gene for this form of dystonia is being actively pursued.
Other global issues that were discussed mainly focused on the collection of patient material. These included a collaborative effort among investigators to collect and share families with later onset dystonia, to develop a registry of dystonia patients and families that can be shared among researchers, and to coordinate efforts to involve patients and family members in brain donations. Particularly in light of the penetrance issues, the brains of non-manifesting carriers of TOR1A may provide clues as to specific morphologic features related to the disease state.
Torsins and chaperone proteins -Phyllis Hanson
Cell biologists and biochemists are working toward understanding what the protein encoded by the TOR1A gene normally does in the cell and how a single amino acid deletion can change its activity and thereby cause early-onset torsion dystonia. The session on torsins and chaperone proteins at the recent DMRF workshop highlighted progress in this area, and generated enthusiastic discussions making it clear that further developments will not be long in coming.
TorsinA belongs to the emerging and essential family of AAA+ ATPases. These enzymes appear to function in a chaperone-like manner, carrying out noncovalent conformational modification of stable proteins and protein-protein complexes. TorsinA is the first AAA+ ATPase localized to the lumen of the secretory pathway. Based on studies of other AAA+ ATPases, including the membrane fusion ATPase, NSF, and the bacterial protein folding chaperone, ClpB, one can hypothesize that torsinA is a chaperone with specific target(s) in the secretory pathway. Other AAA+ ATPases function as hexameric ring-shaped oligomers, and are only active in their oligomeric state. If torsinA shares these properties, this may explain how a mutation in it would generate a dominantly transmitted phenotype.
This session brought together investigators with diverse backgrounds and interests, addressing questions that ranged from how to produce and study torsinA using biochemical and high resolution structural approaches, to where and how torsinA may function normally in cells and in the brain as a whole. In all cases, efforts to understand how the GAG deletion alters torsinA structure and function were emphasized.
Specific progress included descriptions by Phyllis Hanson (Washington Univ.) and Gordon Rule (Carnegie Mellon) of recombinant protein production for biochemical and structural analysis. Initial attempts in E. coli were only partially successful, and both have now turned to developing eukaryotic expression systems with which to generate active protein. Michal Zolkiewski (Kansas State) joined Hanson in describing AAA+ enzymes that can serve as biochemical and structural paradigms for torsinA, and highlighted the critical role likely to be played by the C-terminal domain of torsinA that contains the GAG deletion.
Benjamin Cravatt (Scripps) characterized the localization and cellular effects of torsinA overexpressed in mammalian cells, showing that torsinA is indeed a glycoprotein co-localized predominantly with markers of the endoplasmic reticulum. Interestingly, GAG deleted torsinA exhibits a strikingly different localization to large spheroid structures. Xandra Breakefield (MGH) used electron microscopy to show that these structures consist of membrane whorls, probably derived from the endoplasmic reticulum. Breakefield showed studies of endogenous torsinA in neuronal cell lines, again demonstrating co-localization with markers of the ER as well as the secretory pathway. She also described the minimal effects of various cell stress paradigms on torsinA expression. P. Shashidaran (Mt. Sinai) described a variety of studies of torsinA cell biology, including a clear demonstration by isoelectric focussing that DYT1 mutant patients express both wild-type and mutant protein in neuronal tissue. Craig Blackstone (MGH) completed the cell biology presentations by describing studies of a protein underlying a non-DYT1 form of dystonia, dystonia-deafness, and described its interactions with the mitochondrial dynamin-like protein, DVLP. Interaction of these two proteins is required for proper mitochondrial fissioning. These studies may provide insight into new ways in which mitochondrial dysfunction can give rise to dystonia.
One of the big mysteries in understanding how a mutation in a protein such as torsinA generates a focal neurological disease is why particular neurons are more susceptible to effects of the mutation than others. A possible explanation for such specificity may be found in the expression of this protein at different levels in specific neurons and at particular times during development. Marie-Francoise Chesselet (UCLA) described immunolocalization studies looking at torsin protein expression over the course of development in the rat, noting a striking increase in the expression of torsinA in striatal interneurons at around P14, the time of the highest level of synaptogenesis in the basal ganglia. This raises the interesting possibility that some of torsinA's functions might be critical during neuronal development. David Standaert (MGH) described in situ hybridization studies, as well as antibody localization studies of torsinA in human control brains. Using a monoclonal antibody he noted the interesting finding that although torsinA expression in the adult human brain is highest in dopaminergic neurons, very little immunoreactive protein can be found in their cell bodies, with most appearing in vesicular structures in axons and synaptic terminals. Finally, Nutan Sharma (MGH) described FRET techniques for improving the resolution of double labelling studies in the light microscope, holding out promise for refinement in studies of co-localization of torsinA with potential interacting partners including a-synuclein, also noted by Dr. Shashidaran.
