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9,363 Traditional Laundry Room Design Photos
Popular in Laundry
Northwest Custom Interiors
Iron board in Mudroom — emerlin2
Loudoun County Premier Home Builder
Best of Houzz 2014
Best of Houzz 2014
We have earned a reputation for exquisite design, attention to detail & superior results. Our customers are confident that they have received the best level of service in a courteous professional way.
Suzy Handgraaf
These photos are of the laundry room in my builder spec home. It was a very blah and unfriendly place to do laundry. I added color and accessories to make it a more inviting space. The accordion style drying rack hanging on the wall is an oft used item that I don't see how I ever lived without!
stand alone utility sink — blakejr
Crisp Architects
Floor — mloeck
Studio 76 Kitchens and Baths
Laundry Room/Mudroom off Kitchen
A busy family's back entry was remodeled to include the laundry room, using a former closet behind bifold doors. A stacked washer and dryer saves space. Tile flooring is durable and the larger size tiles eliminate many grout joints.
hamper cabinets — jtopiel
Lloyd's Custom Woodwork, Inc.
Tahoe Charm
This is an exposed laundry area at the top of the hall stairs - the louvered doors hide the washer and dryer!
Photo Credit - Bruce Schneider Photography
Wash we and dryer hidden. — kmeyerselfkah09
Cory Smith Architecture
712 W. Bauer
Perfect way to stay organized for large families!
Cory Smith Architecture
Laundry room — rosiebrooke


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Celebrate the incredible stories and facts behind the Giant's Causeway
Hike cliff paths for unforgettable views of the causeway
Want to know how to take the best possible picture of the Causeway? The experts share their tips
Dramatic cliff headlands & views, secluded bays, castle ruins
Rugged coast, spectacular views over the Mull of Kintyre, crashing waves and historical intrigue ...
A thrilling boat trip to the upsidedown lighthouse on Rathlin Island
Luxury, Four Star Boutique Hotel in the heart of Northern Ireland's Causeway Coast.
Dunluce Castle is dramatically located atop a cliff on the north coast.
This year celebrate all things Irish at the Giant's Causeway Visitor Centre and the Causeway Hotel.
Holiday ideas, news, offers… sign up for our ezine and we'll keep you in touch with Ireland.
By sea or by air, find the best route here
Lose yourself in some of Ireland's most inspiring countryside and vibrant cities.
Got more questions about Ireland? We've got answers
Can we help you with anything else? Find out how to get in touch by email, Facebook and more.
For more information on travel, accommodation and attractions have a look at our brochures and maps.
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If you're a visitor from the island of Ireland you may want to visit one of these sites… Otherwise proceed to Ireland.com

subset: commoncrawl


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More to explore:
kitchen#kitchen decorating #kitchen interior design #kitchen design ideas #kitchen decorating before and after| http://kitchendesignsaz857.blogspot.com
white kitchen with dark cabinets
modern and rustic kitchen
Scandinavian style in Amsterdam6
white kitchen, brass accents, design by Andrew Raquet Interior Design, foto Jose Picayo from Clover Magazine ~~
plan de travail inox
Interior Design
rich, moist chocolate lavender cake with mascarpone earl grey german buttercream by Beth Kirby | {local milk}, via Flickr
Black and white wall
Elizabeth Hudson my old boss xx
Very cool, JEX! :)
simple space with a black accent wall
kitchen space. Hardwood floors, white cabinets, galley kitchen
white plank floors


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Company Overview of Easy Rental Equipment Inc.
Company Overview
Easy Rental Equipment Inc. was founded in 1984 and is based in Ponce, Puerto Rico.
Bo. Vallas Torres
Lote 5
Ponce, PR 00731
United States
Founded in 1984
Key Executives for Easy Rental Equipment Inc.
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Dritz Quilting Washaway Wonder Tape-1/4''W x 10yds
Item # 7140288
Online Availability:
In Stock
Product Actions
5 In Stock
Jo-Ann Fabric and Craft, 0115 (Change)
46301 Potomac Run Plz Ste 160
Sterling, VA 20164
Sun 10a - 6p, Mon - Fri 9a - 9p, Sat 9a - 9p
Additional Information
Create fun craft projects with the Quilting Washaway Wonder Tape. This tape comes with a peel-off backing paper to uncover double-sided adhesive for appliques, pockets and decorative items. Hold fabric and paper pieces together using this wonder tape. Use it to add lovely embellishments to your scrapbooks, journals, greeting cards and home decor accessories. You can also use it to join water-soluble stabilizers and quilting pieces together.
- Brand: Prym Dritz
- Dimensions: 0.25 x 10 yards


text:
Job description reports to and housekeeping departments so that they provide superior customer service and operate the hotel to task checklist (3)
Proper preparation for any inspection is a long term process involving all departments of the hotel, not just the housekeeping the task of preparing the daily
A checklist should outline good housekeeping page 20 compiled by judith soskin in a hotel is no easy task. room attendants' work.
Generic hotel bed bug plan housekeeping (rooms and laundry) and maintenance department headboard task on the guest room preventive maintenance checklist.
Deep cleaning task checklist room attendant daily report establishment such as an inn or hotel, who performs housekeeping
Hotel#housekeeping# • 6)+check+all+checkoutrooms+daily+to+clarify+guesthas+ room+ • 4)
Your hotel is a lot to manage. • the housekeeping book • room attendant's book • goal builder daily checklist initial
Task trainer initials trainee initials date responsible for being the hub of communication for the hotel as well as basic housekeeping operations training


text:
5 days when you won't be bothered by the IRS
- Kent Hoover
- Washington Bureau Chief
- Email | Twitter | Google
The Internal Revenue Service is shutting down – well, for five days, at least.
In response to the budget cuts imposed by sequestration, IRS offices will close on May 24, June 14, July 5, July 22 and Aug. 30. Agency employees will get unpaid time off on these days.
Two of these days off occur shortly before deadlines for certain taxpayers. June 17, for example is the tax-return deadline for Americans living abroad, as well as the due date for estimated-tax payments for the second quarter. Sept. 3 is the deadline for truckers to file a highway-use tax return. In these cases, taxpayers should keep these furlough dates in mind if they need to contact the IRS before these deadlines.
The IRS also noted that it won't be able to accept electronically filed returns on these furlough days.
The Treasury Department's Electronic Federal Tax Payment System, which businesses use to pay excise taxes, will continue to operate on these days.
Taxpayers will get an extra day to comply with requests to provide documents to the IRS, if the deadline falls on a furlough day.
The agency indicated it also may have to close on one or two additional days in September, depending on how much money it has left then.
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- Allegiant Air to offer flights at Memphis International Airport
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- Allegiant's unconventional model keeps airfare low
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- Del-Nat Tire sold
COO and Senior Vice President (SVP) of Operations Ecumen | Saint Paul, MN
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Speed Racer - Neon Speed Posters
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Los Angeles - Lily Allen has given birth to a baby girl.
The Not Fair hitmaker and husband Sam Cooper are believed to have welcomed their first child into the world today on Friday, though her spokesperson declined to comment.
However, a friend told the Mail Online website that the birth had been "all good", while Lily's former boyfriend Seb Chew tweeted: "sending out love to lily, sam and mini cooper......YES.(sic)"
On Friday, Lily also took to her page on the social networking website to simply write: "Totes amaze (sic)"
The news comes just over a year after the 26-year-old star lost the couple's baby six months into her pregnancy.
The singer - who also suffered a miscarriage in January 2008 - and Sam married in June, and the pair broke the news they were expecting a baby at the wedding reception.
A source said at the time: "There were tears all around. This is the happiest day of Lily and Sam's lives and they are so in love. This is the icing on the wedding cake!"
- BangShowBiz
24.com publishes all comments posted on articles provided that they adhere to our Comments Policy. Should you wish to report a comment for editorial review, please do so by clicking the 'Report Comment' button to the right of each comment.
The Top Billing host and radio DJ is working on another secret TV project. Read More »
Taylor Swift has revealed her good friend Jaime King has asked her to be godmother of her and husband Kyle Newman's impending child. Read More »
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AuthorsDen.com Join (Free!) | Login
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Featured Authors: Francis Eaden, iR. Glenn Brown, iBarbara Henry, iM. Pritchard, iShelley Costello, iBettye Johnson, iWill Clark, i
· Become a Fan · Contact me · Success story · Articles · Poetry · Stories · Blog · 29 Titles · 266 Reviews · Save to My Library · Share with Friends! · Member Since: Jul, 2006 Joyce I Devenish, click here to update your pages on AuthorsDen.
Featured Book Deflector by Terry Vinson
Featured Book Eternal Recurrence ...a step out of time by Tom Kitt
Shakespeare is a favourite of mine. In his time the greatest dramatist that England produced. Maybe the best until motion pictures.
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All my days I've tried to write a plot to match the BARD.
Never, ever could I dream a plot of quality,
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Nor poet am I.
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Until I die.
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Sydney: Mar 10, 2014
Australia's Twenty20 opening batsman, Aaron Finch has said that his team is motivated after watching a hobbled Ryan Harris bowl Australia to a thrilling Test series win against South Africa.
Finch has said that the Australia T20 team was huddled around the team room watching the end of the final match against South Africa and it was getting "quite nervous".
According to the Sydney Morning Herald, Finch said that the team was inspired by the way Harris was bowling on one leg and the way Australia won were "amazing scenes". Australia's T20 captain George Bailey, reportedly arrived in Cape Town ahead of Australia's three-match T20 series against South Africa and after meeting Harris, who was in so much pain he could "barely walk".
He said that the "legend" of Harris grows "bigger and bigger". Bailey also said that Harris "just gives so much" to the Australian cricket team despite the pacer's recurring knee injury, the report added.
WC 2015, 25th ODI Match, Pool B
Wed, Mar 4, 2015 at Napier
Pakistan won by 129 runs
WC 2015, 24th ODI Match, Pool B
Tue, Mar 3, 2015 at Canberra
South Africa won by 201 runs
WC 2015, 23rd ODI Match, Pool B
Sun, Mar 1, 2015 at Brisbane
Pakistan won by 20 runs


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Remove leaves from apple trees?
jenn(SoCal 9/19)January 21, 2012
We have 2 apple trees which we planted last spring: Anna and Dorsett Golden. They are planted about 8 feet apart.
Anna has dropped almost all its leaves. Dorsett Golden still has most of its leaves and also a few blossoms.
I own the excellent "Growing Apples in the City" but cannot find instructions specifically saying "Always pull off all the leaves of your apple tree in Spring".
Yes or No: Should we pull the remaining leaves off our Anna and Dorsett Golden every year? If so, is January the best time to do this?


