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An excerpt from www.HouseOfNames.com archives copyright © 2000 - 2017
Wiss Early Origins
family seat from very ancient times. William Wishart had a grant of the mill from the Abbey of Cambus Kenneth in the year 1200, and was associated with lands at Conon and Tulloch in 1254. Another William Wishart was a monk at St. Andrews in 1250. The family also held the lands of Kenny Murchardyn in 1279. Sir John Wishart rendered homage in 1296 to King Edward I of England on his brief conquest of Scotland. "Andrew Wycchard (Wyschard or Wychard) of Scotland, a prisoner of war in Hereford Castle, 1305-07 . George Wischart was burned for heresy at St. Andrews, 1545/6, and a later George Wischart was first bishop of Edinburgh after the Restoration and private chaplain and biographer of the marquess of Montrose." [1]CITATION[CLOSE]
Black, George F., The Surnames of Scotland Their Origin, Meaning and History. New York: New York Public Library, 1946. Print. (ISBN 0-87104-172-3) "The family are of ancient date in Forfarshire, and are descended from Robert, a natural son of David, Earl of Huntingdon, who having gone on a crusade to the Holy Land, was called Guishart, on account of the slaughter he made of the Saracens. Adam Wishart of Logie, was living in 1272." [2]CITATION[CLOSE]
Sims, Clifford Stanley The Origin and Signification of Scottish Surnames. 1862. Print.
Wiss Spelling Variations
Wiss Spelling Variations
Surnames that evolved in Scotland in the Middle Ages often appear under many spelling variations. These are due to the practice of spelling according to sound in the era before dictionaries had standardized the English language. Wiss has appeared as Wishart, Wishard, Wychart, Wisehart, Wisheart, Wiseheart and many more.
Wiss Early History
Wiss Early History
This web page shows only a small excerpt of our Wiss research. Another 129 words (9 lines of text) covering the years 1592, 1596, 1597, 1593, 1596, 1649, 1513, 1546, 1538 and 1546 are included under the topic Early Wiss History in all our PDF Extended History products and printed products wherever possible.
Wiss Early Notables (pre 1700)
Wiss Early Notables (pre 1700)
Notable amongst the family at this time was George Wishart (c.1513-1546) famous reformer and martyr. In 1538, while a schoolmaster at a grammar school in Montrose, he incurred a charge of heresy for teaching the Greek New Testament. After spending some time on...
Another 43 words (3 lines of text) are included under the topic Early Wiss Notables in all our PDF Extended History products and printed products wherever possible.
Wiss In Ireland
Wiss In Ireland
Some of the Wiss family moved to Ireland, but this topic is not covered in this excerpt. More information about their life in Ireland is included in all our PDF Extended History products and printed products wherever possible.
The Great Migration
The Great Migration
The North American colonies beckoned, with their ample land and opportunity as their freedom from the persecution suffered by so many Clan families back home. Many Scots even fought against England in the American War of Independence to gain this freedom. Recently, clan societies have allowed the ancestors of these brave Scottish settlers to rediscover their familial roots. Among them:
Wiss Settlers in United States in the 18th Century
Wiss Settlers in United States in the 19th Century
Contemporary Notables of the name Wiss (post 1700)
Contemporary Notables of the name Wiss (post 1700)
Wiss Family Crest Products
Wiss Family Crest Products
See Also
See Also
Other References
The Wiss Family Crest was acquired from the Houseofnames.com archives. The Wiss Family Crest was drawn according to heraldic standards based on published blazons. We generally include the oldest published family crest once associated with each surname.
This page was last modified on 10 August 2016 at 08:19.
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3-60 Saratoga Ln, Geneva WI 53147 $17,700
Features of 3-60 Saratoga Ln Geneva WI 53147
- Lots for Sale
- Status: Active
- Est. Acreage: 0.4
- School District: Lake Geneva J1
- County: Walworth
- Property Taxes: $540
- Property Tax Year: 2016
- Postal Municipality: Lake Geneva
- Listing Company: Keefe Real Estate, Inc.
Property Description for 3-60 Saratoga Ln Geneva WI 53147
3-60 Saratoga Ln Geneva WI 53147 - Home site in the Barclay Club of Geneva National, sloped for walkout lower level. Owned by a builder and can be hired to build your dream home.
Land Features
- Topography: Association, Sloped
- Zoning: Residential - Single
- Waterfront/Access: N
|MLS Number
|New Status
|Previous Status
|Activity Date
|New List Price
|Previous List Price
|Sold Price
|Oct 13 2017 11:07AM
|May 6 2016 11:21AM
Estimated Monthly Payment
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Receive more detailed information on this propertyAsk a Question
Community Homes Near 3-60 Saratoga Ln
Buyer understands that the information which is contained on Data pages is obtained from a number of different sources and which has not been independently verified or confirmed by the various real estate brokers and agents who have been and are involved in this transaction. If any particular measurement or data element is important or material to buyer, Buyer assumes all responsibility and liability to research, verify and confirm said data element and measurement. REALTOR *MLS* Equal Housing Opportunity

subset: commoncrawl


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- Programs of Study
- Chinese
- French
- Why Study French?
- French Connections
- French Faculty
- Adjuncts & TAs
- Major & Minor
- Graduate Studies
- Courses
- Contact Info
- Resources
- Area Associations
- Lyon Program
- Poitiers Program
- Summer Study Abroad
- German
- Classics, Ancient Greek, and Latin
- Italian
- Latin American Studies
- Russian
- Spanish
Why Study French?
French in Today's World:
- Over 274 million people across the continents speak French.
- French is the world's second most frequently studied language.
- French is the second most widely used "working" language in the EU.
- Thousands of corporations have invested in France. In the U.S., French companies employ over 650,000 workers.
- French is a key language to connecting with other peoples, other cultures, other ways of seeing and thinking.
French is Part of the Global Heritage:
- French is a stepping-stone to other Romance languages such as Spanish, Italian and Portuguese. In English alone, thousands of words are derived from French.
- French is a global language linking the arts: from painting, sculpture, and architecture to music, opera, theatre, cuisine, and fashion.
- French is the language of world-renowned masterpieces of literature: Hugo's Les Misérables, L'Étranger by Camus, La Fontaine's Fables, Voltaire's Candide, Molière's Le Bourgeois gentilhomme, George Sand's Indiana, and many others.
- The foundations of modern science and politics were largely shaped by French thinkers.
- Missouri and the Mississippi Valley were settled by the French as early as the 17th Century. St. Louis was founded by a French businessman, Pierre Laclède.
Need more reasons to study French?
See what our students have to say in "French Connections" !


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Stay positive
I firmly believe a positive attitude helps. When I was diagnosed I was determined to remain positive. I decided I wasn't going to be ill during treatment and other than fatigue I haven't felt too bad. Don't be afraid of it, treat it as an illness to be recovered from rather than a battle you may lose. And don't give the illness any more time than you need to. Live your life as fully as you are able and enjoy yourself at every opportunity
Lorraine M


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When most people think of calling it quits on a career, they immediately think of a move to Florida.
But not everyone wants to spend their golden years in Florida; some of us want to stay close to friends and family and within the great state of Idaho.
But where exactly in Idaho? Well, there's only one place to go for the answer — data.
To that end, we have tried to identify the places in Idaho that are safe, affordable, and have plenty of things to keep you busy well into retirement.
What did we find after pouring through all the data? Let's just say we hope folks in Rupert don't mind us spreading the word. Here are our top ten places in the Gem State to retire:
Why did Rupert take the top spot? And where are Nampa and Meridian? Read on to see see all the golden parachute level details.
How We Determined The Best Places To Retire In the Gem State
To create our list of the best places in Idaho to retire, we first used Census data to find all places in the Gem State with a population of at least 5,000 that aren't townships.
This left us with 22 places from across the state.
For these 22, we looked at the following criteria taken from the Census, the FBI's Crime Report, National Weather Service, and OpenFlights:
- Low cost of living
- Low crime
- Things to do (As measured by population density)
- Nice weather
- Distance to the closest international airport
We then ranked each of these places for each criteria from one to 22, with the lowest number being the best.
Finally, we took the average rank across these criteria. The place, in this case Rupert, with the lowest average rank was crowned the best of the best, a place for you to start your second careers.
1. Rupert
Population: 5,583Cost Of Living: 82
Distance to Closest Airport: 153 miles
Crime Per 100k: 2,901
Rupert ranked 1 overall thanks to its 1 place rank for cost of living and 13 place safety rank.
2. Jerome
Population: 11,046Cost Of Living: 83
Distance to Closest Airport: 186 miles
Crime Per 100k: 2,417
Jerome ranked 2 overall thanks to its 2 place rank for cost of living and 9 place safety rank.
3. Rexburg
Population: 26,301Cost Of Living: 102
Distance to Closest Airport: 209 miles
Crime Per 100k: 866
Rexburg ranked 3 overall thanks to its 18 place rank for cost of living and 1 place safety rank.
4. Emmett
Population: 6,546Cost Of Living: 91
Distance to Closest Airport: 240 miles
Crime Per 100k: 1,344
Emmett ranked 4 overall thanks to its 9 place rank for cost of living and 4 place safety rank.
5. Blackfoot
Population: 11,891Cost Of Living: 87
Distance to Closest Airport: 167 miles
Crime Per 100k: 2,834
Blackfoot ranked 5 overall thanks to its 5 place rank for cost of living and 12 place safety rank.
6. Idaho Falls
Population: 57,935Cost Of Living: 92
Distance to Closest Airport: 186 miles
Crime Per 100k: 2,810
Idaho Falls ranked 6 overall thanks to its 11 place rank for cost of living and 11 place safety rank.
7. Weiser
Population: 5,407Cost Of Living: 84
Distance to Closest Airport: 230 miles
Crime Per 100k: 1,886
Weiser ranked 7 overall thanks to its 3 place rank for cost of living and 6 place safety rank.
8. Chubbuck
Population: 14,117Cost Of Living: 96
Distance to Closest Airport: 149 miles
Crime Per 100k: 4,972
Chubbuck ranked 8 overall thanks to its 13 place rank for cost of living and 22 place safety rank.
9. Mountain Home
Population: 13,871Cost Of Living: 91
Distance to Closest Airport: 250 miles
Crime Per 100k: 2,126
Mountain Home ranked 9 overall thanks to its 9 place rank for cost of living and 8 place safety rank.
10. Twin Falls
Population: 45,362Cost Of Living: 91
Distance to Closest Airport: 177 miles
Crime Per 100k: 3,467
Twin Falls ranked 10 overall thanks to its 9 place rank for cost of living and 18 place safety rank.
Maybe You're Not Ready To Retire Yet…
So there you have it, the best place to retire in Idaho goes to Rupert.
If you're not ready to hang up your office apparel yet, then these places might be up your alley:
- Lewiston
- Sandpoint
- Post Falls
For more Idaho reading , check out:
- A Hilarious Look At The 10 Most Redneck Cities In Idaho
- These Are The 10 Most Dangerous Cities In Idaho
- These Are The 10 Most Redneck Cities In Idaho
Detailed List Of Best Places To Retire In Idaho
|Idaho Falls
|Mountain Home
|Twin Falls
|Coeur D'Alene
|Post Falls


