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From birth, my girls were totally different. Baby A slept well during the night, not well during the day. She was a total mommy's girl, wanted me to hold her all the time and didn't like it when other people did. She was feisty and loud, could never be alone for a second, needing constant attention.
Baby B slept well during the day, not so well during the night. She was totally independent, happily playing by herself, or lying in a bouncy chair or swing and watching the world go by. She would let anyone hold her and wasn't particularly attached to me or anyone else.
Just before their first birthdays, they seemed to switch personalities. Now Baby A sleeps well during the day and not the night, and B is the opposite. B is now loud and feisty and needs constant attention. She cries when I or DH leave the room, is constantly hitting her sister or pulling her hair, and just generally seems really sensitive and/or dramatic. A is the more independent now, more quiet and sweet. A now only needs me when people she doesn't know are close by.
It's so weird. Has anyone had this happen?
Mine totally did. By now, they've "switched personalities" a few times since they were born.
They keep you on your toes, eh?
(And yes, I'm positive I haven't mixed mine up.)


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The purpose of the NSF-DUE Transforming Undergraduate Education in the STEM Disciplines Type I project was to address the disturbing failure rates of college students placed in remedial mathematics. Given the interdisciplinary nature of the project, two educational psychologists collaborated with two mathematicians as the research and development team to direct the two year project.
The grant proposal #1141334 titled The Predictability of Student Attributes and Instructional Milieu on Developmental Math Courses, College Algebra, the CAAP, and Matriculation to Degree was designed to develop, implement, and evaluate impact of a multi-tiered system of supports (MTSS) model with adult learners. Our MTSS model evaluation method demonstrated improved learner outcomes in passing Basic and Intermediate Algebra, and early longitudinal data suggest students go on to pass College Algebra and the mathematics subtest of the Collegiate Assessment of Academic Proficiency (CAAP) examination. Completing these general education requirements shows promise toward graduating with a college degree.
Most importantly, this project promoted use of instructional methods grounded in scientifically-based research. Our findings confirmed the effectiveness of explicit instruction with adult learners who need direct teaching methods to learn fluency with numbers and the procedural knowledge necessary to achieve conceptual understanding. Finally, changes in course structure and instructional methods to prompt student engagement in active learning established within the multi-tiered system of supports model show promise in improved retention and graduation rates for underrepresented and disadvantaged college students placed in remedial education.
During the two year NSF Transforming Undergraduate Education in the STEM Disciplines Type I project:
Dr. Lee Pearce
Special Education
Jonas 231. Unit 9004
Spearfish, SD 57799-9502
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About Us / Catering & EventsSuccess is a guarantee when you have the Waffle Cabin® at your party or event. We offer a high quality product that is unique and at the same time very tasty. Our guests are always positively surprised and the word-of-mouth this creates keeps us busy all year. We have catered Bar/Bat Mitzvah's, sweet sixteen parties, birthday parties, weddings and graduation parties among others. When considering our product we ask that you first forget everything you know about waffles. Ours is a yeast-raised dough made with vanilla and mixed with sugar nibs known as Pearl Sugar. The sugar melts when baked creating sweet pockets in the waffles while caramelizing the outside. The aroma created when baked is enough for anyone to be stricken with we call Waffology - the desire for a Waffle Cabin Waffle. We can accommodate parties of 100 - 300 guests. We work with several party planners and are happy to work with you and/or your representative. Whatever you party might be we'll do our very best to make it a unique experience that your guests will remember.
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20 Moonbrook Drive
Rutland, VT 05701
>>> IN THE NEWS: APRIL, 2013 - The Best Waffle In America • Seersucker [ READ ARTICLE ]
The Waffle Cabin was a concept started by
Peter Creyf and Ingrid Heyrman. We originally
called it "Leo's – Gaufres de Liège" operating
from a pushcart at Quincy Market in Boston in
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one at Boston Common Park we decided to try
a new venue – ski resorts. [ more ]
©2015 Waffle Cabin
Site by: J.E.G. Design, Inc.
• Wachusett Mtn
• Nashoba Valley Ski Area
• Ski Bradford Ski Area
• Blue Mountain
• Camelback Mtn Resort
• Snowshoe Mtn Resort


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(Emily Amsler/AP)
Dog not gone: Rescue in the Columbia Gorge
PORTLAND, Ore. (AP) — A yellow Labrador that got spooked on a Christmas Day hike in the Columbia Gorge, snapping her leash and plunging 150 feet down a cliff, was rescued in the dark by a climber who rappelled to a narrow ledge where the dog was trapped. Full Story
(Ed Araquel/AP)
Box Office: 'Hobbit' wins Christmas, 'Big Eyes' flops
LOS ANGELES (AP) — Christmas saw the arrival of a host of fresh fare for moviegoers, but "Unbroken" and "Into the Woods" were no match for "The Hobbit: The Battle of The Five Armies. Full Story
Has One Direction lost its luster?
'NCIS' repeat and holiday cartoons top British boy band's behind-the-scenes special Tuesday night.


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health and medical information on the World Wide Web comes
from many sources and changes daily. There are likely to be
errors and omissions in this information. This web site, its contributors nor its sponsors represents or
warrants that the information in this Web Site or accessed
through this Web Site is accurate or complete.
direct your medical and health questions to your health care
William J.
Catalona, M.D., answers your questions...
Professor of Urological
Surgery at Washington University Medical Center in St. Louis,
Dr. Catalona has received the
Benhamin Stockwell Barringer Medal from the American Association
of Genito-Urinary surgeons for his cancer research. He was also
by American Health magazine
as one of the Best Doctors in America. Dr. Catalona edits QUEST,
published by the Urologic Research Foundation.
Q. What does
it mean when there is blood in the semen and what should be done to
treat it?
A. The most common
causes of blood in the semen are irritation, inflammation, and/or
infection in the prostate gland (prostatitis). There may be
associated symptoms of pain or burning in the urinary tract, urinary
frequency, slow urinary stream, or urinary urgency. However, in
some cases, bleeding may be the only symptom. In most cases, the
bleeding will eventually clear as the inflammation or infection is
handled by the body's natural immune defenses, but in most cases
taking antibiotics will hasten the return to normal.
Blood in the semen can also occur
with prostate cancer, but this is an unusual cause. In our study of
men who underwent biopsy because they had a PSA higher than 4 or
suspicious findings on digital prostate exam, if they had not other
symptoms, 24 percent had biopsies that showed cancer. If they also
had blood in the semen, 36 percent had biopsies that showed cancer.
This indicates that blood in the semen is more common in patients
with cancer, but it is important to point out that these men also
had other evidence of cancer (PSA ore rectal exam abnormalities).
(Reference: Catalona et al. J Urol 151:1283, 1994.).
I recommend that men with blood in
the semen be treated with a two to four week course of antibiotic
therapy. If the bleeding persists, I recommend that the patient
undergo cystoscopy (looking into the urine channel with a small
flexible scope) and biopsy of the prostate to rule out the presence
of prostate cancer.
Q. Are there
measures that can be taken to prevent the development or progression
of prostate cancer?
A. Prostate cancer is
influenced by both heredity and environmental factors. Prostate
cancer is almost certainly caused by changes (mutations) in the DNA
that affect the regulation of the growth of normal prostate cells.
