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I took the kids to a lovely exhibition at Hove Museum yesterday. It was about the work of Carol McNicoll - wonderful, witty pottery. I'd never seen her stuff before, but apparently she is a leading light in British ceramics since the 1970s. They had a great video of her talking about individual pieces and what was behind them. Often this was a cutting comment on politics and capitalism.
I could have stayed there much longer, but Leo was in a big hurry to get the museum over with and move on to the main business of the day at the museum shop! We have recently introduced pocket money in an attempt to curb his rampant acquisitiveness, but I am not very hopeful about this, judging by my experience yesterday. He left his pocket money at home and then pestered me non-stop to buy him something. So I ended up buying things for both of them (on grounds of fairness) even though P had dutifully brought a pound of her own money.
It's not the money that bothers me so much (although we are a bit skint at the moment, after Christmas), but the fact that his preoccupation with being bought something can overshadow the whole experience of being wherever we are - museum, high street, park, etc. In fact, once he had been bought a very small something, I think he did listen to my frustrated rant and look at the exhibits with a bit more interest. Maybe we should just visit the museum shop first...
Mind you, I don't know if I am a very good judge of what people are taking in from these kind of things. P, L and I went to another exhibition on Wednesday afternoon, at the Brighton University gallery in Grand Parade. It was an exhibition of collections, with accompanying text about the objects, and what collecting them has meant to the collector. Fascinating. I enjoyed it immensely, but felt again that the kids hadn't taken much notice of it.
But yesterday, Leo revived one of his old interests and put together a frog and snake collection in a display case, just like the ones in the gallery. So you never know.


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Democracy Dies in Darkness
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October 22, 2014
Santa Clara, CA – October 22, 2014 – Netronome, the leading provider of data plane processing solutions for software-defined networks (SDN), today announced that they will discuss a processing architecture to address scalability issues facing cloud network providers at The Linley Tech Processor Conference on October 23, 2014, at the Hyatt Regency Hotel in Santa Clara, California.
"Large scale data center operators face new challenges as their next wave of deployments include higher power CPUs and network interconnects moving to 25, 40, and 100 Gigabit Ethernet," said Bob Wheeler, principal analyst at The Linley Group. "A new class of intelligent offload solutions optimally addresses power, cost, throughput and programmability for future-proofed designs."
Sundar Gopalan, senior director of product management at Netronome, will present, "Enabling Cloud Providers for 10/40/100GbE Network Processing Requirements," during the Challenges of Implementing SDN and NFV track. This presentation will focus on the challenges faced by the cloud infrastructure providers and highlight architectural requirements of intelligent interface cards for delivering a scalable and high performance solution on standard servers.
"Cloud infrastructure is experiencing unprecedented growth in terms of new server deployments that require high network performance with heavier compute workloads. As a result network operators are looking at opportunities to re-architect their network and compute infrastructure," said Jarrod Siket, senior vice president and general manager of marketing at Netronome. "Our purpose built flow processors, intelligent NIC cards, combined with software support for SDN and NFV designs provide an ideal solution for the operators to meet the challenges."
The Linley Tech Processor Conference includes dual-track presentations on the latest processor chips, processor IP and other technology required to efficiently process data in embedded, enterprise, and cloud applications. For more information about the conference, please visit:http://www.linleygroup.com/events/event.php?num=29.
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About Netronome
Netronome is the leading developer of high-performance networking solutions for cloud, data center, service provider and enterprise communications products. Netronome flow processing solutions increase the performance of virtualized servers by offloading compute-intensive workloads such as network virtualization, security, flow classification, content processing, deep packet inspection and dynamic load balancing. Netronome products are used by networking OEMs, ODMs worldwide and include FlowProcessors, FlowNICs and FlowEnvironment software. Netronome processors are supported by comprehensive tools and a broad ecosystem of premier partners. Netronome products are developed in labs in Santa Clara, CA, Boxborough, MA and Pittsburgh, PA. To learn more about Netronome and its products, please visit www.netronome.com.
Netronome, the Netronome Logo, "The Flow Processing Company," "Total Performance," "Total Control," "Intelligent to the Core," NanoNetwork, Open Appliance Platform and the Open Appliance Platform Logo are registered trademarks or trademarks of Netronome Systems, Inc. in the United States and other countries.
All other trademarks mentioned in this document (release, ad, etc.) are registered trademarks or trademarks of their respective owners in the United States and other countries.


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How to install and run Zombie Slayer For PC via Andy OS Android emulator
In order to install Zombie Slayer For PC via Andy OS Android emulator, you may want to use the guide: How To Run Apps For PC On Windows, Mac Using Andy OS.


text:
I don't know about you, but headsets are something that I spend a pile of time on. Getting a headset that fits well, sounds good, and has basics like a long enough cord, is not always easy.Â
I've always been a fan of Plantronics sets. They tend to be a little pricy, but they also tend to be very good quality. My dog ate my last one a little while ago (she's a puppy, and chews). Recently I've been using the new .Audio 340 Behind-the-Head Multimedia Headset.
The 340 is sleekly styled, and at $29.95 relatively inexpensive. With an 8 foot cable, I can wear it while sitting at my desk and still move around easily. The integrated mute button and volume controls have a convenient clip on the back as well, so they can be easily fixed to my shirt. And it's comfortable as can be.
Most importantly, 340 has deep rich sound, and does a very credible job of reproducing bass tones. You could easily use these headphones for VoIP calls (as I primarily do), or listening to music, or playing games. They're that good.
What could be better? The biggest single addition would be a USB version. Then I could hear Skype ring through my PC speakers, and use the headphones for the conversation. It's a small complaint.


text:
BCT Day 43
The following information is a portion of "Marck's Boot Camp Diary". The information contains a straight-forward look at Basic Training Life on a daily basis. The information contained within this page is used with the express written permission of the author. For more information about the author view the "Author's Information" at the bottom of the page. Some choice phrases have been censored.
September 5th, 2002
Ow. So much pain. Today was one of the two hardest days of BCT. We learned Individual Tactical Training (ITT). Ow. We did high crawls and low crawls on the dirt. It wasn't a nice soft pit this time. It was hard, rocky, nasty dirt with tree roots and spiders. I went under barbed wire, held my rifle behind logs, and crawled in ditches. All this training is to learn concealed movement in battle. I had my face camouflaged with green and black clay stuff. By the end I was so dirty and nasty, and I do mean nasty, that I looked like that kid from Peanuts who always has a cloud of dirt around him. We all did.
Ow. Then we pissed the DS off for something, so we marched back at an EXTREMELY FAST pace with about 50 lbs on our back. I had to run a whole lot just to keep up with the DS. His back was nice and free of heavy ****, so it was extremely hard to keep up. We had one guy pass out. My arms are barely hanging now. I can't even do 40 decent push-ups. When I get my arms back in a few days, I'll be SO strong for the final PT test next week. I must've done 300 push- ups yesterday. If you punch me on my chest now, I'll probably cringe in muscle pain for five minutes and then get up to beat the crap out of you. I also got some cough drops in the mail. Thanks mom & dad.
Ow. I ate my first Meal Ready to Eat (MRE) today. Those are the military meals that come in a 12x6 box and last years. It was awesome. Ow. I'll tell you more tomorrow, since it hurts to write and breathe at once.


