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Mount Rainier fire kills one, injures firefighters
Updated: July 3, 2013 - 12:50 pm
MOUNT RAINIER, Md. (AP/ABC7) - Prince George's County fire officials say a 70-year-old man is dead after a fire in a Mount Rainer home.
Firefighters called to the home on 36th Street on Tuesday night found fire showing from the third floor and received reports that a person was still inside.
Fire officials say the man found on the top floor was unconscious and suffering from severe burns and paramedics pronounced him dead on the scene. Officials say two other residents heard the smoke alarm and were able to escape unharmed.
Officials say one firefighter sustained second-degree burns to his upper body and another injured his knee when a ladder gave way. Both were treated and released.
The cause of the fire is under investigation.
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Ask TOH users about Befores and Afters
Something for Everyone
The kitchen lost its dropped ceiling as well as its back stairs, while gaining 30 square feet in the form of a cantilevered bumpout—puzzle piece number two. The tiny add-on required no foundation yet was "the small release valve that made everything work," says Clements. Scott, who likes to unwind from his tech job by reading cookbooks, gained extra prep space, a supersize German fridge, and a spiffy induction cooktop. The kids got personal cubbies—meaningful real estate, since they share a bedroom—with hooks for lunch boxes and backpacks, and bins for stray cell phones. "We didn't have space for one big mudroom," Clements explains. "So we figured out what they needed and spread it out."
Shown: Bumping out the sink wall eased traffic flow around the generous new island.
Paint: Benjamin Moore's Sea Haze (walls) and Dill Pickle (island)
Cabinets: Bellmont Cabinet Co.
Cooktop and Wall Oven: Thermador
Range Hood: Vent-A-Hood
Diswasher: Miele
Refrigerator and Freezer: Liebherr -
This Old House To Go

subset: commoncrawl


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I feel vindicated.
I've been saying for a long time that SSBB is being overhyped and doesn't have the universal appeal that the fanatic fanboys claim it does. "No, it's not a fighting game!" "You just don't understand!" Whatever.
A friend of mine got it recently so I'm going to check it out this coming weekend, but so far the reaction from the people I know has been "meh".


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Florida Protective Orders Laws
More Information on Protective Order Laws
Code Section | 741.30 & 31 |
Activity Addressed by Order | Exclude from dwelling; enjoin contact; regarding minor children: grant temporary custody visitations, temporary support; counseling; restrain from committing any acts of domestic violence |
Duration of Order | Ex parte temporary: maximum 15 days. General protective order: maximum 1 year, can reapply but maximum an additional year each time |
Penalty for a Violation of Order | Misdemeanor in 1st degree |
Who May Apply for Order | Any family or household member who is a victim of domestic violence or one who has reasonable cause to believe he or she is about to become victim |
Can Fees Be Waived? | Yes |
Order Transmission to Law Enforcement | Within 24 hours of issuance |
Civil Liability for Violation of Order | Civil contempt |
Note: State laws are constantly changing -- contact an attorney or conduct your own legal research to verify the state law(s) you are researching.
Research the Law:
- Official State Codes - Links to the official online statutes (laws) in all 50 states and DC.
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Full story: El Paso Times
Comments (Page 933)
Judged: 1 1 1 |
Judged: 1 1 1 Not enough, cut off the pay to Congress, President, and all their staff. They have'nt done anything for the last 5 years, but spent more then they have for all the lazyasses. |
Judged: 1 1 1 I don't know if we are saving much of anything as long as the do-nothing Congress and pResident are drawing paychecks. I just hope the crybabies and drama queens that run our government get their crap together before deer season. I like to hunt in a nearby National Forest. |
Judged: 1 1 1 Thanks for agreeing with me you flaming white trash homosexual. Now be a good bitch and stop trolling after my ballsac ya joto. Go play with you daughter, she used to you bothering and molesting her. signed lower valley dave |
Judged: 1 1 1 Someone needs to put some antlers on nashville dc dave and take him out hunting, not humping. |
Judged: 1 1 1 If you go camping and wake up with vasoline on your butt hole, will you tell anyone? I know your answer is NO. lets go camping. I'll show you how to pitch a tent, and we can find some wood. |
I you went camping and woke up with vasoline on your butt hole, would you tell anyone? I know your answer is, NO. Lets go camping, I'll show you how to pitch a tent, and we can look for wood together |
Judged: 1 1 1 If you went camping and woke up with vasoline on your butt hole, would you tell anyone? Your answer would be NO. Lets go camping. I'll show you how to pitch a tent, then we can lok for some wood together. |
United States |
Judged: 1 1 1 What kind of loser third world homosexual still uses AOL? Are you still living with your mommy in her basement? Man stfu you putrid fairy punk! |
United States |
Judged: 1 1 1 Stop stroking my balls ya fairy. Because of your queerazz, now everybody wants to be like me, DC Dave. |
Padre Island Ntl Seashor, TX |
Judged: 1 1 1 Looks like you spooks are going to have to handle Whitey and Brownies all by yourself. Homeland security is out of business for the time being. Good luck spook, you will need it. |
Judged: 1 1 1 I am DC Dave, not you ya smelly snatch queertranny. Always stalking my balls, wanting to get teabaged by my sac, ya pin head vendido joto. |
Judged: 1 1 1 |
Judged: 1 1 1 What a horrible example of a human being much less an American and a governor? My god, America is lost and upside down. The end is near, no it's here. If I had complete control, I'd rally the Mexicans up through Arizona and then head them west and line up across the northern border of Texas. Then I would have the federal Government arm them with automatic weapons and the ammunition that the NRA says I have stockpiled. These Mexicans could earn their citizenship by guarding the border of the penal colony or republic of Tejas (we can think of a name earlier). Those Americans who want out will have their head examined for the tea party gene and then if they have some brains, we can help relocate them comfortably inside the new America , where stupid is a crime...... From then out, we just ship stupid to Tejas and we welcome the primarily Humble God-fearing catholic Christian Mexican Americans who rather than try to tear America down, are so happy to be here that they will make excellent citizens and superior guards. They will have full knowledge that it was those whom they are guarding, were the ones who wanted to shoot them. Men women and children as they crossed the Rio grande. Jmo, |
Padre Island Ntl Seashor, TX |
Judged: 1 1 1 If you had complete control you'd rally the mexicans up through Arizona and then head them west and line up across the northern border of Texas? Your plan sucks just like you, you ignorant buttwipe. If I were to go up through Arizona and head west I would end up in California. Once going west to California the government could arm them with pea shooters, because California wouldn't dare allow anyone with an automatic weapons to guard their borders. So much for your plan assmuncher. |
Judged: 1 1 1 Go take your meds, you frigging left loon nutbag. |
United States |
Judged: 1 1 1 |
Judged: 1 1 1 What do you think of amnesty for all the undocumented workers? Maybe you should organize your brothers and sisters to raise some hell about that? Please come to Bonham and do something with the buster black folks in Fannin, set them straight on this along with the crackers. |
Judged: 1 1 The Governor of Florida is Rick Scott, a Caucasian. Marco Rubio is a US Senator. Both are Republican. |
Judged: 1 1 http://www.nikejordanshoessales.net/ |
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Kiki Morris Raymond
36,763pages on
this wiki
this wiki
Kiki Morris Raymond was a Human. She was born in 2045 in Chicago, Illinois. She married Chris Tezber Raymond, and they had two sons named Jon Grinsberg and Joe Espina Raymond. Kiki died in 2114. (TNG: "The Neutral Zone")
The name of this character was never mentioned in dialog; the name came from the remastered Clare Raymond family tree.


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As the World Wide…
A look at some areas hit hard by the heavy rains.
The views expressed are not those of this company or its affiliated companies. Please note by clicking on "Post" you acknowledge that you have read the Terms of Service and the comment you are posting is in compliance with such terms.


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Meet and Communicate with Fun Singles Near You on BlackSingles.com
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Damn there goes your baby momma
Looking like she wanna Problem
from Wiz Khalifa (Ft. Problem) – Smokin Drink Lyrics on Rap Genius
Your girl seems to be a trouble-maker, always looking for a problem. He'll be happy to provide her with one, if you get my drift.
To help improve the meaning of these lyrics, visit "Smokin Drink" by Wiz Khalifa (Ft. Problem) Lyrics and leave a comment on the lyrics box


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white energy co ltd
(WEC:Australian Stock Exchange Ltd)
Executive Profile*
Neil D. WhittakerChief Executive Officer of River Energy JV Limited, White Energy Company Limited |
Age | Total Calculated Compensation | This person is connected to 0 board members in 1 different organizations across 1 different industries. |
-- | A$569,125 As of Fiscal Year 2012 |
Mr. Neil D. Whittaker serves as the Chief Executive Officer of River Energy JV Limited. Mr. Whittaker serves as a Principal at Hunter Bay Resources. He is a Director of Special Projects at White Energy Company Limited and combines a corporate strategy perspective with management consulting techniques. Mr. Whittaker's experience in leadership and change management provides a solid advisory background. He has had a highly successful career in the business arena and in ...
Corporate Headquarters*Maritime Trade Towers Phone: 61 2 9959 0000Sydney, New South Wales 2000 Australia Fax: 61 2 9959 0099 Board Members Memberships*Director
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text:
US 7076308 B1 Abstract A bionic ear cochlear stimulation system has the capability to stimulate fast enough to induce stochastic neural firing, thereby acting to restore �spontaneous� neural activity. Such neurostimulation involves the use of a high rate pulsitile stimulation signal that is amplitude modulated with sound information. Advantageously, by using such neurostimulation, a fitting system may be utilized that does not normally require T-level threshold measurements. T-level threshold measurements are not required in most instances because the high-rate pulsitile stimulation, even though at levels that would normally be a sub-threshold electrical stimulus, is able to modulate neural firing patterns in a perceptible way.
