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Sunlight in Perspective: Pleasure, Sunlight and the
Socio-sensual Environment
Looking back on the historical assemblage of the body in sunlight it is possible to draw certain conclusions, as much with an eye on current and contemporary developments as to the past. In previous chapters we have examined the role that sunlight and the sun's rays have played in various sociotechnical figurations. One significant motif has been the various assemblages that have coalesced around the nexus made up of sunlight, bodies and health. Thus the outdoors movements, such as the Scouts or campers, the treatment of tuberculosis and rickets, the campaigning of the Sunlight League, and even sunlight architecture are drawn together in a common aim: the production of'healthy bodies'. Indeed the 'healthy body'was a crucial element in the attempts to establish the durability of these projects.
Yet ultimately many of these sociotechnical projects unravelled and their network building reversed. For example, the advocates of both heliotherapy and actinotherapy failed in their attempts to constitute these therapies as mainstream medical practices - today even the names of these treatments appear archaic.1 Resistance to these attempted enrolments came from a variety of sources, for example: as Mellanby observed, it proved far simpler to ensure children had an adequate dietary intake of vitamin D than to establish a complex system of sunlamp provision in schools; tuberculosis microbes proved resistant to the sun's rays with heliotherapy only being effective against certain forms of the disease; patients were often less than patient about the time and expense needed for sunlight treatment; and the medical profession were successfully enrolled by the competing collective of allopathic pharmacological medicine as found in the new antibiotics sponsored by the UK's Medical Research Council.
Yet these different assemblages did succeed in establishing an association between health and sunlight. For example, in the context of possible 'cures' for a widely feared illness, such as rickets or tuberculosis, many experts also sought


text: Rot is supposed to be used for medium to high speed films - gives shorter development times than blau. Red variety has been out of production for a long time but do not worry the stuff will be quite o.k. My long time favourite. Quite sharp and can handle wide contrast range without speed loss.

subset: commoncrawl


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Height: 6-foot-3
Weight: 248 pounds
Class Year: SR
Hometown: Powder Springs, GA
Prior School: UCF
As a Recruit (profile)
Class: 2008 (High School)
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The Latest: O'Leary hopes to avoid burning redshirts — 10/23/2008
UCF's offensive depth has taken a hit in recent weeks, but coach George O'Leary is hoping he can avoid burning the redshirt years of several true freshmen.[details]
Kyle Madden College Videos
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Headlines from Rivals.com Network
O'Leary hopes to avoid burning redshirts — 10/23/2008, from UCFSports.com
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DU 11/12 Weekend Commits — 11/12/2007, from StudentSportsFootball.com
DU 10/19: Weekend travelers, more news — 10/19/2007, from StudentSportsFootball.com
Gameday atmosphere impresses Madden — 9/25/2007, from UCFSports.com
Daily Update (9/10): State Recaps/Commits — 9/10/2007, from StudentSportsFootball.com
Knights at the top for Madden — 7/30/2007, from UCFSports.com
Knights offer Georgia TE — 6/26/2007, from UCFSports.com
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Managing Partner at @SoftTech VC (one of the original micro-vcs, before it was fashionable ;). Invested in 160+ companies in 10 years. And really loving it.
Investor in @Attributor, @Discovery Engine, @Heartly, WebMynd, @Blekko, @Dogster, @ScanScout, @Solve Media, @Sundia,Tremor Video, @Ustream, @Ooyala, @Octopart, @Factery.
Entrepreneur, investor. Founder, @AdForce, @Wink. Worked @ Verity, @Apple. Investor/Advisor: @LinkedIn, @Cloudmark, @Wikia, ICONAircraft, @BrightRoll, @Fundly, GAINFitness
Head of Platform, @Disruption. Full-stack engineer. Worked at @TechCrunch, Edgeio. Mentor at 500 Startups. Co-Author of O'Reilly's Redis Cookbook.


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Lila the Arson Dog Toasted at Retirement Party
Lt. Fabian Jackson removed the badge from around the neck of his partner since 2008 at an emotional ceremonyRead More…
Phone: 503-823-3700
Fax: 503-823-3710
55 SW Ash Street, Portland, OR 97204
Station 7, located at 1500 SE 122nd Avenue in Portland, Oregon, serves the Mill Park and Hazelwood neighborhoods. Home to Portland Fire's HazMat Team, Station 7 is one of Portland's largest stations, which houses one battalion chief, two company officers, five firefighters, and two firefighter paramedics. Crews work in three rotating shifts (A, B, and C shift) with 24 hours on and 48 hours off.
The crews operate a wide variety of apparatus including Battalion Chief 3, Truck 7 (advanced life support), Engine 7 (advanced life support), HazMat 7, HazMat 121, and Recon 7. Station 7 is one of 13 HazMat teams in the State of Oregon. In the past year, these trained technicians have responded to 43 HazMat incidents involving situations such as train and vehicle wrecks, trench rescues, fuel, and chemical spills.
Station 7 is Portland's busiest station. In 2009 it responded to over 6,100 emergency calls and averaged 17 calls per day.
The crews at Station 7 are highly involved in their community, especially in their neighborhood elementary, middle, and high schools. Three firefighters are also graduates of David Douglas High School. During football season, they come out to support the David Douglas Scots. Every time the Scots score, they hit the lights and sirens, which is a thrill for the players, students, and parents in attendance.
With over 3,000 students, David Douglas High School is one of the state's largest high schools and has grown 26% in the past 10 years. This venue gives the crews a chance to build relationships with the adults and children in the diverse community they serve. The crew reports that by fostering these relationships, they are perhaps inspiring the next generation of Portland firefighters. Even when attending community events, the crews stand ready to respond to an emergency call within seconds.
Two of Station 7's crew are incorporating the David Douglas mascot, the Scot, into the station logo which will be proudly displayed on Truck 7 once completed. The crews are also involved in events at the elementary and middle-school level and regularly participate in career days, conduct first aid classes, cheer on students at field days toward the end of the school year, and visit classrooms to teach kids about fire safety and what to do in an emergency.
On the evening of January 14th, Portland Fire's Station 7 was buzzing not with the sounds of alarm bells, but with the laughter and chatter of small children. Led by B-shift Lt. Shon Christensen, this station tour was just one of many outreach events that Station 7 holds for the families and children in the neighborhood it serves. On this particular night, they hosted about 50 students, parents, and a translator from Mt. Hood Community College's Head Start Program.
After talking with the kids and families through a translator about what to do in an emergency, demonstrating their turnout gear and air bottles, and letting kids crawl through the cab of Engine 7, the kids moved on for popcorn and a fire safety video in Station 7's family room before heading home for the night.
Just minutes after the last child left, the alarm sounded and Truck 7 was on its way to a commercial fire on Airport Way - all in a night's work for the men and women of Station 7.
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March 3, 2010
I received a call last night about 1AM regarding an alarm at 1117 SE 122nd. You seemed to think it was my building or my business that was there. My only involvement with that building was as an architect for some remodeling work, so I'm not sure why I was called.
As I understand it, the alarm was triggered, but no problem was found at the site, so I was glad to hear that.
I'll call the owner to let them know, just in case you were not able to do that.
Thanks for the help and I'm glad there was no problem. I'd be interested in finding out, however, why I was called. I'm just wondering if my name is incorrectly entered in some data regarding the building. If so, that should be set straight.
Thanks again
Ernest Goble
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1997 Cadillac STS • 140,000 miles
I replaced mosfets on my speed control board for my blower motor to pass safety. It worked fine for a month and tonite my blower is not blowing ! What gives?
December 3, 2012.
December 3, 2012.
Check out the related content below while we wait for the question to be answered by a professional mechanic.
Need help or step by step pictures on how to pull out fan blower motor. on a 1995 cadillac deville 4.9
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2 answers • 2000 Dodge Neon • 145,000 miles
Whats the fastest and easiest way to replace a heater blower motor mine went out and its the middle of winter!
1 answer • 1992 Chevrolet Truck • 345,000 miles


