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Government cuts drag down U.S. economy
That's the conclusion to draw from the new report on gross domestic product. The U.S. economy grew at a 2.5 percent annual rate in the first three months of the year, which was an improvement from the weak 0.4 percent of the final months of 2012. But Friday's report was nearly a percentage point below analysts' expectations, and for the last six months, number averages to only a 1.45 percent annual rate of growth.
The report should scratch any thought that our economy is heading into "escape velocity" and breaking into a higher, self-reinforcing trajectory of growth. That had appeared to be the case in the first couple of months of the year. But it just isn't so. We're muddling along at basically the same pace we've been at for nearly four straight years of this dismal recovery, with growth too slow to make up the lost economic ground from the 2008-2009 recession.
The report details this stuck-in-neutral economy. It's not without bright spots, but there aren't enough of them, and they aren't bright enough to make up for the forces dragging the recovery, most significantly a drop in government spending.
The biggest culprit in the weak report was the government sector, which fell at a 4.1 percent rate, after a 7 percent pace of decline in the fourth quarter. The fall was universal -- at the federal, state and local levels. The U.S. government is in pullback mode, and whatever one thinks about reducing the size government in the long run, for now it is unequivocally the villain in slowing growth. If there'd been no change in government spending over the last six months, GDP growth would have averaged a respectable 2.55 percent, not the current soft 1.45 percent.
There's also little sign that the private sector is expanding aggressively to make up the difference. Business investment in structures (think factories and office building) fell in the first quarter, while spending on equipment and software rose at a 3 percent annual rate, far below the double-digit pace that was common earlier in the recovery.
The best news in from the report is that Americans spent more these first three months of the year. Personal spending rose at a 3.2 percent annual rate, the strongest since the end of 2010, with particularly good showings in durable goods (such as cars and furniture) and services (everything from restaurant meals to health care). That spending could go down in future quarters, as Americans see their paychecks shrink under the higher payroll taxes. But for now, consumers aren't the problem.
Housing, on the other hand, which many have hoped will be the big driver of growth in 2013, is expanding, but not fast enough. Residential investment rose at a 12.6 percent rate in the first quarter, but that is coming off a very low starting point, and is slower than the 17.6 percent growth rate of the fourth. This housing "boom" added only 0.3 percentage points to the overall rate of GDP growth. The improvement is welcome, but builders need to work more than they did in the first quarter of 2013.
Many see these first months of 2013 as a face-off between a government that's retrenching and a housing sector that's expanding. In the first quarter, government unequivocally took dominance, subtracting 0.8 percentage points from growth, as housing added only a 0.3 percentage point.
So, there you have it. Economics writers are always looking for signs of change, and we excitedly (sometimes too eagerly) leap to identify a new trend in the economy. But this time there isn't one. Government is contracting, as it has in 10 of the last 11 quarters. The private sector is improving quickly enough to counteract that contraction and ensure that GDP growth is expanding - but not fast enough to spur the robust recovery that the country sorely needs.
Neil Irwin writes the Econ Agenda column for The Washington Post and is the economics editor of Wonkblog, The Post's site for policy news and analysis.
China moves to accept imports of all US apple varieties Everett Clinic to lease space in Frontier Building Boeing, SpaceX will beat Russia on price for astronaut rides Employers: Agreement on key issue in port labor dispute Milk industry fights back against �anti-dairy folks� WikiLeaks slams Google over delay in revealing US warrants
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Nvidia and AMD/ATI uses thousends of labour hours for tunning their windows drivers to get high scores on benchmarks. No linux driver get's such attention and Futuremarks test's are developed only for windows so there is no tunning for these tests in linux 3d drivers.
To make meaningful WINE test use something which is developed for windows, but hadn't been so owerly tuned by driver developers!

subset: commoncrawl


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Posted on 8/7/2013 by Crystal D. Holland
Jason T. Graves is a Winston Salem-based author and educator. He will talk about his experiences with writing for traditional and self-publishing, and building an author platform on social media. He will also discuss the craft of writing—learned the hard way—and give selected readings, including excerpts from Blood Roses and his forthcoming novel Morning Stars. Learn more about Mr. Graves at his website: http://www.jasontgraves.com
Come with questions or get your own copies of his books autographed. We look forward to seeing you here! This program is FREE and open to the public. No registration is required. Appropriate for ages 12 and older.
Kernersville Branch Library Paddison Room Monday, August 19th @ 6PM
130 E. Mountain St. Kernersville, NC 27284 (336) 703-2930 (336) 993-5216 FAX
Closed Sunday 10:00 - 9:00 Monday 10:00 - 9:00 Tuesday 10:00 - 9:00 Wednesday 10:00 - 6:00 Thursday 10:00 - 6:00 Friday 10:00 - 5:00 Saturday Hours for All Locations Holiday Closings


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World News and Views :: Top of the News Total Group reports lower sales and earnings; Satorp to be commissioned by year-end10:17 AM MDT | August 5, 2013 | Natasha Alperowicz Total Group has reported lower second-quarter sales and earnings. Group net income was down 3% at €2.7 billion ($3.5 billion) on sales 4% lower at €46.97 billion. Adjusted operating income was €5.08 billion, down 11% on the corresponding period in 2012. Adjusted net operating income at the refining & chemicals business was down 2% to €370 million and adjusted operating income down 22% at €357 million. Refining & chemicals sales were €21.56 billion in the second quarter of this year, compared with €22.59 billion in... This information is only available to Chemical Week subscribers. Forgot your user ID or password?
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SLE314 - Research Project
Unit details
Year | 2015 unit information |
Enrolment modes: | Trimester 1: Burwood (Melbourne), Warrnambool, Waurn Ponds (Geelong) Trimester 2: Burwood (Melbourne), Warrnambool, Waurn Ponds (Geelong) Trimester 3: Burwood (Melbourne), Warrnambool, Waurn Ponds (Geelong) |
Credit point(s): | 1 |
EFTSL value: | 0.125 |
Previously coded as: | SBS311 |
Unit chair: | Anneke Veenstra (B) |
Coordinator: | F Pfeffer (G), A Mainwaring (W) |
Prerequisite: | Students would normally be expected to have a weighted average mark of 70% for all completed units, please contact the Unit Chair prior to enrolling in this unit. |
Corequisite: | Must be enrolled in S382, S393, S398, S321, S323, S324, S320, S399, S394, S369, D311, D321, D351, D372, D331, or D329 |
Incompatible with: | SQA302, SLE344 |
Contact hours: | Students are expected to attend meetings with the Unit Chair and/or supervisor. |
This unit involves a substantial piece of independent research conducted under the supervision of a member of staff. Students will not be permitted to take more than one credit point, within the School of Life and Environmental Sciences, from this offering.
Written report 60%, oral presentation 15%, supervisor mark 10%, research project proposal 15%
Unit Fee Information
Student Contribution Rate* | Student Contribution Rate** | Fee rate - Domestic Students | Fee rate - International students |
$1096 | $1096 | $3863 | $3609 |
* Rate for all CSP students, except for those who commenced Education and Nursing units pre 2010
** Rate for CSP students who commenced Education and Nursing units pre 2010
Please note: Unit fees listed do not apply to Deakin Prime students.


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The Soul Dew (こころのしずく Soul Droplet) is an item held by either Latios or Latias in Pokémon Ruby, Sapphire and Emerald. This held item raises the Special Attack and Special Defense of the Eon Pokémon by 50%, both of which are exclusively available on Southern Island. It is this attributed feature that led to its banning in all official tournaments. The Soul Dew is also available for purchase in Pokemon Battle Revolution for 14,400 Poké Coupons.
In the Games
In the remade versions of Pokémon Gold and Silver, the player is able to receive an item called the Enigma Stone through means of an event. In the Pewter Museum of Science, the player meets Steven Stone, who determines that it is in fact the Soul Dew. Once outside, the player will encounter one of the Eon Pokémon, given the opportunity to capture it. Latios is available in HeartGold, whereas Latias is available in SoulSilver.
In Black2 and white2, soul dew is dropped after latios/latias has been captured in the dreamyard. However, wielding the soul dew will turn latios/latias into legendary status and prevented from tournament entries, such as the PWT.
In the Movies
Soul Dew (こころのしずく Soul Droplet) | |
General | |
Function: | Contains the soul of Latios and activates the DMA |
Debut: | Pokémon Heroes |
The Soul Dew plays a pivotal role in the 5th Pokémon movie, Pokémon Heroes, in which it is able to power a device known as the Defense Mechanism of Alto Mare. The Soul Dew was granted to an elderly couple in the past, after Latios sacrificed its life to save Alto Mare. It is acquired by members of Team Rocket, Annie and Oakley, who wish to use its powers to control the world. The Soul Dew's purpose is to protect the city of Alto Mare from danger, and to this length it is capable of gridlocking the city, controlling the water sources and reviving Pokémon fossils, once connected with the Defense Mechanism. The Soul Dew is said to house the spirit of a Latios.
The Soul Dew is embedded into a water fountain located within the hidden garden. Its sole purpose is to maintain water flow within the city of Alto Mare. Once the Soul Dew is removed from the aforementioned fountain, it is only a matter of time before all of the water drains from the city. When the Soul Dew is placed into the Defense Mechanism, it becomes tainted as a result of overuse. Once a person touches the Soul Dew whilst in this fragile state, it will disintegrate, causing the Defense Mechanism to go berserk.
Name Origin
- In Pokémon Heroes, Oakley reveals that the Soul Dew was named because it holds the soul of a Latios, and that it is "like a giant dew drop".


