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Many people ask about how to do this, so I thought I'd write up a sample. This sample basically searches the inbox of a user and then does an HTTP GET against the DAV:href to get the EML file and then does an HTTP PUT to upload it to a SharePoint document library.
using System; using System.Web; using System.Xml; using System.Net; using System.Text; namespace UploadEmlToSharePoint { /// <summary> /// Summary description for Class1. /// </summary> class Class1 { static System.Net.CredentialCache MyCredentialCache; /// <summary> /// The main entry point for the application. /// </summary> [STAThread] static void Main(string[] args) { System.Net.HttpWebRequest Request; System.Net.WebResponse Response; string strRootURI = "http://exchangeserver/exchange/User1/Inbox/"; string strSPSRootURI = "http://wss/Shared%20Documents/"; string strUserName = "User1"; string strPassword = "Passw0rd"; string strDomain = "DOMAIN"; string strQuery =""; byte[] bytes = null; System.IO.Stream RequestStream = null; System.IO.Stream ResponseStream = null; XmlDocument ResponseXmlDoc = null; XmlNodeList ResponseNodes = null; XmlNamespaceManager xmlnsm = new XmlNamespaceManager(new NameTable()); xmlnsm.AddNamespace("a","DAV:"); try { // Build the SQL query. strQuery = "<?xml version=\"1.0\"?><D:searchrequest xmlns:D = \"DAV:\" >" + "<D:sql>SELECT \"DAV:displayname\" FROM \"" + strRootURI + "\"" + "WHERE \"DAV:ishidden\" = false AND \"DAV:isfolder\" = false" + "</D:sql></D:searchrequest>"; // Create a new CredentialCache object and fill it with the network // credentials required to access the server. MyCredentialCache = new System.Net.CredentialCache(); MyCredentialCache.Add( new System.Uri(strRootURI), "NTLM", new System.Net.NetworkCredential(strUserName, strPassword, strDomain) ); MyCredentialCache.Add(new System.Uri(strSPSRootURI), "NTLM", new System.Net.NetworkCredential(strUserName, strPassword, strDomain)); // Create the HttpWebRequest object. Request = (System.Net.HttpWebRequest)HttpWebRequest.Create(strRootURI); // Add the network credentials to the request. Request.Credentials = MyCredentialCache; // Specify the method. Request.Method = "SEARCH"; // Encode the body using UTF-8. bytes = Encoding.UTF8.GetBytes((string)strQuery); // Set the content header length. This must be // done before writing data to the request stream. Request.ContentLength = bytes.Length; //Set the translate header to false Request.Headers.Add("Translate","f"); // Get a reference to the request stream. RequestStream = Request.GetRequestStream(); // Write the SQL query to the request stream. RequestStream.Write(bytes, 0, bytes.Length); // Close the Stream object to release the connection // for further use. RequestStream.Close(); // Set the content type header. Request.ContentType = "text/xml"; // Send the SEARCH method request and get the // response from the server. Response = (HttpWebResponse)Request.GetResponse(); // Get the XML response stream. ResponseStream = Response.GetResponseStream(); // Create the XmlDocument object from the XML response stream. ResponseXmlDoc = new XmlDocument(); ResponseXmlDoc.Load(ResponseStream);
ResponseNodes = ResponseXmlDoc.GetElementsByTagName("a:response"); if(ResponseNodes.Count > 0) { Console.WriteLine("Non-folder item hrefs..."); // Loop through the display name nodes. for(int i=0; i<ResponseNodes.Count; i++) { // Display the non-folder item displayname. XmlNode responseNode = ResponseNodes[i]; XmlNode hrefNode = responseNode.SelectSingleNode("a:href",xmlnsm); XmlNode displayNameNode = responseNode.SelectSingleNode("a:propstat/a:prop/a:displayname",xmlnsm);
//Downloads the EML file from the specified URL byte[] emlFile = GetBytesFrom(hrefNode.InnerText);
//Uploads the EML file to the SharePoint document library UploadToSPS(emlFile,strSPSRootURI + System.Web.HttpUtility.UrlPathEncode(displayNameNode.InnerText)); } } else { Console.WriteLine("No non-folder items found..."); } // Clean up. ResponseStream.Close(); Response.Close(); } catch(Exception ex) { // Catch any exceptions. Any error codes from the SEARCH // method request on the server will be caught here, also. Console.WriteLine(ex.Message); } Console.WriteLine("Done."); Console.Read(); } static byte[] GetBytesFrom(string DavURL) { Console.WriteLine(DavURL); byte[] buffer; System.Net.HttpWebRequest Request; System.Net.HttpWebResponse Response; System.IO.Stream ResponseStream; Request = (HttpWebRequest)HttpWebRequest.Create(DavURL); Request.Credentials = MyCredentialCache; Request.Headers.Add("Translate","f"); Response = (HttpWebResponse)Request.GetResponse(); ResponseStream = Response.GetResponseStream(); buffer = new byte[Response.ContentLength]; ResponseStream.Read(buffer,0,(int)Response.ContentLength); ResponseStream.Close(); Response.Close(); return buffer; } static void UploadToSPS(byte[] fileBytes, string URL) { Console.WriteLine("Uploading " + fileBytes.Length.ToString() + " bytes to " + URL); System.Net.HttpWebRequest Request; Request = (HttpWebRequest)HttpWebRequest.Create(URL); Request.Credentials = MyCredentialCache; Request.ContentLength = fileBytes.Length; Request.Method = "PUT"; System.IO.Stream str = Request.GetRequestStream(); str.Write(fileBytes,0,fileBytes.Length); str.Close(); Request.GetResponse(); } } }
ResponseNodes = ResponseXmlDoc.GetElementsByTagName("a:response");
PingBack from http://mhinze.com/links-for-2007-02-08/
Currently I can recieve but in eml format, will this fix so I recieve as email to open in my outlook client as a regular email. I need to have this work, we are intergrating the document library to our exchange 2007. I was told I needed a mapi client installed on the sharepoint 2007 server. But not sure if any special configuration is involved. Any help would be great.
mmm.. nice design, I must say..
So the reason you need a MAPI client is that Outlook is a MAPI client and reads MAPI data from a MAPI store. MSG is the file format for MAPI data. These types of files would open in Outlook. It would involve a completely different code path to get the MAPI data from the store and save it as MSG. You can use MAPI Editor's source code available from this KB article (http://support.microsoft.com/kb/291794/) to do so. However, please note that MAPI code is not supported inline with .NET code. You would have to do this in C++ unmanaged code and wrap it in a COM+ component that you could call into from .NET. Outlook Object Model can also save messages as MSG files, but be careful again because Outlooks OMG (Object Model Guard) can prompt you with security prompts. Once you save the msg file to the disk, you can use the same UploadToSPS code I have above to push it into SharePoint. The reason I went after the EML (MIME content) files is that WebDAV is supported under .NET.
We implement a VERY similar technique, although we found you have to loop read the bytes as on larger emails stuff gets lost/truncated. See my blog entry for what I mean.
You will find you'll need to read the email stream in chunks to avoid loosing stuff and ending up with a truncated email (especially true when there are attachments, my blog has code for our very similar implementation.
Hi Patrick,
Good article.
What I am trying to do is write an event handler for an email enabled document library. So, when my document library receives the eml file, I want to open the file and find out who the sender is, the subject, cc fields, body and what the attachments are. Do you know of any way to parse eml files through code?
Hi Pritam. Yes, CDOSYS is an effective API for parsing eml files.
I'm new to webDAV. How would you use this in a project?
Hi Patrick Creehan,
I am also working in SharePoint 2007. I am facing some problem with images while sending it thru mail. Our problem is, we are sending Emails (watch word - with image) to end users on daily basis. Currently we are doing it manually. Now we need to automate this using SharePoint designer work flow. The problem is background images are not displayed while getting the emails in outlook. There is no problem with the text. We are in hot stove now. Could you please help me to solve this?
Hi Saharathendral,
Are you getting the red X where your image should be or are you just seeing a plain text email?
If you are seeing the red X, then it is most likely that your images are stored in a location that requires authentication to access. You should put them somewhere they can be accessed anonymously. You'll also want to look at these articles:
In Outlook 2003, there was an entirely different security mechanism in Tools/Options that governed how and if images were displayed in HTML emails.
your reply helps us a lot to solve our problem. Thanks a lot. ;-)
Hi Patrick!
I'm not sure whether this is a good place to ask about this, so sorry if not, but here goes anyway.
We have a "company-general" email address, that actually goes to just one person's mailbox. Currently the person then forwards the messages to whoever he guesses the message concerns. We would like to have the messages sent to this address be somehow automatically moved to a place in Sharepoint where other people could see them if they like and put alerts to get notification etc. Ideally this place could be a Sharepoint discussion forum so that the messages could be seen similar to as in an email reader.
We could of course just forward the mails coming to the address to all those who want to receive them, but then the decision of whether to receive them or not would be more of an on-off decision, all or nothing. If the messages (even only text body would be enough) would automatically appear on a Sharepoint page, then the people could check them when they want (for example when they expect something concerning them to be inbound) and then ignore them in other times.
Finally, in our location we don't have the Sharepoint server, it is managed by IT in another location so it would be best if something like this could be done without any actual extra coding, just utilizing existing features, adding a suitable web part or something similar. Do you think anything like this would be reasonably easily doable?
I am getting the remote server returned an error 403 forbidden after it reach this line
// Send the SEARCH method request and get the
// response from the server.
Response = (HttpWebResponse)Request.GetResponse();
I am getting a 403 error here on this line
// Get a reference to the request stream.
RequestStream = Request.GetRequestStream();
I have a https URI web email access
string strRootURI = "https://webmail.hhs.gov/exchweb/bin/auth/owalogon.asp?url=https://webmail.hhs.gov/exchange&reason=0";
string strSPSRootURI = "http://samhsa-dmsg:48497/Email%20Import/";