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Launch of the Space Shuttle Atlantis on a repair mission of the Hubble Space Telescope. This was the final mission to the Hubble and one of the last missions of NASA's Space Shuttle program. Kennedy Space Center, Cape Canaveral, Florida, May 11, 2009
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In these experiments, we used a strain of angiotensin converting enzyme (ACE) germline null mutant mice, generated by J. H. Krege and co-workers (J. H. Krege, S. W. M. John, L. L. Langenbach, J. B. Hodgin, J. R. Hagaman, E. S. Bachman, J. C. Jennette, D. A. O'Brien, and O. Smithies. Nature 375: 146–148, 1995), to examine the effect of chronic ACE deficiency on the magnitude of tubuloglomerular feedback (TGF) responses. The genotype was determined by PCR on DNA extracted from the tail and was verified after each experiment by assessment of the blood pressure response to an injection of ANG I. To assess TGF responsiveness, we determined the change in stop-flow pressure (PSF) caused by increasing NaCl concentration at the macula densa by using micropuncture techniques. When loop of Henle flow rate was increased from 0 to 40 nl/min, PSF fell from a mean of 42.3 ± 1.95 to 33.6 ± 2.09 mmHg (n = 6,P = 0.005) in wild-type mice (+/+), fell from 40.6 ± 2.35 to 38.6 ± 1.93 mmHg in heterozygous (+/−) mice (n = 7,P = 0.014), and did not change in homozygous ACE (−/−) mice [36.7 ± 2.02 mmHg vs. 36.4 ± 2.01 mmHg; n = 4,P = not significant (NS)]. During an infusion of ANG II at a dose that did not significantly elevate blood pressure (70 ng ⋅ kg−1 ⋅ min−1), TGF response magnitude (PSF 0 − PSF 40) increased from 6.5 ± 1.4 to 9.8 ± 1.19 mmHg in +/+ (P = 0.006), from 1.14 ± 0.42 to 4.6 ± 1.3 mmHg in +/− (P = 0.016), and from 0.42 ± 0.25 to 4.02 ± 1.06 in −/− mice (P = 0.05). Absence of TGF responses in ACE null mutant mice and restoration of near-normal responses during an acute infusion of ANG II supports previous conclusions that ANG II is an essential component in the signal transmission pathway that links the macula densa with the glomerular vascular pole.
- transgenic mouse
- gene knockout
- micropuncture
- angiotensin converting enzyme
- stop-flow pressure
an increase in NaCl concentration at the macula densa cells causes a prompt increase in the resistance of afferent arterioles and a subsequent decrease in glomerular capillary pressure and glomerular filtration rate (3). There is abundant evidence to indicate that the effect of NaCl on vascular tone is linked to Na-K-2Cl cotransport activity in macula densa cells and that an alteration in transport causes the formation of a paracrine mediator capable of interacting with the vascular smooth muscle cells of the afferent arterioles (3). Identification of this flow-dependent mediator is still incomplete, mainly because interference with the generation or action of a number of potential vasoactive compounds has been demonstrated to affect the tubulovascular response magnitude. Results from a variety of technical approaches lend support to the notion that the renin-angiotensin system is intimately involved in determining the magnitude of the vascular response. Pharmacological inhibition of ANG II type I (AT1A) receptors and of converting enzyme has been shown to reduce the magnitude of the tubulovascular response (8, 16, 18, 22, 25, 28), whereas peritubular and systemic infusion of ANG II was found to enhance the vascular response magnitude (13, 19). The physiological implication of the dependence of feedback regulation on ANG II levels is that a change in the activity of the renin-angiotensin system may be responsible for the alteration in macula densa-dependent responses seen during changes in extracellular fluid volume (3, 21).
The generation of transgenic mice with null mutations in components of the renin-angiotensin system offers a new way to examine the role of ANG II in tubuloglomerular feedback (TGF) coupling. Previous results have shown that the effect of changes in macula densa NaCl concentration on afferent arteriolar tone is virtually abolished in mice with a knockout mutation in the AT1A receptor gene (24). Two laboratories have independently generated mice with a targeted disruption of the angiotensin converting enzyme (ACE) gene that interferes with the production of functional ACE enzymes of both the somatic and testicular isoforms (5, 10). ACE enzyme activity in plasma and tissues such as lung, kidney, heart, and brain, as well as ACE mRNA and protein in various tissues, was virtually undetectable in homozygous ACE-knockout mice (5, 26). The current experiments in one of these ACE-knockout strains were performed to examine whether interference with the renin-angiotensin system by an independent intervention results in the same alteration of the tubulovascular signaling pathway previously found in the AT1A receptor knockout animals (24). The second aim of these studies was to test whether an acute administration of ANG II can restore the ability of increases in distal NaCl concentration to cause afferent vasoconstriction.
Experiments were performed in a strain of ACE germline null mutant mice that has been generated as described by Krege et al. (10). All animals used in this study [wild-type ACE (+/+), heterozygous ACE (+/−), and homozygous ACE (−/−)] were derived from a heterozygous breeder pair from the original North Carolina colony. At weaning, animals were ear-tagged and a short piece of the tail was clipped off. Genomic DNA was extracted from the tail samples with a standard procedure involving digestion with proteinase K and purification of DNA with ethanol. We determined the genotype of each DNA sample by testing for the presence of wild-type or modified ACE sequence using PCR. To identify the presence of the wild-type gene, ACE gene-specific primers upstream and downstream of theHind III site used for the insertion of the neomycin resistance gene (Neor) construct were used to amplify a 443-bp product that included a 207-bp intronic sequence but that was identical in its exon sequences to the published ACE cDNA (2). Primer sequences are sense 5′-AAGCACGGAGGTATCCAATCACACC-3′ (bp 1086–1110) and antisense 5′-AATGGCACTTGTATGCCCCTGGAA-3′ (bp 1297–1321). We detected the mutant ACE gene with primers amplifying a 457-bp product of the Neor by using previously published oligonucleotide sequences (1). An example of PCR genotyping is shown in Fig. 1. A third PCR for β-globin served as control to validate similar amounts of DNA in each sample. Amplification was carried out for 30 cycles (denaturation at 94°C for 40 s, annealing at 58°C for 40 s, and extension at 72°C for 40 s, followed by a final extension at 72°C for 8 min). After genotyping, animals were separated according to genotype and gender.
Animals were maintained on a standard rodent chow and tap water. Male mice (23–30 g) were anesthetized with 100 mg/kg Inactin ip and 100 mg/kg ketamine im. Body temperature was maintained at 38°C by placing the animals on an operating table with a servo-controlled heating plate. The trachea was cannulated and a stream of 100% oxygen was blown toward the tracheal tube throughout the experiment. The femoral artery was cannulated with hand-drawn polyethylene tubing for measurement of arterial blood pressure. The femoral vein was cannulated for an intravenous maintenance infusion of 2.25 g/100 ml BSA in saline at a rate of 0.35 ml/h. This rate of infusion has previously been shown to be associated with a urine flow of ∼2 μl/min and a urine osmolality of ∼1,000 mosmol/kgH2O, suggesting adequate hydration but no overt volume expansion (23).
The left kidney was approached from a flank incision, freed of adherent fat and connective tissue, and placed in a Lucite cup adapted for the size of the mouse kidney. The kidney was covered with mineral oil. Measurements of early proximal stop-flow pressure (PSF) during loop of Henle perfusion were done to assess TGF responsiveness. A late proximal tubule segment was identified from the staining pattern after microperfusion of a randomly selected proximal segment with the FD&C-stained perfusate. The tubule was blocked with wax, the pump was inserted in the last superficial proximal segment, and the pressure pipette was inserted into an early proximal segment, recognizable from the widening of the tubular lumen. When PSF had stabilized, loop of Henle perfusion rate was altered, between 0 and 40 nl/min, in a random fashion. In selected tubules in which the stability of the PSF recording and the position of the recording pipette were optimal, ANG II was added to the venous line through a Y-shaped side-arm at an initial rate of 100 ng ⋅ kg−1 ⋅ min−1. If this infusion caused a discernible increase in arterial pressure, the rate of ANG II infusion was reduced to 50 ng ⋅ kg−1 ⋅ min−1. This reduction in the rate of ANG II delivery was necessary in 14 of the 16 mice infused with angiotensin. Average ANG II infusion rates were 70 ng ⋅ kg−1 ⋅ min−1in wild-type, 75 ng ⋅ kg−1 ⋅ min−1in heterozygous, and 68 ng ⋅ kg−1 ⋅ min−1in homozygous mice. Infusion periods ranged from 7 to 12 min. Several minutes after starting the infusion, TGF responses were tested again in the same nephron. In general, only one ANG II infusion period was performed in a given mouse. In two cases, ANG II was infused twice (one +/+ and one −/−), and in two cases, three infusions were made (both −/− mice). When angiotensin was infused more than once, the infusion periods were spaced at least 45 min apart. The perfusion fluid contained (in mM) 136 NaCl, 4 NaHCO3, 4 KCl, 2 CaCl2, 7.5 urea, and 100 mg/100 ml FD&C green (Keystone). At the end of each experiment, we tested the blood pressure responses to intravenous ANG I (10, 50, and 100 ng) and ANG II (1, 3, 5, and 10 ng) to functionally verify the genotype of the animal.
Micropuncture data are expressed as experimental, not tubule, means. Statistical comparisons were made with ANOVA, in association with the Scheffé test or the Student'st-test, using paired or unpaired comparisons, as appropriate.
Experiments were performed in six +/+ mice, seven +/− mice, and four −/− mice. Average body and kidney weights, respectively, were 27.7 ± 0.6 g and 168 ± 3.8 mg in wild-type, 27.6 ± 2 g and 149 ± 14.7 mg in heterozygous mice, and 30.2 ± 0.95 g and 173 ± 12.8 mg in homozygous mice, values not significantly different between genotypes. Mean arterial blood pressure of the anesthetized mice was 99.3 ± 5.4 mmHg in wild-type (78.5–110 mmHg), 90.5 ± 2.9 mmHg in heterozygous (83–102 mmHg), and 79.5 ± 5.9 mmHg in homozygous mice (73–97 mmHg;P = 0.057 compared with wild-type). To determine whether elevated bradykinin levels may contribute to the low blood pressure in ACE-knockout mice, two ACE −/− and two ACE +/+ mice were injected intravenously with 10 μg of the bradykinin B2-receptor antagonist Hoe-140 immediately after cannulation of the femoral vein. No sustained blood pressure effects were observed in Hoe-140-pretreated mice. Blood pressure responses to exogenous ANG I were similar between ACE +/+ and ACE +/− mice but were markedly blunted in ACE −/− animals, providing an independent verification of the ACE-null genotype (Fig. 2). Blood pressure responses to ANG II were not significantly different between ACE +/+, +/−, and −/− animals (Fig. 2).
In response to an increase in loop of Henle flow rate from 0 to 40 nl/min, PSF fell from a mean of 42.3 ± 1.95 to 33.6 ± 2.09 mmHg (mice/tubules = 6/23;P = 0.005) in wild-type mice and from 40.6 ± 2.35 to 38.6 ± 1.93 mmHg in ACE +/− mice (mice/tubules = 7/16; P = 0.014). In homozygous ACE mutant mice, PSFaveraged 36.7 ± 2.02 at 0 loop flow and 36.4 ± 2.01 mmHg at a flow of 40 nl/min, indicating abolished TGF responsiveness (mice/tubules = 4/15; NS). Examples of original recordings obtained in mice with different ACE genotypes are shown in Fig.3.
Maximum PSF responses during both control and ANG II infusion periods were obtained in six nephrons of wild-type, seven nephrons of heterozygous, and nine nephrons of homozygous ACE transgenic mice. Because results were obtained in the same nephrons, comparison between control and angiotensin infusion periods was done by paired t-test. Data are shown in Fig. 4. It can be seen that ANG II infusion caused a significant increase in the TGF response magnitude in all three groups of mice, from 6.5 ± 1.39 to 9.8 ± 1.2 mmHg in +/+ (mice/tubules = 5/6;P = 0.007), from 1.14 ± 0.42 to 4.6 ± 1.3 mmHg in +/− (mice/tubules = 7/7;P = 0.016), and from 0.42 ± 0.25 to 4.02 ± 1.06 mmHg in −/− mice (mice/tubules = 4/9;P = 0.05). Increments in PSF caused by ANG II infusion were identical among the three groups of animals (3.3 ± 0.63 mmHg in +/+, 3.4 ± 1.03 mmHg in +/−, and 3.6 ± 1.1 mmHg in −/− animals), implying that the degree of increase in responsiveness was greater in the transgenic than in the control mice (1.68 ± 0.18-fold in +/+, 2.7 ± 1-fold in +/−, and 4.7 ± 1.5-fold in −/− mice). Even though TGF responses in heterozygous or homozygous mice during ANG II infusion tended to be somewhat lower than those measured during control in wild-type mice, the difference did not achieve statistical significance, suggesting that angiotensin infusion restored responsiveness to near normal levels. During ANG II infusion, PSF, in the absence of loop perfusion, averaged 46.3 ± 2.2 mmHg in +/+ mice, 39.8 ± 2.2 mmHg in +/− mice, and 41.7 ± 2.07 mmHg in −/− mice, values not significantly different from each other (as determined by ANOVA). Mean arterial blood pressure during ANG II infusion averaged 101 ± 5.6 mmHg in +/+ mice (vs. 99.2 ± 5.6 mmHg before the infusion), 89.5 ± 3.1 mmHg in +/− mice (vs. 88.4 ± 3.3 mmHg before ANG II), and 78.7 ± 2.8 mmHg in −/− mice (vs. 77.1 ± 2.8 mmHg before ANG II). Blood pressures were not significantly elevated by ANG II in any of the three groups of mice.