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Ayesha Ali: Mum and lover found guilty of killing eight-year-old girl
Two women wrapped up in a bizarre relationship based on cyber-fantasies face long prison sentences today for killing an eight-year-old girl.
Two teenagers held on suspicion of murder of schoolboy in Islington
Two teenagers have been arrested on suspicion of murdering a schoolboy who was stabbed to death while riding his bike with friends.
Shocking footage shows moment bus driver stabbed in the eye with Stanley knife in south-east London
This is the moment a bus driver was stabbed in the eye by a man who walked out in front of his vehicle with a child in south-east London.
London councils urged to copy borough merger and save £400m
Related Articles
Published: 18 June 2012
A senior Cabinet minister today called on councils across London to merge to save taxpayers hundreds of millions of pounds.
Eric Pickles wants others to follow the example of Hammersmith and Fulham, Kensington & Chelsea and Westminster councils, which have formed a "tri-borough" agreement, saving tens of millions every year.
Mr Pickles said that if every borough in London followed their example, taxpayers would be £400 million better off. The Secretary of State for Communities and Local Government added that as much as £2 billion could be saved if the scheme were to be rolled out nationally.
Under the tri-borough agreement, the three councils are sharing £300 million of services, cutting management costs by shedding 175 senior posts.
Kensington & Chelsea and Hammersmith and Fulham also share a chief executive. The councils today said they are on track to save £40 million thanks to the partnership, which started last year.
Mr Pickles today said that "no council should even contemplate cutting services before they have considered following this example".
He added: "The success of the tri-borough approach is testament to the innovation and forward thinking of Westminster, Hammersmith and Fulham and Kensington & Chelsea.
"Its councillors and officers are my London 2012 heroes for making such creative savings to protect frontline services, all while keeping council tax bills down, helping families and pensioners with their cost of living."
Mr Pickles said: "No council should even contemplate cutting services before they have considered following this example of how to join forces with neighbours to share back-office services, procure better, slash in-house management and cut overhead costs.
"Potentially £400 million of taxpayers' money could be saved in London if other councils were able to copy such sensible savings."
Nicholas Botterill, the new leader of Hammersmith and Fulham council, said: "A year ago our plans were seen as radical, now they are seen as commonsense. Sharing costs, overheads and expertise is not only bringing down the cost of delivering services it is leading to better and more effective services."
And Philippa Roe, leader of Westminster City Council, said: "Sharing services is not just about reducing cost, important as that is in this age of austerity, it is about improving lives and innovation."
Merrick Cockell, the leader of Kensington & Chelsea, said: "It is up to other councils to decide whether sharing services makes sense.
"Many have already gone down this route. We are anxious to share our learning and experiences to help them in their future thinking."
Top stories in News
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Top stories in News
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"So Much (Scoring) Funk We All Get Seconds!" - 1/8/09 - Nashville vs. Pittsburgh
:nashville vs. http://www.granitegrok.com/pix/The%20Penguin.jpg
@ http://www.orbitcast.com/archives/xm_sommet_center.jpg No previous meetings. Nashville (17-19-3, 37pts, 4th Central, 14th West) About Nashville: Last Game: Colorado 2 @ Nashville 1 Last Ten Games: 3-7-0 Home Record: 10-7-1 OT/Shootout Record: 5-3 Goals For: 94 Goals Against: 114 Power Play: 12.8%, 28th Home Power Play: 12.8%, 25th Penalty Kill: 83.4%, 8th Home Penalty Kill: 86.4%, 4th Goals: Jason Arnott, 15 Assists: Jean-Pierre Dumont, 25 Points: Dumont, 32 (7g+25a) Power Play Goals: Arnott, 5 Power Play Scoring: Dumont Short Handed Goals: Five Tied at 1 Short Handed Scoring: Scott Nichol, 3 (0g+3a) Shots: Weber, 125 Shooting Percentage: Ryan Jones, 16.7% Plus-Minus: Weber, +8 Penalty Minutes: Jordin Tootoo, 72 Hits: Weber, 86 Blocked Shots: Greg Zanon, 111 Giveaways: Dumont, 27 Takeaways: Dan Hamhuis, 30 Turnover Differential: Joel Ward, +19 Faceoffs Won: Arnott, 268-241 Faceoff Percentage: Radek Bonk, 62.8% Time On Ice: Weber, 24:43 Wins: Dan Ellis, 10-14-3 Minutes Played: Ellis, 1571 Goals Against Average: Pekka Rinne, 2.44 Save Percentage: Rinne, .911 Shutouts: Rinne, 3 Nashville Fun Fact: Pending Nashville Player to Watch: Pending Nashville Reason to Worry: Pending Nashville Injury Report Scott Nichol (Concussion, Out) Steve Sullivan, (Back, Out) Nashville Projected Lineup Santorelli-Arnott-Erat Jones-Legwand-Dumont Fiddler-Bonk-Ward/Tootoo Smithson-Peverley-Belak Weber-Suter Hamhuis-Zanon De Vries-Klein Rinne Pittsburgh Penguins (20-17-4, 44pts, 4th Atlantic, 9th East) About Pittsburgh: Last Game: Last Ten Games: 3-7-0 Away Record: 11-8-2 OT/Shootout Record: 7-4 Goals For: 124 Goals Against: 125 Power Play: 17.0%, 19th Away Power Play: 13.1%, 27th Penalty Kill: 80.3%, 21st Away Penalty Kill: 78.1%, 21st Pittsburgh Team Leaders Goals: Sidney Crosby, 16 Assists: Evgeni Malkin, 48 Points: Malkin, 63 (15g+48a) Power Play Goals: Petr Sykora, 10 Power Play Scoring: Malkin, 22 (4g+18a) Short Handed Goals: Malkin, 2 Short Handed Scoring: Malkin, 2 (2g+0a) Shots: Malkin, 142 Shooting Percentage: Sykora, 16.3% Plus-Minus: Malkin, +18 Penalty Minutes: Eric Godard, 106 Hits: Brooks Orpik, 151 Blocked Shots: Rob Scuderi, 102 Giveaways: Malkin, 42 Takeaways: Malkin, 42 Turnover Differential: Scuderi/Hal Gill, +7 Faceoffs Won: Crosby, 419-428 Faceoff Percentage: Maxime Talbot, 55.4% Time On Ice: Ryan Whitney, 24:49 Wins: Marc-Andre Fleury, 12-9-2 Minutes Played: Fleury, 1395 Goals Against Average: Dan Sabourin, 2.72 Save Percentage: Fleury, .905 Shutouts: Fleury, 1 Pittsburgh Fun Fact: Pending Pittsburgh Player to Watch: Petr Sykora has 7 goals and 15 points in 20 games all-time against Nashville, as well as three goals in his last six games. Pittsburgh Reason to Worry: Evgeni Malkin carries a 9 game goal-scoring drought into tonight's game which has shown no signs of slowing down. Pittsburgh Injury Report Mike Zigomanis (Undisclosed, Out) Sergei Gonchar (Shoulder, Out) Pittsburgh Projected Lineup Dupuis-Crosby-Kennedy Fedotenko-Malkin-Sykora Cooke-Staal-Talbot Satan-Jeffrey-Godard Whitney-Orpik Letang-Scuderi Eaton-Gill Fleury |
I remember the last time that the Pens came to town, we got a huge lead and then started screwing around and almost let them come back.
So, WFOME you're expecting Santorelli - the center - to be suddenly placed on the first line wing tonight? I'm personally thinking he'll see some time at wing, but will start as the second line center with Legwand bumped down a peg.
Out lineup has changed considerably with Feds and Dupuis being hurt.
Kennedy-Crosby-Staal Satan-Malkin-Sykora Cooke-Talbot-Goligoski (yikes) Wallace-Jeffrey-Godard I'm preparing the GDT over in the Pens forums, I'd like it if you could come over and make sure my info about the Preds is right. Good luck tonight! |
So good luck on guessing our lines. My guess would look more like this: Fiddler - Arnott - Erat Dumont - Legwand - Jones Ward - Santorelli - Tootoo/Pihlstrom Smithson - Bonk - Belak scratched: Peverly, Tootoo/Pihlstrom |
that Arnott montage was THE ****.
Ridiculously bad giveaway in front of the net leads to an easy goal on what was probably the second shot of the period for the Pens (oh and it came at the 15 minute mark)............*sigh*
A hard clear by Suter directly into Arnott and promptly into the back of the net....Unbelievable.... Edit: Trotz inexplicably changes goalies, and a quick cross ice goal is given up..... |
Did Ellis get pulled or injured?
What is it with this station and its horrible audio?
Wow. If we get the number of good bounces in the next 15 games needed to even out the bad ones in the last 15, this will be the game that we point to that changed the season.
The first time in about two months that we have scored more than 3 goals.......Ironically, it was about two months ago that we sent down both Jones and Hornqvist.....
Hmmmmmmmmm |
But we were giving up 6-8 goals a game so cudos to the staff down there-- they improved Jones' defense by 3-5 per game.;)
Helluva comeback, Ward worked his ass off, shame he missed the open net.
I've been clamoring for Legwand on the wing for two years...it finally happens and he and Erat come alive.
Dumont Santorelli Jones was the best line of the night, however...even if it didn't show on the scoresheet. I'd like never to see Rich Peverley in another game. |
I agree, I thought Santorelli, Dumont and Jones was the best line, they had very little play in their own end and they seemed to generate very good offense even if they didn't bury any. As for Santorelli, I thought he was very impressive he got a little time on the PP and set up a beautiful chance in the slot there and the rebound got denied by Fleury. I wouldnt change a thing going against Chicago on Saturday?
What was everyones thoughts on Santorelli? Santorelli or Peverley? |
ugh....yeah, great win.....but they decide to wake up for an EASTERN CONFERENCE TEAM......gotta do that **** for the western teams
Re scratches: Richy and VK(see reason below) can sit the rest of the year....getting waived or traded would work too. Quote:
The only time Mi. Santorelli's line got pinned in the d-zone is when VK failed to keep a puck in at the blueline resulting in an odd man rush the other way. Sure the puck was bouncing but he played it the same way Zidlicky used to....with his stick(and missed just like Zidlicky used too) instead of his body like Suter, Weber, Hamhuis, Zanon, Klien, DeVries, Sulzer, or Franson(noticing a trend?) would have. Why is it that "offensive" defensemen from Europe don't understand that the most important thing is "keeping the zone" and not making SportCenter? The Preds were inches from giving up a goal because of his failure to hold the zone. Trade VK for a single puck just so that we can bring up Sulzer and sit DeVries for the rest of the year please.?.?.?.? . . . . . . . Okay I'm done now |
He needed that time to play the same game as the NHL only at the pace of the AHL. He seems much more aware and at least equal to the game if not a half step ahead. Before he was almost a full step behind. |
Legwand looked great on Arnott's wing. I like that line. So much so that it makes inserting Sullivan into the lineup that much more difficult. Legwand-Arnott-Erat Jones-Santorelli-Dumont Sullivan-Fiddler/Bonk-Pihlstrom Tootoo-Bonk/Fiddler-Belak/Ward Smithson, Ward/Belak Maybe? I didn't think Smithson and/or Ward played that poorly last night, but I think we need insulation for Sullivan. Quote:
I liked what I saw from Santorelli... his passes were sharp, through traffic.
Erat had a good game, you could tell he was frustrated after he scored. I felt bad for Ellis, I mean to get pulled for the 2 goals that were scored... He was ticked too I read a quote: Quote:
It's going to be interesting to see what Trotzy does with Sully. |
Funny... Ive always inserted a word also starting with "F" and ending with "king" to describe Rich Peverley myself. :laugh: |
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text:
Sweet and Salty Granola Bars
Made This Recipe? Add Your Photo
Total Time
10 minsCook
25 minsHere's a homemade Garnola Bar that offers both the sweet and salty fan in your family something to cheer about. Crunchy seeds and nuts paired with Eagle Brand Sweetened Condensed Milk and semi-sweet chocolate chips make these a summer staple for families on the go!!
- 3 cups oats (Robin Hood or Old Mill)
- 1 cup toasted almond, coarsely chopped
- 1 cup sunflower seeds (or a combination of both) or 1 cup pumpkin seeds (or a combination of both)
- 1 cup raisins
- 1 (300 ml) caneagle brand sweetened condensed milk (regular or low-fat)
- 1⁄2 cup crisco all-vegetable shortening, melted
- 1 tablespoon kosher salt
- 1 cup semi-sweet chocolate chips, melted
- 1⁄4 teaspoon kosher salt
- Preheat oven to 350°F Line a 15x20-inch jelly roll pan with parchment paper.
- BARS: Combine all ingredients for bars in large bowl. Mix well. Press evenly into prepared pan.
- Bake in preheated oven 25-30 minutes or until golden brown. Cool slightly.
- TOPPING: Combine melted chocolate and Kosher salt. Drizzle over bars. Allow chocolate to harden. Cut into bars.
- NOTE; When lining a pan with parchment paper, overlap the sides to allow for easy removal and cutting of squares.