It is probable that several mutations have to occur before a
full-blown cancer occurs. These mutations can accumulate over time,
causing the gradual progression to prostate cancer. Some mutations
may cause a precancerous condition called prostatic intraepithelial
neoplasia (PIN), others may cause a slowly growing form of the
cancer, and still others may cause a rapidly progressive form of the
disease. It may take decades for some prostate cancers to develop,
while others may develop more rapidly if a more serious mutation
occurs. Some men may inherit one or more of these mutations from
either his mother or his father. In this case, fewer additional
mutations would be needed before cancer occurs. Nevertheless, it is
possible that preventive measures might prevent the development or
progression of prostate cancer.
Chemoprevention is treatment
that prevents the development and/or the progression of cancer.
Some potential chemopreventive agents for prostate cancer may be
medicines, herbs, or food supplements. Recently, considerable
attention has been focused on nutrients as possible prostate cancer
preventive agents. The most promising are soy products (called
phytoestrogens and isoflavones), vitamin D, vitamin E, selenium, and
Some researchers have speculated
that the high ingestion of soy products is responsible for the lower
death rate from prostate cancer in Japan, where the death rate is
4-5 times lower than in the United States. Some studies have
suggested that prostate cancers develop and progress more slowly in
Japan. It has been shown that soy slows the proliferation of
prostate cells, and part of this effect may be mediated through
lowering the levels of male hormones. Soy contains steroid-like
chemicals that may alter the chemical reactions in prostate tissues.
Soy proteins are changed by
intestinal bacteria to produce estrogenic and antiestrogenic
compounds. This increases the levels of binding protein levels in
the blood that tie up the male hormone, testosterone, which normally
stimulates prostate cancer cells to grow. Soy proteins also have
numerous other potential actions. They alter enzyme functions,
alter protein synthesis, alter the action of growth factors,
directly affect the growth rate of prostate cells, and alter the
production of blood vessels that allow tumors to grow.
Genistein is one of the most
prominent components in soy. Genistein has been shown in animal
prostate cancers to slow the rate of tumor growth. There is
sufficient experimental evidence to justify the use of soy proteins
in clinical trials for preventing prostate cancer, and these studies
will provide important information in the future.
Lycopene is found in tomato products. It belongs to a type of
chemical called a carotenoid. Lycopene has one of the greatest
capacities for preventing mutations in DNA. Chemicals that have
these properties are called antioxidants, and lycopene is one of the
stronger antioxidants. In addition, lycopene has other biological
activities related to cell-cell communications and growth control.
Experimental animal studies show that lycopene can slow the growth
of prostate cancer cells.
Epidemiologic studies have shown
that consumption of lycopene is associated with a lower risk of
prostate cancer. In one important study, lycopene was the only
antioxidant found in significantly lower levels in prostate cancer
patients than in men without prostate cancer. There are also
studies suggesting that taking lycopene before prostate cancer
surgery may result in more favorable treatment results. Lycopene
concentrates are available at pharmacies and health food stores and
can be purchased over the counter without a prescription.
Vitamin E
Vitamin E is a fat-soluble vitamin that also has the potential to
decrease DNA damage through its antioxidant effects. In addition,
vitamin E affects the immune system. Vitamin E deficiency is
associated with decreased immune function that increases the
susceptibility to cancer and infections. Vitamin E may not only
prevent the development of prostate cancer but also may slow its
progression, especially in smokers. Vitamin E may also lower the
activity of some cellular signals that increase cell growth.
Studies suggest that vitamin E may be especially important in
inhibiting the growth of prostate cancer cells induced by a high-fat
Many fat-soluble vitamins such as
vitamin E can cause side effects if taken in large doses. Some (but
not all) studies suggest that too much vitamin E may interfere with
blood coagulation and lead to post-surgery bleeding due to its
effect on platelet function. Vitamin E exists in several forms; 200
to 800 of the alpha tocopherol form of vitamin E are well
Selenium may also act as an anticancer agent due to its antioxidant
properties. Selenium suppresses cell growth and enhances the immune
response - thus functioning similarly to vitamin E. Studies have
suggested that prostate cancer risk was reduced by 60% among those
who take supplemental selenium. Selenium may also exert its
antitumor effects by other mechanisms, such as inducing programmed
death in cancer cells, and affecting important enzyme systems. The
recommended dose of selenium is 200 ug/day, which is 2 to 3 times
the recommended daily allowance.
Vitamin D.
Vitamin D is important primarily for bone and mineral metabolism.
It induces differentiation of prostate cancer cells into normal
cells and blocks the growth of prostate cancer cells. Vitamin D may
act by blocking the progression of cells through the cell cycle and
initiating programmed cancer cell death. Prostate cancer cells have
receptors for vitamin D. Vitamin D may be important in preventing
prostate cancer and is also being studied as an agent for the
treatment of advanced prostate cancer. The recommended daily dose
is 10 ug/day. Higher doses may cause high blood calcium levels.
Q. What do you
do when Lupron stops working?
A. Hormonal therapy can be
divided into two phases, primary and secondary. Primary Hormonal
Most prostate cancer cells either
die or go into a dormant phase when the blood male hormonal level
(testosterone) level is dramatically lowered. Male hormone levels
can be lowered or rendered less effective by either removal of the
testicles (called castration or orchiectomy) or by injections
(called Lupron [leuprolide], Zoladex [goserelin], or by antiandrogen
pills, such as Megace (megestrol), Eulexin (flutamide), Casodex (bicalutamide),
Nilandron (nilutamide), by estrogens (diethylstilbestrol, estrace),
by inhibitors of androgen synthesis, such as ketoconazole, by
antiestrogens, such as tamoxifen, by herbal estrogen compounds, such
as PC spes, or by glucocorticoids (such as prednisone or
dexamethisone). The phase of so-called "androgen-dependent"
prostate cancer can be quite variable, lasting from only a few
months to as long as 18 years, in my experience.
Secondary Hormonal Therapy
There is a second phase in which the tumor can grow even though the
male hormone level is level is low. During this phase, the tumor
cells still can be controlled by discontinuing some drugs that were
being taken previously or by switching to a different drug. This
administration and withdrawal of hormones is called secondary
hormonal therapy. The principle behind secondary hormonal therapy
is that while the patient is taking a hormone (such as Eulexin) that
blocks the growth of the cancer, the cancer cells can mutate or
change in a way that makes the Eulexin stimulate their growth. In
this situation, simply stopping the Eulexin can have a beneficial
effect and may lower the PSA for a period of 3 to 9 months in 20% to
50% of cases.
After a while, the PSA will again
begin to rise. When this occurs, other similar drugs of the
antiandrogen family, such as Casodex, Nilandron, Megace, may be
started and the PSA will again decrease for a period of time. If
the cancer cells again mutate and again become stimulated to grow by
the new drug, the new drug may be stopped and again, in some cases,
the PSA will again decrease. In this way, it is sometimes possible
to use a variety of drugs, switching from one to another, to achieve
an additional response. Megestrol acetate should be avoided in
patients with heart disease, high blood pressure, or diabetes.
