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Landing Page - Event Registration
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I need a simple landing page for an sign up for an event called "Inspired By Prophet Muhammad (p.b.u.h)
I will need a place to fit in a video, some content and the sign up forms .
Landing page will have to tabs for sign up -> I want to watch (name, email, country) or I want to participate(name, age, email, country)
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I wish she'd chosen to partner with a cloth diaper maker instead. I think it's a fabulous idea, but I won't give my money to Pampers.
OMG, Cynthia Rowley Designs Diapers for Pampers!
Does your little bundle of joy need to look chic n' cheap? Do you want them to get used to designer labels early in life? Then good news. Pampers has a new Diaper that they are about to start selling at Target……..designed by, wait for it…Cynthia Rowley. Above is a picture of the stylish design. Quite understated on an infant don't you agree? Will it make the unseemly task of diaper changing take on a new elegance? Who can say. It's early days for this latest innovation from Proctor and Gamble. I'm not a mommy, but I a dying to find out how much these cost. The products will be on Target store shelves and Target.com in July.
These innovative products will bought to market by Pampers maker Proctor and Gamble. This is an interesting move for them, after recent complaints about Pampers from some Mommy bloggers. According to P&G, Pampers by Cynthia Rowley incorporates a beautiful look and feel – giving discerning parents high performance, value and choice of style. The diaper collection, which will be available in pastel designs including madras, stripes and printed ruffles suited specifically to babies and toddlers, delivers the perfect blend of utility and aesthetics. After all babies need fashion choices too. I mean white is so yesterday.
"As a mom, I wanted other moms and dads to have more options in every part of their lives — even diapers," said Rowley. "It's the first piece of clothing your baby will ever wear, and it should be special."
I just don't know about that…are the diapers really "clothing?" What ever happened to the good old cloth diapers that were washed instead of disposed of in the garbage bins and land fills. Now the talents of Cynthia Rowley will join all the other disposable diapers in the trash heap.
What do you think readers? Will you be just waiting to get these Designer Diapers? Or would you rather wear a fashionable designer on your own stylish shelf? So many questions. This one is even more surprising that Liza going to the Home Shopping Network.
5 Comments
LOL, I blogged about the same topic today 🙂
I don't know….there's pros/cons to both cloth and plastic diapers. I wouldn't criticize her for her choice of partnering with Pampers. But it should be interesting to see whether they cost more, and if so, whether consumers will go for it.
since it is summer, I'd probably buy my son some as long as they weren't seriously more expensive compared to regular Pampers…. they'd be cute w/ just a t-shirt when it's super hot out.
Between this, Stella McCartney for Gap, and all the designer duds I see for babies – I want one!!
Minus the hard stuff, that is 🙂
They're going to to be $6 more than a regular box of Pampers, at least that's what I heard.

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Capital Markets
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Alopecia Areata Treatment & Management
- Author: Chantal Bolduc, MD, FRCPC; Chief Editor: Dirk M Elston, MD more...
Approach Considerations
See the treatment algorithm below.
Treatment is not mandatory because the condition is benign, and spontaneous remissions and recurrences are common. Treatments used are believed to stimulate hair growth, but no evidence indicates they can influence the ultimate natural course of alopecia areata. Treatment modalities usually are considered first according to the extent of hair loss and the patient's age.
Assessment of the efficacy of a treatment must be considered with care because the condition is highly unpredictable in presentation, evolution, and response to treatment. Little data exist regarding the natural evolution of the condition. For example, in patients with less than 40% scalp involvement, a study showed no benefit with treatment (minoxidil 1% and topical immunotherapy) over placebo.[19] The high spontaneous remission rate makes clearly assessing the true efficacy of a therapy difficult unless appropriate controls with placebo treatment are studied.
For patients with extensive alopecia areata (>40% hair loss), little data exist on the natural evolution. The rate of spontaneous remission appears to be less than in patients with less than 40% involvement. Vestey and Savin[20] reviewed 50 patients with extensive alopecia areata. Of the 50 patients, 24% experienced spontaneous complete or nearly complete regrowth at some stage during the observation period of 3-3.5 years. The relapse rate is high in patients with severe forms of alopecia areata.
Patients with alopecia totalis or alopecia universalis usually have a poorer prognosis, and treatment failure is seen in most patients with any therapy.
Because alopecia areata is believed to be an autoimmune condition, different immunomodulators have been used to treat this condition. Additional treatment options for alopecia areata include minoxidil and other treatment modalities.
Topical Treatments
Corticosteroid therapies can include intralesional injections or topical application.
Intralesional steroids
For intralesional steroids, few studies are available regarding efficacy; however, they are used widely in the treatment of alopecia areata. Intralesional steroids are the first-line treatment in localized conditions and are usually superior to topical corticosteroids.[21]
In a study including 84 patients, regrowth on treated areas was present in 92% of patients with patchy alopecia areata and 61% of patients with alopecia totalis. Regrowth persisted 3 months after treatment in 71% of patients with patchy alopecia areata and 28% of patients with alopecia totalis. Regrowth usually is seen within 4-6 weeks in responsive patients. Patients with rapidly progressive, extensive, or long-standing alopecia areata tend to respond poorly.
Another study showed regrowth in most patients (480) treated with intralesional steroids, except in 2 patients with alopecia universalis.
Hair growth may persist for 6-9 months after a single injection. Injections are administered intradermally using a 3-mL syringe and a 30-gauge needle.
Triamcinolone acetonide (Kenalog) is used most commonly; concentrations vary from 2.5-10 mg/mL. The lowest concentration is used on the face. A 2015 study showed no difference in regrowth when using 2.5 mg/mL, 5 mg/mL, or 10 mg/mL and all were superior to placebo. Although all concentrations were well tolerated, more cases of reversible skin atrophy were seen in the 10-mg/ml group.[22] The lowest concentration should always be used on the face to avoid skin atrophy. Caution should be used in patients with glaucoma when treating the eyebrows. It may be best to consult with their ophthalmologists.
Less than 0.1 mL is injected per site, and injections are spread out to cover the affected areas (approximately 1 cm between injection sites; see image below).
Adverse effects mostly include pain during injection and minimal transient atrophy (10%). The presence of atrophy should prompt a reduction in the triamcinolone acetonide concentration and avoidance of the atrophic site.
Injections are administered every 4-6 weeks.
Although intralesional injections of triamcinolone acetonide are usually recommended for alopecia areata with less than 50% involvement, a report showed that 6 of 10 patients had regrowth.[23] Although injections may work in extensive alopecia areata, results are unlikely if no response is observed at 6 months (personal observation).