text:
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Apple iPad a Breeze to Grab at Best Buy in Connecticut Town
Apple iPad a Breeze to Grab at Best Buy in Connecticut Town
Clint Boulton
Posted 2010-04-03
The Best Buy in Trumbull, Conn., began selling the Apple iPad at 10 a.m. April 3, where the retailer offered the easiest route to acquire the tablet. Only five people waited an hour before the store opened, a stark contrast to the hundreds of people camped out in front of Apple Stores and Best Buy locations in New York City and California. Consumers were easily able to play with and purchase the devices, which are available in WiFi only with capacities of 16GB, 32GB and 64GB for $499, $599 and $699, respectively. eWEEK spoke to some of the consumers who waited for the iPad here.
TRUMBULL, Conn.-While people in
New York City
camped out in front of Apple stores for a chance to buy the Apple iPad, the best place to pick up one of the hallowed devices may have been at the
Best Buy in
, a small town in Connecticut's Fairfield County. By 9 a.m. on April 3, Best Buy employees had handed out only a handful of tickets to people interested in purchasing one of the
tablet computers
, which are available in WiFi only with capacities of 16GB, 32GB and 64GB for $499, $599 and $699, respectively. Versions of the iPad with 3G cellular service from AT&T go on sale later this month for $629 (16GB), $729 (32GB) and $829 (64GB).
Analyst estimates for units sold vary, but researcher iSuppli expects Apple to
see sales
of 7.1 million iPads in 2010. The Federal Communications Commission published a complete teardown of the device, which
iFixit details here
Trumbull resident Greg Dowling, vice president of analytics for consulting firm Semphonic, pulled into the Best Buy parking lot around 9 a.m. and was first of about five people to receive a "pre-sale ticket," a sort of lottery invoice for consumers to lay claim to a product. Best Buy didn't open for an hour, so Dowling waited patiently by his car with his ticket, good for one 64GB model. He was checking his smartphone, an Apple iPhone, of course. Dowling felt comfortable that he would have no trouble picking up an iPad in town and did not preorder one for that reason. "I come here because I live in Trumbull. Why bother going to [the Apple Store] in Stamford?"
However, he said he was still surprised he didn't have to wait in a long line, not that he was complaining. Dowling said he had been a PC user for many years because the computers he used in the industry for work were machines based on the Microsoft Windows operating system. "I'm not an Apple fanboy, and overzealot or a Mac Nazi, but the ease of use and the simplicity of the interface are probably what drew me to [Mac computers]," Dowling said. "And then as soon as there was an Intel-based chip inside and I could run both Windows and Mac OS on one box, that's what I did for two years and I haven't looked back." Dowling uses a MacBook for work and an iPod Touch for pleasure. Now he has an iPad with which to play. "Right now, I can't think of doing anything outside the Apple world when it comes to fixed Web, mobile Web and mobile application integration," he added.
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Smooth Creations Hellcat Core i7 965 EE Gaming System
Smooth Creations is back once again and this time we get a look at their most powerful system to date.
Earlier today we took a look at CyberPower's new Black Pearl Core i7 system and it scored high marks for 3.6 GHz performance at an excellent price point. But now it's time to throw all of that to the wind because it doesn't compare to the massive power put out by the Smooth Creations Hellcat, or its price.
The mad scientists at Smooth Creations took Core i7 to a new level, one that only a few will try to achieve and even fewer will succeed. The Smooth Creations Hellcat comes from the factory clocked at an amazing 4.0 GHz, yet doesn't have a problem with stability thanks to a who's who list of Danger Den products. The madness doesn't stop there; the dual 4870 X2 cards running in CrossfireX mode are water cooled as well and we can thank Danger Den for that. But the real congratulations go to Smooth Creations for putting together such a beast and having it ready for launch day. Don't call this just another liquid cooled system; the Hellcat is lubricated on the fat of unbaptized children.
As you already know, today is the day Core i7 and its counterpart, X58 get released into the market. Core i7 is already a big hit with enthusiasts even though so few already have a system. I have been told by some of the shops who accepted preorders that Core i7 parts are the hottest ticket in town and they expect to sell quite a few units today. One way to ensure an early i7 system is to go with a custom system builder.
Smooth Creations, long known for their quality art used by so many system builders and their work with Cooler Master on the CSX line has opened up a boutique shop to satisfy the needs of those looking for the ultimate. Today we are going to take a look at Smooth Creations' flagship system, the Hellcat; a 5,400 Dollar beast that will devour your benchmarks while ingesting bone, skin and your wallet.
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Bill to limit tattoo, piercing artists moves forward Updated: 13 mins ago A state bill to rein in tattoo and piercing artists is moving forward in Santa Fe.
BLM burns acres of Carlsbad land Updated: 20 mins ago The skies near Carlsbad might be grey sometime this week but it won't be because of weather.
Drivers who pay at the pump for gas beware By Crystal Gutierrez Published: March 26, 2014, 6:20 pm Click to share on Twitter Click to share on Google+ Share on Facebook Click to share on Pinterest SANTA FE (KRQE) – Santa Fe police are investigating a massive fraud cause where someone was getting the tools to rip people off while they were filling up at the pump. Detectives said crooks have gotten their hands on the account information of hundreds of people. Police said more than 600 Del Norte Credit Union customers alone were hit. A Del Norte representative told detectives the link is a Giant gas station on Sawmill road off of Saint Francis and I-25. Santa Fe Police Spokesperson Celina Westervelt said detectives believe the criminals possibly did it right under the victims' noses. They think he may have used a device known as a skimmer. "Crooks can use that skimmer and put it right over where you use that gas pump and they're able to obtain all of your personal information," she said. Skimmers read account numbers and the pin or zip code used to activate it. Police aren't sure what kind of skimmer was used. They don't know if it stored the information and the crooks picked it up or if the device sent it electronically. Police said fraudulent charges have already been racked up on 17 accounts. Westervelt said one victim is a Santa Fe police officer. "His wife actually banks at Los Alamos Credit Union," Westervelt said. "They were taken for a $1,000 on that charge." Police worry the number of victims will rise before a suspect is caught. "Scammers don't usually target just one gas station or just one bank. They do it through multiple areas of the city," Westervelt said. "If you think about how much traffic a gas station has within a day – it could be thousands." Police don't know if the crook or crooks are from Santa Fe but said the fraudulent charges were made in Las Vegas and Henderson, Nev. KRQE News 13 called Western Refining, who owns the Giant gas station, for comment but they didn't get back to us. Police are encouraging drivers to pay inside until they figure out who's behind the crime. Share this:Click to share on Reddit (Opens in new window)Share on Facebook (Opens in new window)Click to share on Twitter (Opens in new window)Click to share on Google+ (Opens in new window)Click to share on StumbleUpon (Opens in new window)Click to email this to a friend (Opens in new window)Click to send this to your Kindle device (Opens in new window)Click to print (Opens in new window)MoreClick to share on LinkedIn (Opens in new window)Click to share on Tumblr (Opens in new window)Click to share on Pinterest (Opens in new window)Click to share on Pocket (Opens in new window)Like this:Like Loading... Related Advertisement