Claims(4) 1. A method of fitting a bionic ear implant having an electrode array with multiple electrode contacts through which electrical stimulation may be applied to the patient's cochlea; the method comprising:
measuring M-levels associated with a stimulus applied to selected ones of the multiple electrode contacts, where an M-level comprises a comfortable stimulation intensity;
globally determining a T-level for all of the electrode contacts, where a T-level comprises a minimally perceptable stimulation intensity; and
using the M- and T-levels to help set the intensity of stimuli applied to the electrode contacts after fitting, wherein globally determining a T-level comprises setting the T-level to zero as the determined T-level.
2. A method of fitting a bionic ear implant having an electrode array with multiple electrode contacts through which electrical stimulation may be applied to the patient's cochlea; the method comprising:
measuring M-levels associated with a stimulus applied to selected ones of the multiple electrode contacts, where an M-level comprises a comfortable stimulation intensity;
globally determining a T-level for all of the electrode contacts, where a T-level comprises a minimally perceptable stimulation intensity; and
using the M- and T-levels to help set the intensity of stimuli applied to the electrode contacts after fitting, wherein globally determining a T-level comprises setting the T-level to a value that is a prescribed fraction of the measured M-level.
3. A method of fitting a bionic ear implant having an electrode array with multiple electrode contacts through which electrical stimulation may be applied to the patient's cochlea; the method comprising:
measuring M-levels associated with a stimulus applied to selected ones of the multiple electrode contacts, where an M-level comprises a comfortable stimulation intensity;
globally determining a T-level for all of the electrode contacts, where a T-level comprises a minimally perceptable stimulation intensity; and
using the M- and T-levels to help set the intensity of stimuli applied to the electrode contacts after fitting,
wherein globally determining a T-level comprises setting the T-level to a value that is a prescribed fraction of the measured M-level; and
wherein setting the T-level to a prescribed fraction of the measured M-level comprises setting the T-level to be 1/10 of the measured M-level.
4. A method of fitting a multichannel bionic ear implant, the bionic ear implant having multiple channels coupled to multiple electrode contacts of an electrode array, the bionic ear implant having means for applying electrical stimulation to the patient's cochlea through the electrode contacts of the electrode array; the method comprising:
measuring M-levels associated with a stimulus applied to selected ones of the multiple electrode contacts, where an M-level comprises a comfortable stimulation intensity;
determining a T-level for each channel using live speech, where a T-level comprises a minimally perceptable stimulation intensity; and
using the M- and T-levels of each channel to help set the intensity of stimuli applied to the electrode contacts after fitting,
wherein determining a T-level using live speech comprises:
sweeping tones at a center frequency of each channel while the bionic ear implant is set to a comfortable volume in live speech mode; and
assessing whether the tones are detected in each channel without a T setting, and if so not using a T-setting on that channel, and if not increasing a T-setting on that channel during live speech to give desired detectability.
Description The present application claims the benefit of U.S. Provisional Patent Application Ser. No. 60/313,226, filed Aug. 17, 2001, which application is incorporated herein by reference. The present invention relates to cochlear implants, and more particularly to a cochlear implant and a simplified method for fitting such cochlear implant that, in many instances, does not require the use of measured minimum-perception thresholds. Electrical stimulation of predetermined locations within the cochlea of the human ear through an intra-cochlear electrode array is described, e.g., in U.S. Pat. No. 4,400,590. The electrode array shown in the '590 patent comprises a plurality of exposed electrode pairs spaced along and imbedded in a resilient curved base for implantation in accordance with a method of surgical implantation, e.g., as described in U.S. Pat. No. 3,751,615. The system described in the '590 patent receives audio signals, i.e., sound waves, at a signal processor (or speech processor) located outside the body of a hearing impaired patient. The speech processor converts the received audio signals into modulated RF data signals that are transmitted by a cable connection through the patient's skin to an implanted multi-channel intracochlear electrode array. The modulated RF signals are demodulated into analog signals and are applied to selected ones of the plurality of exposed electrode pairs in the intra-cochlear electrode so as to electrically stimulate predetermined locations of the auditory nerve within the cochlea. U.S. Pat. No. 5,938,691, incorporated herein by reference, shows an improved multi-channel cochlear stimulation system employing an implanted cochlear stimulator (ICS) and an externally wearable speech processor (SP). The speech processor employs a headpiece that is placed adjacent to the ear of the patient, which receives audio signals and transmits the audio signals back to the speech processor. The speech processor receives and processes the audio signals and generates data indicative of the audio signals for transcutaneous transmission to the implantable cochlear stimulator. The implantable cochlear stimulator receives the transmission from the speech processor and applies stimulation signals to a plurality of cochlea stimulating channels, each having a pair of electrodes in an electrode array associated therewith. Each of the cochlea stimulating channels uses a capacitor to couple the electrodes of the electrode array. Other improved features of a cochlear implant system are taught, e.g., in U.S. Pat. Nos. 5,626,629; 6,067,474; 6,157,861; 6,219,580; 6,249,704; and 6,289,247, each of which patents is incorporated herein by reference. The implantable cochlear stimulators described in the '629, '474, '861 and '580 patents are also able to selectively control the pulse width of stimulating pulses that are applied through the electrode array to the cochlea, and the frequency at which the stimulating pulses are applied. The new generation of cochlear implants that have the enhanced processing power, and which can provide multiple platforms for delivering electrical stimuli to the auditory nerve, including high frequency pulsitile stimulation having current pulses of controlled amplitude, width and frequency, are frequently referred to as a �bionic ear� implant. As the art of cochlear stimulation has advanced to produce bionic ear implants, the implanted portion of the cochlear stimulation system, and the externally wearable processor (or speech processor) have become increasingly complicated and sophisticated. It is also noted that much of the circuitry previously employed in the externally wearable processor has been moved to the implanted portion, thereby reducing the amount of information that must be transmitted from the external wearable processor to the implanted portion. The amount of control and discretion exercisable by an audiologist in selecting the modes and methods of operation of the cochlear stimulation system have increased dramatically and it is no longer possible to fully control and customize the operation of the cochlear stimulation system through the use of, for example, switches located on the speech processor. As a result, it has become necessary to utilize an implantable cochlear stimulator fitting system to establish the operating modes and methods of the cochlear stimulation system and then to download such programming into the speech processor. One such fitting system is described in the '629 patent. An improved fitting system is described in the '247 patent. The '247 patent further highlights representative stimulation strategies that may be employed by a multichannel stimulation system. Such strategies represent the manner or technique in which the stimulation current is applied to the electrodes of an electrode array used with the stimulation system. Such stimulation strategies, all of which apply current pulses to selected electrodes, may be broadly classified as: (1) sequential or non-simultaneous (where only one electrode receives a current pulse at the same time); (2) simultaneous (where substantially all of the electrodes receive current stimuli at the same time, thereby approximating an analog signal); or (3) partially simultaneous pulsitile stimulation (where only a select grouping of the electrodes receive stimuli at the same time in accordance with a predefined pattern). Typically, when the fitting systems described in the '629 or '247 patents are employed for multichannel stimulation systems, or when equivalent or similar fitting systems are employed, it is necessary to use directly measured threshold values and/or thresholds derived from the measurement of psycophysically-determined pseudo-comfort levels. That is, for each channel of the multichannel system, a minimum threshold level is measured, typically referred to as a �T� level, which represents the minium stimulation current which when applied to a given electrode associated with the channel produces a sensed perception of sound at least 50% of the time. In a similar manner, an �M� level is determined for each channel, which represents a stimulation current which when applied to the given electrode produces a sensed perception of sound that is moderately loud, or comfortably loud, but not so loud that the perceived sound is uncomfortable. These �T� and �M� levels are then used by the fitting software in order to properly map sensed sound to stimulation current levels that can be perceived by the patient as sound. Disadvantageously, determining the �T� and/or �M� levels (or other required thresholds) associated with each channel of a multichannel stimulation system is an extremely painstaking and time-intensive task. Such determinations require significant time commitments on the part of the clinician, as well as the patient. Moreover, once determined one channel at a time, such levels may not be representative of actual threshold levels that are present during real speech. That is, preliminary data indicate that thresholds set in single channel psychophysics overestimate the actual threshold required when all channels are running during live speech. Such an overestimation appears to penalize patient performance, particularly performance in noise. Hence, neural stimulation parameters which render threshold measurement unnecessary would dramatically reduce the time requirements for programming sequential and/or partially simultaneous pulsitile stimulation, as well as facilitate a higher probability of optimized programming for pediatric as well as adult populations where obtaining such measures are difficult. As the ages of patients into which implantable cochlear stimulators are implanted decreases, it becomes increasingly more important to improve the fitting process and to minimize, or eliminate, the need to make threshold measurements. This is because very young patients, for example, two year olds, are unable to provide adequate subjective feedback to the audiologist for the audiologist to accurately �fit� the cochlear stimulation system optimally for the patient. Thus, what is needed is an improved apparatus and simplified method for fitting a speech processor where many of the threshold measurements previously required are no longer needed. The present invention addresses the above and other needs by providing a cochlear implant that provides auto-conditioning with high resolution neurostimulation. Such neurostimulation involves the use of high rate pulsitile stimulation that is amplitude modulated with sound information. Advantageously, as a result of using such auto-conditioning with high resolution neurostimulation, a fitting system may be utilized that does not normally require T-level threshold measurements. T-level threshold measurements are not