Previous studies from our laboratory have shown that TGF responses are essentially absent in mice homozygous for a null mutation in the AT1A receptor gene (24). The present experiments in ACE transgenic mice complement these previous studies by showing that both homozygosity and heterozygosity for the disrupted ACE gene are associated with a marked reduction in the efficiency of macula densa-mediated feedback control of vascular resistance. The observation that interference with the normal operation of the renin-angiotensin system by two independent genetic interventions causes the same change in the physiological endpoint studied is strong evidence for ANG II being an integral and necessary component of the TGF pathway. It is unlikely that genetic background effects, which would be expected to be different between animals of the AT1A and the ACE mutant strains, are responsible for the strong and systematic influence of functional ANG II deficiency in the juxtaglomerular signaling pathway. Abolition of TGF-mediated effects of luminal NaCl concentration on glomerular hemodynamics in mice homozygous for the disruption in the ACE gene is paralleled by undetectable levels of ACE activity in plasma and by tissue ACE activities in lung, kidney, heart, and brain that approach zero (5, 10, 26). In the same tissues, ACE mRNA expression was found to be extremely low (26). Virtual absence of plasma and tissue ACE activity is supported at the functional level by the almost complete absence of blood pressure responses to intravenously injected ANG I in both the present study and previous observations (26).
Even though only a small number of homozygous ACE mutant animals were studied successfully, we believe that our conclusion of complete TGF blockade in these animals is justified. Results from 16 nephrons in four mice were remarkably homogeneous, with average responses in the four mice being between 0 and 0.3 mmHg. Statistical analysis in this small group of animals is, therefore, not skewed by abnormal responses in one of the mice. Micropuncture experiments in homozygous ACE mutants proved difficult for two main reasons. Consistent with previous observations in conscious mice, most of the −/− animals we attempted to study had blood pressures lower than 65–70 mmHg (5,10, 11, 26). The reliable use of the PSF method, which requires multiple punctures of the same nephron, is virtually impossible at these low pressure levels. Furthermore, previous results in normal rats, as well as in mice, have shown that the TGF response magnitude is directly dependent on blood pressure, with responses becoming essentially undetectable at a level of ∼60 mmHg (20, 27). Thus the effects of pressure per se and of the genetic intervention would be indistinguishable. Preliminary studies with the bradykinin B2-receptor blocker Hoe-140 did not yield evidence that the low blood pressure was due to a tonic blood pressure-lowering effect of elevated bradykinin levels, which one may expect in ACE −/− mice. Enhanced and prolonged blood pressure-lowering effects of bradykinin have been reported in ACE −/− animals in a previous study, but the converse, a blood pressure increase with BK-receptor blockade, was not studied (26). The second problem was related to choosing tubules for study in view of visible inhomogeneities in the appearance of the renal surface. Consistent with the structural abnormalities described earlier (5, 7), the surface of ACE −/− mice showed regions with dilated tubules and cystic tubular enlargements. Although micropuncture was restricted to areas that appeared largely normal, infusion of the perfusate sometimes indicated delays in the clearance of the stained fluid. Thus we cannot exclude the possibility that the absence of feedback-induced vasomotion is, in part, related to the structural abnormalities that have been observed in the renal vasculature (5, 7). Restoration of TGF responses during ANG II infusion to values not significantly different from control would suggest that afferent arterioles have maintained at least some capacity to regulate their tone.
Our present results are in general agreement with several previous studies showing that acute administration of a number of different ACE inhibitors, including captopril, enalapril, teprotide, and CGS-14824, caused a consistent attenuation of TGF responsiveness (8, 18, 22, 25,28). Nevertheless, the degree of TGF inhibition in all of these studies was more modest, with TGF responses being reduced to only 25–50% of control. Even with very high doses of captopril, TGF inhibition did not reach 100% (22). It is likely that the doses utilized in most cases reduced plasma ACE activity to very low levels for the duration of the experiment (4, 9, 12). For example, a single oral dose of 10 mg/kg enalapril has been shown to reduce plasma ACE activity in rats to undetectable levels for longer than 24 h (12). Furthermore, blood pressure responses to ANG I during ACE inhibition were tested in most of the studies referred to above and were found to be largely abolished (8, 18, 25, 28). Because acute and complete ACE inhibition is usually paralleled by a reduction of plasma ANG II concentration to low levels (4, 9), TGF responsiveness does not appear to correlate tightly with plasma ACE activity and plasma ANG II levels. However, modulation of TGF responses by ANG II may primarily depend on the formation of the peptide in the juxtaglomerular interstitium and, therefore, on the activity of renal tissue ACE rather than plasma ACE. It is conceivable that acute administration of ACE inhibitors may inhibit tissue ACE and deplete tissue ANG II levels to a lesser degree than chronic ACE deficiency. Thus it appears that the chronicity of complete ACE inhibition distinguishes the current studies in ACE-knockout mice from the previous studies with pharmacological blockers. Whether long-term adaptations outside the renin-angiotensin system take place that contribute to TGF inhibition remains to be determined. It is possible that chronic ACE deficiency is associated with increases in bradykinin or angiotensin-(1–7) levels in the juxtaglomerular interstitium that may affect TGF responses by nonspecific vasodilatation (4, 29). Furthermore, the contribution of the structural alterations in the homozygous knockout mice to overall vascular reactivity is an issue that requires further exploration.
In view of the inability of even high doses of ACE inhibitors to induce complete inhibition of TGF responses when given acutely, it was somewhat surprising that TGF responses were found to be markedly reduced in heterozygous ACE mutants. In fact, even though loop of Henle flow elevation still caused a significant reduction of PSF, the inhibition of TGF responses in the heterozygous ACE mice appeared greater than that previously observed in mice heterozygous for the AT1A knockout mutation (24). In agreement with earlier observations, blood pressure levels in the +/− animals were not significantly different from those of +/+ animals, even though they tended to be slightly lower (5, 11, 26). Thus heterozygous animals appear to be able to generate sufficient amounts of ANG II to fulfill its role in sustaining blood pressure. Largely normal blood pressure responses to exogenous ANG I in heterozygous animals support the notion of a well-maintained ACE enzymatic activity in plasma and/or endothelial cells. Because blood pressure sustenance may be primarily dependent on plasma ACE, whereas TGF support may mainly come from kidney tissue ACE, it is conceivable that this difference in susceptibility to an ACE reduction in the heterozygous animals may signify a greater suppression of tissue ACE than plasma ACE. However, available data do not strongly support this possibility. Plasma ACE activity has been found to change in a linear fashion when ACE copy number was reduced to 1 or increased to 3 by gene knockout or gene duplication strategies, respectively (11). In agreement with this finding, ACE plasma activity was reduced by ∼50% in the heterozygous ACE animals (5, 10, 26). Similarly, kidney tissue ACE activity, kidney ACE mRNA, and kidney ACE protein expression were also reduced to ∼40–60% of control in ACE mutant heterozygotes (5, 26). It appears that, despite identical reductions in ACE activity, ANG II levels in renal tissue are affected to an extent that limits TGF responsiveness, whereas plasma ANG II is sufficient to support blood pressure. In fact, there is previous evidence to show that the decrease in ANG II levels after the administration of low doses of converting enzyme inhibitors is markedly greater in kidney tissue than in plasma (4, 6). For example, a dose of the ACE inhibitor perindopril, which caused a 50% reduction in ACE activity, did not affect plasma ANG II while it reduced renal tissue ANG II to 40% of control (4). Measurements of ANG II levels in plasma and renal tissue of heterozygous ACE-knockout mice are required to determine if ANG II generation in the kidney is reduced in disproportion to plasma ANG II. Because the appearance of the kidneys in +/− mice was indistinguishable from that of +/+ animals, the reduced TGF activity does not seem to be explainable by changes in renal vascular structure.
The major additional finding in these experiments is the observation that administration of exogenous ANG II caused a restoration of TGF responsiveness to 71% of control in heterozygous and to 62% of control in homozygous ACE mutant mice. Even though TGF responses in the mutant mice during ANG II infusion were not significantly different from those observed in control mice before angiotensin infusion, it appears that full restoration of TGF responsiveness was not achieved. Because arterial blood pressure did not significantly change during the angiotensin infusion period, restoration of TGF responses does not appear to be explained by the hypertensive action of ANG II. Our observations are consistent with previous studies showing that infusion of ANG II in rats treated acutely with enalapril or captopril tended to restore TGF responses without reaching 100% normalization (8, 17). Furthermore, in volume-expanded rats, in which TGF responses were markedly reduced in parallel to a reduction in plasma ANG II concentration, infusion of ANG II caused a restoration of TGF responses to levels normally seen in hydropenic rats (21). It was concluded from these and other results that ANG II sensitizes the TGF target vessels to the constrictor effect of the flow-dependent TGF-mediating agent (3,17, 21). Although in the current studies, the effect of angiotensin was highly significant and consistent, it is noteworthy that full normalization of TGF responses was not achieved. The reason for the tendency of an acute ANG II infusion to not fully correct the response deficit associated with acute or chronic ACE deficiency is unclear. Based on previous determinations of the relation between ANG II infusion rate and plasma ANG II, it seems unlikely that response restoration was limited by insufficiently high plasma ANG II levels (15, 21). Furthermore, the rate of infusion in the present study, as well as in previous studies, was higher than needed to restore TGF responses in the volume-expanded rats (21). These data suggest that the TGF-sensitizing effect of ANG II is only in part dependent on plasma ANG II and needs to be complemented from an extravascular source. In fact, there is strong evidence to suggest that marked differences exist in ANG II levels between the plasma and tissue compartments in the kidney and, most likely, between different regions of the kidney as well (14). The inability to quantitatively restore the functional consequences of chronic ANG II depletion with an acute and short-term infusion may be a consequence of this highly compartmentalized distribution of the renin-angiotensin system in the kidney. It is conceivable that, despite restoration of plasma ANG II concentrations, normalization of peptide levels was not achieved at the site where ANG II interacts with the TGF system, presumably in the juxtaglomerular interstitium.
In summary, marked blunting of TGF responses in mice homozygous or heterozygous for a null mutation of the ACE gene, and restoration of responsiveness in these animals by ANG II, supports the notion that the renin-angiotensin system is a necessary component of macula densa control of renal vascular tone.
This work was supported by National Institutes of Health (NIH) Grants DK-37448 and DK-40042 (to J. Schnermann and J. Briggs), HL-49277 and GM-20069 (to O. Smithies), and HL-03470 (to J. H. Krege). J. H. Krege was a Howard Hughes Postdoctoral Fellow. Support was provided, in part, by the General Clinical Research Center at the University of Michigan, funded by National Center for Research Resources Grant M01-RR-00042.
Present address and address for reprint requests and other correspondence: J. Schnermann, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Bldg. 10, Room 4D 51, 10 Center Dr. MSC 1370 (E-mail: JurgenS{at}intra.niddk.nih.gov).
The costs of publication of this article were defrayed in part by the payment of page charges. The article must therefore be hereby marked "advertisement" in accordance with 18 U.S.C. §1734 solely to indicate this fact.
- Copyright © 1999 the American Physiological Society