Topical steroids
For topical steroids, again, few studies have been performed regarding efficacy in the treatment of alopecia areata; they can however be useful, especially in children who cannot tolerate injections.
Fluocinolone acetonide cream 0.2% (Synalar HP) twice per day induced a satisfactory-to-excellent response in 61% of patients, which was maintained in 71% of patients. Children younger than 10 years responded better, as did patients with a duration of hair loss of less than 1 year.
Betamethasone dipropionate cream 0.05% (Diprosone) showed similar efficacy.
A 2005 study by Tosti et al[24] in patients with alopecia totalis or alopecia universalis showed that the use of 2.5 g of clobetasol propionate under occlusion with a plastic film 6 d/wk for 6 months induced regrowth in 8 (28.5%) of 28 patients. Regrowth was seen 6-14 weeks after the onset of therapy. Regrowth was maintained for at least 6 months after cessation of therapy in 5 (62.5%) of 8 patients. Even though only 17.8% of patients showed long-term benefits from that treatment, it should be kept in mind that the study was performed in a subgroup of patients that is usually refractory to treatment.
Treatment must be continued for a minimum of 3 months before regrowth can be expected, and maintenance therapy often is necessary.
Despite these data, the authors do not believe that monotherapy with a topical steroid has been of great benefit in the authors' practice.
The most common adverse effect is local folliculitis, which appears after a few weeks of treatment. Telangiectases and local atrophy also have been reported. No systemic adverse effects have been reported.
Topical immunotherapy[1] is defined as the induction and periodic elicitation of an allergic contact dermatitis by topical application of potent contact allergens.
Commonly used agents for immunotherapy include squaric acid dibutylester (SADBE) and diphencyprone (DPCP).[2] These 2 sensitizers are not present in the natural or industrial environment. These are compounded investigational agents not approved by the US Food and Drug Administration for use in alopecia. Dinitrochlorobenzene (DNCB) has become less popular as a result of reports that it is mutagenic in the Ames assay (a bacterial assay).
No rigorous toxicologic and pharmacologic studies have been performed on the use of these agents in humans.
Although DPCP and SADBE have not been found mutagenic in the Ames assay, neither is approved by the US Food and Drug Administration, and unknowns still exist concerning their safety profiles.
No contaminants have been found in SADBE. Acetone solutions and alcohol solutions of SADBE are equally stable for 2 months under storage conditions.
DPCP occasionally can contain mutagenic contaminants; therefore, it should be screened periodically to ensure purity. No formal data are available on DPCP regarding its longevity in solution.
Cosmetically acceptable regrowth with topical immunotherapy rates in patients with severe alopecia areata (>50% involvement) varies from 22-68%. Most studies have a success rate of 30-50%. Wiseman et al[25] retrospectively reported the results of a large cohort of 148 consecutive patients treated with DPCP.
Their analysis showed that the cumulative patient response at 32 months was 77.9%. The response rate varied with the extent of the alopecia. Cosmetically acceptable regrowth was seen in 17.4% of patients with alopecia totalis or alopecia universalis, 60.3% in patients with 75-99% hair loss, 88.1% in patients with 50-74% hair loss, and 100% regrowth in those with 25-49% hair loss.
Age at onset was also a significant variable, with older age at onset leading to a better prognosis. A lag period of 3 months was usually present between the onset of therapy and the presence of regrowth.
The median time to achieve significant regrowth was 12.2 months. Some patients showed regrowth on the treated side after 18 months of therapy. No benefit is achieved with continuing therapy after 24 months in the absence of regrowth. The relapse rate after reaching significant regrowth was 62.6%.
In a report of a 5-year experience with the use of DPCP, 97 subjects received continued therapy with DPCP. A response rate of greater than 75% was seen in 15% at 6 months, 49% at 12 months, 53% at 18 months, and 56% at 24 months. The only variable that seemed to affect response to treatment was the baseline extent of the alopecia areata. A greater than 75% response rate was seen in 100% of patients with 25-49% hair loss at baseline, 77% of those with 50-74% loss at baseline, 54% of those with 75-99% loss at baseline, 50% of alopecia totalis patients, and 41% of alopecia universalis patients demonstrated a response. Maintenance treatment (once every 1-4 wk) appeared to reduce the risk of relapse (>25% hair loss). Only 18% of patients experienced relapse on maintenance therapy, compared with 57% in those who discontinued treatment.[26]
The type of alopecia areata before treatment, duration of the disease, and the presence of nail changes were found to predict a lower response to treatment. Age at onset and sex of the patient do not appear to influence the prognosis. Controversy exists concerning whether atopy is an adverse prognosis factor.
Topical immunotherapy has been used for almost 20 years; no serious adverse effects have been reported.
The most common side effect, which is desired, is a mild contact dermatitis (redness, scaling, itching).
Adverse effects include cervical lymphadenopathy and pigment changes. Vitiligo developed on the application site in 6.7-7.5% of patients. Transient leukoderma on a distant untreated area has been reported. Of patients who develop vitiligo, 31% (4 of 13) had a history of vitiligo. Only 0.75% of patients developed hyperpigmentation. Confetti-type dyschromia (ie, hyperpigmentation, hypopigmentation) has been described as an adverse effect of DPCP treatment and occurred in 1.6% of 243 patients treated. Less common adverse effects include erythema multiforme–like eruptions and urticaria, which were reported in 3 patients treated with DPCP.
The mechanism of action of topical immunotherapy is unknown. Antigenic competition has been hypothesized. That is, the introduction of a second antigen can initiate a new infiltrate containing T-suppressor cells and suppressor macrophages that may modify the preexisting infiltrate and allow regrowth.
Because topical immunotherapy involves the production of contact sensitivity in a previously naive patient, it is best to seek approval for the treatment by the ethics review board and to have the patient sign an informed consent.
Both SADBE and DPCP appear to be equally effective. Acetone-based solutions usually are preferred because they evaporate quickly, allowing patients to wear a hat or wig immediately after treatment. Quick drying also decreases the chances of dissemination to other body parts by contact.
Treatment is provided weekly.
The patient first is sensitized directly on the scalp with a 2% concentration on a small area (2 cm).
The following week, a low concentration (0.0001%) is applied.
The concentration is increased slowly every week as needed until a mild tolerable allergic contact dermatitis is elicited. Many concentrations are available that achieve this goal.
