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|"Margaret", "Margorie", "Margery", "Margary of Scotland", "Mary of Bruce", "Princess Marjory", "16182"
|Dundonald, Kyle, Argyllshire, Scotland
|Died in Paisley, Renfrewshire, Scotland
|Cause of death:
|Complications of childbirth following successful caesarian operation
|Managed by:
|Sally Gene Cole
|Last Updated:
Historical records matching Marjorie Bruce, Princess of Scotland
Immediate Family
About Marjorie Bruce, Princess of Scotland
[NOTE: Please maintain this as a separate profile from that of Margaret Bruce, possibly her younger half-sister.]
Margorie Bruce, Princess of Scotland was born circa 1297 and died on 2 March 1316 at Paisley, Renfrewshire, Scotland, in childbirth. She was buried at Paisley Abbey, Paisley, Renfrewshire, Scotland. (3, 4) From 1315, her married name became Stewart. Marjorie was named after her father's mother, Marjorie of Carrick.
- Parents: eldest daughter of Robert I Bruce, King of Scotland by his first wife, Isabella, Lady of Mar (her only child; she died shortly after giving birth). (2)
- Paternal grandparents: Robert de Brus, 6th Lord of Annandale and Marjorie of Carrick, 3rd Countess of Carrick.
- Maternal grandparents: Donald, 6th Earl of Mar and Helen ferch Llywelyn.(1)
- in 1315 to Walter Stewart, 6th High Steward of Scotland, son of James Stewart, 5th High Steward of Scotland and Gille de Burgh.
Child of Margorie Bruce, Princess of Scotland and Walter Stewart, 6th High Steward of Scotland:
- Robert II Stewart, King of Scotland+ b. 2 Mar 1315/16, d. 19 Apr 1390
Her marriage to Walter, High Steward of Scotland united Clan Stewart and the royal House of Bruce, giving rise to the House of Stewart. Her son was the first Stewart monarch, King Robert II of Scotland.
Brief Biography
from: Children of Robert Bruce: Blog, by Laura Vosika, author of the "Blue Bells Trilogy"
Most sources agree Marjory was born in December of 1296, the same month Longshanks invaded Scotland and took Berwick. As an author, I could hardly write better foreshadowing for the life Marjory would lead. In June 1306, at the age of 9, she was captured at St. Duthac in Tain, north of Inverness, while trying to escape to safety in Orkney. It is all too easy to imagine the terror of a 9 year old girl, separated from her father, who she knows is fighting not just for his kingdom, but for his life, seeking safety in a church with her aunts and step-mother, and seeing armed men storm into what should have been a place of refuge and safety. It is easy to imagine the terror of wondering what had become of her Uncle Nigel who had tried to protect them, still under attack back at Kildrummy; or what would become of Sir John of Atholl, who had whisked them away from Kildrummy for safety, or her aunts and step-mother.
We know that two of the women captured in the church that day–Isobel MacDuff and Mary Bruce, Marjory's aunt–would spend years living in cages hung on castle walls. Edward I had a similar cage built for Marjory at the Tower of London, but in a rare moment of softness, reconsidered and instead ordered her held in solitary confinement in the nunnery at Watton. There, the young Marjory lived, virtually alone, for 8 years. She was released after Bannockburn in 1314, when she was still 17, in exchange for English prisoners held by the Scots.
The following year, she married Walter, the 6th High Steward of Scotland, who was only 22 himself at the time, but one of the heroes of Bannockburn. She very quickly became pregnant. The following March, she rode her horse in the late stages pregnancy, fell when it reared, and delivered the future Robert II by c-section on March 2, 1316, according to Electric Scotland. (link6)
She died at the age of barely 19, having spent close to half her life in near-solitary confinement. It hardly gets more tragic than this.
- Medieval Lands: Kings of Scotland (Bruce)
- the peerage.com
- The Bruce Children
- Wikipedia: Marjorie Bruce
- History of Paisley: Marjory Bruce
- Women in History of Scots Descent: Marjory Bruce
- Heir presumptive
- Paisley Abbey: Cradle of the Royal House of Stewart
- Sewell's Descent of the Kings of Scotland
- [S323] Sir James Balfour Paul, The Scots Peerage: founded on Wood's edition of Sir Robert Douglas's The Peerage of Scotland (Edinburgh, Scotland: David Douglas, 1904), volume I, page 14. Hereinafter cited as The Scots Peerage.
- [S323] Sir James Balfour Paul, The Scots Peerage, volume 1, page 8.
- [S11] Alison Weir, Britain's Royal Family: A Complete Genealogy (London, U.K.: The Bodley Head, 1999), page 209. Hereinafter cited as Britain's Royal Family.
- [S11] Alison Weir, Britain's Royal Family, page 214.
- Royal Ancestors of Magna Charta Barons; Page 225; G929.72; C6943ra; Denver Public Library; Genealogy
- The Royal House of Stuart; Page ix; G929.7; A224ro; (oversized) Denver Public Library
Marjorie Bruce
From Wikipedia, the free encyclopedia http://en.wikipedia.org/wiki/Marjorie_Bruce
Spouse Walter Stewart, 6th High Steward of Scotland Issue: Robert II of Scotland
Father Robert I of Scotland Mother Isabella of Mar
Born 1296
Died 2 March 1316 (aged 19–20)
Burial Paisley Abbey
Marjorie Bruce or Marjorie de Brus (1296 – 2 March 1316) was the eldest daughter of Robert the Bruce, King of Scots by his first wife, Isabella of Mar, and the founder of the Stewart dynasty. Her marriage to Walter, High Steward of Scotland gave rise to the House of Stewart. Her son was the first Stewart monarch, King Robert II of Scotland. Her father remarried, after a certain period of time, Elizabeth de Burgh.
Early life
Her mother, Isabella, a nineteen-year-old noblewoman from the Clan Mar, died soon after giving birth to her. Her father was then the Earl of Carrick. Marjorie was named after her father's mother, Marjorie, Countess of Carrick.
According to legend, her parents had been very much in love, and Robert the Bruce did not remarry until Marjorie was six years old. In 1302, a courtier named Elizabeth de Burgh became her stepmother.
On 27 March 1306, her father was crowned King of Scots at Scone, Perthshire, and Marjorie, then nine years old, became a Princess of Scotland.
Imprisonment (1306–1314)
The Priory of Watton, where Marjorie was imprisoned under Edward I Three months after the coronation, in June, 1306, her father was defeated at the Battle of Methven. He sent his female relatives (his wife, two sisters and Marjorie) north with his supporter the Countess of Buchan, but by the end of June the band of Bruce women were captured and betrayed to the English by the Earl of Ross.
As punishment, Edward I sent his hostages to different places in England. Princess Marjorie went to the convent at Watton; her aunt Christina Bruce was sent to another convent; Queen Elizabeth was placed under house arrest at a manor house in Yorkshire (because Edward I needed the support of her father, the powerful Earl of Ulster, her punishment was lighter than the others'); and Marjorie's aunt Mary Bruce and the Countess of Buchan were imprisoned in wooden cages, exposed to public view, Mary's cage at Roxburgh Castle and Countess Isabella's at Berwick Castle. For the next four years, Marjorie, Elizabeth, Christina, Mary and Isabella endured solitary confinement, with daily public humiliation for the latter two. A cage was built for Marjorie at the Tower of London, but Edward I reconsidered and instead sent her to the convent.[1] Christopher Seton, Christina's husband, was executed.
Edward I died on 7 July 1307. He was succeeded by his son, Edward II, who subsequently held Marjorie captive in a convent for about seven more years. She was finally set free around 1314, possibly in exchange for English noblemen captured after the Battle of Bannockburn (23 June – 24 June 1314).
Marriage and death
Walter Stewart, 6th High Steward of Scotland distinguished himself in the battle and was rewarded the hand of the adolescent princess. Her dowry included the Barony of Bathgate in West Lothian. The original site of Bathgate Castle, which was part of the dowry, can be found on the grounds of Bathgate Golf Club. The site is protected by the Historic Scotland organisation and the Club is debarred from carrying out any excavation work on the site without prior permission. Every year on the first Saturday of June, the town of Bathgate celebrates the marriage of Marjorie and Walter in their annual historical pageant, just before the town's procession and Newland festival. Local school children are given the parts of Marjorie, Walter and other members of the court. After the pageant, everyone joins the procession along with Robert the Bruce on horseback.