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Publications (36)135 Total impact
- [Show abstract] [Hide abstract]
ABSTRACT: Progranulin (PGRN), an autocrine growth factor, has multiple physiological functions and is widely involved in the pathogenesis of many types of diseases. The pivotal anti-inflammatory function of PGRN in rheumatoid arthritis encouraged us to examine the role of PGRN in acute kidney injury (AKI). We found that levels of PGRN were significantly reduced in the kidney in a mouse model of renal ischemia/reperfusion injury. We also observed that PGRN deficiency (Grn(-/-) mice) significantly aggravated renal injury as evidenced by higher serum creatinine, more severe morphological injury, increased tubular epithelial cell death, and tubulointerstitial neutrophil and macrophage infiltration versus wild-type mice. In vitro, we found that recombinant human PGRN attenuated hypoxia-induced inflammatory actions and apoptosis in proximal tubule epithelial cells, at least in part associated with a nucleotide-binding oligomerization domain containing 2 (NOD2)-mediated immune response. Importantly, pretreatment with or delayed administration of recombinant human PGRN protected against or promoted recovery from renal ischemia/reperfusion injury in wild-type and Grn(-/-) mice. Similar protective effects were also found in cisplatin-induced AKI. Thus, our findings provide a better understanding of the biological activities of PGRN in the kidney and suggest that PGRN may be an innovative therapeutic strategy for treating patients with AKI.Kidney International advance online publication, 21 January 2015; doi:10.1038/ki.2014.403.Kidney international. 01/2015; - [Show abstract] [Hide abstract]
ABSTRACT: Although our recent studies have demonstrated that NOD2 is one of critical components of a signal transduction pathway that links renal injury to inflammation in diabetic nephropathy (DN), the regulatory mechanisms for NOD2 expression under hyperglycemia have not yet been elucidated. Considering that NOD2 mRNA from different species bearing long 3'-UTR with various AU-rich elements (AREs), the present study was designed to investigate the potential contribution of the RNA binding protein human antigen R (HuR) on the posttranscriptional regulation of NOD2 expression. In this study, we firstly found that the up-regulation of HuR in the kidney from DN subjects, which was correlated with proteinuria, indicating the role of HuR in the pathogenesis of DN. In vitro, high glucose (HG) induced a distinct increase in cytoplasmic HuR in rat glomerular mesangial cells (RMCs). By RNA-EMSA analysis, we found that HuR bind to the 3'-UTR NOD2, while HuR silencing reduced HG-induced NOD2 expression and mRNA stability. Mechanistically, we further found that NADPH oxidase-mediated redox signaling contributed to the expression and translocation of HuR and NOD2 mRNA stability. Finally, we evaluated the role of HuR showing that in vivo gene silencing of HuR by intrarenal lentiviral gene delivery ameliorated renal injury as well as reduced NOD2 expression in diabetic rats. Collectively, our studies demonstrate that HuR acts as a key posttranscriptional regulator of NOD2 expression, suggesting that targeting of HuR-NOD2 signaling might be crucial for the treatment of DN. Copyright © 2014. Published by Elsevier Inc.Free Radical Biology and Medicine 12/2014; 79. · 5.71 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Oxidative stress is central to the pathology of several neurodegenerative diseases, including Parkinson's disease (PD), and therapeutics designed to enhance antioxidant potential could have clinical value. In this study, we investigated whether dimethyl fumarate (DMF) has therapeutic effects in cellular and animal model of PD, and explore the role of nuclear transcription factor related to NF-E2(Nrf2) in this process. Treatment of animals and dopaminergic SH-SY5Y cells with DMF resulted in increased nuclear levels of active Nrf2, with subsequent upregulation of antioxidant target genes. The cytotoxicity of 6-hydroxydopamine (6-OHDA) was reduced by pre-treatment with DMF in SH-SY5Y cells. The increase in the reactive oxygen species caused by 6-OHDA treatment was also attenuated by DMF in SH-SY5Y cells. The neuroprotective effects of DMF against 6-OHDA neurotoxicity was dependent on Nrf2, since treatment with Nrf2 siRNA failed to block against 6-OHDA neurotoxicity and induce Nrf2-dependent cytoprotective genes in SH-SY5Y cells. In vivo, DMF oral administration was shown to upregulate mRNA and protein levels of Nrf2 and Nrf2-regulated cytoprotective genes, attenuate 6-OHDA induced striatal oxidative stress and inflammation. Moreover, DMF ameliorated dopaminergic neurotoxicity in 6-OHDA-induced PD animal models as evidenced by amelioration of locomotor dysfunction, loss in striatal dopamine, and reductions in dopaminergic neurons in substantia nigra and striatum. Taken together, these data strongly suggest that DMF may be beneficial for the treatment of neurodegenerative diseases like PD. Copyright © 2014. Published by Elsevier Ltd.Neuroscience 11/2014; 286. · 3.33 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: A novel tetramethylpyrazine derivative, CXC195, has been recently shown to protect against cerebral ischemia-reperfusion (I/R) injury. However, the detailed mechanisms underlying the neuroprotection of CXC195 are still unclear. The aim of the present study was to investigate the effects of CXC195 on the phosphorylation of endothelial nitric oxide synthase (eNOS) in response to cerebral I/R and to determine whether phosphatidylinositol-3-kinase (PI3K)/Akt signaling pathway might be involved. An in vitro model of oxygen glucose deprivation (OGD) which was performed on primary cultured human aortic endothelial cells (HAECs) and an in vivo middle cerebral artery occlusion (MCAO) model that was performed on Wistar rats were used in this study. CXC195 increased nitric oxide (NO) production and the phosphorylation but not the protein level of eNOS in HAECs subjected to 1 h OGD followed by reperfusion. In addition, CXC195 increased the phosphorylation of Akt; inhibition of PI3K/Akt pathway by a specific inhibitor, wortmannin, suppressed CXC195-induced NO release in HAECs. Consistently, CXC195 treatment significantly restored the phosphorylations of eNOS and Akt in the cortical penumbra of rats subjected to 2 h MCAO followed by reperfusion. Moreover, wortmannin abolished CXC195-induced eNOS phosphorylation and neuroprotection as evidenced by a reversal of the reduction in infarct volume and neurobehavioral outcomes. In conclusion, CXC195 induced phosphorylation of eNOS by activation of PI3K/Akt signaling under pathological cerebral I/R conditions, which provided a novel explanation for the neuroprotective effect of CXC195.Neurochemical Research 11/2014; · 2.55 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Stroke is a devastating clinical condition for which an effective neuroprotective treatment is currently unavailable. S-allyl cysteine (SAC), the most abundant organosulfur compound in aged garlic extract, has been reported to possess neuroprotective effects against stroke. However, the mechanisms underlying its beneficial effects remain poorly defined. The present study tests the hypothesis that SAC attenuates ischemic neuronal injury by activating the nuclear factor erythroid-2-related factor 2 (Nrf2)-dependent antioxidant response in both in vitro and in vivo models. Our findings demonstrate that SAC treatment resulted in an increase in Nrf2 protein levels and subsequent activation of antioxidant response element (ARE) pathway genes in primary cultured neurons and mice. Exposure of primary neurons to SAC provided protection against oxygen and glucose deprivation (OGD)-induced oxidative insults. In wild type (Nrf2(+/+) ) mice, systemic administration of SAC attenuated middle cerebral artery occlusion (MCAO)-induced ischemic damage, a protective effect not observed in Nrf2 knockout (Nrf2(-/-) ) mice. Taken together, these findings provide the first evidence that activation of the Nrf2 antioxidant response by SAC is strongly associated with its neuroprotective effects against experimental stroke and suggest that targeting the Nrf2 pathway may provide therapeutic benefit for the treatment of stroke. This article is protected by copyright. All rights reserved.Journal of Neurochemistry 11/2014; · 4.24 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Studies have highlighted the importance of histone deacetylase (HDAC)-mediated epigenetic processes in the development of diabetic complications. Inhibitors of HDAC are a novel class of therapeutic agents in diabetic nephropathy, but currently available inhibitors are mostly nonselective inhibit multiple HDACs, and different HDACs serve very distinct functions. Therefore, it is essential to determine the role of individual HDACs in diabetic nephropathy and develop HDAC inhibitors with improved specificity. First, we identified the expression patterns of HDACs and found that, among zinc-dependent HDACs, HDAC2/4/5 were upregulated in the kidney from streptozotocin-induced diabetic rats, diabetic db/db mice, and in kidney biopsies from diabetic patients. Podocytes treated with high glucose, advanced glycation end products, or transforming growth factor-β (common detrimental factors in diabetic nephropathy) selectively increased HDAC4 expression. The role of HDAC4 was evaluated by in vivo gene silencing by intrarenal lentiviral gene delivery and found to reduce renal injury in diabetic rats. Podocyte injury was associated with suppressing autophagy and exacerbating inflammation by HDAC4-STAT1 signaling in vitro. Thus, HDAC4 contributes to podocyte injury and is one of critical components of a signal transduction pathway that links renal injury to autophagy in diabetic nephropathy.Kidney International advance online publication, 9 April 2014; doi:10.1038/ki.2014.111.Kidney International 04/2014; · 8.52 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: CXC195 showed strongest protective effects among the ligustrazine derivatives in cells and prevented apoptosis induced by H2O2 injury. We recently demonstrated that CXC195 protected against cerebral ischemia/reperfusion (I/R) injury by its antioxidant activity. However, whether the anti-apoptotic action of CXC195 is involved in cerebral I/R injury is unknown. Here, we investigated the role of CXC195 in apoptotic processes induced by cerebral I/R and the possible signaling pathways. Male Wistar rats were submitted to transient middle cerebral artery occlusion for 2 h, followed by 24 h reperfusion. CXC195 was injected intraperitoneally at 2 h and 12 h after the onset of ischemia. The number of apoptotic cells was measured by TUNEL assay, apoptosis-related protein cleaved caspase-3, Bcl-2, Bax and the phosphorylation levels of Akt and GSK3β in ischemic penumbra were assayed by western blot. The results showed that administration of CXC195 at the doses of 3 mg/kg and 10 mg/kg significantly inhibited the apoptosis by decreasing the number of apoptotic cells, decreasing the level of cleaved caspase-3 and Bax, and increasing the level of Bcl-2 in rats subjected to I/R injury. Simultaneously, CXC195 treatment markedly increased the phosphorylation of Akt and GSK3β. Blockade of PI3K activity by wortmannin, dramatically abolished its anti-apoptotic effect and lowered both Akt and GSK3β phosphorylation levels. Our study firstly demonstrated that CXC195 protected against cerebral I/R injury by reducing apoptosis in vivo and PI3K/Akt/GSK3β pathway involved in the anti-apoptotic effect.Neurochemistry International 01/2014; · 2.65 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Although the innate immune response to induce postischemic inflammation is considered as an essential step in the progression of cerebral ischemia injury, the role of innate immunity mediator NLRP3 in the pathogenesis of ischemic stroke is unknown. In this study, focal ischemia was induced by middle cerebral artery occlusion in NLRP3(-/-), NOX2(-/-), or wild-type (WT) mice. By magnetic resonance imaging (MRI), Evans blue permeability, and electron microscopic analyses, we found that NLRP3 deficiency ameliorated cerebral injury in mice after ischemic stroke by reducing infarcts and blood-brain barrier (BBB) damage. We further showed that the contribution of NLRP3 to neurovascular damage was associated with an autocrine/paracrine pattern of NLRP3-mediated interleukin-1β (IL-1β) release as evidenced by increased brain microvessel endothelial cell permeability and microglia-mediated neurotoxicity. Finally, we found that NOX2 deficiency improved outcomes after ischemic stroke by mediating NLRP3 signaling. This study for the first time shows the contribution of NLRP3 to neurovascular damage and provides direct evidence that NLRP3 as an important target molecule links NOX2-mediated oxidative stress to neurovascular damage in ischemic stroke. Pharmacological targeting of NLRP3-mediated inflammatory response at multiple levels may help design a new approach to develop therapeutic strategies for prevention of deterioration of cerebral function and for the treatment of stroke.Journal of Cerebral Blood Flow & Metabolism advance online publication, 15 January 2014; doi:10.1038/jcbfm.2013.242.Journal of cerebral blood flow and metabolism: official journal of the International Society of Cerebral Blood Flow and Metabolism 01/2014; · 5.46 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Acetylpuerarin (AP), because of its lower water solubility, shows poor absorption that hinders its therapeutic application. Thus, the aim of this study was to prepare nanoemulsions for AP, enhance its oral bioavailability, and thus improve the therapeutic effect.International Journal of Nanomedicine 01/2014; 9:3413-23. · 4.20 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Background/Aims: Although the pathogenesis of myocardial infarction (MI) is multifactorial, activation of innate immune system to induce inflammation has emerged as a key pathophysiological process in MI. NOD2, one member of the NOD-like receptor (NLR) family, plays an important role in the innate immune response. This study was to examine the role of NOD2 during MI. Methods: MI was induced by permanent ligation of the left coronary artery in wild type and NOD2(-/-) mice and cardiac fibroblasts were isolated. Results: NOD2 expression was significantly increased in myocardium in post-MI mice. NOD2 deficiency improved cardiac dysfunction and remodeling after MI as evidenced by echocardiographic analysis, reduced the levels of cytokines, inflammatory cell infiltration and matrix metalloproteinase-9 (MMP-9) activity. In vitro, we further found that NOD2 activation induced the activation of MAPK signaling pathways, production of proinflammatory mediators and MMP-9 activity in cardiac fibroblasts. Conclusions: Our studies demonstrate that NOD2 is a critical component of a signal transduction pathway that links cardiac injury by exacerbation of inflammation and MMP-9 activity. Pharmacological targeting of NOD2-mediated signaling pathways may provide a novel approach to treatment of cardiovascular diseases. © 2014 S. Karger AG, Basel.Cellular Physiology and Biochemistry 12/2013; 32(6):1857-1866. · 3.55 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Stroke is a complex disease that may involve oxidative stress-related pathways in its pathogenesis. The nuclear factor erythroid-2-related factor 2/antioxidant response element (Nrf2/ARE) pathway plays an important role in inducing phase II detoxifying enzymes and antioxidant proteins and thus has been considered a potential target for neuroprotection in stroke. The aim of the present study was to determine whether eriodictyol-7-O-glucoside (E7G), a novel Nrf2 activator, can protect against cerebral ischemic injury and to understand the role of the Nrf2/ARE