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Motorcade cop killed in Florida
A police officer preparing to close a Florida highway for President Obama's motorcade on Sunday was struck and killed by a pickup truck.
The collision occured on I-95 in West Palm Beach ahead of the president's arrival, White House press secretary Jay Carney told reporters traveling with the president. An ambulance from Obama's motorcade sped the officer to a nearby hospital.
"No other vehicle in the motorcade was involved in the accident," Carney said. "As I said, the accident was serious."
The Associated Press identified the officer as Bruce St. Laurent, 55, a 20-year veteran of the Jupiter Police Department.
Carney said the president did not see the accident but was notified of the death. He said "our thoughts and prayers are with the officer's family," the AP reported.


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I volunteered with CAP during 2004- 2005. I had an amazing experience and the organization has truly impacted the people of Eastern KY in a positive manner. CAP provides wonderful programs including housing repair, child care, respite, after school care, adult education, elderly care, and a host of other programs. There is a paid staff that is part of CAP and a board of directors... without which the organization would fall apart. Father Ralph Beiting started the organization 50 years ago and it is a huge undertaking... not everyone can be a volunteer anymore as there is too much work to do and plus... it's providing jobs for people in the area. Please consider volunteering for this organzation... it is a life changing experience.
I have volunteered and continue to do so on a short term basis, in Housing, typically spending 5 months a year with them since 2009. CAP is an excellent organization to volunteer with, and has a direct and effective impact on the lives of those in Appalachia it serves. It also has a life changing impact on the college students who sign up to volunteer each spring on alternative spring breaks, known as Workfest (similar program for high school students, Youthfest).
Like so many charities, it has been severely stressed by the reduction of donations in 2012. It is not alone in this situation, and must try to maintain or solicit new donors to overcome this 25% or so decline.
I cannot say enough about this organization and the opportunity it presents for volunteers to truly engage and impact the needs of the poor in Kentucky.
I participated in Workfest 2005 and 2006. Workfest is an annual gathering of college groups that travel to Kentucky during their spring breaks to help rebuild homes in Eastern Kentucky. I am saddened to see negative reviews because both my experiences were awesome. Workfest was organized and the team leaders were excellent. They provided me and the other volunteers with guidance (most of us had never worked with power tools before this) and they also helped to educate us on some of the problems that people in this area face. It is truly mind-boggling that people can be living in such poverty in this country! The first house my group helped rebuild was owned by an 80-year old woman named Opal. Her walls were insulated with cardboard boxes and her house was basically falling down around her. She was extremely grateful for the work we did and not at all ashamed of her financial situation. We visited with her at the start and end of each work day and she was always thanking and blessing us!
My second experience with WorkFest was just as good as the first. The homeowners were extremely happy we were there and often cooked us traditional southern dishes to show their thanks.
If you want the chance to step outside your comfort zone, learn to use power tools, talk to people whose lives are completely different from yours (even though you live in the same country), meet other volunteers from places all over the US, and see a very beautiful slice of America, I recommend getting involved with CAP.
I started donating small amounts of money years ago to this organization in honor of family members. They treat small donors with the same appreciation of larger ones. After retiring, I decided to increase my donation by becoming a short term volunteer. I have served at WorkFest, YouthFest and Children's Summer Camp. I have observed how the funds are spent. All persons involved are dedicated to stretching the donations, and at the same time having quality programming. I admire what this organization does for the people of Appalachia. They are truly Americans helping Americans---They are 'What's Right with America"
Review from Guidestar