When antiandrogen drugs no longer
work, it is possible in some cases, to achieve additional responses
with other hormonal agents, such as Nizoral (ketoconazole), Cytadren
(aminoglutethimide), Stilphostrol (diethylstilbesterol diphosphate),
PC Spes (Chinese herbal estrogen preparation). Some of these drugs,
such as Nizoral and Cytadren require the additional use of
cortisone-like medications, because they suppress the production of
cortisol by the adrenal glands.
Hormone-Resistant Prostate Cancer
Even after prostate cancer cells reach the stage at which they are
no longer responsive to hormone therapy, they still may be sensitive
to treatment with radiation, chemotherapy or experimental drugs.
Patients may not develop symptoms of metastatic prostate cancer
(such as pain or weight loss) until the PSA has reached a level of
100 to 300. Some patients with very advanced disease have PSA
levels in the thousands. Each tumor is somewhat unique in its
behavior, as is each patient. Measurement of PSA levels is the best
way of determining the response of therapy. In general, if the PSA
level can be decreased by 50%, there is usually a prolongation of
Significant responses can be achieved with chemotherapy, although,
in general, chemotherapy is less effective and has more side effects
than primary hormonal therapy. However, there are some regimens
that are very well tolerated and offer reasonable prospects for
benefit. One such regimen is mitoxantrone plus prednisone. This
regimen may improve the quality of life, but does not appear to
prolong survival. Another combination that is also sometimes
effective is estramustine in combination with vinblastine. Other
combinations include etoposide, doxorubicin, and cyclophosphamide.
Recently taxol drugs (docetaxel)
have been combined with estramustine and hydrocortisone for men with
hormone refractory prostate cancer. One regimen combines these
drugs with thalidomide, which works as an antiangiogenesis agent
that blocks the growth of blood vessels that feed the tumor.
Spot radiation therapy can be
effective is reducing pain from bone metastases and protects against
the development of pathologic fractures. Biphosphonates (pamidronate
and etidronate) inhibit bone resorption and may also be used to
prevent fractures and to treat bone pain.
The goals treating advanced prostate
cancer are to maintain a high quality of life, relieve symptoms, and
prolong survival. These goals can require the use of other
medications such as nonsteroidal anti-inflammatory agents as well as
stronger narcotic medications.
Several new drugs and approaches are
under investigation. Further research will certainly bring new and
more effective treatments for hormone-refractory prostate cancer.
Q. What should
I do when my PSA level begins to rise after radical prostatectomy?
I believe
it is unknown where the cancer cells reside in my body. If they lie
outside of the prostate bed or pelvis, the radiotherapy will not
benefit me. How often does recurrence in the prostatic bed occur?
How many patients have lifelong chronic problems with the rectum or
bladder, including bleeding? Would a lesser dose of radiation
eliminate all of the malignant cells and completely avoid
complications? Is there any way of avoiding loss of sexual
relations after surgery and/or radiotherapy?
A. Depending upon the stage
and grade of the tumor, and the preoperative PSA level, a rising PSA
occurs after radical prostatectomy within 7 years in about 20% of
patients who were thought to have cancer that was contained within
the prostate before surgery. In about 70% of these patients, the
PSA level will return to the undetectable range following radiation
treatment to the bed of the prostate. Based on our data, the PSA
has remained in the undetectable range in about 70% of those
patients for as long as we have followed them (70% of 70% equals
about 50% overall long-term favorable response). These findings
suggest that the cause of the rising PSA is tumor cells remaining in
the bed of the prostate in about 70% of patients. About half of all
patients with a rising PSA level either have the tumor cells that
are outside the pelvis or have tumor cells in the pelvis that are
resistant to the doses of radiotherapy that can be safely given.
With postoperative radiotherapy,
there is concern that the bladder and rectum may be damaged, and
this does occur to a chronic bothersome degree in about 3% to 4% of
patients who receive the prescribed dose of 6400 cGy at 180 cGy per
day. Radiotherapy also significantly reduces erections in about
half of patients and may decrease urinary continence in a small
percentage as well. However, current evidence suggests that a
lesser dose that would completely avoid bladder and rectal
complications would be less effective in eliminating all of the
cancer cells. Therefore, the "worst case scenario" could have the
patient have chronic inflammation of the bladder and rectum with or
without bleeding and still have the PSA rise and have to be treated
with hormonal therapy as well.
For this reason, some patients who
have a very slowly rising PSA after a long interval following
surgery correctly assume that their tumor is not highly aggressive
and elect not to receive radiation therapy, but rather opt for
hormonal therapy either immediately or in the distant future, if
necessary. However, patients with slowly rising PSA long after
surgery are the very patients who respond best to radiation
therapy. So, it is a difficult decision to make. The patient must
balance the potential benefits of the radiation therapy completely
eliminating the cancer against the risks of long-term side effects.
Erections can be restored in nearly
all patients. This may be accomplished with oral Viagra, intra
urethral MUSE suppositories, injections of prostaglandin into the
side of the penis, a vacuum erection device, or a penile prosthesis
(surgery required). Although each of these options has its
drawbacks, the ability to have sexual relations may outweigh the
drawbacks for many patients and their partners.
Q. How does
the doctor determine how many samples to take in a prostate biopsy?
Initially, most doctors take six to
eight samples. However, it is known that the first biopsy procedure
misses the cancer in 20% of patients. Accordingly, in patients who
continue to have a rising PSA level or other features that are
worrisome for prostate cancer, more biopsies are taken during repeat
procedures. The exact number taken depends upon how uncomfortable
the patient finds the procedure and may include 15 to 20 samples.
Q. . Does the
natural size of a man's prostate have any relationship to later
development of an enlarged prostate or prostate cancer?
A. The normal prostate size
for an adult male is 15cc to 30cc (one half ounce to one ounce).
Prostate size may increase because of swelling from inflammation,
benign enlargement which usually begins at age 40-50, and cancer
which also is unusual before age 40-50. Accordingly, men whose
prostate gland is larger than 30 cc are more likely to be diagnosed
with either benign hyperplasia (enlargement) or prostate cancer than
those whose prostate is in the normal range.
Q. What is PIN
and is it a precursor to cancer? What is its relationship to
cancerous tumors and to the prediction for such tumors developing?
Prostate cancer is caused by changes
(mutations) in the DNA that affect the regulation of the growth of
normal prostate cells. It is probable that several mutations (at
least 5 or 6) have to occur before a full-blown cancer occurs.
These mutations may accumulate over time, causing the gradual
progression to prostate cancer. It is likely that certain mutations
may cause a precancerous condition called prostatic intraepithelial
neoplasia (PIN). PIN is also found as a satellite lesion
surrounding a prostate cancer or elsewhere in a cancerous prostate
gland. Approximately 30% to 50% of men who have an elevated PSA
level and high-grade PIN are subsequently found to have prostate
cancer within four years. Accordingly, repeat biopsies are
routinely recommended in men with PIN.
Q. Can the
amount of selenium in a man's body predict if he is more or less
likely to develop prostate cancer? Is there a test for such
predictions and how would the measurements be interpreted?
A. Selenium is a trace
element that is not routinely measured in blood samples to predict
prostate cancer risk. Studies suggest a 50% to 66% reduction in
risk for advance cancer in men with the highest selenium status as
determined by the level of selenium in toenail clippings.(Yoshizawa
K, et al. Study of prediagnostic selenium level in toenails and the
risk of advanced prostate cancer. J Natl Cancer Inst. 1998; 90:
Q. What is a
TUR or TURP and what purpose does it serve?