Treating only half of the head allows the physician to use the untreated half as a control. Once regrowth occurs on the treated half, treatment can be applied to the entire scalp. If regrowth initially occurs on both sides, spontaneous remission is likely, although treatment cannot be excluded as the cause.
Avoid severe contact dermatitis. Patients are advised to avoid light exposure on the scalp for 48 hours because light degrades the chemical. Patients also are advised not to wash the scalp for 48 hours.
Initial regrowth may be seen at weeks 12-24. Once cosmetically acceptable regrowth is achieved, the treatment can be tapered gradually. Almost all patients relapse if the treatment is discontinued, and maintenance treatment is needed.
The efficacy of anthralin was assessed in 3 studies, which unfortunately were uncontrolled.
Both short-contact and overnight treatments have been used. Anthralin concentrations varied from 0.2-1%.
A 2004 study by Tang et al[27] showed no benefit in using anthralin. Other studies showed a response rate of 20-75%, respectively, for patchy alopecia areata and a 25% response rate for alopecia totalis. The mean time to response was 11 weeks, and the mean time to cosmetic response was 23 weeks. Anthralin was used by Tang et al in balding C3H/HeJ mice, which is one animal model for alopecia areata. Half the body was treated with anthralin 0.2%, while the other side was treated with the vehicle ointment. Regrowth was seen on the treated side in 64% of mice after 10 weeks. Four mice had almost complete regrowth. The untreated side showed either no regrowth or continued hair loss. Cytokine studies performed with an RNase protection assay showed that tumor necrosis factor-alpha and -beta were inhibited in mice that responded to treatment.
Most patients experienced irritant contact dermatitis. Whether the dermatitis is necessary for efficacy remains under debate.
Cosmetically acceptable regrowth was maintained during therapy in 71% of responders. No correlation exists between duration of the current episode and response to treatment.
Adverse effects include pruritus, erythema, scaling folliculitis, local pyoderma, and regional lymphadenopathy. Withholding treatment for a few days results in rapid disappearance of adverse effects. Treatment then can be reinstituted, but anthralin should be left on for shorter periods. Staining of clothes and skin can be a concern.
The mechanism of action of anthralin is unknown. Most likely, it creates inflammation by generating free radicals, which have antiproliferative and immunosuppressive actions.
Combination of DPCP and anthralin
A small retrospective study showed better results using both DPCP and anthralin compared with DPCP alone.[28]
Minoxidil appears to be effective in the treatment of alopecia areata in patients with extensive disease (50-99% hair loss). Response rates in that group vary from 8-45%. Minoxidil was of little benefit in patients with alopecia totalis or alopecia universalis.
The 5% solution appears to be more effective.
No more than 25 drops are applied twice per day regardless of the extent of the affected area.
Initial regrowth can be seen within 12 weeks, but continued application is needed to achieve cosmetically acceptable regrowth.
Minoxidil usually is well tolerated. Adverse effects include distant hypertrichosis (5%) and irritation (7%).
The exact mechanism of action of minoxidil remains unclear. Minoxidil does not appear to have either a hormonal or an immunosuppressant effect. Minoxidil most likely has a direct mitogenic effect on epidermal cells, both in vitro and in vivo. Anagen-phase hair bulbs plucked from men applying minoxidil showed a significant increase in proliferation index as measured by DNA flow cytometry. Minoxidil also has been shown to prolong the survival time of keratinocytes in vitro. Finally, minoxidil may oppose intracellular calcium entry. Calcium influx normally enhances epidermal growth factors to inhibit hair growth. Minoxidil is converted to minoxidil sulfate, which is a potassium channel agonist and enhances potassium ion permeability, thus opposing the entry of calcium into cells. Local vasodilatation does not appear to play a primary role in hair growth associated with minoxidil.
Prostaglandin analogs
Retrospective studies using either latanoprost or bimatoprost showed some regrowth of the eyelids while using intralesional triamcinolone concomitantly on the scalp and eyebrows,[29, 30] but all prospective studies using either drugs did not show statistically significant changes when used to treat either the eyelids or eyebrows.[31, 32, 33, 34]
Systemic Treatments
Psoralen plus UV-A
Many studies have been performed regarding the efficacy of psoralen plus UV-A (PUVA) in the treatment of alopecia areata, and the initial response rate varies from 20-73%. The relapse rate, unfortunately, is high (50-88%). Most patients relapse within a few months (mean 4-8 mo) after treatment is stopped.
Both systemic and topical PUVA therapies have been used.
The number of treatments required for regrowth varies, but 20-40 treatments usually are sufficient in most cases.
A younger age at onset, a longer duration of disease, and the presence of alopecia totalis or alopecia universalis appear to indicate a poorer outcome. Taylor and Hawk[35] published 10 years of experience with PUVA. The initial response rate (>90% regrowth) was comparable to other studies and was 43.8% for partial alopecia areata and 50% for alopecia totalis and alopecia universalis. However, after excluding patients with vellus hair regrowth and patients who relapsed rapidly in the follow-up period (approximately 4 mo), they found the success rate to be, at best, 6.3% for partial alopecia areata and 12.5% for alopecia totalis and alopecia universalis. They concluded that PUVA generally is not an effective long-term treatment for alopecia areata. PUVA is a relatively safe treatment modality; adverse effects include burning and, possibly, an increased risk of skin cancer.
The use of systemic steroids for the treatment of alopecia areata is sometimes justifiable, but hair loss frequently follows discontinuation of the medication and benefits must be carefully weighed against long-term risks. Some authors support a beneficial role of systemic steroids in halting the progression of alopecia areata,[36] but many others have had poor results with this form of therapy.
The rate of regrowth varies greatly (27-89%), and many dose regimens have been used in these studies.
Although the initial regrowth appears promising, the prednisone dose necessary to maintain cosmetic growth usually must be high enough that adverse effects are inevitable, and most patients relapse after therapy is discontinued.
Some benefit was shown using minoxidil 2% solution applied twice per day following a 6-week taper of prednisone, but the relapse rate remained at a minimum of 50% at 4 months in the treated group.
Adverse effects from systemic therapy were common in these reports and included diabetes, weight gain, hypertension, psychological changes, osteoporosis, suppression of the adrenocorticotropic axes, striae, acne, hypertrichosis, and purpura.
Systemic steroids most likely are effective via their immunosuppressive effects.
An initial benefit may occur by using systemic prednisone in some patients, but the relapse rate is high, and it does not appear to alter the course of the condition.
Systemic prednisone is not an agent of choice for alopecia areata because of the adverse effects associated with both short- and long-term treatment.