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"You're exactly right. My blade is full of hate. I didn't come here to fight you. I came here to violently hack you to pieces."- Tōshirō Hitsugaya
Basic InformationName: Tōshirō Hitsugaya Yuki
Nickname: Icy Midget, Shiro- Chan, Whitey
Gender: Male
Age: 17
Clan: Yuki
Personality: Hitsugaya is generally mature and serious. Tōshirō is shown to be easily annoyed by others goofing off or drawing unneeded attention to themselves. Hitsugaya hates anything that he deems childish as well as being described as something close to a child, such as an elementary school child. Hitsugaya dislikes summer and warm weather in general.
Looks: Tōshirō is short, with turquoise eyes and short, spiked, white hair. He wears a standard sleeveless white jacket with a green sash around his shoulders, held together by a round, star-like clip. The sash holds his katana's sheath in place on his back and is tied to it at either end.
Village InformationVillage Of Birth: Kirigakure
Village Of Alliance: Konoha
Rank | Chakra InformationNinja-Rank: Kage
Speciality: Water | Lightning | Intelligence | Blue Jays
雷遁 火遁 風遁 水遁
Katon | Mastered
Fuuton | Mastered
Raiton | Mastered
Suiton | Mastered
Doton | C Rank
Hyōton | Needs Training
Ninjutsu: Rasengan + Shadow clones| Taijutsu | Kenjutsu: Started
"Sit Atop The Frozen Heavens, Hyourinmaru!"
Suiton: Due to his Kekkei Genkai Toshiro has a great mastery over Suiton allowing him to perform water jutsu with a single handseal
Intelligence: Toshiro has shown great intelligence to the point where he is highly observant and analytical, allowing himself to see through most deceptions and is rarely caught off-guard.
Raiton: Due to extensive training with lightning Toshiro can perform handseals for lightning jutsu much quicker than normal shinobi
Hyōrinmaru: Hyōrinmaru looks like a normal katana with the exception of the guard, which is in the shape of a four-pointed bronze-colored star. Its hilt is light blue and sheath dark blue. The sword is somewhat longer than most swords, with Hyōrinmaru being at about 1.4 meters long, making it taller than Hitsugaya, preventing him from wearing it at the hip. Instead he carries it on his back by a chain.
Cold Resistance: Being a Yuki and growing up using Ice release, Toshiro has become more resilient to the effects of very cold temperatures.
Background Information
Hitsugaya comes from a relatively poor province in Kirigakure. There, he lived with his grandmother and Momo Hinamori. As a young child, Hitsugaya was a bit of a brat who likes to eat watermelons and poke fun at Hinamori. She calls him "Shiro-chan" and he retorts by calling her "Bed-wetter Momo." The two are close friends and Hitsugaya, despite being younger and much shorter than his friend, always feels that he needs to protect her.
Even at a young untrained age, Hitsugaya could feel the power of his clans KG Ice pumping through his body. Despite this he never told anyone and couldn't accurately determine what was happening other than to assume it was a strange sensation.
When Hinamori left to become shinobi, he portrayed himself as happy to see her go, though it was more of an act than his true feelings. Her next door friends were scared of him, even though he had never done anything to them. His grandmother, "Granny", and Hinamori were the only ones in the district that weren't afraid of him. His personality was supposedly "cold as ice" or at least thought to be, and he often wondered if it was due to his appearance or attitude. When Momo had been in the Ninja Academy for 5 years, he began to notice how he hadn't grown an inch. He noticed that Hinamori's hair grew longer and she visited less often, seemingly having a goal in her life now. He also noticed that "Granny" was getting skinnier.
While out at the market one day a shopkeeper, whom he was buying items from, was treating him disdainfully. He then meets Rangiku Matsumoto, who yells at the shop keeper for being rude to Hitsugaya and reproached him for his poor customer skills. When Hitsugaya turns around, he is knocked down by bumping into Rangiku's bosom. She then yells at him for "lying there and crying about it and not standing up like a man", although it was she who had knocked him down in the first place. He then shakes her off and runs away.
Later that night, he has a vivid dream of a gigantic, icy blue, serpentine dragon with large wings and an echoing voice. This dream is markedly different from the previous ones, where he had only experienced sensations but nothing substantial to what the meaning might be. This time, however, the reason behind the dreams not only materializes, but tries to tell him its name. Hitsugaya cannot hear him, however, as his voice is muffled by strong winds. He then wakes up in a cold sweat. Upon waking up, he sees Rangiku and is surprised to see her in his home. She immediately tells him to stop leaking his reiatsu everywhere and to get some sleep. She further explains that his grandmother looks pretty cold, which causes Hitsugaya to notice for the first time that the room is exceedingly cold and affecting his grandmother. Rangiku then tells him he should become a Shinobi, stating that kids with power as strong as his need to learn how to bring their power under control. She explains that if he stays the way he is, his power will end up killing his grandmother. At first, Hitsugaya isn't sure what she is talking about but she calmly places her hand on his chest and asks him if can feel a strange, cold sensation. She explains to him that once he has truly felt that sensation, he'll understand how to control his power. She then notes that for that to happen he must become a Shinobi.
Soon after, he tells his grandmother of his intentions of becoming a Shinobi. Much to Hitsugaya's surprise, she is happy for him. She tells him that she had always felt that he holds everything in because he didn't want to leave her, but doing so was only hurting him and in turn seeing him that way, hurt her. Hitsugaya then leaves to follow his own path with his grandmother's blessing.
With Hitsugaya's natural talent, he quickly entered the Shinobi Academy and graduated early despite his age. Hinamori continued to call him "Shirō-chan" out of habit and felt she needed to protect him. Because of a promise she had made, she only began to call him "Hitsugaya-kun" after he had achieved Jounin status and was recognized as a Shinobi. Highly capable and knowledgeable, he quickly became the youngest of the Jounin within the village.
Summoning Animal: Blue Jay(Aokakesu)
Scroll Owner: Light...
Other Users who have signed contract: Sage of Darkness
Summoning Boss if existing: Raijin
Other Summoning Animals tied to contract: Suijin ,Odin, Skyrus, Raijin
Kuchiyose no jutsu | Aokakesu:Raijin
Kuchiyose no jutsu Aokakesu | Suijin
Kuchiyose No Jutsu: Odun| Summoning Jutsu Odun
Kuchiyose Skyrus | Summoning SkyrusTheme Song and Background
BattlesWon: Countless
Lost: Countless
Permissions: Permission for a toshiro bio from Toshiro
Permission for a custom ice bio from RokuNR
Credit goes to Kato. for making the bio D, i changed a bit


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REAL SPORTS WITH BRYANT GUMBEL caps its 20th season with a special roundtable review of the show's most memorable moments from 2014.
213: December 16, 2014One-on-one with Mark Cuban; A Watts Bears profile; Six-year-old surfer Quincy Symonds; Ref assaults update
212: November 25, 2014Marketing shooting sports to children; SMU basketball coach Larry Brown; an update on hockey's Yaroslavl Lokomotiv plane crash.
203: February 25, 2014Marijuana use in the NFL; one-on-one with former NFL MVP Boomer Esiason; Sacramento Kings owner Vivek Ranadive; safety and professional bull riding.
202: January 21, 2014Real Sports presents a roundtable review of 2013.
201: December 17, 2013Real Sports examines Russian president Putin's controversial Winter Olympics; Profiles Denver Nuggets rookie head coach Brian Shaw; And, looks at the wild world of live animal mascots.
200: November 19, 2013One-on-one with Reggie Jackson; A profile of�Clippers star Chris Paul; Professional video gaming; And, the inspirational story of Jaclyn Murphy.
199: October 22, 2013NFL accountability in the concussion crisis; the spectacle of a Sounders' game; a profile of one of sailing's best; and, the remarkable baseball comeback story of Steve Delabar.
198: September 17, 2013Red Bull's innovative extreme marketing strategy; a profile of legendary wrestler Diamond Dallas Page; one-on-one with former U.S. Open champ Andy Roddick; and, an update on Donald Trump's new Scottish golf course.
197: August 20, 2013Controversy surrounding the Chivas USA soccer club; a profile of surfing legend Kelly Slater; and, a Bob Hewitt sex abuse scandal update.
196: July 23, 2013A teenager's campaign to rediscover the Negro Leagues; an innovative program for war veterans with PTSD; a profile of former first-round NBA draft-pick Jay Williams; and, reconnecting with daredevil Nik Wallenda of the flying Wallendas.
195: June 25, 2013Real Sports profiles Penn State head football coach Bill O'Brien; tells the dramatic story of two cities bonding over tragedy; explores violent attacks on game officials in America; and revisits the fascinating world of horse breeding.
194: May 21, 2013Ultra Soccer fans and Egypt's revolution; NASCAR female pit crew member Christmas Abbott; Update on the Miami Marlins' controversial stadium deal; and, The heart-warming story of pro-boxer Boyd Melson.
193: April 16, 2013Mike Tyson reinvents himself again; transsexual NCAA athlete Gabrielle Ludwig; extreme sports and going too far; and, reconnecting with Olympic hurdler Lolo Jones.
192: March 19, 2013Mixed martial artist Ronda Rousey opens up; Pakistani squash star Maria Toor Pakay fights oppression; and, former national rugby player Gareth Thomas prepares for "Dancing on Ice."
191: February 19, 2013Luther Campbell gives back to kids through football; Houston Rockets' Royce White and coping with mental illness in the NBA; and,�an updated�Harbaugh brothers profile.�
190: January 22, 2013Bryant Gumbel leads fellow Real Sports correspondents in a spirited roundtable discussion of the year's�story favorites.
189: December 18, 2012Jon Frankel learns how�Magic Johnson�plans to change�L.A.'s baseball landscape; Bernard Goldberg speaks with some of the victims of�fan-on-fan violence�and attempts to find out what can be done to curb it; Armen Keteyian hits the Las Vegas strip to meet some of the superb athletes and stars of Cirque du Soleil; and Jon Frankel returns to Grove City, OH�to�reveal how the community is coping with "pay-to-play" in�scholastic sports, who's back on the field, and who's still on the sidelines.
186: September 18, 2012Keeping up with the high-energy, fast-rising broadcaster Jon Gruden; Jon Frankel talks to Steve Gleason about his battle with ALS-Lou Gehrig's disease and his plans to keep living life to the fullest; Erik Compton's first season on the PGA Tour and the numerous health scares he's endured along the way.
185: August 21, 2012Real Sports goes one-on-one with Dodgers all-star Matt Kemp; tells the improbable story of two gymnasts; and reexamines marching band hazing at historically black colleges.
184: July 17, 2012What's ahead for Phil Jackson in his life and whether a return to an NBA team is in his future; Holley Mangold prepares for the fast-approaching London Olympic Games; an inside look at the world's most dangerous race; and catching up with "Lost Boy of Sudan" runner Lopez Lomong as he prepares for the 2012 London track trials.
183: June 19, 2012The phenomenon of Korean women dominating the LPGA Tour; USA�olympian Lolo Jones;�the inspiring story of Micky Wards nephew Ray Greenhalge;�and reconnecting with New York City fireman Matt Long.
182: May 22, 2012Backcountry skiing; Tampa Bay Rays manager Joe Maddon; and the Los Angeles Lakers' Pau Gasol
181: April 17, 2012NY Rangers goalie Henrik Lundqvist; The Flying Wallendas; Steve Delabar of the Seattle Mariners
180: March 20, 2012The tragedy of Russian hockey team, Yaroslavl Lokomotiv; BASE jumper and wingsuit flyer Jeb Corliss; the fall of NFL agent Leigh Steinberg
179: February 21, 2012The Van Gundy brothers; Toradol in the NFL; a high school football team that never punts; and an update on Barret Robbins
178: January 24, 2012Bryant Gumbel leads the Real Sports correspondents in a spirited roundtable discussion of 2011.
177: December 20, 2011Bob Hewitt; Brad Stevens; Marcus Dixon
176: November 22, 2011Jerry West's battle with depression; Obsessive runners; Sports in polygamist communities
175: October 18, 2011The bowl game money trail; Jay Glazer; Dexter Manley
174: September 20, 2011Plaxico Burress; what can be done to revive American tennis; illegal drug use among surfers; the dangers of equestrian eventing.
173: August 16, 2011The Harbaugh brothers; a softball team with incomparable challenges; the running of the bulls; a revisit of Ashrita Furman.
172: July 19, 2011The return of Tiki Barber, La Rue Martin, the bizarre soccer career of Saadi Qadaffi, Ann Wolfe.
171: June 21, 2011Milwaukee Brewers voice Bob Uecker, Bridget Johnson, smokeless tobacco in baseball, Donald Trump.
170: May 17, 2011Real Sports profiles Phillies All-Star Jimmy Rollins; examines health organization P.A.S.T.; explores mini-cheerleading and revisits paralmpic track champ Marlon Shirley.
169: April 19, 2011A departure from our regular format to present a special hour-long edition dedicated to one topic: the state of big-time college sports in America.
168: March 30, 2011


