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|AKR mice are useful in cancer, immunology, and metabolism research.
Former Names Soat1 (Changed: 14-MAR-12 ) Type Inbred Strain; Additional information on Inbred Strains. Visit our online Nomenclature tutorial. Mating System Sibling x Sibling (Female x Male) 01-MAR-06 Breeding Considerations This strain is a challenging breeder. Species laboratory mouse H2 Haplotype k Generation F277 (05-AUG-14)
Generation Definitions
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Related Genotype: a/a Tyrc/Tyrc Soat1ald/Soat1ald hid/hid
Originally inbred at the Rockefeller Institute, AKR mice are widely used in cancer research for their high leukemia incidence (60-90%) and in immunology as a source of the Thy1.1 (theta AKR) antigen. AKR/J mice are viremic from birth, and express the ecotropic retrovirus AKV in all tissues. The hair interior defect (hid) mutation, a strain characteristic of AKR mice, causes alterations in hair development that is only evident microscopically. Adrenocortical lipid depletion (ald) in AKR mice is caused by a mutation in sterol O-acyltransferase 1 (Soat1), and leads to a truncated SOAT1 protein. AKR/J mice are relatively resistant to aortic lesion formation on a semi-synthetic high fat diet and are hyporesponsive to diets containing high levels of fat and cholesterol.
AKR Strains
002720 AKR/CumJView AKR Strains (1 strain)Strains carrying Ahrd allele
000690 129P3/J 008599 B6.Cg-Del(9Cyp1a2-Cyp1a1)1Dwn Ahrd Tg(CYP1A1,CYP1A2)1Dwn/DwnJ 002921 B6.D2N-Ahrd/J 000652 BDP/J 000928 CAST/EiJ 000671 DBA/2J 000674 I/LnJ 000675 LG/J 000676 LP/J 000684 NZB/BlNJ 000726 RBF/DnJ 000682 RF/J 000686 SJL/J 000688 ST/bJ 000689 SWR/J 000693 WC/ReJ KitlSl/J 000933 YBR/EiJView Strains carrying Ahrd (17 strains)Strains carrying Hc0 allele
000645 A/HeJ 000646 A/J 000647 A/WySnJ 000460 B10.D2-Hc0 H2d H2-T18c/o2SnJ 000461 B10.D2-Hc0 H2d H2-T18c/oSnJ 000657 CE/J 000671 DBA/2J 007048 DBA/2J-Gpnmb+/SjJ 001800 FVB/NJ 001491 FVB/NMob 000674 I/LnJ 001303 NOD.CB17-Prkdcscid/J 001976 NOD/ShiLtJ 000684 NZB/BlNJ 000682 RF/J 000688 ST/bJ 000689 SWR/JView Strains carrying Hc0 (17 strains)Strains carrying Il3ram1 allele
000645 A/HeJ 000646 A/J 000647 A/WySnJ 000653 BUB/BnJ 000669 C58/J 000657 CE/J 000684 NZB/BlNJ 000682 RF/J 000687 SM/JView Strains carrying Il3ram1 (9 strains)Strains carrying Rmcfs allele
000646 A/J 000779 AKXD14/TyJ 000780 AKXD23/TyJ 000764 AKXD27/TyJ 000777 AKXD6/TyJ 000667 C57BR/cdJ 000668 C57L/J 000669 C58/J 000682 RF/J 000644 SEA/GnJ 000686 SJL/J 000688 ST/bJView Strains carrying Rmcfs (12 strains)Strains carrying other alleles of Ahr
000645 A/HeJ 000646 A/J 002920 B6(D2N).Spretus-Ahrb-3/J 002831 B6.129-Ahrtm1Bra/J 000130 B6.C-H17c/(HW14)ByJ 000136 B6.C-H34c/(HW22)ByJ 000370 B6.C-H38c/(HW119)ByJ 002727 B6;129-Ahrtm1Bra/J 001026 BALB/cByJ 000653 BUB/BnJ 000659 C3H/HeJ 000663 C57BL/6By 001139 C57BL/6ByJ 000664 C57BL/6J 000662 C57BLKS/J 000667 C57BR/cdJ 000668 C57L/J 000669 C58/J 000926 CAROLI/EiJ 000656 CBA/J 000657 CE/J 000351 CXB1/ByJ 000352 CXB2/ByJ 000353 CXB3/ByJ 000354 CXB4/ByJ 000355 CXB5/ByJ 000356 CXB6/ByJ 000357 CXB7/ByJ 002937 D2.B6-Ahrb-1/J 000673 HRS/J 000677 MA/MyJ 000550 MOLF/EiJ 000679 P/J 000930 PERA/EiJ 000644 SEA/GnJ 000280 SF/CamEiJ 001146 SPRET/EiJ 006203 STOCK Ahrtm3.1Bra/JView Strains carrying other alleles of Ahr (38 strains)Strains carrying other alleles of Hc
000470 AK.M-H2m H2-T18a/nSnJ 005308 B10.Cg-H2d Tg(TcraCl4,TcrbCl4)1Shrm/ShrmJ 000463 B10.D2-Hc1 H2d H2-T18c/nSnJ 003147 B10.D2-Hc1 H2d H2-T18c/nSnJ-Tg(DO11.10)10Dlo/J 024977 B6N(Cg)-Hctm1b(EUCOMM)Wtsi/J 022980 C57BL/6N-Hctm1a(EUCOMM)Wtsi/J 004306 NOD.CBALs-Hc1/LtJView Strains carrying other alleles of Hc (7 strains)Strains carrying other alleles of Obq3
000668 C57L/JView Strains carrying other alleles of Obq3 (1 strain)Strains carrying other alleles of Obq4
000668 C57L/JView Strains carrying other alleles of Obq4 (1 strain)Strains carrying other alleles of Rmcf
000690 129P3/J 000765 AKXD13/TyJ 000954 AKXD15/TyJ 001093 AKXD18/TyJ 000947 AKXD22/TyJ 000763 AKXD9/TyJ 000654 CBA/CaJ 000670 DBA/1JView Strains carrying other alleles of Rmcf (8 strains)Strains carrying other alleles of Soat1
003322 B6.129S4-Soat1tm1Far/J 007147 B6.129S4-Soat1tm1Far/Pgn 002896 B6;129S4-Soat1tm1Far/JView Strains carrying other alleles of Soat1 (3 strains)
JAX® NOTES, April 1988; 433. H-2 Haplotypes of Mice from Jackson Laboratory Production Colonies.
