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Five golfers commit to Pfeiffer
May 19, 2009
Misenheimer, NC Pfeiffer Universitys Director of Golf Phil Fecteau announces five players have committed to competing for the Falcons next year. Pfeiffer adds one woman and four men to its roster.
Chad Nicholson makes his way to Pfeiffer from Port Carling, Ontario, Canada. Nicholson was the Bracebridge and Muskoka Lakes Secondary School boys golf teams captain in 2008 and 2009. In 2008 he was the recipient of the Scott Self Award and was the Junior Club Champion at the South Muskoka Curling and Golf Club. Nicholson was a CJGA Team East Canada Team member in 2007 and took the title at the Juvenile Pepsi Tour. Nicholson won the title of MVP and Most Dedicated on his schools hockey team for three seasons. He was named to the Academic Awards Honor Roll and is a member of the Student Parliament.
Like Nicholson, Tyson Louth will also be coming to Pfeiffer from Canada. The native of Washago, Ontario, Canada is a four-time Orillia District High School golf MVP. He was also the MVP of the 2006 Regional basketball team and three-time Heart and Hustle award winner for the team. Louth was also MVP of the Orillia hockey and lacrosse teams. He is an honors student and volunteers at his home course to organize and teach junior golfers.
JD Lamm, a native of Port Charlotte, Florida, has been named All-County for the past four years and also earned the county championship title twice. In 2008 Lamm won the Charlotte County Junior Championship. He also won two Greater Sarasota Junior Golf Association Tournaments.

subset: commoncrawl


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This vivid mile-by-mile account takes you on the roads the soldiers used as Robert E. Lee led his Army of Northern Virginia on its last march from Petersburg to Appomattox, April 1865. Eleven detailed road maps and nine battle diagrams by Steve Stanley. Numerous modern and period photographs and contemporary line drawings. By Chris Calkins, 84 pages. Paperback.
Since 1982, the renowned Civil War historian James I.
The definitive travel reference for America's most famous, and hallowed, Civil War battle sites. Contains thorough listings of all major sites, including historical background information, full-color photographs throughout, special features on military and civic leaders, a glossary of Civil War terminology, directions to hard-to-find locations, and helpful listings of restuarants, lodgings, shopping, tours, and special events. Hardcover, 444 pages.
The 18 tours in this book cover all the significant Civil War sites in both Virginia and West Virginia. While the better known sites are covered (Fredericksburg, Spotsylvania, Wilderness, Chancellorsville, Richmond, Petersburg, both Bull Run battles, Appomattox, etc), Johnson also covers lesser-known events (Saltville, Mine Run, Chantilly, etc). By Clint Johnson. Paperback, 407 pages.
Hollywood Cemetary: A Tour, by James E. DuPriest, Jr. History, art and landscape architecture all come together in the magnificent setting of Hollywood Cemetery, perched on the bluffs high above the James River in Richmond, Virginia. Nowhere in America will one find so many prominent 19th Century personalities as those that rest at Hollywood. (28 pages, 8.5 x 6.75, Paperback)
In the Footsteps of Robert E. Lee, by Clint Johnson. This is your guide to the major Lee sites in Virginia and West Virginia and lesser known sites in distant states. Taken together, they will let you - as far as possible - stand where General Lee stood and see what he saw. (186 pages, 8 x 5.5, Paperback)
Appomattox County, by Patrick A. Schroeder and Scott Frantel. Appomattox County, formed in 1845 and named after the nearby river, was originally best known for growing tobacco. However, that dramatically changed in 1865 when Gen. Robert E. Lee surrendered to Gen. Ulysses S. Grant at the McLean House. In the 1930s, efforts began to commemorate Civil War events, and a national park was created. Boasting a unique history abundant with churches, notable citizens, and special events, this photograph collection shows the diverse and memorable history of Appomattox.
An essential to-do list for the Civil War. Inside this book, pictures, places, objects, and stories of the Civil War weave a hard-won lesson of loss and triumph on a continental scale. Ranging across nearly two dozen states, former territories, and the District of Columbia, this simple-to-use guidebook gives concise descriptions and key images for each entry plus clear directions on where to look or how to get there. Paperback, 261 pages.
Contact us | Feedback | Site Map
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Museum of the Confederacy and nearby Richmond hotels
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Class Notes
Class Notes | Obituaries
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Showing class notes submitted in the last year by graduates in 1952
Leigh Josephson '52 is living his golden years on the island of Kauai, after teaching for many years at Iolani School in Honolulu. He has been married for over 60 years and raised his children in Honolulu as well. After being inducted into the Hawaiian Aquatic Hall of Fame, he is looking forward to competing in an annual open water swim in Hanalei Bay.


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In this assignment, you will develop a ROS package for reactive robot soccer control using the Subsumption architecture. You will evaluate your client through (graded) basic skills challenges such as navigating to specific locations and (ungraded) competition against other groups.
Videos of Lisa Miller's subsumption implementation from Spring Semester 2009:
Assigned: Sunday, October 24, 2010
Inter-group soccer competition: Friday, November 5, 2010
Project reports due: Sunday, November 7, 2010 (5:00 pm)
Your previous soccer-playing robots required some notion of state, such as robot pose, a map, ball location, etc., in order to function. State information, stored explicitly in variables, gave your controller a sense of context and immediate history about the robot's current situation. Robot decision making at any moment in time is informed by state information in order to deliberate and plan into the future or take immediate action towards some future benefit. State variables allow our robots to adapt in the face of unexpected changes in the environment and new scenarios <ref>Learning aims to provide a greater level of adaptability such that the robot has long-term context and history towards learning from experience. It could roughly be thought of as never making the same mistake twice.</ref>. However, estimation of such variables comes at the cost of requiring greater computation, latency in decision making, and overall price tag for the robot.
Consider the case where you want to commercialize a soccer playing robot for a mass market <ref>Because we are in Rhode Island, let's say you are making a robot for Hasbro.</ref>. Such a robot should not cost too much money to purchase, on the order of the $150 Roomba Red or $200 Nintendo Wii. Further, every additional component added to the robot, such as memory, processing, sensing, and actuators, increasingly cuts into your profit margin. In such scenarios, reliance upon state variables costs you money<ref>"Money" in this case can be generalized to "cost" that limits practical implementation. So, even if you are not fiscally motivated, such costs limit your ability realizing your technical designs.</ref>, thus you might be inclined to minimize the number of these variables.
Using reactive control, your robot controller in this assignment will rely upon no state variables and simply react to what is sensed at the current instance of time. In contrast to the deliberate intentionality of planning, reactive approaches rely upon emergent behavior where the "intelligence" of the robot results from its decision making, the properties of the environment, and the dynamics of controller-environment interaction. The Subsumption Architecture is one approach to reactive control that uses multiple robot controllers with a priority scheme. In particular, each robot controller is capable of independently producing some robot behavior (such as wandering or obstacle avoidance) given certain applicability conditions (or preconditions) are met (such as whether an obstacle is present). When their preconditions are met, higher priority controllers "subsume" lower priority controllers, whose decisions are then ignored.
You will implement a robot soccer controller that uses only the robot's on-board immediate sensing and perception. Specifically, you are only allowed to use sensing information provided in the current time instant by the robot's bumper, infrared (IR), blobfinding, and AR tag recognition. Timers, odometry, and variables that require history are not allowed in your controller. Timing-based operations in the Create Open Interface also cannot be used by your client. With this information, you are tasked with implementing a Subsumption architecture. Towards this end, you must:
Your robot will need to stay within the general field of play as given by the physical walls of the Roomba Lab and virtual walls placed around the pitch. Additionally, your robot should make reasonable attempts to avoid collisions with other robots, which will be marked with 14.5 x 10 x 10 cm pink rectangular fiducials placed on top of the robot. We will definitely call player pushing penalties for this assignment!
The rear of goals on the FC 148 field will be marked with some combination of solid color rectangles (either green or orange) and AR tags.
Virtual walls (pictured to the right) are devices that emit infrared (IR) light that can be received by the iRobot Create and Roomba platforms. The distribution of space covered by the emitted IR forms a beam that can extend up to 8 feet. These beams form an invisible barrier around the field that your robot should respect. Your control client will ave access to the Creates's IR sensing through the IR proxy. Note that the width of the virtual wall's beam also increases as its length increases, creating a cone-shaped area from the IR wall. It is best to set the range slider to the middle option: 4-7 feet. However, if you find the width of the beam emitted from the IR wall is interfering with your robot too much, the 0-3 feet range may be more appropriate.
You will be asked to demonstrate (either through a demo or in your report) that each control behavior functions independently and properly. In addition, your independent behaviors will need to function together (without code modification) using a subsumption priority for coordination. You will also be asked to randomly (based on the opinion of the course staff) reprioritize your behaviors to evaluate emergent behavior properties.
The goal scoring challenge and inter-group soccer competition from the Path Planning project will be used for this competition. The navigation challenge will have the same format as Assignment 3 except that obstacles will be pink fiducials and the field boundaries will be enforced by the physical and virtual walls. Both the Goal Scoring and Collision-free Navigation tasks must be completed within 120 seconds.
Your group should create a subsumption package that includes a node (soccer_subsumption) that subscribes to your robot's sensing from the Create base (via irobot_create_2_1), cmvision blobfinder, and ar_recog tag recognition and outputs cmd_vel topics for moving the Create base. You are allowed to use any information provided by these subscriptions. However, absolutely no information or variables can be stored and maintained in your controller, it can only react to the current sensing information. soccer_subsumption should use a ros_param to specify subsumption order.
Your group is free to use additional nodes for performing subsumption and/or having controllers specific to each challenge.
You are expected to conduct least 3 trials with 4 different initial conditions for both your subsumption_soccer node (24 trials total). For each trial, measure the properties mentioned for each challenge. All of your trials must use the same controller without modification.
Document your controller and experimental results in a written report based on the structure described in the course missive. You are welcome to experiment with additional techniques and evaluate the relative performance of each. When completed, your report should be committed to the object_seeking/docs/username directory of your repository.
Your grade for this assignment will be determined by equal weighting of your group's implementation (50%) and your individual written report (50%). The weighted breakdown of grading factors for this assignment are as follows:
Note: Demonstrations of your challenges should occur by scheduling an appointment with a TA. Soccer competitions will be held during class and the preceding hour (12-2pm).


