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1. A classic.
2. Drake's geography lesson.
3. Helen's childhood acting catch phrase.
Mom: "Take out the trash, please."
You: " THAT IS NOT MY JOB!"
4. The definition of chowder.
5. This totally legit note.
6. The foam finger fiasco.
7. Uh, whatever the hell this was.
Oh, Crazy Steve…
9. One word that said it all.
10. Megan's retort.
You know, she has a point.
11. This chick.
She's the one who didn't like green gummy bears. STUPID.
12. When Drake spoke the truth.
13. The "Theater Thug" episode.
14. Astronaut Josh Nichols.
My question is where did he even find that cardboard cutout?
15. The sassy delivery man.
I pretty much say this on a regular basis.
16. When Josh attempted football.
17. Liar vs. Truther.
Arguably one of the show's best lines.
18. Helen and Josh's work relationship.
We've all been there, Josh.
20. When Drake threw shade.
21. This wild story.
I still wanna know how this ends.
22. This moment from the pilot episode.
23. The time Crazy Steve actually made sense.
24. Josh's tragic realization.
25. Drake's badass weapon.
26. Josh's mustache.
27. The time Papa Nichols went berserk.
28. Walter's life lesson.
29. The secret salsa ingredient.
We're just…we're not even gonna go there.
30. The time Josh took his life into his own hands.
You never eat Crazy Steve's enchilada. NEVER.
31. The Sass Master 5000.
32. Apologies aren't the end all, cure all.
My mom quotes this line a little too often.
33. When Drake learned a new word.
34. The magic of the Game Sphere.
Oh, how you wished this was real.
Also, someone made this fake Valentine's Day card.
35. And finally, this.
This post was created by a member of BuzzFeed Community, where anyone can post awesome lists and creations. Learn more or post your buzz!
- It's time: Hillary Clinton and Donald Trump will face off tonight at 9 p.m. ET in the first of three US presidential debates.
- Inside the final days of the FARC, Colombia's 50-year-old rebel group, which is on the verge of signing a peace treaty with the government.
- Dozens of children were killed after the Syrian government launched a major offensive in Aleppo over the weekend.


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Four Broadway shows will be taking their last bows today. Two of the closures – Exit the King and Joe Turner's Come and Gone – were limited runs and therefore expected. However, two other closings – Reasons To Be Pretty and Guys and Dolls – were only announced days ago and likely due to their failure to earn any Tony Awards this past Sunday. Post-Tony closing announcements are something of a tradition on Broadway. Many struggling shows make the effort to continue running until Tony time, in the hopes that if they snag a win, it will result in an upswing in ticket sales. Unfortunately Reasons To Be Pretty, which has had a hard time selling tickets despite good buzz and positive reviews, lost the Best Play award to God of Carnage, and it didn't get any actor wins either. Although Guys and Dolls did fail to win Best Musical Revival, its closing announcement was far more surprising, considering it has done respectable (if not impressive) box office sales during its run.
Pawdesh Salawi
Latest posts by Pawdesh Salawi (see all)
- "Heisenberg" Begins Previews with Mary-Louise Parker - September 24, 2016
- "Jersey Boys" Announces Closing, "Groundhog Day" to Replace It - September 16, 2016
- Sean Hayes Wraps Up Run in "An Act of God" - September 3, 2016
- SNL Undergoes Summer Talent and Cast Change - August 24, 2016
- Pat Benatar and Neil Giraldo Musical Headed to Broadway - August 13, 2016


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I love it when big conferences are held in Chicago because I get to see all of my friends without having to get on a plane…and I get to sleep in my own bed.
This past weekend was SOBCon, the mastermind of Liz Strauss and Terry Starbucker St. Marie, and lots of friends – old and new – were here to participate.
On Saturday night, I met some friends for dinner at Michael Jordan's new restaurant (which, even for a vegetarian, was super yummy and the service was superb) and we sat down to talk about the realness (or lack thereof) of social media "experts," the marketing industry as a whole, and whether or not PR firms are really worth their salt.
You see, I am told many times, when I tell a new acquaintance what I do for a living, that the person has worked with PR firms in the past and they haven't gotten their money's worth.
They'll say, "Oh yeah. I know it's hard to measure PR" and they'll roll their eyes.
I mean, I hear this a lot. I usually just shake my head and say something such as, "Yeah, I hear that a lot" and the conversation moves on.
Clients Want Results
But I think it's worth discussing here because, a few days ago, Abbie Fink sent me an article titled, "Your startup's PR strategy might be crusty and outdated."
In it, the author – John Hall, the CEO of Digital Talent Agents – wrote:
In the past, public relations required a client to pay a large retainer in order to be mentioned in a sidebar or get quick features published, promoting a service or product. Today, PR is online, and it includes a combination of content marketing, search engine optimization, and thought leadership. Times are changing, and there are some things you should take into consideration when you're making decisions about your startup's PR.
People want to see results
In the past, PR companies weren't held to meeting specific client expectations. They could always hide behind saying, "How do you measure credibility and authority?" Clients consistently felt screwed over by PR companies because they were paying a sizable retainer without any way to gauge the results.
But this isn't just for a startup's PR. This is for any PR firm working with a client.
Measure PR
We have to measure PR results to business goals.
The article goes on to say you can measure your efforts with tools such as Google analytics,DoubleClick Ad Planner, WhoReTweetedMe, and Klout, but I'd take it one step further.
Yes, Google analytics is a starting point, but that's all it is. (I'm not sure the other tools he mentions do anything more than "measure" brand awareness…and don't really help the case.)
From there, you have to combine the data you get in there with a client's customer relationship management software, their email marketing software, and their ecommerce software. Without access to all of those things, you are only scratching the surface.
An Example
Let me give you an example.
We have a client who uses Hubspot and Mailchimp for lead generation and email, respectively.
They have a pretty robust content program that includes monthly webinars at the top of the funnel, white papers and eBooks at the middle of the funnel, and a free trial at the bottom of the funnel.
Our goal, of course, is to use content to get people into the free trial. From there, they know exactly how far through the trial a person has to go before they become a customer and their email marketing helps motivate them to that level.
We measure PR in a few ways:
- Organic search increases
- Unique visitors to the website
- Unique visitors to the blog
- Top of the funnel leads
- Free trial landing page visits
- Free trial users
- Trials converted to customers
- Monthly recurring revenue (subscription-based)
- New revenue
All of this data can be found by combining analytics, Hubspot, and Mailchimp into a spreadsheet we review weekly. We do not track Facebook fans, Twitter followers, YouTube viewers, or Klout scores.
These are both soft and hard metrics, but they lead to the ultimate goal: More customers.
In this example, we know exactly how many people we need to get to the free trial landing page – through content – in order to get them to take it and then what kinds of content – through email – we need to send them so they'll convert to a customer.
It becomes science and math, combined with art, and it works really, really well.
Become a Partner
What this does is creates an opportunity for us to be partners with our clients. They trust us to not only give us access to their data, but also to help them achieve their goals.
And it also holds us completely accountable to the results that really matter to a business leader, owner, or entrepreneur.
We know every week whether or not something is working and can make tweaks or change paths, if necessary. We also are testing a pay-for-performance/retainer hybrid model with this particular client. I'll let you know by year's end how it worked.
I agree with John Hall clients want results, but the way we measure them go a step further than he took it in his article.
What do you think?
Leave a Reply
65 Comments on "How to Measure PR: Use These Tools"
This gives me so many ideas…great post Gini, as always.
katskrieger I like it when I give you ideas!
ginidietrich katskrieger Always! Besides reading your tips is way better than the task I am currently procrastinating on….packing my desk to move to the other side of the office. And taking photos of receipts to send to Concur.
katskrieger LOL! Yeah…reading sounds way more enjoyable.
ginidietrich katskrieger In all seriousness, this definitely give me some more places to look for good data. Doing a lot of the right things, but I think I need to pull into one big spreadsheet.
katskrieger A lot of what we pull is from Hubspot. If you need help, let me know.
ginidietrich Gini, I'd love to hear more about how you are using Hubspot — and how you are tracking this for clients who DON'T use Hubspot. Likely more than fits in a comment though…
blfarris That'll cost you a bottle of wine!
ginidietrich Deal! I hope you like Red.
blfarris I do, indeed!