Animal models - William Dauer
The animal models section of the meeting focused on three general themes: 1) general issues of animal model construction and analysis, 2) etiologic specific models of DYT1 primary dystonia and 3) animal models of secondary dystonia.
Dr. Don Price (Johns Hopkins), Dr. Michael Lee (Johns Hopkins), Dr. Yuquig Li (University of Illinois), and Dr. Alan Peterson (Royal Victoria Hospital) discussed different examples of animal model construction and analysis. Don Price presented on animal models of Alzheimer's disease and highlighted how etiologic specific mouse models can lead to a fundamental molecular understanding of disease pathogenesis, and suggest rational therapeutic targets. He discussed how these mice have helped to elucidate the interactions between presenilin 1 (PS1) and amyloid precursor protein (APP), and the crucial role of beta secretase in APP processing. Importantly, beta secretase knock out mice do not produce any of the normal APP products, a result which suggests that inhibitors of this protein should slow or prevent the production of amyloid plaques and Alzheimer's disease. Michael Lee presented his work on alpha synuclein transgenic mice, which supported a specific pathogenic effect of the A53T form of mutant alpha synuclein. Mice that overexpress the A53T mutant alpha synuclein develop a progressive motor phenotype consisting of decreased movement, poor balance and dystonia which culminates in early death, but mice that overexpress comparable levels of wild type or A30P mutant alpha synuclein do not develop these abnormalities. The A53T mutant mice develop a progressive astroglial reaction of brainstem and cerebellar nuclei. Yuquig Li and Alan Peterson discussed the novel approaches they are using to generate torsinA mutant models. Yuquig Li detailed his plans to use various lines of Cre recombinase expressing mice to generate region specific knock in mice that express mutant torsinA specifically in the striatum, as well as plans to generate tissue specific knock out mice. Alan Peterson presented a method which allows one to insert a transgene at a defined chromatin site highly permissive for expression of transgenes. This is accomplished by cloning the desired transgene in a genomic vector containing a portion of the HPRT gene, and using homologous recombination to target this vector in ES cells that lack the vector HPRT sequence in one allele. Homologous recombination thus restores HPRT function to the clone, placing the transgene at a defined site. This location has been shown to allow promoters to express in a physiological pattern, and the technique allows precise structure/function studies to be performed that are difficult or impossible using classical transgenic techniques. Alan Peterson plans to make a number of transgenic torsinA lines with this technique, including ones that express torsinA widely, or only in developing neurons.
Etiologic specific models of DYT1 primary dystonia were discussed by Scott Emmons (Albert Einstein), Lesilee Rose(UC at Davis), Jim Gusella (MGH), and William Dauer (Columbia CPS). Emmons and Rose discussed studies in C. elegans. Emmons focused on two torsin related proteins (trp), trp1 and trp2, and another torsin related protein, OOC-5. Using a GFP-tagged transgene, he found trp1 to be expressed in a subset of neurons, muscles and gut cells, and work to define the expression pattern of trp2 is underway. Another set of experiments explored the consequences of blocking trp gene function (using the RNAi technique), and showed that blockade of OOC-5 resulted in an embryonic arrest phenotype, while blockade of trp1 gave no phenotype. Work by Dr. Rose has shown that the torsin-related OOC-5 gene and another gene, OOC-3, are required for normal polarity of a particular cell during the asymmetric divisions of the early C. elegans embryo. Disruption of these genes leads to a specific polarity defect involving an altered distribution of the PAR-3 and PAR-2 proteins, which are required for normal polarity of the early C. elegans embryo. Additionally, her lab has found OOC-5 protein to be localized to all cells of the early embryo, in a pattern reminiscent of the ER and/or secretory pathway in other cell types. Others have reported that the OOC-3 protein is a predicted transmembrane protein localized to the ER. Rose and her colleagues find that OOC-5 and OOC-3 proteins colocalize extensively in early embryos. These localization studies are consistent with the proposed ER localization of human torsinA, suggesting that some essential ER-related function has been conserved throughout evolution in the torsin proteins. Furthermore, it is possible that OOC-3 encodes a protein that interacts with the OOC-5 gene product, and may thus represent a homolog of a torsinA-interacting protein.