text:
Moisture Festival · Comedy · Varietè
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Tamara "The Trapeze Lady"! Gayly spreading love and glitter from above since 1993. Producer of the Fallen Women Follies, the Columbia City Cabaret and the originator of Burlesque Behind The Pink Door, she's dangled from some of the finest rafters in the world. Seattle's original Aerial Burlesque darling and Infamous Cat Lady Of Lading Island can be summed up something like this; She's played London. She's played France. They've all seen her underpants. Thailand, Spain and Italy have all been left more glittery. Her aerial feats leave them breathless. She dazzles with her wit. Her bawdy antics have them pondering, "Who's she kidding with that LADY bit?". She's always a lady, even when she's on the floor. (wink wink)
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Mary Kay Inc. – Officially Amazing!
Seminar 2013 has come to a close, but it was a whirlwind of excitement - full of glitz and glamour. We taught Dallas a little something about going big for our 50th Anniversary! "How big?" you might ask. WORLD RECORD...
Read More
Playing With Makeup
To commemorate our 50th Anniversary, we are setting a world record by creating a mural entirely from makeup. Four students from The University of North Texas were selected to work on the mural. They share their thoughts on working...
Read More
Don't Call It A Comeback
We've been here for years. 50 years in fact. Help us continue our 50 year legacy of giving back to the community by helping us win $10,000 for The Mary Kay Foundation in the Social Madness National Challenge contest from...
Read More
Pink is my signature color
Everyone who knows me will tell you that I like to shake things up a bit. So, who says a 50th anniversary has to be golden? Why can't it be pink? To honor Mary Kay's Inc.'s 50th anniversary and the arrival of...
Read More
Party over here
We love a good party at Mary Kay, and we are throwing the biggest party of the year. Our annual Seminar starts today, and we are celebrating our golden anniversary. 50 years of enriching women's lives. 50 years of offering...
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Pink Carpet
Summertime in Dallas means triple digit heat but it is also the most wonderful time of the year for Mary Kay. We're celebrating bigger than ever before because we will have 50,000 of our closest friends in "Big D"...
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Two To Tango
When you start a brand new internship at a new company, there is always a bit of anxiety about what it will be like. Will it be the experience of a lifetime, or will you be hovering over the copy machine? Maybe you'll even...
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I should have listened
I love makeup. When I was a teenager and in my 20s, I tried every new makeup trend as soon as it came out. Every time I went for a makeover they told me how dry my skin was. I never understood what they were trying to say, I was...
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Along Came A Spider
Someone once told me that you could tell a lot about a company's culture by how people treat you in the elevator. One of my very first mornings as a summer intern at Mary Kay I was about to enter an elevator in the parking...
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"Fake" Up
Independence Day is upon us. For many that means BBQ, pool-time, and fireworks. It also means summer is almost half way over! Can you believe it? I'm sure it's zooming by for some of you, but there are those reading...
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More Than We Bargained For
1963 was a big year. I was born. And, of course, so was Mary Kay Inc. Now, I am quick to admit that the latter event had a MUCH bigger impact on the world than one tiny baby born in the Nebraska cornbelt. Do you ever think Mary...
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Ashley | Chelsea | Erin | Jennifer |Khadijah | Mizzy | Natalie | Sheilly | Sydney
Recent News
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This week's top rock tracks
LISTEN: This week's top rock songs, according to the latest Billboard chart.
Anne Hathaway expecting first child
The Oscar winner is expecting her first child, according to reports.
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Jack White launches new Third Man Records store in Detroit
Rocker Jack White has launched his first Third Man Records venture in his native Detroit.
WATCH: Holiday parodies of the year's biggest hits
From "Bad Blood" and "Watch Me," to "Uptown Funk," here are the year's hottest songs made fit for the holidays.
OPENING WEEKEND: Family-friendly 'The Good Dinosaur,' boxing drama 'Creed' among Thanksgiving releases
Here's a look at some of the films set to open this weekend.


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Gov. John Kitzhaber in his office at the Oregon State Capitol on Thursday, April 18, 2013.
- Filed Under
Gov. John Kitzhaber and legislative leaders presented a plan to legislators Monday night that they think could provide a path to a deal that includes pension cuts, revenue increases and a small business tax cut.
However, much of the plan is similar to what the Oregon Legislature already saw - and turned down - during the regular session.
The tax cut for small businesses is new and was modeled on a plan originally proposed by Sen. Brian Boquist, R-Dallas, and Sen. Larry George, R-Sherwood. It lowers the tax rate for small businesses who make smaller profits. They're currently all taxed at a 9.9 ...


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FC archers prepare for StatePublished 11:20am Thursday, April 10, 2014
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MEADVILLE -The Franklin High Archery team is looking to repeat as State Champions as they prepare to defend their crown in Jackson next week.
The Bulldogs, coached by Todd Haygood and Connie Johnston, will be in action Wednesday April 16. The FC Archers recently placed first in the Tylertown meet last week.
The Top Archers for FC were the following: Jon Wilkinson 281, Josh Owen 279, Dakota Jones 275, Kenneth Davis 275, Erich Buckles 274, Scout Earls 273, Austin Wallace 272, Blake Smith 272, Leigh Beth Hurst 267, Kelsi Barron 265, Brittany Holloway 262, and Morgan Rushing 260.
Times for the State Archery Meet have yet to be announced, but Franklin County will most likely start shoot in the afternoon session.
The Bulldogs will take part in a tune-up for State, when they host Lawrence County and West Lincoln this Friday for a meet in Meadville. The action is set to begin at 5:30 p.m. at the Bulldog's Gymnasium.


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According to the Daily News newspaper, Morrisons mom says he died,"from full blown Aids," ANYONE can contract it, so its BEST to wait for the right woman, marry her, "then" get BUSY!Posted September 7, 2013 4:45 pm
Old Yank
the voice of reason — Your point is well made. However, it is not unreasonable to see a man as a man — as a father, a brother, a son, a carpenter, a Deacon, a volunteer, a cancer survivor and more. And likewise, it is not unreasonable to see Morrison as a man who was also a fighter. He certainly has a legacy as a fighter and that legacy might be very different than the legacy he created for himself as a man. In my opinion there is nothing wrong per se in reflecting on both when a fighter dies.Posted September 7, 2013 8:38 am
It was AIDS/HIV why are the drs lying.Posted September 7, 2013 5:54 am
AIDS drugs are very expensive, maybe Morrison would rather die young than old and impoverished.Posted September 7, 2013 5:52 am
Buba, morrison didnt even have 5000 between him snd his girl to get outa jail s few yesrs ago, so I highly doubtt he was doing as much coke as u claim. Tommys aids was a killer but his will to live was gone aswell. Tommy could never come to terms about all the money he lost, that could have ben a retirment for him and his family. His come back didnt happen and he list all hope. It ben proven a person will to survive is very powerful.Posted September 6, 2013 3:52 pm
I'm not sure why this subject is worth any media reports, No AIDs mentioned on the Death Certificate means……. Morrison may have had AIDS or he might not have…….. in other words nothing!
Fight Aficionado
Whatever the stated cause was, it's no doubt a byproduct of AIDS.Posted September 6, 2013 1:33 pm
Old Yank
AssyrianGod — Well said!Posted September 6, 2013 11:54 am
Old Yank
the voice of reason — I understand your compassion and your reasoning. However I would encourage you to recognize that Tommy was not alone in denying that HIV/AIDS exists. The great tragedy happening is for those in denial — they are signing their own death sentences. So I would suggest to you that other things DO MATTER that you might not be considering. Tommy was in denial. His family lost him young because of his denial. Perhaps it matters that they speak out against HOIV/AIDS denial groups so others do not have to repeat the tragedy. If the voice of Tommy's family could reach only one man or woman living in denial, then perhaps a life could be spared. And I think that matters.Posted September 6, 2013 11:17 am
Jack Jones
Yes He died, because of aids destroying his immune system. He's still lucky. Because we all gone die of something. When he got his diagnosis he still had many years to live. Others are not so lucky when diagnosed. Like Leuekimia, liver cancer, lung, prostate, colon you may last a few months sometimes weeks. Thats what Im scared of! Not Hiv.Posted September 6, 2013 11:17 am
Old Yank
HIV/AIDS denial is a tragedy. Today treatment is available. Without treatment it is a death sentence. Morrison's family has a great opportunity to be of great service to humanity by speaking out on the dangers of HIV/AIDS denial. They have an opportunity to SAVE LIVES! I pray that they do not squander this opportunity.Posted September 6, 2013 8:48 am
Freddy Marcuree
Even in death Tommy's family is being delusional. Sure he was a good boxer in his day but this will be greatly overshadowed by how he selfishly put others at risk.Posted September 6, 2013 8:12 am
He had AIDS imo, and they should have done an auptopsy, because it would be relevant if he has been sleeping with a lot of women.Posted September 6, 2013 7:50 am
He had AIDS and put other fighters at risk by being in denial. Pseudomonas Aeruginosa Septicemia is contracted by those with autoimmune disorders like AIDS. It's sad to see him go, but you gotta protect yourself.Posted September 6, 2013 6:53 am
Does anyone know how two other fighters diagnosed around the same time as Morrision are currently doing – I am talking about Lamar Parks and Paul Banke?Posted September 6, 2013 5:02 am
the voice of reason
whether tommy really had the virus or not is not the f***ing issue – its a very sad day that this great man – a real powerhouse athlete – has died so young. On his day tommy was a beautiful boxer with a fabulous arsenal of punches – he fought and defeated some top-tier men – tillis, thomas, williams, ruddock, foreman etc – and right there that speaks volumes for just how good a fighter this man was. He was a brave and exciting boxer, who bounced back from two terrifying knockouts to go right to the top – defeats that would have crumbled lesser fighters. Remember him with the respect duePosted September 6, 2013 3:34 am
Tommy was a good kid. An all American boy. He had lots of heart. Rest in Peace Tommy. You gave it your allPosted September 6, 2013 2:11 am
So many show so much disrespect. How can anyone claim that Tommy had AIDS for certain? Are they doctors and did they do an autopsy? Judgement about something they know nothing about.
I remember Tommy in the big fight against Razor Ruddock! It was awesome, not only the fight, but the walk-in music! What a knockout!!! I do not judge Tommy about something I know, and could not possibly know about. I really don't care what killed him, it's a human life that has been lost. Life is short, live your own life while it lasts. Quit trying to live and judge someone other than yourself. Your death will come soon enough, perhaps tonight, tomorrow or 10 years from how.Posted September 6, 2013 1:27 am
Joseph Herron
Guys, no one dies of AIDS.
It's always complications due to the lack of immune system derived from AIDS.
The report says nothing.Posted September 6, 2013 12:28 am
rest in peace TommyPosted September 5, 2013 11:28 pm
RIP – I enjoyed watching you fightPosted September 5, 2013 10:58 pm
I don't mean to be a naysayer on articles but this is kinda tasteless anyway for an article topic. I'm not sure of the mistake or moment that brought this illness on but That's not really for me or I would think anyone else to discuss anyway. We'll all have a final judgment, other than that the man was a good boxer and a human being that deserves a right to some level of privacy or at least decency.Posted September 5, 2013 10:05 pm
may he rest in peacePosted September 5, 2013 10:04 pm
Brazilian Boxing Fan
R.I.P Tommy Morrison.Posted September 5, 2013 9:59 pm
It's Me, Ernie
God Bless the Duke…Posted September 5, 2013 9:21 pm
terrible ending for TommyPosted September 5, 2013 9:21 pm
So sad Tommy was cremated "puff "once here and gone today. Life is preciousPosted September 5, 2013 9:20 pm
Seems people are so fast to forget that its not HIV/AIDS that kill you. its your compromised immune system that cant fight off disease as effectively anymore that kills you.Posted September 5, 2013 8:52 pm
That's exactly right. About the DC. If he had pneumonia because of his compromised immune system the certificate would read respiratory or pneumonia, etc…his health records would be where that info would be foundPosted September 5, 2013 8:32 pm
Old Yank
Pseudomonas Aeruginosa Septicemia is associated with about 50% of all deaths related to AIDS.Posted September 5, 2013 7:29 pm
It's Me, Ernie
And you know this how?…Posted September 5, 2013 6:49 pm
He died of something that AIDS enabled him to catch. His immune system was weakened because of it, got really sick and died. Of course you're not going to hear the truth.Posted September 5, 2013 6:45 pm