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The Community Forensic Service Croydon provides community-based assessment, treatment and care for people, aged 18-65, who have severe mental health problems and who may be a risk to themselves and others. They may also be subject to Home Office restrictions, Probation Orders or have judicial proceedings outstanding.
SLaM provides care for people who live in the London Borough of Croydon.
A Forensic Community Team provides care for people with offending histories who, with appropriate care and treatment, pose a low risk to the public. The teams of health and social care professionals work with the police, public protection unit and the local Multi-Agency Public Protection Arrangement (MAPPA) team to manage people in the community.
|Contact: Queens Forensic Community Outreach
Queens Resource Centre
Forensic Community Outreach
66A Queens Road
Tel: 020 3228 5826
Email: firstname.lastname@example.com
Website: www.slam.nhs.uk/


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Available ebook formats: epub mobi pdf rtf lrf pdb txt html
James Hutchings lives in Melbourne, Australia. His work has appeared in Daily Science Fiction, Enchanted Conversation and fiction365 among other markets.
Review by:
ailyn koay
on June 24, 2014 :
(no rating)
I liked the title, and I wanted to know why they left the seas, so I got the book
I did giggle a little, and frown a little as I make my way to the end of the book, there are long poems, cat poems, really short poems. They are enjoyable and it was fun reading them. The whole thing is simple, although it is still a poem, which means that there might be hidden messages.
I am just not smart enough to figure it out, I just liked reading it.
Recommended for: poem beginners or people looking for inspiration. I think this is a cool edition
(reviewed 7 days after purchase)


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30 Day Yoga Challenge
A friend of mine has challenged me to practicing yoga everyday for the next 30 days. Wonderful! I love it! Sometimes I need a little challenge in my life to add a little Umph. For those who'd like similar inspiration, her lovely blog is right here: 30 Days of Yoga… I love yoga. That's no… Continue reading 30 Day Yoga Challenge


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Bigactive have said I'm an outside artist which i love! Here's a definition :
The term outsider art was coined by art critic Roger Cardinal in 1972 as an English synonym for art brut (French: [aʁ bʁyt], "raw art" or "rough art"), a label created by French artist Jean Dubuffet to describe art created outside the boundaries of official culture; Dubuffet focused particularly on art by insane-asylum inmates.[1]
While Dubuffet's term is quite specific, the English term "outsider art" is often applied more broadly, to include certain self-taught or Naïve art makers who were never institutionalized. Typically, those labeled as outsider artists have little or no contact with the mainstream art world or art institutions. In many cases, their work is discovered only after their deaths. Often, outsider art illustrates extreme mental states, unconventional ideas, or elaborate fantasy worlds.
The top image is something im working on with PRINCEY who is a fellow baron. This is for the work we are producing entitled: FEAR OF EVERYTHING. The second image is something I did for FOALS. It was a Metropolis Session, where they played and recorded straight to Vinyl. I produced work in the same building on the same day. There was an audience for some lucky chaps. At the end of the day a record was finished, complete with artwork. Heres a vid of the session. A small bit of me working it, in it, innit x
Thursday, 11 November 2010
Wednesday, 10 November 2010
Her Name Is Calla
Her Name Is Calla, I did some work in collaboration with Liz Johnson for 'her name is calla' my friend Weikie is in the band and he asked me to do one image and as he's been a friend since primary school. He is still today one of my best friends although he live is Leicester for some fucking reason. Anyway I thought this would be sick thing to do! Everyone should check out Hannah McCague's cover and inside cover art though. My work is featured on this label and on the inside booklet.
The Concept is its lots of diferent artists works all mashed together for the booklet. I didnt make the picture disc because im so badly organised.
At the moment I'm working on a Piece of FOALS art for a lucky competition winner.
The Concept is its lots of diferent artists works all mashed together for the booklet. I didnt make the picture disc because im so badly organised.
At the moment I'm working on a Piece of FOALS art for a lucky competition winner.
Posted by tinhead at 06:42 No comments:
Friday, 5 November 2010
So deep right now
Then i Went and Bought some black Pudding and some knock off shit.....
Posted by tinhead at 09:47 No comments:
Subscribe to: Posts (Atom)


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My dad planted a raspberry vine/bush thing, and so I am always picking raspberries and storing them. As a result, I decided to use a whole bunch of them at once by making a white chocolate raspberry cheesecake! It tasted sooo good!
I used the recipe at AllRecipes.com, found http://allrecipes.com/Recipe/White-Chocolate-Raspberry-Cheesecake/Detail.aspx.
I followed the recipe exactly except I used a store bought cake crust pan thing, and so I had some batter left over which I made mini-cheesecake (using vanilla wafers to make a mini crust).


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Narrating from her deathbed, Mathilda tells the story of her unnamed father's confession of incestuous love for her, followed by his suicide by drowning; ...
Discontinued App
Casual | Android
Books & Reference | iPhone, iPad
Narrating from her deathbed, Mathilda tells the story of her unnamed father's confession of incestuous love for her, followed by his suicide by drowning; her relationship with a gifted young poet called Woodville fails to reverse Matilda's emotional withdrawal or prevent her lonely death. When Mathilda is being courted by a young man, her father becomes violently jealous. He can't control his overwhelming passion -'My daughter, I love you' - and flees. From being her God, her father becomes Mathilda's nightmare: "infamy and guilt was mingled with my portion; unlawful and detestable passion has poured its poison into my ears and changed all my blood (in) a cold fountain of bitterness." Although sometimes too sentimental, Mathilda is a strong psychological portrait, brilliantly written by an intelligent and very well read author: 'more lovely than a sunbeam, slighter, quicker than the waving plumage of a bird, dazzling as lightning and like it giving day to night, yet mild and faint, that smile came.' Between 1819 and 1820, Mary Shelley wrote Mathilda, her second novel following the classic Frankenstein. The story is reminiscent of Shelley's own life, if not outright autobiographical, with characters resembling herself, her husband Percy Shelley, and her father William Godwin. Mathilda is an often overlooked literary gem written in the classic Romantic style with Matilda on her deathbed telling her tale full of loss, incest, and suicide. This audio book application contains audio files for all chapters in one download, so you can listen to the book on the go without the need for an internet connection. Hence the application size is a little big, and you may need to download it over WiFi connection or through iTunes on your PC or Mac. Audio Book Features ==> Pause/resume function Track progress slider Volume slider Easy navigation Audio continues to play when your device is locked. Offline viewing and listening without internet connection If you like this audio book, please check out other great audio books at www.awaudiobooks.com! *** On Sale Now! (Normal price $2.99) ***
Version: 1.0
Size: 30.4 MB
Price: 0,88 €
Developed by AppWarrior
Day of release: 2010-05-2
Recommended age: 12+


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Download Full Free Asphalt Xtreme v1.4.2b MOD Apk + OBB Data [All Unlocked/Cars Maxed] – Android Games by Gameloft
Asphalt Xtreme Description
You are about to download the Asphalt Xtreme 1.4.2b apk mod file for Android 4.4 and up From ApkOne.Net With Direct Link
Last studio Gameloft pleased us a new part Asphalt racing series back in 2013, then the developers have released the eighth part. Later was elaborate game called Asphalt Nitro, which received at that time a completely new engine and a number of new features.
Continuation of the same according to the developers will be called Asphalt Xtreme and it's not easy, because you will provide an opportunity porassekat on open areas off-road, while sitting behind the wheel of licensed vehicles such as Jeep, Ford, Mercedes-Benz, Predator, Dodge, Chevrolet and the whole several others.
Support for installation on an external SD card: no
Startup cache: none
Asphalt Xtreme is cranking it up a notch with its first brand-new update!
• 3 New Vehicles: The spectacular Peugeot Hoggar Concept (buggy), the brutal Hummer H1 (monster truck) and the amazing Ford Super Duty F-450 (pickup) will all make their big entrance over the next few weeks.
• 6 new Seasons and 3 new Vehicle Masteries.
• Seasonal events with exclusive boxes!
• New Limited-Time Events to fuel your adrenaline addiction.
• Various game performance improvements and bug fixes.
Direct Download Link
unlocked all events, anti-ban, without fake cars and money
Asphalt Xtreme: Offroad Racing v1.4.2b Apk / Mirror
Asphalt Xtreme: Offroad Racing v1.4.2b OBB Data / Mirror
Google Playstore Link
note: extract obb data folder "com.gameloft.android.ANMP.GloftMOHM" to "internal memory/android/obb/"