Gusella presented his work on torsinA mutants in Drosophila. There is only a single torsin homolog in Drosophila, termed torsin related protein 4 (TORP 4). Null mutants of TORP 4 have been limited by the fact that there is a P element hot spot very close to the TORP 4 gene. In addition, the specific perturbation of TORP4 is likely to be quite challenging, as it is in close proximity to 4 other genes. Overexpression of TORP 4 in the Drosophila eye produces a "rough" phenotype, and abnormalities in the photoreceptor and pigment layer. Work in progress is aimed at expressing wild type and mutant human torsinA in Drosophila, and knocking out TORP 4 by using homologous recombination. It is anticipated that the eye phenotype may be used to screen for TORP 4 interacting or modifying proteins, and similar screening strategies will also be valuable in the C. elegans work described by Emmons and Rose.
In the final talk relating to DYT1 models William Dauer (Columbia) described the generation and characterization of torsinA mutant mice. "Knock down" mice expressing low levels of mutant torsinA (and no wild type protein) display deficient habituation in open field studies. Cyclic voltametry studies measuring striatal dopamine release in these mice show that the magnitude of release and the kinetics of dopamine uptake and vesicle recycling are normal in these mice, as are the levels of the D1 receptor and dopamine transporter. Mice that contain the pathogenic GAG deletion in their native torsinA gene (knock in mice) have not yet been found to have an abnormal behavioral phenotype, and studies of dopamine receptor and transporter level also appear normal in this line. TorsinA knock out mice die at P0, presumably because of a failure to feed, although the precise reason for this inability to feed has not been clarified.
Mark LeDoux completed the animal models section with a talk on the dystonic rat, a naturally occurring autosomal recessive mutation. These rats display dystonic symptoms by 12 days of age, and die from progressive motor disability by approximately 35 days. Biochemical studies have shown increased GAD activity, decreased muscimol binding, and increased glucose utilization within the cerebellar nuclei. These findings suggest increased activity at the level of Purkinje cell synapses within the cerebellar nuclei. In contrast studies of the basal ganglia including those of muscarinic cholinergic, dopamine D2, 5HT2, and benzodiazepine receptor binding have been normal. Behavioral studies with harmaline and electrophysiologic studies of Purkinje cell firing suggest that a defect in the pathway from the inferior olive to climbing fiber synapses on cerebellar Purkinje cells is the primary abnormality in the dystonic rat. These studies support a role for non-basal ganglia related structures in dystonic physiology.
Last Modified April 12, 2011


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JBL Synchros S700 Premium Powered Over-Ear Stereo Headphones
"Hi, I`ve noticed that there aren`t many reviews of these headphones out there and I think they are too underrated, maybe because of it`s price. So, let`s get to it. Style: I think they look beautiful, I would wear the them in the street without any problem, even wearing a suit. Build Quality: Simply the best out there, I`ve never seen a pair..." --Silver01
JBL Synchros Reflect In-Ear Sport Headphones With Android Control (Black)
"Just received my JBL synchros reflect (wired version) with Android control button. Bought it from American seller; from_wow in Ebay which advertised it as "like new, still in retail package". Received it in just 10 days after payment. Truly speedy delivery thanks man. For your information I have tried JBL synchros reflect (bluetooth) before and I am truly impressed with its sound quality. So..." --AzimAkbar
JBL J46BT Bluetooth Wireless In-Ear Stereo Headphone
"These are easy to use and pair quickly with your device. The sound quality - lows, mids, and highs are excellent provided a good seal with the ear. Good bluetooth range, battery life, and comfort. They do not fall out of my ears and I couldn't give them a higher recommendation especially with a price point so far below competitors of similar quality. " --Mark231
JBL J33 Premium In-Ear Headphones - Black
" Okay..well this is my first review..if you may call it so..hence any,,amatuerish mistakes be forgiven..here we go.. So i saved some money(i'm always short of it) for the Vsonic gr02 be..after studying Head-fi posts extensively..and at the moment of commitment, my seller went out of stock. These were just launched on a reputed website here in india..so i decided that i'd get them to satisfy..." --Aakash
JBL E40BT Black High-Performance Wireless On-Ear Bluetooth Stereo Headphone, Black
"hi guys! first of all this is going to be my first headphones to review. and i am new here at head fi. i bought this headphones for my commute from home to work. i got tired from all the wires getting tangled up inside my bag so i decided to try out a bluetooth headphones. built quality. is not too bad i think. its all made with plastic with a bit of aluminum on the..." --saitama