Transurethral resection of the prostate
is an operation performed to relieve symptoms of urinary obstruction
caused by either benign enlargement of the prostate or prostate
cancer. It is performed through a scope and involves removing
prostatic that is obstructing the flow of urine. During this
operation, the "resectoscope" is passed through the urethra into the
prostate gland. Then using an electrified wire loop, dozens to
hundreds of raisin-sized pieces of the prostate gland are removed
from the inside of the prostate gland and washed out through the
scope. This operation is often called a "Roto-Rooter" procedure or
a "scraping" of the prostate. It enlarges the urinary channel and
allows the patient to urinate more freely.
Q. What is the
pattern for return of sexual function after prostate surgery?
Usually erections return slowly beginning 3-6 months after surgery
and continue to improve for 18-24 months. There is a lot of
variation. Usually erections are not as firm as before surgery
unless the patient had "perfect" erections to being with. If the
nerves can be partially or completely saved, Viagra usually improves
the erections. If not, prostaglandin injections, MUSE urethral
suppositories, or a vacuum erection device must be used. Some
patients prefer to solve the problem with a surgically implanted
penile prosthesis.
Q. Regarding
my bladder incontinence, how much better control, can I expect to
have and is it going to change in dramatic steps? (Question from
someone operated on for prostate cancer)
About 92% of patients regain normal control. Of the remaining 8%,
most have mild stress incontinence that requires minimal protection
(a pad). Only 1 – 2% have severe incontinence or implantation of an
artificial urinary sphincter to correct. Usually, continence
returns quickly, but sometimes it returns slowly, over a period of
18 months to two years.
Q. How do the
exercises given after my operation work to help bladder incontinence
work by increasing the bulk and strength of the one remaining
sphincter muscle. This muscle helps to create a seal by
approximating the walls of the urethra. There is a fine line
between being wet and being dry. It is like a leaky faucet that can
be fixed by replacing a washer that differs very little in
appearance from the old washer. It is just a little more plump and
flexible, but that makes the difference between wet and dry.
Q. Please give
sources for information about cancer
A. The Cancer
Information Service (CIS) of the National Cancer Institute (NCI)
provides up-to-date cancer information and can answer questions (in
both English and Spanish) in nontechnical language. To reach CIS,
call 1-800-4-CANCER (1-800-422-6237) or visit
http://rex.nci.nih.gov, the NCI
web site. This site includes information on new and experimental
To contact the American Cancer Society (ACS), visit the ACS Web site
at http://www.cancer.org or call
Q. What about
freezing of the prostate as a treatment for prostate cancer?
A. It's an unproven
treatment. One of the real worries is that the freezing process
starts in the center of the prostate and goes out to the edge. But
most of the prostate cancers arise very near the edge of the
prostate. In order to protect the surrounding tissues (especially
the rectum) from freezing, doctors don't freeze out to the edge in
all area so many patients with the freezing treatment will have
recurrences of the cancer out near the edge of the prostate.
Available studies would suggest that freezing is not as effective as
surgery or radiation therapy.
Q. What could
cause a PSA level to rise other than cancer?
Three things can cause the PSA to rise: cancer, benign enlargement,
and inflammation (prostatitis). With cancer and benign enlargement
the PSA goes up persistently; the slope is steeper with cancer.
With inflammation, the PSA can rise with a flare-up and come back
down with resolution of the inflammation (with or without antibiotic
treatment). Interpreting the pattern is complicated because some
patients have two or all three of these conditions at the same time
Q. Can
frequent sex with a penile implant make a PSA level rise?
Sexual activity can cause a transient rise in the PSA level. This
rise is usually minimal and does not last for more than 6 – 24
hours. Sex with or without a penile implant does not make a
difference. It is ejaculation that causes the PSA to rise. It is
ideal to avoid sex for 24 hours before PSA testing.
Q. What is
nerve-sparing surgery and how does it work?
A. The
nerves are like railroad tracks and the prostate is like a boxcar on
top of the railroad tracks. In this surgery, doctors try to gently
life the "box car" off the railroad tracks without damaging the
"tracks". If the cancer is detected early, then the prostate can
sometimes be removed very nicely without disturbing the nerves. The
nerves usually get bruised and stretched somewhat during the
operation which is why it can take 3 to 6 months for erections to
begin to return after surgery and up to two years for complete
recovery, but with time the nerves regenerate so it's very important
to detect the cancer while still on the inside of the prostate
before it grows out into those nerves. After it's grown out into the
nerves, it's not safe to try to spare the nerves.
Q. What is so
special about the PSA test?
major problem with prostate cancer detection before the PSA test was
that men would not come in for a rectal examination unless they were
really having problems. But the PSA blood test is acceptable to
most men, and it's made prostate cancer detection more acceptable to
men. The PSA blood test is probably the most single accurate test
we have for the detection of prostate cancer.
Q. Why should
men come in for an examination if they take the PSA blood test?
There are some very early cancers that can be felt on the rectal
exam before they cause PSA level to be elevated. There are other
cancers, which will elevate the PSA level before they can be
detected on the rectal exam. To maximize the accuracy of screening,
it's best to have both the test and the examination.
Q. Can certain
vitamins or foods lessen the risk for prostate cancer?
A. Vitamin E
has been shown to improve the effectiveness of the immune system ad
to lower the risk for prostate cancer. Usually the dosage is 200 to
400 units a day. Selenium, taken 200 micrograms a day, has been
shown to lower the risk of prostate cancer. A low fat diet,
especially a diet avoiding animal fat, specifically red meat fat,
has been shown to have an impact on the risk for prostate cancer.
Eating fruits and vegetables is important and it's been shown that a
diet rich in tomato products in also helpful in avoiding prostate
cancer. One of the substances in tomatoes is called lycopene and in
order to get enough of this substance one would have to eat pounds
of tomatoes each day. But now lycopene has come into the market in
pill form. The above recommendations have been evaluated in
published scientific studies. Obviously, many other substances
could be effective but they just haven't been adequately studied at
this time to know if they are helpful or harmful. All of the
factors discussed above are believed to produce relatively minor
effects, and it's still very important to get the PSA blood test and
the prostate check.
Q. What is
external beam radiotherapy and how are its effects different from
those of a radical prostatectomy?
A. In
this method, X-ray treatments are given with a machine that sends
high-energy x-rays through the patient's body, aimed at the prostate
gland and sometimes the pelvic lymph nodes. External beam
radiotherapy is a totally non-invasive treatment used as an option
for men who are considered too old or too ill for a prostatectomy or
who just don't want surgery. It is difficult to determine whether
external beam radiation actually eradicates all of the prostate
cancer, because many patients in whom progression of the tumor is
slowed or halted have what appears to be persistent tumor on
rebiopsy. The best results are obtained when the tumors are less
than 2 centimeters in size at the time of therapy.
Q. What is an
abnormal PSA level and what are grade levels for prostate cancer?
