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MOSCOW, November 12. /TASS/. Russian Helicopters expect to keep the portfolio of orders at the current level in two coming years and to sell 230-280 helicopters a year, deputy CEO of the company Alexander Scherbinin told TASS on Thursday.
Current production and sales of Russian Helicopters are aligned with the global market situation and production capacity of the company, Scherbinin added.
"It [the capacity] is about 230-280 helicopters of a year. We plan to maintain such level and the available portfolio of orders in future year or two," he said.
Russian Helicopters supplied 271 helicopters to customers and produced about 300 in 2014, Scherbinin said. "They were partly supplied to customers in 2015 in accordance with contracts," he added.
"Our firm orders portfolio is over 500 helicopters as of the beginning of 2015. We will produce these helicopters for our traditional and for new partners," the official said.
The company is present in nearly all niches of the global helicopter market, from the light Ansat and Kamov Ka-226T to ultra-heavy Mil Mi-26T2. "Demand and contracts cover all types of helicopters," he added.
Russian Helicopters plans to open a network of Russian equipment maintenance centers in China.
"Asia is the most promising region for us now. The holding plans to set up a network of Russian helicopters' maintenance and repair centers in China in cooperation with the largest Chinese operators," Scherbinin said.
Such Chinese companies as AVIC International, CITIC Offshore Helicopter Co. Ltd, AVICOPTER International, and Poly Technology already showed interest in such cooperation, he added.
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Mohegan — Despite reporting dramatic declines in earnings and profit for the quarter that ended June 30, Mohegan Tribal Gaming Authority executives said Tuesday they'll aggressively continue to pursue out-of-state gaming licenses and expect to break ground by the end of the year on a $110 million hotel at Mohegan Sun.
Prior to a conference call with gaming analysts, the authority announced its net income, or profit, for the quarter was $13.9 million, a 37.7 percent decrease over the same three-month period in fiscal 2013. Adjusted EBITDA — earnings before interest, depreciation, amortization and other payments — was $69.1 million, down 18.7 percent.
The authority, the gaming arm of the Mohegan Tribe, owns and operates Mohegan Sun and Mohegan Sun at Pocono Downs in Wilkes-Barre, Pa. It also includes Mohegan Gaming Advisors, which manages Resorts Casino Hotel in Atlantic City.
Mitchell Etess, the authority's chief executive officer, called the quarterly results "mixed" and attributed declines in gaming revenues at Mohegan Sun to the state's sluggish economic recovery and competition.
"We face the same challenges as many of our peers," he said.
Mohegan Sun derives 59 percent of its "gaming trips" from state residents who also provide 53 percent of the casino's gaming revenue. The casino's quarterly EBITDA of $63.7 million was down 14.8 percent. Slot revenue, the largest single source of casino revenue, was down 9.1 percent.
At Pocono Downs, adjusted EBITDA was $13 million, a 17.3 percent decline.
The authority's corporate expenses increased significantly during the quarter, a reflection of costs incurred in pursuing casino projects with partners in Revere, Mass., Philadelphia and New York's Catskills region.
"If MTGA were to win even one of these licenses, that would go a long way toward helping us achieve our goal of diversifying," Kevin Brown, the tribe's chairman, told analysts.
Closer to home, Mohegan Sun is on the verge of finalizing an agreement with a partner to develop a 400-room hotel at the casino, a $110 million project that would require little capital investment by the Mohegans, said Mario Kontomerkos, the authority's chief financial officer.
"It will be built and financed by a third party and leased back to MTGA," he said.
Asked about a timetable, Kontomerkos said construction would begin by the end of the year, with the hotel expected to open 18 to 20 months thereafter. The hotel would bear the Mohegan "flag," he said.
Mohegan Sun already has 1,200 hotel rooms.
Plans to add a $50 million nongaming addition to the casino also are advancing, according to Etess. The so-called "Downtown District" project, featuring space devoted to retail, entertainment and dining, would be owned by the developers.
Small child abandoned on Bank Street taken to New London fire station
Head of regional energy cooperative defends Kentucky Derby trip
Boyle, a serial rapist and killer, once again eligible for parole
Trump took $17 million in insurance for hurricane damage few remember
Police search for Norwich twin brothers in connection with September bank robbery in Canterbury
19th Senate District Debate: Cathy Osten and Barbara Richardson Crouch
Lunch with the Candidates: Rob Dempsky, Bonnie Hong and Emmett Riley
East Lyme school cancels Halloween parade, many districts scale back activities
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My bf bought an 02 mercury sable. It has 151k miles. The tranny went completely out. It won't go into any gear! It showed no problems as far as slipping or anything? Is there a recall on the tranny of the sable? What could have caused this to happen all of a sudden like that? Help plz
3 Answers
Even if there was a recall its long past any consideration at that mileage. Sometimes trannys jus go out, jus like people just drop dead. Its aroun:-) d that time for it to go. You could check fluids, sensors, linkage but I think it just died on your bf...sorry!