Two years later, on 2 March 1316, Marjorie was riding in Gallowhill, Paisley, Renfrewshire while heavily pregnant. Her horse was suddenly startled and threw her to the ground at a place called "The Knock." She went into premature labour and delivered the child at Paisley Abbey, surviving the birth by a few hours at most.
She was nineteen at the time of her death, like her mother, who was also nineteen years old when she died in childbirth.
At the junction of Renfrew Road and Dundonald Road in Paisley, a cairn marks the spot near to where Marjorie reputedly fell from her horse. While the reputed place of her death is now referred to as Knockhill Road, with nearby roads of Bruce Way, and Marjorie Drive named in her honour. She is buried at Paisley Abbey.
Her son succeeded his childless uncle David II of Scotland in 1371 as King Robert II. Her descendants include the House of Stuart and all their successors on the throne of Scotland, England and the United Kingdom.
Marjorie in fiction
The young adult novel Girl in a Cage by Jane Yolen and Robert J. Harris features Marjorie Bruce as its protagonist. In it, Marjorie is imprisoned in a cage. Although there is a preface stating that it is fictional, many have taken it to be a true story.
- Scott, Ronald McNair, Robert the Bruce, p. 87
External links
Bannockburn article (National Trust for Scotland) contains some information on Marjorie Bruce. Bathgate Castle and Old Hall Knowe
Robert Bruce, King of Scotland, was born the 11th of July 1274, and died June 7, 1329. He married first Isabella, eldest daughter of Donald, tenth Earl of Marr. Their daughter, Marjory, Princess Royal of Scotland, fell into the hands of the English 1306, and was detained a prisoner in charge of Henry Percy till 1314, when she was conducted to Scotland by Walter, the sixth high steward of Scotland, to whom she was married in 1315. She died in March, 1316. Her husband, Walter, born in 1294, brought a noble body of men to the aid of Bruce. In the battle of Bannockburn he and his cousin, Sir James Douglas, commanded the Third division. The same year he was appointed to receive, on the borders, the Queen of King Robert, Marjory, his daughter, and other illustrious Scottish prisoners. On that occasion he formed an attachment for the Princess. He died April 9, 1326. "Had he lived," says an old writer, "he might have equaled Randolph and Douglas; but his course of glory was short." The only child of the Princess Marjory was Robert Stuart, King of Scotland, born March 2, 1316.
Source: The Descent Of General Robert Edward Lee From Robert The Bruce, Of Scotland. By Professor Wm. Winston Fontaine, of Louisville. (From the Southern Historical Society Papers)
Marjorie Bruce or Marjorie de Brus (December, 1296 – 2 March 1316) was the eldest daughter of Robert the Bruce, King of Scots by his first wife, Isabella of Mar, and the founder of the Stewart dynasty. Her marriage to Walter, High Steward of Scotland gave rise to the House of Stewart. Her son was the first Stewart monarch, King Robert II of Scotland.
Source= http://www.patrickspeople.co.uk/ancestors%20of%20isabella%20gordon/2152.htm --------------------
The wife of Donald of the Isles, was Princess Margaret Stuart, granddaughter of King Robert the Bruce, through his daughter, the Princess Marjorie.
The village is mostly known for Dundonald Castle, which was built in the 14th century by king Robert II, on the ruins of a castle built earlier (in 1260 by his grandfather, Alexander Comyn. It served the Scottish kings for 150 years

subset: commoncrawl


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Angels & Demons
by Dan Brown
Publisher Pocket Star
Language English
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It takes guts to write a novel that combines an ancient secret brotherhood, the Swiss Conseil Europ�en pour la Recherche Nucl�aire, a papal conclave, mysterious ambigrams, a plot against the Vatican, a mad scientist in a wheelchair, particles of antimatter, jets that can travel 15,000 miles per hour, crafty assassins, a beautiful Italian physicist, and a Harvard professor of religious iconology. It takes talent to make that novel anything but ridiculous. Kudos to Dan Brown (Digital Fortress) for achieving the nearly impossible. Angels & Demons is a no-holds-barred, pull-out-all-the-stops, breathless tangle of a thriller--think Katherine Neville's The Eight (but cleverer) or Umberto Eco's Foucault's Pendulum (but more accessible).
Harvard symbologist Robert Langdon is shocked to find proof that the legendary secret society, the Illuminati--dedicated since the time of Galileo to promoting the interests of science and condemning the blind faith of Catholicism--is alive, well, and murderously active. Brilliant physicist Leonardo Vetra has been murdered, his eyes plucked out, and the society's ancient symbol branded upon his chest. His final discovery, antimatter, the most powerful and dangerous energy source known to man, has disappeared--only to be hidden somewhere beneath Vatican City on the eve of the election of a new pope. Langdon and Vittoria, Vetra's daughter and colleague, embark on a frantic hunt through the streets, churches, and catacombs of Rome, following a 400-year-old trail to the lair of the Illuminati, to prevent the incineration of civilization.
Brown seems as much juggler as author--there are lots and lots of balls in the air in this novel, yet Brown manages to hurl the reader headlong into an almost surreal suspension of disbelief. While the reader might wish for a little more sardonic humor from Langdon, and a little less bombastic philosophizing on the eternal conflict between religion and science, these are less fatal flaws than niggling annoyances--readers should have no trouble skimming past them and immersing themselves in a heck of a good read. "Brain candy" it may be, but my! It's tasty. --Kelly Flynn [via]


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text: General Wellness Webcast | Summer Health Risk: Lyme DiseaseMedically reviewed by Ed Zimney, MDLyme disease, which is spread by tick bites, can cause abnormalities in the skin, joints, heart and nervous system. Do you know how to protect yourself and what you should do if you get bit? Join us as experts explain what Lyme disease is, how the disease mimics other conditions like chronic fatigue syndrome, lupus, multiple sclerosis and rheumatoid arthritis and which symptoms you should look out for this summer, when ticks are most common. You'll also find out how Lyme disease is diagnosed, which treatments are available and what to do next if you think you may have it. As always, our expert guests answer questions from the audience. Announcer: The opinions expressed on this webcast are solely the views of our guests. They are not necessarily the views of HealthTalk, our sponsors or any outside organization. And, as always, please consult your own physician for the medical advice most appropriate for you. Judy Foreman: Hello and welcome to Health Now. I'm your host, Judy Foreman. Lyme disease, which is spread by tick bites can cause abnormalities in the skin, joints, heart and nervous system. The disease can be difficult to diagnose because it mimics other conditions like chronic fatigue syndrome, lupus, multiple sclerosis and rheumatoid arthritis. Tonight, we will talk about what you can do to prevent Lyme disease, how you can get an accurate diagnosis if you think you have it, and what treatment options are available. I am very pleased to welcome Dr. Lesley Ann Fein. She is the medical director for the Pennsylvania Lyme Disease Society and the Greater Hartford, Connecticut Lyme Disease Support and Action Group. Dr. Lesley Ann Fein, thank you so much for joining us tonight. Dr. Lesley Ann Fein: Sure. I'm honored. Thank you. Judy: I'm also very pleased to welcome Constance Bean. She in 1993 was diagnosed with Lyme disease and has spent the past 14 years researching its treatment while figuring and fighting out the disease herself. She is author of the book, "Beating Lyme: Understanding and Treating This Complex and Often Misdiagnosed Disease." Constance Bean, welcome to Health Now. Ms. Constance A. Bean: Nice to talk with you. Judy: Okay. Constance, I would like to start with you. Can you just get us started by telling us your story and how hard it was for you to get diagnosed properly? Ms. Bean: I knew that I had Lyme disease. I was well aware of the disease. I was at MIT. I was in health education. I was in public health. I knew all about it. I was handing out brochures on Lyme disease. And I had a rash. I had a tick bite. Everything was very classic. And I got the two weeks of standard treatment. Judy: And the treatment was antibiotics? Ms. Bean: Antibiotics, low-dose antibiotics were standard, and I continued to develop symptoms that got worse and worse, and I went to 24 doctors. Judy: Wow. Ms. Bean: And I was hospitalized about three times. Judy: For what? Ms. Bean: Because I had such severe symptoms, I was shaking and had chills. Judy: I am sure a lot of our listeners do know what the symptoms are, but just in case some don't, what were your symptoms, and why did you end up in the hospital? Ms. Bean: I had fatigue, and it was thought it might be chronic fatigue syndrome. I was told maybe it was fibromyalgia because of sore joints. Judy: Where did you get the tick bite? Did you have any idea? Ms. Bean: Oh, yes, at my home on Cape Cod, I think actually I got the tick bite in November because I was not outdoors again until May actually. Judy: Oh, wow. That's kind of late in the year. Ms. Bean: Well, ticks can be active anytime the temperature is above freezing, and it was a lovely day in November, and I was surveying some property that I had out there, walking around and seeing what my new land consisted of, and I had sweatpants on, and they were dark. And I would never have seen a tick anyway, and it was on my back, on the back of my hip. Judy: Now, did you see the tick bite on your skin? Ms. Bean: I discovered it in May. Judy: Oh, wow. Ms. Bean: It had been there all those months. It was deep-embedded, a black spot. And then I got a magnifying glass out, and it looked like it could even have legs. I didn't know. Judy: Oh, my. Ms. Bean: So I rushed off, within 15 minutes I was at the medical center. Judy: But this was five or six months after you probably got it. Ms. Bean: Yes, it was. I had no symptoms at all. I won't say this is typical because there are so many different ways things happen, but the basic pattern was the same: a tick bite and a rash. Judy: Okay. I want to jump now to you, Dr. Lesley Ann Fein. How did you get interested in Lyme disease? Dr. Fein: It was more of an intellectual curiosity. I was at Mount Sinai in New York doing my internship and residency, and we saw a case. Actually, we had one of the first cases. And he was a kid who was incredibly sick with high fevers and massively swollen joints. And on IV penicillin, which was used at the time, he completely resolved. And I just thought that was incredibly fascinating. And then we had grand rounds given by Dr. Steere talking about how it can… Judy: …well, let's just, in case our listeners don't know who Dr. Steere is, can you just give us a real quick… Dr. Fein: …well, he was one of the first rheumatologists who discussed it and published articles about it. So he was talking about it inducing rheumatoid arthritis and inducing lupus and inducing multiple sclerosis, and to me the concept of your immune system reacting against yourself was fascinating and also doesn't make sense from a sort of a historical perspective. Why would you evolve into an illness where you are attacking yourself? It didn't make any sense. So I was curious. Judy: But by then we already knew a lot about autoimmune diseases in general, didn't we? Dr. Fein: No, no, no, no. We didn't. We knew nothing about it. We just knew that it happened, but we didn't know why it happened. And this was the first demonstration of how an infection can bring it on and induce it, so that's why it was so fascinating. And if you go back into the literature like in the early 1920s, '30s, it was discussed in the literature that it was thought these autoimmune diseases could be induced by infections, but this was the first demonstration of an infection causing autoimmune disease, and I thought it was fascinating. Judy: And does it work that your immune system kind of detects the bacterium from a tick and mounts an immune response against that, and then there is some kind of like molecular mimicry thing? Dr. Fein: Well, that could be. I mean, over the years, we have come up with multiple different explanations for it. One would be exactly that, which means that as your immune system is recognizing this foreign invader, there is something about the foreign invader that is similar to your own tissue. And while you are trying to fight the invader, you are also attacking your own tissue. Judy: That makes sense. That kind of makes sense. Dr. Fein: Right. And then there is the genetic predisposition. There is clearly a genetic predisposition. So if you get the infection, does it switch on the gene? And if it switches on the gene, does it then become self-perpetuating, and treating the infection makes no difference if you have triggered the gene? And as this has gone on, we have sort of changed our hypotheses about why it happens, but it's still very interesting, and we still don't actually know all of the answers. Judy: And the genetic thing, is there any suggestion that a susceptibility to Lyme runs in families? Dr. Fein: No. Judy: Okay. Dr. Fein: What I meant by that was if you carry a certain gene, it will trigger a certain manifestation. So if you have the DR2 or the DR4 gene, it will trigger rheumatoid arthritis. If you have let's say B1, I can't remember, A1-BA-DR or I can't remember the DR, but if you have a certain sort of triplicate genetic complex, it will trigger something more like lupus and multiple sclerosis. Judy: I get it. So depending on your genes, you get the Lyme infection, and then those genes determine how it manifests itself? Dr. Fein: It sort of switches on those genes, so to speak. And then, of course, the debate is once it's been switched on, can we switch it off with antibiotics, or do we have to just treat whatever it has switched on? Judy: I see. Dr. Fein: And that's what the big fight is about and what the big debate is. Ms. Bean: Yes. I think that really is, and what's happened is that people have tended to go for the post-Lyme syndrome and ignore the fact of persistent infection. And because continuing antibiotics in many cases, and certainly in mine and many others, it was the length of time and the duration and the intensity of the antibiotics that took me through this. Judy: You mean it helped? Ms. Bean: It helped, definitely. Without it, I would have been in a very sad situation. I was really very, very sick. I think I was in the hospital three more times. Judy: Wow. Ms. Bean: But it took several years of treatment and 18 months of intravenous antibiotics. Judy: That's a long time. Ms. Bean: Plus a lot of oral antibiotics, and that's what helped me. Judy: How are you today, Constance? Ms. Bean: Yeah. I'm fine. I mean, I can relapse very easily, but fine. Judy: You mean you have to stay on antibiotics? Ms. Bean: I do stay on antibiotics. Yes, I do. And I have been off for a few months at a time and discovered that I do get symptoms again, and then they resolve when I take the antibiotics. Judy: Before we get any further into the treatment, I would like to back up for a second and, Dr. Lesley Ann Fein, ask you a little bit about some more of the background. As I understand it, there are about 20,000 Americans every year who are newly diagnosed with Lyme disease. Dr. Fein: Well, Judy, reported to the CDC. That's not really the same thing. Judy: Oh. So you think it's probably more that actually are diagnosed? Dr. Fein: Much more, much more. I mean, the CDC will acknowledge that only one out of 10 cases are actually reported, and it's probably far less than that. Most doctors don't have the time to call up the health department and ask for forms to report the cases. Judy: So there is no law saying they have to report it? Dr. Fein: Well, the labs, theoretically the labs are supposed to send it in to the health department, but in my experience they don't always do that. Judy: Okay. Let's just sort of lay that in the background for a minute more. So we have many more than 20,000 Americans… Dr. Fein: …far more. Judy: Far more, being diagnosed every year. And then something like, from what I have read, almost 2 million living with the disease in its chronic form. Tell us just for background, either one of you, what is the life cycle of these ticks? I mean, how do we get sick from these tick bites? What's the life cycle? Dr. Fein: Well, the life cycle and how you get sick are two different things. Connie, do you want to talk about the life cycle? Ms. Bean: Well, I will try to do that too. It's all interrelated, the disease, the ticks, the animals, the environment in which you live. The ticks are incredibly able to survive and to wait for a blood meal. They can go months without anything. They are just looking for a blood meal. And the ticks go through several stages. They become infected when they take their first blood meal, usually on a mouse or a rodent. Judy: From a mouse who already has the bacterium? Ms. Bean: That's right. Let's say, and each thing infects the next stage. So the uninfected larva becomes infected, and then when it takes its next meal on whatever it is, it infects the next thing that it bites. And if it's you, it's you. If it's a deer, it goes into the deer, and the deer become a reservoir, and every deer that then feeds on that infected deer will then get Lyme disease. So it tends to increase almost exponentially because things go round and round. Now, humans are a dead-end host. We don't take it anywhere else beyond ourselves, but the deer can continue to transmit it wherever they go, to all the ticks. There can be hundreds of ticks at any one deer's neck, and hunters will tell you that anytime. Dr. Fein: The concept of the reservoir means the animal, which are mostly the rodents, the mice, and then obviously the deer, the reservoir means they are not ill as a result of these infections. They just carry it in their blood, and the ticks acquire it from the reservoir, and then when the ticks infect the host, then the host becomes ill, but the host wouldn't transmit it any other way. In other words, the host was fine until it got bitten by the tick, and then it became ill from the tick-borne infection. Judy: But the deer and the mice don't get sick. Dr. Fein: But that's the difference between the reservoir and the host. Judy: I see. So the humans are the host. Ms. Bean: Yeah, that's right. Possibly through pregnancy it might be passed from one human to another, but otherwise it wouldn't. And if there were no human there, the ticks would continue to bite the deer and the mice. But people are out in their gardens now, and they are out a lot. And they are in the woods, and there are many more ticks now. And there are many more infected ticks, and the climate has changed. Judy: So what does a tick bite look like, and does it have to be this kind of classic, bull's-eye thing, or can it also be a more subtle thing? Dr. Fein: There have been a couple of studies on that. There was a study at NYU where Dr. Franks, who is a dermatologist and a rheumatologist, did biopsies on skin lesions that were a variety of shapes and sizes, and of those that were positive on biopsy for Lyme, less than 50 percent of those were the classic bull's-eye lesions. And then the second study was a very large study that was a vaccine trial where there were 10,000 people involved, and every single rash that occurred during the trial period had to be biopsied. And of those that were biopsied positive for Lyme, again less than 50 percent of those were classic bull's-eyes. So, in fact, the rashes, firstly the rashes don't