required in many instances because the high-rate pulsitile stimulation, even though at levels that would normally be a sub-threshold electrical stimulus during a single channel measurement, is able to modulate neural firing patterns in a perceptible way. For purposes of the present application, �high rate� is defined as a stimulation rate greater than about 2 KHz (i.e., stimulation where the period, T, between successive biphasic stimulation pulses is less than about 500 microseconds (μS)). �Pulsitile� stimulation is considered to be any pulsed stimulation, e.g., biphasic stimulation, where the pulse width, PW, of each phase of the biphasic pulse is less than about 21 μS, e.g., 11 μS, although in some instances it may be longer, e.g., 32 μS or as long as 75 μS. These definitional terms are illustrated in the waveform diagram presented in Advantageously, when such high rate pulsitile stimulation parameters are used, the cochlea implant user, in most instances, tends to have better speech performance without mapping low-level signals to single channel pyscophysical threshold levels. Thus, not only is it not necessary to measure, on a channel-by-channel basis, the pyscophysical threshold levels (T-levels) that have heretofore been required for purposes of cochlear implant fitting, thereby greatly reducing the time demands associated with the cochlear fitting process, but by avoiding such measurements, the user's performance improves. In accordance with one aspect of the invention, there is provided a cochlear stimulation system that applies high rate pulsitile stimulation to the users's cochlea, thereby inducing perceptible low-level neuronal firing patterns. In accordance with another aspect of the invention, a cochlear implant fitting process is provided wherein, in most instances, the channel-by-channel T-levels need not be measured. In accordance with still another aspect of the invention, stochastic firing is restored to the auditory nerve, thereby enhancing thresholds, dynamic range and pyscophysical performance. In accordance with yet a further aspect of the invention, individual neurons are stimulated by a cochlear implant at a rate faster than they are able to follow, thereby resulting in a randomization of interspike intervals. Advantageously, when the neuron is no longer phase-locked to the carrier pulse, the firing probability becomes a function of stimulus energy, and becomes much more like �natural� hearing. The above and other aspects, features and advantages of the present invention will be more apparent from the following more particular description thereof, presented in conjunction with the following drawings wherein: Appendix A provides an overview of one embodiment of a Fitting System that may be used with the invention. Corresponding reference characters indicate corresponding components throughout the several views of the drawings. The following description is of the best mode presently contemplated for carrying out the invention. This description is not to be taken in a limiting sense, but is made merely for the purpose of describing the general principles of the invention. The scope of the invention should be determined with reference to the claims. The ICS 21 and the SP 16 are shown in In a typical bionic ear implant system, as shown more particularly below in Typically, where a transcutaneous data link must be established between the external portion and implantable portions of the system, such link is realized by an internal antenna coil within the implantable portion, and an external antenna coil within the external portion. In use, the external antenna coil is positioned so as to be aligned over the location where the internal antenna coil is implanted, allowing such coils to be inductively coupled to each other, thereby allowing data (e.g., the magnitude and polarity of a sensed acoustic signals) and power to be transmitted from the external portion to the implantable portion. Note, in other embodiments of the invention, both the SP 16 and the ICS 21 may be implanted within the patient, either in the same housing or in separate housings. If in the same housing, the link 14 may be realized with a direct wire connection within such housing. If in separate housings, as taught, e.g., in U.S. Pat. No. 6,067,474, incorporated herein by reference, the link 14 may be an inductive link using a coil or a wire loop coupled to the respective parts: The microphone 18 senses acoustic signals and converts such sensed signals to corresponding electrical signals, and may thus be considered as an acoustic transducer. The electrical signals are sent to the SP 16 over a suitable electrical or other link 24. The SP 16 processes these converted acoustic signals in accordance with a selected speech processing strategy in order to generate appropriate control signals for controlling the ICS 21. Such control signals specify or define the polarity, magnitude, location (which electrode pair receives the stimulation current), and timing (when the stimulation current is applied to the electrode pair) of the stimulation current that is generated by the ICS. Such control signals thus combine to produce a desired spatiotemporal pattern of electrical stimuli in accordance with the desired speech processing strategy. Unlike early cochlear implant systems, the bionic ear implant system advantageously confines such control signals to circuitry within the implantable portion of the system, thereby avoiding the need to continually send or transmit such control signals across a transcutaneous link. The speech processing strategy is used, inter alia, to condition the magnitude and polarity of the stimulation current applied to the implanted electrodes of the electrode array 48. Such speech processing strategy involves defining a pattern of stimulation waveforms that are to be applied to the electrodes as controlled electrical currents. In accordance with the present invention, an auto-conditioning with high resolution (ACHR) strategy is used which stimulates the implanted electrodes with a high rate pulsitile pattern that is amplitude modulated by the sound information. If multiple electrode pairs exist, as is the case with a multichannel cochlear implant system, then the types of stimulation patterns applied to the multiple channels may be conveniently categorized as: (1) simultaneous stimulation patterns, or (2) non-simultaneous stimulation patterns. Simultaneous stimulation patterns may be �fully� simultaneous or partially simultaneous. A fully simultaneous stimulation pattern is one wherein stimulation currents, either analog or pulsitile, are applied to the electrodes of all of the available channels at the same time. A partially simultaneous stimulation pattern is one wherein stimulation currents, either analog or pulsitile, are applied to the electrodes of two or more channels, but not necessarily all of the channels, at the same time. Examples of each type are given in U.S. Pat. No. 6,289,247, previously incorporated herein by reference. Analog waveforms used in analog stimulation patterns are typically reconstructed by the generation of continuous short monophasic pulses (samples). The sampling rate is selected to be fast enough to allow for proper reconstruction of the temporal details of the signal. An example of such a sampled analog stimulation pattern is a simultaneous analog sampler (SAS) strategy. Current pulses applied in pulsitile stimulation patterns are generally biphasic pulses, as shown in Turning next to A complete description of the functional block diagram of the bionic ear implant shown in In the manner described in the U.S. Pat. No. 6,219,580, the bionic ear implant functionally shown in Thus it is seen that the system of While it is common to use a one-to-one mapping scheme between the analysis channels and the stimulus channels, wherein n=m, and the signal analyzed in the first analysis channel is mapped to produce a stimulation current at the first stimulation channel, and so on, it is not necessary to do so. Rather, in some instances, a different mapping scheme may prove beneficial to the patient. For example, assume that n is not equal to m (n, for example, could be at least 20 or as high as 32, while m may be no greater than sixteen, e.g., 8 to 16). The signal resulting from analysis in the first analysis channel may be mapped, using appropriate mapping circuitry 41 or equivalent, to the first stimulation channel via a first map link, resulting in a first stimulation site (or first area of neural excitation). Similarly, the signal resulting from analysis in the second analysis channel of the SP may be mapped to the second stimulation channel via a second map link, resulting in a second stimulation site. Also, the signal resulting from analysis in the second analysis channel may be jointly mapped to the first and second stimulation channels via a joint map link. This joint link results in a stimulation site that is somewhere in between the first and second stimulation sites. The �in between site� is sometimes referred to as a virtual stimulation site. Advantageously, this possibility of using different mapping schemes between n SP analysis channels and m ICS stimulation channels to thereby produce a large number of virtual and other stimulation sites provides a great deal of flexibility with respect to positioning the neural excitation areas in a location that proves most beneficial to the patient. Still with reference to In contrast to prior art systems, a modern bionic ear implant, such as the CII Bionic Ear system manufactured by Advanced Bionics Corporation of Sylmar, Calif., advantageously puts at least a portion of the speech processor 16 within the implanted portion of the system. For example, a bionic ear implant may place the Pulse Table 42 and arithmetic logic unit (ALU) 43 inside of the implanted portion, as indicated by the bracket labeled �Imp2� in Future generations of bionic ear implant systems will incorporate more and more of the speech processor 16 within the implanted portion of the system. For example, a fully implanted speech processor 16 would incorporate all of the SP in the implanted portion, as indicated by the bracket labeled Imp3 in The stimulation provided by a bionic ear implant in accordance with the present invention is premised on a stimulus pattern that induces stochastic, i.e., random, neural firing, which stochastic neural firing acts to restore �spontaneous� neural activity. Such stochastic neural firing results through application of a rapid stimulation pulse train applied by the speech processor circuits of the bionic ear cochlear implant. In operation, the high rate stimulus pattern provided by the invention stimulates individual neurons at a rate faster than the individual neurons can follow. This results in a randomization of inter-spike intervals, where the inter-spike interval is the time between successive neural firings for a given auditory neuron; or stated differently, inter-spike intervals represent the �firing patterns� of individual auditory nerve fibers. The inter-spike intervals, or firing patterns, of all auditory nerve fibers, even during the absence of sound, tend to be stochastic (random). Furthermore, these firing patterns are stochastic across the neural population. Advantageously, when the neuron is no longer phase-locked to the carrier pulse, its firing probability becomes a function of stimulus energy, and thus becomes more like �natural� hearing. Such randomization in a neural population better enables the population of neuron fibers to encode the fine details of sound. That is, the population of neuron fibers is able to encode what a single neuron fiber is not able to encode. It should be noted that it is technically not correct to refer to the IHC 101 as �firing� because, as indicated above, �firing� occurs at the nerve fiber synapse 104, not in the transductory IHC 101. The auditory nerve fiber 108 is the �spiking cell�, where the effect of a nerve impulse passing through a synapse 104 is first manifest. As a short-hand way of referencing the chain of events that causes an auditory nerve fiber to become spiked, it is common to refer to the �firing of the auditory nerve�, or the �firing of a ganglion cell� associated with the inner hair cell nerve fiber complex 100, or simply �neural firing�. When the stereocilia 102 are displaced, e.g., bent one