text:
Art Of Todd Lawson: Vol. 1
First Wave Of Offense
(Hardcover Standard Edition)
ONLINE ONLY DIRECT PRICE $59.95 (you save 15% when you buy directly online)
Bookland Barcode on back with RRP Cover Price: $69.95 US / $80.00 CAN
Showcasing a select collection of the most popular as well as lesser known works from Canadian contemporary artist Todd Lawson. For the first time ever a collection of over 70 paintings now contained in one publication. Todd's work has been displayed throughout North America as well as in the UK and continues to grow in popularity with collectors who like a bit of sour in their sweet and edge in their soft. A must have for any fans of outsider contemporary art.
Designed by independent publishing design and branding group Miner Thought, full colour 120 page hardcover edition book. Standard 80# matte paper.
Learn more about the artist at http://www.toddlawson.com
Gallery/store owners -buy multiple copies of this book at a discounted merchant rates up to 35% off the RRP when you buy direct at http://minerthought.com/trade.html
This book is also available in paperback for only $34.95. Learn more: http://www.blurb.com/my/book/detail/936033
This book is also available with 100# premium silk-finish paper in a limited edition of 50 which features an alternate back cover. Buy the Collectors edition while available for only $5.05 dollars more here: http://www.blurb.com/bookstore/detail/799800
Miner: ('mine') to burrow beneath the surface – ('miner'): noun. Thought: To exercise the powers of judgment, conception, or inference. MinerThought's goal is to open the eyes, minds and hearts of our clients and their customers with original content and visual communications. We make advertising, design, books, brands and visual marketing - but we treat it as art.
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subset: commoncrawl


text:
Latest Silversmiths Stories
Central Valley residents introduced to medical breakthrough that can take years off their appearance at a fraction of the cost of traditional surgical facelifts. Visalia, CA (PRWEB) July 29, 2011 By the time Central California men and women reach their 40's, the toll of aging begins to show itself.
LONDON, February 15, 2011 /PRNewswire/ -- Asprey hosted the official BAFTA, Nominees' Party for the third consecutive year this weekend. The prestigious Cocktail Party, held in the luxurious surroundings of Asprey's New Bond Street Flagship Store, celebrated the film nominations and Asprey's continued partnership with BAFTA. To view the Multimedia News Release, please click: http://multivu.prnewswire.com/mnr/prne/asprey/47600/ The store, occupied by Asprey since 1847 and...
Midnight Ride, Industrial Dawn: Paul Revere and the Growth of American Enterprise (Johns Hopkins University Press) is the culmination of more than a decade of research that began in graduate school.
LONDON, November 30, 2010 /PRNewswire/ -- Asprey is pleased to announce that a range of Asprey items have been featured in the highly anticipated film, 'The Tourist', starring Angelina Jolie and Johnny Depp. To view the Multimedia News Release, please click: http://multivu.prnewswire.com/mnr/prne/asprey/46315/ The coolly elegant look achieved for the lead character, played by Angelina Jolie, included Asprey accessories in several of the ensembles, including The Regent clutch in...
WASHINGTON, June 8 /PRNewswire-USNewswire/ -- Governor George Pataki, Chairman of Revere America, announced today that the organization has collected more than 125,000 signatures from Americans in all 50 states who want to repeal and replace ObamaCare.
WASHINGTON, May 26 /PRNewswire-USNewswire/ -- With a new poll by Rasmussen showing that 63% of likely voters want to repeal ObamaCare, the American people have an unparalleled opportunity to make their voices heard by signing Revere America's national petition to repeal and replace the government takeover of healthcare.
-- Governor George Pataki Kicks off National Petition Drive to Repeal and Replace ObamaCare with Six State Swing -- WASHINGTON, April 15 /PRNewswire-USNewswire/ -- Former Governor George Pataki (R-NY), Chairman of Revere America (www.revereamerica.org), announced today the first six stops of the new organization's grassroots tour and national petition drive to gather the signatures of one million Americans who want to repeal and replace ObamaCare with the responsible reforms our health care...
LONDON, February 21, 2010 /PRNewswire/ -- - Asprey Hosts BAFTA Award Nominees at Bond Street Store Asprey hosted the official BAFTA nominee party tonight at its beautiful New Bond Street store, where a star studded guest list included the finest talent from the film world.
Tiffany & Co. Mark L. Aaron, 212-230-5301 email@example.com or Arnold & Porter LLP James B. Swire, 212-715-1013 firstname.lastname@example.com Tiffany & Co.
- The analysis of literature, focusing on the words and grammar to the exclusion of context or literary merit.


text:
Owner of sunken ferry blamed for 5 earlier crashes
SEOUL, South Korea (AP) — In the same narrow waterways where more than 300 people died this spring aboard the ferry Sewol, another ship owned by the same company crashed into an oil tanker 11 years earlier. The ferry's captain had chosen the difficult water path to cut a mere seven miles from its journey.
It was among five crashes, from 2003 to 2011, that government investigators blamed mostly on sailors of Chonghaejin Marine Co. ferries. Three of the incidents occurred within a 12-month span, and after those occurred, a government investigator chided the company for failing to make safety reforms.
None of the crashes caused fatalities, but together, some experts say, they were reason enough for regulators to suspend or even revoke the company's license. That never happened, and in fact Chonghaejin was allowed to expand by adding the Sewol to its fleet last year.
Chonghaejin's punishment for those five failures: two one-month suspensions for sailors, three verbal warnings to captains, one verbal warning to the company and a fine of 7.5 million won ($7,400). The biggest fine that can be issued in ferry-safety cases is just 30 million won ($29,400).
The Korean Maritime Safety Tribunal, an arm of the Ministry of Oceans and Fisheries that serves as a maritime court, recommended safety changes to the company as well, but they were nonbinding.
After the 2003 oil-tanker crash, the tribunal asked that Chonghaejin stop using the Maenggol Channel, but the shortcut saved the company money on fuel. The Sewol sank in the same channel, about 16 kilometers (10 miles) from the 2003 crash.
Chung Yeong-seok, a professor of maritime law at Korea Maritime and Ocean University, said South Korea could have and should have gone further than it did.
"Even though the current law is not strict, the maritime ministry, with its administrative power, could (cancel a ferry license) based on the tribunal's findings. But it didn't do it," Chung said.
Prosecutors blame the CEO and four company employees for causing the April 16 Sewol sinking by overloading the ship with poorly stowed cargo after a risky redesign, and by neglecting safety in other ways. They also accuse 15 crew members of negligence and of failing to perform their duty to rescue passengers. All but one of the crew has pleaded not guilty, and lawyers for the other defendants have said the cause of the sinking remains unclear.
Chonghaejin's license has been revoked, and authorities continue to search for the patriarch of the family that owns the company, 73-year-old billionaire Yoo Byung-eun.
The government has blamed its failure to prevent the disaster on the cozy ties between the industry and regulators. President Park Geun-hye has vowed to get rid of what she and South Korean media have called a "government mafia" consisting of retired public servants who work at private companies.
South Korea had reason not to revoke or suspend Chonghaejin's license before the Sewol sank. The company had a monopoly on South Korea's longest domestic sea route, the one between Incheon and Jeju that the Sewol was plying went it went down. A license suspension or revocation would have affected travel plans for thousands of people and transport for thousands of tons of cargo.