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I've recently realized that not only do I have a large taste for the sense pleasures of samsara, that I honestly have little interest in actually relinquishing any of my drive and craving towards them. I'm rather fond of them it would appear... I'm in the process of trying to turn the mind towards the dharma and reflecting on the meaninglessness of Samsaric activities and the greatness of liberation. I should likely reflect on cause and effect to and the unbearable nature of the lower realms too. However, I feel as if I'm under a spell. I understand that not only do I have a truly golden opportunity of, after having found the dharma, being able to practice it under the instruction of one of the few great masters left however, i watch myself knowingly fall for the same trap every time. I don't really think that anyone could be so thick not to notice the pitfalls associated with the sense pleasures, particularly sexual ones. To be frank, they're much more appealing than meditation and practice. The mind is so gullible to believe that "oh maybe this time they won't deceive me!"
How could one like this ever end up interested in the dharma I ask myself. Perhaps some great past merit and virtue. I don't know. I do know that all I am truly interested in is a spiritual adventure. Even if I arrived at the feet of the most qualified teacher who wholeheartedly would devote himself to my own awakening I'm afraid I would likely be packing my bags (at least mentally) and planning a trip to whereever sounded most exotic and untouched in hopes of learning the language, mating with the women, and bonding with their sages only to write a book about it in hopes of receiving the praise of my peers.
I am fine to take Buddhism, for example the six realms, with a grain of metaphorical salt but even that will not satiate my ceaseless taste for samsara and all its goodies.
This confession was written one lonely evening by a forum member solely for his own self-serving interests. May it not disturb other forum goers but remind them of the pitfalls waiting at the end of the trails which stray from the path.


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Base Camp Duffle - Large 90L
THE standard in tough holdalls, used by everyone who needs to transport large amounts of equipment. From Special Forces personnel to 8000m climbers, if you don't own one, you'll know someone who does.
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|Nice an big
|Reviewed By James, 08 Sep 2013
Big, waterproof and robust, really good bit of kit for any traviler!
|Fantastic strong and Reliable Holdall
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This bag is practically indestructible. Mine has been across the desert in Oman, dumped in bogs on Dartmoor, left on a runway in Dubai, carried around Australia, and it is still going strong. Just wish they did the funky colours when I brought mine (It would make spotting my bag on a luggage carousel a lot easier)! Buy this bag you won't reget it.
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imho,no comparable product, from other manufacturers,comes close to the durability,versatility,and near tank like construction,of these ledgendary duffles.mine have,over the years, been used an abused in military environments and the rigors of worldwide travel, and have always come up smileing.it laughs in the face, of mercyless airport baggage handellers,and is its naturall ellement,strapped to the side,or being stepped on by yaks,on long expeditionsi have sveral of these workhorse duffles, the latest being bought to replace ones "missing in action" :).bought at a gret price ,too boot :).latest ones purchased from cardiff city centre store.special thanks go to anthony and james, for facillitating my various orders, duncan for"communications", and of course to the ever reliable ,friendly, and helpfull catherine and bronwyn,for completeing the sales,and packing my numerous additional order items.ahighly reccomended piece of kit,and a professional, motivated, and enthusiastic group of people :)
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This is the bag you can drop out of a helicopter, then kick down a mountain, ski down, and pick it up at the end, intact. Lockable zips, comfortable carry straps, and can take it anywhere.


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Things To Do
BirdingThis region is known for many species of birds that make their home in Cache Valley or migrate through. Due to our unique geography you can see shorebirds and mountain species all within a 20-minute drive. Cache Valley is nestled between the Bear River and Bear Lake National Wildlife Refuges. Here, you'll spot birds along the Bear River, directly west of Logan. In spring and summer, Cutler Marsh offers viewing in classic wetland habitat. White Pelicans, White-Faced Ibis, Great Blue Herons, Egrets, Western Grebes, plus a variety of ducks, geese, and shorebirds may be seen from roadways or you can rent a canoe or kayak for a close-up experience. The number and diversity of marsh birds is spectacular. In early autumn, visit the north end of the Wellsville Mountain wilderness to watch the annual raptor migration. Northern harriers, kestrels, goshawks, red-tailed hawks, golden eagles, and other species float by, lifted by canyon updrafts that ease their relentless flight south.
A free birding brochure is available at Cache Valley Visitors Bureau, 199 North Main, Logan or can be downloaded by clicking Birding Trails.pdf
For more information on birding in Cache Valley, visit Bridgerland Audubon's website at www.bridgerlandaudubon.org
various locations, Cache Valley & Logan Canyon
Skiing and SnowshoeingNothing beats cross-country skiing and snowshoeing on a crisp winter day in the Wasatch-Cache National Forest or at Sherwood Hills. There are routes for all levels of experience. You're likely to see deer, elk, moose, snowshoe hare, cottontails, ermine, porcupines, and a variety of birds. Beginner areas are located in Sink Hollow, at the entrance of Beaver Mountain Ski Resort, and at Sunrise Campground on U.S. Hwy. 89, which overlooks Bear Lake and is near the Logan Canyon summit. A groomed track in Green Canyon offers excellent skiing just minutes from downtown Logan.
Logan Ranger District (435) 755-3620
SnowmobilingNorthern Utah is one of America's favorite snowmobile playgrounds. Experience more than 300 miles of groomed trails through spectacular scenery with terrain that's as challenging or easy as you want it to be. The Bridgerland snowmobile complex in the Wasatch-Cache National Forest includes Logan Canyon, Monte Cristo, and Hardware Ranch. Some trails extend into Idaho's adjacent Caribou National Forest. Snowmobile maps are available at the Cache Valley Visitors Bureau, 199 N. Main St. in Logan.
The snowmobile season begins in December and often extends into April or May. The average annual snowfall in Logan Canyon is 400+ inches.
For additional info and downloadable map:
Hardware Ranch Wildlife Management AreaA visit to Hardware Ranch Wildlife Management Area is both fun and educational. During winter months, a trip to the ranch makes a great outing for the whole family with the memorable experience of viewing up to 700 wild Rocky Mountain elk. A horse drawn sleigh gives you up close and personal views as it takes you right into the middle of the herd. The area is also an extremely popular snowmobiling area.
During the summer, Hardware Ranch area provides great opportunities for outdoor recreation and wildlife viewing. An extensive network of ATV trails will take you north all the way to Logan Canyon, Bear Lake, and southeast Idaho. Head south and explore the Monte Cristo area all the way to Ogden Valley.
15 miles east of Hyrum on U.S. Hwy 101 in Blacksmith Fork Canyon, Hyrum
(435) 753-6206
mid-December through mid-February
View Website


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Discussion in 'Religious Discussions' started by montezumaz, Nov 14, 2012.
Good. Very thought provoking.
Thanks for sharing.
Will pass it on.
The old saw I heard years ago: "God and I make a majority", so I'm not sure about just being one of the 7%.
Very nice video and I just became a 7 percenter.
Thanks for posting it.
Excellent video, thanks for posting it.
I love it, and sent it on to several friends.
Separate names with a comma.