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Do you have excess growth of hair on your upper lip? Is waxing and threading leaving your skin red, sore and scaly? Then here is a herbal mask for upper lip hair that will get the job done without the pain, plus it will reduce hair growth overtime!
But, before we explore this DIY mask to remove upper lip hair, let us first understand what causes the facial hair.
Increase in the growth of hair on face can be due to spike in androgen count in the body. A male hormone that causes male-pattern hair growth, as well as, other male characteristics like deep voice.
Androgen spike can be triggered by several factors including PCOD, medication, a medical condition called hirsutism or hormonal imbalance.
Natural ingredients used in this mask to remove upper lip hair - gelatin and milk. Gelatin works as a peel off that removes unwanted hair and deeply-embedded impurities in the skin.
Plus it is packed with proteins, which heals and nourishes the skin. Milk, on the other hand, is used to dilute the gelatin. And it has lactic acid which lightens skin tone.
Now that you know precisely how this mask works, here is a step-by-step guide on how to remove upper lip hair naturally!
Step 1
Cleanse your face of the day's dirt and grime. In case you have makeup on, use a mild cleanser and rinse off every last bit of it. Using a clean towel, blot dry your skin. Tie your hair up, away from your face.
Step 2
Take a bowl, add a tablespoon of unflavoured gelatin powder to it. Add one and a half tablespoon of milk. Keep the bowl in the microwave for 20 seconds. Keep stirring it until the gelatin powder melts and you get a smooth paste.
Step 3
Add 3 drops of lemon juice to the mixture. Lemon has citric acid, which will help take off the unwanted hair, without making your upper lip dark. It also has vitamin C, which helps brighten skin.
Step 4
The gelatin mask to remove unwanted facial hair has very pungent odour. To mask the smell, add a few drops of lavender oil to the mixture. Don't add too much oil as it will reduce the effect of the herbal mask to remove upper lip hair.
Step 5
You have to keep stirring the mask, and use it immediately, otherwise it will thicken and become impossible to use. Using a popsickle stick apply a thin coat of the mask on your upper lip.
Step 6
The consistency of the mask has to be just right. If it is too thick, then it will not come off in one strip. If it is too thin, then it will crumble and break into pieces. The consistency should be in between.
Step 7
Let the mask sit for 5 to 10 minutes. Once the mask starts to tighten on the skin, peel it off in one quick motion. It will remove the unwanted hair right off, and give you super-smooth skin.
Step 8
Take your regular face cream in the palm of your hand and massage it onto the skin above your lips. It will soothe any redness. Make sure the cream you use is light and oil-free.


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Week 44 Cinnamon
View Single Post
October 31st, 2012, 03:27 PM
Happy Song
Join Date: Jan 2010
Posts: 11,023
OK I made this last night and didn't take a picture so I'll use a stock picture.
Apple Pork Loin in the crock pot
Pork Loin, 2.5-3 lbs.
Red Delicious Apples, 3 sliced
Honey, approx 4 T
Cinnamon, 2 T
Lay the apple slices (from two of the apples) in the bottom of the crockpot.
Sprinkle with cinnamon.
Cut slits in the pork loin (approx 1/2″-3/4″).
Drizzle some honey into the slits.
Then place apple slices into the slits.
Place the pork loin into the crockpot.
Drizzle the top with the remainder of the honey.
Place the rest of the apples on top.
Then sprinkle the whole thing with cinnamon.
Cook on low for 7 hours.( Mine was over cooked so I'd go a little less on this)
Happy Song
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Find all posts by Happy Song


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Contact Info / Websites
All 25 movie Reviews
3D Engine Box Demo
Rated 1 / 5 stars
The score is normal.
It's just some boxes falling, and you have to refresh the flash to see it again, woo hoo. Maybe if there was some form of functionality it would be good but saying "most people didn't read the coments so i ahve an almost failing scroe." is just ridiculous. People aren't going to shower you with fives just because you made some falling boxes.
ABunchofNESsedUpCrap 2
Rated 1 / 5 stars
Such a clone
It just seemed like a really badly rip off of Sprite TV and some other flashes, the jokes were really old and uninspiring and I almost instantly predicted what the punchline was every time. The only good thing was the animation, but then it was with sprites so that isn't hard to pull off. It's been done a thousand times before and a thousand times better.
Well, in my defense, they're all rip offs of Robot Chicken (including mine). Skit (or sketch, whatever) based comedy is a style. At least try to come up with a better reason for hating this. If you hate skit based comedy, than you are obviously looking in the wrong place for laughs...
Collab of Champions
Rated 0 / 5 stars
Since you just resubmitted it from last week it's an auto 0.
I don't think so.
The iPhone Shuffle
Rated 2 / 5 stars
Overall not very good.
Well the only well done part was the voice work, the animation was basic since there was no mouths. and as i'm sure many others have said that joke was extremely unoriginal.
Sonic Sagas Ep.2
Rated 0.5 / 5 stars
did sonic rip off DBZ or what?
Sonic was around before DragonBall Z dude...
Do you have a waffle
Rated 4.5 / 5 stars
Catchy stuff
Sometimes I can't bring myself to close your submissions, one thing to fix would it seems to lag a bit when the big red waffle text with the waffle background shows up, just seems abit slow. Good job otherwise.
Smb. prank. series intro
Rated 2 / 5 stars
Too short
First off, add buttons for the text. Second, put more into the background and third that was way too short, it wouldn't had been that tough to add in another minute or two.
ur right, but this is only the unfinished intro, i will work on it when i get more resources
MetroidPrimeCatastrophe 2
Rated 2.5 / 5 stars
That was
pretty hard to watch, the art was really good but my god the humor was terrible, I actually closed it because the jokes were so full of child like wonder, I'm sure the younger crowd will find this funny but wow dude, pick it up.
It's just that my humor isn't "for everyone" and more likely for audience with simple type of humor.
I don't really like doing more "mature" jokes for some reason, not to mention I sure hate cock jokes.
Thanks for your opinions anyway.
The Island---- Epi: 1
Rated 5 / 5 stars
Since I can't put both of my votes for that bitch kickle i'll only vote once, KICKLE KICKLE KICKLE!
Your vote has been counted.
Haha thanks for the review, although I can't make your wish come true and use two votes on kickle, I'm glad you enjoyed the movie, thanks for the perfect score :P.
Jerry The Magical Poop
Rated 4 / 5 stars
Just as the title said, this flash is magical poop.


