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|28 posts found
Associate Editor - News Manager
Joined: 6/28/10
In the final installment of our Guild Wars 2 Review in Progress, we take a look at dungeons. We offer our honest opinion about rough spots as well as the good stuff. Read on and then let us know what you think in the comments.
Read more of Bill Murphy's Guild Wars 2: Review in Progress - Dungeons.
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Review here is dead on!
I finished all 8 dungeons story modes with guildies- and had a blast! my final comment on this as you slightly mention wish rewards were a bit more fruitful.
the exploration mode is a huge learning curve(in a good way!)-dont expect it to be like open world events. expect different bosses from story mode and some coordination is key.
still Anet have done a fantastic job on letting the player Explore with their skills in combat -allowing one to use different concepts& ideas.- so none should be telling you 'use this, or do that' ,
I aslo found that the boses arent in script mode where everyone has to stand in 'one' place to avoid something or spreadout to avoid aoe...like other mmo's, the fights are more dynamic-and different everytime.
It might seem like a ressurection fest at first, but if you get a solid group of players together who know how to use combos and crowd control skills, it can go as smooth as butter. Looking forward to more challenging dungeons being introduced in the future.
Novice Member
Joined: 11/05/10
Maybe it would help if players had done mini dungeons before attempting dungeons. Too bad there's only 3 of them that I know of. These mini-dungeons, assuming not zerged, can prepare players for the difficulty spike.
Originally posted by KhinRunite
they are called 'personal story'..try em out. they are pretty close to a mini dungeon which leads up to the story mode dungeon seemlessly.basically when you do your personal story you can invite a friend to participate. no chest loot- but you get the idea...
Advanced Member
Joined: 2/07/12
They drastically increased rewards on tuesday so i think that issue is resolved. As for the trash mob complaint i respect the opinion but i resent the idea that their should ever be trash mobs in any game. There should be hard and harder mobs with scaling rewards accordingly but why play a game that is a cake walk.
Novice Member
Joined: 5/14/10
a few things here. first off nice review
"Side-bar: ArenaNet would do well to give an overview of how different professions' skills combo. Maybe even make an in-game journal that records when and how you actually did something (IE – shooting arrows through a wall of fire). This could only help to promote better grouping and more strategic fighting in dungeons." - YES. this is needed, and tbh i thought the stream yesterday was going to be the combo class they should be putting out.
"It makes the experience feel more like a D&D campaign, like a real RPG, and that's something that's often missing when other companies just say, "We need dungeons to check off this item on the list of what's in an MMO"." - comletely agree. i want my game to pull me in, and not make me feel like i'm going through the motions.
i will say this about your group dying though. this game doesn't have a full on trinity, but going into a 5 man with a "soft" trinity will change it up completely. in guild we do runs all the time, and we run this way. one person is geared towards tanking, and generally tries to gain initial aggro, and stand in the mix to take a few hits. really just to be the target. we're slowly figuring out how aggro works in game this way.
we also run a support build at times. one person will spec into healing a bit more then normal, and with the combination of the tank and support, runs can be a lot easier to manage. the last few runs in guild no one actually died. we use our dodges, watch our range to all targets, and use abilities. none of us go in with our general world pve specs. we at least tend to change up our 6-0 skills.
Experience is the best teacher.. if you can afford the tuition.
Apprentice Member
Joined: 8/06/08
Went through all 4 paths of TA, CM and two paths of HoTW yesterday without any wipes and no deaths on my part, very few on the part of others. However, I was with my guild, who are very good at this kind of thing. We had food buffs, everyone brought support skills (NEVER underestimate Warrior Banners) and we read up before-hand on what other people faced in those dungeons.
In PUG groups, I never have that kind of luck. Dungeons are meant to be super difficult, and that's part of my gripe. While yeah, I nailed it with a really goot 'leet' group, I don't like that that's what it took to enjoy the dungeons. But then again, if you make them easier thnn people like that will be bored and left out...it's a difficult thing to find a balance on. My main gripe is that dungeon bosses take too long to kill; once you figure out the pattern and allow some elbow room for the occasional deviation from it (bosses like to get tricky) it's pretty much just waiting for it to die and, in some cases, the wait is quite long. Like, pop open that mp3 playlist, long.
Anyway, if you're going into a dungeon people, PLEASE TAKE HEED OF THE FOLLOWING:
1 and most important) ALL CLASSES HAVE SUPPORT SKILLS. BRING THEM. That mean Warriors should have their banners. Guardians need their party boon skills. If you see a Mesmer without Feedback, Null Field and Time Warp, smack them (and also smack the fields as they'll aoe chaos armor everyone). Necros should have minimum 3 "dark" combo Wells (when slapped, they aoe blind). Thieves have aoe blinds and poisons for a reason (poison reduces amount enemies can heal drastically). Elementalists and Engineers are self-explanatory, and finally Rangers need their spirits.
2) Food and potion buffs make a HUGE difference. I typically go in with Mango Pie, +70 vitality and constant Regeneration buff. Additionally, there are vendors all over Tyria that offer consumables that stack with food buffs, including one underwater dude in Bloodtide somewhere that sells two orbs: one is a stack of 5 Fury for 30 minutes, the other is a stack of 5 Regeneration for 30 minutes. Buy some, and if you feel like you're having trouble, pop one.
3) Even scaled down, gear and level matters. A level 80 Mesmer in great gear scaled down to 35 will still have more hp, defense and power than a level 35 Warrior, so if you can't manage it, come back to it when you're stronger.
Bottom line, those kamikaze "die and run back" strategies are not part of the game. They're an option, but that's not one you should be relying on, as evidenced by the fact that Anet is steadily removing Waypoints popping up throughout the dungeon so that by the time you run back, the bosses are back to full. If that's your only way of winning, you're doing something wrong.
By the way - I just wanted to point out that, to the people complaining that not enough is explained, that too much has to be found out on your own, this is the way MMOs have worked classically. Information was never spoon-fed, people figured things out the hard way, then that knowledge was passed on through word of mouth, strategy guides, forum posts, and later wikis. This is exactly what a lot of you have been wanting for a while now, so complaining about it now is a bit ironic.
"Forums aren't for intelligent discussion; they're for blow-hards with unwavering opinions."
Novice Member
Joined: 8/30/08
Originally posted by gaeanprayer
The problem is not everyone is like me. I have no issue with figuring things out on my own, but others are going to have a harder time of it. Some sort of editorial or video series on playing your class, field combo's, You and Dungeons, etc. would go along way in helping out those that don't have the time or simply find it difficult to discover these things on their own.
I don't have an issue, but I also have a daughter, son and wife that play and they some times need that explanation. Besides that, if you ever have to pug, or need a few randoms to fill out a party you formed with your guild wouldn't it be better if they knew or at least had an idea of what to do?
Joined: 3/03/12
Maybe if you used food and magic chest buffs it would feel more balanced.
The Elder Scrolls Online
Originally posted by ZeGerman
Thats good news!- I started to re think abt my Op. and I feel they played it smart about the loot find- they have magic find boosts, runes and gear= so that should work. havent tried them but, if all as plan thats a very smart move on the mechanics of the game=everything shouldnt be a 'sure' thing and s'poon fed' to you-then whats the point.