Sheeesh, why don't you just marry Michael Jordan already.
belllindsay He just got married or I would!
ginidietrich belllindsay He is adorable.
KateFinley ginidietrich belllindsay He's married to a chick named Yvette…I think she has a great name 😉
I heart this post for so many reasons! Let me count the ways …
1. You don't measure success by fans/likes/follows.
2. You've just given me a couple more ideas on how I can measure PR back to results.
3. Performance-based retainers … I want to know more about how you are attempting this.
KateFinley I'll fill you in on number three, Kate. It's not perfect yet, but we're attempting it. Right now what we're doing is a break even retainer – this is how much it costs to have my tam work with you without any incentives – and then anything we achieve above the set goals we get a bonus.
ginidietrich KateFinley I was trying to think about how I would adapt that model if I decided to go that route and the basic retainer + bonus makes complete sense. Really looking forward to seeing how this works. Curious about how the bonuses breakdown per team member, etc. Very cool! Thanks!
KateFinley The bonus goes into the entire pool for the company because we're small that nearly everyone is involved. That said, we haven't actually achieved the bonus yet so we'll see.
Joshua Wilner/A Writer Writes ginidietrich I almost said "phewww" out loud when I read that first sentence you quoted from the post. I started, and still "manage", Facebook and Twitter for our college, and despite touting the greater value of engagement over Likes, etc., others keep focusing on numbers, especially even ones ending in 0. Those are apparently sacred in some cultures.
Word Ninja Joshua Wilner/A Writer Writes It's because people understand numbers. What they don't understand is what is behind them. Imagine having 20,000 Facebook fans with no engagement and no sales or 200 Facebook fans with 90 percent engagement and lots of sales. The executives will almost always focus on the fact that your Facebook fan base isn't growing. Which is dumb, but that's human nature.
Joshua Wilner/A Writer Writes It's a deal breaker for us if the client won't share their data and analytics. It's only happened once and we walked away from the deal because of it.
I agree we need to educate clients too, but that's up to us to do it. If we continue to measure media impressions and advertising equivalencies and fans/followers/likes, they'll continue to be frustrated they're throwing money into the wind.
bdorman264 Did you really get an Egg McMuffin?! I never get anything! <pout!>
You're absolutely correct in that there has to be some heavy lifting on the part of the client. We can't work in a vacuum. That's why chemistry is so important, as well. If you like the people, you're more willing to do the work.
ginidietrich bdorman264 I got some Captain Highliner fish. 🙂
You talking about accountability gives me goose bumps. How can anyone in modern PR expect to be taken seriously without the means to demonstrate the work they do translates into meaningful results (i.e. – what their efforts mean to the bottom line)? I really like the concept of tying in analytics, CRM, lead gen, and email platform results into a spreadsheet. Brilliant.
This post has sparked some ideas then I want to experiment with. Thanks!
John_Trader1 Oh … you'd be surprised. I'm shocked every time I talk with a group of highly intelligent PR professionals. Shocked.
lauraclick When I did my keynote last week, I talked about that very thing. How when, in 2010, we said we were no longer a PR firm, the conversation changed. In fact, a client whom we adore just asked us to consider doing a solo media relations program for one of their new projects. We turned it down because we can't guarantee results and because you can't measure those results. I think they were a little surprised, but told me at least four times they respect our candidness.
lauraclick I do see where you're going with this. I think it's up to you to decide if you want to pursue it…and if the client is willing to take some risk. For us, we're too small and I'm too vocal about data-driven results to take that risk. That, and we no longer have anyone here (besides me) who has media relations expertise. So I'd have to hire someone, which is an entirely different conversation.
Howie Goldfarb ginidietrich Yup. Totally. A lot of the work I'm doing for clients right now is out the foundational level (strategy, branding, web design, etc.) Those things have to happen first BEFORE you can start getting into social media. I never just DO social media for clients. Never Ever. But, that's a whole other conversation entirely!
JodiEchakowitz I've spent A LOT of time digging into why people are not moving toward the Barcelona Principles and why they still use AVEs. It's because their clients and bosses require it. And we all have only 24 hours in the day so, unless you're REALLY passionate about finding a different way of doing things, you're not going to spend the non-work hours that are required to do it. So, until the industry can come up with a set way to view metrics and measure efforts, I don't think it's going to happen.
All for just $19.99!
What drives me bonkers is what measurement services do measure. Because often they have no real bearing on telling whether results were good or bad. Really we laid out for folks Ms ginidietrich
Who is Michael Jordan is he the one who is the chef that yells at people?
Howie Goldfarb It's a good thing I know you as well as I do because I'd hate for that question to be real.
tayloraldredge I like how this grasshopper views it! And I agree.
tayloraldredge Oh sorry. Now I realize it should be capitalized Grasshopper.
Thank you for laying the details out, Gini. It's interesting, because last week I had a colleague tell me that an intern wants to learn more about PR and communications. I immediate reaction was, "number one, don't use the term PR'.
susancellura If we just said communications, we'd get rid of a lot of the typical spin doctor responses.
Isn't the key to layout ahead of time clear expectations and KPIs? Wouldn't that solve some headaches?
ClayMorgan It would IF the client either doesn't already think media impressions and AVEs are the end all, be all. In most cases, though, you have to slowly educate them on what really matters and how it's measured. And it's pretty fluid so you have to stay ahead of it.
"We do not track Facebook fans, Twitter followers, YouTube viewers, or Klout scores." This can not be said enough. I think there is a general misconception when it comes to this type of metric. The only thing social, I measure is click-throughs and conversion rate.
I also really like your breakdown of the funnel process. I've been trying to explain this process to a client for several months now, (they take forever to approve anything). The CEO doesn't see the value in webinars, he prefers face to face interactions.Hopefully I can convince him that one step leads to another.
stevenmcoyle I remember the days when I didn't believe in using the Internet to build my business, too. Then a very good friend said to me, "Wouldn't you rather meet 100 people 'face-to-face' at once than do one at a time?" Um. Yes.
Is it too basic to "ask at the door"? We ask virtually every customer how they heard of us!
jdrobertson I don't think it is, but you can't always ask that, particularly if your customers are tens of thousands. You ask it, but not everyone tells you. Or they say, "Oh a friend told me" when really they saw an ad.
This assumes you are driving all traffic to the digital platforms that start the funnel, yes? So all the social platforms you use are driving to web-hosted content that's all part of that "pretty robust content marketing program?"
rustyspeidel We actually don't use social in this example….so no. But yes, it could be in some cases.
I'm confused. I thought you just wrote press releases. 🙂
Adam | Customer Experience I'm shaking my fist at you right now!
I just about choked on my chicken sandwich. Klout as a measure of success? Thanks Gini for highlighting the metrics that matter.
ThePRCoach Well, we can't win them all. Yet.
Like it a lot. Keep banging the drum . I know you were having fun in Paris at the time, but your star colleague, allenmireles did highlight why "measurement is huge" and how "Inkybee Measures Your Campaigns" using a Google Analytics plug-in, so we're right with you.
HughAnderson Yep! I read all of the blog posts before I left so I saw it!
Soulati | B2B Social Media Marketing You're so funny! I always say, "I'm going to tell this story and it's going to make me sound old, but I really started my career when I was 12."
hopwood I 100% agree with you, which is why I mention this should be used in combination with the "soft metrics." The problem, of course, is (at least in the U.S.) chief executives don't care about brand awareness if it's not affecting sales. And…thank you for the compliment!
[…] do you measure results? We talk about measurement often on Spin Sucks. That's because it's essential to the success of an agency in retaining […]
[…] We talk a lot here about how to provide the metrics that show you are an investment, instead of an expense. […]
[…] are the days where it's sufficient to report on media impressions and advertising equivalencies—clients want REAL, tangible metrics that demonstrate return on investment and progress. They want […]


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Home > Graphic Apps::Other > Aurigma Photo Kiosk 7.0
Aurigma Photo Kiosk 7.0
Photo kiosk software to create powerful print order station from a common PCCompany: Aurigma Inc.