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|Home | About | Journals | Submit | Contact Us | Français
Autoimmune diseases affect a significant segment of the population and are typically thought to be multifactorial in etiology. Autoimmune diseases due to single gene defects are rare, but offer an invaluable window into understanding how defects in the immune system can lead to autoimmunity. In this review, we will focus on autoimmune polyendocrinopathy syndrome type 1 and recent advances in our understanding of this disease. We will also discuss two other monogenic autoimmune diseases: immunodysregulation, polyendocrinopathy, and enteropathy, X-linked and Autoimmune lymphoproliferative syndrome. Importantly, the knowledge and principles gained from studying these diseases have been applicable to more common autoimmune diseases and have opened the door to better diagnostic and therapeutic modalities.
Autoimmune disease affects approximately 3% of Americans, with many of these diseases arising in childhood (1). Despite its prevalence, the complex, polygenic inheritance of most autoimmune disorders has been an obstacle in understanding the pathogenesis of these diseases. Although recent technical advances, including genome-wide association studies, have allowed for the identification of genetic factors contributing to autoimmunity, the individual impact of each of these genetic factors is low. For example, 8 of the 10 chromosomal regions associated with type I diabetes have an odds ratio of <2.0 (2). The low impact of these genetic factors has made them difficult to study. In contrast, studies of rare monogenic autoimmune diseases have more readily yielded mechanistic insights into autoimmune pathophysiology because of the obvious impact of mutations in these single genes in provoking disease.
An example of a rare monogenic autoimmune diseases is autoimmune polyendocrinopathy syndrome (APS) type I (OMIM 240300), also known as autoimmune polyendocrinopathy candidiasis-ectodermal dystrophy, which is due to a defect in autoimmune regulator (Aire). Studies of monogenic diseases, like APS type I, have played a critical role in informing us on how the immune system normally recognizes self from nonself and how breakdowns in this system can result in autoimmune disease. In this review, we will discuss recent advances in our understanding of the clinical features, genetics, pathophysiology, and diagnosis of this disease. In addition, we will also touch on two other monogenic autoimmune diseases, Immunodysregulation, polyendocrinopathy, and enteropathy, X-linked (IPEX) (OMIM 304790) and auto-immune lymphoproliferative syndrome (ALPS) (OMIM 601859) (Table 1), and discuss recent advances in the study of these syndromes.1
APS type I is a rare disease, but is more common in certain populations, including Iranian Jews (3), Finns (4), Sardinians (5), and Norwegians (6). Clinically, APS type I is characterized by three major features: adrenal insufficiency, hypoparathyroidism, and mucocutaneous candidiasis (7). A clinical diagnosis is based on the presence of two of these three features. APS type I often manifests with mucocutaneous candidiasis in infancy, followed by hypoparathyroidism and adrenal insufficiency in childhood (7–9). In addition, a number of other organ-specific autoimmune diseases, including ovarian failure, testicular failure, autoimmune thyroiditis, autoimmune diabetes, autoimmune gastritis, and vitiligo, develop in these patients at a lower incidence (8,10). The pace at which disease manifestations are seen is quite variable, requiring patients to be screened throughout their lifetime (7,8). For example, in one series in Norway, the first clinical manifestation of APS type I ranged from the first year of life to age 43 y (6).
Because APS type I is a monogenic disease, classical positional cloning techniques could be used to map the gene defect. In 1997, this defect was mapped to the Aire gene (11,12). Interestingly, the genotype–phenotype correlation is quite variable, with patients in the same family with the same Aire mutation manifesting different organ-specific autoimmune diseases (6). More than 58 mutations have been described in Aire (4). The two most common mutations are the R257X mutation and a 13 bp deletion in exon 8. The frequency of these mutations is population specific, with the R257X mutation seen in the majority of Finnish APS type I patients (12), and a 13 bp deletion in exon 8 (1094–1106del) seen in the majority of APS type I patients in Britain (13) and North America (14). Heterozygous carriers of these mutations in general have not been found to manifest autoimmunity (15).
Interestingly, a G228W point mutation in Aire has been described in an Italian family with an autoimmune syndrome that is inherited in an autosomal dominant manner (16). This autoimmune syndrome differs from the classic APS type I in that some family members have only autoimmune thyroiditis. A mouse model with a knockin of this G228W point mutation in Aire recapitulates the disease seen in this family (17). Thus, Aire-associated mutations can also result in an autoimmune syndrome that differs from APS type I in phenotype and inheritance pattern.
Most studies looking at common, isolated autoimmune endocrinopathies, however, have not identified Aire as a susceptibility gene. Polymorphisms in Aire did not seem to be associated with isolated adrenal insufficiency (18), type I diabetes (19), or vitiligo (20). Additionally, the two most common mutations in Aire were not seen in a large group of patients with isolated adrenal insufficiency, type I diabetes, or autoimmune thyroiditis (21). One small study suggested an association between an Aire intronic polymorphism and systemic sclerosis and thyroiditis (22). Other studies have suggested an association between Aire polymorphisms and alopecia areata (23) and vitiligo (24). Larger scale studies will have to be done to validate these findings because these associations could be due to chance alone.
Aire is predominantly expressed in the thymus, suggesting an important role in immune regulation (25). Within the thymus, Aire is expressed in a subset of stromal cells known as medullary thymic epithelial cells (mTECs). mTECs have the unusual property of displaying a variety of peripheral self-antigens, including known autoimmune targets such as insulin and thyroglobulin (26–28) (Fig. 1A). This expression of peripheral self-antigens had been proposed to be important in allowing self-reactive T cells that recognize these self-antigens to be deleted. In this process, termed negative selection, T cells that recognize self-antigen in the thymus would be prevented from being released into the body and thus prevented from causing autoimmune disease. It was, therefore, hypothesized that Aire might be playing an important role in driving the expression of these peripheral self-antigens in mTECs.
Indeed, Aire-deficient mice, developed as a model of human APS type I, have decreased expression of a number of organ-specific self antigens in mTECs (29). Additionally, these mice also have defective negative selection of T cells in the thymus (30,31). Like humans with homozygous null mutations in Aire, these mice develop tissue-specific autoimmune disease in multiple organs. The autoimmune disease is characterized both by a lymphocytic infiltrate in the targeted organ and the presence of serum autoantibodies reactive against the targeted organ.
These data are consistent with a model in which self-reactive T cells in the thymus are normally removed from the T-cell repertoire (Fig. 1A). Aire plays an important role in the normal thymus in that it increases the expression of thymic self-antigens, and the presentation of these antigens drive the negative selection of self-reactive T cells (32). Without Aire, a number of self-antigens are no longer transcribed in mTECs and self-reactive T cells can escape into the periphery to cause autoimmunity (Fig. 1B). These self-antigens have been shown to be enriched for organ-specific self-antigens (29), perhaps explaining why APS type I patients develop a number of organ-specific autoimmune diseases.
More recently, Aire has been proposed to play an additional role in guarding against autoimmunity in extrathymic immune sites (33,34). Studies in a mouse that expresses a fluorescent tag in Aire-expressing cells showed that a unique subset of cells in the lymph node and spleen also express Aire (33). Like mTECs, these cells, termed extrathymic Aire-expressing cells (eTACs), are able to delete autoreactive T cells in a transgenic system. Furthermore, these eTACs, like mTECs, express a number of organ-specific antigens in an Aire-dependent manner. The antigens expressed in eTACs were nonoverlapping with mTECs, suggesting that these two cell types may have nonredundant functions and protect against a different repertoire of self-reactive T cells.
The mechanism by which Aire functions to up-regulate thymic self-antigen expression in mTECs remains to be fully elucidated. The Aire protein contains a number of domains that suggest that it plays a role in transcription (32). In particular, Aire contains two PHD (plant homeodomain) domains, which have recently been shown in a number of proteins to bind trimethylated histone H3 (H3K4me3) (35,36). Unlike the PHD domains in these other proteins, PHD1 in Aire seems to bind histone H3 in the unmethylated state (37,38). Despite this difference, this finding nevertheless suggests a link between Aire and chromatin pattern recognition. Additionally, Aire has been shown to bind to three proteins with described roles in transcription: 1) positive transcription elongation factor b (pTEFb) (39), 2) DNA-dependent protein kinase (DNA-PK) (40), and 3) cAMP response element-binding protein (CBP) (41,42). How these proteins interact together in Aire-mediated transcription remains to be determined.
Much headway has recently been made in identifying the autoantigens associated with the clinical features seen in APS type I. Utilizing sera from APS type I patients with hypoparathryoidism, investigators recently identified NACHT (NAIP [neuronal apoptosis inhibitory protein], CIITA [MHC class II transcription activator], HET-E [incompatibility locus protein from Podospora anserina] and TP1 [telomerase-associated protein]) leucine-rich-repeat and pyrin protein 5 (NALP5) as a parathyroid autoantigen (43). Autoantibodies to NALP5 seem to be present specifically in APS type I patients with hypoparathyroidism, making the autoantibody response against NALP5 a potential diagnostic marker for parathyroid involvement in APS type I. Whether Aire-deficient mice also share serum reactivity against NALP5 and whether NALP5 is Aire regulated in the thymus remain to be addressed.
In Aire-deficient mice, a number of antigens important in preventing organ-specific autoimmunity have been identified (44–46). Identification of these antigens in mice have played an important role in understanding the pathophysiology of APS type I. Interphotoreceptor retinoid-binding protein, IRBP, for example, has been identified as the antigen important in preventing eye autoimmunity (44). Remarkably, loss of expression of this antigen in the thymus results in eye auto-immunity in mice, demonstrating that lack of a single thymic antigen is sufficient to result in disease.
Interestingly, the presence of autoantibodies against type I interferon (IFN-α and ω) has been independently reported by five groups (6,47–49). In all five studies, there was a striking correlation (nearly 100%) between the presence of antitype I interferon antibodies and the presence of Aire mutations. Notably, patients heterozygous for the dominant negative G228W point mutation also demonstrated type I interferon autoantibodies (49). These antibodies seem to appear early in the disease course, thus making this a useful diagnostic tool in identifying APS type I patients.
The actual contribution of anti-interferon autoantibodies to APS type I has been the subject of much speculation. Type I interferons are important in defense against pathogens and adaptive immunity (50). Given that many APS type I patients present with mucocutaneous candidiasis, the possible role of anti-interferon autoantibodies in causing an immune dysregulation that results in this infection has been proposed (51). In support of this proposal, monocytes and dendritic cells from APS type I patients have been shown to down-regulate a number of genes that are known to be driven by interferon (52). Furthermore, sera from APS type I patients seem to down-regulate these genes in monocytes in a cell culture system. However, this hypothesis does not explain why APS type I patients are not more susceptible to other fungal or viral infections. Thus, further study will be needed to elucidate the role of these autoantibodies in disease pathogenesis.
The importance of identifying thymic self-antigens extends beyond understanding the pathogenesis of APS type I. In addition to APS type I, more common autoimmune diseases have also been linked to decreases in thymic antigen expression. In type I diabetes, decreased levels of thymic insulin expression are linked to disease. Polymorphisms in the Variable Number of Tandem Repeats region upstream of the insulin promoter result in variation in the amount of insulin expression in the thymus. Population studies show that alleles that result in higher levels of insulin expression in the thymus is associated with protection against the development of type I diabetes (53,54). This protection is thought to be conferred by the promotion of negative selection of T cells reactive to the autoantigen insulin. Furthermore, in mice, the loss of thymic expression of proinsulin 2 also predisposes to autoimmune diabetes (55).
An analogous situation occurs with the α chain of the acetylcholine receptor (CHRNA1) and Myasthenia Gravis. The relative thymic expression level of this antigen varies relative to a single nucleotide polymorphism in the promoter region. Additionally, increased expression of this thymic antigen is correlated with later age of onset of disease (56,57). Interestingly, a correlation seemed to exist between level of Aire expression and CHRNA1 expression in purified human mTEC samples, suggesting that Aire may play a role in thymic expression of CHRNA1 in the thymus. In vitro studies also indicated that Aire is able to drive the expression of a CHRNA1 reporter construct.
IPEX is a very rare X-linked disorder that results in a severe autoimmunity syndrome that is lethal in infancy unless treated with immunosupression (58) and/or bone marrow transplantation (59). This severe autoimmunity manifests as a constellation of diarrhea, dermatitis, diabetes mellitus, thyroiditis, and hemolytic anemia (Table 1) (60,61). Importantly, an exaggerated response to viral infections is seen, further suggesting an over-activation of the immune system in general.
In 2001, patients with the IPEX syndrome were found to harbor mutations in the Foxp3 gene (62,63). These patients were noted to have mutations in the same gene as scurfy mice, a mouse strain with severe autoimmunity and lymphoproliferation (64). Studies in mice have shown that Foxp3 plays an important role in the development and function of regulatory T (Treg; [65,66]) cells. This regulatory T-cell population has been shown to dampen immune responses in a variety of settings, including autoimmune diseases.
More recently, the pathogenesis of IPEX has been further defined. First, selective ablation of Foxp3-expressing cells in mice results in severe autoimmunity and lymphoproliferation, demonstrating that a decrease in the number of these Foxp3-expressing cells is sufficient to cause severe autoimmune disease (67). Second, the suppressive function of Tregs seems to depend on the expression of cytotoxic T lymphocyte antigen (CTLA)-4 by Tregs (68), because selective loss of CTLA-4 in FoxP3-expressing cells results in a severe auto-immunity. Third, loss of expression of interleukin (IL)-10 by Foxp3-expressing cells results in inflammation in the gut and lung (69), suggesting that expression of specific cytokines by Tregs may have specific, nonredundant functions.
A number of studies have investigated whether Treg defects can also contribute to more common autoimmune diseases. Although the loss of Treg cells due to Foxp3 mutations have been well linked to the IPEX syndrome, subtle changes in the number and function of Tregs in other more common, polygenic autoimmune diseases are less clear. For example, the role of Tregs in the pathogenesis of type I diabetes mellitus has been controversial. One article reported a reduction in the percentage of Tregs in CD4+ lymphocytes in patients with type I diabetes (70), whereas several others have reported no differences in Treg frequency between these patients and controls (71–73). Thus, the generalizability of decreased Treg numbers as a common pathogenic mechanism in autoimmune disease remains to be determined.
A syndrome related to IPEX has been described in two patients with mutations in the IL-2 receptor alpha (CD25) gene (74,75). The Foxp3 gene was found to be wild type in both of these patients. In one patient, homozygous mutations in CD25 resulted in defective secretion of IL-10 by CD4+ T lymphocytes (75). Because IL-10 is important in the down-regulation of inflammation, this finding suggests a possible mechanism by which homozygous mutations in CD25 may phenocopy IPEX.
As its name suggests, ALPS is characterized by lymphadenopathy, splenomegaly, hypergammaglobulinemia, and autoimmune diseases (76). The autoimmune features often manifest as hemolytic anemia and thrombocytopenia, and can also include a number of additional autoimmune diseases such as hepatitis, uveitis, and vasculitis. Like APS type I and IPEX, the pathophysiology of ALPS was largely worked out utilizing a mouse model that phenotypically resembled patients with ALPS (76).
This mouse model, the MRL lpr/lpr mouse, played a key role in demonstrating that ALPS patients have a defect in the ability of their lymphocytes to undergo apoptosis. Patients with ALPS were noted to have an increase in double-negative T cells (developing T cells that have not yet expressed CD4 or CD8 on their cell surface) much like MRL lpr/lpr mice (77). Because MRL lpr/lpr mice have defective expression of Fas, a key mediator of apoptosis, ALPS patients were also suspected of having this defect (78,79). Indeed, subsets of patients were found to have heterozygous mutations in TNFRSF6, the gene encoding Fas (ALPS type Ia) (78) (Table 1).
Additionally, mutations in the Fas-mediated apoptosis pathway were also found (Table 1). A patient with Systemic Lupus Erythematosus and lymphoproliferation was found to have a mutation in the TNFSF6 gene encoding Fas ligand, the binding partner for Fas. This disease subtype was termed ALPS type Ib (80). Patients with ALPS type IIa have mutations in the gene encoding caspase 10, a protein downstream of Fas in the apoptotic pathway (81). ALPS type IIa patients seem to be distinguishable from the other subtypes in that they manifest a more severe lymphoproliferation and autoimmunity, and a resistance to apoptosis in both lymphocytes and dendritic cells is seen. Finally, patients with homozygous mutations in caspase 8, another downstream mediator of the apoptosis pathway, have been termed ALPS type IIb (82). Unexpectedly, however, these patients seem to have immunodeficiency rather than autoimmunity (83).
The mechanism by which defects in apoptosis results in autoimmunity still awaits clarification. The increased numbers of double negative T cells points to an aberrant contraction of the immune response. It has been postulated that lymphocytes that recognize self-antigens may be more dependent on Fas-mediated apoptosis than lymphocytes that recognize external antigen (82). This assertion, however, remains to be proven.
Despite the rarity of monogenic autoimmune diseases, these diseases have played an invaluable role in our understanding of the pathophysiology of autoimmune diseases in general. Mouse models of these diseases have been instrumental in unraveling the cellular and molecular pathways at play in these diseases. Out of these basic science advances may come more targeted diagnostic and therapeutic approaches to auto-immune diseases. An example of a promising antigen-targeted therapeutic is the utilization of Tregs as a treatment for type I diabetes. In a mouse model for type I diabetes, transfer of a small number of antigen-specific Tregs could reverse disease (84,85). Similarly, studies in Aire-deficient mice and APS type I patients have resulted in the identification of new antigens that may be useful in the diagnosis and prediction of autoimmune disease. Taken together, these basic science advances in the study of monogenic autoimmune diseases are making major contributions to our ability to diagnose and treat patients with autoimmunity.
PubMed Central Canada is a service of the Canadian Institutes of Health Research (CIHR) working in partnership with the National Research Council's national science library in cooperation with the National Center for Biotechnology Information at the U.S. National Library of Medicine(NCBI/NLM). It includes content provided to the PubMed Central International archive by participating publishers.