I was with you in that Dungeon, Bill, and I think it was all your fault we wiped 30 times ;). Kidding of course. That was our first time in that dungeon and as a group, and I think that fact we had 3 engineers and 2 warriors didnt provide the right amount of skills, in addition to our lack of coordination. But we did finish eventually!
I agree, the trash mobs are unbalanced. They dont need to be trash, but 3 trash mobs shouldnt wipe our entire group, when one boss cant. In addition, we should be rewarded for the difficulty of the challenge, otherwise, there is no incentive to play, as the experience itself isnt that dynamic.
One major problem as well is the distance from waypoints to bosses. Some of them are a good 5 mins run. And I think they should not allow rezzing at a WP and getting back into the fight.
Advanced Member
Joined: 7/03/04
I liked MMOs better when gamers didn't play them, and just geeks did.
The group of MMO vets I play with have all been playing together for years. We are obviuosly awful at AC and CM, because we were wiping constantly. I'm not saying there's no room to improve for us, as there obviously is. All I'm saying is, if we are having trouble and we were playing these types of games for years, I can't imagine how rough it must be for noobs.
Played: AA, AC1, AC2, Aion, AO, AoC, CO, CoX, DAoC, DCUO, DN, EVE, EQ1, EQ2,
Hard Core Member
Joined: 7/18/10
Veni, Vidi, Converti
" I love that the game's eight dungeons see the players through the drama surrounding the Destiny's Edge guild. You get to party and adventure with people like Eir Stagalkin, Rytlock Brimstone, and the like. The entire series is geared towards telling this story, and I absolutely love the way it does so."
"A big part of the challenge of many dungeons is disarming traps, avoiding pratfalls, and solving puzzles."
Definitely 2 things dungeons should be designed to do well: Story and "Puzzle". :)
Spotlight Poster
Joined: 6/15/04
Running the dungeons is fun, to a point. Ascalon Catacombs can be torched to the ground lol for all I care, I hate that place. But loved the other dungeons. However, explorable mode to me, is not worth the headache and time invested. The rewards IN the dungeon are pathetic and the gear from the vendors in LA while some are cool, most aren't and the length of time to get them is just stupid. So there goes another aspect of the endgame just not worth doing. It seems only playing alts or PVP is where endgame play is right now. That needs to change IMO, as longevity is becoming a real issue.
Hard Core Member
Joined: 5/28/04
I walked away from GW2 after my first dungeon experience there. It basically took everything that I was "borderline" on about GW2 and red-flagged it. Repair costs were about 20% of my total cash. That was it for me. Haven't logged in for several weeks now. And haven't missed it.
Apprentice Member
Joined: 9/03/10
I love lack of trinity and requirement of paying attention to enemy, fellow players and skills in order to be successful. It so refreshing after being dps, standing in spot X and pressing 1-3-4-2-2-5 in order as it's best rotation.
As for rewards/difficulty ANet is still balancing them. I remember duo-ing Caudicus Mannor story at lvl 43 bout 2 weeks after launch. Then I went there much later with 5 peeps and it was nightmare, with mobs much stronger and with many annoying skills. Folks say it's actually more difficult than exploration mode. So experience may vary...
Novice Member
Joined: 11/05/10
Originally posted by orbitxo
That too. Although personally I'm in the level 40 of my PS and I never needed a group, but I can't say the same when I did mini dungeons.
Novice Member
Joined: 2/28/12
Well, that review is a bit off, bacically you dislike the gameplay of try and error, graveyard rushing, the tedious high--hit-point trash and the lack of desirable rewards, but the story mode is presented well. So the overall experience is positive??? Realy?!
I would agree on most said above, but my judgement would be fundamentally different, the dungeons are by design the weakest and least fun aspect of an otherwise great game. They are overly long, boring, tedious and unrewarding. The lack of trinity makes it horrible for Pugging since most people won't know right away what they are supposed to do to be a decent team. There is next to no tutorial or intro or learning curve within and throughout the dungeons.
As someone else said in the threat where I questioned if dungeons with the trinity are more fun then without, PVE in GW2 feels more like PVP, this is by design and working as intended, however, this is off-putting for all the folks who mainly play(ed) PvE in other MMO's they don't like the hectic reaction based gameplay and prefer beeting though but predictable encounters, learning patterns and strategies. While strategies or kind of possible and required it mostly end up being a deperate struggle for survival on every trash group and most bosses. You never feel like you are in control of the situation. Maybe fun for a couple of encounters, but all the time it is very tedious.
Oh and while talking about dungeons maybe talk about how much penalized melee are over ranged in this game for a majority of encounters. So everyone requires range to increase survivbillity. Not a good balance...
Elite Member
Joined: 10/29/07
Originally posted by orbitxo
Yeah, but the problem is that the PS lacks cooperation(unless you have a friend help you out but the PS is not really made for that), I wish there was a group version of the PS, like a guild story or something as well.


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|C57BL/6J is the most widely used inbred strain and the first to have its genome sequenced. Although this strain is refractory to many tumors, it is a permissive background for maximal expression of most mutations. C57BL/6J mice are resistant to audiogenic seizures, have a relatively low bone density, and develop age related hearing loss. They are also susceptible to diet-induced obesity, type 2 diabetes, and atherosclerosis. Macrophages from this strain are resistant to the effects of anthrax lethal toxin.
Type Inbred Strain; Additional information on Inbred Strains. Visit our online Nomenclature tutorial. Mating System Sibling x Sibling (Female x Male) 01-MAR-06 Breeding Considerations This strain is a good breeder. Species laboratory mouse H2 Haplotype b Generation F226pF230 (14-AUG-14)
Generation Definitions
View larger image
Related Genotype: a/a
Important Note
This strain is homozygous for Cdh23ahl, the age related hearing loss 1 mutation, which on this background results in progressive hearing loss with onset after 10 months of age.
C57BL/6 is the most widely used inbred strain. It is commonly used as a general purpose strain and background strain for the generation of congenics carrying both spontaneous and induced mutations. Although this strain is refractory to many tumors, it is a permissive background for maximal expression of most mutations. C57BL/6J mice are used in a wide variety of research areas including cardiovascular biology, developmental biology, diabetes and obesity, genetics, immunology, neurobiology, and sensorineural research. C57BL/6J mice are also commonly used in the production of transgenic mice. Overall, C57BL/6 mice breed well, are long-lived, and have a low susceptibility to tumors. Primitive hematopoietic stem cells from C57BL/6J mice show greatly delayed senescence relative to BALB/c and DBA/2J. This is a dominant trait. Other characteristics include: 1) a high susceptibility to diet-induced obesity, type 2 diabetes, and atherosclerosis; 2) a high incidence of microphthalmia and other associated eye abnormalities; 3) resistance to audiogenic seizures; 4) low bone density; 5) hereditary hydrocephalus (early reports indicate 1 - 4 %); 6) hairloss associated with overgrooming, 7) a preference for alcohol and morphine; 8) late-onset hearing loss; and 9) increased incidence of hydrocephalus and malocclusion.