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Randy Winn discusses his work with B.A.T. charity
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|Pseudobombax (soo-doh-BOM-baks) (Info)
|longiflorum (lon-jee-FLO-rum) (Info)
This plant is attractive to bees, butterflies and/or birds
Flowers are fragrant
Average Water Needs; Water regularly; do not overwater
6-8 ft. (1.8-2.4 m)
8-10 ft. (2.4-3 m)
10-12 ft. (3-3.6 m)
12-15 ft. (3.6-4.7 m)
15-20 ft. (4.7-6 m)
Full Sun
Pollen may cause allergic reaction
White/Near White
Late Summer/Early Fall
Mid Fall
Late Fall/Early Winter
This plant may be considered a protected species; check before digging or gathering seeds
From woody stem cuttings
From softwood cuttings
From semi-hardwood cuttings
From hardwood cuttings
This plant has been said to grow in the following regions:
Captiva, Florida
Lakeland, Florida
Corpus Christi, Texas
On May 27, 2012, popper1 from Lakeland, FL (Zone 10a) wrote:
Flowers are amazing but short lived. Watching the buds grow can be interesting too. Can get a large caudex at the base.
On Jan 18, 2008, sldel702 from Corpus Christi, TX wrote:
I purchased this tree at an auction and it took 6 years to identify it. I did not know it was a tree. I accidentally sprayed it with round-up one spring. I saturated it from top to bottom. The solution was in the wrong bottle in case you think I wanted to kill him. We realized the mistake about 2 days later so my husband chopped it to the ground. It came back bigger and with many more branches, so we routinely cut it back each spring until I found out it was a tree. It loses all of it's leaves when the flower pods start to develop 2-3 times a year. This tree grew 6-10 feet per year depending on whether or not it was cut back. Growth in height slowed significantly when allowed to reach about 15 ft. and we no longer cut it back. It tolerates the rare light freezes and even rarer snow here. I... read more
On Sep 12, 2004, Monocromatico from Rio de Janeiro
Short (up to 4 meters tall, often less), relatively slow grower, desciduous tree from the brazilian coastal areas.


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Recently I suffered three unexpected losses. I use the word suffered on purpose here. In December I needed to get a flight review. I had scheduled this with three instructors, but due to the holidays, I was unable to get it done. In early January I contacted a local CFI that I know only socially. He knew about the losses in my life. After talking with me a few moments, he gently suggested that I was not well enough emotionally to fly that day. Of course, I burst into tears because he was number four on my list of instructors.
After I got done crying about it, I got to thinking about how, as a professional psychotherapist, I was seemingly unable to see the state of my own mental health. Below is an excerpt of an article I wrote for AOPA Pilot as well as a link for online screening tools for depression, anxiety, bi-polar and PTSD.
Here are some simple ways to put you and your emotional health on the pre-flight checklist as well as some ideas on when to get support if needed.
Mood: Think back over the past week. Rate your mood on a 1 to 5 scale with 1 being the lowest, and 5 being a happy mood. What is your average? Has anyone told you that you look tired, depressed, or nervous? Sometimes our spouse or families are the greatest mirrors for us. We might not see our mood, but to them it is written all over our faces.
Sleep: Have you been sleeping well? The average person in a lab setting will sleep a 6-7 hour stretch and take a 1-2 hour nap in the afternoon. Think back and check whether you have had any difficulties falling or staying asleep. Our deep restorative delta sleep typically happens well into an uninterrupted sleep cycle. Think about performing a go-round on every approach, with sleep we simply cannot get down to delta if the cycle is continually disrupted.
Energy: Has your get up and go, got up and went? Do you find yourself drinking coffee or energy drinks just to get through the day? Some pilots find they have too much energy and are unable to relax into a healthy focus. Between the tortoise and the hare, somewhere in the middle of the two is the most efficient.
Anxiety and Worry: Someone once told me that worry is interest on a debt we don't yet owe. An interesting study on worry shows that it can be healthy in small doses. Worry is a high brain function, one that can help us sort through possibilities and strategies. Too much worry shuts down the function and we can find ourselves in a lower brain: fight, flight, or freeze. 30 minutes of worry once per week is effective. How many minutes this week have you racked up?
Concentration/Focus: Particularly important in being pilot-in command [PIC] is the ability to concentrate and stay focused. If you are noticing that your mind is wandering or you are distracted by worry, it might be best to keep yourself and the aircraft on the ground.
Sex Drive: This might seem a strange item to have on your personal checklist, but the fact is a person's sex drive can be indicative of emotional health. A lack of desire can be suggestive of a mood problem.
Appetite: Does your favorite food taste good to you? Are you eating for comfort or to excess? Healthy food is fuel for the brain and the body. Make sure that you do not fly without fuel on board.
Bumper Sticker: Ask yourself this question and pay attention to the answer: If you had to summarize your attitude about life to fit on a bumper sticker, what would yours say? Is your bumper sticker upbeat and optimistic, or doubtful and negative?
Below is a link for the Mental Health America online screening tools. These screening tools are for use with adults only. If your screening indicates a problem, it is best to contact a licensed mental health counselor in your community for follow-up.
A few days after my crying spell, I completed my flight review and had a great time doing it. My instructor had not flown in a Mooney for some time, and after the necessary maneuvers, I was able to show him a lot about my airplane.
One of my losses was the death of my father who was a primary flight instructor in the Army Air Corp and a Mooney pilot for 30 plus years. The day I was to leave for his memorial I was checking and double-checking the weather. I thought to myself, "I wonder if I am okay to fly?" That was the only question I needed to ask. If you wonder if you are okay, you are not okay. I packed up the car and made the five-hour drive with my son. While an hour and a half in the air is quicker, for me, that day, the drive was safer.
Our mental health is equally important as our physical health. We are all subject to the same rules of stress and loss. I am happy that CFI #3 told me he didn't think I should be flying. His insight could have saved us from a bad outcome. I believe we all do need to have eyes and ears on our fellow pilots. We are a small community and we all get to do something that we love to do. Let's all make sure we are up to the task emotionally too. Thanks for listening.