text:
Attract new customers with our unique line of PURESTRENGTH plate-loaded strength training equipment.
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Performance + Ergonomics + Biomechanics
The objective of PURESTRENGTH is to replicate the feeling of using free weights, while optimizing the performance of each routine through the ergonomic and biomechanical design of the equipment.
The application of advanced biomechanics guarantees that our equipment respects the physiological movement of the body without the stress or trauma that using free weights may cause.
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Thanks to the ergonomic design, the push and pull movements become more effective and comfortable due to distributing load more evenly.
The surface provides a grained texture to increase grip and prevent lateral slipping.
The markings indicate correct hand position and the special aluminum, copper and silicon alloy ensures maximum durability.
Body Print System
The special high-density upholstery filling adapts to the shape of the body, providing a stabilizing effect and maximum comfort during exercise.
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There is a large, un-served market of customers who want all the benefits of free weight training, without any of the perceived risks. PURESTRENGTH addresses this market.


text:
Citigroup Global Mkt - Form 8.5 (EPT/RI) RNS Number : 3181S Citigroup Global Markets Limited 29 November 2012 FORM 8.5 (EPT/RI) PUBLIC DEALING DISCLOSURE BY AN EXEMPT PRINCIPAL TRADER WITH RECOGNISED INTERMEDIARY STATUS DEALING IN A CLIENT-SERVING CAPACITY Rule 8.5 of the Takeover Code (the "Code") 1. KEY INFORMATION (a) Name of exempt principal trader: Citigroup Global Markets Limited (b) Name of offeror/offeree in relation to whose Sportingbet Plc relevant securities this form relates: Use a separate form for each offeror/offeree (c) Name of the party to the offer with which exempt William Hill Plc principal trader is connected: (d) Date dealing undertaken: 28 November 2012 (e) Has the EPT previously disclosed, or is it today Yes disclosing, under the Code in respect of any other party to this offer? 2. DEALINGS BY THE EXEMPT PRINCIPAL TRADER (a) Purchases and sales Class of Purchases/ Total number Highest price per unit Lowest price per relevant sales of securities paid/received unit security paid/received GBP GBP 0.1p Sale 52,000 0.4450 0.4450 ordinary (b) Derivatives transactions (other than options) Class of Product Nature of dealing Number of Price relevant description reference per security e.g. opening/closing a long/short securities unit e.g. TRS position, increasing/reducing a long/short position (GBP) (c) Options transactions in respect of existing securities (i) Writing, selling, purchasing or varying Class of Product Writing, Number of Exercise Type Expiry Option relevant description purchasing, securities price date money security e.g. call selling, to which per unit e.g. paid/ option varying option American, received etc. relates European per unit etc. (GBP) Class of relevant Product description Number of Exercise price per unit (GBP) security securities e.g. call option (d) Other dealings (including subscribing for new securities) Class of relevant Nature of dealing Details Price per unit (if security applicable) e.g. subscription, conversion The currency of all prices and other monetary amounts should be stated. Where there have been dealings in more than one class of relevant securities of the offeror or offeree named in 1(b), copy table 2(a), (b), (c) or (d) (as appropriate) for each additional class of relevant security dealt in. 3. OTHER INFORMATION (a) Indemnity and other dealing arrangements Details of any indemnity or option arrangement, or any agreement or understanding, formal or informal, relating to relevant securities which may be an inducement to deal or refrain from dealing entered into by the exempt principal trader making the disclosure and any party to the offer or any person acting in concert with a party to the offer: If there are no such agreements, arrangements or understandings, state "none" None (b) Agreements, arrangements or understandings relating to options or derivatives Details of any agreement, arrangement or understanding, formal or informal, between the exempt principal trader making the disclosure and any other person relating to: (i) the voting rights of any relevant securities under any option; or (ii) the voting rights or future acquisition or disposal of any relevant securities to which any derivative is referenced: If there are no such agreements, arrangements or understandings, state "none" None Date of disclosure: 29 November 2012 Contact name: Cathal McKenna Telephone number: 02890 409544 Public disclosures under Rule 8 of the Code must be made to a Regulatory Information Service and must also be emailed to the Takeover Panel at firstname.lastname@example.com. The Panel's Market Surveillance Unit is available for consultation in relation to the Code's dealing disclosure requirements on +44 (0)20 7638 0129. The Code can be viewed on the Panel's website at www.thetakeoverpanel.org.uk. This information is provided by RNS The company news service from the London Stock Exchange END FERMMMZMMLKGZZM -0- Nov/29/2012 11:17 GMT
Citigroup Global Mkt Form 8.5 (EPT/RI)
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text:
At a Glance
Why Get Tested?
To evaluate kidney function; to monitor the effectiveness of dialysis and other treatments related to kidney disease or damage
When to Get Tested?
As part of a routine comprehensive or basic metabolic panel or when you are acutely or chronically ill with a condition that may cause or be worsened by kidney dysfunction
Sample Required?
A blood sample drawn from a vein in your arm
Test Preparation Needed?
The Test Sample
What is being tested?
This test measures the amount of urea nitrogen in the blood. Urea is produced in the liver when protein is broken into its component parts (amino acids) and metabolized. This process produces ammonia, which is then converted into the less toxic waste product urea.
Nitrogen is a component of both ammonia and urea. Urea and urea nitrogen are referred to somewhat interchangeably because urea contains nitrogen and because urea/urea nitrogen is the "transport method" used by the body to rid itself of excess nitrogen. Urea is released by the liver into the bloodstream and is carried to the kidneys, where it is filtered out of the blood and excreted in the urine. Since this is an ongoing process, there is usually a small but stable amount of urea nitrogen in the blood.
Most diseases or conditions that affect the kidneys or liver have the potential to affect the amount of urea present in the blood. If increased amounts of urea are produced by the liver or decreased amounts are excreted by the kidneys, then urea concentrations will rise. If significant liver damage or disease inhibits the production of urea, then BUN concentrations may fall.
How is the sample collected for testing?
A blood sample is drawn from a vein in the arm.
NOTE: If undergoing medical tests makes you or someone you care for anxious, embarrassed, or even difficult to manage, you might consider reading one or more of the following articles: Coping with Test Pain, Discomfort, and Anxiety, Tips on Blood Testing, Tips to Help Children through Their Medical Tests, and Tips to Help the Elderly through Their Medical Tests.
Another article, Follow That Sample, provides a glimpse at the collection and processing of a blood sample and throat culture.
Is any test preparation needed to ensure the quality of the sample?
No test preparation is needed.
The Test
Common Questions
Ask a Laboratory Scientist
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Due to a dramatic increase in the number of questions submitted to the volunteer laboratory scientists who respond to our users, we have had to limit the number of questions that can be submitted each day. Unfortunately, we have reached that limit today and are unable to accept your inquiry now. We understand that your questions are vital to your health and peace of mind, and recommend instead that you speak with your doctor or another healthcare professional. We apologize for this inconvenience.
This was not an easy step for us to take, as the volunteers on the response team are dedicated to the work they do and are often inspired by the help they can provide. We are actively seeking to expand our capability so that we can again accept and answer all user questions. We will accept and respond to the same limited number of questions tomorrow, but expect to resume the service, 24/7, as soon as possible.
Article Sources
NOTE: This article is based on research that utilizes the sources cited here as well as the collective experience of the Lab Tests Online Editorial Review Board. This article is periodically reviewed by the Editorial Board and may be updated as a result of the review. Any new sources cited will be added to the list and distinguished from the original sources used.
Sources Used in Current Review
Lerma, E. (Updated 2012 October 30). Blood Urea Nitrogen (BUN). Medscape Reference [On-line information]. Available online at http://emedicine.medscape.com/article/2073979-overview through http://emedicine.medscape.com. Accessed November 2012.
Dugdale, D. (2011 May 30). BUN - blood test. MedlinePlus Medical Encyclopedia [On-line information]. Available online at http://www.nlm.nih.gov/medlineplus/ency/article/003474.htm. Accessed November 2012.
(© 1995-2012). Blood Urea Nitrogen (BUN), Serum. Mayo Clinic Mayo Medical Laboratories [On-line information]. Available online at http://www.mayomedicallaboratories.com/test-catalog/Overview/81793 through http://www.mayomedicallaboratories.com. Accessed November 2012.
(Updated 2012 March 23). The Kidneys and How They Work. National Kidney and Urologic Diseases Information Clearinghouse (NKUDIC) [On-line information]. Available online at http://kidney.niddk.nih.gov/kudiseases/pubs/yourkidneys/ through http://kidney.niddk.nih.gov. Accessed November 2012.
McMillan, J. (Modified 2010 January). Chronic Kidney Disease. Merck Manual for Healthcare Professionals [On-line information]. Available online through http://www.merckmanuals.com. Accessed November 2012.
Arora, P. (Updated 2012 March 28). Chronic Kidney Disease. Medscape Reference [On-line information]. Available online at http://emedicine.medscape.com/article/238798-overview through http://emedicine.medscape.com. Accessed November 2012.
Pagana, K. D. & Pagana, T. J. (© 2011). Mosby's Diagnostic and Laboratory Test Reference 10th Edition: Mosby, Inc., Saint Louis, MO. Pp 993-995.
Sources Used in Previous Reviews
Thomas, Clayton L., Editor (1997). Taber's Cyclopedic Medical Dictionary. F.A. Davis Company, Philadelphia, PA [18th Edition].
Pagana, Kathleen D. & Pagana, Timothy J. (2001). Mosby's Diagnostic and Laboratory Test Reference 5th Edition: Mosby, Inc., Saint Louis, MO.
(2002 March). Medical Tests of Kidney Function. National Kidney and Urologic Diseases Information Clearinghouse, NIH Publication No. 02–4623 [On-line information]. Available online at http://kidney.niddk.nih.gov/kudiseases/pubs/kidneytests/index.htm through http://kidney.niddk.nih.gov.
Esson, M. and Schrier, R. (2002). Diagnosis and Treatment of Acute Tubular Necrosis. Ann Intern Med 2002;137:744-752 [On-line journal]. PDF available for download at http://www.annals.org/cgi/reprint/137/9/744.pdf through http://www.annals.org.
Agha, Irfan (2003 August 7, Updated). BUN. MedlinePlus Medical Encyclopedia [On-line information]. Available online at http://www.nlm.nih.gov/medlineplus/ency/article/003474.htm.
Agrawal, M. and Swartz, R. (2000 April 1). Acute Renal Failure. American Family Physician [On-line journal]. Available online at http://www.aafp.org/afp/20000401/2077.html through http://www.aafp.org.
Physician's Reference Laboratory: Kidney Function Panel. Available online at http://www.prlnet.com/Kidney.htm through http://www.prlnet.com.
August 2007) National Kidney and Urological Disease Information Clearinghouse (NKUDIC). Your Kidneys and How They Work. Available online at http://www.kidney.niddk.nih.gov/kudiseases/pubs/yourkidneys/index.htm#rate through http://www.kidney.niddk.nih.gov. Accessed November 2008.
(Update May 15, 2007) MedlinePlus Medical Encyclopedia. BUN. Available online at http://www.nlm.nih.gov/medlineplus/ency/article/003474.htm. Accessed November 2008.
American Urological Association. UrologyHealth.org, Kidney (renal) Failure. Available online at http://www.urologyhealth.org/adult/index.cfm?cat=02&topic=120#top through http://www.urologyhealth.org. Accessed November 2008.
Clarke, W. and Dufour, D. R., Editors (2006). Contemporary Practice in Clinical Chemistry, AACC Press, Washington, DC. Pp. 312-313.
Pagana K, Pagana T. Mosby's Manual of Diagnostic and Laboratory Tests. 3rd Edition, St. Louis: Mosby Elsevier; 2006 Pp. 525-527.