1 out of 1 people think this is helpful.
Take a look at the transmission fluid if it looks brown or black most likely never had the fluid and filter changed and that is why it has gone bad.
Could the shift cable be the problem? Have it looked at by a trusted mechanic.
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In firebug I am setting break points which allows me to see names and properties of some web analytics that are being sent by a javascript. We are trying to build an automation suite to verify all of these analytic events.
There is a requirement that I log in to the application, which is easily done using Selenium, but if I attempt to grab any Firebug data then that is not noticed by Selenium.
Before I went down the road of looking at possibly creating a Javascript to try and pull this information from the other JS that is passing the events I wanted to see if maybe there is something else that might exist.
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There should be functions in Selenium to grab the innerHTML of a target element.
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Cardioprotective Role of AT2 Receptor in Postinfarction Left Ventricular Remodeling
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The aim of this study was to determine the role of the AT2 receptor (AT2R) in left ventricular (LV) remodeling after myocardial infarction (MI). The left anterior descending arteries were ligated in AT2R gene knockout (Agtr2-) and wild-type (Agtr2+) mice. The LV remodeling was evaluated by echocardiography and histology over a period of 2 weeks after MI. The infarct sizes in hearts excised from Agtr2+ and Agtr2- mice on day 1 were similar. The mortality rate of Agtr2- mice (62.9%) on day 14 after MI was significantly (P<0.05) higher than that of Agtr2+ mice (39.7%). Accordingly, LV/body weight ratios (3.7±0.2 versus 3.0±0.1 on day 14) and LV end-diastolic (4.8±0.3 versus 3.9±0.4 mm on day 7) and end-systolic (4.4±0.3 versus 3.2±0.6 mm on day 7) dimensions evaluated by echocardiography were significantly greater in Agtr2- than in Agtr2+ mice. The rates of ventricular arrhythmia, rates of cardiac rupture, and blood pressures in the 2 strains were similar after MI. Myocyte cross-sectional areas were increased after MI, but the magnitudes were similar in Agtr2+ and Agtr2- mice, indicating the greater increases in LV dimensions and weight in Agtr2- mice are due to elongation of myocyte length and/or an increase in the interstitial weight (including vasculatures, infiltrated cells, and interstitial fluid). Interstitial fibrosis in remote myocardium was not evident in either strain. These results indicate AT2R plays a significant role in the protection against early development of LV dilation, thereby reducing the early mortality rate after MI.
Myocardial infarction induces global changes in ventricular architecture, called post–myocardial infarction (MI) left ventricular (LV) remodeling, which involves LV dilation, myocyte hypertrophy, and interstitial fibrosis, leading to a deterioration in LV function that is associated with increased rate of mortality.1,2 A substantial body of evidence1,3–5 has suggested that angiotensin II (Ang II) plays a critical role in the development of post-MI LV remodeling. Ang II has 2 major receptor subtypes, AT1 receptor (AT1R) and AT2R, both of which are expressed in the heart.6 It has been suggested that AT1R signaling mediates vasoconstriction, aldosterone secretion, cardiomyocyte hypertrophy, proliferation of fibroblasts, interstitial collagen deposition, and catecholamine release, all of which are implicated in progression of LV remodeling.7,8 Harada et al3 recently reported that AT1R gene-knockout mice had less LV remodeling and improved survival after MI. A deletion of AT1R or a blockade of AT1R with specific antagonists may result in selective binding of Ang II to the AT2R, indicating that the effect of AT1R blockade could be in part mediated by the effect of AT2R.7 However, little is known about the role played by AT2R in post-MI LV remodeling. The AT2R is thought to have opposite effect to AT1R and has been shown to suppress myocardial hypertrophy,9–11 fibroblast proliferation,10,12 and vascular cell hyperplasia.13,14 We have recently demonstrated in gene-engineered mice that interstitial fibrosis associated with LV hypertrophy was enhanced by a deletion of AT2R15 and inhibited by cardiac-specific overexpression of AT2R.16 Therefore, it is hypothesized that the AT2R plays a cardioprotective role in LV remodeling after MI. In the present study, to test this hypothesis, we induced MI on Agtr2- and Agtr2+ mice and compared the survival rates, cardiac geometries and functions, and histological findings in the 2 strains of mice over a period of 2 weeks after MI. We present a finding that the AT2R plays an essential role in protection against LV dilation and in improving survival rate in the early phase after MI.
Pairs of adult male Agtr2-17 and Agtr2+ littermates were used in this study. Because the AT2R gene is located on the X chromosome, heterozygous females were mated with Agtr2+ males to obtain hemizygous and Agtr2+ males. These mice were back-crossed for at least 6 generations onto a FVB/N background.15 All experimental procedures were approved and carried out in accordance with the Guidelines of Hiroshima University Graduate School of Biomedical Sciences.