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Rich Dad Education
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Rich Dad® Education's Free Workshop can help you put the lessons of Rich Dad Poor Dad into action in your life. Channel your desire for wealth into actual money-making investment opportunities... help you take that final step from saying "I can" to saying "I am"... give you the courage to succeed and the know-how to achieve it! Rich Dad® Education offers you, and those who have been inspired to become smarter about money and investing, a clear path to the next level through hands-on, detailed strategies and techniques proven effective in every area of the country and any market conditions. Rich Dad® Education was developed from the ground up to help you advance your financial goals through the greatest wealth creation tool of all time - real estate. With detailed, step-by-step action plans, we help you identify the right opportunities and educate you on how to capitalize on them. Money doesn't make people rich. Knowledge makes people rich. Everyone wants to be rich, but only a select few will ever TAKE ACTION and get the financial knowledge they need to make their dreams come true. You can be one of that select few!
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A: You are not required to purchase anything. As we stress, not only is the workshop free, but when you attend, you will also receive a FREE GIFT. We do offer additional educational products and services at our workshops, but you are never under any obligation to buy.
Q: Will there be food at the workshop?
A: Food will not be provided at the free workshop. As we cover a lot of ground, we will need your undivided attention.
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There is plenty of free parking at the four-storey building, set in landscaped gardens just five minutes' drive from the A14 junction 7. Our team will greet you in the open-plan lobby where you can surf the web via our free Wi-Fi. Residents can help themselves to our generous breakfast of delicious cereals, freshly baked pastries and fruit in the
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There is plenty of free parking at the four-storey building, set in landscaped gardens just five minutes' drive from the A14 junction 7. Our team will greet you in the open-plan lobby where you can surf the web via our free Wi-Fi. Residents can help themselves to our generous breakfast of delicious cereals, freshly baked pastries and fruit in the bright Great Room each morning.
Our 24-hour residents' bar provides a stylish spot to socialise with friends or colleagues over drinks, and you can head to the popular Harvester restaurant for dinner just across the car park or order in your favourite take away. Then make yourself at home in your air-conditioned guest room with a cosy duvet to help you get a good night's sleep.
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Hourly trains arrive and depart at Kettering train station to and from London which is just a short taxi ride away.
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10 Things to Know for Friday - Comments
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Luke Janes
September 16, 2013
Luke Janes was pivotal in Pfeiffer's 2-1 victory over North Georgia on Saturday afternoon. The junior erased a late 1-1 tie in the 88th minute against the Nighthawks as he took a direct kick from teammate Jaime Sanclemente and put a header in the back of the net for the win. It was Janes' second career goal and first ever game-winner.


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Religious traditions identify themselves with and draw sustenance from the texts that they hold sacred. In Sarah Lawrence College religion courses, these texts command and hold our attention. As students explore the sacred text of a particular religion, whether studying Buddhism, early Christianity, or the origins of Islam, they gain insight into the social and historical context of its creation. Using critical, hermeneutical, and intellectual historical approaches, they enter into the writings in such depth as to touch what might be the foundation of that religion. In addition, work with contemporary texts (such as those by religious activists on the Internet) gives students insight into what most moves and motivates religious groups today. The College's religion courses provide an important complement to courses in Asian studies and history.
Religion courses
- Chan and Zen Buddhism
- Christianity and the Roman Empire
- Classical Sufi Texts
- Contemporary Muslim Novels and Creative Nonfiction
- First-Year Studies: Islam
- Jewish Mysticism From Antiquity to the Present
- Pariah Lives: Modern Jewish Fiction and Autobiography
- The Buddhist Tradition in East Asia
- The Buddhist Tradition in India, Tibet, and Southeast Asia
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These were perfect to attach to the bubble favors to invite people to the reception!
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I hope everyone has been staying warm this week. I have to admit that when I left Washington after the House adjourned last week, I didn't necessarily think I would really need to bring my scarf and gloves down to North Carolina.
I, however, am certainly not about to complain too much as I was so inspired to see thousands of people brave the sub-zero temperatures in Washington to support the 41st annual March for Life. Each year, this is such a powerful event and I am very sorry I was not able to attend this year's important anniversary.
Unfortunately, the March for Life also marks the anniversary of a very tragic event in our nation's history—a devastating Supreme Court decision that has led to the death of nearly 56 million innocent children. Roe v. Wade threw aside the very ideals that our nation stands upon — that all people are created equal, and that we are endowed by our Creator with the right to life, liberty, and the pursuit of happiness. This decision ripped away these rights from the most vulnerable in our society — children in the womb.
As a country, we have a moral obligation to stand up to protect the most innocent in our society and to be a voice for those who are not given the opportunity to speak. Life is the most sacred, precious gift from God. Life begins at conception and ends with natural death, which means it is our responsibility to care for each and every life. As long as I am your elected Representative, it will be a priority of mine to fight for the end of abortion on demand. To that end, I cosponsored the bipartisan Prenatal Nondiscrimination Act (PRENDA), H.R. 447, introduced by Representative Trent Franks. This would restrict sex-selection abortion and race-selection abortion, as well as the coercion of a woman to obtain either. Any individual working at an abortion clinic who carries out these disgusting practices should be punished to the full extent of the law.
I strongly believe it is absolutely wrong to use your hard-earned taxpayer dollars to finance abortions and clinics that provide them like Planned Parenthood. Next week, the House will vote on H.R. 7, the No Taxpayer Funding for Abortion Act, which prohibits the use of federal funds for any abortions or any health benefits coverage that includes coverage of abortion. This is a commonsense proposal I will be proud to support.
Advances in technology and modern medicine have deepened our understanding of the stages of pregnancy. The facts are clear — babies in the womb can feel pain, we can detect heartbeats at earlier stages than ever before, and we can see the growth of these precious children at each step along the way. This evidence should settle the debate once and for all that abortions after 20 weeks are particularly cruel and should be strictly prohibited.
We've come a long way in shifting public opinion on this debate, and our position is stronger now than ever before. This is thanks entirely to all of you who have supported and worked so hard on behalf of the pro-life movement. One need not look no further than this week's March for Life to see the incredible display of compassion and support for the defense of the unborn.


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Associated Press
The price of oil rose by more than a dollar Monday, to above $94 a barrel, as it recovered from sharp drops last week and echoed strong gains in European stock markets. By early afternoon in Europe, benchmark crude for April delivery was up $1.11 to $94.24 a barrel in electronic trading on the New York Mercantile Exchange. The contract rose 29 cents to settle at $93.13 in New York on Friday. Oil dived last week on the possibility that the Federal Reserve might stop offering monetary stimulus, which its most recently released minutes suggested was possible. That would strengthen the dollar, making crude more expensive for international investors, and shut off a supply of money that some say had been fueling speculative trades in commodity markets. The price of crude stabilized F
Read the Full Article


