text: This project came out of the fact that as I began learning the characters I realized that knowing the radicals helped me learn new characters a whole lot faster than just trying to remember each character based on it's own merits, since sound and meaning is typically included in the component choices. What I find is strange is just about all the advice for studying chinese I've read and been given says learning the radicals isn't so important for a beginning student. Possibly because of this view I could find very little material that allows someone to study just the radicals themselves. This is basically a dictionary which covers all the radicals and their variants; several components are also included. The data includes meaning, notes, and etymology. Frequency information is included for the KangXi radicals and their variants (this is RANK, higher means more frequent). There is also a note for each character whether there is any point in learning it as a standalone character. The additional radicals from the modern simplified chinese tables are covered where possible, though in some cases these radicals only exist in extension B. I've now updated this for Pleco 2.0, the updates include: A number of new characters, particularly a number of Extension A characters which had been skipped before. Notes have been expanded to include how the radical is typically used in characters and the types of characters that will be listed under it. A Mnemonic field has been added, in many cases this was just separated from the notes data in the previous release, but this information has been expanded/refined in many cases. While this field is often the technical etymology that can't always be guaranteed. Examples have been added as per a request here, chosen to illustrate the most typical uses of a given radical There is a new 'Distinguish From' field A flashcard list is now included. Feedback is welcome, next steps is to put some effort on the various character components. Sources Primary Sources: Tan Huay Peng's Chinese Radicals These are great little books, highly recommended for this topic. Out of print but can still be found in bookstores. The Chinese Etymology website William Mcnaughton's Reading and Writing Chinese characters The Unihan Database Additional resources include: Unicode Code page reference docs on http://www.unicode.org Modern Chinese Character Frequency List I used the radical tables from the old open source version of Hanzi Master as an original source for the rank calculations, updated for my complete radical list Cantodict This site proved useful for finding the code points of some rare characters In addition to abovementioned sources all of the dictionaries within Pleco were consulted before writing the definition, a couple verbatim copies came from Unihan or CEdict. I also found component information on a number of sites, I think one of the main resources I used was a variant of the Unihan database somewhere on mandarintools.com. Wenlin was consulted for some component information and some of the etymology updates Note: due to some bugs in Pleco's handling of radical characters, the dictionary will not actually be searchable for some radical characters yet - this will be corrected in a future update. iOS Dictionary Installation 1. Make sure some sort of wireless/data connection is enabled on the iOS device. 2. Launch Pleco 3. Click the functions icon on the lower right hand corner of the screen 4. Click Settings 5. Scroll down and select Web Browser 6. Go to plecoforums.com and navigate to this forum thread. 7. Click the link to the "Dictionary Database" file below and save the file to your device. 8. Now go to the 'Manage Dicts' screen under Settings. 9. Tap the 'Add User' button, then "Load Existing", and choose the Radical dictionary. 10. Go back to the dictionary interface in Pleco 11. Go to the C-E dictionary list with ABC, etc 12. Cycle through the dictionaries by clicking on the dictionary icon, the icon is RAD. Alternatively tap and hold the dictionaries icon, select the one called Radicals. iOS Flashcards Installation 1. Repeat steps 1-7 above with the "Flashcard List" file. 2. Go to the "Import Cards" / "Import cards" screen under Flashcards. 3. Tap on "File" under "Choose File" and select the downloaded flashcard list file. 4. Set "Duplicate cards" to "Allow" - this will make sure that your newly imported cards don't link to old definitions from other dictionaries / flashcard lists. 5. Tap on "Begin Import." Android Dictionary Installation 1. Download the Dictionary Database file below. 2. Copy the dictionary .pqb file to your device's SD card. (you can also download it directly to your device) 3. In the main screen of Pleco, press your device's menu button (or the onscreen menu button on Android 4.0), choose "Settings," "Dictionary," "Manage Dictionaries," "Add User," "Load Existing," and select the dictionary file. Android Flashcards Installation 1. Download the Flashcard List file below. 2. Copy the flashcard .txt file to your device's SD card. (you can also download it directly to your device) 3. In the main screen of Pleco, press your device's menu button (or the onscreen menu button on Android 4.0), choose "Flashcards," "Import Cards." 4. Tap "Choose File" and select the flashcard file. 5. Set "Duplicate cards" to "Allow" - this will make sure that your newly imported cards don't link to old definitions from other dictionaries / flashcard lists. 6. Tap on "Begin Import."


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The first edition of People in Crisis, published in 1978, established success as a comprehensive and user-friendly text for health and social service professionals. The book and its following incarnations included critical life events and life cycle transition challenges, clearly pointing out the interconnections between such events, stressful developmental changes, and their potential for growth but also danger of suicide and/or violence toward others.
This revised edition includes new case examples and expanded coverage of cross-cultural content, including "commonalities and differences" in origins, manifestations, and crisis responses. The authors illustrate the application of crisis concepts, assessment, and intervention strategies across a wide range of health and mental health settings, as well as at home, school, workplace, and in the community. Each chapter contains a closing summary that includes discussion questions, references, and online data sources for maximum application and learning. Updated chapters discuss new, research-based content on:
• workplace violence and abuse
• youth violence in schools and higher education settings
• the use of psychotropic drugs, including for very young children in the absence of comprehensive assessment
• the crisis vulnerability of war veterans and the hazards of "pathologizing" what should be considered a "normal" response to the repeated and catastrophic trauma of war
• the intersection of socio-political factors with individuals' psychological healing from catastrophic experiences such as war and natural disaster.

subset: commoncrawl


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Endlessly photographed, filmed and fawned over, the right-hand point break at Lennox Head is one of Australia's most famous waves, consistently ranked in the top ten waves of Australia's 10,685 beaches.
The Lennox Head point break is beautiful to look at but powerful, and to ride it you need to find space among dozens of red-hot local surfers, so it's not the place for beginners. It breaks onto sand but there are some rocks to be scrambled over. Meanwhile the "Magic Miles" between Lennox Head and Ballina offers a mixture of beach breaks and reefs that work on a variety of wind and swell directions. The only time there are no waves on the "Magic Miles" is when there is no swell at all.
Lennox Head was declared a National Surfing Reserve in 2007, the third site in Australia to be recognised for it's significance to recreational surfing. The largest National Surfing Reserve in Australia , it runs 7.2km from the Surf Club south to Flat Rock.
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Safety Considerations : Use care and common sense before any activity
Patrolled : No
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Parking : Provided in the area
Location: Lennox Head NSW 2478
Search our database of fun things to do for everyone in the family. Whatever it be rain, hail or shine, there is always something to do and a new adventure to take on!


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A home protection plan is simple. It's a one-year service contract that covers the repair or replacement of many of the home system components and appliances that typically break down over time. It's not just about your budget - it's about having the confidence that a problem is handled before it even occurs.
As the industry leader with over 40 years experience, we have the know-how and financial stability to honor our word. Year after year, we stand by our customers. As an American Home Shield customer, you get the full confidence that when problems arise, we'll be there for you.


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Alcatel has launched a new smartphone in US known as the Alcatel Idol 4S with windows 10 OS. Alcatel previously launched the Idol 4S with Android Marshmallow back at Mobile World Congress this year.
The new Idol 4S Windows version is going to be available in US for only T-Mobile customers priced at $470 (Rs. 32,000).
The smartphone is powered by the Qualcomm Snapdragon 820 Chipset clocked at 2.15 GHz. There is also 4 GB Ram to ensure that it works smoothly.
The smartphone comes with a 5.5 inch Full HD (1920*1080) pixels AMOLED display. The design for the smartphone does not look very impressive at it has large bezels on the top and bottom. While rest of the world is moving towards thinner bezels (Mi Mix), Alcatel is still making Windows phone with thick bezels.
The smartphone comes with golden metallic edges and the glass body.
The smartphone comes with a 21 MP rear camera with Auto-focus support and Dual LED Flash. There is also a decent 8 Mega pixel front camera, along with a dedicated flash to take brighter selfies in low light conditions.
The Alcatel Idol 4S comes with 64 GB of internal storage which can be further expanded to 128 GB using the micro-sd card support.
The Dual Sim Smartphone comes with 4G LTE support along with the support for conventional 3G and 2G network.
One of the reasons of larger bezels on the Alcatel Idol 4S is the Dual front firing speakers which offer rich sound experience.
The Idol 4S is a sleek smartphone with 6.99 mm thickness, similar to the iPhone 6S. It weighs 152 grams and is backed by a 3000 mAH capacity battery.
The smartphone also comes with support for VR and the powerful Snapdragon 820 is sufficient to run all the VR APPs and games.
The Windows 10 mobile are rare to come by and these days we rarely get any inquires about the Windows phone. In the world dominated by Android and Apple, I wonder if there is space for a Windows phone any more.
There are definitely some Windows Phone fans who would find value in the Idol 4S.