Most doctors would say the PSA level is normal if it's below 4 and
above normal if it's above 4. We feel that the cutoff of 4 is to
high and we have used 2.5 as the cut off in our PSA screening study
since 1995. We have proven that if the PSA is between 2.5 and 4,
there is a 22 percent chance that the man will have prostate
cancer. We routinely recommend a biopsy if the PSA is above 2.5 and
if the free PSA test confirms the advisability of performing a
biopsy. Prostate cancers when looked at under the microscope can
look like almost normal prostate tissue or they can look very wild
and disorganized. They are graded on a scale called the Gleason
grade of between 2 and 10 where 2 is the best, i.e., a low grade,
slow growing prostate cancer, and a 10 would e the worst, i.e., a
rapidly progressing, very aggressive prostate cancer. It turns out
that grade level statistics for prostate cancer fall into a bell
shaped curve. Only about 10 percent of tumors are grade 2,3 and 4
and only about 10 percent of tumors are 8,9 and 10 with the vast
majority being Gleason grades 5,6 and 7.
Q. What is the
free PSA test?
A. One
of the problems with PSA blood tests is that if the PSA level is
elevated, only about 25 to 60 percent (depending on how high the PSA
level is) of those men are actually found to have prostate cancer.
The flip side of that fact is that roughly 40 to 80 percent of men
who have elevated PSA levels don't have prostate cancer and they go
through the biopsy unnecessarily. The free PSA blood test can
eliminate at least 20 percent of these unnecessary biopsies and
still detect 95 percent of these cancers. Another new test is
coming down the road in a year or two called the HK2 test which can
further eliminate unnecessary biopsies, especially in men with PSA
levels between 2.5 and 4. A lot of medical research is being done
to improve the accuracy of PSA testing to avoid biopsies in men who
really don't have prostate cancer and so don't need a biopsy. But
it's always going to be an imperfect world, and it is better to
accept some false alarms to save some lives than it would be to
avoid all false alarms and not save any lives.
Q. Why do some
men not get treated for prostate cancer?
Part of the reason is that they misunderstand some of the public
information about prostate cancer. Some studies have been presented
in the media and lay press that suggest prostate cancer is a
toothless lion, that more people die with it than of it, and that
prostate cancer does not require treatment. So, not wanting to be
treated, some men latch on to the position of watchful waiting or
diet therapy only. Watchful waiting is appropriate in some
patients, but it's generally only for older men with low-grade
tumors that are not very threatening.
Q. Is the PSA
reading different for a man before and after having a prostate
cancer removed?
is a low PSA reading for a man with a prostate, but it is a high
reading for a man whose prostate has been removed. It means there
are cancer cells left behind that are producing PSA.
Q. Is it true
that all men at some pointing their lives will develop prostate
A. No,
even though that is often said. It is true that if the prostate
gland were removed and carefully examined under the microscope, up
to 80 percent of men in their 80's would have some microscopic
traces of prostate cancer, but only about 15 to 20 percent of men
area actually diagnosed with prostate cancer during their lifetime.
Prostate cancer will be large enough to detect with a biopsy in only
10 to 15 percent of men.
Q. Do
vasectomies have any effect on prostate cancer?
was stated in the past that men who had vasectomies were at an
increased risk for prostate cancer, but this subsequently has been
disproved. It was a myth. What seems to be true is that
vasectomies are more likely to be seen by a urologist and if a
urologist sees them, then they are more likely to be screened and
have early detection of prostate cancer.
Q. Is prostate
cancer a fatal disease if not treated?
rostate cancer kills more men than any other cancer except lung
cancer. In some patients, particularly elderly patients, the
prostate cancer might progress very slowly, and they may not need to
be treated right away, but most men who have a life expectancy of at
least 10 years need to have treatment or they are risking death from
prostate cancer.
went to see a urologist a couple of months ago for symptoms of
enlarged prostate. After the PSA and rectal examination, my
urologist told me that nothing needed to be worried about and all I
need to do was to come back for yearly examination. My urination was
not too bad and there is little inconvenience in daily life.
However, the major impact is sex. For the past two years, there has
been a rather quick deterioration. The sensation of ejaculation has
mostly lost and the volume of semen has become quite reduced. I
mentioned this to my urologist, but he told me that I should not get
too concerned as long as I still can have sex. I am 51 and have a
younger wife. I feel I should do something about it. Is it true that
people with prostate enlargement normally have pretty bad
deterioration in sex pleasure, even in pretty early stages? Is there
anything I can do about it?
A. It is true
that beginning around age 50, many men begin to develop overgrowth of the
central part of the prostate gland, called benign prostatic hyperplasia.
This can cause compression of the prostatic ductal system that
carries the prostatic fluid from the glands located in the
peripheral regions of the prostate to the urethra, which is fairly
centrally located. Because 90% of the ejaculate is composed of
prostatic fluid, it is common for the volume of the ejaculate to
decrease in men with benign prostatic hyperplasia. This condition
does not cause a decrease in sex drive or erections, however. There
are hormonal, blood vessel changes, and other diseases such as
diabetes or high blood pressure that can occur around age 50 that
can cause decreased sexual function. Other possibilities are
medications that may interfere with sexual function. The best advice
you can be given is to find a urologist whom you have confidence in
and follow your prostate closely with PSA tests and digital prostate
William J.
Catalona, M.D.,
Real Estate
information contained in this site is not up to date,
Consult a professional
ProstateAction.com is NOT a medical
website. It was developed to provide what we hope
will be useful information for men who have been diagnosed
with prostate disease…and their family members. We do NOT
have doctors to answer your questions, we do NOT make
medical referrals or offer second opinions, and we will
not reply to questions about any specific case. Instead, we
hope that you will use our LINKS section to locate other sites
of interest; utilize our message boards to discover prostate
cancer screenings and related events; and to use the Forum
area to "discuss" prostate cancer issues with others who share
your interests and concerns. We reserve the right to delete
any objectionable postings.
health and medical information on the World Wide Web comes
from many sources and changes daily. There are likely to be
errors and omissions in this information. This web site, its contributors nor its sponsors represents or
warrants that the information in this Web Site or accessed
through this Web Site is accurate or complete.
direct your medical and health questions to your health care
provider.It is our objective to promote an exchange of information
about prostate health. We do not endorse or recommend specific
medical treatments, but we encourage visitors to our site to
explore a variety of points of view.
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text:
The Arsenio Hall Show Canceled
Farewell, sweet dog pound.
Skip to content, or skip to search.
Skip to content, or skip to search.
Farewell, sweet dog pound.
It's his biggest pet peeve.
Andre announced the news on The Arsenio Hall Show, before taking his clothes off.
The first episode of The ArScheerio Paul Show.
It's ... happening!
To succeed, she needs to be liked; to be liked, she needs to temper her success.
We ♥ the nineties.
In the fall of 2013.
Plus: Take Your Child to Work Day was awkward for Matthew Morrison, whose father is a midwife, and more, on our daily late-night roundup.
He'd like one please, thanks.
Live on 'The Arsenio Hall Show'!
Only Arsenio Hall had the kind of chops to pull something like this off.


text:
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Catalogue No: LL259
Shop Product Code: 256381N
Usually despatched within 24 - 48 hours
Department: Education & Exams - Piano Exams: LCM
Publisher: London College Of Music
This product includes pieces featured in grade exams. Click on a grade to view the full syllabus.
|LCM Piano 2013-2017
* Estimated prices
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text:
The Romero Laced
Black Black$54.99
This updated version of the classic Emerica Laced was designed, tested and 100% skateboarding approved by Emerica pro Leo Romero.