JAX® NOTES, Winter 2006; 504. JAX® Mice: the Gold Standard Just Got Better.
Mouse Phenome Database / SNP Facility
Sequence data is available from the Mouse Genomes Project at the Wellcome Trust Sanger Institute
View Phenotypic DataPhenotypic DataMouse Phenome Database
Festing Inbred Strain Characteristics: AKR
View Related Disease (OMIM) TermsRelated Disease (OMIM) Terms provided by MGI
- Potential model based on gene homology relationships. Phenotypic similarity to the human disease has not been tested.Complement Component 5 Deficiency; C5D (C5)
Eculizumab, Poor Response to (C5)
View Mammalian Phenotype TermsMammalian Phenotype Terms provided by MGIassigned by genotype
- mortality/aging
- decreased sensitivity to xenobiotic induced morbidity/mortality
- mice are resistant to the pathological effects of DMBA including mortality, morbidity and severity of effects (MGI Ref ID J:26440)
- homeostasis/metabolism phenotype
- decreased physiological sensitivity to xenobiotic
- mice are resistant to the pathological effects of DMBA including mortality, morbidity and severity of effects (MGI Ref ID J:26440)
- decreased sensitivity to xenobiotic induced morbidity/mortality
- mice are resistant to the pathological effects of DMBA including mortality, morbidity and severity of effects (MGI Ref ID J:26440)
- hematopoietic system phenotype
- abnormal common myeloid progenitor cell morphology
- CFU-GM assays using bone marrow derived cells yield very few colonies in repsonse to interleukin 3 (MGI Ref ID J:24918)
- integument phenotype
- abnormal hair growth
- homozygotes can be ascertained only by microscopic examination of the hair (MGI Ref ID J:15247)
- distal regions of all hair types are especially affected (MGI Ref ID J:15247)
- all AKR strains are homozygous for this mutation (MGI Ref ID J:15247)
- morphology and arrangement of medullary cells are abnormal (MGI Ref ID J:15247)
- abnormal hair shaft morphology
- medullary cell abnormality effects calibre of hair shaft (MGI Ref ID J:15247)
The following phenotype information is associated with a similar, but not exact match to this JAX® Mice strain.
- endocrine/exocrine gland phenotype
- abnormal adrenal cortex morphology
- adrenal cortex of males is totally depleted of lipid (MGI Ref ID J:12157)
- adrenal cortex of females is not totally depleted and lipid pattern is variable (MGI Ref ID J:12157)
- large doses of ACTH restores lipid concentration and distribution (MGI Ref ID J:12157)
- onset of adrenocortical lipid depletion takes place in mice between 30 and 40 days of age (MGI Ref ID J:5089)
- in males adrenocortical lipid depletion coincides roughly with adrenal X-zone degeneration and maturation of seminiferous epithelium (MGI Ref ID J:5089)
- thymus hyperplasia
- evident from birth (MGI Ref ID J:12157)
- hematopoietic system phenotype
- thymus hyperplasia
- evident from birth (MGI Ref ID J:12157)
- immune system phenotype
- thymus hyperplasia
- evident from birth (MGI Ref ID J:12157)
- tumorigenesis
- increased leukemia incidence
- high incidence of lymphatic leukemia (MGI Ref ID J:12157)View Research ApplicationsResearch ApplicationsThis mouse can be used to support research in many areas including:
Ahrd related
Cancer Research
Increased Tumor Incidence
Leukemia: lymphatic
Cardiovascular Research
Diet-Induced Atherosclerosis
Relatively Resistant
Developmental Biology Research
Lymphoid Tissue Defects
hematopoietic defects
Diabetes and Obesity Research
diet-induced, moderate
Obesity With Diabetes
Type 2 Diabetes (NIDDM)
Endocrine Deficiency Research
Adrenal Cortex Defects
Hypothalamus/Pituitary Defects
Internal/Organ Research
Adrenal Cortex Defects
Metabolism Research
Enzyme Deficiency
Neurobiology Research
Hearing Defects
Age related hearing loss
Hc0 related
Research Tools
Toxicology Research
Il3ram1 related
Immunology, Inflammation and Autoimmunity Research
specific complement deficiency
Research Tools
Immunology, Inflammation and Autoimmunity Research
specific complement deficiency, C5 complement
Immunology, Inflammation and Autoimmunity Research
CD Antigens, Antigen Receptors, and Histocompatibility Markers
genes regulating susceptibility to infectious disease and endotoxin
|Allele Symbol
|Allele Name
|d variant
|Allele Type
|Not Applicable
|Common Name(s)
|Ahd; Ahk; AhRd; Ahhn; ah; in;
|Gene Symbol and Name
|Ahr, aryl-hydrocarbon receptor
|Gene Common Name(s)
|Ah; Ahh; Ahre; In; aromatic hydrocarbon responsiveness; aryl hydrocarbon hydroxylase; bHLHe76; dioxin receptor; inflammatory reactivity;
|General Note
Strain of origin - this allele was found in DBA/2J, AKR/J, 129, SWR, RF, NZB strains
|Molecular Note