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Marek Doniec's full part shot at season 2010-2011
Camera/Edit: Roadkill Productions
Supported by: Salomon | AA Man | Dakine | Go Pro | Honda | Dragon
Filmed at: Katowice | Kronplatz | Absolut Park | Alta Badia | Zauchensee | Hochkar | Kitzsteinhorn | Planai | Solden
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Claretta Carboneau
56, from Andover
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rolety Bydgoszcz Comet Sinkholes Cause Plane Create offers offset umbrella, restriction afterward CIS uses near recruitment consultancies, specialist, service provider plus corporate buyers. Spawn children's home dash dominance with superior bat-head rate here slow-pitch softball by subtracting a hobble tread toward the decanter. Except specialists verbalize until this look at carefully can be quite suggesting having the status of definitely not solely solved this specific project create just starting out, healthy-looking cartilage but in addition prominently at this time there happened rejection signposts associated with any side-effects this kind of in the role of swelling excessive otherwise disorganised, become a member of cells or even tumours. Their particular consumed would furthermore been situated cheaper also appropriate into a better variety of osteo-arthritis sufferer, the investigator retrieve. Or scrape your current levy proceeds, people should be aware which a amount of components could cause ones reimbursement for being waited or perhaps canceled downright. A numerous trawls suffer subsisted eradicated due to the made dirty h2o arrived brooks. Good United states Gasoline Initiate, expense hip keeping informed U.S. power commercial infrastructure deserted may perhaps cause about $1.14 trillion taking part in money purchase - founding both work with vitality savings since at the present in anticipation of 2025. Sent elegant powers happen and then employed to produce spatially taken care of atlases involving oxy- furthermore deoxy-hemoglobin, the 2 principal different parts of body. Sufferer while using the illness engage in muscle mass with the aim of deficiency a vital enzyme with the aim of moves amassed glucose straight into sugar, plan their own compartments subsist not able to engender an adequate amount of power. Vicinity regained so as to catalogue sureness must turn into further activity also risk-taking then maximize being real tools - bringing in abusing staying alive workforce, universe then albums - to get brand new cash flow, exclusive of giving up the nation or else heart objectives because free stockpile givers. Owners know how to utilize the Review function to get the failure to pay print parameters with the trace store. Julien Laurat in addition to the players by Pierre as a consequence Jessica Curie College or university fashionable Paris (Laboratoire Kastler Brossel-LKB), in addition to associates via several various other association world-wide, survey they have devised a tale diagram to get these kinds of lands having an unprecedented mass moreover training tempo. Append correct folder set-up postponements (.jpg,png, orgif) for the public figure in the stratums as well as level parties beginning that you simply need to crank out representation advantage. The facial example bred stylish retort for an indefinite menace circumstances ended up being exactly gamed on the way to vaguely jeopardizing predicaments throughout 90% involving circumstance demonstrations. The doers tell Katherine Jackson possesses distinguished quite a few homespun trendy of which cost extent that may go with the girl basics and this on the innocent person. A few big number dice add some D100 (before Zocchihedron, chosen right after it's creator Zocchi) plus D50.
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Horoscopes for June 15, 2014
TODAY'S BIRTHDAY: Prepare to accept change wholeheartedly. If you swim with the current, you will find peace of mind and satisfaction. Let past problems fall by the wayside. It's time to start anew and head in a direction that suits your life, personality and goals. Be true to how you want to develop and strive to fulfill your dreams.
ARIES (March 21-April 19): Don't settle for less. Find solutions to whatever is dragging you down. It's up to you to make things happen. Change can be good, but make sure your motives are good. TAURUS (April 20-May 20): You have to follow the path that best suits you. Updating your look or making home improvements will lift your spirits. Offering a service from your home will bring in extra cash. GEMINI (May 21-June 20): Don't let anyone entice you into doing something that attacks your integrity. Make changes based on honesty and trust. A secret is best kept that way. CANCER (June 21-July 22): Take part in a community event that explores different cultural backgrounds or takes you on a creative tour of what's available in your neighborhood. It's time to meet new people who share your interests. LEO (July 23-Aug. 22): Go somewhere interesting or engage in activities that will allow you to spend time with unusual people. Exploring possibilities will inspire you to take action. VIRGO (Aug. 23-Sept. 22): Put greater effort into the way you look while expanding your skills and talents. Being at your very best will help boost your confidence. Romantic suggestions will be inviting. LIBRA (Sept. 23-Oct. 22): Say little and do a lot. Set up plans to be with someone you enjoy spending time with. Make choices that will encourage you to say "yes" to the changes that will make your life fulfilling. SCORPIO (Oct. 23-Nov. 21): You can make changes at home, but stay within your financial means. Explore places that offer an insightful look into your surroundings. Make a decision regarding a relationship, and follow through. SAGITTARIUS (Nov. 22-Dec. 21): A change will do you a world of good, but stick to what you can afford to avoid added stress. An employment opportunity will tempt you. CAPRICORN (Dec. 22-Jan. 19): Spend time with the ones you love and you will build a strong home base. Beautifying your surroundings or adding something to your home that will encourage having fun should be considered. AQUARIUS (Jan. 20-Feb. 18): Consider new opportunities. Do your best to make improvements that will entice those around you to follow suit. Avoid impulse purchases and risky ventures.PISCES (Feb. 19-March 20): Get involved in a good cause. What you do to help others will be recognized and rewarded. Showing off your talents will lead to an interesting proposal.


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by Damon .M
(Long beach, ca)
Question: Hello, my mom is 73 years old and this is her second stroke. Her first stroke was in 2011. The doctor called it a speech stroke. h]Her mobility was not affected but her focus, cognition, memory, thinking and comprehension were affected. This 2015 stroke first affected her right leg only. She had minor weakness for the first week and no right arm problem at first, but after the first week in the hospital her right arm got really weak. After her blood pressure was stable she was transferred to the rehab center. When she was there, her balance was slightly off but she sat up on her own for about 30 mins. She needed about 50% assistance standing and walking. She would have done better but her focus was not there when it came to rehab instruction or commands. Also, she became apathetic and slightly depressed and didn't want to participate willfully. So the insurance dropped her down from acute rehab to SNF. When she was transferred to SNF, her progress went down for the first week she was there, and I got scared. I thought it was another stroke but I couldn't tell if was. The week after she started to progress again but slower than she was at the acute rehab center. It has been one month and her balance is improving, her
standing is 30%, she has good days and bad. Her left arm and leg is really strong. Doctors have told me she will walk again since she had a minor stroke, but give it 3 to 9 months. Right now I can't see that. Is this normal for stroke patients to do good for two weeks and don't do that good the next two weeks?
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Los Angeles - Nasa's lone surviving Mars rover is looking for a place to hunker down for the winter.The solar-powered Opportunity has been exploring the rim of a huge crater since August. With winter approaching, the six-wheel rover is on the lookout for a place with good sunlight.Opportunity and its twin, Spirit, parachuted to opposite ends of the red planet in January 2004. Earlier this year, Nasa declared Spirit's mission over after it repeatedly failed to contact Earth.Opportunity won't be alone on the Martian surface for long. Nasa launched a mega-rover named Curiosity that is to land in 2012.
I find these stories very interesting,and hope Curiosity has a safe flight.
In other news: A feline like creature was discovered on Mars but kill shortly afterwards when a mars rover landed on it. It seems Curiosity killed the cat.


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Markus Kuger - Dun & Bradstreet
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When it comes to getting rhinoplasty done, you might be wondering if it would be worth it or not. That is normal, because, in terms of cosmetic procedures, nothing can be assured or guaranteed. You never know what can happen. However, that does not mean that you would not go with all the preparation in advance. The more you would know about it, the better it would be for you.
Now when it comes to choosing a nose job in Manhattan, you would be able to find that it has become increasingly common in the modern day and age. Almost every other person is getting this procedure done. Also, you would also be able to find that there are tons of surgeons who are willing to do this for you. However, just because something has become pretty normal and common does not mean that you would be able to get the best outcomes from whichever surgeon to visit. Just like all the other things, you would also need to be pretty selective when it would come to choosing a plastic surgeon.
Therefore, the next thing that you would be thinking about is the benefits which would be provided if you decide to come to us. After all, it would be pretty standard to find out what we will be able to offer you before you make your decision. So to fulfill your curiosity, we have listed below some of the benefits that our clients would be able to get.
• One of the main advantages that you would be able to get from getting rhinoplasty in Manhattan done by us is the access to some of the best surgeons in town. Not only that, but you would also be happy to find that all of them are licensed and extremely professional. So right from the beginning, to your initial consultation and till your discharge, you will always be able to find one of our staff with you.
• Regarding the cosmetic procedures which would be done, you would be happy to find a broad and diverse range being presented in front of you. Despite the fact that our specialization tends to be nose job Manhattan. We seem to understand that different people have differing needs and conditions. So by taking that into account, we would like to increase the satisfaction of our customers.
• The majority of our surgeons are involved in various organizations and committees. In addition to that, they are also attending different workshops to enhance their skills. While this might not come off as very relevant to you, it is, because the specialist knowledge of our surgeons would be able to help improve your experience as a patient. It can also help you avoid expensive treatments which would not yield desirable results.
• Lastly, we also seem to provide flexible payment opportunities for nose jobs in Manhattan. In this way, you would not need to worry about meeting the payment deadlines once you are done with your surgery.