pathway in neuroprotection. In primary cultured astrocytes, E7G increased the nuclear localization of Nrf2 and induced the expression of the Nrf2/ARE-dependent genes. Exposure of astrocytes to E7G provided protection against oxygen and glucose deprivation (OGD)-induced oxidative insult. The protective effect of E7G was abolished by RNA interference-mediated knockdown of Nrf2 expression. In vivo administration of E7G in a rat model of focal cerebral ischemia significantly reduced the amount of brain damage and ameliorated neurological deficits. These data demonstrate that activation of Nrf2/ARE signaling by E7G is directly associated with its neuroprotection against oxidative stress-induced ischemic injury and suggest that targeting the Nrf2/ARE pathway may be a promising approach for therapeutic intervention in stroke.Toxicology and Applied Pharmacology 12/2013; 273(3):672-9. · 3.98 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: There is increasing evidence that oxidative stress is critically involved in the pathogenesis of Parkinson's disease (PD), suggesting that pharmacological targeting of the antioxidant machinery may have therapeutic value. Naringenin, a natural flavonoid compound, has been reported to possess neuroprotective effect against PD related pathology; however the mechanisms underlying its beneficial effects are poorly defined. Thus, the purpose of the present study was to investigate the potential neuroprotective role of naringenin and to delineate its mechanism of action against 6-hydroxydopamine (6-OHDA)-induced neurotoxicity in models of PD both in vitro and in vivo. Naringenin treatment resulted in an increase in nuclear factor E2-related factor 2 (Nrf2) protein levels and subsequent activation of antioxidant response element (ARE) pathway genes in SH-SY5Y cells and in mice. Exposure of SH-SY5Y cells to naringenin provided protection against 6-OHDA-induced oxidative insults that was dependent on Nrf2, since treatment with Nrf2 siRNA failed to block against 6-OHDA neurotoxicity or induce Nrf2-dependent cytoprotective genes in SH-SY5Y cells. In mice, oral administration of naringenin resulted in significant protection against 6-OHDA-induced nigrostriatal dopaminergic neurodegeneration and oxidative damage. Our results indicate that activation of Nrf2/ARE signaling by naringenin is strongly associated with its neuroprotective effects against 6-OHDA neurotoxicity and suggest that targeting the Nrf2/ARE pathway may be a promising approach for therapeutic intervention in PD.Neuropharmacology 12/2013; · 4.82 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Neuroinflammation is involved in various central nervous system (CNS) disorders, including brain infections, ischemia, trauma, stroke, and degenerative CNS diseases. In the CNS inflammation, secretory phospholipase A2-IIA (sPLA2-IIA) acts as a mediator, resulting in the generation of the precursors of pro-inflammatory lipid mediators, such as prostaglandins (PGs) and leukotrienes (LTs). However, the role of sPLA2-IIA in neuroinflammation is more complicated and remains unclear yet. In the present study, we investigated the effect of sPLA2-IIA inhibition by specific inhibitor SC-215 on the inflammation in LPS-induced mice cerebral cortex and primary astrocytes. Our results showed that the inhibition of sPLA2-IIA alleviated the release of PGE2 by suppressing the activation of ERK1/2, cPLA2α, COX-2 and mPGES-1. These findings demonstrated that sPLA2-IIA showed the potential to regulate the neuroinflammation in vivo and in vitro, indicating that sPLA2-IIA might be a novel target for the treatment of acute neuroinflammation.PLoS ONE 10/2013; 8(10):e77909. · 3.53 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Aims: This study was conducted to investigate the protective effects of CXC195, a tetramethylpyrazine analogue, in acute focal cerebral ischemia/reperfusion (I/R) injury in rats and to elucidate the potential mechanism. Methods: Middle cerebral artery occlusion for 2 h followed by reperfusion for 24 h was conducted in male Wistar rats and different doses of tetramethylpyrazine and CXC195 were intraperitoneally injected at 30 min after reperfusion. Results: Our results demonstrated that CXC195 at the dosage of 3 and 10 mg/kg significantly reduced the neurological deficit score and the infarct volume compared to the vehicle-treated group. In addition, CXC195 exhibited a protective effect against hippocampus neuronal cell death and significantly restored the brain ATP content. The activities of superoxide dismutase (SOD), glutathione peroxidase (GPx) and total antioxidative capability (T-AOC), as well as production of malondialdehyde (MDA) and reactive oxygen species (ROS) were assayed in ipsilateral hemisphere homogenates to evaluate the redox status after I/R injury. Treatment with CXC195 significantly attenuated the decrease of SOD, GPx and T-AOC activities and inhibited the elevation of MDA content and ROS generation. Furthermore, CXC195 prevented the upregulation of the NADPH oxidase (NOX) 2 and NOX4, and reduced inducible nitric oxide synthase (iNOS) induction and production of nitric oxide induced by I/R. Conclusion: These results suggest that CXC195 has a neuroprotective effect in transient focal ischemia, which is most likely due to its antioxidant activity by inhibiting NOX and iNOS expression. © 2013 S. Karger AG, Basel.Pharmacology 10/2013; 92(3-4):198-206. · 1.58 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Astrocytes activation has been implicated in the inflammatory responses underlying brain injury and neurodegenerative diseases including bacterial infections, cerebral ischemia, and Parkinson's diseases. Acetylpuerarin is a newly modified isoflavone based on puerarin that has neuroprotective and antioxidant effects. In this study, we investigated the anti-inflammatory action of acetylpuerarin in regulating the eicosanoids generation and its underlying molecular mechanisms in lipopolysaccharide (LPS)-induced production of arachidonic acid (AA) metabolites in primary rat astrocytes. The results showed that acetylpuerarin concentration dependently inhibited the LPS-induced production of AA metabolites such as prostaglandin E2 (PGE2) and leukotriene C4 (LTC4), and acetylpuerarin significantly attenuated the expression and immunoreactivity of group V secretory phospholipase A2 (sPLA2) protein induced by LPS in astrocytes. Furthermore, in astrocytes pretreated with acetylpuerarin, the time course of phosphorylation of extracellular signal-regulated kinase (ERK)1/2 and of cytosolic PLA2 alpha (cPLA2α) and expression of transcription factors, nuclear factor kappa B (NF-κB), was markedly truncated. Acetylpuerarin concentration dependently abolished the LPS-induced expressions of AA-metabolizing enzymes including cyclooxygenase-2 (COX-2) and lipooxygenase-5 (LOX-5). This study indicates that acetylpuerarin inhibited LPS-induced AA-metabolizing enzymes and AA metabolites in astrocytes via downregulation expression of group V sPLA2 and phosphorylation of ERK1/2, cPLA2α, and NF-κB. These findings reveal, in part, the molecular basis underlying the anti-inflammatory properties of acetylpuerarin.Journal of Molecular Neuroscience 09/2013; · 2.76 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Hydroxysafflor yellow A (HSYA) is the major active chemical component of the flower of the safflower plant, Carthamus tinctorius L. Previously, its neuroprotection against cerebral ischemia-reperfusion (I/R) injury was reported by anti-oxidant action and suppression of thrombin generation. Here, we investigate the role of HSYA in cerebral I/R-mediated apoptosis and possible signaling pathways. Male Wistar rats were subjected to transient middle cerebral artery occlusion for 2 h, followed by 24 h reperfusion. HSYA was administered via tail-vein injection just 15 min after occlusion. The number of apoptotic cells was measured by TUNEL assay, apoptosis-related proteins Bcl-2, Bax and the phosphorylation levels of Akt and GSK3β in ischemic penumbra were assayed by western blot. The results showed that administration of HSYA at the doses of 4 and 8 mg/kg significantly inhibited the apoptosis by decreasing the number of apoptotic cells and increasing the Bcl-2/Bax ratio in rats subjected to I/R injury. Simultaneously, HSYA treatment markedly increased the phosphorylations of Akt and GSK3β. Blockade of PI3K activity by wortmannin dramatically abolished its anti-apoptotic effect and lowered both Akt and GSK3β phosphorylation levels. Taken together, these results suggest that HSYA protects against cerebral I/R injury partly by reducing apoptosis via PI3K/Akt/GSK3β signaling pathway.Neurochemical Research 08/2013; 38(11). · 2.55 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Although NADPH oxidase-mediated oxidative stress is considered as one of the major mechanisms triggering the pathogenic actions of ischemic stroke and very recent studies have indicated that NADPH oxidase is a major source of reactive oxygen species (ROS) production controlling glutamate release, how neuronal NADPH oxidase activation is coupled to glutamate release is not well understood. Therefore, in this study, we used an in vivo transient middle cerebral artery occlusion (MCAO) model and in vitro primary cell cultures to test whether complexins, the regulators of soluble N-ethylmaleimide-sensitive factor attachment protein receptor (SNARE) complexes necessary for vesicle fusion, are associated with NOX2-derived ROS and contribute to glutamate-mediated excitotoxicity in ischemic stroke. In this study, we first identified the upregulation of complexin II in the ischemic brain and evaluated the potential role in ischemic stroke showing that gene silencing of complexin II ameliorated cerebral injury as evidenced by reduced infarction volume, neurological deficit, neuron necrosis accompanied by decreased glutamate levels in consistent with the results from NOX2(-/-) mice with ischemic stroke. We further demonstrated complexin II expression was mediated by NOX2 in primary cultured neurons subjected to oxygen-glucose deprivation (OGD) and contributed to OGD-induced glutamate release and neuron necrosis via SNARE signaling. Taken together, these findings for the first time provide evidence that complexin II as a central target molecule that links NADPH oxidase-derived ROS to glutamate-mediated neuronal excitotoxicity in ischemic stroke.Free Radical Biology and Medicine 08/2013; · 5.27 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Previously 13 nm gold nanoparticles (GNPs) have been shown to display cytotoxicity to lung cancer cells when l-buthionine-sulfoximine (BSO) was used to decrease the expression of intracellular glutathione (GSH). In this study, we investigated how the GNPs induced cell death at the molecular level. Dual staining with fluorescent annexin V, and propidium iodide was used to discriminate apoptotic and necrotic cell death. We found that GNPs induced apoptosis and necrosis in lung cancer cells with low level of intracellular GSH. The disruption of F-actin and phosphorylation of H2AX induced by GNPs were both associated with apoptosis. The ER stress was caused, mitochondrial membrane potential was disrupted, intracellular calcium was elevated and intracellular caspase-3 was activated by GNPs in lung cancer cells with low intracellular GSH, while cell death could not be prevented by the pan-caspase inhibitor N-benzyloxycarbonyl-Val-Ala-Asp-fluoromethylketone. The cells were further examined for caspase-independent death. After GNPs and BSO exposure, apoptosis inducing factor, endonuclease G, and glyceraldehyde-3-phosphate dehydrogenase translocated into the nuclei of apoptotic cells. Receptor-interacting protein 1 kinase inhibitor necrostatin-1 significantly decreased the PI positive cells that were induced by GNPs and BSO. Taken together, our results suggest that multiple modes of cell death are concurrently induced in GNPs-exposed lung cancer cells with low intracellular GSH, including apoptosis and necrosis. These results have important implications for GNPs in anticancer applications.Journal of Nanoparticle Research 08/2013; 15(8). · 2.28 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: Although hyperhomocysteinemia (hHcys) has been recognized as an important independent risk factor in the progression of end-stage renal disease and in the development of cardiovascular complications related to end-stage renal disease, the mechanisms triggering the pathogenic actions of hHcys are not yet fully understood. The present study was designed to investigate the contribution of nucleotide-binding oligomerization domain containing 2 (NOD2), an intracellular innate immunity mediator, to the development of glomerulosclerosis in hHcys. Our results showed that NOD2 deficiency ameliorated renal injury in mice with hHcys. We further discovered the novel role of NOD2 in mediating Ca(2+) signaling and found that homocysteine-induced NOD2 expression enhanced transient receptor potential cation channel 6 (TRPC6) expression and TRPC6-mediated calcium influx and currents, leading to intracellular Ca(2+) release, ultimately resulting in podocyte cytoskeleton rearrangement and apoptosis. Moreover, we found that nephrin expression was downregulated dependently by NOD2, and overexpression of nephrin attenuated homocysteine-induced TRPC6 expression in podocytes. The results add evidence to support the essential role of nephrin in mediating NOD2-induced TRPC6 expression in hHcys. In conclusion, our results for the first time establish a previously unknown function of NOD2 for the regulation of TRPC6 channels, suggesting that TRPC6-dependent Ca(2+) signaling is one of the critical signal transduction pathways that links innate immunity mediator NOD2 to podocyte injury. Pharmacological targeting of NOD2 signaling pathways at multiple levels may help design a new approach to develop therapeutic strategies for treatment of hHcys-associated end-stage renal disease.Hypertension 07/2013; · 7.63 Impact Factor - [Show abstract] [Hide abstract]
ABSTRACT: An increasing number of clinical and animal model studies indicate that activation of the innate immune system and inflammatory mechanisms are important in the pathogenesis of diabetic nephropathy. Nucleotide-binding oligomerization domain containing 2 (NOD2), a member of the NOD-like receptor family, plays an important role in innate immune response. Here we explore the contribution of NOD2 to the pathogenesis of diabetic nephropathy and found that it was upregulated in kidney biopsies from diabetic patients and high-fat diet/streptozotocin-induced diabetic mice. Further, NOD2 deficiency ameliorated renal injury in diabetic mice. In vitro, NOD2 induced proinflammatory response and impaired insulin signaling and insulin-induced glucose uptake in podocytes. Moreover, podocytes treated with high glucose, advanced glycation end-products, tumor necrosis factor-α, or transforming growth factor-β (common detrimental factors in diabetic nephropathy) significantly increased NOD2 expression. NOD2 knockout diabetic mice were protected from the hyperglycemia-induced reduction in nephrin expression. Further, knockdown of NOD2 expression attenuated high glucose-induced nephrin downregulation in vitro, supporting an essential role of NOD2 in mediating hyperglycemia-induced podocyte dysfunction. Thus, NOD2 is one of the critical components of a signal transduction pathway that links renal injury to inflammation and podocyte insulin resistance in diabetic nephropathy.Kidney International advance online publication, 17 April 2013; doi:10.1038/ki.2013.113.Kidney International 04/2013; · 8.52 Impact Factor
Publication Stats
343 | Citations | |
135.00 | Total Impact Points | |
Top Journals
University of Jinan (Jinan, China)
Chi-nan-shih, Shandong Sheng, China
Shandong University
- Department of Pharmacology
Chi-nan-shih, Shandong Sheng, China