Methylprednisolone administered at a dose of 500 mg/day for 3 days or 5 mg/kg twice a day over 3 days has been used in patients with widespread disease causing severe emotional distress.[37, 38] Risks and benefits of systemic steroid therapy must be weighed carefully. Predictors of response include disease duration of 6 months or less, age younger than 10 years at disease onset, and multifocal disease.
Cyclosporine has been used both topically and systemically in the treatment of alopecia areata.
Topical cyclosporine has not proven to be effective in severe alopecia areata because no patient (0 of 10) showed benefit with application of a 10% cyclosporin A (CsA) solution twice per day for 12 months.
Another study of 14 patients using a 5% solution of cyclosporine twice per day for 4-6 months reported vellus growth in 3 of 14 patients and normal hair growth in 3 patients with patchy alopecia areata. No regrowth was seen in 8 of the patients.
Neither study showed systemic absorption of CsA, and routine blood examination showed only a transient increase of hepatic enzymes in 1 patient.
Oral cyclosporine was effective in the DEBR model for alopecia areata. All rats had a full pelage by 5 weeks of treatment with 10 mg/kg/d, 5 d/wk for 7 weeks. Studies in humans also have proven efficacy with doses of 6 mg/kg/d for 3 months in 6 patients. All patients experienced regrowth, and cosmetically acceptable regrowth was seen in 3 of 6 patients. Unfortunately, all patients relapsed within 3 months of discontinuation of cyclosporine. No evidence indicates that CsA can prevent hair loss during an active episode because reports have described patients taking CsA who developed alopecia areata while they were under treatment for unrelated conditions.
The mechanism of action of cyclosporine remains unclear. It may act through its immunosuppressive effect, because, in patients who regrew hair, clearance of immune cells from the hair follicles and alteration in the balance of regulatory lymphocytes occurred (ie, decrease of the CD4/CD8 ratio). Cyclosporine causes hypertrichosis in patients treated for conditions unrelated to hair loss. The mechanism by which cyclosporine stimulates hair growth remains unknown.
In conclusion, topical cyclosporine has shown limited efficacy. Although systemic CsA appears to be effective in alopecia areata, the adverse effect profile, the recurrence rate after treatment discontinuation, and thus, the inability to produce long-term remissions, make CsA unattractive for the treatment of alopecia areata.
Regrowth was shown on the application site of topical tacrolimus in 2 studies using the DEBR model. Oral tacrolimus was ineffective. No benefit was shown in the use of topical tacrolimus for alopecia areata in a small 2005 study by Price et al that included 11 patients.[39]
A study of 11 patients with alopecia areata ranging from patchy alopecia areata to alopecia universalis showed no benefit using intralesional interferon alfa-2 (1.5 million IU, 3 times per wk for 3 wk).
Dapsone at 50 mg twice per day was used in a 6-month, double-blind, placebo-controlled study. Of patients in the study, 54% (7 of 13) withdrew from the dapsone group because of adverse effects such as malaise. Of the remaining 6 patients, 3 experienced generalized growth of terminal hair, compared with 4 (4 of 13) patients in the placebo group, who experienced only sparse patchy regrowth of vellus hair. The authors concluded that although dapsone showed some efficacy, the high incidence of adverse effects rendered it unacceptable. Another study showed a rate of success comparable to the occurrence of spontaneous regrowth reported in the literature.
Joly[40] reported 22 patients with long-standing, severe alopecia areata who responded well to methotrexate, with or without systemic corticosteroids. Although the results from that study are surprisingly good, a more standardized study involving more patients is needed because other dermatologists have not had such good efficacy with methotrexate. A 2016 study suggests that long-term maintenance treatment is usually required to maintain hair growth.[41] Risks versus benefits must be carefully weighed.
Other Treatment Modalities
Many other modalities have been reported to have variable response rates in small studies. These include latanoprost,[42] nitrogen mustard, massage and relaxation, isoprinosine, acupuncture, and aromatherapy, among others. The efficacy of these treatments needs to be demonstrated in larger, placebo-controlled trials before they can be recommended.
Biological agents
Data are mixed, with many prospective, randomized placebo controlled studies[43, 44] and case reports published in the last few years regarding the use of biologic agents (including adalimumab, alefacept, etanercept and infliximab) in the treatment of alopecia areata, failing to show efficacy, and some patients developed alopecia areata while under treatment with biologic agents for other conditions.
There are only case reports regarding the use of tofacitinib and baricitinib in the treatment of alopecia areata.[45, 46] Clinical trials are ongoing to assess the true efficacy of tofacitinib.
A prospective, open-label pilot study using simvastatin/ezetimibe showed some response that seemed to be maintained while taking the drug, but most relapsed once it was discontinued.[47] Only 19 of 29 patients completed the study. One patient was noncompliant, but reasons for lost to follow-up or withdrawal are not specified for the other patients.
Platelet-rich plasma (PRP)
PRP has both proliferation-inducing as well as anti-inflammatory effects. A small randomized, double-blind, placebo- and active-controlled, half-head study has shown PRP to be superior to both intra-lesional triamcinolone acetonide and placebo.[48]
One study showed a correlation between low serum zinc levels and disease severity, as well as duration and resistance to therapies,{ref} 26147750 anda few reports show some benefit to using zinc gluconate (30-50 mg/day) in the treatment of alopecia areata.[49, 50] However, another study did not find a statistically significant difference in zinc concentration in serum and hair between alopecia areata patients and controls.[51]
Nonpharmacologic methods
A systematic MEDLINE search could not find any study with sufficient validity to provide scientific evidence of benefit with complementary and alternative medicine therapies for alopecia areata.[52]
A study on hypnosis for refractory alopecia areata did not show efficacy of regrowth, but it did show that hypnosis can improve depression, anxiety, and quality of life in affected patients.[53]
Cosmetic treatments for patients with alopecia areata include dermatography and hairpieces. Dermatography has been used to camouflage the eyebrows of patients with alopecia areata. Follow-up visits at 4 years showed that 30 of 39 of patients demonstrated excellent cosmetic results and 3 had good results. On average, 2-3 sessions lasting 1 hour each were required for each patient. No adverse effects were reported. Hairpieces are useful for patients with extensive disease and allow them to carry on their usual social life. Reassure patients about the natural look provided by hairpieces.
Surgical Care
Surgical intervention has no role in the treatment of alopecia areata.
Alopecia areata is highly unpredictable. No treatment is effective in preventing or halting progression of the condition. No trigger can be found to explain disease exacerbation in most patients.