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Information and FAQs regarding FMP
View Single Post
2005-12-19, 00:11
Gomen asobase desuwa!
Join Date: Nov 2003
Age: 36
Copyright: Fujimi Fantasia Bunko, Gatoh Shoji, Shiki Doji
VOLUME 10: "Dounimo Naranai Gori Muchuu"
Original publishing date: 2001.10.25
Category: Side Story Compilations
Jun de Fujun na Grappler
"FMP? Fumoffu" Ep. 6 Part A covers this story
Chidori and Kyoko are running away from some hooligans when a guy saves them with martial arts. The next day, Sousuke and Chidori head to a school clubhouse to persuade their members to leave the premises as it is to be demolished. The head of the martial arts club happens to be the same person who saved Chidori.... Tsubaki Issei makes his first appearance in this story.
Zen'i no Trespass
"FMP? Fumoffu" Ep. 6 Part B covers this story
Sousuke and Issei got off to a wrong start, and they have become rivals at school. Everywhere they go, they wreak havoc, causing stress and pain to the school caretaker, Oonuki Zenji. As punishment, Sousuke and Issei are given orders to help out Mr. Oonuki on his daily activities. Things go wrong when they accidentally prepare Mr. Oonuki's dinner by killing the koi he has taken care of.
Jingi Naki Fancy
"FMP? Fumoffu" Ep. 10 covers this story
The Mikihara yakuza clan is on the brink of extinguishment. Their only remaining hope is to hire a military master (Bonta-kun) to help the inept Mikihara clan members to shape up. When Oren-san and Chidori are kidnapped by the rival Ryuujinkai, Sousuke leads a mass army of Bonta-kuns on a rescue mission.
Houkago no Peacekeeper
This story has not been animated
Chidori and Sousuke are called in to help resolve a quarrel between two rival elementary school "gangs" for their territorial rights of a nearby playzone by Amemiya Takami. Sousuke plays the role of a diplomatic mission, going back and forth between the rival students' headquarters to try to resolve this matter peacefully. However, both ends do not back down on what they want. In the end, Sousuke took his matter to his own hands by blowing up the cause of the quarrel – the playground itself.
Maigo no Old Dog
This story has not been animated
Sousuke helps meets an old man named Komura Shujiro, a former lieutenant in the old Imperial Japanese Navy. Mr. Komura asks Sousuke to help him find a carry-on bag that some hooligans have stolen from him. Due to Sousuke's usual misconceptions, he believes that the bag contains some lethal toxic agent and frantically joins Mr. Komura finding the bag "before things become too late." Little did Sousuke know that Mr. Komura Shujiro is Chidori's grandfather…
Wari to Hima na Sentaichou no Ichi Nichi
"FMP! TSR" OVA covers this story
A diary depicting the everyday life of Teletha Testarosssa's role as a captain of the De Danaan. Journal entries include Tessa being forced to drink alcohol with Mao, getting involved in Kurtz' usual lecherous antics, being lectured by Commander Mardukas, eating Lt. Commander Kalinin's dinner which tastes very great except for his borscht, etc. etc…
Last edited by kj1980; 2009-11-05 at
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PMD provides a wonderful outlet for those who want to volunteer but can't commit to doing so on a regular schedule, and/or who have not selected (or do not want to narrow themselves to) a single specific cause. And it provides a wonderful resource for organizations which need a burst of reliable manpower for a specific task. It can also be a great way to learn about organizations operating in the area, and the causes they support. if you *are* shopping around for a longer-term volunteering gig.
People who volunteer with People Making a Difference (PMD) are super motivated and reliable. They arrive ready to help where they're needed most, and come with tools and manager(s). PMD volunteers make an effort to get to know charities and the issues our clients face. PMD volunteers also contribute financially to help purchase materials when needed (and when they can afford to do so), which makes all the difference.


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Wednesday, May 29, 2013
Photo a Day in May: 29/31
This is my Hello Kitty suitcase where I keep my zine collection. I feel like a Hello Kitty suitcase is an appropriate place to keep a zine collection, no? I got it from a garage sale literally right across the street from where I'm sitting. It was one of those days where I was just like "I should go out an explore more often" and I made it across the street and found this bad boy. I feel awkward going to garage sales by myself, cause if it's a bunch of crap you always feel bad walking away empty handed..so I like bringing a buddy as a buffer. But this garage sale had the good kinda crap so it was a success!
2 comments:
- Remi Germaine said...
Thats adorable! What a steal!
- May 29, 2013 at 9:29 PM
- pigeon said...
so pretty! gee, i'm jealous (of the suitcase AND zine collection!). cute photo!
[new flowery post in my weirdoland - www.pigeongray.blogspot.com]
- May 30, 2013 at 8:24 AM
Subscribe to: Post Comments (Atom)


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spuds 17:13 01 Jul 2011
Mainly for Convenience-- They take your order over the phone and payment at the same time. Quite a number of companies still do this, using trusted employees?.
The newspaper companies that I use, also have a on-line order and secure server payment procedures, especially for out of hours service. Did you inquire about this when you placed your order?.


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When I execute the sencha command I get this error.. I am using the latest version of sencha touch 2.1.0 and Sencha Cmd.
I am totally stuck because I also can't generate an app.. please help! ThanksCode:sencha: line 4: dirname: command not found Sencha Cmd folder (/Users/pietermoeyersons/Desktop/HTDOCS/sencha-touch-2) is missing sencha.cfg - aborting!


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2016 has seen numerous TV programs and television jodis who have made an exceptional place in our heart. From Ek Duje Ke Vaaste's Suman-Shravan to Pardes Mein Hai Mera Dil's Naina-Raghav, among others are the most recent group satisfying couples.
Also Read: Nikita Dutta : Shop When Happy, Shop When Sad, Don't Follow Trends!
Also Read: #THROWBACK – The Top 3 Best Ways To Be 'Sorry' – Taking Tips From Indian TV!
From the affection despise scenes to the lighthearted elements or even the sentimental minutes, TV jodis have made us their fervent fans. Once the romantic tale starts, the on-screen jodi turns into the most cutest match!