text:
This just isn't right.
What Italo did in this 60-minute session is hard for any of us to comprehend. The turns, the airs, the sheer ARGHHHHHHH in everything he did is simply baffling. All in an hour, at shitty Off the Wall.
In the background, you can hear plenty of people ooohing and ahhhing nearly every time Italo stood up. You may be thinking, "With everyone watching him tear it to pieces, why didn't anyone else paddle out and join in the fun?"
Let me ask you one question: Would you want to be compared to that guy during this session?
Didn't think so.


text:
Authorities say the driver of a car that struck a hotel shuttle van near Boston's airport and killed two visitors from Texas was drunk.
Steven Birenbaum, of Swampscott, was released on $5,000 bail after pleading not guilty at his arraignment Monday to several charges, including two counts each of manslaughter and motor vehicle homicide while driving drunk. Prosecutors said the 27-year-old was also under the influence of marijuana and prescription medications at the time of the accident.
Birenbaum was operating a Subaru Legacy at speeds of almost 80 mph, prosecutors said, when he struck the rear of a Hampton Inn airport shuttle on Route 90 east at about 1 a.m. on Aug. 31, causing both vehicles to collide with highway barriers. The van was heading for the hotel after picking up travelers from Logan International Airport.
Two van passengers, 59-year-old Joseph Rodriguez and 47-year-old Sandra Arreola, both of El Paso, Texas, were ejected and died at the scene. Two other passengers and the shuttle driver were hospitalized with less serious injuries.
Toxicology screens revealed the presence of THC in Birenbaum's blood and urine, as well as a blood-alcohol content of .15, nearly double the legal limit of .08.
Birenbaum's attorney, Joseph Moynihan, called the crash an "unfortunate, tragic accident" and said his client is remorseful.
His next court date is Feb. 22.


text:
Picture: Glen Ballard and Dave Stewart Broadway opening night of 'Ghost The Musical' at the Lunt-Fontanne Theatre - Arrivals. New York City, USA - 23.04.12
EURYTHMICS star Dave Stewart is singing British teenager Joss Stone's praises after she nailed a revamp of Burt Bacharach's classic movie theme Alfie in just two takes.
Stewart and pal SIR Mick Jagger, who have been working on the score for the remake, were left speechless by the young soul diva's talents when they invited her to join them in the studio.
Stewart says, "The song has been recorded about 76 times and it's probably one of the most covered songs, with heavy orchestrations and a lot of them leaning towards the cabaret sort of feeling.
"This didn't fit at all with the kind of music that Mick and I had been making in the movie, so Joss Stone was great.
"She's 17-years-old and obsessed with blues, gospel, R+B singing and so we just stripped the thing right down with no
orchestration and a Hammond organ and a piano.
"She's an incredible singer and she did it in two takes after learning it in an hour. Amazing."03/10/2004 21:11