have to occur at all. But if they do occur, they can be completely what we call atypical and are more than 50 percent likely to be atypical rather than typical. Ms. Bean: That's right. I have seen very few actual bull's-eyes myself. But if you have a spreading, pink, red rash that just continues to expand, and I know mine continued to expand for a number of days after the tick was removed, I didn't have it until the tick was removed, and suddenly everything kind of came to life, and anyway it continued to spread in all directions. Judy: Dr. Lesley Fein, is there anything that a person can look at their own skin or their kid's skin and say, "Oh, gee, that probably is a tick bite?" Or is there just absolutely no way? Dr. Fein: Oh, I mean, if you are lucky enough to have a circle, a red circle with a pale center that expands over a period of days to weeks, then that's the so-called bull's-eye rash, and it can have multiple circles, so it can sort of look like a dart board. But most people, as I say, aren't going to have that. And the ticks like places like the nape of the neck and the armpits and the groin, and those are places where you don't even look. Judy: Right. Dr. Fein: So even if you had the rash, you probably wouldn't notice it because it's painless usually, although that's another myth. I see a lot of rashes that are very intensely painful, and those might be the rashes where the tick is also transmitting some of the co-infections. But the rashes can be very pale and transient and just last for hours to days, or they can be intensely red and nasty and oozy and pusy. There is a tremendous variety of manifestations. Ms. Bean: Yeah. Only half of the people with Lyme disease ever get a rash at all or ever see a rash. Dr. Fein: Well, that could be a retrospective analysis. That's the thing. There are no prospective analyses. I mean, nobody is going to enroll themselves in a trial where they say, "All right, well, let me get infected, and let's see what happens." When you do retrospective analysis of people who have been diagnosed with Lyme, less than 50 percent of those people recall having had a rash, and they may have had it, but they don't recall seeing it. Judy: Dr. Fein, if you have a rash that you think might be Lyme, should you go right to the ER or wait a week to call your doctor or what? Dr. Fein: Yeah. You see, that's where it becomes very difficult for me to answer those questions because a lot of family doctors and internists aren't aware of the various manifestations that these rashes can present, and a lot of people are told that they have spider bites when it's actually a tick bite. And clearly if somebody has an unexplained rash, especially during the what we would call most endemic seasons and areas, they should be suspicious, and they should have it looked at. But sometimes a doctor has to just treat empirically or present to the patient the option saying, "I don't know for sure. But since you live wherever you live, and since you could easily have been exposed, we can treat you empirically or we can wait." I mean, I don't force people to be treated at all. Because if I don't know for sure, I am not going to force them. But if I have somebody, let's say, has a rash and then a swollen lymph node draining that rash, then I am going to say to them, "There is a very high likelihood this was a bite and that you should be treated." Judy: Okay. The bacterium, how do you say it, the Borrelia burgdorferi, gets into the bloodstream, and I have read that it can get into the brain within hours, even while the person is still completely asymptomatic. Dr. Fein, how can that happen that fast? Dr. Fein: It just, it does. We have isolated it in the spinal fluid of completely asymptomatic people within 24 hours. If it spreads through the bloodstream, it just needs sort of the one pass go around to infect any part of the body. So why those people were asymptomatic is interesting, but it's a fact, so. It goes wherever it wants to go. It usually spreads locally first. It burrows into the skin, and then it goes into the lymphatics and the veins and then spreads to the circulation. And then it chooses. Where it wants to go seems to be a mystery. We don't know for sure. Is it partly the different subspecies prefer different parts of the body? Is it genetic? We know that it goes to previously injured joints, for example. So it ends up in multiple places, but it has been isolated from spinal fluid within 24 hours of the bite. Judy: So you get a bite. Constance, let me just pursue the chronology of this for a second. You get a bite. The bacteria end up in your bloodstream, and then I would assume, Dr. Fein, that your immune system catches on quickly and starts making antibodies. Is that right? How long does it take to start making antibodies? Dr. Fein: That's again, nobody has done a study. Well, we did, we did a little study in the '90s. And we started with people who came in with ticks and rashes, and we actually did serial Western blots, and some of them… Judy: …oh, wait. Whoa, whoa. Can you just explain what a serial western blot is? Dr. Fein: I'm sorry. It's a blood test. Judy: Yeah. Dr. Fein: And some people did start to make antibodies pretty quickly, but if you treat them, they may not make antibodies at all. So if you treat right from the start, you may never have a positive test. But actually in those days, we were detecting antibodies as quickly as two weeks after the removal of the ticks. Judy: So let's just run through, start with Dr. Fein and then, Constance, you can add on if you would. Dr. Fein, so what are the tests that can potentially diagnose Lyme? I know they are flawed, but what's the logic there? What are the tests? What are they? Dr. Fein: Well, it depends on the doctor. If somebody walks into my office tomorrow, and they have got a tick, an engorged deer tick still on them, for example, I will remove the tick, and I will send it out for testing, which is what's called PCR, which is looking for the actual infections. And I will also send their blood out for PCR because you can find a positive PCR within hours of a bite. Judy: PCR, just for our listeners, is polymerase chain reaction, and it's basically amplifies the DNA of whatever you are looking at. Dr. Fein: So that would be looking for the various infections, and I test for all the infections. I test for five tick-borne infections. Judy: Okay. Dr. Fein: And then in those patients, I will put them on antibiotics, and then repeat, then start doing antibodies. So then the antibody tests would start to come up, maybe or maybe not, in a couple of months. But the antibody tests that are available are called ELISAs, and you can have an IgM or an IgG ELISA. Judy: Wait. I am not sure all our listeners are quite as up to speed as you two, but those would be tests for antibodies to whatever infection there is. Dr. Fein: Antibodies, antibodies. Right. Judy: Okay. Right. Dr. Fein: There is something called a C6 peptide assay, which is highly accurate, but most people don't do it, and most labs don't do it, but it would be highly accurate for early Lyme. There is something called a Western blot, which should be done on everybody but is almost never done if the ELISA is negative. And that's a mistake. That's a huge mistake because in about 30 percent of people who have Lyme, they can have a negative ELISA and a positive Western blot. So everybody should have a Western blot. Judy: Let me just ask you, Constance, in your experience as an advocate, what tests should people ask for? Ms. Bean: Well, what I say in "Beating Lyme: Understanding and Treating This Complex and Often Misdiagnosed Disease," in the book, is I explain the tests, exactly what they do and what they don't do and how they work. I put it in very simple terms. Judy: Dr. Lesley Ann Fein, back to you for a second, and this is a pretty straight medical question, how does Lyme disease mimic these other autoimmune diseases or the autoimmune diseases like lupus or rheumatoid arthritis? Dr. Fein: Well, that would be more in the chronic phase, although I have seen Lyme induce, let's say, a lupus-like syndrome within weeks. But normally it's in the chronic, undiagnosed patients that it slowly turns into what would look like, let's say, rheumatoid arthritis or lupus or multiple sclerosis. And in those cases, by the time the autoimmune disease has been initiated, the blood test for Lyme is most likely going to be negative. So in those people, you are most likely going to find positive lupus tests and maybe positive rheumatoid factor and a negative Lyme test, and those are the ones that are the most difficult to diagnose and the most controversial. Judy: So are you saying that a tick bite with Lyme can actually cause lupus? Dr. Fein: Right. In a certain strain of mice, for example, if you inject Lyme spirochetes into them, they turn into lupus mice. And if you test them, they don't show positive antibodies for Lyme. They show lupus. It actually turns into lupus, and that's the point. The point is that somebody can walk into your office and look like they have lupus or look like they have rheumatoid arthritis, and it was first triggered by Lyme, and that's where diagnostic skills come into play. You need to listen to the history. You also need to test for all the other infections. So let's say I have got a patient who has a 10-, 11-, 12-year history of a progressive illness, and it did start off with maybe a flu-like illness, and maybe not, but if they have a positive test for lupus but they also have a positive ehrlichia test, or a Rocky Mountain spotted fever test, or a Babesia test or something like that, which would tell me that that person had been exposed to a tick bite, and depending upon the symptoms that they had over the past 10 or 11 years, I can build a case to say to them, "Your initial infection was a tick bite, and it has now initiated the progression of symptoms that has brought you to this point, and you are being treated for lupus." But I think if we try and treat the underlying