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Devi M, Rashid A, Ghafoor R. Intralesional Triamcinolone Acetonide Versus Topical Betamethasone Valearate in the Management of Localized Alopecia Areata. J Coll Physicians Surg Pak. 2015 Dec. 25 (12):860-2. [Medline].
Chu TW, AlJasser M, Alharbi A, Abahussein O, McElwee K, Shapiro J. Benefit of different concentrations of intralesional triamcinolone acetonide in alopecia areata: An intrasubject pilot study. J Am Acad Dermatol. 2015 Aug. 73 (2):338-40. [Medline].
Chang KH, Rojhirunsakool S, Goldberg LJ. Treatment of severe alopecia areata with intralesional steroid injections. J Drugs Dermatol. 2009 Oct. 8(10):909-12. [Medline].
Tosti A, Piraccini BM, Pazzaglia M, Vincenzi C. Clobetasol propionate 0.05% under occlusion in the treatment of alopecia totalis/universalis. J Am Acad Dermatol. 2003 Jul. 49(1):96-8. [Medline].
Wiseman MC, Shapiro J, MacDonald N, Lui H. Predictive model for immunotherapy of alopecia areata with diphencyprone. Arch Dermatol. 2001 Aug. 137(8):1063-8. [Medline].
El-Zawahry BM, Bassiouny DA, Khella A, Zaki NS. Five-year experience in the treatment of alopecia areata with DPC. J Eur Acad Dermatol Venereol. 2010 Mar. 24(3):264-9. [Medline].
Tang L, Cao L, Sundberg JP, Lui H, Shapiro J. Restoration of hair growth in mice with an alopecia areata-like disease using topical anthralin. Exp Dermatol. 2004 Jan. 13(1):5-10. [Medline].
Durdu M, Özcan D, Baba M, Seçkin D. Efficacy and safety of diphenylcyclopropenone alone or in combination with anthralin in the treatment of chronic extensive alopecia areata: a retrospective case series. J Am Acad Dermatol. 2015 Apr. 72 (4):640-50. [Medline].
Vila TO, Camacho Martinez FM. Bimatoprost in the treatment of eyelash universalis alopecia areata. Int J Trichology. 2010 Jul. 2 (2):86-8. [Medline].
Coronel-Pérez IM, Rodríguez-Rey EM, Camacho-Martínez FM. Latanoprost in the treatment of eyelash alopecia in alopecia areata universalis. J Eur Acad Dermatol Venereol. 2010 Apr. 24 (4):481-5. [Medline].
Ross EK, Bolduc C, Lui H, Shapiro J. Lack of efficacy of topical latanoprost in the treatment of eyebrow alopecia areata. J Am Acad Dermatol. 2005 Dec. 53 (6):1095-6. [Medline].
Faghihi G, Andalib F, Asilian A. The efficacy of latanoprost in the treatment of alopecia areata of eyelashes and eyebrows. Eur J Dermatol. 2009 Nov-Dec. 19 (6):586-7. [Medline].
Roseborough I, Lee H, Chwalek J, Stamper RL, Price VH. Lack of efficacy of topical latanoprost and bimatoprost ophthalmic solutions in promoting eyelash growth in patients with alopecia areata. J Am Acad Dermatol. 2009 Apr. 60 (4):705-6. [Medline].
Ochoa BE, Sah D, Wang G, Stamper R, Price VH. Instilled bimatoprost ophthalmic solution in patients with eyelash alopecia areata. J Am Acad Dermatol. 2009 Sep. 61 (3):530-2. [Medline].
Taylor CR, Hawk JL. PUVA treatment of alopecia areata partialis, totalis and universalis: audit of 10 years' experience at St John's Institute of Dermatology. Br J Dermatol. 1995 Dec. 133(6):914-8. [Medline].
Sharma VK. Pulsed administration of corticosteroids in the treatment of alopecia areata. Int J Dermatol. 1996 Feb. 35(2):133-6. [Medline].
Acikgoz G, Ozmen I, Cayirli M, Yeniay Y, Kose O. Pulse methylprednisolone therapy for the treatment of extensive alopecia areata. J Dermatolog Treat. 2014 Apr. 25(2):164-6. [Medline].
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Price VH, Willey A, Chen BK. Topical tacrolimus in alopecia areata. J Am Acad Dermatol. 2005 Jan. 52(1):138-9. [Medline].
Joly P. The use of methotrexate alone or in combination with low doses of oral corticosteroids in the treatment of alopecia totalis or universalis. J Am Acad Dermatol. 2006 Oct. 55(4):632-6. [Medline].
Anuset D, Perceau G, Bernard P, Reguiai Z. Efficacy and Safety of Methotrexate Combined with Low- to Moderate-Dose Corticosteroids for Severe Alopecia Areata. Dermatology. 2016. 232 (2):242-8. [Medline].
Ross EK, Bolduc C, Lui H, Shapiro J. Lack of efficacy of topical latanoprost in the treatment of eyebrow alopecia areata. J Am Acad Dermatol. 2005 Dec. 53(6):1095-6. [Medline].
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Strober BE, Siu K, Alexis AF, Kim G, Washenik K, Sinha A, et al. Etanercept does not effectively treat moderate to severe alopecia areata: an open-label study. J Am Acad Dermatol. 2005 Jun. 52(6):1082-4. [Medline].
Craiglow BG, King BA. Killing two birds with one stone: oral tofacitinib reverses alopecia universalis in a patient with plaque psoriasis. J Invest Dermatol. 2014 Dec. 134 (12):2988-90. [Medline].
Jabbari A, Dai Z, Xing L, Cerise JE, Ramot Y, Berkun Y, et al. Reversal of Alopecia Areata Following Treatment With the JAK1/2 Inhibitor Baricitinib. EBioMedicine. 2015 Apr. 2 (4):351-5. [Medline].
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Trink A, Sorbellini E, Bezzola P, Rodella L, Rezzani R, Ramot Y, et al. A randomized, double-blind, placebo- and active-controlled, half-head study to evaluate the effects of platelet-rich plasma on alopecia areata. Br J Dermatol. 2013 Sep. 169 (3):690-4. [Medline].
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Lux-Battistelli C. Combination therapy with zinc gluconate and PUVA for alopecia areata totalis: an adjunctive but crucial role of zinc supplementation. Dermatol Ther. 2015 Jul-Aug. 28 (4):235-8. [Medline].