Surgical Procedure
Male mice at ages of 10 to 12 weeks were anesthetized with pentobarbital (50 μg/g), intubated, and artificially ventilated with a rodent respirator. A left thoracotomy was performed, and MI was induced by permanent ligation of the left anterior descending artery (LAD) with a 7–0 nylon surgical suture, 1 to 2 mm from the tip of the left auricle. Successful ligation of the LAD was verified by color change of the ischemic area and by ST-segment elevation on the ECG. Some mice were killed 1 day after ligation and the infarct size was calculated as described previously,18 with some modification. Briefly, the hearts were cut into 2 pieces transversely at the middle portion of the site of ligation and apex. In cross sections, infarct size was calculated as the ratio of infarction length to the circumference of the endocardium. For the sham procedure, the same procedure was performed except for the LAD ligation.
Agtr2+ and Agtr2 mice were randomly allocated to 1 of 4 study groups. In one group, the natural prognosis after myocardial infarction was examined. The mice in this group were observed over a period of 2 weeks after MI, and the causes of death were estimated by autopsy (31 Agtr2+ mice and 35 Agtr2- mice). In another group, physiological profiles were examined. Transthoracic echocardiography was performed before (day 0) and on days 3, 7, and 14 after surgery or sham operation (18 Agtr2+ mice with MI, 4 Agtr2+ mice underwent sham-operation, 15 Agtr2-mice with MI, and 6 Agtr2- mice underwent sham-operation). Blood pressure was measured on days 0 and 3 by the tail-cuff method as previously described.6 In another group, histologic and morphometric examinations were performed. The mice in this group were killed on days 0, 3, and 14, and the hearts and lungs were excised and weighed. The hearts were subjected to histological assessment. The numbers of animals are indicated in tables and in figure legends. In the final group, Western blot analysis was performed. Three mice of each strain were killed on day 3 after MI.
Physiological and Morphological Assessment
Cardiac geometry and function were evaluated with the use of an echocardiographic system (Toshiba SSA 550A) equipped with a 14-MHz linear transducer. All studies were performed under spontaneous respiration and light anesthesia with an intraperitoneal tribromoethanol/amylene hydrate (Avertin) 2.5% wt/vol solution (5μL/g of mouse). Avertin was chosen for its negligible hemodynamic effects at this dose. LV end-diastolic dimension (LVDd) and end-systolic dimension (LVDs) were measured at the distal level of the papillary muscle by using short-axis M-mode images. Three beats were averaged for each measurement. Percent fractional shortening (%FS) was calculated as [(LVDd−LVDs)/LVDd]×100.
Histopathological Assessment
Mice were killed with KCL injection through the jugular vein. The hearts were fixed with 10% buffered formalin and embedded in paraffin. One to two-micrometer-thick sections were cut and stained with Masson's trichrome for measurement of myocyte cross-sectional area and perivascular fibrosis as previously described.6 For measurement of cross-sectional area, 100 myocytes (per mouse) having circular profiles were chosen from a noninfarcted area and the areas were traced. Perivascular fibrosis was assessed by calculating the ratio of the collagen area to total vessel area. The rate was defined as the perivascular fibrosis index. More than 10 coronary arteries were measured per mouse. All the parameters were quantitatively analyzed with Scion Image 1.62 software (National Institutes of Health Service Branch).
Western Blotting of AT1R
The amount of AT1R was examined by Western blot analysis with the use of an anti-AT1R antibody (Santa Cruz, AT1 N10, sc1173), as previously described.6 Membrane fractions were prepared from the left ventricles of Agtr2+ and Agtr2- mice 3 days after MI or sham operation. The samples were subjected to SDS-PAGE and transferred to nitrocellulose membranes. The membranes were probed with the anti-AT1R antibody.
Statistical Analysis
All results are expressed as mean±SEM. Analyses of survival after MI were carried out by the Kaplan-Meier method with the Breslow-Gehan-Wilcoxon method. Multiple comparisons among ≥3 groups were carried out by 1-way ANOVA and the Fisher exact test for post hoc analyses. Statistical significance was accepted at a value of P<0.05.
Survival Rate After Myocardial Infarction
There was no difference between the infarct sizes in Agtr2+ (62.7±6%, n= 10) and Agtr2- mice (66.3±5%, n= 9) on day 1. The results of Kaplan-Meier analysis in 31 Agtr2+ and 35 Agtr2- mice showed that the survival rate after MI over the 2-week period was significantly (P<0.05) lower in Agtr2- mice than in Agtr2+ mice (Figure 1.). Postmortem examination of the hearts showed that cardiac rupture had occurred in 11 (48%) of the 23 Agtr2- mice and 7 (50%) of the 14 Agtr2+ mice, but other causes of death were not identifiable. Electrocardiograms of randomly selected mice with MI were monitored for 2 hours, but no arrhythmia was evident in either strain of mice.