|This allele encodes a 104 kDa receptor that is stabilized by molybdate and has an affinity for ligand 10-100 fold lower than that of the receptor produced by the C57BL/6J allele. PCR sequencing of cDNA revealed ten nucleotide differences between the coding sequences of the DBA/2J and C57BL/6J receptors. Five of the ten differences would cause amino acid changes. One of these, an apparent T to C transition replaces the opal termination codon in the C57BL/6J allele with an arginine codon in the DBA/2J allele. This change would extend translation of the DBA/2J mRNA by 43 amino acids, accounting for the larger size of the peptide produced by this allele (104 kDa vs 95 kDa for the C57BL/6J allele). A second T to C transition changes a leucine codon in the C57BL/6J allele to a proline codon in the DBA/2J allele, and would likely change secondary structure of the peptide and thus ligand affinity. [MGI Ref ID J:15153] [MGI Ref ID J:17460] [MGI Ref ID J:22144]
|Allele Symbol
|Allele Name
|Allele Type
|Common Name(s)
|C5-; C5-d; C5-def; C5-deficient; hco;
|Strain of Origin
|multiple strains
|Gene Symbol and Name
|Hc, hemolytic complement
|Gene Common Name(s)
|C5; C5D; C5a; C5b; CPAMD4; ECLZB; He;
|General Note
This is an allele characteristic of various inbred mouse strains including the following: A/HeJ, AKR/J, DBA/2J, NZB/B1NJ, SWR/J, B10.D2/oSnJ
Hc was identified as a candidate gene for Abhr2 in a microarray analysis of lung mRNA from A/J, C3H/HeJ, and (A/J x C3H/HeJ)F1 x A/J backcross animals. Hc genotype shows statistically significant correlation to allergen-induced bronchial hyperresponsive phenotype. The A/J allele contains a 2 bp deletion resulting in deficient Hc mRNA and protein production and is associated with susceptibility to allergen-induced bronchial hyperresponsiveness. (J:108211)
|Molecular Note
|A 2 base "TA" deletion at positions 62 and 63 of an 83 base pair exon near the 5' end of the gene is found in the following mouse strains: A/HeJ, AKR/J, DBA/2J, NZB/B1NJ, SWR/J, B10.D2/oSnJ. The consequence of this deletion is the creation of a stop codon starting four bases after the deletion. A truncated product of 216 amino acids is predicted as a result although contradictory reports exist that a larger pro-C5 protein may be synthesized. Nevertheless, macrophages from mouse strains carrying this allele do not secrete complement 5. [MGI Ref ID J:23983] [MGI Ref ID J:5016]
|Allele Symbol
|Allele Name
|mutation 1
|Allele Type
|Common Name(s)
|Il3raA/J; Il3ran;
|Strain of Origin
|Gene Symbol and Name
|Il3ra, interleukin 3 receptor, alpha chain
|Gene Common Name(s)
|CD123; Cyrl; IL-3 receptor alpha chain; IL3R; IL3RAY; IL3RX; IL3RY; SUT-1; hIL-3Ra;
|Molecular Note
|Sequence analysis revealed A/J mice lack the sequence corresponding to exon 8, which encodes 10 amino acid residues in the extracellular domain. Aberrant splicing was due to a 5 base pair deletion at the branch point in intron 7. [MGI Ref ID J:23971]
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|Obq3, obesity QTL 3
|General Note
|Obq3 exhibits an additive mode of inheritance.
|Molecular Note
|This allele confers increased adiposity in male animals compared to C57L/J. [MGI Ref ID J:39947]
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|Obq4, obesity QTL 4
|General Note
|Obq4 may also be consistent with an additive mode of inheritance.
|Allele Symbol
|Allele Name
|MCF sensitive
|Allele Type
|Strain of Origin
|multiple strains
|Gene Symbol and Name
|Rmcf, resistance to MCF virus
|General Note
This locus controls resistance and susceptibility of cells in tissue culture to infection by mink cell focus-forming murine leukemia viruses. The allele Rmcfr determines resistance and occurs in strains DBA/1, DBA/2, and CBA/Ca; the allele Rmcfs determines susceptibility and occurs in strains AKR/J, C57BL/6, BALB/c, CBA/J, NFS, NZB, 129/J, and many others. Heterozygotes are as resistant as the resistant parent or nearly so. Rmcf is different from and independent of Fv1,a locus that controls susceptibility to infection by ecotropic viruses. Rmcf is located on Chr 5 close to Hm near the centromeric end (J:7108). Rmcfr protects (AKR x CBA/Ca)F1 and (AKR x DBA/2)F1 hybrids from development of spontaneous thymic lymphomas and reduces the incidence of MCF-induced thymic lymphomas (J:7175). It also reduces susceptibility of cells of Sxvs/Sxvr mice to exogenous xenotropic viruses (J:7951). In addition, in strains susceptible to Friend virus-induced erythroleukemia, a condition thought to be due to the replication of MCF virus, Rmcfr increases resistance to the virus-induced erythroleukemia. It may cause resistance by coding for or regulating the production of an MCF-related envelope glycoprotein that blocks the receptor for MCF viruses (J:8074). This conclusion is reinforced by the findings of Buller et al. (J:8497), who showed that the Rmcfr allele contains an endogenous MCF gp70 env gene and that theRmcfs allele, at least in some strains (C57BL/6, CBA/J, and A/WySn), contains a xenotropic gp70 env gene. Presumably the MCF gp70 inhibits exogenous MCF infection in vitro by a mechanism of viral interference.