text:
Ezreal Build Guide by Qfish
Not Updated For Current Season
This guide has not yet been updated for the current season. Please keep this in mind while reading. You can see the most recently updated guides on the browse guides page.
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AP- yep im still here
Ezreal Build
Normal start aginhst most ap's
average purchace order
if you are been beaten
if you just want to wreck people
general full build
Ability Sequence
Not Updated For Current Season
The masteries shown here are not yet updated for the current season, the guide author needs to set up the new masteries. As such, they will be different than the masteries you see in-game.
Not fully done but this is one hours of me putting out my thoughts, please put in constructive criticism but I would rather you not vote yet, but you can if you must, but enjoy till then.
Hi, I'm Qfish ere with my first build on Moab fire, so any tips would be greatly appreciated when it comes to this, or you can tell me anything that you want me to add, and I'll test it out then hopefully post it within a week at the most. Anyway here is my guide to my faviroute way to play ez. Ap if you haven't guessed already. when I started out playing lol back in 2011 I didn't know what I was doing, AT ALL, buying whatever the hell I wanted - e.g. Ap on cait or flat attack speed on malphite. that happened until I picked up teemo and then looked a phreaks guides, then I understood (not). but anyway not long after that I pick up my second champ ez. when I first looked at his skill ratios I thought he was an ap. so I started playing him that way even till today. it has been about 2 and 1/2 years of experimenting to get me to where I think this build is just right. but do note I DO NOT recommened this build to anyone who can't land skill shots or love to play master yi (mainly because I can't play master yi or tryndamere). you must be able to know how far you can go in if you were to play this in ranked (I got through bronze, into silver then into gold with this build, so I know it works) but anyway enough of me blabbering on and lets get on with the build.
Pros/ Cons
Extremely high burst damage to entire teams
Capable of blowing many mid laners to shreds
nice poke
nobody will think your going ap
he hates noxians like darius
No cc
squishy pretty much throughout the game if he doesn't have someone to protect him
his taunt actually works ;(
these runes I chose because they give him some resistance against ad which is his main weakness early but do give him the capability to scare of ganks with his high damage out put even if he is facing someone who's brought Mr runes thanks to the ap quints and gylphs and MP marks.
these masteries I chose just because they sinergise well with his play style of being a buuuuuurrrrssssttttt mage. it also has mana and manaregen so you can keep firing your W.
these items I chose because it mean that the enemies cannot stop your damage even if they buy one mr item. with these items you'll end up forcing the enemies to waste a lot of gold buying as much mr as possible instead of damage.
Skill Sequence
this should be your general sequence as you shouldn't have to work on your q for little to know improvement since your not going ad. you can change it if enemies are playing to smart to get hurt by your e.
Ranked Play
for ranked play I would not recommend this until your happy that you can play consistently and not get greedy, as If you do you'll end up cost your team the mid lane and/or a loss. so DON'T get GREEDY.
Team Work
as ap ez your want to follow up all initiates done by your tank ( but only if your hitting at least 3+ important enemies e.g. enemy adc, mid laner and support or top) then make sure to get behind someone who has some cc then just poke until you can jump in for the clean up. (or if your that far ahead that once you ult though them you could probably e and w for the instant penta - which you should be able to do - but remember don't get GREEDY) in short your goingf to be able to duel anyone and practically deal a tonne of damage in a fight but you will die if a master yi ignores everyone and hes really fed and he dives on you. (from that it means fed yi + enemy ap ez = dead ap ez) but you get the jist, base your team fight off what your capiable of doing at the time and remember don't flame your teammates you need them to protect you when you do stupid things.
farming is very important (as always) on ap ez as your probably not going to start racking up your kills really quickly until levels 6-9 ( this was an accident that I can't fix) but even them you must try and keep up with your cs and use your q often if you've just used your ujlti as that cd that you get from that is nice. in short just farm and get your items ( make sure to poke with your w) then own everybody.
Fighting/ your combo
you combo should be e in, w, q and then ulti to kill someone ( dfg at the start if you have built one) ( oh and make sure to hit the skills)
in summary enjoy this build, its a lot of fun and you will surprise allies and friend and maybe even some enemies. but don't get GREEDY, make sure to land your skills, remember to farm with your q and autos and poke with your w but don't spam your abilities so fast that you run out of mana and have a good time.


text:
Something I want to tell you my readers I am not a religious kind of guy. I believe in some stuff but I am more in to science and technology than old stuff. One thing that always happens to me is that most of my friends try to prove me wrong that there is GOD and there is heaven and there is hell. I am always having debates with friends and family but something that amazes me is that most of my friends who try to make me believe in God, don't know anything about GOD themselves lol. So if you are someone like me who is always getting into being religious or atheist here is a good question to ask your religious friend I bet he will be lost. And then you can go on your rant..
Question to ask —>
Name the seven last Plagues?
Answer to expect —>
1. Soires
2. Sea turns to blood
3. Rivers turn to blood
4. People scorched by intense heat of sun
5. Darkness
6. River Euphrates dries up
7. Earthquake
I bet you, most of your friends will run away after you ask this question, I have more of these stuff, so sign up for my RSS feed or bookmark me as I am always adding my daily rant and random stuff. Comments are always welcome, be friendly though.


text:
Cholesterol enables nerve cells to connect
While cholesterol has a bad reputation for clogging up arteries and causing heart disease, this fatty molecule is an essential part of all cell membranes. Scientists have now found to their surprise that cholesterol may also regulate when and where nerve cells in the brain form the vital junctions known as synapses.
Equally unforeseen, say investigators, is their finding that non-nerve cells called glia seem to provide the cholesterol that controls synapse building.
"We were definitely shocked," says Frank W. Pfrieger of the Max-Delbrück Center for Molecular Medicine in Berlin. He leads a French-German collaboration that reports the new findings at the Society for Neuroscience meeting in San Diego this week and in the Nov. 9 Science.


text: This is my first year in the lawn and landscape business,and I do it part time.I don't usually deal with contracts unless requested by a customer.I'm not naive,but maybe a little to trusting,and so far with the exception of one customer I have'nt had any problems.A little background I mow this customers yard for the price of $135.00 first mow was $180.00 he paid me $200.00.Property is expensive here in Northern VA and he owns 20 acres with a 3500 sf house pool ,play ground ,basketball court probably worth $1.5 mil. or sther than mowing he requested mulching ,leaf removal,gutter cleaning,pruning etc.So I thought cool 5 min. from the house well off easy going customer with a ton of work at our agreed upon price of $45.00 per man hour.Well- I have'nt been able to get in touch with him for over 2 months after he accumulated a bill of over $600.00 with the exception of when I handed him the first bill ( he asked at that time if he could mail me the payment because he had his business check book at his office ) I called the #'s he gave me prob. 50 times literally (no response) he told me early on he gets about 300 voice mails a day in his line of work and cant possibly check them all. For his second notice I handed it to his wife 1 month ago ( no response).I'm going to drop off one more notice advising him he has 1 week to pay his debt or else There will be legal action taken.My question is and especially for you LCO's in VA but open to all- what are my options ? small claims court?With the info. I've given what are my chances of collecting ? What's it cost to do that in VA? How well does a verbal contract hold up in court? I have'nt done any work there for almost two months and informed his wife that was due to lack of payment and my unsuccessful attempts to contact him.Sorry for the length of this post there's a lot to explain here.Any help or advice would be appreciated , with the exception of busting my 00 about the lack of a contract.The bills I gave him were itemized bills the second stating it was his second notice.


text:
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text: correct me if i'm wrong but i didn't see a thread started regarding EVO apps. list your favorite EVO app's in this thread....if you really like the app give a brief summary of it as well. free or paid? Free 1. Pandora 2. Handcent - (SMS like iphone, much better than stock messaging app) 3. Twidroid - (twitter app) 4. ShopSavvy - (turns EVO into a bar code scanner, really works well.) 5. Flikie - (free hd wallpapers) 6. Ringdroid - (make your own ringtones)


text:
|September 23
|Senior Pilgrimage
|September 30
|Senior Cruise
|October 18
|Graduation Presentation
|National Achiever Services graduation presentation
|November 8
|Place orders for graduation regalia on campus
|National Achiever Services will return to campus to answer questions and collect orders.
ORDER ONLINE: You can order your cap and gown unit (which comes with a tassel and sash) on the Herff Jones website.
|December 1
|Senior Pictures Due
|Senior Pictures are due to the yearbook staff (via email firstname.lastname@example.com, email@example.com; or via eshare: hjeshare.com, school code GoCru). Please label your photo: Senior Photo with (Student Full Name). Specs for your photographer: 2"w x 3"w, RGB color, 300 dpi.
|December 4
|Senior "Shout-out"
|Senior "Shout-out" (created by senior) due to yearbook staff - email@example.com. The following items are included in the shout-outs: quotation, shout-outs, favorite memory, activities and future plans.
|December 12
|Senior Ads due
|Ads (optional and first come, first served) must be turned in to yearbook staff from the beginning of school until December 12. We are working to accommodate as many students as possible. This year only 1/4 ($100) or 1/2 ($200) page ads will be sold. Use this form to order.
|December 15
|Deadline for online National Achiever orders
|Last day to place your online order for graduation regalia at nationalachiever.com
|January 12
|Yearbook Orders due
|Purchase from HerffJones via mailer or at yearbookordercenter.com, school #6287.
|January 19-21
|Senior Emmaus Retreat
|Student Baby Pictures due
|Deadline to submit student baby pictures for senior tribute slideshow. Email or deliver to the Attendance Office.
|April 11
|Formal Group Portrait
|This photo has become an annual tradition at EC and is used to congratulate the students and for marketing materials. Liturgy dress will be required on this day. Girls will be seated and should plan to wear a dress or skirt that reaches to the knee or below. Long skirts/maxis are encouraged. More information will be sent regarding this photo.
|April TBD
|Caps and Gowns delivered
|National Achiever Services will be on campus to deliver caps, gowns and other ordered items.
|May 15
|Tuition, Fees and Fines paid
|All tuition, fees, and fines must be paid by this date
|May 19
|Junior/Senior Prom
|May 23 (tentative)
|Senior Tribute
All seniors are expected to attend and senior parents will not want to miss it! Seniors must arrive by 7:15 a.m. for their individual head-shot picture. Liturgy dress is required. A reception hosted by the sophomore class will follow the Senior Tribute. After Senior Tribute and the reception, seniors will be dismissed with their families for the day.
|May 29 - June 1
|Final Exams for seniors
|May 31
|Senior Lunch and Graduation Rehearsal
|A hosted lunch will follow rehearsal. Seniors must be at rehearsal in order to walk at graduation. Any honor cords that are earned will be distributed at rehearsal.
|June 1
|Baccalaureate Mass
|Baccalaureate Mass is held on Friday, June 1 at 7 p.m. at Mary, Queen of Peace Catholic Church in Sammamish. Seniors will wear their graduation gowns and sashes, but not caps.
|June 4
|The ceremony takes place Monday, June 4 at 7:30 p.m. at Benaroya Hall in downtown Seattle. Students will need to arrive no later than 6:30 p.m. - please account for rush hour traffic. Further details will be emailed this spring. A student seating chart will be posted in May.
|June 5
|Grad Night 2018
|This event is parent-sponsored and encourages 100% student participation. Seniors will be picked up at Benaroya Hall after graduation. Buses will depart promptly at 10:30 p.m. The destination is a surprise but a sweatshirt and closed-toed shoes are recommended. Buses will return to EC campus Tuesday, June 6 with a stop at the Mercer Island Park and Ride at 5:45 a.m. and EC by 6:15 a.m. Parents should plan to pick up at one of these locations so students are not driving after being up all night. As a reminder, no cell phones, bags, large oversized jackets or backpacks will be allowed. More information will be forthcoming from the planning committee in the spring.