Platform: WinXP
Size: 9572 KB
Price: USD $199.00
Release Date: 2012-01-20
Screenshot URL: http://photokiosk.aurigma.com/PAD/v2/photo-kiosk-screenshot.png
Category: Graphic Apps::Other
Photo Kiosk is an ideal photo print order tool for a variety of points of sale, including but not limited to: print order stations, instant print stations, internet cafe PCs, etc. It has easy-to-use Web 2.0 user interface with on-screen keyboard, it easily integrates with other software components and is easily localizable into other languages. It has rich portable media support (USB Flash drives, Memory cards, DVD) and small memory footprint.
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Fair photos from this past weekend are now available! Click here for a look at the full day of events… judges meeting students with their projects, the Getting Discrete with Calculating & Concrete workshops as they unfolded, 3D Printing and Additive Manufacturing presentations, and the final award ceremony.
Again, thanks to all who were involved! The fair is a rewarding endeavor for all of us here at SUNY Broome as well as the local sponsors and other participants from the community.


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|Home | About | Journals | Submit | Contact Us | Français
The rising incidence of obesity and insulin resistance to epidemic proportions has closely paralleled the surge in the prevalence of diabetes and outpaced therapeutic advances in diabetes prevention and treatment. Current evidence points to obesity induced oxidative stress and chronic inflammation as the common denominators in the evolution of insulin resistance and diabetes. Of all the hypoglycemic agents in the pharmacological arsenal against diabetes, thiazolidinediones, in particular pioglitazone, as well as metformin appear to have additional effects in ameliorating oxidative stress and inflammation; rendering them attractive tools for prevention of insulin resistance and diabetes. In addition to their hypoglycemic and lipid modifying properties, pioglitazone and metformin have been shown to exert anti-oxidative and anti-inflammatory effects in vascular beds, potentially slowing the accelerated atherosclerosis in diabetes, which is the major cause of morbidity and mortality in the affected population. The combination of pioglitazone and metformin would thus appear to be an effective pharmacological intervention in prevention and treatment of diabetes. Finally, this review will address the currently available evidence on diabetic cardiomyopathy and the potential role of combination therapy with pioglitazone and metformin.
The recent escalation of obesity from an individual health problem to a major public health issue reaching epidemic proportions has drawn attention to a constellation of abnormalities (abdominal obesity, hypertension, dyslipidemia) collectively referred to as metabolic syndrome. As an indicator of insulin resistance and a harbinger of diabetes, this syndrome has been associated with major cardiovascular mortality and morbidity (Galassi et al 2006). The surge in the prevalence of diabetes to epidemic proportions around the world has followed the same trends as the obesity and metabolic syndrome. The current global prevalence of type 2 DM is approximately 150 million with a predicted increase to 215 million by 2010 and 300 million by 2025 reported by CDC (2006). Therefore, it would seem intuitive to blame excess body weight and excess adipose tissue mass for causing insulin resistance and diabetes. Yet, the exact pathophysiological events leading to the development of diabetes or cardiovascular disease in metabolic syndrome remain unknown. This review aims to go over the current literature on the pathogenesis of diabetes and effects of two different insulin sensitizers, on the treatment of diabetes and cardiovascular complications of diabetes.
Subjects with type 2 diabetes (T2DM) are characterized by chronic insulin resistance with poor β-cell compensation. Although there is much descriptive information on the pathogenesis of type 2 diabetes (T2DM), the underlying mechanisms remain unknown. The initial pathophysiologic event is usually insulin resistance associated with obesity and a sedentary lifestyle. Along with the development of insulin resistance, insulin secretory decompensation develops, leading to impaired glucose tolerance, and eventually T2DM (Martin et al 1992). It is not well understood why obesity causes insulin resistance. However, the hypotheses linking obesity to insulin resistance generally fall into the following categories: inflammation, lipotoxicity, oxidative stress and endoplasmic reticulum stress.
Obesity is associated with increased accumulation of macrophages in adipose tissues and increased levels of pro-inflammatory cytokines (Di Gregorio et al 2005). In addition, obesity results in excess lipid accumulation not only in adipose tissue but also in tissues such as the liver, skeletal muscle, heart and pancreas (Unger 2003). There is a strong correlation between intramyocellular fat content (IMCL) and insulin resistance (Perseghin et al 1999; Friedman 2002). Ectopic lipid accumulation with resultant functional impairment has been called lipotoxicity, a pathophysiologic event preceding diabetes (Friedman 2002). Increased level of reactive oxygen species (ROS) has been also known as a culprit in the pathogenesis of insulin resistance and diabetic complications (Petersen et al 2004; Schrauwen and Hesselink 2004; Houstis et al 2006; Mehta et al 2006). Insulin resistance and diabetes are associated with disruption and uncoupling of several key oxidative reactions that result in excessive production of ROS at mitochondrial and cellular levels (Nishikawa et al 2000). Increased production of reactive oxygen species in mitochondria, accumulation of mitochondrial DNA damage, and progressive respiratory chain dysfunction are associated with atherosclerosis or cardiomyopathy in human investigations and animal models of oxidative stress (Mehta et al 2006; Madamanchi and Runge 2007). More recently, obesity-induced endoplasmic reticulum stress (increased unfolded protein response due to the disruption of the smooth operation of endoplasmic reticulum) has been demonstrated to result in peripheral insulin resistance (Hotamisligil 2005). Lipotoxicity, oxidative stress and endoplasmic reticulum stress are associated with activation of nuclear factor-κB (NF-κB), chronic inflammation and insulin resistance (Figure 1). Interventions aimed at modulating any of the above mentioned pathways are potential therapies for diabetes and its complications.
Because insulin resistance is the key feature of type 2 diabetes, much drug development has been focused on insulin sensitizers. At present, two classes of drugs are used as insulin sensitizers: biguanides (metformin), and thiazolidinediones (rosiglitazone and pioglitazone). The mechanism of action of these drugs is not well understood. Metformin is relatively more active in the suppression of hepatic glucose production (DeFronzo et al 1991; Stumvoll et al 1995), and thiazolidinediones are more active in stimulation of muscle glucose uptake (Nolan et al 1994; Yki-Jarvinen 2004).
Metformin is the only biguanide available in the U.S. and it is one of the most commonly used anti-diabetic medications. Several studies have demonstrated improved peripheral insulin sensitivity following metformin treatment in diabetic patients (Widen et al 1992; Velazquez et al 1994; Dorella et al 1996; Li et al 1999; Moghetti et al 2000). In one study, metformin produced an improvement in basal, but not insulin stimulated, glucose transport, and no effect on insulin secretion (Morel et al 1999) yet in another study, metformin did not have any effect on peripheral insulin sensitivity (Rasouli et al 2005). The improvement in insulin sensitivity by metformin was less than that observed with thiazolidinedione in a recent study in diabetic patients receiving a sulfonylurea (Kim et al 2002; Rasouli et al 2005). Hence, the data suggest that metformin has a primary action on hepatic glucose production, either through direct activation of insulin receptor (Gunton et al 2003) or inhibition of key enzymes in the gluconeogentic pathways (Cusi et al 1996; Dorella et al 1996). Recent studies have shown that metformin stimulated AMP kinase activity (Zhou et al 2001), and enhanced the peroxisome proliferator-activated receptor-gamma coactivator-1alpha (PGC-1α) expression (Suwa et al 2006). Both AMP kinase and PGC are major regulators of fatty acid oxidation; hence, metfomin might improve lipotoxicity. Indeed metformin decreased muscle fat content in diabetic subjects (Mathieu-Costello et al 2003), however, we did not detect any change in IMCL in our study, with metformin (Rasouli et al 2005). Metformin has also favorable effects on some inflammatory markers such as CRP levels. However, the effect of thiazolidinedione or weight loss on lowering CRP was more pronounced (Chu et al 2002; The Diabetes Prevention Program Research Group 2005). We have previously reported no change in plasma levels of TNFα or adiponectin in subject with impaired glucose tolerance after treatment with metformin for 10 weeks, however, resistin level decreased significantly in that study (Rasouli et al 2006). In addition, metformin did not change the number of adipose tissue macrophages as markers of inflammation (Bodles et al 2006; Di Gregorio et al 2005).