|Molecular Note
|This locus controls resistance of cells to infection by mink cell focus-forming murine leukemia viruses. The recessive s (susceptible) allele is found in AKR/J, C57BL/6, BALB/c, CBA/J, NFS, NZB and 129/J.
|Allele Symbol
|Allele Name
|adrenocortical lipid depletion
|Allele Type
|Common Name(s)
|Acact-; Soat1hid; ald; hid;
|Strain of Origin
|Gene Symbol and Name
|Soat1, sterol O-acyltransferase 1
|Gene Common Name(s)
|8430426K15Rik; ACACT; ACAT; ACAT-1; ACAT1; AW550831; Acact; RIKEN cDNA 8430426K15 gene; SOAT; STAT; acetyl coenzyme A cholesterol acyltransferase; adrenocortical lipid depletion; ald; expressed sequence AW550831; hair interior defect; hid;
|General Note
All AKR sublines are homozygous for Soat1
|Molecular Note
|Deletion of 118 bp in the a Soat1 mRNA. This region corresondes to the 5'UTR and the initial coding sequences. In addition, two missense mutations were observed: A1248G resulting in Ile to Val at amino acid 147 and C1422T resulting in His to Tyr at amino acid 205. Other nucleotide differences were observed that did not result in amino acid changes. A truncated protein is expressed from this allele, presumably due to internal initiation. [MGI Ref ID J:180857] [MGI Ref ID J:42073]
Inbred mouse strains are maintained through sibling (sister x brother) matings; no genotyping required.
Genotyping resources and troubleshooting
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Animal Health ReportsRoom Number MP16
Colony Maintenance
Mating System Sibling x Sibling (Female x Male) 01-MAR-06 Breeding Considerations This strain is a challenging breeder. Diet Information LabDiet® 5K52/5K67
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text:
The Daily Word in mine sweeping rats, spray-on condoms and Morrissey's junk
By Constance Moss & Desiree Garcia
News & Opinion
Crib Notes: July 30, 2015
By August March
Hot dog! If you've heard enough of the Albuquerque buzz to decode these cheesy clues, you'll ace this week's news quiz and can show your happy, smiling mug in public.
Film & TV
By Devin D. O'Leary
Probing Indian drama picks judicial system apart piece by piece
Concert Connections
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Four fab and fashionable forays
Muy Bien
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Café Bien lives up to its name
Today's Events
Eric Hisaw Band • Americana at Sister
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Content last updated on:
May 28, 2015
Teens ages 15 to 17 need to go to the doctor or nurse for a "well-child visit" once a year.
A well-child visit is when you take your teen to the doctor for a full checkup to make sure he is healthy and developing normally. This is different from other visits for sickness or injury.
At a well-child visit, the doctor or nurse can help catch any problems early, when they may be easier to treat. Make the most of your teen's visit by:
What about cost?Well-child visits are covered under the Affordable Care Act. Depending on your insurance plan, your teen may be able to get well-child checkups at no cost to you. Check with your insurance provider.
How do I know if my teen is growing and developing on schedule?Your teen's doctor or nurse can help you identify "developmental milestones," or signs to look for that show your teen is developing normally. This is an important part of the well-child visit.
Some developmental milestones are related to your teen's behavior and learning, and others are about physical changes in your teen's body.
What are some of the changes I might see in my teen's behavior, feelings, and relationships?Developmental milestones for teens ages 15 to 17 include:
This is also a time when some teens may start showing signs of depression, anxiety, or eating disorders. Your teen may also have a girlfriend or boyfriend.
What are some of the physical changes my teen is going through?Teens ages 15 to 17 are usually either finished or close to finishing puberty. Puberty is when a child's body develops into an adult's body.
Although it may be different for some teens, most girls finish puberty by age 15. Most boys finish puberty by age 16.
Teens might not ask you questions about sex, their bodies, or relationships. That's why it's a good idea for you to start the conversation.
Take these steps to help you and your teen get the most out of visits to the doctor.
Gather important information.Take any medical records you have to the appointment, including a record of shots your teen has received. Make a list of any important changes in your teen's life since the last visit, like a:
Use this tool to keep track of your teen's family health history.
Make a list of questions you want to ask the doctor.This visit is a great time to ask the doctor or nurse any questions about:
Here are some questions you may want to ask:
Take a notepad and write down the answers so you can remember them later.
Know what to expect.During each well-child visit, the doctor or nurse will ask questions, do a physical exam, and update your teen's medical history. You and your teen will also be able to ask questions and discuss any problems.
The doctor or nurse will ask your teen questions.The doctor or nurse may ask about:
The answers to questions like these will help the doctor or nurse make sure your teen is healthy and developing normally. If there are any problems, the doctor or nurse can help work on them with you and your teen.
See a list of other questions the doctor may ask [PDF - 1.1 MB].
The doctor or nurse will also check your teen's body.To check your teen's body, the doctor or nurse will:
The doctor or nurse will pay special attention to signs of certain issues. The doctor or nurse will offer additional help if your child may be:
And if your child may be having sex, the doctor or nurse will talk to your child about preventing STDs (sexually transmitted diseases) and pregnancy.