C57BL/6J mice fed a high-fat diet develop obesity, mild to moderate hyperglycemia, and hyperinsulinemia (see JAX® Diet-induced Obesity (DIO) Models). C57BL/6J mice fed an atherogenic diet (1.25% cholesterol, 0.5% cholic acid and 15% fat) for 14 weeks develop lesions in the range of 4500 to 8000 um2 atherosclerotic aortic lesions/aortic cross-section. The variation in aortic lesions found among various inbred strains has led to the identification of the existence of eight genes affecting atherosclerosis, Ath1 to Ath8. C57BL/6J mice also develop severe and progressive hearing loss later in life. Histopathological changes associated with age-related hearing loss include the disruption of both outer and inner hair cells. C57BL/6 mice are also more susceptible to noise-induced hearing loss. Age related hearing loss 1 (Ahl), a major gene responsible for this hearing loss, was mapped in an intersubspecific backcross by measuring elevated auditory-evoked brainstem response (ABR) thresholds. Ahl is located on Chromosome 10 near marker D10Mit5. A naturally occurring deletion in nicotinamide nucleotide transhydrogenase (Nnt) exons 7-11 occurred in C57BL/6J sometime prior to 1984. This deletion results in the absence of the NNT protein, and is associated with impaired glucose homeostasis control and reduced insulin secretion. This mutation is not found in C57BL/6JEi, C57BL/6N, C57BL/6NJ, C57BL/6ByJ, C57BL/10J, C57L/J, or C58/J (Toye AA, et al, Diabetologia, 2005). Since C57BL/6JEi separated from C57BL/6J in 1976, the Nnt deletion arose sometime between 1976 and 1984. Because of the n-Tr20m1J point mutation, which is also present in C57BL/6JEiJ but not C57BL/6NJ or C57BL/6ByJ, extracts from the cerebellum of C57BL/6J mice have increased ribosomal pausing at AGA codons compared with that of other inbred strains (Ishimura et al., 2014).
C57BL/6J was the DNA source for the international collaboration that generated the first high quality draft sequence of the mouse genome. 5 SNP differences have been identified that distinguish C57BL/6J from C57BL/6ByJ and C57BL/6NJ. Both C57BL/6ByJ and C57BL/6NJ type as follows: 08-015199792-M (rs3709624) is C; 11-004367508-M (rs3659787) is A; 13-041017317-M (rs3722313) is C; 15-057561875-M (rs3702158) is G; 19-049914266-M (rs3724876) is T. C57BL/6J types as follows: 08-015199792-M (rs3709624) is T; 11-004367508-M (rs3659787) is G; 13-041017317-M (rs3722313) is T; 15-057561875-M (rs3702158) is A; 19-049914266-M (rs3724876) is G (Petkov and Wiles, 2004.) Others have subsequently identified further SNP differences between sublines of C57BL/6 (Mekada et al., 2009, Zurita et al., 2010).
The C57BL/6J inbred strain was created by Dr. CC Little from the mating of female 57 with male 52 from Miss Abbie Lathrop's stock. The same cross gave rise to the C57L and C57BR strains.
C57BL Strains
000665 C57BL/10J 003752 C57BL/10ScNJ 000476 C57BL/10ScSnJ 000666 C57BL/10SnJ 001822 C57BL/10SxJ 001197 C57BL/10WtRkJ 000663 C57BL/6By 001139 C57BL/6ByJ 009123 C57BL/6HaJ 000924 C57BL/6JEiJ 005304 C57BL/6NJView C57BL Strains (11 strains)Strains carrying Ahrb-1 allele
000136 B6.C-H34c/(HW22)ByJ 000663 C57BL/6By 001139 C57BL/6ByJ 000662 C57BLKS/J 000667 C57BR/cdJ 000668 C57L/J 000669 C58/J 000351 CXB1/ByJ 000356 CXB6/ByJ 002937 D2.B6-Ahrb-1/J 000677 MA/MyJView Strains carrying Ahrb-1 (11 strains)Strains carrying Cdh23ahl allele
001137 129P1/ReJ 000690 129P3/J 000691 129X1/SvJ 000646 A/J 000647 A/WySnJ 003070 ALR/LtJ 003072 ALS/LtJ 004502 B6;AKR-Lxl2/GrsrJ 001026 BALB/cByJ 000653 BUB/BnJ 005494 C3.129S1(B6)-Grm1rcw/J 004764 C57BL/6J-Cdh23v-8J/J 003129 C57BL/6J-Epha4rb-2J/GrsrJ 004820 C57BL/6J-Kcne12J/J 004703 C57BL/6J-Kcnq2Nmf134/J 004811 C57BL/6J-nmf110/J 004812 C57BL/6J-nmf111/J 004747 C57BL/6J-nmf118/J 004656 C57BL/6J-nmf88/J 004391 C57BL/6J-Chr 13A/J/NaJ 004385 C57BL/6J-Chr 7A/J/NaJ 000662 C57BLKS/J 000667 C57BR/cdJ 000668 C57L/J 000669 C58/J 010614 CBACa.B6-Cdh23ahl/Kjn 000657 CE/J 000670 DBA/1J 001140 DBA/1LacJ 000671 DBA/2J 007048 DBA/2J-Gpnmb+/SjJ 002106 KK/HlJ 000675 LG/J 000676 LP/J 000677 MA/MyJ 001976 NOD/ShiLtJ 002050 NOR/LtJ 000679 P/J 002747 SENCARB/PtJ 002335 SKH2/J 003392 STOCK Crb1rd8/JView Strains carrying Cdh23ahl (41 strains)Strains carrying Micrln allele
000646 A/J 000651 BALB/cJ 000009 BXH14/TyJ 000038 BXH6/TyJ 000014 BXH7/TyJ 000665 C57BL/10J 000669 C58/JView Strains carrying Micrln (7 strains)View Strains carrying P2rx7P451L (3 strains)Strains carrying n-Tr20m1J allele
004823 C57BL/6J-Gtpbp2nmf205/J 000924 C57BL/6JEiJView Strains carrying n-Tr20m1J (2 strains)Strains carrying other alleles of Ahr
000690 129P3/J 000645 A/HeJ 000646 A/J 000648 AKR/J 002920 B6(D2N).Spretus-Ahrb-3/J 002831 B6.129-Ahrtm1Bra/J 000130 B6.C-H17c/(HW14)ByJ 000370 B6.C-H38c/(HW119)ByJ 008599 B6.Cg-Del(9Cyp1a2-Cyp1a1)1Dwn Ahrd Tg(CYP1A1,CYP1A2)1Dwn/DwnJ 002921 B6.D2N-Ahrd/J 002727 B6;129-Ahrtm1Bra/J 001026 BALB/cByJ 000652 BDP/J 000653 BUB/BnJ 000659 C3H/HeJ 000926 CAROLI/EiJ 000928 CAST/EiJ 000656 CBA/J 000657 CE/J 000352 CXB2/ByJ 000353 CXB3/ByJ 000354 CXB4/ByJ 000355 CXB5/ByJ 000357 CXB7/ByJ 000671 DBA/2J 000673 HRS/J 000674 I/LnJ 000675 LG/J 000676 LP/J 000550 MOLF/EiJ 000684 NZB/BlNJ 000679 P/J 000930 PERA/EiJ 000726 RBF/DnJ 000682 RF/J 000644 SEA/GnJ 000280 SF/CamEiJ 000686 SJL/J 001146 SPRET/EiJ 000688 ST/bJ 006203 STOCK Ahrtm3.1Bra/J 000689 SWR/J 000693 WC/ReJ KitlSl/J 000933 YBR/EiJView Strains carrying other alleles of Ahr (44 strains)Strains carrying other alleles of Cdh23
002552 B6(V)-Cdh23v-2J/J 002756 B6.CAST-Cdh23Ahl+/Kjn 010615 B6.CBACa-Cdh23CBA/CaJ/Kjn 002432 B6J x B6.C-H2-Kbm1/ByJ-Cdh23v-J/J 004764 C57BL/6J-Cdh23v-8J/J 004819 C57BL/6J-Cdh23v-9J/J 005016 CByJ;B6-Cdh23v-10J/J 000275 V/LeJView Strains carrying other alleles of Cdh23 (8 strains)Strains carrying other alleles of Fbrwt1
000671 DBA/2JView Strains carrying other alleles of Fbrwt1 (1 strain)Strains carrying other alleles of Fbrwt2
000671 DBA/2JView Strains carrying other alleles of Fbrwt2 (1 strain)Strains carrying other alleles of Micrl
000032 BXH10/TyJ 000033 BXH19/TyJ 000011 BXH4/TyJ 000076 BXH8/TyJ 000008 BXH9/TyJ 000260 JGBF/LeJ 000072 JGBF/LeTyJView Strains carrying other alleles of Micrl (7 strains)Strains carrying other alleles of P2rx7
005576 B6.129P2-P2rx7tm1Gab/J 015809 NOD.129P2(B6)-P2rx7tm1Gab/DvsJView Strains carrying other alleles of P2rx7 (2 strains)
JAX® NOTES, April 1988; 433. H-2 Haplotypes of Mice from Jackson Laboratory Production Colonies.