The impact of metformin on oxidative stress has also been investigated in several studies. It has been shown that metformin can quench the surge in reactive oxidant species in insulin resistance and diabetes. In a study by Rosen et al on Goto-Kakizaki (GK) rat model of type 2 diabetes, Metformin was noted to ameliorate mitochondrial oxidative stress as demonstrated by a reduction in mitochondrial aconitase activity. In addition, metformin prevented the rise in oxidized proteins (carbonyl moieties) and lipid peroxides that are usually observed in this model of type 2 diabetes (Rosen and Wiernsperger 2006). Furthermore, metformin has been shown to reduce ROS levels in human leukocytes by either directly scavenging the free radicals or modulating their intracellular production (Bonnefont-Rousselot et al 2003).
The thiazolidinedione derivatives, pioglitazone and rosiglitazone, are synthetic ligands for peroxisome proliferative-activated receptorγ (PPARγ) which improve insulin sensitivity. PPARγ is mainly expressed in adipose tissue; however, improved insulin sensitivity is a function that primarily involves glucose transport into skeletal muscle. This effect is poorly understood, but it is thought to be due to redistribution of lipid from ectopic sites to subcutaneous adipose tissue. PPARγ agonists promote adipocyte differentiation, and they promote FFA uptake and storage in subcutaneous adipose rather than visceral adipose tissue or ectopic sites (muscle or liver) (Bajaj et al 2003; Rasouli et al 2005). In addition to the favorable effect on lipotoxicity, we have previously reported that thiazolidinediones decreased inflammatory responses in insulin resistance (Di Gregorio et al 2005; Bodles et al 2006) by reducing the number of adipose tissue macrophage. Pioglitazone promotes apoptosis in adipose tissue macrophages along with improvement in insulin sensitivity (Bodles et al 2006). PPAR ligands have been shown to regulate inflammatory cytokines like TNFα, CRP, PAI and adiponectin as well (Chu et al 2002; Yki-Jarvinen 2004; Rasouli et al 2005, 2006). Thiazolidinediones have also been shown to exert potent antioxidant effects. In a study by Hwang and colleagues, rosiglitazone reduced NADPH oxidase activity and superoxide ion O2− generation in the vascular tissues obtained from obese, diabetic, leptin receptor-deficient (db(−)/db(−)) mice (Hwang et al 2007). Similarly, pioglitazone has been found to reduce superoxide ion O2− generation in human coronary artery endothelial cells (Mehta et al 2003).
Cardiovascular disease is the major cause of mortality and morbidity in diabetes and is usually due to atherosclerosis in the coronary, cerebral and other peripheral vascular beds (Tziakas et al 2005; Pambianco et al 2006). Yet, myocardial dysfunction in the absence of epicardial coronary atherosclerosis has also been described and accounts for some cases of congestive heart failure in diabetics (Rubler et al 1972).
Several lines of evidence point to endothelial dysfunction and loss of endothelial vasoreactivity as one of the initial events in atherosclerotic plaque formation (Davignon and Ganz 2004). The dysfunctional endothelium subsequently expresses a host of cell surface receptors including intercellular adhesion molecule-1 (ICAM-1) and vascular adhesion molecule-1 (VCAM-1) as well as receptors for oxidized lipid particles such as leptin-like oxLDL receptor (LOX-1) which facilitate inflammatory cell recruitment and oxidized LDL transport to the intimal layer. The ingestion of oxidized lipid particles by these cells results in further activation of inflammatory cells and release of cytokines and growth factors which perpetuate the vicious cycle of inflammation, cell recruitment, lipid particle ingestion and growth; ultimately leading to an atherosclerotic plaque. The acceleration of this process in diabetes could be attributed to several potential mechanisms. There is growing evidence that excessive ROS formation in diabetes leads to endothelial dysfunction (Vincent and Taylor 2006). The first evidence of endothelial dysfunction in human type 1 and type 2 diabetes were reported from examination of penile corpura cavernosa (Saenz de Tejada et al 1989). Subsequent studies confirmed loss of endothelial vasoreactivity in other vascular beds including coronary arteries (Nitenberg et al 1993). Although the exact mechanism of endothelial dysfunction in diabetics has not been elucidated, several studies point to reduced NO bioavailability and that tight glycemic control can improve endothelial reactivity (Rodriguez-Manas et al 2003). Our laboratory data indicates that increased ROS induces LOX-1 expression which not only facilitates ox-LDL incorporation into endothelium but also enhances expression of adhesion molecules ICAM-1 and VCAM (Li and Mehta 2000; Chen et al 2001; Li et al 2003). The dyslipidemic milieu of diabetes ensures a continued flux, oxidation and uptake of LDL particles into the eventual atherosclerotic plaque.
Thiazolidiones and in particular pioglitazone have been shown to improve endothelial function in diabetes. In a randomized controlled trial, Martens and colleagues demonstrated that four weeks of therapy with pioglitazone improves shear stress-induced flow mediated vasodilation (an endothelial dependent phenomenon) of brachial artery in patients with type 2 diabetes with no effect on endothelium independent vasodilation (Martens et al 2005). In addition, pioglitazone has also been shown to reduce LOX-1 as well as VCAM expression which can potentially prevent plaque formation (Imamoto et al 1920; Mehta et al 2003). The metabolic effects of pioglitazone in improving insulin sensitivity, reducing blood glucose, LDL, triglyceride and certain inflammatory mediators such as C-reactive protein levels further modulates the substrate for plaque formation and halts atheroma progression (Berhanu et al 2006; Szapary et al 2006). Metformin has also been noted to effectively reduce oxidative stress through inhibition of the PKC and NADPH oxidase pathways (Ouslimani et al 2005; Mahrouf et al 2006). While reducing the oxidative stress burden, metformin can ultimately improve endothelial function (De et al 2005). Metformin has also been shown to exert antiproliferative effects on vascular smooth muscle cells through inhibition of PKC pathway which can also slow the pace of plaque formation (Li et al 2005). Several human studies have indeed indicated that macrovascular complications of diabetes are reduced by metformin (Johnson et al 2005). Given the evidence, it appears that thiazolidinediones and metformin modulate endothelial function and plaque formation through distinctly separate pathways rendering their combination an attractive agent in decelerating diabetic atherosclerosis.
Approximately 34 years ago, Rubler and colleagues reported a series of four diabetic patients with congestive heart failure, normal epicardial coronary arteries and no other known causes of congestive heart failure in association with diabetic glomerulosclerosis (Rubler et al 1972). This new entity termed diabetic cardiomyopathy is frequently associated with diastolic dysfunction, left ventricular hypertrophy and fibrosis (Di Bonito et al 1996). Subsequent attempts in elucidating the putative mechanisms in animal models indicated a direct correlation between myocardial fat content and systolic and diastolic dysfunction. In a transgenic mice model which over expressed long chain Acyl-CO A synthase (ACS), Chiu and Schaffer were able to demonstrate dilated cardiomyopathy by sequential echocardiography along with excessive triglyceride accumulation in cardiomyocytes by both light and electron microscopy (Chiu et al 2001). In addition, incubation of cardiomyocytes with long-chain fatty acids resulted in DNA fragmentation and cardiomyocyte apoptosis (Kong and Rabkin 2000). The obese (fa/fa) rat which develops diabetes with aging is the closest animal model to human metabolic syndrome. A recent study by Zhao and colleagues demonstrated that the obese (fa/fa) rats will accumulate significantly higher amounts of cardiac triglycerides as early as 7 weeks of age as compared to the normal lean rats. Further evaluation of myocardial performance by echocardiography at 20 weeks of age revealed a dilated cardiomypoathy along with systolic and diastolic dysfunction in the obese (fa/fa) rats while the lean rats maintained the normal cardiac function at 20 weeks in this study (Zhou et al 2000). Despite these well-described animal models of myocardial lipotoxicity, the prevalence and mechanisms of cardiac lipotoxicity in humans remain unknown.