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Mendocino County's bounty is hardly limited to its scenic splendors. This is a gourmet mecca as well, with a crab and wine festival that draws crowds every January, the world's largest salmon barbecue in July, and a mushroom extravaganza every November. And the region's scores of wineries, inns and breweries stand ready to slake your thirst and hunger for all things, whether it's a sublime chardonnay, hoppy ale or briny shellfish — or a sumptuous apple cake with a French Laundry pedigree.
Philo Apple Farm's Sally Schmitt owned The French Laundry in the 1980s and early '90s, when her beautiful apple-cranberry kuchen was a mainstay on the menu. The dessert was so popular, Thomas Keller included it in his "French Laundry Cookbook" (Artisan, 1999). Today it's a favorite at Philo Apple Farm, where the cooking school, rustic cottages and heirloom orchards are run by Schmitt's daughter, Karen.
Cooking is a collaborative act at Renata Dorn's Mar Vista Cottages in Gualala, as well. Guests can collect mussels at Anchor Bay Beach, clean them at the fish cleaning station on the property and bake them up in the cottage kitchen — with a recipe provided by frequent guests Susan and Harry Squires.
Here's just a taste of those flavors.
— Jackie Burrell and Ann Tatko-Peterson


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Results 1 to 5 of 5
Thread: mpc files
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11-04-2004 #1
- Join Date
- Aug 2004
- Posts
- 24
mpc files
I have downloaded files from Azureus, these files are all mpc files. How to play this files in AmaroK? http://www.musepack.net This link is where to find the codec but the problem is I do not know how to relate it with my AmaroK.
Thank you for your offers.
p.s.: If there is converter to MP3 which can run under linux, it will work for me too.
I found a prgram MPC2MP3.exe, executed with wine, but the program did not work as I expected
11-04-2004 #2
have you tried xmms? (not to sway you from AmaroK)
with xmms you can install xmms-mp3 xmms-mpc......
11-06-2004 #3
- Join Date
- Aug 2004
- Posts
- 24
I have installed the xmmss' rpm, I can play files, but you know, I can not ./compile.....make....make install anything. I have installed althe g++, cc etc. rpms, anything you can imagine, but this lovely distro does not wil to compile anyting on my pc. Thats why I am planning to pass to Suse
11-12-2004 #4
- Join Date
- Nov 2004
- Posts
- 1
Musepack is being worked into the gstreamer plugins CVS right now, so support is forthcoming.
11-12-2004 #5
- Join Date
- Mar 2003
- Location
- Wisconsin
- Posts
- 1,907
Cevis, I had troubles compliling some programs with MDK 10.1C myself. Try upgrading to 10.1 O.
JeremyRegistered Linux user #346571
"All The Dude ever wanted was his rug back" - The Dude


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Contests & Giveaways
Win tickets to GUIDED BY VOICES at Irving Plaza on 7/11!
June 30, 2014
We have a pair of tickets to giveaway to see GUIDED BY VOICES with CRYSTAL STILTS at Irving Plaza on Friday, July 11th!
For show information and tickets, click here.
Enter your name and email address below for a chance to win. Only one entry per email address will be accepted. Winner will be randomly selected on 7/9. Good luck!


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Things v3.1.5 macOS | 12.3 MB
Languages: English, French, German, Italian, Japanese, Russian,
Simplified Chinese, Traditional Chinese, Spanish
Meet the all-new Things! A complete rethinking of the original, award-winning task manager - with a perfect balance between simplicity and power, incredible new features, delightful interactions, and a timeless new design.
If you're new to Things, this is the basic workflow:
1. Collect Your Thoughts
Get things off your mind quickly: no matter what app you're in, a keyboard shortcut instantly reveals Quick Entry. Type your thoughts and save them to the Inbox. Or just talk to Siri on any device ("Remind me to...") and import from Reminders.
2. Get Organized
Create a project for each of your goals, then add the steps to reach them. For clarity, add structure with headings. Then group your projects by areas of responsibility, such as "Family", "Work", or "Health". Review these regularly to stay on top of things.
3. Plan Your Time
See your calendar events alongside your to-dos and plan your time effectively. Create repeating to-dos for things you do every few days, weeks, or months - Things will remind you on the right day.
4. Make the Most of Your Day
Every morning, grab a coffee and prepare your list for "Today": review previously planned to-dos and make quick decisions on what to tackle. Pick some more steps from your projects and then get going. The Today list is the only place you'll need to look for the rest of the day.
5. Customize Your Workflow
Use tags to categorize your to-dos or add context. For example, tag places like "Office" or "Home", or tag all your "Errands", or everything you're working on with "Kate". You can easily find everything you've tagged via filtering or search.
1. It stays out of your way.
As you start using Things, you'll quickly notice how seamlessly all the features fit together to give you an uncluttered, focused experience. There are no unnecessary frills. No pointless gimmicks or useless controls. In fact, you'll hardly notice the app - it's just you and your to-dos.
2. Everything revolves around your to-dos.
In Things, each of your to-dos are special. In a list, they simply show a checkbox and the title of your to-do. But when you open them, they extend into a beautiful white piece of paper that's ready to hold your thoughts. Additional details (checklists, tags, reminders, deadline) are nicely tucked away in the corner until you need them.
3. It's got a touch of magic.
Things removes friction in magical ways. As an example, take Type Travel, a remarkable new way to navigate your lists. You just start typing where you want to go - the name of any project, area, or to-do - and instantly you're transported there. Or look at the Natural Date Input - just type "Tom(orrow)", "Sat(urday)", "in fou(r days)", "Au(gust 1)"., and it will jump to all the right conclusions.
4. It's got all the power of macOS.
Things is fully integrated with all the latest macOS technologies: Touch Bar, Today Widget, Calendars, Siri & Reminders, Share Extension, Handoff, and AppleScript.
What's New in Version 3.1.5We now support opening new windows via AppleScript on macOS.Made some improvements for the Todoist importer.Made some improvements to the natural language date parser for the Chinese localization.Fixed an issue which prevented setting a to-do's start date to the same date as its deadline.Fixed an issue that could sometimes cause the Where button inside to-dos to display an incorrect state.Fixed an issue where dragging a Someday item with a deadline wouldn't work correctly.Fixed an issue where repeating to-dos might have a distant future deadline after the repeating template was stopped.
Download link:
Links are Interchangeable - No Password - Single ExtractionCode:http://nitroflare.com/view/B5A89B9EB366327/4cwnh.Things.v3.1.5.macOS.dmg


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By Natasha Ofosu
United Kingdom | Friday 5 August 2016: 9:09 BST
It's been a long time coming, but finally one of soca's most consistent exponents is about to release his debut album. Trinidad-born, New York-based singer/songwriter Devon 'Lyrikal' Martin brings together an impressive body of music on his collection, The Journey. The 20 track recording includes an All Access DVD (which follows Lyrikal at Trinidad Carnival), hit digital stores on 22 July. On it you will find fresh material in the form of World's Greatest, his 2016 Crop Over collaboration with King Bubba FM on the Tamu Tamu riddim, along with his Trinidad Carnival offerings Dip and Roll, Unlimited Vibes (with Kes the Band), and the song which took him to the final of the International Soca Monarch competition in February, Freedom. Relive carnivals past through tracks such as All Over De Gyal, 25/8, Ramajay and Conquer Meh, as well as the original and remixed versions of Loner and Cloud 9. Lyrikal says that the album captures the maturing he has undergone in the past ten years. "I chose the name The Journey because the album shows my growth and experiences over the past decade," he said. He explained further, in an interview with New York radio station Hot 97's DJ Young Chow in June, "My journey was a long time coming, and it was a hard-fought journey. But I like things that don't come easy. I like to be the underdog, per se." He added, "When I migrated to the States, to go back to Trinidad it was a kind of hassle and a little difficult because more times they used to say you should be living in Trinidad to sing soca. But, it was no problem for me. I like work and I always used to go back to promote my music. I [kept] going... and I never gave up." Lyrikal was born in Port of Spain, and left for Brooklyn at the age of 13. As a child he sang in the church choir, and was heavily into Jamaican dancehall in his youth and early career, making his own chants and performing in school talent shows and neighbourhood block parties. He began to focus seriously on his recording career in 2003, and caused a stir when he jumped on the 2005 Pot O' Gold riddim produced by Shawn 'Mastamind' Noel; his song was More Gyal. With more than 40 releases since then, Lyrikal has emerged as a singer with a penchant for mellow, danceable grooves and good storylines. He is an accomplished songwriter and deft freestyler, too. In the last two years his music has stepped up a gear and in 2014, for the first time, he reached the final of the International Power Soca Monarch competition in T&T performing Lockdown, his duet with Kerwin Du Bois. He began to receive tangible recognition in November 2015 when Cloud 9 was voted the People's Choice Most Popular Song at the Copyright Organisation of T&T's annual music awards. While The Journey is strictly a soca collection, Lyrikal said he intends to show off his versatility through different genres on his sophomore album. And in the meantime, he plans to release some hip-hop and dancehall mix tapes to do just that. Fans can now purchase their copy of The Journey on iTunes.
Soca News
November 2017


text: Order By: Last Active Most Members Newly Created Alphabetical All Groups 4 Viewing 1 - 4 of 4 groups Achimota Basic 2017 Grp1 Private Course Groupactive 5 months, 3 weeks ago Students of Achimota Basic School forms 3b, 3c and 3e of 2016/17 academic year. Private Course Group / 9 members UNNRULY Private Course Groupactive 5 months, 3 weeks ago CLIQUE Private Course Group / 5 members Coloring Pages Easter Religious Public Groupactive 9 months, 1 week ago Being launched as a Nintendo video game for youngsters, Mario quickly ended up being really prominent amongst the various other animated personalities. Mario coloring pages showcases the adventures and situations […] Public Group / 1 member Akotoshie M/A Private Course Groupactive 1 year ago Akotoshie M/A Basic Private Course Group / 2 members Viewing 1 - 4 of 4 groups


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2012 Financial Statement
04 April 2012
Our 2012 Financial Statement provides an overview of the Haribon Foundation's assets, liabilities, and fund balance.
The Haribon Foundation is a non-stock, non-profit organization incorporated and registered with the Philippines Securities and Exchange Commission (SEC). It is accredited by the Philippine Council for NGO Certification.