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Kidney Dysplasia
What is kidney dysplasia?
The kidneys are a pair of bean-shaped organs that filter fluid and wastes from the blood to form urine. Urine flows from the kidneys to the bladder through tubes called ureters. Kidney dysplasia is a condition that can occur in babies while they are growing in the womb. Other terms that health care providers and scientists use to describe this condition are renal dysplasia and multicystic dysplastic kidney (MCDK).
In kidney dysplasia, the internal structures of one or both of the baby's kidneys do not develop normally. Fluid-filled sacs called cysts replace normal kidney tissue. Kidney dysplasia usually happens in only one kidney. A baby with one working kidney can grow normally and has few, if any, health problems. Babies with kidney dysplasia affecting both kidneys generally do not survive pregnancy, and those who do survive need dialysis and kidney transplant very early in life.
How does kidney dysplasia happen?
Ureters are tubes that grow into the kidneys and branch out to form a network of tubules that will collect urine when the fetus is developing in the womb. In kidney dysplasia, the tubules fail to branch out completely. The urine that would normally flow through small tubules has nowhere to go, so it collects inside the affected kidney and forms cysts.
Patient Comments
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Kidney Dysplasia - Treatment Question: What did your doctor prescribe for kidney dysplasia treatment?
Kidney Dysplasia - In Children Question: Does your child have kidney dysplasia?
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Sigma announced this new version of its very large focal range zoom at the beginning of October 2011. Let's see how this new version compares with its predecessors and competitors.
To provide photographers with a broader perspective about mobiles, lenses and cameras, here are links to articles, reviews, and analyses of photographic equipment produced by DxOMark, renown websites, magazines or blogs.
Sigma announced its 18-200 mm f/3.5-6.3 DC OS HSM II lens at the beginning of October 2011. While there's not much new in terms of its specs compared to the previous version (the Sigma 18-200mm F3.5-6.3 DC OS —see 18-200mm DxOMark reviews), the publication of its measurement results gives us the chance to take stock of all the Sigma offerings for this lens type.


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Choosing a Law Firm
Our website is full of information about our law firm and the lawyers here at Herrick and Hart. You can find out what kind of work we do, learn about our qualifications and experience, or even see if we have handled cases like yours. In short, you can do the research necessary to make this important decision and learn more about the law and how it might affect you. Choosing the right lawyer is just too important to be flipping the pages of the phone book and seeing where your finger lands.
We serve clients all across western Wisconsin, including Eau Claire, Chippewa Falls, Menomonie, Hudson, Rice Lake, Superior, Wausau, LaCrosse, and everywhere in between.


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Palo Alto Medical Foundation
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Well Well well....AH HA Ha ha....
Leave it to Bill Maher to use all the crazy people to make religion look ridiculous. I didn't see Billy Graham in there, or Franklin Graham, or Pat Robertson (he's pretty lucid during an interview), or one of those people are highly education or scientists who are pretty rational and still believe in God and/or the bible.
Apparently you've seen the full movie and these claims are based on facts? The Grahams don't appear in the film, nor does Pat Robertson, no believer scientists—you know all this for a fact? Did you time-travel to 10/3/2008? Any tips about what to watch out for between now and then? Is gas $8.00 a gallon in the USA?
I mean, hey, if you want a comedy, hey, by all means, show the most ridiculous people you can find. But it won't change any minds, because all rational people who are believers/non-believers know people like me, normal law abiding citizens, funny, rational, cool, hip (nah…....nerd more like it) who HAPPEN TO BELIEVE in the bible.
My position on this issue is this: The delusion of religious belief runs from it being a very mild delusion that most rational people simply compartmentalize outside of their work-a-day functioning world, to the severely deluded who are firmly entrenched that not only is their belief system accurate, but their gods even speak to them directly and tell them what to do.
The average person who goes to church every once in awhile is an example of the former, while a jihadist flying a plane full of screaming people into a skyscraper is an exmaple of the latter. But make no mistake: The category is a broad umbrella, but both types (and all in betweeners) are under the same umbrella
Hey, that's the way it is. And there are a billion or two like me out there. But here is the bottom line. At the end of the day, really, I…..think…..Bill Maher (though I'm fond of his ability to make me laugh) and all you atheists are ridiculous.
Bill Maher doesn't say he doesn't believe in a god by the way. He said he thinks religion is ridiculous. Do you actually know Maher's beliefs?
I mean, COME ONE, a creator is self evident to you just by looking at our universe and the myrid complexities of life, I mean, COME ON, don't be ridiculous!
An undemonstrated assertion isn't a fact. You're argument is jettisoned as you reach a conclusion. You cite the universe as complex, you cite the myriad complexities of life—and then you posit a more complex being who could design it all in the first place as the cause. I prefer something more consistent in the line of logic. If the universe is complex and needs an explanation, and life within the universe is complex and needs an explanation, and you assert something more complex as that explanation, I'm forced to ask you why you've abandoned the core foundation of your own assertion?


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Order calls for interagency cooperation on veterans' mental health
Tuesday - 9/4/2012, 5:33am EDT
President Obama signed an executive order Friday telling the Department of Veterans Affairs to add more personnel to respond to the mental health needs of veterans and telling other agencies to help, an effort to fight a rising tide of suicides in the military services.
Among its many provisions, the order tells VA to boost the capacity of its veterans' crisis hotline by 50 percent and to ensure that a veteran who identifies him or herself as in emotional distress gets seen by a mental health professional within 24 hours.
"I know you join me in saying to everyone who's worn the uniform, if you're hurting, it's not a sign of weakness to seek help. It's a sign of strength," Obama told soldiers at Fort Bliss, Texas, just after signing the order on Friday. "We're here to help you stay strong. That's the commitment I'm making to you."
The order also tells the departments of Defense, Veterans Affairs, Health and Human Services and Education to develop a national research action plan to improve early diagnosis and treatment capabilities for post-traumatic stress and traumatic brain injury. DoD and HHS will also undertake a comprehensive nationwide study on mental health to improve treatment outcomes.
Upward trend of suicides among servicemembers
The White House announcement comes amid an upward trend of suicides among servicemembers, both on active duty and in the reserve component. The Army alone reported a spike of 38 potential suicides in July — double the month before.
"We are now having more deaths from suicide than in combat losses," said Lt. Gen. Jeffrey Talley, chief of the Army Reserve, in a recent interview with Federal News Radio. "All the leaders of the Army are working every day to defeat that enemy. But there's no simple solution and there's no silver bullet."
While there's no silver bullet, everyone agrees there's a shortage of mental health professionals who are trained and ready to address mental health issues when they arise among servicemembers and veterans. The order tries to address that problem by telling VA to use HHS' network of private mental health providers that receive grants.
VA has already committed to hiring an additional 1,600 mental health professionals and 300 support staff, but has struggled to train and place new staff, especially in rural areas.
"Staffing is one of the major issues VA has as far as meeting veterans' mental health needs," said Brian Bertges, a rehabilitation policy expert at the American Legion. "At one point, VA claimed that 95 percent of the people that needed to be screened for mental health were seen within 14 days. In reality, it was about 64 percent."
Veterans groups mostly pleased with executive order
Still, Bertges and other veterans' service organizations were largely pleased with the White House's order.
"Long, repeat deployments through more than a decade of war have taken an immeasurable toll on our nation's newest generation of heroes," said John Hamilton, national commander of the Veterans of Foreign Wars. "We have an obligation to care for these brave men and women when they return from the front lines, and we think this executive order will be a critical step in fulfilling that obligation."
The order mirrors some recent legislative proposals sponsored by Sen. Patty Murray (D-Wash.), chairwoman of the Veterans Affairs Committee, and other lawmakers. Those proposals have not passed the full Congress. Putting them in the form of an executive order allows them to take effect more quickly, but lacks the ability to appropriate additional money to pay for the programs.
"There are a lot of competing interests for resources. If Congress doesn't fully fund the VA or the DoD in reference to these initiatives, yeah, there's a possibility none of this will ever happen," Bertges said. "But understanding the president's directive in this executive order, I think agencies are going to do everything in their power to make this happen."
There's no immediate prospect for extra VA funding next year. Congress is expected to pass a continuing resolution that will simply extend this year's funding levels for all government agencies into 2013. On the bright side, the Office of Management and Budget says the $1.2 trillion in automatic 10-year budget cuts that could happen at the end of the year under sequestration won't directly apply to veterans' benefits.
But Obama told soldiers at Fort Bliss that Congress needs to act to make sure sequestration doesn't cut funding for the rest of the government's programs, including the military.
"There's no reason those cuts should happen," he said. "Folks in Congress ought to come together and agree on a responsible plan that reduces the deficit and keeps our military strong. That's a pledge that we need to make to you."
Army suicides doubled last month from June's total
VA to add about 1,900 to mental health staff
Report: VA failing to provide timely mental care