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I was driving down interstate doing about 65 and I went to pass a car and when I did that I felt the engine shut off. I wasn't sure so I pulled over. I assumed it was the alternator and checked it and came back fine. After further inspecting I realized that the engine oil was not even showing on the oil stick. I try and start it and it just clicks like if it where the starter but its not the starter. If anybody could help me figure it out id appreciate it.
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Diabetes Health Center
Gestational Diabetes: Can I Lower My Risk?
In this article
Your doctor may have told you that you are at high risk for a condition known as gestational diabetes mellitus (GDM). If you develop GDM, you are more likely to have problems during pregnancy and delivery. As many as 18 of every 100 pregnant women will develop GDM.
Exactly how does GDM develop? When you become pregnant, your cells become slightly more resistant to insulin. This causes the amount of glucose or sugar in your blood to rise. The extra sugar helps make more nutrients available to the baby. But if the glucose can't get into cells and levels become too high, it can cause problems for you and your baby.
Learn why you are at high risk for GDM and what you can do to reduce your risk.
Why Am I at Increased Risk?
Even if your doctor explained that you were at high risk for GDM, you may not have completely understood why. Certain risk factors increase the chances you will get GDM. But no one can say for sure you will get it, just based on risk factors.
You are at increased risk if you:
- Are Hispanic, African-American, Native American, Asian American, or Pacific Islander
- Were overweight before your pregnancy
- Have a family member with diabetes
- Are 25 or older
- Had GDM in a previous pregnancy
- Had a previous very large baby (9 pounds or more) or a stillbirth
- Have had abnormal blood sugar tests before
Talk to your doctor to better evaluate your risk.
How to Lower Your Risk--Diet
If your risk of GDM is high, your doctor or a nutritionist may suggest you eat more healthfully. They can help you choose foods designed to keep your blood glucose within a healthy range.
- Your health care professional can tell you which foods to eat and which to avoid. She or he can tell you about ideal portions and meal timing.
- You should also limit sweets and track how many carbohydrate-rich foods you eat.
- It's also a good idea to include fiber in your meals. This can be from fruits, vegetables, whole-grain breads, whole-grain crackers, and cereals. One large study looked at diets of women before they got pregnant. Each 10-gram increase in fiber a day reduced their risk of GDM by 26%.
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Super 8 Saanichton Victoria Airport - Saanichton
- Free continental breakfast, free WiFi, and free parking
Main amenities
- 50 smoke-free guestrooms
- 24-hour front desk
- Air conditioning
- Free newspapers in the lobby
- Luggage storage
What's around
- Panorama Recreation Centre (2.8 mi / 4.4 km)
- Butchart Gardens (4.9 mi / 7.9 km)
- John Dean Provincial Park (3.7 mi / 6 km)
Rooms were clean and comfortable and close to food
Old original motel, very worn out but clean with friendly staff.
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Hotel Info: from Rs2,904
- Double Room, 2 Double Beds, Non Smoking
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- Double Room, 2 Double Beds, Non Smoking
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- This hotel has 50 rooms
- This hotel is arranged over 2 floors
- Check-in time starts at 3 PM
- Check-out time is 11 AM
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Required at check in
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- Minimum check-in age is 18
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- Pets allowed *
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- Up to 0 lb
- Free WiFi in public areas
- Free WiFi in rooms
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- Free continental breakfast daily
- 24-hour front desk
- Free newspapers in lobby
- Luggage storage
- Number of buildings/towers - 1
- Disabled parking
- Accessible path of travel
- Accessibility equipment for the deaf
- Accessible bathroom
- In-room accessibility
In the room
- Climate control
- Air conditioning
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- Private bathroom
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- Hair dryer
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Deposit: CAD $100 per stay
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Published by: American Academy of Orthopaedic Surgeons
Edited by: Leesa M. Galatz, MD
OKU: Shoulder and Elbow 3 is a thorough overview of relevant shoulder and elbow research and new clinical advances from 2002 – 2006. Developed by American Shoulder and Elbow Surgeons and published by AAOS, this new edition features the latest clinical studies, classifications, and approaches to treatment.
Expand your knowledge of current and evolving treatment and management issues for shoulder and elbow conditions and support your treatment recommendations.
Shoulder and Elbow 3 reviews relevant topics in the following areas:
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Nikola Tesla. One of the greatest minds of the 20th century. He devised plans for worldwide peace, had invented hundreds of inventions, and fathered no children. Why? Because he was a firm believer in eugenics, and did not believe he was good enough to carry on his genes to the next generation.
Now, if Nikola Tesla wasn't good enough to reproduce, where does that leave most of human society? In the sphere of inferiority.
As all biologists will tell you, evolution is the slow process of adaptation and weeding out of those that can't survive. In our first world society, where almost anyone can breed and have children this is a setback. To insure the survival of our species, we need to test every new generation for susceptibility to disease, intelligence, and muscle gain. By sterilizing those who are weak in all three areas, we will insure that the next generation is smarter, stronger, and less likely to die from disease.
Now the most foreseeable problem with this, is that it takes away the freedom to bear children. However, if sterilized women or men want to raise children, they can easily do so as a babysitter or a professional nanny.
By limiting the amount of people that can have children, this world will have a good start on stopping overpopulation. There is also a new argument against racism, because if you exist in this future and can have children, then you are not inferior. Also, the fact that there is diversity in this future proves that all "races" are equal.
By eliminating the issue of overpopulation, we can give more aid to those who need it. Education and healthcare will be available to more, and crime will go down as well. If this future is enacted, we're looking at a society of intellectuals with plentiful resources and with little violence. This was the vision of 20th century eugenicists, without the arrogance of racism and racial superiority or the idea that white people would dominate everyone else.
This ideal world is the human dream. Evidence of this can be seen in many different religious doctrines as ascension from one form to another. Whether judeo-christian or pagan, or atheist, or Buddhist, living in a higher plane of being is one of the main attractions. This is also why immigrants came to North America, and why barbarians conquered other civilizations. This is why democracy and communism and ignorance and fascism were created, so that people could live in a perfect world, and that perfect world isn't going to be created by violence and hatred, it's going to be created by higher human beings.
The cost of this perfect world is small. Compulsory sterilization for those that fail the tests and obedience. But, with a few speeches and the knowledge that they're putting future generations ahead of themselves I believe that loyalty will be easy to gain.
No longer will one people take advantage of the rest in this world of scholars and doctors. No more will education have to choose between two students, and no longer will anyone be left behind.
In this perfect world of perfect humans, there will be enough food for everyone, because of the current rate we're farming now. Jails and prisons will be similar to Norway, meaning rehabilitation over punishment, and in a few thousand years, perhaps the average Intelligence Quotient will be 120 instead of 100, because links have been found between genes and Intelligence Quotient.
As the future looms in the distance, I see my home and planet in the hands of these versatile supermen, where resources are spent researching technology to further improve the human condition instead of nuclear death fleets.
I also see an alternate future in the hands of our children. There is still war, and struggles, but the world does improve, just at a slower more primitive pace. Progress is never halted until our extinction.