JAX® NOTES, Fall 1995; 463. Inbred C57 Black Mice: Microphthalmia and Ocular Infections.
JAX® NOTES, Fall 2003; 491. The Importance of Understanding Substrains in the Genomic Age.
JAX® NOTES, Fall 2006; 503. Cause of Glucose Intolerance in C57BL/6J Mice Discovered.
JAX® NOTES, Fall 2008; 511. Influence of Nnt alleles on DIO in C57BL/6 JAX® Mice.
JAX® NOTES, January 1988; 432. Arthritis Models in the Mouse.
JAX® NOTES, July 1989; 438. Profile: C57BL/6J.
JAX® NOTES, October 1987; 431. Alopecia (loss of hair) in C57BL/6J and Related Strains.
JAX® NOTES, October 1989; 439. Splenic Melanosis in Black Mice.
JAX® NOTES, Spring 1990; 441. Imperforate Vagina and Mucometra in Mice.
JAX® NOTES, Spring 2003; 489. Malocclusion in the Laboratory Mouse.
JAX® NOTES, Spring 2003; 489. Mouse Genome Sequence Released.
JAX® NOTES, Spring 2004; 493. Chromosome Substitution Strain Panel: A New Tool for Quantitative Trait Loci Analysis.
JAX® NOTES, Spring 2007; 505. C57BL/6J Strain Used to Construct the Allen Brain Atlas.
JAX® NOTES, Spring 2009; 513. JAX® Mice help reveal potential health benefits of resveratrol.
JAX® NOTES, Summer 1994; 458. Ly5 Gene Nomenclature, C57BL/6J and SJL/J - A History of Change.
JAX® NOTES, Summer 2003; 490. Charles River Japan Now Breeding JAX® Mice Strain C57BL/6J.
JAX® NOTES, Summer 2003; 490. Hydrocephalus in Laboratory Mice.
JAX® NOTES, Summer 2006; 502. Characterization of DIO Model is Refined.
JAX® NOTES, Summer 2009; 514. JAX® Mice Study: Starvation Diet Does Not Extend Life.
JAX® NOTES, Summer 2009; 514. Another Obesity-modulating Gene Revealed.
JAX® NOTES, Summer 2009; 514. Defective Gene Plays Unexpected Role in Fat Metabolism.
JAX® NOTES, Summer 2009; 514. Non-Invasive Technique - Potential Parkinson's Therapy.
JAX® NOTES, Winter 2002; 488. Colony Expansions Completed for JAX® Mice Strains C57BL/6J and BALB/cJ.
JAX® NOTES, Winter 2006; 504. JAX® Mice: the Gold Standard Just Got Better.
JAX® NOTES, Winter 2008; 512. New resource illustrates divergence of C57BL/6 laboratory mouse substrains.
Mouse Phenome Database / SNP Facility
National Center for Biotechnology Information / SNP Data
View Phenotypic DataPhenotypic DataBody Weight Information - JAX® Mice Strain C57BL/6J (000664)Mouse Phenome Database
(This chart reflects the typical correlation between body weight and age for mice maintained in production colonies at The Jackson Laboratory.)
Festing Inbred Strain Characteristics: C57BL
JAX® Physiological Data Summary [pdf]
JAX® Physiological Data Protocol [pdf]
View Related Disease (OMIM) TermsRelated Disease (OMIM) Terms provided by MGI
- Potential model based on gene homology relationships. Phenotypic similarity to the human disease has not been tested.Deafness, Autosomal Recessive 12; DFNB12 (CDH23)
Glucocorticoid Deficiency 4; GCCD4 (NNT)
Usher Syndrome, Type ID; USH1D (CDH23)
View Mammalian Phenotype TermsMammalian Phenotype Terms provided by MGIassigned by genotype
- cardiovascular system phenotype
- decreased cardiac muscle contractility
- decreased heart rate
- average 433 beats per minute versus 524 in A/J (MGI Ref ID J:76471)
- increased heart rate
- average heart rate is approximately 50 beats per minute higher than that in A/J (MGI Ref ID J:76471)
- increased left ventricle weight
- average left ventricular weight of 46.2mg is significantly higher than control A/J and results from an increase in the end-diastolic dimension and a proportional increase in wall thickness (MGI Ref ID J:76471)
- muscle phenotype
- decreased cardiac muscle contractility
- homeostasis/metabolism phenotype
- enhanced exercise endurance
- exercise time on a treadmill is significantly greater than that of A/J (MGI Ref ID J:76471)
- mortality/aging
- increased sensitivity to xenobiotic induced morbidity/mortality
- mice are susceptible to DMBA induced lethality (MGI Ref ID J:26440)
- homeostasis/metabolism phenotype
- *normal* homeostasis/metabolism phenotype
- topical application of the polycyclic hydrocarbon DMBA induced hepatic aryl hydrocarbon hydroxylase activity (MGI Ref ID J:5387)
- increased physiological sensitivity to xenobiotic
- mice are susepctible to the teratogenic effects of TCDD with 100% of mice developing cleft palates and 90% of mice developing severe hydronephrosis (MGI Ref ID J:132380)
- mice are susceptible to the pathological effects of DMBA and exhibit lethality, weight loss, peritonitis, decreased spleen weight, and decreased thymus weight (MGI Ref ID J:26440)
- mice exposed to DMBA exhibit decreased lymphocyte counts, increased polymorphic cells, decreased bone marrow cell counts and ascites formation (MGI Ref ID J:26440)
- increased sensitivity to xenobiotic induced morbidity/mortality
- mice are susceptible to DMBA induced lethality (MGI Ref ID J:26440)
- immune system phenotype
- increased inflammatory response
- mortality/aging
- increased sensitivity to xenobiotic induced morbidity/mortality
- mice exhibit neonatal lethality in response to in utero exposure to cHBB unlike mice homozygous for the Ahrd allele (MGI Ref ID J:113285)
- homeostasis/metabolism phenotype
- increased physiological sensitivity to xenobiotic
- when mice are fostered by control C57BL/6 dams following in utero exposure to cHBB, 21% of neonates survive (MGI Ref ID J:113285)