way or the other as illustrated in At any instant of time, in response to sensed sound that causes the stereocilia 102 to be displaced, or in response to silence, where the stereocilia 102 remain substantially at rest, the nerve fiber synapses 104 fire in a stochastic (random) manner, causing nerve impulses to be sent along the respective auditory nerve fibers. As the stimulus energy increases, the probability that more nerve fibers will fire increases, but the firing remains stochastic, or random. Next, with reference to When the inner hair cells 101 are damaged or missing, as is usually the case for a patient experiencing sensorial hearing loss, then the auditory nerve fibers are silenced, and the patient has no hearing function. In order to awaken such auditory nerve fibers, and provide the patient with the sensation of hearing, it is known in the art to electrically stimulate the auditory nerve fibers using a cochlear implant. Disadvantageously, the stimulation patterns employed by most cochlear implants result in a very narrow system dynamic range for the patient. This is because, as seen in One approach known in the art for expanding the dynamic range achieved with a cochlear implant is to apply a high rate conditioning signal, e.g., a 5000 Hz pulse train, in combination with analog stimulation to the electrode contacts. The 5000 Hz pulse train functions as a conditioner. See, Rubinstein et al., Second Quarterly Progress Report NO1-DC-6-2111. See also U.S. Pat. No. 6,078,838, incorporated herein by reference. This approach, and the results achieved thereby, are illustrated in In contrast to the analog approach proposed by Rubinstein et al. (see One of the key advantages made possible through use of the present invention is that T levels need not be measured for many, if not most, patients. Rather, the T level may be assumed to be 0 or some other value, e.g., 1/10 of the M value for a given channel. To further explain why T levels may not be needed in all patients, consider that a threshold represents the intensity of signal X that meets an arbitrary detection criteria by system Y. The threshold (T) of X by Y is only valid for signal X. The system Y has inherent noise. If the system noise is altered, the T for X is likewise altered. In other words, a given T is specific for both the signal X as well as for the system noise of Y. In traditional cochlear implant fittings, the value of T is determined by the activation of a single channel using a simple signal. The intensity of this signal that meets the detection criterion is called T. This T is then assumed to be appropriate for the system during �live speech� mode in which all channels are activated. However, the system noise of the neural detector Y is substantially different from single channel to live speech mode, resulting in a T mismatch. This means that all the effort undertaken to determine the T level may, in fact, be wasted effort for many patients because the T level thus determined doesn't fit the live speech mode. Patent data indicate that thresholds set in single channel psychophysics, painstakingly measured, overestimate the actual threshold required when all channels are running during live speech. Such an overestimation appears to penalize patient performance, particularly performance in noise. It has been empirically determined that for many patients, because the T level is apparently very close to the noise level, the T level can simply be set to 0, or some other arbitrary level, thereby eliminating the need to go through the painstaking process of measuring T. Patient data further indicate that when using high stimulation rates, the detectability of low level signals is enhanced such that certain patients actually gain performance benefits without the use of T levels during live speech mode. A similar effect appears to be present for simultaneous analog stimulation (SAS) in which T values are not used. Since single channel measurements at high rates and at narrow pulse widths are made difficult by possible adaptation and shallow loudness growth, the relative shape of the upper level contour (most comfortable level M) may be determined at a wider pulse width. This upper level contour may then be linearly scaled to give the patient the appropriate volume. In addition to setting T levels at zero or some other relatively low level, e.g., 1/10 of the measured M-levels, thereby significantly saving fitting time and complexity, the present invention further recognizes that T levels may be set using live speech. One method of setting T levels using live speech is as follows: Detectability is assessed by sweeping tones at the center frequencies (CF) of each channel's filter band while the patient is at a comfortable volume in live speech mode. If detectability meets the audiogram criteria without a T setting, then no T setting is used; otherwise, T is increased during live speech to give desired detectability on a channel-by-channel basis. Another manner in which T levels may be set using live speech is as follows: The relative T and M contours are determined at a wider pulse width. M is globally adjusted in live speech to patient comfort. M is then set to be equal to T by setting the volume to zero on the sound processor. Live-speech is then pulsed on and off and the T contour is then globally shifted until a threshold criteria of detecting the global T pulse is determined. Using the techniques of the present invention, significant enhancements in patient performance have been observed. For example, Appendix A provides an overview of one type of fitting software that may be used with the invention, e.g., to set the T levels to some low value. By way of example, the T-levels may be set to a small fraction of the measured M-levels, e.g., 1/10 of the measured M levels. By not having to measure individual T levels on each channel, significant time is saved during the fitting process. As described above, it is seen that a bionic ear implant allows populations of neuron fibers to be stimulated at a rate that is faster than an individual neuron fiber is able to follow. Advantageously, such fast stimulation results in a randomization of interspike intervals, or a randomization of when the individual neuron fibers fire. When the neuron is no longer phase-locked to the carrier pulse, its firing probability becomes a function of stimulus energy, and thus becomes more like �natural� hearing. Such randomization in a neural population better enables the population of neuron fibers to encode the fine details of sound. That is, the population of neuron fibers is able to encode what a single neuron fiber is not able to encode. Further, as described above, it is seen that by restoring stochastic firing to the auditory nerve, thresholds, dynamic range and pyscophysical performance may be significantly enhanced. While the invention herein disclosed has been described by means of specific embodiments and applications thereof, numerous modifications and variations could be made thereto by those skilled in the art without departing from the scope of the invention set forth in the claims. Patent Citations
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Recipe of the Day
Fish Fillets Italiano
Cod and haddock fillets work well for this braised dish. An extra easy and quick way to fix a superb tasting fish dish! Serve over rice. — MyPlate
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Wednesday, November 17, 1999
Cultural and civilizational differences will replace political and ideological ones as a source of conflict in the coming century, said Stephanie Lawson, a professor of international relations at the University of East Anglia.
Lawson discussed the consequences of local culture compared to a more global culture Tuesday in her lecture "A New Age for World Politics?"
"[In the future, the] principle source of conflict will be cultural," Lawson said. She particularly emphasized the culture of the many Asian-Pacific countries under the influence of Western, democratic culture.
"Due to the fact that democracy is generally based on ideas of the west, they do not translate well into Asian countries," Lawson said. She offered Taiwan, South Korea, the Philippines and Japan as countries who have adopted democracy despite their cultural and natural tendencies.
Lawson also discussed the definition of culture and its varying subcategories, such as world culture and state culture.
"Culture is something loose," she said. "It's about change and if it's not moving it's dead."
After defining culture, Lawson challenged the more specific nature of the term.
"Is there one culture in a culture or multiple interpretations?" she asked. "Is the idea of culture used to exploit? To what extent can a culture be claimed?"
She also discussed moments in the course of the century that have shaped world politics, especially the Cold War.
1999 marks the 10 year anniversary of the fall of communism. With this landmark came a certain amount of relieved tension within the West, giving democracy a boost world-wide. Democracy was once again celebrated with the coming of Operation Desert Storm. "Removal of Iraq replaced euphoria from the end of the cold war," Lawson said.
Stephanie Lawson has written on the politics in the South Pacific region. Her research focuses on the politics of culture and democratization in Southeast Asia.


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Results 1 to 10 of 12
Dupli-Color Shadow for Black Chrome Look
I'm trying to black out or greatly reduce the appearance of chrome on my 2011 Silverado and have been toying with many ideas and accessories that would help me accomplish this. A lot of people will paint their various chrome accessories black so I was wondering if anyone has ever used the Dupli-Color Shadow to achieve a black chrome look. I was considering using it for the silverado lettering on the doors and tailgate.
04-01-2012, 05:57 AM #2The thing to remember with the Duplicolor is that the more coats you put on the darker it is going to be. I was debating on using this same product on my rims to get the same look. The first reason I didn't was because I am not sure how the chrome cladding will handle it. I would hate to spray them and have the cladding peel off. I don't know why it would but it is just a fear of mine. The other reason is if I was to do my rims than I would have to do the grill and trim on the front end. If you do it please post some pics and let us know how it went.
04-01-2012, 11:05 PM #3That's an intriguing product. I've not heard of it before you posted about it so I can't be any help there. I'd be curious to try it for some of the same applications you are. I'm trying to, piece by piece, get rid of all my chrome and replace it with black so this might be a good option for some of the emblems.
To Be Completed (Eventually):
6" BDS Suspension lift | KingShocks Front and Rear Coil Overs | 4.56 Gears | 35" A/Ts (TBD) | Cervinis 07-12 Silverado Ram Air Hood | Fab Fours Front Winch Bumper (CS07-H2051-1) | Fab Fours Full Size Rear Bumper (CS07-W1850-1) | RX-2 Series Main Grille (Black) | Line-x Bed and wheel wells | Skid Plates | Rizor Automated Tonneau Cover | Custom Bed Insert | RBP RX-3 Series Nerf Bars | Kenwood Excelon DNX9990HD | JL Audio Stealthbox (2x500RMS) | JL Audio HD 1200/1 Class D Mono Amp | JL Audio C5-653 Components (Front) | JL Audio C5-650 Components (Rear) | JL Audio Slash v2 300/4v2 Class A/B Amp
04-01-2012, 11:13 PM #4well my fellow black chrome lover, I wouldn't dare to it to my rims since they are OEM and in very nice condition (good for resale value if desired). I am however contemplating it for the silverado lettering as soon as I figure out how to make sure they go on in the same position.
04-10-2012, 07:27 PM #5well I found a video sponsored by dupli-color. In the example they made the wheel really dark. I'm still indecisive about my bumpers and don't want to pay the money if I can do something like this. My dad used to do body work so I'll talk to him to see what he thinks.
04-10-2012, 08:43 PM #6I tried it out on a chrome hitch cover as a test. I may not have prepped it right and it took a few coats to cover and subsequently is darker than I had hoped. If you are good with paint, I think it will be a good way to give you the effect you want. Me on the other hand will need to get better at it before I try any major parts.