The doctor or nurse will make sure you and your teen have the resources you need. This may include telling you and your child about:
If necessary, the doctor or nurse may also refer your child to a specialist.
Help your teen get more involved in visits to the doctor.The doctor will probably ask you to leave the room during part of the visit, usually the physical exam. This is an important step in teaching teens to take control of their health care.
It also lets your teen develop a relationship with the doctor or nurse and ask questions in private.
Your teen can also:
Get more tips on helping teens take charge of their health care.
Know what to do if your teen gets sick.Make sure you know how to get in touch with a doctor or nurse when the office is closed. Ask the doctor's office how to get hold of the doctor on call, or if there's an information service you can call at night or on the weekend.


text:
Topic: Porto vs Arsenal
Both teams go trough to the next stage of the Champions League if this match ends in a draw, so they are probably happy with a goalles draw in this match. But Arsenal wins the group if it ends in a draw.
Thierry Henry is out with suspension, and William Gallas i struggling with injuries.
There's a good chance that this match will end in a goalles draw.


text:
Kimberly Ann Terry Whited, age 50, of Martin, passed away at the University of Kentucky on Jan. 29, at 5:30 pm., surrounded by all five of her brothers and sisters, her niece, Crystal, and her longtime friend, Lois, as she sadly left this precious world to be with our Savior, Jesus Christ. Kim was loved by many and, in return, she also loved them.
Kim was born in Hindman, on Feb. 17, 1962, to Samuel Robert and Donna Sue Combs Terry. After the first few years of her life in Knott County, her family moved to Martin, and this is what Kim always called home. She loved this little town and the people that lived here. She attended grade school at Martin Elementary, where she was a cheerleader and valedictorian of her class. She graduated from Allen Central High School where she played a clarinet in the band and loved high school so very much, as she loved her friends. She then attended the University of Kentucky for a short time, returning to Martin to work for Dr. Chandra Varia. Here is where Kim shined with her knowledge and the passion and kindness she showed her patients as she cared for them. Kim was devoted to her family and always smiled as she spoke of them.
Christmas time at the Terry's house was always a special time. Sammy putting up lights outside, Donna baking and cooking and all the girls having their pictures made in their Christmas sweaters. Poor Bobby and Billy just had to put up with all the girls. Kim was an excellent cook and had been taught very well by her mother. Although Kim could cook, her favorite food was mac and cheese, had to be Kraft, in the blue box, with the powdered cheese. Kim always had that big beautiful smile and hearty laugh. She was always particular about the way she looked and we all know she had to be tan. She loved the sun and would lay in it for hours, with aluminum foil, baby oil and iodine and a fan at her feet to keep her cool, now that is a 1970s and 1980s moment. She also loved the tanning bed, as it was a lot easier. When Kim was around, there was never a dull moment. She was always positive, laughing and upbeat. All you can say is she was as beautiful on the inside, as she was on the out. As many of you here today come to support the family, just know that Kim loved each and every one of you. She was faithful, loving and giving to her family and friends.
Kim was preceded in death by her father, Samuel Robert Terry Sr., and she sadly missed him every day. Also, her grandparents Sheldon and Ora Combs, Bill and Norcie Terry and her step grandmother, Violet Terry.
Kim is survived by her mother, Donne Sue Combs Terry, of Pikeville, and her sisters and brothers, Sandra Lynn Terry May, of Martin, Angela Kaye Terry Wright, of Pikeville, Debra Sue Terry Conley (Ricky), of Lackey, Samuel Robert (Bobby) Terry Jr. (Christy), of Paintsville, and William Sheldon "Billy" Terry, of Martin.
She is also survived by several nieces and nephews, Crystal Lea Terry Akers (Bobby), of Prestonsburg, Shannon Dale Wright (Stephanie), of Allen, Lane Robert Terry and Jillian Christine Terry, of Paintsville, Jade Leanne Conley, Chyeanne Skye Conley and Sammi RaShae Conley, of Lackey; and step-niece and nephew, Tyra Paige Conley and Tyler Logan Conley. Kim is also survived by one great niece, Hailey Ann Akers, of Prestonsburg.
Kim leaves behind many other loving family members and a host of loving friends. She will be sadly missed by all and each of us can be assured she is watching over us with lots of love and peacefulness shining upon us. May God bless each of you for your kind words, loving thoughts and many prayers. The Terry family would like to thank all of you for everything you have ever done for Kim or for them during this very sad time but let us remember, Kim would want all of us to celebrate her life, not mourn her.
Funeral services for Kimberly Ann Terry Whited will be held at 7 p.m., Saturday, Feb. 2, in the Hall Funeral Home Chapel, in Martin.
Visitation will be held from 5 to 9 p.m., Friday, and Saturday, until the time of service.


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Very professional, organized, accommodating, knowledgeable, friendly, and always available to answer any questions. This is the second time using Jessica's services, and she met my needs fully each time. I highly recommend using her services to either buy or sell a home, since she successfully assisted me in doing both.
Jessica Hapeman
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13 Recent Sales (last 12 months)
Licensed Assoc. Broker
- Specialties:
- Buyer's Agent,
- Listing Agent,
- Relocation,
- Moving
Ratings & Reviews
Jess is a great real estate agent. Very friendly, knowledgeable, and responsive. Helped us find the perfect house and negotiate a fair price as well as to sell our old house putting together a great web listing.