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Originally published by Oregon Music News
When I uploaded World's Greatest Ghosts' new album, No Magic, into my iTunes, its genre automatically came up as "Alternative & Punk." Who knows why Apple does this for artists that couldn't be further from the label, but Andrew Bird, Captain Beefheart, Fleet Foxes and Glenn Kotche have all received this misnomer. To my surprise, World's Greatest Ghosts aren't out of place this time.
No Magic is a refreshing return to raucousness. It's indie-spazz rock in rapid-fire common time. The upbeat, energetic rhythms are made for dancing with your arms flailing wildly like Elwood Blues doing the "running man". Sing-along melodies standout from start to finish, and you can just picture a guy doing a handstand on a Moog while the guitarist repeatedly scissor kicks. Reggie and the Full Effect will be jealous of the warm, playful synths on tracks like "Mazes and Monsters," "The Royal Court" and "On the Shore".
Journey-ed rock-outs prove that WGG has a lot of musical ideas. No Magic is an album for anyone who just wants music to be fun again. The lyrics are occasionally cringe-worthy ("Can you show me a way/A way to wake up/ It's time for me to wake up/We're on a mission/To find our way/Out of these nightmares") and the melodies are overly sugary, but it's all too sincere to refuse.
The Magic themes are prominent throughout the album and an apt compliment to the wildly spastic twists and turns from one harmonized guitar riff to the next. It's like the musical version of playing the Legend of Zelda for the NES and occasionally finding secret levels in the next screen over. There are a lot of surprises on No Magic, none of which are unpleasant. The vocal/guitar duo breakdown near the album's end comes out of nowhere, but kicks the energy up to a level that can only be reached in the poppiest moments of punk rock.
Coming in at just over a half hour, the album is over too soon. The nine tracks blaze by like a fire-breathing dragon dousing the countryside, villages aflame. But it's just as easy to put it on again for a second ride. Or, even better, this is an album that beckons its listener to come out to a WGG show. I'd expect full LARPer garb from these kids.


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GraphicRiver Back To The 80 s 8430294
Photoshop \ Print Template \ Flyers \ Events \ Clubs & Parties
League \ Gothic \ Feedback \ Assistance \ Questions \ Theleagueofmoveabletype
Print Template - Back To The 80 s 8430294 by thenewage @ GraphicRiver
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text:
Usher's two sons mean everything to him and he describes the two boys as his ''most prized possessions''.
Usher's sons are his ''most prized possession''.
The 'Scream' hitmaker is father to Usher, six, and five-year-old Naviyd - who he had with ex-wife Tameka Foster - and delights in the talents the two boys are already showing.
He said: ''They are definitely musical, Naviyd plays piano, he had his first recital not too long ago, but Usher is more active and athletic.
''They are my greatest investment, my most prized possession, it's everything.''
The 35-year-old singer portrays boxer Sugar Ray Leonard in new movie 'Hands of Stone' and loved working on the film - particularly as he learned how to fight and improved his physique.
Speaking on 'Live with Kelly and Michael', he said: ''I've been working on that for the last year and we just completed it. It's really incredible. I did train like a boxer, I'm actually rather good with my hands now.
''That's one of the great things about working on a boxing film, you definitely get the physique together, but greater than that was to act opposite Edgar Ramirez, Robert DeNiro, I mean, it was just incredible.''
Though he has an impressive body from making the movie, Usher admits to having a ''weakness'' for junk food.
Asked which of the coaches on 'The Voice' eats the most junk food, he admitted it was himself and added: ''My favourite junk food is Australian liquorice, I have a weakness.''
Take a look back at October's inaugural event.
The film is expected to continue without Mendes' involvement.
Roberto "Manos de Piedra" Duran was fierce in the ring, his poor upbringing on the...
When Justin Bieber was discovered by Scooter Braun and Usher Raymond after performing his songs...
Clearly designed with his 12-year-old female fanbase in mind, this backstage myth-making doc wants to...


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Despite much bluster about running against Democratic incumbent Gray Davis for California governor in 2004, Arnold Schwarzenegger has ruled it out, the Los Angeles Times reports. "The timing's not right," says a spokesperson for the staunchly Republican actor. "If there were a time in the future when he did not have commitments and felt he could do more good for the country in an official capacity, he would make a determination to run at that time. At this time, he can't and he's not."
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- Description
2-Pack Armani classical boxer briefs. The support and the comfort they offer are in line with the tradition of Emporio Aramni boxer briefs.
2-Pack black Emporio Armani boxer briefs that discreetly bear the Armani eagle on the waistband, as well as the initials of the brand: an E on the left and an A on the right. What could be simpler? Two very elegant and discreet men's boxer briefs about to become classics of the Armani brand.
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The Emporio Armani products include ready-to-wear clothes, glasses, perfumes, accessories and watches. This line is considered a "linking line" with finer finishing touches than those of Armani Exchange or Armani Jeans.
Emporio Armani has had David Beckham, Christiano Ronaldo and even Rafal Nadal as ambassadors.
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The Lazarus Project Paperback
by Aleksandar Hemon
On 2 March 1908, Lazarus Averbuch, a young Russian Jewish immigrant to Chicago, tried to deliver a letter to the city's Chief of Police.
He was shot dead. After the shooting, it was claimed he was an anarchist assassin and an agent of foreign operatives who wanted to bring the United States to its knees.
His sister, Olga, was left alone and bereft in a city seething with tension.
A century later, two friends become obsessed with the truth about Lazarus and decide to travel to his birthplace.
As the stories intertwine, a world emerges in which everything and nothing has changed . . . 'Prose this powerful could wake the dead' Observer 'This is easily Hemon's best work to date, an intricately tessellated portrait of flight, emigration, and the meaning of home' Evening Standard
- Format: Paperback
- Pages: 304 pages
- Publisher: Pan Macmillan
- Publication Date: 26/08/2008
- Category: Modern & contemporary fiction (post c 1945)
Other Formats
- EPUB from £7.19
Free Home Delivery
on all orders
Pick up orders
from local bookshops
Showing 1 - 4 of 4 reviews.
Review by wandering_star
This stunning novel interleaves the story of Lazarus Averbuch, a young Jewish immigrant, killed in Chicago in 1908 and framed as part of the "anarchist menace", with that of Vladimir Brik, an early-21st century writer, a Bosnian immigrant to the US (like Hemon himself) who becomes interested in the case and travels through the Balkans and Eastern Europe researching Lazarus' family history, hoping to get a novel out of it.The 1908 section - the anti-anarchist panic, the callous framing of an innocent young man and the media response to Lazarus' supposed evil crime - is properly angry-making, although it does lose its way a little as the novel goes on. The sections set in the modern-day, desperate and gangster-ridden Eastern Europe are more depressing than angering. But the prose is incredible, and the book stirs up all sorts of ideas, about home, freedom, the old world and the new, and what it means to know someone else. The echoes between the events of both sections lead the reader to think about twenty-first century genocides and xenophobias, as well as the nineteenth- and twentieth-century ones which are directly explored. I had to keep forcing myself to slow down so that I could really get the most out of the writing itself and also the ideas behind it.Sample: One morning in Chicago I had tiptoed to the kitchen with the intention of making some coffee. While customarily spilling coffee grounds all over the counter, I spotted a can in the corner whose red label read SADNESS. Was there so much of it they could can it and sell it? A bolt of pain went through my intestines before I realized that it was not SADNESS but SARDINES. It was too late for recovery, for sadness was now the dark matter in the universe of still objects around me: the salt and pepper shakers; the honey jar; the bag of sun-dried tomatoes; the blunt knife; a desiccated loaf of bread; the two coffee cups, waiting. My country's main exports are stolen cars and sadness.
Review by AlexDraven
Shapeless, pointless, depressing, but individual stories and scenes funny/entertaining. No conclusions, Bosnia is life, America death, his return his rebirth? Multiple interpretations of the Lazarus mythsIntense loneliness and dislocation of young men who have come to America to escape European conflicts, whether it's 1903 or 1992, Jewish Lazarus, or the Bosnian columnist who plans to write about him. Fluid and murky, Ideas about truth, history, propaganda, and what emotive terms like 'know' and 'self, and 'home' might mean. Patterns are set up, but not resolved, parallels drawn but not cleanly, instead everyone is drawn into a spiders web of overlapping stories with overlapping levels of 'truth'.Outside perspective as well as the style keeps us distant from the characters, even as the narrator longs for closeness with his wife, his friend, his current life, his history.
Review by labeet
A very strange book, I found. In one way it gripped me in another it didn't. It's a strange loose story that doesn't quite come together. I realise it was probably the author's intention that it doesn't come together. But I'm afraid I wanted it to and it failed me there...
Review by Daftboy1
This is not the normal book I would go for, but I am glad I did. Part history lesson, story of friendship and travelogue this is well worth a read.
Also by Aleksandar Hemon | View all