Hemodynamic and Morphological Assessments
Systolic blood pressure tended to decrease after MI in both Agtr2+ and Agtr2- mice, but there was no significant difference in systolic and diastolic blood pressures between the 2 strains of mice either before or after MI (Table 1). Echocardiography was performed on days 0, 3, 7, and 14 after surgery or sham operation (Figure 2). LVDd and LVDs were progressively enlarged in both Agtr2+ and Agtr2- mice after MI. However, on days 3 and 7 after MI, both LVDd and LVDs were significantly larger in Agtr2- mice than in Agtr2+ mice, indicating that early expansion of infarct and/or remote myocardium after MI was enhanced in Agtr2- mice. There was no significant difference between %FS in Agtr2+ mice and that in Agtr2- mice with MI throughout the study period (Figure 2C). LVDd, LVDs, and %FS in Agtr2+ mice and those in Agtr2- mice before surgery were similar and were not affected by the sham operation.
Body weights, LV weights, and lung weights of mice killed on days 0, 3, and 14 after surgery are shown in Table 2. Consistent with the results of echocardiography showing exaggerated LV dilation after MI in Agtr2- mice, the LV/body weight ratio in Agtr2- mice was significantly (P<0.05) larger than that in Agtr2+ mice on days 3 and 14. The lung/body weight ratio, an index for heart failure, tended to be larger in Agtr2- mice than in Agtr2+ mice on days 3 and 14, but the difference did not reach statistical significance.
Myocyte Hypertrophy, Interstitial Fibrosis, and Histological Assessment
The myocyte cross-sectional area in the myocardium remote from infarction progressively increased after MI (from day 3 to day 14) both in Agtr2+ and Agtr2- mice, but the magnitude of increase in the 2 strains of mice was not different (Figure 3). On the other hand, LV weight and LV dimension were larger in Agtr2- mice during the 2 weeks after MI, indicating that cardiomyocytes were elongated to a larger extent in Agtr2- mice. Other explanations for this finding may be an increase in the weight of interstitium, including those of remodeled vasculature, infiltrated cells, and interstitial fluid, in Agtr2- mice.
MI did not increase the perivascular fibrosis index until day 14 either in Agtr2+ or Agtr2- mice (Figure 4).
Infiltration of the infarct zone and the border zone by neutrophils and macrophages did not appear to be different in Agtr2+ and Agtr2- mice (data not shown).
AT1R Expression
As shown in Figure 5, the amounts of AT1R determined by Western blot analysis were not different in Agtr2+ and Agtr2- mice on day 3, excluding the possibility that the LV dilation and increased mortality rate after MI in Agtr2- mice were a result of upregulation of the number of AT1R in the myocardium. However, we cannot exclude a possibility that AT1R activity, not the number, was increased in Agtr2- mice, based on a recent report by AbdAlla et al that AT2R binds directly to the AT1R and thereby antagonizes the function of the AT1R.19
The major findings in the present study are (1) the survival rate of Agtr2- mice was reduced in the first 2 weeks after MI and (2) LV dilation on days 3 and 7 after MI was greater in Agtr2- mice. Since there was no apparent difference between the rates of occurrences of cardiac rupture or fatal arrhythmia in Agtr2+ and Agtr2- mice, the most likely cause of the increased mortality rate of Agtr2- mice is heart failure. These observations suggest that the AT2R plays a cardioprotective role in the mechanism of preventing early development of LV dilation/LV failure after acute MI. Recently, Xu et al20 reported that post-MI survival rate was not different between AT2R-null mice and wild-type mice. The discrepancy between the results of our study and theirs may be explained by the differences in the introduced infarct size (62% to 66% of the LV circumferential length in our study versus 40% in Xu's study) and the background strains of mice on which the gene targeting was established (FVB/N versus C57/BL).
Post-MI LV remodeling has been divided into an early phase (within 72 hours) and a late phase (after 72 hours).1,21 The early phase mainly involves expansion of the infarct zone. In the present study, LV dilation and decreased survival rate persisted beyond 3 days. However, in cases of extended transmural MI, the noninfarct zone also undergoes a progressive lengthening that is consistent with secondary volume-overloaded hypertrophy and that causes persistent LV dilation associated with a reduction in survival.1 It is thought that such processes of early remodeling may have been exaggerated in Agtr2- mice for some reasons related to the absence of AT2R. On the other hand, late remodeling progresses 1 to 2 months after MI, involving LV dilation, myocyte hypertrophy, and interstitial fibrosis.3,21,22 In the present study, an interstitial fibrosis, which characterizes the late remodeling, was not evident either in Agtr2+ or Agtr2- mice, supporting that the process of the late LV remodeling had not started in these mice. In fact, the interstitial fibrosis developed in the 4th week after MI, the extent of which was significantly larger in Agtr2- mice than in Agtr2+ mice (data not shown).