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November 29, 2015
WASHINGTON (AP) -- The Treasury Department, facing huge borrowing needs because of the government's $700 billion rescue program, says it will increase the size of Treasury securities being sold to the public.
The Treasury said Wednesday it will expand the number of Treasury securities which are currently being traded as a way to boost borrowing and fund programs such as the $700 billion financial bailout plan.
In a statement, the department said this will also deal with what it called "severe dislocations in the market." The demand for safe Treasury securities has soared during the current credit crisis.
(Copyright 2008 by The Associated Press. All Rights Reserved.)
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Katcoff, Hannah BA*; Wenzlaff, Angela S. MPH†; Schwartz, Ann G. PhD, MPH†
*Department of Epidemiology, University of Michigan School of Public Health, Ann Arbor, Michigan; and †Karmanos Cancer Institute, Department of Oncology, Wayne State University School of Medicine, Detroit, Michigan.
Disclosure: The authors declare no conflict of interest.
Address for correspondence: Ann G. Schwartz, PhD, MPH, Karmanos Cancer Institute, 4100 John R, Detroit, MI 48201. E-mail: email@example.com
Introduction: Although lung cancer is the leading cause of cancer death in women, few studies have investigated the hormonal influence on survival after a lung cancer diagnosis and results have been inconsistent. We evaluated the role of reproductive and hormonal factors in predicting overall survival in women with non–small-cell lung cancer (NSCLC).
Methods: Population-based lung cancer cases diagnosed between November 1, 2001 and October 31, 2005 were identified through the Metropolitan Detroit Surveillance, Epidemiology, and End Results Registry. Interview and follow-up data were collected for 485 women. Cox proportional hazard regression models were used to determine hazard ratios (HRs) for death after an NSCLC diagnosis associated with reproductive and hormonal variables.
Results: Use of hormone therapy (HT) was associated with improved survival (HR, 0.69; 95% confidence interval, 0.54–0.89), adjusting for stage, surgery, radiation, education level, pack-years of smoking, age at diagnosis, race, and a multiplicative interaction between stage and radiation. No other reproductive or hormonal factor was associated with survival after an NSCLC diagnosis. Increased duration of HT use before the lung cancer diagnosis (132 months or longer) was associated with improved survival (HR, 0.54; 95% confidence interval, 0.37–0.78), and this finding remained significant in women taking either estrogen alone or progesterone plus estrogen, never smokers, and smokers.
Conclusion: These findings suggest that HT use, in particular use of estrogen plus progesterone, and long-term HT use are associated with improved survival of NSCLC.
In the United States, lung cancer is the leading cause of cancer death in both men and women. It is estimated that in 2012, 116,470 men and 109,690 women were diagnosed with lung cancer and 87,750 men and 72,590 women died from the disease.1 Since 1990, lung cancer mortality in men has been decreasing, whereas in women mortality has only seen a slight decrease since 2004.2
Many studies have explored sex differences in lung cancer incidence. Although differences in smoking patterns in men and women contribute to the variations in lung cancer risk, these differences do not fully explain incidence rate variation.3 That women are more likely to develop adenocarcinomas and develop cancer at a younger age suggests alternative biological explanations.4 Reproductive and hormonal factors contributing to the risk of lung cancer have been studied, particularly the role of estrogens in cancer development, with inconsistent results.5–13
Sex differences also exist in survival; female lung cancer patients have better survival compared with the survival in male lung cancer patients.14–16 The influence of reproductive factors on lung cancer survival has not been extensively studied. Moore et al. investigated the influence of menopausal status on outcomes of lung cancer and found that even though premenopausal women had tumors of an advanced clinical stage, overall survival was not significantly different from survival of postmenopausal women. The authors suggest that lifelong exposure to estrogen may offer a protective effect in lung cancer progression.17
Research on hormone therapy (HT) use and outcomes from lung cancer is limited, and results have been mixed. In a retrospective study, Ganti et al.18 found that women taking HT had decreased survival compared with women who had never used HT. The Women's Health Initiative (WHI) randomized control trial study showed that the use of estrogen-only HT was not associated with incidence or mortality from lung cancer.8 In the WHI study, combined estrogen and progesterone HT use was associated with an increased mortality, but not associated with incidence of lung cancer among postmenopausal women.9 Two other studies did not reveal an association between HT use and lung cancer outcomes.19,20
The purpose of our study was to investigate whether reproductive factors are associated with overall survival in female non–small-cell lung cancer (NSCLC) patients and to further explore the role of HT use on survival of women with NSCLC.
Lung cancer cases diagnosed between November 1, 2001 and October 31, 2005 were identified through the population-based Metropolitan Detroit Cancer Surveillance System, a member of the National Cancer Institute's Surveillance, Epidemiology, and End Results program. Women aged 18 to 74, diagnosed with NSCLC, and residing in Wayne, Macomb, and Oakland were eligible to participate. Eligibility criteria were originally restricted to adenocarcinoma cases, but were extended after November 1, 2004 to include all other types of NSCLC as many histologic diagnoses were not more specific.
Detailed in-person interviews were completed for 577 women (60%); 273 women refused participation and 129 reported being too ill to participate. Participation rates were 58% in white and 63% in African American women. Cases with reported race other than African American or white (n = 13), women with unknown menstrual status or those whose menses were possibly affected by previous lung cancer treatment (n = 20), and women with a previous breast cancer diagnosis (n = 59) were excluded. After applying these exclusion criteria, 485 women with NSCLC were included in our analysis.
The study was approved by the institutional review board and all participants signed a written informed consent. Surveys collected demographic information, medical history, smoking history, reproductive history, and environmental tobacco exposure. Reproductive history included age at first birth, age of menarche, age of menopause, oral contraceptive use and duration before lung cancer diagnosis, and hormone use and duration before lung cancer diagnosis. Details of HT type (estrogen only, estrogen and progesterone combined, and progesterone only) and dose were collected. Data collected on a number of risk factors for NSCLC have been published previously.21–24 NSCLC diagnoses dates, histology, and treatment data were collected through the Metropolitan Detroit Cancer Surveillance System.
Statistical analyses were carried out using SAS version 9.2 (SAS Institute, Cary, NC). Student's t tests were used to compare means of continuous variables, whereas comparisons of categorical variables were performed using χ2 tests. A Wilcoxon rank-sum test was used to compare medians between groups. All demographic, treatment, reproductive, and hormonal variables were first included in a univariate Cox proportional hazards regression model to assess the influence of the variable on survival. Nonhormonal and nonreproductive factors such as stage, treatment with surgery, treatment with radiation, income, age at diagnosis, pack-years, and race were included in all models. Stepwise regression methods were used to identify reproductive and hormonal variables associated with outcome. Reproductive and hormonal factors that met significance at the p value less than 0.05 level remained in the model. Additional models included HT duration (categorized: 0 months, 1–41 months, 42–131 months, 132 months, or longer before lung cancer diagnosis). HT duration was selected as the measure of exposure for additional analyses because dose data were missing for 41% of women reporting progesterone use and 45% of women reporting estrogen use. Of those women reporting doses, the majority reported using doses of 2.5 mg of progesterone and 0.625 mg of estrogen. With little variation in reported dose, duration of use was used to evaluate cumulative exposure. Models stratifying on menopausal status, HT type, and smoking status were also developed. No interaction between ever smoking and hormone use was found in any of the models. A level of 0.05 was used to assess the statistical significance of p values in all analyses.
Subject characteristics are listed in Table 1. Of the 485 women, 76.9% of the cases were white, and most were current or former smokers (92.3%); 91.6% of the women were postmenopausal and 72.0% had used oral contraceptives; 72.8% of the cases presented with an adenocarcinoma histology. Stage of diagnosis was balanced with about one-third of the women diagnosed in each of the local, regional, and distant stages.
Of the 485 women, 230 women (47.4%) had taken HT. Women who had taken HT were more likely to be white (p< 0.001), to have quit smoking (p = 0.03), had a higher education level (p < 0.001), and were at a higher income level (p = 0.02) compared with those who had never taken HT. HT type was determined for 187 women of the 230 who used hormones. Of those who used hormones, 99 women had taken estrogen only, three had taken progesterone only, and 85 had taken the combined estrogen and progesterone formulation. The women taking the combined therapy were older at menopause (mean years 46 versus 41.4; p < 0.001), but there were no other significant differences between the users of each hormone formulation in race, smoking status, pack-years, HT duration, or education level.
After adjusting for stage at diagnosis, surgery, radiation, education level, pack-years, age, race, and a multiplicative interaction between stage and radiation, the only reproductive or hormonal factor that predicted survival after NSCLC diagnosis was hormone use (hazard ratio [HR], 0.69; 95% confidence interval [CI], 0.54–0.89) (Table 2). Although HRs were reduced for all duration of use categories, hormone use of 132 months (11 years) or more before lung cancer diagnosis was significantly associated with better survival (HR, 0.54; 95% CI, 0.37–0.78). Age at first birth, age of first menstrual period, age of last menstrual period, number of pregnancies, number of children, oral contraceptive use and duration, infertility, or average length of days between menstrual cycles did not predict survival.
Ever use of HT did not significantly predict survival for women who took estrogen-only HT (HR, 0.79; 95% CI, 0.59–1.04), unless use of estrogen was for 11 years or longer in which case survival was improved (HR, 0.58; 95% CI, 0.37–0.92). Hormone use for those taking combined estrogen and progesterone was associated with significantly improved survival (HR, 0.64; 95% CI, 0.47–0.86). Although HRs were reduced for all duration of use categories, duration of HT was only statistically significantly associated with survival among those with 11 years or more of combined estrogen and progesterone use (HR, 0.50; 95% CI, 0.30–0.83) (Fig. 1).
The significance of hormone use did not change when restricting our analyses to postmenopausal women (n = 444). As in our analysis of all women, duration of more than 11 years of HT use was significant in this subgroup analysis. Hormone use was also predictive of survival in both never smokers and in ever smokers, and duration of HT use of 11 years was significantly protective in both groups.
Lung cancer was the recorded cause of death for 84.5% of the cases who died. Ever use of hormones remained significant when restricting the analyses to women with a lung cancer–specific cause of death (HR, 0.69; 95% CI, 0.53–0.90). Ever use of hormones was also significantly associated with survival when the analysis was restricted to women with adenocarcinoma of the lung (HR, 0.65; 95% CI, 0.49–0.87).
In this retrospective study, several reproductive factors were tested for their influence on survival after an NSCLC diagnosis. Age at first birth, number of children born, number of pregnancies, oral contraceptive use and duration, age of first or last menstrual period, infertility, and average length of days between menstrual cycles did not predict survival of NSCLC. However, increased duration of HT use was associated with a decreased risk of death after an NSCLC diagnosis. These findings suggest a complex relationship between exposure to exogenous hormones and lung cancer outcomes.
Research examining the relationship between reproductive factors and lung cancer survival in women has been limited. Skuladottir and Olsen25 examined whether reproductive patterns could predict outcomes of lung cancer in both men and women. They observed that women without children had worse prognoses than did parous women. The authors found a similar result in men without children, and concluded that the finding was not the result of hormones, but likely the result of lifestyle factors such as socioeconomic status. A more recent cohort study explored the impact of parity on the risk of death from lung cancer in Taiwan. With increasing parity, there was an increased risk of death although this trend was not significant (p = 0.25).26 That study was limited to premenopausal women, whereas our study is composed mainly of postmenopausal women. We did not find a significant association between parity and increased risk of death. Plasma estrogen levels are increased during pregnancy,27 but the precise role of estrogens in lung cancer development and progression is still not fully understood.28 To our knowledge, to date, there have been no other findings on reproductive factors and effect on survival outcomes for lung cancer.
Observational studies on HT use and lung cancer outcomes have been few and have resulted in conflicting findings. Table 3 summarizes the literature in this area. Our study supports the findings of an observational study that reported reduced lung cancer mortality among long-term hormone users (defined as use of 15 or more years) (relative risk, 0.22; 95% CI, 0.04–1.15).29 Huang et al. observed that postmenopausal women with lung cancer and a history of HT use had an increased survival time, but the difference was not statistically significant (p = 0.12). The authors did find significantly better survival among smokers who had taken HT compared with smokers who had not taken HT (median survival, 16.2 versus 10.4 months; p = 0.04); however, this result was not significant in a multivariate analysis.20 In our study, hormone use and longer duration of use were associated with an increased survival of NSCLC among both smokers and never smokers in multivariate analysis. Conversely, another more recent study found that women with lung cancer taking HT were at significantly greater risk of death compared with women with lung cancer who had never taken HT (HR = 1.97; 95% CI, 1.14–3.39), and this relationship was even more dramatic among those with a smoking history.18 None of these studies reported HT duration or distinguish between the types of HT taken.
In our study, HT use did not predict survival among those who took estrogen only, except among long-term users. HT use was associated with better survival among women who had taken combined estrogen plus progesterone and this result was pronounced for women taking combined HT for 11 years or longer. Our results contrast those of the WHI's randomized controlled trial study which assessed use of estrogen alone among postmenopausal women who had a hysterectomy and combined estrogen plus progesterone in postmenopausal women with no previous hysterectomy.8,9 These studies concluded that although estrogen-only therapy did not influence lung cancer incidence or mortality, combined estrogen plus progesterone use increased the number of deaths from lung cancer. There are advantages to the WHI trial including the randomized double-blind design with standardized dosing and centralized review of lung cancer outcomes. The clinical trial design more effectively controls for potential confounding associated with factors that affect HT use and outcomes such as race and smoking history. There are also limitations to the WHI studies such as a lack of treatment data and limited time of HT use.
The epidemiologic literature as described remains inconsistent. None of the studies directly address the same question because timing of use, type used, dose and duration of use, and unmeasured confounders vary between studies limiting the conclusions that can be drawn. The role of hormone exposures 11 or more years before a lung cancer diagnosis may impact outcomes differently than shorter exposures closer to the time of diagnosis, or use after diagnosis. Women using HT may also be different from women not using HT in terms of unmeasured confounders such as comorbidities, socioeconomic status, body mass index, or differential interactions with the medical system that affect outcomes.
The mechanisms underlying the association between estrogens and lung cancer are being evaluated in preclinical and clinical settings. Estrogens promote both cell proliferation and the transcription of estrogen-responsive genes. Estrogen action is through two distinct receptors, estrogen receptor (ER)-α and ER-β, both of which are expressed in the lung and localized in the cytoplasm and in the nucleus.30,31 Estrogen can be synthesized in the lung by aromatase, and high aromatase expression has been associated with poor prognosis in postmenopausal women with early lung cancer.32 Progesterone action, mediated by progesterone receptor (PR), is thought to stimulate tissue differentiation and inhibit cell proliferation.31 Progesterone supplementation has been shown to inhibit the growth of PR-positive lung tumors in mice.33 The interplay between estrogen and progesterone in lung carcinogenesis is not well understood and, in the epidemiologic literature, combined HT seems to be driving findings.
Stabile et al.34 report that both cytoplasmic and nuclear expression of ERs and PR predict overall survival and time to progression. High cytoplasmic ER-β is associated with reduced survival, whereas low total PR is a negative predictor of time to progression, even after adjustment for age, stage, sex, and smoking. Patients with the combined expression characteristics of low ER-β, low aromatase, low EGFR, and high PR had shorter overall survival compared with patients with the opposite pattern (HR, 6.6; 95% CI, 1.7–25.2). Expression varied by smoking status with never and former smokers having higher expression of nuclear ER-α, cytoplasmic PR, and total PR. Overall survival was higher in women compared with men, and no survival differences were noted by menopausal status. In an earlier study we conducted, which included some of the women from this current study, we evaluated the role of ER-β expression in lung tumors on survival; we also had HT information for some of the women.35 In that study, ER-β expression was not associated with use of HT before diagnosis or with any reproductive factors, but this was a small study with limited follow-up. If outcomes are truly mediated by tumor expression of ERs and PR, the role of HT in lung cancer mortality may be dependent on tumor characteristics that have not been evaluated in the epidemiologic literature.
Strengths of our study include the use of a large population-based sample, detailed information on HT type, duration of HT, the inclusion of a large proportion of cases with a history of HT use (n = 230, 47.5%), and the long-term follow-up of cases. Some limitations are of note. Reproductive and hormonal factors are based on recollection, so there is a potential for recall bias. We did use a calendar to trigger memories of important events in the woman's life to minimize this bias. Overall, the study had 80% power to detect an HR associated with the use of HT of 0.73. Some of the stratified analyses included smaller numbers of women, so those results should be interpreted with caution. Women in the study were healthy enough to participate in the survey and this is reflected in longer overall survival than might be expected for lung cancer cases, so this study may not be generalizable to all women with NSCLC.
In conclusion, our study examines the influence of both reproductive and hormonal factors on NSCLC outcomes. Hormone use was associated with increased survival, particularly with use for 11 or more years, and especially among users of combined estrogen plus progesterone. With few consistent results in the literature, more research examining the biological significance of long-term HT use on lung cancer outcomes is needed, with better characterization of tumors in terms of ER and PR expression.
This research was funded in part by NIH grants R01-CA87895, and contracts N01-PC35145 and P30CA22453.
Non–small-cell lung cancer; Hormone use; Survival
Copyright © 2014 by the European Lung Cancer Conference and the International Association for the Study of Lung Cancer.
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Wesley Carroll Matthews
Position: WR
Height: 5-10 Weight: 180 lbs.
Born: October 10, 1943 in San Antonio, TX
High School: Muskogee
College: Oklahoma St., NE State (OK)
Weighted Career AV (100-95-...): 0 (14739th overall since 1950)
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Chris Berman has done just about everything during his ESPN career.
Even for him, this will be a first.
ESPN announced Wednesday the broadcaster will do the play-by-play for the Chargers' "Monday Night Football" season opener on Sept. 10 against the Raiders.
Hired in 1979, Berman has gone hand-in-hand with the network's NFL coverage, hosting "Sunday NFL Countdown" for going-on 28 years. But he's never been in the commentator's booth to call a game.
Berman said he was surprised upon learning of his Oakland assignment.
"I went, 'Wow. Now there's something I hadn't really thought about,' " Berman said in a podcast, part of ESPN's official release. "Of course, I've thought about calling NFL games my whole life, but given what I do and the Sunday duties ... I didn't see it coming."
After Berman was told, word spread. Sports By Brooks first reported his forthcoming booth debut last week, the news drawing a mix of jeers and cheers.
Sports Illustrated reporter Richard Deitsch wrote on Twitter last week: "If Chris Berman calls the game, ask yourself if you are being best served as an NFL fan by that assignment."
Berman and ESPN analyst Trent Dilfer, who will provide color commentary, have a warmup assignment of sorts, hosting a "Monday Night Football" preseason game Aug. 23 between the Cardinals and Titans.
Berman still will host ESPN's three-hour "Countdown" show on opening Sunday. He'll then fly cross-country for his first regular-season shot in the booth, the second part of a "MNF" double-header.
The AFC West rivalry game is set for 7:15 p.m. PT.
"It's nice to be surprised after 32 years," Berman said with a laugh. "It really is."
firstname.lastname@example.com (619) 293-1853 Twitter: UTGehlken