infection, we can get rid of the lupus as well. Judy: Yeah. Dr. Fein: But sometimes you actually treat both. Judy: I was going to say it sounds like you probably have to treat both. Dr. Fein: I end up treating both, but it's a matter of explaining to people how they got there. And with me, I take three to four hours with a new patient because it takes a very long time to go through the history and to look at the records and to build a case to decide if this is definitely just what I would say is pure lupus and rheumatoid arthritis or whether it's not. So it's complicated. Judy: We have a couple of e-mail questions that have come in that I would like to get to. We do have an e-mail from Joan in Lynchburg, Virginia, and she writes, "My husband was misdiagnosed, and the doctor actually refused to give him a Lyme test because he said it was not common in our area." That's Virginia. "The Lyme disease was found in his spinal cord. He still has his share of aches and pains, but is there any possibility that he could have dementia or neurological problems? He did undergo intensive antibiotics, intravenous therapy for a month or so. Dr. Fein, I know you can't diagnose someone you have never even talked to, but do you have any suggestions for this couple? Dr. Fein: If the patient still has symptoms, I do what's called a SPECT scan, which looks at inflammation of the brain. It's much more sensitive than an MRI, and it's done in a couple of places. By far, the best place would be Columbia University in New York because they have done all of the studies, and they have done the most cases. And SPECT scans can differentiate between Lyme and other neurological illnesses. And I would do MRIs of the brain and the spinal cord, and I would do spinal taps, and then, as I say, look for all the co-infections. I would do lupus panels, order immune panels, and we look for other sort of inflammatory markers that would be atypical for, let's say, dementia. So it's a very sophisticated workup, but you need to have a sophisticated workup at a point where you are already in what's called a chronic neurologic phase. Judy: Yeah. It sounds like it. I want to move on to a slightly different topic, and again this is for Lesley Fein. How does vitamin D play into all this? Does vitamin D protect a person? Dr. Fein: No. The only data that there is on vitamin D is that a subgroup of people who have chronic intracellular infections get activation of vitamin D, and they get rapid transformation of 25D into 125D, and that's triggers an inflammatory response. And in those individuals, that part of the inflammatory response needs to be addressed, and those people should not be given vitamin D because it can actually make them worse, not better, and should avoid the sunlight because that can also prolong or promote the inflammatory pathway. But that's not a process that's specific to Lyme. It's seen in multiple chronic intracellular infections. Judy: So you don't want someone with that to be taking extra vitamin D supplements? Dr. Fein: No, no. So if anybody has the vitamin D tested, they have got to have 25 and 125, and the 125 has to be frozen and has to be transported frozen. And that's why nobody bothers to do it because it's a pain in the neck to do, but you need to because otherwise you shall be going to miss that particular inflammatory pathway. Judy: Okay. This is a question I would like to ask both of you, one at a time obviously, and, Constance, why don't we start with you. I want to hear a little bit more about the role of long-term antibiotics for chronic Lyme. And, for our listeners, Constance is not a doctor, but she has been there in the trenches. Ms. Bean: Oh, okay. So once you know you have got Lyme, and you really know that it can be nothing else, you have ruled out everything else, and you know it's most likely if you have been bitten and that you didn't get well in four weeks of treatment, and you are still getting sick. It's probably the same disease, except that you didn't get it soon enough and so on. Judy: Well, do most patients want to have very long-term antibiotics? We are talking years, I guess. Ms. Bean: No, nobody wants them. Nobody wants them. It's a matter of treating until well, and you don't know. Many people are well in a month. Many are well in two months. More sometimes it's four months. Judy: Well, how long did you take antibiotics for? Ms. Bean: Well, as I told you, I relapsed when I went off them, so let's see. The initial treatment, I don't know that I can quite… Judy: …well, have you been taking them for 14 years? Ms. Bean: No. Well, for five years, I was taking them intensively and gradually getting better and better and better and better, and that's why I took them. I went off for a little bit, and then I had to go back because the symptoms recurred. And I know I had the infection. It was no question I am taking them just to take them. It's only to treat the symptoms. Judy: Oh, of course. Ms. Bean: Yeah. I think it's resolved, but if it isn't resolved – because it can hide – it can go dormant, nothing happens. The disease is not active. The bacteria don't replicate very quickly, so you can go quite a while with no symptoms, but it's not gone. Judy: Right. So, Dr. Lesley Ann Fein, speaking as a doctor, how long do some people have to be on antibiotics for with a chronic form of Lyme? Dr. Fein: Well, as she said, it's very controversial, and I think doctors who are dogmatic about stating that they know what the duration of treatment is are making inappropriate statements. We don't know what the appropriate treatment is. We haven't done enough studies, and there are no prospective studies. There is one, but he only used one drug, and I think you need to use multiple. So the thing is if somebody walks into my office now, I am not willing to tell them how long they are going to need to be on. They are going to need to be on as long as they need to be on until I think that the infection is under control. And it may take a long time. It may take months to years of different combinations. And you do sometimes do what's called an antibiotic holiday where you take people off and see what happens. But I am never, ever willing to commit to a specific time period. The truth is we don't know how long it takes. Each case is different. Judy: Constance, this is for you, why do you think some patients are so insistent that they have Lyme when they really may not? Ms. Bean: Well, we don't have the gold standard test. And even if we did, there are still those say who really don't want Lyme disease to be treated for a long period of time, and insurance companies, of course, are all a part of this, they will say even if you find the spirochetes, it doesn't mean that you are still sick. It just means that they are there, but that may not be causing your symptoms. Well, it's sort of a stretch of a belief system if you feel that you are at risk and you don't want any risk. So we don't know when all the spirochetes are gone, and we are not sure from any tests that you have it. Judy: Okay. Ms. Bean: But when you put everything together, the symptoms, the exposure, the possible rash, the way the symptoms go, the fact that they make no sense, they don't fit any other disease, there is no other explanation. Dr. Fein: The diagnosis is basically a clinical one. It's got to be based upon the clinical manifestations and, like Connie said, what makes sense and what doesn't make sense. If you have a kid with, let's say, lesions on a brain MRI and swollen joints, there are not that many diseases that cause that. So then it's a matter of analyzing the case. Judy: How do you reduce the risk of getting Lyme, Dr. Fein? Dr. Fein: I would rather Connie answer that question. Judy: Well, I would like to stick with the actual medical thing for a second. What steps should you take, Dr. Fein, to try to avoid getting a tick bite? Dr. Fein: What I recommend people do is go to the Web site of the Connecticut Health Department. The Department of Health in Connecticut has done the most extensive research. They have books and brochures on how to treat your property, how to protect yourself. Ms. Bean: Well, I would say we have the standard precautions. All the state health departments have them. They are many brochures, at the national seashores, everywhere else. In the book, "Beating Lyme: Understanding and Treating This Complex and Often Misdiagnosed Disease," I do make it more specific because many people do follow the standard precautions, and they are not enough. For instance, tuck your pants into your socks. Well, ticks usually first are low to the ground and wherever they come out of your shoes, they will go right up the pants, and they will find skin no matter how much you tuck your pants into your socks. Judy: Well, just, I mean, to back up for just one second, the ticks, you get them from being in the bushes, right? Ms. Bean: Yeah. They are usually in the low foliage. They don't drop from trees. And as you walk through long grass, beach grass, shrubs, garden-type foliage, they sense the carbon dioxide which you are exhaling, and they are just waiting and questing to find the human blood meal or any warm-blooded meal, and so then they are ready to attach. Judy: So if you are in an open area like a lawn where the grass is cut or… Ms. Bean: …yeah, but people sit on lawns. And sometimes they are shaded. As long as there is moisture, if you sit on a park bench, if you sit on a stone wall or a tree stump, all of those places are very risky. Dr. Fein: Well, I mean they have shown that the center of your property has a lower tick density than the periphery, so the periphery of your property has a much higher tick density. Ms. Bean: Between the lawn and the woods, yes. As you go, what you can do is have a six-foot width of wood chips all around, between your lawn and the woods surrounding it. Judy: Well, does that work? Ms. Bean: Yes. These are the kinds of things you can do for your property. There are also various chemicals and so on too. We have deer