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Inspired by the world famous Plaza Hotel on the edge of central park in New York City, this majestic patio collection is the finest available. With a square stock powder coated aluminum frame and UV protected N-dura™ had woven resin wicker, it is not only a thing of beauty it is engineered to heirloom standards.
Summer Classics is a premier manufacturer of outdoor patio furniture and was founded on the principle that truly classic design, no matter how simple or elaborate, can and will be embraced for years. They aspire to create exquisite, timeless patio furniture, which offers a fresh level of sophistication and quality to outdoor living. Often called the "Ralph Lauren" of premier garden furniture, from comfortable deep seating to upscale outdoor dining, the craftsmanship and quality of Summer Classics surely will not disappoint.
- Patented N-Dura All Weather Hand Woven Wicker
- Rust Proof Maintenance Free Aluminum
- Old World Casting Techniques
- Hand Carved Designs
- State Of The Art Finishing Process
- Baked On Powder Coating
- Hand Applied Antique Accenting
- All Weather Indoor / Outdoor Cushions
- Available in Ancient Earth #2
Featured Patio Set Includes: Option (A) Sofa, Lounge Chairs, Swivel Glider, Ottoman, Coffee Table & End Table
Also Available Individually:
- Plaza Sofa # 33852
W 73" x D 34" x H 38" SEAT HEIGHT 18"
- Plaza Love seat # 33824
W 46" x D 33" x H 38" SEAT HEIGHT 18"
- Plaza Swivel Glider # 33892
W 27" x D 27" x H 38" SEAT HEIGHT 18"
- Plaza Lounge Chair # 33872
W 27" x D 27 x H 38" SEAT HEIGHT 18"
- Plaza Ottoman # 33912
W 24" x D 22" x H 13" SEAT HEIGHT 17"
- Plaza End Table # 33862
W 20" x D 20" x H 22"
- Plaza Coffee Table # 33822
W 40" x D 28 x H 28"
- Plaza Arm Chair # 33802
W 22" x D 26" x H 37"
- Plaza Side Chair # 33812
W 21" x D 26" x H 37"
- Plaza Chaise Lounge # 33832
W 31.5" x D 84" x H 47" SEAT HEIGHT 20"
Also Available in a Dining Collection
Plaza Sofa W73"D34"H38"
Plaza Lounge Chair W27"D27"H38"
Plaza Ottoman W24"D22"H13"
Plaza End Table W20"D20"H22"
Plaza Coffee Table W40"D28"H28"
Plaza Swivel Glider W27"D27"H38"
Plaza Chaise Lounge W31.5"D84"H23.5"
Free Shipping
This item requires freight delivery. This shipping method uses a full size semi - truck and product can be delivered to a street side location or into a room of choice based on the level of service requested. A representative will call to schedule your delivery which will require a signature upon receipt. Most delivery times within the Continental U.S. range from 5-7 days on in stock merchandise. Special order patio furniture ranges from 2-6 weeks depending on manufacturer.
Shipping Details & Conditions


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I believe strongly that the U.S. Constitution provides citizens and the states with certain protections from federal intrusion, and have used this belief as the basis for many of my decisions as a representative to the U.S. Congress. Free speech rights, the right to Bear Arms, the protections from warrantless searches and seizures by the government all must be protected, not just by U.S. courts, but by all representatives of the government.
The Patriot Act put a tremendous amount of effort into protecting our homeland from terrorists. With these new protections came a number of new policies that were seen by some as an infringement on our civil rights. The Act gave law enforcement authorities and the intelligence community the flexibility they need to pursue terrorists and bring them to justice. It was intended to do so while continuing to protect the constitutional rights of our citizens. The Patriot Act allows law enforcement to use many of the same tools that have been used to fight organized crime and drug dealers to fight terrorists. Specifically, the Patriot Act allows law enforcement to conduct surveillance against more crimes of terror, such as the use of chemical weapons and other weapons of mass destruction, allows Federal agents to follow sophisticated terrorists trained to evade detection, and allows law enforcement to conduct investigations without tipping off terrorists.
With regards to emminent domain, our Fifth Amendment rights are stated clearly in the Constitution - private property cannot be taken for public use, without just compensation.
Source: http://burgess.house.gov/Issues/Issue/?IssueID=1234