text:
Frank-ter Haar syndrome is a rare disorder associated with skeletal, cardiac, ocular and craniofacial features including hypertelorism and brachycephaly. The most common underlying genetic defect in Frank-ter Haar syndrome appears to be a mutation in the SH3PXD2B gene on chromosome 5q35.1. Craniosynostosis, or premature fusion of the calvarial sutures, has not previously been described in Frank-ter Haar syndrome.
Case presentation
We present a family of three affected siblings born to consanguineous parents with clinical features in keeping with a diagnosis of Frank-ter Haar syndrome. All three siblings have a novel mutation caused by the deletion of exon 13 of the SH3PXD2B gene. Two of the three siblings also have non-scaphocephalic sagittal synostosis associated with raised intracranial pressure.
The clinical features of craniosynostosis and raised intracranial pressure in this family with a confirmed diagnosis of Frank-ter Haar syndrome expand the clinical spectrum of the disease. The abnormal cranial proportions in a mouse model of the disease suggests that the association is not coincidental. The possibility of craniosynostosis should be considered in individuals with a suspected diagnosis of Frank-ter Haar syndrome.
Keywords:Frank-ter Haar syndrome; Craniosynostosis; Sagittal synostosis; Intracranial pressure
Frank-ter Haar syndrome is a rare disorder comprising cardiovascular, skeletal and craniofacial anomalies including hypertelorism, brachycephaly and a wide anterior fontanelle [1]. The 'ter Haar' syndrome was initially described in three related children, all of whom developed severe cardiovascular complications [2,3]. These patients appeared to share many of the craniofacial and skeletal features normally associated with Melnick-Needles Syndrome, an X-linked congenital disorder of skeletal dysplasia. However the autosomal recessive pattern of inheritance and congenital cardiac defects distinguished the syndrome as a separate entity [4,5]. The condition affecting this subgroup of patients was renamed Frank-ter Haar syndrome following the realisation that ter Haar's three patients shared many of the features described previously by Frank in an 18 month old female with megalocornea, skeletal dysplasia and developmental delay [1,6].
A genetic basis for Frank-ter Haar syndrome has recently been established through homozygosity mapping studies in patients from 12 affected families, identifying homozygous mutations in the SH3PXD2B gene on chromosome 5q35.1 as the most common cause [7]. The analysis of patients from 13 families identified 4 different intragenic mutations, and one complete deletion of SH3PXD2B, accounting for the phenotype in 7 of the families [7]. Clinical features characterising this group of mutation positive patients include brachydactyly megalocornea, hypertelorism, a prominent forehead, brachycephaly, a wide anterior fontanelle, micrognathia, a broad mouth and full cheeks [7]. Sh3pxd2b null mice appear to share many of the skeletal, craniofacial, cardiac and ocular defects described in Frank-ter Haar syndrome, supporting the link between this gene and the syndrome [7].
The craniofacial features of Frank-ter Haar syndrome are numerous however to our knowledge craniosynostosis has not been reported previously. Craniosynostosis refers to the premature fusion of one or more of the calvarial sutures, affecting 1 in 2500 individuals [8]. The calvarial sutures arise where cranial ossification centres meet at approximately 18 weeks of embryonic development. From this stage onwards further skull growth is appositional, and deposition of new mineralised bone matrix occurs along the margins of the sutures [8]. Synchronised closure of these sutures during the postnatal period allows the calvarium to achieve its full size and shape, facilitating normal expansion and development of the underlying brain [9]. Growth restriction along a given margin may result in compensatory, excessive growth at other sutures leading to skull deformity [10]. Furthermore as the brain continues to develop and expand within a limited cranial capacity, raised intracranial pressure (ICP) may complicate craniosynostosis, with potential neurodevelopmental consequences [11].
Here we describe three siblings with the Frank-ter Haar syndrome phenotype, all of whom are homozygous for a complete deletion of exon 13 of the SH3PXD2B gene. They demonstrate many of the classic features associated with Frank-ter Haar syndrome, however two of them also have presented with non-scaphocephalic sagittal synostosis complicated by raised ICP.
Case presentation
We present a family of four children born to consanguineous (first cousin) parents originating from Kashmir, Pakistan. Three siblings (two males and one female) have Frank-ter Haar syndrome (Table 1, Figure 1) confirmed by genetic analysis. The youngest male sibling appears to be unaffected.
Table 1. Comparison of clinical features in patients 1–3 with previous cases with prove SH3PXD2B mutations
Figure 1. A-D: Patient 1.A: Antero-posterior (AP) view showing facial features including hypertelorism. B: Lateral view showing brachycephaly and micrognathia. C: X-ray (XR) right radius/ulna showing broad appearance of the radius at the junction between the proximal and middle thirds. D: XR left hand showing crowding of the carpal bones, broad metacarpals, proximal and middle phalanges, and flexion at the MCP and PIP joints. E-G: Patient 2.E: AP view showing facial features including hypertelorism. F: Lateral view showing brachycephaly. G: 3D CT scan showing absence of the sagittal suture. H-J: Patient 3.H: AP view showing facial features including hypertelorism. I: Lateral view showing class III malocclusion and brachycephaly. J: 3D CT scan showing absence of the sagittal suture.
Patient 1
This male patient was the firstborn child, delivered by caesarean section for macrocephaly and cephalopelvic disproportion. Hypertelorism and dysmorphia were noted at birth, without evidence of fetal head constraint. He was referred to the Plastic Surgery Department for severe hypertelorism and forehead bossing. On examination at 20 months the head circumference was 49 cm (>99.6th centile), with marked hypertelorism, frontal skull bossing, a wide open anterior fontanelle, and a persistent metopic suture. The cephalic index was 86% confirming the clinical appearance of mild brachycephaly (normal range 76-85%). Limb examination revealed severe bilateral cavo varus of the feet and bilateral metacarpo-phalangeal (MCP) joint hyperextension and contractures.
CT head and 3D studies at age 1 year and 10 months demonstrated patency of the major cranial sutures and confirmed the clinical findings of hypertelorism and an open anterior fontanelle. Surgical correction for bilateral cavo varus at age 7 years and 4 months was complicated by cardiopulmonary decompensation, pulmonary hypertension and a marked oxygen requirement during the post-operative period. Echocardiography demonstrated severe mitral valve prolapse with moderate mitral regurgitation, mild tricuspid valve prolapse and mild aortic regurgitation, however ventricular function was good at the time.
The diagnosis of Frank-ter Haar syndrome was initially suspected at the age of 18 months. The G-banded karyotype was normal (46,XY). Alternative clinical diagnoses considered included Winchester syndrome and geleophysic dysplasia, but genetic testing for these disorders was normal. The diagnosis was confirmed following the identification of the SH3PXD2B mutation at age 12 years and 4 months (Figure 2, Figure 3).
Figure 2. Genome and sequence context of deletion in SH3PXD2B. The upper panel shows a schematic representation (not to scale) of SH3PXD2B around the deleted region. Exons are shown as rectangles with coding sequence filled black and the 3′ UTR unfilled. Affected individuals were homozygous for a 7,625 bp deletion (double-headed red arrow). The breakpoint (dotted red lines) occurred at the position shown in the sequence chromatogram from patient 2, with an ambiguity of one nucleotide because a cytosine is located at both breakpoints. The lower panel shows the results of PCR with the primer pairs indicated. Individuals homozygous for the deletion failed to amplify using primer pair 12F/13-8R (upper gel image, 2,268 bp product); all family members yielded a truncated product using primer pair 12F/13-7R (lower gel image; non-deleted product would be 11,464 bp) indicating that unaffected family members are heterozygous for the deletion. C: control DNA from an unaffected, unrelated individual. N: negative control. The hg19 co-ordinates of the deleted region are chr5:171,763,754-171,771,378.
Figure 3. Schematic plan of SH3PXD2B (TKS4) protein showing all mutations identified to date and their location in relation to domains defined by PROSITE (prosite.expasy.org). Note the extent of the deletion identified in this family. A deletion of the entire encoding SH3PXD2B gene has also been reported in another affected family [7].
The mitral valve function subsequently deteriorated resulting in ventricular dilatation and requiring mitral valve repair at 12 years and 5 months. Examination at age 12 years and 11 months revealed a progressive lower limb oedema, which had been developing over a 2 year period, and was initially suspected to be a complication of his cardiac condition. Originally affecting the right leg only, it had progressed to involve the left groin, left leg and genitalia, and appeared to be lymphatic in origin. Lymphoscintigraphy studies at age 13 years and 10 months demonstrated profound lymphatic abnormalities in both lower legs and functional hypoplasia of the main draining lymphatics.
This patient has a left-sided astigmatism and meibomian gland dysfunction; however he does not have any of the ocular complications classically associated with Frank-ter Haar syndrome, such as megalocornea and glaucoma [7,16]. Intraocular pressures have been within the normal range, without clinical evidence of glaucoma. He has been attending a mainstream school with learning support, and is under occupational therapy for difficulties with fine motor skills, washing and dressing as a result of the severe hand contractures and deformity.
Patient 2
This is the second affected sibling in the family, delivered by caesarean section for maternal proteinuria without evidence of fetal head constraint. Facial features including hypertelorism were noticed at birth. Examination on presentation to the craniofacial department at age 6 years and 6 months demonstrated hypertelorism, brachycephaly (cephalic index 89%), a flat nasal bridge, prominent ears, a class III malocclusion and hypoplastic teeth. Her fingers and toes appeared shortened and exostoses on the dorsal aspect of the radius and ulna were visible on examination of the arms.
CT head and 3D studies were carried out at age 6 years and 10 months to further delineate the craniofacial features. This demonstrated sagittal synostosis, however the skull was not scaphocephalic in shape, but instead appeared brachycephalic in keeping with the clinical examination findings. There was evidence of limited space within the supratentorial compartment, and small ventricles. Although neurological development had previously been within normal limits, difficulties with literacy and recurrent headaches were reported at this time, also suggestive of raised ICP. ICP monitoring (Codman Microsensor, Codman and Shurtleff Inc., Raynham, Mass.) demonstrated elevated pressures with a mean of 22 mmHg and 5 significant associated B-waves. Calvarial expansion was successfully undertaken. She made a good post-operative recovery, and the headaches subsequently resolved.