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Events Calendar
Jewel Winds Quintet |
Date(s) | 09/12/2012 - 12:30 p.m. |
Location | Library Concourse |
Presenter | The Professor Harry F. and Mary Ruth Jackson Lunch Hour Series |
Description | Sponsored by the Utica College Social Cultural Committee, programs begin at 12:30 p.m. Musical performances are held in the Library Concourse. Literary readings are held in Macfarlane Auditorium, Deperno Hall, unless otherwise noted.
For details about this event, click here or call (315) 792-3028. Literary programs are made possible with public funds from the New York State Council on the Arts, a State Agency. |
Cost | Free |
Link | http://www.utica.edu/jackson |
Contact | email@example.com |
View Only Cultural Events


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Earth Day Celebration
Sunday, April 26, 2015
1 - 4 p.m.
Kent State University at Stark
Campus Center Lot & Pond Area
Kent State University at Stark is pleased to welcome the community to campus for our annual Earth Day Celebration on Sunday, April 26, 2015.
Enjoy a FREE family-friendly afternoon filled with environmental educational activities, nature walks, games, crafts, mock fossil digs, entertainment, giveaways and much more!
Reservations are not required.
2014 Earth Day Celebration Participants:
- Beech Creek Botanical Garden & Nature Preserve
- Canton Audubon Society
- Canton Country Day School
- College Advantage
- Earth Philosophy
- Kent State University at Stark Student Groups
- Kent State University Environmental Conservation Group
- Kent State University Environmental Health & Safety
- ODNR Division of Forestry
- Project Coyote
- Stark County Health Department
- Stark Parks
- Stark Soil and Water Conservation District
- Stark-Tuscarawas-Wayne Joint Solid Waste Management District
- The Ohio State University Extension
- Vegetarian Club of Canton
- Wildlife Habitat Conservancy
Earth Day Contact:
Kelly Simonis
Special Events Coordinator
Office of External Affairs