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PROVINCETOWN, Mass. -- After failing in their fifth effort to sedate a right whale entangled in rope, rescuers say they are still not ready to give up.
Scientists returned home early Sunday, after a third day of frustrations.
''Nothing has worked,'' said National Marine Fisheries Service spokeswoman Teri Frady. ''What we have to do now is decide if there's anything else we can try.''
Heavy nylon fishing rope entangled around the whale's jaw has caused a serious infection that veterinarians say is likely to eventually cause its death.
Rescuers said the whale's skin color had faded from black to almost completely white, indicating its condition had worsened.
But Frady said there is hope because the whale is still swimming.
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CLICK HERE to read Mosiah 18.
While King Noah and his people are busy burning Abinadi the prophet at the stake, Alma is busy writing down all that he learned from Abinadi. He really learns it, not only recording his words but also repenting "of his sins and iniquities" (verse 1).
When we truly repent, we also testify.
So Alma picks up where Abinadi left off, "privately among the people, (teaching) the words of Abinadi" (verse 1).
These teachings were "concerning that which was to come, and also concerning the resurrection of the dead, and the redemption of the people, which was to be brought to pass through the power, and sufferings, and death of Christ, and his resurrection and ascension into heaven" (verse 2).
He taught all the people willing to listen, but he taught them privately so they would be out of danger of the king. "And many did believe his words" (verse 3).
Near where they met was a place called Mormon, named after a king in the land that bordered the water (verse 4). This is where Alma taught the people (verse 5), and all who believed him came to this place to hear the teachings (verse 6). He taught them, "and did preach unto them repentance, and redemption, and faith in the Lord" (verse 7).
In this way, the people were prepared for baptism, and they were baptized in the waters of Mormon.
These were their covenants of baptism:
"… as ye are desirous to come into the fold of God, and to be called his people, and are willing to bear one another's burdens, that they may be light; yea, and are willing to mourn with those that mourn; yea, and comfort those that stand in need of comfort, and to stand as witnesses of God at all times and in all things and in all places…" (verses 8-9).
So Alma asked the people:
"… if this be the desire of your hearts, what have you against being baptized in the name of the Lord, as a witness before him that ye have entered into a covenant with him, that ye will serve him and keep his commandments, that he may pour out his Spirit more abundantly upon you?" (verse 10).
The people "clapped their hands for joy, and exclaimed: This is the desire of our hearts" (verse 11).
And so Alma began to baptize the people.
He brought Helam into the waters first, saying "O Lord, pour out thy Spirit upon thy servant, that he may do this work with holiness of heart" (verse 12).
And then he baptized him:
"I baptize thee, having authority from the Almighty God, as a testimony that ye have entered into a covenant to serve him until you are dead as to the mortal body; and may the Spirit of the Lord be poured out upon you; and may he grant unto you eternal life, through the redemption of Christ, whom he has prepared from the foundation of the world" (verse 13).
(Note: prepared from the foundation of the world. Just as Christ was prepared premortally for his mission on Earth, so were each of us also prepared for our purpose in mortality. We were all premortally taught and prepared for making covenants on Earth – and for keeping them.)
When Alma baptized the people, he baptized them by immersion, and they "arose and came forth out of the water rejoicing, being filled with the Spirit" (verses 14 and 15) and they "were filled with the grace of God" (verse 16).
"Grace" is God doing for us what we cannot do for ourselves.
That's why there is rejoicing.
We cannot save ourselves.
We cannot be good.
We cannot do it on our own.
But we can be made perfect in Him, by letting go of what is not of God and embracing who He is.
When He becomes a part of us, when we are filled with His righteousness, then we are made perfect, whole, and complete. It is then that we are at-one… but it is because of what HE has done for us, not what we have done ourselves. We cannot take any credit, but we can rejoice in the confidence in His ability to do in us and for us what He has promised.
"And they were called the church of God, or the church of Christ, from that time forward" (verse 17).
As the church was being established, Alma ("having authority from God" (verse 18)) organized the priesthood as well, so that those ordained as priests could teach the people "the things pertaining to the kingdom of God" (verse 18).
What did they teach?
"…nothing save it were the things which he had taught, and which had been spoken by the mouth of the holy prophets…. nothing save it were repentance and faith in the Lord, who had redeemed his people" (verses 19 and 20).
These are the most important concepts for all of us, because everything else builds upon them.
If we can do these things: repent and have faith in the Lord – continually, then all other problems will be prevented or resolved or we will be strengthened to endure through the experience.
If we can follow the words of the holy prophets, then we will be who we need to be and where we need to be. We will be protected, provided for, and blessed for our efforts and beyond (grace).
If we do these three things: follow the words of the holy prophets, repent, and have faith in the Lord, then we will – together – establish Zion now. We will be at-one with God and at-one with each other, and this is where our power lies.
That is why contention causes so much destruction, because it makes us not-at-one.
"And he commanded them that there should be no contention one with another, but that they should look forward (to celestialness with God)… in unity and in love one towards another" (verse 21).
This is what he taught them, and that is how they established Zion, by becoming "the children of God" (verse 22).
They became the people of Holiness.
And as the people of Holiness, remembering Who made them holy, "he commanded them that they should observe the Sabbath day, and keep it holy, and also every day they should give thanks to the Lord their God" (verse 23).
We cannot keep the Holy Sabbath Day holy without working hard the other days, so that we are prepared for it. Keeping the Sabbath Day holy doesn't start on Sunday morning. It starts the whole week before, when we work hard and play hard on the days intended for those activities. It starts by finishing on Saturday what ought not be done on Sunday. We have to work hard so that we are prepared to take the day off on Sunday, and taking the day off helps us to work hard the rest of the week.
"And he also commanded them… (that they) should labor with their own hands for their support" (verse 24).
We should be as self-reliant as possible, able to use our own resources to gather our own provisions and care for our families in this way.
But then, after our hard week of work, we should rest from that work and use the time to worship.
"And there was one day in ever week that was set apart that they should gather themselves together to teach the people, and to worship the Lord their God…" (verse 25).
Going to Sunday School and Sacrament meeting are so important that they were included in the same chapter as baptism. That's because Sacrament renews our baptismal covenants as this chapter teaches, and then Sunday School answers the "now what" question by studying the covenant so we can live it.
"And (they)… were not to depend upon the people for their support; but for their labor they were to receive the grace of God, that they might wax strong in the Spirit, having the knowledge of God, that they might teach with power and authority from God" (verse 26).
This is living the covenant: working hard to provide for our own temporal needs and those of our family, and working hard to develop our own spiritual strength through knowledge (study that becomes faith that becomes testimony) and then testifying of that knowledge.
All is to be shared with everyone.
We should work hard not only to meet our own temporal needs, but also so that we can share with others. We should work hard not only to develop our own spiritual strength, but also so that we can strengthen others.
"And again Alma commanded that the people of the church should impart of their substance, every one according to that which he had.." (verse 27).
Just as tithing my ten percent means a different actual amount than the people sitting in the next pew over, so does my ability to give differ. I should give what I am able, but that may not be the same as what other people have to give.
This is not to say that I do not have to give as much. I am commanded to be industrious and come up with a way to use what resources I have – and multiply them so that I am able to give more.
In the same way, what I am able to share may different not only in amount but also in what it is I can give. I can give words or teaching, while my friend can give practical compassion. I can give knowledge, while my friend can give understanding. I have food storage and flip flops to share, while my friend has water and socks to share.
That's why we need to be at-one.
"And thus they should impart of their substance of their own free will and good desires towards God, and to… every needy, naked soul" (verse 28).
We are commanded to serve others in both temporal and spiritual ways.
Neither will count without the other.
Our physical presence and practical friendship means nothing if we do not also testify in some way.
Our spiritual gifts mean nothing if we are not also caring for their physical needs.
We must do both.
That is how to establish Zion.
"And this he said unto them, having been commanded of God; and they did walk uprightly before God, imparting to one another both temporally and spiritually according to their needs and their wants" (verse 29).
This is how Alma taught the people in the thicket of trees, the forest, near the waters of Mormon (verse 30), on the borders of the land so that King Noah would not find them and kill them (verse 31).
But the kind did finally find them, and sent spies to watch them (verse 32).
King Noah used this to accuse Alma of "stirring up the people to rebellion against him; therefore he sent his army to destroy them" (verse 33).
Not so different from what happened to Joseph Smith, is it?
But Alma and church were warned of the king's army on its way, "therefore they took their tents and their families and departed into the wilderness" (verse 34), about 450 of them (verse 35).


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City Paradise Trailer (2004)
09 November 2004 Adventure, Animated 6 mins
A young Japanese woman moves to a new city where she doesn't speak the language. Intimidated by her new home, she seeks out common bonds with people who share her love of diving, but ultimately jumps into a unexpected dream-like experience that opens her eyes to a whole new world.
Watch the official City Paradise 2004 trailer in HD below or find more City Paradise videos on Vidimovie.


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WWE Royal Rumble 2008 Trailer (2008)
27 January 2008 Drama, Action 180 mins
Royal Rumble (2008) was the twenty-first annual Royal Rumble PPV. It took place on January 27, 2008 at Madison Square Garden in New York, New York and featured talent from the Raw, SmackDown and ECW brands. The event was also the first WWE pay-per-view broadcast in high definition. As has been customary since 1993, the Royal Rumble match winner received a match at that year's WrestleMania. The main event was the annual 30-man Royal Rumble match, which featured wrestlers from all three brands. The primary match on the Raw brand was Randy Orton versus Jeff Hardy for the WWE Championship. The primary match on the SmackDown brand was Edge versus Rey Mysterio for the World Heavyweight Championship. The featured matches on the undercard were Montel Vontavious Porter versus Ric Flair in a Career Threatening match and John "Bradshaw" Layfield versus Chris Jericho.
Watch the official WWE Royal Rumble 2008 2008 trailer in HD below or find more WWE Royal Rumble 2008 videos on Vidimovie.


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kolagen z elastyną Drilling Brunt Charge May possibly Produce 15% Take away With regard to Philadelphia Many of us suffer pressure in several methods in the same way as entities, even so the solution toward splitting with pressure in point of fact is based on to be able to make out the sort of pressure you're encountering. Li exclaimed Greenpeace motivation promote the online video media from first to last the on the net ducts, also chances it'll engender telephone call proceeding China community media. This implies another epoch something like, you're about to necessitate a lot more mister to create other dopamine to come up with a reduced amount of compensate, and many others, right up until you're taking in a lot regarding mister afterward obtaining virtually naught for this." Straightforwardly elucidated, foods theater company be aware that after they affix glucose, you might be likely to accept a lot more. 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Guitar Port - any1 tell me about it?
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Equipment forum
Sat 19th Apr 2003, 3:35 pm
i.e. is it worth £150 plus the monthly subscription fee which i think is £30? not sure on the fee
anyone know of any good reviews on the net?
nick Edited by: nickhealy at: 4/19/03 4:36:35 pm
Mon 21st Apr 2003, 7:48 pm
excellent for a bit of fun and if you want to learn a lot of other
people's songs and play along to the records, which can be fun from time to time so.......whatever. still, in no way is it essential, it has no appeal for me for instance, it has no live application and just duplicating other stuff gets a bit stale, anyway, depends what you want to do with the guitar. really if you want to do something like that, stick the cd in a hifi, crank it, and play along is just as good, and cheaper too.
Tue 22nd Apr 2003, 1:11 am
£30 a month?!! So by this time next year you will have spent £510 on this device. It would need to be able to do the dishes or something.
Thu 24th Apr 2003, 3:27 pm
sorry, correction, its about a fiver a month (optional)
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website server change
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just a warning:
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