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Wow. Very...revealing. I like.
TiPb TV 21: The 2011 Comicon mobile survey
TiPb Nation, Comicon finally hit Montreal so Rene and I hit Comicon to find out just which mobile device -- iPhone, BlackBerry or Android reigns supreme with geeks and nerds, cosplayers and booth babes alike! From Batman to Electra, Greedo to Garfield, I asked them all which cell phone they used, and the answers may surprise you! (Especially how many didn't use one at all -- what's up with that, data rates on Krypton just to high?)
So sit back, grab your popcorn, hit play, and see if the iPhone is strong at Comicon!
Have something to say about this story? Leave a comment! Need help with something else? Ask in our forums!
Reader comments
TiPb TV 21: The 2011 Comicon mobile survey
Haha...nice. :-D Love the blue streaks Georgia. :-D
It does show the "I see more [iphone|android|blackberry|etc]s in my area" is not a valid metric, from the other thread last week or so.
I would argue that the people who cosplay at a comic book convention don't represent the average, and Canadians are no different.
Who doesn't make the average of what ? Comicon people don't represent the average of comicon attendees. And canadians are no different ? They don't represent the average of canadians ?
Wow, I'm lost here.
All I know is that the hot girls that we see in the video do represent the average montrealer girl.
And that I know. I live here. :)
How do so many Americans not know who their President is? ; ) LOL
At this point I'd chalk it up to denial!
We know who he (the president) is. That's why we want him in prison! If you knew the facts, and can think rationally, you'd agree unequivocally!
To many folks said "Blackberry". LOL!!!
Not a single palm phone. Very telling. Not surprising in the least but probably a rude awakening for the Webos Taliban that insisted for a long time that webos phones were selling great. A ok o.s. but it really never caught on. And two years on i've only seen three others in real life.
The blonde is Marie-Claude Bourbonnais, by the way. And no, they're not real: http://mcbourbonnais.com/
Does anyone really care? They look fabulous!
It's spelled too. Not to.
Too high.
i love how the closer was "take that kevin!"... my name's kevin >.>
and Georgia looks really good in that zelda shirt lol
I'd like to B my L on her T's.
Really? That's a pretty sexist remark, why are you afraid to fill in the rest of the letters?
Georgia is a cutie. First time I have seen her in video.
Also, what's up with all the blackberry people? Seriously?
Georgia is so HOTTTT!!!
i phone's are de best phones very people should buy them more often
R2D2, it uses Android of course.


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There is only one player we NEED to get in FA and thats Josh Smith.
Josh Smith
JS and AD together would be unbelieveable.
Apparently being a true center means just being big.
Now I do voice overs. Finally!
Click here for my first voice demo!
There is only one player we NEED to get in FA and thats Josh Smith.
Josh Smith
JS and AD together would be unbelieveable.
Being an All-Star doesn't make you the paragon of what it is to be your position. Cousins would rather shot 22 foot jumpers than play back down, near the goal game. Asik is just a better Aaron Gray. Monroe is a PF. And Brook Lopez is softer than tissue paper. McGee and Jordan don't have half a brain to rub together between them.
And I'm not just thinking in terms of who can Davis guard. There's two ends of the court. I don't see any of those players I mentioned being able to contain him. Won't be easy, but going forward? With an offseason to continue to bulk up and perfect his game?
BTW---What does The PARAGON have to do with anything ? You certainly dont have to carry that moniker for a DEcent #5 to guard AD . There arent to many Paragons in the NBA, so i dont understand why that point of reference was used !
Last edited by Tinman; 12-04-2012 at 12:43 PM.
Paragon, epitome, gold standard, quintessence, prime example. Choose another name for it. Bottom line is getting an All-Star game bid doesn't mean much.
And AD doesn't have to guard them if they don't want to. Rendering this whole discussion meaningless. Quite conveniently, I might add. So...yeah, whatever brah.
I dont know but with all those fa's coming available. we should be able to come away with some nice players considering our cap room. Doesnt that rashard lewis contract come off the books next year??
I mean there's a reason they've gotten rid of the C position in the All-Star game. The role of the C has expanded past its traditional function of backing up their man and standing in the paint. There isn't one specific prototype center anymore; in fact, the opposite is more revered. The more diverse and well-rounded a big fella is, the more valuable he becomes. Davis has all the potential in the world to succeed at the C position with that in mind.
Last edited by PelsFan2313; 12-04-2012 at 12:58 PM.
"I'm not going to allow my putative owner to answer that question, this is an NBA related press conference. Paul Tagliabue and Roger Goodell have collectively sung their praises of Tom and if uh ESPN has a problem with that tell Mr. Skipper to call me at my office."
Since 2000, how many teams that dont have Lebron James on them have won a championship without a dominate big man ? Shaq 4---Duncan---3 Gasol/Bynum 2----Chandler and the 6'9" (BAD MAN) Ben Wallace ! He certainly wasnt your long lean athletic type !
Let's stop playing around here the most realistic scenario is that CP3 comes back and Dwight follows(both at lesser salary). Boom untouchable. If you're going to dream...dream big.
Not to mention that the All-Star ballot doesn't even have a slot for centers anymore. It's all guards and frontcourt.
Tinman makes reading this forum hard. I'd rather hear Da Thrones crap than try to read this jacked up paragraphs about having a fat center instead of Anthony Davis.
I'd take Anthony Davis those "true" 5s.
anthony davis is a 4, an incredible 4, and needs to stay at the 4. we need a SF and Center
I'm intrigued by just how much he can bulk up without losing anything in the way of athleticism.
Your right that he will be a center but I see it later in his career kind of like what Boston did with kg last year. He has too much offensive potential to sit in the post.
Just because you're a C doesn't mean they'll just stick you in the post.
I know I'm not a forum mod, but Tinman, please stop posting back to back to back.
Just edit your first post to include whatever additional information you would like to contribute.
It's a lot harder to read these threads when one user constitutes 50%+ of its posts. There's no back and forth, just one user making multiple arguments.
Not trying to be a *******, but that's just standard forum decency/policy.
There are currently 1 users browsing this thread. (0 members and 1 guests)


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One student at West Point High School, Oak Hill Academy and Hebron Christian School is going to get a little help with college next fall, providing they are planning to attend Mississippi State University.
The MSU Alumni Chapter in West Point will be hosting its fourth-annual golf tournament to raise money for scholarships that will go to one Bulldog-bound student from each area high school.
The golf tournament, which will be played on May 6, 2013 is the primary way the chapter raises money for these scholarships.
The tournament will be played at the Old Waverly Golf Club, and it will be a Four-Man Scramble.
Registration that day will be 9 a.m.
The price to enter is $150 per golfer and $600 per team. This includes lunch, cart, green fees and prizes.
There will be a first, second and third-place prize, along with a hole-in-one prize.
Sponsorships are open for Hole Sponsor: $200, White Level - $500
(includes 2 players) and Maroon Level - $750 (includes 4 players)
Registration and payment deadline is April 29.
|MSU Golf Touney 2013.pdf
|MSU Golf Tourney 2013-2.pdf


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Limited Edition Featured Artists
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You'll always know your piece is unique to you!
Whether you're an aficionado or a collector, there's a print to fit your budget and your passions — from fine art and fashion to football. Each print is museum-quality, printed in small batches, numbered and signed by the artist.
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The Power Cordz Prime Brake Cable System for road and mountain bikes allows you to have super light weight housing and cables that are corrosion free and offer smooth and responsive braking feel. Power Cordz uses a synthetic material called Zylon HM that is stronger then steel and 2x stronger then Kevlar. Once installed, the Power Cordz cables don't stretch out or require re-adjustment. Available for road and mountain bike brakes from all manufacturers.
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Get free Standard Shipping with your $50 purchase today! Just choose Standard Shipping during checkout.
Please note this offer is only available to physical shipping addresses in the 48 continental United States (no PO Boxes), and oversize charges still apply on some heavy/large items.