- mice exhibit neonatal lethality in response to in utero exposure to cHBB and decreased thymus and spleen weights compared to untreated mice (MGI Ref ID J:113285)
- following exposure to cHBB in utero, neonates exhibit an increase in volume density of hematopoietic islands in the liver (MGI Ref ID J:113285)
- increased sensitivity to xenobiotic induced morbidity/mortality
- mice exhibit neonatal lethality in response to in utero exposure to cHBB unlike mice homozygous for the Ahrd allele (MGI Ref ID J:113285)
- hearing/vestibular/ear phenotype
- organ of Corti degeneration
- in 3 of 4 cochleae assesed at 12 months of age the organ of Corti had totally degenerated (MGI Ref ID J:87783)
- nervous system phenotype
- cochlear ganglion hypoplasia
- at 9 months of age there is a decrease in the ganglion cell density for each turn over time with the largest cell loss in the basal turn (MGI Ref ID J:87783)
- endocrine/exocrine gland phenotype
- decreased insulin secretion
- this phenotype is rescued by transgenic expression of full-length Nnt sequence derived from 129S6/SvEvTac (MGI Ref ID J:109356)
- homeostasis/metabolism phenotype
- decreased insulin secretion
- this phenotype is rescued by transgenic expression of full-length Nnt sequence derived from 129S6/SvEvTac (MGI Ref ID J:109356)
- impaired glucose tolerance
- this phenotype is rescued by transgenic expression of full-length Nnt sequence derived from 129S6/SvEvTac (MGI Ref ID J:109356)
Myo5ad/Myo5adinvolves: C57BL/6J
- pigmentation phenotype
- abnormal melanocyte morphology
- cultured primary melanocytes display clustering of melanosomes but are otherwise normal (MGI Ref ID J:37976)
- endocrine/exocrine gland phenotype
- decreased insulin secretion
- this phenotype is rescued by transgenic expression of full-length Nnt sequence derived from 129S6/SvEvTac (MGI Ref ID J:109356)
- homeostasis/metabolism phenotype
- decreased insulin secretion
- this phenotype is rescued by transgenic expression of full-length Nnt sequence derived from 129S6/SvEvTac (MGI Ref ID J:109356)
- impaired glucose tolerance
- this phenotype is rescued by transgenic expression of full-length Nnt sequence derived from 129S6/SvEvTac (MGI Ref ID J:109356)
- cellular phenotype
- abnormal cell death
- lysis triggered by extracellular ATP is reduced 45% in C57BL/6 mice as compared to BALB/c (MGI Ref ID J:89513)
- abnormal apoptosis
- sensitivity to extracellular ATP induced apoptosis is reduced in C57BL/6 relative to BALB/c (MGI Ref ID J:89513)
- cellular phenotype
- abnormal translational elongation
- compared with C57BL/6J congenic for wildtype n-Tr20 from C57BL/6NJ, extracts from the cerebellum of C57BL/6J inbred mice have more ribosomal pausing at AGA codons and 1.6 times the pausing magnitude at AGA codons (MGI Ref ID J:211326)
- homeostasis/metabolism phenotype
- abnormal translational elongation
- compared with C57BL/6J congenic for wildtype n-Tr20 from C57BL/6NJ, extracts from the cerebellum of C57BL/6J inbred mice have more ribosomal pausing at AGA codons and 1.6 times the pausing magnitude at AGA codons (MGI Ref ID J:211326)View Research ApplicationsResearch ApplicationsThis mouse can be used to support research in many areas including:
Ahrb-1 related
Cardiovascular Research
Diet-Induced Atherosclerosis
Developmental Biology Research
Eye Defects
Lymphoid Tissue Defects
hematopoietic defects
Skeletal Defects
Diabetes and Obesity Research
diet-induced, moderate
Insulin Resistance
Obesity With Diabetes
diet-induced, moderate
Type 2 Diabetes (NIDDM)
Hematological Research
Hematopoietic Defects
Immunology, Inflammation and Autoimmunity Research
Lymphoid Tissue Defects
hematopoietic development
Neurobiology Research
Behavioral and Learning Defects
Hearing Defects
Age related hearing loss
Research Tools
General Purpose
Genetics Research
Mutagenesis and Transgenesis
Mutagenesis and Transgenesis: Production of Transgenic Mice
Hematological Research
Immunology, Inflammation and Autoimmunity Research
background strain for histocompatibility congenics
Infectious Disease
Tuberculosis (TB)
Sensorineural Research
Eye Defects
Hearing Defects
Age related hearing loss
Cdh23ahl related
Research Tools
Toxicology Research
Neurobiology Research
Hearing Defects
Age related hearing loss
Sensorineural Research
Hearing Defects
Age related hearing loss
|Allele Symbol
|Allele Name
|b-1 variant
|Allele Type
|Not Applicable (Not Applicable)
|Common Name(s)
|Ah; Ahb-1; Ahb; Ahhi; Ahrb; In;
|Strain of Origin
|Gene Symbol and Name
|Ahr, aryl-hydrocarbon receptor
|Gene Common Name(s)
|Ah; Ahh; Ahre; In; aromatic hydrocarbon responsiveness; aryl hydrocarbon hydroxylase; bHLHe76; dioxin receptor; inflammatory reactivity;
|General Note
C57BL/6 carries the responsive Ahrb allele; DBA/2 carries nonresponsive Ahrd. Heterozygotes (Ahrb/Ahrd) are responsive (J:5282). Later work identified a second (J:8895) and later a third (J:22144) allele conferring response. Thus the allele in C57, C58, and MA/My strains is now Ahrb-1; Ahrb-2 is carried by BALB/cBy, A, and C3H; and Ahrb-3 by Mus spretus, M. caroli, and MOLF/Ei. The nonresponsive strains AKR, DBA/2, and 129 carry Ahrd (J:22144). Nucleotide and amino acid sequence differences between Ahrb-1 and Ahrd have been determined (J:17460).