Dastgheib L, Mostafavi-Pour Z, Abdorazagh AA, Khoshdel Z, Sadati MS, Ahrari I, et al. Comparison of zn, cu, and fe content in hair and serum in alopecia areata patients with normal group. Dermatol Res Pract. 2014. 2014:784863. [Medline].
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Willemsen R, Haentjens P, Roseeuw D, Vanderlinden J. Hypnosis in refractory alopecia areata significantly improves depression, anxiety, and life quality but not hair regrowth. J Am Acad Dermatol. 2010 Mar. 62(3):517-8. [Medline].
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Hussian College Open House - Apr. 8, 2017
Hussian's Open House
Open House at Hussian College, School of Art provides students and their families the opportunity to visit campus and discover more about our Bachelor of Fine Arts program.
Hussian offers four years of professional development that enables you to be confident and competent as you embark on your career journey in the visual arts. We welcome you to bring guests and if you choose, your artwork to share with the Hussian family. Admissions and career representatives will be available to answer questions about Hussian's program.
- Date and Time
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Hussian College, School of Art
Suite 300, The Bourse • 111 S. Independence Mall East
Philadelphia, PA
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Luke Cage
Separate names with a comma.
its not up for my ps3 either
yes you can watch it here
i hate that
Is there a way to load live rosters on to your offline franchise?
please help
this sounds dumb but who are the unlockables again?
yes i hope they put in a i quit match
no i would guess that he would be in the first downloadable roster update
yes its confirmed by the designer its like last year
thats what i want dont you toyy
Please Rate! tell me the ones i missed [face_grin]
Alicia Fox
Beth Pheonix
Big Show
Chavo Guerrero
Chris Masters
today is the big reveal of WWE SVR 2010 at 3:00
Go to http://www.smackdownvsraw.com/
What new matches do u want to have in 2010?
"I Quit" Match
Steel Cage
Barbed Wire Steel Cage
Empty Arena Match
Falls Count Anywhere
Flag Match
Handicap Match
SHAWNDAMIAN Has great ideas
if those are put into the next game it will be the top wrestling game of the year no doubt
I think it should be Matt Ryan
NBA live 09 no doubt
this is a great game this is the most realistic nba game ever
how do u lure rosco away from the gate
i knocked over trash cans and i tried to knock over the statue but it doesnt work
this game sucks on ps2 it is great on psp the graphics are extremely better and there is alot more mini games
do u agree?
do u do these in career mode or exibition
do u do this in career mode or exhibition


text:
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- Product:
- Stone-leader Beige Floor tile
- Family:
- Stone-leader
- Manufacturer:
- Refin
- Architonic ID:
- 1130327
- Country:
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Product description
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Richardson TX Real Estate & Homes for Sale
53 Properties Found
The median home value in Richardson, TX is $208,100.
This is
higher than
the county median home value of $115,300.
The national median home value is $177,414.
The average price of homes sold in Richardson, TX is $208,100.
Approximately 58.44% of Richardson homes are owned,
compared to 36.04% rented, while
5.53% are vacant.
Richardson real estate listings include condos, townhomes, and single family homes for sale.
Commercial properties are also available.
If you see a property you're interested in, contact a Richardson real estate agent to arrange a tour today!
Sort Results:
1 - 25 of 53 Properties Displayed
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Courtesy: Keller Williams Realty Plano Marybeth Tiemeyer
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Cul-de-sac location. One owner beautifully maintained. Updates include nailed down hardwood floors, recent carpet, updated master bath tiles, marble vanity top and floor in guest bath. Walk in wet bar, 2 stair cases, two fireplaces. 2nd bedroom downstairs, currently use as study. Large covered patio with flagstone floors. B on B fence. A wonderful home. Must see!
Courtesy: Ebby Halliday, Realtors McK Jackie Schor
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Fabulous hardwoods: Entry, Formal dining, Family Room, Study, Master and hallway. Full wall of windows from family room, offer view of private rear yard, patio and deck. Split spacious Master,oversized walk-in closet, huge jetted tub, separate shower. Kitchen features cooking island with seating, breakfast area, coffee bar, walk-in pantry. Study off the entry features built-ins. FRIDGE,WASHER,DRYER,GAS GRILL REMAIN. NO SURVEY. Close to Dart,PGB
Courtesy: Jck Realty Joe Killingsworth
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Courtesy: Texasbesthomes.Net Suzanne Burns
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Gorgeous! Hand scraped hardwood floors, patterned concrete floors in kit. 1 half Acre with plenty of yard & pool, spa, slide, dive board. Fish pond. Granite counterops, bathrms are updated, as well. Enclosed patio. Loads of parking & drive thru front gate to back with electronic gate. Many copper hammered sinks. Wrought iron ballustrade on stairway with Juliet balcony. 3 Fireplaces.Seller having pool resurfaced. Home Warranty.
Courtesy: Keller Williams Central Christian Dao
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Beautifully REMODELED!!! 5 Bedroom, 4 Bathroom home located in the Wyndsor Estates subdivision. With PLENTY of room this house boasts 2 living areas, with one on the first and second floor. This house is loaded with upgrades and is ready for you to call it home!
Courtesy: Virea Realty David Maisch
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Amazing home in coveted Creek Hollow Estates, wood floors, plantation shutters, and granite thru-out. Apartment sized master retreat with custom closet. Kitchen has brand new designer SS appliances. Or cook outside while the kids play in the pool or spa. Large game-media room upstairs. close to GBT and Breckinridge Park. Walk to Stinson Elem.
Courtesy: William Davis Realty Frisco Binh Nguyen
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Beautiful one story home on corner premium lot in desirable Richardson neighborhood with award winning PISD Schools. New roof in 2016 with warranty and new fence.Tile in entry way and laminate hardwoods throughout the rest of home. Kitchen is updated with granite countertops and c-tile backsplash plus white cabinets make this the modern dream kitchen. Home has gutters and sprinklers in the lavishly landscaped backyard
Courtesy: Novus Real Estate Shyh-Chang Kou
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GORGEOUS, SPACIOUS, LIGHT, BRIGHT, solid and OPEN 4-beds estate available in a scenic area and in a highly rated school district. New hardwood floor, new roof, new paint in and out, new belt drive garage opener. Next to Breckenridge park can see July 4 firework at home. Some of the recent updates include kitchen granite counter top, water heaters, board on board cedar fence. Check out the room size, kitchen, media room and big size back yard!