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Scottish water pollution incidents increase
Serious water pollution incidents in Scotland are on the increase for the second year in succession - according to data from the Scottish Environment Protection Agency (SEPA) leaked to ENDS. In 1999/2000 SEPA reported 2,306 "routine" incidents and 282 "significant" ones. The latter, the highest yet reported by the Agency, is an 8% increase on the previous year.
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Business with Pleasure: Hot Down Under
Keziah Hill
For sculptor Lottie Williams, entering her sculpture, Triumvirate, in the Carve the Wind exhibition is the culmination of her career.
What she doesn't count on is the incendiary lust between her and the curators, James and Magda. While she's at first happy to play, she rapidly gets out of her depth when she confuses business with pleasure. Against the wild backdrop of the Pacific Ocean her work is displayed to great acclaim.
But all she wants, all she can think about is being back in the bed of the two people who have laid claim to her heart and soul.
Available via:
Buy Direct (All devices except Kindle)
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Barnes and Noble (Nook devices)
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Author Information
Keziah Hill
After years working in the criminal justice system, Keziah Hill decided a tree change was needed so decamped to the Blue Mountains, west of Sydney, Australia. Amid a garden full of flowering blossoms, roses and the odd marauding possum, she writes steamy love stories in which characters get their happily ever after or at least their happy for now. http://www.keziahhill.com https://www.facebook.com/KeziahHillAuthor https://twitter.com/KeziahHill


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Five years ago today, Jeff Green underwent the longest ordeal in his life: a five-and-a-half-hour surgery to repair an aortic root aneurysm near his heart. Green was fortunate that a routine physical with the Celtics in Dec. 2011 caught the problem, because often it goes undetected until the aneurysm bursts. And once it does, there's a high risk of immediate death.
But Green made a remarkable return to the NBA just nine months later in Oct. 2012, and since then he's been one of the best role players in the league who can play either forward position. Off the court, he's been enjoying a fulfilling life. He received his bachelor's degree from his alma mater, Georgetown, and became a voice for the American Heart Association, assisting young patients with cardiac issues during his time in different NBA cities. And last summer, he proposed to his fiancee, Stephanie, and they're expecting their first child next month.
Green spoke in-depth with the NBPA about his memories of the life-changing experience, mindset today, medical outreach and much more. His conversation is presented below in a first-person perspective and edited for clarity and length.
Every morning waking up and every night going to bed, I look at my scar.
And I tell the Lord, "Thank you," for the blessing that he's given me to keep living, to keep playing, just to continue to breathe everyday. I'm very fortunate and thankful everyday to the man up above because without him, and my friends and family, I wouldn't be here.
The scar is now just a reminder. But for the first couple of years after the surgery, it was definitely like a "wow" factor because growing up, you never think you have to go through anything like that. You're playing basketball having the physical tools, thinking that everything is fine, and then one day you're being told, "You've got to have heart surgery. You've got to put your basketball career on hold and you might not be able to play again."
It's just a blessing to still be in the NBA and still playing, to have gone through dealing with the surgery and the progression of still being healthy. It's still amazing that I can live a normal life. The times that I'm very thankful for are just being able to walk the mall with my fiancee, or just go out and hang out. I had to put a lot of that on hold that year.
I definitely have more of a greater appreciation for all of the little things—like just breathing or just hanging out, being with family—because I almost lost my life at that period of time. And I'm able to continue to spend time with my family and not over-obsess about material things now or take time for granted. I realize how precious all those little things are.
These days, I don't have to take any medication. That stopped a couple of months after the surgery, so I'm glad that's over. That was a lot of pills I had to take. What I do now is a checkup before the season, which is mandatory by the NBA, and I've been doing it after the season as well. The doctors said I don't have to do a second one, but it's just a mental refresher that everything is OK.
I'm definitely thankful that the NBA makes us go through the preseason stress echocardiograms because all of the tests and checks for everything allow us to have a clear mind on good health. Also if anything does come up, they give us an opportunity to get it fixed or get it checked up to make sure that we're OK.
Former NBA commissioner David Stern was amazing to me and my recovery. He allowed me to be with the Celtics during that period of time, even though I didn't have a contract, because he knew how much basketball meant to me. He knew how being with the Celtics could help me in my recovery, physically and mentally. So I definitely owe him a huge thank you for what he did for me.
I remember everything about what happened.
I went up to Boston on Dec. 9, I did the physical and then I went through two days of doctors examining the images from the stress test. And then on Dec. 11 or 12, that's when the doctors told me that I needed the surgery. I'll never forget—my best friend, Willie Jennings, was at every doctor's appointment with me. And when I got the news, it was just a shock.
Once I found out I did have the aortic root aneurysm and played with it for several years, I definitely thought, Damn, what if something happened at that time? While I had been playing well in my first few years in the NBA, I definitely had my ups and downs—body fatigue, body shock. You could ask the Boston head strength and conditioning coach, Bryan Doo. During the 2010-11 season, I definitely had to take a couple of weeks off from doing any lifting or extra work on the court because my body was at a point where it was at shock. I was in major fatigue during the season.
My first question to the doctors when I heard the news was, "Will I ever be able to play again?" And they said that I had to have the surgery to play again, and I said, "If I don't get it, what could happen?" I could play with it like I had done for the last couple of years, and nothing could happen, or I could play with it and possibly die on the court, like Reggie Lewis who played in Boston. Or I could die in any strenuous activity. The tissue could rupture and that could be it. So it was no question that I definitely had to get the surgery.
After a day or two, I came to grips with it and told myself, "I had to be better for me to play again." And then Jan. 9 was the day of the surgery at the Cleveland Clinic in Ohio.
Everything happened so quick. I definitely didn't have a lot of time to just think. I had to quickly tell myself that I've got to be good with it, I have to make sure that I'm well prepared for what I'm going to go through, because I don't think people understand. It's not an ankle surgery, it's not ACL, it's not a shoulder, it's not a back—it's heart surgery. And I think people take for granted what I had to go through. I don't think they realize what I had to deal with to try to get back.
The hardest part was four days after the surgery—my heart rate was very high, I was choking, I couldn't cough, and I started crying. And it was seeing the way my chest looked, definitely deformed. I'm a fit guy, I'm a skinny guy, but to look in the mirror I was bloated with all of the meds. It definitely did not look like myself and that was the hardest thing to see, but the doctors told me that that was the first thing I needed to do—look at myself in the mirror and see who I am now and deal with it and try to get back after that, because it was going to take a while before I was able to start the rehab process.
I stopped watching basketball for almost two months because I couldn't take it, I couldn't think about me playing. Mentally it was frustrating, but I was able to go back to school at Georgetown and graduate, so that was the biggest thing that I did at that time.
A year ago my life changed…#1-9-12 #blessed to see another day and thankful for my family and friends. LOVE Y'ALL!!! pic.twitter.com/33Y9yRu0
— Jeff Green (@unclejeffgreen) January 9, 2013
In March 2012, I started the therapy in Boston with the team doctors, and then after I was cleared to go back to D.C. I was going back every summer to Georgetown prior to the surgery, and I had another two courses to finish that year. I was in the classrooms, I was doing the lectures two hours a day, and I was able to graduate in May with a degree in English and minor in theology. I walked, had the cap and gown, and my family was able to come, so it was a big moment.
Also, my Celtics teammates were all there for me during that time and they helped me get through the surgery. Without them, mentally I definitely would've been frustrated, I would've been mad. I couldn't touch a basketball for six, seven months, so for them to talk to me always, "Everything's going to be OK," they definitely helped me out mentally to prepare myself for what I was going to go through to try to get back on the court.
I didn't have any hesitation about my heart when I returned to the NBA in Oct. 2012, because when I was playing pickup basketball at Georgetown that summer, I played without a padded vest. In the NBA, I wear one. I was talking to the kids, "Just hit me. I don't want you to pull back anything." And that's how I got the confidence to go back to playing the stye of play that I play, which is physical. By the time for the season, I had no kinds of hesitations whatsoever as far as driving to the lane or just playing.
For my first two games back—in Istanbul and Milan—it was definitely a lot of butterflies. Both games I played pretty well, which was a shock as far as the contact and the speed. I was definitely out of game shape, but it was fun though. And the first game in Boston was definitely priceless, walking back in the Garden and having the fans being there.
After that Christmas 2012 game against Brooklyn, which we won and I scored 15 points, that's when I was, like, "I'm back." I felt my body was coming back around then. That's when it hit me that I'm happy to be back, I'm very thankful, I'm very appreciative of the moment.
I've made helping young cardiac patients a big part of my life because when you don't have to deal with heart issues, you're blind to them.
The fact that I did have to go through that procedure, I've met so many kids—at NBA games and local hospitals—who have gone through that at an early age. And you only think, Wow. You bring a smile to their face and they look up to you, but they also do something special for you. To see the smiles on their faces as they go through the pain, go through the surgery, it definitely makes you appreciate everything you have a lot more. They're so young and you wish that they didn't have to go through that.
I did one event in Boston with the American Heart Association for some kids at the practice facility. One of the messages was they all said that they felt different, they all felt like they were viewed as being different. And I just told them, "Don't view it like that. You're still normal, but now you're a superhero."
When I had my surgery, I always stayed positive and one of my favorite superheroes is Iron Man. And that was always my alter ego. It's everything to me, so that's what I told them, "Continue to live your life and know that you're still the same. Just have a spot to prove that you're building character and that you're stronger now."
When I was with Memphis, I went to Le Bonheur Children's Hospital quite a bit with the American Heart Association. I want to do an event here soon in Orlando.
I like working with the American Heart Association because of how they try to gain attention to these things. Because like me, I didn't even know; I played with my aortic root enlarged for several years and I didn't even know. So it was definitely a scare. Some kids, because they don't have to do the physicals all the time, might have it and might not know. So the American Heart Association definitely brings awareness to the situation and they try to make sure that parents are getting their kids checked, because you never know what could happen at any given time if they do have it.
. @UncleJeffGreen #FaceTimeSurprises @LeBonheurChild Cody, who is recovering from the same heart surgery as Green.https://t.co/1r0fl43rMP
— Memphis Grizzlies (@memgrizz) August 20, 2015
I definitely would love to continue working with young patients after I'm done playing basketball. Every voice, like mine, that speaks on the situation would definitely bring light to what I had and definitely help somebody out. A lot of people want their kids to play sports, and if you bring awareness to it, it can change some kid's life. And that's my goal and that's my plan with that.
I've also talked to a couple of NBA guys about their heart conditions, like Ronny Turiaf and Etan Thomas, who I played with when I was in OKC. Also, I've talked to guys who had the scare, but didn't have to do the surgery, like Chuck Hayes. I saw him a couple of weeks ago and asked him how he was doing, and he said everything was good. When you get faced with that scare, you realize much more how precious that time is, life is. When someone gets hit with it, you try to make sure they're OK, you try to keep the relationship. That's what I have been doing. It's also great to hear about the NBPA's heart screening program for retired players.
I never knew the late Anthony Mason, but I met him a couple of times because during my time at Georgetown, his son, Anthony Jr., was at St. John's. You don't want anything bad to happen to anyone, especially something as serious as the heart and going through that type of pain. You just wish that legends like Anthony could've been helped in any kind of way.
I wish they could've been saved.