feeding stations where we try to disinfect the deer as they feed. They put their necks, and they go through rollers, and the insecticide takes the ticks. So there are various things like that, but you really have to protect yourself. Judy: Sounds like a lot of trouble. If you have been in an area where say you have been hiking or something in an area where there could be ticks, I guess you should go back home and take a shower. Ms. Bean: Get those shoes off, yeah, and those socks off. Judy: And then in your book, Constance, you suggest putting your outdoor clothes in the dryer for 30 minutes on high. Ms. Bean: Yeah. Judy: Is there actual evidence that that will kill? Ms. Bean: Oh, yes. There is a study on that, yeah. Because ticks, one thing, they can live forever in your suitcase, it seems like just about anywhere, but they cannot stand being dried out. Judy: Well, what if you put yourself in a sauna or something, in a really hot shower, would that kill them? Ms. Bean: I guess the best thing would be to get in a shower and shampoo and absolutely scrub yourself down. But once they have attached, you cannot wash them off. And you can't brush them off. Dr. Fein: Right. And temperature, you can't raise body temperature enough to kill them off without killing yourself. Judy: Well, Dr. Fein, speaking of body temperature, years ago, I wrote about what seems like a totally crazy treatment. But if I am remembering it right, it was Heimlich, the guy who invented the Heimlich maneuver, I think it was he who suggested that people go to the tropics and intentionally get… Dr. Fein: …they got malaria, yes. Judy: They got malaria, and the high fever actually cured the Lyme. Is there any… Dr. Fein: …well, you have to go up to about 106 degrees, so that's a little dangerous. Ms. Bean: So even your sauna is not going to take the body temperature up there. But many people do take saunas. Dr. Fein: But that's not really going to do anything. I mean, it might make you feel better. Judy: Yeah, but if you are still stuck with it. Ms. Bean: So I think being very vigilant, you can't see the ticks in many areas the body. It just means a total scrub-down and not going very many hours between either because it only takes a few hours before they don't brush off anymore. Dr. Fein: Yes. And I recommend to parents always to look at the nape of the neck. You would be amazed how many kids have tiny little rings and circles in the nape of their neck. You have to look. The girls have to wear their hair up and wear a hat, and you have to look behind the ears, at the hairline, axillae, in the armpits and in the groin. Judy: It's been a fascinating show, and my guests have been Dr. Lesley Ann Fein, who is the medical director for the Pennsylvania Lyme Disease Society and the Greater Hartford Connecticut Lyme Disease Support Action Group, and Constance Bean, who has been a patient and is author of the book "Beating Lyme: Understanding and Treating This Complex and Often Misdiagnosed Disease." I would like to thank you both very much, and I would like to thank you, the listeners, for joining us. Until next week, I am Judy Foreman. Good night. Last Updated: 07/09/2008 | Last Reviewed: 07/09/2008This section created and produced exclusively by the editorial staff of HealthTalk. © 2009 EverydayHealth.com; all rights reserved. | Next Article: Sign Up For Newsletters At Everyday Health »


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Anaheim >Food & Dining > Rainforest Cafe
1515 S Disneyland Dr, Anaheim, CA | Directions 9280233.809501 -117.924842
Call for hours
Neighborhoods: Disneyland
This is a great place to take the kids - just be prepared to wait OR call in advance to get a reservation. The highlight is they are acutely aware of food allergies and are more than accommodating. My pick so far is actually the burger. Predictable and easy.
All these locations are the same as far as I can tell. They are all amazing too. This one's HUGE and is very cool. The food is okay, but the atmosphere is all tropical jungle and bull frogs. Amazing.
Awesome place to take kids! Good food and fun atmosphere to keep the kids busy. They have standard American faire and a large drink menu. The animals that are strategically placed around the restaurant come to life every 30 minutes. So just be aware if you sitting in front of one of the displays!
Whenever we visit the Disney parks in Anaheim we eat at the Rainforest Cafe.
Never a disappointment.
We were seated immediately, our server (LQ) brought me coffee when she took our order. I ordered the 'cisco and pancho' with rare steak, scrambled eggs, home potatoes, and a buttered english muffin. Wife and granddaughter ordered 'breakfast of bandits' with bacon, scrambled eggs,home potatoes, and toast (wife asked to substitute the toast with fresh fruit and they were happy to accomodate) The manager (Stacy) came by to ask how everything was right when I was cutting into my steak, and I was happy to say "Perfect". Another manager came by (Linda) to check, and we were all real happy to say everything was excellent. Needless to say, breakfast was fantastic, as usual when we come to the Rainforest Cafe. Our server checked on us at regular intervals, and gave better than expected service. I can't speak highly enough about this place, as it says on their coffee cups, "it's a great place to eat!"
May 31, 2013
Come here every few months, and never disappointed. Come here every few months, and never disappointed. My wife and I enjoy the ambiance, but the food is why we come. It's normally breakfast, and I order the steak (rare) and scrambled eggs, potatoes, and english muffin. My steak is always good, but sometimes it's absolutely fantastic. Our server (LQ) is the greatest, always super attentive to our needs, even to making sure my english muffin is buttered before coming to tjhe table, and that I get a great perfectly cooked steak every time we come. The managers (Stacy and Nancy) go above and beyond too, checking on us to be sure we are having a great experience. My wife orders the bacon, scrambled eggs, potatoes, and they substitute her toast with fresh fruit. When we bring the grandkids they always get great meals too, as well as enjoy the scenery in the restaurant. I highly recommend this Rainforest Cafe over all the others we have been to, the staff shows they have real teamwork, and it makes such a huge difference.
Good food, crowded place – I went to this Rainforest Cafe in Anaheim and the one in Las Vegas and thye both were the same. The food was good(not great, but ok) and it was slightly overpriced. We waited about 45 for a table for 4, but looking around the gift shop made the time go by alittle quicker. The restaurant looks like something out of Indiana Jones, full of tropical animals, and some african animals and alot of vegetation, a huge aquarium, and fire shooting out the top of the building with acouple of fater falls, overall i think i might go back but its not my first choice in restaurants.
This cartoon jungle is a child's wonderland, with waterfalls, swinging monkeys and a canopy of faux flora and fauna. – In Short
While waiting for a table, hungry diners prowl the retail village, where cash-register ka-chings compete with chirping birds, roaring lions and chest-thumping gorillas. When the lights start to flicker, never fear--thunderstorms are part of the show. The menu is American comfort food--pastas, burgers, pizzas and salads--served with a tropical flair. There are plenty options that the kids will recognize, but enough novelty to keep the parents happy.
avoid weekends – the food is great i can't rave enough about the rasta pasta and the awesome appetizer. However I have had two bad experinces with the rain forest cafe the first was when my mother came into town we went to rain forest and we also bought a cake with us the resturant offered to place it in their refrigarator while we enjoyed our dinner when it was time to eat our cake the waitress laughed and stated she had dropped our cake, the resturant did provide us with another cake, and we were compensated on the bill for the price of the cake, however my husband went with some of our friends this weekend and we had the best service from our waiter at the time brett but his breaker was very rude he snatched the plates out of our hands and he also didn't ask was everything okay or how did we enjoy our food it was very dissapointing to say that my mother and our friends had experinced both bad experinces.
Great Family Fun!! – The Food is average (burgers, salads, ribs) but tasty. The wait can get a little long but for parents of young children the atmosphere & experience is well worth it. The place is so much fun. My family goes every time we are at Downtown Disney & never regret it.....lotsa family fun
Fun place! – The wait can be a bit long, but it just feels like you are somewhere else dining here..I love the fish tank sorrounding the place...the food is just your basic staples with a bit of a twist...a tropical twist like adding cucumbers to a chicken plate or the coconut chicken strips...it may be a bit pricey but a great, cheery experience
love the atmosphere! – My boyfriend and I met up with some friends here for lunch. I love the atmosphere; there's a simulated tropical thunderstorm every half hour. Also, they have animatronic animals too. It's so much fun! Their food is great: I love the coconut battered chicken strips and their chinese chicken salad. The service was slow. We were put in the back and were forgotten. Our drinks were rarely refilled and it took awhile to get the bill. But, the food was wonderful.
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Numbers 12:6 (New International Version)
Numbers 12:6
New International Version (NIV)
6 he said, "Listen to my words:
"When there is a prophet among you,
I, the Lord, reveal myself to them in visions,
I speak to them in dreams.
Holy Bible, New International Version®, NIV® Copyright © 1973, 1978, 1984, 2011 by Biblica, Inc.® Used by permission. All rights reserved worldwide.