The mechanisms whereby a deletion of the AT2R results in severer postinfarction heart failure is not clear. In support of our observation, Yang et al23 recently suggested that overexpression of the AT2R in cardiomyocytes improved LV contractile function in postischemia/reperfusion LV remodeling in transgenic mice. We could not detect a difference between the LV contractile functions in Agtr2+ and Agtr2- mice by %FS; this may have been due to the limited capability of our method for evaluating global LV function. In the same transgenic mice as used by Yang et al,23 we have recently observed that stimulation of the AT2R overexpressed in cardiomyocytes activated kinin formation from the myocytes.16 Results of other studies also implicate the AT2R in activation of the kinin/NO system.24,25 It is possible that the LV dilation and increased mortality rates in Agtr2- mice are mediated by the loss of kinin/NO activation after MI, although the role of kinin/NO system in post-MI remodeling remains incompletely understood.26 It is also possible that the exaggerated heart failure in Agtr2- mice could be a secondary phenomenon caused by a compromised systemic hemodynamics. Since AT2Rs in the kidney and systemic vasculature are involved in the mechanisms of natriuresis27 and a vasorelaxation,27,28 respectively, a deletion of AT2R may causes volume overload and increased systemic vascular resistance, leading to a deterioration of systemic hemodynamics after MI. More studies are needed to clarify the mechanism of cardioprotection mediated by the AT2R.
In the present study, we demonstrated that deletion of AT2R resulted in exaggerated early development of LV dilation and a reduction in the survival rate after MI in mice. Although the underlying mechanism is unclear, a cardioprotective role of the AT2R unrelated to myocyte hypertrophy and interstitial fibrosis in post MI remodeling has never been described. The clinical relevance of this observation may be important since administration of an AT1-receptor antagonist causes elevation in the plasma level of Ang II,29 which specifically binds to AT2Rs in the heart and may serve as an AT2R agonist. It would be an intriguing topic whether or not this class of drug could benefit the early mortality rate after MI.
This study was supported by Grants-in Aid for Scientific Research (Nos. 08457639, 07407065, 11470518, 11771511, and 14572183) from the Ministry of Education, Japan, and a grant from the Charitable Trust Clinical Pathology Research Foundation of Japan.
- Received October 2, 2002.
- Revision received October 30, 2002.
- Accepted November 12, 2002.
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Harada K, Sugaya T, Murakami K, Yazaki Y, Komuro I. Angiotensin II type 1A receptor knockout mice display less left ventricular remodeling and improved survival after myocardial infarction. Circulation. 1999; 100: 2093–2099.
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- Cardioprotective Role of AT2 Receptor in Postinfarction Left Ventricular RemodelingYoshihiko Oishi, Ryoji Ozono, Yoko Yano, Yasuhiro Teranishi, Masahiro Akishita, Masatsugu Horiuchi, Tetsuya Oshima and Masayuki KambeHypertension. 2003;41:814-818, originally published March 1, 2003http://dx.doi.org/10.1161/01.HYP.0000048340.53100.43
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- Cardioprotective Role of AT2 Receptor in Postinfarction Left Ventricular RemodelingYoshihiko Oishi, Ryoji Ozono, Yoko Yano, Yasuhiro Teranishi, Masahiro Akishita, Masatsugu Horiuchi, Tetsuya Oshima and Masayuki KambeHypertension. 2003;41:814-818, originally published March 1, 2003http://dx.doi.org/10.1161/01.HYP.0000048340.53100.43
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This video will show you how to correctly remove the shift knob from various Mercedes models of the same generation. 1. Undo the shift knob boot by grasping it firmly and turning it counter clockwise until it loosens up. The shift knob boot must not detach from the shift knob itself! 2. Proceed to pull the shift knob straight up. If this is your first time removing the shift knob - very mild "wiggling" may help in this process, but do not twist the shift knob beyond that! 3. Installation is opposite of removal. Like this video? Be sure to thumbs up and subscribe :D
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Jana Kramer Sustains Injury During 'Dancing With the Stars' Performance
Jana Kramer suffered an injury during her first 'Dancing With the Stars' performance.
Jana Kramer suffered an injury during her first 'Dancing With the Stars' performance.