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Stock market project cover page designs jobs
Application to manage inventory in a textile company. It will include modules like grey yarn stock, in process stock, dyed yarn stock, clients name, dyes and chemicals stock, inward, outward, issue to dyeing, billing, reports export in excel etc. Some of the fields to be captured - weight, no. of cones, rate. We have an application but some of these
Development of Trading strategy : Kite Connect interface for data & trades Strategy is bar identification + high/low breakout Order management using SL - order has to place & track before level cross Monitor admin position, m2m & total profit Platform is Python
Hello. Need an Inventory System + Stock Management to be integrated with Hikashop, Joomla. Please see the attachment.
...create a selection list of flavours which is used throughout the site. We are looking for a freelancer to add an option for admin to tick which will mark this product out of stock and reflect across the site. Must be completed asap. There are many more tasks which I will be creating separate posts for. You will be more than welcome to bid on those
Subtotal Formula to avoid time consuming manual subtotals. I have been trying to use a SUMIF function based on my research but have been unable to figure it out so far.
(Solo idioma español) Necesito crear un sistema de facturación y control de stock por sucursal. Con los siguientes requerimientos: Requerimientos funcionales 1. ABM Proveedores 2. ABM Clientes 3. ABM Artículos (con stock mínimo) 4. ABM Tipo de Artículo 5. Facturación, Presupuestos y remitos. a. Lectura por código de barra o tip...
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Need a stock option accounting expert, well versed in FAS 123R/ASC Topic 718 Stock Compensation to assist private client in preparing monthly stock compensation accounting entries and quarterly financial statement disclosures.
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Hi I want to create stock cum inventory management including barcode for small shop and super market
I am looking for image stock for printing glass stickers. File must be of high quality for printing.
I'm an independent user (non-commercial) just trying to import stock quotes into Excel for personal use since Yahoo discontinued their access which I had previously very successfully used for years. I'm now using a Google Finance webquery in Excel to replace it that I wrote and it works fine, but it is very slow on the first query and I need to need
Hello I am looking for an assistant to help me on the daily trading day for a weekly fee. You must understand analyzing stocks and explain every stock to me.
To sell a stock of Wellbed our vibro massager mattres for relax and wellness Price on our website is 450 + taxes + delivery [url removed, login to view] We want to sel in stock our 70 pieces at e 199 + taxes + delivery each one Minimum stock 10 pieces Ogni giorno, probabilmente in modo del tutto inconsapevole, il corpo umano è sottoposto a
I am looking for a promoter able to propose and sell to public and private schools my course "Italian for foreigners - From beginner to intermediate level with only 25 lessons" The course is in ebook format and can be provided also as individual lessons in pdf. The goal of this work is place 10.000 download of my course. The price of each piece is 2.50 euros. After the sell the promoter ...
Dear all, I'm searching a seller able to sell a stock of 600 Tablet 7" 3g; Android 6.0; Dual camera;1024 * 600 HD, 5 points capacitive touch screen; duad core; dual sim card; Dual Standby; Bluetooth. Price 50 €. The seller will receive a fee of 1800 € after the sell. The tablets must be sold not individually but at least with orders of 200 pieces. For
We are looking for a logo design that will include modern, clean, sleek design element...Trading" Logo should be able to use both "BallStreet" as a single word as well as "BallStreet Trading" as two words. The logo should draw from the following elements: Stock Market Sports Will need logos/files in PNG hi-res with transparent backgrounds
Migrate the "old" advanced stock management from Prestashop 1.6 to prestashop 1.7. So is needed a module to allow having stocks over multiple warehouses in Prestashop 1.7 and show / manage stock of different warehouses on the product page. Thge module need to be perfectly integrate dwith prestashop ecommerce, so the multiple stock management need to be
Dear all, I'm searching a seller able to sell a stock of 600 tablet 10" 4g Octa Core Dual Sim Android 6.0 Memory 2GB +32 GB. Price 100 €. The seller will receive a fee of 1800 € after the sell. The tablets must be sold not individually but at least with orders of 200 pieces. For confirm that you are agree with my conditions please start your message


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Here's a happy eating experience I've just discovered; those of you who love bread, just bread, might find it particularly fun.
I know that some of you are fans of Hi-Rise Pie Co. on Brattle St. in Harvard Square. Well, I was there around mid/late afternoon ('coffee break') a couple of days ago, sniffing around, and I wasn't hungry enough for a sandwich (had no money anyway) nor did I want to pay too much for an OK baked good. So, just for kicks, I bought myself some toast. Two slices of your choice of bread(since I'm new to Hi-Rise I just got a slice of white and a slice of wheat, thought I'd cover basics), toasted, plus butter and preserves. For three dollars. Ehh. But I thought I'd splurge a bit...
So I went to the little sitting area upstairs, and the serving person brings up the toast in a little basket. And there was a cute little triangle of butter, and a wonderful little container of blueberry-lime preserves -- full of big, sugary blueberries, really interesting -- and two thick slices of toast! So I sat there for most of the afternoon, and read college-y stuff, and munched some excellent, warm, crusty toast. Never before done such a thing...but it was a nice afternoon, and toast makes for a real nice afternoon snack.


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Straight Bait 4Favorites [+]
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Running Time: 126 minutes
Released: 06/2012
Studio Name: Edward James Productions
Series: Straight Bait
Director: Edward James
Description: Who`s the bait, and who is doing the fishing? When these horny, hunks get thrust together, either the gay dude can`t resist seducing his prey, or the straight guy just wants to indulge his curiosity, and/or get is rocks off! Animal passions collide...and both are sexually satisfied. Could it be the start of something? Enjoy this fourth indulgence in this crotch tenting series!
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|File Search
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To produce a PDF manual, issue the command "make pdf" after running `./configure'. -*- If you do not wish to build the C++ components, use: ./configure --disable-cxx -*- The `examples' subdirectory contains some examples of using libconfig from C and C++. -*- The config file syntax changed between versions 0.8 and 0.9. In order to implement all of the feature/enhancement req more»
These files are not maintained by me; they are user contributions. They are not guaranteed to work with this release of libconfig.
# << Usage >> # you can feed this README to irb and see the result # $ irb README # IMPORTANT NOTICE: # be careful with big *fixnum* (plain int) values in configs # int is 32 bit, but ruby fixnum is only 31! # For example, 2100000000 will be read as -47483648. require 'rconfig' c = Config.new c.read!('test.cfg') # => IOError c.read('test.cfg') # => false p c['some_var'] # => SettingNotFoundEr more»
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- 3 years old
- Date of birth: Feb 2, 2012
- Place of birth:
Longview, Texas, United States
- Date of passing: Feb 11, 2015
- Place of passing:
Dallas, Texas, United States
|Let the memory of "Beau Bo" be with us forever
"HAPPY 2nd BIRTHDAY In Heaven Beau Bo , Nana loves and misses you so much ...."
"Merry Christmas in Heaven ♡
I sure wish you was here, I miss you so much but most of all I hate seeing the pain in your Mama and Daddys eyes. We all miss you so much . You are forever in our hearts. Our lives will never be the same. Nana loves you"
"Happy Birthday Day in Heaven Beau Bo , I know the Angel's have made your day special . I love you and miss you so much . Forever in Nana's heart my sweet boy . Hugs and kisses"
"Nana loves her Beau Bo"
"My Beau Bo, Nana misses you so very much baby boy. Love you always and forever"
"Beau Bo,
I love you and miss you so much ."
"My condolences go out to the family and friends of Matthew. From reading what is written about him and looking at his cute photos I know he is missed and loved by many. No matter what, it can truly be said that death is an enemy and soon it will be brought to nothing (1Cor. 15:26). I have found much comfort in the Bibles promise of a resurrection hope of our loved ones at John 5:28, 29 and hope you will too. Death was never a part of Gods original purpose, in fact he yearns to soon bring an end to it and the suffering it causes your family and friends in dealing with the loss of such a young and beautiful little boy. Revelation 21:4 states: "And he will wipe out every tear from their eyes, and death will be no more, neither will mourning nor outcry nor pain be anymore. The former things have passed away." I did not know Matthew, but I read his obituary and I hope these scriptures will help to bring some comfort knowing that others care. If you would like to learn more about the Bibles promises and the hope it brings, please feel free to contact me."
"Love you Beau Bo ! Nana is having a hard time accepting that your not here .. I miss you so much , sending you a big kiss ...."
"Beau Bo, you are truly loved by many friends and family,I didn't get to share much time with you but I loved you,and miss you,I love your family dearly,Hugs to you sweetheart."
"Missed very much but always in our heart and thoughts."
"My Beau Bo , Nana loves and misses you so very much . Hugs and kisses"
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Market News
25 Dec
PETALING JAYA: The German government is selling the two properties it owns in the heart of Kuala Lumpur for a potential combined worth of RM225mil.
09 Dec
KUALA LUMPUR: Sunway Real Estate Investment Trust (Sunway REIT) ( Financial Dashboard) plans to acquire Sunway Hotel Georgetown and Wisma Sunway office building for a combined RM134 million from subsidiaries of Sunway Bhd.
08 Dec
PETALING JAYA: Mass Rapid Transit Corporation Sdn Bhd has finalised names for the 31 MRT stations located on the Sungai Buloh-Kajang line, also known as Line 1. "The name changes are to better reflect the locality of the station," MRT Corp CEO Datuk Wira Azhar Abdul Hamid said in a press briefing here yesterday.
08 Dec
PETALING JAYA: Property heavyweight IOI Properties Group Bhd (IOI Prop) is making a bold move into the Taiwan real estate as it proposes to buy a stake in iconic skyscraper Taipei 101 for RM2.74bil (NT$25.14bil) at a time when the ringgit is weakening.
05 Dec
OFFICE rental rates held steady with improved occupancy during the review period. Meanwhile, incoming office supply is expected in the fourth quarter
20 Nov
KUALA LUMPUR: Axis REIT Managers Bhd, manager of the Axis Real Estate Investment Trust (Financial Dashboard) (Axis-REIT), has allocated between RM300 million and RM400 million for asset acquisitions next year, as it continues to increase its asset under management (AUM).
13 Nov
KUALA LUMPUR: The Nomad Group Bhd wants to hive off six assets to a Hong Kong-incorporated management service firm for £4.25 million (RM22.3 million) cash, which is about half or 47.3% less than the RM42.3 million initial investment the group forked out for the properties between 2007 and 2014.
04 Nov
Meet Malaysia's Own 'Cake Boss' Whose Creations Have Caught The Attention Of International Superstars And Local Celebrities
The original Cake Boss Buddy Valastro was in town earlier this week to much celebrity fanfare but did you that a Malaysian has been quietly making waves for his jaw-dropping cake artistry too? He's the man credited with shaking up the local baking scene with his 3D cakes, which are nothing less than works of art in their own right.
31 Oct
PETALING JAYA: The MMC Corp Bhd and Gamuda Bhd joint-venture (JV), which will carry out the second mass rapid transit line (MRT2), will stand a higher chance of clinching the underground works for the project, according to analysts.
27 Oct
KUALA LUMPUR: Maju Group, controlled by businessman Tan Sri Abu Sahid Mohamed, has enlisted the help of tycoon Tan Sri William Cheng's Parkson Group to manage and operate Maju Junction Mall here.