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TUESDAY, February 25, 2014 7:15pm, Legislative Chambers
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Resolution No. 92-a (2013) Local Law "B" Establishing the Telephone Directory Reduction and Recycling Act
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The left-handed slugger's first All-Star berth was yet another sign of how far he has elevated his game since coming to the Red Sox last season.
Ortiz, the American League leader in RBIs, will ride shotgun with teammates Manny Ramirez and Curt Schilling in Houston on July 13.
"I feel great, especially for David," said Red Sox ace Pedro Martinez. "Curt and Manny have always been there. I'm happy for them. But for David, his first time, he was praying he'd make it. I just can't describe how happy I am for him."
Ramirez, Boston's lethal cleanup man, was voted in by the fans as a starter for the fourth time in his four seasons with the Sox. It was Ramirez's eighth career selection to the All-Star team.
The left fielder became just the third player in Red Sox history to be voted in four consecutive times, joining Hall of Famer Carl Yastrzemski (1967-72) and five-time batting champion Wade Boggs (1986-92).
"I'm just very proud to be selected to the All-Star Game once again. That's something that, when you're not playing anymore, that's always going to be there for your kids," said Ramirez. "You're always going to remember that. I just want to thank all my fans. They're always good to me. It is fun, especially when the fans vote for you. It's going to be real fun."
While other Sox players have been in and out of the lineup with injuries, Ramirez and Ortiz have carried the offense at times.
Ramirez is hitting .340 with 22 homers, 65 RBIs and a slugging percentage of .656.
Ortiz has proved that his breakout last year, when he finished fifth in the American League MVP voting, was no fluke. He's elevated his game even more this year, hitting .306 with 31 doubles, 22 homers and a league-leading 76 RBIs. The Sox signed Ortiz in January 2003, after he was released by the Twins.
"He's been unbelievable," Sox manager Terry Francona said of Ortiz. "I just think he's gotten to the point in his career where I think he knows he belongs. He made a splash last year kind of and now he realizes he belongs. I guess the thing I really appreciate is he's taking this and kind of running with it. He understands that not only being a good hitter, but being a good player, there's lot of responsibility and accountability that comes with it. I see him learning that and getting better at it."
For Ortiz, it was the culmination of years of hard work.
"I worked for it, man," said Ortiz. "I try my best all the time. I had the hope that one day I was going to have the chance and here we are. Now I'll just keep trying to have more opportunities."
His work this season was recognized by his peers, as Ortiz was selected by a combination of players, coaches and managers. He had 480 votes, beating out Kevin Harvey (189) by nearly 300 votes at first base.
As for Schilling, he has been just the rock for the rotation the Sox envisioned when they traded for him the day after Thanksgiving and then signed him to a two-year extension. The right-hander and six-time All-Star is 11-4, placing him in a tie with Kenny Rogers for the league lead in wins. His 3.08 ERA was fourth at the start of play on Sunday. His 120 innings lead the league.
Even at the age of 37, the thrill of going to the All-Star Game has not worn off.
"It's never gotten old," said Schilling. "It's the best players on the planet. To be included in that is something I'm very proud of."
Schilling's worth has never been more evident than in the last few weeks, as the Sox have been in a rut as a team. He's taken the ball seven consecutive times following a Boston loss, and the Sox have won six times.
"Obviously, Curt is having a Schilling-type year," said Red Sox closer Keith Foulke. "He's going out there and dominating the game. He's definitely an All-Star."
Like Ortiz, Schilling was voted in by his peers. With 243 votes, he ranked second behind Oakland's Mark Mulder (289) in the pitcher balloting.
Other Sox who made strong All-Star bids but didn't make the team were Martinez and Foulke. Martinez, a three-time Cy Young Award winner and six-time All-Star, is 8-3 with a 3.74 ERA and has 105 strikeouts.
"I'm an old goat," quipped Martinez. "There's a lot of people to choose. A lot of people are having great years. To me, it's not a big deal."
Foulke has been dominant in his first year with the Sox, posting a 1.28 ERA over his first 36 outings. Hurting his bid was a lack of save opportunities. He is 13 for 16 and has had just one save opportunity since June 13.
"I'll have three days of nice tan from Arizona," said Foulke. "That all goes back to, it's kind of a numbers game sometimes, I'm happy with the way I'm throwing the ball. I didn't make it. Arizona is nice this time of year."
And for three members of the Red Sox, so, too, will Houston.
Ian Browne is a reporter for MLB.com. This story was not subject to the approval of Major League Baseball or its clubs.
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text: NASCAR Race Sunday By Billy Goforth February 28, 2011 3:27 PM Share on Facebook Share on Twitter Photos from the NASCAR race in Phoneix Arizona on Sunday. Filed Under: arizona, nascar, phoneix Category: Sports Share on Facebook Share on Twitter More Leave a comment Send to a friend! Print this page Pin it! Reddit This! Share on Tumblr! More From Voice Of Amarillo SpaceX To Attempt Launch On Friday Three Rescued From Sinking Tow Boat In New Orleans Grain And Cattle Higher, Pork Lower On Chicago Board Of Trade El Doctorow To Recieve Library Of Congress Prize For Fiction Best of the Web


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The scope of the Conservation Services Programme (CSP) is the impact of commercial fishing on protected species in New Zealand fisheries waters.
Protected marine species include all marine mammals and reptiles; sea birds (except black backed gulls); seven species of fish; all black corals, gorgonian corals, stony corals and hydrocorals.
Direct commercial fishing impacts include species being caught, killed or injured in nets or on lines (bycatch). Impacts may also be indirect through the modification of habitat or food availability.
CSP monitors the effects of commercial fishing on protected species, studies species populations and looks at ways to limit bycatch.
Find out more about the Conservation Services Programme.
Review agendas, minutes and presentations from meetings and project progress reports.
View the latest Conservation Services strategic plan, annual plans and an archive of older planning documents
Review the latest reports from the Conservation Services Programme and an archive of older project reports
View identification guides for seabirds and coral species written for the Conservation Services Programme.
Find other reports, plans and guides related to the Conservation Services Programme.
Read the latest issue of Bycatch Bylines, a newsletter dedicated to the latest protected species information relevant to trawl and longline fishing methods.
National Plan of Action - Seabirds 2013
CITES endangered species
Conservation Services ProgrammeMarine Conservation ServicesDepartment of ConservationPO Box 10 420Wellington 6143Email: firstname.lastname@example.com


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PuertoNico, on 09 April 2012 - 07:30 AM, said:
Just like I said, simple(ly) beautiful.
Sometimes its okay to let the loop ride and vibe the fuck out not everything needs to be a magnum opus. And if a rapper can pack that much emotion in one 16 then I'm not even going to try to front.


text:
2009 Market Perspectives
The U.S. Grains Council's weekly market newsletter, Market Perspectives, provides timely information on coarse grain markets including price, weather and freight information. The newsletter is a trade and customer servicing tool utilized by U.S. agribusinesses as well international feed and food grains/feed ingredient importers, processors and end-users. Read Market Perspectives in Spanish.


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HOME MLB Atlanta Braves Gear Atlanta Braves Mens Atlanta Braves Mens Jackets over $60 (37 items) Product ID: 1101997
Main Content
You like to brag about wins and rep your Braves to the fullest. Boast your team spirit with this Clean Up Hitter half-zip jacket. This team-colored jacket features a team logo on the front and team name on the back, so anyone from any angle can see your Atlanta devotion. When you wear this jacket, everyone will see that you're proud to be a Braves fan!
- 100% Polyester fabric
- Embroidered graphics on front and back
- 13" Zip opening on left side
- Two front pockets
- Half-zip jacket
- Elastic cuffs
- Imported
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Braves Fanatics also liked:
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Manufacturer direct items are shipped directly from the manufacturer. These items are not available for international or expedited shipping.
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A rugged, self-illuminating Smith & Wesson® Watch, made military service-tough, with Diver features. This Watch is probably the easiest in the world to read in low light, even in total darkness, thanks to its tritium light source that requires no battery power or any other form of charging, and never needs servicing or maintenance. A minute quantity of tritium lights up the dye in tiny, airtight vials, sealed under high pressure. The self-illuminating technology is guaranteed to deliver 25 years of consistent glow!