2009 GMC Sierra 5.3L running on E85
Black and chrome
Volant CAI
Borla dual exhaust
Chrome Billet grill
Tonneau cover
4300k High/Low Beam HIDs
Westin Ultimate Bull Bar with PIAA 540s
Blacked-out tail and third brake light
04-10-2012, 08:53 PM #7I've been experimenting with small pieces before I do my bumpers and grill. I've gotten better each time I use it and I think I have the process down. Follow the Duplicolor instructions exactly. I also had really good luck clearing it afterwards for longer durability. I'm doing my bumpers, grill, and I'm buying the chrome handle covers etc and doing them.
04-11-2012, 12:56 AM #9Dupli-Color Chrome comes out looking like silver paint even if you do a gloss black primer, first. This is very obvious if you put Dupli-Color Chrome next to actual chrome.
I half expect Dupli-Color Shadow to come out looking like dull/dark silver instead of black chrome. Do you have pics of something done in Dupli-Color Shadow? Have you put it next to real black chrome to compare?
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Stuart Edmonds - Super Slow-mo 3
This video was shot to highlight the super slow mo capabilities of my FS 700. Not many sports suit super slow motion like motocross and Professional rider Stuart Edmonds put on a great show in front of camera. The majority of shots were filmed at 240 frames per second and the piece was graded in magic bullet looks.
Credit: Mike Neale
ocscottie's videos 743 - 749 of 2122 « Previous Next »
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Mikey Neale made an amazing video out of a guy riding his new bike.. what a shock! that dirt looked mint


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Capital flows and domestic market integration in China
This paper examines whether reforms have created a substantial commercial sector with market characteristics in China's financial system. Many studies conclude that the operation of China's capital markets lacks market features, resulting in fragmentation. Yet, this lack of integration coincides with rapid and continuous economic growth. We study the dynamic pattern of domestic capital mobility with a unique data set on provincial savings and investment, which allows us to examine components as well as aggregates. After stripping out foreign and officially influenced funds, we find that the behavior of capital flows in what we define as China's commercial sector looks increasingly like that of interstate flows in the US and other advanced nations. Tracking the direction and size of capital flows also highlights the different behavior of commercial and non-commercial funds transfers in China. This result undercuts the widespread view of China's economy as lacking domestic integration.Download Info
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Article provided by Taylor & Francis Journals in its journal Journal of Chinese Economic and Business Studies.
Volume (Year): 8 (2010)
Issue (Month): 1 ()
Pages: 67-94
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Keywords: China's economy; capital market; financial integration;References
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Made in Britain
can be personalised
Welcome to 'Olivia sticks with Layla' collection- illustrations of childhood that can be personalised to make that extra special gift.
These charming, timeless illustrations capture the innocence and freedom of a nostalgic childhood.
The perfect art for children whether it be a little girl or new born baby. This print makes a unique gift for for christenings, new baby gifts, birthday gifts or for parents who love the traditional look for their little one's nurseries, bedrooms and playrooms.
Little one's are sure to treasure their piece of art as it becomes part of the fabric of their childhood.
You can personalise this print by adding baby's name to the print!
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Only the highest quality materials are used for this Giclee fine art. It is printed onto Hahnemühle 410 gsm woven canvas cloth with archival inks, designed to last.
Comes with premium quality ph neutral Fine Art Guilds approved white mount & cream backing board. Your print is then protected in a cellopane presentation bag.
- The dimensions of the print & mount are 20 x 30cm (A4 size)
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Publications (6)14.94 Total impact
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ABSTRACT: Cardiac hypertrophy is the first visible sign of cardiac remodeling. Oxidative stress is implicated in the etiopathogenesis of cardiac hypertrophy. In vitro studies have shown that exposure of cardiomyocytes to free radical generators induce cell hypertrophy. However, there are no studies to show that in vivo redox status can influence cardiomyocyte growth. Blood samples were collected from healthy volunteers and serum lipid peroxidation was determined as a measure of oxidative stress. Cardiac myocytes cultured from newborn rat were exposed to serum samples. A significant correlation was observed between serum lipid peroxidation and cardiomyocyte volume, indicating that in vivo oxidative stress can act as an important co-factor in mediating the hypertrophic response. This experimental system also envisages a novel approach to identify patients prone to left ventricular remodeling and identification of humoral factors mediating the changes.International journal of cardiology 03/2007; 115(2):246-8. · 7.08 Impact Factor - [show abstract] [hide abstract]
ABSTRACT: Magnesium (Mg) deficiency and oxidative stress are independently implicated in the etiopathogenesis of various cardiovascular disorders. This study was undertaken to examine the hypothesis that Mg deficiency augments the myocardial response to oxidative stress. Electrically stimulated rat papillary muscle was used for recording the contractile variation. Biochemical variables of energy metabolism (adenosine triphosphate (ATP) and creatine phosphate) and markers of tissue injury (lactate dehydrogenase (LDH) release and lipidperoxidation), which can affect myocardial contractility, were assayed in Langendorff-perfused rat hearts. Hydrogen peroxide (100 micromol/L) was used as the source of reactive oxygen species. The negative inotropic response to H2O2 was significantly higher in Mg deficiency (0.48 mmol Mg/L) than in Mg sufficiency (1.2 mmol Mg/L). Low Mg levels did not affect ATP levels or tissue lipid peroxidation. However, H2O2 induced a decrease in ATP; enhanced lipid peroxidation and the release of LDH were augmented by Mg deficiency. Increased lipid peroxidation associated with a decrease in available energy might be responsible for the augmentation of the negative inotropic response to H2O2 in Mg deficiency. The observations from this study validate the hypothesis that myocardial response to oxidative stress is augmented by Mg deficiency. This observation has significance in ischemia-reperfusion injury, where Mg deficiency can have an additive effect on the debilitating consequences.Canadian Journal of Physiology and Pharmacology 07/2006; 84(6):617-24. · 1.56 Impact Factor - [show abstract] [hide abstract]
ABSTRACT: Magnesium has a significant role in the regulation of ion transport. Marginal deficiency of Mg can therefore affect myocardial excitability and contractility. This study was taken up with the objective of examining the inotropic response of the myocardium to variation in extracellular [Mg]o and identifying the ion channels and pumps mediating the inotropic changes. Electrically stimulated rat papillary muscle was used as the experimental model and mechanical changes were recorded using a physiograph. Channel specific antagonists were used to identify the channels mediating the functional changes. Diastolic Ca2+ levels were determined in isolated myocytes by the ratiometric method using the fluorescent indicator Fura2-AM. A negative association was observed between the level of [Mg]o and force of contraction, with a peak at 0.48 mM Mg. The force of contraction in Mg deficient medium (0.48 mM) was 158% of control (1.2 mM Mg) (p < 0.001). Inotropic response to the L-type channel antagonist (verapamil-1 microm) and NaK ATPase inhibitor (Ouabain-0.3 mM) was augmented in Mg deficiency (p < 0.005), indicating activation of the channel and the pump. The response to T-type channel inhibitor (NiCl2-40 microM) was attenuated in Mg deficiency (p < 0.05). The response to the sarcoplasmic reticular Ca pump inhibitor (caffeine-10 mM) and the SR Ca2+ release channel inhibitor (ryanodine-1 microM) were not significantly affected by Mg deficiency. Diastolic level of Ca2+ increased with a decrease in Mg (p < 0.05). The observations of the study lead to the conclusion that the positive inotropic response in Mg deficiency is mediated by an increase in basal Ca2+ combined with Ca-induced-Ca release consequent to Ca2+ influx through L-type Ca channel. Variation in sensitivity to Ca channel blockers and NaK ATPase inhibitor in Mg deficiency can have pharmacological implications.Magnesium research: official organ of the International Society for the Development of Research on Magnesium 10/2005; 18(3):163-9. · 1.38 Impact Factor - [show abstract] [hide abstract]