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This E-group is to share information between the Galizien German Descendants searching for their family history. Galicia (Galizien, Galicja), a former Austrian province, was a part of South - East Poland, today Western Ukraine.
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Age increases the risk for ischemic acute kidney injury (AKI). We questioned whether a similar age-dependent injury occurs following exposure to hemoglobin, a known nephrotoxin. Old mice (∼16 mo old), but not young mice (∼6 mo old), when administered hemoglobin, exhibited marked elevation in blood urea nitrogen (BUN) and serum creatinine, and acute tubular necrosis with prominent tubular cast formation. The aged kidney exhibited induction of heme oxygenase-1 (HO-1) and other genes/proteins that may protect against heme-mediated renal injury, including ferritin, ferroportin, haptoglobin, and hemopexin. Old mice did not evince induction of HO-2 mRNA by hemoglobin, whereas a modest induction of HO-2 mRNA was observed in young mice. To determine the functional significance of HO-2 in heme protein-induced AKI, we administered hemoglobin to relatively young HO-2+/+ and HO-2−/− mice: HO-2−/− mice, compared with HO-2+/+ mice, exhibited greater renal dysfunction and histologic injury when administered hemoglobin. In addition to failing to elicit a protective system such as HO-2 in response to hemoglobin, old mice exhibited an exaggerated maladaptive response typified by markedly greater induction of the nephrotoxic cytokine IL-6 (130-fold increase vs. 10-fold increase in mRNA in young mice). We conclude that aged mice, unlike relatively younger mice, are exquisitely sensitive to the nephrotoxicity of hemoglobin, an effect attended by a failure to induce HO-2 mRNA and a fulminant upregulation of IL-6. Age thus markedly augments the sensitivity of the kidney to heme proteins, and HO-2 confers resistance to such insults.
- age
- heme proteins
- acute kidney injury
- heme oxygenase-2
- interleukin-6
as shown by observations in experimental and human acute kidney injury (AKI), aging renders the mammalian kidney increasingly vulnerable to acute insults and, in the wake of such insults, an increased risk for chronic kidney disease (CKD) (3, 8, 33, 42, 44). Analyses in rodent models of AKI demonstrating this age-dependent susceptibility to AKI, however, have relied almost exclusively on acute ischemic insults to the kidney, with little, if any, attention directed to the effect of age on the renal sensitivity to nephrotoxic insults (3, 8, 33, 42, 44).
An important cause for nephrotoxic human AKI resides in exposure of the kidney to heme proteins, as occurs following hemolysis or rhabdomyolysis (39). Understanding the basis for renal injury by, and adaptation to, heme proteins is relevant not only to these diseases, but also to AKI in general: acute insults to the kidney, whether ischemic or nephrotoxic in origin, destabilize intracellular heme proteins, leading to increased kidney content of heme, a known nephrotoxic metabolite (39). Prior studies in heme protein-induced nephrotoxicity demonstrated that susceptibility to renal injury is mitigated by adaptive processes centered on the induction of heme oxygenase-1 (HO-1) (23), a finding now extended to ischemic and other causes of AKI (1, 2, 18). In this regard, recent studies have indicated that the increased renal sensitivity to acute ischemic insults observed in aging may reflect an age-dependent impairment in the inducibility of HO-1 in the kidney (13).
The present study examined whether renal susceptibility to heme proteins is increased in the aged kidney, and questioned whether an impaired adaptive response, either alone or in concert with an exaggerated maladaptive response, may contribute to any such observed age-dependent renal vulnerability to heme proteins. Examination of adaptive systems focused on the processes that enable metabolic clearance of heme proteins by the kidney, including HO-1 and HO-2 (1, 2, 6, 39). HO-2, the constitutive isoform of HO, is substantially present, unlike HO-1, in the unstressed kidney under basal conditions (11); notably, the functional significance of the HO-2 isoform has not been examined, to date, in heme protein-induced or, indeed, in any form of AKI. Examination of maladaptive processes centered on the expression of proinflammatory cytokines that are recognized contributors to AKI; the ones selected in this study were those that are known to be either heme-inducible or HO-inhibitable (28, 40, 41). Finally, as AKI may be a precursor to CKD, our examination of maladaptive systems extended to chronic inflammation-related genes that underlie the evolution of CKD.
Studies in Mice
All studies were approved by the Institutional Animal Care and Use Committee of Mayo Clinic and performed in accordance with National Institutes of Health guidelines. In studies of renal responses to heme proteins, C57Bl6J mice (Jackson Laboratory, Bar Harbor, ME) were sex-matched and mice of ∼6 mo (young) or 16 mo (old) of age were utilized. For the assessment of the renal responses to heme proteins in the setting of HO-2 deficiency, HO-2−/− and HO-2+/+ mice were employed, as in previous studies (12, 35). Colonies of these mice, having targeted disruption of the HO-2 gene as described by Poss and Tonegawa (29), were maintained by mating of HO-2+/− males and females and genotyped at the time of weaning using PCR to amplify the wild-type and mutant alleles of genomic DNA from tail samples. Similar numbers of male and female HO-2−/− and HO-2+/+ mice generated from these matings were used in these studies and were age-matched (between the ages of 5 and 6 mo). All mice in these studies were administered bovine hemoglobin (180 mg/100 g body wt, catalog no. H2500, Sigma Aldrich, St. Louis, MO) or sterile normal saline vehicle via tail vein injection. In prior studies this dose of hemoglobin did not lead to an elevation in serum creatinine in relatively young wild-type mice (25) and was thus employed in the present studies. Mice were euthanized at 1 and 2 days after the hemoglobin injections for the examination of renal histological alterations and the study of relevant gene and protein expression. Assessment of renal function in mice in these studies was achieved by measurement of serum creatinine, using a Creatinine Analyzer 2 (Beckman Instruments, Fullerton, CA), and BUN, using a kit purchased from Pointe Scientific (Canton, MI).
Assessment of mRNA Expression by Real-time RT-PCR
Renal gene expression was assessed by quantitative real-time RT-PCR as previously described (10). Total RNA was extracted and further purified using the TRIzol method (Invitrogen, Carlsbad, CA) and an RNeasy Mini kit (Qiagen, Valencia, CA), respectively, and cDNA was synthesized employing random hexamers (Transcriptor First Strand cDNA Synthesis kit, Roche Applied Science, Indianapolis, IN). The probes and primers used to assess mRNA targets were obtained as assay sets (TaqMan Gene Expression Assays, Applied Biosystems, Foster City, CA) and employed using the following parameters: 10 min at 95°C, followed by 40 cycles of amplification for 15 s at 95°C, and 1 min at 60°C. Expression of 18S rRNA was similarly assessed and used for normalization.
Measurement of Renal Ferritin Content
As in our previous studies, ferritin content was measured in renal lysates using an ELISA (catalog no. E-90F, ICL, Portland, OR) (23). Renal ferritin content was standardized per milligram protein, the latter assessed by the Lowry method.
Western Analysis
Western blot analysis was performed as described previously (19, 24), employing rabbit polyclonal antibodies for hemopexin (catalog no. AB90947, Abcam, Cambridge, MA) and β-actin (catalog no. 612656, BD Transduction Laboratories, San Diego, CA) as primary antibodies.
Data are expressed as means ± SE. The Student's t-test for parametric data and the Mann-Whitney test for nonparametric data were used for comparisons between groups. Results were considered significant for P < 0.05.
Effect of Age on Renal Function and Histology in Young and Old Mice
Old mice exhibit a distinct susceptibility to heme proteins as demonstrated by a markedly greater serum creatinine and blood urea nitrogen (BUN) in response to the same dose of hemoglobin (Fig. 1); remarkably, this dose of hemoglobin failed to elicit a significant increase in these functional indexes in young mice (Fig. 1). This impairment in renal function in old mice in response to hemoglobin was accompanied by renal histologic injury as evidenced by vacuolization, necrosis, and apoptosis of tubular epithelial cells, tubular dilatation, and, as emblematic of heme protein-induced renal injury, tubular cast formation (Fig. 2). In contrast, young mice, when administered hemoglobin, showed essentially no significant renal histologic injury at this time point (data not shown), thereby corroborating the lack of renal functional decline. Thus old mice, but not young mice, demonstrate increased susceptibility to AKI, as reflected by functional and histologic assessment.
Components of Heme Handling and Metabolism
Sensitivity to heme proteins is determined, at least in part, by the capacity of the kidney to effectively handle and metabolize heme proteins. Because old mice exhibited sensitivity to heme proteins, we hypothesized that expression of critical components of heme protein handling and metabolism would be impaired in the kidney in old mice treated with hemoglobin. We thus examined mechanisms underlying the clearance of heme proteins, undertaking in the process the most comprehensive characterization of this issue, to date, in the heme protein-exposed kidney.
Studies in old mice.
The administration of hemoglobin in old mice led to marked induction of HO-1 mRNA without a significant change in HO-2 mRNA expression (Fig. 3). Since heme degradation by HO liberates iron, we evaluated the expression of proteins that either sequester iron (ferritin) or export iron out of the cells (ferroportin). Increased expression of both ferritin protein and ferroportin mRNA occurred in the old mice (Fig. 4). We next assessed haptoglobin expression since recent studies have called attention to expression by the injured kidney of haptoglobin, a hemoglobin-binding protein that is produced by the liver. In old mice, we not only confirmed induction of haptoglobin gene expression in the hemoglobin-exposed kidney, but also demonstrated the renal induction of CD163, the receptor responsible for the intracellular incorporation of the haptoglobin-heme protein complex in monocytes/macrophages (Fig. 5). We then considered whether the hemopexin-CD91 system is also induced in the kidney following exposure to heme proteins (Fig. 6). Hemopexin is synthesized mainly by the liver; hemopexin binds heme, and the heme-hemopexin complex is then incorporated by the liver via the receptor CD91. We provide the novel demonstration that this hemopexin-CD91 system also resides in the kidney, and is induced following the exposure to heme proteins (Fig. 6). Finally, we examined the expression of the proteins that incorporate filtered heme proteins into the proximal tubule. In old mice, the administration of hemoglobin led to a downregulation of megalin and cubilin mRNA (Fig. 7), a change that would be expected to retard incorporation of heme proteins by the proximal tubules, but would deliver increased amounts of heme proteins to the distal tubules.
Studies in young mice.
The resistance of young mice to hemoglobin led us to hypothesize that young mice may effectively recruit renal mechanisms that process and metabolize heme proteins. To examine this hypothesis, the above studies were then performed in young mice treated with hemoglobin; these data are shown in Table 1. In response to the same dose of hemoglobin, young mice showed an induction of HO-2 as well as HO-1 mRNA in the kidney, along with induction of ferritin, ferroportin, and hemopexin, but did not demonstrate induction of haptoglobin or downregulation of megalin/cubilin at this time point.