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Subjects and Verbs 1, 2, 3, 4 to Infinity by Ralph Duncans Jr
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Recent Florida South West reviews
"We arrived around midnight due to a late flight into Tampa. The owner had kindly left lights on for us and arranged it so we could let ourselves in. We had a good night's sleep on the king-sized mattress. The pillows on the king were a little tall and stiff to our liking, but there were other pillows in the closest that we used and slept comfortably on. As mentioned, the property location is perfect! We walked to the coffee shop for breakfast and lunch a couple times. The walk to the beach is no more than 1 block. You can also walk to the Sandbar, which is our favorite restaurant on Anna Maria. The patio area/pool is very private and very nice. The owner has a tarp over part of the pool, which nicely shaded us from the hot sun. The pool was a great temperature and very clean. We had an pleasant, relaxing stay at this property and would definitely rent from them again."Guest: Jill C. Date of Stay: September 2016 Review Submitted: September 27, 2016View listing
"My family of 5 including 2 college and one high school age, had a great time staying at this location. It was stocked with everything we needed and there was plenty of space for everyone. The location was perfect for the beach, bike riding, the pool, tennis and just overall relaxing. I would definitely recommend going here again!"Guest: Ruta L. Date of Stay: December 2015 Review Submitted: December 30, 2015View listing
"My husband and I just returned from "Ibis Paradise". The condo was fantastic and extremely clean, it was stocked with everything you might need ! The sunsets were beautiful !"Guest: Anonymous Date of Stay: April 2016 Review Submitted: May 6, 2016View listing
"The pictures looked good but mostly we rented for the proximity to our friends who had recently moved nearby. We loved our little beach cottage. It was clean and adequately furnished. In Iowa the kitchen is the "heart of the home" and this one was beautiful! Roomy enough for several people to work and and a great counter height granite bar with numerous seating that wraps around the kitchen making entertaining perfect! There is also a small outdoor entryway/courtyard that is a perfect place to store all your sandy beach gear. It's located one block off the main " beach "drag so everything is nearby yet it's very quiet. There is an outdoor patio with table and gas grill. (Ok, the backyard turf is not in great shape, it's bit scraggly but that didn't matter to us. There is a pond with some fish. ) I really don't understand the previous "not good" review as we found none of those deficiencies. We found the owners Drew & Mia very friendly and accommodating. My message to all renters, should you have a problem or concern, contact the owner and give them a chance to resolve any issues. We enjoyed our stay in Bonita Springs and hope to return and stay at this property."Guest: Robert/Lynda M. Date of Stay: June 2016 Review Submitted: June 19, 2016View listing
Featured Destinations: Dauphin Island, Isle of Palms, Bald Head Island, Destin Condo Rentals


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The clip miri hitting on guy from Zack and Miri Make a Porno (2008)
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Product DescriptionGet ready for cards and casino night with friends when you use our Casino Club Serving Tray/ Serve up tasty treats as poker is played in this black 12" Club Card Suit Tray. Perfect for card night with the ladies or poker night with the guys and looks great as an accent to your Vegas and Casino decorations with a little Wonderland thrown in. Hand Wash Only. Each tray is approximately 2" deep.
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Keep Your Child Safe This Halloween with Light Up Accessories
Be seen, be safe. Add light up accessories to your child's Halloween costume that will make them visible to motorists and other pedestrians while trick-or-treating this Halloween.
5 Steps to Keep Zombies Away During the Zombie Apocalypse
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This is a special program. Hurricane Irene is coming to the East Coast and the New England area. President Obama is saying it could be "historic." Meteorologists refer to hurricanes as coming from "Mother Nature." "Mother Nature" is God. Why do they come from God and why does He allow other natural disasters like earthquakes to occur?
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Rush Training Send Aways at 11 Months of Age
I filmed this video back in August of 2008, when Cindy's dog, Rush, was just 11 months old. We were doing our normal training when this cat walked up. It is not our house cat. Rush makes a few mistakes in his send away training, but for an 11 mont...2 Minutes, 27 Seconds
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Sending out ahead in Mondioring
One of the exercises in the Mondio is the SEND AWAY. This video explains the proceedure handlers need to know for the SEND AWAY. It explains how far the dogs must go for Mondio ONE; Mondio TWO: and Mondio THREE. The video also shows different hand...2 Minutes, 56 Seconds
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Part 2 of the Michael Ellis E-collar Series Coming Mid-May 2013
In this short video, Michael discusses what will be covered in the second DVD on the electric collar. This DVD is titled Common Application of the Electric Collar, and will cover just that. It is broken down into 3 main categories: obedience, high...2 Minutes, 56 Seconds
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Michael Ellis Lecture -- Part Seven
It's one thing to train dogs and quite another to train people how to train dogs. In 2009, Michael Ellis started to collaborate with Leerburg to make videos for dog training. He is one of the best sport dog trainer and instructor in the world. Thi...5 Minutes, 30 Seconds
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Michael Ellis Works Young Dog on the Object Guard
Donna Matey and her dog, Eager, attended the Michael Ellis Ecollar class held at Leerburg on December 1 & 2, 2012. At the end of the class Michael and Donna demonstrated the early stages of training the object guard with Eager.11 Minutes, 45 Seconds
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Using Dog Parks to Proof Obedience with Michael Ellis
Dog parks can be dangerous but they can serve a purpose. We use dog parks as distractions in order to proof our obedience exercises. In this video, Michael Ellis demonstrates how to utilize dog parks for this without putting his dog in harm's way....2 Minutes, 9 Seconds
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Muzzle Attacks Off Object Guard with Michael Ellis and JacksonPosted: 06-01-2011 • Length: 5 Minutes, 21 Seconds
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9th Annual Bivona Child Abuse Summit
Thursday, April 27 and Friday, April 28, 2017
at the Joseph A. Floreano Rochester Riverside Convention Center
Bivona's annual Child Abuse Summit is nationally recognized as the premier conference in the Northeast region. This cutting-edge multidisciplinary training is for social workers, law enforcement, prosecutors, mental health and medical professionals, school personnel, day care providers, the faith-based community and more. Workshops are being presented by national and local experts.