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|Introduction | Noise in shadows | Noise in mid-tones | Noise in highlights | Summary | Annex
Why does photonic noise variance have to be proportional to the signal?
It is worth noting that photonic noise is due to the light signal itself and is independent of the camera. It is mathematically modeled by a Poisson process, and consequently, the mean signal is equal to its variance. (This is a mathematical property of a Poisson process.) It is possible to prove this property without any assumptions; however:
Assume that a pixel receives photons and has a noise standard deviation . Let'snow cut this pixel into four smaller pixels of equal size. Each small pixel receives an average number of photons and has a noise standard deviation . Since the variance is additive, the variance of the large pixel value is the sum of the variances of the small pixel values. Thus:
From this, it is not very difficult to see that has to be proportional to .
Photonic noise is the main source of noise in the midtones, and follows the rule of 3dB per EV:
To provide photographers with a broader perspective about mobiles, lenses and cameras, here are links to articles, reviews, and analyses of photographic equipment produced by DxOMark, renown websites, magazines or blogs.


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First Hill - Treatment Center
The Swedish Cancer Institute's medical-oncology team includes physicians, oncology nurses and other oncology specialists with many years of experience treating a wide variety of cancers. At the Cancer Institute's Medical Treatment Centers, physicians treat patients with the most current anti-cancer drugs and treatment protocols available. Many medical oncologists here are also involved with promising clinical research treatments — ranging from national research trials to locally based studies they have initiated themselves.
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Cancer services are provided at the Arnold Pavilion and at our locations in Ballard, Bellevue, Cherry Hill, Edmonds, Redmond, Mill Creek, Issaquah and Burien (Highline partnership).
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Monogenic Forms of Diabetes: Neonatal Diabetes Mellitus and Maturity-onset Diabetes of the Young
On this page:
- Monogenic Forms of Diabetes
- What is neonatal diabetes mellitus (NDM)?
- What is maturity-onset diabetes of the young (MODY)?
- What do I need to know about genetic testing and counseling?
- Hope through Research
- Points to Remember
- For More Information
- Appendix and Selected References
The most common forms of diabetes, type 1 and type 2, are polygenic, meaning the risk of developing these forms of diabetes is related to multiple genes. Environmental factors, such as obesity in the case of type 2 diabetes, also play a part in the development of polygenic forms of diabetes. Polygenic forms of diabetes often run in families. Doctors diagnose polygenic forms of diabetes by testing blood glucose in individuals with risk factors or symptoms of diabetes.
Genes provide the instructions for making proteins within the cell. If a gene has a mutation, the protein may not function properly. Genetic mutations that cause diabetes affect proteins that play a role in the ability of the body to produce insulin or in the ability of insulin to lower blood glucose. People have two copies of most genes; one gene is inherited from each parent.
Monogenic Forms of Diabetes
Some rare forms of diabetes result from mutations in a single gene and are called monogenic. Monogenic forms of diabetes account for about 1 to 5 percent of all cases of diabetes in young people. In most cases of monogenic diabetes, the gene mutation is inherited; in the remaining cases the gene mutation develops spontaneously. Most mutations in monogenic diabetes reduce the body's ability to produce insulin, a protein produced in the pancreas that helps the body use glucose for energy. Neonatal diabetes mellitus (NDM) and maturity-onset diabetes of the young (MODY) are the two main forms of monogenic diabetes. MODY is much more common than NDM. NDM first occurs in newborns and young infants; MODY usually first occurs in children or adolescents but may be mild and not detected until adulthood.
Genetic testing can diagnose most forms of monogenic diabetes. If genetic testing is not performed, people with monogenic diabetes may appear to have one of the polygenic forms of diabetes. When hyperglycemia is first detected in adulthood, type 2 is often diagnosed instead of monogenic diabetes. Some monogenic forms of diabetes can be treated with oral diabetes medications while other forms require insulin injections. A correct diagnosis that allows the proper treatment to be selected should lead to better glucose control and improved health in the long term. Testing of other family members may also be indicated to determine whether they are at risk for diabetes.
Read more about diabetes in Your Guide to Diabetes: Type 1 and Type 2 at www.diabetes.niddk.nih.gov and in National Diabetes Statistics Report, 2014 at www.cdc.gov.
What is neonatal diabetes mellitus (NDM)?
NDM is a monogenic form of diabetes that occurs in the first 6 months of life. It is a rare condition occurring in only one in 100,000 to 500,000 live births. Infants with NDM do not produce enough insulin, leading to an increase in blood glucose. NDM can be mistaken for the much more common type 1 diabetes, but type 1 diabetes usually occurs later than the first 6 months of life. In about half of those with NDM, the condition is lifelong and is called permanent neonatal diabetes mellitus (PNDM). In the rest of those with NDM, the condition is transient and disappears during infancy but can reappear later in life; this type of NDM is called transient neonatal diabetes mellitus (TNDM). Specific genes that can cause NDM have been identified. More information about each type of NDM is provided in the appendix.
Symptoms of NDM include thirst, frequent urination, and dehydration. NDM can be diagnosed by finding elevated levels of glucose in blood or urine. In severe cases, the deficiency of insulin may cause the body to produce an excess of acid, resulting in a potentially life-threatening condition called ketoacidosis. Most fetuses with NDM do not grow well in the womb and newborns are much smaller than those of the same gestational age, a condition called intrauterine growth restriction. After birth, some infants fail to gain weight and grow as rapidly as other infants of the same age and sex. Appropriate therapy improves and may normalize growth and development.
What is maturity-onset diabetes of the young (MODY)?
MODY is a monogenic form of diabetes that usually first occurs during adolescence or early adulthood. However, MODY sometimes remains undiagnosed until later in life. A number of different gene mutations have been shown to cause MODY, all of which limit the ability of the pancreas to produce insulin. This process leads to the high blood glucose levels characteristic of diabetes and, in time, may damage body tissues, particularly the eyes, kidneys, nerves, and blood vessels. MODY accounts for about 1 to 5 percent of all cases of diabetes in the United States. Family members of people with MODY are at greatly increased risk for the condition.
People with MODY may have only mild or no symptoms of diabetes and their hyperglycemia may only be discovered during routine blood tests. MODY may be confused with type 1 or type 2 diabetes. People with MODY are generally not overweight and do not have other risk factors for type 2 diabetes, such as high blood pressure or abnormal blood fat levels. While both type 2 diabetes and MODY can run in families, people with MODY typically have a family history of diabetes in multiple successive generations, meaning that MODY is present in a grandparent, a parent, and a child. Unlike people with type 1 diabetes who always require insulin, people with MODY can often be treated with oral diabetes medications. Treatment varies depending on the genetic mutation that has caused the MODY. More information about each type of MODY is provided in the appendix.
What do I need to know about genetic testing and counseling?
Testing for monogenic diabetes involves providing a blood sample from which DNA is isolated. The DNA is analyzed for changes in the genes that cause monogenic diabetes. Abnormal results can determine the gene responsible for diabetes in a particular individual or show whether someone is likely to develop a monogenic form of diabetes in the future. Genetic testing can also be helpful in selecting the most appropriate treatment for individuals with monogenic diabetes. Prenatal testing can diagnose these conditions in unborn children.
Most forms of monogenic diabetes are caused by dominant mutations, meaning that the condition can be passed on to children when only one parent is affected. In contrast, if the mutation is a recessive mutation, a disease gene must be inherited from both parents for diabetes to occur. For recessive forms of monogenic diabetes, testing can indicate whether parents or siblings without disease are carriers for recessive genetic conditions that could be inherited by their children.
If you suspect that you or a member of your family may have a monogenic form of diabetes, you should seek help from health care professionals-physicians and genetic counselors-who have specialized knowledge and experience in this area. They can determine whether genetic testing is appropriate, select the genetic tests that should be performed, and provide information about the basic principles of genetics, genetic testing options, and confidentiality issues. They also can review the test results with the patient or parent after testing, make recommendations about how to proceed, and discuss testing options for other family members.
Hope through Research
Researchers are studying the genetic causes of and metabolic processes related to diabetes. Discoveries about monogenic forms of diabetes may contribute to the search for the causes of and treatments for type 1 and type 2 diabetes. For information about clinical trials related to diabetes and genetics, see www.ClinicalTrials.gov.
Points to Remember
- Mutations in single genes can cause rare forms of diabetes.
- Genetic testing can identify many forms of monogenic diabetes.
- A physician evaluates whether genetic testing is appropriate.
- A correct diagnosis aided by genetic testing can lead to optimal treatment.
- Recent research results show that people with certain forms of monogenic diabetes can be treated with oral diabetes medications instead of insulin injections.
For More Information
Information About Genetic Testing, Evaluation, and Counseling, Funded by the National Institutes of Health (NIH)
The GeneTests website provides information about medical genetics, an international directory of genetic testing laboratories, and an international directory of genetics clinics providing genetic evaluation and genetic counseling.
Information About Genetics
National Human Genome Research Institute
Genetic and Rare Diseases Information Center
P.O. Box 8126
Gaithersburg, MD 20898-8126
Phone: 1-888-205-2311
Fax: 240-632-9164
Email: email@example.com
Internet: www.genome.gov/health
The Genetics Home Reference website provides consumer-friendly information about genetic conditions. A service of the National Library of Medicine (NLM), NIH.
The Online Mendelian Inheritance in Man database is a catalog of human genes and genetic disorders with references and related links. A service of the National Center for Biotechnology Information, NLM, NIH.
Entrez Gene is a searchable database of genes with extensive information and related links. A service of the National Center for Biotechnology Information, NLM, NIH.
The Diabetes Research department and the Centre for Molecular Genetics at the Peninsula Medical School and Royal Devon and Exeter Hospital, Exeter, United Kingdom provides information for patients and health care professionals about genetic forms of diabetes.