ABSTRACT: Familial clustering is common in epilepsies, but pedigree patterns suggest a multi-factorial inheritance. Genetic liability for multi-factorial inheritance is population specific and such data are not available for the population of Kerala or other states in south India. In this study, we have attempted to determine the genetic liability to epilepsy based on an adult population of this state. Pedigrees were recorded for probands who reported to the Kerala Registry of Epilepsy and Pregnancy. In order to obtain a genetically matched sample for comparison and estimation of empiric risks, we have used the family history of the spouse except when the spouse was proband's relative. The ILAE criteria were followed for diagnosis and classification of epilepsy. Data were collected on 18,419 family members of 505 probands with epilepsy (82 men and 423 women) and 10,231 family members of spouses (control). The frequency of epilepsy in first and second-degree relatives of the spouses was comparable to the population frequency (0.5%), justifying the use of this sample as control. Positive family history was observed in 22.2% of probands and 8.24% of controls (Odd's Ratio 3.2, 95% Confidence Interval 2.12-4.73). An affected first-degree relative was observed in 7.5% of probands. The corresponding figure for GE, LRE and other epileptic syndromes were 10.2%, 5.8% and 5.12%, respectively. The segregation ratio for Juvenile Myoclonic Epilepsy (JME) (1:19) was higher than that for other types of Generalized Epilepsy (GE) (1:24) and Localization Related Epilepsy (LRE) (1:52). Prevalence of epilepsy among the first-degree relatives (1.96%) was greater than the square root of the population frequency (0.51%) and was higher than that for second-degree (1.24%) and third-degree (0.64%) relatives for the probands. Probands had higher parental consanguinity (13.07%) compared to controls (6.64%). The above factors support a complex inheritance. Genetic liability to epilepsy (heritability) is greater for GE (0.6) and significantly higher for JME (0.7) compared to LRE (0.4). A limitation of this study is that the inferences are based on a predominantly adult female proband sample but no gender specific differences were identified. The observations of this study indicate complex inheritance and the liability values are useful for genetic counseling in the local population. Further studies involving more individuals from younger age group and male gender are envisaged.Epilepsy Research 01/2005; 62(2-3):163-70. · 2.24 Impact Factor - [show abstract] [hide abstract]
ABSTRACT: This study was performed with the objective of assessing the mechanical response of the myocardium to different levels of cerium and delineation of the mechanism underlying the mediation of the functional changes. Rat ventricular papillary muscle was used as the experimental model. Isolated papillary muscles were exposed to different concentrations of CeCl3 and the force of contraction was measured using a force transducer. Experiments have revealed that the negative inotropic response to CeCl3 was proportional to its concentration. The inotropic changes were found to be completely reversible at concentrations < or =5 microM, and partially reversible at higher concentrations. Neutralization of cerium-induced inotropic changes by the superoxide anion scavenger superoxide dismutase (SOD) at concentrations < or =5 microM indicates that the mechanical changes are mediated by reactive oxygen species. At higher concentrations of Ce3+, SOD partially reversed the contractile changes. The beneficial effect of SOD was seen only if the muscles were pretreated with the scavenger prior to the addition of cerium chloride.Biological Trace Element Research 02/2003; 96(1-3):203-8. · 1.31 Impact Factor - [show abstract] [hide abstract]
ABSTRACT: Magnesium has attracted attention as an essential element with diverse roles in the regulation of cardiac contraction. Chronic suboptimal intake of the element results in hypomagnesaemia. Experimental and clinical studies indicate the possibility of a marginal decrease in myocardial magnesium compared to those with sufficient intake. Reduction in extracellular magnesium affects myocardial excitability and contractility predominantly, by modulation of the levels of other ions that have an influence on cardiac mechanics. Majority of the in vitro experiments in isolated ventricular tissue or myocytes record an inverse relation between Mg concentration and inotropic response, mediated probably by enhanced influx of Ca2+ promoting sarcoplasmic reticular Ca2+ release. Paradoxically myocardial contractility is usually compromised in animals on Mg deficient diet or on perfusion of whole heart with low Mg (< 0.5 mM) buffer. In the whole animal or organ, magnesium deficiency induced coronary vasospasm, defective energy metabolism and excessive free radical generation may be important variables acting in concert or independently to affect myocardial function. Electrical excitability is enhanced in magnesium deficiency, and arrhythmic changes are presumed to be mediated by disturbance in K+ homeostasis. Magnesium deficiency has not received the attention it deserves probably due to absence of clinical symptoms. Magnesium deficiency concomitant with stress may be of clinical significance, leading to arrhythmic, hemodynamic and ischaemic changes in the heart. Chronic magnesium deficiency is accompanied by increased free radical generation. Free radicals are known to influence myocardial excitability and contractility. Physiologic and pathologic stress also promotes free radical generation. The additive action of free radical generation in magnesium deficiency and any form of stress may be one of the reasons for enhanced sensitivity to stress in magnesium deficiency. Clinical and experimental data on the cardiac consequences of marginal magnesium deficiency being limited, a number of factors need experimental validation. For example--the extent of change in total and ionized magnesium in the serum and heart, mechanical response of the myocardium to decrease of total and ionized magnesium in the intra- and extracellular milieu; the extent of free radical generation in magnesium deficiency and the cardiac consequence; and also the additive effect of magnesium deficiency and different forms of stress.Magnesium research: official organ of the International Society for the Development of Research on Magnesium 12/2002; 15(3-4):287-306. · 1.38 Impact Factor
Publication Stats
20 | Citations | |
192 | Views | |
14.94 | Total Impact Points | |
Top Journals
Sree Chitra Tirunal Institute for Medical Sciences and Technology
- Department of Cellular and Molecular Cardiology
Thiruvananthapuram, Kerala, India


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I love the female form and have now found a way to draw with my embellisher machine. This is 1 in a series they are approx 1/2m long in diff poses from my life drawing classes. I have embellished them from cream merino, then detailed in fine wisps of black and red to give the look of a sketched piece each is padded and 3d'd -sadly photos dont show this!!!
Wonderful ! go on !
Fantastic - Keep creating :-))
Thanks guys I am loving it and now incorporate them into my art quilts
Holy cow, this is amazing.
This is absolutely amazing, I wish I could see the real thing to see the 3-D effect. Thank you for posting your "art" for us all to see.
Excellent work. Doing work this fine with an embellisher isn't easy and this looks like a soft charcoal sketch. Beautiful.
thanks for the comments royce creative --Im glad it looks like a sketch that was the aim.
I am new to this today. What is an embellisher machine? And how can I incorporate this into my quilting arts?
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WMNF: The Blog
Full text of Governor Rick Scott's first State of the State address
Sean Kinane about about 3 years ago
Florida Governor Rick Scott's State of the State Address. March 8, 2011. Tell us what you think.
Remarks prepared for delivery.
President Haridopolos, Speaker Cannon, Chief Justice Canady, members of the Supreme Court, members of the Cabine...
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' | Steven G. Waguespack, M.D. |
Present Title & Affiliation
Primary Appointment
Dual/Joint/Adjunct Appointment
Bio Statement
Dr. Steven Waguespack is an Associate Professor of internal medicine and pediatrics at the University of Texas MD Anderson Cancer Center in Houston. A native of Louisiana, Dr. Waguespack graduated summa cum laude from Loyola University in New Orleans, where he also participated in the presidential scholars honor program. He graduated from the University of Texas Medical School at Houston with honors and pursued training in internal medicine/pediatrics at the Indiana University Medical Center in Indianapolis. He completed a combined adult and pediatric endocrinology fellowship at Indiana University and is board certified in both subspecialties. Recognized as one of "America's Top Doctors for Cancer" in endocrinology, his major clinical and research interests are thyroid cancer, pituitary tumors, and genetic syndromes of endocrine neoplasia. At MD Anderson, where he has been on faculty since 2002, Dr. Waguespack enjoys providing endocrine care to patients of all ages.
Research Interests
Thyroid Cancer, Pituitary Tumors, Multiple Endocrine Neoplasia, and Pediatric Endocrine Tumors