This sibling has also developed cardiac complications but has been less severely affected than her older sibling. Echocardiography at age 7 years and 6 months demonstrated mild-moderate mitral regurgitation with mitral valve prolapse, and minimal aortic regurgitation. Left ventricular function is currently stable. The diagnosis of Frank-ter Haar syndrome was made at age 7 years and 7 months following genetic analysis of the family (Figure 2).
This patient has not developed any of the ocular complications classically associated with Frank-ter Haar syndrome and intraocular pressures and appearance of the retina have been within normal limits. She has been attending a mainstream school, but has experienced some difficulties with literacy.
Patient 3
Patient 3 is the younger affected male sibling. He was delivered by caesarean section following a normal pregnancy, without evidence of fetal head constraint. Facial features in keeping with Frank-ter Haar syndrome were noticed soon after birth including hypertelorism, a broad forehead, wide anterior fontanelle, brachycephaly and prominent ears. The diagnosis of Frank-ter Haar syndrome was made at age 6 years and 5 months, following genetic analysis of the family (Figure 2).
CT head and 3D studies were performed at age 6 years and 9 months following the outcome of these investigations in Patient 2. Evidence of sagittal synostosis without scaphocephaly, a flattened occiput and shortened AP diameter were identified on CT. There was hypoplasia of the inferior vermis but an otherwise normal appearance of the brain. Given the history of raised ICP in his sibling, ICP monitoring was undertaken at age 6 years and 10 months, demonstrating a mean pressure of 10 mmHg but 7 B waves, indicating raised ICP.
Examination at age 7 years and 1 month demonstrated an anterior open bite, class III malocclusion, hypoplasic teeth and skeletal features including short fingers and toes, and a mild positional talipes. The cephalic index was 88% in keeping with the clinical appearance of brachycephaly. Calvarial expansion was performed at age 7 years and 7 months.
Cardiac complications have also been documented, but have been less severe than the other affected siblings, with evidence of mild mitral regurgitation on echocardiography. No ocular complications have been reported, and intraocular pressures have been within the normal range. He has required some learning support for literacy and numeracy, but developmental progress and performance at school have been within normal limits.
Genetic analysis
We genotyped DNA samples from three affected patients, one unaffected sibling and their parents using the Affymetrix GeneChip® Human Mapping 250K Sty Array. Linkage analysis was performed assuming autosomal recessive inheritance, full penetrance, consanguinity and a disease allele frequency of 0.0001. Multipoint LOD scores were calculated using the program ALLEGRO [17]. We identified three regions of shared homozygosity on chromosomes 5 (150943995–173861414; hg18 coordinates), 8 (134041593–146264218) and 12 (115552170–123960781). Following the identification of mutations in Frank-ter Haar syndrome [7], it was noted that the region of homozygosity on chromosome 5 included the SH3PXD2B gene. All coding exons of SH3PXD2B were therefore PCR amplified in samples from the family (primer sequences available on request). As exon 13 failed to amplify (using primers 13F, 5′-AACATCTCCATTGGTGGTCC-3′ and 13R, 5′-GATGTGTTTGGCTGGCATC-3′, positions of primers are shown in Figure 2) in the affected individuals, an alternative primer pair (13-2F, 5′-CATGTAAGATATTCCCGGAACATGGT-3′; 13-2R, 5′-CCCCATGTCATTTTTCAGCTGGAACA-3′) was designed to rule out the possibility of a SNP underlying the primer and preventing annealing. This also failed to amplify in the 3 affected individuals, confirming a homozygous deletion of exon 13 of SH3PXD2B. Further primer pairs were designed throughout the 3′ untranslated region (UTR) to identify the extent of the deletion (primer sequences available on request), a primer pair near the end of the 3′ UTR successfully amplified in the affected individuals (13-7F, 5′-TACCTTACATCCCAGGGCAAACGGACAGCT-3′; 13-7R, 5′-GCGTGGCCTTGTGGCAGAGGTTTAAAATGAC-3′), the reverse primer of which was then used in a long PCR with the 12F primer (5′-AATCCAACTAGGTCCCCAGC-3′). This yielded a truncated product of ~3.5 kb rather than the normal 11,464 kb, which was sequenced to identify the exact breakpoint. All family members produced this smaller PCR product, demonstrating that unaffected members are heterozygous carriers of the allele bearing the deletion, which preferentially amplifies during PCR. The non-deleted allele was demonstrated by PCR between 12F and 13-8R (5′-CATAACACATCCTGAAGATAAACAGCCTAGACA-3′). The complete deletion of the coding part of exon 13 is predicted to abolish over half of the protein, including the SH3 domains and a proline-rich domain (Figure 3).
Frank-ter Haar syndrome is a rare, autosomal recessive disorder. We present three siblings born to consanguineous parents, who are homozygous for a deletion of exon 13 of the SH3PXD2B gene. These three children share many of the characteristic physical features of the Frank-ter Haar phenotype, however two of the three siblings also developed non-scaphocephalic sagittal synostosis, both cases being complicated by raised ICP. Craniosynostosis and raised ICP have not been described in previous reports of Frank ter-Haar syndrome.
The sagittal suture is involved in 50-60% of cases of craniosynostosis, and is therefore the most common suture to be synostosed [8,10]. Familial or genetic factors are less significant in sagittal synostosis than in other forms of craniosynostosis. In a large study of over 500 patients with isolated sagittal synostosis familial factors were identified in only 6% [18]. Most such cases are likely to be multifactorial, as causative genetic mutations are only rarely identified [19-21]. In comparison genetic factors comprising chromosomal abnormalities and gene mutations appear to account for at least 21% of cases of craniosynostosis overall [22]. Intrauterine fetal head constraint has been implicated as an important causative factor in sagittal synostosis [23], however to our knowledge neither of our two Frank-ter Haar siblings with sagittal synostosis had a history of intrauterine fetal head constraint.
It is widely acknowledged that premature fusion of the calvarial sutures may restrict normal growth and development of the underlying brain especially in syndromic cases. This can be complicated by raised ICP [10]. Raised ICP may be defined as an elevated baseline ≥15 mmHg, or >3 B waves (levels of 20–50 mmHg lasting 5–10 minutes before returning to baseline) in a 24 hour period [24,25]. In early studies, raised ICP was demonstrated in only 14% of cases of single suture synostosis but affected almost 50% of cases involving more than one suture [11]. The discovery of raised ICP in both of our patients with Frank-ter Haar syndrome and sagittal synostosis is unusual. Scaphocephaly is a predictable consequence of sagittal synostosis and refers to a boat-shaped skull deformity, with an increased anteroposterior diameter, decreased biparietal diameter and reduced cephalic index (<76%) In contrast, non-scaphocephalic sagittal synostosis is a rare diagnosis [25]. In the first reported cases of non-scaphocephalic sagittal synostosis only eight patients were identified out of 193 patients with isolated sagittal synostosis [25]. It is interesting to note that a diagnosis of raised ICP was made in 66% of these non-scaphocephalic cases, much higher than the reported incidence of 14% for all single suture synostosis [11].
An underlying genetic defect has recently been identified in Frank-ter Haar syndrome following homozygosity mapping in 16 patients from 12 affected families [7]. The mutated gene, SH3PXD2B on chromosome 5q35.1, encodes tyrosine kinase substrate with four Src homology 3 [SH3] domains (TKS4), a protein considered integral to the formation and functioning of podosomes. These are cell membrane protrusions which facilitate cell adhesion, migration, and extracellular matrix degradation [7]. Two mouse models with Sh3pxd2b loss of function mutations have been described. The first is a spontaneous nee mutation resulting from the deletion of a single base pair in exon 13 of the Sh3pxd2b gene (1303delA), which is thought to encode a truncated protein lacking part of the third and all of the fourth SH3 domains [16,26]. The second is an engineered null mutation created by the insertion of a gene trap vector between exons 3 and 4 of the Sh3pxd2b gene, which appears to result in a complete loss of gene expression [7]. Mice with the engineered null mutation appear to share many of the craniofacial, skeletal and cardiac features described in patients with Frank-ter Haar syndrome including mitral valve defects, hypertelorism and micrognathia [7,26,27]. Significantly, abnormal cranial proportions were described in these Sh3pxd2b null mice, which had a short snout, brachycephaly and hypertelorism, associated with persistently open sagittal sutures [7]. Both Sh3pxd2b null and Sh3pxd2b nee mice appear to develop ocular features including corneal opacities and anterior segment dysgenesis resulting in early-onset glaucoma. Sh3pxd2b nee mice also have abnormal craniofacial features including a short snout, brachycephalic skull, and Eustachian tube dysmorphology [16,26,27]. Surprisingly the ocular features associated with Frank-ter Haar syndrome were not identified in any of the three affected siblings, which contrasts both with the occurrence of megalcornea in all previous patients with identified SH3PXD2B mutations and with the ocular abnormalities described in both mouse models.
A variety of specific SH3PXD2B gene mutations have been reported in families with the Frank-ter Haar syndrome phenotype, but none so far have reported specific involvement of exon 13, the region of the gene affected in this family (Figure 3). However deletion of the entire SH3PXD2B gene has been described in one family [7]. Our three children have many features classically associated with Frank-ter Haar syndrome however they also have additional features which to our knowledge have not been described in previous reports (Table 1). These include lymphoedema, class III malocclusion, anterior open bite, open metopic suture, and non-scaphocephalic sagittal synostosis. Although we cannot completely exclude the possibility that these novel phenotypes are related to the two other regions of shared homozygosity identified on chromosomes 8 and 12 in this family, the involvement of the sagittal suture and additional craniofacial features in mouse models of the disease provides a precedent. The clinical features of craniosynostosis and raised ICP in this family therefore expand the clinical spectrum of Frank-ter Haar syndrome.
Written informed consent was obtained from the family for publication of this case report and any accompanying images. A copy of the written consent is available for review by the Series Editor of this journal.
Competing interests
The authors declare that they have no competing interests.
Authors' contributions
CLB collected the clinical data and wrote the paper. ALF performed the mutation analysis. JAH made the clinical diagnosis in the patients. GN and PN performed the homozygosity mapping. SAW supervised the clinical study. AOMW supervised the genetic studies and wrote the paper. DJ supervised the clinical study and wrote the paper. All authors read and approved the final manuscript.
The family for their cooperation.
Wellcome Trust (078666) for funding (AOMW).
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Pre-publication history
The pre-publication history for this paper can be accessed here:


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The ethics of database marketing: personalization and database marketing--if done correctly--can serve both the organization and the customer. (Business Matters).
This article:
* Examines the ethics of storing and using customer information
* Discusses privacy as an essential business strategy
Today, every information manager and technologist faces the challenge and added responsibility of safeguarding the corporation's greatest asset: customer trust.
Technology has advanced to a state where collection, enhancement, and aggregation of data are instantaneous. Corporations now have the technology to analyze the finest details about each customer. They can determine the most profitable clients and tailor their marketing messages accordingly. Information can be collaborated upon across the enterprise so the customer hears a single voice.
While this ability is a positive development for the corporation and means better services for the client, it also adds a level of anxiety if the aggregation is not performed correctly and appropriately. As personalization and data collection technologies become more advanced, the expectations of consumers are shifting rapidly. Customers' patience levels are getting shorter and shorter. Mistakes are not quickly forgiven.
Privacy and customer permission have become the cornerstone to customer trust. Most importantly, trust has become the cornerstone to a continuing relationship. A recent IMG Strategies study reported that "having basic permission and privacy policies (e.g., an opt-in policy) is becoming just the price of entry for marketers as opposed to the differentiator it used to be a year earlier."
Enter CRM
Companies want to "know" their customers; customer relationship management (CRM) is the key to that knowledge. IDC Research estimated that the worldwide market for CRM applications would more than double between 2000 and 2005, from $6.23 billion to $14.04 billion. CRM, however, is more than technology, software, and hardware. The real essence of CRM is in the acronym itself: customer relationship management--the relationship between the customer and those who manage the dialogue with the customer.
The database has become the strategic enabler for developing and maintaining this relationship. The role of database management as the vehicle for customer interaction has become a central strategic issue that reaches far beyond technology. Establishing and maintaining customer trust must be consistent across all customers' touch points. Each piece of information made available at the various interactions with the customer must be made available throughout the enterprise.
The capability and role of the database in marketing has drastically changed in the last decade. In times past, organizations maintained numerous databases. Customer information was stored in accounting, customer service, service, shipping, sales, and marketing databases. Each of these contained information important to that department's function. Unfortunately, it was rare when even the account number was consistent across the various departments. To add to the complexity, the formats were not standardized, and access was limited. The customer was seen as fragmented pieces of information. It also was commonplace for customers to receive multiple pieces of correspondence about the same topic and to have to present information about their accounts each time they contacted the company.
The information age brought with it technology and innovation that enabled a blending of these diverse databases that provides companies a more complete picture of the customer.
Personalization has emerged as one way to send the right message to the right person at the right time. Personalization brings with it the question of just how much information is too much. Corporations face the challenge of making appropriate information available at all customer touch points while protecting privacy at the same time. As a safeguard, sensitizing and training must occur at all levels of the organization. A company could have highly publicized privacy policies, but it only takes one bad interaction with a customer service representative to lose a customer.
Although personalization and privacy seem to be in conflict, the bottom line is that personalization benefits all involved: company, customer, and supplier. It gives the company a way to serve the customer better. Better service means better customer retention. Commonly, a five percent increase in customer retention can translate into a 25- to 125-percent increase in company profitability in the business-to-business marketplace, according to Fred Reichheld's book, The Loyalty Effect.
The customer gets products and services tailored to his or her preference. No longer do consumers have to provide information over and over to the companies they do business with. And suppliers can control their inventories and product development cycles.
Author Bruce Kasanoff sums up the seeming inconsistency between service and privacy: "It is a strange time. Privacy advocates are up in arms, but most people still are more upset over what companies forget about them than what they remember."
There is no lack of creativity in the application of technology to customer services. Some of the most significant advances in CRM can be found in the travel industry. Airlines, for example, closely track passenger history and preferences to better serve customers. One airline, for example, has begun using flight information to have travelers' cars ready at valet parking when they return from a trip. Changes in flight information can be sent via e-mail to a personal digital assistant (PDA) or cellular phone. In the future, flight delays can be reported directly to a destination hotel.
Privacy and Business Strategy
Privacy has become a top-down business strategy. At executive levels, companies are introducing the new position of chief privacy officer (CPO). (Editor's Note: Also see article by Pemberton, page 57.) CPOs serve as a liaison between the corporation and consumer. They often have veto power over product launches, marketing campaigns or strategic partnerships that they feel would interfere with the privacy of customers, employees, or suppliers.
Due to the never-ending media coverage of the legal aspect of consumer privacy, people are more aware of the types of information being collected about them. Most people, however, do not mind revealing personal information if it is used to better serve their needs and makes their interaction with a given company more convenient. A recent study conducted by Ipsos-Reid, a research and polling firm, reported that 47 percent of adults aged 18 to 34 have visited health-related Web sites and provided personal information, and 42 percent of those 35 to 54 years old have done so. Several of the most popular information-gathering and distribution sites on the Web are actually those of state governments. The sites contain information on everything from local government rules and regulations to birth, death, adoption, and marriage records.
Use with Caution
Employees and employers must become the custodians of customer trust and protect the privacy of their customers. They must learn to think beyond the proverbial box about the implications of the process of collecting and using information. Some organizations, however, are still learning how to apply CRM techniques appropriately as well as effectively. A dental group, for example, decided to apply personalization and database marketing techniques to send reminder postcards to current clients. Each postcard was properly personalized with the client information, including the amount of money toward dental care that they still had available from their insurance provider. Although technology was applied effectively, recipients were not pleased to see their personal information printed on postcards that could be read by postal carriers and anyone else handling the mail.
The customer relationship is built on more than the pieces of data and collected history. Merely collecting data and storing it in data warehouses can be a tremendous waste of corporate resources. Personalization and database marketing--conducted correctly--serve both the organizations and the customers.
Business cultures must grasp the concept that every single point of contact within the organization and throughout the supply chain is a key to the success of customer relationships. Technology is merely the enabler. The application of the technology--based on understanding the impact each and every dialogue has on the total customer relationship--is quickly becoming the highest priority within corporations. Businesses no longer focus on product share alone. The shift has moved toward customer share. This trend shows no sign of slowing down.
Kasanoff, Bruce. Making it Personal: How to Profit from Personalization Without Invading Privacy. Cambridge, MA: Perseus Books, 2001.
Reichheld, Fred. The Loyalty Effect. Cambridge, MA: Harvard Business School Press, 1996.
Debbie A. Cannon is President of DAC Enterprises, a firm that specializes in database management as well as one-to-one marketing. She may be reached at email@example.com.
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A young woman falls in love with and marries a handsome young man. Her twin sister sets out to break up the marriage and frames her sister's husband to make it look like he is an infamous bandit who has been plaguing the area.
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Assemblyman Smyth joins Sheriff's Department in event for kids.
Friday, October 30, will be all about keeping kids safe, as Assemblyman Cameron Smyth and the Los Angeles County Sheriff's Department join together for a child safety event.
From 3:00 p.m. to 6:00 p.m. kids from all across the Santa Clarita Valley are encouraged to stop by the Westfield Valencia Town Center near the Atrium for a free fingerprinting. Safety demonstrations will be put on by local deputies and patrol cars will be available for kids to tour.
Assemblyman Smyth will be on hand throughout the event helping to give out child safety kits. These kits have space for a child's fingerprints, a photo, and some critical information. Parents are directed to take the kits home, add a recent picture of their child, fill out the information, and store the kit in a safe place.
KHTS AM-1220 will also be conducting live broadcasts from the Community Resource Station, located at the entrance to the Atrium, near Forever 21.
The event aims to provide an opportunity for parents to prepare their children to be safe, especially right before Halloween.