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Building relationships with donors
Legacy fundraising, big-gift seeking are all part of the professional fundraiser's role...
Legacy fundraising, big-gift seeking are all part of the professional fundraiser's role. This unit will help you to gain the skills necessary to persuade individuals to become donors. How do you change people's ideas about methods of giving, moving them from casual street donations to regular direct debit giving?
After studying this unit you should:
- appraise your own skills in asking for contributions;
- identify ways of sustaining and developing donor involvement;
- make recommendations on how your organisation might most appropriately acknowledge contributions;
- contribute to thinking on the actual or potential role of 'big gifts' in your organisation's approach to fundraising;
- enhance your approach to legacy fundraising.
- Duration 6 hours
- Updated Thursday 4th July 2013
- Masters level
- Posted under Business Studies
- Current section: Introduction
- Learning outcomes
- 1 Donor motivation
- 2 Asking someone for something: the core skill
- 3 Asking lots of people for something: key issues and choices
- 3.1 Introduction
- 3.2 Knowing who to approach
- 3.3 Deciding what to ask for
- 3.4 Communicating your request
- 3.5 Building the relationship: developing your donors
- 3.6 Extending and sustaining involvement
- 3.6.1 Saying thank you and acknowledging current contribution
- 3.6.2 Listening and responding to what donors and supporters say
- 3.6.3 Balancing emotional commitment with awareness and understanding
- 3.6.4 Taking account of 'external' perceptions
- 3.6.5 Using 'involvement devices' sensitively and appropriately
- 4 Seeking big gifts
- 5 Legacy fundraising
- 6 Summary
- Next steps
- References
- Module team
- Acknowledgements
Building relationships with donors
The starting point for this session is the very simple proposition that the vast majority of people become donors only when they are asked. You have to turn people into donors by providing them with suitable opportunities to contribute. It is your request and your approach that lay the foundations for each individual donor relationship; it is your subsequent actions that will sustain – or otherwise – the nature and amount of any further donations and support from that donor.
So our aim in this unit is to provide you with an overview: of approaches you can adopt and issues you are likely to face in providing the right sorts of opportunities for people to become donors and contributors to your organisation.
You will look at what is involved in asking for donations on an individual face-to-face basis and also look at the process of asking numbers of people for support, and at how to extend the involvement of your donors and supporters once you have established contact. You will also look more closely at how to tackle the business of securing major individual contributions. We finish with a brief overview of legacy fundraising – the culmination for many donors of a lifetime's generosity.
This material is from our archive and is an adapted extract from Winning resources and support, (B625) which is no longer taught by The Open University. If you want to study formally with us, you may wish to explore other courses we offer in this subject area [Tip: hold Ctrl and click a link to open it in a new tab. (Hide tip)] .
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This is an extract from an Open University course which is no longer available to new students. If you found this interesting you could explore more free Business Studies course units or view the range of currently available OU Business Studies courses.
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Author: ZenKimchi
Joe McPherson founded ZenKimchi in 2004. He has been featured and sourced in The New York Times, The Wall Street Journal, The Los Angeles Times, The Washington Post, CNN, KBS, MBC, SBS, Le Figaro, Travel + Leisure Southeast Asia, Harper's Bazaar Korea, The Chosun Weekly, and other Korean and international media. He has consulted for "Parts Unknown with Anthony Bourdain," The Travel Channel's "Bizarre Foods with Andrew Zimmern," Lonely Planet, National Geographic, Conde Nast Traveler, the PBS documentary series "Kimchi Chronicles," and other projects in the UK, Canadan, and Australia featuring celebrity chefs such as Gizzi Erskine and Gary Mehigan. Mr. McPherson has written for multiple Korean and international publications, including SEOUL Magazine, JoongAng Daily, The Korea Herald, Newsweek Korea and wrote the feature article for U.S. National publication Plate magazine's all-Korean food issue. He has acted as dining editor for 10 Magazine and was on the judging panel for Korea for the Miele Guide. He spoke at TEDx Seoul on Korean food globalization, at TED Worldwide Talent Search on the rise of Korean cuisine, and in New York City on Korean Buddhist temple cuisine. The company ZenKimchi International organizes food tours for tourists and corporations and acts as a media liaison for foreign and Korean media and local restaurants and producers.
The Food Map
CLICK to go to The Food Map.
All of our restaurants in one handy convenient map.
Promoting Korean Food, Chapter 1: Know Your Market Jan 30, 2015
Promoting Korean Food, Intro: I'm Your Market Jan 29, 2015
A Culture of Copying Aug 31, 2012
Under the Bridge Jul 12, 2009
The Wedding Oct 11, 2009
Andong Mar 9, 2009
Wonder Girls as Korean Food Ambassadors Sep 6, 2011
ON TV: ZenKimchi on "Chef on the Road" Nov 18, 2013
Video: Gourmet Ramyeon at Eat Your Kimchi Feb 19, 2011
Roasted Turkeys at E-Mart Dec 8, 2009