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Rep. Kirkland is fighting at the state level to help fund Chester Upland School District. He introduced a bill that would provide $25 million in state funding to Chester Upland for the purpose of easing the school's structural deficit. Once the deficit would be closed, the remaining balance would be used for capital improvement projects. Read more
Chester Children's Chorus takes center stage; public safety tops my legislative goals; annual Christmas luncheon a continued success and much more!
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Pennsylvania's children have a new protection thanks to Hannah's Law, named after Hannah Ginion of Bristol Township, who suffered from the rare genetic disorder called Krabbe disease. Hannah passed away on Dec. 28, 2014. Read more
Pennsylvania has a Children's Health Insurance Program that provides free and low-cost health insurance to children. CHIP is not welfare – it covers children of working parents whose employers don't offer coverage and who can't afford private insurance. If you can't afford health insurance for your children, chances are they are eligible for CHIP. Read more
Each year, the Pennsylvania Treasury receives millions of dollars of unclaimed property. It is estimated that roughly one in 10 Pennsylvanians has unclaimed property. Read more
Landmark legislation passed during the 2013-14 Legislative Session now allows first responders, including law enforcement, fire fighters, EMS or other organizations the ability to administer a medication known as naloxone, a life-saving opioid-overdose antidote.
The law also allows individuals such as friends or family members who might be in a position to help a person at risk of experiencing an opioid-related overdose to obtain a prescription for naloxone. Additionally, Act 139 provides immunity from prosecution for those responding to and reporting overdoses.
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House Bill 993, which renamed the Pennsylvania Department of Public Welfare as the Pennsylvania Department of Human Services, was signed into law as Act 132 during the 2013-14 Legislative Session.
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- Product Information
Product Description
Our Black Ticket custom invitation is the perfect choice to invite guests to a special celebration! Our Black Ticket invitation is great customize for birthday parties, anniversary parties, sports theme events, Bar or Bat Mitzvahs and Hollywood parties. Black Ticket invitations are available in 2 sizes, 4" x 6" or 5" x 7" with matching size envelopes. Choose from 12 type styles and 24 colors when you customize this invitation for a special party. All custom invitations are printed on hiqh quality card stock and are sold in a minimum of 10 invitations. Additional invitations can be ordered in 2 invitation increments.
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- Whimsical B&W Ticket Invite Review by SweetSWGrl
Great value for ticket invites; love the quality and design. The only drawback is it took longer, but I didn't want to pay the higher shipping costs. Everyone loved these invites.
(Posted on 10/20/13)
- Very professional looking Review by Nicky
The invitations were super cute and really fit with our theme. Wish we would have had more flexibility in what we could write on them but other then that they were great.
(Posted on 8/23/12)
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10 Ways to Decorate Your Office or Classroom this Holiday Season
Add Christmas decorations to your office, cubicle or classroom to bring some festivity into the work place this holiday season! Get inspiration from some of these decorating ideas.
How to Make Your New Year's Eve Party the Easiest Party You've Hosted
Don't sweat the small stuff this New Year's Eve—planning for a party becomes a piece of cake with the help of New Year's Party Kits!
Star Wars Theme Party Ideas
Celebrate the opening of the new Star Wars film with a Star Wars theme party. Put together an out-of-this-world affair with the help of these party tips!