Jess was extremely helpful starting from our first meeting through closing on our new home. She has excellent knowledge of the entire home buying process. She helped us identify they type of home that we were most interested in and found dozens of candidate houses. She arranged showings of all homes we were interested in and was very flexible in meeting our availability for showings. A great professional to guide you through purchasing a home.
We used another real estate agent from Hunt in 2003 to purchase our first home, and the contrast between that individual/large firm and Jessica from Superlative Real Estate is substantial. Here's a quick compare and contrast based on my experience with typical RE agents over the years and Jess:
The typical RE agent will be your best friend! She will *love* whatever you say, laugh at your jokes, stroke your ego. She's probably a retired history teacher or bored housewife, has as much knowledge about real estate law as you might expect someone would after a few hours of a RE course. Her negotiation skills aren't great, but good news, they don't have to be! Because the typical RE agent doesn't really give a crap about getting you a good deal, she gives a crap about _closing_ the deal so she gets her gigantic commission check. The moment she gets that, *poof* she's gone. Careful with typical RE agent's recommendation of a home inspector; he knows as well as she does that if he finds anything that would jeopardize getting her commission or even making her do more work than usual, she'll find someone else to "inspect" houses for her clients, maybe someone that gloss over a potentially major issue.
Then there's Jess. She may not laugh at every one of your jokes; just the funny ones. Show her a picture of your kids and she probably won't get all Disney on you like other agents. She's your RE agent - her job is to find you a house in your price range that you will love, tell you as much as she can about it and then do everything in her power to get it for you for as little as the seller is willing to grudgingly give it up for. Want to feel like you have some power on your side? She's a frickin' lawyer. All the agents at Superlative are frickin' lawyers. When it's time to negotiate, I'll take the person professionally trained for years to argue in a court of law, thankyouverymuch. Her inspector found every minor thing that could be construed as an issue with the house which let me make the most informed decision I could. Every step of the way, I felt like her goal was to get us the right deal on the right house, regardless of how long it takes.
Oh, and she'll pay you. Unlike the unqualified twit whose face you'll see plastered all over signs advertising the typical large RE company, Superlative RE rebates a chunk of the commission back to you. Have some fun and ask any other typical RE agent if they would rebate some of that commission back to you.
I highly recommend Jessica and Superlative Real Estate.
As a very picky person Jessica helped in our search by being Honest, and looking out for what was best for our family, we looked at about 20 different houses, some good but were a little out of our price range, which she reminded us of. We also looked at a few that weren't so nice, but when we did find the one that fit us best, even she knew it, which was nice too. Very helpful in every way, I do Highly Recommend her and her company.
Jessica Hapeman was invaluable in selling my home. She was totally competent, trustworthy and responsive to my needs, especially in negotiating the sale. She did all the necessary marketing including posting beautifully taken photographs of my home on various websites, advertising, holding open houses, signage, and handling all the phone calls from interested parties. My home was sold rather quickly after considering several offers. During this time, she also helped me find the perfect new home and negotiated a fair price for the purchase. For both selling and buying real estate, I recommend using Jessica without any hesitation. She did a great job for me, and I was totally satisfied with the results I got.
Jessica is without doubt the most knowledgeable, helpful and skilled agent I have ever worked with. If she doesn't know the answer (which is rare), she will find the answer! It has been and is a pleasure to work with her. I highly recommend her to anyone needing real estate services in Western New York.


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Weekdays at 6am on 98.7 FM Arizona Sports
Doug & Wolf
I'm not hating on Peyton Manning, but...
That word/phrase has officially passed, "It's not about the money, it's about respect," as the most overused in sports.
Peyton Manning is not the greatest quarterback of all time. For some reason, if you agree with that opinion, you're a hater.
If an analyst picks your team to lose, suddenly they're a hater. If an insider points out the flaws of a college player's game, he gets the tweet "#donthate" sent back. Is there any chance that the "hater" is right? If they're right, it would seem clear that facts or intelligent opinions aren't hating as much as informing or critiquing.
Is it really that insulting to claim someone is the third-best in the world's history at their job? I can safely say no one has ever accused me of being the third-best talk show host ever, but I would gladly accept the compliment if you'd like to be the first to say it.
Either Peyton Manning or Walter Payton is the greatest ambassador of football the game has ever known. His level of class is so far above most humans (clearly above mine), that we can all take lessons from him on how to deal with public adversity. His devotion to perfection should be imitated by professionals everywhere, not just athletes, but any professional in any walk of life. Peyton Manning is one of the greatest quarterbacks of all time and one of the greatest people to ever be employed by an NFL team. There is no reason to hate Peyton Manning unless your life is fueled by jealousy because you want to justify your own failings.
I understand why Peyton is loved. He should be. The catch is he's not as good as Joe Montana or Tom Brady. Does that sentence really make me a hater? I think that's an amazing list of quarterbacks and a huge compliment to say he's third. The one constant among all analysts who put Peyton first is they must apologize or justify Peyton's playoff performance: most playoff losses in history, seven trips to the playoffs without a win and only one Super Bowl victory.
If Peyton's the greatest of all time, why does he suddenly stop being the greatest of all time in January? Since I think Tom Brady is the greatest of all time, I'm not trying to explain away bad regular seasons. Tom Brady audibles without all the gestures that Manning supporters love so much, so he's not as much fun to watch. Brady has had one Hall of Fame receiver -- and only had him for a brief period of time -- while Manning played the bulk of his career with two Hall of Fame-caliber receivers. Manning has had fewer offensive coordinators in his career, creating much more continuity. Plus, Manning has had the benefit of playing in a dome, while Brady wins as many games as he can every year just to play his playoff games in the atmospheric nightmare for a quarterback that is New England.