Courtesy: Re/Max Dallas Suburbs Katie Laquey
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Backyard Oasis in Plano ISD! Spacious kitchen with granite counters, custom cabinets, tumbled marble backsplash, undercabinet lighting, sunroom, media room & large game room with balcony. Large master suite with sitting area, dual sinks, separate tub & shower. Outdoor kitchen with granite counters, sink & refrigerator overlooking pool with extensive deck, beautiful landscaping, oversized 3 car garage, 8' BOB fence with automatic gate & opener.
Courtesy: Keller Williams Central Scott Bernard
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Lush landscaping invites you into the foyer with soaring ceilings, split formals provide options, there is a second bedroom down with a full bath off the hallway. Family room opens to the kitchen, stone fireplace and views to a beautiful back yard with pool, spa area. Kitchen offers ss appliance package. Master shower remodeled. Second floor has a centered game room, two bedroom share a Jack & Jill bath, third bedroom has its own private bath.
Courtesy: Ebby Halliday, Realtors Shelly Seltzer
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This home has all the space you have been looking for AND the elementary school is award winning Stinson, within walking distance. MOVE IN condition with extensive hardwood floors, carpets recently replaced, granite in kitchen & baths. 2 Beds & baths 1st level, 2-3 living areas. HUGE upstairs with 3 beds, 2 baths Game & Media. Large pie shaped backyard with outdoor living space & firepit. 3 car garage + additional parking. Breckinridge Park!
2682 Mum Drive Richardson, TX 75082
Courtesy: Ebby Halliday, Realtors Denise Lowry
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Beautiful Brick home w plenty of living areas and bedrooms. Wood floors thru living, light filled w lots of windows, multiple built-ins for storage. Large kitchen w stainless appliances, eat at bar and breakfast room. Huge 1st floor master bedroom and bath. 3 bedrooms, 2 baths and game room up. Separate utility w sink. Sparkling pool and covered patio for outdoor living. Quiet street and great Spring Park neighborhood. Come see this beauty soon.
Courtesy: Arnold Elite Macie Bailey
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Move in ready!! Very spacious 5 BR, 3.5 BA, with 2 Living areas, 2 Dining, and a huge Gameroom upstairs. New Roof 2016, and two new HVAC units 2016. Front formal living-dining as you walk in. Large Master on the main floor with dual sinks, Garden tub, and two closets with one being a huge walk-in closet. The Kitchen offers a gas cook-top, and a huge island that overlooks into the Living area. Four BR upstairs have joining bathrooms for each.
Courtesy: Donnelli Realty Llc Richard Donnelli
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Unique opportunity to own a 5,700+ sqft golf course lot home in the desirable Richardson neighborhood of the Fairways of Sherrill Park. This 5 bedroom and 3.5 bathroom home has 3 large separate living areas. Spectacular views from your covered backyard porch looking out over the golf course and 2 small lakes with water features. Well manicured landscaped lot. Did I mention that every room is big. Plenty of room for the family in this home.
Courtesy: Ebby Halliday, Realtors Shana Cummings
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Beautiful Landstar home overlooking jogging trails and Breckinridge Park. Inviting front porch, Plantation Shutters, Wood Flooring and other custom details. Fantastic floorplan with Downstairs Master and private Study. Cheerful Kitchen with breakfast bar and butler's pantry featuring granite counters, island, abundant storage space and walk-in pantry. The Loft with a wall of built-in shelves is the perfect place to quietly relax or exercise.
Courtesy: Excalibur Luxury Properties Laura Dytko
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Luxury home in gated Toll Bros community on corner lot. Offers a dramatic entry with circular rotunda, graceful curved staircase, wood floors. Kitchen is a cooks delight with SS appls, gas cooktop, upgraded cabinets and granite countertops with lrg walk in pantry The mstr bedroom and study on the first floor. 2nd floor features two bedrms, lrg game rm and media rm. HOA maintains front yard, great location near George Bush. See media for upgrades.
Courtesy: Custom Real Estate Company Randy Rhew
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Pristine home on corner lot in established neighborhood within walking distance to Breckenridge Park. This home features 4 bedrooms, 3 baths, study and gameroom with wetbar. Oversized kitchen has granite, double oven and is open to family rooms great for entertaining. Roof has recently been replaced with a 40 year architectural roof.
3209 Grantham Richardson, TX 75082
Courtesy: Virginia Cook, Realtors Carolyn Pearson
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One owner home near City Line in Breckenridge Pk - Wyndsor Est. Updated kitchen with granite countertops, tile backsplash, SS appl. pkg, gas cooktop, refrig., micro. Washer-dryer remain. Jetted tub in M.bath. Oversized M. Bedrm. Media Center. 2016 roof. One AC unit replaced. Fence restained. Near Bike trails, golf, soccer & new St. Farm & Raytheon offices. PISD
Courtesy: Keller Williams Central Joyce Lynn Chandler
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Fabulous, move in ready home just a block from Breckinridge Park, with exemplary schools & comm. pool & playground. Enjoy new roof, gutters, front door & exterior paint [summer 2016]. Plus recent fence & deck, & gorgeous hardwood floors in entry, combined formals & family room. Also, lots of fresh paint, remodeled powder bath, & granite kitchen with 2 ovens, cooktop plumbed for gas and vented to exterior. Master & Study down, 4 bed & game rm. up
Courtesy: Keller Williams Realty Plano Denton Aguam
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Outdoor oasis with huge pool, attached spa & waterfall. Large backyard with plenty of grass for the dogs and kids. Many updates to include: interior paint, exterior paint, updated bath, stained patio, stained BOB fence, exterior siding, roof replaced, hot water heaters, Pool remodeled 3 years ago, silica filter. 5 beds & 4 full bathrooms. Large upstairs game room has closet which could be used as 6th bedroom. 3 car garage. Heavily landscaped.
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Gorgeous NEW DAVID WEEKLEY HOME. Fabulous Model Plan -2 story with 4 bedrooms and a huge master bedroom and bath. Oversized island in kitchen perfect for entertaining and cozy side patio. Hardwoods on the main level along with upgraded granite and stainless steel appliances. A must see and right across the street from Schell Elementary School. Ask about our 1-2-10 Year Warranty and SAVE BIG with our EnergySaver Program!
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Beautifully maintained Sotherby home, just steps away from 400 acre Breckinridge Park. Updates include paint, carpet, porcelain floors which look like wood. Kitchen has granite and new back splash. Master, guest room, and study downstairs. Open floor plan, 2 staircases, jack and jill bath upstairs plus 3 bedrooms, game room and media.