JBL Synchros S300 Premium On-Ear Stereo Headphones
"I have tried the JBL Synchros line of s300, s500, and s700. As it turns out, the low end model sounds the best to me. These are on ear design, cups are about 3 inches wide. Headband and yokes are metal, hinges and cups are plastic. I would like metal for the hinges for durability myself. The sound is very nice for a portable, a V shaped sound curve with bass that hits very very low...." --ourfpshero
JBL J55 High-Performance On-Ear Headphones with JBL Drivers and Rotatable Ear-Cups - Black
" In 2012, there was a number of newly launched headphone products by JBL. My friend happens to to have bought one of the J55is. Here is brief review on it. Packaging case for J55i For a headphone which costs only $79, packaging is up to standard. At least, it doesn't appear too cheap. Various items inside the case A small pocket inside..." --JamesM
JBL Tempo On-ear Headphones
"I was looking for a sennheiser HD201, and I can't find it in the market. Finally I choose this headphones because of JBL name and the simple construction. This are very good for a mobile devices that are used to listed music, radio, recordings, etc, they don't have volume or mic to talk. The sound is amazing for a bottom line model. Here the sensitivy factor is very important for a mobile..." --rickheadfi
JBL Everest Elite 300 Noise Canceling Bluetooth Headphones
"I recently picked up the JBL Everest Elite 300 On-Ear Bluetooth Noise Canceling Headphones for $209.99 from the local Best Buy. They are a new product, and since they haven't been discussed around here, I thought I'd post my brief impressions for the holiday shopping season. TLDR; In short, I really like them. They are going to be my go-to travel and portable headphones for quite..." --hodgjy
JBL Synchros 400BT Bluetooth Wireless On-Ear Stereo Headphones
"I have tested all JBL BT Headphones. I am not even sure why. These are the ONLY one which is worth considering if you look for a good sound. But NOT for the actual price of $270, EVER. They do not sound so great either. But not bad like the other JBL. I have found them somehow exciting, with good dynamic, good bass, good highs and decent mids. They surely are not the most balanced..." --Giogio


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Prints - Paper Types
Standard prints are available in a large variety of sizes and crops, including square and panoramic.
Glossy & Matte
Glossy is a shiny finish and prints appear brighter than Matte. Both are printed on Fuji Crystal Archive Paper Type II.
Lustre is a premium finish on a heavier paper. It offers the vibrant colors of glossy with the fingerprint-resistant finish of matte. Printed on Fujicolor Crystal Archive Professional Paper Super Type PD. More info
Rich, distinctive metallic look with a huge WOW! factor. Printed on Fujicolor Crystal Archive Pearl Paper. More info
Your photos. Your words. Perfectly presented on high quality card stock. Create one now.
Printed on heavy Matte stock. Envelopes included. Make one in minutes.
Wrapped over a wood frame, then finished with a UV and water resistant dry laminate. More info
Metallic or Lustre print mounted onto lightweight gatorboard. More info
High-quality canvas prints made more affordable by eliminating the wooden frame for mounting. More info
Wrapped over a wood frame, then finished with a UV and water resistant dry laminate. More info
Great Gift Ideas
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|3-inch and 4-inch high-quality pin-on buttons. (Some of your image is trimmed away.) More.
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Stuff You Wear
|Photo Apron
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Greeting Cards
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Two years before seeking help from physicians at The Mount Sinai Medical Center, Rosemary McGinn, 53, was diagnosed with hypoglycemia. She went everywhere with an arsenal of snacks and juices that she ingested frequently to keep her blood sugar from dropping.
"When my sugar would suddenly crash, it was like I was drunk," she says. "I would become very combative, not knowing what I was saying, and sway back and forth."


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Authorities arrested a teen driver who they said caused two traffic accidents in Puhi on Friday.
Kauai police said the first accident happened at 2:35 p.m. while the 17-year-old driver was driving a Jeep west on Kaumualii Highway near Kukui Grove Center. Officers said the teen rear-ended a Chevrolet sedan driven by a 48-year-old man from Colorado.
The teen drove away from the scene and was about to turn left onto the Anonue Road junction past Kauai Community College when police said he crashed onto a Nissan MPV that was heading east in the outermost lane of Kaumualii Highway.