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Quick Links
Planning and Development/Bylaw Services
Email Us
Phone: 250-561-7622
Bylaw Services1100 Patricia BlvdPrince George, British ColumbiaV2L 3V9
The City of Prince George's Bylaw Services Division enforces a variety of City Bylaws designed to improve the quality of living in Prince George.
Animal Control Officers respond to animal related complaints and enforce the
Animal Control Bylaw.
Learn more about animal control within the City of Prince George.
Office Hours: 24 hours a day, seven days a weekPhone: 250-561-7622
Parking Control Officers enforce on-street and off-street parking bylaws.
Learn more about parking control within the City of Prince George.
Bylaw Enforcement Officers enforce the provisions of bylaws such as
Using the online report a concern form.
If you make a complaint, your personal information is collected under the authority of section 26(b) of the
Freedom of Information and Protection of Privacy Act, for the purposes of processing the complaint and for administration and enforcement. Complaint submissions become part of City of Prince George's records and are subject to release under the
Freedom of Information and Protection of Privacy Act. The information will be used to process your complaint but your name and address will not be made available to the public, unless releasing that information is required for a legal action to prosecute the bylaw infraction.
If you have any questions about the collection of information, please contact:
Fred Crittenden, Manager of Bylaw Services
By mail or in person:
1100 Patricia BoulevardPrince George, BC V2L 3V9
By telephone: 250-561-7623
By email:
City of Prince George Bylaw Notice Enforcement Bylaw, No. 8813, 2016 and the Local Government Bylaw Notice Enforcement Act established the use of and a method for disputing a Bylaw Offence Notice and a Parking Offence Notice. This procedure will outline the process for disputing a Bylaw Offence Notice and a Parking Offence Notice. A dispute will be reviewed by a Screening Officer and if necessary referred to Adjudication. The Adjudication system allows the City to manage bylaw disputes locally rather than through the provincial court system. Violation notices can be disputed out of court with a private adjudicator appointed by the Provincial Attorney General Office.
City of Prince GeorgeBylaw Services1100 Patricia Blvd.Prince George, BC V2L 3V9Fax#: 250-561-7745
City of Prince GeorgeBylaw Services1100 Patricia Blvd.Prince George, BC V2L 3V9
Fax#: 250-561-7745


text:
Jim Barbey Automotive
Review for Jim Barbey Automotive
Anthony G. on 11/21/2011
Anthony said they were able to get him in right away and the work was done on time. He mentioned it was a little expensive.
Category: Service
Service Date: 10/19/2011
Review Created: 11/21/2011
This review was collected via phone interview by Customer Research Inc.


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GraphicRiver Colored Balloons 10305050
Category: Stock Vector | Comment: ( 0 )
GraphicRiver Colored Balloons 10305050
Stock Vector \ Conceptual \ Seasons \ Holidays \ Birthdays
Realistic \ Pink \ Play \ Photo \ Party \ Isolated \ Orange \ Ribbons \ Background
Stock Vector - Colored Balloons 10305050 by andegro4ka @ GraphicRiver
Stock Vector \ Conceptual \ Seasons \ Holidays \ Birthdays
Layered: No; Graphics Files Included: Photoshop PSD, JPG Image, Vector EPS; Minimum Adobe CS Version: CS.
Colored balloons isolated on white photo-realistic vector illustration
Zip file includes.
- eps10, editable vector, RGB
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- psd
Flashlight; Basketball Hoop; Grave; American Flag Background; Opened Manhole; Table Lamp; Street Lantern;
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Short-eared Owl Relaxing by ClaudeGWatch
©2013-2017 ClaudeG
This is my favourite subject for the last days...
Seems like a wierd possession to be relaxing in
That is beautiful!
beautiful pose !!
Just amazing
This pose is so unique - and this owl is adorable :3
Looks like he's doing Yoga - great shot!
so lovely
uh i love it :3 these are my fav owls *__* this shoot is awsome ^^


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Results 1 to 1 of 1
Thread: Problem with ASE 12.03 and JAVA
07-29-04, 02:39 #1Registered User
- Join Date
- Jun 2004
- Posts
- 3
Unanswered: Problem with ASE 12.03 and JAVA
We use a JAVA-Class in a Stored Procedure like this:
-- declare the JAVA-Calss
declare @Calc_object tarif.Calc
-- create the class
select @Calc_object = new tarif.Calc()
-- call a method of that class
select @out_java = @Calc_object>>int_main (@parameter)
Problem 1:
When the connection to the database is lost while the JAVA-Class is running within the Stored Procedure, we cannot drop the Stored Procedure (Error: "Procedure is currently in use"). A look with sp_who shows no process.
Only thing that helps seems to be shut down and reboot the server.
Any better ideas?
Problem 2:
I couldn't find how to destroy a Java-Class created in the Stored procedure. GarbageCollection seems not to work.
Thanks for any help!


text:
The PACS Health Clinic provides primary care, meaning you can get set up with a regular go-to doctor to help you manage your health. It is available exclusively to low-income individuals who are struggling to afford the cost of primary health care.
The clinic sees patients by appointment only. If you are not already a patient, call 503.252.4589 to find out if we have any openings for new patients. Current patients can call the same number to schedule an appointment.
The clinic does not currently provide dental care. However, a campaign has been launched to add dental to our health care services. Visit PACSDENTAL.ORG to learn more and follow the progress of the project.


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Synopsis of the novel Vampire Diaries - The Return: Midnight
Damon is now human. He uses Misao's star ball to reopen the gate to the Dark Dimension and takes Bonnie with him by accident. Damon is then turned back into a vampire by Princess Jessalyn. Bonnie is arrested as a runaway slave and taken to an auction house to be sold. Shinichi buys Bonnie to extract information about Misao's star ball. Since the group has drained Power out of it, Misao is dying. Shinichi throws Bonnie out of a window, but Damon saves her.
Meanwhile back in Fell's Church, Matt is arrested for assaulting Caroline, and Stefan, Elena and Mrs. Flowers have to get him out. They bring in Saber, a hell hound Sage left to protect them. Caroline changes into a werewolf at the sight of him. All the other officials at the court all turn into werewolves as well. When Matt is free, Stefan and Elena go into the Dark Dimension to find their friends, which they do at Lady Ulma's house. Soon after the four of them travel to the Nether World in search of the largest star ball ever made in hopes that it will save their town from The Last Midnight (the Last Midnight is then revealed to be all the possessed children attacking their parents).
Sage is the guardian of the Gate where the treasures are, including the star ball. He lets the group pass and visit one of the 7 gateways. Meredith discovers that she had a twin brother named Cristian, who almost died, but Klaus had taken him when they were only 3 years old when the "attack" went on with her grandfather. She also tells the group what she really is, which is a hunter-slayer, and a new type of vampire with small fangs. Her parents have been feeding her amounts of blood in their meals.
The kitsunes say they are working for someone who told them to destroy Fell's Church. It turns out to be Inari, Isobel's Grandmother. She is the Japanese goddess of rice and foxes; she is the most powerful Kitsune in the world and the star ball is hers.
In the Nether World, on the smallest moon, the group finds the star ball in a large tree. Damon is unexpectedly killed by the tree when it stakes him in the heart while he saved Bonnie from falling to her death. Elena uses her "Wings Of Destruction" to destroy the tree and star ball, killing Inari. Shinichi had died earlier from the "Sin of Regret" and Misao dies when all the Power from her star ball has been drained.
Mrs. Flowers changed into a younger version of herself before Inari died. Everyone, including Stefan, Bonnie, and especially Elena, mourns the death of Damon, while Elena promises to never forget him or how she loved him. Sage tells them that maybe the Celestial Court can reverse all the damage done in Fell's Church. There it is revealed by the leader of the blonde Guardians that Elena was destined to become a guardian and when she and her family were driving in their car they appeared to her on earth.
Young Elena was more talented then they expected and was able to see them passing and she distracted her father when she told him about what she saw. Elena was the one meant to have died in the accident (although they had planned on simply kidnapping her) so she could become a Guardian in the Dark Dimension. The Celestial Court agrees to reverse what was done in Fell's Church (in return for the treasures they had collected) and erase the memory of what happened there.
Elena can go back home as if she had never died and Stefan will be accepted as her boyfriend. Vicki Bennett, Sue Carson, and Mr. Tanner all get their lives back, but unfortunately, there is nothing that can be done for Damon. When angered that the Celestial Court will not do anything to save Damon she begins to unleash her wings of destruction. However Lenea, a soldier at the court, stops her from speaking the words and therefore the power is not unleashed.
Angered by her attempt to kill them The three Guardians of the Celestial Court decide Elena is unfit to wield the great power of her wings and so they clip her of all six of them. Elena quickly asks to just be sent home to Fell's Church after they have undid everything. However her wings and telpathy (with everyone but Stefan) are lost and she is practically fully human once again with no supernatural aspect about her. Stefan and Elena sit in her old room and cherish the memories that they have of Damon. The book ends on the Nether World's smallest moon. It is raining from the tree Elena had destroyed and liquid drips onto a lifeless body. Only it seems to not be lifeless; it thinks of a girl with golden hair and blue eyes and a girl with a small face and brown eyes; Elena and Bonnie. It says if he can only remember who he is, he can live again, and with time a self revelation came, a name, Damon.