The International Society for Pediatric and Adolescent Diabetes is an international society for health care professionals and others interested in childhood diabetes. They publish consensus guidelines; see the list of selected references.
Information About DiabetesNational Diabetes Information Clearinghouse
1 Information Way
Bethesda, MD 20892-3560
Phone: 1-800-860-8747
Fax: 703-738-4929
Email: firstname.lastname@example.com
Internet: www.diabetes.niddk.nih.gov
National Diabetes Education Program
1 Diabetes Way
Bethesda, MD 20892-3560
Phone: 1-800-438-5383
Fax: 703-738-4929
Email: firstname.lastname@example.com
Internet: www.ndep.nih.gov
American Diabetes Association
National Call Center
1701 North Beauregard Street
Alexandria, VA 22311-1742
Phone: 1-800-DIABETES (342-2383)
Fax: 703-549-6995
Email: firstname.lastname@example.com
Internet: www.diabetes.org
Juvenile Diabetes Research Foundation International
26 Broadway, 14th Floor
New York, NY 10004
Phone: 1-800-533-CURE (2873)
Fax: 212-785-9595
Email: firstname.lastname@example.com
Internet: www.jdrf.org
You may also find additional information about this topic by visiting MedlinePlus at www.medlineplus.gov.
This publication may contain information about medications. When prepared, this publication included the most current information available. For updates or for questions about any medications, contact the U.S. Food and Drug Administration toll-free at 1-888-INFO-FDA (1-888-463-6332) or visit www.fda.gov. Consult your doctor for more information.
The U.S. Government does not endorse or favor any specific commercial product or company. Trade, proprietary, or company names appearing in this document are used only because they are considered necessary in the context of the information provided. If a product is not mentioned, the omission does not mean or imply that the product is unsatisfactory.
Appendix and Selected References
Appendix: Characteristics of Monogenic Forms of Diabetes
|Type of Diabetes
|Gene or Syndrome*
|Affected Protein
|How Common
|Usual Age of Onset
|Type of Inheritance or Mutation**
|Causes Intrauterine Growth Restriction?
|Transient or Permanent?***
|Neonatal Diabetes Mellitus (NDM)
|Rare; occurs in about one of every 100,000 to 500,000 live births
|Permanent Neonatal Diabetes
Mellitus (PNDM)
|50% of all cases of NDM
|Most common type of PNDM
|3 to 6 months
|Autosomal dominant (10%)
|Permanent (This gene also causes a transient form of NDM; see TNDM section)
|Treated with insulin in the past but often can be treated with oral sulfonylureas
|SUR1-sulfonylurea receptor 1
|1 to 3 months
|Autosomal dominant (12% of NDM)
|Permanent (This gene also causes a transient form of NDM; see TNDM section)
|Treated with insulin in the past but often can be treated with oral sulfonylureas
|1 week
|Autosomal recessive
|IPF1; also known as PDX1
|insulin promoter factor 1
|1 week
|Autosomal recessive
|Treat to replace endocrine and exocrine pancreas functions
|pancreas transcription factor 1 A
|At birth
|Autosomal recessive
|Treat to replace endocrine and exocrine pancreas functions
|FOXP3, IPEX syndrome
|forkhead box P3
|Sometimes present at birth
|EIF2AK3, Wolcott-Rallison syndrome
|eukaryotic translation initiation factor 2-alpha kinase 3
|3 months
|Autosomal recessive
|Insulin and treatment for associated conditions
|Transient Neonatal Diabetes
Mellitus (TNDM)
|50% of all cases of NDM
|ZAC: pleomorphic
adenoma gene-like 1 or PLAG1
HYMAI: hydatiform mole-associated and imprinted transcript
|Most common form of NDM
|Birth to 3 months
|Autosomal dominant
|Initially, treat with insulin; reduce dosage as needed; when diabetes recurs, treat with diet modification and physical activity; may also require insulin
|SUR1-sulfonylurea receptor 1
|Birth to 6 months
|Autosomal dominant
|Transient (This gene also causes a permanent form of NDM; see PNDM section)
|Oral sulfonylureas
|Uncommon cause of TNDM but most common cause of PNDM
|Birth to 6 months
|Autosomal dominant
|Transient (This gene also causes a permanent form of NDM; see PNDM section)
|Oral sulfonylureas
|HNF1 β (beta); also known as HNF1B
|hepatocyte nuclear factor 1B
|Birth to 6 months
|Autosomal dominant (60%)
|Maturity-onset Diabetes of the
Young (MODY)
|1 to 5% of all cases of diabetes in the United States
|hepatocyte nuclear factor 4ɑ (alpha)
|Adolescence or early adulthood
|Autosomal dominant
|For most, oral sulfonylureas; some patients may need insulin
|MODY 2 and MODY 3 account for about two-thirds of all cases of MODY
MODY 2 is the second-most common form of MODY
|Mild hyperglycemia may be present at birth; otherwise, early childhood
|Autosomal dominant
|Lower than normal birth-weight can occur
|Diet modification and physical activity; medications usually not required; some patients do not require any treatment during childhood
|hepatic nuclear factor 1ɑ (alpha) or HNF1ɑ (alpha) or HNF1A
|MODY 3 is the most common form of MODY
|Adolescence or early adulthood
|Autosomal dominant
|Initially, treat with diet modification; can be treated with oral sulfonylureas; some patients may need insulin
|IPF1; also known as PDX1
|insulin promoter factor 1
|Early adulthood; can present later
|Autosomal dominant
|Oral sulfonylureas; some patients may need insulin
|hepatic nuclear factor 1β (beta) or HNF1B
|Adolescence or early adulthood
|Autosomal dominant
|Insulin; patients also may need treatment for related conditions such as kidney failure or cysts
|NeuroD1, or BETA2
|neurogenic differentiation factor 1
|In the fourth decade of life
|Autosomal dominant
*Gene or Syndrome: the name of the gene with the mutation or the syndrome-a grouping of conditions that occur together and indicate a specific disease-caused by the mutated gene
**Type of Inheritance or Mutation:
- Autosomal dominant. Normally, every cell has two copies of each gene-one that comes from the mother and one from the father. An autosomal dominant inheritance pattern means that a mutation happens in only one copy of the gene, and a parent with a mutation can pass on a copy of their working gene or a copy of their damaged gene. In autosomal dominant inheritance, a child who has a parent with a mutation has a 50% chance of inheriting that mutation.
- Autosomal recessive. Normally, every cell has two copies of each gene-one that comes from the mother and one from the father. An autosomal recessive inheritance pattern means a mutation must be present in both copies of the gene in order for a person to be affected, and each parent must pass on a gene mutation for a child to be affected. If a person only has one copy of the gene mutation, that person is called a carrier. If both parents are carriers of a recessive gene mutation, each child has a 25% chance of being affected.
- Spontaneous. A new mutation, or change, in a gene
- X-linked. When a trait or a disease occurs in a person who has inherited a mutated gene on the X chromosome, one of the sex chromosomes
***Transient or Permanent: whether the form of diabetes goes away after some time, called transient, or is permanent
Selected References
Babenko AP, Polak M, Cavé H, Busiah K, Czernichow P, Scharfmann R, Bryan J, Aguilar-Bryan L, Vaxillaire M, Froguel P. Activating mutations in the ABCC8 gene in neonatal diabetes mellitus. New England Journal of Medicine. 2006;355(5):456-466.
Colombo C, Delvecchio M, Zecchino C, Falenza MF, Cavallo L, Barbetti F. Transient neonatal diabetes mellitus is associated with a recurrent (R201H) KCNJ11 (Kir6.2) mutation. Diabetologia. 2005;48:2439-2441.
Craig ME, Hattersley A, Donaghue K. International Society for Pediatric and Adolescent Diabetes (ISPAD) Clinical Practice Consensus Guidelines 2006-2007. Definition, epidemiology and classification. Pediatric Diabetes. 2006;7:343-351.
Fajans SS, Bell GI, Polonsky KS. Molecular mechanisms and clinical pathophysiology of maturity-onset diabetes of the young. New England Journal of Medicine. 2001;345(13):971-980.
Gloyn AL, Pearson ER, Antcliff JF, Proks P, Bruining GJ, Slingerland AS, Howard N, Srinivasan S, Silva JM, Molnes J, Edghill EL, Frayling TM, Temple IK, Mackay D, Shield JP, Sumnik Z, van Rhijn A, Wales JK, Clark P, Gorman S, Aisenberg J, Ellard S, Njølstad PR, Ashcroft FM, Hattersley AT. Activating mutations in the gene encoding the ATP-sensitive potassium-channel subunit Kir6.2 and permanent neonatal diabetes. New England Journal of Medicine. 2004;350(18):1838-1849.
Hattersley A, Bruining J, Shield J, Njølstad P, Donaghue K. International Society for Pediatric and Adolescent Diabetes (ISPAD) Clinical Practice Consensus Guidelines 2006-2007. The diagnosis and management of monogenic diabetes in children. Pediatric Diabetes. 2006;7:352-360.
Hattersley AT. Beyond the beta cell in diabetes. Nature Genetics. 2006;38(1):12-13.
Hattersley AT, Ashcroft FM. Activating mutations in Kir6.2 and neonatal diabetes: new clinical syndromes, new scientific insights, and new therapy. Diabetes. 2005;54:2503-2513.
Hattersley AT, Pearson, ER. Minireview: pharmacogenetics and beyond: the interaction of therapeutic response, beta-cell physiology, and genetics in diabetes. Endocrinology. 2006;147:2657-2663.
National Center for Biotechnology Information, National Library of Medicine, National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases. The genetic landscape of diabetes. Available at: www.ncbi.nlm.nih.gov/books/bv.fcgi?call=bv.View..ShowTOC&rid=diabetes.TOC&depth=2. Posted 2004. Accessed January 2, 2007.
Pearson ER, Flechtner I, Njølstad PR, Malecki MT, Flanagan SE, Larkin B, Ashcroft FM, Klimes I, Codner E, Iotova V, Slingerland AS, Shield J, Robert JJ, Holst JJ, Clark PM, Ellard S, Søvik O, Polak M, Hattersley AT for the Neonatal Diabetes International Collaborative Group. Switching from insulin to oral sulfonylureas in patients with diabetes due to Kir6.2 mutations. New England Journal of Medicine. 2006;355(5):467-477.
Ræder H, Johansson S, Holm PI, Haldorsen IS, Mas E, Sbarra V, Nermoen I, Eide SA, Grevle L, Bjorkhaug L, Sagen JV, Aksnes L, Søvik O, Lombardo D, Molven A, Njølstad PR. Mutations in the CEL VNTR cause a syndrome of diabetes and pancreatic exocrine dysfunction. Nature Genetics. 2006;38(1):54-62.
Sperling MA. ATP-sensitive potassium channels-neonatal diabetes mellitus and beyond. New England Journal of Medicine. 2006;355(5):507-510.
Sperling MA. Neonatal diabetes mellitus: from understudy to center stage. Current Opinion in Pediatrics. 2005;17:512-518.
Vaxillaire M, Froguel P. Genetic basis of maturity-onset diabetes of the young. Endocrinology and Metabolism Clinics of North America. 2006;35:371-384.
National Diabetes Information Clearinghouse
1 Information Way
Bethesda, MD 20892–3560
Phone: 1–800–860–8747
Fax: 703–738–4929
Email: email@example.com
Internet: www.diabetes.niddk.nih.gov
The National Diabetes Information Clearinghouse (NDIC) is a service of the National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK). The NIDDK is part of the National Institutes of Health of the U.S. Department of Health and Human Services. Established in 1978, the Clearinghouse provides information about diabetes to people with diabetes and to their families, health care professionals, and the public. The NDIC answers inquiries, develops and distributes publications, and works closely with professional and patient organizations and Government agencies to coordinate resources about diabetes.
Publications produced by the Clearinghouse are carefully reviewed by both NIDDK scientists and outside experts. This publication was reviewed by Mark A. Sperling, M.D., Department of Pediatrics, Children's Hospital, University of Pittsburgh; Kenneth S. Polonsky, M.D., Department of Medicine, Washington University School of Medicine; and Concepcion R. Nierras, Ph.D., Juvenile Diabetes Research Foundation International.
This publication is not copyrighted. The Clearinghouse encourages users of this publication to duplicate and distribute as many copies as desired.
NIH Publication No. 07-6141
March 2007
Page last updated August 27, 2014