Animal studies indicate that pioglitazone can prevent diastolic dysfunction in advanced diabetes by improving myocardial fatty acid metabolism (Kim et al 2003). While these studies supported the cardiac lipotoxicity hypothesis and pointed to an emerging potential therapy, early use of thiazolidinediones in humans was associated with several reports of fluid retention and exacerbation of congestive heart failure (CHF) (Kermani and Garg 2003; Cheng and Fantus 2004). A large multicenter clinical trial on 16,000 individuals with CHF aimed to assess potential benefits of pioglitazone in preventing complications of diabetes yielded mixed results; indicating that the favorable effects of pioglitaozne in reducing mortality are offset by increased number of hospital admissions for CHF exacerbations (Dormandy et al 2005). Therefore, further clinical studies are needed to elucidate the role of Thiazolidinediones in diabetic cardiomyopathy. Similarly, the initial concerns that metformin can cause lactic acidosis in patients with diabetes and congestive heart failure were revisited in a large retrospective study of over 12,000 patients (Eurich et al 2005). This study indicated that metformin alone or in combination therapy is associated with lower morbidity and mortality in patients with heart failure and type 2 diabetes as compared to sulfonylureas (Eurich et al 2005).
The increasing incidence of diabetes has paralleled the epidemic of obesity and insulin resistance. The exact mechanism(s) linking obesity to insulin resistance and diabetes remain to be explored but the evidence points to the three axes of oxidative stress, lipotoxicity and ER stress. Given the multiple putative pathways involved in the pathogenesis of diabetes and its complications, the treatment of this condition calls for simultaneous disruption of such pathologic phenomena. Therefore, it is anticipated that combination therapy with two or more agents will surpass the currently accepted indication of achieving optimal glucose control and expand to include most patients with diabetes. Pioglitazone and metformin are two insulin sensitizers with different mechanisms of action and distinctly separate effects on oxidative stress and lipotoxicity rendering their combination a potentially powerful tool in treating diabetes. In addition, the favorable effects of pioglitazone on endothelial function and cell surface receptor expression are in synergy with some of the anti-atherosclerotic effects of metformin; thus rendering combination therapy an attractive tool against accelerated atherosclerosis of diabetes.
This work was supported by the Research Service of the Department of Veterans Affairs through VA Merit Review grant to NR, and by grant M01RR14288 from National Center for Research Resources (National Institutes of Health) to the University of Arkansas for Medical Sciences to fund the General Clinical Research Center.
PubMed Central Canada is a service of the Canadian Institutes of Health Research (CIHR) working in partnership with the National Research Council's national science library in cooperation with the National Center for Biotechnology Information at the U.S. National Library of Medicine(NCBI/NLM). It includes content provided to the PubMed Central International archive by participating publishers.


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I train MMA.
I read books and write about them so you don't have to!
And I love to live.
If you have any questions or want to get in contact with me, email me at email@example.com
I train MMA.
I read books and write about them so you don't have to!
And I love to live.
If you have any questions or want to get in contact with me, email me at firstname.lastname@example.com
7/7 – 7/17
Nothing, but therapy, assisted and on my own, in the past two weeks. Everytime I master an exercise, I move onto a tougher, more painful and challenging one, but that is what I have to do to improve as solidly and rapidly as possible.
I still haven't got my stitches out which limits my knee's ability to bend. Because of this, I can't drive and can only walk short distances very slowly. Just being able to walk at all, however, feels amazing. I'm still trapped on a mattress the majority of the day, but when I want to, I can actually fend for myself now.
I have been reading fanatically, learning to play the keyboard, and spending time with great friends and family to pass the time. All I can see in my mind, all the time, is MMA. Slowly, but surely, I will get back there.
"I only see my goals, I don't believe in failure"
6/28 – 7/6
Surgery on 6/28.
I remember getting wheeled into the operating room, already dosed on a few painkillers and loopy. I remember talking to the nurse and giggling a lot and suddenly, I woke up and my leg felt horrible. They gave me some fentanyl and wheeled me out to my mother and then to the backseat of her car.
Who knew that pain could get this bad. I couldn't feel my leg, but I could feel a searing knife cutting into it at all times. I had to pop 2 percocets every 3 hours to stop myself from going insane. That pain continued throughout that night into the following night, where it began to dull from excruciating to simply severe. I couldn't move my leg without sharp, stabbing pains so I laid in bed, trying to use the bathroom as little as possible. To get to the bathroom, I had to use my crutches to carry my drugged out self and hope I didn't fall (I did. A few times.)
I had to go from the guy who people leaned on, who helped everybody, who never asked for help. To the guy who needed someone to hold his hand through the pain, who needed to be carried around, who had to ask for help to get fed and clothed.
But I honestly couldn't have asked for a better blessing. If you asked me what my biggest weaknesses were before this, I would tell you I was too prideful, too impatient, too obsessive, too focused on the big picture and willing to let the details slide, too driven. Recovering from this surgery has been all about swallowing my pride, being mind-numbingly patient, paying attention to the tiniest details, figuring out balance, and an opportunity to train my mind and spirit as opposed to my body.
Derrick rose and countless other athletes have compared this surgery and the physical therapy following it to "hell", "the closest thing to death", and "I wouldn't wish it upon my worst enemy." That is all true, but it is exactly what I needed and when I needed it. Through all of the pain, weakness, and discomfort, I always try to remember that and it keeps a smile on my face.
Now those are my thoughts on that – let's get down to business. I have had two therapy sessions in the week since my surgery. The number one priority for therapy is to achieve full knee/leg extension and be able to perform leg raises. The first therapy session was only 3 days after and the first time I removed this horrible brace off my leg, temporarily of course. The therapist led me through a list of exercises of which I wasn't able to really do many of them yet. I couldn't even do a damn leg raise or bend my knee.
I kept working at all of them, multiple times a day, and I was finally able to get all of them down. I had my 2nd therapy session exactly a week after surgery on 7/5. I have almost achieved my previous level of leg extension which is slightly past hyperextension. The therapist was impressed at how fast my knee is recovering and how much my flexibility and movement has improved. Then gave me a list of harder exercises. Time to get to work on those.
"We all hang on to our sad life stories, thinking that they give us the right to be unhappy. They fucking don't."
6/24 – 6/27
Two workouts in the last few days:
Bench Press – 165 lbs, 3×8
Military Press – 95 lbs, 3×8
Barbell Rows – 145 lbs, 3×8
Pull ups – 14 reps, 10 reps, 6 reps
Bicep Curls – 30 lb Dumbbells, 3×8
Skull Crushers – 55 lbs, 3×8
Lateral Raises – 17.5 lbs, 3×8
Surgery tomorrow morning. Let's do it.
6/15 – 6/23
The damage is the terrible triad. Torn ACL, Medial Meniscus, and partially torn MCL. I am getting surgery on June 28th and then staying in South Florida as I recover and undergo physical therapy.
No real working out for me in the next couple months. I have done 3 gym sessions focused on upper body this past week. I will keep updating my numbers and hopefully strengthen one part of my body while the other rests.
Last workout:
Bench Press – 160 lbs, 3×8
Military Press – 95 lbs, 3×8
Barbell Rows – 145 lbs, 3×8
Pull ups – 12 reps, 8 reps, 6 reps
Bicep Curls – 70 lb Barbell, 3×8
Skull Crushers – 50 lbs, 3×8
Lateral and Anterior Raise – 15 lbs, 3×8
"It might be that to surrender to happiness was to accept defeat, but it was a defeat better than many victories"
11am – 1 pm
I arrive to st. pete and start drilling some wrestling for a few minutes. Everybody else goes to lift, so I go for a 30 minute run. My legs hurt and it is extremely hot outside so my pace kind of sucks, but all that matters is I took right action. We then go into drilling and live wrestling, and although it just mostly drills, my wrestling feels the best it's ever been. Good work for the day.
9am – 10am
Helping out Lin, a new student today in class. Teaching and learning from the teaching.
10am – 10:30am
Tempo intervals for conditioning, I did 22 minutes today on the heavy bag and shadowboxing.