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LEGO Harry Potter: Years 1-4
Question from newgamer28
How do you get the house crest in level A Jinxed Broom?
There is part of the house crest up above where you build a lego thing & i have no idea how you get it! Any suggestions?
5 years ago
When you get to the part where the Slytherin Prefect is there is a fence to the left of him. You need to jump the other side of it and continue left.
5 years ago
As crago2000 says, to the left of where the prefect was is a stack of baskets and other assorted trash next to a wall. Do not destroy the items and use them to jump over the wall and head left. This was actually the last one I needed and I felt quite annoyed when I realized how easy it was.
5 years ago
Are you talking about the fence that is right before you go up the ladder towards the end? if so how do you get over it ive tried and it wont let me....
5 years ago
Actually, you are right muntfutter, but if you have trouble finding the house crests, then go to the owl emporium and buy the hogwarts crest decetor and mant other detectors.
3 years ago
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|I need help finding/getting to the Slytherin Crest in a Jinxed Broom?
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Somali endorses new parliamentComment on this story
Mogadishu -
Somalia's new parliament endorsed on Tuesday all 10 ministers appointed by the prime minister, approving one of the smallest ever cabinets for the war-ravaged nation.
Two women are among the 10, including Somalia's first female foreign minister, Fowsiyo Yusuf Haji Adan, who hails from the self-declared independent state of Somaliland.
"Lawmakers have endorsed the new cabinet with a majority vote, 219 members out of the 225 who attended the session gave the 'yes' vote to the new cabinet," said parliament speaker Mohamed Osman Jawari.
Three others rejected the cabinet, and three abstained.
"The lawmakers have endorsed the new cabinet, and now they have to face the difficult tasks ahead," lawmaker Aweys Al-Qarni told AFP.
The new government faces tough challenges as it seeks to install order in a country racked by decades of war, and with al-Qaeda-linked Shebab insurgents vowing to overthrow the Western-backed administration.
Somalia has been in political chaos and deprived of an effective central government since the fall of President Siad Barre in 1991.
The new administration led by President Hassan Sheikh Mohamud - who took office in September - ended eight years of transitional rule by the corruption-riddled government. - Sapa-AFP
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Funny Or Die
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Keyboard Cat Videos, Keyboard Cat Pictures, Keyboard Cat Articles
Play Corey Haim Off, Keyboard Cat
Cory Haim likes drugs. Keyboard Cat likes Corey Haim.
Play B Boy Off, Keyboard Cat
Pop and Lock with the Keyboard Cat
J-Stache - Official Trailer
Official Trailer for J-Stache, premiering exclusively on Funny or Die.com!
Kato Kaelin Keyboard Cat
Frankly, here at Tosh.0, we're getting a little sick of keyboard cat popping up everywhere. That's why we decided to make a better, new and improved keyboard...
Play Helen and Hall & Oates Off, Keyboard Cat
Sometimes an internet meme doesn't know when to stop, remixes with Hall & Oates might be the end of the keyboard cat as we know it.
Play Colbert Off, Keyboard Cat
Stephen used to be as cool as a cucumber in sunglasses before Keyboard Cat. Now he's challenging him to keyboards at dawn.
Play Him Off, Keyboard Cat
You almost start thinking he's going to be fine right before the keyboard cat takes him out.
Play Cheney Off, Keyboard Cat
Watch poor Dick Cheney squirm and try his best to defend the Bush record. And yet, the keyboard cat somehow makes it all good. Take it away, Keyboard Cat.
Play Kirk Out, Keyboard Cat
The wrath of keyboard cat


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3 to 6 days
Free delivery when you spend over £75 (GBP).
Will be calculated at checkout
Knitted jumpers - Blue
You must select a size
Knitted jumper from MORRIS. Rounded neckline and appliqués to the front. Ribbed neck, cuffs and hem.
Made of 100% Cotton.
Morris designs clothing for men and women that are classic, with a British influence. They seek a more raw, preppy style, that appeals to a younger clientele, through vivid colors and fashionable cuts. They create fashion like polo shirts, dress shirts, jackets, coats, pants, and much more. Shop Morris here at Nelly.com UK.
Free delivery when you spend over £75. Standard shipping £3.95. Delivery time: 3-6 days. We deliver with UPS.
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7100 Wayzata Blvd
Minneapolis, MN 55426
Sunday closed (show)
Monday 8:00am - 8:00pm
Tuesday 8:00am - 8:00pm
Wednesday 8:00am - 8:00pm
Thursday 8:00am - 8:00pm
Friday 8:00am - 6:00pm
Saturday 8:00am - 5:30pm
Sunday closed (hide)
Committed to Excellence-Dedicated to Caring
|MON 8:00am - 8:00pm
|TUE 8:00am - 8:00pm
|WED 8:00am - 8:00pm
|THURS 8:00am - 8:00pm
|FRI 8:00am - 6:00pm
|SAT 8:00am - 5:30pm
|SUN closed
Category: Car Dealers
Impressed with Service and Price
by lg725 , 09/10/2007
I purchased a vehicle from Lupient about 7 months ago and was amazed by the customer service and the ease of making the purchase. We were in and out in what seemed like minutes, and drove away in our new car. I would definitely recommend Lupient. They go above and beyond to make sure their customers are happy.
View all reviews for Jim Lupient's Buick-GMC of Golden valley