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- Our Village
- Events
- Good Food
- Sleep Over
- Recipes
- Buy Online
Take home a piece of Lancaster County!
Rich trendy fabrics sewn into purses and bags...locally mixed clays hand thrown into gorgeous and practical pottery...wood and stone carved into made-to-order signs and key chains...artwork painted by local artists...quilts lovingly hand stitched by local quilters...
The variety of authentic Locally Made items you'll find right here in our Village is almost limitless!
Diane, of Cloverfields, makes all her fabric bags
Many of the shopkeepers in our 40 specialty shops make their items right here before your eyes. Or they purchase them from someone who makes them close by in Lancaster County.
Steve hand carves wood signs at Village Signcrafters
Made-from-scratch cookies from the Bake Shop
And then there's the food!
The heart of our Village is the Jam & Relish Kitchen where we started making jams and jellies in 1954. We're still cannin' and jammin' today in our kitchen - right before your eyes. And you can sample our close to 90 varieties every day for free!
You'll also find Lancaster County meats and cheeses, hand paddled fudge made right here every day, hand dipped chocolate treats, freshly popped kettle corn, luscious ice cream made on the Lapp Valley family farm just down the road, hand squeezed lemonade, and more taste-tempting goodies throughout the Village. Deb hand paddles fudge
Look for the Locally Made logo,
click here for a listing of Locally Made gifts,
and get ready to experience Lancaster County at Kitchen Kettle Village.
You're sure to find a special treasure to take home!
Barrick Design candles at By Candle Light Hand-carved ornamental eggs from Grande Place