7pm – 8:30pm
I got out of work early so I head over to PAC and get some striking on! I warm up with some kickboxing at the end of the fundamentals class and go right into sparring drills and free sparring with Chris and Tyson in the intermediate class. Fun night, I leave the gym very pumped up and in love with the process of getting better!
9am – 10am
Taught a great class to a couple older faces and some beginners. Always fun to help people improve.
10:30am – 11am
Drilling wrestling/ground and pound positions with coach Vince.
2pm – 3pm
Strength maintenance workout, still harder than most strength building workouts! 3×8 with 215 lbs on squat, 3×6 with 275 lbs on deadlift, and 3×8 with 155 lbs on bench.
3pm – 4pm
Nothing but a run in the books for me today. I get 4 miles in at a good pace.
I showed up to st. pete to train with coach Vince and others, only to realize it was closed down today for a seminar at Gracie Bradenton. I make the drive home and go to crunch for some work.
2:30pm – 3pm
Tempo Intervals for conditioning at Crunch fitness, 25 minutes, working on the heavy bag.
11am – 12pm
MMA class at PAC with Chris, Amy, Vince, and Bobby. In my last sparring round, Chris attempts a judo throw and I resist. In a freak accident, I end up falling over my knee and hear the worst sound I have heard in my life. Like the sound of a toaster being blended at 1000% intensity. I fall to the ground in pain and looks I have my first serious knee injury.
I am back home, unable to walk or drive, waiting to see a doctor to move on with life. Whatever happens, I accept it completely.
"Do not let anything that happens in life be important enough that you're willing to close your heart over it"
12pm – 2pm
I drive through the Howard Frankland bridge and a tropical storm to make it to St. Pete for some MMA work! I have to give my training partners and coaches some sparring.
I warm up and we get right into 5, 5 minute rounds. I do two rounds with Chris, one with Bobby, one with Ethan, and one with Gabe. I perform well enough, but I am aware of lots of mistakes I am making and I can improve on, and I get caught with a good counter that gives me a small mouse under my left eye.
Amazing work, regardless.
4pm – 5pm
I head to crunch fitness to get some cardio in since the rain is stopping me from running outdoors. I do 10 minutes on the elliptical, 5 on the bike, 5 on the treadmill, and then decide to say screw it and go for a 25 minute run through the rain.
I am exhausted this morning, but I pull myself out of bed for class and to begin a personal "life experiment" I have been putting off for a while. Let's see how the day goes.
9am – 10am
BMT class was great, working with Steph and Amy. I was a little unfocused in class, but when I tuned out the distractions, I was performing just as good as I ever have.
10am – 10:30am
Tempo Intervals for conditioning. I do 15 seconds of moderate intensity work on the heavy bag and shadowbox lightly for a minute. Repeat for 20 minutes. Afterwards, I go get some chiropractic work in.
"You have good days. You have bad days. The important thing is to grow mentally"
2pm – 3pm
Great 3 mile run. I kept my heart rate at the perfect range and actually got a great pace in. When I'm well-rested, there are very few people who are as aerobically fit as me.
1pm – 2pm
I had a rough morning so I skipped out on going to st. pete. Not my best decision, but I made up for it by a great weight training session. I am just trying to maintain strength, but working out like this, I might actually get a little stronger as I drop weight and increase my cardio.
"Doesn't matter what the press says. Doesn't matter what the politicians or the mobs say. Doesn't matter if the whole country decides that something wrong is something right. This nation was founded on one principle above all else: the requirement that we stand up for what we believe, no matter the odds or the consequences. When the mob and the press and the whole world tell you to move, your job is to plant yourself like a tree beside the river of truth, and tell the whole world — "No, you move.""
9am – 10am
BMT in the morning, working with Jason again!
9am – 10am
Teaching BMT to 3 awesome ninjas in the morning, staying present, and having fun!
12pm – 2pm
Great dutch drills and MMA sparring with Ethan, Chris, Vince, and Gabe at St. Pete. The team is strong.
"Money is numbers and numbers never end. If it takes money to be happy, your search for happiness will never end."
After near perfect water loading/cutting and cutting 4 pounds in the hot bath yesterday night, I awoke at 150 lbs this morning. I have a few sips of cold water and a couple low-weight foods and endure one more hour of hot bath work to cut the final 4 lbs. I am drained, but not the worst I ever been when I weigh in at 146 lbs on the dot.
Afterwards, rehydration and relaxing for my fight tomorrow.
11 – 11:30am
Light work the morning of the fight to shake my adrenaline off and get used to moving with my weight back on my frame.
At night, I am feeling heavier and more tired than usual. I am not sure if this is because of the heavy load of workouts this week, the weight cut, or nerves. Most likely all 3. I go out there and dominate the striking for the first two and a half rounds, only really getting hurt at the end of the 2nd round. I finish the fight on top almost cinching a kimura/armbar and win a one sided unanimous decision. The happiest I've ever been after a fight because I truly earned this win!
5/22 – 5/28
I'm back home to relax after a difficult camp, with my family. I get a 3 mile run in with my brother on monday morning and a 5 mile run in myself on thursday morning. I get a heavy weights workout done on friday morning and my strength feels the same as before. Great. I'm coming off this fight in the best shape I've ever been, ready to get better than ever!
12pm – 2pm
Sunday mornings at st. pete. I can't grapple because my face is still quite swollen, but I get some good striking in with Anton.
12pm – 2pm
The gym is closed for memorial day, but that doesn't stop us from getting in a class going anyway. We work on some bump techniques, getting out to the side when we initiate attacks. Then we do some really fantastic wrestling drills.
6:30pm – 7:30pm
BMT class in the evening. Back at it, good to see familiar faces!
9am – 10am
BMT with Jason as my partner. I am exhausted at the end of this class, I think I have been dieting too hard and not sleeping enough. But it's alright because I have no fight coming up!
3pm – 4pm
Great run in the afternoon, about 5 miles at a good, steady pace.
"A paradox of life: The problem with patience and discipline is that developing each of them requires both of them."
9am – 10am
BMT morning rounds with Jason
I had a chiropractic appointment after class to get my neck fixed up.
7:30pm – 8:30pm
Grappling with Dan Martinez. Had a great and focused BJJ class. Good rolls and my Jitz feels awesome.
11am – 1pm
Padwork, wrestling work, and sprints to finish todays workout.
10am – 11am
Final rounds of padwork before this weekend.
"Someday, we will all die."
"True, but on all the other days, we will not."


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If skydiving is on your 'bucket list' you really need to check out tandem skydiving with us here at El Paso Skydiving! Join us here at El Paso Skydiving for the most breathtaking ways to experience the fun and excitement of complete freefall sky diving over Texas. Skydiving is among life's greatest and best adventures. The feeling is indescribable, and the feeling of euphoria lasts for days. Discovering skydiving definitely will change your life!
El Paso Skydiving School skydiving centers provide two method of starting on your way to skydive certification, Accelerated Freefall (AFF) and Tandem Progression, making sure that you signed up for the curriculum that is right for you. Whether you want to learn to skydive solo or are interested in a career in skydiving, El Paso Skydiving School can assist you!
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Have you ever really wanted to know how it feels to fly? Parachuting describes the process of skydiving out of an airplane and descending slightly to the earth. While it may feel like flying, skydiving is a bit different. The degree of security provided by having a parachute is the predominate difference. Your chute slows your descent, not only making your freefall safer, but also providing you with a beautiful canopy tour thousands of feet in the air!
They call the first jump a "tandem skydive", due to the presence of a skydive instructor. Your tandem instructor will be with you the entire time! This gives you the opportunity to focus more on the scenery rather than focusing on saving your own life. Once your free fall is over, the instructor will launch your parachute providing a beautiful glide before a safe landing.
When it comes to skydiving, there's a an altitude range that's necessary to reach. Typically this range is between 10,000 and 14,000 feet. That's between 2.5 and 3 miles straight off the ground! Limitations remain in place for jumping as the higher you go the less breathable oxygen you have.
You'll be taught on how each of your parachutes are used before you leave the ground.