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Top Definition
Gwan is a Pattwa' word, It is mainly a Jamaican term. It can mean two things to *go away or *what is going on
Go away - "Tell da boys dem fi gwan."
what is going on? - "Whatta gwan?"
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dodane przez sacha maj 11, 2004
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slang from Cork city in Ireland - short for "go on". The term made it's made to the Carribean while Irish slaves were shipped there in the 16th and 17th centuries. Slang Irish terms like this then made their way into the Carribean "patois", particularly Jamaica, where "gwan" is pronounced and used in exactly the same way as in Cork city in Ireland.
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idiom: going on
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Guyanese word for "going on"
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Jamaican for "go on"
Jamaican: "Whatta gwan?"
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1.A word meaning going, i picked it up from a old movie called The Defiant Ones
1.I'm gwan to go home.
dodane przez EthanB. listopad 30, 2005


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Get an overview of this hotelHostel in Mumbai with free parking
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All 15 rooms offer room service and TVs.Information missing or incorrect? Tell us!Opens in a new window
Hotel Amenities
Hotel Amenities
Onsite self parking is complimentary.
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- Metro Dormitory Hostel Mumbai
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Harvard has Raised $230 Million for House Renewal as of March
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UC Vice President and member of Dunster House Danny V. Banks '17 adjusts his "antlers" following the release of the housing letters.
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dunster bedroom
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Ten Stories That Shaped 2015
2015 was a tumultuous year for Harvard. Final clubs faced immense administrative pressure to go co-ed, faculty saw a proposal to overhaul the College's General Education program, and perhaps most consequential of all, a University-wide survey revealed what administrators called a "troubling" climate of sexual assault on campus. Amidst a fast-paced capital campaign and Title IX scrutiny, Harvard's top administrators were called to respond to perceived racial injustice on campus and a graduate student unionization movement. Divest Harvard protesters even blockaded University President Drew G. Faust's office for a week. At the close of a particularly turbulent year, the Crimson looks back on the ten stories that most shaped Harvard in 2015.
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Thanksgiving Dinner in Dunster House
Students staying on campus during Thanksgiving break celebrate over dinner in Dunster House on Thursday evening.
Thanksgiving Dinner in Dunster House
Students were able to request specific parts of the turkey being served for Thanksgiving dinner in Dunster House.
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Daniel Banks '17 colors an intricately designed elephant at the Relaxation Study Break hosted by the Asian American and Pacific Islanders Community as part of Mental Health Awareness Week in Dunster House. With elephant and Pokemon coloring sheets, family board games, healthy snacks from CVS, and Christmas music, attendees were encouraged to relax and destress from midterms.
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Dunster Common Rooms
Parijat Lal '17 does his work in a Dunster House common room on Wednesday afternoon. Many undergraduates are critical of the hallway common rooms featured in recent house renovations.
Houses Compete for Straus Cup
Students from Quincy and Dunster houses compete at Cumnock Field for the Straus Cup. The fall intramural season runs through November and includes tennis, ultimate frisbee, and flag football.


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Star Tracks: Monday, May 16, 2016 42 years, 2,191 covers and 55,436 stories from PEOPLE magazine's history for you to enjoy
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- Read the Cover Story: Brad & Angelina Split After 12 Years: It's Over
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People Top 5
LAST UPDATE: Tuesday February 10, 2015 01:10PM EST
PEOPLE Top 5 are the most-viewed stories on the site over the past three days, updated every 60 minutes
- December 01, 2003
- Vol. 60
- No. 22
Hello, New Man!
Seinfeld Alum Wayne Knight Has Dropped 117 Lbs. (and Counting) with a Little Help from His Friends
Known to Seinfeld fans as Jerry's nemesis Newman, Knight, 48, has traded grazing for shopping: "I now have the privilege of going into pricey boutiques and spending too much," he says. "Who knew regular people were spending so much money on clothes?"
Chubby since his Georgia childhood, Knight says, "I was overweight so long, it became part of who I was." Playing plus-size funny guys in regional theater and later on Seinfeld and 3rd Rock from the Sun, he had tried amphetamines, fen-phen, Atkins and the Zone. But when he did shed pounds, he felt "exposed." Says Knight: "My weight was like a room of flesh I hid myself in."
Hiding, of course, had its risks. In 1993 a Seinfeld scene in which his character ran through a field left the actor gasping for breath. The doctor who examined Knight soon after "read me the riot act," he says. "He basically told me, 'You're going to die.' " The message hit home; Knight's own father, who had had heart disease, suffered a fatal stroke at the dinner table in 1992.
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Professionally, the trimmer Knight is thriving; in December he'll be seen in the Steve Martin comedy Cheaper by the Dozen. His romantic life, though, is in transition. Married for seven years to makeup artist Paula Sutor, 51, he's now in mid-divorce. But he looks forward to dating again, and his new confidence (and sleeker wardrobe) can only help. "As a fat person," says Knight, "I was isolated. There's a lot of self-loathing attached to being that size."
Will he lose work because he's dropped so much weight? Maybe. But Knight's willing to take that risk. He reports that when casting directors call, "I ask them, 'Do you know what I look like now?' The commodity I was selling before, I'm no longer selling," he says. "Now, I'm selling the guy inside."
Alison Singh Gee in Los Angeles
- Contributors:
- Alison Singh Gee.
More in the Archive
Today's Photos
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1. The act of being as cool as the Neaners and Nabbers Pair.
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Forum > Classic Mustang Technical forum
emergency brake assembly for 1971-1973 mustang
(1/3) > >>
Mine is no longer locking when i try to engage the brake. as my car is a 4 speed this creates a bit of an issue at times.
I vaguely remember my 72 doing the same thing 20 years ago and I think I was able to fix it, but I can't remember how.
Are there any guides for either repair or is there a replacement assembly available? I doubt it would be hard to make another year part work and the 1970 is available, but I don't want to rig it if I can possibly do it right.
Thanks again for any help
Jeff, on my 65 there is an adjustment screw on the "Y" that is in the neighborhood of the transmission that has a nut you can turn to tighten up the cable. If that doesn't fix it, then you may need to replace the shoes on the rear.
this problem is with the pedal assembly it pops out after being engaged as if the ratchet inside is bad.
Yeah, I had that problem too, so just replace the assembly all the way from the handle (foot pedal) to the "Y" Mine is a pull handle, but basically the same principal.
My first car was a 72 Maverick with the pull handle and I still think that they are the coolest.
I can find the 1970 pedal assembly, but not the 71-73. I guess i'll keep looking
[0] Message Index
[#] Next pageGo to full version


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