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Jay shares a few things and people that he is very thankful for, this year Read more.
Jay shares a few things and people that he is very thankful for, this year Read more.
Bill Apter reminisces … Read more.
Apter knew the man and has a very unusual theory … Read more.
Taped on July 11, 2013 Bill Apter with Sheamus @The Apter Chat! Read more.
Jay Shannon spends a part of his birthday sharing a few of the people, places and things that have made wrestling special to him for all these years. Read more.
This video was shot in late 2011 and was the video i sent to Bill Apter to get Big Ray's Wrestling Show on 1wrestling.com. This is my opinion…What your top 5? Read more.
Big Ray chats with one of Youtube's / The Internet Wrestling communities most interesting personalities, Dameon Nelson of the Pro Wrestling Report. Sit back and enjoy this fun and passionate talk about Pro Wrestling of today and yesterday! Read more.


text: Whether or not the Boston Bruins win the 2013 Stanley Cup, this organization should be considered among the elite franchises in the NHL. With president Cam Neely, general manager Peter Chiarelli, coach Claude Julien and most importantly, the players, the Bruins have become a perennial championship-caliber hockey club. The 2011 Stanley Cup title was no fluke. This team is built on character and knowing how to play Boston Bruins hockey.


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Although some fear its effects, the option is likely to continue growing in popularity, especially in California
One borrower's story
Marisabel Garcia of Oak Park has supported two kids and a mortgage on one income following a divorce. Her financial worries worsened after a series of home repairs surfaced: a broken front door, a faulty heater and electrical wiring that was acting up.
After hearing about the Keep Your Home California program on Spanish radio, Garcia, 47, immediately applied to improve her chances of keeping her home.
After a thorough five-month process that involved reams of paperwork, she emerged with a permanent mortgage reduction that is saving her $600 a month.
In Garcia's case, her principal reduction was paired with a loan extension to 45 years, up from the original 30-year fixed-rate loan she had before.
"The loan modification, it was perfect," said Garcia, who plans to live in her home long-term. "I didn't notice the (loan extension) until later … I will worry about that later, I guess … I don't like that it prolongs the terms."
As part of the program, a lien is placed on the home and is forgiven in five years if the borrower is in good standing. The amount that is forgiven could be taxable, but that depends on the borrowers' situation, state officials said.
To reduce, or not reduce?
San Diego County's largest lenders, Bank of America, Wells Fargo and Chase, say they have forgiven more than $6 billion in mortgage principal and plan to do more. It appears lenders still have some ways to go, factoring in their obligations to their 2012 settlement with 49 states and the federal government, and the fact that $7 trillion of home equity in the nation has been lost between 2005 and 2007. The last estimate from DataQuick showed that about one-third of homeowners in San Diego County owed more on their mortgages than their homes are worth. The reality of lost equity continues to push borrowers toward strategic defaults, in which homeowners decide to stop paying the mortgage.
"Principal reduction is a life ring to underwater homeowners from drowning in debt," said Murtaza Baxamusa, who directs planning and development for the Family Housing Corporation, of the San Diego Building Trades in Mission Valley. "With a third of San Diego mortgages underwater, the attorneys general settlement will directly benefit our region."
Others like Kurt Branstetter, loan officer and mortgage manager at W.J. Bradley Mortgage in San Diego, say principal cuts are not the answer.
"There is a moral hazard with selective principal reductions that cannot be overcome," he said. "Bank of America requiring homeowners to be 60 days late on their payment to qualify will result in the worst possible outcome and most likely be the straw that breaks the camel's back for the millions of homeowners who have honored their commitment by making their payments."
Regardless of your position, more principal reductions are expected to happen in the nation, especially in the hard-hit state of California.
Keep Your Home California, the state program, no longer requires servicers to match program money dollar-for-dollar in order for a principal reduction to happen, a change that program officials hope will lure more servicers to the table.


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About Schools in Kempton, IL
Kempton offers a range of educational opportunities with one elementary school.
Contact a Coldwell Banker® real estate agent to learn more about the schools and school districts in Kempton, IL.FIND AN AGENT
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i can see guida winning this by decision, if he does he deserves a shot at the winner of bj vs florian
The Ultimate Fighter season 1 winner Diego "The Nightmare" Sanchez (22-2) has agreed to fight Clay Guida (25-9) in the main event at the TUF 9 Finale on June 20th at "The Pearl" inside the Palms Resort in Las Vegas, Nevada, according to Sherdog.
Sanchez is coming off his first win as a lightweight, defeating Joe "Daddy" Stevenson by unanimous decision at UFC 95.
Guida, who's on a 3 fight wining streak, scored a close split decision win over Nate Diaz at UFC 94.
Both fighters are intense fast pace fighters, with good wrestling skills.
For more on the Ultimate Fighter 9 finale, check out our UFC Rumors section.
i can see guida winning this by decision, if he does he deserves a shot at the winner of bj vs florian
I woulda' preferred to See Diego fight Nick Diaz… See Nick get some payback for his Big Bro!
I think Sanchez will win this, i also think Sanchez is gonna tear ass in the lightweight division.
Sanchez vs. Guida will be a solid fight to look forward to.
i would prefer a different match up, but clay has been winning and he wants to step up in comp. this should be diego's fight no problem but you never know with clay.if clay did win i would say that really moves him up in the ranks.
maybe he will repeat the stupidest entrance in mma history. Please win guida, even though I don't think you have a prayer.
haha, right on steveo, he looked like a complete retard eh
Nightmare will walk throw clay..I use to love clay but after his lAST fight with Nate(which in my eyes Nate won that with easy) all he did in that fight was hold on to Nate so he coudnt Get sub, he didnt even throw punchs or anything..When nate was on the ground Nate was looking for subs the hole time and Clay didnt even throw punchs he just holded on..when they were standing clay would just slip punch then hold Nate then Nate would go for a standing Kermora and then Clay would scrable and then just sit in Nates gaurd..You could even hear clays corner men saying its not just a wrestling match throw some punches…
Nightmare will has just as good wrestling and The nightmare is better on the feet..
hopefully sanches beats the shit out of clay "hug me gentley" Guida
yeah i feel the same way as tom i used to like clay but i can't stand hugging your way to victory.
I agree Diego has showed more talent and ability recently while Guida has just layed on top of people huggning them but i still think the guy is intesting.
im going to take Clay in a split decision …it will be a decent fight i think
I see Sanchez winning by decision, possibly by knockout. Diego is gonna keep it standing. Not to take anything away from Nate, but Diego has better take down defense. I still think that Guida won the fight against Nate. Even though he hugged him to victory, Nate should have just defended the take down. He knew it was coming but allowed it anyway so Nate lost. Diego however will defend the take down successfully and force Clay to stand and eat punches
I think Sanchez could win anywhere the fight goes, striking or on the ground. If Sanchez's power returns like when he fought at 170, it will could be a shot night for Clay


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- I suppose we'll be seeing Simon Gagne in place of Dustin Penner in Edmonton.
- Jeff Carter made several weak passes. Justin Williams, at least one.
- Jake Muzzin looked pretty good. There was one shaky shift.
- Once again, the fourth line was best.
- Kyle Clifford, Colin Fraser, Jordan Nolan — all +1.
- Clifford, with a goal in the game and two assists in the opener, leads the team in points.
- Nolan is second in points, with 2.
- Scuderi, Martinez and Greene each have 1 point. Everyone else has 0.
- Trevor Lewis once again led the team with 4 shots, and made a great save, too.
- NHL Network just accidentally called Quick "Johnny Glove."
- The big guns were a non-factor.
- Dwight King, Jarret Stoll and Lewis were all even in plus/minus.
- Drew Doughty and Rob Scuderi were -1. The rest of the D were even.
- Doughty missed his assignment on the second rebound-off-the-boards goal.
- Kopitar failed to clear the puck, leading to a goal.
- Penner coughed it up inside the blue line, leading to another.
- Quick was better than in game 1.
- But his numbers still suck. 4.10 GAA, .849 SV%.
- Over two games, Brown and Richards are a team low -4.
- The fourth line is a team high +4.
- Kopitar, Gagne and Stoll have a combined 1 shot.
- Kings are 0 for 10 (I think it's 10, it's definitely 0) on the power play.
- PK was good though.
- I wonder if Sutter will scratch both Penner and Carter in favor of Richardson and Gagne. I can picture it. Not sure I would do it. But I can picture it. (I might do it.)
- I think I would also return Bodnarchuk and recall Tyler Toffoli. Then let him play for whichever millionaire winger isn't getting it done. Keep them honest.
- Kings lost their first two games back in '08, en route to a 5th overall draft pick. They haven't lost the first three, going back at least as far as '88.
- Rich Clune got in a fight in the NAS/MIN game. Glad to see he's playing.
- NHL Network just said "Davis Drewsky."
- Yeah, I'd also play Jonathan Bernier in the next game.
3 comments for "COL 3, LAK 1 — Post-Game Bullets (game 2)"
I'm kinda seeing the love/hate relationship the fans in Philadelphia had with Jeff Carter.
In fairness, the D is not what it might be. Penner always sucks early season…as far back as the Ducks. I just can't see Gagne playing or how he would make a difference…I like him but never saw him as a good fit for LA's style.
Toffoli? Will take Quisp's word for it. An idea but its still early and the Kings still have a bit of adjustment ahead with the absence of Greene and Mitchell.
Carter wasn't too bad in game 1, same seemed true for Penner (but please, no more of the blue line stuff…saw too much of it last year).
Season picked up where it left off…one mistake and its in the net.
After being one of the Kings' best forwards in the first game, Penner sure sucked tonight. I would also expect him to be scratched Thursday. I'd put Gagne with Kopi and Brown and move Williams to a line with Richards and Carter. So far the top 6 has been terrible so maybe a minor shakeup like that will help.


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Dentures Center - Rosemead, CA
WebMD Physician Directory
Rosemead, California
What are dentures?
When a person is missing teeth, he or she may experience a variety of problems. The person may become less confident in their smile and have difficulty speaking and eating certain foods. Dentures are appliances that ar...
Other Related Dentures Articles
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Printable View
Romulus: That agent of anarchy, Kayden James, had better keep tight vigilance of his surroundings lest he know the Wrath of Remus.
What do you think?
IC: Romulus wasn't able to get his usual brazilian wax due to time constraints with the EWNCW schedule so... it's a bit of a situation for him.