Strain of origin - this allele was found in C57BL/6, C58/J, C57BR, MA/My strains
|Molecular Note
|This allele encodes a high affinity, relatively heat stabile, 95 kDa receptor. PCR sequencing of cDNA revealed ten nucleotide differences between the coding sequences of the DBA/2J and C57BL/6J receptors. Five of the ten differences would cause amino acid changes. One of these, a C to T transition in exon 11 would change the arginine codon in the DBA/2J allele to an opal termination codon in the C57BL/6J allele. This change would prevent the 43 amino acid extension of mRNA translation predicted for the DBA/2J allele and account for the smaller size of the peptide produced by this allele (95 kDa vs 104 kDa for the DBA/2J allele). A second C to T transition changes a proline codon in the DBA/2J allele to leucine codon in the C57BL/6J allele, and would likely change secondary structure of the peptide and thus ligand affinity. [MGI Ref ID J:15153] [MGI Ref ID J:17460] [MGI Ref ID J:477]
|Allele Symbol
|Allele Name
|age related hearing loss 1
|Allele Type
|Common Name(s)
|Cdh23753A; mdfw;
|Strain of Origin
|multiple strains
|Gene Symbol and Name
|Cdh23, cadherin 23 (otocadherin)
|Gene Common Name(s)
|4930542A03Rik; CDHR23; RIKEN cDNA 4930542A03 gene; USH1D; W; age related hearing loss 1; ahl; bob; bobby; bus; bustling; mdfw; modifier of deaf waddler; neuroscience mutagenesis facility, 112; neuroscience mutagenesis facility, 181; neuroscience mutagenesis facility, 252; nmf112; nmf181; nmf252; sals; salsa; v; waltzer;
|Molecular Note
|Genetic complementation tests have shown allelism between the mdfw (modifier of deaf waddler) locus and the ahl locus. Further analysis has identified an association between ahl and a G to A transition at nucleotide position 753 of Cdh23. This hypomorphic allele causes in frame skipping of exon 7 and reduced message stability. Twenty-seven strains classified with ahl and carrying the 753A allele include: CD1, RBF/DnJ, PL/J, AKR/J, RF/J, BALB/cBy, A/WySnJ, P/J, SENCARA/PtJ, DBA/1J, ALS/LtJ, C58/J, C57BLKS/J, 129P1/ReJ, C57BR/cd, SKH2/J, BUB/Bn, MA/MyJ, LP/J, 129X1/SvJ, NOR/LtJ, A/J, C57BL/6, NOD/LtJ, DBA/2J, ALR/LtJ, C57L/J. Strains classified with ahl that DO NOT carry this mutation include: C3H/HeSnJ, I/LnJ,YBR/Ei, MRL/MpJ. [MGI Ref ID J:86905]
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|Fbrwt1, forebrain weight 1
|Molecular Note
|This allele confers increased forebrain weight compared to DBA/2J. [MGI Ref ID J:143361]
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|Fbrwt2, forebrain weight 2
|Gene Common Name(s)
|Molecular Note
|This allele confers increased forebrain weight compared to DBA/2J. [MGI Ref ID J:143361]
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|Gluchos1, glucose homeostasis QTL 1
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|Gluchos2, glucose homeostasis QTL 2
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|Gluchos3, glucose homeostasis QTL 3
|Molecular Note
|This allele confers 50% decreased plasma insulin at T30 during the intraperitoneal glucose tolerance test compared to C3H/HeH. [MGI Ref ID J:106692]
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|multiple strains
|Gene Symbol and Name
|Micrl, microwave induced increase in complement receptor B cells
|General Note
The following inbred strains are homozygous for the recessive QTL, Micrl
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|Nnt, nicotinamide nucleotide transhydrogenase
|Gene Common Name(s)
|4930423F13Rik; AI323702; BB168308; GCCD4; RIKEN cDNA 4930423F13 gene; expressed sequence AI323702; expressed sequence BB168308;
|Molecular Note
|This allele contains a stretch of 17,814 bp missing between exons 6 and 12. RT-PCR demonstrated cDNA corresponding to exons 7-11 was absent. Mature protein was not detected in these mutants. [MGI Ref ID J:106692] [MGI Ref ID J:108213]
|Allele Symbol
|Allele Name
|Allele Type
|Strain of Origin
|Gene Symbol and Name
|P2rx7, purinergic receptor P2X, ligand-gated ion channel, 7
|Gene Common Name(s)
|AI467586; P2X(7); P2X7; P2X7 receptor; P2X7R; expressed sequence AI467586;
|Molecular Note
|A proline to leucine change at amino acid residue 451 is caused by a T to C transition at nucleic acid base 1352. This mutation is found in C57BL/6, C57BL/10, DBA/1, and DBA/2 and contrasts with the condition in BALB/c, NZW, NOD, 129, Mus caroli, M. spretus, M. musculus, and M. poschiavinus. The mutation lies within a C-terminal cytoplasmic domain homologous with the TNFR 1-death domain and with an SH3 binding protein. [MGI Ref ID J:79540]
|Allele Symbol
|Allele Name
|mutation 1, Jackson
|Allele Type
|Spontaneous (Not Specified)
|Common Name(s)
|n-Tr20C57BL/6J; rs46447118;
|Strain of Origin
|Gene Symbol and Name
|n-Tr20, nuclear encoded tRNA arginine 20 (anticodon TCT)
|Gene Common Name(s)
|Molecular Note
|This C to T transition, found in C57BL/6J and C57BL/6JEiJ but not other inbred strains, is in nucleotide 50 in the stem of the T loop and impairs maturation, leading to accumulation of a 105 nucleotide pre-tRNA band with leader and trailer sequences instead of the usual 115 nucleotide pre-tRNA, and also diminishes aminoacylation of this isodecoder [MGI Ref ID J:211326]
Genotyping ProtocolsNntC57BL/6J, Separated MCA
NntC57BL/6J, Standard PCR
Inbred mouse strains are maintained through sibling (sister x brother) matings; no genotyping required.
Helpful LinksGenotyping resources and troubleshooting
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Animal Health ReportsRoom Number AX1
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Colony Maintenance
Mating System Sibling x Sibling (Female x Male) 01-MAR-06 Breeding Considerations This strain is a good breeder. Diet Information LabDiet® 5K52/5K67
|Pricing for USA, Canada and Mexico shipping destinations
Weeks of Age Price per mouse (US dollars $) Gender 3 weeks $21.05 Female $19.25 Male 4 weeks $21.68 Female $20.02 Male 5 weeks $23.23 Female $22.94 Male 6 weeks $23.90 Female $23.58 Male 7 weeks $25.25 Female $25.40 Male 8 weeks $26.00 Female $26.16 Male 9 weeks $26.27 Female $26.16 Male 10 weeks $35.24 Female or Male 11 weeks $36.17 Female $36.51 Male 12 weeks $37.20 Female $37.15 Male 13 weeks $37.45 Female $41.65 Male 14 weeks $38.73 Female $45.19 Male 15 weeks $41.30 Female $50.25 Male 16 weeks $44.08 Female $50.25 Male 17 weeks $46.87 Female $53.30 Male 18 weeks $49.54 Female $56.29 Male 19 weeks $52.12 Female $59.23 Male 20 weeks $54.80 Female $62.27 Male 21 weeks $57.47 Female $65.38 Male 22 weeks $60.36 Female $68.60 Male 23 weeks $63.04 Female $74.76 Male 24 weeks $65.71 Female $80.41 Male
Standard Supply
JAX Ready Strain®. Most popular strains. Readily available in any quantity you need.
Supply Notes
Volume pricing is available for this strain. View our Volume Pricing Program for information.
- Timed pregnant females are available from our Bar Harbor, Maine and Sacramento, Calif. facilities.
- This strain is available from both the Bar Harbor, Maine and Sacramento, Calif. facilities.
- Shipped at a specific age in weeks. Mice at a precise age in days, littermates and retired breeders are also available.
Frozen Products
Price (US dollars $) Frozen Embryo $1725.00
Standard Supply
JAX Ready Strain®. Most popular strains. Readily available in any quantity you need.