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Sara Rubin a day ago 73 responses
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The biggest stars in the world's most insane sport answer the cutest questions imaginable.
We followed a hair donation through the Wigs For Kids process to find out.
We asked the top scientists in the world about this very important topic...
♫ Playin' Hungry, Hungry Humans ♫
It's pretty powerful to see how their responses change over time.
Do you want to text a snowman?
"I feel like the substitute teacher who can't pronounce anyone's name."
"It's like a blacksmith who made transformers fell into the culture of pleasure." What?
Get that shot before it disappears forever. Welcome to a typical day in the life of a full-time Snapchatter.
Because you better break it down barney style if you wanna cover yer six.
Jared and Clyde, best friends forever.
Hopefully this will help ease the pain of your Parks and Rec withdrawl.
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He knows more of the words than you.
Sea creatures battle it out on dry land!
Mihaela Noroc traveled all around the world to show that stunning female beauty is everywhere.
She was nominated by her daughter for a local radio station's "Giveback" program.
"I'd love to see Meryl, like, slutdropping on the dance floor."
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text:
"My name is Joe," says the familiar droopy voice on the soundtrack. "And this is what I do." He's making reference to being a hitman, but the actor might as well be talking about himself: His name is Nicolas Cage, and this is what he does — take the big bucks to star in slovenly, inert pulp of the sort no actor of his magnitude should be stooping to. In Bangkok Dangerous, the Pang brothers, remaking their 1999 Thai thriller, turn Cage into a scruffy, blank-eyed cipher of an assassin, who has been hired to kill four men; we learn so little about them that they flash by like the bad guys in a trailer. The filmmakers even manage to turn seamy Bangkok into the least exotic setting imaginable. F
Bangkok Dangerous''My name is Joe,'' says the familiar droopy voice on the soundtrack. ''And this is what I do.'' He's making reference to being a hitman, but the actor...Bangkok DangerousAction/AdventureJason RichmanPT100MR''My name is Joe,'' says the familiar droopy voice on the soundtrack. ''And this is what I do.'' He's making reference to being a hitman, but the actor...2008-09-15Jason WithLions Gate Films
Bangkok Dangerous
Genre: Action/Adventure; Starring: Nicolas Cage, Jason With; Director: Oxide Pang Chun, Danny Pang; Author: Jason Richman; Release Date Wide: 09/05/2008; Runtime (in minutes): 100; MPAA Rating: R; Distributor: Lions Gate Films
Posted September 15 2008 — 12:00 AM EDT
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How cute is daisy?
Her current rating is 4.23 out of 5 with 57 vote(s).
All about daisy
Pet Tip
Vaccines and Allergic Reactions – Vaccination Reactions in Cats and Dogs – Pet tip 221
Vaccinating our pets is necessary to protect them from harmful diseases. Most veterinarians in North America recommend giving almost all cats and dogs certain vaccines when they are brought in for the first time as puppies or kittens. If they …
- Owner:
- jennifer mckay
- City:
- prince albert
- Province, Country:
- SK, Canada
- Type/Breed:
- black lab cross
- Date of Birth:
- july 2002
- Sex:
- Female
- Height:
- Not Provided
- Weight:
- 75
- Coat Colour:
- black
- Eye Colour:
- brown
chewing everything in sight
buffalo bones
anything food, not very fussy, as long as it's not my IAM's
Sleeping in the sunlight
Hogging the bed
The neighbour's cat
getting socks/shoes/boots/magazines/newspaper taken away
not getting my way!
the winter
I am very happy in my house. And it is MY house. My Mommy seems to think that she is the owner of the house, but really it is MINE. I love to spend my days lounging around in my backyard, lying in the sunlight on my deck. I like to dig lots of holes under my deck too, just to annoy my Mommy.
I am a very pretty Black Labrador Retriever. I'm just over a year old right now, so I'm still young and have TONS of energy.
My favorite memory is the day I left the SPCA animal shelter. As I was driving to my new house, and I saw that Mommy had just bought a few groceries. She had a couple bags on the floor of the vehicle, so I decided to take a look in one of the bags. There was something squishy and soft and smelt sooooo good!! I tore open the bag and threw bread all over the vehicle!! It was so much fun...........and it's only getting better.