Your second chute is there in case the first one malfunctions. This chute will release when you hit low altitude with a malfunctioned main canopy. Often in professional training, skydivers can practice and learn the release times of a parachute while being connected to the airplane. This method of parachute training is referred to as "Static line jumping" which is much like bungee jumping, but you are high in the air rather than on a stationary structure.
Light planes such as the Cessna and Otter are operated most often for skydiving. Apart from recreational sport use, light aircraft are excellent for freight transport and aerial photography. However, most of these planes are made and designed exclusively for skydiving as they climb much faster than usual aircraft allowing divers to jump up to 4 times in one hour!
It can take anywhere between 10 and 20 minutes to reach diving altitude, depending on the plane. Unsurprisingly, the size of the engine determines how fast it gets up to this point. The quicker planes, like the King Airs with PT6A-20 engines, are regarded as the fastest in the world in their ascent.
Not only do these particular planes ascend rapidly, they can hold up to 15 people. {The King Airs, however, are only the second most popular aircraft used for diving.} The most prominent twin and single engine planes for skydivers are the Havilland Canada DHC-6-Twin Otter, and the Pacific Aerospace PAC P-750 XSTOL, respectively. Whether piston-powered or turbine-powered, aircraft optimized for skydivers have wide doors. Turbine and piston-powered planes are separated by a key difference: turbine planes are lightweight and use their fuel more efficiently. While they supply a similar horsepower as a traditional piston engine, crafts with turbine engines are typically more expensive.
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Blog Entry# 352844
Posted: Feb 18 2012 (8:33PM)
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Last Response: Feb 22 2012 (3:16PM)
Feb 18 2012 (8:33PM) 22817/Howrah - Mysuru Weekly SF Express (PT)
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Finally HWH-MYS starting its journey.
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Feb 18 2012 (11:04PM)
Begins Falls Rises ™*~ 21448 blog posts 1207 correct pred (63% accurate)
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its error in the TT!! can update it properly!!
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Morning star Mercury and a nightly stellar lineup this week
Additional rain heightens flood threat on B.C.'s North Coast
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"I am still convinced that a good, simple, homemade cookie is preferable to all the store-bought cookies one can find." – James Beard
More in News
No mechanical issues found with Legacy High School bus that crashed at DIAPolice say they have found no signs of mechanical issues with the Legacy High School bus that crashed earlier this month at the Denver International Airport, killing the driver and injuring 18 others aboard.
Fourth day of search for climber in Maroon Bells area underwayDavid Cook, 49, planned to summit Pyramid Peak and Maroon and North Maroon peaks last week. When he failed to return as planned, a woman reported him missing.
Police shoot, kill man in San Diego area; protesters gatherA black man reportedly acting erratically at a strip mall in suburban San Diego was shot and killed by police after pulling an object from his pocket, pointing it at officers and assuming a "shooting stance," authorities said.
Deal reached to keep U.S. government running, help FlintCongressional leaders have broken a stalemate over money to address the Flint, Michigan, water crisis, clearing the way for a spending bill needed to keep the government running until December.


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Starburst launched an aggressive U.S.-based mobile marketing promo on Friday as part of a new integrated campaign.
Grey Interactive and mobile marketing partner ipsh! are behind the cross-carrier effort, which follows a relaunch of the MasterFoods candy brand. The new identity promises "More Juice. More Burst."
Last week, MasterFoods began distributing the first of 100 million Starburst packs with unique wrapper codes that will eventually reach store shelves. Consumers can enter to win prizes by texting their unique numbers to the short code "JUICY." One pack design shows a teen holding out his phone and offers the copy, "Text. You Could Win Juicy Prizes." Smaller type below it says, "Look under wrapper. Text game code to JUICY (58429) or log on to starburst.com."
"Starburst is the most progressive brand for mobile marketing," said Nihal Mehta, ipsh! founder and president. "This is the largest cross-carrier mobile campaign for a consumer packaged goods [brand] in the U.S. to date."
Sweepstake giveaways include a 42-inch flat-screen TV, portable audio centers, and music downloads. The promotion continues through September.
Grey Interactive has handled Web and interactive advertising for Starburst since the late '90s. Last year, the MasterFoods brand gave its offline business to TBWAChiatDay, but Grey Interactive retained some digital assignments.
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Training Course
Introduction to Finding Grants
Self-Paced eLearning
Live Webinar
Recorded Webinar
Are you new to the field of grantseeking?
Discover what funders are looking for in nonprofits seeking grants and how to find potential funders in this introductory course.
You will learn the 10 most important things you need to know about finding grants, including:
- Who funds nonprofits and what are their motivations.
- What do funders really want to know about the organizations they are interested in funding.
- How do you identify potential funders and make the first approach.
- In-person classes will end with 30 minutes of hands-on, guided online grant research. It is advisable, but not necessary, to bring a laptop/tablet for this portion of the class.
Please Note:
- This orientation is not intended for individuals seeking grants for their own use. Please refer to our Resources for Individual Grantseekers.
This course qualifies for 1.5 CFRE Education Credits.
Online Classes
Introduction to Finding Grants WebinarRecorded Webinar
This brief recording captures the key concepts of the course. For more expansive discussion of the topic, we suggest attending a live webinar or in-person session. Note the recording is captioned for individuals with hearing impairments.
Introduction to Finding Grants OnlineSelf-Paced eLearning
This online course is self-paced and includes video, graphics, and interactive quizzes. Compatible with updated versions of all browsers and mobile-friendly!
Upcoming Course Dates
5:00pm - 6:00pm
9:30am - 11:30am
9:00am - 10:00am
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10:00am - 11:30am
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Training Event Calendar
Attend in-person grantseeker training courses that combine the Foundation Center's expert advice on grantseeking strategies with writing and research activities that put theory into practice. Learn more and sign up»


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Gipsy Lane, Headington
£500,000 Guide
Frenchay Road, The Central...
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I rented a flat for two years through Scott Fraser, and they were exceptionally responsive, organized, and professional. My property manager was Gareth Thomas, who was very friendly, attentive, and supportive throughout my tenancy. He was always quick to respond to any inquiries or issues that arose, and he made an extra effort to help me navigate an international move. I recommend Gareth and Scott Fraser most highly
I recently had to sell my mother's house to pay for her care home . This was a complex sale as the property was owned jointly by a trust and myself in the capacity of Attorney for my mother. Oliver very tactfully and thoroughly kept tabs on all parties to the proceedings including the solicitors. He brought to fruition a very satisfactory and speedy sale. His professionalism was matched with his cheery nature. Nothing was too much trouble and he negotiated the maze of detail very...
Jenny and the team at Scott Fraser provided a calm, friendly and professional service throughout the process of purchasing our new home. Nothing was too much trouble for the team and despite being part of a lengthy chain they concluded the purchase with minimal stress usually associated with moving house. When I sell again in Oxford I will go straight to Scott Fraser....
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Great customer service from Scott Fraser. It was a pleasure to work with them. Oliver Saxton was very helpful and made great suggestions. We had a great experience from this professional estate agent and would highly recommend them. Thanks to all at the Summertown branch and Oliver especially. You will be the first agency to call when we buy/sell a house again!
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A huge thank you, Mr T & Miss B


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Punishment History and Spillover Effects: A Laboratory Investigation of Behavior in a Social Dilemma
Punishment has been shown to be an effective reinforcement mechanism. Intentional or not, punishment will likely generate spillover effects that extend beyond one's immediate decision environment, and these spillovers are not as well understood. We seek to understand these secondary spillover effects in a controlled lab setting using a standard social dilemma: the voluntary contributions mechanism game. We find that spillovers from punishment lead to either more or less cooperative behavior depending on the history of play. If subjects have direct experience with a punishment mechanism, they will contribute more to the public good after observing others' punishment. The reverse is true of those who observe others' punishment but have no exposure to direct experience with punishment. Key Words:
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| Postal: Thelma C. Raley Hall, Boone, North Carolina 28608
Phone: 828-262-2148
Fax: 828-262-6105
Web page: http://economics.appstate.edu/
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References listed on IDEAS
Please report citation or reference errors to , or , if you are the registered author of the cited work, log in to your RePEc Author Service profile, click on "citations" and make appropriate adjustments.:
- David Dickinson & Marie Claire Villeval, 2008. "Does Monitoring Decrease Work Effort?," Post-Print halshs-00276284, HAL.