Supply Notes
- Cryopreserved Embryos
Available to most shipping destinations1
This strain is also available as cryopreserved embryos2. Orders for cryopreserved embryos may be placed with our Customer Service Department. Experienced technicians at The Jackson Laboratory have recovered frozen embryos of this strain successfully. We will provide you enough embryos to perform two embryo transfers. The Jackson Laboratory does not guarantee successful recovery at your facility. For complete information on purchasing embryos, please visit our Cryopreserved Embryos web page.
1 Shipments cannot be made to Australia due to Australian government import restrictions.
2 Embryos for most strains are cryopreserved at the two cell stage while some strains are cryopreserved at the eight cell stage. If this information is important to you, please contact Customer Service.
|Pricing for International shipping destinations
Weeks of Age Price per mouse (US dollars $) Gender 3 weeks $27.40 Female $25.10 Male 4 weeks $28.20 Female $26.10 Male 5 weeks $30.20 Female $29.90 Male 6 weeks $31.10 Female $30.70 Male 7 weeks $32.90 Female $33.10 Male 8 weeks $33.80 Female $34.10 Male 9 weeks $34.20 Female $34.10 Male 10 weeks $45.90 Female or Male 11 weeks $47.10 Female $47.50 Male 12 weeks $48.40 Female $48.30 Male 13 weeks $48.70 Female $54.20 Male 14 weeks $50.40 Female $58.80 Male 15 weeks $53.70 Female $65.40 Male 16 weeks $57.40 Female $65.40 Male 17 weeks $61.00 Female $69.30 Male 18 weeks $64.50 Female $73.20 Male 19 weeks $67.80 Female $77.00 Male 20 weeks $71.30 Female $81.00 Male 21 weeks $74.80 Female $85.00 Male 22 weeks $78.50 Female $89.20 Male 23 weeks $82.00 Female $97.20 Male 24 weeks $85.50 Female $104.60 Male
Standard Supply
JAX Ready Strain®. Most popular strains. Readily available in any quantity you need.
Supply Notes
- Shipped at a specific age in weeks. Mice at a precise age in days, littermates and retired breeders are also available.
- This strain is available from some international Charles River (CR) breeding facilities in Japan and/or Europe. For more information, see the Worldwide Distributor List for JAX® Mice.
Frozen Products
Price (US dollars $) Frozen Embryo $2242.50
Standard Supply
JAX Ready Strain®. Most popular strains. Readily available in any quantity you need.
Supply Notes
- Cryopreserved Embryos
Available to most shipping destinations1
This strain is also available as cryopreserved embryos2. Orders for cryopreserved embryos may be placed with our Customer Service Department. Experienced technicians at The Jackson Laboratory have recovered frozen embryos of this strain successfully. We will provide you enough embryos to perform two embryo transfers. The Jackson Laboratory does not guarantee successful recovery at your facility. For complete information on purchasing embryos, please visit our Cryopreserved Embryos web page.
1 Shipments cannot be made to Australia due to Australian government import restrictions.
2 Embryos for most strains are cryopreserved at the two cell stage while some strains are cryopreserved at the eight cell stage. If this information is important to you, please contact Customer Service.
In case of dissatisfaction for a valid reason and claimed in writing by a purchaser within ninety (90) days of receipt of mice, products or services, JACKSON will, at its option, provide credit or replacement for the mice or product received or the services provided.
In no event shall JACKSON, its trustees, directors, officers, employees, and affiliates be liable for any causes of action or damages, including any direct, indirect, special, or consequential damages, arising out of the provision of MICE, PRODUCTS or services, including economic damage or injury to property and lost profits, and including any damage arising from acts or negligence on the part of JACKSON, its agents or employees. Unless prohibited by law, in purchasing or receiving MICE, PRODUCTS or services from JACKSON, purchaser or recipient, or any party claiming by or through them, expressly releases and discharges JACKSON from all such causes of action or damages, and further agrees to defend and indemnify JACKSON from any costs or damages arising out of any third party claims.
MICE and PRODUCTS are to be used in a safe manner and in accordance with all applicable governmental rules and regulations.
The foregoing represents the General Terms and Conditions applicable to JACKSON's MICE, PRODUCTS or services. In addition, special terms and conditions of sale of certain MICE, PRODUCTS or services may be set forth separately in JACKSON web pages, catalogs, price lists, contracts, and/or other documents, and these special terms and conditions shall also govern the sale of these MICE, PRODUCTS and services by JACKSON, and by its licensees and distributors.
Acceptance of delivery of MICE, PRODUCTS or services shall be deemed agreement to these terms and conditions. No purchase order or other document transmitted by purchaser or recipient that may modify the terms and conditions hereof, shall be in any way binding on JACKSON, and instead the terms and conditions set forth herein, including any special terms and conditions set forth separately, shall govern the sale of MICE, PRODUCTS or services by JACKSON.


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Pink bebe Logo Mug
Style # 215124
AVAILABILITY: Unavailable for immediate shipment.
There is not other way to start the day than with a cup of tea or coffee in a pretty pink bebe mug! Also makes a cute gift.
- Ceramic, magnesia porcelain, silicone
- Microwave safe
- Dishwasher safe
- Imported
Pink bebe Logo Mug 5 5
Pink Coffee Mugs! I purchased 2 of these mugs. Pink is my favorite color and these are hard to find. They are a little bigger than I thought which is great! You can even put them in the microwave or dishwasher....definitely suggest them...makes a great gift! December 14, 2013
Cute I got the pink & the black ones so I can mix it up when I drink coffee, tea, etc. Sturdy & microwave safe. April 25, 2013
nothing special about this mug It looks great online, I thought the bebe logo was carved, but it looks like a cheap sticker. On the positive side, the color is really nice. April 20, 2013
This product is biutiful This product is great I love the color pink, I love logo bebe is fantastic March 26, 2013
Fun Stuff OK, went on a Bebe retail bender and got both water bottles, the cold water cup, the travel coffee mug and this the coffee cup. I keep it a work for that afternoon pick me up, it's super cute and everyone knows how much I loooovvveee Bebe!!! February 27, 2013
1-5 of 5


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PEORIA, Ill.---Right wing Spencer Machacek lit the lamp 2:07 into overtime to lift the Chicago Wolves to a 3-2 victory over the Peoria Rivermen at Carver Arena Friday night.
Defenseman Paul Postma and center Jared Ross potted the other goals for Chicago, which picked up its fourth straight win.
Machacek scored the game-winning goal when he put home a rebound from the side of the crease past Peoria goaltender Ben Bishop 2:07 into the extra session.
Peoria (12-7-1-1) opened scoring under a minute into the first period when right wing Derek Nesbitt took a rebound off of Chicago goaltender Peter Mannino and shot it into the open side of the net.
Chicago (10-11-1-0) tied the score at one at the seven minute mark of the first period when Postma fired a wrist shot from the top of the right faceoff circle past Bishop, stick side.
The Wolves took a 2-1 lead 10:36 into the second period when Ross scored on a breakaway sending the puck past Bishop, top shelf.
Bishop (6-7-1) recorded 25 saves.
The Rivermen tied the score 2-2, 4:43 into the third frame when center Dave Scatchard grabbed the rebound off of the glass and pushed the puck into the net past an outstretched Mannino.