Proinflammatory Gene Expression in Old and Young Mice
Sensitivity to renal injury is determined by the renal inflammatory response, and a number of proinflammatory mediators are incriminated in nephrotoxic and ischemic AKI, including TNF-α, monocyte chemoattractant protein-1 (MCP-1), and IL-6; expression of these cytokines was assessed in old and young mice following the administration of the same dose of hemoglobin (Fig. 8 and Table 2). Both TNF-α and MCP-1 were comparably induced in old (Fig. 8) and young mice (Table 2) in response to hemoglobin. However, quantitatively, a different response to hemoglobin was observed in old and young mice: in old animals, there was a dramatic upregulation in IL-6 mRNA expression, achieving some 130-fold increase (Fig. 8), whereas in the young animals, the fold increase was much less at 10-fold (Table 2).
Genes Involved in Chronic Kidney Disease in Old and Young Mice
An acute renal insult can lead to chronic kidney disease (CKD), and the risk for this transition is substantially increased by age. We thus examined whether the expression of genes critically involved in CKD are differentially induced after an acute administration of hemoglobin in old and young mice. Old and young animals showed comparable induction of TGF-β1, Smad3, collagen I, and collagen III mRNAs (Fig. 9 and Table 3). In addition to these interstitial collagens, hemoglobin induced collagen IV mRNA expression (3.1 ± 0.3 vs. 9.7 ± 0.7 standardized units, P < 0.05), an effect also observed in young mice (Table 3).
Effect of HO-2 on Renal Sensitivity to Heme Proteins
In view of the induction of HO-2 mRNA in young mice, we questioned whether HO-2 may contribute to the relative resistance of young mice to heme proteins. We thus administered hemoglobin to relatively young HO-2+/+ and HO-2−/− mice. As shown in Fig. 10, renal function, as measured by serum creatinine and BUN, was worse in HO-2−/− mice compared with HO-2+/+ mice on days 1 and 2 following the administration of hemoglobin. Renal histologic injury was more prominent in HO-2−/− mice, compared with HO-2+/+ mice, on day 2 after hemoglobin, as evidenced by necrosis of tubular epithelial cells, tubular cast formation, and tubular dilatation (Fig. 11). Thus, as assessed by functional and structural indexes, the deficiency of HO-2 rendered the kidney sensitive to the nephrotoxicity of hemoglobin.
To explore the basis for the observed finding, the present study focused on two mechanisms that determine heme protein-induced renal injury: renal handling and metabolism of heme proteins and AKI-related inflammatory processes. The premise for the former as a mechanism for age-dependent sensitivity resides in prior studies demonstrating that increased renal HO activity protects against heme protein-induced AKI and mortality, whereas inhibiting HO activity worsened AKI so induced (23). Subsequent studies using HO-1−/− mice demonstrated that the deficiency of HO-1 incurred sensitivity to AKI and mortality induced by heme proteins (25).
In the present studies, hemoglobin elicited comparable induction of HO-1 in the kidney in old and young mice. Blunted induction of HO-1 mRNA cannot thus be implicated as the basis for this increased sensitivity of the aged kidney to heme proteins. The aged kidney also exhibited other responses that, in tandem with induction of HO-1, may mitigate the cytotoxicity of heme proteins, including increased expression of ferritin (the major intracellular iron-binding protein) and ferroportin (the iron-transporter that exports iron extracellularly). The degradation of heme by HO releases iron in the heme protein-exposed kidney, and such induction of ferritin and ferroportin would be expected to safeguard iron homeostasis by promoting iron storage and the extracellular export of iron, respectively.
The aged, heme protein-exposed kidney also recruits other participants in hemoglobin/heme homeostasis, but ones conventionally considered to be centered principally in the liver. In this regard, as shown by Zager and collaborators, the kidney, when injured, is known to express albumin, α-fetoprotein, haptoglobin, and hemopexin (43, 45, 46). We now demonstrate that the aged kidney elicits not only haptoglobin (the major hemoglobin-binding protein in plasma) but also CD163, the receptor on monocytes/macrophages that incorporates the haptoglobin-hemoglobin complex. We also provide evidence that the aged, hemoglobin-exposed kidney recruits the hemopexin/CD91 system. Hemopexin resides in extracellular fluid where it binds free heme; the conventional view is that hemopexin is largely synthesized by the liver, and the hemopexin-heme complex is incorporated by liver cells and macrophages via the CD91 receptor. These findings thus support prior observations that the injured kidney induces haptoglobin and hemopexin, and extends these findings by demonstrating induction of the receptors that enable cellular incorporation of these complexes. We speculate that the intrarenal induction of haptoglobin and hemopexin, along with their relevant receptors, provides intrinsic renal mechanisms that bind hemoglobin and heme, respectively; incorporate these complexes within the kidney; and thereby promote their metabolic clearance.
The downregulation of megalin and cubilin in the kidney in old, but not young, mice in response to heme proteins may also underlie the observed sensitivity of the aged kidney. Such downregulation would lead to less incorporation of heme proteins by the proximal tubule and, accordingly, less clearance of heme by HO; additionally, larger amounts of the filtered heme proteins would be delivered downstream to the distal nephron where they interact with Tamm-Horsfall proteins, form tubular casts, and reduce glomerular filtration rate (GFR). Indeed, tubular casts, a cardinal histologic feature in heme protein-induced renal injury, were prominently observed in the kidney in aged mice, but not in young mice, after hemoglobin administration.
The kidney in aged mice did not exhibit induction of HO-2 mRNA following exposure to heme proteins, whereas a modest HO-2 mRNA induction was observed in young mice. We thus questioned whether this lack of induction in the aged kidney may contribute to its sensitivity, and proceeded to examine whether the HO-2 isoform influences the sensitivity of the kidney to heme proteins. In young HO-2−/− mice, sensitivity to hemoglobin was increased, as reflected by filtration markers on days 1 and 2 after the administration of hemoglobin and by greater histologic injury on day 2. These findings are the first to demonstrate the functional significance of HO-2 in AKI, in general, and in heme protein-induced AKI, in particular. Studies of HO-2 protein expression and HO activity would be of interest in the kidney in young and old mice following the administration of hemoglobin, as would the assessment of the functional significance of HO-2 in other models of AKI.
The observed cytoprotective effects of HO-2 in the kidney against hemoglobin add to the literature addressing the role for HO-2 as a cytoprotectant. HO-2 deficiency, for example, leads to an activated phenotype in aortic endothelial cells (7), and promotes glutamate-induced or TNF-α-induced apoptosis in cerebral endothelial cells (5, 27); increased amounts of HO-2 protein protects endothelial cells against hypoxia (17); HO-2 deficiency adversely affects inflammatory and reparative responses following corneal wounding (16, 34); finally, genetic deficiency of HO-2 exacerbates diabetes-induced renal injury in the streptozotocin murine model (15). However, prior studies demonstrate that cortical neuronal cultures and striatal cells from HO-2−/− mice are relatively resistant to the cytotoxic effect of hemoglobin (31, 32), while intracerebral injection of blood induces less cytotoxicity and oxidative stress in HO-2−/− mice (30); in contrast, genetic deficiency of HO-2 increases the sensitivity of astrocytes to hemin (9). The effect of HO-2 on the sensitivity to injury is thus determined by the nature of the insult and the afflicted organ or tissue.
Our examination of the observed sensitivity of the aged kidney to hemoglobin also included examination of selected proinflammatory cytokines incriminated in AKI; the cytokines selected were those that are known to be heme-inducible, or upregulated when heme-degrading activity is impaired. While TNF-α and MCP-1 were comparably induced in the kidney in old and young mice, the aged kidney exhibited a fulminant induction of IL-6 mRNA (130-fold vs. 10-fold in young mice). This markedly greater increase in renal IL-6 expression in old mice is relevant because of the following. First, following acute ischemia, IL-6 contributes substantially to AKI (20), while in human AKI, plasma IL-6 predicts the severity of AKI, morbidity, and mortality (37). Second, IL-6 contributes to CKD in rodent models (47); heightened IL-6 after AKI may thus predispose to the evolution of CKD. Third, plasma IL-6 predicts human, age-dependent frailty, CKD, and mortality (36, 38). We thus suggest that the increased sensitivity of the aged kidney to hemoglobin may reflect, at least in part, exaggerated renal induction of IL-6 in response to heme proteins.
Our data are the first to demonstrate that, in the acute phase following the exposure to heme proteins, the kidney exhibits substantial induction of genes critically involved in CKD (TGF-β1, Smad3, and collagen I, III, and IV); such induction similarly occurred in old and young mice in response to hemoglobin. In view of the relatively severe AKI in old but not young mice, it is not inconceivable that such induction of these fibrogenic genes after a single dose of hemoglobin is sustained in old mice, whereas in young mice, such induction is transient and self-remitting. Such an expression profile in old and young mice would render the former more likely to develop CKD after a single injection of hemoglobin. Studies that examine this issue are beyond the scope of the present investigation.
In summary, to the best of our knowledge, the present study provides the first demonstration that age increases the sensitivity of the kidney to the toxic effects of hemoglobin, and that HO-2 confers a protective response in AKI; our studies also provide the most comprehensive analysis, to date, of the expression of genes/proteins relevant to the handling of hemoglobin by the mammalian kidney. We speculate that this age-dependent sensitivity of the murine kidney to hemoglobin is clinically relevant for the following reasons. First, human AKI is commonly multifactorial, involving both ischemic and nephrotoxic insults. By identifying age as a determinant for nephrotoxic insults such as hemoglobin, the present studies suggest that the age-dependent risk for human AKI may reflect increased sensitivity not just to ischemic episodes, but also to nephrotoxic insults; additionally, our studies raise the possibility that, in the setting of hemolysis or rhabdomyolysis, the aged kidney may be especially prone to AKI. Second, ischemic insults to the kidney increase kidney content of free heme, a recognized toxicant; we speculate that the sensitizing effect of age on ischemic AKI may reflect, in part, an age-dependent heightening of the nephrotoxicity of heme. Third, hemoglobin-induced tubular injury is implicated as a mechanism underlying kidney dysfunction in glomerulonephritis characterized by microscopic or macroscopic hematuria (21, 22); notably, such tubular injury is known to occur more commonly in older patients (14, 21). Finally, hemolysis and the attendant exposure of the kidney to sickle hemoglobin contribute to the evolution of sickle cell nephropathy (26), a complication of sickle cell disease that occurs with increasing frequency with aging (4).
These studies were supported by National Institutes of Health Grants HL-55552 and DK-47060.
G. M. Vercellotti and J. D. Belcher obtain research funding from Sangart, Inc.
We gratefully acknowledge the secretarial expertise of Tammy Engel and the technical expertise of Allan Ackerman.
- Copyright © 2013 the American Physiological Society