Smooth Suede
- Tongue stabilizing wing for a snug fit if you decide to grind up a rail. Boo-yah!
- Low-profile cupsole for increased board feel.
- Emerica Triangle Tread pattern with high-density rubber heel drag pod for maximum grip and durability.
- Item code: 6102000089/003
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text:
WASHINGTON - Atticus Lane-Dupre, an 8-year-old with cancer, made his wish, and the Portland Timbers fulfilled it.
The boy found out about his cancer in 2011 and missed the last game with his soccer team.
As part of wish through Make-A-Wish Oregon -- and before a crowd of more than 3,000 at JELD-WEN Field this week, the Portland Timbers faced off with The Green Machine, Lane-Dupre's team.
Lane-Dupre netted four goals -- including the game-winner -- in the 10-9 victory.
Check out the video:
© 2013 WTOP. All Rights Reserved.


text:
The Good Wife Recap: Current Affairs
When you're writing a show based on a politician's downfall, there must be no greater pain than watching a germane and spectacular scandal happen while you're on summer break. Anthony Weiner's Twitter-gate began just ten days after The Good Wife season-two finale aired and became old news two months before the start of season three. You can almost hear the frustration in this week's episode, when Eli's new political strategist frenemy Mickey Gunn (played by the vibrant Michael Kelly) asks where Eli was when that mess was going on, and the only response Eli can muster is, "God, the day politicians discovered Twitter
" Guess it made more sense than having him go into the copy room and rage about that ill-timed summer hiatus.
It wasn't until the subject of Weiner came up that it became clear how far beyond anything else on television The Good Wife is in terms of topicality. Not since The West Wing has a show so artfully weaved a fictional world into the current news cycle. So if last week's attempt to tackle the Arab Spring, Israel-Palestine relations, and gay college romances gone wrong all at once came out as confused and overly ambitious, at least it comes from a good place. The Kings had to have been chomping at the bit of current events.
This week, current events, specifically the 2012 presidential campaign, play much better, since they have finally (finally!) brought together a pairing hotter than Will "going hard on" Alicia: Eli and Kalinda. Is there anyone whose breath didn't shorten when Eli asked Alicia, "Who's the investigator around here?" It makes sense that they've never met. When would they have? Kalinda worked for Peter before Eli did, and they hadn't had occasion to cross paths at the campaign office or Alicia's apartment. Everything about their first interaction pointed to the best chemistry ever on a show known for great chemistry. Eli enlisted her because he needed to pitch Mickey on how to manage a scandal that Mickey would tell him nothing about. It took Kalinda two seconds to figure out Eli's motives: "You want the information so he's forced to hire you or you'll expose it." When Eli asked her, in awe, "How have I never met you before?" he seemed to be speaking for all of us.
There is so much potential in this coupling built on a mutual love of finding dirt and winning. It is, of course, not sexual, but if their banter — Eli: "I'll need you again" Kalinda: "Then invoice" — isn't foreplay of some kind, I don't know what is. They have wonderful odd couple rhythms, like when Eli told Kalinda "Mazel tov" and she stopped dead in her tracks, having no idea what to do with that. But Eli keeps asking Kalinda where he knows her, and that is troubling. Did they cross paths in another context? And how will Eli react when he finds out his new ally is the reason why his future governor and Alicia are no longer together, a possible catastrophic revelation for Pete's gubernatorial campaign? There was also a moment when Will was marching through the office going, "Where's Kalinda?" and she was with Eli. Given their bonding session over being emotionless voids at the bar last week, and how many times Will has fought to get Kalinda to stay at the firm, there's no way Will and Eli aren't fighting over her in the future. Or that Will isn't going to have a freak-out when he sees how much extra cash Kalinda is making from invoicing Eli. Didn't she just get a massive raise?
The introduction of Mickey to the Eli posse was also pretty intriguing, and not just because he was a guy who claimed to have no money for a campaign but was somehow able to spring for a couple of burly actor colleagues to come to meetings with him in an attempt to intimidate the un-intimidatable Eli Gold. Mickey is a delightful snake; he tricked Eli into vetting his candidate for him by hinting that he'd hire him if he could find any skeletons in the guy's closet. Eli may not like being used, as he said last episode, but he seems to respect those who can (almost) pull one over on him. As Eli and genius Kalinda figure out, Mickey's candidate is not local, but presidential, and he's (gasp!) a Republican who seems to be a rarity among politicians in that he has not slept with any nannies or interns yet. We see Kalinda showing Mickey a picture of the guy but don't see the photo ourselves, which is a nifty trick by the show to not get locked into casting. Mickey also slips into a New Orleans drawl and starts going on about crawfish, which must mean he's based on a young James Carville. In that, there's potential for a Mary Matalin, sleeping-with-the-enemy story line. Or, better yet, perhaps the story line will be with Eli and a Republican lady. Oh, sweet anticipation!
The Mickey interaction was also useful in that it gave a number of insights into Eli. Eli isn't getting laid and Mickey hasn't gotten laid since the summer of '08, which would be during the Obama campaign, apparently the last time any Democrat in politics wasn't in a sexless, double-dip recession mood. Eli and Alicia are still allies; he makes her race back from court for a "meeting" with Mickey that consists of only walking in the room so he can see that Eli has "Saint Alicia" on his team. How that would be a boon to any candidate who isn't Peter is a bit of a mystery. We learn how Eli would have handled the Weiner scandal, by getting out front and capitalizing on the "entertainment value" of someone who can Facebook message so well about "Jewish girls who suck " you know the rest. The only false note is when Eli says he'll join up with the campaign, provided Mickey gets hired, because "I don't mind this guy [the candidate]. He's not bad." Why would Eli blow his wad like that after having just told Mickey he would only reveal what he found out about the candidate if Mickey hired him? And if Taub 2.0 and the Jewish community were furious with Eli for working on a pro-Muslim campaign, what kind of wrath would rain down on him if he tried to get a REPUBLICAN elected PRESIDENT? Would he really risk getting blackballed like that?
Meanwhile, New Alicia is still kicking ass in court. She gets a libel case dismissed in the first few minutes of the episode, but not before whispering to her client, "Nothing personal, but I have to rip your friend apart." The case is basically the controversy surrounding Into Thin Air with a few twists. The author of the book, Danny Lambros, was climbing Mount Everest with his brother, a mountaineering guide, who died in the "death zone," the area above 26,000 feet where oxygen is not sufficient to sustain life. Rich guy Oliver Cardiff (yet another Wire alum, John Doman) sues Danny for having written that he stepped over Danny's dying brother and took his oxygen tank on the way to the summit. Since the oxygen-deprived "untrustworthy perceptions" of all witnesses can't be proven false, the book isn't libelous. Then Eddie Izzard walks in and there's no way this case is over that quickly.