text:
JON CARDINELLI says the decision to move the South African leg of the IRB Sevens Series to Cape Town is a no-brainer.
According to Die Burger, plans are afoot to move the annual sevens spectacle from Outeniqua Park in George to one of the World Cup stadiums in Cape Town, Port Elizabeth or Rustenberg.
Saru hopes that the South African leg will take on the shine and glamour of the Hong Kong event, and while Outeniqua Park has its charms, it cannot compete with the World Cup venues.
George's contract to host the tournament expires this year, meaning this weekend's competition is likely to be the last time the southern Cape city stages the event. Come next year, the world's best sevens players should be competing in an arena worthy of the occasion, arenas that house at least five times as many people as the 8 000-seater Outeniqua Park.
It's a no-brainer to move away from George. Moving to a bigger stadium will mean a bigger occasion, with more people filtering through the gates and adding to the spectacle. It also makes sense to stage the event in a more accessible centre, as international visitors need to fly to Cape Town before connecting to George.
So the only decision that remains is whether to move the event to the Cape Town Stadium, the Nelson Mandela Bay Stadium, or the Royal Bafokeng Stadium. These venues were largely successful during the 2010 soccer World Cup, and two of those venues have enjoyed further use.
The Royal Bafokeng Stadium is home to the Platinum Stars while the Eastern Province Kings have taken residence at the Nelson Mandela Bay Stadium. The Cape Town Stadium, however, is still struggling to acquire a resident sports side with the Stormers and Western Province refusing to move away from Newlands. Negotiations are ongoing, but for the moment, the magnificent venue in Green Point remains vacant.
These reasons alone won't be enough to swing the vote, but Cape Town may crack the nod by virtue of their sports-mad fan base.
The crowd numbers at Newlands in the past couple of years have been record-breaking, while the Stormers' first-ever match at the Cape Town Stadium in February (a warm-up game against Boland) drew a crowd of 40 000. Fifty thousand Capetonians watched Bafana Bafana play USA in November, a fixture that witnessed an unfavourable 1-0 result to the Americans, but was once again an outstanding occasion.
It makes the most commercial sense to pick Cape Town as the host city. Apart from the resident fan base, it's easily the most popular tourist destination of the three cities lobbying for the event.
Like George, Rustenberg, or more accurately, Phokeng, is not as accessible to international travellers as Cape Town. Tourists from overseas will also need to connect to Port Elizabeth from one of South Africa's international airports if they want to attend an event at the Nelson Mandela Bay Stadium.
If Saru's aim to is to make the South African leg as successful and as popular as the Hong Kong Sevens, they need to pick a venue as well as a city that is popular with foreign and local fans alike. Saru needs to take the Sevens Series to one of the World Cup stadiums, and they need to take it to the venue situated in one of the world's favourite cities.


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