This Watch is extremely robust and classically reliable. Check the specs:
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Countdown Timer until specified Date-Time starts a countdown to a time defined in forms. It can take a given time for the end of a countdown from form inputs which define the year, month, day, hour, minute, and second. The object can also take the end time from static page elements with identifiers which start with a given prefix. The object can start the countdown and keep updating page elements which show the remaining time to the end.
Simple PHP Chat Script is an AJAX chatting system which does not require a database. It works with text files saved on the server, so it is fast and requires very few server resources. It can be included in both PHP or HTML files (in blogs, forums), but the server must run PHP. The script can easily be changed to be used in other languages, just by translating the texts in a single file. It can have one or more chat rooms. The user can add smiles in chat, and can format the text with Bold, Italic, an Underline.
Splunk Hogs is a quick Perl script that was written to find what systems are sending too many events to Splunk and using up all the licenses. This is good for several reasons: developers will turn debugging on and use up all the licenses, which will help catch it early; INFO messages get turned on and useless events will be sent, filling up the licenses; issues will be detected that were being ignored by others. The advantage over Splunk's SaveSearch is that it outputs the raw data, so you can import it into Nagios or other tools. It collects raw data from Splunk that consists of the host, Splunk server, and the count of events that took place.
BSF4ooRexx is a Java language binding for the scripting language ooRexx. It allows ooRexx programmers to directly use the Java Runtime Environment (JRE) libraries. It allows, for example, implementation of Java methods in ooRexx and callbacks from Java to ooRexx. It camouflages Java so that it resembles ooRexx by being dynamically typed and caseless. BSF4ooRexx comes with built-in support for programming OpenOffice.org/LibreOffice.org and allows ooRexx to be used as a macro language.
Nfsight is a Netflow processing and visualization application designed to offer a comprehensive network awareness solution. Developed as a Nfsen plugin, it constructs bidirectional flows from unidirectional flows and leverages these bidirectional flows to provide client/server identification.
YMSG is a PHP class that can be used to send messages and manage Yahoo! Messenger accounts. It can connect to a Yahoo! Messenger server, authenticate as a given user, and perform several types of operations on behalf of that user. Currently, it can get the user buddy list, add a new buddy, deny a new buddy request, send a buddy request, remove a buddy, get an user avatar URL, and send a private message.
Penumbra is a first person horror adventure focusing on story, immersion, and puzzle solving. Violence and combat are hardly an option: the player has to use wits to guide Philip through his final test, which makes the series unique in offering a truly dangerous and terrifying experience. Played from a first person perspective and powered by a 3D engine utilizing cutting edge technology, Penumbra is a true advancement to the adventure genre. The unique physics system allows for true environmental interaction: The player can open drawers, pull levers, pick up objects and manipulate those objects in 3D space, all using natural mouse movements. This creates a highly interactive and immersive game world.
A Web resource optimizer for Java.
A package manager plugin for Rodent Filemanager.


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Thursday, April 24, 2014
The Associated Press
CARSON CITY, Nev. — The tale of a mysterious Nevada recluse's gold reached a new chapter Tuesday when a portion of the trove raked in more than $3.5 million at auction.
Mexican 50 peso pieces were among the $3.5 million in gold coins auctioned off in Carson City, Nev., on Tuesday, Feb. 26, 2013. Recluse Walter Samaszko died in June 2012, leaving thousands of coins hidden in his garage. (AP Photo/Las Vegas Review-Journal, Cathleen Allison)
This Monday, Dec. 17, 2012 photo shows the Carson City, Nev. home of Walter Samaszko Jr. A crew sent to clean out Samaszko's house found more than $7.4 million worth of gold coins, bars and bullion left behind in the garage after his death in June. About 135 pounds (61 kilograms) of Walter Samaszko Jr.'s gold coins will be auctioned off in 11 lots Tuesday, Feb. 26, 2013, in a courtroom to pay government taxes and fees. Carson City Clerk Recorder Alan Glover says the auction will only include about half of the trove left behind when the reclusive man died in his modest Carson City home last year. An exhaustive search identified California substitute teacher Arlene Magdanz as Samaszko's first cousin and sole heir to the fortune that was decades in the making. (AP Photo/Scott Sonner)
It was the allure of mystery that pulled some bidders to the courtroom, but for others, it was the sheer value of a collection unknown to the public before Walter Samaszko Jr. was found dead in his modest ranch-style home last year.
Regardless of motivation, those who converged on the auction could sense the immense value of the treasure upon arriving.
There were numerous guards at the entrance, then more in the hallway outside the courtroom, and finally several with bullet proof vests and others with helmets inside the room holding the gold.
Five bidders diligently inspected the 11 lots of gold displayed in plastic sleeves, tubes and felt jewelry display boxes heavily guarded room before the bidding wars began.
By the time all sales were final, however, one bidder had secured nine of the 11 lots for sale.
Carson City's Alan Rowe of Northern Nevada Coin spent $617,000 from his own company, and another $2 million on behalf of the Illinois-based Rare Coin Company of America Inc. It was the uniqueness of the gold that drove his bidding, he said.
"Every one of us has a little hoarder nature in our culture and we all like to have things, but to this degree is quite a story," Rowe told reporters after the auction, adding that the metal value "is not as exciting as the story itself, there's actually value to the story."
He added that some of the coins will be available in the store or online for locals hoping to snag a piece of history. Others, he said, will be marketed nationally and likely on television.
This auction was only for the bullion coins — items that are not necessarily rare, just expensive because they are made of gold. There will likely be a second auction for the larger portion of the collection which is comprised of the rare coins, said Alan Glover, the public administrator for Samaszko's estate.
"They're buying and bidding on an ounce of gold, pure gold by the weight," Glover said.
In total, about 150 pounds of gold was sold at Tuesday's auction. About $800,000 will pay various fees and estate taxes, and the rest of the profits go to a substitute teacher in San Rafael, Calif., who is the first cousin and sole heir to the trove of Walter Samaszko Jr.
Because of the other coins' rarity, that sale is expected to net higher profits.
James Mitchell of Reno's Silver State Coin and a California-based group named Spectrum Group International Inc. grabbed the two lots not purchased by Rowe or his partners.
Mitchell landed the lot of 4,600 Mexican dos pesos, the largest number of coins in a single lot. He said the story posed no additional value to him.
"It had the most potential for profit," Mitchell said of his purchase. "There was one lot I wanted more, but this one will have to do."
That lot, a collection of 620 Canadian Maple Leafs, was the largest in terms of weight and the coins were the purest gold available. It fetched $1.16 million from Rowe and the Rare Coin Company of America.
No one knows exactly when the collection began, or why Samaszko never sold it. Frankly, no one knew anything about him even though he lived in the same neighborhood for decades. Weeks passed before authorities even discovered he had died in his modest Carson City home. A coroner said he died of heart problems.
(Continued on page 2)


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1 person found this helpful
I worked at Panera Bread full-time for more than 3 years
Pros – Core values taught and repeated enough to make a positive impact throughout cafes
Competitive salary (for established locations*)
Five day work weeks (6 day "unofficial" requirement for first 2 weeks of new cafe openings)
45 scheduled hours per week
Great training resources for the entire operation - any and all questions can be answered within minutes after looking through the online database of training docs or after a phone call to the "help desk" IT support which is on call 24/7.
Cons – Promotions and new job openings given to "friends of friends" like many other companies...it's still very annoying
45 scheduled hours can see a 30% spike during new cafe openings and this is very high. I have seen higher jumps in weekly work hours during new cafe openings and the workload is often unfairly put on the shoulders of the few people who seem to make the best of things with the least amount of help.
There are shift supervisors working for the company that make more money than some assistant managers.
New positions that emerge within the company are often filled before an opportunity arises for anyone to apply. The "opportunity for growth" is always mentioned in cafes to anyone interested in "climbing the corporate ladder", but it is something that eludes many talented individuals in the company.
Advice to Senior Management – Corporate phrases that are repeated over and over again mean nothing to managers that want to know how to work through everything you need them to. More talk of strategy, goal setting and accountability need to take place in every cafe. Special treatment with a blatant disregard for management structure should be fixed immediately and giving praise more often than "never" will help more than you know!
Yes, I would recommend this company to a friend – I'm not optimistic about the outlook for this company
I have been working at Panera Bread full-time for more than 3 years
I worked at Panera Bread part-time for more than a year
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New book now available!
Check out my brand new sketchbook! See the full series | Buy the book on Amazon | Read on your Kindle | Download the PDF | Buy a Print
Read my ongoing comic, My Sister the Freak, a story about two sisters as they battle life, family, and alien invaders.
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