Clinical Interests
Thyroid Cancer, Pituitary Tumors, Multiple Endocrine Neoplasia, and Pediatric Endocrine Tumors
For general clinic information and referrals, please visit the Department of Endocrine Neoplasia and Hormonal Disorders' contact information webpage at:
Office Address
1400 Pressler Street
Unit Number: 1461
Houston, TX 77030-3722
Room Number: FCT12.5030
Phone: (713) 792-2841
Fax: (713) 794-4065
Education & Training
Degree-Granting Education | |
1994 | University of Texas Medical School at Houston, Houston, TX, MD, Medicine |
1990 | Loyola University, New Orleans, LA, BS, Summa cum laude, Presidential Scholar, Biology |
Postgraduate Training | |
6/1998-6/2002 | Fellowship, Adult and Pediatric Endocrinology, Indiana University Medical Center, Indianapolis, IN |
6/1994-6/1998 | Internship and Residency, Internal Medicine/ Pediatrics, Indiana University Medical Center, Indianapolis, IN |
Board Certifications
3/2013 | Pediatric Endocrinology |
10/2012 | Endocrinology, Diabetes, and Metabolism |
Administrative Appointments/Responsibilities
Honors and Awards
2013 | Best Doctors in America, Best Doctors, Inc. |
2013 | Castle Connolly America's Top Doctors |
2012 | Castle Connolly America's Top Doctors for Cancer |
2012 | Compassionate Doctor Recognition |
2011 | Patients' Choice Award |
2010 | America's Top Pediatricians, Consumers' Research Council of America |
2005 | Fellow, American College of Endocrinology |
2005 | Fellow, American Academy of Pediatrics |
2002 | Richard B. Schnute Award in recognition of outstanding compassion, professionalism, and clinical care as exemplified by Dr. Schnute, Indiana University School of Medicine |
1994 | Walter G. Sterling Award for Excellence, University of Texas Medical School at Houston |
1990 | The Ruth & Lee Faust Award for outstanding academic achievement by a graduate in biological sciences, Loyola University |
Professional Memberships
Alpha Omega Alpha Honor Medical Society Member, 1993-present |
American Association of Clinical Endocrinologists Member, 2002-present |
American Thyroid Association Member, Medullary Thyroid Cancer Guidelines Committee, 11/2012-present Member, ATA Task Force on Childhood Thyroid Cancer Treatment Guidelines, 8/2009-present Member, 2008-present |
National Cancer Comprehensive Network, Fort Washington, PA Member, NCCN Thyroid Carcinoma Panel, 2007-present |
Pediatric Endocrine Society (formerly known as the Lawson Wilkins Pediatric Endocrine Society) Member, 1999-present |
The Endocrine Society Member, 1999-present |
Selected Publications
Peer-Reviewed Original Research Articles | ||
1. | Yip L, Lee JE, Shapiro SE, Waguespack SG, Sherman SI, Hoff AO, Gagel RF, Arens JF, Evans DB. Surgical management of hereditary pheochromocytoma. J Am Coll Surg 198(4):525-34; discussion 534-5, 4/2004. PMID: 15051000. | |
2. | Busaidy NL, Jimenez C, Habra MA, Schultz PN, El-Naggar AK, Clayman GL, Asper JA, Diaz EM, Evans DB, Gagel RF, Garden A, Hoff AO, Lee JE, Morrison WH, Rosenthal DI, Sherman SI, Sturgis EM, Waguespack SG, Weber RS, Wirfel K, Vassilopoulou-Sellin R. Parathyroid carcinoma: a 22-year experience. Head Neck 26(8):716-26, 8/2004. PMID: 15287039. | |
3. | Kouvaraki MA, Shapiro SE, Cote GJ, Lee JE, Yao JC, Waguespack SG, Gagel RF, Evans DB, Perrier ND. Management of pancreatic endocrine tumors in multiple endocrine neoplasia type 1. World J Surg 30(5):643-53, 5/2006. PMID: 16680581. | |
4. | Waguespack SG, Hui SL, Dimeglio LA, Econs MJ. Autosomal Dominant Osteopetrosis: Clinical Severity and Natural History of 94 Subjects with a Chloride Channel 7 Gene Mutation. J Clin Endocrinol Metab 92(3):771-8, 3/2007. e-Pub 12/2006. PMID: 17164308. | |
5. | Shaw SA, Camacho LH, McCutcheon IE, Waguespack SG. Transient Hypophysitis after Cytotoxic T Lymphocyte-Associated Antigen 4 (CTLA4) Blockade. J Clin Endocrinol Metab 92(4):1201-2, 4/2007. PMID: 17409339. | |
6. | Wray CJ, Rich TA, Waguespack SG, Lee JE, Perrier ND, Evans DB. Failure to recognize multiple endocrine neoplasia 2b: more common than we think? Ann Surg Oncol 15(1):293-301, 1/2008. e-Pub 10/2007. PMID: 17963006. | |
7. | Jimenez C, Cabanillas ME, Santarpia L, Jonasch E, Kyle KL, Lano EA, Matin SF, Nunez RF, Perrier ND, Phan A, Rich TA, Shah B, Williams MD, Waguespack SG. Use of the Tyrosine Kinase Inhibitor Sunitinib in a Patient with von Hippel-Lindau Disease: Targeting Angiogenic Factors in Pheochromocytoma and other von Hippel-Lindau Disease-related Tumors. J Clin Endocrinol Metab (*Cover Illustration) 94(2):386-91, 2/2009. e-Pub 11/2008. PMID: 19017755. | |
8. | Waguespack SG, Williams MD, Clayman GL, Herzog CE. The Successful Use of Sorafenib to Treat Pediatric Papillary Thyroid Carcinoma. Thyroid 19(4):407-12, 4/2009. PMID: 19355831. | |
9. | Waguespack SG, Rich T, Grubbs E, Ying AK, Perrier ND, Ayala-Ramirez M, Jimenez C. A Current Review of the Etiology, Diagnosis, and Treatment of Pediatric Pheochromocytoma and Paraganglioma. J Clin Endocrinol Metab 95(5):2023-37, 5/2010. e-Pub 3/2010. PMID: 20215394. | |
10. | Amhaz HH, Chamoun RB, Waguespack SG, Shah K, McCutcheon IE. Spontaneous involution of Rathke cleft cysts: is it rare or just underreported? J Neurosurg 112(6):1327-32, 6/2010. e-Pub 11/2009. PMID: 19929199. | |
11. | Cabanillas ME, Waguespack SG, Bronstein Y, Williams MD, Feng L, Hernandez M, Lopez A, Sherman SI, Busaidy NL. Treatment with Tyrosine Kinase Inhibitors for Patients with Differentiated Thyroid Cancer: the M. D. Anderson Experience. J Clin Endocrinol Metab 95(6):2588-95, 6/2010. e-Pub 4/2010. PMID: 20392874. | |
12. | Grubbs EG, Waguespack SG, Rich TA, Xing Y, Ying AK, Evans DB, Lee JE, Perrier ND. Do the recent American Thyroid Association (ATA) Guidelines accurately guide the timing of prophylactic thyroidectomy in MEN2A? Surgery 148(6):1302-10, 12/2010. PMID: 21134565. | |
13. | Jasim S, Ying AK, Waguespack SG, Rich TA, Grubbs EG, Jimenez C, Hu MI, Cote G, Habra MA. Multiple endocrine neoplasia type 2B with a RET proto-oncogene A883F mutation displays a more indolent form of medullary thyroid carcinoma compared with a RET M918T mutation. Thyroid 21(2):189-92, 2/2011. e-Pub 12/27/2010. PMCID: PMC3025175. | |
14. | Ayala-Ramirez M, Feng L, Johnson MM, Ejaz S, Habra MA, Rich T, Busaidy N, Cote GJ, Perrier N, Phan A, Patel S, Waguespack S, Jimenez C. Clinical Risk Factors for Malignancy and Overall Survival in Patients with Pheochromocytomas and Sympathetic Paragangliomas: Primary Tumor Size and Primary Tumor Location as Prognostic Indicators. J Clin Endocrinol Metab 96(3):717-25, 3/2011. e-Pub 12/29/2010. PMID: 21190975. | |
15. | Ejaz S, Vassilopoulou-Sellin R, Busaidy NL, Hu MI, Waguespack SG, Jimenez C, Ying AK, Cabanillas M, Abbara M, Habra MA. Cushing syndrome secondary to ectopic adrenocorticotropic hormone secretion: The University of Texas MD Anderson Cancer Center Experience. Cancer 117(19):4381-9, 10/1/2011. e-Pub 3/15/2011. PMCID: PMC3134535. | |
16. | Hong DS, Cabanillas ME, Wheler J, Naing A, Tsimberidou AM, Ye L, Waguespack SG, Hernandez M, El Naggar AK, Bidyasar S, Wright J, Sherman SI, Kurzrock R. Inhibition of the Ras/Raf/MEK/ERK and RET Kinase Pathways with the Combination of the Multikinase Inhibitor Sorafenib and the Farnesyltransferase Inhibitor Tipifarnib in Medullary and Differentiated Thyroid Malignancies. J Clin Endocrinol Metab 96(4):997-1005, 4/2011. e-Pub 2/2/2011. PMCID: PMC3070247. | |
17. | Khan MI, Habra MA, McCutcheon IE, Nogueras-González GM, Devin JK, Busaidy NL, Levine NB, Lindstrom W, Kagan D, Jimenez C, Waguespack SG. Random postoperative day 3 cortisol as a predictor of hypothalamic-pituitary-adrenal axis integrity after transsphenoidal surgery. Endocr Pract 17(5):1-25, 9/1/2011. e-Pub 3/2011. PMID: 21454228. | |
18. | Stephen MD, Zage PE, Waguespack SG. Gonadotropin-Dependent Precocious Puberty: Neoplastic Causes and Endocrine Considerations. Int J Pediatr Endocrinol (*article selected and evaluated by the Faculty of 1000) 2011(Article ID 184502):184502, 2011. e-Pub 3/2011. PMCID: PMC3095257. | |
19. | Hightower E, Cabanillas ME, Fuller GN, McCutcheon IE, Hess KR, Shah K, Waguespack SG, Corley LJ, Devin JK. Phospho-histone H3 (pHH3) immuno-reactivity as a prognostic marker in non-functioning pituitary adenomas. Pituitary 15(4):556-61, 12/2012. e-Pub 11/2011. PMID: 22120760. | |
20. | Clayman GL, Agarwal G, Edeiken BS, Waguespack SG, Roberts DB, Sherman SI. Long-term outcome of comprehensive central compartment dissection in patients with recurrent/persistent papillary thyroid carcinoma. Thyroid 21(12):1309-16, 12/2011. PMID: 22136266. | |
21. | Jonasch E, McCutcheon IE, Waguespack SG, Wen S, Davis DW, Smith LA, Tannir NM, Gombos DS, Fuller GN, Matin SF. Pilot trial of sunitinib therapy in patients with von Hippel-Lindau disease. Ann Oncol 22(12):2661-6, 12/2011. PMID: 22105611. | |
22. | Morris LF, Waguespack SG, Edeiken-Monroe BS, Lee JE, Rich TA, Ying AK, Warneke CL, Evans DB, Perrier ND, Grubbs EG. Ultrasonography Should Not Guide the Timing of Thyroidectomy in Pediatric Patients Diagnosed with Multiple Endocrine Neoplasia Syndrome 2A through Genetic Screening. Ann Surg Oncol 20(1):53-9, 1/2013. e-Pub 8/2012. PMID: 22890595. | |