Mannino (5-3-0) kicked out 33 of 35 shots to collect his fourth straight win.
The Wolves play the Grand Rapids Griffins at the Allstate Arena on Saturday at 7 p.m.
Chicago 1 1 0 1 -- 3
Peoria 1 0 1 0 -- 2
First Period---1, Peoria, Nesbitt 2 (Scatchard, Spina), 0:55; 2, Chicago, Postma 2 (Galivan, Kozek), 7:00. Penalties---Cole, Peoria (double minor – high sticking), 16:12.
Second Period---3, Chicago, Ross 5 (Dawes), 10:36. Penalties---Krog, Chicago (hooking), 6:54; Peltier, Peoria (interference), 14:56; Cracknell, Peoria (hooking), 17:28.
Third Period---4, Peoria, Scatchard 3 (Nesbitt, Spina), 4:43. Penalties---None.
Overtime---5, Chicago, Machacek 10 (Davies), 2:07. Penalties---None.
Shots on goal---Chicago: 6-12-8-2--28. Peoria: 10-10-15-0--35. Power plays---Chicago: 0-3. Peoria: 0-1. Goalies--Chicago, Mannino (33-35). Peoria, Bishop (25-28). A---6,018. Referee---Keith Kaval. Linesmen---Aaron Mills and Al Stensland.


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Thursday, September 5, 2013
Members of the Summerville Evening Rotary Club conducted their July "Rotary RAK" (Random Acts of Kindness) weeding and planting flowers beds at The Village at Summerville, formerly known as the Presbyterian Home of Summerville. The club attempts to conduct a project every month within the community. The Summerville Evening Rotary Club is the newest Rotary club in District 7770 and meets at Montreux Bar & Grill Restaurant, 127 West Richard Ave, Summerville, SC Monday's at 5:30 p.m. All are welcomed.090
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Yes, yes, truth be told, yesterday was indeed my birthday. But older? Naw! Who you kidding? What's that old, creepy and very dated saying? "She's not getting older, she's getting better!" * sigh * as he looks at her adoringly…..
But though, after a certain * ahem * age we dread birthdays, who doesn't love a party? Or the attention? Or the gifts! I certainly will never scoff at any of those! And this year was no different (okay, so I never have a party… at least I get lots of attention and at least a few gifts).
Well, at least as the baker in the family I get to choose my own cake. For years I have been making Rose Levy Beranbaum's Chestnut Cake made with chestnut flour, which I just adore. But even frosted with our favorite chocolate buttercream frosting, they just don't get it!
So this year, I returned to an old favorite of mine : a deep, dense, dark devilish chocolate cake spread with the chocolate buttercream and filled with a slightly bitter coffee-flavored whipped cream. Oh so decandent!
1 ¾ cup flour
2 cups sugar
¾ cup unsweetened cocoa powder
1 ½ tsp baking powder
1 ½ tsp baking soda
1 tsp salt
2 large eggs
1 cup milk or buttermilk
½ cup vegetable oil
2 tsps vanilla
1 cup boiling water, coffee, fruit juice or a combination of water and something else
Preheat oven to 350°F (180°C). Oil and flour two 10-inch round cake pans generously.
Combine all of the dry ingredients in a large mixing bowl. I whiz them with the electric mixer on low speed for 30 seconds until everything is well combined.
Add the eggs, milk, oil and vanilla. Beat on low until well blended then increase the mixer speed to medium and beat for 2 minutes.
Stir in the boiling water (today I used about ¾ cup strong coffee with ¼ cup water). When I make a Forêt Noire, I use ½ cup juice from jarred cherries mixed with ½ cup water, then I use the cherries layered with plain, slightly sweetened whipped cream in between the two chocolate cake layers.
Carefully divide the batter between the two prepared cake pans – it will be watery.
Bake in the preheated oven for 35 – 40 minutes or until the center is set.
Remove from oven and allow to cool for 10 – 15 minutes on cooling racks before turning them out onto the racks to cool completely.
Mine look a bit funny because I have one of those rinky-dink tiny French ovens and two round cake pans do not fit on the oven rack at the same time. I end up having to juggle the cake pans – up and down, back and forth – throughout the baking in order to make sure that each layer cooks fairly evenly and that neither burns. But that takes nothing away from their flavor! And I need a new oven.
1 to 2 cups heavy cream
4 tsps instant coffee or espresso powder
4 – 8 tsps powdered or confectioner's sugar, depending on strength of your coffee as well as your personal taste
Make sure your glass bowl and beaters are well chilled.
Start whipping the cream on high speed until it starts to thicken, then gradually add the coffee and the sugar. Continue to beat until very thick. Put in the fridge until ready to use.
If you taste this now, it may taste too bitter or strange. True. But sandwiched in between the layers of chocolate cake gives an incredible, fabulous flavor sensation!
A little under 3 cups (11 ½ oz, 350 g) powdered/confectioner's sugar
8 Tbs (120 g) unsalted butter softened to room temperature
2 oz (50 g) unsweetened cocoa powder
4 Tbs boiling water
With an electric mixer, beat the butter with the sugar until light and fluffy.
Add the cocoa powder and the boiling water and beat until the ingredients are completely and well blended and the frosting is light and fluffy. If it is too thin just pop it in the fridge for a few minutes.
Assemble the cake :
Put one of the chocolate cake layers on a serving platter. Spread the coffee whipped cream evenly over the layer, putting as much or as little as desired. (Any left over can be refrigerated for another use or served with the cake).
Carefully place the second cake layer on the whipped cream, then frost the sides and top of the cake with the chocolate buttercream.
I pressed – or attempted to press – chocolate sprinkles/jimmies all over the sides. Now, this may seem to be overkill, but hey it's my birthday and I can do what I want! I also never claimed to be the Cake Decorating Queen and my attempts at anything fancy end up looking sorry indeed.
But this cake is so fabulously delicious, rich and decadent, chocolaty to the extreme without going overboard, as well as being so easy to make! The cake texture is close to one of a flourless cake, chewy, dense and moist. The coffee in the batter added to the coffee whipped cream just gives you a delicate coffee sensation in each mouthful.
Let's just say that this recipe is my birthday gift to you! Who's opening the champagne….?


text: Fire pits lend warm ambiance to any outdoor space, and allow you to linger longer outside at night. They're an inviting, safe way to add heat and light to evening get-togethers with your family and friends. Watching firelight flicker inside an outdoor fire pit is so relaxing, and the mesmerizing glow inspires singing, storytelling, and meditative silence. Our outdoor fire pits are carefully crafted to provide season after season of use, with safety features like built-in guardrails and spark guards to contain flying embers. Many come with a cooking grill and poker. Our steel fire rings are a unique way to add safety and style to campfires and bonfires.


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fewer cars on the road is a gift for everyone.
Give someone a shiny new Zipcar membership, a few hours behind the wheel or both.
They'll really appreciate it. Hey, we all will.
Give the freedom of Zipcar. Beats a fruit basket every time.
It's better to give a hint than receive a Chia Pet.
Want to redeem a gift certificate? Go here.


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Things Antiques
Things Antiques is located in the heart of Hollywood. For 20 years we have been working and supplying the top designers in Los Angeles with one-of-a-kind pieces of furniture, lighting, and art.