text:
Bruce E. Radford
Position: NT-DE
Height: 6-5 Weight: 257 lbs.
Born: October 5, 1955 in Pineville, LA
Died: May 23, 2011 in New Orleans, LA
High School: Tioga
College: Grambling St.
Drafted by the Denver Broncos in the 3rd round (77th overall) of the 1979 NFL Draft.
Weighted Career AV (100-95-...): 6 (8603rd overall since 1950)
Let people know you like this site. Sponsor this page for $5. Your message will replace this ad.
Output includes all games from 1960 to 2014.
Career Game Log: Regular Season · Playoffs · vs.


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Possessing knockout beauty as well as distinctive detailing, this rosetone watch complements your singular sense of style. An opulent mother-of-pearl dial and clear, round, pave-style crystal accents shine against the classic, embossed leather strap for a sophisticated look with a dash of difference. From Melania(R) Timepieces & Fashion Jewelry.Brown or White.
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Latest RIM-162 ESSM Stories
2011-06-22 02:00:00
PARIS, June 22, 2011 /PRNewswire/ -- Raytheon Company (NYSE: RTN) and its international partners will introduce a new ground-based Evolved SeaSparrow Missile at the Paris Air Show. The ground-based ESSM features multi-use capabilities for both naval and land-based applications.
Word of the Day
- A person who stands up for something, as contrasted to a bystander who remains inactive.
- One of the upright handlebars on a traditional Inuit sled.
The word 'upstander' in the first sense here is a play on the word 'bystander' and the idea of 'standing up' for something.


text:
Counting the CostAutomotives get eco-friendly The European manager of General Motors joins the show, plus a spring of discontent in Kyrgyzstan. 05 Mar 2011 08:14 GMT | Business & Economy, Kyrgyzstan " /> The bold and the beautiful of the automotive industry get eco-friendly at the Geneva Motor Show. We speak to General Motors' European boss.Also, a spring of discontent blossoms in Kyrgyzstan as the country grapples with huge economic debt.And Ipad, the sequel - Apple unveils version two of the tablet computer but was it upstaged?This episode of Counting the Cost aired from Friday, March 4, 2011.Source: Al Jazeera
GM automaker makes market return US president hails end of government's role as majority shareholder in car manufacturer as it raises $20.1bn. Business & Economy, US & Canada, China, Japan, United States
GM turnaround on the cards US government's role as majority shareholder ends as car maker raises $20.1bn from markets. Business & Economy, US & Canada, China, Japan, United States
The other side of Somalia's pirates Poisoned waters and illegal fishing drove Somalis into sea piracy to feed families, according to relatives. Human Rights, Poverty & Development, Environment
The most powerful mafia you've never heard of Italy's 'Ndrangheta crime syndicate is believed haul in more revenue than many Fortune 500 companies. Italy, Crime, Europe
New Afghan militia sets its sights on ISIL 'Marg' vigilante group vows to safeguard northern Afghanistan from armed factions amid reports of ISIL infiltration. War & Conflict, Asia, Afghanistan
Devastation and disease after deadly Malawi floods Tens of thousands stranded after southern Africa country's record floods face rising risk of disease outbreak. Humanitarian crises, Africa, Malawi
The SystemProsecutorial MisconductThe System examines two cases where prosecutorial misconduct may have led to wrongful imprisonment. Crime, Law, United States
Talk to Al JazeeraPrince Ali of Jordan: 'Bring back FIFA's reputation'The FIFA presidential candidate explains why he is challenging Sepp Blatter and how he plans to reform the organisation.Sport, Middle East, FIFA
Al Jazeera WorldAhalil: Music of the Red OasisThe music of the Algerian Sahara, known as Ahalil, is a key part of a way of life that has survived for centuries.Arts & Culture, Algeria, Music
People & PowerKilling the BallPeople & Power investigates how a match-fixer and his syndicate corrupted global football.Sport, Football, Corruption


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Shotgun Jaxon
We're Playing The Most Country Music in KC!
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Wes Poe
Back on the air this week
by Wes Poe
posted Mar 1 2010 10:56AM
I was out last week for CRS which stands for Country Radio Seminar. It's the better part of a week, and country programmers and station personnel head to Nashville for networking, education, and familiarizing ourselves with the new best music coming our way in the next year. There are some folks that are worth checking out. Most of these folks are pretty unknown but you will most likely hear a lot more about them in the year, and years to come.
Tyler Dickerson, a new artist on Lyric Street. Only 16 years old, but the attitude and song writing ability of someone a lot older, you can listen to his stuff on
Emily West: She was recently in town for a show at Fuel and I saw her again with her entire band. She's on the verge of turning into something big, you can listen to some of her songs on her
page also. We're currently playing Blue Sky featuring Keith Urban on the Wolf.
Josh Thompson: also makes my list for possible superstars, many of you may have heard "Beer On The Table" which was his first single. I have pretty good feeling he'll be around for at while. He has a album available now and should be available at Walmart and Target. You can hear songs from it on
Last but not least, Mallary Hope She's a new artist on MCA Records. The trend has been going toward singer song writers in Nashville for the last 5 years or so. More and more labels are searching for talented singers and songwriters. Mallary fits the bill, great voice and a great song writer. Check out some of her stuff on her
There are more that I'm missing but the good news there is a lot of fresh talent out there and some great new music you'll be hearing on the Wolf soon! (WP)
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