23. | Morris LF, Waguespack SG, Warneke CL, Ryu H, Ying AK, Anderson BJ, Sturgis EM, Clayman GL, Lee JE, Evans DB, Grubbs EG, Perrier ND. Long-Term Follow- Up Data May Help Manage Patient and Parent Expectations for Pediatric Patients Undergoing Thyroidectomy. Surgery 152(6):1165-71, 12/2012. PMID: 23158186. | |
24. | Carhill AA, Cabanillas ME, Jimenez C, Waguespack SG, Habra MA, Hu M, Ying A, Vassilopoulou-Sellin R, Gagel RF, Sherman SI, Busaidy NL. The Non-investigational Use of Tyrosine Kinase inhibitors in Thyroid Cancer: Establishing a Standard for Patient Safety and Monitoring. J Clin Endocrinol Metab 98(1):31-42, 1/2013. e-Pub 11/2012. PMCID: PMC3537108. | |
25. | Shawa H, Elsayes KM, Javadi S, Morani A, Williams MD, Lee JE, Waguespack SG, Busaidy NL, Vassilopoulou-Sellin R, Jimenez C, Habra MA. Adrenal ganglioneuroma: features and outcomes of 27 cases at a referral cancer centre. Clin Endocrinol (Oxf). e-Pub 8/2013. PMID: 24033606. | |
26. | Thosani S, Ayala-Ramirez M, Palmer L, Hu MI, Rich T, Gagel RF, Cote G, Waguespack SG, Habra MA, Jimenez C. The Characterization of Pheochromocytoma and Its Impact on Overall Survival in Multiple Endocrine Neoplasia Type 2. J Clin Endocrinol Metab. e-Pub 9/2013. PMID: 24030942. | |
Invited Articles | ||
1. | Kalkan E, Waguespack SG. Endocrine Tumors Associated with Neurofibromatosis Type 1, Peutz- Jeghers Syndrome and Other Familial Neoplasia Syndromes. Front Horm Res 41:166-181, 2013. e-Pub 3/2013. PMID: 23652677. | |
2. | Waguespack SG, Rich TA, Perrier ND, Jimenez C, Cote GJ. Management of medullary thyroid carcinoma and MEN2 syndromes in childhood. (Selected as a Medscape CME article). Nat Rev Endocrinol 7(10):596-607, 10/2011. e-Pub 8/2011. PMID: 21862994. | |
3. | Khan MI, Waguespack SG, Hu MI. Medical Management of Postsurgical Hypoparathyroidism. Endocr Pract 17 Suppl 1:1-19, Mar-Apr, 3/2011. e-Pub 12/2010. PMID: 21134871. | |
4. | Francis G and Waguespack SG. An Individualized Approach to the Child With Thyroid Cancer. Expert Review of Endocrinology & Metabolism 6(1):85-92, 1/2011. | |
5. | Waguespack SG, Francis G. Initial Management and Follow-up of Differentiated Thyroid Cancer in Children. J Natl Compr Canc Netw 8(11):1289-1300, 11/2010. PMID: 21081785. | |
6. | Tuttle RM, Ball DW, Byrd D, Dilawari RA, Doherty GM, Duh QY, Ehya H, Farrar WB, Haddad RI, Kandeel F, Kloos RT, Kopp P, Lamonica DM, Loree TR, Lydiatt WM, McCaffrey JC, Olson JA, Parks L, Ridge JA, Shah JP, Sherman SI, Sturgeon C, Waguespack SG, Wang TN, Wirth LJ. Thyroid Carcinoma. J Natl Compr Canc Netw 8(11):1228-1274, 11/2010. PMID: 21081783. | |
7. | Tuttle RM, Ball DW, Byrd D, Daniels GH, Dilawari RA, Doherty GM, Duh QY, Ehya H, Farrar WB, Haddad RI, Kandeel F, Kloos RT, Kopp P, Lamonica DM, Loree TR, Lydiatt WM, McCaffrey J, Olson JA, Parks L, Ridge JA, Shah JP, Sherman SI, Sturgeon C, Waguespack SG, Wang TN, Wirth LJ. Medullary carcinoma. J Natl Compr Canc Netw 8(5):512-30, 5/2010. PMID: 20495082. | |
8. | Ying AK, Huh W, Bottomley S, Evans DB, Waguespack SG. Thyroid cancer in young adults. Semin Oncol 36(3):258-74, 6/2009. PMID: 19460583. | |
9. | Rich TA, Hu MI, Martin JW, Perrier ND, Waguespack SG. CDC73-Related Disorders [Includes: CDC73-Related Familial Isolated Hyperparathyroidism, Hyperparathyroidism-Jaw Tumor Syndrome, CDC73-Related Parathyroid Carcinoma] [Last Update May 24, 2012]. In: Pagon RA, Bird TC, Dolan CR, Stephens K, editors. GeneReviews [Internet]. Seattle (WA): University of Washington, Seattle; 1993-. e-Pub 12/2008. PMID: 20301744. | |
Editorials | ||
1. | Waguespack SG. A perspective from pediatric endocrinology on the hereditary medullary thyroid carcinoma syndromes. Thyroid 19(6):543-6, 6/2009. PMID: 19499985. | |
Book Chapters | ||
1. | Waguespack S, Walvoord E, Pescovitz OH. Precocious and Delayed Puberty. In: Principles and Practice of Endocrinology and Metabolism, 3rd. Ed(s) Becker KL. Lippincott Williams & Wilkins: Philadelphia, 893-908, 2001. | |
2. | Waguespack S, Maldonado M, Sekhar R, Gagel R, Balasubramanyam A. Endocrine Evaluation and Management of the Perioperative Cancer Patient. In: Acute Care of the Cancer Patient. Ed(s) Shaw A, Riedel B, Burton A, Fields A, Feeley T. Taylor & Francis: Boca Raton, FL, 43-67, 2005. | |
3. | Waguespack S, Wells S, Ross J, Bleyer A. Thyroid Cancer. In: Cancer epidemiology in older adolescents and young adults 15 to 29 years of age, including SEER incidence and survival: 1975-2000. Ed(s) Bleyer A, O'Leary M, Barr R, Ries LAG. National Cancer Institute, NIH Pub No 06-5767: Bethesda, MD, 143-54, 2006. | |
4. | Waguespack S, Wells SA. Thyroid Carcinoma. In: Cancer in Adolescents and Young Adults. 1. Ed(s) Bleyer A and Barr R. Springer Verlag, 259-68, 2007. ISBN: 978-3-540-40842-0. | |
5. | Waguespack SG, Bauer AJ, Huh W, Ying AK. Chapter 36 Endocrine Tumors. In: Principles and Practices of Pediatric Oncology, 6th. Ed(s) PA Pizzo and DG Poplack. Lippincott Williams & Wilkins, 1068-97, 2010. | |
6. | Waguespack SG. Chapter 500 Rare Tumors (Thyroid Tumors and Adrenal Tumors). In: Nelson Textbook of Pediatrics, 19th edition. Ed(s) RM Kliegman, BF Stanton, JW St. Geme, NF Schor, RE Behrman. Elsevier Saunders: Philadelphia, 1772-73, 2011. | |
7. | Bauer AJ, Waguespack SG, Grover A, Francis GL. Thyroid Neoplasia. In: Pediatric Endocrinology: A Practical Clinical Guide, 2nd. Ed(s) S Radovick, M MacGillivray. Humana Press, 319-338, 2013. | |
Letters to the Editor | ||
1. | Waguespack SG, Bauer AJ, Francis GL, Vassilopoulou-Sellin R. Radioactive Iodine for Pediatric Differentiated Thyroid Carcinoma: Sometimes "Less is More". J Clin Endocrinol Metab, http://jcem.endojournals.org/content/96/8/E1268/reply#jcem_el_89932, 10/2011. | |
2. | Waguespack SG, Rich TA. Multiple endocrine neoplasia syndrome type 2B in early childhood: long-term benefit of prophylactic thyroidectomy. Cancer 116(9):2284, 5/2010. e-Pub 2/2010. PMID: 20166211. | |
Other | ||
1. | Web-based CME Activity. "Mini-Case: Signs, Symptoms, and Treatment of Acromegaly in Adults." A Medscape CME Activity. August 29, 2011. |
Last updated: 10/10/2013
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Seesmic Desktop Is Ahead of the Game with Twitter Lists Integration
http://www.wchingya.com -
There has been a wild guess going on between Tweetdeck and Seesmic Desktop on who will launch the FIRST Twitter Lists integration? I was ecstatic to learn about Seesmic determination to be ahead of the game with Twitter Lists !
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Carillion awaits Middle East contract flurrySupport services and construction firm Carillion is in line for potential contracts in the Middle East worth more than GBP3 billion ($4.82 billion) in the next four months as infrastructure projects in the region show signs of life. More… |
Msheireb awards $650m deal to Carillion, QBCMsheireb Properties has awarded a joint venture of Carillion (Qatar) and Qatar Building Company (QBC) a construction contract valued at QR2.37 billion ($650.7 million) for the key phase of its flagship project, Msheireb Downtown Doha. Unde More… |
Carillion wins $158m Dubai contractBritish support services group Carillion said it won another contract in the Middle East and confirmed it expects to deliver improved earnings in 2009, sending its shares higher. Carillion said its 49 percent-owned Al Futtaim Carillion joi More… |


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Dr DHANA LAKSHMI (15th sept / kannur)
MuDdY......... wAtEr............! !
MuDdY......... wAtEr............! !
Pouring muddy water?
I clean my self
Again he throws
Muddy water
I throw him out again
There walks a new person
Sprinkles little mud around
I dust them here n there
And I walk towards life
He stops me there
I turn back at him
He splashes the muddy water
I wash them again with
With waters of tears!
Words of apology
Gestures of forgiveness
He takes my crying face
And paints me again
Again with muddy water
No tears this time
Its blood I use again
To clear the muddy water
And I limit the days I want to live
In distress he pats my pats my back
With his right hand
But alas he was staining me
With his left hand
Muddy water was it again
I clear my seat
I tidy myself
Painstakingly not with
Brush nor broom
But with scalpel and knife
Cutting and chistlling
Callous and critical
Every inch of my individuality
But still he would silently take me
Take me to the corners of love
& again teem me with muddy water
There I attempt to kill myself
& he pacifies me again
With explanations of muddy water
Now I know scrubbing and scouring
Will never help
Virtuous and untainted
Spotless and shipshape do I wannu be
But its muddy water this lfe
So i`l go beneath heaps of mud
Cause however much of muddy water
Muddy water he pours
I`l be mud all the time
My blood minced in mud
My bones decayed in mud
My flesh sink in mud
Every cell of mine
Crumble into the muddy mud! !
Comments about this poem (MuDdY......... wAtEr............! ! by Dr DHANA LAKSHMI )
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2012 Shazam FS Footwear (NEW) | Sombrio WEB SHOP | Canada + International
Has anyone wore these yet or seen them in person? I want to get some hightopped shoes for the summer to keep dirt/rocks out. These are a little cheaper than the 5.10 High Impacts so thought I'd ask. Don't want to spend that much dough when I could go a little more with a shoe that is proven.....
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