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a d
J. Taylor Whaley, MD
The Abramson Cancer Center of the University of Pennsylvania
Ultima Vez Modificado: 25 de octubre del 2012
Myelofibrosis is a rare cancer of the bone marrow in which the marrow is replaced by scar tissue and is not able to make healthy blood cells. It is classified as a type of chronic leukemia and belongs to a group of blood disorders called myeloproliferative diseases. It may also be called primary myelofibrosis, chronic idiopathic myelofibrosis or myelosclerosis with myeloid metaplasia.
A myeloproliferative disorder is a disease in which the bone marrow produces either too many or too few of certain blood cells, most of which do not function properly (each disorder's name comes from the cells that are affected). This group of diseases also includes chronic myelogenous leukemia, polycythemia vera (PV), essential thrombocythemia (ET), chronic neutrophillic leukemia and chronic eosinophillic leukemia. PV and ET can progress to myelofibrosis and account for 10-15% of myelofibrosis cases. Myelofibrosis can progress to other, more aggressive, types of leukemia. The disease can have a variable prognosis with some patients living decades while others progress to more aggressive forms with survival times less than 2 years.
In order to understand myelofibrosis, it is helpful to understand how normal bone marrow functions. The bone marrow is a spongy area in the center of our bones. Its role is to produce blood cells.
Larger bones have more bone marrow, and therefore produce more blood cells. The larger bones include the femur (top part of the leg or thigh), the hip bones, and parts of the rib cage. The bone marrow contains hematopoietic stem cells (or blood stem cells), which are cells that can produce one type of blood cell over and over again. It also contains a small percentage of cells that are in development and are not yet mature. These immature cells are called blasts. Once the cell has matured, it moves out of the bone marrow and into the circulating blood. The body has mechanisms to know when more cells are needed and has the ability to produce them in an orderly fashion.
In the case of myelofibrosis, one blood stem cell acquires the ability to reproduce without regulation, producing large numbers of immature blood cells. When looked at under a microscope, these abnormally produced cells look different then the healthy cells and do not function properly. The body continues to produce these abnormal, non-functional cells, leaving little space for healthy cells. At the same time, these cells release chemicals that cause the bone marrow to become "fibrous" or fill with scar tissue, further interfering with the ability to produce healthy blood cells. In addition, these abnormal cells may be produced in other areas of the body, most often the spleen or liver, which results in an enlarged spleen or liver that can be felt by the physician.
Myelofibrosis is a rare diagnosis, though the exact incidence is not known. It is likely underestimated as less severe and/or asymptomatic disease may go undetected. However, estimates suggest the annual incidence in the United States population to range from 0.3 to 1.5 cases per 100,000 persons.
Aging is the major risk factor for developing myelofibrosis, as the disorder usually develops in people over age 50, with the average age at the time of diagnosis being about 60 years old. About 15% of all patients diagnosed with myelofibrosis are under the age of 50 and about 6% under 40. There are no other known risk factors.
Myelofibrosis primarily develops without a known cause (called primary myelofibrosis);however, other myeloproliferative diseases can progress into myelofibrosis. Ten to fifteen percent of myelofibrosis cases have developed from a diagnosis of polycythemia vera or essential thrombocythemia (called secondary myelofibrosis). Though some families have a predisposition for the disease, it is not passed on or inherited. The disease appears to be caused by a change in a gene that occurs during a person's lifetime. Perhaps this change is due to exposure to something in our environment (which family members may have in common).
Currently, there is no known intervention to prevent myelofibrosis. Additionally, there is no screening test for the disease. There is not a specific test for myelofibrosis, however, routine blood work can be used as a screen to check the red and white blood cell counts as well as the platelet count. These tests can prompt further, more invasive testing, such as a bone marrow biopsy.
About one third of individuals with myelofibrosis have no symptoms, which can make the disease difficult to detect. As the number of abnormal cells increase and healthy cells decrease, symptoms tend to develop, most often related to a low red blood cell count (anemia) or enlarged spleen. Anemia commonly presents as paleness, generalized fatigue, and shortness of breath during activity. Approximately 50-70% of patients report fatigue at the time of diagnosis. People may also develop chest pain or dizziness as the heart has to work harder to get adequate oxygen to the brain and other organs when the red blood cell count is low. A minority of patients report "constitutional symptoms", which include weight loss, low-grade fever, and night sweats. Many patients will also experience intense itching, known as pruritis, secondary to generalized inflammation.
Severe anemia, easy bruising or bleeding and multiple infections can result because of the lack of healthy blood cells. An enlarged spleen (splenomegaly) and/or liver (hepatomegaly) may also occur. When the bone marrow scars, the liver and spleen try to make blood cells to compensate (called extramedullary hematopoiesis), causing these organs to swell. Approximately 25-50% of patients will have symptoms from an enlarged spleen at diagnosis, including pain with deep breaths, loss of appetite and feeling full after eating a small amount (early satiety). Extramedullary hematopoiesis can also occur in other parts of the body (lymph nodes, spinal cord, lungs), causing swelling in these areas, leading to symptoms.
The World Health Organization (WHO) has developed the following criteria for the diagnosis of myelofibrosis. A patient must meet all 3 major criteria as well as 2 minor criteria for a diagnosis of primary myelofibrosis.
Major criteria:
Minor criteria:
CBC is the most common blood test. It uses a machine to evaluate the number of red blood cells, platelets, and white blood cells circulating in the blood. The amount of hemoglobin, the substance which carries oxygen in red blood cells, is also assessed on a CBC. A low red blood cell count is known as anemia, which is common in this disease. Additionally, the platelet counts and white blood cells counts can either be elevated or low.
A small sample of blood can be smeared on a slide and examined under a microscope to count cells by hand as well as examine them for defects.
The body's normal response to anemia is to form new blood cells. Because this occurs poorly in myelofibrosis, immature blood cells are seen in the circulating blood. This is known as leukoerythroblastosis. Red blood cells frequently appear abnormal in myelofibrosis on a peripheral smear with unusual shapes and sizes, reflecting defective production of the red blood cells.
Defects in the appearance of the platelets and white blood cells can also be seen on a peripheral smear. Similar to red blood cells, immature platelets and white blood cells may be seen on a blood smear.
A bone marrow biopsy is required for the diagnosis of myelofibrosis. Generally, this involves local anesthetic being injected into the buttock region and a small core of bone marrow taken out using a needle. A bone marrow aspiration, which is less invasive, is frequently unsuccessful because of the scarring. The scarring will cause the aspiration to be "dry."
In myelofibrosis, the bone marrow typically finds fibrosis, or scarring, of the marrow. Hyperplasia, which is an increase in the precursor cells (the cells that develop into mature red & white blood cells and platelets), is nearly always seen. This increase in precursor cells can be in one or multiple cell lines. For example an increase in the number of megakaryocytes, which will go on to form platelets in the blood, is known as megakaryocyte hyperplasia. These megakaryocytes may also appear abnormal under the microscope. Increased (thrombocytosis) or decreased (thrombocytopenia) platelets can be seen in the peripheral blood of individuals as well.
In 2005, it was discovered that many patients with myelofibrosis have a gene mutation that can be detected with testing. The most common mutation, occurring in 50-60% of cases, is found in the JAK2 (Janus Kinase 2) gene. This gene is important to the system our body uses to signal the need for more blood cells. When the "JAK2V617F" mutation is present, the gene can no longer say "stop making cells", causing the overgrowth of immature, non-functional cells.
For another 5-10% of patients, the abnormality is found in the MPL gene, which is also involved in the pathway that signals for more blood cells to be produced.
The median survival of all patients with myelofibrosis is slightly higher than 5 years. Because survival times range from decades to less than 2 years, it was necessary to create a scoring system to assist physicians in making informed decisions regarding prognosis and treatment options. The most widely used classification system is the International Prognosis Score System (IPSS). The IPSS assigns a point value based on various abnormalities. These points are added up to calculate a score specific to each person. This score can be used as a measure of the aggressiveness of the disease. Each of the following indicators of aggressive disease are given 1 point in the IPSS:
These are tallied to achieve a score, which corresponds to a risk group – low risk (score 0), intermediate 1 (score 1), intermediate 2 (score 2), high risk (score ≥3). Other scoring systems include the Dynamic IPSS and the Dynamic IPSS Plus, which incorporate the need for transfusion of red blood cells as well as unfavorable genetic changes. Additionally, the age at diagnosis and how generally healthy the person is at the time of diagnosis are critical when selecting a therapy. Median survival is up to 15 years for low risk disease (less than 1 risk factor) versus 2-3 years for patients with high risk disease (3 or more adverse features).
Because there are very few curative options for myelofibrosis, most therapies are aimed at minimizing symptoms. Many patients do not meet criteria for aggressive therapies, and treatment is focused on palliation with primary goals of limiting the symptoms associated with the decreased blood counts, improving quality of life, and decreasing the risk of progression to acute leukemia. Generally speaking, patients that are not candidates for stem cell transplant (see below) and do not have symptoms are not treated until symptoms develop. Selecting the appropriate treatment for a patient is very important and a number of studies have demonstrated ways to determine which treatment is appropriate for a given person.
The discovery of JAK2 mutations in 2005 opened the door to development of a targeted therapy for people with myelofibrosis. Following this discovery, several clinical trials began, hoping to take advantage of the mutation and develop a new treatment. Although these agents have proven useful in alleviating symptoms associated with myelofibrosis, none have proven effective as a cure. Collectively, these therapies are known as JAK2 inhibitors.
Ruloxitinib (Brand name- Jakafi) is a JAK2 inhibitor and became the first FDA-approved drug for treating MF patients in 2011. This approval was based on a trial published in the New England Journal of Medicine in 2010, which demonstrated that the drug is well tolerated and led to reductions in symptoms including spleen size, abdominal discomfort, early satiety, bone pain, night sweats and itching. Although Jakafi did not decrease the burden of myelofibrosis in the bone marrow, it did reduce the level of pro-inflammatory cytokines in the blood. It is currently only used in patients with symptomatic intermediate and high risk disease.
Pomalidomide, a new immunomodulator, has also shown some promise. In a study from the Mayo Clinic, it demonstrated improvement in anemia related to myelofibrosis in patients with the JAK2 mutation.
Many medications are the subject of clinical trials for the treatment of myelofibrosis. JAK2 inhibitors continue to be explored in clinical trials. Additionally, immunomodulators, including lenalidomide and thalidomide, appear to have some activity in symptomatic myelofibrosis. mTOR inhibitors, including everolimus, are also currently of interest as well. A trial with everolimus demonstrated improved symptoms in patients with symptomatic high-risk disease. Etanercept, a Tumor Necrosis Factor (TNF) receptor, appears to improve constitutional symptoms in myelofibrosis as well. All of these medications remain under investigation.
Interferon is an immune therapy that works by reducing the overabundance of unhealthy blood cells and reduces the cytokines that lead to fibrosis in the marrow. It appears to work best in those with early myelofibrosis secondary to PV or ET. Interferon has significant side effects that can be difficult to tolerate.
Currently, the only curative treatment is allogeneic stem cell transplant (where the bone marrow comes from a donor). Stem cell transplants have risks related to complications. For this reason, they are generally reserved for people in good health, who are younger than 60 and have a "matched" donor. Current guidelines recommend stem cell transplant only in young patients with high risk disease.
Given that most people present with MF at an older age and that MF is a chronic disease, supportive care is extremely important to limit symptoms and maintain a high quality of life. Supportive care treatments include the following:
A combination of androgen preparation (fluoxymestrone, Halotestin) and corticosteroids (prednisone) can be used to improve anemia. Exogenous Erythropoietin administration has also been shown to improve anemia in 30-50% of patients.
Hydroxyurea: is a drug that is thought to interfere with the synthesis of DNA and is used in the treatment of other blood disorders. It has been shown to decrease the size of the spleen and help control platelet and WBC counts.
Because the enlarged spleen can be a source of discomfort, its removal can alleviate symptoms. Surgical removal is associated with risks as the spleen has an important role in infection fighting. Indications for splenectomy in patients who continue to have splenomegaly despite drug therapy include thrombocytopenia, frequent red cell transfusions and symptomatic portal hypertension. One study of splenectomy in 314 patients with myelofibrosis found that 75% benefitted from the procedure with resolution of severe thrombocytopenia and becoming transfusion-independent. In this study, the benefit lasted for a median of 1 year. If surgical removal of the spleen is not possible, a short course of radiation therapy to the spleen can be given to transiently improve symptoms.


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Jabra GN2000 Series
Jabra GN2000 Series are the perfect headset for blended office environments.
Whether your office uses VoIP, mobile telephony or a combination of the two, the Jabra GN2000 Series enables your employees to enjoy the freedom of hands-free phone calls. Comfortable headsets mean happy employees. Create a more professional office environment. Use headsets that work with your existing systems,seamlessly – thus increasing user adoption and satisfaction.
Check Jabra Compatibility Guide here for compatible headets and adapters needed. (Pop-up Window)


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On Sunday, March 9, 2014, we'll "spring ahead" and return to daylight saving time. What's the best way to prepare for the year's worst Monday morning and recalibrate sleep cycles?
In the short essay below, sleep researcher Michael V. Vitiello, a University of Washington professor, shares advice and his perpsective on the time change. Prof. Vitiello serves as co-director of the Center for Research in the Management of Sleep Disturbance and editor-in-chief (for the Americas) of Sleep Medicine Reviews. He is a content specialist for Wellbody Academy.
Originally proposed by Benjamin Franklin in 1784, (although not adopted until World War I in an effort to save energy) daylight saving time is observed in most of North America and Europe.
This means that the twice annual changing of clock time is again upon us and most of us will lose an hour of clock time as time "springs forward", which means most of us will have one less hour of sleep when we awaken on Monday morning, March 10th.