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Moon Lake was not being drained, the water level was taken down approximately 5 feet. After this happened is when the car was discovered. I haven't heard of anything official.Where is all this info coming from cause I am not finding any info but what is posted here. Also, I've been to moon lake and I am finding a tiny bit hard to believe that a car was submerged in that tiny "lake" for 5 years and no one noticed. My husband and his squad use to run around moon lake like every... tuesday... for several years. We had Org days with people canoeing(sp?) and no one noticed. I don't know... I just would like to some kinda of report or official update.
Body Found In Submerged Car In Fort Riley Lake
There are 31 comments on the KAKE story from Apr 15, 2010, titled Body Found In Submerged Car In Fort Riley Lake. In it, KAKE reports that:
Fort Riley authorities are investigating after a body was found in a car submerged in Moon Lake on the northeast Kansas Army post.
Join the discussion below, or Read more at KAKE.
#22 Sep 23, 2010
#23 Sep 23, 2010
Classified means information about the incident has not been released to the public. Anything that is being said is speculation.
#24 Oct 8, 2010
it did happen.. not to sure about all the details I wish I could give them to you.. but I lived here when they found the car.. I am with you Idk how it went un-noticed for so long.. but I know they found the car with bodies in it.. I hope this helps..
#25 Nov 2, 2010
Her name was Echo. and she was my friends ex wife.. He did not do it. He was over seas when she came up missing.
#26 Nov 7, 2010
If she came up missing while he was away over seas is it possable to say maybe she like so many other army wives had a love life on the side that went haywire? i know its a stretch something like that just never happens now does it?
#27 Nov 21, 2010
Seen the comment about abuse going unpunished by military and the disappointment in the Military for not punishing the suspect....bottom line is this if it happens off post it is hard for the Chain of Command to do anything (hands are tied)..then even when it happens on post and is reported to social services there is an investigation and the Chain of Command has to wait the results...last situation is that the victem either doesnt report it or doesnt want to press charges. Dont blame the Military in an entirety. As a person of Seniority I will punish my Sodliers to the fullest extent if I have the authority..as for Moon Lake I went to the lake about a month after the car was discovered and it was being drained (not completely) for some sort of construction and someone seen the antennea...also it was put out that a womans body was in the car...the rest I know is all hear say or rumors...like the husband reported to his next duty station single (Alaska)...also that he had reported her missing and that this had happened like 5 years earlier. Bottom line I only know what was put out while I was on Fort Riley..
#28 Feb 3, 2011
Read up on Army Times and Moon Lake. You will have all your answers. Boy were a lot of you wrong in your predictions. Those who assumed the husband was guilty, well you were wrong. Not to sound rude, but honestly a lot of you have nothing better to do then to act like you KNOW everything. I worked on this case and I know for a fact it was CONFIDENTIAL!!!!
#29 Feb 16, 2011
if you needed to know you would know stay out of the buisness of those that dont concern you
#30 Feb 22, 2011
hey buttout, why don't YOU buttout?... no one asked your opinion...
#31 May 4, 2011
I new him but not her i had no idea he was a murder till his ex girlfrends sitster told me
that he got picked up by the u.s marshalls his life is over
#32 Oct 19, 2012
That was my sister in law that was found in that car. While my brother was off fighting for our country, this man took her life while my brother thought she just ran off and left him. Our thoughts and prayers should go out to him and his children.
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For this subject and predicate identification worksheet, students determine the complete subject and predicate and the simple subject and predicate in a group of sentences. Definitions of the terms are at the top of the page for reference. Students underline the complete subject once and the complete predicate twice.
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Bibliografische Details
Titel: Post-War on the Liners: 1945-1977
Verlag: Fonthill Media
Erscheinungsdatum: 2014
Einband: Soft cover
Zustand: Used
This Book is in Good Condition. Clean Copy With Light Amount of Wear. 100% Guaranteed. Buchnummer des Verkäufers ABE_book_usedgood_1781553599
Inhaltsangabe: From the end of the Second World War through three decades, to the 1970s, traditional port-to-port, class-divided passenger ship business carried on. This meant all kinds of ships ? from big liners to small, often rebuilt, ex-coastal steamers. Accommodations varied from luxurious suites with bedrooms, sitting rooms & marble baths in upper-deck accommodations to Spartan dormitories with as many as 50 berths and communal facilities. But the purpose was all but the same: to go from A to B. It was about the destination, whether with 100 pieces of baggage like the Duke & Duchess of Windsor on 5-night Atlantic crossings to little more than an overnight bag for a immigrant on a 6-week voyage from Europe out to Australia. This book examines, through anecdotes & collected experiences, the many passenger ship services of now a bygone era. It is about the diversity and the contrast. There are of course the Atlantic crossings, but also three & four class ships to South America, combination passenger-cargo types carrying only 100 or so travelers, fast mail ships to South Africa, colonial passenger vessels to Mombasa, crowded migrant sailings to Sydney and Auckland, and trans-suez and trans-Pacific passages. All sorts of ships appear: big Cunarders like the Queen Mary, Italy?s Augustus and Britain?s Kenya & Uganda, the Oronsay and Southern Cross and even more remote ships such as the Cap Salinas, Tjinegara, Changsha and Hikawa Maru. It concludes with the closing down, in 1977, of the Union Castle Line?s run between Southampton and the South African Cape, the last regular big liner service in the world.