- Charles Noussair & Steven Tucker, 2005. "Combining Monetary and Social Sanctions to Promote Cooperation," Economic Inquiry, Western Economic Association International, vol. 43(3), pages 649-660, July.
- David Masclet & Charles Noussair & Steve Tucker & Marie Claire Villeval, 2003.
"Monetary and non Monetary Punishment in the Voluntary Contributions Mechanism,"
halshs-00144848, HAL.
- David Masclet & Charles Noussair & Steven Tucker & Marie-Claire Villeval, 2003. "Monetary and Nonmonetary Punishment in the Voluntary Contributions Mechanism," American Economic Review, American Economic Association, vol. 93(1), pages 366-380, March.
- David Masclet & Charles Noussair & Steven Tucker & Marie Claire Villeval, 2002. "Monetary and non Monetary Punishment in the Voluntary Contribution Mechanism," Post-Print halshs-00176878, HAL.
- David Masclet & Charles Noussair & Steven Tucker & Marie Claire Villeval, 2001. "Monetary and Non-Monetary Punishment in the Voluntary Contributions Mechanism," Post-Print halshs-00151423, HAL.
- Masclet, D. & Noussair, C. & Tucker, S. & Villeval, M.C., 2001. "Monetary and Non-monetary Punishment in the Voluntary Contributions Mechanism," Purdue University Economics Working Papers 1141, Purdue University, Department of Economics.
- David Masclet & Charles Noussair & Steven Tucker & Marie Claire Villeval, 2003. "Monetary and non Monetary Punishment in the Voluntary Contributions Mechanism," Post-Print halshs-00175251, HAL.
- Xiao, Erte & Houser, Daniel, 2011.
"Punish in public,"
Journal of Public Economics,
Elsevier, vol. 95(7-8), pages 1006-1017, August.
- Xiao, Erte & Houser, Daniel, 2011. "Punish in public," Journal of Public Economics, Elsevier, vol. 95(7), pages 1006-1017.
- Bendor, Jonathan & Mookherjee, Dilip, 1990. "Norms, Third-Party Sanctions, and Cooperation," Journal of Law, Economics and Organization, Oxford University Press, vol. 6(1), pages 33-63, Spring.
- Haigner, Stefan & Kocher, Martin & Sutter, Matthias, 2006. "Choosing the Stick or the Carrot? Endogenous Institutional Choice in Social Dilemma Situations," CEPR Discussion Papers 5497, C.E.P.R. Discussion Papers.
- David Dickinson, 2001.
"The Carrot vs. the Stick in Work Team Motivation,"
Experimental Economics,
Springer, vol. 4(1), pages 107-124, June.
- David Dickinson, . "The carrot vs. the stick in work team motivation," Working Papers 2000-06, Utah State University, Department of Economics.
- Anderson, Lisa R & Stafford, Sarah L, 2003. "Punishment in a Regulatory Setting: Experimental Evidence from the VCM," Journal of Regulatory Economics, Springer, vol. 24(1), pages 91-110, July.
- Urs Fischbacher, 2007. "z-Tree: Zurich toolbox for ready-made economic experiments," Experimental Economics, Springer, vol. 10(2), pages 171-178, June.
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HourlyHours to be determined
Entry Level
Start DateJune 8, 2014
We maintain a tracking solution for our fleet-customers based on the project traccar.
We need for our Tracking Solution a modification for the iphone and android client:
- Fixed IP-Adress defined to access our tracking server from the Apps and other small defined modifications.
About the Client
Rapperswil 03:48 AM
Jobs Posted
49% Hire Rate, 1 Open Job
Over $20,000
Total Spent
60 Hires, 13 Active
Avg Hourly Rate Paid
2,792 Hours


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Text reader spy app, tracking skype messages
Text reader spy app, tracking skype messages
Text reader spy app - ufone sms tracking
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Salesforce blurs the marketing, CRM lines
At Salesforce Connections, 2015, speakers joined the dots between marketing and CRM
Meet The Marketer: Sahana Jayaraman, Eastwick's head of digital and content marketing
Comms agency Eastwick's top social marketer reveals her tools of the trade.
Review: WeLink is a social media listening platform that can target hyper local conversations
Customer review of social media listening platform WeLink.
Marketing clouds vs. open platforms: Which is better for your company?
Is it better to buy all your marketing tools from a single source, or to pick the best from several different vendors?
With "Social Studio," Salesforce introduces a powerful integration of Buddy Media and Radian6
Integrated social media marketing for its ExactTarget Marketing Cloud
Who is winning the Marketing Cloud wars?
Adobe, Salesforce, Oracle, IBM and HP battle it out in the quest for digital marketing cloud supremacy.
Salesforce Radian6 enhances Insights for Big Data
Salesforce Radian6 announced July 17 enhancements to its Insights solution stack designed to structure and analyze Big Data in the social media space.
Kana Software acquires social listening company Overtone
Kana Software, a service experience management technology provider, acquired Overtone, a maker of social media and customer listening platforms, on April 5. The deal will allow Kana clients to understand and analyze customer sentiment, emerging issues and trends across social networks.
First-quarter marketing and interactive services M&A activity up 13%
The total value of marketing and interactive services mergers and acquisitions increased 13% year-over-year to $2 billion during the first quarter of 2011, according to a report by investment banking firm Jordan, Edmiston Group. Notable Q1 marketing and interactive services transactions included Salesforce.com's acquisition of Radian6 for $326 million; Teradata's acquisition of Aster Data Systems, a data and analytics platform, for $263 million; and Adobe's acquisition of data management platform Demdex.
Salesforce.com to acquire Radian6
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Hot Topics
Chicken Fingers
Note for babysitters:
This recipe contains milk or eggs.
Ask parents about any food allergies before
making this recipe for kids.
Using high-fiber cereal in place of traditional breadcrumbs makes this a healthier version of a kid-favorite meal.
Prep time: 10 minutes
- 1 4-oz. boneless, skinless chicken breast, rinsed, patted dry, and sliced into strips or 4 1-oz boneless, skinless chicken tenders
- 1 egg or ¼ cup egg substitute or ¼ cup skim milk
- 1/3 cup cereal flakes, crushed (preferably cereal with 3g or more of fiber per serving)
- Preheat oven to 350º F (176º C).
- Dip chicken strips into egg, egg substitute, or skim milk.
- Roll dipped chicken in high-fiber cereal to coat.
- Place coated strips on nonstick baking sheet.
- Bake for 18 to 20 minutes, turning after 9 minutes, until chicken is done (it must be white, not pink, inside).
Serves: 1
Serving size: 1 chicken breast
Nutritional analysis (per serving):
227 calories
30 g protein
3 g fat
0.8 g sat. fat
20 g carbohydrate
8 g fiber
75 mg cholesterol
239 mg sodium
44 mg calcium
10 mg iron
185 mcg folic acid
Note: Analysis may vary depending on ingredients used.
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© 1995- The Nemours Foundation. All rights reserved.
Images provided by The Nemours Foundation, iStock, Getty Images, Corbis, Veer, Science Photo Library, Science Source Images, Shutterstock, and Clipart.com


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Hey guys...
I will soon come to a crossroad in my academic career as I only have 3 more semesters to earning my B.S. in Aerospace Engineering. Looking back, I am having trouble justifying why I chose my major even though I've had an inherent passion for commercial aviation.
Like many of you, I want to one day be in the left seat of an airliner. What is the best route you would recommend? Is there some kind of program which earns you a Master of Science degree while training you to fly?
While many of my school budies go on to work in the industry for Boeing, Lockheed Martin, and such or go on to grad school, I really want to start racking up my hours.
Basically, I am trying to build on my current education. Not start over. Too many all-nighters, too much crap I've put up with these past 4 years in school, and most of all, too much money invested
Thanks for any advice.
Alex Ly '01