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Long Corduroy Shirtdress: Long, flattering A-line dress in soft, fine wale corduroy with a touch of stretch. Pair this beautiful shirtdress with tall boots for an instant outfit!
Read what customers are saying about this product:
Rating: 4.33 out of 6 votes cast
Heavily perfumed
I'm chemically sensitive. The scent on this dress is so heavy I can't get near it without a carbon mask. So I haven't tried it on yet. I hung it in the spare room several weeks ago but it still hasn't outgassed. I don't even dare put it in my washing machine 'til it's less smelly.
Townsend, MA
Best buy!!!
I ordered one in chocolate and loved it!!!!!! I then ordered the black and jade as well, these are fabulous vale for the money, the colors are rich, the sizing has enough ease so that it is truly comfortable to wear all day and I get soo many compliments, every time. You can dress it up with jewelry or go more casual with a scarf. I will LIVE in these all winter. Can't say anything else except Chadwick PLEASE make this dress in a navy, a eggplant or a dark purple, a rich olive or forest green, or burdundy paisley like the short version. I will buy every single one!!!!!!!!!It is so classic with boots as well.
A Dress with Flair
This dress is terrific and the best clothng buy I've made in a long time.Ditch the unimaginative uniform of black tights and tops and go with this dress.The fabric quality is excellent, the color I ordered (chocolate) rich and the patch-pocket style is what dressing well is all about: flattering, chic and for me, age-appropriate.I wasn't sure which size to order, given conflicting comments.It's a matter of what's comfortable, how you want to wear the dress (belted or not) and whether a bit looser is more forgiving.The size turned out well: I'd rather the dress have slightly more 'give' than not.I just wore it for the first time with a scarf and boots and I can see this is going to be the dress I reach for time and again. Chadwicks: broaden the color options for this dress to include rust/cinnamon, khaki or a dusty green. Attention Canadian shoppers: ask to speak to a supervisor in Customer Service to place an order, as the regular phone reps are not set up to do it.
Best winter dress!
Great winter dress! The fabric is soft and warm. I wish they had it in more colors like navy blue and ivory.
Comfortable dress
Very comfortable fit. Size runs a bit big, though. May want to order 1 size smaller. Really wish they had more colors in stock. This is definitely a keeper!
Hampton VA
Great casual dress
This dress is exactly what I hoped it would be. I tend to be pear shaped, and this dress fits my small top and passes smoothly over the hip. It begs for accessories and looks great with a chunky necklace or bright scarf, with heels or flats. I nearly always belt it but it is cut to be worn without a belt without looking sloppy. The 50" length makes it perfect for women with religious conventions requiring skirts well below the knee but I took 7" off the hem to make it mainstream (the short version would be way too short for a woman of my age).
New Jersey
Click here to read all of this product's reviews


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No Warning, A Jake Stone Thriller (Book 18), is now available from the following retailers:
Barnes& Noble
Jake and Snowflake encounter their most bizarre challenge yet in this action-packed new installment of the popular Jake Stone series.
"Wild action, loved Snowflake. Peters hit it out of the park in this one."
"Great, fast-paced read. I finished it in one night."
"Peters just keeps them coming harder and faster all the time."
"Loved it, just loved it."


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Ok, but did it taste good?
Phoronix: Celebrating The Phoronix Birthday With A High-Tech Dinner
As many are aware, tomorrow Phoronix will turning be seven years old. A site that many technology enthusiasts and other media publications refer to as being the leading source for Linux hardware information and news/reviews, along with being the home to the Phoronix Test Suite, OpenBenchmarking.org, and other web properties. Sunday will also mark the three year anniversary of the release of Phoronix Test Suite 1.0...
Ok, but did it taste good?
Well, judging by the modern art composition of the plates, and the really scarce amounts of actual food in them, that restaurant's bill won't be light.
For such price, one would at least enjoy what he eats.
So no Dampfnudel...
Should we be worried now?
i'm sure the "Dampfnudeln" will be magic today...
you get what? you can do your own with a Frying pan and a Pan Lid
just make a dough with powder then put water 1-2cm in the pan put the lid on heat it up if it boils put small round pieces of the dough in the 0,5-1cm hot water do it fast put the lid on low the heat to an minimum now wait the pieces inflate now wait for 30 minutes.
now the sausage get milk heat it up put 1-2 spoons of powder into it mix it fast put vanilla into it and sugar or Stevia after that put this into the fridge cool it down to 1-5C.
now back to the pan now finish your job get an melted buttera and paint the dampfnudeln with that now pudere sugar and cinnamon on the balls now pull the stuff out on an plate and put the vanilla sausage around the dampfnudeln and a little bit over it.
and now eat the qaridarium style Dampfnudel!
the most important question that most geeks would think under this circumstance - performance of frestaurant.
Did you get to score?
Wait, no benchmarks?


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Showing 1-24 of 180 items found in Sports & Gaming
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Addison Ice Arena
Addison Ice Arena is one of the premier family ice sports complexes in the Chicagoland area, featuring one NHL rink, one Olympic rink, the Grace St. Bar and Grill, and an Advanced Pro Shop.
Oak Meadows Golf & Banquet
Professional caliber 18-hole golf course. Banquets/meetings up to 400 year-round. Four distinct rooms for meetings, seminars or receptions. Accessible from O'Hare Airport and major expressways.
Dave & Buster's
There's no place quite like it! From hand-crafted tables and world class pocket billiards to the vast array of games in the Million Dollar Midway, D&B will provide one of the most unique entertainment outings you'll ever experience.
K1 Speed
This indoor kart racing and entertainment facility features CyberSport, a multiplayer game that blends bumper cars, lacrosse and basketball into an alternative to racing for groups of 10 to 400.
Golf Club of Illinois
Hailed by Golf Digest as "The best example of target golf yet built," the Golf Club includes pot bunkers, double greens, fescue-covered mounds and water hazards.
McHenry County Pirates Football
Catch all the summer gridiron action of this minor league football team during Saturday night home games played at Jacobs High School in Algonquin.
Paradise Raceway
This slot car racing establishment has three fast tracks and a drag strip.
CITY:Arlington Heights
Arlington Lakes Golf Club
The challenging 18-hole layout spans 90 acres of superbly manicured fairways and greens with 14 beautiful lakes to negotiate during a round. Features clubhouse, snack bar area, pro shop, and patio area.
CITY:Arlington Heights
Nickol Knoll Golf Course
Large bentgrass tees and greens give this fun layout a big course feel. Several shots from elevated tees provide a beautiful view of the area. Features pro-shop, snack bar and club house.
CITY:Arlington Heights
Arlington International Racecourse
At Arlington International, we aim to make every day of the season special by combining the excitement of thoroughbred racing, gorgeous grounds and fantastic food to create a winning track experience for our guests! Whether you're looking for a fun and exciting day out with friends and family, a casual and charming afternoon at the races, or a sophisticated and stylish get-together, Arlington International offers an extraordinary experience for everyone! Live racing and entertainment from May through September or find a full calendar of race dates and events at arlingtonpark.com.
CITY:Arlington Heights
Laser Quest - Arlington Heights
Laser Quest is a family entertainment venue combining the classic games of hide-and-seek and tag with a high tech twist. Our multi-level arenas have specialty lighting, swirling fog and energetic music to add to the excitement for up to 30 or more players in each game.
CITY:Arlington Heights
Fox River Games Paintball
Bring a group of friends to play a round of paintball at Fox River Games. Open play or "walk-on" play is recreational play for all ages (age restrictions) and skill levels. Play as an individual or play in groups. Typically we are open every weekend Saturday and Sunday and during the summer we operate on Wednesdays and Fridays too. Please check our website for our operating schedule and specific hours of operation.
Luigi's Pizza and Fun Center
Enjoy the fun here that includes laser tag, an arcade room and great pizza.
Vaughn Athletic Center and Indoor Water Park
The Vaughan Athletic Center is dedicated to fitness excellence with quality equipment and programs designed to offer members the best value for their fitness dollar. The newest facility in the Fox Valley Park District is the Vaughan Athletic Center located on Indian Trail near Orchard Road on the far west side of Aurora. The Vaughan Athletic Center features 225,000 square feet of athletic, fitness, and aquatic fun. The Vaughan Athletic Center is home to an aquatic center with three indoor pools. The aquatic center offers lap swimming, diving boards, warm water therapy, swim lessons, and aquatic fitness classes. In addition, there is an indoor water park featuring two water slides, a lazy river, and spray and play areas. In the Vaughan Athletic Center's 10,000-square-foot, full-service fitness center, members can use a wide variety of equipment, attend the Fox Valley area's best group exercise classes, and receive personalized, one-on-one help from certified trainers. This year-round indoor water park has tube slides, zero-depth entry and interactive water play.
Aurora Skate Center
Skate to the sounds of Top 40 music. The skate center also features special live organ music dance sessions.
Ultimate Sports Dome
This sports spot features go-karts, batting cages, miniature golf and an indoor driving range.
Airtastic Playland
A fun and exciting play land offering 10 inflatable play structures, four party rooms and two age-appropriate play areas.
Hollywood Casino Aurora
Hollywood Casino Aurora has all of the newest and hottest slots. Denominations ranging from 1 cent to $100 including multi-denomination machines. One blockbuster hit after another, plus find all traditional favorites, popular themes and progressive machines that deliver big pay outs, plus the best variety of table games and Poker Room.
Bartlett HIlls Golf Course
This 18-hole course features a practice range, pro shop, snack shop and banquet facilities.
Apple Orchard Golf Course
Apple Orchard is a par 27, nine-hole course with a pro shop and snack bar.
Villa Olivia Country Club
This Killian/Nugent-designed course features five water holes. An Olympic-size pool is available for free to golfers, along with sand volleyball, tennis, a softball area and more.
Funway Entertainment Center
Illinois' largest family entertainment center. Year round: bowling, bumper cars, roller skating, arcade, lazer tag and Rock-n-Roll mini golf. Seasonal: western trails miniature golf, go-karts, batting cages, soccer cages, jumpshot, Water Wars and bumper boats.