Über den Autor: Known as "Mr Ocean Liner," Bill Miller has written over 80 books about passenger ships. He has covered the liners large and small, in war and peace, and as collectibles and in post card form. Miller has also penned ovrer 1000 articles about ships and sea travel. He has been a popular guest lecturer aboard such liners as the QE2, QUEEN MARY 2, ROTTERDAM & CRYSTAL SERENITY for over 30 years. Born in Hoboken, New Jersey, just across from the great liner piers of New York City, Miller has appeared in numerous TV documentaries, & news programs and himself has been the subject of a 60-min documentary called "Mr Ocean Liner". Miller has been the recipient of numerous maritime awards.
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Sofia's yellow submarine
Want to travel in your own yellow submarine? The owners of 42 metre Sofia, Moonen's largest yacht yet, sure did. The yacht's owner is an avid diver, so the addition of the sub was a welcome toy to Sofia's arsenal. The submarine-carrying Moonen yacht was specifically designed around the two-person submersible – a C-Quester 3 from the Dutch firm U-Boat Worx. The stern platform had to be lengthened to carry the sub on the transom and weight was redistributed forward to accommodate the added weight. Designed by Rene van der Velden, this elegant explorer yacht offers luxe accommodations and the bonus option to dive deep thanks to the sub.


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Now, smart fridge that tells you what to buy
Sydney: LG has unveiled a voice-controlled fridge that helps you diet and keeps track of your groceries, as well as ovens and washing machines that can be controlled via smartphone.
LG's Smart Refrigerator, released at the Consumer Electronics Show in Las Vegas, comes with a "food management system" that allows users to check what's in their fridge from their smartphone, with granular details right down to the expiration date, the Syney Morning Herald reported.
There are multiple ways to tell the fridge what items are inside. One way is to select from a range of preset items using the fridge's touchscreen but there is also voice recognition and the ability to scan grocery receipts or barcodes using your smartphone.
The Smart Refrigerator also includes a "health manager", which will tell you recipes for things you can make based on what is in your fridge and the personal profiles of family members.
It takes into account age, gender, weight and body mass index to develop daily or weekly meal plans based on personal profiles, says LG.
Users can also order food directly from the touch screen on the fridge, but for such a feature to work LG would need to form partnerships with the big grocery chains.
LG's smart fridge also interacts with LG's "Smart Oven" and if you use the fridge to select a recipe of something to cook, it can automatically send the relevant information and settings to the oven.
On the other hand the company's Smart Washing Machine can download new washing cycle programs from the internet such as "super rinse" and "baby sanitise".
The best part is that LG's smart oven, fridge and washing machine can be monitored – and controlled - remotely from a smartphone or TV set, regardless of the user's geographical location.
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7th Pay Commission: Central govt staff acquiring higher qualification to get enhanced incentives


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*Title: C# .NET Developer * *Location:Cranberry, PA.* *Duration- Long term* *Rate:35-40$/hr on c2c* * * * Need Healthcare EXP* *Must have- 5 yrs of C#* * * *Understanding business requirement document and writing technical write-ups * Designing and developing ASP.Net <http://asp.net/> pages in C# * Create rich user experience screens using WPF and host WPF services in IIS * Performing unit test (NUnit) using Visual Studio Test * Writing technical write-ups and creating SQL Server stored procedures. * Extensive use of stored procedures for performing Selects, Inserts, Updates and Deletes * Optimizing SQL queries for performance. * Participating in code reviews, Team meetings and status review meetings * Implementing the solution on live environment, fixing the problems and working on enhancements for the desired changes.* * * *-- *
** *Thanks!* * SCOTT . Sr Associate Recruiter* *Sriven Technologies LLC* *Office: 703.842.4866X302* *sc...@sriventech.com * * "Technology is dominated by two types of people: those who understand what they do not manage, and those who manage what they do not understand." * -- You received this message because you are subscribed to the Google Groups "REQSRESUMES" group. To post to this group, send email to firstname.lastname@example.com. To unsubscribe from this group, send email to reqsresumes+unsubscr...@googlegroups.com. For more options, visit this group at http://groups.google.com/group/reqsresumes?hl=en.