Police said the driver of the Nissan, a 32-year-old Kapa'a woman, had to be extricated from her vehicle by Kauai Fire Department personnel. The woman, who sustained non-life-threatening injuries to her neck, arms and legs, was taken to Wilcox Hospital.
The teen driver and the Colorado man were not injured, police said.
Officers said the teen was charged with second-degree negligent injury, accidents involving bodily injury, leaving the scene of an accident with damages and no insurance. He was released to his guardian.
The two crashes caused traffic congestion in the area for about an hour, police said.


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The Shift Collection features classic design interpreted in a contemporary fashion, fusing traditional pattern with modern styling. Fabrics are designed to be mixed and matched by layering texture, color and pattern, creating an eclectic familiar-meets-new mood.
|FR Standards Met:
|California Technical Bulletin 117-2013 Class 1
|Sunbrella® is a registered trademark of Glen Raven, Inc.


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WASHINGTON, D.C. - The U.S. Consumer Product Safety Commission (CPSC) voted unanimously (3-0) to approve a new federal mandatory safety standard to improve the safety of infant swings to prevent injuries and deaths to children.
Infant swings are stationary juvenile products with a frame and powered mechanism that enables an infant to swing in a seated position. An infant swing is intended for use with infants from birth until a child is able to sit up unassisted. Cradle and travel swings are also included in the standard.
The new federal standard, which incorporates provisions in the voluntary standard ASTM F2088 - 12a, requires the following:
Between May 2011 and May 2012, CPSC received reports of 351 infant swing-related incidents that occurred between 2009 and 2012. Two of the 351 incidents resulted in fatalities, and 349 incidents were nonfatal; 24 of the nonfatal incidents resulted in injuries.
The effective date for the mandatory infant swing standard is May 7, 2013. The Danny Keysar Child Product Safety Notification Act, Section 104 of the Consumer Product Safety Improvement Act of 2008, requires CPSC to issue safety standards for durable infant or toddler products, including infant swings. In addition to infant swings, CPSC has issued mandatory safety standards for full-size and non-full-size cribs, play yards, children's bed rails, baby bath seats, baby walkers, and toddler beds.
The U.S. Consumer Product Safety Commission is charged with protecting the public from unreasonable risks of injury or death associated with the use of
thousands of types of consumer products under the agency's jurisdiction. Deaths, injuries, and property damage from consumer product incidents cost the
nation more than $1 trillion annually. CPSC is committed to protecting consumers and families from products that pose a fire, electrical, chemical or
mechanical hazard. CPSC's work to help ensure the safety of consumer products - such as toys, cribs, power tools, cigarette lighters and household
chemicals -– contributed to a decline in the rate of deaths and injuries associated with consumer products over the past 40 years.
Federal law bars any person from selling products subject to a publicly-announced voluntary recall by a manufacturer or a mandatory recall ordered by the
To report a dangerous product or a product-related injury go online to www.SaferProducts.gov or call CPSC's Hotline at 800-638-2772 or teletypewriter at
301-595-7054 for the hearing impaired. Consumers can obtain news release and recall information at www.cpsc.gov, on Twitter @USCPSC or by subscribing
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NASCAR Acceleration Weekend
I am going and just wondering if any Garage Gang Members are gonna be there.
Whatever goes... remember, this is a family forum
BlackFlag wrote:I would have liked to have gone, but I'm too lazy to get up early on Saturday to go wait in line. LOL
I guess this replaces the old Winston Cup Preview they use to hold in Winston-Salem years ago.
Hall of Fame is definitely a better venue for it. I hope you had a good time!
"Days of Thunder" ??? Why didn't they show a good movie, like Ricky Bobby???
michaeljohn wrote:now, now, danica is just misunderstood, she is the princess of nascar <hurl>
chip28018 wrote:43 different autos and about 87 total for me. I brought my dad and a friend of mine to go in different lines. Then I met up with jaclyn (one of the miss sprint cups) I had become good friends with her last year and she had saved me wristbands for almost all the lines! Will post pictures soon
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Are kids making good choices in lunch line?
Published On: Jul 23 2011 08:18:15 PM CDT Updated On: Aug 14 2013 09:13:05 AM CDT
Your child's school lunch could be high in calories and fat, but there are healthy alternatives that can help them maintain a healthier daily diet. Find out some alternatives here.