What follows is basically a big fat excuse to give Izzard some great lines as barrister James Thrush, as well as give us didactic lessons on the legal quirks of England. British libel laws are the opposite of American libel laws; it's up to the person libeling to prove what he says is true. And since Izzard, uh, Thrush, bought the book on Amazon, and since eighteen copies have been sold in England, all bought by Cardiff's staff, he can sue Danny in British court for 20 million pounds. It turns out there are two different classes of lawyers in the U.K.: posh, bombastic barristers like Thrush, and the work-a-day solicitors like the marvelous underdog Timothy Ash Brannon (Simon Delaney) that Danny hires, who may not have fancy credentials but does have an impressive nervous habit of making up anagrams of people's names. And since Danny's business and wife's house are in Toronto and it's a British commonwealth, the suit has frozen his assets.
The deposition, or letter rogatory, takes place over video with a judge who proves perhaps the most annoying of The Good Wife's quirky judges, demanding the lawyers call him "Your lordship," and greet him with "Good morning" even though it's evening in Chicago. Will blows it by doing his usual American pontificating, leaving the more deferential Alicia and Brannon to take over. They touch on the issue of "libel tourism," which is apparently something Roman Polanski and Arnold Schwarzenegger did. And we learn what I assume is true, that Parliament was going to change their libel laws to eliminate libel tourism, until the recent News of the World brouhaha — yet another current event the Kings are probably kicking themselves for not being able to address over the hiatus.
Thrush is pure asshole, consistently undermining Brannon. Then he wanders around Will's office carrying a baseball bat and giving a speech prompted by nothing about how he's not the England of "doilies and cucumber sandwiches," but is instead the England of "football hooligans and Jack the Ripper." It just seemed like an excuse to give Izzard something to do since he's likely never coming back; it's not like Lockhart Gardner are going to be trying a series of cases by video conference in the U.K. Thrush's other main function is to spar with Alicia, which turns Will on with "American revolution" fantasies. She playfully offers to dress up. He wants to "take" her in his bathroom on a ten-minute break, but Diane spots them, though she doesn't suspect anything yet, and inadvertently spoils the plan. Between that and their brazen fondling of pinkies during the deposition, they're asking to get caught.
Lockhart Gardner wins points by playing a tape of the dying man talking on satellite phone to his wife, who creepily recorded the conversation so their son can listen to it one day. Then Alicia discredits a female witness whose vision was blurry from having gotten Lasik right before the climb; why she shows up to court wearing glasses is never explained. The case seems almost won when they hit another impasse. Though Cardiff allegedly did the same thing to a dying Japanese climber on Trango II in Pakistan, he has, through another quirk of British law, won a superinjunction against Trango II ever being mentioned or even discussed. "It is as if the book [about Trango II] and its events and even events questioning the events never happened," explains Thrush. "We would ask for a superinjunction to prevent the mention of the superinjunction." The only way to discuss the events of Trango II in court is if they are a matter of discussion in the British press. Alicia takes care of that by getting Eli and his "geek squad" of "Internet trolls who tweet anonymously" (Becca????) to take up the cause. Brannon questions Cardiff and gets him to admit that he did indeed bypass a dying Japanese climber and take his gear. And since Cardiff is about to head off on another mountain-climbing trip to Patagonia, the book isn't subject to libel laws since it serves as a warning to future climbers of Cardiff's pattern of abuse.
The warning flags don't go up nearly fast enough, though, in Diane's section of the office where Peter has asked the firm to interview for being the state's attorney's outside counsel on civil matters. It would bring in $20 million a year and Diane wants to go for it. Her first hint at something amiss should have been when Will told her she ought to do the pitch without him. She's worried there's something he isn't telling her; she doesn't want to get blindsided. "Have you noticed you're turning into me? All those sports metaphors," Will asks. "I'm a better version of you," Diane rebuts. They have a nice laugh.
But when she gets to the SA's office, she is indeed blindsided. What starts out as a civil conversation turns into one filled with menace, as Peter and Cary ask Diane to voluntarily submit the firm to an official audit by the state board of taxation in order to prove that they're not running off of Lamond Bishop's drug money. The scene is masterful, with Noth playing the bomb dropping perfectly; there's no menace in his tone, and he seems reasonable and friendly, but as the camera does a well-timed zoom in on Christine Baranski's face, its clear she knows that something isn't right.
But she suppresses her better instincts and goes back to Will to see if he has other ideas. He suggests asking Peter if he'd accept an audit by an independent accounting firm. "Let's not expose ourselves on a subway platform unless we have to," says Will. "What a colorful and pointless metaphor," Diane replies. They have another laugh. But Peter wants a departmental audit only, and he hasn't asked any of the other two firms up for the job to do the same. Diane says she'd never butt into Alicia's personal life, but does just that, dropping by her apartment unexpectedly, transparently looking for a clue to what's going on in her marriage. She'd called the office and Peter said he was home; that he's not there sends up red flags. Alicia knows she's been made but refuses to answer direct questions about Peter. So Diane tries another tack: "If you were assisting me in this decision, how would you advise? Agree to the audit or not?" Alicia the Moral advises not, and the look on Julianna Margulies's face at that moment expresses exactly why she got that Emmy: She's humiliated, her job is now at stake, her boss sees her as a liability, the husband that she kicked out of her life is still controlling it from afar, and she's just found out that Peter has moved on from hurt to vengeful.
Diane turns down the offer to get audited. In her mind, Alicia is why the firm just lost a potential $20 million. Alicia's relationship with Peter was supposed to benefit the firm; it's why they kept her instead of Cary, and now they're losing money and they may soon start losing cases. She gets Will to agree to let Alicia go if things get worse. But what will Diane do when she finds out that Will could soon make Peter even more wrathful? Could this be the real beginning of the end for their partnership?


text:
ISPConfig Quota problem
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25th October 2009, 03:27
Join Date: Apr 2009
Posts: 33
Thanks: 7
Thanked 5 Times in 4 Posts
try this
Hey kextra1, i just noticed u were logged into my VPN, ill shell into your dedicated server and take a looksie of that and ur ispconfig.
FYI for falko, yes repquota -avug shows the correct value for the web which im assuming is 15gigs set by kextra1, but for the actual ispconfig user which is the admin for that web it shows a quota of 10mb.
kextra1, I dont want to change any of your configuration without permission however here may be something.
Have you checked /etc/quotagrpadmins to make sure the user and group are set to root root?
I dunno, im not to smart with quota systems, falko would be the better advisor than me.
I hope this helps!