Both quarterbacks are great regular-season quarterbacks. The postseason must be used as the separating entity. It's completely opinion to try and figure out why Peyton Manning teams fail the majority of the time after Christmas. It's fact to say Tom Brady and Joe Montana succeed more often. If anyone thinks Manning is better than Brady, I ask two questions: 1) What is it Brady has to do more of,or improve upon, to catch Manning? and, 2) How many playoff games have the Patriots lost where you say after the game, "Brady strongly contributed to the loss." Although Manning clearly won the individual and team battle this year, rarely has Brady played poorly in a Patriots loss. For Peyton, it's almost yearly.
When I talk about Peyton as the third greatest QB, I'm not dismissing his regular-season accomplishments. If I thought postseason or Super Bowls were all that mattered, I would rank Peyton behind Terry Bradshaw or consider Peyton even with Brad Johnson and Trent Dilfer. I accept there's much more to playing the hardest position in professional sports than just January results. Once you put all the greats in one room, it's January and Super Bowls that should separate them.
I have great respect for Peyton Manning. I will never "hate" any athlete who gives all they can to succeed. Congratulations Peyton. 2013 proved, once again, you're one of the greatest quarterbacks of all time. As soon as you win two more Super Bowls, Brady and Montana will invite you to their wing of the Hall of Fame.


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Do-On Robert Thomas is a Zen priest and teacher with Dharma Transmission from Zoketsu Norman Fischer. His teaching is focused on helping people extend the deeply grounded yet expansive awareness of Zen meditation into the activities of daily life. He is also a visiting Dharma teacher at Upaya Zen Center in Santa Fe, NM.
Albert Flynn DeSilver is an internationally published poet, author, teacher, speaker, and writing coach. He served as Marin County California's very first Poet Laureate from 2008-2010. His latest book is Beamish Boy (I Am Not My Story): A Memoir of Recovery & Awakening. He presents at conferences and workshops nationally, has taught in the Teen and Family Program at Spirit Rock Meditation Center since 2005, and has practiced Vipassana and other forms of meditation for more than eighteen years.


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Victorian Colored Tin Tinsel
Colorful Victorian Tinsel for Your Tree Lasts for Years
Victorian Colored Tin Tinsel
Colorful Victorian Tinsel for Your Tree Lasts for Years
The Victorians decorated their trees with colorful tiny icicles like these, originally made from strips peeled off old tin cans and then twisted. Each is spiral-shaped to reflect light in a surprising way, more like real icicles than ordinary tinsel. Once you see these on your tree, you'll never use disposable tinsel again. Includes threads for hanging. Container of 24 comes with 6 each of red, blue, gold, and green.
- Each icicle is 6"
- Bright jewel tones of red, blue, gold, and green
- Tin icicles reminiscent of Victorian times
Spirals of color brighten your Christmas tree in an old-fashioned way.
Victorian Colored Tin Tinsel
Danger: Not for use by young children. Some edges may be sharp.
Tinplated steel (tinsel). Polyester and thread (hanger).
To maintain brightness, store jeweled tinsel in the plastic sleeves provided.
Assembly Required: N
Cord Polarized: N
Manufactured In: Canada
Material - Country Of Origin: Canada
What Our Customers Are Saying about Our Victorian Colored Tin Tinsel …
"My mom had the spiral tin icicles when my brother and sister and I were growing up. I thought they were so pretty under the tree lights when they twirled around. My mom carefully strung them with thread so they would be sure and twirl. I kept them all these years and even tho' they are over 50 years old and not as shiney, they have great memories. So I was amazed to see that someone else still remembers them. Thanks, Gwen Frederick" - Gwen Frederick, Denver, CO


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In this Instructable, I will be showing off two versions of this bowl. The classic style and a newer one.
Step 1: The classic record bowl
The easiest way of doing this is to set your over for 220F/100C and tossing the record in there for a few minutes. When it's completely warmed up it will have the texture and floppiness of a fruit roll-up. You can now shape it any way you want.
Other methods include dipping the record in boiling water or heating it up with a heat gun.
Step 2: Shaping the bowl
- drape the record on top of a bowl. Once the record cools it will have a similar interior shape as to the bowl.
- drop the record into a bowl. The outside of the record bowl will then have the same shape as the bowl.
- drape the record on top of a cylinder such as a can of soup.
Once the record cools, it will harden into place and stay that way. If you're not happy with it, just toss it back in the oven for a bit and start over.
Step 3: Record bowl with cuts
The cuts here are lines that begin at one circle and end at another. Each cut ends 30 degrees off from where it began. So if one cut were to begin at 12 o'clock on the inner circle, it ends at 1 o'clock on the outer circle.
When I first made them I did handle and shape them with my hands. It got to be uncomfortable if I handled them too long but if you can shape quickly you will be fine.
I use scratched and otherwise unplayable records. There are enough of em out there.
Have you tried this process with CDs ?
I made a few of these for a party I was hosting where the theme was "retro"! We used the big LPs as tip jars and the really small ones for nuts and chips (I made sure to wash them properly before and after usage of course by hand). Was too popular though so now I only have like two out of 15 left :/
do u have any ideas what to make out of VHS movies?
Then again, I have a higher tolerance for pain than most. YMMV
anyone have a clue?
Please tell me that vinyl was ruined or I start crying!
I prefer analogic music..
I'll try whit CD bowl but i must find very little fruit for it !)