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Berlin - The owner of the heavily used YouPorn and Pornhub websites has been extradited to Germany after being arrested in Belgium on suspicion of tax evasion.
Fabian Thylmann, a German who lives in Belgium, was extradited on Thursday. Alf Willwacher, a spokesman for Cologne prosecutors, said Friday that Thylmann didn't contest extradition. It wasn't clear whether he has responded to the accusations against him.
Thylmann was arrested last week on a German warrant, and several properties including his home were searched. Prosecutors have declined to give details of the case against him.
Thylmann's company, Manwin, owns several sites including YouPorn and Pornhub which, according to Internet information company Alexa, are among the most popular websites on the planet. Manwin has offices in Luxembourg, Britain, Germany, Canada, Cyprus and the United States. - Sapa-AP
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text: Rollover Accident Closes Minnesota Avenue By Tea Storm Chasers April 5, 2013 1:43 PM Share on Facebook Share on Twitter Tea Storm Chasers/KSOO-AM A rollover accident has closed Minnesota Avenue from 18th Street to 22nd Street. At this time the cause of the accident is unknown. More information as it becomes available. Filed Under: Accident | Minnesota Avenue | Sioux Falls Category: Articles Share on Facebook Share on Twitter More Leave a comment Send to a friend! Print this page Pin it! Reddit This! Share on Tumblr! Best of KSOO-AM Recommended For You Best of the Web


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On last night's Gary Unmarried, Gary discovered that his daughter doesn't just excel at music, but something he actually cares about... golf!
However, when Gary realizes Allison will veto his decision to let Louise play golf, he has to try and trick Allison into wasting her veto. Find out what happened in our "Gary Uses His Veto" recap.
Here's some of our favorite quotes from the episode:
Allison [about riding a dirt bike]: Get on there, what's the worst that could happen?
Tom: I dunno, having to learn to type with a pencil taped to my head? | permalink
Gary: When are you gonna take time to be a kid
Louise: In my late 30's like you
Gary: I'd ground you for that, but then I wouldn't have anyone to play with | permalink


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Registration takes less than 1 minute.
The Surface Transportation Reauthorization and Reform Act of 2015 attempts to fund highway and infrastructure spending while remaining budget neutral through a series of offsets, including the application of some customs user fees for highway aid.
Combined with the sweeping Trans-Pacific Partnership free trade agreement signed last month, the Chinese policy could help "ignite new trade," according to Federal Maritime Commissioner Richard A. Lidinsky.
Legislators had hoped to pass a multi-year bill by Thanksgiving, but instead opted for a measure to extend federal transportation spending through Dec. 4 to avoid a funding shutdown when the current three-week authorization expires Friday.


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CDPI Virtual Job Fair
sponsored by the Career Development Professionals of Indiana
Posted: Thursday, April 12, 2012
April 18
If you're looking for a job or looking to hire, don't miss this alumni-only virtual job fair. IU is joining several other colleges and universities from the state of Indiana to offer this event. Please join us online!
Looking for new career opportunities? You won't want to miss this unique opportunity to meet with many of the state's most respected employers in varying industries.
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Word of their captivating, high-energy performances has spread, gaining Jet Edison recognition throughout the Rockies and into the Midwest. At home, the group was recently nominated as Colorado's Best Rock Band in the "Jam/Improv" category for the second year in a row by a popular Denver publication. With an ever-expanding tour regimen, JE has been billed with many top national artists including Particle, 7 Walkers feat. Bill Kreutzmann, Orgone, Cornmeal, and Marco Benevento, and has played with members of Colorado's most established bands, a few being Leftover Salmon, The Motet, Elephant Revival, and The Phix. The band has also performed at numerous festivals, such as ShoeFest, Denver Day of Rock, Phibstock, Colorado's famous NedFest, and more.
The future has much in store for Jet Edison, as the band hits the studio to record and release their second full-length album and tour through many more cities nationwide. A new year, and a new album, means new tunes and a solid look at a band very much in their element with this upcoming studio release, due out early this Summer 2013.


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Possibly buying 5 spoke SVT's
This will be the first set of wheels that I am going to buy, so I want some opinions. Seller is willing to balance them for me as he works at a shop. He did paint them over with Rustoleum wheel paint to cover up some rash. They are not perfect, but the price is very generous. Just looking for opinions on if you would buy them or not, any questions to ask prior to buying them, and how much you would pay for them. They have the tires on them, but he said they need to be replaced, maybe 25% tread on 2 of 'em, and the other 2 are bald. The center caps are wrapped with Vinyl. He's selling because he can't afford a new set of tires for them.
his asking price is a deal,look at some of the pricing here and you will agree. if the rash bugs you fix them and either repaint a color you like or get them powdercoated. just see if they are bent. but if you wanted five spokes jump on the deal before somebody else grabs them and flips them for more
Bent rims will be your most likely issue. 5-spokes are relatively soft wheels and are easy to bend. That said, I ran my 5-spokes with two bent wheels for about a year before upgrading. The only problem I had was a slight vibration through the steering wheel (kind of like a wheel out of balance but not as bad) and a thumping noise that could easily be drowned out by the radio.
I plan on painting them black - would it make sense to remove the paint currently on them, or just paint over it?
Best prep is to strip, sand, lay down a base, and paint....
Or get PC... Some places you can get a full set restored and PC for about 60/ wheel
┓┏ 凵 =╱⊿┌┬┐
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Paper Chase is a research database designed to offer abstracts of research articles published in journals that have a highly rated impact factor as determined by ISI Impact Factor and PageRank. Abstracts are organized by date, with the most recently published papers listed first.
Paper Chase
Large-scale genetic study in East Asians identifies six new loci associated with colorectal cancer risk.
Division of Epidemiology, Department of Medicine, Vanderbilt-Ingram Cancer Center, Vanderbilt Epidemiology Center, Vanderbilt University School of Medicine, Nashville, Tennessee, USA.
Known genetic loci explain only a small proportion of the familial relative risk of colorectal cancer (CRC). We conducted a genome-wide association study of CRC in East Asians with 14,963 cases and 31,945 controls and identified 6 new loci associated with CRC risk (P = 3.42 × 10(-8) to 9.22 × 10(-21)) at 10q22.3, 10q25.2, 11q12.2, 12p13.31, 17p13.3 and 19q13.2. Two of these loci map to genes (TCF7L2 and TGFB1) with established roles in colorectal tumorigenesis. Four other loci are located in or near genes involved in transcriptional regulation (ZMIZ1), genome maintenance (FEN1), fatty acid metabolism (FADS1 and FADS2), cancer cell motility and metastasis (CD9), and cell growth and differentiation (NXN). We also found suggestive evidence for three additional loci associated with CRC risk near genome-wide significance at 8q24.11, 10q21.1 and 10q24.2. Furthermore, we replicated 22 previously reported CRC-associated loci. Our study provides insights into the genetic basis of CRC and suggests the involvement of new biological pathways.
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- Schepens Eye Research Institute
- Spaulding Rehabilitation Hospital
- VA Boston Healthcare System


text:
I went to JoAnn earlier this week to purchase Burda 7738, but they didn't have it in stock... it wasn't even in the Burda Spring pattern book. I thought this was weird, because it doesn't show as out-of-print on the Simplicity website. So... is it actually out of print, and the Simplicity website isn't up-to-date, or does JoAnn not carry all the Burda envelope patterns?
My JAs carries the full line...which is not as many patterns they used to have. If I can't find it in the book, it is probably discontinued. When this has happened to me, I ordered from the simplicity web site before they don't have it. I am finding Burda discontinues patterns faster than they used to.HTH
If the pattern is not in the Burda book, it's discontinued. We pull the patterns for discard when we get the instructions to do so. Which means we have to pull out the old before we put in the new. btw at our Joann's, we basically only have one of each pattern, because the pattern includes all sizes.
Thanks y'all! I guess I'll be ordering it from the website so I can grab it before it's gone.
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1995 Audi A6 • 165,000 miles
My ignition sometimes doesnt turn when I put in the key. But it eventually does. And I recently had my alternator and my battery go out. So are these signs that this car might not make it?
September 13, 2011.
September 13, 2011.
Well sometimes the key wont turn. It starts always. But it sometimes gets stuck.I think it might be the steering wheel locking or something but im not sure. But my major concern which I just remembered is that once every couple months or so. Ill make a stop at a light or a stop sign and when I go to accellerate and press the pedal the car doesnt move and theres no power at all. But if I press the pedal a second time. It goes. Whats going on with it?
Sep 13, 2011.
Sep 13, 2011.
How do i properly remove the alternator? Step by step please, I'm kind of slow.
1 answer • 2000 Jeep Wrangler • 137,000 miles
Do you have a walkthrough for replacing an alternator on a 97' Dodge Caravan with a 2.4L frontwheel drive engine? If you can help me Please include all the tools necessary for doing ...
1 answer • 1997 Dodge Caravan