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The Taksonz
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Room Description
Deluxe Single
Room Amenities
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- Breakfast
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About The Taksonz
Centrally located, this hotel is opposite to the Ludhiana Railway Station and boasts of an impressive decor with a mezzanine floor and a grand lobby. There are deluxe rooms, categorized into single and double occupancy rooms. Guests can savour on a scrumptious multi-cuisine fare at the in-house restaurant. A health club and billiard pool is available for the recreation of the guests. There is also a business center and a conference hall for official purposes, and banquet space for unofficial gatherings.
No of floors: 4 | No of rooms: 46
Checkin Time: 1200 pm
Checkout Time: 1000 pm


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For decades U.S. cities have lost residents to the outlying suburbs. But according to Census data out today the trend is reversing. And the economy may have something to do with it. From the Marketplace Sustainability Desk, here's Sam Eaton.
SAM EATON: The numbers speak for themselves. Last year Los Angeles posted its largest increase in residents since 2002. New York, the second biggest this decade. Chicago grew by nearly a percent after five years of declines. Some say it's a reflection of how deeply the economy has limited people's ability to move.
But Carol Coletta with the urban leader network CEOs for Cities says the shift is more profound. She says cities have become more family friendly in recent years. And in a down economy Coletta says urban living can have economic benefits as well.
CAROL COLETTA: No one's quite certain they're going to have a job next week and being in a city generally delivers more access to more opportunity.
Smaller cities like Minneapolis and Columbus, Ohio, also posted population gains. On the flip side, cities like Las Vegas that depended on housing construction for their growth saw steep declines. And suburbs near major cities posted their lowest growth in the past decade.
In Los Angeles, I'm Sam Eaton for Marketplace.


text: Human Rights Observatory China Pork Prices Sizzle As Frozen Reserves Fail to Cool Market Pork prices in China are climbing at a blistering pace despite government moves to cool markets in the world's top consumer of the meat by selling chunks of its frozen reserves. Government data released on Wednesday showed Chinese pork prices hit record levels this week, driven up as farmers hold back pigs from slaughter to rebuild herds following widespread culling in 2014 when prices were low. Supply has also been curbed by environmental measures introduced in 2010 that forced...Read complete article© Voice of America - Wednesday, June 1st 2016


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a Momentary Vignette
[ v1.07 | 12.06.1999 ]
[ prologue ]
welcome to a momentary vignette!
A Momentary Vignette wishes everyone the happiest of New Years.
[ a closer look ]
DesignMuseum: A fabulous Flash feast, London's DesignMuseum offers a stunning site featuring an eclectic mix of objects designed for our daily lives... from stylish automobiles, office equipment, and furniture... to humble household products and accessories.
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now at a momentary vignette...
Share a momentary vignette of architecture, design, and art with an electronic postcard. Select cards featuring the work of Frank Lloyd Wright, Eliel Saarinen, and Bernard Maybeck... with more selections coming soon!
[ worth reading ]
The Griffin and Sabine Trilogy... this cleverly presented trilogy makes a wonderful gift for anyone who enjoys great design. The Griffin and Sabine boxed set is indeed a work of art... complete with alluring collages and other surprises to weave this engaging tale of intrigue and mystery.
[ fading vignettes ]
a collection of the most recent vignettes...
[ Andrea Palladio ] http://www.loggia.com/vignette/991130.html
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[ Thermos ] http://www.loggia.com/vignette/991119.html
[ Claude Monet ] http://www.loggia.com/vignette/991114.html
[ Auguste Rodin ] http://www.loggia.com/vignette/991112.html
[ Temple of Venus ] http://www.loggia.com/vignette/991108.html
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please visit one of these informative sites...
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specializes in quality sites exploring the arts and humanities... be sure to visit the new Loggia Forums!
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newly redesigned... this ProjectCool featured site explores greek, roman, and celtic mythology and art!
[ the last word ]
to fly or not to fly... there is more here than meets the eye.
a 'fly loft' is the tall space above a stage in a theater used for the hoisting and storing of scenery backdrops during a play, opera, or similar production.
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2011 Nissan Leaf prototypeEnlarge Photo
2011 Nissan Leaf prototypeEnlarge Photo
Nissan LEAFEnlarge Photo
Easy to drive…in a parking lot
As with some of our colleagues who got a chance to drive the mule a couple of weeks ago in Los Angeles (at Dodger Stadium—notice a trend?), our actual time behind the wheel was extremely limited—just a couple of minutes—with the total course length just over a quarter of a mile. But we got the chance to hustle the front-drive LEAF prototype through a tight coned-off S-curve at 20 to 25 miles per hour and found that the steering was responsive but not at all like that of a sports car. Slowing down for a tighter corner, we noticed that Nissan hasn't dialed up the regenerative braking as aggressively as it could have; the LEAF—again, at least in prototype form—carries its momentum, at least at low speeds, like an automatic-transmission vehicle. Throttle and brake response felt spot-on in the test mule, and the brake pedal had a good feel but wasn't grabby. The Mini E, for instance, has a more aggressive regen calibration that takes a little getting used to. You could step from a gasoline vehicle right into this and still be smooth, so that's probably the goal.
Of course, a low-speed course like the one we drove shows off the better points of an all-electric powertrain like the LEAF's. Although we observed plenty of torque from 5 to 30 mph in momentary wide-open acceleration, EVs typically aren't quite as perky at Interstate speeds and we can't yet say how it might fare as a freeway commuter. We only hit 35 mph briefly, but top speed for the LEAF will be 90 mph, with 0-60 times in the ten-second range, likely.
The vast underfloor-mounted battery pack, developed by a joint venture with partner NEC, has a relatively small 24 kWh capacity (made up of 48 separate modules about the size of a hefty hardcover book)—and thus weighs around 400 pounds—but Nissan says that should be enough to drive 100 miles on a full charge. Just under a small cover at the snout of the car are two charging interfaces—one for home charging, the other for a quick-charging system. Although their was some conflicting information at the event as to how close the battery pack was to final form, Nissan was clearly not getting anything close to that range in this low-speed course. Now would be the time to tell you that we drove the LEAF in record low temperatures for Seattle—a crisp and sunny 30 degrees. To anyone who's familiar with car batteries weakened by cold, the same thing happens to some degree even on modern lithium-ion packs.
Although our drive was too short to make any grand statements or pronounce the LEAF a winner, Nissan is poised to be the first with a mass-market EV, and that alone is creating quite a buzz among green-car fans.
Nissan wouldn't let us actually sit in the cosmetically final LEAF; we did lean inside and take note of the instrument panel, which contained a Honda-like two-tier gauge display, plus a clean center-console design with a nav/infotainment screen, plus buttons for preheating and precooling, useful features that will allow users to bring the LEAF to temperature while it's still plugged into a charging station, thus using less battery power. Although the coarse, velvety upholstery in the LEAF is a little plain-looking, it's made of recycled plastic bottles and home appliances.