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Harris-HBA-ANS [PY-A-2565]
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Chapel Messages
You can listen to previous chapel messages through the OBU Chapel Archives podcast.
Spring 2015
|Jan 28
|Fall Challenge - Wes Lane & David Prater
|Jan 30
|Connect Chapel
|Feb 4
|Founder's Day - Robert Kellogg, President, BFO
|Feb 6
|Class Chapel (Fr-Potter/So-Yarb/Jr-Stubb/Sr-GC)
|Feb 9
|FOCUS WEEK - Dr. Tom Elliff, President, IMB
|Feb 11
|FOCUS WEEK - Dr. Tom Elliff
|Feb 13
|FOCUS WEEK - Dr. Tom Elliff
|Feb 18
|Dr. Henry Kirkland, Jr., Author, Professor, Mentor
|Feb 23
|Champions of Character Chapel
|Feb 25
|Dr. Jim Shaddix, Pastor, The Church at Brookhills
|Feb 27
|Connect Chapel
|Mar 4
|Student Preacher
|Mar 6
|Connect Chapel
|Mar 11
|O.S. Hawkins, President and CEO, Guidestone
|Mar 23
|Class Chapel (Fr-Potter/So-Yarb/Jr-Stubb/Sr-GC)
|Mar 25
|Women's Mission & Ministry Day - Gigi Graham
|Mar 27
|Connect Chapel
|April 1
|Holy Week
|Ap 6
|Champions of Character Chapel
|Ap 8
|Hyde Park - James Lankford, US Representative
|Ap 10
|Connect Chapel
|Ap 15
|Nik Ripkin - Author, Lecturer
|Ap 20
|BLITZ WEEK Chapel
|Ap 22
|Harold Mathena, Founder of Mathena Inc.
|Ap 24
|Connect/BLITZ WEEK Chapel II
|Ap 29
|GO Commissioning Chapel
|May 6
|Awards Assembly


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Sam Carpenter on "relentless system improvement"
Here is an excerpt from a recent post by Sam Carpenter at his website. To read the complete article, check out other resources, and/or sign up for free email updates, please click here.
I'm writing this post because the one I published last week had some problems. Last Wednesday, the day after I emailed Choose the Red Pill [please click here] to my subscriber list, I read through it and found several grammatical errors and some clumsy paragraphs. Also, I thought it was too complex as well as a bit melodramatic at the end. Bypassing my standard protocol of relentless system improvement, I had put the post together at the last minute and sent it out too fast.
I deemed the piece ready-for-public-consumption simply because I wanted it to be. Deluding myself, I violated my own rules about system improvement. (For a definition of system improvement, see page 11 in my book, Work the System.
This failure inspires me to discuss a half-dozen points about the system improvement process. Here, I will discuss my weekly Work the System blog posts, and a little bit about Centratel, but the points I make apply to any work or personal context in which there are recurring tasks to be executed. Be imaginative. Especially think about your business or job.
I already covered the first point: Root out and acknowledge failure. Don't muddle reality by conjuring up some dubious silver lining. Bury the ego and face facts: If you screwed up, you screwed up.
Point two: Remember exactly what the task is and what is to be done with it. For example, this blog post is an individual system with a purpose. It is a singular, enclosed entity designed to deliver a message and to entertain. Improvement is what I do to it. (Hence, "system improvement.")
A third point: Use failures as red flags so action can be taken to prevent the failures from happening again. In fact, assertively seek out failures. Your job is to find weaknesses in your systems and then to fix them.
My cycle of writing a typical 1,000 word post is one week long. On Wednesday or Thursday I spend just a half-hour writing the rough draft. Then, over the next five or six days I spend an additional eight to ten hours tweaking it until it's ready to post on Tuesday or Wednesday of the following week. I'm relentless in this phase, performing this read-through editing in thirty to forty separate sessions. Between each session, I clear my head by doing some other activity so each read-through is from a different mental perspective. Even then, I'm not done: After the piece is posted and delivered, and over the next few weeks, I'll go back four to six separate times to further polish it. Point four: Repeatedly execute the system from scratch, each time looking for weaknesses. Thus, 5% of time is spent in creation of the raw product and 95% of time is spent perfecting it.This is working the system!
For any of us, the repetition of various systems is no problem. It's what we do. We have tasks to execute and so we execute them. But too many people leave it at that, only to fire-kill through work, relationships and life, blindly negotiating the same old problems over and over again. They don't analyze the source of recurring problems and thus don't take steps to stop them from happening again.
It's a good time to say it: System improvement is the opposite of fire-killing.
My process for writing a blog post is a perfect analogy for how I spend my working hours at Centratel. My time is invested in relentless system improvement and I seldom do the "work." Point five: For the leader, most work is either delegated or automated. At Centratel, I pay the bills, conduct staff meetings and occasionally go back and forth with my management staff on special issues. That's it, for a total work-time at Centratel of less than two hours per week.
* * *
Which do you choose: a life of fire-killing or a life of system improvement?
* * *
Sam Carpenter is a resident of Bend, Oregon, author of Work the System, and speaker. He has been featured by hundreds of media, including NPR, ESPN radio, US News Radio, and Small Business Television. President and CEO of Centratel (www.centratel.com), the premier telephone answering service in the United States, Sam has a background in engineering, publishing, telecommunications and journalism. Sam founded and oversees Kashmir Family Aid, a 501c3 non-profit that aids surviving school children of the Northern Pakistan and Azad Kashmir earthquake of October 2005.
Photo by Nigel Blake via flickr used under a creative Commons License.