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Post permanent important notices in this area. the news to the right is just for the most recent, happening now sort of thing. This section might have stuff about things that have happened over time. A business might post all their press releases and other press related material here.
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Patient Security
Cottage Health System has increased its security in a number of ways in an effort to provide the safest possible environment for our patients, employees, physicians, and volunteers. Our view is that our responsibility for patient safety begins at the front door.
Patient Identification
Accurate patient identification is essential day and night so the medical and nursing staff can easily see who you are. While you are in the hospital please wear your plastic identification bracelet at all times. Make sure your nurse or caregiver verifies your identification before giving you any medicine or performing any tests or procedures.
Employee Identification
All hospital personnel must wear a picture ID badge at all times. If someone without a badge claims to be a hospital employee, notify a nurse or security personnel immediately.


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Treatments for Rosacea
Rosacea generally cannot be cured, but it can be controlled. Treatment goals are to control the condition and improve appearance. There are several ways to treat rosacea. Your doctor may recommend more than one treatment, depending on your condition. Most dermatologists believe that early treatment prevents rosacea from getting worse. Early treatment may prevent blood vessels from enlarging and rhinophyma (enlarged nose) from developing.
Treatment of rosacea involves the following:
Lifestyle changesMedicationsSurgery
National Rosacea Society website. Available at: http://www.rosacea.org/index.php.


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Main Content
While your opponents move sluggishly in their smothering clothes, you can school those chumps with your Florida style by sporting these super comfy Replica Lacrosse performance shorts from Nike. Granted there are other mesh shorts out there, but not many of them come with a double layer mesh construction and Nike's moisture-wicking Dri-FIT technology to keep you at the top of your game all day long. In addition, these stylish shorts feature contrast-colored mesh panels at the sides, two slash pockets and an embroidered team logo on the right leg for a solid Gators representation.
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Manufacturer direct items are shipped directly from the manufacturer. These items
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Customized items can be personalized with options such as your name, your favorite
number, and/or designs. Some options may be limited by league rules.


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Grocery Retailers in South Africa
Grocery Retailers in South Africa
Published: May 2013
Publisher: Euromonitor International
Product ref: 179884
Pages: 52
Format: PDF
Delivery: Immediate download
Grocery retailers was the least affected by shrinking consumer purchasing power in 2012. The general cost of living significantly increased, with fuel and electricity being the key drivers of a fall in consumers' purchasing power. Grocery retailers succeeded despite the presence of these factors, as can be seen by the 10% current value growth during the year.
Euromonitor International's Grocery Retailers in South Africa report offers insight into key trends and developments driving the industry. The report examines all retail channels to provide sector insight. Channels include hypermarkets, supermarkets, discounters, convenience stores, mixed retailers, health and beauty retailers, clothing and footwear retailers, furniture and furnishing stores, DIY and hardware stores, durable goods retailers, leisure and personal goods retailers. There are profiles of leading retailers, with analysis of their performance and the challenges they face. There is also analysis of non-store retailing: vending; homeshopping; internet retailing; direct selling, as available.
Product coverage: Modern Grocery Retailers, Traditional Grocery Retailers.
Data coverage: market sizes (historic and forecasts), company shares, brand shares and distribution data.
Why buy this report?
- Get a detailed picture of the Grocery Retailers market;
- Pinpoint growth sectors and identify factors driving change;
- Understand the competitive environment, the market's major players and leading brands;
- Use five-year forecasts to assess how the market is predicted to develop.
Euromonitor International has over 40 years' experience of publishing market research reports, business reference books and online information systems. With offices in London, Chicago, Singapore, Shanghai, Vilnius, Dubai, Cape Town, Santiago, Sydney, Tokyo and Bangalore and a network of over 800 analysts worldwide, Euromonitor International has a unique capability to develop reliable information resources to help drive informed strategic planning.
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By market: South Africa (in Africa), Africa
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Analysis of POC Marketplace Enables Identification...
Better In Vivo Models Are Urgently...
Assay Makers Study Markers by Marrying...
Has CRISPR/Cas9 Technology Forced the Question?
(Page 1 of 1)
Major biotech headlines last May included Shire's purchase of Advanced BioHealing, Thermo Fisher Scientific's buy of Phadia, and Geron winning a $25 million loan to advance its human embryonic stem cell (hESC) product for spinal cord injury through clinical development.
Through the takeover of Advanced BioHealing, Shire became a regenerative medicine company, at least partially. Thermo Fisher's deal extended and strengthened its current product line, while Geron morphed into a cancer company from a hESC therapeutic company.
On May 18, 2011, Shire shelled out $750 million for Advanced BioHealing, which agreed to sell itself one day after it planned to go public. That acquisition bought Shire Dermagraft, approved to help heal diabetic foot ulcers. It is an artificial skin product consisting of foreskin-derived human fibroblasts grown on a mesh scaffold.
Advanced Tissue Sciences (ATS), Dermagraft's developer, went out of business in 2002, filing for bankruptcy. Canaan, among other investors, obtained rights to the project and started Advanced BioHealing to commercialize it.
This April 11, Shire made a second acquisition moving it further into the regenerative medicine space. The company reported an agreement to acquire substantially all the assets of Pervasis Therapeutics for its Vascugel product. This endothelial cell-based therapy is being developed to improve the function of arteriovenous (AV) access sites, dialysis access points that connect veins and arteries with tubing. Vascugel has received orphan drug designation from the FDA and EMA.
"About 50 percent of those grafts don't last the first year," Xconomy quoted Kevin Raking, former CEO and chairman of Advanced BioHealing and now head of Shire's Regenerative Medicine, as saying. "But when Pervasis' cells are wrapped around those access points, it improves healing."
Advanced BioHealing was a leader in the field of regenerative medicine with its living cell product Dermagraft, Jeff Jonas, M.D., Shire's svp of R&D, specialty pharmaceuticals and regenerative medicine, pointed out to GEN. The acquisition brought in biomaterials, experience in cell-based manufacturing, and an understanding of the complications of diabetes, he added.
"Shire already had an internal familiarity with the complications of the disease, and we thought we might have some competitive advantage because of our work with Renovo, even though we decided not to move forward with Juvista, so this was a natural fit.
"Pervasis provides a natural evolution of a product line that leverages both our experience with Dermagraft and our manufacturing skill. We already know how to develop a matrix and put cells on it, and now we have another cell-based technology platform that we can leverage to develop additional regenerative medicine therapies.
"Pervasis gives us an opportunity to conduct a clear Phase II clinical trial that will give us a clear go/no-go outcome. This is in line with Shire's desire to make decisions early on in the clinical development process regarding whether or not to move forward with a technology. These products are also in line with our focus on specialist physicians because each of them will be delivered to patients by physicians in specialty practices.
Shire, however, has experienced some bad luck in moving from pills to proteins, following its other acquisitions of biologics companies. In 2005, Shire acquired Transkaryotic Therapies (TKT), now Shire Human Genetic Therapies, in an all-cash transaction valued at $1.6 billion. At the time, Shire said the acquisition was consistent with its strategy and highly complementary to its business model to develop and market products for specialty pharmaceutical markets.
The deal brought Shire two approved recombinant protein products, an enzyme replacement therapy for Fabry disease called Replagal, and Dynepo, a treatment for anemia in patients with chronic kidney disease.
Shire decided to stop marketing Dynepo, citing "changes in the external environment," including the launch of several biosimilars at lower prices. And while Replagal was approved in Europe in 2001 and is avalaible in 46 countries, this March Shire decided to pull its BLA in the U.S. Shire commented that "recent interactions with the FDA have led the company to believe that the agency will require additional controlled trials for approval."
Another big acquisition deal from last May was Thermo Fisher Scientific's takeover of Phadia. On May 19, 2011, the firm ponied up $3.5 billion in cash for the Swedish diagnostics maker, which concentrates on allergy and autoimmunity tests. Phadia is expected to add $190 million to Thermo's 2011 revenue results.
Thermo Fisher Scientific also picked up Dionex, Sterilin, and TREK in 2011, president Marc N. Casper told GEN. "Each of these 2011 acquisitions added key capabilities to both strengthen our competitive position and create value for our customers and shareholders.
"The Dionex acquisition, finalized in May 2011, has enabled Thermo Fisher to continue its leadership in mass spectrometry, gain market share in HPLC, and strengthen its presence in the high-growth life sciences and applied markets," Casper added. "Regarding revenue synergies, we are well ahead of our goal and believe that we'll meet our three-year targets as early as one year ahead of plan. We feel good about the progress on Dionex, and it's great to be tracking ahead of expectations."
Thermo Fisher Scientific may also be hedging against potentially weakening segments and finding high-margin value in its acquisitions. "Thermo Fisher Scientific's operational leverage, diversity, and improving business mix (toward higher-margin businesses) provide superior earnings per share insulation and strong 13–15% growth, both standouts during a challenging funding environment," noted Morgan Stanley analyst Daniel Brennan in The Life Sciences Report.
Last year around this time, the pioneer in human embryonic stem cell (hESC) therapeutics, Geron, seemed to be doing well. On May 5, 2011, it got a $25 million loan from the California Institute for Regenerative Medicine (CIRM) to support clinical study of its Phase I-stage spinal cord injury (SCI) treatment, GRNOPC1. A year later, though, the company is no longer developing GRNOPC1.
In November 2011, Geron halted the trial. The firm said that it hadn't lost faith in the potential of hESC-based therapeutics but had decided, given the scarcity of funds, to focus on its cancer therapeutics pipeline. Geron returned the $6.5 million it had borrowed to date from CIRM plus interest.
"In the current environment of capital scarcity and uncertain economic conditions, we intend to focus our resources on advancing our Phase II clinical trials of imetelstat and GRN1005," CEO John A. Scarlett, M.D., told GEN last November.
Imetelstat, an oligonucleotide, acts as a direct enzyme inhibitor of telomerase. It is currently being tested in cancers including breast cancer, advanced non-small-cell lung cancer (NSCLC), essential thrombocythemia, and multiple myeloma. GRN1005, a peptide-drug conjugate designed to transport paclitaxel across the blood-brain barrier, targets low-density lipoprotein receptor-related proteins (LRPs), specifically LRP-1. GRN1005 is being evaluated in two Phase II trials: brain metastases arising from breast cancer and brain metastases arising from NSCLC.
Geron's departure from the hESC field leaves Advanced Cell Technology (ACT) as the only company testing hESCs. Its hESC-derived RPE product is in two ongoing clinical trials, one in Stargardt macular dystrophy and the other in advanced dry age-related macular degeneration.
Shire, Thermo Fisher, and Geron all continue to look for growth opportunities. Among various strategies, each will evaluate acquisitions that bring them either unique product lines or opportunities to leverage their existing business in challenging economic times. At the same time, they will have to work hard to move drug candidates through the development and regulatory maze, to develop tools and services that target key market segments, as well as to promote existing commercial products.
To change public attitudes about dealing with health issues do you agree that the public officials should rely on fear and disgust instead of encouraging and promoting healthy behaviors?
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The New York City-based media arts center that produces documentary films, provides education courses and has spent decades using the power of the moving image to open eyes and grip hearts, is celebrating its 40th anniversary this week.
Continue reading this entry »You can follow Tom on Twitter @DocSoupMan.
lower east side
Upcoming Events
Herman's House
April 16, 2015
7:00 pm
Brooklyn, New York
This spring, the Brooklyn Public Library is hosting a series of events around Herman's House. On April 16th at 7pm, join the library for a free screening of Herman's House, followed by a Q&A with director Angad Singh Bhalla. The library will also host a full exhibition of Jackie Summell's "The House That Herman Built" from April 16th-June 5th. The exhibit will feature a model of Herman's dream house, a life-size 6x9 foot solitary confinement cell, books from Herman's reading list, blueprints of his dream house, and original letters between Sumell and Wallace. There will also be a number of panel discussions in May on the criminal justice system in New York and the effects of long-term prison sentences and solitary confinement. For more information on the events and to RSVP for the screening, visit the Brooklyn Public Library event listing.
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- ITVS Beyond the Box
- The Chutry Experiment
- The House Next Door
- The Independent Eye
- Tribeca: The Future of Film