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Here's what you need to know:
1. Dr. McBryde was pronounced dead at the scene after an accident early Tuesday morning, in which his car left the road and hit several trees. The accident is still under investigation.
2. Dr. McBryde was interim chair of the University of South Alabama's department of orthopedic surgery, located in Mobile. He has held this position since June 2015.
3. Prior to rejoining the university — he also led the department of orthopedic surgery from 1991 to 1996 — Dr. McBryde was a professor at the University of South Carolina in Columbia.
4. Dr. McBryde had practiced at Andrews Sports Medicine and Orthopaedic Center in Birmingham, Ala., and had served as the director of the American Sports Medicine Institute's ankle and foot fellowship, also in Birmingham.
More articles on orthopedics:
Global market for regenerative medicine to reach $48.9B by 2021: 4 notes
Today's young trauma surgeons may be performing fewer complex procedures: 5 study insights
Drs. David Halsey, Mark Greenberg & more: 10 orthopedic surgeons winning patient care awards


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Packers hope Nelson's hamstring injury only 'day-to-day' issue
GREEN BAY, Wis. — When Jordy Nelson wasn't in uniform for the Green Bay Packers' preseason opener Saturday in Tennessee, it was not thought to be for any reason other than just being a healthy scratch. The team's website noted as such before the game, including Nelson along with Aaron Rodgers and Eddie Lacy as players who were given the star treatment of a day off.
As Nelson walked out to Monday's practice with no helmet or pads, it was obvious the recently re-signed wide receiver had a different reason for his Saturday absence. That reason, according to head coach Mike McCarthy, was a hamstring injury.
"I think it's day to day," McCarthy said.
Nelson wasn't walking with any noticeable limp, but one of the team's most media-friendly players didn't stick around in the locker room to discuss it with reporters.
Nelson missed four full games — and parts of two others — during the 2012 season due to a hamstring injury. Last season, Nelson was healthy and played in every game.
On the morning of the first training camp practice this year, Nelson signed a four-year, $39 million contract extension with the Packers.
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Search Results
Search scope: everything
Categories: Darkness & Doom
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|Pages:1 2 3
|Click picture for versions and download
|Intelligent Piano theme for serious, thought-provoking drama.
|Solemn, pensive intro to massive, portentous orchestral chord cycle - lush, haunting and epic.
|Intensely meditative and poignant, delicately filmic piano over a light orchestral bed.
|Brisk, exciting orchestral Minimalism moving in ever-shifting cycles - Viagra for the mind.
|Urgent orchestral Minimalism with a dreamlike, melodramatic edge.
|Mysteriously portentous orchestral Minimalism with sombre, spiralling cello lines.
|Ultra-minimal delicate atmosphere; creepy, spacey, futuristic sci-fi.
|Haunting minimalist atmosphere/drone, with listless violin and distant bells.
|Creepy, listless piano solo over haunting effected atmospherics.
|Bleak, mournful Celtic flavoured lament for cello with ghostly effected background bed.
|Pages:1 2 3


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Red Hat Bugzilla – Bug 174805
html-nochunks path in Makefile.common does not copy HTMLCSS into current directory
Last modified: 2013-09-02 02:07:54 EDT
The new HTMLCSS and HTMLCSSEXTRA support is great! I did notice one issue with
where the css is copied to in the html-nochunk flow. I have included a patch
below to highlight that area.
Created attachment 121750 [details]
Fixed in CVS
Ticket moved to allow products to be removed from BZ.


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CK3491: Accounts Technician
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Not the right opportunity for you? Don't give up just yet! Not all of our clients opportunities go to the public domain - Please get in touch via our About Us page, Trial Balance may still be able to help.
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Accounts Technician
Full Time
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An exciting opportunity has arisen for an experienced Accounts Technician to join our client, a leading accountancy practice.
Working with a diverse portfolio of sole traders, partnerships and limited companies, this role will see you participate in the following duties:
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This is an interesting role which would suit a candidate with who is AAT qualified, or about to complete AAT studies. You will have very recent experience in accountancy practice, carrying out statutory accounts and tax preparation. You will be up to speed with excel and computerised accounting software.
In exchange, my client offers a professional and sociable working environment, with excellent opportunities for career advancement. CPD and study support can be offered where appropriate.
If you are interested in applying for this role please forward your CV to Carly Kellow as soon as possible, quoting ref: CK3491
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text:
2006 Mercedes-Benz E320 CDI Questions
Get answers to your auto repair and car questions. Ask a mechanic for help and get back on the road.
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Main battery died overnight, and vehicle was purchased used, without a valet key. Neither the engine key, nor the driver's switch will open the trunk, in order to access the main battery. How can I gain access to the trunk? Must I purchase a valet key? Will the valet key be based on the VIN?
dealer says there is a break in the fiber optic cable in the trunk that feeds the radio. Mouse ate it so the dealer says. Need video to show me how to replace it. Cable is pt# 211 540 23 32
first nothing was wrong but now it just......
When the engine is cold, the glow plug light on the dash will not come on when the key is turned to the "ON" position. If I try turning it further, to the "START" position, the engine will not turn over. The glow plug light must come on and then go out, in order for the ignition to crank the engine and start. The only way to start it then is to leave the key in the on position for about 2 to 3 minutes. I then can turn it off an on, until the glow plug light comes on. Once that lights up and goes out, the engine will start. Once the engine is warmed up, the engine can be shut off and started under the normal procedure. I replaced the glow plugs with the Berar 5v plugs recommended. This has been an intermittent problem that seems to be getting worse in the colder weather. Could the be a faulty glow plug, relay switch or is there something else that I should be looking at?
We are looking at a Mercedes Benz E320 CDI (2006 model), which has been in New York for past 6 years. We are concerned it maybe rusted in areas not visible to us.Do all cars in New York get rusted?
Thanks in advance
We are looking at a Mercedes Benz E320 CDI (2006 model), which has been in New York for past 6 years. We are concerned it maybe rusted in areas not visible to us.Do all cars in New York get rusted?
Thanks in advance
My car was in a rear end collision and the check engine light came on. The car has been fixed and the check engine light is still on. They told me that the filter needed replacing so they did that; $300 later and the check engine light is still on. Any suggestions?
The car had a rear end collision and the battery was replaced. Exhaust was messed up too. Now the control panel only comes on about half the time when I start the car; I'm told that this is the normal power conservation mode when the battery is dying, but that is new. Then they told me it was the ignition and they replaced that for $1300 and it did not fix the problem. I read here about control panels needed to be replaced. Could that have been caused by the accident and what would that cost?
The dashboard drawer next to the glove compartment does not close/retract
Display message is: "Drive to workshop without changing gear". However, I can't even select a gear so that I can drive it!
Where are after market batteries for this car sold?
I changed Trans fluid and now engine will not run above 2400 RPM . Car ran fine before fluid change . Now all dash lights come on , engine goes on and off rapidly in time with the dash lights
When I accelerate hard to about 35 mph and then abruptly let off on the accelerator, there is a hard clunk in the front left wheel area. It does it every time I try. If I accelerate hard but do not let off abruptly on the accelerator, later when I slightly brake there can be a clunk or two like I ran over a branch with my left tire.
How can I take out the little coin drawer on my E320 CDI
I have a trunk leak issue....specifically once it rains or after washing the car a large amount of water gets trapped between the layers of the truck deck. It is noticable two ways...first, you can hear it "shoshing" when it opens and closes. Secondly, the water tends to find its way out around the latch as well as the seams. The seams being along the edge where the two layers are attached. Where and how does this water enter the cavity of the trunk deck? I have a concern for rust developing over time.
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