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How to get a Free iPhone 6
I am going to explain to you how you can get a sim Free iPhone 6 with no contract for nothing at all! If you don't have faith in my words why not check my 'Proof' page where I have some personal pictures of the Free Gifts I have received.
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One spy's view of '08-09 regular season
Updated: April 10, 2009, 5:35 PM ETBy Damien Cox | Special to ESPN.com
We stumbled across one league spy's "notes" from the 2008-09 regular season. The spy went under the name "twitterHockeyMole," but the spy's "bio" wasn't readily available. We still think it's legit. You be the judge.
Made it in. Have infiltrated the NHL's NY offices. Nobody knows; spying for Paul Kelly. Wonder if Ted Saskin can read my e-mail? 10:22 a.m., Sept. 15 Sent from twitterHockeyMole
RT @JohnDavidson: What did they teach Erik Johnson at the NDTP? You'd think getting ...
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Metal Mafia Steampunk Inspired Gun Belly Bar
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Double duty for Huey at Flushing Meadows
Filipino Treat Huey and British partner Dominic Inglot, seeded 16th in the men's doubles, get their US Open bid going against an Austrian pair in Flushing Meadows, New York.
Huey and Inglot, who reached the third round in 2011, will battle Julian Knowle and Jurgen Melzer in a first round match yet to be scheduled. Huey and Inglo also made it to the third round in this year's French Open and Wimbledon.
The winners of two titles last year, Huey and Inglot are looking for breakthrough performance in a Grand Slam event. They were runners-up in Indian Wells earlier this year and in the Salem Open which was a buildup for the US Open.
But Huey, the 27-year-old cornerstone of the Philippine team, will first see action late Wednesday in the mixed doubles competitions with American Raquel Kops-Jones.
Huey and Kops-Jones will take on Victoria Duval and Donald Young of the United States.
Ranked 32nd in the world in doubles, Huey will lead the PH Davis Cup team against New Zealand on Sept. 13 to 15 at Plantation Bay Spa and Resort in Lapu Lapu City. Marc Anthony Reyes
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Our lives are filled with them - inadequate nutrition, occasional sleeplessness, and daily stress leave us feeling zapped of energy.
More Than a Multivitamin!
Many traditional multivitamins can only supply a fraction of the nutrients you need to feel energized for an entire day. Vitality could be as easy as choosing the right solution — one that packs a powerful punch for nutrition AND energy.*
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‡Typical Amino Acid Content per serving, including Amounts for Whey Protein: L-Alanine 300 mg, L-Arginine 145 mg, L-Aspartic Acid 665 mg, L-Cystine 65 mg, L-Glutamic Acid 1,090 mg, Glycine 500 mg, L-Histidine 105 mg, L-Isoleucine 350 mg, L-Leucine 630 mg, L-Lysine 550 mg, L-Methionine 125 mg, L-Phenylalanine 180 mg, L-Proline 385 mg, L-Serine 540 mg, Taurine 500 mg, L-Threonine 415 mg, L-Tryptophan 115 mg, L-Tyrosine 525 mg, L-Valine 340 mg, Total 7,525 mg
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† Based on 2000 calorie diet.
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The whey protein concentrate provides a minimal amount of dairy products, namely, lactose and casein. It contains a maximum of 7% lactose and 15% casein. This calculates to a maximum of 0.6 grams of lactose and approximately 0.9 grams of casein per serving of Fatigued to Fantastic! Energy Revitalization System. For comparison purposes, there are 10 to 16 grams of lactose in an 8-oz glass of milk.
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Can Fatigued to Fantastic! Energy Revitalization System be used as a daily multivitamin?
†† For homeopathic products: these indications are based solely on traditional homeopathic use. They have not been evaluated by the Food & Drug Administration.
* For dietary supplements: this statement has not been evaluated by the Food & Drug Administration. This product is not intended to diagnose, treat, cure or prevent any disease.


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It has symptoms similar to a heart attack but the main cause is sudden shock or stress.
The broken heart is a serious topic not just in novels and films, but also in the not-so-romantic medical field of Cardiology. The "symptoms" are sudden acute pain in the chest, in the neck and left arm, accompanied by breathlessness, nausea and/or vomiting; similar to the symptoms of acute myocardial infarction or Heart Attack. At this point, it ceases to be romantically exciting and the situation demands a totally different type of attention.
A typical case involves a female patient around 50+ years, who is brought to the Emergency Room of a hospital. The ECG and ultrasound scan show that the left ventricle is practically non-functional. But there is no block in the coronary arteries and no stoppage/reduction of circulation to the heart muscles. No valve lesions and no circulatory interruption, but the muscles of the left ventricle ceased to function as though paralysed. Why? The heart is "broken"! This patient reacted to the sudden break up of her marriage.
Takotsubo Cardiomyopathy is the medical term for the condition described above. Since the beginning of the 1990s, it is also known as "Broken Heart Syndrome". It is the shape created by the "apical ballooning" or the bulging out of the apex of the heart with preserved function of the base that earned the syndrome its name "tako tsubo" or octopus-trap in Japan, where it was first described. The cause for this heart-attack like illness is usually acute stress or an extreme shock, which could be negative as in the case above or positive like a lottery win.
It has been observed that women's hearts "break" more often than those of men. The New England Journal of Medicine reported the study of a series of such cases and came to the conclusion that more than 90 per cent of the patients were women between the ages of 50 and 70 years. Some cardiologists believe that the reduction in oestrogen levels in women after menopause could lead to the activation of the autonomous nervous system. This could result in the production of too much stress-hormone, which in turn may have a negative effect on the heart muscles. There are nonspecific changes in the electrocardiogram and some elevation of cardiac enzymes (Troponin and Creatine kinaso) in blood.
The patient needs to be kept under close observation, usually in the Intensive Care Unit, for any further sign of shock-symptoms. What is critical is that the attending physician recognises the symptoms of the "fake-attack" and does not start the patient on medication meant for heart attack. This could lead to deleterious consequences.
The patient is advised bed-rest in quiet, calm surroundings away from excitement and stress. The dysfunction of the left-heart muscles disappear normally within days or weeks. Unlike in the case of a real heart-attack, no permanent damage is left over on the heart muscle. Most patients survive the initial acute presentation of the so-called Broken Heart Syndrome, with very low rate of mortality or complication. Direct preventive measures are difficult because shock and stress are not easily preventable.
Keywords: broken heart syndrome, medical condition, stress


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Product: Mobile > Cell Phones > AT&T Cell Phones > SGH-I857
AT&T Samsung DoubleTime™
Frequency Spectrum
2402.0 - 2480.0 MHz
This profile defines how high quality audio (stereo or mono) can be streamed from one device to another over a Bluetooth connection. For example, music can be streamed from a mobile phone, to a wireless headset, hearing aid & cochlear implant streamer, or car audio or from a laptop/desktop to a wireless headset.
Currently in version 1.5, this is commonly used to allow car hands-free kits to communicate with mobile phones in the car. It uses SCO (Synchronous Connection-Oriented) to carry a monaural audio channel with continuously variable slope delta modulation or pulse-code modulation, and with logarithmic a-law or µ-law quantization.
Most bluetooth headsets implement both Hands-Free Profile and Headset Profile (HSP), because of the extra features in HFP for use with a mobile phone, such as last number redial, call waiting and voice dialing.
This profile is designed to provide a standard interface to control TVs, Hi-fi equipment, etc. to allow a single remote control (or other device) to control all of the A/V equipment to which a user has access. It may be used in concert with A2DP or VDP.
A basic profile for sending "objects" such as pictures, virtual business cards, or appointment details. It is called push because the transfers are always instigated by the sender (client), not the receiver (server).
OPP uses the APIs of OBEX profile and the OBEX operations which are used in OPP are connect, disconnect, put, get and abort. By using these API the OPP layer will reside over OBEX and hence follow the specifications of the Bluetooth stack.
Phone Book Access (PBA) or Phone Book Access Profile (PBAP) is a profile that allows exchange of Phone Book Objects between devices. It is likely to be used between a car kit and a mobile phone to:


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Sign Up ForFREE Pattern Info!
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Piece Code: PPRB
Piece Name: Silverplate Paul Revere Bowl
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Piece Code: PPRB6
Piece Name: 6" Paul Revere Bowl
Size: 6 in
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