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text: Last year the staff did a great job in identifying a QB early, pursing him and getting a commit before he blew up. I believe that the staff should and will do the same thing this year. The state of California is loaded with QB's in 2015 and those prospects are getting a ton of interest. Hopefully we stay out of that mess and just find one of the best QB's in the south. We don't need to get the number 1 QB in the nation we just need to make sure we get a top 15 or 20 QB. The staff needs to make sure we are not taking the 30th or 40th best QB as a fallback plan like we had to do for the last few years before Grier. Here is a list of some names we might hear over the next few weeks. Christian Alexander Lakeland Christian High School - Lakeland, FL Threw for Pease last year at camp and did the same this weekend 6/8. Alexander is coached by former Gator QB Wayne Pease http://www.hudl.com/athlete/1385399/highlights/21839375 Tyler Queen 6-3 233 Kennesaw, GA (North Cobb) Joker visited him during spring evaluations he has offers from Bama UNC Clemson Auburn. Will throw for the staff sometime this week. http://www.hudl.com/athlete/419536/highlights/39039393 Chris Riddle Just transferred to Sandalwood he threw For Pease this weekend. TBH not a fan from the film I have seen http://www.youtube.com/watch?feature=player_embedded&v=EQl5SeTY3fE#! Brett Rypien 6'2 174 Shadle Park High School - Spokane, WA Pease went to see him during spring evaluations. http://www.hudl.com/athlete/451767/highlights/27234377 Just throwing a few names out there James Pensyl Land O' Lakes FL 2015 6'5-202 http://www.youtube.com/watch?feature=player_embedded&v=aa2ZuZIAONY Brad Mayes 6-2 180 Tampa, FL (Berkeley Prep) http://www.youtube.com/watch?feature=player_embedded&v=rjirlKkM-hI Drew Lock 6-4 175 http://www.hudl.com/athlete/403144/highlights/38722381 Kevin Johnson 6-0' 171 Atlanta, GA (Pace Academy) http://www.youtube.com/watch?feature=player_embedded&v=C25mMKNOjGo


text:
Current Articles
By Darin Fager
-- About a month before I found myself with a free afternoon to bowhunt, I spotted a massive 8-pointer while driving past my father's ground on the way to pick up some flu medicine for my 3-year-old. The buck was about 150 yards off the road.
I pulled over to get a better look at it through my binoculars, but it was too dark to actually count points. Even so, I saw enough to know he was worth hunting.
The big whitetail was in an area that would be almost impossible to hunt with a prevailing west wind. The only way around it might be to erect a ground blind, which normally isn't my cup of tea.
I didn't quite know what to do, and I was chasing other deer. So I really didn't do anything at all.
About a week later, my friend Praveen saw the buck at dusk in another area. The news was encouraging, but I still wasn't sure where he would be each night.
Another week later, my friend Matt saw him again in the same spot. And it was seen there twice more on Oct. 5 and 6.
On the night of Oct. 6, we went out to dinner and started plotting. The next day, a Saturday, I met Matt at the field and we went to where he and Praveen had been seeing the deer. We found half a dozen scrapes within 80 yards that were at least 36 inches in diameter.
The smell of buck urine was unmistakable.
To add to my excitement, the wind that day was perfect for hunting there. As Matt and I were leaving, I started thinking about how nice it would be to find a babysitter and go hunting that evening. I had never had a buck patterned like that, with so much fresh sign and a perfect wind.
Well, everything fell into place. In no time at all, I was taking my scent-free shower and rushing out the door. After I got all the way to the hunting property and got all my gear out, I realized I didn't have my arrows. I was making pretty good time to that point, but I lost precious minutes when I had to go back for them.
While on my way home, I thought to myself that this might be a good omen because things like this have happened to me before … Just when I think everything is starting off completely wrong, good things happen.
After about 30 minutes, I was back at the field and walking to the tree I had picked for my climber. It was about 70 degrees, and I had some trimming to do.
As I got situated, I realized that the wind had picked up, though it was blowing steadily from the same direction. At 4:43 p.m., half an hour before legal shooting time would expire, I got a text from Matt, asking how things were going.
At that very minute, I looked down the tree line and saw this buck walking straight toward me at 40 yards. He was in the woods instead of the bean field, probably because it was so breezy. He walked to within 30 yards and stopped.
He stood there for about two minutes, looking my way, and then he bedded down. I immediately got a lump in my throat. This was definitely not a good thing: bedded down, facing me, 25 more minutes to shoot, and windy. I sat ready to draw for about 10 minutes, wondering what was going to happen.
I finally decided to draw slowly, and then grunt as softly as possible, hoping he would stand up and present a better shot. I also was concerned about possibly spooking him. Only 30 yards separated us!
Before I could execute my plan, the buck heard a noise or something behind him and looked away from me. That was my chance to draw and shoot. Even though he was bedded down, I believed I could make the shot and sent the arrow flying.
I thought it hit its mark, but I was not sure. (I actually hit about 6 inches higher than where I was aiming). He immediately jumped up and ran downhill toward me about 15 yards, broadside. He looked like he had just had the sense knocked out of him.
He stood with his legs spread apart, looking dumbfounded. I nocked another arrow and put it through both lungs.
He took two more steps behind a bush, and I nocked again and sent yet a third broadhead in his direction.
He expired about 16 yards below me.
I was terribly excited and couldn't wait to get down and see him up close. He was a symmetrical 4x4 with a split brow that scored approximately 140 inches by Buckmasters and 149 4/8 inches Pope and Young. I was on cloud nine!
This is the first time a hunt like this came together for me on the first try. But, in hindsight, there were many things that fell into place that seem very clear now.
The key factors were locating a mature buck, patterning him, finding fresh scrapes, timing (two weeks before the rut), a helpful wind and one mighty dose of luck.
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Working with Muslim Clients
|Shehzi Yusaf, Clinical Psychologist
|This seminar will assist psychologists specialising and interested in counselling / working with Muslim clients. It will give psychologists an awareness of the issues that Muslim clients face. We will discuss the issues encountered, and the skills and attitudes required to effectively provide psychological services.
This session will be case study driven and there will be time for discussion and review with a panel including Jasmine Sliger; Cross Cultural Counselling & Organisational Psychologist who will facilitate the workshop and outline the challenges of working with Muslim Clients and Trisha Carter Cross Cultural and Corporate Psychologist
The learning will achieve the following outcomes; Participants will:
1 .Learn about the basic tenets of Islam.
2. Gain understanding of the challenges that face Muslim populations in Australia
3. Recognise key issues faced for a psychologist working with Muslim clients
4. Understand how mental health is perceived for Muslim populations
5. Gain an awareness of possible assessment approaches /tools
6. Identify therapeutic approaches that may be more helpful with clients where different values around gender and religion are critical issues.
|About the presenter(s)
|Shehzi Yusaf, a clinical psychologist and convenor of the Psychology from an Islamic perspective Interest Group will be our speaker.
|NSW Metro, Australia
|Rydges Parramatta,
116 James Ruse Drive, Rosehill, Parking Free VENUE MAP http://www.rydges.com/hotel/RNPARR/Rydges-Parramatta-Sydney/location.htm
Please contact the event organiser to confirm if this venue is wheelchair accessible
|Start/End Date
|20 Nov 2012
|5:30 for a 6pm start
|$35 for members, $45 for non-members
|IG - Psychology & Cultures Interest Group (NSW)
|Contact Name
|Jasmine Sliger
|02 9417 6211
|02 9417 6911
|Register online


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Imperial Las Perlas Reviews
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Yeah, located between town and hotels. Off everything. Breakfast was ok, but don't exspect too much. Beach was beautiful. Our toilet was dripping, Service was ok. Overall ok.
- Novice Nomad
- magnusjagla
- Germany
- All Male Group, 25-30
- (3 reviews)


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Dancing Flag, "Happy 4th of July!" - 11K
|ARG! Cartoon Animation
75 Commerce Street
Colorado Springs, CO 80907 USA
(720) 989-1251
Copyright © 2013 ARG! Cartoon Animation.
Permission granted for use of dancing flag, happy 4th of July cartoons on non-commercial web pages with a credit and
link to www.artie.com. Updated 2/26/2005.


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