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ALLFROZENSEAFOOD.com $20 shipping anywhere in the USA!!
Bring home the same smoked whitefish salad that creates a creamy sensation at many New York City bagel stores and delis. Our salad is made of smoked whitefish blended with mayo and is produced for us by Acme smoked fish, New York's leading smokehouse. This scrumptious smoked whitefish salad is great on bagels, bread, matzos, crackers and more. Add some garnish and make a beautiful presentation at your breakfast, brunch or lunch. Half a pound is only $6.99


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Javier Esposito and Kevin Ryan are two individual homicide detectives, but had they been written as Siamese twins, not much would have changed in terms of the show's trajectory. When they're not making jokes and offering a nice balance to Castle and Beckett's complicated dynamic, these partners are often given the very annoying task of getting statements from every person at a crime scene, despite the fact that there's an actual writer working with them now. Moreover, they are almost always sent to investigate something about the case that doesn't pan out, while Castle and Beckett head right to the source of the problem. We imagine they find this frustrating.
Castle Season 6, Episode 3: "Need to Know" Spoiler Photos — Beckett's in NYC!
Spoiler! Castle Season 6 Spoilers: When Will We See Esposito and Lanie?
Castle Season 6 Official Cast Promo Pictures — First Look! (PHOTOS)
Spoiler! Castle Season 6 Spoiler: Problems Ahead For Lanie and Esposito?
Spoiler! Castle Season 6: Does Ryan's Baby Mean the End of Bromance With Esposito?


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William T. and Gay F. Solomon General Internal Medicine Clinic
Professional Office Building 1
5959 Harry Hines Blvd
11th Floor
Dallas, Texas 75390-9124
Mildred Wyatt and Ivor P. Wold Center for Geriatric Care
James W. Aston Ambulatory Care Center
5303 Harry Hines Blvd.
8th Floor, Suite 200
Dallas, TX 75390-9124
At UT Southwestern Medical Center, our primary care physicians strive to help our patients maintain optimal health and well-being throughout their lives. Our physicians are dedicated to treating patients with attentive and compassionate care in the following primary care services:
UT Southwestern has pioneered many important advances in medicine and patient care, and our primary care physicians are widely regarded as among the best in the nation.
UT Southwestern has one of the largest academic physician practices in the nation and is a major referral center for patients with the most complex conditions. Our commitment to primary care is exemplified by the comprehensive specialties we offer.
Our physicians have the extensive knowledge and expertise that comes from evaluating and treating thousands of patients. On an annual basis, our clinicians provide care to patients during more than 2 million outpatient visits and 89,000 inpatient visits.


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Returning Customers
If you are a registered user, please enter your ART portal username and password.
Quad Cable
Item# 82857
Registered Price [?]
Non Registered Price
- Sales Unit: each
- Dimensions: 12'
Use this 12 cable to plug up to four Metal Pendant
Buttons into the Quizbowl Quizbox.
Additional cables may be purchased to extend the length between
the box and the quizzers.


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The Ad Hoc Charles Houston Recreation Center Naming Committee, established to honor individuals who made outstanding contributions to Alexandria's African American community by dedicating recreation center rooms in their honor, has extended the deadline to submit nominations of honorees for a second round of dedications to Wednesday, October 20.
Specifically, the Committee seeks nominees who fit the following criteria: People who participated in the struggle to advance African Americans economically, socially, educationally, and religiously from the 1920s to 1965, who have made
The goal of the committee, composed mostly of former students of the Parker-Gray School, and other Alexandria schools attended by African American students before integration, is to preserve the history of those whose hard work left a legacy for others to emulate.
Nomination packets may be mailed or dropped off to RPCA, c/o Jack Browand, Division Chief, 1108 Jefferson St., Alexandria, VA 22314. Nominations must be received in person or postmarked by Wednesday, October 20. For more information or to receive assistance in submitting an application, contact Jack Browand at 703.746.5504 or by email at firstname.lastname@example.com.
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First, who's gonna win?
Second, what are the implications for the tournament timetabling if this match goes well into tomorrow afternoon
Third, what kind of rule should be implemented to curtail such ridiculously long games in the future?
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Sputum direct fluorescent antibody (DFA) is a test that looks for microorganisms in lung secretions.
Direct immunofluorescence test; Direct fluorescent antibody - sputum
You will produce a sputum sample from your lungs by coughing up mucus from deep inside your lungs. (The mucus is not the same as saliva or spit from the mouth.)
In the laboratory, antibodies that have been chemically linked to a fluorescent dye are added to the sample. These antibodies are considered "tagged." They will attach to specific antigens -- in this case, the microorganism against which they were formed. If the specific microorganism is present, a bright glow (fluorescence) can be seen in the sputum sample using a special microscope.
If coughing does not produce sputum, a breathing treatment may be given before the test to trigger sputum production.
There is no discomfort.
Your doctor may order this test if you have signs of certain lung infections or pneumonias.
Normally, there is no antigen-antibody reaction.
Abnormal results may be due to an infection such as Legionnaire's disease, mycoplasma pneumonia , or chlamydia pneumonia.
There are no risks.
Limper AH. Overview of pneumonia. In: Goldman L, Schafer AI, eds. Cecil Medicine. 24th ed. Philadelphia, PA: Saunders Elsevier; 2011:chap 97.


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Rising Interest Rates Will End Current Home Affordability
NEW YORK ( TheStreet) -- Home prices are rising, but Americans are paying less.
How's that possible? Because low interest rates have dramatically reduced the size of the typical mortgage payment, according to an by Zillow
"Thanks to historically low interest rates, American homeowners paid almost 37% less per month in mortgage payments in the fourth quarter
In the pre-bubble period of 1985-99, the 30-year fixed-rate mortgage charged between 6% and 13%, compared with just over 3% today. During the pre-bubble years, Americans spent 19.9% of the median household income on mortgage payments for the typical median-priced home. In the fourth quarter of 2012 they spent 12.6%.
At the same time, home prices have gone up relative to income. In the pre-bubble years, buyers spent 2.6 times their median annual income on a home. At the end of 2012 the figure was three times income, due to rising prices and stagnant income. Buyers can spend more when low interest rates allow them to qualify for larger loans.
The national figures mask wide variations around the country. In San Jose, for example, it takes 29.5% of monthly income to make the typical home payment, with the median home price at a whopping seven times median income.
At the other extreme, monthly payments cost just 6.5% of median income in Detroit, where the median home costs just 1.5 times annual income.
What's it all add up to?
Well, this is a pretty good time to buy . Rates remain extraordinarily low, and prices are still well below their bubble-era peaks despite gains over the past year or so. People who have stayed on the sidelines should think about getting that next home while the getting is good .
"The days of historically high levels of housing affordability are numbered," Zillow Chief Economist Stan Humphries says. "Current affordability is almost entirely dependent on low interest rates, and there's no doubt that rates will begin to rise in the next few years."
Higher rates will cause larger payments on loans of a given size, making it harder for buyers to qualify.
"Home values will have to either remain stagnant while incomes catch up or, quite possibly, home values will have to fall in some markets," Humphries said. "This will especially be the case in some markets that have seen strong home value appreciation."
No one expects another nationwide collapse in home prices like we experienced in the middle of the past decade. But it's not uncommon for prices to dip in individual markets. Though today's rates allow borrowers to qualify for large loans, buying the most expensive home you can afford would deepen your losses if prices were to decline.